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You searched for subject:( B lymphocyte). Showing records 1 – 30 of 104 total matches.

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University of Otago

1. McKelvey, Kelly Joanne. The B-lymphocyte Component of Inflammation .

Degree: 2010, University of Otago

B-lymphocytes have been implicated as key mediators of inflammation and autoimmune disease. However, the mechanisms involved have yet to be fully elucidated. What is known… (more)

Subjects/Keywords: Inflammation; B-lymphocyte; Rheumatoid arthritis

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APA (6th Edition):

McKelvey, K. J. (2010). The B-lymphocyte Component of Inflammation . (Doctoral Dissertation). University of Otago. Retrieved from http://hdl.handle.net/10523/389

Chicago Manual of Style (16th Edition):

McKelvey, Kelly Joanne. “The B-lymphocyte Component of Inflammation .” 2010. Doctoral Dissertation, University of Otago. Accessed July 03, 2020. http://hdl.handle.net/10523/389.

MLA Handbook (7th Edition):

McKelvey, Kelly Joanne. “The B-lymphocyte Component of Inflammation .” 2010. Web. 03 Jul 2020.

Vancouver:

McKelvey KJ. The B-lymphocyte Component of Inflammation . [Internet] [Doctoral dissertation]. University of Otago; 2010. [cited 2020 Jul 03]. Available from: http://hdl.handle.net/10523/389.

Council of Science Editors:

McKelvey KJ. The B-lymphocyte Component of Inflammation . [Doctoral Dissertation]. University of Otago; 2010. Available from: http://hdl.handle.net/10523/389

2. Quinquenel, Anne. Analyse des sous-populations lymphocytaires B dans la leucémie lymphoïde chronique et de l'Impact de l'ibrutinib sur ces sous-populations : Analysis of immunoregulatory CD5⁺CD19⁺ subpopulations in chronic lymphocytic leukemia and of the impact of ibrutinib therapy on these subpopulations.

Degree: Docteur es, Biologie cellulaire et moléculaire, 2018, Sorbonne Paris Cité

La leucémie lymphoïde chronique (LLC) est une hémopathie B qui se caractérise par une évolution clinique hétérogène et un dysfonctionnement du système immunitaire. Des travaux… (more)

Subjects/Keywords: Lymphocyte B régulateur; Ibrutinib; Ibrutinib

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APA (6th Edition):

Quinquenel, A. (2018). Analyse des sous-populations lymphocytaires B dans la leucémie lymphoïde chronique et de l'Impact de l'ibrutinib sur ces sous-populations : Analysis of immunoregulatory CD5⁺CD19⁺ subpopulations in chronic lymphocytic leukemia and of the impact of ibrutinib therapy on these subpopulations. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2018USPCD072

Chicago Manual of Style (16th Edition):

Quinquenel, Anne. “Analyse des sous-populations lymphocytaires B dans la leucémie lymphoïde chronique et de l'Impact de l'ibrutinib sur ces sous-populations : Analysis of immunoregulatory CD5⁺CD19⁺ subpopulations in chronic lymphocytic leukemia and of the impact of ibrutinib therapy on these subpopulations.” 2018. Doctoral Dissertation, Sorbonne Paris Cité. Accessed July 03, 2020. http://www.theses.fr/2018USPCD072.

MLA Handbook (7th Edition):

Quinquenel, Anne. “Analyse des sous-populations lymphocytaires B dans la leucémie lymphoïde chronique et de l'Impact de l'ibrutinib sur ces sous-populations : Analysis of immunoregulatory CD5⁺CD19⁺ subpopulations in chronic lymphocytic leukemia and of the impact of ibrutinib therapy on these subpopulations.” 2018. Web. 03 Jul 2020.

Vancouver:

Quinquenel A. Analyse des sous-populations lymphocytaires B dans la leucémie lymphoïde chronique et de l'Impact de l'ibrutinib sur ces sous-populations : Analysis of immunoregulatory CD5⁺CD19⁺ subpopulations in chronic lymphocytic leukemia and of the impact of ibrutinib therapy on these subpopulations. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2018. [cited 2020 Jul 03]. Available from: http://www.theses.fr/2018USPCD072.

Council of Science Editors:

Quinquenel A. Analyse des sous-populations lymphocytaires B dans la leucémie lymphoïde chronique et de l'Impact de l'ibrutinib sur ces sous-populations : Analysis of immunoregulatory CD5⁺CD19⁺ subpopulations in chronic lymphocytic leukemia and of the impact of ibrutinib therapy on these subpopulations. [Doctoral Dissertation]. Sorbonne Paris Cité; 2018. Available from: http://www.theses.fr/2018USPCD072


Boston University

3. Lee, Joseph. B lymphocyte RANKL enhances periodontal disease in type 2 diabetes.

Degree: MS, Nutrition and Metabolism, 2018, Boston University

 Periodontitis (PD) and type 2 diabetes (T2D) are inflammatory diseases, which have B lymphocyte dysfunction in subjects with both conditions. B lymphocytes, when activated, release… (more)

Subjects/Keywords: Nutrition; B lymphocyte; RANKL; T lymphocyte; Periodontitis; Type 2 diabetes

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APA (6th Edition):

Lee, J. (2018). B lymphocyte RANKL enhances periodontal disease in type 2 diabetes. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/33016

Chicago Manual of Style (16th Edition):

Lee, Joseph. “B lymphocyte RANKL enhances periodontal disease in type 2 diabetes.” 2018. Masters Thesis, Boston University. Accessed July 03, 2020. http://hdl.handle.net/2144/33016.

MLA Handbook (7th Edition):

Lee, Joseph. “B lymphocyte RANKL enhances periodontal disease in type 2 diabetes.” 2018. Web. 03 Jul 2020.

Vancouver:

Lee J. B lymphocyte RANKL enhances periodontal disease in type 2 diabetes. [Internet] [Masters thesis]. Boston University; 2018. [cited 2020 Jul 03]. Available from: http://hdl.handle.net/2144/33016.

Council of Science Editors:

Lee J. B lymphocyte RANKL enhances periodontal disease in type 2 diabetes. [Masters Thesis]. Boston University; 2018. Available from: http://hdl.handle.net/2144/33016


Washington University in St. Louis

4. Miller, Brian. The Functions of Autophagy Genes in Lymphocytes and Osteoclasts.

Degree: PhD, Biology and Biomedical Sciences: Immunology, 2011, Washington University in St. Louis

 Macroautophagy: herein autophagy) is a process by which cells degrade long-lived proteins and organelles. The autophagy pathway and autophagy genes have been implicated in many… (more)

Subjects/Keywords: Immunology; Cellular Biology; Autophagy, B Lymphocyte, Osteoclast, T Lymphocyte

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APA (6th Edition):

Miller, B. (2011). The Functions of Autophagy Genes in Lymphocytes and Osteoclasts. (Doctoral Dissertation). Washington University in St. Louis. Retrieved from https://openscholarship.wustl.edu/etd/245

Chicago Manual of Style (16th Edition):

Miller, Brian. “The Functions of Autophagy Genes in Lymphocytes and Osteoclasts.” 2011. Doctoral Dissertation, Washington University in St. Louis. Accessed July 03, 2020. https://openscholarship.wustl.edu/etd/245.

MLA Handbook (7th Edition):

Miller, Brian. “The Functions of Autophagy Genes in Lymphocytes and Osteoclasts.” 2011. Web. 03 Jul 2020.

Vancouver:

Miller B. The Functions of Autophagy Genes in Lymphocytes and Osteoclasts. [Internet] [Doctoral dissertation]. Washington University in St. Louis; 2011. [cited 2020 Jul 03]. Available from: https://openscholarship.wustl.edu/etd/245.

Council of Science Editors:

Miller B. The Functions of Autophagy Genes in Lymphocytes and Osteoclasts. [Doctoral Dissertation]. Washington University in St. Louis; 2011. Available from: https://openscholarship.wustl.edu/etd/245


Université du Québec à Montréal

5. Cueva Vargas, Jorge Luis. Possible rôle du MIF dans l'activation polyclonale non spécifique des cellules B pendant l'infection par Plasmodium chabaudi adami DK.

Degree: 2009, Université du Québec à Montréal

 Les infections par Plasmodium induisent des changements importants sur la réponse effectrice et la régulation des cellules B. Ces effets se caractérisent par une suppression… (more)

Subjects/Keywords: Paludisme; Lymphocyte B; Facteur chimiotactique; Macrophage

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APA (6th Edition):

Cueva Vargas, J. L. (2009). Possible rôle du MIF dans l'activation polyclonale non spécifique des cellules B pendant l'infection par Plasmodium chabaudi adami DK. (Thesis). Université du Québec à Montréal. Retrieved from http://www.archipel.uqam.ca/2278/1/M10890.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cueva Vargas, Jorge Luis. “Possible rôle du MIF dans l'activation polyclonale non spécifique des cellules B pendant l'infection par Plasmodium chabaudi adami DK.” 2009. Thesis, Université du Québec à Montréal. Accessed July 03, 2020. http://www.archipel.uqam.ca/2278/1/M10890.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cueva Vargas, Jorge Luis. “Possible rôle du MIF dans l'activation polyclonale non spécifique des cellules B pendant l'infection par Plasmodium chabaudi adami DK.” 2009. Web. 03 Jul 2020.

Vancouver:

Cueva Vargas JL. Possible rôle du MIF dans l'activation polyclonale non spécifique des cellules B pendant l'infection par Plasmodium chabaudi adami DK. [Internet] [Thesis]. Université du Québec à Montréal; 2009. [cited 2020 Jul 03]. Available from: http://www.archipel.uqam.ca/2278/1/M10890.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cueva Vargas JL. Possible rôle du MIF dans l'activation polyclonale non spécifique des cellules B pendant l'infection par Plasmodium chabaudi adami DK. [Thesis]. Université du Québec à Montréal; 2009. Available from: http://www.archipel.uqam.ca/2278/1/M10890.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

6. Roy, Shuchismita. Regulation of B lymphocyte differentiation; -.

Degree: 2007, Jawaharlal Nehru University

Subjects/Keywords: Regulation; B Lymphocyte

Page 1

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APA (6th Edition):

Roy, S. (2007). Regulation of B lymphocyte differentiation; -. (Thesis). Jawaharlal Nehru University. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/29830

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Roy, Shuchismita. “Regulation of B lymphocyte differentiation; -.” 2007. Thesis, Jawaharlal Nehru University. Accessed July 03, 2020. http://shodhganga.inflibnet.ac.in/handle/10603/29830.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Roy, Shuchismita. “Regulation of B lymphocyte differentiation; -.” 2007. Web. 03 Jul 2020.

Vancouver:

Roy S. Regulation of B lymphocyte differentiation; -. [Internet] [Thesis]. Jawaharlal Nehru University; 2007. [cited 2020 Jul 03]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/29830.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Roy S. Regulation of B lymphocyte differentiation; -. [Thesis]. Jawaharlal Nehru University; 2007. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/29830

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université du Québec à Montréal

7. Cueva Vargas, Jorge Luis. Possible rôle du MIF dans l'activation polyclonale non spécifique des cellules B pendant l'infection par Plasmodium chabaudi adami DK.

Degree: 2009, Université du Québec à Montréal

 Les infections par Plasmodium induisent des changements importants sur la réponse effectrice et la régulation des cellules B. Ces effets se caractérisent par une suppression… (more)

Subjects/Keywords: Paludisme; Lymphocyte B; Facteur chimiotactique; Macrophage

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APA (6th Edition):

Cueva Vargas, J. L. (2009). Possible rôle du MIF dans l'activation polyclonale non spécifique des cellules B pendant l'infection par Plasmodium chabaudi adami DK. (Thesis). Université du Québec à Montréal. Retrieved from http://archipel.uqam.ca/2278/1/M10890.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cueva Vargas, Jorge Luis. “Possible rôle du MIF dans l'activation polyclonale non spécifique des cellules B pendant l'infection par Plasmodium chabaudi adami DK.” 2009. Thesis, Université du Québec à Montréal. Accessed July 03, 2020. http://archipel.uqam.ca/2278/1/M10890.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cueva Vargas, Jorge Luis. “Possible rôle du MIF dans l'activation polyclonale non spécifique des cellules B pendant l'infection par Plasmodium chabaudi adami DK.” 2009. Web. 03 Jul 2020.

Vancouver:

Cueva Vargas JL. Possible rôle du MIF dans l'activation polyclonale non spécifique des cellules B pendant l'infection par Plasmodium chabaudi adami DK. [Internet] [Thesis]. Université du Québec à Montréal; 2009. [cited 2020 Jul 03]. Available from: http://archipel.uqam.ca/2278/1/M10890.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cueva Vargas JL. Possible rôle du MIF dans l'activation polyclonale non spécifique des cellules B pendant l'infection par Plasmodium chabaudi adami DK. [Thesis]. Université du Québec à Montréal; 2009. Available from: http://archipel.uqam.ca/2278/1/M10890.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Depaul University

8. O'Laughlin, John Peter. Genetic diversity of the chB6 alloantigen.

Degree: Biological Sciences, 2010, Depaul University

 Chickens are one of the classic models of vertebrate immunity. We have been interested in the role of the alloantigen chB6 (formerly called BU1) in… (more)

Subjects/Keywords: B Lymphocyte; Apoptosis; chB6; Genetics; Birds

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APA (6th Edition):

O'Laughlin, J. P. (2010). Genetic diversity of the chB6 alloantigen. (Thesis). Depaul University. Retrieved from https://via.library.depaul.edu/etd/18

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

O'Laughlin, John Peter. “Genetic diversity of the chB6 alloantigen.” 2010. Thesis, Depaul University. Accessed July 03, 2020. https://via.library.depaul.edu/etd/18.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

O'Laughlin, John Peter. “Genetic diversity of the chB6 alloantigen.” 2010. Web. 03 Jul 2020.

Vancouver:

O'Laughlin JP. Genetic diversity of the chB6 alloantigen. [Internet] [Thesis]. Depaul University; 2010. [cited 2020 Jul 03]. Available from: https://via.library.depaul.edu/etd/18.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

O'Laughlin JP. Genetic diversity of the chB6 alloantigen. [Thesis]. Depaul University; 2010. Available from: https://via.library.depaul.edu/etd/18

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston College

9. Dufort, Fay Josephine. Contribution of Glucose Metabolism to the B Lymphocyte Responses.

Degree: PhD, Biology, 2012, Boston College

B-lymphocytes respond to environmental cues for their survival, growth, and differentiation through receptor-mediated signaling pathways. Naïve Blymphocytes must acquire and metabolize external glucose in order… (more)

Subjects/Keywords: ATP citrate lyase; B lymphocyte; glucose

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APA (6th Edition):

Dufort, F. J. (2012). Contribution of Glucose Metabolism to the B Lymphocyte Responses. (Doctoral Dissertation). Boston College. Retrieved from http://dlib.bc.edu/islandora/object/bc-ir:101190

Chicago Manual of Style (16th Edition):

Dufort, Fay Josephine. “Contribution of Glucose Metabolism to the B Lymphocyte Responses.” 2012. Doctoral Dissertation, Boston College. Accessed July 03, 2020. http://dlib.bc.edu/islandora/object/bc-ir:101190.

MLA Handbook (7th Edition):

Dufort, Fay Josephine. “Contribution of Glucose Metabolism to the B Lymphocyte Responses.” 2012. Web. 03 Jul 2020.

Vancouver:

Dufort FJ. Contribution of Glucose Metabolism to the B Lymphocyte Responses. [Internet] [Doctoral dissertation]. Boston College; 2012. [cited 2020 Jul 03]. Available from: http://dlib.bc.edu/islandora/object/bc-ir:101190.

Council of Science Editors:

Dufort FJ. Contribution of Glucose Metabolism to the B Lymphocyte Responses. [Doctoral Dissertation]. Boston College; 2012. Available from: http://dlib.bc.edu/islandora/object/bc-ir:101190


Boston College

10. Argueta, Shannon A. The Nutrients L-Glutamine and Glucose Have Unique Roles in B Lymphocyte Growth and Proliferation Responses.

Degree: PhD, Biology, 2016, Boston College

B cell activation is an energetically demanding process during which B lymphocytes undergo reprogramming and shift from a resting state to a highly proliferative, metabolically… (more)

Subjects/Keywords: B cell; Glucose; Glutamine; Lymphocyte; Metabolism

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APA (6th Edition):

Argueta, S. A. (2016). The Nutrients L-Glutamine and Glucose Have Unique Roles in B Lymphocyte Growth and Proliferation Responses. (Doctoral Dissertation). Boston College. Retrieved from http://dlib.bc.edu/islandora/object/bc-ir:107246

Chicago Manual of Style (16th Edition):

Argueta, Shannon A. “The Nutrients L-Glutamine and Glucose Have Unique Roles in B Lymphocyte Growth and Proliferation Responses.” 2016. Doctoral Dissertation, Boston College. Accessed July 03, 2020. http://dlib.bc.edu/islandora/object/bc-ir:107246.

MLA Handbook (7th Edition):

Argueta, Shannon A. “The Nutrients L-Glutamine and Glucose Have Unique Roles in B Lymphocyte Growth and Proliferation Responses.” 2016. Web. 03 Jul 2020.

Vancouver:

Argueta SA. The Nutrients L-Glutamine and Glucose Have Unique Roles in B Lymphocyte Growth and Proliferation Responses. [Internet] [Doctoral dissertation]. Boston College; 2016. [cited 2020 Jul 03]. Available from: http://dlib.bc.edu/islandora/object/bc-ir:107246.

Council of Science Editors:

Argueta SA. The Nutrients L-Glutamine and Glucose Have Unique Roles in B Lymphocyte Growth and Proliferation Responses. [Doctoral Dissertation]. Boston College; 2016. Available from: http://dlib.bc.edu/islandora/object/bc-ir:107246

11. Symington, Hannah Lucy. Mechanism of IL-2 mediated BACH2 regulation in the control of Human naive B cell differentiation into plasma cells : Mécanisme de régulation de BACH2 par la voie IL-2 lors de la différenciation des lymphocytes B humains en plasmocytes.

Degree: Docteur es, Biologie, 2016, Rennes 1

La différenciation terminale des lymphocytes B qui se déroule dans les centres germinatifs des organes lymphoïdes secondaires est l’étape ultime de la réponse T dépendante… (more)

Subjects/Keywords: Lymphocyte B; Plasmocyte; Différenciation; Signalisation cellulaire; Cytokine; B lymphocyte; Plasma cell; Differentiation; Cell signalling; Cytokine

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APA (6th Edition):

Symington, H. L. (2016). Mechanism of IL-2 mediated BACH2 regulation in the control of Human naive B cell differentiation into plasma cells : Mécanisme de régulation de BACH2 par la voie IL-2 lors de la différenciation des lymphocytes B humains en plasmocytes. (Doctoral Dissertation). Rennes 1. Retrieved from http://www.theses.fr/2016REN1B009

Chicago Manual of Style (16th Edition):

Symington, Hannah Lucy. “Mechanism of IL-2 mediated BACH2 regulation in the control of Human naive B cell differentiation into plasma cells : Mécanisme de régulation de BACH2 par la voie IL-2 lors de la différenciation des lymphocytes B humains en plasmocytes.” 2016. Doctoral Dissertation, Rennes 1. Accessed July 03, 2020. http://www.theses.fr/2016REN1B009.

MLA Handbook (7th Edition):

Symington, Hannah Lucy. “Mechanism of IL-2 mediated BACH2 regulation in the control of Human naive B cell differentiation into plasma cells : Mécanisme de régulation de BACH2 par la voie IL-2 lors de la différenciation des lymphocytes B humains en plasmocytes.” 2016. Web. 03 Jul 2020.

Vancouver:

Symington HL. Mechanism of IL-2 mediated BACH2 regulation in the control of Human naive B cell differentiation into plasma cells : Mécanisme de régulation de BACH2 par la voie IL-2 lors de la différenciation des lymphocytes B humains en plasmocytes. [Internet] [Doctoral dissertation]. Rennes 1; 2016. [cited 2020 Jul 03]. Available from: http://www.theses.fr/2016REN1B009.

Council of Science Editors:

Symington HL. Mechanism of IL-2 mediated BACH2 regulation in the control of Human naive B cell differentiation into plasma cells : Mécanisme de régulation de BACH2 par la voie IL-2 lors de la différenciation des lymphocytes B humains en plasmocytes. [Doctoral Dissertation]. Rennes 1; 2016. Available from: http://www.theses.fr/2016REN1B009

12. Leibler-Romand, Claire. Action du CTLA4-Ig sur la réponse humorale en Transplantation : Impact of the immunosuppressive treatment CTLA4-Ig on B-cells responses.

Degree: Docteur es, Biologie cellulaire et moléculaire, 2017, Université Paris-Est

La réponse alloimmune de type humorale et en particulier l'apparition de novo d’anticorps anti HLA dirigés contre le donneur (dnDSA) après une transplantation rénale sont… (more)

Subjects/Keywords: CTLA4-Ig; Lymphocyte B; Transplantation; CTLA4-Ig; B-Cells; Transplantation; 617.9

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APA (6th Edition):

Leibler-Romand, C. (2017). Action du CTLA4-Ig sur la réponse humorale en Transplantation : Impact of the immunosuppressive treatment CTLA4-Ig on B-cells responses. (Doctoral Dissertation). Université Paris-Est. Retrieved from http://www.theses.fr/2017PESC0015

Chicago Manual of Style (16th Edition):

Leibler-Romand, Claire. “Action du CTLA4-Ig sur la réponse humorale en Transplantation : Impact of the immunosuppressive treatment CTLA4-Ig on B-cells responses.” 2017. Doctoral Dissertation, Université Paris-Est. Accessed July 03, 2020. http://www.theses.fr/2017PESC0015.

MLA Handbook (7th Edition):

Leibler-Romand, Claire. “Action du CTLA4-Ig sur la réponse humorale en Transplantation : Impact of the immunosuppressive treatment CTLA4-Ig on B-cells responses.” 2017. Web. 03 Jul 2020.

Vancouver:

Leibler-Romand C. Action du CTLA4-Ig sur la réponse humorale en Transplantation : Impact of the immunosuppressive treatment CTLA4-Ig on B-cells responses. [Internet] [Doctoral dissertation]. Université Paris-Est; 2017. [cited 2020 Jul 03]. Available from: http://www.theses.fr/2017PESC0015.

Council of Science Editors:

Leibler-Romand C. Action du CTLA4-Ig sur la réponse humorale en Transplantation : Impact of the immunosuppressive treatment CTLA4-Ig on B-cells responses. [Doctoral Dissertation]. Université Paris-Est; 2017. Available from: http://www.theses.fr/2017PESC0015

13. Bouaziz, Jean-David. Dualité fonctionnelle cellulaire des lymphocytes B dans les maladies inflammatoires : le concept des lymphocytes B effecteurs et régulateurs : Counter regulatory B cell functions during autoimmune and inflammatory diseases.

Degree: Docteur es, Immunologie, 2008, Université Paris-Est

La reconnaissance des antigènes du soi et du non soi par les lymphocytes B et T est à l'origine des maladies auto-immunes et inflammatoires. Les… (more)

Subjects/Keywords: Lymphocyte B; Lymphocyte T; Présentation antigénique; Tolérance; Interleukine 10; Auto-immunité; Diabète; Sclérose en plaques

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APA (6th Edition):

Bouaziz, J. (2008). Dualité fonctionnelle cellulaire des lymphocytes B dans les maladies inflammatoires : le concept des lymphocytes B effecteurs et régulateurs : Counter regulatory B cell functions during autoimmune and inflammatory diseases. (Doctoral Dissertation). Université Paris-Est. Retrieved from http://www.theses.fr/2008PEST0031

Chicago Manual of Style (16th Edition):

Bouaziz, Jean-David. “Dualité fonctionnelle cellulaire des lymphocytes B dans les maladies inflammatoires : le concept des lymphocytes B effecteurs et régulateurs : Counter regulatory B cell functions during autoimmune and inflammatory diseases.” 2008. Doctoral Dissertation, Université Paris-Est. Accessed July 03, 2020. http://www.theses.fr/2008PEST0031.

MLA Handbook (7th Edition):

Bouaziz, Jean-David. “Dualité fonctionnelle cellulaire des lymphocytes B dans les maladies inflammatoires : le concept des lymphocytes B effecteurs et régulateurs : Counter regulatory B cell functions during autoimmune and inflammatory diseases.” 2008. Web. 03 Jul 2020.

Vancouver:

Bouaziz J. Dualité fonctionnelle cellulaire des lymphocytes B dans les maladies inflammatoires : le concept des lymphocytes B effecteurs et régulateurs : Counter regulatory B cell functions during autoimmune and inflammatory diseases. [Internet] [Doctoral dissertation]. Université Paris-Est; 2008. [cited 2020 Jul 03]. Available from: http://www.theses.fr/2008PEST0031.

Council of Science Editors:

Bouaziz J. Dualité fonctionnelle cellulaire des lymphocytes B dans les maladies inflammatoires : le concept des lymphocytes B effecteurs et régulateurs : Counter regulatory B cell functions during autoimmune and inflammatory diseases. [Doctoral Dissertation]. Université Paris-Est; 2008. Available from: http://www.theses.fr/2008PEST0031

14. Le Dantec, Christelle. Intérêt du couple CD5/CD6 dans les lymphocytes B humains : Interest of the CD5 / CD6 couple in human B lymphocytes.

Degree: Docteur es, Immunologie, 2012, Brest

Issues d'un gène ancestral commun, les molécules CD5 et CD6 sont présentes à la surface de tous les lymphocytes T (LT) matures ainsi qu'à la… (more)

Subjects/Keywords: CD5; CD6; Lymphocyte B; Épigénétique; Auto immunité; CD5; CD6; B lymphocyte; Epigenetic; Autoimmunity; 571.967; 616.079 8

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APA (6th Edition):

Le Dantec, C. (2012). Intérêt du couple CD5/CD6 dans les lymphocytes B humains : Interest of the CD5 / CD6 couple in human B lymphocytes. (Doctoral Dissertation). Brest. Retrieved from http://www.theses.fr/2012BRES0090

Chicago Manual of Style (16th Edition):

Le Dantec, Christelle. “Intérêt du couple CD5/CD6 dans les lymphocytes B humains : Interest of the CD5 / CD6 couple in human B lymphocytes.” 2012. Doctoral Dissertation, Brest. Accessed July 03, 2020. http://www.theses.fr/2012BRES0090.

MLA Handbook (7th Edition):

Le Dantec, Christelle. “Intérêt du couple CD5/CD6 dans les lymphocytes B humains : Interest of the CD5 / CD6 couple in human B lymphocytes.” 2012. Web. 03 Jul 2020.

Vancouver:

Le Dantec C. Intérêt du couple CD5/CD6 dans les lymphocytes B humains : Interest of the CD5 / CD6 couple in human B lymphocytes. [Internet] [Doctoral dissertation]. Brest; 2012. [cited 2020 Jul 03]. Available from: http://www.theses.fr/2012BRES0090.

Council of Science Editors:

Le Dantec C. Intérêt du couple CD5/CD6 dans les lymphocytes B humains : Interest of the CD5 / CD6 couple in human B lymphocytes. [Doctoral Dissertation]. Brest; 2012. Available from: http://www.theses.fr/2012BRES0090


Université de Montréal

15. Valcke, Han Sang. Étude du dysfonctionnement du compartiment des cellules B chez des patients à différents stades d’infection par le virus d’immunodéficience humaine (VIH) .

Degree: 2010, Université de Montréal

 Les anomalies phénotypiques et fonctionnelles des lymphocytes B (LB) sont typiques d'une infection au VIH et se traduisent principalement par une activation polyclonale, une perte… (more)

Subjects/Keywords: Vih; Sida; Ltnps; Phi; cellule dendritique; lymphocyte B; CD4+; Blys; hyperglobulinémie; Hiv; Aids; dendritic cell; B lymphocyte; hyperglobulinemia

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APA (6th Edition):

Valcke, H. S. (2010). Étude du dysfonctionnement du compartiment des cellules B chez des patients à différents stades d’infection par le virus d’immunodéficience humaine (VIH) . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/3556

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Valcke, Han Sang. “Étude du dysfonctionnement du compartiment des cellules B chez des patients à différents stades d’infection par le virus d’immunodéficience humaine (VIH) .” 2010. Thesis, Université de Montréal. Accessed July 03, 2020. http://hdl.handle.net/1866/3556.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Valcke, Han Sang. “Étude du dysfonctionnement du compartiment des cellules B chez des patients à différents stades d’infection par le virus d’immunodéficience humaine (VIH) .” 2010. Web. 03 Jul 2020.

Vancouver:

Valcke HS. Étude du dysfonctionnement du compartiment des cellules B chez des patients à différents stades d’infection par le virus d’immunodéficience humaine (VIH) . [Internet] [Thesis]. Université de Montréal; 2010. [cited 2020 Jul 03]. Available from: http://hdl.handle.net/1866/3556.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Valcke HS. Étude du dysfonctionnement du compartiment des cellules B chez des patients à différents stades d’infection par le virus d’immunodéficience humaine (VIH) . [Thesis]. Université de Montréal; 2010. Available from: http://hdl.handle.net/1866/3556

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

16. Debant, Marjolaine. Etude de la dérégulation des entrées calciques du lymphocyte B de leucémie lymphoïde chronique : mise en évidence d'une nouvelle piste thérapeutique : Study of calcium entries deregulation in chronic lymphocytic leukemia B-cells : evidence for a new therapeutic target.

Degree: Docteur es, Immunologie, 2017, Brest

La leucémie lymphoïde chronique (LLC) constitue l'hémopathie maligne la plus fréquente dans les pays occidentaux et résulte d’une accumulation de lymphocytes B (LB) monoclonaux matures… (more)

Subjects/Keywords: Signalisation calcique; Leucémie lymphoïde chronique; Lymphocyte B; STIM; Thérapie; Calcium signaling; Chronic lymphocytic leukemia; B lymphocyte; STIM1; Therapy; 571.967; 616.15

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APA (6th Edition):

Debant, M. (2017). Etude de la dérégulation des entrées calciques du lymphocyte B de leucémie lymphoïde chronique : mise en évidence d'une nouvelle piste thérapeutique : Study of calcium entries deregulation in chronic lymphocytic leukemia B-cells : evidence for a new therapeutic target. (Doctoral Dissertation). Brest. Retrieved from http://www.theses.fr/2017BRES0150

Chicago Manual of Style (16th Edition):

Debant, Marjolaine. “Etude de la dérégulation des entrées calciques du lymphocyte B de leucémie lymphoïde chronique : mise en évidence d'une nouvelle piste thérapeutique : Study of calcium entries deregulation in chronic lymphocytic leukemia B-cells : evidence for a new therapeutic target.” 2017. Doctoral Dissertation, Brest. Accessed July 03, 2020. http://www.theses.fr/2017BRES0150.

MLA Handbook (7th Edition):

Debant, Marjolaine. “Etude de la dérégulation des entrées calciques du lymphocyte B de leucémie lymphoïde chronique : mise en évidence d'une nouvelle piste thérapeutique : Study of calcium entries deregulation in chronic lymphocytic leukemia B-cells : evidence for a new therapeutic target.” 2017. Web. 03 Jul 2020.

Vancouver:

Debant M. Etude de la dérégulation des entrées calciques du lymphocyte B de leucémie lymphoïde chronique : mise en évidence d'une nouvelle piste thérapeutique : Study of calcium entries deregulation in chronic lymphocytic leukemia B-cells : evidence for a new therapeutic target. [Internet] [Doctoral dissertation]. Brest; 2017. [cited 2020 Jul 03]. Available from: http://www.theses.fr/2017BRES0150.

Council of Science Editors:

Debant M. Etude de la dérégulation des entrées calciques du lymphocyte B de leucémie lymphoïde chronique : mise en évidence d'une nouvelle piste thérapeutique : Study of calcium entries deregulation in chronic lymphocytic leukemia B-cells : evidence for a new therapeutic target. [Doctoral Dissertation]. Brest; 2017. Available from: http://www.theses.fr/2017BRES0150


Université de Lorraine

17. Harlé, Guillaume. Régulations des systèmes nerveux central et immunitaire en condition de stress : rôle de la corticotropin-releasing hormone et de ses récepteurs : Central nervous system and immune system regulation in stress condition : role of corticoprin-releasing hormone ans its receptors.

Degree: Docteur es, Sciences de la vie et de la santé, 2016, Université de Lorraine

Lors d’un stress, l’activation de l’axe hypothalamo-hypophyso-surrénalien (HHS) conduit à une augmentation de la production de glucocorticoïdes (tel que la corticostérone) par les glandes surrénales.… (more)

Subjects/Keywords: Corticotropin-Releasing hormone (CRH); Stress; Cervelet; Lymphocyte B; Neuroimmunologie; Corticotropin-Releasing hormone (CRH); Stress; Cervelet; Lymphocyte B; Neuroimmunologie; 616.079; 573.837 4

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APA (6th Edition):

Harlé, G. (2016). Régulations des systèmes nerveux central et immunitaire en condition de stress : rôle de la corticotropin-releasing hormone et de ses récepteurs : Central nervous system and immune system regulation in stress condition : role of corticoprin-releasing hormone ans its receptors. (Doctoral Dissertation). Université de Lorraine. Retrieved from http://www.theses.fr/2016LORR0126

Chicago Manual of Style (16th Edition):

Harlé, Guillaume. “Régulations des systèmes nerveux central et immunitaire en condition de stress : rôle de la corticotropin-releasing hormone et de ses récepteurs : Central nervous system and immune system regulation in stress condition : role of corticoprin-releasing hormone ans its receptors.” 2016. Doctoral Dissertation, Université de Lorraine. Accessed July 03, 2020. http://www.theses.fr/2016LORR0126.

MLA Handbook (7th Edition):

Harlé, Guillaume. “Régulations des systèmes nerveux central et immunitaire en condition de stress : rôle de la corticotropin-releasing hormone et de ses récepteurs : Central nervous system and immune system regulation in stress condition : role of corticoprin-releasing hormone ans its receptors.” 2016. Web. 03 Jul 2020.

Vancouver:

Harlé G. Régulations des systèmes nerveux central et immunitaire en condition de stress : rôle de la corticotropin-releasing hormone et de ses récepteurs : Central nervous system and immune system regulation in stress condition : role of corticoprin-releasing hormone ans its receptors. [Internet] [Doctoral dissertation]. Université de Lorraine; 2016. [cited 2020 Jul 03]. Available from: http://www.theses.fr/2016LORR0126.

Council of Science Editors:

Harlé G. Régulations des systèmes nerveux central et immunitaire en condition de stress : rôle de la corticotropin-releasing hormone et de ses récepteurs : Central nervous system and immune system regulation in stress condition : role of corticoprin-releasing hormone ans its receptors. [Doctoral Dissertation]. Université de Lorraine; 2016. Available from: http://www.theses.fr/2016LORR0126

18. Martin, Mickael. Expression de ZAP-70 dans les lymphocytes B non tumoraux : implications dans la rupture de tolérance et la transformation néoplasique : ZAP-70 expression in non tumoral B lymphocytes : implications in tolerance breakdown and neoplastic transformation.

Degree: Docteur es, Immunologie, 2018, Université de Strasbourg

L’expression de ZAP-70 dans la leucémie lymphoïde chronique (LLC) est associée à une hypersignalisation du BCR et à la survenue de cytopénies auto-immunes (CAI). Les… (more)

Subjects/Keywords: Leucémie lymphoïde chronique; ZAP-70; Auto-immunité; Lymphocyte B; Chronic lymphocytic leukemia; ZAP-70; Autoimmunity; B lymphocyte; 571.96; 572.8; 616.99

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APA (6th Edition):

Martin, M. (2018). Expression de ZAP-70 dans les lymphocytes B non tumoraux : implications dans la rupture de tolérance et la transformation néoplasique : ZAP-70 expression in non tumoral B lymphocytes : implications in tolerance breakdown and neoplastic transformation. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2018STRAJ079

Chicago Manual of Style (16th Edition):

Martin, Mickael. “Expression de ZAP-70 dans les lymphocytes B non tumoraux : implications dans la rupture de tolérance et la transformation néoplasique : ZAP-70 expression in non tumoral B lymphocytes : implications in tolerance breakdown and neoplastic transformation.” 2018. Doctoral Dissertation, Université de Strasbourg. Accessed July 03, 2020. http://www.theses.fr/2018STRAJ079.

MLA Handbook (7th Edition):

Martin, Mickael. “Expression de ZAP-70 dans les lymphocytes B non tumoraux : implications dans la rupture de tolérance et la transformation néoplasique : ZAP-70 expression in non tumoral B lymphocytes : implications in tolerance breakdown and neoplastic transformation.” 2018. Web. 03 Jul 2020.

Vancouver:

Martin M. Expression de ZAP-70 dans les lymphocytes B non tumoraux : implications dans la rupture de tolérance et la transformation néoplasique : ZAP-70 expression in non tumoral B lymphocytes : implications in tolerance breakdown and neoplastic transformation. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2018. [cited 2020 Jul 03]. Available from: http://www.theses.fr/2018STRAJ079.

Council of Science Editors:

Martin M. Expression de ZAP-70 dans les lymphocytes B non tumoraux : implications dans la rupture de tolérance et la transformation néoplasique : ZAP-70 expression in non tumoral B lymphocytes : implications in tolerance breakdown and neoplastic transformation. [Doctoral Dissertation]. Université de Strasbourg; 2018. Available from: http://www.theses.fr/2018STRAJ079

19. Dzangué Tchoupou, Gaëlle. Caractérisation des réponses immunitaires chez les patients atteints de myopathies auto-immunes idiopathiques : Characterization of immune responses in patients with autoimmune idiopathic myopathies.

Degree: Docteur es, Immunologie, 2018, Sorbonne université

Les myosites sont des maladies auto-immunes, caractérisées par des atteintes musculaires et extra musculaires. Le diagnostic des myosites peut être difficile et nécessite de l’expertise,… (more)

Subjects/Keywords: Myosites; Cytométrie de masse; Lymphocyte T CD8+; Lymphocyte B; Biomarqueurs; Analyses multidimensionnelles; Myositis; Mass cytometry; CD8+T cells; B-lymphocyte; Biomarkers; Multidimensional analysis; 616.748; 616.0797; 616.0798

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APA (6th Edition):

Dzangué Tchoupou, G. (2018). Caractérisation des réponses immunitaires chez les patients atteints de myopathies auto-immunes idiopathiques : Characterization of immune responses in patients with autoimmune idiopathic myopathies. (Doctoral Dissertation). Sorbonne université. Retrieved from http://www.theses.fr/2018SORUS171

Chicago Manual of Style (16th Edition):

Dzangué Tchoupou, Gaëlle. “Caractérisation des réponses immunitaires chez les patients atteints de myopathies auto-immunes idiopathiques : Characterization of immune responses in patients with autoimmune idiopathic myopathies.” 2018. Doctoral Dissertation, Sorbonne université. Accessed July 03, 2020. http://www.theses.fr/2018SORUS171.

MLA Handbook (7th Edition):

Dzangué Tchoupou, Gaëlle. “Caractérisation des réponses immunitaires chez les patients atteints de myopathies auto-immunes idiopathiques : Characterization of immune responses in patients with autoimmune idiopathic myopathies.” 2018. Web. 03 Jul 2020.

Vancouver:

Dzangué Tchoupou G. Caractérisation des réponses immunitaires chez les patients atteints de myopathies auto-immunes idiopathiques : Characterization of immune responses in patients with autoimmune idiopathic myopathies. [Internet] [Doctoral dissertation]. Sorbonne université; 2018. [cited 2020 Jul 03]. Available from: http://www.theses.fr/2018SORUS171.

Council of Science Editors:

Dzangué Tchoupou G. Caractérisation des réponses immunitaires chez les patients atteints de myopathies auto-immunes idiopathiques : Characterization of immune responses in patients with autoimmune idiopathic myopathies. [Doctoral Dissertation]. Sorbonne université; 2018. Available from: http://www.theses.fr/2018SORUS171

20. Jung, Sophie. Agents infectieux et rupture de tolérance lymphocytaire B : étude des processus de maturation d'affinité et de différenciation plasmocytaire au cours d'une infection bactérienne dans un nouveau modèle knock-in autoréactif : Infectious agents and B cell tolerance breakdown : study of affinity maturation and plasma-cell differentiation processes during bacterial infection in a new autoreactive knock-in mouse model.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2013, Université de Strasbourg

Les maladies auto-immunes, qui touchent plus de 5% de la population, sont induites par une perte de la tolérance aux antigènes du Soi. Ces pathologies,… (more)

Subjects/Keywords: Auto-immunité; Lymphocyte B; Infection bactérienne; Modèles murins transgéniques; Tolérance lymphocytaire B; Autoimmunity; B lymphocyte; Bacterial infection; Transgenic mouse models; B cell tolerance; 571.96; 572.8; 616.97

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APA (6th Edition):

Jung, S. (2013). Agents infectieux et rupture de tolérance lymphocytaire B : étude des processus de maturation d'affinité et de différenciation plasmocytaire au cours d'une infection bactérienne dans un nouveau modèle knock-in autoréactif : Infectious agents and B cell tolerance breakdown : study of affinity maturation and plasma-cell differentiation processes during bacterial infection in a new autoreactive knock-in mouse model. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2013STRAJ067

Chicago Manual of Style (16th Edition):

Jung, Sophie. “Agents infectieux et rupture de tolérance lymphocytaire B : étude des processus de maturation d'affinité et de différenciation plasmocytaire au cours d'une infection bactérienne dans un nouveau modèle knock-in autoréactif : Infectious agents and B cell tolerance breakdown : study of affinity maturation and plasma-cell differentiation processes during bacterial infection in a new autoreactive knock-in mouse model.” 2013. Doctoral Dissertation, Université de Strasbourg. Accessed July 03, 2020. http://www.theses.fr/2013STRAJ067.

MLA Handbook (7th Edition):

Jung, Sophie. “Agents infectieux et rupture de tolérance lymphocytaire B : étude des processus de maturation d'affinité et de différenciation plasmocytaire au cours d'une infection bactérienne dans un nouveau modèle knock-in autoréactif : Infectious agents and B cell tolerance breakdown : study of affinity maturation and plasma-cell differentiation processes during bacterial infection in a new autoreactive knock-in mouse model.” 2013. Web. 03 Jul 2020.

Vancouver:

Jung S. Agents infectieux et rupture de tolérance lymphocytaire B : étude des processus de maturation d'affinité et de différenciation plasmocytaire au cours d'une infection bactérienne dans un nouveau modèle knock-in autoréactif : Infectious agents and B cell tolerance breakdown : study of affinity maturation and plasma-cell differentiation processes during bacterial infection in a new autoreactive knock-in mouse model. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2013. [cited 2020 Jul 03]. Available from: http://www.theses.fr/2013STRAJ067.

Council of Science Editors:

Jung S. Agents infectieux et rupture de tolérance lymphocytaire B : étude des processus de maturation d'affinité et de différenciation plasmocytaire au cours d'une infection bactérienne dans un nouveau modèle knock-in autoréactif : Infectious agents and B cell tolerance breakdown : study of affinity maturation and plasma-cell differentiation processes during bacterial infection in a new autoreactive knock-in mouse model. [Doctoral Dissertation]. Université de Strasbourg; 2013. Available from: http://www.theses.fr/2013STRAJ067

21. Radouani, Fatima-Ezzahra. Polymorphismes des récepteurs des Fc des Immunoglobulines G et maladies autoimmunes en Martinique : impact du FcγRIIb sur la régulation du Lymphocyte B : Fc receptor polymorphisms of immunoglobulin G and autoimmune diseases in Martinique : Impact of FcγRIIb on the regulation of the B lymphocyte.

Degree: Docteur es, Biologie et Santé / immunologie, 2016, Antilles

Les récepteurs du Fc des Immunoglobulines G (FcγR) sont impliqués dans de nombreuses réactions immunitaires. Deux groupes de faible affinité existent : les FcγRIIa/b/c et… (more)

Subjects/Keywords: Autoimmunité; FcγRs; Lymphocyte B; Neuromyélite optique; Lupus; Autoimmunity; FcγRs; B cell; Lupus; Neuromyelitis optica

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APA (6th Edition):

Radouani, F. (2016). Polymorphismes des récepteurs des Fc des Immunoglobulines G et maladies autoimmunes en Martinique : impact du FcγRIIb sur la régulation du Lymphocyte B : Fc receptor polymorphisms of immunoglobulin G and autoimmune diseases in Martinique : Impact of FcγRIIb on the regulation of the B lymphocyte. (Doctoral Dissertation). Antilles. Retrieved from http://www.theses.fr/2016ANTI0129

Chicago Manual of Style (16th Edition):

Radouani, Fatima-Ezzahra. “Polymorphismes des récepteurs des Fc des Immunoglobulines G et maladies autoimmunes en Martinique : impact du FcγRIIb sur la régulation du Lymphocyte B : Fc receptor polymorphisms of immunoglobulin G and autoimmune diseases in Martinique : Impact of FcγRIIb on the regulation of the B lymphocyte.” 2016. Doctoral Dissertation, Antilles. Accessed July 03, 2020. http://www.theses.fr/2016ANTI0129.

MLA Handbook (7th Edition):

Radouani, Fatima-Ezzahra. “Polymorphismes des récepteurs des Fc des Immunoglobulines G et maladies autoimmunes en Martinique : impact du FcγRIIb sur la régulation du Lymphocyte B : Fc receptor polymorphisms of immunoglobulin G and autoimmune diseases in Martinique : Impact of FcγRIIb on the regulation of the B lymphocyte.” 2016. Web. 03 Jul 2020.

Vancouver:

Radouani F. Polymorphismes des récepteurs des Fc des Immunoglobulines G et maladies autoimmunes en Martinique : impact du FcγRIIb sur la régulation du Lymphocyte B : Fc receptor polymorphisms of immunoglobulin G and autoimmune diseases in Martinique : Impact of FcγRIIb on the regulation of the B lymphocyte. [Internet] [Doctoral dissertation]. Antilles; 2016. [cited 2020 Jul 03]. Available from: http://www.theses.fr/2016ANTI0129.

Council of Science Editors:

Radouani F. Polymorphismes des récepteurs des Fc des Immunoglobulines G et maladies autoimmunes en Martinique : impact du FcγRIIb sur la régulation du Lymphocyte B : Fc receptor polymorphisms of immunoglobulin G and autoimmune diseases in Martinique : Impact of FcγRIIb on the regulation of the B lymphocyte. [Doctoral Dissertation]. Antilles; 2016. Available from: http://www.theses.fr/2016ANTI0129

22. Denis-Lagache, Nicolas. Commutation ou extinction de l'expression du BCR et impact sur la cellule B : Commutation or extinction of BCR expression and impact on B cell.

Degree: Docteur es, Immunogénétique, 2015, Limoges

Lors de la reconnaissance de leur antigène spécifique, les lymphocytes B vont s’activer et interagir avec les autres cellules des organes lymphoïdes secondaires (cellules folliculaires… (more)

Subjects/Keywords: Lymphocyte B; AID; CSR; LSR; Apoptose; B cell; AID; CSR; LSR; Apoptosis; 616.079 6

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APA (6th Edition):

Denis-Lagache, N. (2015). Commutation ou extinction de l'expression du BCR et impact sur la cellule B : Commutation or extinction of BCR expression and impact on B cell. (Doctoral Dissertation). Limoges. Retrieved from http://www.theses.fr/2015LIMO0071

Chicago Manual of Style (16th Edition):

Denis-Lagache, Nicolas. “Commutation ou extinction de l'expression du BCR et impact sur la cellule B : Commutation or extinction of BCR expression and impact on B cell.” 2015. Doctoral Dissertation, Limoges. Accessed July 03, 2020. http://www.theses.fr/2015LIMO0071.

MLA Handbook (7th Edition):

Denis-Lagache, Nicolas. “Commutation ou extinction de l'expression du BCR et impact sur la cellule B : Commutation or extinction of BCR expression and impact on B cell.” 2015. Web. 03 Jul 2020.

Vancouver:

Denis-Lagache N. Commutation ou extinction de l'expression du BCR et impact sur la cellule B : Commutation or extinction of BCR expression and impact on B cell. [Internet] [Doctoral dissertation]. Limoges; 2015. [cited 2020 Jul 03]. Available from: http://www.theses.fr/2015LIMO0071.

Council of Science Editors:

Denis-Lagache N. Commutation ou extinction de l'expression du BCR et impact sur la cellule B : Commutation or extinction of BCR expression and impact on B cell. [Doctoral Dissertation]. Limoges; 2015. Available from: http://www.theses.fr/2015LIMO0071

23. Gamonet, Clémentine. Identification de nouveaux transcrits alternatifs du gène CD20 humain, différentiellement exprimés dans les hémopathies impliquant le lymphocyte B : Identification of new alternative splicfng variants of CD20 human gene, differentially expressed in pathologies involving b lymphocyte.

Degree: Docteur es, Sciences de la vie et de la santé, 2015, Besançon

La protéine D393-CD20, codée par un transcrit alternatif du gène cd20 découvert au laboratoire en 2010, est expriméedans les lymphocytes B (LB) tumoraux et surexprimée… (more)

Subjects/Keywords: CD20; Epissage; Rituximab; Lymphocyte B; Dérégulation; CD20; Splicing; Rituximab; Involving B; Deregulation; 616; QW 541

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APA (6th Edition):

Gamonet, C. (2015). Identification de nouveaux transcrits alternatifs du gène CD20 humain, différentiellement exprimés dans les hémopathies impliquant le lymphocyte B : Identification of new alternative splicfng variants of CD20 human gene, differentially expressed in pathologies involving b lymphocyte. (Doctoral Dissertation). Besançon. Retrieved from http://www.theses.fr/2015BESA3003

Chicago Manual of Style (16th Edition):

Gamonet, Clémentine. “Identification de nouveaux transcrits alternatifs du gène CD20 humain, différentiellement exprimés dans les hémopathies impliquant le lymphocyte B : Identification of new alternative splicfng variants of CD20 human gene, differentially expressed in pathologies involving b lymphocyte.” 2015. Doctoral Dissertation, Besançon. Accessed July 03, 2020. http://www.theses.fr/2015BESA3003.

MLA Handbook (7th Edition):

Gamonet, Clémentine. “Identification de nouveaux transcrits alternatifs du gène CD20 humain, différentiellement exprimés dans les hémopathies impliquant le lymphocyte B : Identification of new alternative splicfng variants of CD20 human gene, differentially expressed in pathologies involving b lymphocyte.” 2015. Web. 03 Jul 2020.

Vancouver:

Gamonet C. Identification de nouveaux transcrits alternatifs du gène CD20 humain, différentiellement exprimés dans les hémopathies impliquant le lymphocyte B : Identification of new alternative splicfng variants of CD20 human gene, differentially expressed in pathologies involving b lymphocyte. [Internet] [Doctoral dissertation]. Besançon; 2015. [cited 2020 Jul 03]. Available from: http://www.theses.fr/2015BESA3003.

Council of Science Editors:

Gamonet C. Identification de nouveaux transcrits alternatifs du gène CD20 humain, différentiellement exprimés dans les hémopathies impliquant le lymphocyte B : Identification of new alternative splicfng variants of CD20 human gene, differentially expressed in pathologies involving b lymphocyte. [Doctoral Dissertation]. Besançon; 2015. Available from: http://www.theses.fr/2015BESA3003

24. Bigot, Jérémy. Caractérisation fonctionnelle et phénotypique de lymphocytes B transitionnels CD24hi CD38hi associés à un phénotype régulateur chez des patients transplantés rénaux traités par Belatacept : Functional and phenotypic characterization of CD24hi CD38hi human transitional B cells associated with an immunoregulatory phenotype in renal transplant recipients treated with Belatacept.

Degree: Docteur es, Biologie cellulaire et moléculaire, 2016, Université Paris-Est

A l’instar des lymphocytes T régulateurs, l’étude de populations lymphocytaires B au potentiel immunosuppresseur a émergé ces dernières années. La capacité immunosuppressive des lymphocytes B(more)

Subjects/Keywords: Lymphocyte B regulateur; Il-10; Tolerance; Transplantation; Regulatory B cells; Il-10; Tolerance; Transplantation; 610

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APA (6th Edition):

Bigot, J. (2016). Caractérisation fonctionnelle et phénotypique de lymphocytes B transitionnels CD24hi CD38hi associés à un phénotype régulateur chez des patients transplantés rénaux traités par Belatacept : Functional and phenotypic characterization of CD24hi CD38hi human transitional B cells associated with an immunoregulatory phenotype in renal transplant recipients treated with Belatacept. (Doctoral Dissertation). Université Paris-Est. Retrieved from http://www.theses.fr/2016PESC0071

Chicago Manual of Style (16th Edition):

Bigot, Jérémy. “Caractérisation fonctionnelle et phénotypique de lymphocytes B transitionnels CD24hi CD38hi associés à un phénotype régulateur chez des patients transplantés rénaux traités par Belatacept : Functional and phenotypic characterization of CD24hi CD38hi human transitional B cells associated with an immunoregulatory phenotype in renal transplant recipients treated with Belatacept.” 2016. Doctoral Dissertation, Université Paris-Est. Accessed July 03, 2020. http://www.theses.fr/2016PESC0071.

MLA Handbook (7th Edition):

Bigot, Jérémy. “Caractérisation fonctionnelle et phénotypique de lymphocytes B transitionnels CD24hi CD38hi associés à un phénotype régulateur chez des patients transplantés rénaux traités par Belatacept : Functional and phenotypic characterization of CD24hi CD38hi human transitional B cells associated with an immunoregulatory phenotype in renal transplant recipients treated with Belatacept.” 2016. Web. 03 Jul 2020.

Vancouver:

Bigot J. Caractérisation fonctionnelle et phénotypique de lymphocytes B transitionnels CD24hi CD38hi associés à un phénotype régulateur chez des patients transplantés rénaux traités par Belatacept : Functional and phenotypic characterization of CD24hi CD38hi human transitional B cells associated with an immunoregulatory phenotype in renal transplant recipients treated with Belatacept. [Internet] [Doctoral dissertation]. Université Paris-Est; 2016. [cited 2020 Jul 03]. Available from: http://www.theses.fr/2016PESC0071.

Council of Science Editors:

Bigot J. Caractérisation fonctionnelle et phénotypique de lymphocytes B transitionnels CD24hi CD38hi associés à un phénotype régulateur chez des patients transplantés rénaux traités par Belatacept : Functional and phenotypic characterization of CD24hi CD38hi human transitional B cells associated with an immunoregulatory phenotype in renal transplant recipients treated with Belatacept. [Doctoral Dissertation]. Université Paris-Est; 2016. Available from: http://www.theses.fr/2016PESC0071


University of California – San Francisco

25. Wheeler, Matthew Lewis. Fine-tuning B cell antigen receptor signaling by reactive oxygen species and diacylglycerol metabolism.

Degree: Biomedical Sciences, 2013, University of California – San Francisco

 Recognition of cognate antigen by the B cell antigen receptor (BCR) is one of the primary events that ultimately facilitate the production of pathogen-specific antibodies… (more)

Subjects/Keywords: Immunology; antibody response; B cell receptor; B lymphocyte; diacylglycerol; reactive oxygen species; signal transduction

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APA (6th Edition):

Wheeler, M. L. (2013). Fine-tuning B cell antigen receptor signaling by reactive oxygen species and diacylglycerol metabolism. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/3657t2n7

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wheeler, Matthew Lewis. “Fine-tuning B cell antigen receptor signaling by reactive oxygen species and diacylglycerol metabolism.” 2013. Thesis, University of California – San Francisco. Accessed July 03, 2020. http://www.escholarship.org/uc/item/3657t2n7.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wheeler, Matthew Lewis. “Fine-tuning B cell antigen receptor signaling by reactive oxygen species and diacylglycerol metabolism.” 2013. Web. 03 Jul 2020.

Vancouver:

Wheeler ML. Fine-tuning B cell antigen receptor signaling by reactive oxygen species and diacylglycerol metabolism. [Internet] [Thesis]. University of California – San Francisco; 2013. [cited 2020 Jul 03]. Available from: http://www.escholarship.org/uc/item/3657t2n7.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wheeler ML. Fine-tuning B cell antigen receptor signaling by reactive oxygen species and diacylglycerol metabolism. [Thesis]. University of California – San Francisco; 2013. Available from: http://www.escholarship.org/uc/item/3657t2n7

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. Roy, Shuchismita. Regulation of B lymphocyte differentiation; -.

Degree: Immunology, 2007, Jawaharlal Nehru University

None

Bibilography p.17-91, Abbrevations

Advisors/Committee Members: George, Anna.

Subjects/Keywords: Regulation; B Lymphocyte; ligagtion; differentiation

Page 1

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APA (6th Edition):

Roy, S. (2007). Regulation of B lymphocyte differentiation; -. (Thesis). Jawaharlal Nehru University. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/29823

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Roy, Shuchismita. “Regulation of B lymphocyte differentiation; -.” 2007. Thesis, Jawaharlal Nehru University. Accessed July 03, 2020. http://shodhganga.inflibnet.ac.in/handle/10603/29823.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Roy, Shuchismita. “Regulation of B lymphocyte differentiation; -.” 2007. Web. 03 Jul 2020.

Vancouver:

Roy S. Regulation of B lymphocyte differentiation; -. [Internet] [Thesis]. Jawaharlal Nehru University; 2007. [cited 2020 Jul 03]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/29823.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Roy S. Regulation of B lymphocyte differentiation; -. [Thesis]. Jawaharlal Nehru University; 2007. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/29823

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

27. Gardam, Sandra. Regulation of lymphocyte development and function by TRAF2 and TRAF3.

Degree: Clinical School - St Vincent's Hospital, 2008, University of New South Wales

 Tumour necrosis factor receptor (TNFR) family members are widely expressed in cells of the immune system and are essential for the development and function of… (more)

Subjects/Keywords: B lymphocyte; Immunology; Signalling; TRAF

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APA (6th Edition):

Gardam, S. (2008). Regulation of lymphocyte development and function by TRAF2 and TRAF3. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/42104 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:3686/SOURCE2?view=true

Chicago Manual of Style (16th Edition):

Gardam, Sandra. “Regulation of lymphocyte development and function by TRAF2 and TRAF3.” 2008. Doctoral Dissertation, University of New South Wales. Accessed July 03, 2020. http://handle.unsw.edu.au/1959.4/42104 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:3686/SOURCE2?view=true.

MLA Handbook (7th Edition):

Gardam, Sandra. “Regulation of lymphocyte development and function by TRAF2 and TRAF3.” 2008. Web. 03 Jul 2020.

Vancouver:

Gardam S. Regulation of lymphocyte development and function by TRAF2 and TRAF3. [Internet] [Doctoral dissertation]. University of New South Wales; 2008. [cited 2020 Jul 03]. Available from: http://handle.unsw.edu.au/1959.4/42104 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:3686/SOURCE2?view=true.

Council of Science Editors:

Gardam S. Regulation of lymphocyte development and function by TRAF2 and TRAF3. [Doctoral Dissertation]. University of New South Wales; 2008. Available from: http://handle.unsw.edu.au/1959.4/42104 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:3686/SOURCE2?view=true


University of Oxford

28. Cheng, Daian. An investigation into the biology and function of protein Icb-1.

Degree: PhD, 2013, University of Oxford

 In this thesis I describe an investigation into the function of the protein Icb-1, a homologue of Themis1 in B cells and monocytes. Themis1 is… (more)

Subjects/Keywords: 612.01575; Immunology; B lymphocyte; Icb-1; Themis2; Themis1; BCR signalling

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APA (6th Edition):

Cheng, D. (2013). An investigation into the biology and function of protein Icb-1. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:e6be188d-3a6f-4e95-aa3a-6f319d791f8c ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581294

Chicago Manual of Style (16th Edition):

Cheng, Daian. “An investigation into the biology and function of protein Icb-1.” 2013. Doctoral Dissertation, University of Oxford. Accessed July 03, 2020. http://ora.ox.ac.uk/objects/uuid:e6be188d-3a6f-4e95-aa3a-6f319d791f8c ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581294.

MLA Handbook (7th Edition):

Cheng, Daian. “An investigation into the biology and function of protein Icb-1.” 2013. Web. 03 Jul 2020.

Vancouver:

Cheng D. An investigation into the biology and function of protein Icb-1. [Internet] [Doctoral dissertation]. University of Oxford; 2013. [cited 2020 Jul 03]. Available from: http://ora.ox.ac.uk/objects/uuid:e6be188d-3a6f-4e95-aa3a-6f319d791f8c ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581294.

Council of Science Editors:

Cheng D. An investigation into the biology and function of protein Icb-1. [Doctoral Dissertation]. University of Oxford; 2013. Available from: http://ora.ox.ac.uk/objects/uuid:e6be188d-3a6f-4e95-aa3a-6f319d791f8c ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581294


University of Iowa

29. Buchta, Claire Marie. Mechanisms of TLR signaling and cooperation in B lymphocytes.

Degree: PhD, Immunology, 2014, University of Iowa

B lymphocytes play important roles in antibody production, cytokine production, and antigen presentation to T cells. Ligation of Toll-like receptors (TLRs) on B cells… (more)

Subjects/Keywords: B lymphocyte; Toll-like receptor; TRAF5; Immunology of Infectious Disease

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APA (6th Edition):

Buchta, C. M. (2014). Mechanisms of TLR signaling and cooperation in B lymphocytes. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/4584

Chicago Manual of Style (16th Edition):

Buchta, Claire Marie. “Mechanisms of TLR signaling and cooperation in B lymphocytes.” 2014. Doctoral Dissertation, University of Iowa. Accessed July 03, 2020. https://ir.uiowa.edu/etd/4584.

MLA Handbook (7th Edition):

Buchta, Claire Marie. “Mechanisms of TLR signaling and cooperation in B lymphocytes.” 2014. Web. 03 Jul 2020.

Vancouver:

Buchta CM. Mechanisms of TLR signaling and cooperation in B lymphocytes. [Internet] [Doctoral dissertation]. University of Iowa; 2014. [cited 2020 Jul 03]. Available from: https://ir.uiowa.edu/etd/4584.

Council of Science Editors:

Buchta CM. Mechanisms of TLR signaling and cooperation in B lymphocytes. [Doctoral Dissertation]. University of Iowa; 2014. Available from: https://ir.uiowa.edu/etd/4584


University of Melbourne

30. Motazedian, Ali. Lymphocyte differentiation from pluripotent stem cells.

Degree: 2017, University of Melbourne

 Human pluripotent stem cells (PSCs), including embryonic stem cells (ESCs) and induced pluripotent stem cells (iPSCs), are cell types that can undergo unlimited self-renewal and,… (more)

Subjects/Keywords: pluripotent stem cells; iPS; ES; lymphocyte; T-cell; B-cell

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APA (6th Edition):

Motazedian, A. (2017). Lymphocyte differentiation from pluripotent stem cells. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/191658

Chicago Manual of Style (16th Edition):

Motazedian, Ali. “Lymphocyte differentiation from pluripotent stem cells.” 2017. Doctoral Dissertation, University of Melbourne. Accessed July 03, 2020. http://hdl.handle.net/11343/191658.

MLA Handbook (7th Edition):

Motazedian, Ali. “Lymphocyte differentiation from pluripotent stem cells.” 2017. Web. 03 Jul 2020.

Vancouver:

Motazedian A. Lymphocyte differentiation from pluripotent stem cells. [Internet] [Doctoral dissertation]. University of Melbourne; 2017. [cited 2020 Jul 03]. Available from: http://hdl.handle.net/11343/191658.

Council of Science Editors:

Motazedian A. Lymphocyte differentiation from pluripotent stem cells. [Doctoral Dissertation]. University of Melbourne; 2017. Available from: http://hdl.handle.net/11343/191658

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