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You searched for subject:( GTPase Activating Proteins metabolism 60). Showing records 1 – 30 of 21425 total matches.

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1. Funk, Adam J. Intracellular signaling abnormalities in schizophrenia.

Degree: PhD, 2011, University of Alabama – Birmingham

The pathophysiology of schizophrenia is complex and diverse, with many classes of receptors, neurotransmitters, and brain regions implicated in this illness. The many hypotheses proposed… (more)

Subjects/Keywords: Carrier Proteins  – metabolism<; br>; GTPase-Activating Proteins  – metabolism<; br>; Gyrus Cinguli  – metabolism<; br>; Intracellular Signaling Peptides and Proteins  – metabolism<; br>; Membrane Proteins  – metabolism<; br>; Prefrontal Cortex  – metabolism<; br>; Schizophrenia  – metabolism

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APA (6th Edition):

Funk, A. J. (2011). Intracellular signaling abnormalities in schizophrenia. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1151

Chicago Manual of Style (16th Edition):

Funk, Adam J. “Intracellular signaling abnormalities in schizophrenia.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 21, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1151.

MLA Handbook (7th Edition):

Funk, Adam J. “Intracellular signaling abnormalities in schizophrenia.” 2011. Web. 21 Oct 2019.

Vancouver:

Funk AJ. Intracellular signaling abnormalities in schizophrenia. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Oct 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1151.

Council of Science Editors:

Funk AJ. Intracellular signaling abnormalities in schizophrenia. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1151

2. Moore, Carlene Drucilla. The role of centaurin alpha-1 in the regulation of neuronal differentiation.

Degree: PhD, 2008, University of Alabama – Birmingham

In the nervous system, PI 3-kinase has been implicated in neuronal differentiation. My studies have focused on a candidate neuronal PI 3-kinase target centaurin alpha-1,… (more)

Subjects/Keywords: 1-Phosphatidylinositol 3-Kinase <; br>; Adaptor Proteins, Signal Transducing  – metabolism <; br>; Dendrites  – metabolism <; br>; Dendrites  – ultrastructure <; br>; GTPase-Activating Proteins  – metabolism <; br>; Hippocampus  – cytology <; br>; Nerve Tissue Proteins  – metabolism <; br>; Neurons  – metabolism

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APA (6th Edition):

Moore, C. D. (2008). The role of centaurin alpha-1 in the regulation of neuronal differentiation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,208

Chicago Manual of Style (16th Edition):

Moore, Carlene Drucilla. “The role of centaurin alpha-1 in the regulation of neuronal differentiation.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 21, 2019. http://contentdm.mhsl.uab.edu/u?/etd,208.

MLA Handbook (7th Edition):

Moore, Carlene Drucilla. “The role of centaurin alpha-1 in the regulation of neuronal differentiation.” 2008. Web. 21 Oct 2019.

Vancouver:

Moore CD. The role of centaurin alpha-1 in the regulation of neuronal differentiation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,208.

Council of Science Editors:

Moore CD. The role of centaurin alpha-1 in the regulation of neuronal differentiation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,208

3. Larimore, Jennifer Lynn. The role of centaurins in vesicular trafficking and neuronal differentiation.

Degree: PhD, 2008, University of Alabama – Birmingham

In order to better understand the role of the phosphoinositide (PI) 3-Kinase (PI3K) pathway in neuronal differentiation and plasticity, I am studying two centaurins that… (more)

Subjects/Keywords: Adaptor Proteins, Signal Transducing  – metabolism<; br>; Dendrites  – metabolism<; br>; Dentrites  – ultrastructure<; br>; GTPase-Activating Proteins  – metabolism<; br>; Hippocampus  – cytology<; br>; Neurons  – metabolism

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APA (6th Edition):

Larimore, J. L. (2008). The role of centaurins in vesicular trafficking and neuronal differentiation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,823

Chicago Manual of Style (16th Edition):

Larimore, Jennifer Lynn. “The role of centaurins in vesicular trafficking and neuronal differentiation.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 21, 2019. http://contentdm.mhsl.uab.edu/u?/etd,823.

MLA Handbook (7th Edition):

Larimore, Jennifer Lynn. “The role of centaurins in vesicular trafficking and neuronal differentiation.” 2008. Web. 21 Oct 2019.

Vancouver:

Larimore JL. The role of centaurins in vesicular trafficking and neuronal differentiation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,823.

Council of Science Editors:

Larimore JL. The role of centaurins in vesicular trafficking and neuronal differentiation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,823


University of Texas Southwestern Medical Center

4. Peru Y Colón de Portugal, Raniero Leonette. Brain Molecules under the Influence: Intracellular Regulation of Behavioral Responses Induced by Ethanol.

Degree: 2013, University of Texas Southwestern Medical Center

 Alcohol abuse is a devastating condition affecting millions of individuals. Regulation of insulin receptor (InR) signaling is critical for ethanol-induced responses and consumatory ethanol behavior.… (more)

Subjects/Keywords: Ethanol; ADP-Ribosylation Factors; GTPase-Activating Proteins

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APA (6th Edition):

Peru Y Colón de Portugal, R. L. (2013). Brain Molecules under the Influence: Intracellular Regulation of Behavioral Responses Induced by Ethanol. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1253

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Peru Y Colón de Portugal, Raniero Leonette. “Brain Molecules under the Influence: Intracellular Regulation of Behavioral Responses Induced by Ethanol.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed October 21, 2019. http://hdl.handle.net/2152.5/1253.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Peru Y Colón de Portugal, Raniero Leonette. “Brain Molecules under the Influence: Intracellular Regulation of Behavioral Responses Induced by Ethanol.” 2013. Web. 21 Oct 2019.

Vancouver:

Peru Y Colón de Portugal RL. Brain Molecules under the Influence: Intracellular Regulation of Behavioral Responses Induced by Ethanol. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/2152.5/1253.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Peru Y Colón de Portugal RL. Brain Molecules under the Influence: Intracellular Regulation of Behavioral Responses Induced by Ethanol. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1253

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

5. Lowery, Jason. Regulation Of Membrane Traffic By The Big2 Member Of Guanine Nucleotide Exchange Factors.

Degree: PhD, 2012, University of Alabama – Birmingham

Vesicular transport is an essential cellular process that facilitates the movement of molecules within a cell. The importance of vesicular transport is highlighted by numerous… (more)

Subjects/Keywords: ADP-Ribosylation Factors – chemistry.<; br>; GTPase-Activating Proteins – chemistry.<; br>; Guanine Nucleotide Exchange Factors – chemistry.<; br>; Guanosine Diphosphate – chemistry.<; br>; Guanosine Triphosphate – chemistry.<; br>; Protein Structure, Tertiary

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APA (6th Edition):

Lowery, J. (2012). Regulation Of Membrane Traffic By The Big2 Member Of Guanine Nucleotide Exchange Factors. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1432

Chicago Manual of Style (16th Edition):

Lowery, Jason. “Regulation Of Membrane Traffic By The Big2 Member Of Guanine Nucleotide Exchange Factors.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 21, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1432.

MLA Handbook (7th Edition):

Lowery, Jason. “Regulation Of Membrane Traffic By The Big2 Member Of Guanine Nucleotide Exchange Factors.” 2012. Web. 21 Oct 2019.

Vancouver:

Lowery J. Regulation Of Membrane Traffic By The Big2 Member Of Guanine Nucleotide Exchange Factors. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2019 Oct 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1432.

Council of Science Editors:

Lowery J. Regulation Of Membrane Traffic By The Big2 Member Of Guanine Nucleotide Exchange Factors. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1432


Columbia University

6. Chang, Donald Dao-Yuan. The Regulation and Function of RGK Proteins on Voltage-Gated Calcium Channel Physiology.

Degree: 2015, Columbia University

 Rad/Rem/Rem2/Gem/Kir (RGK) proteins are Ras-like GTPases with diverse (and expanding) functions including: regulating cytoskeleton dynamics, cell proliferation, synaptogenesis, and inhibition of high voltage-dependent calcium (CaV)… (more)

Subjects/Keywords: GTPase-activating protein; Proteins – Analysis; Calcium channels; Physiology; Biophysics

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APA (6th Edition):

Chang, D. D. (2015). The Regulation and Function of RGK Proteins on Voltage-Gated Calcium Channel Physiology. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8251H2V

Chicago Manual of Style (16th Edition):

Chang, Donald Dao-Yuan. “The Regulation and Function of RGK Proteins on Voltage-Gated Calcium Channel Physiology.” 2015. Doctoral Dissertation, Columbia University. Accessed October 21, 2019. https://doi.org/10.7916/D8251H2V.

MLA Handbook (7th Edition):

Chang, Donald Dao-Yuan. “The Regulation and Function of RGK Proteins on Voltage-Gated Calcium Channel Physiology.” 2015. Web. 21 Oct 2019.

Vancouver:

Chang DD. The Regulation and Function of RGK Proteins on Voltage-Gated Calcium Channel Physiology. [Internet] [Doctoral dissertation]. Columbia University; 2015. [cited 2019 Oct 21]. Available from: https://doi.org/10.7916/D8251H2V.

Council of Science Editors:

Chang DD. The Regulation and Function of RGK Proteins on Voltage-Gated Calcium Channel Physiology. [Doctoral Dissertation]. Columbia University; 2015. Available from: https://doi.org/10.7916/D8251H2V


McGill University

7. Kalantari, Fariba. A cellular and molecular approach to investigate pathological calcification in liver.

Degree: PhD, Department of Anatomy and Cell Biology., 2008, McGill University

The liver is a vital organ, playing numerous critical roles in the body. The liver's ability to perform essential functions is disturbed by injuries that… (more)

Subjects/Keywords: Calcinosis  – etiology.; Liver Diseases  – pathology.; Liver Transplantation  – pathology.; ras GTPase-Activating Proteins  – metabolism.

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APA (6th Edition):

Kalantari, F. (2008). A cellular and molecular approach to investigate pathological calcification in liver. (Doctoral Dissertation). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile111907.pdf

Chicago Manual of Style (16th Edition):

Kalantari, Fariba. “A cellular and molecular approach to investigate pathological calcification in liver.” 2008. Doctoral Dissertation, McGill University. Accessed October 21, 2019. http://digitool.library.mcgill.ca/thesisfile111907.pdf.

MLA Handbook (7th Edition):

Kalantari, Fariba. “A cellular and molecular approach to investigate pathological calcification in liver.” 2008. Web. 21 Oct 2019.

Vancouver:

Kalantari F. A cellular and molecular approach to investigate pathological calcification in liver. [Internet] [Doctoral dissertation]. McGill University; 2008. [cited 2019 Oct 21]. Available from: http://digitool.library.mcgill.ca/thesisfile111907.pdf.

Council of Science Editors:

Kalantari F. A cellular and molecular approach to investigate pathological calcification in liver. [Doctoral Dissertation]. McGill University; 2008. Available from: http://digitool.library.mcgill.ca/thesisfile111907.pdf


University of Texas Southwestern Medical Center

8. Koo, Yeon Seung. Blood Vessel Development.

Degree: 2014, University of Texas Southwestern Medical Center

 Cardiovascular system is the first developing functional organ in vertebrates, and one of the fundamental organ systems through adulthoods. Cardiovascular function depends on patent blood… (more)

Subjects/Keywords: Blood Vessels; Endothelium, Vascular; GTPase-Activating Proteins; Intracellular Signaling Peptides and Proteins

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APA (6th Edition):

Koo, Y. S. (2014). Blood Vessel Development. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3949

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Koo, Yeon Seung. “Blood Vessel Development.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed October 21, 2019. http://hdl.handle.net/2152.5/3949.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Koo, Yeon Seung. “Blood Vessel Development.” 2014. Web. 21 Oct 2019.

Vancouver:

Koo YS. Blood Vessel Development. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/2152.5/3949.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Koo YS. Blood Vessel Development. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3949

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. Li, Xinrui. Regulation of B cell biology by FCγ receptors.

Degree: PhD, 2009, University of Alabama – Birmingham

B cell development, maturation and proliferation are all strictly regulated processes. Disruption of the regulatory network at any step would lead to immune disorders. The… (more)

Subjects/Keywords: B-Cell Activating Factor  – metabolism<; br>; B-Lymphocytes  – immunology<; br>; Myeloid Cells  – immunology<; br>; Opsonin Proteins  – immunology<; br>; Polymorphism, Single Nucleotide<; br>; Receptors, IgG  – immunology

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APA (6th Edition):

Li, X. (2009). Regulation of B cell biology by FCγ receptors. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,915

Chicago Manual of Style (16th Edition):

Li, Xinrui. “Regulation of B cell biology by FCγ receptors.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 21, 2019. http://contentdm.mhsl.uab.edu/u?/etd,915.

MLA Handbook (7th Edition):

Li, Xinrui. “Regulation of B cell biology by FCγ receptors.” 2009. Web. 21 Oct 2019.

Vancouver:

Li X. Regulation of B cell biology by FCγ receptors. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,915.

Council of Science Editors:

Li X. Regulation of B cell biology by FCγ receptors. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,915


McGill University

10. Danek, Eric Ian. Characterization of regulatory mechanisms of CdGAP, a negative regulator of the small GTPases Rac1 and Cdc42.

Degree: PhD, Department of Anatomy and Cell Biology., 2008, McGill University

 The Rho GTPases form a diverse family of proteins which regulates numerous cell processes from cytoskeletal reorganization to control of gene expression. They are negatively… (more)

Subjects/Keywords: GTPase-Activating Proteins  – metabolism.; Mitogen-Activated Protein Kinase 3  – metabolism.; Glycogen Synthase Kinase 3  – metabolism.; Phosphorylation.; rac1 GTP-Binding Protein  – metabolism.; cdc42 GTP-Binding Protein  – metabolism.

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APA (6th Edition):

Danek, E. I. (2008). Characterization of regulatory mechanisms of CdGAP, a negative regulator of the small GTPases Rac1 and Cdc42. (Doctoral Dissertation). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile113853.pdf

Chicago Manual of Style (16th Edition):

Danek, Eric Ian. “Characterization of regulatory mechanisms of CdGAP, a negative regulator of the small GTPases Rac1 and Cdc42.” 2008. Doctoral Dissertation, McGill University. Accessed October 21, 2019. http://digitool.library.mcgill.ca/thesisfile113853.pdf.

MLA Handbook (7th Edition):

Danek, Eric Ian. “Characterization of regulatory mechanisms of CdGAP, a negative regulator of the small GTPases Rac1 and Cdc42.” 2008. Web. 21 Oct 2019.

Vancouver:

Danek EI. Characterization of regulatory mechanisms of CdGAP, a negative regulator of the small GTPases Rac1 and Cdc42. [Internet] [Doctoral dissertation]. McGill University; 2008. [cited 2019 Oct 21]. Available from: http://digitool.library.mcgill.ca/thesisfile113853.pdf.

Council of Science Editors:

Danek EI. Characterization of regulatory mechanisms of CdGAP, a negative regulator of the small GTPases Rac1 and Cdc42. [Doctoral Dissertation]. McGill University; 2008. Available from: http://digitool.library.mcgill.ca/thesisfile113853.pdf

11. Beagle, Brandon Richard. Canonical Wnt signaling by the proteolytic processing of LRP6.

Degree: PhD, 2010, University of Alabama – Birmingham

Low density Lipoprotein receptor Related 6 (LRP6) functions as an essential coreceptor for Wnt/β-catenin signaling as pathway activation, reflected by cytosolic β- catenin stabilization and… (more)

Subjects/Keywords: beta Catenin  – metabolism<; br>; Glycogen Synthase Kinase 3  – metabolism<; br>; LDL-Receptor Related Proteins  – metabolism<; br>; Lymphoid Enhancer-Binding Factor 1  – metabolism<; br>; Repressor Proteins  – metabolism<; br>; Transcription Factors  – metabolism<; br>; Wnt Proteins  – metabolism

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APA (6th Edition):

Beagle, B. R. (2010). Canonical Wnt signaling by the proteolytic processing of LRP6. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,857

Chicago Manual of Style (16th Edition):

Beagle, Brandon Richard. “Canonical Wnt signaling by the proteolytic processing of LRP6.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 21, 2019. http://contentdm.mhsl.uab.edu/u?/etd,857.

MLA Handbook (7th Edition):

Beagle, Brandon Richard. “Canonical Wnt signaling by the proteolytic processing of LRP6.” 2010. Web. 21 Oct 2019.

Vancouver:

Beagle BR. Canonical Wnt signaling by the proteolytic processing of LRP6. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,857.

Council of Science Editors:

Beagle BR. Canonical Wnt signaling by the proteolytic processing of LRP6. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,857

12. Danilchanka, Olga V. Diffusion pathways through the outer membrane of mycobacteria.

Degree: PhD, 2009, University of Alabama – Birmingham

The extraordinary capacity of Mycobacterium tuberculosis (Mtb) to adapt to environmental changes during infection contributes to its success as a pathogen. While the unique outer… (more)

Subjects/Keywords: Anti-Bacterial Agents  – metabolism<; br>; Bacterial Proteins  – metabolism<; br>; Chloramphenicol  – metabolism<; br>; Fluoroquinolones  – metabolism<; br>; Membrane Transport Proteins  – metabolism<; br>; Mycobacterium smegmatis<; br>; Mycobacterium tuberculosis  – metabolism<; br>; Porins  – metabolism

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APA (6th Edition):

Danilchanka, O. V. (2009). Diffusion pathways through the outer membrane of mycobacteria. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1150

Chicago Manual of Style (16th Edition):

Danilchanka, Olga V. “Diffusion pathways through the outer membrane of mycobacteria.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 21, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1150.

MLA Handbook (7th Edition):

Danilchanka, Olga V. “Diffusion pathways through the outer membrane of mycobacteria.” 2009. Web. 21 Oct 2019.

Vancouver:

Danilchanka OV. Diffusion pathways through the outer membrane of mycobacteria. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1150.

Council of Science Editors:

Danilchanka OV. Diffusion pathways through the outer membrane of mycobacteria. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1150

13. Joo, Heui Yun. Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions.

Degree: PhD, 2009, University of Alabama – Birmingham

Posttranslational modifications of histones regulate important chromatin and cellular functions. Among them, ubiquitination of histone H2A is correlated to transcriptional repression, such as HOX gene… (more)

Subjects/Keywords: Chromatin  – physiology<; br>; Endopeptidases  – metabolism<; br>; Histones  – metabolism<; br>; Ubiquitin Thiolesterase  – metabolism<; br>; Xenopus Proteins  – metabolism<; br>; Xenopus laevis  – embryology

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APA (6th Edition):

Joo, H. Y. (2009). Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1101

Chicago Manual of Style (16th Edition):

Joo, Heui Yun. “Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 21, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1101.

MLA Handbook (7th Edition):

Joo, Heui Yun. “Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions.” 2009. Web. 21 Oct 2019.

Vancouver:

Joo HY. Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1101.

Council of Science Editors:

Joo HY. Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1101

14. Grabski, Robert. Using RNA interference to study the function of the tethering protein p115 in ER-Golgi traffic.

Degree: PhD, 2008, University of Alabama – Birmingham

Metazoan cells are characterized with elaborate network of intracellular membranous compartments. These membranes allow the cell to spatially separate antagonistic processes and environments, and maintain… (more)

Subjects/Keywords: Carrier Proteins  – metabolism <; br>; Golgi Apparatus  – metabolism <; br>; Membrane Proteins  – metabolism <; br>; Protein Transport <; br>; RNA Interference

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APA (6th Edition):

Grabski, R. (2008). Using RNA interference to study the function of the tethering protein p115 in ER-Golgi traffic. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,328

Chicago Manual of Style (16th Edition):

Grabski, Robert. “Using RNA interference to study the function of the tethering protein p115 in ER-Golgi traffic.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 21, 2019. http://contentdm.mhsl.uab.edu/u?/etd,328.

MLA Handbook (7th Edition):

Grabski, Robert. “Using RNA interference to study the function of the tethering protein p115 in ER-Golgi traffic.” 2008. Web. 21 Oct 2019.

Vancouver:

Grabski R. Using RNA interference to study the function of the tethering protein p115 in ER-Golgi traffic. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,328.

Council of Science Editors:

Grabski R. Using RNA interference to study the function of the tethering protein p115 in ER-Golgi traffic. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,328

15. Cui, Wenjun. Structural and mechanistic studies on ER UPR sensor PERK and mitochondrial translocon element TIM44.

Degree: PhD, 2010, University of Alabama – Birmingham

The unfolded protein response is one mechanism utilized by endoplasmic reticulum (ER) to maintain the homeostasis between ER protein folding machinery and ER proteins. UPR… (more)

Subjects/Keywords: Mitochondrial Membrane Transport Proteins  – chemistry<; br>; Mitochondrial Membrane Transport Proteins  – metabolism<; br>; Mitochondrial Membranes  – metabolism<; br>; Saccharomyces cerevisiae  – metabolism<; br>; Saccharomyces cerevisiae Proteins  – chemistry<; br>; Saccharomyces cerevisiae Proteins  – metabolism<; br>; Unfolded Protein Response

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APA (6th Edition):

Cui, W. (2010). Structural and mechanistic studies on ER UPR sensor PERK and mitochondrial translocon element TIM44. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1088

Chicago Manual of Style (16th Edition):

Cui, Wenjun. “Structural and mechanistic studies on ER UPR sensor PERK and mitochondrial translocon element TIM44.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 21, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1088.

MLA Handbook (7th Edition):

Cui, Wenjun. “Structural and mechanistic studies on ER UPR sensor PERK and mitochondrial translocon element TIM44.” 2010. Web. 21 Oct 2019.

Vancouver:

Cui W. Structural and mechanistic studies on ER UPR sensor PERK and mitochondrial translocon element TIM44. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1088.

Council of Science Editors:

Cui W. Structural and mechanistic studies on ER UPR sensor PERK and mitochondrial translocon element TIM44. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1088

16. Indran, Sabarish Vellatheri. Role of the human cytomegalovirus tegument protein pp150 in the trafficking and assembly of infectious virions.

Degree: PhD, 2010, University of Alabama – Birmingham

Human cytomegalovirus, a ubiquitous human pathogen, establishes a persistent infection in the infected host. HCMV assembly takes place in the nucleus and cytoplasm of infected… (more)

Subjects/Keywords: Adaptor Proteins, Signal Transducing – physiology.<; br>; Cytomegalovirus<; br>; Cytoskeletal Proteins – physiology<; br>; Host-Pathogen Interactions.<; br>; Phosphoproteins – metabolism<; br>; Viral Matrix Proteins – metabolism.<; br>; Virus Assembly.<; br>; rab GTP-Binding Proteins – metabolism.

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APA (6th Edition):

Indran, S. V. (2010). Role of the human cytomegalovirus tegument protein pp150 in the trafficking and assembly of infectious virions. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1351

Chicago Manual of Style (16th Edition):

Indran, Sabarish Vellatheri. “Role of the human cytomegalovirus tegument protein pp150 in the trafficking and assembly of infectious virions.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 21, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1351.

MLA Handbook (7th Edition):

Indran, Sabarish Vellatheri. “Role of the human cytomegalovirus tegument protein pp150 in the trafficking and assembly of infectious virions.” 2010. Web. 21 Oct 2019.

Vancouver:

Indran SV. Role of the human cytomegalovirus tegument protein pp150 in the trafficking and assembly of infectious virions. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1351.

Council of Science Editors:

Indran SV. Role of the human cytomegalovirus tegument protein pp150 in the trafficking and assembly of infectious virions. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1351

17. Balasubramani, Anand. Evolutionarily Conserved Cis-Acting Elements Regulate Lineage-Specific Expression Of Ifng.

Degree: PhD, 2010, University of Alabama – Birmingham

The ability to differentially manipulate available genetic information in order to generate diverse cellular identities represents an innovation of complex multicellular eukaryotic organisms. Cis-acting modules… (more)

Subjects/Keywords: DNA Replication – physiology.<; br>; Drosophila – metabolism.<; br>; Drosophila Proteins – metabolism.<; br>; GTP Phosphohydrolases – metabolism.<; br>; Microfilament Proteins – metabolism.<; br>; Multiprotein Complexes – metabolism.<; br>; Origin Recognition Complex – metabolism.

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APA (6th Edition):

Balasubramani, A. (2010). Evolutionarily Conserved Cis-Acting Elements Regulate Lineage-Specific Expression Of Ifng. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1428

Chicago Manual of Style (16th Edition):

Balasubramani, Anand. “Evolutionarily Conserved Cis-Acting Elements Regulate Lineage-Specific Expression Of Ifng.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 21, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1428.

MLA Handbook (7th Edition):

Balasubramani, Anand. “Evolutionarily Conserved Cis-Acting Elements Regulate Lineage-Specific Expression Of Ifng.” 2010. Web. 21 Oct 2019.

Vancouver:

Balasubramani A. Evolutionarily Conserved Cis-Acting Elements Regulate Lineage-Specific Expression Of Ifng. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1428.

Council of Science Editors:

Balasubramani A. Evolutionarily Conserved Cis-Acting Elements Regulate Lineage-Specific Expression Of Ifng. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1428

18. Matthews, Tori A. Pathological modifications of tau induce toxicity and facilitate cell death.

Degree: PhD, 2009, University of Alabama – Birmingham

lzheimer’s disease (AD) is a progressive neurodegenerative disorder characterized by two major pathophysiological hallmarks, beta-amyloid (A[Beta]) plaques and tau tangles. In AD and other tau… (more)

Subjects/Keywords: Caspases  – metabolism<; br>; Cell Death  – physiology<; br>; Microtubules  – metabolism<; br>; tau Proteins  – physiology

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APA (6th Edition):

Matthews, T. A. (2009). Pathological modifications of tau induce toxicity and facilitate cell death. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,607

Chicago Manual of Style (16th Edition):

Matthews, Tori A. “Pathological modifications of tau induce toxicity and facilitate cell death.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 21, 2019. http://contentdm.mhsl.uab.edu/u?/etd,607.

MLA Handbook (7th Edition):

Matthews, Tori A. “Pathological modifications of tau induce toxicity and facilitate cell death.” 2009. Web. 21 Oct 2019.

Vancouver:

Matthews TA. Pathological modifications of tau induce toxicity and facilitate cell death. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,607.

Council of Science Editors:

Matthews TA. Pathological modifications of tau induce toxicity and facilitate cell death. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,607

19. Masyukova, Svetlana V. Analysis of NPHP complex genetic interactions associated with human cilia disorders.

Degree: PhD, 2011, University of Alabama – Birmingham

Primary cilia are antenna-like organelles that extend from the surface of almost all mammalian cell types. They regulate many signaling pathways and sense physical and… (more)

Subjects/Keywords: Caenorhabditis elegans Proteins – metabolism.<; br>; Cilia – metabolism.<; br>; Membrane Proteins – metabolism<; br>; Mutation, Missense – physiology.<; br>; Proteins – genetics<; br>; Proteins – metabolism

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APA (6th Edition):

Masyukova, S. V. (2011). Analysis of NPHP complex genetic interactions associated with human cilia disorders. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1352

Chicago Manual of Style (16th Edition):

Masyukova, Svetlana V. “Analysis of NPHP complex genetic interactions associated with human cilia disorders.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 21, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1352.

MLA Handbook (7th Edition):

Masyukova, Svetlana V. “Analysis of NPHP complex genetic interactions associated with human cilia disorders.” 2011. Web. 21 Oct 2019.

Vancouver:

Masyukova SV. Analysis of NPHP complex genetic interactions associated with human cilia disorders. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Oct 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1352.

Council of Science Editors:

Masyukova SV. Analysis of NPHP complex genetic interactions associated with human cilia disorders. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1352

20. Silveira, Alexandra C. Characterization of SUDS3 as a BRMS1 family member in breast cancer.

Degree: PhD, 2008, University of Alabama – Birmingham

BRMS1 and SUDS3 belong to a protein family characterized by the Sds3-like domain. These proteins are core members of SIN3-HDAC chromatin remodeling complexes and thus,… (more)

Subjects/Keywords: Breast Neoplasms  – metabolism <; br>; Carrier Proteins  – metabolism <; br>; Chromatin Assembly and Disassembly <; br>; Histone Deacetylases  – metabolism <; br>; Neoplasm Proteins  – metabolism <; br>; Repressor Proteins/metabolism

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APA (6th Edition):

Silveira, A. C. (2008). Characterization of SUDS3 as a BRMS1 family member in breast cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,339

Chicago Manual of Style (16th Edition):

Silveira, Alexandra C. “Characterization of SUDS3 as a BRMS1 family member in breast cancer.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 21, 2019. http://contentdm.mhsl.uab.edu/u?/etd,339.

MLA Handbook (7th Edition):

Silveira, Alexandra C. “Characterization of SUDS3 as a BRMS1 family member in breast cancer.” 2008. Web. 21 Oct 2019.

Vancouver:

Silveira AC. Characterization of SUDS3 as a BRMS1 family member in breast cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,339.

Council of Science Editors:

Silveira AC. Characterization of SUDS3 as a BRMS1 family member in breast cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,339

21. Ding, Huiping. Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6.

Degree: PhD, 2008, University of Alabama – Birmingham

Alzheimer’s disease (AD), the most common neurodegenerative disease, is pathologically characterized by senile plaques composed of amyloid [beta] peptide and neurofibrillary tangles composed of hyperphosphorylated… (more)

Subjects/Keywords: Alzheimer Disease  – metabolism<; br>; Brain  – metabolism<; br>; Caspases  – metabolism<; br>; Histone Deacetylases  – metabolism<; br>; Microtubules  – metabolism<; br>; Neurons  – metabolism<; br>; tau Proteins  – metabolism

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APA (6th Edition):

Ding, H. (2008). Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,439

Chicago Manual of Style (16th Edition):

Ding, Huiping. “Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 21, 2019. http://contentdm.mhsl.uab.edu/u?/etd,439.

MLA Handbook (7th Edition):

Ding, Huiping. “Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6.” 2008. Web. 21 Oct 2019.

Vancouver:

Ding H. Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,439.

Council of Science Editors:

Ding H. Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,439

22. Yu, Jei-Hwa. MAPKs regulate nuclear import of human papillomavirus type 11 replicative helicase E1.

Degree: PhD, 2008, University of Alabama – Birmingham

Papillomaviruses (PV) are prevalent pathogens that infect human or animal squamous epithelia. Its genome is a double strand circular DNA of approximately 7.9 kb. It… (more)

Subjects/Keywords: DNA Helicases  – metabolism <; br>; DNA-Binding Proteins  – metabolism <; br>; Human papillomavirus 11  – physiology <; br>; Mitogen-Activated Protein Kinases  – metabolism <; br>; Nuclear Localization Signals  – metabolism <; br>; Replication Origin <; br>; Viral Proteins  – metabolism

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APA (6th Edition):

Yu, J. (2008). MAPKs regulate nuclear import of human papillomavirus type 11 replicative helicase E1. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,213

Chicago Manual of Style (16th Edition):

Yu, Jei-Hwa. “MAPKs regulate nuclear import of human papillomavirus type 11 replicative helicase E1.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 21, 2019. http://contentdm.mhsl.uab.edu/u?/etd,213.

MLA Handbook (7th Edition):

Yu, Jei-Hwa. “MAPKs regulate nuclear import of human papillomavirus type 11 replicative helicase E1.” 2008. Web. 21 Oct 2019.

Vancouver:

Yu J. MAPKs regulate nuclear import of human papillomavirus type 11 replicative helicase E1. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,213.

Council of Science Editors:

Yu J. MAPKs regulate nuclear import of human papillomavirus type 11 replicative helicase E1. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,213


Ryerson University

23. Tucholska, Monika. RAP, a member of the Ras superfamily of small GTPases and its putative GTPase activating proteins and guanine nucleotide exchange factors in raw 264.7 macrophages.

Degree: 2008, Ryerson University

 The Fcγ receptor is a cell surface protein essential in the immune response that binds IgG-opsonized particles resulting in phagocytosis. Phagocytosis is a process used… (more)

Subjects/Keywords: Ras proteins; Guanosine triphosphatase genes; GTPase-activating protein; Fc receptors; Cellular immunity; Phagocytosis

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APA (6th Edition):

Tucholska, M. (2008). RAP, a member of the Ras superfamily of small GTPases and its putative GTPase activating proteins and guanine nucleotide exchange factors in raw 264.7 macrophages. (Thesis). Ryerson University. Retrieved from https://digital.library.ryerson.ca/islandora/object/RULA%3A970

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tucholska, Monika. “RAP, a member of the Ras superfamily of small GTPases and its putative GTPase activating proteins and guanine nucleotide exchange factors in raw 264.7 macrophages.” 2008. Thesis, Ryerson University. Accessed October 21, 2019. https://digital.library.ryerson.ca/islandora/object/RULA%3A970.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tucholska, Monika. “RAP, a member of the Ras superfamily of small GTPases and its putative GTPase activating proteins and guanine nucleotide exchange factors in raw 264.7 macrophages.” 2008. Web. 21 Oct 2019.

Vancouver:

Tucholska M. RAP, a member of the Ras superfamily of small GTPases and its putative GTPase activating proteins and guanine nucleotide exchange factors in raw 264.7 macrophages. [Internet] [Thesis]. Ryerson University; 2008. [cited 2019 Oct 21]. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A970.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tucholska M. RAP, a member of the Ras superfamily of small GTPases and its putative GTPase activating proteins and guanine nucleotide exchange factors in raw 264.7 macrophages. [Thesis]. Ryerson University; 2008. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A970

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cambridge

24. Martynyuk, Nataly. Actions of alpha-chimaerins in mechanisms relevant to dendritic spine formation and neurodegeneration.

Degree: PhD, 2019, University of Cambridge

 Rho GTPases and their regulators such as guanosine exchange factors (GEFs) and GTPase activating proteins (GAPs) represent an important class of molecules controlling dendritic spine… (more)

Subjects/Keywords: Chimaerin; Alpha-chimaerin; Dendritic spines; Neurodegeneration; Neuroplasticity; RhoGTPases; Rac; Rho; GTPase activating proteins; GAP; RacGAP; Cdc42; Cytoskeleton; Endocytosis; HEK293; HeLa; Chimerin; CHIN; ROCK; EphA4; C1 domain

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APA (6th Edition):

Martynyuk, N. (2019). Actions of alpha-chimaerins in mechanisms relevant to dendritic spine formation and neurodegeneration. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/287938 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.763874

Chicago Manual of Style (16th Edition):

Martynyuk, Nataly. “Actions of alpha-chimaerins in mechanisms relevant to dendritic spine formation and neurodegeneration.” 2019. Doctoral Dissertation, University of Cambridge. Accessed October 21, 2019. https://www.repository.cam.ac.uk/handle/1810/287938 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.763874.

MLA Handbook (7th Edition):

Martynyuk, Nataly. “Actions of alpha-chimaerins in mechanisms relevant to dendritic spine formation and neurodegeneration.” 2019. Web. 21 Oct 2019.

Vancouver:

Martynyuk N. Actions of alpha-chimaerins in mechanisms relevant to dendritic spine formation and neurodegeneration. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2019 Oct 21]. Available from: https://www.repository.cam.ac.uk/handle/1810/287938 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.763874.

Council of Science Editors:

Martynyuk N. Actions of alpha-chimaerins in mechanisms relevant to dendritic spine formation and neurodegeneration. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/287938 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.763874

25. Harms, Paul William. Modulation of cell signaling by Tomoregulins in embryogenesis and cancer.

Degree: PhD, 2006, University of Alabama – Birmingham

Growth factor signals often regulate similar cellular processes both during embryogenesis and in adult homeostasis. Stringent control of these signals ensures proper embryonic development and… (more)

Subjects/Keywords: Homeodomain Proteins <; br>; Membrane Proteins  – metabolism <; br>; Neoplasm Proteins  – metabolism <; br>; Proteins <; br>; Signal Transduction  – physiology <; br>; Transcription Factors <; br>; Xenopus Proteins

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APA (6th Edition):

Harms, P. W. (2006). Modulation of cell signaling by Tomoregulins in embryogenesis and cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,369

Chicago Manual of Style (16th Edition):

Harms, Paul William. “Modulation of cell signaling by Tomoregulins in embryogenesis and cancer.” 2006. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 21, 2019. http://contentdm.mhsl.uab.edu/u?/etd,369.

MLA Handbook (7th Edition):

Harms, Paul William. “Modulation of cell signaling by Tomoregulins in embryogenesis and cancer.” 2006. Web. 21 Oct 2019.

Vancouver:

Harms PW. Modulation of cell signaling by Tomoregulins in embryogenesis and cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2006. [cited 2019 Oct 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,369.

Council of Science Editors:

Harms PW. Modulation of cell signaling by Tomoregulins in embryogenesis and cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2006. Available from: http://contentdm.mhsl.uab.edu/u?/etd,369

26. Parish, Lindsay A. Protein trafficking and 4.1R relocalization in Plasmodium falciparum-infected erythrocytes.

Degree: PhD, 2009, University of Alabama – Birmingham

Malaria is a mosquito-borne infectious disease that is caused by parasites in the genus Plasmodium. There are four species of malaria that routinely infect humans,… (more)

Subjects/Keywords: Erythrocytes  – metabolism<; br>; Plasmodium falciparum  – metabolism<; br>; Protozoan Proteins  – genetics<; br>; Qa-SNARE Proteins  – genetics<; br>; Secretory Pathway  – genetics

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APA (6th Edition):

Parish, L. A. (2009). Protein trafficking and 4.1R relocalization in Plasmodium falciparum-infected erythrocytes. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,503

Chicago Manual of Style (16th Edition):

Parish, Lindsay A. “Protein trafficking and 4.1R relocalization in Plasmodium falciparum-infected erythrocytes.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 21, 2019. http://contentdm.mhsl.uab.edu/u?/etd,503.

MLA Handbook (7th Edition):

Parish, Lindsay A. “Protein trafficking and 4.1R relocalization in Plasmodium falciparum-infected erythrocytes.” 2009. Web. 21 Oct 2019.

Vancouver:

Parish LA. Protein trafficking and 4.1R relocalization in Plasmodium falciparum-infected erythrocytes. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,503.

Council of Science Editors:

Parish LA. Protein trafficking and 4.1R relocalization in Plasmodium falciparum-infected erythrocytes. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,503

27. Qadri, Yawar J. Small molecule inhibitors of acid sensing ion channel-1.

Degree: PhD, 2009, University of Alabama – Birmingham

Acid Sensing Ion Channel 1 is one of the many proteins in the Epithelial Sodium Channel/Degenerin family. The proteins in this family interact to form… (more)

Subjects/Keywords: Amiloride  – pharmacology<; br>; Nerve Tissue Proteins  – chemistry<; br>; Nerve Tissue Proteins  – metabolism<; br>; Sodium Channels  – chemistry<; br>; Spider Venoms  – metabolism

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APA (6th Edition):

Qadri, Y. J. (2009). Small molecule inhibitors of acid sensing ion channel-1. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1174

Chicago Manual of Style (16th Edition):

Qadri, Yawar J. “Small molecule inhibitors of acid sensing ion channel-1.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 21, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1174.

MLA Handbook (7th Edition):

Qadri, Yawar J. “Small molecule inhibitors of acid sensing ion channel-1.” 2009. Web. 21 Oct 2019.

Vancouver:

Qadri YJ. Small molecule inhibitors of acid sensing ion channel-1. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1174.

Council of Science Editors:

Qadri YJ. Small molecule inhibitors of acid sensing ion channel-1. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1174

28. Parks, Brian W. Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A.

Degree: PhD, 2008, University of Alabama – Birmingham

The G protein-coupled receptor, G2A, is expressed by multiple cell-types involved in atherosclerosis and is activated by structurally related lysophospholipids generated during low-density lipoprotein (LDL)… (more)

Subjects/Keywords: Apolipoproteins E  – metabolism<; br>; Arteriosclerosis<; br>; Bone Marrow Cells  – metabolism<; br>; Cell Cycle Proteins<; br>; Hypercholesterolemia  – metabolism<; br>; Lysophosphatidylcholines  – metabolism<; br>; Macrophages  – physiology<; br>; Receptors, G-Protein-Coupled<; br>; Receptors, LDL

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APA (6th Edition):

Parks, B. W. (2008). Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,768

Chicago Manual of Style (16th Edition):

Parks, Brian W. “Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 21, 2019. http://contentdm.mhsl.uab.edu/u?/etd,768.

MLA Handbook (7th Edition):

Parks, Brian W. “Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A.” 2008. Web. 21 Oct 2019.

Vancouver:

Parks BW. Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,768.

Council of Science Editors:

Parks BW. Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,768

29. Davenport, James Robert. The role of the primary cilium in energy and glucose metabolism.

Degree: PhD, 2007, University of Alabama – Birmingham

Virtually ignored for years as a useless organelle, the primary cilium has emerged as an essential signaling center in both development and maintenance of tissues… (more)

Subjects/Keywords: Cilia  – metabolism<; br>; Flagella  – metabolism<; br>; Kidney  – metabolism<; br>; Kidney Diseases, Cystic  – metabolism<; br>; Obesity  – metabolism<; br>; Pancreas  – abnormalities<; br>; Pancreas  – pathology<; br>; Tumor Suppressor Proteins

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Davenport, J. R. (2007). The role of the primary cilium in energy and glucose metabolism. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,516

Chicago Manual of Style (16th Edition):

Davenport, James Robert. “The role of the primary cilium in energy and glucose metabolism.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 21, 2019. http://contentdm.mhsl.uab.edu/u?/etd,516.

MLA Handbook (7th Edition):

Davenport, James Robert. “The role of the primary cilium in energy and glucose metabolism.” 2007. Web. 21 Oct 2019.

Vancouver:

Davenport JR. The role of the primary cilium in energy and glucose metabolism. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Oct 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,516.

Council of Science Editors:

Davenport JR. The role of the primary cilium in energy and glucose metabolism. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,516

30. Steg, Adam. Analysis of the hedgehog pathway in pancreatic adenocarcinoma.

Degree: PhD, 2008, University of Alabama – Birmingham

Pancreatic adenocarcinoma (PAC) is the fourth leading cause of cancer mortality in the United States. Despite the use of highly aggressive treatment regimens (surgery, chemotherapy… (more)

Subjects/Keywords: Adenocarcinoma  – metabolism <; br>; Adenocarcinoma  – pathology <; br>; Hedgehog Proteins  – metabolism <; br>; Pancreatic Neoplasms  – metabolism <; br>; Pancreatic Neoplasms  – pathology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Steg, A. (2008). Analysis of the hedgehog pathway in pancreatic adenocarcinoma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,290

Chicago Manual of Style (16th Edition):

Steg, Adam. “Analysis of the hedgehog pathway in pancreatic adenocarcinoma.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 21, 2019. http://contentdm.mhsl.uab.edu/u?/etd,290.

MLA Handbook (7th Edition):

Steg, Adam. “Analysis of the hedgehog pathway in pancreatic adenocarcinoma.” 2008. Web. 21 Oct 2019.

Vancouver:

Steg A. Analysis of the hedgehog pathway in pancreatic adenocarcinoma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,290.

Council of Science Editors:

Steg A. Analysis of the hedgehog pathway in pancreatic adenocarcinoma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,290

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