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You searched for subject:( GTPase Activating Proteins metabolism 60). Showing records 121 – 150 of 21507 total matches.

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121. Salman, Emily Deanna. Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain.

Degree: PhD, 2011, University of Alabama – Birmingham

The human cytosolic sulfotransferases are a family of phase II drug-metabolizing enzymes that conjugate a sulfonate moiety from 3’-phosphoadenosine 5’-phosphosulfate (PAPS) to a hydroxyl moeity… (more)

Subjects/Keywords: Arylsulfotransferase  – metabolism<; br>; Brain  – enzymology<; br>; Cytosol  – enzymology<; br>; Immunohistochemistry<; br>; Sulfotransferases  – metabolism

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APA (6th Edition):

Salman, E. D. (2011). Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,960

Chicago Manual of Style (16th Edition):

Salman, Emily Deanna. “Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed November 17, 2019. http://contentdm.mhsl.uab.edu/u?/etd,960.

MLA Handbook (7th Edition):

Salman, Emily Deanna. “Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain.” 2011. Web. 17 Nov 2019.

Vancouver:

Salman ED. Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Nov 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,960.

Council of Science Editors:

Salman ED. Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,960

122. Yezdani, Gulam. Role of VDR in host immune response to Porphyromonas gingivalis infection.

Degree: MS, 2011, University of Alabama – Birmingham

Porphyromonas gingivalis is one of the etiologic factors of periodontal disease, a chronic inflammatory disorder characterized by the destruction of periodontal connective tissue and the… (more)

Subjects/Keywords: Mice<; br>; Porphyromonas gingivalis – metabolism<; br>; Receptors, Calcitriol – metabolism<; br>; Vitamin D – metabolism

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APA (6th Edition):

Yezdani, G. (2011). Role of VDR in host immune response to Porphyromonas gingivalis infection. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,991

Chicago Manual of Style (16th Edition):

Yezdani, Gulam. “Role of VDR in host immune response to Porphyromonas gingivalis infection.” 2011. Masters Thesis, University of Alabama – Birmingham. Accessed November 17, 2019. http://contentdm.mhsl.uab.edu/u?/etd,991.

MLA Handbook (7th Edition):

Yezdani, Gulam. “Role of VDR in host immune response to Porphyromonas gingivalis infection.” 2011. Web. 17 Nov 2019.

Vancouver:

Yezdani G. Role of VDR in host immune response to Porphyromonas gingivalis infection. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2011. [cited 2019 Nov 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,991.

Council of Science Editors:

Yezdani G. Role of VDR in host immune response to Porphyromonas gingivalis infection. [Masters Thesis]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,991

123. Guidry, Loni Ann. Analysis Of Ergothioneine Production In Mycobacterium Tuberculosis.

Degree: PhD, 2012, University of Alabama – Birmingham

Mycobacterium tuberculosis (Mtb), the etiological agent of tuberculosis, currently infects more than two billion people worldwide. Mtb is a highly adaptable pathogen, capable of modifying… (more)

Subjects/Keywords: Bacterial Proteins – metabolism<; br>; Ergothioneine.<; br>; Homeostasis<; br>; Lipid Metabolism – physiology.<; br>; Mycobacterium tuberculosis<; br>; Tuberculosis – microbiology

…been developed in the last 60 years (1). It is therefore critical to identify new… …Aerobic metabolism/respiration results in the formation of reactive oxygen species (ROS… …include: (a) formation of disulfide bonds in proteins, which may inhibit their… …proteins in the cluster revealed that recombinant Msm EgtB catalyzes Fe-dependent oxidative… …while other LMW thiols tend to form disulfides (73). The Eo′ value of ergo is -60 mV… 

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APA (6th Edition):

Guidry, L. A. (2012). Analysis Of Ergothioneine Production In Mycobacterium Tuberculosis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1420

Chicago Manual of Style (16th Edition):

Guidry, Loni Ann. “Analysis Of Ergothioneine Production In Mycobacterium Tuberculosis.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed November 17, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1420.

MLA Handbook (7th Edition):

Guidry, Loni Ann. “Analysis Of Ergothioneine Production In Mycobacterium Tuberculosis.” 2012. Web. 17 Nov 2019.

Vancouver:

Guidry LA. Analysis Of Ergothioneine Production In Mycobacterium Tuberculosis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2019 Nov 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1420.

Council of Science Editors:

Guidry LA. Analysis Of Ergothioneine Production In Mycobacterium Tuberculosis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1420


Michigan State University

124. MacDonald, Alex Bruce. Guanosine triphosphate metabolism in rabbit reticulocytes.

Degree: PhD, Department of Biochemistry, 1967, Michigan State University

Subjects/Keywords: Proteins; Proteins – Metabolism

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APA (6th Edition):

MacDonald, A. B. (1967). Guanosine triphosphate metabolism in rabbit reticulocytes. (Doctoral Dissertation). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:26689

Chicago Manual of Style (16th Edition):

MacDonald, Alex Bruce. “Guanosine triphosphate metabolism in rabbit reticulocytes.” 1967. Doctoral Dissertation, Michigan State University. Accessed November 17, 2019. http://etd.lib.msu.edu/islandora/object/etd:26689.

MLA Handbook (7th Edition):

MacDonald, Alex Bruce. “Guanosine triphosphate metabolism in rabbit reticulocytes.” 1967. Web. 17 Nov 2019.

Vancouver:

MacDonald AB. Guanosine triphosphate metabolism in rabbit reticulocytes. [Internet] [Doctoral dissertation]. Michigan State University; 1967. [cited 2019 Nov 17]. Available from: http://etd.lib.msu.edu/islandora/object/etd:26689.

Council of Science Editors:

MacDonald AB. Guanosine triphosphate metabolism in rabbit reticulocytes. [Doctoral Dissertation]. Michigan State University; 1967. Available from: http://etd.lib.msu.edu/islandora/object/etd:26689


University of Utah

125. Chin, David Tung-ball. The role of ubiquitin in ATP-dependent proteolysis.

Degree: PhD, Biochemistry;, 1984, University of Utah

 Radiolabeled ubiquitin was introduced into HeLa cells by red cell-mediated fusion procedure. The nuclei contained two major labeled proteins, ubiquitin and protein A24. The cytosol… (more)

Subjects/Keywords: Metabolism; Proteins

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APA (6th Edition):

Chin, D. T. (1984). The role of ubiquitin in ATP-dependent proteolysis. (Doctoral Dissertation). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/193/rec/1223

Chicago Manual of Style (16th Edition):

Chin, David Tung-ball. “The role of ubiquitin in ATP-dependent proteolysis.” 1984. Doctoral Dissertation, University of Utah. Accessed November 17, 2019. http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/193/rec/1223.

MLA Handbook (7th Edition):

Chin, David Tung-ball. “The role of ubiquitin in ATP-dependent proteolysis.” 1984. Web. 17 Nov 2019.

Vancouver:

Chin DT. The role of ubiquitin in ATP-dependent proteolysis. [Internet] [Doctoral dissertation]. University of Utah; 1984. [cited 2019 Nov 17]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/193/rec/1223.

Council of Science Editors:

Chin DT. The role of ubiquitin in ATP-dependent proteolysis. [Doctoral Dissertation]. University of Utah; 1984. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/193/rec/1223


Oregon State University

126. Alyan, Mohammad Atta. Calcium dependent proteinase (calpain) and muscle protein degradation : molecular approach.

Degree: PhD, Animal Science, 1991, Oregon State University

Subjects/Keywords: Proteins  – Metabolism

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APA (6th Edition):

Alyan, M. A. (1991). Calcium dependent proteinase (calpain) and muscle protein degradation : molecular approach. (Doctoral Dissertation). Oregon State University. Retrieved from http://hdl.handle.net/1957/36155

Chicago Manual of Style (16th Edition):

Alyan, Mohammad Atta. “Calcium dependent proteinase (calpain) and muscle protein degradation : molecular approach.” 1991. Doctoral Dissertation, Oregon State University. Accessed November 17, 2019. http://hdl.handle.net/1957/36155.

MLA Handbook (7th Edition):

Alyan, Mohammad Atta. “Calcium dependent proteinase (calpain) and muscle protein degradation : molecular approach.” 1991. Web. 17 Nov 2019.

Vancouver:

Alyan MA. Calcium dependent proteinase (calpain) and muscle protein degradation : molecular approach. [Internet] [Doctoral dissertation]. Oregon State University; 1991. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/1957/36155.

Council of Science Editors:

Alyan MA. Calcium dependent proteinase (calpain) and muscle protein degradation : molecular approach. [Doctoral Dissertation]. Oregon State University; 1991. Available from: http://hdl.handle.net/1957/36155


Hong Kong University of Science and Technology

127. Ji, Xianglin LIFS. Nutrient dependent turnover of lipid droplet associated proteins.

Degree: 2015, Hong Kong University of Science and Technology

 Lipid droplets (LDs) are conserved organelles for cellular fat storage. In C.elegans, numerous LDs can be found in intestinal cells, where fat is primarily stored.… (more)

Subjects/Keywords: Lipids; Metabolism; Proteins; Caenorhabditis elegans

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APA (6th Edition):

Ji, X. L. (2015). Nutrient dependent turnover of lipid droplet associated proteins. (Thesis). Hong Kong University of Science and Technology. Retrieved from https://doi.org/10.14711/thesis-b1514897 ; http://repository.ust.hk/ir/bitstream/1783.1-94920/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ji, Xianglin LIFS. “Nutrient dependent turnover of lipid droplet associated proteins.” 2015. Thesis, Hong Kong University of Science and Technology. Accessed November 17, 2019. https://doi.org/10.14711/thesis-b1514897 ; http://repository.ust.hk/ir/bitstream/1783.1-94920/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ji, Xianglin LIFS. “Nutrient dependent turnover of lipid droplet associated proteins.” 2015. Web. 17 Nov 2019.

Vancouver:

Ji XL. Nutrient dependent turnover of lipid droplet associated proteins. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2015. [cited 2019 Nov 17]. Available from: https://doi.org/10.14711/thesis-b1514897 ; http://repository.ust.hk/ir/bitstream/1783.1-94920/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ji XL. Nutrient dependent turnover of lipid droplet associated proteins. [Thesis]. Hong Kong University of Science and Technology; 2015. Available from: https://doi.org/10.14711/thesis-b1514897 ; http://repository.ust.hk/ir/bitstream/1783.1-94920/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Michigan State University

128. Long, Charles Herman, 1935-. Factors which influence gastrointestinal absorption in the immediate post-natal period.

Degree: PhD, Department of Animal Husbandry, 1964, Michigan State University

Subjects/Keywords: Proteins – Metabolism

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APA (6th Edition):

Long, Charles Herman, 1. (1964). Factors which influence gastrointestinal absorption in the immediate post-natal period. (Doctoral Dissertation). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:35740

Chicago Manual of Style (16th Edition):

Long, Charles Herman, 1935-. “Factors which influence gastrointestinal absorption in the immediate post-natal period.” 1964. Doctoral Dissertation, Michigan State University. Accessed November 17, 2019. http://etd.lib.msu.edu/islandora/object/etd:35740.

MLA Handbook (7th Edition):

Long, Charles Herman, 1935-. “Factors which influence gastrointestinal absorption in the immediate post-natal period.” 1964. Web. 17 Nov 2019.

Vancouver:

Long, Charles Herman 1. Factors which influence gastrointestinal absorption in the immediate post-natal period. [Internet] [Doctoral dissertation]. Michigan State University; 1964. [cited 2019 Nov 17]. Available from: http://etd.lib.msu.edu/islandora/object/etd:35740.

Council of Science Editors:

Long, Charles Herman 1. Factors which influence gastrointestinal absorption in the immediate post-natal period. [Doctoral Dissertation]. Michigan State University; 1964. Available from: http://etd.lib.msu.edu/islandora/object/etd:35740


Michigan State University

129. Ferguson, Mary H. Studies on high protein diets.

Degree: MS, 1930, Michigan State University

Subjects/Keywords: Metabolism; Proteins

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APA (6th Edition):

Ferguson, M. H. (1930). Studies on high protein diets. (Masters Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:14811

Chicago Manual of Style (16th Edition):

Ferguson, Mary H. “Studies on high protein diets.” 1930. Masters Thesis, Michigan State University. Accessed November 17, 2019. http://etd.lib.msu.edu/islandora/object/etd:14811.

MLA Handbook (7th Edition):

Ferguson, Mary H. “Studies on high protein diets.” 1930. Web. 17 Nov 2019.

Vancouver:

Ferguson MH. Studies on high protein diets. [Internet] [Masters thesis]. Michigan State University; 1930. [cited 2019 Nov 17]. Available from: http://etd.lib.msu.edu/islandora/object/etd:14811.

Council of Science Editors:

Ferguson MH. Studies on high protein diets. [Masters Thesis]. Michigan State University; 1930. Available from: http://etd.lib.msu.edu/islandora/object/etd:14811


Michigan State University

130. Dennett, Linnea C. The magnesium metabolism and its relation to calcium retention of two normal preschool children on high and medium protein diets.

Degree: MS, 1933, Michigan State University

Subjects/Keywords: Metabolism; Proteins

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APA (6th Edition):

Dennett, L. C. (1933). The magnesium metabolism and its relation to calcium retention of two normal preschool children on high and medium protein diets. (Masters Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:10464

Chicago Manual of Style (16th Edition):

Dennett, Linnea C. “The magnesium metabolism and its relation to calcium retention of two normal preschool children on high and medium protein diets.” 1933. Masters Thesis, Michigan State University. Accessed November 17, 2019. http://etd.lib.msu.edu/islandora/object/etd:10464.

MLA Handbook (7th Edition):

Dennett, Linnea C. “The magnesium metabolism and its relation to calcium retention of two normal preschool children on high and medium protein diets.” 1933. Web. 17 Nov 2019.

Vancouver:

Dennett LC. The magnesium metabolism and its relation to calcium retention of two normal preschool children on high and medium protein diets. [Internet] [Masters thesis]. Michigan State University; 1933. [cited 2019 Nov 17]. Available from: http://etd.lib.msu.edu/islandora/object/etd:10464.

Council of Science Editors:

Dennett LC. The magnesium metabolism and its relation to calcium retention of two normal preschool children on high and medium protein diets. [Masters Thesis]. Michigan State University; 1933. Available from: http://etd.lib.msu.edu/islandora/object/etd:10464

131. Xayarath, Bobbi. Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence.

Degree: PhD, 2007, University of Alabama – Birmingham

 The polysaccharide capsules of Streptococcus pneumoniae represent the most important virulence determinant produced by this organism. Ninety-one different serotypes have been identified, but only a… (more)

Subjects/Keywords: Bacterial Capsules  – metabolism <; br>; Cell Wall  – metabolism <; br>; Genes, Essential <; br>; Mutation <; br>; Polysaccharides, Bacterial  – metabolism <; br>; Streptococcus pneumoniae  – physiology

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APA (6th Edition):

Xayarath, B. (2007). Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,190

Chicago Manual of Style (16th Edition):

Xayarath, Bobbi. “Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed November 17, 2019. http://contentdm.mhsl.uab.edu/u?/etd,190.

MLA Handbook (7th Edition):

Xayarath, Bobbi. “Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence.” 2007. Web. 17 Nov 2019.

Vancouver:

Xayarath B. Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Nov 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,190.

Council of Science Editors:

Xayarath B. Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,190

132. Durham, Carolyn G. Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma.

Degree: PhD, 2010, University of Alabama – Birmingham

The inverse correlation between the industrialization and disease prevalence is termed the "hygiene hypothesis." Supporting this, immunological studies show Th1 cytokines modulate Th2 immune responses.… (more)

Subjects/Keywords: Asthma<; br>; Gastric Mucosa  – metabolism<; br>; Gastritis<; br>; Helicobacter Infections  – metabolism<; br>; Helicobacter felis<; br>; Mucus  – metabolism

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APA (6th Edition):

Durham, C. G. (2010). Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,707

Chicago Manual of Style (16th Edition):

Durham, Carolyn G. “Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed November 17, 2019. http://contentdm.mhsl.uab.edu/u?/etd,707.

MLA Handbook (7th Edition):

Durham, Carolyn G. “Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma.” 2010. Web. 17 Nov 2019.

Vancouver:

Durham CG. Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Nov 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,707.

Council of Science Editors:

Durham CG. Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,707

133. Cook, Ian Thomas. Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate.

Degree: PhD, 2011, University of Alabama – Birmingham

Sulfation is an important Phase II drug metabolism reaction catalyzed by the cytosolic sulfotransferases (SULTs). SULT2A1 is a major SULT in liver and adrenal cortex… (more)

Subjects/Keywords: Cytosol  – enzymology<; br>; Enzyme Inhibitors  – pharmacology<; br>; Liver<; br>; Sulfatases  – metabolism<; br>; Sulfates  – metabolism<; br>; Sulfotransferases  – metabolism

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APA (6th Edition):

Cook, I. T. (2011). Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1047

Chicago Manual of Style (16th Edition):

Cook, Ian Thomas. “Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed November 17, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1047.

MLA Handbook (7th Edition):

Cook, Ian Thomas. “Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate.” 2011. Web. 17 Nov 2019.

Vancouver:

Cook IT. Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Nov 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1047.

Council of Science Editors:

Cook IT. Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1047

134. Ghosh, Arindam P. Regulation of neuronal cell death by BH3-only molecules.

Degree: PhD, 2012, University of Alabama – Birmingham

Apoptosis in metazoan organisms plays critical roles in normal development, tissue homeostasis and immunity, and its disturbed regulation leads to many pathological states, including cancer,… (more)

Subjects/Keywords: Apoptosis  – physiology<; br>; Apoptosis Regulatory Proteins  – metabolism<; br>; Ethanol  – toxicity<; br>; Nervous System  – embryology<; br>; Neurons  – physiology<; br>; Neuropeptides  – metabolism<; br>; Tumor Suppressor Protein p53  – metabolism<; br>; Tumor Suppressor Proteins  – metabolism<; br>; bcl-2-Associated X Protein  – metabolism<; br>; bcl-X Protein  – metabolism

…Non-apoptotic functions of BH3-only proteins… …103 Role of BH3-only proteins in autophagy and autophagic cell death… …104 Role of BH-3 only proteins in tumorigenesis… …104 Role of BH3-only proteins in cancer therapeutics… …7 3. Sub-families of BCL-2 proteins… 

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APA (6th Edition):

Ghosh, A. P. (2012). Regulation of neuronal cell death by BH3-only molecules. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1138

Chicago Manual of Style (16th Edition):

Ghosh, Arindam P. “Regulation of neuronal cell death by BH3-only molecules.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed November 17, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1138.

MLA Handbook (7th Edition):

Ghosh, Arindam P. “Regulation of neuronal cell death by BH3-only molecules.” 2012. Web. 17 Nov 2019.

Vancouver:

Ghosh AP. Regulation of neuronal cell death by BH3-only molecules. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2019 Nov 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1138.

Council of Science Editors:

Ghosh AP. Regulation of neuronal cell death by BH3-only molecules. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1138

135. Jiang, Wen, Ph.D. KLF4 and retinoid receptor signaling in cancer.

Degree: PhD, 2009, University of Alabama – Birmingham

The fight against cancer has generated wide interest in understanding the genetic mechanisms behind the disease. One group of oncogenes – transcription factors – offers… (more)

Subjects/Keywords: Carcinoma, Squamous Cell  – metabolism<; br>; Cell Nucleus  – metabolism<; br>; Kruppel-Like Transcription Factors  – metabolism<; br>; Mice, Transgenic<; br>; Skin Neoplasms  – metabolism

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APA (6th Edition):

Jiang, Wen, P. D. (2009). KLF4 and retinoid receptor signaling in cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,598

Chicago Manual of Style (16th Edition):

Jiang, Wen, Ph D. “KLF4 and retinoid receptor signaling in cancer.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed November 17, 2019. http://contentdm.mhsl.uab.edu/u?/etd,598.

MLA Handbook (7th Edition):

Jiang, Wen, Ph D. “KLF4 and retinoid receptor signaling in cancer.” 2009. Web. 17 Nov 2019.

Vancouver:

Jiang, Wen PD. KLF4 and retinoid receptor signaling in cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Nov 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,598.

Council of Science Editors:

Jiang, Wen PD. KLF4 and retinoid receptor signaling in cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,598

136. Lee, Seung-Ah. Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism.

Degree: PhD, 2008, University of Alabama – Birmingham

Retinol dehydrogenase 12 (RDH12) is a member of the microsomal short-chain dehydrogenase/reductase superfamily of proteins that is highly expressed in photorecep-tor cells. Mutations in RDH12… (more)

Subjects/Keywords: Alcohol Oxidoreductases  – metabolism<; br>; Genetic Diseases, Inborn  – enzymology<; br>; Lipid Peroxidation<; br>; Mutation, Missense<; br>; Photoreceptor Cells  – enzymology<; br>; Retinal Diseases  – enzymology<; br>; Retinaldehyde  – metabolism<; br>; Retinoids  – metabolism<; br>; Tretinoin  – metabolism

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APA (6th Edition):

Lee, S. (2008). Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,814

Chicago Manual of Style (16th Edition):

Lee, Seung-Ah. “Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed November 17, 2019. http://contentdm.mhsl.uab.edu/u?/etd,814.

MLA Handbook (7th Edition):

Lee, Seung-Ah. “Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism.” 2008. Web. 17 Nov 2019.

Vancouver:

Lee S. Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Nov 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,814.

Council of Science Editors:

Lee S. Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,814

137. Champattanachai, Voraratt. Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia.

Degree: PhD, 2008, University of Alabama – Birmingham

Increased levels of protein O-linked-N-acetylglucosamine (O-GlcNAc) have been correlated with increased cell survival following stress. Therefore the goal of this study was to determine whether… (more)

Subjects/Keywords: Acetylglucosamine  – metabolism <; br>; Glucosamine  – pharmacology <; br>; Glycoproteins  – metabolism <; br>; Mitochondria  – metabolism <; br>; Myocardial Reperfusion Injury  – metabolism <; br>; Myocardial Reperfusion Injury  – pathology <; br>; Myocytes, Cardiac  – metabolism <; br>; Myocytes, Cardiac  – pathology

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APA (6th Edition):

Champattanachai, V. (2008). Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,194

Chicago Manual of Style (16th Edition):

Champattanachai, Voraratt. “Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed November 17, 2019. http://contentdm.mhsl.uab.edu/u?/etd,194.

MLA Handbook (7th Edition):

Champattanachai, Voraratt. “Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia.” 2008. Web. 17 Nov 2019.

Vancouver:

Champattanachai V. Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Nov 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,194.

Council of Science Editors:

Champattanachai V. Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,194

138. Mavalli, Mahendra D. Mechanisms of growth hormone action in skeletal muscle.

Degree: PhD, 2009, University of Alabama – Birmingham

Growth hormone (GH) and insulin like growth factor-1 (IGF-1) exert profound growth promoting actions during pre and postnatal skeletal muscle development. GH and IGF-1 seem… (more)

Subjects/Keywords: Growth Hormone  – metabolism<; br>; Insulin  – metabolism<; br>; Insulin Resistance<; br>; Muscle, Skeletal  – growth & development<; br>; Muscle, Skeletal  – metabolism<; br>; Receptor, IGF Type 1  – metabolism<; br>; Receptors, Somatotropin  – metabolism<; br>; Sarcopenia

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APA (6th Edition):

Mavalli, M. D. (2009). Mechanisms of growth hormone action in skeletal muscle. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1103

Chicago Manual of Style (16th Edition):

Mavalli, Mahendra D. “Mechanisms of growth hormone action in skeletal muscle.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed November 17, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1103.

MLA Handbook (7th Edition):

Mavalli, Mahendra D. “Mechanisms of growth hormone action in skeletal muscle.” 2009. Web. 17 Nov 2019.

Vancouver:

Mavalli MD. Mechanisms of growth hormone action in skeletal muscle. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Nov 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1103.

Council of Science Editors:

Mavalli MD. Mechanisms of growth hormone action in skeletal muscle. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1103

139. Tang, Yi, Ph.D. The role of transforming growth factor beta-1 in bone remodeling.

Degree: PhD, 2009, University of Alabama – Birmingham

Bone remodeling depends on the precise coordination of bone resorption by the osteoclasts and bone formation by the osteoblasts. It is proposed that osteoclastic bone… (more)

Subjects/Keywords: beta Catenin  – metabolism<; br>; Bone Regeneration  – drug effects<; br>; Cadherins  – metabolism<; br>; Cell Membrane  – metabolism<; br>; Multiprotein Complexes  – metabolism<; br>; Smad7 Protein  – metabolism<; br>; Transforming Growth Factor beta  – metabolism

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APA (6th Edition):

Tang, Yi, P. D. (2009). The role of transforming growth factor beta-1 in bone remodeling. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,405

Chicago Manual of Style (16th Edition):

Tang, Yi, Ph D. “The role of transforming growth factor beta-1 in bone remodeling.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed November 17, 2019. http://contentdm.mhsl.uab.edu/u?/etd,405.

MLA Handbook (7th Edition):

Tang, Yi, Ph D. “The role of transforming growth factor beta-1 in bone remodeling.” 2009. Web. 17 Nov 2019.

Vancouver:

Tang, Yi PD. The role of transforming growth factor beta-1 in bone remodeling. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Nov 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,405.

Council of Science Editors:

Tang, Yi PD. The role of transforming growth factor beta-1 in bone remodeling. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,405

140. Calderon, Christopher. O-Linked Beta-N-Acetylglucosamine (o-Glcnac) And The Mitochondrion.

Degree: 2013, University of Alabama – Birmingham

O-linked beta-N-acetylglucosamine (O-GlcNAc) is a dynamic and ubiquitous posttranslational modification of serine and threonine residues on nuclear and cytoplasmic proteins. O-GlcNAc has emerged as an… (more)

Subjects/Keywords: Acetylglucosamine – metabolism<; /br>; Glycosylation<; /br>; Hexosamines – biosynthesis<; /br>; Mitochondrial Membrane Transport Proteins – metabolism.<; /br>; Oxidative Stress.

…52 O-GlcNAcylation of Mitochondrial Proteins ...........................................52… …x29;, onto proteins. The removal of O-GlcNAc is regulated via a unique hexosaminidase, β… …neurodegenerative diseases [9], and many oncogenic proteins and tumor suppressor proteins are… …x28;OGT) catalyzes the addition of UDP‐GlcNAc, onto ser/thr residues of naked proteins… …cytoplasmic proteins in an attempt to characterize potential differentiation and histocompatibility… 

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APA (6th Edition):

Calderon, C. (2013). O-Linked Beta-N-Acetylglucosamine (o-Glcnac) And The Mitochondrion. (Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1660

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Calderon, Christopher. “O-Linked Beta-N-Acetylglucosamine (o-Glcnac) And The Mitochondrion.” 2013. Thesis, University of Alabama – Birmingham. Accessed November 17, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1660.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Calderon, Christopher. “O-Linked Beta-N-Acetylglucosamine (o-Glcnac) And The Mitochondrion.” 2013. Web. 17 Nov 2019.

Vancouver:

Calderon C. O-Linked Beta-N-Acetylglucosamine (o-Glcnac) And The Mitochondrion. [Internet] [Thesis]. University of Alabama – Birmingham; 2013. [cited 2019 Nov 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1660.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Calderon C. O-Linked Beta-N-Acetylglucosamine (o-Glcnac) And The Mitochondrion. [Thesis]. University of Alabama – Birmingham; 2013. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1660

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

141. 이, 수진. Studies on the Molecular Mechanisms of Tumorigenesis in Neurofibromatosis Type 1.

Degree: 2008, Ajou University

"Neurofibromatosis type 1 (NF1) is one of the most common inherited autosomal dominant disorders, with an estimated incidence of 1 per 3,500 births. NF1 is… (more)

Subjects/Keywords: 신경섬유종증; 세포자멸사; 뉴로파이브로민; GTPase activating protein(GAP); p120RasGAP; Ras; RASA1; Apoptosis; neurofibromin; NF1

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APA (6th Edition):

이, . (2008). Studies on the Molecular Mechanisms of Tumorigenesis in Neurofibromatosis Type 1. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/1836 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000006846

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

이, 수진. “Studies on the Molecular Mechanisms of Tumorigenesis in Neurofibromatosis Type 1.” 2008. Thesis, Ajou University. Accessed November 17, 2019. http://repository.ajou.ac.kr/handle/201003/1836 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000006846.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

이, 수진. “Studies on the Molecular Mechanisms of Tumorigenesis in Neurofibromatosis Type 1.” 2008. Web. 17 Nov 2019.

Vancouver:

이 . Studies on the Molecular Mechanisms of Tumorigenesis in Neurofibromatosis Type 1. [Internet] [Thesis]. Ajou University; 2008. [cited 2019 Nov 17]. Available from: http://repository.ajou.ac.kr/handle/201003/1836 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000006846.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

이 . Studies on the Molecular Mechanisms of Tumorigenesis in Neurofibromatosis Type 1. [Thesis]. Ajou University; 2008. Available from: http://repository.ajou.ac.kr/handle/201003/1836 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000006846

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

142. Bergman, Cody. Interaction between p85 and Rab5 in the presences and absence of phosphorylated PDGFR peptide.

Degree: 2012, University of Saskatchewan

 The adaptor subunit of phosphatidylinositol 3'-kinases (PI3K), p85, is involved in many different biological processes. Recent studies have shown that one of these functions is… (more)

Subjects/Keywords: PI3K; Rab5; Endocytosis; p85; PDGFR; Signalling; Peptide; GTPase Activating Protein

…dimerize and has sequence similarity to GTPase activating proteins (GAP), discussed… …endosomes (Touchot et al., 1987). There are more than 60 Rab proteins reported in humans… …small monomeric G proteins, contain a weak intrinsic GTPase activity which can be enhanced by… …a GTPase activating protein (GAP), to hydrolyze bound GTP to GDP. There have… …73 5.4.1.1 Removal of Possible GTPase Contamination From p85 Samples… 

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APA (6th Edition):

Bergman, C. (2012). Interaction between p85 and Rab5 in the presences and absence of phosphorylated PDGFR peptide. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/ETD-2012-01-315

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bergman, Cody. “Interaction between p85 and Rab5 in the presences and absence of phosphorylated PDGFR peptide.” 2012. Thesis, University of Saskatchewan. Accessed November 17, 2019. http://hdl.handle.net/10388/ETD-2012-01-315.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bergman, Cody. “Interaction between p85 and Rab5 in the presences and absence of phosphorylated PDGFR peptide.” 2012. Web. 17 Nov 2019.

Vancouver:

Bergman C. Interaction between p85 and Rab5 in the presences and absence of phosphorylated PDGFR peptide. [Internet] [Thesis]. University of Saskatchewan; 2012. [cited 2019 Nov 17]. Available from: http://hdl.handle.net/10388/ETD-2012-01-315.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bergman C. Interaction between p85 and Rab5 in the presences and absence of phosphorylated PDGFR peptide. [Thesis]. University of Saskatchewan; 2012. Available from: http://hdl.handle.net/10388/ETD-2012-01-315

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

143. Yeung, Ching-Yan. Identification of Arhgap28, a New Regulator of Stress Fibre Formation in Cells Assembling a Fibrous Extracellular Matrix.

Degree: 2012, University of Manchester

 The motivation for this PhD thesis was to understand the molecular basis of how cells regulate the formation of an organised and mechanically strong extracellular… (more)

Subjects/Keywords: tendon; Rho GTPase activating protein; extracellular matrix; Arhgap28; tendon

…specifically by Rho GTPase activating proteins (RhoGAPs). By… …small GTPase RhoA is the major regulator of actin… …novel RhoGTPase activating protein (GAP) that is present… 

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APA (6th Edition):

Yeung, C. (2012). Identification of Arhgap28, a New Regulator of Stress Fibre Formation in Cells Assembling a Fibrous Extracellular Matrix. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:170516

Chicago Manual of Style (16th Edition):

Yeung, Ching-Yan. “Identification of Arhgap28, a New Regulator of Stress Fibre Formation in Cells Assembling a Fibrous Extracellular Matrix.” 2012. Doctoral Dissertation, University of Manchester. Accessed November 17, 2019. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:170516.

MLA Handbook (7th Edition):

Yeung, Ching-Yan. “Identification of Arhgap28, a New Regulator of Stress Fibre Formation in Cells Assembling a Fibrous Extracellular Matrix.” 2012. Web. 17 Nov 2019.

Vancouver:

Yeung C. Identification of Arhgap28, a New Regulator of Stress Fibre Formation in Cells Assembling a Fibrous Extracellular Matrix. [Internet] [Doctoral dissertation]. University of Manchester; 2012. [cited 2019 Nov 17]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:170516.

Council of Science Editors:

Yeung C. Identification of Arhgap28, a New Regulator of Stress Fibre Formation in Cells Assembling a Fibrous Extracellular Matrix. [Doctoral Dissertation]. University of Manchester; 2012. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:170516


Vanderbilt University

144. Mehta, Sapna. Characterization of Byr4 and Cdc7 functional domains affecting the septation initiation network in &#60;i>Schizosaccharomyces pombe&#60;/i>.

Degree: PhD, Cell and Developmental Biology, 2005, Vanderbilt University

 The study of cytokinesis in fission yeast S.pombe has revealed a signaling network, the septation initiation network (SIN) that serves to coordinate cytokinesis with mitotic… (more)

Subjects/Keywords: kinase; cytokinesis; GTPase activating protein; septation initiation network; Fission yeast

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APA (6th Edition):

Mehta, S. (2005). Characterization of Byr4 and Cdc7 functional domains affecting the septation initiation network in <i>Schizosaccharomyces pombe</i>. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-06212005-165627/ ;

Chicago Manual of Style (16th Edition):

Mehta, Sapna. “Characterization of Byr4 and Cdc7 functional domains affecting the septation initiation network in <i>Schizosaccharomyces pombe</i>.” 2005. Doctoral Dissertation, Vanderbilt University. Accessed November 17, 2019. http://etd.library.vanderbilt.edu/available/etd-06212005-165627/ ;.

MLA Handbook (7th Edition):

Mehta, Sapna. “Characterization of Byr4 and Cdc7 functional domains affecting the septation initiation network in <i>Schizosaccharomyces pombe</i>.” 2005. Web. 17 Nov 2019.

Vancouver:

Mehta S. Characterization of Byr4 and Cdc7 functional domains affecting the septation initiation network in <i>Schizosaccharomyces pombe</i>. [Internet] [Doctoral dissertation]. Vanderbilt University; 2005. [cited 2019 Nov 17]. Available from: http://etd.library.vanderbilt.edu/available/etd-06212005-165627/ ;.

Council of Science Editors:

Mehta S. Characterization of Byr4 and Cdc7 functional domains affecting the septation initiation network in <i>Schizosaccharomyces pombe</i>. [Doctoral Dissertation]. Vanderbilt University; 2005. Available from: http://etd.library.vanderbilt.edu/available/etd-06212005-165627/ ;

145. Jiang, Shaoning. Molecular mechanisms of hepatic insulin resistance following injury.

Degree: PhD, 2010, University of Alabama – Birmingham

Insulin resistance commonly occurs following injuries or critical illness independent of previous diabetic status. The development of insulin resistance and hyperglycemia is associated with increased… (more)

Subjects/Keywords: Adenoviridae – metabolism<; br>; Adenoviridae Infections – metabolism<; br>; Diabetes Mellitus, Type 2 – physiopathology<; br>; Glucose – metabolism<; br>; I-kappa B Kinase – physiology <; br>; Insulin – metabolism<; br>; JNK Mitogen-Activated Protein Kinases – physiology <; br>; Liver

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APA (6th Edition):

Jiang, S. (2010). Molecular mechanisms of hepatic insulin resistance following injury. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1374

Chicago Manual of Style (16th Edition):

Jiang, Shaoning. “Molecular mechanisms of hepatic insulin resistance following injury.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed November 17, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1374.

MLA Handbook (7th Edition):

Jiang, Shaoning. “Molecular mechanisms of hepatic insulin resistance following injury.” 2010. Web. 17 Nov 2019.

Vancouver:

Jiang S. Molecular mechanisms of hepatic insulin resistance following injury. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Nov 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1374.

Council of Science Editors:

Jiang S. Molecular mechanisms of hepatic insulin resistance following injury. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1374

146. Zhai, Lidong. Involvement of reactive oxygen species in the acute development of insulin resistance following trauma and hemorrhage.

Degree: PhD, 2009, University of Alabama – Birmingham

Hyperglycemia develops in the intensive care unit in many patients following injury, infection and critical illness. However, little is known about the mechanism of acute… (more)

Subjects/Keywords: Aging  – metabolism<; br>; Insulin Resistance<; br>; Liver  – metabolism<; br>; Muscle, Skeletal  – metabolism<; br>; Oxidative Stress<; br>; Shock, Hemorrhagic  – metabolism<; br>; Wounds and Injuries  – metabolism

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APA (6th Edition):

Zhai, L. (2009). Involvement of reactive oxygen species in the acute development of insulin resistance following trauma and hemorrhage. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,834

Chicago Manual of Style (16th Edition):

Zhai, Lidong. “Involvement of reactive oxygen species in the acute development of insulin resistance following trauma and hemorrhage.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed November 17, 2019. http://contentdm.mhsl.uab.edu/u?/etd,834.

MLA Handbook (7th Edition):

Zhai, Lidong. “Involvement of reactive oxygen species in the acute development of insulin resistance following trauma and hemorrhage.” 2009. Web. 17 Nov 2019.

Vancouver:

Zhai L. Involvement of reactive oxygen species in the acute development of insulin resistance following trauma and hemorrhage. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Nov 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,834.

Council of Science Editors:

Zhai L. Involvement of reactive oxygen species in the acute development of insulin resistance following trauma and hemorrhage. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,834

147. Fulzele, Keertik. Insulin signaling and function in osteoblasts.

Degree: PhD, 2009, University of Alabama – Birmingham

Insulin and insulin-like growth factor-1 (IGF-1) are evolutionarily conserved hormonal signaling pathways with structurally similar ligands and receptors. Recent studies suggest that that insulin and… (more)

Subjects/Keywords: Insulin  – metabolism<; br>; Osteoblasts  – metabolism<; br>; Osteogenesis  – physiology<; br>; Receptor, IGF Type 1  – metabolism<; br>; Receptor, Insulin  – metabolism<; br>; Signal Transduction  – physiology<; br>; Skull  – metabolism

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APA (6th Edition):

Fulzele, K. (2009). Insulin signaling and function in osteoblasts. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,999

Chicago Manual of Style (16th Edition):

Fulzele, Keertik. “Insulin signaling and function in osteoblasts.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed November 17, 2019. http://contentdm.mhsl.uab.edu/u?/etd,999.

MLA Handbook (7th Edition):

Fulzele, Keertik. “Insulin signaling and function in osteoblasts.” 2009. Web. 17 Nov 2019.

Vancouver:

Fulzele K. Insulin signaling and function in osteoblasts. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Nov 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,999.

Council of Science Editors:

Fulzele K. Insulin signaling and function in osteoblasts. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,999

148. Vitturi, Dario A. Red blood cell and hemoglobin dependent modulation of reactive species metabolism: implications for vascular homeostasis.

Degree: PhD, 2010, University of Alabama – Birmingham

The recent discovery that red blood cells might mediate hypoxic blood flow together with accumulating evidence that suggests a role for the modulation of vascular… (more)

Subjects/Keywords: Anoxia  – blood<; br>; Antihypertensive Agents<; br>; Blood Substitutes<; br>; Erythrocytes  – metabolism<; br>; Hemoglobins  – metabolism<; br>; Hypertension  – drug therapy<; br>; Nitrites  – blood<; br>; Oxygen  – blood<; br>; Oxyhemoglobins  – metabolism

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APA (6th Edition):

Vitturi, D. A. (2010). Red blood cell and hemoglobin dependent modulation of reactive species metabolism: implications for vascular homeostasis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1004

Chicago Manual of Style (16th Edition):

Vitturi, Dario A. “Red blood cell and hemoglobin dependent modulation of reactive species metabolism: implications for vascular homeostasis.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed November 17, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1004.

MLA Handbook (7th Edition):

Vitturi, Dario A. “Red blood cell and hemoglobin dependent modulation of reactive species metabolism: implications for vascular homeostasis.” 2010. Web. 17 Nov 2019.

Vancouver:

Vitturi DA. Red blood cell and hemoglobin dependent modulation of reactive species metabolism: implications for vascular homeostasis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Nov 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1004.

Council of Science Editors:

Vitturi DA. Red blood cell and hemoglobin dependent modulation of reactive species metabolism: implications for vascular homeostasis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1004

149. Pawar, Pritish Subhash. Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma.

Degree: PhD, 2008, University of Alabama – Birmingham

Cholangiocarcinoma, a fatal tumor arising from biliary epithelium, has very poor 5-year survival rate due to lack of early diagnosis and effective therapies. Induction of… (more)

Subjects/Keywords: Antigens, CD95  – metabolism <; br>; CASP8 and FADD-Like Apoptosis Regulating Protein  – metabolism <; br>; Calmodulin  – metabolism <; br>; Cholangiocarcinoma <; br>; Signal Transduction

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APA (6th Edition):

Pawar, P. S. (2008). Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,301

Chicago Manual of Style (16th Edition):

Pawar, Pritish Subhash. “Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed November 17, 2019. http://contentdm.mhsl.uab.edu/u?/etd,301.

MLA Handbook (7th Edition):

Pawar, Pritish Subhash. “Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma.” 2008. Web. 17 Nov 2019.

Vancouver:

Pawar PS. Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Nov 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,301.

Council of Science Editors:

Pawar PS. Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,301

150. Ritchie, Joseph P. Heparanase drives the aggressive myeloma phenotype: preclinical development of a heparanase inhibitor for the treatment of multiple myeloma.

Degree: PhD, 2010, University of Alabama – Birmingham

Heparanase, an endoglycosidase which cleaves heparan sulfate chains at specific sites, is rarely expressed in normal tissues but becomes evident in many human cancers. We… (more)

Subjects/Keywords: Heparin  – metabolism<; br>; Heparitin Sulfate  – metabolism<; br>; Multiple Myeloma  – pathology<; br>; Neoplasm Invasiveness<; br>; Syndecan-1  – metabolism

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ritchie, J. P. (2010). Heparanase drives the aggressive myeloma phenotype: preclinical development of a heparanase inhibitor for the treatment of multiple myeloma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,852

Chicago Manual of Style (16th Edition):

Ritchie, Joseph P. “Heparanase drives the aggressive myeloma phenotype: preclinical development of a heparanase inhibitor for the treatment of multiple myeloma.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed November 17, 2019. http://contentdm.mhsl.uab.edu/u?/etd,852.

MLA Handbook (7th Edition):

Ritchie, Joseph P. “Heparanase drives the aggressive myeloma phenotype: preclinical development of a heparanase inhibitor for the treatment of multiple myeloma.” 2010. Web. 17 Nov 2019.

Vancouver:

Ritchie JP. Heparanase drives the aggressive myeloma phenotype: preclinical development of a heparanase inhibitor for the treatment of multiple myeloma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Nov 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,852.

Council of Science Editors:

Ritchie JP. Heparanase drives the aggressive myeloma phenotype: preclinical development of a heparanase inhibitor for the treatment of multiple myeloma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,852

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