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You searched for subject:( Filamin A). Showing records 1 – 18 of 18 total matches.

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University of Otago

1. van Kogelenberg, Margriet. Studies into the Underlying Aetiology of Periventricular Heterotopia .

Degree: 2010, University of Otago

 Understanding the genetic and molecular components involved in cortical development will enhance the understanding of the pathogenesis of malformations affecting the brain. This project encompasses… (more)

Subjects/Keywords: Periventricular heterotopia; Filamin A

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APA (6th Edition):

van Kogelenberg, M. (2010). Studies into the Underlying Aetiology of Periventricular Heterotopia . (Doctoral Dissertation). University of Otago. Retrieved from http://hdl.handle.net/10523/455

Chicago Manual of Style (16th Edition):

van Kogelenberg, Margriet. “Studies into the Underlying Aetiology of Periventricular Heterotopia .” 2010. Doctoral Dissertation, University of Otago. Accessed July 07, 2020. http://hdl.handle.net/10523/455.

MLA Handbook (7th Edition):

van Kogelenberg, Margriet. “Studies into the Underlying Aetiology of Periventricular Heterotopia .” 2010. Web. 07 Jul 2020.

Vancouver:

van Kogelenberg M. Studies into the Underlying Aetiology of Periventricular Heterotopia . [Internet] [Doctoral dissertation]. University of Otago; 2010. [cited 2020 Jul 07]. Available from: http://hdl.handle.net/10523/455.

Council of Science Editors:

van Kogelenberg M. Studies into the Underlying Aetiology of Periventricular Heterotopia . [Doctoral Dissertation]. University of Otago; 2010. Available from: http://hdl.handle.net/10523/455


University of Manchester

2. Lopez Camacho, Cesar. A new role for Filamin A as a regulator of Runx2 function.

Degree: 2011, University of Manchester

 AbstractThe University of Manchester, Cesar Lopez Camacho PhD in Molecular BiologyThesis title: A new role for Filamin A as a regulator of Runx2 functionJanuary 2011Filamin… (more)

Subjects/Keywords: Filamin A; Runx2; osteoblastic differentiation; nucleolus

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APA (6th Edition):

Lopez Camacho, C. (2011). A new role for Filamin A as a regulator of Runx2 function. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:120116

Chicago Manual of Style (16th Edition):

Lopez Camacho, Cesar. “A new role for Filamin A as a regulator of Runx2 function.” 2011. Doctoral Dissertation, University of Manchester. Accessed July 07, 2020. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:120116.

MLA Handbook (7th Edition):

Lopez Camacho, Cesar. “A new role for Filamin A as a regulator of Runx2 function.” 2011. Web. 07 Jul 2020.

Vancouver:

Lopez Camacho C. A new role for Filamin A as a regulator of Runx2 function. [Internet] [Doctoral dissertation]. University of Manchester; 2011. [cited 2020 Jul 07]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:120116.

Council of Science Editors:

Lopez Camacho C. A new role for Filamin A as a regulator of Runx2 function. [Doctoral Dissertation]. University of Manchester; 2011. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:120116


University of Manchester

3. Lopez Camacho, Cesar. A new role for Filamin A as a regulator of Runx2 function.

Degree: PhD, 2011, University of Manchester

Filamin A is a well-characterised cytoskeletal protein which regulates cell shape and migration by cross-linking with actin. Filamin A mutations cause a number of human… (more)

Subjects/Keywords: 612; Filamin A, Runx2, osteoblastic differentiation, nucleolus

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APA (6th Edition):

Lopez Camacho, C. (2011). A new role for Filamin A as a regulator of Runx2 function. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/a-new-role-for-filamin-a-as-a-regulator-of-runx2-function(88321064-5c82-4f2b-a755-911ed3b42b2e).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.538413

Chicago Manual of Style (16th Edition):

Lopez Camacho, Cesar. “A new role for Filamin A as a regulator of Runx2 function.” 2011. Doctoral Dissertation, University of Manchester. Accessed July 07, 2020. https://www.research.manchester.ac.uk/portal/en/theses/a-new-role-for-filamin-a-as-a-regulator-of-runx2-function(88321064-5c82-4f2b-a755-911ed3b42b2e).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.538413.

MLA Handbook (7th Edition):

Lopez Camacho, Cesar. “A new role for Filamin A as a regulator of Runx2 function.” 2011. Web. 07 Jul 2020.

Vancouver:

Lopez Camacho C. A new role for Filamin A as a regulator of Runx2 function. [Internet] [Doctoral dissertation]. University of Manchester; 2011. [cited 2020 Jul 07]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/a-new-role-for-filamin-a-as-a-regulator-of-runx2-function(88321064-5c82-4f2b-a755-911ed3b42b2e).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.538413.

Council of Science Editors:

Lopez Camacho C. A new role for Filamin A as a regulator of Runx2 function. [Doctoral Dissertation]. University of Manchester; 2011. Available from: https://www.research.manchester.ac.uk/portal/en/theses/a-new-role-for-filamin-a-as-a-regulator-of-runx2-function(88321064-5c82-4f2b-a755-911ed3b42b2e).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.538413

4. Bandaru, Sashidhar. Filamin A in Cardiovascular Remodeling.

Degree: 2018, University of Gothenburg / Göteborgs Universitet

Filamin A (FLNA) is a large actin-binding cytoskeletal protein that stabilizes actin networks and integrates them with cell membranes. FLNA is therefore important for cell… (more)

Subjects/Keywords: Filamin A; Cardiovascular remodelling

…Deficiency of filamin A in endothelial cells impairs left ventricular remodeling after myocardial… …Akyürek LM. Cardiovascular Research 2014, 105:151-9 Study II Deficiency of filamin A impairs… …2018 Additional publications not included in this thesis 1. Targeting the protein filamin A… …e7. 6. Blocking the cleavage of filamin A by calpain inhibitor decreases tumor cell growth… …13 Filamin A… 

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APA (6th Edition):

Bandaru, S. (2018). Filamin A in Cardiovascular Remodeling. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/56922

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bandaru, Sashidhar. “Filamin A in Cardiovascular Remodeling.” 2018. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed July 07, 2020. http://hdl.handle.net/2077/56922.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bandaru, Sashidhar. “Filamin A in Cardiovascular Remodeling.” 2018. Web. 07 Jul 2020.

Vancouver:

Bandaru S. Filamin A in Cardiovascular Remodeling. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2018. [cited 2020 Jul 07]. Available from: http://hdl.handle.net/2077/56922.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bandaru S. Filamin A in Cardiovascular Remodeling. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2018. Available from: http://hdl.handle.net/2077/56922

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

5. Mohammadi, Hamid. Complex Mechanics of Constrained Connective Tissue Matrices and their Impact on Cellular Mechanosensation.

Degree: PhD, 2014, University of Toronto

 Physical interactions of cells with matrix microenvironments are critical steps in many cellular processes such as mechanosensation and metastatic invasion. Mechanosensory processes are strongly influenced… (more)

Subjects/Keywords: Collagen; Filamin A; Integrin; Mechanobiology; Nonlinearity; Remodeling; 0786

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APA (6th Edition):

Mohammadi, H. (2014). Complex Mechanics of Constrained Connective Tissue Matrices and their Impact on Cellular Mechanosensation. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/73795

Chicago Manual of Style (16th Edition):

Mohammadi, Hamid. “Complex Mechanics of Constrained Connective Tissue Matrices and their Impact on Cellular Mechanosensation.” 2014. Doctoral Dissertation, University of Toronto. Accessed July 07, 2020. http://hdl.handle.net/1807/73795.

MLA Handbook (7th Edition):

Mohammadi, Hamid. “Complex Mechanics of Constrained Connective Tissue Matrices and their Impact on Cellular Mechanosensation.” 2014. Web. 07 Jul 2020.

Vancouver:

Mohammadi H. Complex Mechanics of Constrained Connective Tissue Matrices and their Impact on Cellular Mechanosensation. [Internet] [Doctoral dissertation]. University of Toronto; 2014. [cited 2020 Jul 07]. Available from: http://hdl.handle.net/1807/73795.

Council of Science Editors:

Mohammadi H. Complex Mechanics of Constrained Connective Tissue Matrices and their Impact on Cellular Mechanosensation. [Doctoral Dissertation]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/73795

6. Carabalona, Aurélie. Origine et physiopathologie d' une malformation du cortex cérébral : L' hétérotopie nodulaire périventriculaire liée à des mutations du gène Filamine A. : Origin and physiopathology of cortical malformation : periventricular nodular heterotopia due to mutations in FLNA gene.

Degree: Docteur es, Neurosciences, 2012, Aix Marseille Université

Les hétérotopies nodulaires périventriculaires (HNP) correspondent aux malformations cérébrales les plus fréquemment découvertes à l'âge adulte. Survenant au cours de la migration, elles consistent en… (more)

Subjects/Keywords: Migration neuronale; Glie radiaire; Adhésion; Filamine A; Neuronal migration; Radial glia; Adhesion; Filamin A

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APA (6th Edition):

Carabalona, A. (2012). Origine et physiopathologie d' une malformation du cortex cérébral : L' hétérotopie nodulaire périventriculaire liée à des mutations du gène Filamine A. : Origin and physiopathology of cortical malformation : periventricular nodular heterotopia due to mutations in FLNA gene. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2012AIXM4052

Chicago Manual of Style (16th Edition):

Carabalona, Aurélie. “Origine et physiopathologie d' une malformation du cortex cérébral : L' hétérotopie nodulaire périventriculaire liée à des mutations du gène Filamine A. : Origin and physiopathology of cortical malformation : periventricular nodular heterotopia due to mutations in FLNA gene.” 2012. Doctoral Dissertation, Aix Marseille Université. Accessed July 07, 2020. http://www.theses.fr/2012AIXM4052.

MLA Handbook (7th Edition):

Carabalona, Aurélie. “Origine et physiopathologie d' une malformation du cortex cérébral : L' hétérotopie nodulaire périventriculaire liée à des mutations du gène Filamine A. : Origin and physiopathology of cortical malformation : periventricular nodular heterotopia due to mutations in FLNA gene.” 2012. Web. 07 Jul 2020.

Vancouver:

Carabalona A. Origine et physiopathologie d' une malformation du cortex cérébral : L' hétérotopie nodulaire périventriculaire liée à des mutations du gène Filamine A. : Origin and physiopathology of cortical malformation : periventricular nodular heterotopia due to mutations in FLNA gene. [Internet] [Doctoral dissertation]. Aix Marseille Université 2012. [cited 2020 Jul 07]. Available from: http://www.theses.fr/2012AIXM4052.

Council of Science Editors:

Carabalona A. Origine et physiopathologie d' une malformation du cortex cérébral : L' hétérotopie nodulaire périventriculaire liée à des mutations du gène Filamine A. : Origin and physiopathology of cortical malformation : periventricular nodular heterotopia due to mutations in FLNA gene. [Doctoral Dissertation]. Aix Marseille Université 2012. Available from: http://www.theses.fr/2012AIXM4052

7. Mutel, Alexandre. La Filamine A, acteur central relayant les mécanismes d'invasion des gliomes de haut-grades : implication dans la signalisation et le trafic du récepteur chimiotactique de l'urotensine II : Filamin A, a key regulatory actor of high-grade glioma invasion mechanisms : involvement in urotensin II chemokine receptor signalization and trafficking.

Degree: Docteur es, Aspects moleculaires et cellulaires de la biologie, 2019, Normandie

Les glioblastomes multiformes (GBM) sont les tumeurs les plus fréquentes et agressives du système nerveux central. Le traitement standard des GBM suit le « Protocole… (more)

Subjects/Keywords: Filamine A; Urotensine II; RCPG; Glioblastome; Filamin A; Urotensin II; GPCR; Glioblastoma; 616.994

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APA (6th Edition):

Mutel, A. (2019). La Filamine A, acteur central relayant les mécanismes d'invasion des gliomes de haut-grades : implication dans la signalisation et le trafic du récepteur chimiotactique de l'urotensine II : Filamin A, a key regulatory actor of high-grade glioma invasion mechanisms : involvement in urotensin II chemokine receptor signalization and trafficking. (Doctoral Dissertation). Normandie. Retrieved from http://www.theses.fr/2019NORMR081

Chicago Manual of Style (16th Edition):

Mutel, Alexandre. “La Filamine A, acteur central relayant les mécanismes d'invasion des gliomes de haut-grades : implication dans la signalisation et le trafic du récepteur chimiotactique de l'urotensine II : Filamin A, a key regulatory actor of high-grade glioma invasion mechanisms : involvement in urotensin II chemokine receptor signalization and trafficking.” 2019. Doctoral Dissertation, Normandie. Accessed July 07, 2020. http://www.theses.fr/2019NORMR081.

MLA Handbook (7th Edition):

Mutel, Alexandre. “La Filamine A, acteur central relayant les mécanismes d'invasion des gliomes de haut-grades : implication dans la signalisation et le trafic du récepteur chimiotactique de l'urotensine II : Filamin A, a key regulatory actor of high-grade glioma invasion mechanisms : involvement in urotensin II chemokine receptor signalization and trafficking.” 2019. Web. 07 Jul 2020.

Vancouver:

Mutel A. La Filamine A, acteur central relayant les mécanismes d'invasion des gliomes de haut-grades : implication dans la signalisation et le trafic du récepteur chimiotactique de l'urotensine II : Filamin A, a key regulatory actor of high-grade glioma invasion mechanisms : involvement in urotensin II chemokine receptor signalization and trafficking. [Internet] [Doctoral dissertation]. Normandie; 2019. [cited 2020 Jul 07]. Available from: http://www.theses.fr/2019NORMR081.

Council of Science Editors:

Mutel A. La Filamine A, acteur central relayant les mécanismes d'invasion des gliomes de haut-grades : implication dans la signalisation et le trafic du récepteur chimiotactique de l'urotensine II : Filamin A, a key regulatory actor of high-grade glioma invasion mechanisms : involvement in urotensin II chemokine receptor signalization and trafficking. [Doctoral Dissertation]. Normandie; 2019. Available from: http://www.theses.fr/2019NORMR081


UCLA

8. CHANTARAVISOOT, NAPHAT. Characterizing the Mechanistic Target of Rapamycin Complex 2 Pathway in Glioblastoma Multiforme.

Degree: Microbiology, Immunology, & Molecular Genetics, 2015, UCLA

 ABSTRACT OF THE DISSERTATIONCharacterizing the Mechanistic Target of Rapamycin Complex 2 Pathway in Glioblastoma MultiformebyNaphat Chantaravisoot Doctor of Philosophy in Microbiology, Immunology, and Molecular GeneticsUniversity… (more)

Subjects/Keywords: Molecular biology; Biochemistry; Cellular biology; cancer; cell migration and invasion; Filamin A; Glioblastoma multiforme; mTORC2

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APA (6th Edition):

CHANTARAVISOOT, N. (2015). Characterizing the Mechanistic Target of Rapamycin Complex 2 Pathway in Glioblastoma Multiforme. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/94z7n746

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

CHANTARAVISOOT, NAPHAT. “Characterizing the Mechanistic Target of Rapamycin Complex 2 Pathway in Glioblastoma Multiforme.” 2015. Thesis, UCLA. Accessed July 07, 2020. http://www.escholarship.org/uc/item/94z7n746.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

CHANTARAVISOOT, NAPHAT. “Characterizing the Mechanistic Target of Rapamycin Complex 2 Pathway in Glioblastoma Multiforme.” 2015. Web. 07 Jul 2020.

Vancouver:

CHANTARAVISOOT N. Characterizing the Mechanistic Target of Rapamycin Complex 2 Pathway in Glioblastoma Multiforme. [Internet] [Thesis]. UCLA; 2015. [cited 2020 Jul 07]. Available from: http://www.escholarship.org/uc/item/94z7n746.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

CHANTARAVISOOT N. Characterizing the Mechanistic Target of Rapamycin Complex 2 Pathway in Glioblastoma Multiforme. [Thesis]. UCLA; 2015. Available from: http://www.escholarship.org/uc/item/94z7n746

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Missouri – Columbia

9. Yu, Ningpu, 1979-. The role of P2Y₂ nucleotide receptors in vascular inflammation.

Degree: PhD, 2007, University of Missouri – Columbia

 Extracellular nucleotides act on P2 receptors to regulate vascular tone. Large amounts of extracellular nucleotides are released at the sites of tissue injury and may… (more)

Subjects/Keywords: filamin A; Vasculitis; Chemotaxis; Endothelins; Phosphorylation

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APA (6th Edition):

Yu, Ningpu, 1. (2007). The role of P2Y₂ nucleotide receptors in vascular inflammation. (Doctoral Dissertation). University of Missouri – Columbia. Retrieved from https://doi.org/10.32469/10355/4664

Chicago Manual of Style (16th Edition):

Yu, Ningpu, 1979-. “The role of P2Y₂ nucleotide receptors in vascular inflammation.” 2007. Doctoral Dissertation, University of Missouri – Columbia. Accessed July 07, 2020. https://doi.org/10.32469/10355/4664.

MLA Handbook (7th Edition):

Yu, Ningpu, 1979-. “The role of P2Y₂ nucleotide receptors in vascular inflammation.” 2007. Web. 07 Jul 2020.

Vancouver:

Yu, Ningpu 1. The role of P2Y₂ nucleotide receptors in vascular inflammation. [Internet] [Doctoral dissertation]. University of Missouri – Columbia; 2007. [cited 2020 Jul 07]. Available from: https://doi.org/10.32469/10355/4664.

Council of Science Editors:

Yu, Ningpu 1. The role of P2Y₂ nucleotide receptors in vascular inflammation. [Doctoral Dissertation]. University of Missouri – Columbia; 2007. Available from: https://doi.org/10.32469/10355/4664


University of Toledo

10. Rider, Leah Catherine. Prolactin-Dependent Regulation of the Actin Cytoskeleton by JAK2, SH2B1ß, PAK1 and Filamin A.

Degree: PhD, Biology (Cell-Molecular Biology), 2011, University of Toledo

 JAK2 is a receptor-associated tyrosine kinase responsible for signaling involving most of the cytokine family of receptors, including the prolactin (PRL) receptor (Murata, Noguchi et… (more)

Subjects/Keywords: Cellular Biology; prolactin; actin; SH2B1&946; Filamin A; JAK2; PAK1; breast cancer; ruffling; invasion; migration

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APA (6th Edition):

Rider, L. C. (2011). Prolactin-Dependent Regulation of the Actin Cytoskeleton by JAK2, SH2B1ß, PAK1 and Filamin A. (Doctoral Dissertation). University of Toledo. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=toledo1310079439

Chicago Manual of Style (16th Edition):

Rider, Leah Catherine. “Prolactin-Dependent Regulation of the Actin Cytoskeleton by JAK2, SH2B1ß, PAK1 and Filamin A.” 2011. Doctoral Dissertation, University of Toledo. Accessed July 07, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=toledo1310079439.

MLA Handbook (7th Edition):

Rider, Leah Catherine. “Prolactin-Dependent Regulation of the Actin Cytoskeleton by JAK2, SH2B1ß, PAK1 and Filamin A.” 2011. Web. 07 Jul 2020.

Vancouver:

Rider LC. Prolactin-Dependent Regulation of the Actin Cytoskeleton by JAK2, SH2B1ß, PAK1 and Filamin A. [Internet] [Doctoral dissertation]. University of Toledo; 2011. [cited 2020 Jul 07]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=toledo1310079439.

Council of Science Editors:

Rider LC. Prolactin-Dependent Regulation of the Actin Cytoskeleton by JAK2, SH2B1ß, PAK1 and Filamin A. [Doctoral Dissertation]. University of Toledo; 2011. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=toledo1310079439


Massey University

11. Anandasayanan, Manickavasagar. A structural and functional investigation into the filamin A G2593E mutation : implications for neurological disease : a thesis presented in partial fulfillment of a Master of Philosophy (Science) (Biochemistry) at the Institute of Molecular BioSciences, Massey University, Palmerston North .

Degree: 2012, Massey University

 he dimeric F-actin cross-linking protein human filamin a (hsFLNa) is an actin binding protein, which modulates the properties of the actin cytoskeleton and plays a… (more)

Subjects/Keywords: Filamin A; Actin-binding protein; Protein mutation; Periventricular Nodular Heterotopia (PVNH); Mutant protein

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APA (6th Edition):

Anandasayanan, M. (2012). A structural and functional investigation into the filamin A G2593E mutation : implications for neurological disease : a thesis presented in partial fulfillment of a Master of Philosophy (Science) (Biochemistry) at the Institute of Molecular BioSciences, Massey University, Palmerston North . (Thesis). Massey University. Retrieved from http://hdl.handle.net/10179/4023

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Anandasayanan, Manickavasagar. “A structural and functional investigation into the filamin A G2593E mutation : implications for neurological disease : a thesis presented in partial fulfillment of a Master of Philosophy (Science) (Biochemistry) at the Institute of Molecular BioSciences, Massey University, Palmerston North .” 2012. Thesis, Massey University. Accessed July 07, 2020. http://hdl.handle.net/10179/4023.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Anandasayanan, Manickavasagar. “A structural and functional investigation into the filamin A G2593E mutation : implications for neurological disease : a thesis presented in partial fulfillment of a Master of Philosophy (Science) (Biochemistry) at the Institute of Molecular BioSciences, Massey University, Palmerston North .” 2012. Web. 07 Jul 2020.

Vancouver:

Anandasayanan M. A structural and functional investigation into the filamin A G2593E mutation : implications for neurological disease : a thesis presented in partial fulfillment of a Master of Philosophy (Science) (Biochemistry) at the Institute of Molecular BioSciences, Massey University, Palmerston North . [Internet] [Thesis]. Massey University; 2012. [cited 2020 Jul 07]. Available from: http://hdl.handle.net/10179/4023.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Anandasayanan M. A structural and functional investigation into the filamin A G2593E mutation : implications for neurological disease : a thesis presented in partial fulfillment of a Master of Philosophy (Science) (Biochemistry) at the Institute of Molecular BioSciences, Massey University, Palmerston North . [Thesis]. Massey University; 2012. Available from: http://hdl.handle.net/10179/4023

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Ottawa

12. Szeto, Sandy. Phosphorylation of Filamin A by Cdk1/cyclin B1 Regulates Filamin A Subcellular Localization and is Important for Daughter Cell Separation .

Degree: 2014, University of Ottawa

 In cell culture, entry into mitosis of many adherent mammalian cells is accompanied by substantial changes in cellular architecture. Flat, spread-out interphase cells detach from… (more)

Subjects/Keywords: Actin cytoskeleton remodeling; Mitotic cell rounding; Filamin A; Cyclin-dependent kinase 1; Mitosis; Phosphorylation

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APA (6th Edition):

Szeto, S. (2014). Phosphorylation of Filamin A by Cdk1/cyclin B1 Regulates Filamin A Subcellular Localization and is Important for Daughter Cell Separation . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/31732

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Szeto, Sandy. “Phosphorylation of Filamin A by Cdk1/cyclin B1 Regulates Filamin A Subcellular Localization and is Important for Daughter Cell Separation .” 2014. Thesis, University of Ottawa. Accessed July 07, 2020. http://hdl.handle.net/10393/31732.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Szeto, Sandy. “Phosphorylation of Filamin A by Cdk1/cyclin B1 Regulates Filamin A Subcellular Localization and is Important for Daughter Cell Separation .” 2014. Web. 07 Jul 2020.

Vancouver:

Szeto S. Phosphorylation of Filamin A by Cdk1/cyclin B1 Regulates Filamin A Subcellular Localization and is Important for Daughter Cell Separation . [Internet] [Thesis]. University of Ottawa; 2014. [cited 2020 Jul 07]. Available from: http://hdl.handle.net/10393/31732.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Szeto S. Phosphorylation of Filamin A by Cdk1/cyclin B1 Regulates Filamin A Subcellular Localization and is Important for Daughter Cell Separation . [Thesis]. University of Ottawa; 2014. Available from: http://hdl.handle.net/10393/31732

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Massey University

13. Waite, Ben. Expression and purification of CFM2 and filamin A repeat 10 domain.

Degree: MS, Biochemistry, 2012, Massey University

 Filamins are a group of proteins that interact with over 60 other proteins. Mutations to the Filamin A gene results in a spectrum of disorders… (more)

Subjects/Keywords: Filamins; Protein purification; Genetic mutation; Otoplatodigital spectrum disorder; Otopalatodigital spectrum disorder; Frontometaphyseal dysplasia; Melnick-Needles syndrome; Filamin A

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APA (6th Edition):

Waite, B. (2012). Expression and purification of CFM2 and filamin A repeat 10 domain. (Masters Thesis). Massey University. Retrieved from http://hdl.handle.net/10179/4325

Chicago Manual of Style (16th Edition):

Waite, Ben. “Expression and purification of CFM2 and filamin A repeat 10 domain.” 2012. Masters Thesis, Massey University. Accessed July 07, 2020. http://hdl.handle.net/10179/4325.

MLA Handbook (7th Edition):

Waite, Ben. “Expression and purification of CFM2 and filamin A repeat 10 domain.” 2012. Web. 07 Jul 2020.

Vancouver:

Waite B. Expression and purification of CFM2 and filamin A repeat 10 domain. [Internet] [Masters thesis]. Massey University; 2012. [cited 2020 Jul 07]. Available from: http://hdl.handle.net/10179/4325.

Council of Science Editors:

Waite B. Expression and purification of CFM2 and filamin A repeat 10 domain. [Masters Thesis]. Massey University; 2012. Available from: http://hdl.handle.net/10179/4325


Massey University

14. Clark, Alice Rosemary. The filamin A actin binding domain structure and function: implications for a gain-of-function mechanism for the otopalatodigital syndrome: a thesis presented in partial fulfillment of the requirements for the degree of Doctor of Philosophy in Biochemistry at Massey University, Palmerston North, New Zealand .

Degree: 2010, Massey University

 The filamin family act as scaffolding proteins associating with actin filmanents, acting through a highly conserved actin binding domain (ABD). The ABD of the filamins… (more)

Subjects/Keywords: Filamin A gene; Actin binding domain; Microfilament proteins; Otopalatodigital syndrome spectrum disorders; Periventricular nodular heterotopia; Biochemistry

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APA (6th Edition):

Clark, A. R. (2010). The filamin A actin binding domain structure and function: implications for a gain-of-function mechanism for the otopalatodigital syndrome: a thesis presented in partial fulfillment of the requirements for the degree of Doctor of Philosophy in Biochemistry at Massey University, Palmerston North, New Zealand . (Thesis). Massey University. Retrieved from http://hdl.handle.net/10179/1185

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Clark, Alice Rosemary. “The filamin A actin binding domain structure and function: implications for a gain-of-function mechanism for the otopalatodigital syndrome: a thesis presented in partial fulfillment of the requirements for the degree of Doctor of Philosophy in Biochemistry at Massey University, Palmerston North, New Zealand .” 2010. Thesis, Massey University. Accessed July 07, 2020. http://hdl.handle.net/10179/1185.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Clark, Alice Rosemary. “The filamin A actin binding domain structure and function: implications for a gain-of-function mechanism for the otopalatodigital syndrome: a thesis presented in partial fulfillment of the requirements for the degree of Doctor of Philosophy in Biochemistry at Massey University, Palmerston North, New Zealand .” 2010. Web. 07 Jul 2020.

Vancouver:

Clark AR. The filamin A actin binding domain structure and function: implications for a gain-of-function mechanism for the otopalatodigital syndrome: a thesis presented in partial fulfillment of the requirements for the degree of Doctor of Philosophy in Biochemistry at Massey University, Palmerston North, New Zealand . [Internet] [Thesis]. Massey University; 2010. [cited 2020 Jul 07]. Available from: http://hdl.handle.net/10179/1185.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Clark AR. The filamin A actin binding domain structure and function: implications for a gain-of-function mechanism for the otopalatodigital syndrome: a thesis presented in partial fulfillment of the requirements for the degree of Doctor of Philosophy in Biochemistry at Massey University, Palmerston North, New Zealand . [Thesis]. Massey University; 2010. Available from: http://hdl.handle.net/10179/1185

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of South Florida

15. Velkova, Aneliya. The Role of BRCA1 Domains and Motifs in Tumor Suppression.

Degree: 2011, University of South Florida

 ABSTRACT Individuals that carry deleterious mutations in the breast and ovarian cancer susceptibility gene 1 (BRCA1) have much more elevated risk to develop breast and/or… (more)

Subjects/Keywords: Breast cancer; DNA damage response; DNA-PKcs; Filamin A; Varints of Uncertain Signifficance; American Studies; Arts and Humanities; Molecular Biology

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APA (6th Edition):

Velkova, A. (2011). The Role of BRCA1 Domains and Motifs in Tumor Suppression. (Thesis). University of South Florida. Retrieved from https://scholarcommons.usf.edu/etd/3391

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Velkova, Aneliya. “The Role of BRCA1 Domains and Motifs in Tumor Suppression.” 2011. Thesis, University of South Florida. Accessed July 07, 2020. https://scholarcommons.usf.edu/etd/3391.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Velkova, Aneliya. “The Role of BRCA1 Domains and Motifs in Tumor Suppression.” 2011. Web. 07 Jul 2020.

Vancouver:

Velkova A. The Role of BRCA1 Domains and Motifs in Tumor Suppression. [Internet] [Thesis]. University of South Florida; 2011. [cited 2020 Jul 07]. Available from: https://scholarcommons.usf.edu/etd/3391.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Velkova A. The Role of BRCA1 Domains and Motifs in Tumor Suppression. [Thesis]. University of South Florida; 2011. Available from: https://scholarcommons.usf.edu/etd/3391

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toledo

16. Dayal, Shubham. Novel Roles of RNase L in Prostate Cancer.

Degree: PhD, Biology (Cell-Molecular Biology), 2017, University of Toledo

 Prostate cancer is the second leading cause of cancer-related death in men in the U.S. Hereditary Prostate Cancer (HPC) accounts for 43% of early onset… (more)

Subjects/Keywords: Biology; Prostate Cancer; Cell Motility; Cell Migration; Androgen Receptor; RNase L; Hereditary Prostate Cancer 1; Filamin A; 2-5 A; Androgen; PSA; Integrin beta 1; FAK-Src; Rac-GTPase

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APA (6th Edition):

Dayal, S. (2017). Novel Roles of RNase L in Prostate Cancer. (Doctoral Dissertation). University of Toledo. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=toledo1492793747213566

Chicago Manual of Style (16th Edition):

Dayal, Shubham. “Novel Roles of RNase L in Prostate Cancer.” 2017. Doctoral Dissertation, University of Toledo. Accessed July 07, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=toledo1492793747213566.

MLA Handbook (7th Edition):

Dayal, Shubham. “Novel Roles of RNase L in Prostate Cancer.” 2017. Web. 07 Jul 2020.

Vancouver:

Dayal S. Novel Roles of RNase L in Prostate Cancer. [Internet] [Doctoral dissertation]. University of Toledo; 2017. [cited 2020 Jul 07]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=toledo1492793747213566.

Council of Science Editors:

Dayal S. Novel Roles of RNase L in Prostate Cancer. [Doctoral Dissertation]. University of Toledo; 2017. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=toledo1492793747213566

17. Narain, Niven Rajin. Identification and Clinical Validation of Novel Prostate Cancer Biomarkers Filamin A (flna), Filamin B (flnb), and Keratin 19 (krt19) Using Patient-Derived Omics and Bayesian Artificial Intelligence.

Degree: PhD, Biochemistry and Molecular Biology (Medicine), 2018, University of Miami

  This dissertation herein describes a novel strategy for Prostate Cancer (PCa) biomarker discovery employing use of systems biology causal modeling using omics, molecular phenotyping,… (more)

Subjects/Keywords: Bayesian artificial intelligence, Systems biology and Omics; Biomarkers; Prostate Cancer and Benign prostate hypertrophy; keratin-19; filamin-A; filamin-B

…Of interest, FLNB showed a causal link to another filamin protein, FLNA (see Appendix… …effectively is firstly dedicated to God for his choice to use me as a vessel to shed light onto the… …chasm of blind faith of belief of a Supreme Being juxtaposed against the need to observe and… …allowing me to be who I am and supporting a bold cause to make medicine better. Many nights… …holding my hands and wiping my tears on this path would have created a bond of truth and legacy… 

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APA (6th Edition):

Narain, N. R. (2018). Identification and Clinical Validation of Novel Prostate Cancer Biomarkers Filamin A (flna), Filamin B (flnb), and Keratin 19 (krt19) Using Patient-Derived Omics and Bayesian Artificial Intelligence. (Doctoral Dissertation). University of Miami. Retrieved from https://scholarlyrepository.miami.edu/oa_dissertations/2186

Chicago Manual of Style (16th Edition):

Narain, Niven Rajin. “Identification and Clinical Validation of Novel Prostate Cancer Biomarkers Filamin A (flna), Filamin B (flnb), and Keratin 19 (krt19) Using Patient-Derived Omics and Bayesian Artificial Intelligence.” 2018. Doctoral Dissertation, University of Miami. Accessed July 07, 2020. https://scholarlyrepository.miami.edu/oa_dissertations/2186.

MLA Handbook (7th Edition):

Narain, Niven Rajin. “Identification and Clinical Validation of Novel Prostate Cancer Biomarkers Filamin A (flna), Filamin B (flnb), and Keratin 19 (krt19) Using Patient-Derived Omics and Bayesian Artificial Intelligence.” 2018. Web. 07 Jul 2020.

Vancouver:

Narain NR. Identification and Clinical Validation of Novel Prostate Cancer Biomarkers Filamin A (flna), Filamin B (flnb), and Keratin 19 (krt19) Using Patient-Derived Omics and Bayesian Artificial Intelligence. [Internet] [Doctoral dissertation]. University of Miami; 2018. [cited 2020 Jul 07]. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/2186.

Council of Science Editors:

Narain NR. Identification and Clinical Validation of Novel Prostate Cancer Biomarkers Filamin A (flna), Filamin B (flnb), and Keratin 19 (krt19) Using Patient-Derived Omics and Bayesian Artificial Intelligence. [Doctoral Dissertation]. University of Miami; 2018. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/2186

18. LUO WEIWEI. CHARACTERIZATION OF NODAL ACTIN NETWORK: FUNCTIONS OF MYOSIN II, FILAMIN A AND FORMIN DAAM1.

Degree: 2014, National University of Singapore

Subjects/Keywords: Actin nodes; self-organized network; cytoskeleton cohesiveness; Myosin II; Filamin A; DAAM1; discrete stochastic computational model

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APA (6th Edition):

WEIWEI, L. (2014). CHARACTERIZATION OF NODAL ACTIN NETWORK: FUNCTIONS OF MYOSIN II, FILAMIN A AND FORMIN DAAM1. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/118262

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

WEIWEI, LUO. “CHARACTERIZATION OF NODAL ACTIN NETWORK: FUNCTIONS OF MYOSIN II, FILAMIN A AND FORMIN DAAM1.” 2014. Thesis, National University of Singapore. Accessed July 07, 2020. http://scholarbank.nus.edu.sg/handle/10635/118262.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

WEIWEI, LUO. “CHARACTERIZATION OF NODAL ACTIN NETWORK: FUNCTIONS OF MYOSIN II, FILAMIN A AND FORMIN DAAM1.” 2014. Web. 07 Jul 2020.

Vancouver:

WEIWEI L. CHARACTERIZATION OF NODAL ACTIN NETWORK: FUNCTIONS OF MYOSIN II, FILAMIN A AND FORMIN DAAM1. [Internet] [Thesis]. National University of Singapore; 2014. [cited 2020 Jul 07]. Available from: http://scholarbank.nus.edu.sg/handle/10635/118262.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

WEIWEI L. CHARACTERIZATION OF NODAL ACTIN NETWORK: FUNCTIONS OF MYOSIN II, FILAMIN A AND FORMIN DAAM1. [Thesis]. National University of Singapore; 2014. Available from: http://scholarbank.nus.edu.sg/handle/10635/118262

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.