Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:( Extracellular signal regulated kinase). Showing records 1 – 30 of 13630 total matches.

[1] [2] [3] [4] [5] … [455]

Search Limiters

Last 2 Years | English Only

Languages

Country

▼ Search Limiters


University of Texas Southwestern Medical Center

1. Piala, Alexander Townshend. Kinetics and Regulation of Protein Kinases.

Degree: 2015, University of Texas Southwestern Medical Center

 One of the major functions of kinases in biological systems is the relay of signal from an effector to a downstream target. Kinases are regulated(more)

Subjects/Keywords: Extracellular Signal-Regulated MAP Kinases; MAP Kinase Signaling System; MAP Kinase Kinase Kinases; Signal Transduction

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Piala, A. T. (2015). Kinetics and Regulation of Protein Kinases. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4206

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Piala, Alexander Townshend. “Kinetics and Regulation of Protein Kinases.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed October 23, 2019. http://hdl.handle.net/2152.5/4206.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Piala, Alexander Townshend. “Kinetics and Regulation of Protein Kinases.” 2015. Web. 23 Oct 2019.

Vancouver:

Piala AT. Kinetics and Regulation of Protein Kinases. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2019 Oct 23]. Available from: http://hdl.handle.net/2152.5/4206.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Piala AT. Kinetics and Regulation of Protein Kinases. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4206

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

2. Salery, Marine. Rôle de la protéine Arc (Activity-regulated cytoskeleton-associated protein) dans les adaptations moléculaires et comportementales induites par la cocaïne : Role of Arc protein (Activity-regulated cytoskeleton-associated protein) in molecular and behavioral adaptations to cocaine.

Degree: Docteur es, Neurosciences, 2015, Université Pierre et Marie Curie – Paris VI

Les adaptations cellulaires et moléculaires induites par les drogues jouent un rôle central dans les altérations comportementales à long terme observées dans l’addiction. Cette étude… (more)

Subjects/Keywords: Arc (Activity-Regulated Cytoskeleton-Associated protein); ERK (Extracellular-Signal Regulated Kinase); Striatum; Cocaïne; Chromatine; Plasticité; Arc (Activity-Regulated Cytoskeleton-Associated protein); ERK (Extracellular-Signal Regulated Kinase); Cocaine; 573.8

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Salery, M. (2015). Rôle de la protéine Arc (Activity-regulated cytoskeleton-associated protein) dans les adaptations moléculaires et comportementales induites par la cocaïne : Role of Arc protein (Activity-regulated cytoskeleton-associated protein) in molecular and behavioral adaptations to cocaine. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2015PA066400

Chicago Manual of Style (16th Edition):

Salery, Marine. “Rôle de la protéine Arc (Activity-regulated cytoskeleton-associated protein) dans les adaptations moléculaires et comportementales induites par la cocaïne : Role of Arc protein (Activity-regulated cytoskeleton-associated protein) in molecular and behavioral adaptations to cocaine.” 2015. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed October 23, 2019. http://www.theses.fr/2015PA066400.

MLA Handbook (7th Edition):

Salery, Marine. “Rôle de la protéine Arc (Activity-regulated cytoskeleton-associated protein) dans les adaptations moléculaires et comportementales induites par la cocaïne : Role of Arc protein (Activity-regulated cytoskeleton-associated protein) in molecular and behavioral adaptations to cocaine.” 2015. Web. 23 Oct 2019.

Vancouver:

Salery M. Rôle de la protéine Arc (Activity-regulated cytoskeleton-associated protein) dans les adaptations moléculaires et comportementales induites par la cocaïne : Role of Arc protein (Activity-regulated cytoskeleton-associated protein) in molecular and behavioral adaptations to cocaine. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2015. [cited 2019 Oct 23]. Available from: http://www.theses.fr/2015PA066400.

Council of Science Editors:

Salery M. Rôle de la protéine Arc (Activity-regulated cytoskeleton-associated protein) dans les adaptations moléculaires et comportementales induites par la cocaïne : Role of Arc protein (Activity-regulated cytoskeleton-associated protein) in molecular and behavioral adaptations to cocaine. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2015. Available from: http://www.theses.fr/2015PA066400


University of Illinois – Urbana-Champaign

3. Cervantes, David. Modulation of mitogenic signaling and growth by sympathetic adrenergic regulation.

Degree: PhD, 0325, 2012, University of Illinois – Urbana-Champaign

 Heart disease and diabetes mellitus are growing epidemics, consistently ranking within the top ten causes of death in the United States. Both diseases are associated… (more)

Subjects/Keywords: adrenergic receptor; insulin receptor; extracellular signal-regulated kinase (ERK); heart; arrestin

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cervantes, D. (2012). Modulation of mitogenic signaling and growth by sympathetic adrenergic regulation. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/29837

Chicago Manual of Style (16th Edition):

Cervantes, David. “Modulation of mitogenic signaling and growth by sympathetic adrenergic regulation.” 2012. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed October 23, 2019. http://hdl.handle.net/2142/29837.

MLA Handbook (7th Edition):

Cervantes, David. “Modulation of mitogenic signaling and growth by sympathetic adrenergic regulation.” 2012. Web. 23 Oct 2019.

Vancouver:

Cervantes D. Modulation of mitogenic signaling and growth by sympathetic adrenergic regulation. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2012. [cited 2019 Oct 23]. Available from: http://hdl.handle.net/2142/29837.

Council of Science Editors:

Cervantes D. Modulation of mitogenic signaling and growth by sympathetic adrenergic regulation. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2012. Available from: http://hdl.handle.net/2142/29837


University of Minnesota

4. Zeliadt, Nicholette A. Negative regulation of oncogenic Ras signaling by mitogen-activated protein kinase phosphatase-3.

Degree: PhD, Environmental Health, 2010, University of Minnesota

 Activating mutations in the Ras oncogene are found in ~30% of all human cancers. Ras is a central regulator of intracellular signaling pathways, such as… (more)

Subjects/Keywords: Extracellular signal regulated kinase; Mitogen activated protein kinase phosphatase-3; Prenylation; Ras; Environmental Health

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zeliadt, N. A. (2010). Negative regulation of oncogenic Ras signaling by mitogen-activated protein kinase phosphatase-3. (Doctoral Dissertation). University of Minnesota. Retrieved from http://purl.umn.edu/95772

Chicago Manual of Style (16th Edition):

Zeliadt, Nicholette A. “Negative regulation of oncogenic Ras signaling by mitogen-activated protein kinase phosphatase-3.” 2010. Doctoral Dissertation, University of Minnesota. Accessed October 23, 2019. http://purl.umn.edu/95772.

MLA Handbook (7th Edition):

Zeliadt, Nicholette A. “Negative regulation of oncogenic Ras signaling by mitogen-activated protein kinase phosphatase-3.” 2010. Web. 23 Oct 2019.

Vancouver:

Zeliadt NA. Negative regulation of oncogenic Ras signaling by mitogen-activated protein kinase phosphatase-3. [Internet] [Doctoral dissertation]. University of Minnesota; 2010. [cited 2019 Oct 23]. Available from: http://purl.umn.edu/95772.

Council of Science Editors:

Zeliadt NA. Negative regulation of oncogenic Ras signaling by mitogen-activated protein kinase phosphatase-3. [Doctoral Dissertation]. University of Minnesota; 2010. Available from: http://purl.umn.edu/95772


University of Illinois – Urbana-Champaign

5. Weng, Ning. Dysregulated ERK signal pathway and immune profiles in Fragile X Syndrome.

Degree: PhD, 4094, 2012, University of Illinois – Urbana-Champaign

 Fragile X Syndrome (FXS) is the leading cause of inherited mental retardation, and the most common identified genetic cause of autism. Lack of production of… (more)

Subjects/Keywords: Fragile X Syndrome; extracellular-signal regulated kinase; extracellular-signal regulated kinase (ERK); Mitogen-activated protein kinases (MAPK); biomarker; cytokine profiles; flow cytometry

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Weng, N. (2012). Dysregulated ERK signal pathway and immune profiles in Fragile X Syndrome. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/32022

Chicago Manual of Style (16th Edition):

Weng, Ning. “Dysregulated ERK signal pathway and immune profiles in Fragile X Syndrome.” 2012. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed October 23, 2019. http://hdl.handle.net/2142/32022.

MLA Handbook (7th Edition):

Weng, Ning. “Dysregulated ERK signal pathway and immune profiles in Fragile X Syndrome.” 2012. Web. 23 Oct 2019.

Vancouver:

Weng N. Dysregulated ERK signal pathway and immune profiles in Fragile X Syndrome. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2012. [cited 2019 Oct 23]. Available from: http://hdl.handle.net/2142/32022.

Council of Science Editors:

Weng N. Dysregulated ERK signal pathway and immune profiles in Fragile X Syndrome. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2012. Available from: http://hdl.handle.net/2142/32022


University of Pennsylvania

6. Dumoulin, Michelle Claire. The Role of REM Sleep in Ocular Dominance Plasticity Consolidation.

Degree: 2013, University of Pennsylvania

 Despite decades of research, the function of sleep remains controversial. One theory is that sleep plays a role in consolidating plasticity induced during prior waking.… (more)

Subjects/Keywords: consolidation; extracellular signal-regulated kinase; MAP kinase-interacting kinase 1; ocular dominance plasticity; protein synthesis; REM sleep; Neuroscience and Neurobiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dumoulin, M. C. (2013). The Role of REM Sleep in Ocular Dominance Plasticity Consolidation. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/852

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dumoulin, Michelle Claire. “The Role of REM Sleep in Ocular Dominance Plasticity Consolidation.” 2013. Thesis, University of Pennsylvania. Accessed October 23, 2019. https://repository.upenn.edu/edissertations/852.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dumoulin, Michelle Claire. “The Role of REM Sleep in Ocular Dominance Plasticity Consolidation.” 2013. Web. 23 Oct 2019.

Vancouver:

Dumoulin MC. The Role of REM Sleep in Ocular Dominance Plasticity Consolidation. [Internet] [Thesis]. University of Pennsylvania; 2013. [cited 2019 Oct 23]. Available from: https://repository.upenn.edu/edissertations/852.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dumoulin MC. The Role of REM Sleep in Ocular Dominance Plasticity Consolidation. [Thesis]. University of Pennsylvania; 2013. Available from: https://repository.upenn.edu/edissertations/852

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Edinburgh

7. Chan, Wai Kit. Role of differential heparan sulphate sulphation in Fgf/Erk signalling during mouse telencephalic development.

Degree: PhD, 2016, University of Edinburgh

 Heparan sulphate proteoglycans (HSPGs) are cell surface/secreted molecules expressed by all cells. HSPGs consist of carbohydrate side-chains attached to a core protein and are involved… (more)

Subjects/Keywords: heparan sulphate; Fibroblast growth factors; Fgf; Erk; extracellular signal-regulated kinase; morphogen; development

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chan, W. K. (2016). Role of differential heparan sulphate sulphation in Fgf/Erk signalling during mouse telencephalic development. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/25718

Chicago Manual of Style (16th Edition):

Chan, Wai Kit. “Role of differential heparan sulphate sulphation in Fgf/Erk signalling during mouse telencephalic development.” 2016. Doctoral Dissertation, University of Edinburgh. Accessed October 23, 2019. http://hdl.handle.net/1842/25718.

MLA Handbook (7th Edition):

Chan, Wai Kit. “Role of differential heparan sulphate sulphation in Fgf/Erk signalling during mouse telencephalic development.” 2016. Web. 23 Oct 2019.

Vancouver:

Chan WK. Role of differential heparan sulphate sulphation in Fgf/Erk signalling during mouse telencephalic development. [Internet] [Doctoral dissertation]. University of Edinburgh; 2016. [cited 2019 Oct 23]. Available from: http://hdl.handle.net/1842/25718.

Council of Science Editors:

Chan WK. Role of differential heparan sulphate sulphation in Fgf/Erk signalling during mouse telencephalic development. [Doctoral Dissertation]. University of Edinburgh; 2016. Available from: http://hdl.handle.net/1842/25718


Kyoto University / 京都大学

8. Sano, Takeshi. Intravital imaging of mouse urothelium reveals activation of extracellular signal-regulated kinase by stretch-induced intravesical release of ATP : マウス尿路上皮生体イメージングが解明したストレッチ誘導性ATP膀胱腔内分泌による細胞外シグナル調節キナーゼの活性化.

Degree: 博士(医学), 2017, Kyoto University / 京都大学

新制・課程博士

甲第20242号

医博第4201号

Subjects/Keywords: extracellular signal-regulated kinase; intravital imaging; mouse; urothelium; ATP

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sano, T. (2017). Intravital imaging of mouse urothelium reveals activation of extracellular signal-regulated kinase by stretch-induced intravesical release of ATP : マウス尿路上皮生体イメージングが解明したストレッチ誘導性ATP膀胱腔内分泌による細胞外シグナル調節キナーゼの活性化. (Thesis). Kyoto University / 京都大学. Retrieved from http://hdl.handle.net/2433/225467 ; http://dx.doi.org/10.14989/doctor.k20242

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sano, Takeshi. “Intravital imaging of mouse urothelium reveals activation of extracellular signal-regulated kinase by stretch-induced intravesical release of ATP : マウス尿路上皮生体イメージングが解明したストレッチ誘導性ATP膀胱腔内分泌による細胞外シグナル調節キナーゼの活性化.” 2017. Thesis, Kyoto University / 京都大学. Accessed October 23, 2019. http://hdl.handle.net/2433/225467 ; http://dx.doi.org/10.14989/doctor.k20242.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sano, Takeshi. “Intravital imaging of mouse urothelium reveals activation of extracellular signal-regulated kinase by stretch-induced intravesical release of ATP : マウス尿路上皮生体イメージングが解明したストレッチ誘導性ATP膀胱腔内分泌による細胞外シグナル調節キナーゼの活性化.” 2017. Web. 23 Oct 2019.

Vancouver:

Sano T. Intravital imaging of mouse urothelium reveals activation of extracellular signal-regulated kinase by stretch-induced intravesical release of ATP : マウス尿路上皮生体イメージングが解明したストレッチ誘導性ATP膀胱腔内分泌による細胞外シグナル調節キナーゼの活性化. [Internet] [Thesis]. Kyoto University / 京都大学; 2017. [cited 2019 Oct 23]. Available from: http://hdl.handle.net/2433/225467 ; http://dx.doi.org/10.14989/doctor.k20242.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sano T. Intravital imaging of mouse urothelium reveals activation of extracellular signal-regulated kinase by stretch-induced intravesical release of ATP : マウス尿路上皮生体イメージングが解明したストレッチ誘導性ATP膀胱腔内分泌による細胞外シグナル調節キナーゼの活性化. [Thesis]. Kyoto University / 京都大学; 2017. Available from: http://hdl.handle.net/2433/225467 ; http://dx.doi.org/10.14989/doctor.k20242

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

9. Wu, Yuexiu. Role of ERK5 in Diabetic Vascular Complications.

Degree: 2012, University of Western Ontario

 Chronic complications are the leading cause of the mortality and morbidity in diabetic patients. Endothelial cell dysfunction plays an important role in the pathogenesis of… (more)

Subjects/Keywords: Diabetic Complications; extracellular signal-regulated kinase 5; endothelin-1; vascular endothelial growth factor; fibronectin; Medicine and Health Sciences

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wu, Y. (2012). Role of ERK5 in Diabetic Vascular Complications. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/586

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wu, Yuexiu. “Role of ERK5 in Diabetic Vascular Complications.” 2012. Thesis, University of Western Ontario. Accessed October 23, 2019. https://ir.lib.uwo.ca/etd/586.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wu, Yuexiu. “Role of ERK5 in Diabetic Vascular Complications.” 2012. Web. 23 Oct 2019.

Vancouver:

Wu Y. Role of ERK5 in Diabetic Vascular Complications. [Internet] [Thesis]. University of Western Ontario; 2012. [cited 2019 Oct 23]. Available from: https://ir.lib.uwo.ca/etd/586.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wu Y. Role of ERK5 in Diabetic Vascular Complications. [Thesis]. University of Western Ontario; 2012. Available from: https://ir.lib.uwo.ca/etd/586

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Kyoto University

10. Sano, Takeshi. Intravital imaging of mouse urothelium reveals activation of extracellular signal-regulated kinase by stretch-induced intravesical release of ATP .

Degree: 2017, Kyoto University

Subjects/Keywords: extracellular signal-regulated kinase; intravital imaging; mouse; urothelium; ATP

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sano, T. (2017). Intravital imaging of mouse urothelium reveals activation of extracellular signal-regulated kinase by stretch-induced intravesical release of ATP . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/225467

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sano, Takeshi. “Intravital imaging of mouse urothelium reveals activation of extracellular signal-regulated kinase by stretch-induced intravesical release of ATP .” 2017. Thesis, Kyoto University. Accessed October 23, 2019. http://hdl.handle.net/2433/225467.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sano, Takeshi. “Intravital imaging of mouse urothelium reveals activation of extracellular signal-regulated kinase by stretch-induced intravesical release of ATP .” 2017. Web. 23 Oct 2019.

Vancouver:

Sano T. Intravital imaging of mouse urothelium reveals activation of extracellular signal-regulated kinase by stretch-induced intravesical release of ATP . [Internet] [Thesis]. Kyoto University; 2017. [cited 2019 Oct 23]. Available from: http://hdl.handle.net/2433/225467.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sano T. Intravital imaging of mouse urothelium reveals activation of extracellular signal-regulated kinase by stretch-induced intravesical release of ATP . [Thesis]. Kyoto University; 2017. Available from: http://hdl.handle.net/2433/225467

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Commonwealth University

11. Thompson, Lauren. Intracellular Signaling Contributions to Behaviors Relevant to Nicotine Addiction.

Degree: MS, Pharmacology & Toxicology, 2011, Virginia Commonwealth University

 Nicotine is the primary addictive substance in tobacco, and most smokers who quit will relapse within a year. Evidence shows that cigarette craving increases over… (more)

Subjects/Keywords: nicotine; incubation; protracted abstinence; self-administration; ERK; extracellular signal regulated kinase; Medical Pharmacology; Medical Sciences; Medicine and Health Sciences

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Thompson, L. (2011). Intracellular Signaling Contributions to Behaviors Relevant to Nicotine Addiction. (Thesis). Virginia Commonwealth University. Retrieved from https://scholarscompass.vcu.edu/etd/253

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Thompson, Lauren. “Intracellular Signaling Contributions to Behaviors Relevant to Nicotine Addiction.” 2011. Thesis, Virginia Commonwealth University. Accessed October 23, 2019. https://scholarscompass.vcu.edu/etd/253.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Thompson, Lauren. “Intracellular Signaling Contributions to Behaviors Relevant to Nicotine Addiction.” 2011. Web. 23 Oct 2019.

Vancouver:

Thompson L. Intracellular Signaling Contributions to Behaviors Relevant to Nicotine Addiction. [Internet] [Thesis]. Virginia Commonwealth University; 2011. [cited 2019 Oct 23]. Available from: https://scholarscompass.vcu.edu/etd/253.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Thompson L. Intracellular Signaling Contributions to Behaviors Relevant to Nicotine Addiction. [Thesis]. Virginia Commonwealth University; 2011. Available from: https://scholarscompass.vcu.edu/etd/253

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pennsylvania

12. Furcht, Christopher. The Protein Tyrosine Phosphatase SHP2 as a Mediator of Differential Cellular Sensitivity to EGFR Kinase Inhibitors.

Degree: 2014, University of Pennsylvania

 SRC homology 2 domain-containing phosphatase 2 (SHP2) is a ubiquitously expressed cytosolic protein tyrosine phosphatase. Downstream of epidermal growth factor receptor (EGFR) and other receptors,… (more)

Subjects/Keywords: extracellular signal-regulated kinase; gefitinib; glioblastoma multiforme; GRB2-associated binder 1; non-small cell lung cancer; Chemical Engineering

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Furcht, C. (2014). The Protein Tyrosine Phosphatase SHP2 as a Mediator of Differential Cellular Sensitivity to EGFR Kinase Inhibitors. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/1279

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Furcht, Christopher. “The Protein Tyrosine Phosphatase SHP2 as a Mediator of Differential Cellular Sensitivity to EGFR Kinase Inhibitors.” 2014. Thesis, University of Pennsylvania. Accessed October 23, 2019. https://repository.upenn.edu/edissertations/1279.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Furcht, Christopher. “The Protein Tyrosine Phosphatase SHP2 as a Mediator of Differential Cellular Sensitivity to EGFR Kinase Inhibitors.” 2014. Web. 23 Oct 2019.

Vancouver:

Furcht C. The Protein Tyrosine Phosphatase SHP2 as a Mediator of Differential Cellular Sensitivity to EGFR Kinase Inhibitors. [Internet] [Thesis]. University of Pennsylvania; 2014. [cited 2019 Oct 23]. Available from: https://repository.upenn.edu/edissertations/1279.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Furcht C. The Protein Tyrosine Phosphatase SHP2 as a Mediator of Differential Cellular Sensitivity to EGFR Kinase Inhibitors. [Thesis]. University of Pennsylvania; 2014. Available from: https://repository.upenn.edu/edissertations/1279

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

13. Chang, Shunzhi. Role of Extracellular-signal Regulated Kinase (ERK) and cAMP Response Element Binding Protein (CREB) in the Incubation of Nicotine Craving.

Degree: 2013, University of Toronto

Nicotine Addiction is a chronic relapsing disorder. Relapse risk persists despite years of abstinence. Drug-associated cues have been demonstrated to induce craving and provoke relapse.… (more)

Subjects/Keywords: nicotine addiction; Extracellular-signal Regulated Kinase (ERK); cAMP Response Element Binding Protein (CREB); Fos; incubation of nicotine craving; 0419; 0347

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chang, S. (2013). Role of Extracellular-signal Regulated Kinase (ERK) and cAMP Response Element Binding Protein (CREB) in the Incubation of Nicotine Craving. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/42709

Chicago Manual of Style (16th Edition):

Chang, Shunzhi. “Role of Extracellular-signal Regulated Kinase (ERK) and cAMP Response Element Binding Protein (CREB) in the Incubation of Nicotine Craving.” 2013. Masters Thesis, University of Toronto. Accessed October 23, 2019. http://hdl.handle.net/1807/42709.

MLA Handbook (7th Edition):

Chang, Shunzhi. “Role of Extracellular-signal Regulated Kinase (ERK) and cAMP Response Element Binding Protein (CREB) in the Incubation of Nicotine Craving.” 2013. Web. 23 Oct 2019.

Vancouver:

Chang S. Role of Extracellular-signal Regulated Kinase (ERK) and cAMP Response Element Binding Protein (CREB) in the Incubation of Nicotine Craving. [Internet] [Masters thesis]. University of Toronto; 2013. [cited 2019 Oct 23]. Available from: http://hdl.handle.net/1807/42709.

Council of Science Editors:

Chang S. Role of Extracellular-signal Regulated Kinase (ERK) and cAMP Response Element Binding Protein (CREB) in the Incubation of Nicotine Craving. [Masters Thesis]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/42709


University of Lund

14. Mostajeran, Maryam. A Path for Improving Stroke Recovery. Effects of MEK-ERK1/2 Inhibition.

Degree: 2018, University of Lund

 The present thesis aimed to shed more light on the notion of acute inhibition of extracellular-signal regulated kinase (ERK)1/2 pathway as a treatment to improve… (more)

Subjects/Keywords: Medicin och hälsovetenskap; Ischemic stroke, Extracellular-signal regulated kinase (ERK)1/2, Mitogen activated protein kinase kinase (MEK)1/2, Recovery, Functional outcome, sex

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mostajeran, M. (2018). A Path for Improving Stroke Recovery. Effects of MEK-ERK1/2 Inhibition. (Doctoral Dissertation). University of Lund. Retrieved from http://lup.lub.lu.se/record/7381fb75-7d14-4876-b8b7-12a9ea11f156 ; http://portal.research.lu.se/ws/files/55026035/maryam_e_nailing.pdf

Chicago Manual of Style (16th Edition):

Mostajeran, Maryam. “A Path for Improving Stroke Recovery. Effects of MEK-ERK1/2 Inhibition.” 2018. Doctoral Dissertation, University of Lund. Accessed October 23, 2019. http://lup.lub.lu.se/record/7381fb75-7d14-4876-b8b7-12a9ea11f156 ; http://portal.research.lu.se/ws/files/55026035/maryam_e_nailing.pdf.

MLA Handbook (7th Edition):

Mostajeran, Maryam. “A Path for Improving Stroke Recovery. Effects of MEK-ERK1/2 Inhibition.” 2018. Web. 23 Oct 2019.

Vancouver:

Mostajeran M. A Path for Improving Stroke Recovery. Effects of MEK-ERK1/2 Inhibition. [Internet] [Doctoral dissertation]. University of Lund; 2018. [cited 2019 Oct 23]. Available from: http://lup.lub.lu.se/record/7381fb75-7d14-4876-b8b7-12a9ea11f156 ; http://portal.research.lu.se/ws/files/55026035/maryam_e_nailing.pdf.

Council of Science Editors:

Mostajeran M. A Path for Improving Stroke Recovery. Effects of MEK-ERK1/2 Inhibition. [Doctoral Dissertation]. University of Lund; 2018. Available from: http://lup.lub.lu.se/record/7381fb75-7d14-4876-b8b7-12a9ea11f156 ; http://portal.research.lu.se/ws/files/55026035/maryam_e_nailing.pdf


University of Texas Southwestern Medical Center

15. McReynolds, Andrea Christine. Functional Significance of Extracellular Signal Regulated Kinase (ERK2) Phosphorylation States: Implications for DNA Binding.

Degree: 2013, University of Texas Southwestern Medical Center

 The protein kinase extracellular signal-regulated kinase 2 (ERK2) has been well understood structurally for nearly twenty years. New insight is emerging about its structure and… (more)

Subjects/Keywords: DNA; Extracellular Signal-Regulated MAP Kinases; Phosphorylation

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

McReynolds, A. C. (2013). Functional Significance of Extracellular Signal Regulated Kinase (ERK2) Phosphorylation States: Implications for DNA Binding. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/2725

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

McReynolds, Andrea Christine. “Functional Significance of Extracellular Signal Regulated Kinase (ERK2) Phosphorylation States: Implications for DNA Binding.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed October 23, 2019. http://hdl.handle.net/2152.5/2725.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

McReynolds, Andrea Christine. “Functional Significance of Extracellular Signal Regulated Kinase (ERK2) Phosphorylation States: Implications for DNA Binding.” 2013. Web. 23 Oct 2019.

Vancouver:

McReynolds AC. Functional Significance of Extracellular Signal Regulated Kinase (ERK2) Phosphorylation States: Implications for DNA Binding. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2019 Oct 23]. Available from: http://hdl.handle.net/2152.5/2725.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

McReynolds AC. Functional Significance of Extracellular Signal Regulated Kinase (ERK2) Phosphorylation States: Implications for DNA Binding. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/2725

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

16. 이, 중선. The Effects of PPAR-γ Activators on the Growth and Apoptosis of Melanocytes.

Degree: 2005, Ajou University

"Purpose : Peroxisome proliferator-activated receptors (PPARs) play an important role in cellular responses including proliferation and differentiation. It was reported that 3 subtypes of PPAR… (more)

Subjects/Keywords: Melanocyte; Peroxisome proliferator-activated receptors-γ; Ciglitazone; Growth; Apoptosis; Extracellular signal regulated kinase; 멜라닌세포; 세포고사; 세포성장; extracellular signal-regulated protein kinase

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

이, . (2005). The Effects of PPAR-γ Activators on the Growth and Apoptosis of Melanocytes. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/1279 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000000326

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

이, 중선. “The Effects of PPAR-γ Activators on the Growth and Apoptosis of Melanocytes.” 2005. Thesis, Ajou University. Accessed October 23, 2019. http://repository.ajou.ac.kr/handle/201003/1279 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000000326.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

이, 중선. “The Effects of PPAR-γ Activators on the Growth and Apoptosis of Melanocytes.” 2005. Web. 23 Oct 2019.

Vancouver:

이 . The Effects of PPAR-γ Activators on the Growth and Apoptosis of Melanocytes. [Internet] [Thesis]. Ajou University; 2005. [cited 2019 Oct 23]. Available from: http://repository.ajou.ac.kr/handle/201003/1279 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000000326.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

이 . The Effects of PPAR-γ Activators on the Growth and Apoptosis of Melanocytes. [Thesis]. Ajou University; 2005. Available from: http://repository.ajou.ac.kr/handle/201003/1279 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000000326

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

17. Bouallegue, Ali. Endothelin-1 and H2O2-induced signaling in vascular smooth muscle cells : modulation by CaMKII and Nitric oxide .

Degree: 2010, Université de Montréal

 L’endothéline-1 (ET-1) est un peptide vasoactif extrêmement puissant qui possède une forte activité mitogénique dans les cellules du muscle lisse vasculaire (VSMCs). Il a été… (more)

Subjects/Keywords: Endothéline; Peroxyde d'hydrogène; CaMKII; Monoxyde d’azote; Système vasculaire; PKB; ERK1/2; IGF-1R; Hypertrophie; Endothelin; Hydrogen peroxide; Nitric oxide; Vascular; Protein; Kinase B; Extracellular Signal-Regulated Kinase 1/2; Growth

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bouallegue, A. (2010). Endothelin-1 and H2O2-induced signaling in vascular smooth muscle cells : modulation by CaMKII and Nitric oxide . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/3858

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bouallegue, Ali. “Endothelin-1 and H2O2-induced signaling in vascular smooth muscle cells : modulation by CaMKII and Nitric oxide .” 2010. Thesis, Université de Montréal. Accessed October 23, 2019. http://hdl.handle.net/1866/3858.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bouallegue, Ali. “Endothelin-1 and H2O2-induced signaling in vascular smooth muscle cells : modulation by CaMKII and Nitric oxide .” 2010. Web. 23 Oct 2019.

Vancouver:

Bouallegue A. Endothelin-1 and H2O2-induced signaling in vascular smooth muscle cells : modulation by CaMKII and Nitric oxide . [Internet] [Thesis]. Université de Montréal; 2010. [cited 2019 Oct 23]. Available from: http://hdl.handle.net/1866/3858.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bouallegue A. Endothelin-1 and H2O2-induced signaling in vascular smooth muscle cells : modulation by CaMKII and Nitric oxide . [Thesis]. Université de Montréal; 2010. Available from: http://hdl.handle.net/1866/3858

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Kyoto University

18. Muta, Yu. Composite regulation of ERK activity dynamics underlying tumour-specific traits in the intestine .

Degree: 2018, Kyoto University

Subjects/Keywords: Live imaging; Gastrointestinal cancer; extracellular signal-regulated kinase; Signal transduction; Intestinal organoid

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Muta, Y. (2018). Composite regulation of ERK activity dynamics underlying tumour-specific traits in the intestine . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/235061

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Muta, Yu. “Composite regulation of ERK activity dynamics underlying tumour-specific traits in the intestine .” 2018. Thesis, Kyoto University. Accessed October 23, 2019. http://hdl.handle.net/2433/235061.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Muta, Yu. “Composite regulation of ERK activity dynamics underlying tumour-specific traits in the intestine .” 2018. Web. 23 Oct 2019.

Vancouver:

Muta Y. Composite regulation of ERK activity dynamics underlying tumour-specific traits in the intestine . [Internet] [Thesis]. Kyoto University; 2018. [cited 2019 Oct 23]. Available from: http://hdl.handle.net/2433/235061.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Muta Y. Composite regulation of ERK activity dynamics underlying tumour-specific traits in the intestine . [Thesis]. Kyoto University; 2018. Available from: http://hdl.handle.net/2433/235061

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. 永井, 隆. Differential regulation of angiotensin II-induced extracellular signal regulated kinase-1/2 and -5 in progressive glomerulonephritis : 進行性腎炎におけるアンジオテンシンIIにより活性化された細胞外シグナル調節キナーゼ-1/2および5の病態制御機構.

Degree: 博士(医学), 2016, Tokushima University / 徳島大学

Subjects/Keywords: Extracellular signal-regulated kinase; glomerulonephritis; renin-angiotensin system; macrophage infiltration; fibrosis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

永井, . (2016). Differential regulation of angiotensin II-induced extracellular signal regulated kinase-1/2 and -5 in progressive glomerulonephritis : 進行性腎炎におけるアンジオテンシンIIにより活性化された細胞外シグナル調節キナーゼ-1/2および5の病態制御機構. (Thesis). Tokushima University / 徳島大学. Retrieved from http://repo.lib.tokushima-u.ac.jp/109686

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

永井, 隆. “Differential regulation of angiotensin II-induced extracellular signal regulated kinase-1/2 and -5 in progressive glomerulonephritis : 進行性腎炎におけるアンジオテンシンIIにより活性化された細胞外シグナル調節キナーゼ-1/2および5の病態制御機構.” 2016. Thesis, Tokushima University / 徳島大学. Accessed October 23, 2019. http://repo.lib.tokushima-u.ac.jp/109686.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

永井, 隆. “Differential regulation of angiotensin II-induced extracellular signal regulated kinase-1/2 and -5 in progressive glomerulonephritis : 進行性腎炎におけるアンジオテンシンIIにより活性化された細胞外シグナル調節キナーゼ-1/2および5の病態制御機構.” 2016. Web. 23 Oct 2019.

Vancouver:

永井 . Differential regulation of angiotensin II-induced extracellular signal regulated kinase-1/2 and -5 in progressive glomerulonephritis : 進行性腎炎におけるアンジオテンシンIIにより活性化された細胞外シグナル調節キナーゼ-1/2および5の病態制御機構. [Internet] [Thesis]. Tokushima University / 徳島大学; 2016. [cited 2019 Oct 23]. Available from: http://repo.lib.tokushima-u.ac.jp/109686.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

永井 . Differential regulation of angiotensin II-induced extracellular signal regulated kinase-1/2 and -5 in progressive glomerulonephritis : 進行性腎炎におけるアンジオテンシンIIにより活性化された細胞外シグナル調節キナーゼ-1/2および5の病態制御機構. [Thesis]. Tokushima University / 徳島大学; 2016. Available from: http://repo.lib.tokushima-u.ac.jp/109686

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Xavier University

20. Carter, Molly H. Relationships Among Eye Gaze, Social Ability and Extracellular Signal-Regulated Kinase Pathway Activation in Children and Adolescents with Autistic Disorder.

Degree: Doctor of Psychology (Psy.D.), Psychology, 2016, Xavier University

 Previous eye tracking research suggested that children and adolescents with Autistic Disorder (AD) exhibit atypical gaze patterns while observing dynamic social interactions. The relationship among… (more)

Subjects/Keywords: Behavioral Psychology; Cellular Biology; Developmental Psychology; Autism Spectrum Disorder; eye tracking; extracellular signal-regulated kinase pathway; social responsiveness; fixation duration on faces; latency to first face fixation; play scene; biomarker

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Carter, M. H. (2016). Relationships Among Eye Gaze, Social Ability and Extracellular Signal-Regulated Kinase Pathway Activation in Children and Adolescents with Autistic Disorder. (Doctoral Dissertation). Xavier University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=xavier1461587486

Chicago Manual of Style (16th Edition):

Carter, Molly H. “Relationships Among Eye Gaze, Social Ability and Extracellular Signal-Regulated Kinase Pathway Activation in Children and Adolescents with Autistic Disorder.” 2016. Doctoral Dissertation, Xavier University. Accessed October 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=xavier1461587486.

MLA Handbook (7th Edition):

Carter, Molly H. “Relationships Among Eye Gaze, Social Ability and Extracellular Signal-Regulated Kinase Pathway Activation in Children and Adolescents with Autistic Disorder.” 2016. Web. 23 Oct 2019.

Vancouver:

Carter MH. Relationships Among Eye Gaze, Social Ability and Extracellular Signal-Regulated Kinase Pathway Activation in Children and Adolescents with Autistic Disorder. [Internet] [Doctoral dissertation]. Xavier University; 2016. [cited 2019 Oct 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=xavier1461587486.

Council of Science Editors:

Carter MH. Relationships Among Eye Gaze, Social Ability and Extracellular Signal-Regulated Kinase Pathway Activation in Children and Adolescents with Autistic Disorder. [Doctoral Dissertation]. Xavier University; 2016. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=xavier1461587486


University of Illinois – Urbana-Champaign

21. Ebaugh, Jason D. Extracellular Signal-Regulated Kinase 7, A Fast Evolving Map Kinase Under Positive Selection, Is Associated With Multiple Levels Of Honeybee Behavioral Plasticity.

Degree: Neuroscience, 2009, University of Illinois – Urbana-Champaign

Subjects/Keywords: honeybee; Erk7; behavioral plasticity; fast evolving; positive selection; extracellular signal-regulated kinase 7

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ebaugh, J. D. (2009). Extracellular Signal-Regulated Kinase 7, A Fast Evolving Map Kinase Under Positive Selection, Is Associated With Multiple Levels Of Honeybee Behavioral Plasticity. (Thesis). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/13005

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ebaugh, Jason D. “Extracellular Signal-Regulated Kinase 7, A Fast Evolving Map Kinase Under Positive Selection, Is Associated With Multiple Levels Of Honeybee Behavioral Plasticity.” 2009. Thesis, University of Illinois – Urbana-Champaign. Accessed October 23, 2019. http://hdl.handle.net/2142/13005.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ebaugh, Jason D. “Extracellular Signal-Regulated Kinase 7, A Fast Evolving Map Kinase Under Positive Selection, Is Associated With Multiple Levels Of Honeybee Behavioral Plasticity.” 2009. Web. 23 Oct 2019.

Vancouver:

Ebaugh JD. Extracellular Signal-Regulated Kinase 7, A Fast Evolving Map Kinase Under Positive Selection, Is Associated With Multiple Levels Of Honeybee Behavioral Plasticity. [Internet] [Thesis]. University of Illinois – Urbana-Champaign; 2009. [cited 2019 Oct 23]. Available from: http://hdl.handle.net/2142/13005.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ebaugh JD. Extracellular Signal-Regulated Kinase 7, A Fast Evolving Map Kinase Under Positive Selection, Is Associated With Multiple Levels Of Honeybee Behavioral Plasticity. [Thesis]. University of Illinois – Urbana-Champaign; 2009. Available from: http://hdl.handle.net/2142/13005

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Kyoto University

22. Konishi, Yoshinobu. Live-cell FRET imaging reveals a role of extracellular signal-regulated kinase activity dynamics in thymocyte motility .

Degree: 2019, Kyoto University

Subjects/Keywords: FRET biosensor; Live imaging; Extracellular signal-regulated kinase (ERK); Cell motility; Thymocyte

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Konishi, Y. (2019). Live-cell FRET imaging reveals a role of extracellular signal-regulated kinase activity dynamics in thymocyte motility . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/242374

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Konishi, Yoshinobu. “Live-cell FRET imaging reveals a role of extracellular signal-regulated kinase activity dynamics in thymocyte motility .” 2019. Thesis, Kyoto University. Accessed October 23, 2019. http://hdl.handle.net/2433/242374.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Konishi, Yoshinobu. “Live-cell FRET imaging reveals a role of extracellular signal-regulated kinase activity dynamics in thymocyte motility .” 2019. Web. 23 Oct 2019.

Vancouver:

Konishi Y. Live-cell FRET imaging reveals a role of extracellular signal-regulated kinase activity dynamics in thymocyte motility . [Internet] [Thesis]. Kyoto University; 2019. [cited 2019 Oct 23]. Available from: http://hdl.handle.net/2433/242374.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Konishi Y. Live-cell FRET imaging reveals a role of extracellular signal-regulated kinase activity dynamics in thymocyte motility . [Thesis]. Kyoto University; 2019. Available from: http://hdl.handle.net/2433/242374

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


North Carolina State University

23. Wang, Chun-Chao. Crosstalk Between Phosphoinositide 3-Kinase and Extracellular Signal-regulated Kinase Pathways in Platelet-derived Growth Factor Receptor-mediated Signaling.

Degree: MS, Chemical Engineering, 2005, North Carolina State University

 Cells control their behavior by maintaining a balance between signal transduction pathways. The disruption of this balance has been implicated in the evolution of normal… (more)

Subjects/Keywords: Erk; platelet-derived growth factor; PDGF; signal transduction; extracellular signal-regulated kinase; PI 3-kinase; crosstalk; phosphoinositide 3-kinase

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wang, C. (2005). Crosstalk Between Phosphoinositide 3-Kinase and Extracellular Signal-regulated Kinase Pathways in Platelet-derived Growth Factor Receptor-mediated Signaling. (Thesis). North Carolina State University. Retrieved from http://www.lib.ncsu.edu/resolver/1840.16/1376

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wang, Chun-Chao. “Crosstalk Between Phosphoinositide 3-Kinase and Extracellular Signal-regulated Kinase Pathways in Platelet-derived Growth Factor Receptor-mediated Signaling.” 2005. Thesis, North Carolina State University. Accessed October 23, 2019. http://www.lib.ncsu.edu/resolver/1840.16/1376.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wang, Chun-Chao. “Crosstalk Between Phosphoinositide 3-Kinase and Extracellular Signal-regulated Kinase Pathways in Platelet-derived Growth Factor Receptor-mediated Signaling.” 2005. Web. 23 Oct 2019.

Vancouver:

Wang C. Crosstalk Between Phosphoinositide 3-Kinase and Extracellular Signal-regulated Kinase Pathways in Platelet-derived Growth Factor Receptor-mediated Signaling. [Internet] [Thesis]. North Carolina State University; 2005. [cited 2019 Oct 23]. Available from: http://www.lib.ncsu.edu/resolver/1840.16/1376.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wang C. Crosstalk Between Phosphoinositide 3-Kinase and Extracellular Signal-regulated Kinase Pathways in Platelet-derived Growth Factor Receptor-mediated Signaling. [Thesis]. North Carolina State University; 2005. Available from: http://www.lib.ncsu.edu/resolver/1840.16/1376

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

24. Qi, Baowen. Novel surface-tethered estrogen polymeric platforms in cardiovascular regenerative medicine .

Degree: 2016, Université de Montréal

 L’estradiol (E2) est une hormone femelle qui joue un rôle essentiel, à la fois dans la régulation et dans la détermination de certaines conditions physiologiques… (more)

Subjects/Keywords: Estradiol; Effet non-génomique; Approches ascendantes et descendantes; Substrats en micromotifs; Chitosan; Phosphorylcholine; Oxyde nitrique; Médecine cardiovasculaire régénérative; Kinase régulée par signal extracellulaire; Non-genomic effect; Top-down and bottom-up approaches; Micropatterned substrates; Nitric oxide; Cardiovascular regenerative medicine; Extracellular signal-regulated kinase

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Qi, B. (2016). Novel surface-tethered estrogen polymeric platforms in cardiovascular regenerative medicine . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/14118

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Qi, Baowen. “Novel surface-tethered estrogen polymeric platforms in cardiovascular regenerative medicine .” 2016. Thesis, Université de Montréal. Accessed October 23, 2019. http://hdl.handle.net/1866/14118.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Qi, Baowen. “Novel surface-tethered estrogen polymeric platforms in cardiovascular regenerative medicine .” 2016. Web. 23 Oct 2019.

Vancouver:

Qi B. Novel surface-tethered estrogen polymeric platforms in cardiovascular regenerative medicine . [Internet] [Thesis]. Université de Montréal; 2016. [cited 2019 Oct 23]. Available from: http://hdl.handle.net/1866/14118.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Qi B. Novel surface-tethered estrogen polymeric platforms in cardiovascular regenerative medicine . [Thesis]. Université de Montréal; 2016. Available from: http://hdl.handle.net/1866/14118

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

25. Zaganjor, Elma 1981-. Regulation by ERK1/2 of Novel Substrates, Kinesins KIF2A and KIF2C.

Degree: 2013, University of Texas Southwestern Medical Center

 The kinesin-like protein KIF2A is a microtubule-associated motor protein thatauses microtubule depolymerization by inducing a conformational change in tubulin. The depolymerase function of KIF2A is… (more)

Subjects/Keywords: Cell Transformation, Neoplastic; Extracellular Signal-Regulated MAP Kinases; Kinesin

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zaganjor, E. 1. (2013). Regulation by ERK1/2 of Novel Substrates, Kinesins KIF2A and KIF2C. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-124

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zaganjor, Elma 1981-. “Regulation by ERK1/2 of Novel Substrates, Kinesins KIF2A and KIF2C.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed October 23, 2019. http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-124.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zaganjor, Elma 1981-. “Regulation by ERK1/2 of Novel Substrates, Kinesins KIF2A and KIF2C.” 2013. Web. 23 Oct 2019.

Vancouver:

Zaganjor E1. Regulation by ERK1/2 of Novel Substrates, Kinesins KIF2A and KIF2C. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2019 Oct 23]. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-124.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zaganjor E1. Regulation by ERK1/2 of Novel Substrates, Kinesins KIF2A and KIF2C. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-124

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Kyoto University / 京都大学

26. Okuno, Masayuki. Regorafenib suppresses sinusoidal obstruction syndrome in rats : レゴラフェニブはラット類洞閉塞症候群を緩和する.

Degree: 博士(医学), 2016, Kyoto University / 京都大学

新制・課程博士

甲第19587号

医博第4094号

Subjects/Keywords: colorectal liver metastasis; extracellular signal-regulated kinase; kinase inhibitor; drug-induced liver injury; metalloproteinase-9

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Okuno, M. (2016). Regorafenib suppresses sinusoidal obstruction syndrome in rats : レゴラフェニブはラット類洞閉塞症候群を緩和する. (Thesis). Kyoto University / 京都大学. Retrieved from http://hdl.handle.net/2433/215413 ; http://dx.doi.org/10.14989/doctor.k19587

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Okuno, Masayuki. “Regorafenib suppresses sinusoidal obstruction syndrome in rats : レゴラフェニブはラット類洞閉塞症候群を緩和する.” 2016. Thesis, Kyoto University / 京都大学. Accessed October 23, 2019. http://hdl.handle.net/2433/215413 ; http://dx.doi.org/10.14989/doctor.k19587.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Okuno, Masayuki. “Regorafenib suppresses sinusoidal obstruction syndrome in rats : レゴラフェニブはラット類洞閉塞症候群を緩和する.” 2016. Web. 23 Oct 2019.

Vancouver:

Okuno M. Regorafenib suppresses sinusoidal obstruction syndrome in rats : レゴラフェニブはラット類洞閉塞症候群を緩和する. [Internet] [Thesis]. Kyoto University / 京都大学; 2016. [cited 2019 Oct 23]. Available from: http://hdl.handle.net/2433/215413 ; http://dx.doi.org/10.14989/doctor.k19587.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Okuno M. Regorafenib suppresses sinusoidal obstruction syndrome in rats : レゴラフェニブはラット類洞閉塞症候群を緩和する. [Thesis]. Kyoto University / 京都大学; 2016. Available from: http://hdl.handle.net/2433/215413 ; http://dx.doi.org/10.14989/doctor.k19587

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

27. Okuno, Masayuki. Regorafenib suppresses sinusoidal obstruction syndrome in rats .

Degree: 2016, Kyoto University

Subjects/Keywords: colorectal liver metastasis; extracellular signal-regulated kinase; kinase inhibitor; drug-induced liver injury; metalloproteinase-9

Page 1 Page 2

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Okuno, M. (2016). Regorafenib suppresses sinusoidal obstruction syndrome in rats . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/215413

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Okuno, Masayuki. “Regorafenib suppresses sinusoidal obstruction syndrome in rats .” 2016. Thesis, Kyoto University. Accessed October 23, 2019. http://hdl.handle.net/2433/215413.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Okuno, Masayuki. “Regorafenib suppresses sinusoidal obstruction syndrome in rats .” 2016. Web. 23 Oct 2019.

Vancouver:

Okuno M. Regorafenib suppresses sinusoidal obstruction syndrome in rats . [Internet] [Thesis]. Kyoto University; 2016. [cited 2019 Oct 23]. Available from: http://hdl.handle.net/2433/215413.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Okuno M. Regorafenib suppresses sinusoidal obstruction syndrome in rats . [Thesis]. Kyoto University; 2016. Available from: http://hdl.handle.net/2433/215413

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. Dos Santos, Marc. Dynamique et mécanismes moléculaires de la plasticité structurale des neurones du noyau Accumbens en réponse à la cocaïne : Dynamics and molecular mechanisms of the cocaine-induced structural plasticity of nucleus Accumbens neurons.

Degree: Docteur es, Neurosciences, 2016, Université Pierre et Marie Curie – Paris VI

Les événements vécus peuvent laisser une trace durable au niveau des réseaux cérébraux. Ces réseaux sont constitués de neurones connectés par des synapses, dont l'efficacité… (more)

Subjects/Keywords: Épines dendritiques; Cocaïne; Synaptogénèse; Erk; Mnk; Striatum; Dendritic spines; Cocaine; Extracellular Regulated Kinase; 616.863

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dos Santos, M. (2016). Dynamique et mécanismes moléculaires de la plasticité structurale des neurones du noyau Accumbens en réponse à la cocaïne : Dynamics and molecular mechanisms of the cocaine-induced structural plasticity of nucleus Accumbens neurons. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2016PA066378

Chicago Manual of Style (16th Edition):

Dos Santos, Marc. “Dynamique et mécanismes moléculaires de la plasticité structurale des neurones du noyau Accumbens en réponse à la cocaïne : Dynamics and molecular mechanisms of the cocaine-induced structural plasticity of nucleus Accumbens neurons.” 2016. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed October 23, 2019. http://www.theses.fr/2016PA066378.

MLA Handbook (7th Edition):

Dos Santos, Marc. “Dynamique et mécanismes moléculaires de la plasticité structurale des neurones du noyau Accumbens en réponse à la cocaïne : Dynamics and molecular mechanisms of the cocaine-induced structural plasticity of nucleus Accumbens neurons.” 2016. Web. 23 Oct 2019.

Vancouver:

Dos Santos M. Dynamique et mécanismes moléculaires de la plasticité structurale des neurones du noyau Accumbens en réponse à la cocaïne : Dynamics and molecular mechanisms of the cocaine-induced structural plasticity of nucleus Accumbens neurons. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2016. [cited 2019 Oct 23]. Available from: http://www.theses.fr/2016PA066378.

Council of Science Editors:

Dos Santos M. Dynamique et mécanismes moléculaires de la plasticité structurale des neurones du noyau Accumbens en réponse à la cocaïne : Dynamics and molecular mechanisms of the cocaine-induced structural plasticity of nucleus Accumbens neurons. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2016. Available from: http://www.theses.fr/2016PA066378


Université de Montréal

29. Gobeil, Mélanie P. Étude du rôle de la tyrosine kinase Src dans la régulation de la signalisation des récepteurs opioïdes delta (∆OR) .

Degree: 2009, Université de Montréal

 Les opioïdes sont les analgésiques les plus efficaces mais leur utilisation est limitée par la tolérance, un processus lié en partie à la désensibilisation des… (more)

Subjects/Keywords: Désensibilisation; Desensitization; récepteur mutant delta opioïde tronqué (DOR344T); truncated mutant delta opioid receptor (DOR344T); Src; PP2; DPDPE; Internalisation; Internalization; Tolérance; Tolerance; kinase activant les protéines mitogéniques (MAPK); mitogen-activated protein kinase (MAPK); kinase régulatrice de signaux extracellulaires (ERK); Extracellular Signal-Regulated Protein Kinase (ERK); Adénosine monophosphate cyclique (AMPc); cyclic adenosine monophosphate (cAMP)

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gobeil, M. P. (2009). Étude du rôle de la tyrosine kinase Src dans la régulation de la signalisation des récepteurs opioïdes delta (∆OR) . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/8851

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gobeil, Mélanie P. “Étude du rôle de la tyrosine kinase Src dans la régulation de la signalisation des récepteurs opioïdes delta (∆OR) .” 2009. Thesis, Université de Montréal. Accessed October 23, 2019. http://hdl.handle.net/1866/8851.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gobeil, Mélanie P. “Étude du rôle de la tyrosine kinase Src dans la régulation de la signalisation des récepteurs opioïdes delta (∆OR) .” 2009. Web. 23 Oct 2019.

Vancouver:

Gobeil MP. Étude du rôle de la tyrosine kinase Src dans la régulation de la signalisation des récepteurs opioïdes delta (∆OR) . [Internet] [Thesis]. Université de Montréal; 2009. [cited 2019 Oct 23]. Available from: http://hdl.handle.net/1866/8851.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gobeil MP. Étude du rôle de la tyrosine kinase Src dans la régulation de la signalisation des récepteurs opioïdes delta (∆OR) . [Thesis]. Université de Montréal; 2009. Available from: http://hdl.handle.net/1866/8851

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Alberta

30. Coccaro, Ersilia. Sustained acidosis and phenylephrine activate the myocardial Na+/H+ exchanger through phosphorylation of Ser770 and Ser771.

Degree: PhD, Department of Biochemistry, 2009, University of Alberta

 The mammalian Na+/H+ exchanger isoform 1 (NHE1) is a ubiquitously expressed membrane protein that regulates myocardial intracellular pH. Inhibition of NHE1 prevents hypertrophy and reduces… (more)

Subjects/Keywords: sustained acidosis; phenylephrine; myocardium; p90 risobomal S6 kinase; extracellular regulated kinase 1/2; sodium hydrogen exchanger

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Coccaro, E. (2009). Sustained acidosis and phenylephrine activate the myocardial Na+/H+ exchanger through phosphorylation of Ser770 and Ser771. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/m900nt79n

Chicago Manual of Style (16th Edition):

Coccaro, Ersilia. “Sustained acidosis and phenylephrine activate the myocardial Na+/H+ exchanger through phosphorylation of Ser770 and Ser771.” 2009. Doctoral Dissertation, University of Alberta. Accessed October 23, 2019. https://era.library.ualberta.ca/files/m900nt79n.

MLA Handbook (7th Edition):

Coccaro, Ersilia. “Sustained acidosis and phenylephrine activate the myocardial Na+/H+ exchanger through phosphorylation of Ser770 and Ser771.” 2009. Web. 23 Oct 2019.

Vancouver:

Coccaro E. Sustained acidosis and phenylephrine activate the myocardial Na+/H+ exchanger through phosphorylation of Ser770 and Ser771. [Internet] [Doctoral dissertation]. University of Alberta; 2009. [cited 2019 Oct 23]. Available from: https://era.library.ualberta.ca/files/m900nt79n.

Council of Science Editors:

Coccaro E. Sustained acidosis and phenylephrine activate the myocardial Na+/H+ exchanger through phosphorylation of Ser770 and Ser771. [Doctoral Dissertation]. University of Alberta; 2009. Available from: https://era.library.ualberta.ca/files/m900nt79n

[1] [2] [3] [4] [5] … [455]

.