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You searched for subject:( Epistasis Genetic 60). Showing records 1 – 30 of 17206 total matches.

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1. Banerjee, Samprit. Bayesian genome-wide QTL mapping for multiple traits.

Degree: PhD, 2008, University of Alabama – Birmingham

Identifying genetic loci responsible for variation in traits which are quantitative in nature poses a formidable challenge to geneticists and statisticians. We focus our attention… (more)

Subjects/Keywords: Bayes Theorem<; br>; Chromosome Mapping  – methods<; br>; Epistasis, Genetic<; br>; Models, Genetic<; br>; Quantitative Trait Loci<; br>

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Banerjee, S. (2008). Bayesian genome-wide QTL mapping for multiple traits. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,431

Chicago Manual of Style (16th Edition):

Banerjee, Samprit. “Bayesian genome-wide QTL mapping for multiple traits.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed August 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,431.

MLA Handbook (7th Edition):

Banerjee, Samprit. “Bayesian genome-wide QTL mapping for multiple traits.” 2008. Web. 20 Aug 2019.

Vancouver:

Banerjee S. Bayesian genome-wide QTL mapping for multiple traits. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Aug 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,431.

Council of Science Editors:

Banerjee S. Bayesian genome-wide QTL mapping for multiple traits. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,431


University of Oxford

2. Bell, Jordana Tzenova. Epistasis in complex human traits.

Degree: 2006, University of Oxford

Finally, two main extensions of this approach were considered - linkage approaches to examine more than two loci, and extending the method in this study to include a test of association.

Subjects/Keywords: 611.01816; Epistasis (Genetics) : Diabetes : Genetic aspects : Research

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APA (6th Edition):

Bell, J. T. (2006). Epistasis in complex human traits. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:547db446-c84c-4a6c-8b5c-ce960f7765c5 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.432552

Chicago Manual of Style (16th Edition):

Bell, Jordana Tzenova. “Epistasis in complex human traits.” 2006. Doctoral Dissertation, University of Oxford. Accessed August 20, 2019. http://ora.ox.ac.uk/objects/uuid:547db446-c84c-4a6c-8b5c-ce960f7765c5 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.432552.

MLA Handbook (7th Edition):

Bell, Jordana Tzenova. “Epistasis in complex human traits.” 2006. Web. 20 Aug 2019.

Vancouver:

Bell JT. Epistasis in complex human traits. [Internet] [Doctoral dissertation]. University of Oxford; 2006. [cited 2019 Aug 20]. Available from: http://ora.ox.ac.uk/objects/uuid:547db446-c84c-4a6c-8b5c-ce960f7765c5 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.432552.

Council of Science Editors:

Bell JT. Epistasis in complex human traits. [Doctoral Dissertation]. University of Oxford; 2006. Available from: http://ora.ox.ac.uk/objects/uuid:547db446-c84c-4a6c-8b5c-ce960f7765c5 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.432552


University of Texas Southwestern Medical Center

3. Belobrajdic, Katherine Ann. Narrowing of the SLES1 Internal Reveals Complex Epistatic Interactions in the Suppression of Autoimmunity.

Degree: 2010, University of Texas Southwestern Medical Center

 Sle1 is a potent susceptibility locus for spontaneous systemic autoimmunity derived from the NZM2410 mouse strain. The NZW-derived suppressive modifier locus, Sles1, specifically prevents the… (more)

Subjects/Keywords: Lupus Erythematosus, Systemic; Epistasis, Genetic; Mice

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APA (6th Edition):

Belobrajdic, K. A. (2010). Narrowing of the SLES1 Internal Reveals Complex Epistatic Interactions in the Suppression of Autoimmunity. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/240

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Belobrajdic, Katherine Ann. “Narrowing of the SLES1 Internal Reveals Complex Epistatic Interactions in the Suppression of Autoimmunity.” 2010. Thesis, University of Texas Southwestern Medical Center. Accessed August 20, 2019. http://hdl.handle.net/2152.5/240.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Belobrajdic, Katherine Ann. “Narrowing of the SLES1 Internal Reveals Complex Epistatic Interactions in the Suppression of Autoimmunity.” 2010. Web. 20 Aug 2019.

Vancouver:

Belobrajdic KA. Narrowing of the SLES1 Internal Reveals Complex Epistatic Interactions in the Suppression of Autoimmunity. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2010. [cited 2019 Aug 20]. Available from: http://hdl.handle.net/2152.5/240.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Belobrajdic KA. Narrowing of the SLES1 Internal Reveals Complex Epistatic Interactions in the Suppression of Autoimmunity. [Thesis]. University of Texas Southwestern Medical Center; 2010. Available from: http://hdl.handle.net/2152.5/240

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Univerzitet u Beogradu

4. Grčić, Nikola M., 1981-. Genetička analiza nasleđivanja kvantitativnih osobina kukuruza primenom metoda dialela i generacijskih proseka.

Degree: Poljoprivredni fakultet, 2016, Univerzitet u Beogradu

BIOTEHNIČKE NAUKE - RATARSTVO I POVRTARSTVO / BIOTECHNICAL SCIENCES - FIELD AND VEGETABLE CROPS

U ovom istraživanju ispitivana je genetička osnova nasleđivanja važnih agronomskih osobina… (more)

Subjects/Keywords: diallel; combinig ability; genetic variance; epistasis; generation mean; genetic distance

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Grčić, Nikola M., 1. (2016). Genetička analiza nasleđivanja kvantitativnih osobina kukuruza primenom metoda dialela i generacijskih proseka. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:12785/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Grčić, Nikola M., 1981-. “Genetička analiza nasleđivanja kvantitativnih osobina kukuruza primenom metoda dialela i generacijskih proseka.” 2016. Thesis, Univerzitet u Beogradu. Accessed August 20, 2019. https://fedorabg.bg.ac.rs/fedora/get/o:12785/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Grčić, Nikola M., 1981-. “Genetička analiza nasleđivanja kvantitativnih osobina kukuruza primenom metoda dialela i generacijskih proseka.” 2016. Web. 20 Aug 2019.

Vancouver:

Grčić, Nikola M. 1. Genetička analiza nasleđivanja kvantitativnih osobina kukuruza primenom metoda dialela i generacijskih proseka. [Internet] [Thesis]. Univerzitet u Beogradu; 2016. [cited 2019 Aug 20]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:12785/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Grčić, Nikola M. 1. Genetička analiza nasleđivanja kvantitativnih osobina kukuruza primenom metoda dialela i generacijskih proseka. [Thesis]. Univerzitet u Beogradu; 2016. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:12785/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

5. Oliveira, Paulo Tadeu Meira e Silva de. Aplicação do algorítmo genético no mapeamento de genes epistáticos em cruzamentos controlados.

Degree: PhD, Estatística, 2008, University of São Paulo

O mapeamento genético é constituído por procedimentos experimentais e estatísticos que buscam detectar genes associados à etiologia e regulação de doenças, além de estimar os… (more)

Subjects/Keywords: algoritmo genético; epistasia; epistasis; genetic algorithms; model selection; seleção de modelos

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Oliveira, P. T. M. e. S. d. (2008). Aplicação do algorítmo genético no mapeamento de genes epistáticos em cruzamentos controlados. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/45/45133/tde-29092008-110907/ ;

Chicago Manual of Style (16th Edition):

Oliveira, Paulo Tadeu Meira e Silva de. “Aplicação do algorítmo genético no mapeamento de genes epistáticos em cruzamentos controlados.” 2008. Doctoral Dissertation, University of São Paulo. Accessed August 20, 2019. http://www.teses.usp.br/teses/disponiveis/45/45133/tde-29092008-110907/ ;.

MLA Handbook (7th Edition):

Oliveira, Paulo Tadeu Meira e Silva de. “Aplicação do algorítmo genético no mapeamento de genes epistáticos em cruzamentos controlados.” 2008. Web. 20 Aug 2019.

Vancouver:

Oliveira PTMeSd. Aplicação do algorítmo genético no mapeamento de genes epistáticos em cruzamentos controlados. [Internet] [Doctoral dissertation]. University of São Paulo; 2008. [cited 2019 Aug 20]. Available from: http://www.teses.usp.br/teses/disponiveis/45/45133/tde-29092008-110907/ ;.

Council of Science Editors:

Oliveira PTMeSd. Aplicação do algorítmo genético no mapeamento de genes epistáticos em cruzamentos controlados. [Doctoral Dissertation]. University of São Paulo; 2008. Available from: http://www.teses.usp.br/teses/disponiveis/45/45133/tde-29092008-110907/ ;


University of Kansas

6. Monnahan, Patrick Joseph. EVOLUTIONARY IMPLICATIONS OF THE STANDING GENETIC VARIATION IN NATURAL POPULATIONS OF MIMULUS GUTTATUS.

Degree: PhD, Ecology & Evolutionary Biology, 2016, University of Kansas

 The standing genetic variation present in a population is the raw material on which natural selection can act. The study of the architecture of this… (more)

Subjects/Keywords: Evolution & development; Genetics; Plant sciences; epistasis; genetic architecture; Mimulus; selection

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Monnahan, P. J. (2016). EVOLUTIONARY IMPLICATIONS OF THE STANDING GENETIC VARIATION IN NATURAL POPULATIONS OF MIMULUS GUTTATUS. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/25744

Chicago Manual of Style (16th Edition):

Monnahan, Patrick Joseph. “EVOLUTIONARY IMPLICATIONS OF THE STANDING GENETIC VARIATION IN NATURAL POPULATIONS OF MIMULUS GUTTATUS.” 2016. Doctoral Dissertation, University of Kansas. Accessed August 20, 2019. http://hdl.handle.net/1808/25744.

MLA Handbook (7th Edition):

Monnahan, Patrick Joseph. “EVOLUTIONARY IMPLICATIONS OF THE STANDING GENETIC VARIATION IN NATURAL POPULATIONS OF MIMULUS GUTTATUS.” 2016. Web. 20 Aug 2019.

Vancouver:

Monnahan PJ. EVOLUTIONARY IMPLICATIONS OF THE STANDING GENETIC VARIATION IN NATURAL POPULATIONS OF MIMULUS GUTTATUS. [Internet] [Doctoral dissertation]. University of Kansas; 2016. [cited 2019 Aug 20]. Available from: http://hdl.handle.net/1808/25744.

Council of Science Editors:

Monnahan PJ. EVOLUTIONARY IMPLICATIONS OF THE STANDING GENETIC VARIATION IN NATURAL POPULATIONS OF MIMULUS GUTTATUS. [Doctoral Dissertation]. University of Kansas; 2016. Available from: http://hdl.handle.net/1808/25744


University of Sydney

7. Ali, Abdirahman. Quantitative genetics and genomics of production and disease in beef cattle .

Degree: 2013, University of Sydney

 Cattle grazing in extensive open pasture of tropical areas are subjected to several environmental stressors such as heat and humidity, high incidence of disease, cattle… (more)

Subjects/Keywords: Beef cattle; Disease resistance; Epistasis; Genetic markers; Production traits; Quantitative genetics

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APA (6th Edition):

Ali, A. (2013). Quantitative genetics and genomics of production and disease in beef cattle . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/12296

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ali, Abdirahman. “Quantitative genetics and genomics of production and disease in beef cattle .” 2013. Thesis, University of Sydney. Accessed August 20, 2019. http://hdl.handle.net/2123/12296.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ali, Abdirahman. “Quantitative genetics and genomics of production and disease in beef cattle .” 2013. Web. 20 Aug 2019.

Vancouver:

Ali A. Quantitative genetics and genomics of production and disease in beef cattle . [Internet] [Thesis]. University of Sydney; 2013. [cited 2019 Aug 20]. Available from: http://hdl.handle.net/2123/12296.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ali A. Quantitative genetics and genomics of production and disease in beef cattle . [Thesis]. University of Sydney; 2013. Available from: http://hdl.handle.net/2123/12296

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

8. Morais, Alexandre Augusto de. Epistasia na herança da resistência do milho ao gorgulho Sitophilus zeamais (Coleoptera: Curculionidae).

Degree: PhD, Genética e Melhoramento de Plantas, 2012, University of São Paulo

Considerado um dos aspectos mais complexos da genética quantitativa, a epistasia tem sido ignorada pelos melhoristas nos estudos de herança dos caracteres, principalmente os da… (more)

Subjects/Keywords: Epistasia; Epistasis; Gorgulhos; Maize; Milho; Plant Genetic Resistance; Resistência genética vegetal; Weevils

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Morais, A. A. d. (2012). Epistasia na herança da resistência do milho ao gorgulho Sitophilus zeamais (Coleoptera: Curculionidae). (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/11/11137/tde-18092012-103000/ ;

Chicago Manual of Style (16th Edition):

Morais, Alexandre Augusto de. “Epistasia na herança da resistência do milho ao gorgulho Sitophilus zeamais (Coleoptera: Curculionidae).” 2012. Doctoral Dissertation, University of São Paulo. Accessed August 20, 2019. http://www.teses.usp.br/teses/disponiveis/11/11137/tde-18092012-103000/ ;.

MLA Handbook (7th Edition):

Morais, Alexandre Augusto de. “Epistasia na herança da resistência do milho ao gorgulho Sitophilus zeamais (Coleoptera: Curculionidae).” 2012. Web. 20 Aug 2019.

Vancouver:

Morais AAd. Epistasia na herança da resistência do milho ao gorgulho Sitophilus zeamais (Coleoptera: Curculionidae). [Internet] [Doctoral dissertation]. University of São Paulo; 2012. [cited 2019 Aug 20]. Available from: http://www.teses.usp.br/teses/disponiveis/11/11137/tde-18092012-103000/ ;.

Council of Science Editors:

Morais AAd. Epistasia na herança da resistência do milho ao gorgulho Sitophilus zeamais (Coleoptera: Curculionidae). [Doctoral Dissertation]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/11/11137/tde-18092012-103000/ ;

9. Hallin, Johan Henning. Élucider les facteurs génétiques à l'origine de la variabilité des populations par phénomique et génomique de masse : Elucidating the genetic basis of variation in populations by large scale phenomics and genomics.

Degree: Docteur es, Biologie des interactions et écologie, 2018, Côte d'Azur

La variabilité phénotypique existante au sein d’une population est d’une importance cruciale ; elle permet l’adaptation à de nouvelles conditions par la sélection naturelle de… (more)

Subjects/Keywords: Caractères quantitatifs; Variation génétique; Épistasie; Hétérosis; Prédictions; Quantitative traits; Genetic variation; Epistasis; Heterosis; Predictions

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hallin, J. H. (2018). Élucider les facteurs génétiques à l'origine de la variabilité des populations par phénomique et génomique de masse : Elucidating the genetic basis of variation in populations by large scale phenomics and genomics. (Doctoral Dissertation). Côte d'Azur. Retrieved from http://www.theses.fr/2018AZUR4010

Chicago Manual of Style (16th Edition):

Hallin, Johan Henning. “Élucider les facteurs génétiques à l'origine de la variabilité des populations par phénomique et génomique de masse : Elucidating the genetic basis of variation in populations by large scale phenomics and genomics.” 2018. Doctoral Dissertation, Côte d'Azur. Accessed August 20, 2019. http://www.theses.fr/2018AZUR4010.

MLA Handbook (7th Edition):

Hallin, Johan Henning. “Élucider les facteurs génétiques à l'origine de la variabilité des populations par phénomique et génomique de masse : Elucidating the genetic basis of variation in populations by large scale phenomics and genomics.” 2018. Web. 20 Aug 2019.

Vancouver:

Hallin JH. Élucider les facteurs génétiques à l'origine de la variabilité des populations par phénomique et génomique de masse : Elucidating the genetic basis of variation in populations by large scale phenomics and genomics. [Internet] [Doctoral dissertation]. Côte d'Azur; 2018. [cited 2019 Aug 20]. Available from: http://www.theses.fr/2018AZUR4010.

Council of Science Editors:

Hallin JH. Élucider les facteurs génétiques à l'origine de la variabilité des populations par phénomique et génomique de masse : Elucidating the genetic basis of variation in populations by large scale phenomics and genomics. [Doctoral Dissertation]. Côte d'Azur; 2018. Available from: http://www.theses.fr/2018AZUR4010


UCLA

10. Rodriguez, Alejandra. Integrating molecular phenotypes and gene expression to characterize DNA variants for cardiometabolic traits.

Degree: Human Genetics, 2018, UCLA

 ABSTRACT OF THE DISSERTATIONIntegrating molecular phenotypes and gene expression to characterize DNA variants for cardiometabolic traitsbyAlejandra RodriguezDoctor of Philosophy in Human GeneticsUniversity of California, Los… (more)

Subjects/Keywords: Genetics; Bioinformatics; Molecular biology; Canalization; Epistasis; Gene regulation; Genetic architecture; GWAS; RNA-Seq

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rodriguez, A. (2018). Integrating molecular phenotypes and gene expression to characterize DNA variants for cardiometabolic traits. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/5bb221xm

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rodriguez, Alejandra. “Integrating molecular phenotypes and gene expression to characterize DNA variants for cardiometabolic traits.” 2018. Thesis, UCLA. Accessed August 20, 2019. http://www.escholarship.org/uc/item/5bb221xm.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rodriguez, Alejandra. “Integrating molecular phenotypes and gene expression to characterize DNA variants for cardiometabolic traits.” 2018. Web. 20 Aug 2019.

Vancouver:

Rodriguez A. Integrating molecular phenotypes and gene expression to characterize DNA variants for cardiometabolic traits. [Internet] [Thesis]. UCLA; 2018. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/5bb221xm.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rodriguez A. Integrating molecular phenotypes and gene expression to characterize DNA variants for cardiometabolic traits. [Thesis]. UCLA; 2018. Available from: http://www.escholarship.org/uc/item/5bb221xm

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Ottawa

11. Phenix, Hilary. Derivation and Use of Gene Network Models to Make Quantitative Predictions of Genetic Interaction Data .

Degree: 2017, University of Ottawa

 This thesis investigates how pairwise combinatorial gene and stimulus perturbation experiments are conducted and interpreted. In particular, I investigate gene perturbation in the form of… (more)

Subjects/Keywords: Epistasis; DNA damage checkpoint; Genetic interaction; Gene regulatory network inference; Methyl methanesulfonate

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Phenix, H. (2017). Derivation and Use of Gene Network Models to Make Quantitative Predictions of Genetic Interaction Data . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/37031

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Phenix, Hilary. “Derivation and Use of Gene Network Models to Make Quantitative Predictions of Genetic Interaction Data .” 2017. Thesis, University of Ottawa. Accessed August 20, 2019. http://hdl.handle.net/10393/37031.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Phenix, Hilary. “Derivation and Use of Gene Network Models to Make Quantitative Predictions of Genetic Interaction Data .” 2017. Web. 20 Aug 2019.

Vancouver:

Phenix H. Derivation and Use of Gene Network Models to Make Quantitative Predictions of Genetic Interaction Data . [Internet] [Thesis]. University of Ottawa; 2017. [cited 2019 Aug 20]. Available from: http://hdl.handle.net/10393/37031.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Phenix H. Derivation and Use of Gene Network Models to Make Quantitative Predictions of Genetic Interaction Data . [Thesis]. University of Ottawa; 2017. Available from: http://hdl.handle.net/10393/37031

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

12. Verma, Shefali. INVESTIGATING COMPUTATIONAL METHODS TO MODEL THE GENOTYPIC AND PHENOTYPIC COMPLEXITY OF ADVERSE HEALTH OUTCOMES: UNDERSTANDING UNDERCOVER HERITABILITY.

Degree: 2018, Penn State University

 Genome-wide association studies (GWAS) are the most popular and widely conducted experiments to understand the genetic architecture of common diseases. Though GWAS have been successful… (more)

Subjects/Keywords: Association Studies; GWAS; Rare Variants; Common Variants; Epistasis; Genetic Etiology; Complex Traits; Heritability

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Verma, S. (2018). INVESTIGATING COMPUTATIONAL METHODS TO MODEL THE GENOTYPIC AND PHENOTYPIC COMPLEXITY OF ADVERSE HEALTH OUTCOMES: UNDERSTANDING UNDERCOVER HERITABILITY. (Thesis). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/15006szs14

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Verma, Shefali. “INVESTIGATING COMPUTATIONAL METHODS TO MODEL THE GENOTYPIC AND PHENOTYPIC COMPLEXITY OF ADVERSE HEALTH OUTCOMES: UNDERSTANDING UNDERCOVER HERITABILITY.” 2018. Thesis, Penn State University. Accessed August 20, 2019. https://etda.libraries.psu.edu/catalog/15006szs14.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Verma, Shefali. “INVESTIGATING COMPUTATIONAL METHODS TO MODEL THE GENOTYPIC AND PHENOTYPIC COMPLEXITY OF ADVERSE HEALTH OUTCOMES: UNDERSTANDING UNDERCOVER HERITABILITY.” 2018. Web. 20 Aug 2019.

Vancouver:

Verma S. INVESTIGATING COMPUTATIONAL METHODS TO MODEL THE GENOTYPIC AND PHENOTYPIC COMPLEXITY OF ADVERSE HEALTH OUTCOMES: UNDERSTANDING UNDERCOVER HERITABILITY. [Internet] [Thesis]. Penn State University; 2018. [cited 2019 Aug 20]. Available from: https://etda.libraries.psu.edu/catalog/15006szs14.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Verma S. INVESTIGATING COMPUTATIONAL METHODS TO MODEL THE GENOTYPIC AND PHENOTYPIC COMPLEXITY OF ADVERSE HEALTH OUTCOMES: UNDERSTANDING UNDERCOVER HERITABILITY. [Thesis]. Penn State University; 2018. Available from: https://etda.libraries.psu.edu/catalog/15006szs14

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

13. Rünneburger, Estelle. Évolution de la canalisation génétique dans un modèle quantitatif de réseau de régulation : Evolution of genetic canalization in a quantitative model of gene regulatory networks.

Degree: Docteur es, Sciences de la vie et de la santé, 2016, Paris Saclay

La canalisation génétique est définie comme la capacité d’un organisme à avoir un développement constant en dépit des mutations qui l’affectent. A l’heure actuelle, trois… (more)

Subjects/Keywords: Architecture génétique; Génétique quantitative; Modélisation; Épistasie; Genetic architecture; Quantitative genetics; Modelling; Epistasis

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APA (6th Edition):

Rünneburger, E. (2016). Évolution de la canalisation génétique dans un modèle quantitatif de réseau de régulation : Evolution of genetic canalization in a quantitative model of gene regulatory networks. (Doctoral Dissertation). Paris Saclay. Retrieved from http://www.theses.fr/2016SACLS547

Chicago Manual of Style (16th Edition):

Rünneburger, Estelle. “Évolution de la canalisation génétique dans un modèle quantitatif de réseau de régulation : Evolution of genetic canalization in a quantitative model of gene regulatory networks.” 2016. Doctoral Dissertation, Paris Saclay. Accessed August 20, 2019. http://www.theses.fr/2016SACLS547.

MLA Handbook (7th Edition):

Rünneburger, Estelle. “Évolution de la canalisation génétique dans un modèle quantitatif de réseau de régulation : Evolution of genetic canalization in a quantitative model of gene regulatory networks.” 2016. Web. 20 Aug 2019.

Vancouver:

Rünneburger E. Évolution de la canalisation génétique dans un modèle quantitatif de réseau de régulation : Evolution of genetic canalization in a quantitative model of gene regulatory networks. [Internet] [Doctoral dissertation]. Paris Saclay; 2016. [cited 2019 Aug 20]. Available from: http://www.theses.fr/2016SACLS547.

Council of Science Editors:

Rünneburger E. Évolution de la canalisation génétique dans un modèle quantitatif de réseau de régulation : Evolution of genetic canalization in a quantitative model of gene regulatory networks. [Doctoral Dissertation]. Paris Saclay; 2016. Available from: http://www.theses.fr/2016SACLS547


University of California – San Francisco

14. Breslow, David King. Development of a High-Resolution Growth Assay for Yeast Functional Genomics and Identification of Orm Family Proteins as Key Mediators of Sphingolipid Homeostasis.

Degree: Chemistry and Chemical Biology, 2010, University of California – San Francisco

 Advances in DNA sequencing have provided the complete genome sequence for many organisms. However, understanding the functions of each gene product encoded by the genome… (more)

Subjects/Keywords: Biology, Cell; Biology, Genetics; epistasis; functional genomics; genetic interaction; homeostasis; ORM; sphingolipid

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APA (6th Edition):

Breslow, D. K. (2010). Development of a High-Resolution Growth Assay for Yeast Functional Genomics and Identification of Orm Family Proteins as Key Mediators of Sphingolipid Homeostasis. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/6ds216x8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Breslow, David King. “Development of a High-Resolution Growth Assay for Yeast Functional Genomics and Identification of Orm Family Proteins as Key Mediators of Sphingolipid Homeostasis.” 2010. Thesis, University of California – San Francisco. Accessed August 20, 2019. http://www.escholarship.org/uc/item/6ds216x8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Breslow, David King. “Development of a High-Resolution Growth Assay for Yeast Functional Genomics and Identification of Orm Family Proteins as Key Mediators of Sphingolipid Homeostasis.” 2010. Web. 20 Aug 2019.

Vancouver:

Breslow DK. Development of a High-Resolution Growth Assay for Yeast Functional Genomics and Identification of Orm Family Proteins as Key Mediators of Sphingolipid Homeostasis. [Internet] [Thesis]. University of California – San Francisco; 2010. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/6ds216x8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Breslow DK. Development of a High-Resolution Growth Assay for Yeast Functional Genomics and Identification of Orm Family Proteins as Key Mediators of Sphingolipid Homeostasis. [Thesis]. University of California – San Francisco; 2010. Available from: http://www.escholarship.org/uc/item/6ds216x8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. Mitchell, Natalie E. Genome-wide DNA methylation changes during breast tumorigenesis.

Degree: MS, 2012, University of Alabama – Birmingham

Neoplastic reprogramming generates cancerous cells from normal derivatives, enabling researchers to create a model of the early stages of oncogenesis and analyze molecular changes in… (more)

Subjects/Keywords: Breast – Cancer – Genetic aspects<; br>; DNA – Methylation<; br>; Carcinogenesis<; br>; Breast – Precancerous conditions – Genetic aspects

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APA (6th Edition):

Mitchell, N. E. (2012). Genome-wide DNA methylation changes during breast tumorigenesis. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1309

Chicago Manual of Style (16th Edition):

Mitchell, Natalie E. “Genome-wide DNA methylation changes during breast tumorigenesis.” 2012. Masters Thesis, University of Alabama – Birmingham. Accessed August 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1309.

MLA Handbook (7th Edition):

Mitchell, Natalie E. “Genome-wide DNA methylation changes during breast tumorigenesis.” 2012. Web. 20 Aug 2019.

Vancouver:

Mitchell NE. Genome-wide DNA methylation changes during breast tumorigenesis. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2012. [cited 2019 Aug 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1309.

Council of Science Editors:

Mitchell NE. Genome-wide DNA methylation changes during breast tumorigenesis. [Masters Thesis]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1309

16. Booker, Betty M. Analyzing DNA topology and transcription in Salmonella enterica serovar Typhimurium during dichotomous growth.

Degree: PhD, 2010, University of Alabama – Birmingham

The bacterial chromosome is dynamic. The principle goal of my research is to understand how DNA topology is altered by transcription in Salmonella enterica serovar… (more)

Subjects/Keywords: Chromosomes<; br>; DNA<; br>; RNA, Ribosomal<; br>; Salmonella typhimurium  – genetics<; br>; Transcription, Genetic

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APA (6th Edition):

Booker, B. M. (2010). Analyzing DNA topology and transcription in Salmonella enterica serovar Typhimurium during dichotomous growth. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,699

Chicago Manual of Style (16th Edition):

Booker, Betty M. “Analyzing DNA topology and transcription in Salmonella enterica serovar Typhimurium during dichotomous growth.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed August 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,699.

MLA Handbook (7th Edition):

Booker, Betty M. “Analyzing DNA topology and transcription in Salmonella enterica serovar Typhimurium during dichotomous growth.” 2010. Web. 20 Aug 2019.

Vancouver:

Booker BM. Analyzing DNA topology and transcription in Salmonella enterica serovar Typhimurium during dichotomous growth. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Aug 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,699.

Council of Science Editors:

Booker BM. Analyzing DNA topology and transcription in Salmonella enterica serovar Typhimurium during dichotomous growth. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,699

17. Phipps, Sharla Marion Ostein. Genetic and epigenetic modulation of telomerase activity in development and disease.

Degree: PhD, 2007, University of Alabama – Birmingham

The end replication problem of linear chromosomes leads to the erosion of telomeric DNA with each cell division. A mechanism to counteract telomeric attrition involves… (more)

Subjects/Keywords: Telomerase  – Physiological effect <; br>; Telomerase  – Inhibitors <; br>; Genetic regulation <; br>; Enzymes  – Regulation <; br>; Genetic transcription <; br>; Reverse transcriptase <; br>; Breast  – Cancer  – Genetic aspects <; br>; Tretinoin  – Physiological effect <; br>; Epigenesis.

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APA (6th Edition):

Phipps, S. M. O. (2007). Genetic and epigenetic modulation of telomerase activity in development and disease. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,260

Chicago Manual of Style (16th Edition):

Phipps, Sharla Marion Ostein. “Genetic and epigenetic modulation of telomerase activity in development and disease.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed August 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,260.

MLA Handbook (7th Edition):

Phipps, Sharla Marion Ostein. “Genetic and epigenetic modulation of telomerase activity in development and disease.” 2007. Web. 20 Aug 2019.

Vancouver:

Phipps SMO. Genetic and epigenetic modulation of telomerase activity in development and disease. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Aug 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,260.

Council of Science Editors:

Phipps SMO. Genetic and epigenetic modulation of telomerase activity in development and disease. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,260

18. Dupraw, Diana Lee. Evolution and genetic diversity in streptococcus pneumoniae.

Degree: PhD, 2010, University of Alabama – Birmingham

The relative contributions of point mutations and intergenomic recombination via lateral gene transfer (LGT) determine the population structure and evolutionary style for a given bacterial… (more)

Subjects/Keywords: Evolution, Molecular<; br>; Gene Transfer, Horizontal<; br>; Genetic Variation<; br>; Pneumonia, Pneumococcal  – microbiology<; br>; Recombination, Genetic<; br>; Streptococcus pneumoniae  – genetics

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APA (6th Edition):

Dupraw, D. L. (2010). Evolution and genetic diversity in streptococcus pneumoniae. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1000

Chicago Manual of Style (16th Edition):

Dupraw, Diana Lee. “Evolution and genetic diversity in streptococcus pneumoniae.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed August 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1000.

MLA Handbook (7th Edition):

Dupraw, Diana Lee. “Evolution and genetic diversity in streptococcus pneumoniae.” 2010. Web. 20 Aug 2019.

Vancouver:

Dupraw DL. Evolution and genetic diversity in streptococcus pneumoniae. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Aug 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1000.

Council of Science Editors:

Dupraw DL. Evolution and genetic diversity in streptococcus pneumoniae. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1000

19. Silva, Diego Velásquez Faleiro e. Epistasia em testecrosses de milho.

Degree: PhD, Genética e Melhoramento de Plantas, 2011, University of São Paulo

A epistasia já é conhecida desde o início dos estudos em genética, porém sua contribuição para as estimativas dos componentes da variância genética e para… (more)

Subjects/Keywords: Cruzamento vegetal - Testes; Epistasia; Epistasis; Genetic variance in plants.; Maize; Milho; Plant cross tests; Variação genética em plantas.

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APA (6th Edition):

Silva, D. V. F. e. (2011). Epistasia em testecrosses de milho. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/11/11137/tde-13092011-081029/ ;

Chicago Manual of Style (16th Edition):

Silva, Diego Velásquez Faleiro e. “Epistasia em testecrosses de milho.” 2011. Doctoral Dissertation, University of São Paulo. Accessed August 20, 2019. http://www.teses.usp.br/teses/disponiveis/11/11137/tde-13092011-081029/ ;.

MLA Handbook (7th Edition):

Silva, Diego Velásquez Faleiro e. “Epistasia em testecrosses de milho.” 2011. Web. 20 Aug 2019.

Vancouver:

Silva DVFe. Epistasia em testecrosses de milho. [Internet] [Doctoral dissertation]. University of São Paulo; 2011. [cited 2019 Aug 20]. Available from: http://www.teses.usp.br/teses/disponiveis/11/11137/tde-13092011-081029/ ;.

Council of Science Editors:

Silva DVFe. Epistasia em testecrosses de milho. [Doctoral Dissertation]. University of São Paulo; 2011. Available from: http://www.teses.usp.br/teses/disponiveis/11/11137/tde-13092011-081029/ ;


University of Michigan

20. Wei, Xinzhu. Genetic Interactions and Gene-by-Environment Interactions in Evolution.

Degree: PhD, Ecology and Evolutionary Biology, 2018, University of Michigan

 The phenotypic effect of a mutation depends on both genetic interactions (G×G) and gene-by-environment interactions (G×E). G×G and G×E can distort the additive relationship between… (more)

Subjects/Keywords: gene by environment interaction; genetic interaction; dominance; diminishing returns epistasis; heterosis; pleiotropy; Ecology and Evolutionary Biology; Genetics; Science

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APA (6th Edition):

Wei, X. (2018). Genetic Interactions and Gene-by-Environment Interactions in Evolution. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/144160

Chicago Manual of Style (16th Edition):

Wei, Xinzhu. “Genetic Interactions and Gene-by-Environment Interactions in Evolution.” 2018. Doctoral Dissertation, University of Michigan. Accessed August 20, 2019. http://hdl.handle.net/2027.42/144160.

MLA Handbook (7th Edition):

Wei, Xinzhu. “Genetic Interactions and Gene-by-Environment Interactions in Evolution.” 2018. Web. 20 Aug 2019.

Vancouver:

Wei X. Genetic Interactions and Gene-by-Environment Interactions in Evolution. [Internet] [Doctoral dissertation]. University of Michigan; 2018. [cited 2019 Aug 20]. Available from: http://hdl.handle.net/2027.42/144160.

Council of Science Editors:

Wei X. Genetic Interactions and Gene-by-Environment Interactions in Evolution. [Doctoral Dissertation]. University of Michigan; 2018. Available from: http://hdl.handle.net/2027.42/144160

21. Rio, Simon. Contributions to genomic selection and association mapping in structured and admixed populations : application to maize : Contributions à la sélection génomique et à la génétique d'association en populations structurées et admixées : application au maïs.

Degree: Docteur es, Sciences agronomiques, 2019, Paris Saclay

 L'essor des marqueurs moléculaires (SNPs) a révolutionné les méthodes de génétique quantitative en permettant l'identification de régions impliquées dans le déterminisme génétique des caractères (QTLs)… (more)

Subjects/Keywords: Admixture; Coefficient de Détermination (CD); Prédiction génomique; Structure génétique; Gwas; Epistasie; Admixture; Coefficient of Determination; Genomic Prediction; Genetic Structure; Gwas; Epistasis

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APA (6th Edition):

Rio, S. (2019). Contributions to genomic selection and association mapping in structured and admixed populations : application to maize : Contributions à la sélection génomique et à la génétique d'association en populations structurées et admixées : application au maïs. (Doctoral Dissertation). Paris Saclay. Retrieved from http://www.theses.fr/2019SACLS097

Chicago Manual of Style (16th Edition):

Rio, Simon. “Contributions to genomic selection and association mapping in structured and admixed populations : application to maize : Contributions à la sélection génomique et à la génétique d'association en populations structurées et admixées : application au maïs.” 2019. Doctoral Dissertation, Paris Saclay. Accessed August 20, 2019. http://www.theses.fr/2019SACLS097.

MLA Handbook (7th Edition):

Rio, Simon. “Contributions to genomic selection and association mapping in structured and admixed populations : application to maize : Contributions à la sélection génomique et à la génétique d'association en populations structurées et admixées : application au maïs.” 2019. Web. 20 Aug 2019.

Vancouver:

Rio S. Contributions to genomic selection and association mapping in structured and admixed populations : application to maize : Contributions à la sélection génomique et à la génétique d'association en populations structurées et admixées : application au maïs. [Internet] [Doctoral dissertation]. Paris Saclay; 2019. [cited 2019 Aug 20]. Available from: http://www.theses.fr/2019SACLS097.

Council of Science Editors:

Rio S. Contributions to genomic selection and association mapping in structured and admixed populations : application to maize : Contributions à la sélection génomique et à la génétique d'association en populations structurées et admixées : application au maïs. [Doctoral Dissertation]. Paris Saclay; 2019. Available from: http://www.theses.fr/2019SACLS097

22. Guo, Jingyu. A Yeast Model For High Throughput Screening Of Gene-Gene Interactions Relevant To Cftr-Δf508 Protein Biogenesis.

Degree: 2012, University of Alabama – Birmingham

Much remains unknown about gene interaction in the context of human disease. In cystic fibrosis (CF) a single mutation of the cystic fibrosis transmembrane conductance… (more)

Subjects/Keywords: Cluster Analysis<; br>; Data Mining.<; br>; Epistasis, Genetic.<; br>; Gene Regulatory Networks.<; br>; Genetic Association Studies.<; br>; Models, Genetic

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APA (6th Edition):

Guo, J. (2012). A Yeast Model For High Throughput Screening Of Gene-Gene Interactions Relevant To Cftr-Δf508 Protein Biogenesis. (Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1799

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Guo, Jingyu. “A Yeast Model For High Throughput Screening Of Gene-Gene Interactions Relevant To Cftr-Δf508 Protein Biogenesis.” 2012. Thesis, University of Alabama – Birmingham. Accessed August 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1799.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Guo, Jingyu. “A Yeast Model For High Throughput Screening Of Gene-Gene Interactions Relevant To Cftr-Δf508 Protein Biogenesis.” 2012. Web. 20 Aug 2019.

Vancouver:

Guo J. A Yeast Model For High Throughput Screening Of Gene-Gene Interactions Relevant To Cftr-Δf508 Protein Biogenesis. [Internet] [Thesis]. University of Alabama – Birmingham; 2012. [cited 2019 Aug 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1799.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Guo J. A Yeast Model For High Throughput Screening Of Gene-Gene Interactions Relevant To Cftr-Δf508 Protein Biogenesis. [Thesis]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1799

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. Heath, Cara Hope. Determinants that confer stop codon specificity to Tetrahymena thermophila eRF1.

Degree: MS, 2007, University of Alabama – Birmingham

In eukaryotes, translation termination is a process which is initiated by the presence of a stop codon in the A site of the ribosome and… (more)

Subjects/Keywords: Tetrahymena  – Genetics <; br>; Saccharomyces cerevisiae  – Genetics <; br>; Genetic translation.

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APA (6th Edition):

Heath, C. H. (2007). Determinants that confer stop codon specificity to Tetrahymena thermophila eRF1. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,77

Chicago Manual of Style (16th Edition):

Heath, Cara Hope. “Determinants that confer stop codon specificity to Tetrahymena thermophila eRF1.” 2007. Masters Thesis, University of Alabama – Birmingham. Accessed August 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,77.

MLA Handbook (7th Edition):

Heath, Cara Hope. “Determinants that confer stop codon specificity to Tetrahymena thermophila eRF1.” 2007. Web. 20 Aug 2019.

Vancouver:

Heath CH. Determinants that confer stop codon specificity to Tetrahymena thermophila eRF1. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2007. [cited 2019 Aug 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,77.

Council of Science Editors:

Heath CH. Determinants that confer stop codon specificity to Tetrahymena thermophila eRF1. [Masters Thesis]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,77

24. Blaize, Marie Antionette. Degradation of the Huntington polyglutamine domain by the proteasome : implication to Huntington's disease.

Degree: MS, 2008, University of Alabama – Birmingham

Huntington’s disease (HD) is a progressive autosomal dominant neurodegenerative disorder. HD results from the genetic mutation that leads to an abnormally expanded polyglutamine (PolyQ) sequence… (more)

Subjects/Keywords: Huntington's chorea  – Genetic aspects <; br>; Peptides  – Antagonists <; br>; Protease inhibitors

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APA (6th Edition):

Blaize, M. A. (2008). Degradation of the Huntington polyglutamine domain by the proteasome : implication to Huntington's disease. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,216

Chicago Manual of Style (16th Edition):

Blaize, Marie Antionette. “Degradation of the Huntington polyglutamine domain by the proteasome : implication to Huntington's disease.” 2008. Masters Thesis, University of Alabama – Birmingham. Accessed August 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,216.

MLA Handbook (7th Edition):

Blaize, Marie Antionette. “Degradation of the Huntington polyglutamine domain by the proteasome : implication to Huntington's disease.” 2008. Web. 20 Aug 2019.

Vancouver:

Blaize MA. Degradation of the Huntington polyglutamine domain by the proteasome : implication to Huntington's disease. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2008. [cited 2019 Aug 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,216.

Council of Science Editors:

Blaize MA. Degradation of the Huntington polyglutamine domain by the proteasome : implication to Huntington's disease. [Masters Thesis]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,216

25. Azuero, Andres. Comparisons of sequential testing approaches for detection of association between disease and haplotype blocks.

Degree: PhD, 2008, University of Alabama – Birmingham

Since Wald's development of the Sequential Probability Ratio Test (SPRT) in 1945, sequential analysis has evolved into a rich and well-developed field. Sequential methods are… (more)

Subjects/Keywords: Algorithms<; br>; Genetic Predisposition to Disease<; br>; Genome, Human<; br>; Haplotypes<; br>; Linkage Disequilibrium<; br>; Polymorphism, Single Nucleotide.

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APA (6th Edition):

Azuero, A. (2008). Comparisons of sequential testing approaches for detection of association between disease and haplotype blocks. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,430

Chicago Manual of Style (16th Edition):

Azuero, Andres. “Comparisons of sequential testing approaches for detection of association between disease and haplotype blocks.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed August 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,430.

MLA Handbook (7th Edition):

Azuero, Andres. “Comparisons of sequential testing approaches for detection of association between disease and haplotype blocks.” 2008. Web. 20 Aug 2019.

Vancouver:

Azuero A. Comparisons of sequential testing approaches for detection of association between disease and haplotype blocks. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Aug 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,430.

Council of Science Editors:

Azuero A. Comparisons of sequential testing approaches for detection of association between disease and haplotype blocks. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,430

26. McAlear, Suzanne D. (Suzanne DuChai). Electrogenic Na/Bicarbonate cotransporter (NBCE1) variants expressed in Xenopus oocytes : protein regions involved in function, expression, and ion translocation.

Degree: PhD, 2007, University of Alabama – Birmingham

Electrogenic Na/bicarbonate cotransporters (NBCes) are important regulators of intracellular pH in many tissues including the kidney, brain, and pancreas, and are members of a superfamily… (more)

Subjects/Keywords: Cysteine  – metabolism<; br>; Gene Expression Regulation  – physiology<; br>; Genetic Variation  – physiology<; br>; Mutagenesis<; br>; Oocytes<; br>; Sodium-Bicarbonate Symporters

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APA (6th Edition):

McAlear, S. D. (. D. (2007). Electrogenic Na/Bicarbonate cotransporter (NBCE1) variants expressed in Xenopus oocytes : protein regions involved in function, expression, and ion translocation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,456

Chicago Manual of Style (16th Edition):

McAlear, Suzanne D (Suzanne DuChai). “Electrogenic Na/Bicarbonate cotransporter (NBCE1) variants expressed in Xenopus oocytes : protein regions involved in function, expression, and ion translocation.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed August 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,456.

MLA Handbook (7th Edition):

McAlear, Suzanne D (Suzanne DuChai). “Electrogenic Na/Bicarbonate cotransporter (NBCE1) variants expressed in Xenopus oocytes : protein regions involved in function, expression, and ion translocation.” 2007. Web. 20 Aug 2019.

Vancouver:

McAlear SD(D. Electrogenic Na/Bicarbonate cotransporter (NBCE1) variants expressed in Xenopus oocytes : protein regions involved in function, expression, and ion translocation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Aug 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,456.

Council of Science Editors:

McAlear SD(D. Electrogenic Na/Bicarbonate cotransporter (NBCE1) variants expressed in Xenopus oocytes : protein regions involved in function, expression, and ion translocation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,456

27. Murakami, Miho. Fiber modification of adenoviral vectors for cancer gene therapy.

Degree: PhD, 2010, University of Alabama – Birmingham

Cancer still remains a major public health concern despite improvements in primary prevention, early detection and advanced treatments. Cancer gene therapy using human adenovirus serotype… (more)

Subjects/Keywords: Adenoviruses, Human<; br>; Capsid Proteins<; br>; Gene Therapy  – methods<; br>; Genetic Vectors<; br>; Prostatic Neoplasms  – genetics<; br>; Recombinant Fusion Proteins  – metabolism<; br>; Transduction, Genetic<; br>; Viral Tropism  – physiology

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APA (6th Edition):

Murakami, M. (2010). Fiber modification of adenoviral vectors for cancer gene therapy. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1166

Chicago Manual of Style (16th Edition):

Murakami, Miho. “Fiber modification of adenoviral vectors for cancer gene therapy.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed August 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1166.

MLA Handbook (7th Edition):

Murakami, Miho. “Fiber modification of adenoviral vectors for cancer gene therapy.” 2010. Web. 20 Aug 2019.

Vancouver:

Murakami M. Fiber modification of adenoviral vectors for cancer gene therapy. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Aug 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1166.

Council of Science Editors:

Murakami M. Fiber modification of adenoviral vectors for cancer gene therapy. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1166

28. Badiga, Suguna. Altered Dna Methylation Of Repetitive Elements May Explain The Modified Risk Of Cervical Intraepithelial Neoplasia (cin) Associated With Genetic Polymorphisms Of The Folate Metabolic Pathway In The Us Post Folic Acid Fortification Era.

Degree: PhD, 2011, University of Alabama – Birmingham

Despite the controversy regarding the adverse effects of exposure to higher folate concentrations on health outcomes particularly cancer, there has been paucity of research to… (more)

Subjects/Keywords: Cervical Intraepithelial Neoplasia – genetics.<; br>; DNA Methylation.<; br>; Diet<; br>; Folic Acid – blood<; br>; Genetic Variation – genetics.<; br>; Polymorphism, Genetic<; br>; Uterine Cervical Neoplasms – genetics.<; br>; Vitamin B 12 – blood.

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APA (6th Edition):

Badiga, S. (2011). Altered Dna Methylation Of Repetitive Elements May Explain The Modified Risk Of Cervical Intraepithelial Neoplasia (cin) Associated With Genetic Polymorphisms Of The Folate Metabolic Pathway In The Us Post Folic Acid Fortification Era. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1406

Chicago Manual of Style (16th Edition):

Badiga, Suguna. “Altered Dna Methylation Of Repetitive Elements May Explain The Modified Risk Of Cervical Intraepithelial Neoplasia (cin) Associated With Genetic Polymorphisms Of The Folate Metabolic Pathway In The Us Post Folic Acid Fortification Era.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed August 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1406.

MLA Handbook (7th Edition):

Badiga, Suguna. “Altered Dna Methylation Of Repetitive Elements May Explain The Modified Risk Of Cervical Intraepithelial Neoplasia (cin) Associated With Genetic Polymorphisms Of The Folate Metabolic Pathway In The Us Post Folic Acid Fortification Era.” 2011. Web. 20 Aug 2019.

Vancouver:

Badiga S. Altered Dna Methylation Of Repetitive Elements May Explain The Modified Risk Of Cervical Intraepithelial Neoplasia (cin) Associated With Genetic Polymorphisms Of The Folate Metabolic Pathway In The Us Post Folic Acid Fortification Era. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Aug 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1406.

Council of Science Editors:

Badiga S. Altered Dna Methylation Of Repetitive Elements May Explain The Modified Risk Of Cervical Intraepithelial Neoplasia (cin) Associated With Genetic Polymorphisms Of The Folate Metabolic Pathway In The Us Post Folic Acid Fortification Era. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1406

29. Lee, Seung-Ah. Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism.

Degree: PhD, 2008, University of Alabama – Birmingham

Retinol dehydrogenase 12 (RDH12) is a member of the microsomal short-chain dehydrogenase/reductase superfamily of proteins that is highly expressed in photorecep-tor cells. Mutations in RDH12… (more)

Subjects/Keywords: Alcohol Oxidoreductases  – metabolism<; br>; Genetic Diseases, Inborn  – enzymology<; br>; Lipid Peroxidation<; br>; Mutation, Missense<; br>; Photoreceptor Cells  – enzymology<; br>; Retinal Diseases  – enzymology<; br>; Retinaldehyde  – metabolism<; br>; Retinoids  – metabolism<; br>; Tretinoin  – metabolism

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lee, S. (2008). Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,814

Chicago Manual of Style (16th Edition):

Lee, Seung-Ah. “Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed August 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,814.

MLA Handbook (7th Edition):

Lee, Seung-Ah. “Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism.” 2008. Web. 20 Aug 2019.

Vancouver:

Lee S. Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Aug 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,814.

Council of Science Editors:

Lee S. Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,814

30. Willig, Amanda L. (Amanda Lynn). Contributions of the atrial natriuretic peptide gene system to the relationship between pediatric body fat, free fatty acids, and blood pressure.

Degree: PhD, 2010, University of Alabama – Birmingham

Elevated blood pressure and nonesterified fatty acids (NEFA) in children can lead to increased chronic disease risk during adulthood. The atrial natriuretic peptide precursor A… (more)

Subjects/Keywords: Adiposity<; br>; African Americans<; br>; Blood Pressure  – physiology<; br>; Body Height  – physiology<; br>; Body Weight<; br>; Child<; br>; European Continental Ancestry Group<; br>; Genetic Predisposition to Disease<; br>; Hypertension  – etiology<; br>; Indians, North American<; br>; Physical Fitness  – physiology<; br>

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Willig, A. L. (. L. (2010). Contributions of the atrial natriuretic peptide gene system to the relationship between pediatric body fat, free fatty acids, and blood pressure. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,934

Chicago Manual of Style (16th Edition):

Willig, Amanda L (Amanda Lynn). “Contributions of the atrial natriuretic peptide gene system to the relationship between pediatric body fat, free fatty acids, and blood pressure.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed August 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,934.

MLA Handbook (7th Edition):

Willig, Amanda L (Amanda Lynn). “Contributions of the atrial natriuretic peptide gene system to the relationship between pediatric body fat, free fatty acids, and blood pressure.” 2010. Web. 20 Aug 2019.

Vancouver:

Willig AL(L. Contributions of the atrial natriuretic peptide gene system to the relationship between pediatric body fat, free fatty acids, and blood pressure. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Aug 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,934.

Council of Science Editors:

Willig AL(L. Contributions of the atrial natriuretic peptide gene system to the relationship between pediatric body fat, free fatty acids, and blood pressure. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,934

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