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You searched for subject:( Bone Neoplasms 60). Showing records 1 – 30 of 12597 total matches.

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1. Szafran, April Adams. The use of molecular imaging to evaluate the efficacy of treatments for metastatic breast cancer.

Degree: PhD, 2008, University of Alabama – Birmingham

The development of molecular imaging technologies has allowed biomedical researchers to study the process of cancer metastasis in animal models of disease. Bioluminescence imaging has… (more)

Subjects/Keywords: Antibodies, Monoclonal  – pharmacology<; br>; Antineoplastic Combined Chemotherapy<; br>; Protocols  – pharmacology<; br>; Bone Neoplasms<; br>; Breast Neoplasms<; br>; Diphosphonates  – pharmacology<; br>; Imidazoles  – pharmacology<; br>; Mammary Neoplasms, Experimental  – pathology<; br>; Receptors, TNF-Related Apoptosis-Inducing Ligand  – agonists

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APA (6th Edition):

Szafran, A. A. (2008). The use of molecular imaging to evaluate the efficacy of treatments for metastatic breast cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,927

Chicago Manual of Style (16th Edition):

Szafran, April Adams. “The use of molecular imaging to evaluate the efficacy of treatments for metastatic breast cancer.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,927.

MLA Handbook (7th Edition):

Szafran, April Adams. “The use of molecular imaging to evaluate the efficacy of treatments for metastatic breast cancer.” 2008. Web. 29 Mar 2020.

Vancouver:

Szafran AA. The use of molecular imaging to evaluate the efficacy of treatments for metastatic breast cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,927.

Council of Science Editors:

Szafran AA. The use of molecular imaging to evaluate the efficacy of treatments for metastatic breast cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,927

2. Cody, James Joseph. A dual-action, armed replicating adenovirus for the treatment of bone metastases of breast cancer.

Degree: PhD, 2008, University of Alabama – Birmingham

Most patients with advanced breast cancer develop osteolytic bone metastases, which have numerous complications. Because current therapies are not curative, new treatments are needed. Conditionally… (more)

Subjects/Keywords: Adenoviridae  – genetics <; br>; Bone Neoplasms  – secondary <; br>; Breast Neoplasms <; br>; Gene Expression Regulation, Neoplastic <; br>; Neoplasm Metastasis  – genetics <; br>; Neoplasm Metastasis  – therapy <; br>; Virus Replication  – genetics

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APA (6th Edition):

Cody, J. J. (2008). A dual-action, armed replicating adenovirus for the treatment of bone metastases of breast cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,320

Chicago Manual of Style (16th Edition):

Cody, James Joseph. “A dual-action, armed replicating adenovirus for the treatment of bone metastases of breast cancer.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,320.

MLA Handbook (7th Edition):

Cody, James Joseph. “A dual-action, armed replicating adenovirus for the treatment of bone metastases of breast cancer.” 2008. Web. 29 Mar 2020.

Vancouver:

Cody JJ. A dual-action, armed replicating adenovirus for the treatment of bone metastases of breast cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,320.

Council of Science Editors:

Cody JJ. A dual-action, armed replicating adenovirus for the treatment of bone metastases of breast cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,320

3. Gavin, Cristin. Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation.

Degree: PhD, 2012, University of Alabama – Birmingham

Dynamic changes to the actin cytoskeleton are required for synaptic plasticity and long-term memory formation. However, the molecular mechanisms that mediate filamentous actin (F-actin) dynamics… (more)

Subjects/Keywords: Bone Neoplasms – secondary<; br>; Breast Neoplasms – pathology<; br>; Cell Communication<; br>; Cyclic AMP-Dependent Protein Kinases – metabolism.<; br>; Gap Junctions – metabolism.<; br>; Osteoblasts – cytology.<; br>; Phosphatidylinositol 3-Kinases – metabolism.

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APA (6th Edition):

Gavin, C. (2012). Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1396

Chicago Manual of Style (16th Edition):

Gavin, Cristin. “Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1396.

MLA Handbook (7th Edition):

Gavin, Cristin. “Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation.” 2012. Web. 29 Mar 2020.

Vancouver:

Gavin C. Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1396.

Council of Science Editors:

Gavin C. Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1396

4. Bispo Júnior, Rosalvo Zosimo. Fatores prognósticos da sobrevida no osteossarcoma primário: grau I versus II de Huvos.

Degree: PhD, Ortopedia e Traumatologia, 2009, University of São Paulo

O objetivo deste trabalho foi comparar o prognóstico de sobrevida da graduação histológica após efeito da quimioterapia (graus I versus II de Huvos), visando também… (more)

Subjects/Keywords: Bone neoplasms/drug therapy; Bone neoplasms/epidemiology; Bone neoplasms/pathology; Bone neoplasms/surgery; Neoplasias ósseas/cirurgia; Neoplasias ósseas/epidemiologia; Neoplasias ósseas/patologia; Neoplasias ósseas/quimioterapia

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APA (6th Edition):

Bispo Júnior, R. Z. (2009). Fatores prognósticos da sobrevida no osteossarcoma primário: grau I versus II de Huvos. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5140/tde-24022010-153548/ ;

Chicago Manual of Style (16th Edition):

Bispo Júnior, Rosalvo Zosimo. “Fatores prognósticos da sobrevida no osteossarcoma primário: grau I versus II de Huvos.” 2009. Doctoral Dissertation, University of São Paulo. Accessed March 29, 2020. http://www.teses.usp.br/teses/disponiveis/5/5140/tde-24022010-153548/ ;.

MLA Handbook (7th Edition):

Bispo Júnior, Rosalvo Zosimo. “Fatores prognósticos da sobrevida no osteossarcoma primário: grau I versus II de Huvos.” 2009. Web. 29 Mar 2020.

Vancouver:

Bispo Júnior RZ. Fatores prognósticos da sobrevida no osteossarcoma primário: grau I versus II de Huvos. [Internet] [Doctoral dissertation]. University of São Paulo; 2009. [cited 2020 Mar 29]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5140/tde-24022010-153548/ ;.

Council of Science Editors:

Bispo Júnior RZ. Fatores prognósticos da sobrevida no osteossarcoma primário: grau I versus II de Huvos. [Doctoral Dissertation]. University of São Paulo; 2009. Available from: http://www.teses.usp.br/teses/disponiveis/5/5140/tde-24022010-153548/ ;

5. Hensel, Jonathan Adam. The role of LL-37 in prostate cancer and its potential as a therapeutic target.

Degree: 2011, University of Alabama – Birmingham

LL-37 is the only cathelicidin-derived anti-microbial peptide in humans and has been shown to stimulate proliferation, angiogenesis, cellular migration and inhibit apoptosis, in addition to… (more)

Subjects/Keywords: Antimicrobial Cationic Peptides  – metabolism<; br>; Cathelicidins<; br>; Molecular Targeted Therapy  – methods<; br>; Neoplasms, Hormone-Dependent<; br>; Prostatic Neoplasms  – genetics<; br>; Prostatic Neoplasms  – metabolism<; br>; Prostatic Neoplasms  – pathology

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APA (6th Edition):

Hensel, J. A. (2011). The role of LL-37 in prostate cancer and its potential as a therapeutic target. (Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,875

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hensel, Jonathan Adam. “The role of LL-37 in prostate cancer and its potential as a therapeutic target.” 2011. Thesis, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,875.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hensel, Jonathan Adam. “The role of LL-37 in prostate cancer and its potential as a therapeutic target.” 2011. Web. 29 Mar 2020.

Vancouver:

Hensel JA. The role of LL-37 in prostate cancer and its potential as a therapeutic target. [Internet] [Thesis]. University of Alabama – Birmingham; 2011. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,875.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hensel JA. The role of LL-37 in prostate cancer and its potential as a therapeutic target. [Thesis]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,875

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

6. Anderson, Joel M. (Joel Matthew). Biomimetic self-assembled nanomatrix for bone tissue regeneration.

Degree: MS, 2008, University of Alabama – Birmingham

Bone is a dense, connective tissue with an extracellular matrix (ECM) comprised of biological and organic/inorganic biphasic matrices. For this study, novel peptide amphiphile (PA)… (more)

Subjects/Keywords: Bone regeneration <; br>; Bone  – Differentiation <; br>; Extracellular matrix <; br>; Stem cells <; br>; Nanostructured materials <; br>; Biomimetics

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APA (6th Edition):

Anderson, J. M. (. M. (2008). Biomimetic self-assembled nanomatrix for bone tissue regeneration. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,342

Chicago Manual of Style (16th Edition):

Anderson, Joel M (Joel Matthew). “Biomimetic self-assembled nanomatrix for bone tissue regeneration.” 2008. Masters Thesis, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,342.

MLA Handbook (7th Edition):

Anderson, Joel M (Joel Matthew). “Biomimetic self-assembled nanomatrix for bone tissue regeneration.” 2008. Web. 29 Mar 2020.

Vancouver:

Anderson JM(M. Biomimetic self-assembled nanomatrix for bone tissue regeneration. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2008. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,342.

Council of Science Editors:

Anderson JM(M. Biomimetic self-assembled nanomatrix for bone tissue regeneration. [Masters Thesis]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,342

7. Steg, Adam. Analysis of the hedgehog pathway in pancreatic adenocarcinoma.

Degree: PhD, 2008, University of Alabama – Birmingham

Pancreatic adenocarcinoma (PAC) is the fourth leading cause of cancer mortality in the United States. Despite the use of highly aggressive treatment regimens (surgery, chemotherapy… (more)

Subjects/Keywords: Adenocarcinoma  – metabolism <; br>; Adenocarcinoma  – pathology <; br>; Hedgehog Proteins  – metabolism <; br>; Pancreatic Neoplasms  – metabolism <; br>; Pancreatic Neoplasms  – pathology

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APA (6th Edition):

Steg, A. (2008). Analysis of the hedgehog pathway in pancreatic adenocarcinoma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,290

Chicago Manual of Style (16th Edition):

Steg, Adam. “Analysis of the hedgehog pathway in pancreatic adenocarcinoma.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,290.

MLA Handbook (7th Edition):

Steg, Adam. “Analysis of the hedgehog pathway in pancreatic adenocarcinoma.” 2008. Web. 29 Mar 2020.

Vancouver:

Steg A. Analysis of the hedgehog pathway in pancreatic adenocarcinoma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,290.

Council of Science Editors:

Steg A. Analysis of the hedgehog pathway in pancreatic adenocarcinoma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,290

8. Prakash, Anand, M.S. Characterization of synergistic effect of iododeoxyuridine and clofarabine in cisplatin-resistant ovarian cancer cells .

Degree: MS, 2009, University of Alabama – Birmingham

Epithelial ovarian cancer (EOC) is the most common gynecological cancer and the fifth most frequent cause of cancer deaths in women. Cytoreductive surgery (CRS) followed… (more)

Subjects/Keywords: Cisplatin  – pharmacology<; br>; Drug Resistance, Neoplasm<; br>; Drug Synergism<; br>; Ovarian Neoplasms  – drug therapy

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APA (6th Edition):

Prakash, Anand, M. S. (2009). Characterization of synergistic effect of iododeoxyuridine and clofarabine in cisplatin-resistant ovarian cancer cells . (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,421

Chicago Manual of Style (16th Edition):

Prakash, Anand, M S. “Characterization of synergistic effect of iododeoxyuridine and clofarabine in cisplatin-resistant ovarian cancer cells .” 2009. Masters Thesis, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,421.

MLA Handbook (7th Edition):

Prakash, Anand, M S. “Characterization of synergistic effect of iododeoxyuridine and clofarabine in cisplatin-resistant ovarian cancer cells .” 2009. Web. 29 Mar 2020.

Vancouver:

Prakash, Anand MS. Characterization of synergistic effect of iododeoxyuridine and clofarabine in cisplatin-resistant ovarian cancer cells . [Internet] [Masters thesis]. University of Alabama – Birmingham; 2009. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,421.

Council of Science Editors:

Prakash, Anand MS. Characterization of synergistic effect of iododeoxyuridine and clofarabine in cisplatin-resistant ovarian cancer cells . [Masters Thesis]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,421

9. Evans, Lynda Michele. The effect of stearic acid on breast cancer development and progression.

Degree: PhD, 2009, University of Alabama – Birmingham

Stearate is an 18-carbon saturated fatty acid that is found in many foods in the western diet including beef and chocolate. Cell culture studies indicate… (more)

Subjects/Keywords: Antineoplastic Agents  – administration & dosage<; br>; Apoptosis<; br>; Breast Neoplasms  – metabolism<; br>; Breast Neoplasms  – pathology<; br>; Dietary Fats<; br>; Neoplasm Metastasis<; br>; Stearates  – therapeutic use<; br>; Stearic Acids  – administration & dosage

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APA (6th Edition):

Evans, L. M. (2009). The effect of stearic acid on breast cancer development and progression. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,906

Chicago Manual of Style (16th Edition):

Evans, Lynda Michele. “The effect of stearic acid on breast cancer development and progression.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,906.

MLA Handbook (7th Edition):

Evans, Lynda Michele. “The effect of stearic acid on breast cancer development and progression.” 2009. Web. 29 Mar 2020.

Vancouver:

Evans LM. The effect of stearic acid on breast cancer development and progression. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,906.

Council of Science Editors:

Evans LM. The effect of stearic acid on breast cancer development and progression. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,906

10. Shaikh, Faheem M. Role of variant sialylation in regulating tumor cell behavior.

Degree: PhD, 2008, University of Alabama – Birmingham

Many different tumors have been documented to have elevated levels of the enzyme ST6Gal I, a Golgi glycosyltransferase that adds [alpha]2-6 sialic acids to glycoproteins.… (more)

Subjects/Keywords: Cell Movement  – physiology <; br>; Colonic Neoplasms <; br>; Integrins  – chemistry <; br>; Integrins  – metabolism <; br>; Neoplasm Invasiveness <; br>; Sialic Acids  – metabolism

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APA (6th Edition):

Shaikh, F. M. (2008). Role of variant sialylation in regulating tumor cell behavior. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,266

Chicago Manual of Style (16th Edition):

Shaikh, Faheem M. “Role of variant sialylation in regulating tumor cell behavior.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,266.

MLA Handbook (7th Edition):

Shaikh, Faheem M. “Role of variant sialylation in regulating tumor cell behavior.” 2008. Web. 29 Mar 2020.

Vancouver:

Shaikh FM. Role of variant sialylation in regulating tumor cell behavior. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,266.

Council of Science Editors:

Shaikh FM. Role of variant sialylation in regulating tumor cell behavior. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,266

11. Montgomery, Michele. Physical therapy and chiropractic use in survivors of childhood cancer: impact on health-related quality of life.

Degree: PhD, 2009, University of Alabama – Birmingham

The purpose of this study to was examine prevalence of physical therapy (PT) and chiropractic use in survivors of childhood cancer and which factors are… (more)

Subjects/Keywords: Exercise<; br>; Musculoskeletal Manipulations<; br>; Neoplasms  – therapy<; br>; Quality of Life<; br>; Retrospective Studies<; br>; Survivors

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APA (6th Edition):

Montgomery, M. (2009). Physical therapy and chiropractic use in survivors of childhood cancer: impact on health-related quality of life. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,837

Chicago Manual of Style (16th Edition):

Montgomery, Michele. “Physical therapy and chiropractic use in survivors of childhood cancer: impact on health-related quality of life.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,837.

MLA Handbook (7th Edition):

Montgomery, Michele. “Physical therapy and chiropractic use in survivors of childhood cancer: impact on health-related quality of life.” 2009. Web. 29 Mar 2020.

Vancouver:

Montgomery M. Physical therapy and chiropractic use in survivors of childhood cancer: impact on health-related quality of life. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,837.

Council of Science Editors:

Montgomery M. Physical therapy and chiropractic use in survivors of childhood cancer: impact on health-related quality of life. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,837

12. Volker, Susan E. Development and validation of a palliative care needs assessment instrument-English/Arabic versions (PCNA - EAV), for use with patients with advanced cancer.

Degree: PhD, 2010, University of Alabama – Birmingham

The aim of this study is to develop and translate a psychometrically valid and reliable, population-based, needs assessment instrument, the PCNA-EAV, to measure the health… (more)

Subjects/Keywords: Health Services Needs and Demand<; br>; Needs Assessment<; br>; Neoplasms<; br>; Palliative Care<; br>; Questionnaires<; br>; Reproducibility of Results

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APA (6th Edition):

Volker, S. E. (2010). Development and validation of a palliative care needs assessment instrument-English/Arabic versions (PCNA - EAV), for use with patients with advanced cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,938

Chicago Manual of Style (16th Edition):

Volker, Susan E. “Development and validation of a palliative care needs assessment instrument-English/Arabic versions (PCNA - EAV), for use with patients with advanced cancer.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,938.

MLA Handbook (7th Edition):

Volker, Susan E. “Development and validation of a palliative care needs assessment instrument-English/Arabic versions (PCNA - EAV), for use with patients with advanced cancer.” 2010. Web. 29 Mar 2020.

Vancouver:

Volker SE. Development and validation of a palliative care needs assessment instrument-English/Arabic versions (PCNA - EAV), for use with patients with advanced cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,938.

Council of Science Editors:

Volker SE. Development and validation of a palliative care needs assessment instrument-English/Arabic versions (PCNA - EAV), for use with patients with advanced cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,938

13. Ogunrinu, Toyin Adeyemi. Role of the cystine-glutamate exchanger in glioma cell biology.

Degree: PhD, 2010, University of Alabama – Birmingham

Changes in the glioma microenvironment including oxygen (O2) levels, supply of amino acid such as L-glutamate and L-cystine and glutathione (GSH) concentrations play a critical… (more)

Subjects/Keywords: Anoxia  – metabolism<; br>; Brain Neoplasms  – metabolism<; br>; Glioblastoma  – metabolism<; br>; Glioma  – metabolism<; br>; Glutathione  – metabolism<; br>; Glutamic Acid  – metabolism

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APA (6th Edition):

Ogunrinu, T. A. (2010). Role of the cystine-glutamate exchanger in glioma cell biology. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,956

Chicago Manual of Style (16th Edition):

Ogunrinu, Toyin Adeyemi. “Role of the cystine-glutamate exchanger in glioma cell biology.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,956.

MLA Handbook (7th Edition):

Ogunrinu, Toyin Adeyemi. “Role of the cystine-glutamate exchanger in glioma cell biology.” 2010. Web. 29 Mar 2020.

Vancouver:

Ogunrinu TA. Role of the cystine-glutamate exchanger in glioma cell biology. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,956.

Council of Science Editors:

Ogunrinu TA. Role of the cystine-glutamate exchanger in glioma cell biology. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,956

14. Robert, Stephanie Marie. Cystine/glutamate Transporters As Prognostic & Therapeutic Markers Of Primary Brain Tumors.

Degree: 2012, University of Alabama – Birmingham

Glioblastoma multiforme (GBM) are the most prevalent and aggressive malignant brain tumors. Current treatment - a combination of radiation, chemotherapy and resection - has limited… (more)

Subjects/Keywords: Amino Acid Transport System X-AG – metabolism.<; br>; Brain Neoplasms – metabolism<; br>; Glioblastoma.<; br>; Glutathione<; br>; Seizures<; br>; Sulfasalazine.

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APA (6th Edition):

Robert, S. M. (2012). Cystine/glutamate Transporters As Prognostic & Therapeutic Markers Of Primary Brain Tumors. (Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1791

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Robert, Stephanie Marie. “Cystine/glutamate Transporters As Prognostic & Therapeutic Markers Of Primary Brain Tumors.” 2012. Thesis, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1791.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Robert, Stephanie Marie. “Cystine/glutamate Transporters As Prognostic & Therapeutic Markers Of Primary Brain Tumors.” 2012. Web. 29 Mar 2020.

Vancouver:

Robert SM. Cystine/glutamate Transporters As Prognostic & Therapeutic Markers Of Primary Brain Tumors. [Internet] [Thesis]. University of Alabama – Birmingham; 2012. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1791.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Robert SM. Cystine/glutamate Transporters As Prognostic & Therapeutic Markers Of Primary Brain Tumors. [Thesis]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1791

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. Zamani, Ali. Serotonin signaling in angiogenesis.

Degree: PhD, 2012, University of Alabama – Birmingham

Serotonin (5-hydroxytryptamine, 5-HT) is a well-known "happy" neurotransmitter. Selective serotonin reuptake inhibitors (SSRIs) are the most prescribed class of antidepressant medications to raise the serotonin… (more)

Subjects/Keywords: Angiogenesis Inducing Agents – pharmacology <; br>; Blood Platelets<; br>; HTR1B protein, human<; br>; Neoplasms<; br>; Serotonin<; br>; Vascular Endothelial Growth Factors

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APA (6th Edition):

Zamani, A. (2012). Serotonin signaling in angiogenesis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1383

Chicago Manual of Style (16th Edition):

Zamani, Ali. “Serotonin signaling in angiogenesis.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1383.

MLA Handbook (7th Edition):

Zamani, Ali. “Serotonin signaling in angiogenesis.” 2012. Web. 29 Mar 2020.

Vancouver:

Zamani A. Serotonin signaling in angiogenesis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1383.

Council of Science Editors:

Zamani A. Serotonin signaling in angiogenesis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1383

16. Leavitt, Curry H. Correlation of bone density using histomophometric and cone beam computed tomographic analysis.

Degree: MS, 2010, University of Alabama – Birmingham

Purpose: The aim of this study is to evaluate bone density measured by both histomorphometric analysis of bone biopsies at healed, previously grafted extraction sockets… (more)

Subjects/Keywords: Analysis of Variance<; br>; Bone and Bones  – analysis<; br>; Bone Density<; br>; Dental Implants<; br>; Tomography, X-Ray Computed

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APA (6th Edition):

Leavitt, C. H. (2010). Correlation of bone density using histomophometric and cone beam computed tomographic analysis. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,733

Chicago Manual of Style (16th Edition):

Leavitt, Curry H. “Correlation of bone density using histomophometric and cone beam computed tomographic analysis.” 2010. Masters Thesis, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,733.

MLA Handbook (7th Edition):

Leavitt, Curry H. “Correlation of bone density using histomophometric and cone beam computed tomographic analysis.” 2010. Web. 29 Mar 2020.

Vancouver:

Leavitt CH. Correlation of bone density using histomophometric and cone beam computed tomographic analysis. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2010. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,733.

Council of Science Editors:

Leavitt CH. Correlation of bone density using histomophometric and cone beam computed tomographic analysis. [Masters Thesis]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,733


University of Western Australia

17. Eager, Katrise Mary. Rehabilitation of unilateral profound sensorineural hearing loss with a bone anchored hearing aid.

Degree: M. Med. Sc., 2009, University of Western Australia

The long-term outcomes of subjects fitted with a bone anchored hearing aid (BAHA) for a unilateral profound sensorineural hearing loss (UPSHL) are still evolving. Previous… (more)

Subjects/Keywords: Audiology; Bone-anchored hearing aids; Deafness; Hearing aids; Hearing disorders; <; bone anchored hearing aid>;

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APA (6th Edition):

Eager, K. M. (2009). Rehabilitation of unilateral profound sensorineural hearing loss with a bone anchored hearing aid. (Masters Thesis). University of Western Australia. Retrieved from http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=12832&local_base=GEN01-INS01

Chicago Manual of Style (16th Edition):

Eager, Katrise Mary. “Rehabilitation of unilateral profound sensorineural hearing loss with a bone anchored hearing aid.” 2009. Masters Thesis, University of Western Australia. Accessed March 29, 2020. http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=12832&local_base=GEN01-INS01.

MLA Handbook (7th Edition):

Eager, Katrise Mary. “Rehabilitation of unilateral profound sensorineural hearing loss with a bone anchored hearing aid.” 2009. Web. 29 Mar 2020.

Vancouver:

Eager KM. Rehabilitation of unilateral profound sensorineural hearing loss with a bone anchored hearing aid. [Internet] [Masters thesis]. University of Western Australia; 2009. [cited 2020 Mar 29]. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=12832&local_base=GEN01-INS01.

Council of Science Editors:

Eager KM. Rehabilitation of unilateral profound sensorineural hearing loss with a bone anchored hearing aid. [Masters Thesis]. University of Western Australia; 2009. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=12832&local_base=GEN01-INS01

18. Cuddapah, Vishnu Anand. Regulation Of Clc-3 In Human Malignant Glioma.

Degree: PhD, 2012, University of Alabama – Birmingham

Malignant gliomas are the most common and deadly form of primary brain cancer afflicting adults. Current treatment regimens, including surgical debulking, radiotherapy, and chemotherapy, have… (more)

Subjects/Keywords: Brain Neoplasms – metabolism<; br>; Calcium-Calmodulin-Dependent Protein Kinase Type 2 – metabolism.<; br>; Cell Movement – physiology<; br>; Chloride Channels – metabolism.<; br>; Gene Expression Regulation<; br>; Gene Expression Regulation, Enzymologic<; br>; Gene Expression Regulation, Neoplastic<; br>; Glioma – metabolism<; br>; Ion Channels – metabolism<; br>; Membrane Transport Proteins – metabolism.<; br>; Mitosis<; br>; Neoplasms – metabolism<; br>; Neoplasms – pathology

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APA (6th Edition):

Cuddapah, V. A. (2012). Regulation Of Clc-3 In Human Malignant Glioma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1394

Chicago Manual of Style (16th Edition):

Cuddapah, Vishnu Anand. “Regulation Of Clc-3 In Human Malignant Glioma.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1394.

MLA Handbook (7th Edition):

Cuddapah, Vishnu Anand. “Regulation Of Clc-3 In Human Malignant Glioma.” 2012. Web. 29 Mar 2020.

Vancouver:

Cuddapah VA. Regulation Of Clc-3 In Human Malignant Glioma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1394.

Council of Science Editors:

Cuddapah VA. Regulation Of Clc-3 In Human Malignant Glioma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1394

19. Livada, Rania. Histomorphometric and bone densitometric analysis of dental implant sites.

Degree: MS, 2009, University of Alabama – Birmingham

Purpose: The purpose of this study was to assess bone density at implant sites by four types of means: a) clinical measurements b) radiographic measurements… (more)

Subjects/Keywords: Analysis of Variance<; br>; Bone Density  – physiology<; br>; Dental Implants<; br>; Tomography, X-Ray Computed

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APA (6th Edition):

Livada, R. (2009). Histomorphometric and bone densitometric analysis of dental implant sites. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1023

Chicago Manual of Style (16th Edition):

Livada, Rania. “Histomorphometric and bone densitometric analysis of dental implant sites.” 2009. Masters Thesis, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1023.

MLA Handbook (7th Edition):

Livada, Rania. “Histomorphometric and bone densitometric analysis of dental implant sites.” 2009. Web. 29 Mar 2020.

Vancouver:

Livada R. Histomorphometric and bone densitometric analysis of dental implant sites. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2009. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1023.

Council of Science Editors:

Livada R. Histomorphometric and bone densitometric analysis of dental implant sites. [Masters Thesis]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1023

20. Kojic, Dave D. Histomorphometry and quantative analysis of long-term inplant interfaces.

Degree: MS, 2012, University of Alabama – Birmingham

Objective: This investigation was to evaluate (>10 year) bone response around custom (HA plasma coated) dental implants and HA particulate. Materials and Methods: Three individual… (more)

Subjects/Keywords: Bone and Bones – anatomy & histology<; br>; Dental Implants<; br>; Hydroxyapatites<; br>; Osteoclasts – cytology

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APA (6th Edition):

Kojic, D. D. (2012). Histomorphometry and quantative analysis of long-term inplant interfaces. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1372

Chicago Manual of Style (16th Edition):

Kojic, Dave D. “Histomorphometry and quantative analysis of long-term inplant interfaces.” 2012. Masters Thesis, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1372.

MLA Handbook (7th Edition):

Kojic, Dave D. “Histomorphometry and quantative analysis of long-term inplant interfaces.” 2012. Web. 29 Mar 2020.

Vancouver:

Kojic DD. Histomorphometry and quantative analysis of long-term inplant interfaces. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2012. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1372.

Council of Science Editors:

Kojic DD. Histomorphometry and quantative analysis of long-term inplant interfaces. [Masters Thesis]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1372

21. Kojima, Kyoko, Ph.D. Mouse modeling of pancreatic ductal adenocarcinoma (PDAC): search for early diagnostic markers and therapeutic targets.

Degree: PhD, 2009, University of Alabama – Birmingham

PDAC is a highly malignant neoplasm that carries a very poor prognosis. PDAC development is a multistage transformation process that involves multiple genetic alterations that… (more)

Subjects/Keywords: Carcinoma in Situ  – genetics<; br>; Genes, ras<; br>; Pancreatic Neoplasms  – diagnosis<; br>; Pancreatic Neoplasms  – genetics<; br>; Proteomics<; br>; Smad4 Protein  – genetics<; br>; Tumor Markers, Biological  – analysis

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APA (6th Edition):

Kojima, Kyoko, P. D. (2009). Mouse modeling of pancreatic ductal adenocarcinoma (PDAC): search for early diagnostic markers and therapeutic targets. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,396

Chicago Manual of Style (16th Edition):

Kojima, Kyoko, Ph D. “Mouse modeling of pancreatic ductal adenocarcinoma (PDAC): search for early diagnostic markers and therapeutic targets.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,396.

MLA Handbook (7th Edition):

Kojima, Kyoko, Ph D. “Mouse modeling of pancreatic ductal adenocarcinoma (PDAC): search for early diagnostic markers and therapeutic targets.” 2009. Web. 29 Mar 2020.

Vancouver:

Kojima, Kyoko PD. Mouse modeling of pancreatic ductal adenocarcinoma (PDAC): search for early diagnostic markers and therapeutic targets. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,396.

Council of Science Editors:

Kojima, Kyoko PD. Mouse modeling of pancreatic ductal adenocarcinoma (PDAC): search for early diagnostic markers and therapeutic targets. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,396

22. Anderson, Rashaan. The role of community-based programs in addressing health disparities as it relates to breast and cervical cancer in African American women : systematic review of studies.

Degree: PhD, 2008, University of Alabama – Birmingham

In an effort to extrapolate the detail intricacies of how community-based programs impact public health a systematic review of the literature was performed. This research… (more)

Subjects/Keywords: African Americans<; br>; Breast Neoplasms<; br>; Community Health Services<; br>; Health Services Accessibility<; br>; Health Status Disparities<; br>; Uterine Cervical Neoplasms<; br>; Women

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APA (6th Edition):

Anderson, R. (2008). The role of community-based programs in addressing health disparities as it relates to breast and cervical cancer in African American women : systematic review of studies. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,429

Chicago Manual of Style (16th Edition):

Anderson, Rashaan. “The role of community-based programs in addressing health disparities as it relates to breast and cervical cancer in African American women : systematic review of studies.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,429.

MLA Handbook (7th Edition):

Anderson, Rashaan. “The role of community-based programs in addressing health disparities as it relates to breast and cervical cancer in African American women : systematic review of studies.” 2008. Web. 29 Mar 2020.

Vancouver:

Anderson R. The role of community-based programs in addressing health disparities as it relates to breast and cervical cancer in African American women : systematic review of studies. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,429.

Council of Science Editors:

Anderson R. The role of community-based programs in addressing health disparities as it relates to breast and cervical cancer in African American women : systematic review of studies. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,429

23. Kwon, Yeon-Jin. The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis.

Degree: PhD, 2010, University of Alabama – Birmingham

Glioma-associated oncogene homolog 1 (Gli1) is a well-known oncogene and a transcription factor that mediates several signaling pathways important for tumor progression, such as hedgehog,… (more)

Subjects/Keywords: Breast Neoplasms  – metabolism<; br>; Breast Neoplasms  – pathology<; br>; Cell Movement<; br>; Estrogen Receptor alpha  – deficiency<; br>; Matrix Metalloproteinase 11  – metabolism<; br>; Transcription Factors  – metabolism<; br>; Up-Regulation

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APA (6th Edition):

Kwon, Y. (2010). The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,913

Chicago Manual of Style (16th Edition):

Kwon, Yeon-Jin. “The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,913.

MLA Handbook (7th Edition):

Kwon, Yeon-Jin. “The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis.” 2010. Web. 29 Mar 2020.

Vancouver:

Kwon Y. The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,913.

Council of Science Editors:

Kwon Y. The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,913

24. Cook, Leah M. (Leah Marie). Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice.

Degree: PhD, 2011, University of Alabama – Birmingham

Morbidity and mortality of breast cancer patients are drastically increased when primary tumor cells metastasize to distant organ sites. Effective treatment of metastatic disease has… (more)

Subjects/Keywords: Breast Neoplasms  – pathology<; br>; Mammary Neoplasms, Animal<; br>; Mice, Transgenic<; br>; Neoplasm Metastasis  – pathology<; br>; Tumor Microenvironment<; br>; Tumor Suppressor Proteins  – genetics<; br>; Tumor Suppressor Proteins  – metabolism

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APA (6th Edition):

Cook, L. M. (. M. (2011). Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1045

Chicago Manual of Style (16th Edition):

Cook, Leah M (Leah Marie). “Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1045.

MLA Handbook (7th Edition):

Cook, Leah M (Leah Marie). “Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice.” 2011. Web. 29 Mar 2020.

Vancouver:

Cook LM(M. Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1045.

Council of Science Editors:

Cook LM(M. Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1045

25. Ling, Shiyun. Role Of 14-3-3&tau In Autophagy And Role Of Edd In P53 Regulation.

Degree: PhD, 2010, University of Alabama – Birmingham

Unrestricted cell proliferation and suppression of cell death are two essential events for tumor development. My dissertation research involves two proteins, 14-3-3 &tau and EDD… (more)

Subjects/Keywords: 14-3-3 Proteins – metabolism.<; br>; Breast Neoplasms<; br>; Down-Regulation<; br>; Membrane Proteins – genetics<; br>; Ovarian Neoplasms<; br>; Protein Processing, Post-Translational<; br>; Transcriptional Activation

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APA (6th Edition):

Ling, S. (2010). Role Of 14-3-3&tau In Autophagy And Role Of Edd In P53 Regulation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1419

Chicago Manual of Style (16th Edition):

Ling, Shiyun. “Role Of 14-3-3&tau In Autophagy And Role Of Edd In P53 Regulation.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1419.

MLA Handbook (7th Edition):

Ling, Shiyun. “Role Of 14-3-3&tau In Autophagy And Role Of Edd In P53 Regulation.” 2010. Web. 29 Mar 2020.

Vancouver:

Ling S. Role Of 14-3-3&tau In Autophagy And Role Of Edd In P53 Regulation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1419.

Council of Science Editors:

Ling S. Role Of 14-3-3&tau In Autophagy And Role Of Edd In P53 Regulation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1419

26. Virk-Baker, Mandeep K. Soy And Breast Cancer.

Degree: PhD, 2012, University of Alabama – Birmingham

The incidence of breast cancer is lower in Asia as compared to Western countries. However, the breast cancer risk among Asian immigrants living in Western… (more)

Subjects/Keywords: Antineoplastic Agents, Phytogenic – therapeutic use.<; br>; Biopsy<; br>; Breast Neoplasms – prevention & control<; br>; Isoflavones – therapeutic use.<; br>; Neoplasms – drug therapy<; br>; Phytoestrogens – therapeutic use.<; br>; Risk Factors

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APA (6th Edition):

Virk-Baker, M. K. (2012). Soy And Breast Cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1430

Chicago Manual of Style (16th Edition):

Virk-Baker, Mandeep K. “Soy And Breast Cancer.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1430.

MLA Handbook (7th Edition):

Virk-Baker, Mandeep K. “Soy And Breast Cancer.” 2012. Web. 29 Mar 2020.

Vancouver:

Virk-Baker MK. Soy And Breast Cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1430.

Council of Science Editors:

Virk-Baker MK. Soy And Breast Cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1430

27. Saxena, Ritu. Mechanisms and countermeasures of microgravity-induced bone loss: in vitro and in vivo model systems.

Degree: PhD, 2009, University of Alabama – Birmingham

Rapid bone loss occurs during prolonged periods of weightlessness experienced by astronauts during spaceflights which leads to osteopenia and increased fracture risk upon return to… (more)

Subjects/Keywords: Bone Resorption<; br>; Cell Differentiation<; br>; Osteoblasts  – cytology<; br>; Osteoclasts  – cytology<; br>; Parathyroid Hormone<; br>; Weightlessness

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APA (6th Edition):

Saxena, R. (2009). Mechanisms and countermeasures of microgravity-induced bone loss: in vitro and in vivo model systems. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,689

Chicago Manual of Style (16th Edition):

Saxena, Ritu. “Mechanisms and countermeasures of microgravity-induced bone loss: in vitro and in vivo model systems.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,689.

MLA Handbook (7th Edition):

Saxena, Ritu. “Mechanisms and countermeasures of microgravity-induced bone loss: in vitro and in vivo model systems.” 2009. Web. 29 Mar 2020.

Vancouver:

Saxena R. Mechanisms and countermeasures of microgravity-induced bone loss: in vitro and in vivo model systems. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,689.

Council of Science Editors:

Saxena R. Mechanisms and countermeasures of microgravity-induced bone loss: in vitro and in vivo model systems. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,689

28. Smith, Rebecca Burt. Axonal trafficking of BMP signals in Drosophila motoneurons.

Degree: PhD, 2009, University of Alabama – Birmingham

The Drosophila Bone Morphogenetic Protein (BMP) Glass bottom boat (Gbb) is a ligand for the BMP type II receptor, Wishful thinking (Wit). Mutations in either… (more)

Subjects/Keywords: Axonal Transport<; br>; Bone Morphogenetic Proteins  – genetics<; br>; Drosophila  – genetics<; br>; Motor Neurons<; br>; Neuronal Plasticity<; br>; Synapses  – physiology

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APA (6th Edition):

Smith, R. B. (2009). Axonal trafficking of BMP signals in Drosophila motoneurons. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,930

Chicago Manual of Style (16th Edition):

Smith, Rebecca Burt. “Axonal trafficking of BMP signals in Drosophila motoneurons.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,930.

MLA Handbook (7th Edition):

Smith, Rebecca Burt. “Axonal trafficking of BMP signals in Drosophila motoneurons.” 2009. Web. 29 Mar 2020.

Vancouver:

Smith RB. Axonal trafficking of BMP signals in Drosophila motoneurons. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,930.

Council of Science Editors:

Smith RB. Axonal trafficking of BMP signals in Drosophila motoneurons. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,930

29. Anderson, Joel M. (Joel Matthew). Biomimetic self-assembled nanomatrix for bone tissue regeneration.

Degree: PhD, 2011, University of Alabama – Birmingham

Formation of the native bone extracellular matrix (ECM) provides an attractive template for bone tissue engineering. At the cellular level, structural support and biological complexity… (more)

Subjects/Keywords: Bone regeneration <; br>; Bone  – Differentiation <; br>; Extracellular matrix <; br>; Nanostructured materials <; br>; Tissue engineering <; br>; Biomimetics <; br>; Colloids <; br>; Self-assembly (Chemistry)

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Anderson, J. M. (. M. (2011). Biomimetic self-assembled nanomatrix for bone tissue regeneration. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1036

Chicago Manual of Style (16th Edition):

Anderson, Joel M (Joel Matthew). “Biomimetic self-assembled nanomatrix for bone tissue regeneration.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1036.

MLA Handbook (7th Edition):

Anderson, Joel M (Joel Matthew). “Biomimetic self-assembled nanomatrix for bone tissue regeneration.” 2011. Web. 29 Mar 2020.

Vancouver:

Anderson JM(M. Biomimetic self-assembled nanomatrix for bone tissue regeneration. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1036.

Council of Science Editors:

Anderson JM(M. Biomimetic self-assembled nanomatrix for bone tissue regeneration. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1036

30. Ernest, Nola Jean Sieber. The role of chloride in the volume regulation of human glioma cells.

Degree: PhD, 2007, University of Alabama – Birmingham

According to the Central Brain Tumor Registry of the United States, the most common primary brain tumors are gliomas, tumors composed of cells of glial… (more)

Subjects/Keywords: Brain Neoplasms  – physiopathology <; br>; Cell Size <; br>; Chloride Channels  – physiology <; br>; Glioma  – physiopathology <; br>

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ernest, N. J. S. (2007). The role of chloride in the volume regulation of human glioma cells. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,86

Chicago Manual of Style (16th Edition):

Ernest, Nola Jean Sieber. “The role of chloride in the volume regulation of human glioma cells.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,86.

MLA Handbook (7th Edition):

Ernest, Nola Jean Sieber. “The role of chloride in the volume regulation of human glioma cells.” 2007. Web. 29 Mar 2020.

Vancouver:

Ernest NJS. The role of chloride in the volume regulation of human glioma cells. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,86.

Council of Science Editors:

Ernest NJS. The role of chloride in the volume regulation of human glioma cells. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,86

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