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You searched for subject:( Brain Neoplasms genetics Cerebellum cytology 60). Showing records 1 – 30 of 35553 total matches.

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1. Geng, Ying. p53-dependent neural stem cell death regulation.

Degree: PhD, 2008, University of Alabama – Birmingham

Regulation of neural precursor cell (NPC) death is important for both normal brain development and prevention of brain tumor formation. The tumor suppressor p53 is… (more)

Subjects/Keywords: Antineoplastic Agents  – pharmacology<; br>; Apoptosis  – physiology bcl-2-Associated X Protein  – physiology<; br>; Brain Neoplasms  – genetics Cerebellum  – cytology<; br>; Chloroquine  – pharmacology<; br>; Cytoplasm  – metabolism<; br>; Glioma  – genetics Neurons  – cytology<; br>; Stem Cells  – cytology Transcriptional Activation  – physiology<; br>; Tumor Suppressor Protein p53  – genetics

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APA (6th Edition):

Geng, Y. (2008). p53-dependent neural stem cell death regulation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,767

Chicago Manual of Style (16th Edition):

Geng, Ying. “p53-dependent neural stem cell death regulation.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,767.

MLA Handbook (7th Edition):

Geng, Ying. “p53-dependent neural stem cell death regulation.” 2008. Web. 29 Jan 2020.

Vancouver:

Geng Y. p53-dependent neural stem cell death regulation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Jan 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,767.

Council of Science Editors:

Geng Y. p53-dependent neural stem cell death regulation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,767

2. Brantley, Emily Claire. Loss of PIAS3 expression in glioblastoma multiforme tumors : implications for STAT-3 activation and gene expression.

Degree: PhD, 2007, University of Alabama – Birmingham

Glioblastoma multiforme (GBM) is the most common form of cancer in the central nervous system in adults. Because of the infiltrative and aggressive nature of… (more)

Subjects/Keywords: Brain Neoplasms  – metabolism <; br>; Brain Neoplasms  – physiopathology <; br>; Glioblastoma  – metabolism <; br>; Glioblastoma  – physiopathology <; br>; Protein Transport  – genetics <; br>; STAT3 Transcription Factor

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APA (6th Edition):

Brantley, E. C. (2007). Loss of PIAS3 expression in glioblastoma multiforme tumors : implications for STAT-3 activation and gene expression. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,193

Chicago Manual of Style (16th Edition):

Brantley, Emily Claire. “Loss of PIAS3 expression in glioblastoma multiforme tumors : implications for STAT-3 activation and gene expression.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,193.

MLA Handbook (7th Edition):

Brantley, Emily Claire. “Loss of PIAS3 expression in glioblastoma multiforme tumors : implications for STAT-3 activation and gene expression.” 2007. Web. 29 Jan 2020.

Vancouver:

Brantley EC. Loss of PIAS3 expression in glioblastoma multiforme tumors : implications for STAT-3 activation and gene expression. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Jan 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,193.

Council of Science Editors:

Brantley EC. Loss of PIAS3 expression in glioblastoma multiforme tumors : implications for STAT-3 activation and gene expression. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,193

3. Tang, Yizhe. Modification of adenovirus capsid proteins for gene therapy applications.

Degree: PhD, 2009, University of Alabama – Birmingham

Adenovirus (Ad) is the most commonly used viral vector in gene therapy applications to date for a broad range of diseases. Although Ad-based viral vectors… (more)

Subjects/Keywords: Adenoviridae  – genetics<; br>; Adenoviridae Infections  – metabolism<; br>; Adenoviruses, Human  – genetics<; br>; Blood-Brain Barrier  – virology<; br>; Capsid Proteins  – chemistry<; br>; Gene Products, tat<; br>; Gene Therapy  – methods<; br>; Genetic Engineering<; br>; Neoplasms<; br>; Transduction, Genetic  – methods

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APA (6th Edition):

Tang, Y. (2009). Modification of adenovirus capsid proteins for gene therapy applications. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,675

Chicago Manual of Style (16th Edition):

Tang, Yizhe. “Modification of adenovirus capsid proteins for gene therapy applications.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,675.

MLA Handbook (7th Edition):

Tang, Yizhe. “Modification of adenovirus capsid proteins for gene therapy applications.” 2009. Web. 29 Jan 2020.

Vancouver:

Tang Y. Modification of adenovirus capsid proteins for gene therapy applications. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Jan 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,675.

Council of Science Editors:

Tang Y. Modification of adenovirus capsid proteins for gene therapy applications. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,675

4. Grunda, Jessica M. Identification of new strategies for the treatment of glioblastoma multiforme utilizing a pharmacogenomic approach: genetic profiles associated with patient prognosis and outcome to capecitabine treatment.

Degree: PhD, 2009, University of Alabama – Birmingham

Glioblastoma multiforme (GBM) is the most lethal form of primary brain neoplasm with average patient survival between 9 and 15 months even with the most… (more)

Subjects/Keywords: Brain Neoplasms<; br>; Drug Resistance, Neoplasm  – genetics<; br>; Endopeptidases  – metabolism<; br>; Gene Expression Profiling<; br>; Glioblastoma<; br>; Microtubule-Associated Proteins  – metabolism<; br>; Neoplasm Proteins  – metabolism<; br>; Thymidylate Synthase  – metabolism<; br>; Tumor Markers, Biological  – metabolism

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APA (6th Edition):

Grunda, J. M. (2009). Identification of new strategies for the treatment of glioblastoma multiforme utilizing a pharmacogenomic approach: genetic profiles associated with patient prognosis and outcome to capecitabine treatment. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1085

Chicago Manual of Style (16th Edition):

Grunda, Jessica M. “Identification of new strategies for the treatment of glioblastoma multiforme utilizing a pharmacogenomic approach: genetic profiles associated with patient prognosis and outcome to capecitabine treatment.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1085.

MLA Handbook (7th Edition):

Grunda, Jessica M. “Identification of new strategies for the treatment of glioblastoma multiforme utilizing a pharmacogenomic approach: genetic profiles associated with patient prognosis and outcome to capecitabine treatment.” 2009. Web. 29 Jan 2020.

Vancouver:

Grunda JM. Identification of new strategies for the treatment of glioblastoma multiforme utilizing a pharmacogenomic approach: genetic profiles associated with patient prognosis and outcome to capecitabine treatment. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Jan 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1085.

Council of Science Editors:

Grunda JM. Identification of new strategies for the treatment of glioblastoma multiforme utilizing a pharmacogenomic approach: genetic profiles associated with patient prognosis and outcome to capecitabine treatment. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1085

5. Lai, Yi-Shin. Sox2/PARylated Parp1 complexes regulate pluripotency.

Degree: PhD, 2011, University of Alabama – Birmingham

Embryonic stem cells (ESCs) were first derived from the inner cell mass (ICM) of blastocyst-stage embryos. ESCs are distinguished from other cell types by their… (more)

Subjects/Keywords: Embryonic Stem Cells.<; br>; Fibroblasts – cytology<; br>; Genetic Vectors – genetics.<; br>; Lentivirus – genetics.<; br>; Nuclear Reprogramming – genetics.<; br>; Pluripotent Stem Cells – cytology.<; br>; Skin – cytology

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APA (6th Edition):

Lai, Y. (2011). Sox2/PARylated Parp1 complexes regulate pluripotency. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1361

Chicago Manual of Style (16th Edition):

Lai, Yi-Shin. “Sox2/PARylated Parp1 complexes regulate pluripotency.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1361.

MLA Handbook (7th Edition):

Lai, Yi-Shin. “Sox2/PARylated Parp1 complexes regulate pluripotency.” 2011. Web. 29 Jan 2020.

Vancouver:

Lai Y. Sox2/PARylated Parp1 complexes regulate pluripotency. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Jan 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1361.

Council of Science Editors:

Lai Y. Sox2/PARylated Parp1 complexes regulate pluripotency. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1361

6. Chang, Chia-Wei. Polycistronic lentiviral vector for hit and run reprogramming of mouse and human somatic cells to induced pluripotent stem cell.

Degree: PhD, 2009, University of Alabama – Birmingham

Embryonic stem (ES) cells are pluripotent and, therefore, can differentiate into most if not all somatic cell types. Because of this characteristic, ES cells have… (more)

Subjects/Keywords: Fibroblasts  – cytology<; br>; Genetic Vectors  – genetics<; br>; Lentivirus  – genetics<; br>; Nuclear Reprogramming  – genetics<; br>; Pluripotent Stem Cells  – cytology<; br>; Skin  – cytology

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APA (6th Edition):

Chang, C. (2009). Polycistronic lentiviral vector for hit and run reprogramming of mouse and human somatic cells to induced pluripotent stem cell. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,561

Chicago Manual of Style (16th Edition):

Chang, Chia-Wei. “Polycistronic lentiviral vector for hit and run reprogramming of mouse and human somatic cells to induced pluripotent stem cell.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,561.

MLA Handbook (7th Edition):

Chang, Chia-Wei. “Polycistronic lentiviral vector for hit and run reprogramming of mouse and human somatic cells to induced pluripotent stem cell.” 2009. Web. 29 Jan 2020.

Vancouver:

Chang C. Polycistronic lentiviral vector for hit and run reprogramming of mouse and human somatic cells to induced pluripotent stem cell. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Jan 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,561.

Council of Science Editors:

Chang C. Polycistronic lentiviral vector for hit and run reprogramming of mouse and human somatic cells to induced pluripotent stem cell. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,561

7. Eliuk, Shannon M. (Shannon MIchelle). Oxidative post-translational modification of the brain isoform of creatine kinase: structural and functional consequences.

Degree: PhD, 2008, University of Alabama – Birmingham

Oxidative modification of proteins, including modification by the reactive aldehyde 4-hydroxy-2-nonenal (4HNE), has been implicated in the pathogenesis of Alzheimer’s disease (AD). Initial reports indicated… (more)

Subjects/Keywords: Alzheimer Disease  – metabolism<; br>; Brain  – cytology<; br>; Creatine Kinase  – metabolism<; br>; Mass Spectrometry<; br>; Oxidative Stress<; br>; Protein Isoforms

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APA (6th Edition):

Eliuk, S. M. (. M. (2008). Oxidative post-translational modification of the brain isoform of creatine kinase: structural and functional consequences. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,817

Chicago Manual of Style (16th Edition):

Eliuk, Shannon M (Shannon MIchelle). “Oxidative post-translational modification of the brain isoform of creatine kinase: structural and functional consequences.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,817.

MLA Handbook (7th Edition):

Eliuk, Shannon M (Shannon MIchelle). “Oxidative post-translational modification of the brain isoform of creatine kinase: structural and functional consequences.” 2008. Web. 29 Jan 2020.

Vancouver:

Eliuk SM(M. Oxidative post-translational modification of the brain isoform of creatine kinase: structural and functional consequences. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Jan 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,817.

Council of Science Editors:

Eliuk SM(M. Oxidative post-translational modification of the brain isoform of creatine kinase: structural and functional consequences. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,817

8. Hensel, Jonathan Adam. The role of LL-37 in prostate cancer and its potential as a therapeutic target.

Degree: 2011, University of Alabama – Birmingham

LL-37 is the only cathelicidin-derived anti-microbial peptide in humans and has been shown to stimulate proliferation, angiogenesis, cellular migration and inhibit apoptosis, in addition to… (more)

Subjects/Keywords: Antimicrobial Cationic Peptides  – metabolism<; br>; Cathelicidins<; br>; Molecular Targeted Therapy  – methods<; br>; Neoplasms, Hormone-Dependent<; br>; Prostatic Neoplasms  – genetics<; br>; Prostatic Neoplasms  – metabolism<; br>; Prostatic Neoplasms  – pathology

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APA (6th Edition):

Hensel, J. A. (2011). The role of LL-37 in prostate cancer and its potential as a therapeutic target. (Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,875

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hensel, Jonathan Adam. “The role of LL-37 in prostate cancer and its potential as a therapeutic target.” 2011. Thesis, University of Alabama – Birmingham. Accessed January 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,875.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hensel, Jonathan Adam. “The role of LL-37 in prostate cancer and its potential as a therapeutic target.” 2011. Web. 29 Jan 2020.

Vancouver:

Hensel JA. The role of LL-37 in prostate cancer and its potential as a therapeutic target. [Internet] [Thesis]. University of Alabama – Birmingham; 2011. [cited 2020 Jan 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,875.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hensel JA. The role of LL-37 in prostate cancer and its potential as a therapeutic target. [Thesis]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,875

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. Gavin, Cristin. Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation.

Degree: PhD, 2012, University of Alabama – Birmingham

Dynamic changes to the actin cytoskeleton are required for synaptic plasticity and long-term memory formation. However, the molecular mechanisms that mediate filamentous actin (F-actin) dynamics… (more)

Subjects/Keywords: Bone Neoplasms – secondary<; br>; Breast Neoplasms – pathology<; br>; Cell Communication<; br>; Cyclic AMP-Dependent Protein Kinases – metabolism.<; br>; Gap Junctions – metabolism.<; br>; Osteoblasts – cytology.<; br>; Phosphatidylinositol 3-Kinases – metabolism.

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APA (6th Edition):

Gavin, C. (2012). Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1396

Chicago Manual of Style (16th Edition):

Gavin, Cristin. “Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1396.

MLA Handbook (7th Edition):

Gavin, Cristin. “Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation.” 2012. Web. 29 Jan 2020.

Vancouver:

Gavin C. Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2020 Jan 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1396.

Council of Science Editors:

Gavin C. Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1396

10. Mao,Weiming. The role of bHLH gene ash1 in the developing chick eye.

Degree: PhD, 2008, University of Alabama – Birmingham

Development of the vertebrate eye is controlled by a network of regulatory genes including those in the basic loop-helix-loop (bHLH) family. In this study, we… (more)

Subjects/Keywords: Amacrine Cells  – physiology<; br>; Avian Proteins  – metabolism<; br>; Basic Helix-Loop-Helix Transcription Factors  – metabolism<; br>; Gene Expression Regulation, Developmental  – physiology<; br>; Nerve Tissue Proteins  – genetics<; br>; Nuclear Reprogramming<; br>; Retina  – cytology<; br>; Retinal Neurons  – cytology Retinal Pigment Epithelium  – cytology<; br>; Stem Cells  – cytology

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APA (6th Edition):

Mao,Weiming. (2008). The role of bHLH gene ash1 in the developing chick eye. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,527

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

Mao,Weiming. “The role of bHLH gene ash1 in the developing chick eye.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,527.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

Mao,Weiming. “The role of bHLH gene ash1 in the developing chick eye.” 2008. Web. 29 Jan 2020.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

Mao,Weiming. The role of bHLH gene ash1 in the developing chick eye. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Jan 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,527.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

Mao,Weiming. The role of bHLH gene ash1 in the developing chick eye. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,527

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

11. Ogunrinu, Toyin Adeyemi. Role of the cystine-glutamate exchanger in glioma cell biology.

Degree: PhD, 2010, University of Alabama – Birmingham

Changes in the glioma microenvironment including oxygen (O2) levels, supply of amino acid such as L-glutamate and L-cystine and glutathione (GSH) concentrations play a critical… (more)

Subjects/Keywords: Anoxia  – metabolism<; br>; Brain Neoplasms  – metabolism<; br>; Glioblastoma  – metabolism<; br>; Glioma  – metabolism<; br>; Glutathione  – metabolism<; br>; Glutamic Acid  – metabolism

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ogunrinu, T. A. (2010). Role of the cystine-glutamate exchanger in glioma cell biology. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,956

Chicago Manual of Style (16th Edition):

Ogunrinu, Toyin Adeyemi. “Role of the cystine-glutamate exchanger in glioma cell biology.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,956.

MLA Handbook (7th Edition):

Ogunrinu, Toyin Adeyemi. “Role of the cystine-glutamate exchanger in glioma cell biology.” 2010. Web. 29 Jan 2020.

Vancouver:

Ogunrinu TA. Role of the cystine-glutamate exchanger in glioma cell biology. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Jan 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,956.

Council of Science Editors:

Ogunrinu TA. Role of the cystine-glutamate exchanger in glioma cell biology. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,956

12. Robert, Stephanie Marie. Cystine/glutamate Transporters As Prognostic & Therapeutic Markers Of Primary Brain Tumors.

Degree: 2012, University of Alabama – Birmingham

Glioblastoma multiforme (GBM) are the most prevalent and aggressive malignant brain tumors. Current treatment - a combination of radiation, chemotherapy and resection - has limited… (more)

Subjects/Keywords: Amino Acid Transport System X-AG – metabolism.<; br>; Brain Neoplasms – metabolism<; br>; Glioblastoma.<; br>; Glutathione<; br>; Seizures<; br>; Sulfasalazine.

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APA (6th Edition):

Robert, S. M. (2012). Cystine/glutamate Transporters As Prognostic & Therapeutic Markers Of Primary Brain Tumors. (Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1791

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Robert, Stephanie Marie. “Cystine/glutamate Transporters As Prognostic & Therapeutic Markers Of Primary Brain Tumors.” 2012. Thesis, University of Alabama – Birmingham. Accessed January 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1791.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Robert, Stephanie Marie. “Cystine/glutamate Transporters As Prognostic & Therapeutic Markers Of Primary Brain Tumors.” 2012. Web. 29 Jan 2020.

Vancouver:

Robert SM. Cystine/glutamate Transporters As Prognostic & Therapeutic Markers Of Primary Brain Tumors. [Internet] [Thesis]. University of Alabama – Birmingham; 2012. [cited 2020 Jan 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1791.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Robert SM. Cystine/glutamate Transporters As Prognostic & Therapeutic Markers Of Primary Brain Tumors. [Thesis]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1791

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

13. Cody, James Joseph. A dual-action, armed replicating adenovirus for the treatment of bone metastases of breast cancer.

Degree: PhD, 2008, University of Alabama – Birmingham

Most patients with advanced breast cancer develop osteolytic bone metastases, which have numerous complications. Because current therapies are not curative, new treatments are needed. Conditionally… (more)

Subjects/Keywords: Adenoviridae  – genetics <; br>; Bone Neoplasms  – secondary <; br>; Breast Neoplasms <; br>; Gene Expression Regulation, Neoplastic <; br>; Neoplasm Metastasis  – genetics <; br>; Neoplasm Metastasis  – therapy <; br>; Virus Replication  – genetics

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APA (6th Edition):

Cody, J. J. (2008). A dual-action, armed replicating adenovirus for the treatment of bone metastases of breast cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,320

Chicago Manual of Style (16th Edition):

Cody, James Joseph. “A dual-action, armed replicating adenovirus for the treatment of bone metastases of breast cancer.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,320.

MLA Handbook (7th Edition):

Cody, James Joseph. “A dual-action, armed replicating adenovirus for the treatment of bone metastases of breast cancer.” 2008. Web. 29 Jan 2020.

Vancouver:

Cody JJ. A dual-action, armed replicating adenovirus for the treatment of bone metastases of breast cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Jan 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,320.

Council of Science Editors:

Cody JJ. A dual-action, armed replicating adenovirus for the treatment of bone metastases of breast cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,320

14. Kojima, Kyoko, Ph.D. Mouse modeling of pancreatic ductal adenocarcinoma (PDAC): search for early diagnostic markers and therapeutic targets.

Degree: PhD, 2009, University of Alabama – Birmingham

PDAC is a highly malignant neoplasm that carries a very poor prognosis. PDAC development is a multistage transformation process that involves multiple genetic alterations that… (more)

Subjects/Keywords: Carcinoma in Situ  – genetics<; br>; Genes, ras<; br>; Pancreatic Neoplasms  – diagnosis<; br>; Pancreatic Neoplasms  – genetics<; br>; Proteomics<; br>; Smad4 Protein  – genetics<; br>; Tumor Markers, Biological  – analysis

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APA (6th Edition):

Kojima, Kyoko, P. D. (2009). Mouse modeling of pancreatic ductal adenocarcinoma (PDAC): search for early diagnostic markers and therapeutic targets. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,396

Chicago Manual of Style (16th Edition):

Kojima, Kyoko, Ph D. “Mouse modeling of pancreatic ductal adenocarcinoma (PDAC): search for early diagnostic markers and therapeutic targets.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,396.

MLA Handbook (7th Edition):

Kojima, Kyoko, Ph D. “Mouse modeling of pancreatic ductal adenocarcinoma (PDAC): search for early diagnostic markers and therapeutic targets.” 2009. Web. 29 Jan 2020.

Vancouver:

Kojima, Kyoko PD. Mouse modeling of pancreatic ductal adenocarcinoma (PDAC): search for early diagnostic markers and therapeutic targets. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Jan 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,396.

Council of Science Editors:

Kojima, Kyoko PD. Mouse modeling of pancreatic ductal adenocarcinoma (PDAC): search for early diagnostic markers and therapeutic targets. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,396

15. Moore, Lakisha Dionne. Modulation of Transforming Growth Factor (TGF)-[beta]1 and its implications in breast cancer metastasis.

Degree: PhD, 2008, University of Alabama – Birmingham

Overexpresssion of transforming growth factor (TGF)-[beta] has been implicated in promoting immune suppression, tumor angiogenesis, tumor cell migration, and invasion in many cancers including carcinoma… (more)

Subjects/Keywords: Breast Neoplasms  – genetics <; br>; Neoplasm Metastasis <; br>; RNA Interference <; br>; Transforming Growth Factor beta1  – genetics <; br>; Transforming Growth Factor beta1  – metabolism

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APA (6th Edition):

Moore, L. D. (2008). Modulation of Transforming Growth Factor (TGF)-[beta]1 and its implications in breast cancer metastasis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,233

Chicago Manual of Style (16th Edition):

Moore, Lakisha Dionne. “Modulation of Transforming Growth Factor (TGF)-[beta]1 and its implications in breast cancer metastasis.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,233.

MLA Handbook (7th Edition):

Moore, Lakisha Dionne. “Modulation of Transforming Growth Factor (TGF)-[beta]1 and its implications in breast cancer metastasis.” 2008. Web. 29 Jan 2020.

Vancouver:

Moore LD. Modulation of Transforming Growth Factor (TGF)-[beta]1 and its implications in breast cancer metastasis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Jan 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,233.

Council of Science Editors:

Moore LD. Modulation of Transforming Growth Factor (TGF)-[beta]1 and its implications in breast cancer metastasis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,233

16. McCoy, Eric. Expression and function of aquaporins in malignant and non-malignant astrocytes.

Degree: PhD, 2008, University of Alabama – Birmingham

Aquaporins (AQP) constitute the primary pathway for water movement across cellular membrances. As a result, their expression and function are important for regulating cell volume.… (more)

Subjects/Keywords: Aquaporin 1 Aquaporin 4<; br>; Aquaporins  – metabolism<; br>; Astrocytes  – physiology<; br>; Brain Injuries  – physiopathology<; br>; Brain Neoplasms  – metabolism<; br>; Cell Movement<; br>; Neoplasm Invasiveness<; br>; Wounds, Stab  – physiopathology

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APA (6th Edition):

McCoy, E. (2008). Expression and function of aquaporins in malignant and non-malignant astrocytes. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,774

Chicago Manual of Style (16th Edition):

McCoy, Eric. “Expression and function of aquaporins in malignant and non-malignant astrocytes.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,774.

MLA Handbook (7th Edition):

McCoy, Eric. “Expression and function of aquaporins in malignant and non-malignant astrocytes.” 2008. Web. 29 Jan 2020.

Vancouver:

McCoy E. Expression and function of aquaporins in malignant and non-malignant astrocytes. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Jan 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,774.

Council of Science Editors:

McCoy E. Expression and function of aquaporins in malignant and non-malignant astrocytes. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,774

17. Risner, Michael Lance. Response dynamics of retinal ganglion and early visual cortical cells to simulated optical blur.

Degree: PhD, 2010, University of Alabama – Birmingham

Visual neurons are highly sensitive to changes in luminance around the mean level of light. The effects of changes in luminance contrast on the responses… (more)

Subjects/Keywords: Brain Mapping<; br>; Action Potentials  – physiology<; br>; Light<; br>; Neurons  – physiology<; br>; Nonlinear Dynamics<; br>; Primates  – physiology<; br>; Retina  – cytology<; br>; Retinal Ganglion Cells  – physiology<; br>; Visual Cortex  – cytology<; br>; Visual Fields  – physiology

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APA (6th Edition):

Risner, M. L. (2010). Response dynamics of retinal ganglion and early visual cortical cells to simulated optical blur. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,931

Chicago Manual of Style (16th Edition):

Risner, Michael Lance. “Response dynamics of retinal ganglion and early visual cortical cells to simulated optical blur.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,931.

MLA Handbook (7th Edition):

Risner, Michael Lance. “Response dynamics of retinal ganglion and early visual cortical cells to simulated optical blur.” 2010. Web. 29 Jan 2020.

Vancouver:

Risner ML. Response dynamics of retinal ganglion and early visual cortical cells to simulated optical blur. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Jan 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,931.

Council of Science Editors:

Risner ML. Response dynamics of retinal ganglion and early visual cortical cells to simulated optical blur. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,931

18. Ernest, Nola Jean Sieber. The role of chloride in the volume regulation of human glioma cells.

Degree: PhD, 2007, University of Alabama – Birmingham

According to the Central Brain Tumor Registry of the United States, the most common primary brain tumors are gliomas, tumors composed of cells of glial… (more)

Subjects/Keywords: Brain Neoplasms  – physiopathology <; br>; Cell Size <; br>; Chloride Channels  – physiology <; br>; Glioma  – physiopathology <; br>

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APA (6th Edition):

Ernest, N. J. S. (2007). The role of chloride in the volume regulation of human glioma cells. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,86

Chicago Manual of Style (16th Edition):

Ernest, Nola Jean Sieber. “The role of chloride in the volume regulation of human glioma cells.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,86.

MLA Handbook (7th Edition):

Ernest, Nola Jean Sieber. “The role of chloride in the volume regulation of human glioma cells.” 2007. Web. 29 Jan 2020.

Vancouver:

Ernest NJS. The role of chloride in the volume regulation of human glioma cells. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Jan 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,86.

Council of Science Editors:

Ernest NJS. The role of chloride in the volume regulation of human glioma cells. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,86


Columbia University

19. Pan, Kally Zhang. Cell Size Control in Fission Yeast.

Degree: 2013, Columbia University

 Among all living organisms, there is almost much variety in cell size as there is for cell function and cell type. However, within each cell… (more)

Subjects/Keywords: Cytology; Genetics

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APA (6th Edition):

Pan, K. Z. (2013). Cell Size Control in Fission Yeast. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8028ZXF

Chicago Manual of Style (16th Edition):

Pan, Kally Zhang. “Cell Size Control in Fission Yeast.” 2013. Doctoral Dissertation, Columbia University. Accessed January 29, 2020. https://doi.org/10.7916/D8028ZXF.

MLA Handbook (7th Edition):

Pan, Kally Zhang. “Cell Size Control in Fission Yeast.” 2013. Web. 29 Jan 2020.

Vancouver:

Pan KZ. Cell Size Control in Fission Yeast. [Internet] [Doctoral dissertation]. Columbia University; 2013. [cited 2020 Jan 29]. Available from: https://doi.org/10.7916/D8028ZXF.

Council of Science Editors:

Pan KZ. Cell Size Control in Fission Yeast. [Doctoral Dissertation]. Columbia University; 2013. Available from: https://doi.org/10.7916/D8028ZXF


Columbia University

20. Palmer, Colin James. The transcription factor Zfx is required for tumorigenesis caused by Hedgehog pathway activation.

Degree: 2013, Columbia University

 The Hedgehog (Hh) signaling pathway regulates normal development and cell proliferation across the metazoa. Upon its aberrant activation, mammalian Hh signaling can also cause tumor… (more)

Subjects/Keywords: Cytology; Genetics

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APA (6th Edition):

Palmer, C. J. (2013). The transcription factor Zfx is required for tumorigenesis caused by Hedgehog pathway activation. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8VQ391B

Chicago Manual of Style (16th Edition):

Palmer, Colin James. “The transcription factor Zfx is required for tumorigenesis caused by Hedgehog pathway activation.” 2013. Doctoral Dissertation, Columbia University. Accessed January 29, 2020. https://doi.org/10.7916/D8VQ391B.

MLA Handbook (7th Edition):

Palmer, Colin James. “The transcription factor Zfx is required for tumorigenesis caused by Hedgehog pathway activation.” 2013. Web. 29 Jan 2020.

Vancouver:

Palmer CJ. The transcription factor Zfx is required for tumorigenesis caused by Hedgehog pathway activation. [Internet] [Doctoral dissertation]. Columbia University; 2013. [cited 2020 Jan 29]. Available from: https://doi.org/10.7916/D8VQ391B.

Council of Science Editors:

Palmer CJ. The transcription factor Zfx is required for tumorigenesis caused by Hedgehog pathway activation. [Doctoral Dissertation]. Columbia University; 2013. Available from: https://doi.org/10.7916/D8VQ391B

21. Yang, Rui. High resolution analysis of clonal pluripotent stem cell lentiviral gene therapy in a mouse model of beta-thalassemia.

Degree: PhD, 2011, University of Alabama – Birmingham

Gene therapy for hematopoietic disorders using viral vectors has achieved significant clinical benefit. However this approach has been held back by adverse genotoxic events due… (more)

Subjects/Keywords: Gene Therapy – adverse effects.<; br>; Lentivirus – genetics.<; br>; Mice<; br>; Stem Cells – cytology<; br>; beta-Thalassemia – therapy.

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APA (6th Edition):

Yang, R. (2011). High resolution analysis of clonal pluripotent stem cell lentiviral gene therapy in a mouse model of beta-thalassemia. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1349

Chicago Manual of Style (16th Edition):

Yang, Rui. “High resolution analysis of clonal pluripotent stem cell lentiviral gene therapy in a mouse model of beta-thalassemia.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1349.

MLA Handbook (7th Edition):

Yang, Rui. “High resolution analysis of clonal pluripotent stem cell lentiviral gene therapy in a mouse model of beta-thalassemia.” 2011. Web. 29 Jan 2020.

Vancouver:

Yang R. High resolution analysis of clonal pluripotent stem cell lentiviral gene therapy in a mouse model of beta-thalassemia. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Jan 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1349.

Council of Science Editors:

Yang R. High resolution analysis of clonal pluripotent stem cell lentiviral gene therapy in a mouse model of beta-thalassemia. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1349

22. Badiga, Suguna. Altered Dna Methylation Of Repetitive Elements May Explain The Modified Risk Of Cervical Intraepithelial Neoplasia (cin) Associated With Genetic Polymorphisms Of The Folate Metabolic Pathway In The Us Post Folic Acid Fortification Era.

Degree: PhD, 2011, University of Alabama – Birmingham

Despite the controversy regarding the adverse effects of exposure to higher folate concentrations on health outcomes particularly cancer, there has been paucity of research to… (more)

Subjects/Keywords: Cervical Intraepithelial Neoplasia – genetics.<; br>; DNA Methylation.<; br>; Diet<; br>; Folic Acid – blood<; br>; Genetic Variation – genetics.<; br>; Polymorphism, Genetic<; br>; Uterine Cervical Neoplasms – genetics.<; br>; Vitamin B 12 – blood.

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APA (6th Edition):

Badiga, S. (2011). Altered Dna Methylation Of Repetitive Elements May Explain The Modified Risk Of Cervical Intraepithelial Neoplasia (cin) Associated With Genetic Polymorphisms Of The Folate Metabolic Pathway In The Us Post Folic Acid Fortification Era. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1406

Chicago Manual of Style (16th Edition):

Badiga, Suguna. “Altered Dna Methylation Of Repetitive Elements May Explain The Modified Risk Of Cervical Intraepithelial Neoplasia (cin) Associated With Genetic Polymorphisms Of The Folate Metabolic Pathway In The Us Post Folic Acid Fortification Era.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1406.

MLA Handbook (7th Edition):

Badiga, Suguna. “Altered Dna Methylation Of Repetitive Elements May Explain The Modified Risk Of Cervical Intraepithelial Neoplasia (cin) Associated With Genetic Polymorphisms Of The Folate Metabolic Pathway In The Us Post Folic Acid Fortification Era.” 2011. Web. 29 Jan 2020.

Vancouver:

Badiga S. Altered Dna Methylation Of Repetitive Elements May Explain The Modified Risk Of Cervical Intraepithelial Neoplasia (cin) Associated With Genetic Polymorphisms Of The Folate Metabolic Pathway In The Us Post Folic Acid Fortification Era. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Jan 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1406.

Council of Science Editors:

Badiga S. Altered Dna Methylation Of Repetitive Elements May Explain The Modified Risk Of Cervical Intraepithelial Neoplasia (cin) Associated With Genetic Polymorphisms Of The Folate Metabolic Pathway In The Us Post Folic Acid Fortification Era. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1406

23. Belenky, Michael L. Sensitization of glioma to death receptor 5-mediated apoptosis through genotoxic stress and cell cycle disruption: a study of mechanism.

Degree: PhD, 2011, University of Alabama – Birmingham

Our laboratory reported that a combination of ionizing radiation (IR) or temozolomide (Tmz), a DNA methylating agent clinically approved against glioblastoma multiforme (GBM), a type… (more)

Subjects/Keywords: Antibodies, Monoclonal  – therapeutic use<; br>; Apoptosis  – physiology<; br>; Brain Neoplasms  – drug therapy<; br>; Brain Neoplasms  – radiotherapy<; br>; Cell Cycle<; br>; Glioma  – drug therapy<; br>; Glioma  – radiotherapy<; br>; Receptors, TNF-Related Apoptosis-Inducing Ligand  – agonists

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APA (6th Edition):

Belenky, M. L. (2011). Sensitization of glioma to death receptor 5-mediated apoptosis through genotoxic stress and cell cycle disruption: a study of mechanism. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1032

Chicago Manual of Style (16th Edition):

Belenky, Michael L. “Sensitization of glioma to death receptor 5-mediated apoptosis through genotoxic stress and cell cycle disruption: a study of mechanism.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1032.

MLA Handbook (7th Edition):

Belenky, Michael L. “Sensitization of glioma to death receptor 5-mediated apoptosis through genotoxic stress and cell cycle disruption: a study of mechanism.” 2011. Web. 29 Jan 2020.

Vancouver:

Belenky ML. Sensitization of glioma to death receptor 5-mediated apoptosis through genotoxic stress and cell cycle disruption: a study of mechanism. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Jan 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1032.

Council of Science Editors:

Belenky ML. Sensitization of glioma to death receptor 5-mediated apoptosis through genotoxic stress and cell cycle disruption: a study of mechanism. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1032

24. Warram, Jason M. (Jason Morgan). Strategies for combined screening and imaging of breast cancer.

Degree: PhD, 2011, University of Alabama – Birmingham

Successful treatment of breast cancer directly correlates with tumor stage and grade at diagnosis. Early diagnosis leads to a five-year relative survival rate of 96.8%… (more)

Subjects/Keywords: Adenoviridae  – genetics<; br>; Breast Neoplasms  – diagnosis<; br>; Gene Therapy<; br>; Mass Screening  – methods<; br>; Microbubbles<; br>; Neoplasm Metastasis<; br>; Promoter Regions, Genetic  – genetics<; br>; Vascular Endothelial Growth Factor Receptor-2  – immunology

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APA (6th Edition):

Warram, J. M. (. M. (2011). Strategies for combined screening and imaging of breast cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,881

Chicago Manual of Style (16th Edition):

Warram, Jason M (Jason Morgan). “Strategies for combined screening and imaging of breast cancer.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,881.

MLA Handbook (7th Edition):

Warram, Jason M (Jason Morgan). “Strategies for combined screening and imaging of breast cancer.” 2011. Web. 29 Jan 2020.

Vancouver:

Warram JM(M. Strategies for combined screening and imaging of breast cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Jan 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,881.

Council of Science Editors:

Warram JM(M. Strategies for combined screening and imaging of breast cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,881

25. Cuddapah, Vishnu Anand. Regulation Of Clc-3 In Human Malignant Glioma.

Degree: PhD, 2012, University of Alabama – Birmingham

Malignant gliomas are the most common and deadly form of primary brain cancer afflicting adults. Current treatment regimens, including surgical debulking, radiotherapy, and chemotherapy, have… (more)

Subjects/Keywords: Brain Neoplasms – metabolism<; br>; Calcium-Calmodulin-Dependent Protein Kinase Type 2 – metabolism.<; br>; Cell Movement – physiology<; br>; Chloride Channels – metabolism.<; br>; Gene Expression Regulation<; br>; Gene Expression Regulation, Enzymologic<; br>; Gene Expression Regulation, Neoplastic<; br>; Glioma – metabolism<; br>; Ion Channels – metabolism<; br>; Membrane Transport Proteins – metabolism.<; br>; Mitosis<; br>; Neoplasms – metabolism<; br>; Neoplasms – pathology

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APA (6th Edition):

Cuddapah, V. A. (2012). Regulation Of Clc-3 In Human Malignant Glioma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1394

Chicago Manual of Style (16th Edition):

Cuddapah, Vishnu Anand. “Regulation Of Clc-3 In Human Malignant Glioma.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1394.

MLA Handbook (7th Edition):

Cuddapah, Vishnu Anand. “Regulation Of Clc-3 In Human Malignant Glioma.” 2012. Web. 29 Jan 2020.

Vancouver:

Cuddapah VA. Regulation Of Clc-3 In Human Malignant Glioma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2020 Jan 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1394.

Council of Science Editors:

Cuddapah VA. Regulation Of Clc-3 In Human Malignant Glioma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1394

26. Cook, Leah M. (Leah Marie). Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice.

Degree: PhD, 2011, University of Alabama – Birmingham

Morbidity and mortality of breast cancer patients are drastically increased when primary tumor cells metastasize to distant organ sites. Effective treatment of metastatic disease has… (more)

Subjects/Keywords: Breast Neoplasms  – pathology<; br>; Mammary Neoplasms, Animal<; br>; Mice, Transgenic<; br>; Neoplasm Metastasis  – pathology<; br>; Tumor Microenvironment<; br>; Tumor Suppressor Proteins  – genetics<; br>; Tumor Suppressor Proteins  – metabolism

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APA (6th Edition):

Cook, L. M. (. M. (2011). Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1045

Chicago Manual of Style (16th Edition):

Cook, Leah M (Leah Marie). “Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1045.

MLA Handbook (7th Edition):

Cook, Leah M (Leah Marie). “Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice.” 2011. Web. 29 Jan 2020.

Vancouver:

Cook LM(M. Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Jan 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1045.

Council of Science Editors:

Cook LM(M. Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1045

27. Ling, Shiyun. Role Of 14-3-3&tau In Autophagy And Role Of Edd In P53 Regulation.

Degree: PhD, 2010, University of Alabama – Birmingham

Unrestricted cell proliferation and suppression of cell death are two essential events for tumor development. My dissertation research involves two proteins, 14-3-3 &tau and EDD… (more)

Subjects/Keywords: 14-3-3 Proteins – metabolism.<; br>; Breast Neoplasms<; br>; Down-Regulation<; br>; Membrane Proteins – genetics<; br>; Ovarian Neoplasms<; br>; Protein Processing, Post-Translational<; br>; Transcriptional Activation

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APA (6th Edition):

Ling, S. (2010). Role Of 14-3-3&tau In Autophagy And Role Of Edd In P53 Regulation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1419

Chicago Manual of Style (16th Edition):

Ling, Shiyun. “Role Of 14-3-3&tau In Autophagy And Role Of Edd In P53 Regulation.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1419.

MLA Handbook (7th Edition):

Ling, Shiyun. “Role Of 14-3-3&tau In Autophagy And Role Of Edd In P53 Regulation.” 2010. Web. 29 Jan 2020.

Vancouver:

Ling S. Role Of 14-3-3&tau In Autophagy And Role Of Edd In P53 Regulation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Jan 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1419.

Council of Science Editors:

Ling S. Role Of 14-3-3&tau In Autophagy And Role Of Edd In P53 Regulation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1419

28. Thal, Melissa Ann. Characterization of the induction and regulation of early B cell development.

Degree: PhD, 2009, University of Alabama – Birmingham

Hematopoiesis is a highly regulated process directed by the microenvironment or niche in the bone marrow and the transcription factors those signals activate. Gene knockout… (more)

Subjects/Keywords: B-Lymphocytes  – cytology<; br>; Down-Regulation<; br>; Inhibitor of Differentiation Protein 2  – genetics<; br>; Inhibitor of Differentiation Proteins  – genetics<; br>; Receptors, Interleukin-7  – deficiency<; br>; TCF Transcription Factors  – physiology<; br>; Trans-Activators  – physiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Thal, M. A. (2009). Characterization of the induction and regulation of early B cell development. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,578

Chicago Manual of Style (16th Edition):

Thal, Melissa Ann. “Characterization of the induction and regulation of early B cell development.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,578.

MLA Handbook (7th Edition):

Thal, Melissa Ann. “Characterization of the induction and regulation of early B cell development.” 2009. Web. 29 Jan 2020.

Vancouver:

Thal MA. Characterization of the induction and regulation of early B cell development. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Jan 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,578.

Council of Science Editors:

Thal MA. Characterization of the induction and regulation of early B cell development. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,578

29. Farmer, Tyesha Latrece. Genome copy number of African-American and European-American early-onset breast cancer.

Degree: PhD, 2009, University of Alabama – Birmingham

Breast cancer is the second leading cause of cancer related death among women in the United States, surpassed only by lung cancer. It is estimated… (more)

Subjects/Keywords: African Americans  – genetics<; br>; Breast Neoplasms  – genetics<; br>; DNA Copy Number Variations  – genetics<; br>; European Continental Ancestry Group  – genetics<; br>; Genetic Predisposition to Disease<; br>; Genomics

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Farmer, T. L. (2009). Genome copy number of African-American and European-American early-onset breast cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1001

Chicago Manual of Style (16th Edition):

Farmer, Tyesha Latrece. “Genome copy number of African-American and European-American early-onset breast cancer.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1001.

MLA Handbook (7th Edition):

Farmer, Tyesha Latrece. “Genome copy number of African-American and European-American early-onset breast cancer.” 2009. Web. 29 Jan 2020.

Vancouver:

Farmer TL. Genome copy number of African-American and European-American early-onset breast cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Jan 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1001.

Council of Science Editors:

Farmer TL. Genome copy number of African-American and European-American early-onset breast cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1001

30. Bomben, Valerie Christine. Role of transient receptor potential canonical channels in glioma cell biology.

Degree: PhD, 2010, University of Alabama – Birmingham

Gliomas, primary brain tumors derived from glial cells, constitute the majority of malignant tumors within the central nervous system. The most malignant of these tumors,… (more)

Subjects/Keywords: Brain Neoplasms  – pathology<; br>; Cell Cycle<; br>; Glioma  – pathology<; br>; Transient Receptor Potential Channels  – antagonists & inhibitors

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bomben, V. C. (2010). Role of transient receptor potential canonical channels in glioma cell biology. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,642

Chicago Manual of Style (16th Edition):

Bomben, Valerie Christine. “Role of transient receptor potential canonical channels in glioma cell biology.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,642.

MLA Handbook (7th Edition):

Bomben, Valerie Christine. “Role of transient receptor potential canonical channels in glioma cell biology.” 2010. Web. 29 Jan 2020.

Vancouver:

Bomben VC. Role of transient receptor potential canonical channels in glioma cell biology. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Jan 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,642.

Council of Science Editors:

Bomben VC. Role of transient receptor potential canonical channels in glioma cell biology. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,642

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