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You searched for subject:( Apoptosis 60). Showing records 1 – 30 of 37 total matches.

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1. Turk, Amy Nicole. Cholangiocarcinoma combination therapy : Tamoxifen and Gemcitabine produce an additive effect in vitro due to differing mechanisms and will be an effective combination in vivo.

Degree: MS, 2010, University of Alabama – Birmingham

Cholangiocarcinoma, a fatal tumor arising from the biliary epithelium, has a very poor 5-year survival rate due to the lack of early diagnosis and effective… (more)

Subjects/Keywords: Apoptosis  – drug effects<; br>; Cholangiocarcinoma  – drug therapy<; br>; Deoxycytidine  – therapeutic use<; br>; Tamoxifen  – therapeutic use

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APA (6th Edition):

Turk, A. N. (2010). Cholangiocarcinoma combination therapy : Tamoxifen and Gemcitabine produce an additive effect in vitro due to differing mechanisms and will be an effective combination in vivo. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,639

Chicago Manual of Style (16th Edition):

Turk, Amy Nicole. “Cholangiocarcinoma combination therapy : Tamoxifen and Gemcitabine produce an additive effect in vitro due to differing mechanisms and will be an effective combination in vivo.” 2010. Masters Thesis, University of Alabama – Birmingham. Accessed January 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,639.

MLA Handbook (7th Edition):

Turk, Amy Nicole. “Cholangiocarcinoma combination therapy : Tamoxifen and Gemcitabine produce an additive effect in vitro due to differing mechanisms and will be an effective combination in vivo.” 2010. Web. 22 Jan 2020.

Vancouver:

Turk AN. Cholangiocarcinoma combination therapy : Tamoxifen and Gemcitabine produce an additive effect in vitro due to differing mechanisms and will be an effective combination in vivo. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2010. [cited 2020 Jan 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,639.

Council of Science Editors:

Turk AN. Cholangiocarcinoma combination therapy : Tamoxifen and Gemcitabine produce an additive effect in vitro due to differing mechanisms and will be an effective combination in vivo. [Masters Thesis]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,639

2. Walls, Ken C. Molecular characterization of neural apoptosis.

Degree: PhD, 2009, University of Alabama – Birmingham

Neural cell death plays a critical role in normal nervous system development and dysregulated neural stem cell death contributes to brain malformation, tumorgenesis, and possibly,… (more)

Subjects/Keywords: Apoptosis<; br>; Autophagy  – genetics<; br>; Cell Death<; br>; Lysosomes <; br>; physiology Neurons  – pathology<; br>; Tumor Suppressor Protein p53

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APA (6th Edition):

Walls, K. C. (2009). Molecular characterization of neural apoptosis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,504

Chicago Manual of Style (16th Edition):

Walls, Ken C. “Molecular characterization of neural apoptosis.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,504.

MLA Handbook (7th Edition):

Walls, Ken C. “Molecular characterization of neural apoptosis.” 2009. Web. 22 Jan 2020.

Vancouver:

Walls KC. Molecular characterization of neural apoptosis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Jan 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,504.

Council of Science Editors:

Walls KC. Molecular characterization of neural apoptosis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,504


Marshall University

3. Gadde, Murali K. Effects of aging on regulators of muscle apoptosis in the female F344BN rat.

Degree: 2009, Marshall University

 Age-related muscle atrophy is a consequence of normal aging characterized by decreases in muscle mass and strength. The mechanism(s) underlying the loss of muscle mass… (more)

Subjects/Keywords: aging; Apoptosis; F344BN; Female; Bax; Bcl-2; Muscle; <; p>; Apoptosis.<; /p>; <; p>; Cell death. <; /p>; <; p>; Aging.<; /p>;

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APA (6th Edition):

Gadde, M. K. (2009). Effects of aging on regulators of muscle apoptosis in the female F344BN rat. (Thesis). Marshall University. Retrieved from http://mds.marshall.edu/etd/596

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gadde, Murali K. “Effects of aging on regulators of muscle apoptosis in the female F344BN rat.” 2009. Thesis, Marshall University. Accessed January 22, 2020. http://mds.marshall.edu/etd/596.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gadde, Murali K. “Effects of aging on regulators of muscle apoptosis in the female F344BN rat.” 2009. Web. 22 Jan 2020.

Vancouver:

Gadde MK. Effects of aging on regulators of muscle apoptosis in the female F344BN rat. [Internet] [Thesis]. Marshall University; 2009. [cited 2020 Jan 22]. Available from: http://mds.marshall.edu/etd/596.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gadde MK. Effects of aging on regulators of muscle apoptosis in the female F344BN rat. [Thesis]. Marshall University; 2009. Available from: http://mds.marshall.edu/etd/596

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. Pawar, Pritish Subhash. Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma.

Degree: PhD, 2008, University of Alabama – Birmingham

Cholangiocarcinoma, a fatal tumor arising from biliary epithelium, has very poor 5-year survival rate due to lack of early diagnosis and effective therapies. Induction of… (more)

Subjects/Keywords: Antigens, CD95  – metabolism <; br>; CASP8 and FADD-Like Apoptosis Regulating Protein  – metabolism <; br>; Calmodulin  – metabolism <; br>; Cholangiocarcinoma <; br>; Signal Transduction

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APA (6th Edition):

Pawar, P. S. (2008). Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,301

Chicago Manual of Style (16th Edition):

Pawar, Pritish Subhash. “Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,301.

MLA Handbook (7th Edition):

Pawar, Pritish Subhash. “Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma.” 2008. Web. 22 Jan 2020.

Vancouver:

Pawar PS. Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Jan 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,301.

Council of Science Editors:

Pawar PS. Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,301

5. McFarland, Braden Cox. Targeting angiogenesis with plasminogen kringle 5.

Degree: PhD, 2009, University of Alabama – Birmingham

The recombinant fifth kringle domain of plasminogen (rK5) has been shown to induce apoptosis of dermal microvessel endothelial cells (MvEC), and this pro-apoptotic effect required… (more)

Subjects/Keywords: Angiogenesis Inhibitors  – metabolism<; br>; Apoptosis<; br>; Glioblastoma  – blood supply<; br>; Neovascularization, Pathologic  – drug therapy<; br>; Plasminogen  – therapeutic use

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APA (6th Edition):

McFarland, B. C. (2009). Targeting angiogenesis with plasminogen kringle 5. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,399

Chicago Manual of Style (16th Edition):

McFarland, Braden Cox. “Targeting angiogenesis with plasminogen kringle 5.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,399.

MLA Handbook (7th Edition):

McFarland, Braden Cox. “Targeting angiogenesis with plasminogen kringle 5.” 2009. Web. 22 Jan 2020.

Vancouver:

McFarland BC. Targeting angiogenesis with plasminogen kringle 5. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Jan 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,399.

Council of Science Editors:

McFarland BC. Targeting angiogenesis with plasminogen kringle 5. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,399


Marshall University

6. Arvapalli, Ravi K. Deferasirox decreases age-associated iron accumulation in the aging F344BN rat heart.

Degree: 2009, Marshall University

 It is thought that aging in rats and humans is associated with increases in iron accumulation and these increases in iron may be associated with… (more)

Subjects/Keywords: <; p>; Apoptosis.<; /p>; <; p>; Aging.<; /p>;

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APA (6th Edition):

Arvapalli, R. K. (2009). Deferasirox decreases age-associated iron accumulation in the aging F344BN rat heart. (Thesis). Marshall University. Retrieved from http://mds.marshall.edu/etd/445

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Arvapalli, Ravi K. “Deferasirox decreases age-associated iron accumulation in the aging F344BN rat heart.” 2009. Thesis, Marshall University. Accessed January 22, 2020. http://mds.marshall.edu/etd/445.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Arvapalli, Ravi K. “Deferasirox decreases age-associated iron accumulation in the aging F344BN rat heart.” 2009. Web. 22 Jan 2020.

Vancouver:

Arvapalli RK. Deferasirox decreases age-associated iron accumulation in the aging F344BN rat heart. [Internet] [Thesis]. Marshall University; 2009. [cited 2020 Jan 22]. Available from: http://mds.marshall.edu/etd/445.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Arvapalli RK. Deferasirox decreases age-associated iron accumulation in the aging F344BN rat heart. [Thesis]. Marshall University; 2009. Available from: http://mds.marshall.edu/etd/445

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. Evans, Lynda Michele. The effect of stearic acid on breast cancer development and progression.

Degree: PhD, 2009, University of Alabama – Birmingham

Stearate is an 18-carbon saturated fatty acid that is found in many foods in the western diet including beef and chocolate. Cell culture studies indicate… (more)

Subjects/Keywords: Antineoplastic Agents  – administration & dosage<; br>; Apoptosis<; br>; Breast Neoplasms  – metabolism<; br>; Breast Neoplasms  – pathology<; br>; Dietary Fats<; br>; Neoplasm Metastasis<; br>; Stearates  – therapeutic use<; br>; Stearic Acids  – administration & dosage

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APA (6th Edition):

Evans, L. M. (2009). The effect of stearic acid on breast cancer development and progression. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,906

Chicago Manual of Style (16th Edition):

Evans, Lynda Michele. “The effect of stearic acid on breast cancer development and progression.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,906.

MLA Handbook (7th Edition):

Evans, Lynda Michele. “The effect of stearic acid on breast cancer development and progression.” 2009. Web. 22 Jan 2020.

Vancouver:

Evans LM. The effect of stearic acid on breast cancer development and progression. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Jan 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,906.

Council of Science Editors:

Evans LM. The effect of stearic acid on breast cancer development and progression. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,906

8. Crowder, Roslyn Nicole. Death receptor 5 (DR5) and CD19 signaling in B cells.

Degree: PhD, 2008, University of Alabama – Birmingham

This dissertation research describes the independent examination of B cell signaling mediated by two cell surface molecules, CD19 and Death receptor 5 (DR5). In previous… (more)

Subjects/Keywords: Antigens, CD19  – immunology<; br>; Apoptosis<; br>; Lupus Erythematosus, Systemic<; br>; Neoplasms<; br>; Receptors, TNF-Related Apoptosis-Inducing Ligand<; br>; Signal Transduction  – immunology

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APA (6th Edition):

Crowder, R. N. (2008). Death receptor 5 (DR5) and CD19 signaling in B cells. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,751

Chicago Manual of Style (16th Edition):

Crowder, Roslyn Nicole. “Death receptor 5 (DR5) and CD19 signaling in B cells.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,751.

MLA Handbook (7th Edition):

Crowder, Roslyn Nicole. “Death receptor 5 (DR5) and CD19 signaling in B cells.” 2008. Web. 22 Jan 2020.

Vancouver:

Crowder RN. Death receptor 5 (DR5) and CD19 signaling in B cells. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Jan 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,751.

Council of Science Editors:

Crowder RN. Death receptor 5 (DR5) and CD19 signaling in B cells. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,751

9. Sun, Mianen. The role of DDX3 in regulating apoptosis, p53 and Snail.

Degree: PhD, 2008, University of Alabama – Birmingham

Cancer is a common disease that causes high rates of lethality. Resistance to cancer therapy is one major obstacle for producing an effective cancer treatment.… (more)

Subjects/Keywords: Apoptosis<; br>; Genes, p53<; br. Glycogen Synthase Kinase 3  – metabolism<; br>; Inhibitor of Apoptosis Proteins  – metabolism<; br>; Protein Kinase Inhibitors  – pharmacology<; br>; Receptors, Death Domain  – metabolism

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APA (6th Edition):

Sun, M. (2008). The role of DDX3 in regulating apoptosis, p53 and Snail. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,827

Chicago Manual of Style (16th Edition):

Sun, Mianen. “The role of DDX3 in regulating apoptosis, p53 and Snail.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,827.

MLA Handbook (7th Edition):

Sun, Mianen. “The role of DDX3 in regulating apoptosis, p53 and Snail.” 2008. Web. 22 Jan 2020.

Vancouver:

Sun M. The role of DDX3 in regulating apoptosis, p53 and Snail. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Jan 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,827.

Council of Science Editors:

Sun M. The role of DDX3 in regulating apoptosis, p53 and Snail. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,827


Universidad Nacional de La Plata

10. Scrochi, Mariela Rita. &#60;i>Herpesvirus equino&#60;/i> 1: evaluación de la apoptosis en cultivo de células heterólogas y homólogas, y en pulmón de ratones BALB/c, como mecanismo patogénico y de evasión de la respuesta inmune del hospedador.

Degree: 2016, Universidad Nacional de La Plata

El herpesvirus equino 1 (EHV-1) es uno de los patógenos más importantes de los equinos, debido a que puede causar signología respiratoria, nerviosa y abortos.… (more)

Subjects/Keywords: Ciencias Veterinarias; Caballos; Herpesvirus Équido 1; <; i>; herpesvirus equino<; /i>; 1; apoptosis; células MDBK; células ED; pulmones

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APA (6th Edition):

Scrochi, M. R. (2016). <i>Herpesvirus equino</i> 1: evaluación de la apoptosis en cultivo de células heterólogas y homólogas, y en pulmón de ratones BALB/c, como mecanismo patogénico y de evasión de la respuesta inmune del hospedador. (Thesis). Universidad Nacional de La Plata. Retrieved from http://hdl.handle.net/10915/53833

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Scrochi, Mariela Rita. “<i>Herpesvirus equino</i> 1: evaluación de la apoptosis en cultivo de células heterólogas y homólogas, y en pulmón de ratones BALB/c, como mecanismo patogénico y de evasión de la respuesta inmune del hospedador.” 2016. Thesis, Universidad Nacional de La Plata. Accessed January 22, 2020. http://hdl.handle.net/10915/53833.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Scrochi, Mariela Rita. “<i>Herpesvirus equino</i> 1: evaluación de la apoptosis en cultivo de células heterólogas y homólogas, y en pulmón de ratones BALB/c, como mecanismo patogénico y de evasión de la respuesta inmune del hospedador.” 2016. Web. 22 Jan 2020.

Vancouver:

Scrochi MR. <i>Herpesvirus equino</i> 1: evaluación de la apoptosis en cultivo de células heterólogas y homólogas, y en pulmón de ratones BALB/c, como mecanismo patogénico y de evasión de la respuesta inmune del hospedador. [Internet] [Thesis]. Universidad Nacional de La Plata; 2016. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/10915/53833.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Scrochi MR. <i>Herpesvirus equino</i> 1: evaluación de la apoptosis en cultivo de células heterólogas y homólogas, y en pulmón de ratones BALB/c, como mecanismo patogénico y de evasión de la respuesta inmune del hospedador. [Thesis]. Universidad Nacional de La Plata; 2016. Available from: http://hdl.handle.net/10915/53833

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

11. Belenky, Michael L. Sensitization of glioma to death receptor 5-mediated apoptosis through genotoxic stress and cell cycle disruption: a study of mechanism.

Degree: PhD, 2011, University of Alabama – Birmingham

Our laboratory reported that a combination of ionizing radiation (IR) or temozolomide (Tmz), a DNA methylating agent clinically approved against glioblastoma multiforme (GBM), a type… (more)

Subjects/Keywords: Antibodies, Monoclonal  – therapeutic use<; br>; Apoptosis  – physiology<; br>; Brain Neoplasms  – drug therapy<; br>; Brain Neoplasms  – radiotherapy<; br>; Cell Cycle<; br>; Glioma  – drug therapy<; br>; Glioma  – radiotherapy<; br>; Receptors, TNF-Related Apoptosis-Inducing Ligand  – agonists

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APA (6th Edition):

Belenky, M. L. (2011). Sensitization of glioma to death receptor 5-mediated apoptosis through genotoxic stress and cell cycle disruption: a study of mechanism. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1032

Chicago Manual of Style (16th Edition):

Belenky, Michael L. “Sensitization of glioma to death receptor 5-mediated apoptosis through genotoxic stress and cell cycle disruption: a study of mechanism.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1032.

MLA Handbook (7th Edition):

Belenky, Michael L. “Sensitization of glioma to death receptor 5-mediated apoptosis through genotoxic stress and cell cycle disruption: a study of mechanism.” 2011. Web. 22 Jan 2020.

Vancouver:

Belenky ML. Sensitization of glioma to death receptor 5-mediated apoptosis through genotoxic stress and cell cycle disruption: a study of mechanism. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Jan 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1032.

Council of Science Editors:

Belenky ML. Sensitization of glioma to death receptor 5-mediated apoptosis through genotoxic stress and cell cycle disruption: a study of mechanism. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1032


University of Alberta

12. Baghdasarian, Argishti. An investigation of aminoglutethimide cytotoxicity and its role in activation of cell death signaling pathways.

Degree: MS, 2013, University of Alberta

 Aminoglutethimide (AG), a drug used for the treatment of breast and ovarian cancers, is known to cause toxicities such as agranulocytosis. To investigate its toxicity… (more)

Subjects/Keywords: Drug-induced agranulocytosis; Idiosyncratic drug reactions; HL-60 cells; Free radicals; Proteomics; Apoptosis; Aminoglutethimide; SILAC

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APA (6th Edition):

Baghdasarian, A. (2013). An investigation of aminoglutethimide cytotoxicity and its role in activation of cell death signaling pathways. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/w9505177q

Chicago Manual of Style (16th Edition):

Baghdasarian, Argishti. “An investigation of aminoglutethimide cytotoxicity and its role in activation of cell death signaling pathways.” 2013. Masters Thesis, University of Alberta. Accessed January 22, 2020. https://era.library.ualberta.ca/files/w9505177q.

MLA Handbook (7th Edition):

Baghdasarian, Argishti. “An investigation of aminoglutethimide cytotoxicity and its role in activation of cell death signaling pathways.” 2013. Web. 22 Jan 2020.

Vancouver:

Baghdasarian A. An investigation of aminoglutethimide cytotoxicity and its role in activation of cell death signaling pathways. [Internet] [Masters thesis]. University of Alberta; 2013. [cited 2020 Jan 22]. Available from: https://era.library.ualberta.ca/files/w9505177q.

Council of Science Editors:

Baghdasarian A. An investigation of aminoglutethimide cytotoxicity and its role in activation of cell death signaling pathways. [Masters Thesis]. University of Alberta; 2013. Available from: https://era.library.ualberta.ca/files/w9505177q

13. Eom, Tae-Yeon. Regulation of neural precursor cell apoptosis and proliferation by glycogen synthase kinase-3.

Degree: PhD, 2009, University of Alabama – Birmingham

Neurogenesis is a crucial process for development, plasticity, and regenerative capacity of the developing and adult brain. Impairment of neurogenesis has been implicated in the… (more)

Subjects/Keywords: Apoptosis<; br>; Glycogen Synthase Kinase 3  – metabolism<; br>; Mice, Inbred C57BL<; br>; Mice, Knockout<; br>; Mice, Transgenic<; br>; Neurons  – cytology<; br>; Phosphorylation

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APA (6th Edition):

Eom, T. (2009). Regulation of neural precursor cell apoptosis and proliferation by glycogen synthase kinase-3. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,390

Chicago Manual of Style (16th Edition):

Eom, Tae-Yeon. “Regulation of neural precursor cell apoptosis and proliferation by glycogen synthase kinase-3.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,390.

MLA Handbook (7th Edition):

Eom, Tae-Yeon. “Regulation of neural precursor cell apoptosis and proliferation by glycogen synthase kinase-3.” 2009. Web. 22 Jan 2020.

Vancouver:

Eom T. Regulation of neural precursor cell apoptosis and proliferation by glycogen synthase kinase-3. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Jan 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,390.

Council of Science Editors:

Eom T. Regulation of neural precursor cell apoptosis and proliferation by glycogen synthase kinase-3. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,390

14. Ontiveros, Steven J. Intracellular trafficking of the hantaviral nucleocapsid protein and its function in modulation of immune signaling.

Degree: PhD, 2009, University of Alabama – Birmingham

Old World and New World hantaviruses, family Bunyaviridae, mature intracellularly within cellular compartments. Although it is generally accepted they assemble and bud in the Golgi… (more)

Subjects/Keywords: Apoptosis<; br>; Capsid Proteins  – physiology<; br>; Hantaan virus  – pathogenicity<; br>; Hantavirus  – pathogenicity<; br>; Signal Transduction<; br>; Viral Core Proteins  – physiology<; br>; Virulence Factors  – physiology

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APA (6th Edition):

Ontiveros, S. J. (2009). Intracellular trafficking of the hantaviral nucleocapsid protein and its function in modulation of immune signaling. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,687

Chicago Manual of Style (16th Edition):

Ontiveros, Steven J. “Intracellular trafficking of the hantaviral nucleocapsid protein and its function in modulation of immune signaling.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,687.

MLA Handbook (7th Edition):

Ontiveros, Steven J. “Intracellular trafficking of the hantaviral nucleocapsid protein and its function in modulation of immune signaling.” 2009. Web. 22 Jan 2020.

Vancouver:

Ontiveros SJ. Intracellular trafficking of the hantaviral nucleocapsid protein and its function in modulation of immune signaling. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Jan 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,687.

Council of Science Editors:

Ontiveros SJ. Intracellular trafficking of the hantaviral nucleocapsid protein and its function in modulation of immune signaling. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,687

15. Swindall, Amanda F. The Role Of St6gal-I Sialylation In Fas (cd95) Death Receptor Function And Tumorigenesis.

Degree: PhD, 2012, University of Alabama – Birmingham

The golgi glycosyltransferase, ST6Gal-I, adds a negatively-charged sialic acid in an alpha-2-6 linkage to N-linked glycans. ST6Gal-I is upregulated in many cancers, and is associated… (more)

Subjects/Keywords: Antigens, CD – metabolism.<; br>; Antigens, CD95 – metabolism.<; br>; Apoptosis<; br>; Tumor Cells, Cultured<; br>; Colonic Neoplasms<; br>; Neoplasm Proteins – metabolism.<; br>; Sialyltransferases – metabolism.

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APA (6th Edition):

Swindall, A. F. (2012). The Role Of St6gal-I Sialylation In Fas (cd95) Death Receptor Function And Tumorigenesis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1392

Chicago Manual of Style (16th Edition):

Swindall, Amanda F. “The Role Of St6gal-I Sialylation In Fas (cd95) Death Receptor Function And Tumorigenesis.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1392.

MLA Handbook (7th Edition):

Swindall, Amanda F. “The Role Of St6gal-I Sialylation In Fas (cd95) Death Receptor Function And Tumorigenesis.” 2012. Web. 22 Jan 2020.

Vancouver:

Swindall AF. The Role Of St6gal-I Sialylation In Fas (cd95) Death Receptor Function And Tumorigenesis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2020 Jan 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1392.

Council of Science Editors:

Swindall AF. The Role Of St6gal-I Sialylation In Fas (cd95) Death Receptor Function And Tumorigenesis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1392

16. Szafran, April Adams. The use of molecular imaging to evaluate the efficacy of treatments for metastatic breast cancer.

Degree: PhD, 2008, University of Alabama – Birmingham

The development of molecular imaging technologies has allowed biomedical researchers to study the process of cancer metastasis in animal models of disease. Bioluminescence imaging has… (more)

Subjects/Keywords: Antibodies, Monoclonal  – pharmacology<; br>; Antineoplastic Combined Chemotherapy<; br>; Protocols  – pharmacology<; br>; Bone Neoplasms<; br>; Breast Neoplasms<; br>; Diphosphonates  – pharmacology<; br>; Imidazoles  – pharmacology<; br>; Mammary Neoplasms, Experimental  – pathology<; br>; Receptors, TNF-Related Apoptosis-Inducing Ligand  – agonists

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APA (6th Edition):

Szafran, A. A. (2008). The use of molecular imaging to evaluate the efficacy of treatments for metastatic breast cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,927

Chicago Manual of Style (16th Edition):

Szafran, April Adams. “The use of molecular imaging to evaluate the efficacy of treatments for metastatic breast cancer.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,927.

MLA Handbook (7th Edition):

Szafran, April Adams. “The use of molecular imaging to evaluate the efficacy of treatments for metastatic breast cancer.” 2008. Web. 22 Jan 2020.

Vancouver:

Szafran AA. The use of molecular imaging to evaluate the efficacy of treatments for metastatic breast cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Jan 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,927.

Council of Science Editors:

Szafran AA. The use of molecular imaging to evaluate the efficacy of treatments for metastatic breast cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,927


Marshall University

17. Nalabotu, Siva Krishna. Evaluation of the Role of Oxidative Stress, Inflammation and Apoptosis in the Pulmonary and the Hepatic Toxicity Induced by Cerium Oxide Nanoparticles Following Intratracheal Instillation in Male Sprague-Dawley Rats.

Degree: 2012, Marshall University

 The field of nanotechnology is rapidly progressing with potential applications in the automobile, healthcare, electronics, cosmetics, textiles, information technology, and environmental sectors. Nanomaterials are engineered… (more)

Subjects/Keywords: apoptosis; Cerium oxide nanoparticles; inflammation; liver toxicity; lung toxicity; oxidative stress; <; p>; Nanoparticles - Health aspects.<; /p>; <; p>; Cerium oxides.<; /p>; <; p>; Medicine<; strong>; - <; /strong>; Research.<; /p>;

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APA (6th Edition):

Nalabotu, S. K. (2012). Evaluation of the Role of Oxidative Stress, Inflammation and Apoptosis in the Pulmonary and the Hepatic Toxicity Induced by Cerium Oxide Nanoparticles Following Intratracheal Instillation in Male Sprague-Dawley Rats. (Thesis). Marshall University. Retrieved from http://mds.marshall.edu/etd/228

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nalabotu, Siva Krishna. “Evaluation of the Role of Oxidative Stress, Inflammation and Apoptosis in the Pulmonary and the Hepatic Toxicity Induced by Cerium Oxide Nanoparticles Following Intratracheal Instillation in Male Sprague-Dawley Rats.” 2012. Thesis, Marshall University. Accessed January 22, 2020. http://mds.marshall.edu/etd/228.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nalabotu, Siva Krishna. “Evaluation of the Role of Oxidative Stress, Inflammation and Apoptosis in the Pulmonary and the Hepatic Toxicity Induced by Cerium Oxide Nanoparticles Following Intratracheal Instillation in Male Sprague-Dawley Rats.” 2012. Web. 22 Jan 2020.

Vancouver:

Nalabotu SK. Evaluation of the Role of Oxidative Stress, Inflammation and Apoptosis in the Pulmonary and the Hepatic Toxicity Induced by Cerium Oxide Nanoparticles Following Intratracheal Instillation in Male Sprague-Dawley Rats. [Internet] [Thesis]. Marshall University; 2012. [cited 2020 Jan 22]. Available from: http://mds.marshall.edu/etd/228.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nalabotu SK. Evaluation of the Role of Oxidative Stress, Inflammation and Apoptosis in the Pulmonary and the Hepatic Toxicity Induced by Cerium Oxide Nanoparticles Following Intratracheal Instillation in Male Sprague-Dawley Rats. [Thesis]. Marshall University; 2012. Available from: http://mds.marshall.edu/etd/228

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. Phipps, Matthew Christopher. Development Of Electrospun Bone-Mimetic Matrices For Bone Regenerative Applications.

Degree: PhD, 2012, University of Alabama – Birmingham

Although bone has a dramatic capacity for regeneration, certain injuries and procedures present defects that are unable to heal properly, requiring surgical intervention to induce… (more)

Subjects/Keywords: Apoptosis – drug effects<; br>; beta Catenin – metabolism.<; br>; Breast Neoplasms – drug therapy<; br>; Cell Proliferation – drug effects.<; br>; Cyclic GMP-Dependent Protein Kinases – metabolism.<; br>; Cyclic Nucleotide Phosphodiesterases, Type 5 – genetics.<; br>; Phosphodiesterase 5 Inhibitors.<; br>; Sulindac – analogs & derivatives.<; br>; Wnt Proteins – metabolism.

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APA (6th Edition):

Phipps, M. C. (2012). Development Of Electrospun Bone-Mimetic Matrices For Bone Regenerative Applications. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1410

Chicago Manual of Style (16th Edition):

Phipps, Matthew Christopher. “Development Of Electrospun Bone-Mimetic Matrices For Bone Regenerative Applications.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1410.

MLA Handbook (7th Edition):

Phipps, Matthew Christopher. “Development Of Electrospun Bone-Mimetic Matrices For Bone Regenerative Applications.” 2012. Web. 22 Jan 2020.

Vancouver:

Phipps MC. Development Of Electrospun Bone-Mimetic Matrices For Bone Regenerative Applications. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2020 Jan 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1410.

Council of Science Editors:

Phipps MC. Development Of Electrospun Bone-Mimetic Matrices For Bone Regenerative Applications. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1410

19. Rayburn, Elizabeth R. Novel immunomodulatory oligonucleotides for cancer therapy.

Degree: PhD, 2007, University of Alabama – Birmingham

Prostate and colon cancer are two of the most prevalent cancers in the United States. Cancer therapy, including surgery, chemotherapy, radiotherapy and biological therapy, often… (more)

Subjects/Keywords: Adenocarcinoma  – drug therapy<; br>; Antineoplastic Agents  – therapeutic use<; br>; Antineoplastic Combined Chemotherapy Protocols  – therapeutic use<; br>; Apoptosis  – drug effects<; br>; Colonic Neoplasms  – drug therapy<; br>; Deoxycytidine  – analogs & derivatives<; br>; Immunologic Factors  – therapeutic use<; br>; Oligonucleotides  – therapeutic use<; br>; Prostatic Neoplasms  – drug therapy<; br>; Taxoids  – therapeutic use<; br>; Toll-Like Receptor 9  – agonists<; br>; Tumor Suppressor Protein p53  – metabolism

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APA (6th Edition):

Rayburn, E. R. (2007). Novel immunomodulatory oligonucleotides for cancer therapy. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,463

Chicago Manual of Style (16th Edition):

Rayburn, Elizabeth R. “Novel immunomodulatory oligonucleotides for cancer therapy.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,463.

MLA Handbook (7th Edition):

Rayburn, Elizabeth R. “Novel immunomodulatory oligonucleotides for cancer therapy.” 2007. Web. 22 Jan 2020.

Vancouver:

Rayburn ER. Novel immunomodulatory oligonucleotides for cancer therapy. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Jan 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,463.

Council of Science Editors:

Rayburn ER. Novel immunomodulatory oligonucleotides for cancer therapy. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,463

20. Ezell, Scharri J. Preclinical pharmacology of novel synthetic iminoquinones as anticancer agents.

Degree: PhD, 2010, University of Alabama – Birmingham

Prostate cancer is the most common male malignancy and the second leading cause of cancer related death in the United States. Despite recent advances in… (more)

Subjects/Keywords: Antineoplastic Agents  – therapeutic<; br>; Apoptosis  – drug effects<; br>; Prostatic Neoplasms  – drug therapy<; br>; Pyrroles  – therapeutic use<; br>; Quinolones  – therapeutic use<; br>; Receptors, Androgen  – metabolism

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APA (6th Edition):

Ezell, S. J. (2010). Preclinical pharmacology of novel synthetic iminoquinones as anticancer agents. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,711

Chicago Manual of Style (16th Edition):

Ezell, Scharri J. “Preclinical pharmacology of novel synthetic iminoquinones as anticancer agents.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,711.

MLA Handbook (7th Edition):

Ezell, Scharri J. “Preclinical pharmacology of novel synthetic iminoquinones as anticancer agents.” 2010. Web. 22 Jan 2020.

Vancouver:

Ezell SJ. Preclinical pharmacology of novel synthetic iminoquinones as anticancer agents. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Jan 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,711.

Council of Science Editors:

Ezell SJ. Preclinical pharmacology of novel synthetic iminoquinones as anticancer agents. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,711

21. Burke, Stephen P. Regulation of apoptosis by Smac, IAPS, and the ubiquitin proteasome system.

Degree: PhD, 2010, University of Alabama – Birmingham

Apoptosis, or programmed cell death, is essential for the development and maintenance of mammalian tissues. Activation of cysteinyl aspartate specific proteases, called caspases, is crucial… (more)

Subjects/Keywords: Apoptosomes  – genetics<; br>; Caspase 3  – metabolism<; br>; Caspase 9  – metabolism<; br>; Inhibitor of Apoptosis Proteins  – metabolism<; br>; Mitochondrial Proteins<; br>; Protein Multimerization  – metabolism

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APA (6th Edition):

Burke, S. P. (2010). Regulation of apoptosis by Smac, IAPS, and the ubiquitin proteasome system. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,818

Chicago Manual of Style (16th Edition):

Burke, Stephen P. “Regulation of apoptosis by Smac, IAPS, and the ubiquitin proteasome system.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,818.

MLA Handbook (7th Edition):

Burke, Stephen P. “Regulation of apoptosis by Smac, IAPS, and the ubiquitin proteasome system.” 2010. Web. 22 Jan 2020.

Vancouver:

Burke SP. Regulation of apoptosis by Smac, IAPS, and the ubiquitin proteasome system. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Jan 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,818.

Council of Science Editors:

Burke SP. Regulation of apoptosis by Smac, IAPS, and the ubiquitin proteasome system. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,818

22. Diers, Anne R. Regulation of redox signaling by lipid electrophiles in breast cancer.

Degree: PhD, 2009, University of Alabama – Birmingham

A number of steps in breast cancer progression and metastasis are regulated by redox signaling pathways. Electrophilic lipids such as 15-deoxy-[delta]12,14-prostaglandin J2 (15d-PGJ2) are mediators… (more)

Subjects/Keywords: Antineoplastic Agents  – pharmacology<; br>; Apoptosis  – drug effects<; br>; Breast Neoplasms<; br>; Mitochondria  – drug effects<; br>; Mitochondria  – metabolism<; br>; Prostaglandin D2  – analogs & derivatives

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APA (6th Edition):

Diers, A. R. (2009). Regulation of redox signaling by lipid electrophiles in breast cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,908

Chicago Manual of Style (16th Edition):

Diers, Anne R. “Regulation of redox signaling by lipid electrophiles in breast cancer.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,908.

MLA Handbook (7th Edition):

Diers, Anne R. “Regulation of redox signaling by lipid electrophiles in breast cancer.” 2009. Web. 22 Jan 2020.

Vancouver:

Diers AR. Regulation of redox signaling by lipid electrophiles in breast cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Jan 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,908.

Council of Science Editors:

Diers AR. Regulation of redox signaling by lipid electrophiles in breast cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,908

23. Paik, Jason Chang. A nucleolar specificity factor for E2F1-induced cell death.

Degree: PhD, 2010, University of Alabama – Birmingham

The E2F family of transcription factors are important regulators of cell proliferation, and are often dysregulated in cancers. One member of the E2F family, E2F1,… (more)

Subjects/Keywords: Apoptosis<; br>; Chromosomal Proteins, Non-Histone  – physiology<; br>; E2F1 Transcription Factor  – physiology<; br>; Nuclear Proteins  – physiology<; br>; Transcription, Genetic<; br>; Transcriptional Activation

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APA (6th Edition):

Paik, J. C. (2010). A nucleolar specificity factor for E2F1-induced cell death. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1171

Chicago Manual of Style (16th Edition):

Paik, Jason Chang. “A nucleolar specificity factor for E2F1-induced cell death.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1171.

MLA Handbook (7th Edition):

Paik, Jason Chang. “A nucleolar specificity factor for E2F1-induced cell death.” 2010. Web. 22 Jan 2020.

Vancouver:

Paik JC. A nucleolar specificity factor for E2F1-induced cell death. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Jan 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1171.

Council of Science Editors:

Paik JC. A nucleolar specificity factor for E2F1-induced cell death. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1171

24. Meares, Gordon P. Directing Akt and GSK3[beta] : molecular insights into cell signaling and survival.

Degree: PhD, 2007, University of Alabama – Birmingham

Proper regulation of survival signaling is critical for all organisms. One important signaling cascade involved in the coordinated effort to control signals influencing cell fate… (more)

Subjects/Keywords: Apoptosis  – physiology <; br>; Cell Nucleus  – metabolism <; br>; Glycogen Synthase Kinase 3  – metabolism <; br>; HSP90 Heat-Shock Proteins  – physiology <; br>; Insulin  – physiology <; br>; Insulin-Like Growth Factor I  – physiology <; br>; Nuclear Localization Signals <; br>; Signal Transduction  – physiology

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APA (6th Edition):

Meares, G. P. (2007). Directing Akt and GSK3[beta] : molecular insights into cell signaling and survival. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,134

Chicago Manual of Style (16th Edition):

Meares, Gordon P. “Directing Akt and GSK3[beta] : molecular insights into cell signaling and survival.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,134.

MLA Handbook (7th Edition):

Meares, Gordon P. “Directing Akt and GSK3[beta] : molecular insights into cell signaling and survival.” 2007. Web. 22 Jan 2020.

Vancouver:

Meares GP. Directing Akt and GSK3[beta] : molecular insights into cell signaling and survival. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Jan 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,134.

Council of Science Editors:

Meares GP. Directing Akt and GSK3[beta] : molecular insights into cell signaling and survival. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,134

25. Harms, Kelly Lynn. Mechanisms of P53-mediated apoptosis.

Degree: PhD, 2007, University of Alabama – Birmingham

The p53 tumor suppressor is the most commonly inactivated gene in human cancers. The ability of p53, a transcription factor, to regulate genes that mediate… (more)

Subjects/Keywords: Apoptosis  – genetics<; br>; DNA, Neoplasm  – metabolism<; br>; Gene Expression Regulation  – physiology<; br>; Histone Deacetylases  – metabolism<; br>; Insulin-Like Growth Factor Binding Protein 3  – genetics<; br>; Repressor Proteins  – metabolism<; br>; Transcriptional Activation  – physiology<; br>; Tumor Suppressor Protein p53

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APA (6th Edition):

Harms, K. L. (2007). Mechanisms of P53-mediated apoptosis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,446

Chicago Manual of Style (16th Edition):

Harms, Kelly Lynn. “Mechanisms of P53-mediated apoptosis.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,446.

MLA Handbook (7th Edition):

Harms, Kelly Lynn. “Mechanisms of P53-mediated apoptosis.” 2007. Web. 22 Jan 2020.

Vancouver:

Harms KL. Mechanisms of P53-mediated apoptosis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Jan 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,446.

Council of Science Editors:

Harms KL. Mechanisms of P53-mediated apoptosis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,446

26. Geng, Ying. p53-dependent neural stem cell death regulation.

Degree: PhD, 2008, University of Alabama – Birmingham

Regulation of neural precursor cell (NPC) death is important for both normal brain development and prevention of brain tumor formation. The tumor suppressor p53 is… (more)

Subjects/Keywords: Antineoplastic Agents  – pharmacology<; br>; Apoptosis  – physiology bcl-2-Associated X Protein  – physiology<; br>; Brain Neoplasms  – genetics Cerebellum  – cytology<; br>; Chloroquine  – pharmacology<; br>; Cytoplasm  – metabolism<; br>; Glioma  – genetics Neurons  – cytology<; br>; Stem Cells  – cytology Transcriptional Activation  – physiology<; br>; Tumor Suppressor Protein p53  – genetics

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APA (6th Edition):

Geng, Y. (2008). p53-dependent neural stem cell death regulation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,767

Chicago Manual of Style (16th Edition):

Geng, Ying. “p53-dependent neural stem cell death regulation.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,767.

MLA Handbook (7th Edition):

Geng, Ying. “p53-dependent neural stem cell death regulation.” 2008. Web. 22 Jan 2020.

Vancouver:

Geng Y. p53-dependent neural stem cell death regulation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Jan 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,767.

Council of Science Editors:

Geng Y. p53-dependent neural stem cell death regulation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,767

27. Amm, Hope. Mechanisms by which TRA-8 anti-DR5 antibody and chemotherapy enhance cytotoxicity in breast cancer.

Degree: PhD, 2010, University of Alabama – Birmingham

Breast cancer is the second leading cause of cancer-related death in American women and metastatic breast cancer has a 5-year survival rate of only 26%.… (more)

Subjects/Keywords: Antibodies, Monoclonal  – therapeutic use<; br>; Antineoplastic Combined Chemotherapy Protocols<; br>; Boronic Acids  – administration & dosage<; br>; Breast Neoplasms  – drug therapy<; br>; Doxorubicin  – administration & dosage<; br>; Drug Resistance, Neoplasm<; br>; Pyrazines  – administration & dosage<; br>; Receptors, TNF-Related Apoptosis-Inducing Ligand  – agonists

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APA (6th Edition):

Amm, H. (2010). Mechanisms by which TRA-8 anti-DR5 antibody and chemotherapy enhance cytotoxicity in breast cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,997

Chicago Manual of Style (16th Edition):

Amm, Hope. “Mechanisms by which TRA-8 anti-DR5 antibody and chemotherapy enhance cytotoxicity in breast cancer.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,997.

MLA Handbook (7th Edition):

Amm, Hope. “Mechanisms by which TRA-8 anti-DR5 antibody and chemotherapy enhance cytotoxicity in breast cancer.” 2010. Web. 22 Jan 2020.

Vancouver:

Amm H. Mechanisms by which TRA-8 anti-DR5 antibody and chemotherapy enhance cytotoxicity in breast cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Jan 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,997.

Council of Science Editors:

Amm H. Mechanisms by which TRA-8 anti-DR5 antibody and chemotherapy enhance cytotoxicity in breast cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,997

28. Felipe Augusto Rocha Rodrigues. Propriedades citotÃxica, genotÃxica e antitumoral de um benzil-isotiocianato isolado de Moringa oleifera (MORINGACEAE).

Degree: Master, 2010, Universidade Federal do Ceará

 Moringa oleifera Lam. à uma planta tropical com grande importÃncia por seus usos medicinais. VÃrios compostos jà foram isolados de diferentes partes da planta, e… (more)

Subjects/Keywords: FARMACOLOGIA; Moringa oleifera; Benzilisotiocianato; PeroxidaÃÃo lipÃdica; Apoptose; HL-60; Moringa oleifera; Benzylisothiocyanate; Lipid peroxidation; Apoptosis; HL-60; Apoptose; Estresse Oxidativo; Leucemia MielomonocÃtica CrÃnica

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rodrigues, F. A. R. (2010). Propriedades citotÃxica, genotÃxica e antitumoral de um benzil-isotiocianato isolado de Moringa oleifera (MORINGACEAE). (Masters Thesis). Universidade Federal do Ceará. Retrieved from http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=5979 ;

Chicago Manual of Style (16th Edition):

Rodrigues, Felipe Augusto Rocha. “Propriedades citotÃxica, genotÃxica e antitumoral de um benzil-isotiocianato isolado de Moringa oleifera (MORINGACEAE).” 2010. Masters Thesis, Universidade Federal do Ceará. Accessed January 22, 2020. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=5979 ;.

MLA Handbook (7th Edition):

Rodrigues, Felipe Augusto Rocha. “Propriedades citotÃxica, genotÃxica e antitumoral de um benzil-isotiocianato isolado de Moringa oleifera (MORINGACEAE).” 2010. Web. 22 Jan 2020.

Vancouver:

Rodrigues FAR. Propriedades citotÃxica, genotÃxica e antitumoral de um benzil-isotiocianato isolado de Moringa oleifera (MORINGACEAE). [Internet] [Masters thesis]. Universidade Federal do Ceará 2010. [cited 2020 Jan 22]. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=5979 ;.

Council of Science Editors:

Rodrigues FAR. Propriedades citotÃxica, genotÃxica e antitumoral de um benzil-isotiocianato isolado de Moringa oleifera (MORINGACEAE). [Masters Thesis]. Universidade Federal do Ceará 2010. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=5979 ;

29. Josà Delano Barreto Marinho Filho. ContribuiÃÃo das espÃcies reativas de oxigÃnio na atividade anti-leucÃmica da cordiaquinona J.

Degree: Master, 2009, Universidade Federal do Ceará

Cordiaquinona J à uma 1,4-naftoquinona isolada das raÃzes de Cordia leucocephala, que apresenta atividade antifÃngica e larvicida. No entanto, nÃo hà relatos quanto a sua… (more)

Subjects/Keywords: FARMACOLOGIA; Cordiaquinona J; citotoxicidade; apoptose; cÃlulas HL-60; estresse oxidativo; Cordiaquinone J; cytotoxicity; apoptosis; HL-60 cells; oxidative stress; Leucemia MielomonocÃtica CrÃnica; Testes de Toxicidade

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Filho, J. D. B. M. (2009). ContribuiÃÃo das espÃcies reativas de oxigÃnio na atividade anti-leucÃmica da cordiaquinona J. (Masters Thesis). Universidade Federal do Ceará. Retrieved from http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=3152 ;

Chicago Manual of Style (16th Edition):

Filho, Josà Delano Barreto Marinho. “ContribuiÃÃo das espÃcies reativas de oxigÃnio na atividade anti-leucÃmica da cordiaquinona J.” 2009. Masters Thesis, Universidade Federal do Ceará. Accessed January 22, 2020. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=3152 ;.

MLA Handbook (7th Edition):

Filho, Josà Delano Barreto Marinho. “ContribuiÃÃo das espÃcies reativas de oxigÃnio na atividade anti-leucÃmica da cordiaquinona J.” 2009. Web. 22 Jan 2020.

Vancouver:

Filho JDBM. ContribuiÃÃo das espÃcies reativas de oxigÃnio na atividade anti-leucÃmica da cordiaquinona J. [Internet] [Masters thesis]. Universidade Federal do Ceará 2009. [cited 2020 Jan 22]. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=3152 ;.

Council of Science Editors:

Filho JDBM. ContribuiÃÃo das espÃcies reativas de oxigÃnio na atividade anti-leucÃmica da cordiaquinona J. [Masters Thesis]. Universidade Federal do Ceará 2009. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=3152 ;

30. McFerrin, Michael Bryan. Role of calcium-activated potassium channels in glioblastoma cell volume regulation.

Degree: PhD, 2011, University of Alabama – Birmingham

The most common and most malignant gliomas are the Grade IV Glioblastoma Multiforme (GBM), characterized by a highly proliferative tumor mass and extremely invasive phenotype… (more)

Subjects/Keywords: Apoptosis<; br>; Glioblastoma  – metabolism<; br>; Intermediate-Conductance Calcium-Activated Potassium Channels<; br>; Large-Conductance Calcium-Activated Potassium Channels<; br>; Potassium Channels, Calcium-Activated  – metabolism

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

McFerrin, M. B. (2011). Role of calcium-activated potassium channels in glioblastoma cell volume regulation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1054

Chicago Manual of Style (16th Edition):

McFerrin, Michael Bryan. “Role of calcium-activated potassium channels in glioblastoma cell volume regulation.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 22, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1054.

MLA Handbook (7th Edition):

McFerrin, Michael Bryan. “Role of calcium-activated potassium channels in glioblastoma cell volume regulation.” 2011. Web. 22 Jan 2020.

Vancouver:

McFerrin MB. Role of calcium-activated potassium channels in glioblastoma cell volume regulation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Jan 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1054.

Council of Science Editors:

McFerrin MB. Role of calcium-activated potassium channels in glioblastoma cell volume regulation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1054

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