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You searched for subject:( Apoptosis physiology bcl 2 Associated X Protein physiology 60). Showing records 1 – 30 of 60950 total matches.

[1] [2] [3] [4] [5] … [2032]

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1. Geng, Ying. p53-dependent neural stem cell death regulation.

Degree: PhD, 2008, University of Alabama – Birmingham

Regulation of neural precursor cell (NPC) death is important for both normal brain development and prevention of brain tumor formation. The tumor suppressor p53 is… (more)

Subjects/Keywords: Antineoplastic Agents  – pharmacology<; br>; Apoptosis  – physiology bcl-2-Associated X Protein  – physiology<; br>; Brain Neoplasms  – genetics Cerebellum  – cytology<; br>; Chloroquine  – pharmacology<; br>; Cytoplasm  – metabolism<; br>; Glioma  – genetics Neurons  – cytology<; br>; Stem Cells  – cytology Transcriptional Activation  – physiology<; br>; Tumor Suppressor Protein p53  – genetics

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APA (6th Edition):

Geng, Y. (2008). p53-dependent neural stem cell death regulation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,767

Chicago Manual of Style (16th Edition):

Geng, Ying. “p53-dependent neural stem cell death regulation.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,767.

MLA Handbook (7th Edition):

Geng, Ying. “p53-dependent neural stem cell death regulation.” 2008. Web. 21 Jan 2020.

Vancouver:

Geng Y. p53-dependent neural stem cell death regulation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,767.

Council of Science Editors:

Geng Y. p53-dependent neural stem cell death regulation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,767


University of Central Florida

2. Garg, Pranav. Biophysical Characterization of the Membrane Binding Domain of the Pro-apoptotic Protein Bax.

Degree: 2011, University of Central Florida

 The BCL-2 family of proteins tightly regulates the delicate balance between life and death. The pore forming Bax is a pro-apoptotic member belonging to this… (more)

Subjects/Keywords: Apoptosis; Circular dichroism; Fluorescence; Proteins; bcl 2-Associated X Protein

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APA (6th Edition):

Garg, P. (2011). Biophysical Characterization of the Membrane Binding Domain of the Pro-apoptotic Protein Bax. (Masters Thesis). University of Central Florida. Retrieved from https://stars.library.ucf.edu/etd/6652

Chicago Manual of Style (16th Edition):

Garg, Pranav. “Biophysical Characterization of the Membrane Binding Domain of the Pro-apoptotic Protein Bax.” 2011. Masters Thesis, University of Central Florida. Accessed January 21, 2020. https://stars.library.ucf.edu/etd/6652.

MLA Handbook (7th Edition):

Garg, Pranav. “Biophysical Characterization of the Membrane Binding Domain of the Pro-apoptotic Protein Bax.” 2011. Web. 21 Jan 2020.

Vancouver:

Garg P. Biophysical Characterization of the Membrane Binding Domain of the Pro-apoptotic Protein Bax. [Internet] [Masters thesis]. University of Central Florida; 2011. [cited 2020 Jan 21]. Available from: https://stars.library.ucf.edu/etd/6652.

Council of Science Editors:

Garg P. Biophysical Characterization of the Membrane Binding Domain of the Pro-apoptotic Protein Bax. [Masters Thesis]. University of Central Florida; 2011. Available from: https://stars.library.ucf.edu/etd/6652


University of Central Florida

3. Zhang, Ge. Distinct Domains of Bax are Involved in Mitochondrial Bioenergetics and Apoptosis.

Degree: 2011, University of Central Florida

Apoptosis is essential for cellular homeostasis and is also a pathologic feature of various diseases. The balance between Bcl-2 family proteins determines whether a cell… (more)

Subjects/Keywords: Apoptosis; Bioenergetics; Mitochondria; bcl 2-Associated X Protein

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APA (6th Edition):

Zhang, G. (2011). Distinct Domains of Bax are Involved in Mitochondrial Bioenergetics and Apoptosis. (Masters Thesis). University of Central Florida. Retrieved from https://stars.library.ucf.edu/etd/6653

Chicago Manual of Style (16th Edition):

Zhang, Ge. “Distinct Domains of Bax are Involved in Mitochondrial Bioenergetics and Apoptosis.” 2011. Masters Thesis, University of Central Florida. Accessed January 21, 2020. https://stars.library.ucf.edu/etd/6653.

MLA Handbook (7th Edition):

Zhang, Ge. “Distinct Domains of Bax are Involved in Mitochondrial Bioenergetics and Apoptosis.” 2011. Web. 21 Jan 2020.

Vancouver:

Zhang G. Distinct Domains of Bax are Involved in Mitochondrial Bioenergetics and Apoptosis. [Internet] [Masters thesis]. University of Central Florida; 2011. [cited 2020 Jan 21]. Available from: https://stars.library.ucf.edu/etd/6653.

Council of Science Editors:

Zhang G. Distinct Domains of Bax are Involved in Mitochondrial Bioenergetics and Apoptosis. [Masters Thesis]. University of Central Florida; 2011. Available from: https://stars.library.ucf.edu/etd/6653


McGill University

4. Ring, Giselle Natasha. Identification and characterization of TMEM 85, a novel suppressor of bax-mediated cell death in yeast.

Degree: MS, Division of Experimental Medicine., 2007, McGill University

 The ability to evade apoptosis is an acquired characteristic associated with many normal and pathophysiological processes. TMEM 85 represents a novel transmembrane domain containing human… (more)

Subjects/Keywords: Apoptosis  – physiology.; bcl-2-Associated X Protein  – metabolism.; Membrane Proteins  – metabolism.; Saccharomyces cerevisiae Proteins  – metabolism.

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APA (6th Edition):

Ring, G. N. (2007). Identification and characterization of TMEM 85, a novel suppressor of bax-mediated cell death in yeast. (Masters Thesis). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile112352.pdf

Chicago Manual of Style (16th Edition):

Ring, Giselle Natasha. “Identification and characterization of TMEM 85, a novel suppressor of bax-mediated cell death in yeast.” 2007. Masters Thesis, McGill University. Accessed January 21, 2020. http://digitool.library.mcgill.ca/thesisfile112352.pdf.

MLA Handbook (7th Edition):

Ring, Giselle Natasha. “Identification and characterization of TMEM 85, a novel suppressor of bax-mediated cell death in yeast.” 2007. Web. 21 Jan 2020.

Vancouver:

Ring GN. Identification and characterization of TMEM 85, a novel suppressor of bax-mediated cell death in yeast. [Internet] [Masters thesis]. McGill University; 2007. [cited 2020 Jan 21]. Available from: http://digitool.library.mcgill.ca/thesisfile112352.pdf.

Council of Science Editors:

Ring GN. Identification and characterization of TMEM 85, a novel suppressor of bax-mediated cell death in yeast. [Masters Thesis]. McGill University; 2007. Available from: http://digitool.library.mcgill.ca/thesisfile112352.pdf

5. Ghosh, Arindam P. Regulation of neuronal cell death by BH3-only molecules.

Degree: PhD, 2012, University of Alabama – Birmingham

Apoptosis in metazoan organisms plays critical roles in normal development, tissue homeostasis and immunity, and its disturbed regulation leads to many pathological states, including cancer,… (more)

Subjects/Keywords: Apoptosis  – physiology<; br>; Apoptosis Regulatory Proteins  – metabolism<; br>; Ethanol  – toxicity<; br>; Nervous System  – embryology<; br>; Neurons  – physiology<; br>; Neuropeptides  – metabolism<; br>; Tumor Suppressor Protein p53  – metabolism<; br>; Tumor Suppressor Proteins  – metabolism<; br>; bcl-2-Associated X Protein  – metabolism<; br>; bcl-X Protein  – metabolism

associated X protein BCA Bicinchoninic acid assay BCL-w BCL2 like 2 BCL-X BCL2 related gene… …AFFECT BCL-X DEFICIENCY INDUCED NEURON APOPTOSIS 1. Genotypes of viable mice generated from… …SYSTEM BUT DOES NOT AFFECT BCL-X DEFICIENCY INDUCED NEURON APOPTOSIS 1. HRK loss significantly… …system but does not affect BCL-X deficiency-induced neuron apoptosis” examines the effect of… …apoptosis seen in the developing brain and spinal cord of BCL-X-deficient 14 mice. The third… 

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APA (6th Edition):

Ghosh, A. P. (2012). Regulation of neuronal cell death by BH3-only molecules. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1138

Chicago Manual of Style (16th Edition):

Ghosh, Arindam P. “Regulation of neuronal cell death by BH3-only molecules.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1138.

MLA Handbook (7th Edition):

Ghosh, Arindam P. “Regulation of neuronal cell death by BH3-only molecules.” 2012. Web. 21 Jan 2020.

Vancouver:

Ghosh AP. Regulation of neuronal cell death by BH3-only molecules. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1138.

Council of Science Editors:

Ghosh AP. Regulation of neuronal cell death by BH3-only molecules. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1138

6. Cescato, Valter Angelo Sperling. Expressão dos genes relacionados à apoptose, Bcl-2, bax, e caspase-3 nos adenomas hipofisários clinicamente não funcionantes e seu potencial como marcador do comportamento tumoral.

Degree: PhD, Neurologia, 2010, University of São Paulo

 Adenomas hipofisários são tumores benignos, de crescimento lento, originados no interior da sela túrcica e constituem de 10% a 15% dos tumores intracranianos, Os adenomas… (more)

Subjects/Keywords: Adenoma hipofisário; Antígeno Ki-67; Apoptose; Apoptosis; bcl-2-associated X protein; Caspase 3; Caspase 3; Genes bcl-2; Genes bcl-2; Hipófise; Ki-67 antigen; Pituitary; Pituitary tumors; Proteína X associada a bcl-2

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APA (6th Edition):

Cescato, V. A. S. (2010). Expressão dos genes relacionados à apoptose, Bcl-2, bax, e caspase-3 nos adenomas hipofisários clinicamente não funcionantes e seu potencial como marcador do comportamento tumoral. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5138/tde-10052010-151708/ ;

Chicago Manual of Style (16th Edition):

Cescato, Valter Angelo Sperling. “Expressão dos genes relacionados à apoptose, Bcl-2, bax, e caspase-3 nos adenomas hipofisários clinicamente não funcionantes e seu potencial como marcador do comportamento tumoral.” 2010. Doctoral Dissertation, University of São Paulo. Accessed January 21, 2020. http://www.teses.usp.br/teses/disponiveis/5/5138/tde-10052010-151708/ ;.

MLA Handbook (7th Edition):

Cescato, Valter Angelo Sperling. “Expressão dos genes relacionados à apoptose, Bcl-2, bax, e caspase-3 nos adenomas hipofisários clinicamente não funcionantes e seu potencial como marcador do comportamento tumoral.” 2010. Web. 21 Jan 2020.

Vancouver:

Cescato VAS. Expressão dos genes relacionados à apoptose, Bcl-2, bax, e caspase-3 nos adenomas hipofisários clinicamente não funcionantes e seu potencial como marcador do comportamento tumoral. [Internet] [Doctoral dissertation]. University of São Paulo; 2010. [cited 2020 Jan 21]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5138/tde-10052010-151708/ ;.

Council of Science Editors:

Cescato VAS. Expressão dos genes relacionados à apoptose, Bcl-2, bax, e caspase-3 nos adenomas hipofisários clinicamente não funcionantes e seu potencial como marcador do comportamento tumoral. [Doctoral Dissertation]. University of São Paulo; 2010. Available from: http://www.teses.usp.br/teses/disponiveis/5/5138/tde-10052010-151708/ ;


Wright State University

7. Alqahtani, Tariq M. Interaction between Na/ K-ATPase and BCL-2 Proteins BCLXL and BAK.

Degree: MS, Pharmacology and Toxicology, 2014, Wright State University

 In silico analysis predicts interaction between the Na/ K-ATPase (NKA) and Bcl-2 protein canonical motifs BH3 and BH1. Such interaction is consistent with NKA inhibition… (more)

Subjects/Keywords: Pharmacology; Physiology; Biophysics; Apoptosis; Na,K-ATPase; Bcl-2; BclXL; Bak

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APA (6th Edition):

Alqahtani, T. M. (2014). Interaction between Na/ K-ATPase and BCL-2 Proteins BCLXL and BAK. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1421072560

Chicago Manual of Style (16th Edition):

Alqahtani, Tariq M. “Interaction between Na/ K-ATPase and BCL-2 Proteins BCLXL and BAK.” 2014. Masters Thesis, Wright State University. Accessed January 21, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=wright1421072560.

MLA Handbook (7th Edition):

Alqahtani, Tariq M. “Interaction between Na/ K-ATPase and BCL-2 Proteins BCLXL and BAK.” 2014. Web. 21 Jan 2020.

Vancouver:

Alqahtani TM. Interaction between Na/ K-ATPase and BCL-2 Proteins BCLXL and BAK. [Internet] [Masters thesis]. Wright State University; 2014. [cited 2020 Jan 21]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1421072560.

Council of Science Editors:

Alqahtani TM. Interaction between Na/ K-ATPase and BCL-2 Proteins BCLXL and BAK. [Masters Thesis]. Wright State University; 2014. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1421072560


Case Western Reserve University

8. Gomez, Jose A. Development of Cell Penetrating Bax Inhibiting Peptides (BIP).

Degree: PhD, Pharmacology, 2010, Case Western Reserve University

 Bax is a pro-apoptotic protein that mediates intrinsic cell-death signaling. Using a yeast-based functional screening approach, interferon gamma receptor beta chain (IFN¿R2) and the DNA… (more)

Subjects/Keywords: Biomedical Research; Cellular Biology; Pharmacology; Interferon gamma receptor beta chain (IFN&947; R2); Bcl-2 associated protein X (Bax), Apoptosis; Cytosolic IFN&947; R2; antiapoptotic protein; Bcl-2 family of proteins; Interferon gamma (IFN&947; ); Cell Penetrating Pepta-Peptides (CPP5)

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APA (6th Edition):

Gomez, J. A. (2010). Development of Cell Penetrating Bax Inhibiting Peptides (BIP). (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1252025677

Chicago Manual of Style (16th Edition):

Gomez, Jose A. “Development of Cell Penetrating Bax Inhibiting Peptides (BIP).” 2010. Doctoral Dissertation, Case Western Reserve University. Accessed January 21, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=case1252025677.

MLA Handbook (7th Edition):

Gomez, Jose A. “Development of Cell Penetrating Bax Inhibiting Peptides (BIP).” 2010. Web. 21 Jan 2020.

Vancouver:

Gomez JA. Development of Cell Penetrating Bax Inhibiting Peptides (BIP). [Internet] [Doctoral dissertation]. Case Western Reserve University; 2010. [cited 2020 Jan 21]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1252025677.

Council of Science Editors:

Gomez JA. Development of Cell Penetrating Bax Inhibiting Peptides (BIP). [Doctoral Dissertation]. Case Western Reserve University; 2010. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1252025677

9. Van Duyn, Lauren B. Calrecticulin and its role in collagen regulation.

Degree: PhD, 2009, University of Alabama – Birmingham

Calreticulin (CRT), classically known for its chaperone functions in the endoplasmic reticulum and as a Ca2+ regulator, also is found on the cell surface and… (more)

Subjects/Keywords: Calreticulin  – physiology<; br>; Collagen<; br>; Endoplasmic Reticulum  – metabolism<; br>; LDL-Receptor Related Protein-Associated<; br>; Protein  – metabolism<; br>; Thrombospondin 1

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APA (6th Edition):

Van Duyn, L. B. (2009). Calrecticulin and its role in collagen regulation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1118

Chicago Manual of Style (16th Edition):

Van Duyn, Lauren B. “Calrecticulin and its role in collagen regulation.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1118.

MLA Handbook (7th Edition):

Van Duyn, Lauren B. “Calrecticulin and its role in collagen regulation.” 2009. Web. 21 Jan 2020.

Vancouver:

Van Duyn LB. Calrecticulin and its role in collagen regulation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1118.

Council of Science Editors:

Van Duyn LB. Calrecticulin and its role in collagen regulation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1118


University of Manchester

10. King, Louise. Understanding how the Bcl-2 interactome contributes to apoptotic sensitivity.

Degree: 2019, University of Manchester

 Apoptotic sensitivity between individual cells within a population is varied and the molecular basis of this variation is unclear. Bcl-2 family proteins are the central… (more)

Subjects/Keywords: Apoptosis; Bcl-2

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APA (6th Edition):

King, L. (2019). Understanding how the Bcl-2 interactome contributes to apoptotic sensitivity. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:322785

Chicago Manual of Style (16th Edition):

King, Louise. “Understanding how the Bcl-2 interactome contributes to apoptotic sensitivity.” 2019. Doctoral Dissertation, University of Manchester. Accessed January 21, 2020. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:322785.

MLA Handbook (7th Edition):

King, Louise. “Understanding how the Bcl-2 interactome contributes to apoptotic sensitivity.” 2019. Web. 21 Jan 2020.

Vancouver:

King L. Understanding how the Bcl-2 interactome contributes to apoptotic sensitivity. [Internet] [Doctoral dissertation]. University of Manchester; 2019. [cited 2020 Jan 21]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:322785.

Council of Science Editors:

King L. Understanding how the Bcl-2 interactome contributes to apoptotic sensitivity. [Doctoral Dissertation]. University of Manchester; 2019. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:322785


University of Western Ontario

11. Guadagno, Jennifer. Mechanisms of neural precursor cell apoptosis by microglia-derived cytokines.

Degree: 2015, University of Western Ontario

 The persistence of neural precursor cells (NPCs) in distinct niches of the adult brain and spinal cord provides an important opportunity for regeneration in the… (more)

Subjects/Keywords: Neural precursor cells; microglia; neuroinflammation; apoptosis; pro-inflammatory cytokines; Bcl-2 family; Puma; Cellular and Molecular Physiology; Molecular and Cellular Neuroscience

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APA (6th Edition):

Guadagno, J. (2015). Mechanisms of neural precursor cell apoptosis by microglia-derived cytokines. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/2678

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Guadagno, Jennifer. “Mechanisms of neural precursor cell apoptosis by microglia-derived cytokines.” 2015. Thesis, University of Western Ontario. Accessed January 21, 2020. https://ir.lib.uwo.ca/etd/2678.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Guadagno, Jennifer. “Mechanisms of neural precursor cell apoptosis by microglia-derived cytokines.” 2015. Web. 21 Jan 2020.

Vancouver:

Guadagno J. Mechanisms of neural precursor cell apoptosis by microglia-derived cytokines. [Internet] [Thesis]. University of Western Ontario; 2015. [cited 2020 Jan 21]. Available from: https://ir.lib.uwo.ca/etd/2678.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Guadagno J. Mechanisms of neural precursor cell apoptosis by microglia-derived cytokines. [Thesis]. University of Western Ontario; 2015. Available from: https://ir.lib.uwo.ca/etd/2678

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

12. Walls, Ken C. Molecular characterization of neural apoptosis.

Degree: PhD, 2009, University of Alabama – Birmingham

Neural cell death plays a critical role in normal nervous system development and dysregulated neural stem cell death contributes to brain malformation, tumorgenesis, and possibly,… (more)

Subjects/Keywords: Apoptosis<; br>; Autophagy  – genetics<; br>; Cell Death<; br>; Lysosomes <; br>; physiology Neurons  – pathology<; br>; Tumor Suppressor Protein p53

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APA (6th Edition):

Walls, K. C. (2009). Molecular characterization of neural apoptosis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,504

Chicago Manual of Style (16th Edition):

Walls, Ken C. “Molecular characterization of neural apoptosis.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,504.

MLA Handbook (7th Edition):

Walls, Ken C. “Molecular characterization of neural apoptosis.” 2009. Web. 21 Jan 2020.

Vancouver:

Walls KC. Molecular characterization of neural apoptosis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,504.

Council of Science Editors:

Walls KC. Molecular characterization of neural apoptosis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,504


Seton Hall University

13. St.Angelo, Erin T. Localization and Binding Characteristics of Kaposi's Sarcoma-Associated Herpesvirus Bcl-2 Protein in the Prevention of Apoptosis.

Degree: MS Biology, Biology, 2010, Seton Hall University

  Kaposi's sarcoma-associated herpesvirus (KSHV) is the causative agent of various diseases, and it encodes a Bcl-2 homolog, KS-Bcl-2. The Bcl-2 family is important in… (more)

Subjects/Keywords: Kaposi's sarcoma; Herpesvirus; Bcl-2 protein; Apoptosis; Biology; Diseases; Life Sciences

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APA (6th Edition):

St.Angelo, E. T. (2010). Localization and Binding Characteristics of Kaposi's Sarcoma-Associated Herpesvirus Bcl-2 Protein in the Prevention of Apoptosis. (Doctoral Dissertation). Seton Hall University. Retrieved from http://scholarship.shu.edu/dissertations/2456

Chicago Manual of Style (16th Edition):

St.Angelo, Erin T. “Localization and Binding Characteristics of Kaposi's Sarcoma-Associated Herpesvirus Bcl-2 Protein in the Prevention of Apoptosis.” 2010. Doctoral Dissertation, Seton Hall University. Accessed January 21, 2020. http://scholarship.shu.edu/dissertations/2456.

MLA Handbook (7th Edition):

St.Angelo, Erin T. “Localization and Binding Characteristics of Kaposi's Sarcoma-Associated Herpesvirus Bcl-2 Protein in the Prevention of Apoptosis.” 2010. Web. 21 Jan 2020.

Vancouver:

St.Angelo ET. Localization and Binding Characteristics of Kaposi's Sarcoma-Associated Herpesvirus Bcl-2 Protein in the Prevention of Apoptosis. [Internet] [Doctoral dissertation]. Seton Hall University; 2010. [cited 2020 Jan 21]. Available from: http://scholarship.shu.edu/dissertations/2456.

Council of Science Editors:

St.Angelo ET. Localization and Binding Characteristics of Kaposi's Sarcoma-Associated Herpesvirus Bcl-2 Protein in the Prevention of Apoptosis. [Doctoral Dissertation]. Seton Hall University; 2010. Available from: http://scholarship.shu.edu/dissertations/2456


University of Melbourne

14. GRABOW, STEPHANIE. Which pro-survival Bcl-2 family members are required for lymphoma development and sustained lymphoma growth.

Degree: 2011, University of Melbourne

Apoptosis is a form of programmed cell death that is critical for embryonic development, tissue homeostasis and regulation of the immune system of multi-cellular organisms.… (more)

Subjects/Keywords: Bcl-2 family; apoptosis; lymphoma

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APA (6th Edition):

GRABOW, S. (2011). Which pro-survival Bcl-2 family members are required for lymphoma development and sustained lymphoma growth. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/37023

Chicago Manual of Style (16th Edition):

GRABOW, STEPHANIE. “Which pro-survival Bcl-2 family members are required for lymphoma development and sustained lymphoma growth.” 2011. Doctoral Dissertation, University of Melbourne. Accessed January 21, 2020. http://hdl.handle.net/11343/37023.

MLA Handbook (7th Edition):

GRABOW, STEPHANIE. “Which pro-survival Bcl-2 family members are required for lymphoma development and sustained lymphoma growth.” 2011. Web. 21 Jan 2020.

Vancouver:

GRABOW S. Which pro-survival Bcl-2 family members are required for lymphoma development and sustained lymphoma growth. [Internet] [Doctoral dissertation]. University of Melbourne; 2011. [cited 2020 Jan 21]. Available from: http://hdl.handle.net/11343/37023.

Council of Science Editors:

GRABOW S. Which pro-survival Bcl-2 family members are required for lymphoma development and sustained lymphoma growth. [Doctoral Dissertation]. University of Melbourne; 2011. Available from: http://hdl.handle.net/11343/37023


Harvard University

15. Pritz, Jonathan R. Allosteric Sensitization of Pro-Apoptotic BAX.

Degree: PhD, 2018, Harvard University

BCL-2 family proteins are critical regulators of mitochondrial apoptosis and thus serve as prime targets for therapeutic modulation in diseases of deregulated cell death. Whereas… (more)

Subjects/Keywords: apoptosis; BCL-2 family

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APA (6th Edition):

Pritz, J. R. (2018). Allosteric Sensitization of Pro-Apoptotic BAX. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:42015988

Chicago Manual of Style (16th Edition):

Pritz, Jonathan R. “Allosteric Sensitization of Pro-Apoptotic BAX.” 2018. Doctoral Dissertation, Harvard University. Accessed January 21, 2020. http://nrs.harvard.edu/urn-3:HUL.InstRepos:42015988.

MLA Handbook (7th Edition):

Pritz, Jonathan R. “Allosteric Sensitization of Pro-Apoptotic BAX.” 2018. Web. 21 Jan 2020.

Vancouver:

Pritz JR. Allosteric Sensitization of Pro-Apoptotic BAX. [Internet] [Doctoral dissertation]. Harvard University; 2018. [cited 2020 Jan 21]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:42015988.

Council of Science Editors:

Pritz JR. Allosteric Sensitization of Pro-Apoptotic BAX. [Doctoral Dissertation]. Harvard University; 2018. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:42015988

16. Jiang, Shaoning. Molecular mechanisms of hepatic insulin resistance following injury.

Degree: PhD, 2010, University of Alabama – Birmingham

Insulin resistance commonly occurs following injuries or critical illness independent of previous diabetic status. The development of insulin resistance and hyperglycemia is associated with increased… (more)

Subjects/Keywords: Adenoviridae – metabolism<; br>; Adenoviridae Infections – metabolism<; br>; Diabetes Mellitus, Type 2 – physiopathology<; br>; Glucose – metabolism<; br>; I-kappa B Kinase – physiology <; br>; Insulin – metabolism<; br>; JNK Mitogen-Activated Protein Kinases – physiology <; br>; Liver

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APA (6th Edition):

Jiang, S. (2010). Molecular mechanisms of hepatic insulin resistance following injury. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1374

Chicago Manual of Style (16th Edition):

Jiang, Shaoning. “Molecular mechanisms of hepatic insulin resistance following injury.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1374.

MLA Handbook (7th Edition):

Jiang, Shaoning. “Molecular mechanisms of hepatic insulin resistance following injury.” 2010. Web. 21 Jan 2020.

Vancouver:

Jiang S. Molecular mechanisms of hepatic insulin resistance following injury. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1374.

Council of Science Editors:

Jiang S. Molecular mechanisms of hepatic insulin resistance following injury. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1374

17. Rech de Laval, Valentine. Analyse bioinformatique des protéines BCL-2 et développement de la base de connaissance dédiée, BCL2DB : Bioinformatic analysis of BCL-2 proteins and development of the dedicated knowledge database, BCL2DB.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2013, Université Claude Bernard – Lyon I

Les protéines BCL-2 jouent un rôle essentiel dans la décision de vie ou de mort des cellules. Elles contrôlent l'induction de l'apoptose (mort cellulaire programmée)… (more)

Subjects/Keywords: Famille BCL-2; Motifs Protéiques; Base de données; Apoptose; Évolution moléculaire; BCL-2 family; Protein motifs; Database; Apoptosis; Molecular evolution; 570.15

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APA (6th Edition):

Rech de Laval, V. (2013). Analyse bioinformatique des protéines BCL-2 et développement de la base de connaissance dédiée, BCL2DB : Bioinformatic analysis of BCL-2 proteins and development of the dedicated knowledge database, BCL2DB. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2013LYO10273

Chicago Manual of Style (16th Edition):

Rech de Laval, Valentine. “Analyse bioinformatique des protéines BCL-2 et développement de la base de connaissance dédiée, BCL2DB : Bioinformatic analysis of BCL-2 proteins and development of the dedicated knowledge database, BCL2DB.” 2013. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed January 21, 2020. http://www.theses.fr/2013LYO10273.

MLA Handbook (7th Edition):

Rech de Laval, Valentine. “Analyse bioinformatique des protéines BCL-2 et développement de la base de connaissance dédiée, BCL2DB : Bioinformatic analysis of BCL-2 proteins and development of the dedicated knowledge database, BCL2DB.” 2013. Web. 21 Jan 2020.

Vancouver:

Rech de Laval V. Analyse bioinformatique des protéines BCL-2 et développement de la base de connaissance dédiée, BCL2DB : Bioinformatic analysis of BCL-2 proteins and development of the dedicated knowledge database, BCL2DB. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2013. [cited 2020 Jan 21]. Available from: http://www.theses.fr/2013LYO10273.

Council of Science Editors:

Rech de Laval V. Analyse bioinformatique des protéines BCL-2 et développement de la base de connaissance dédiée, BCL2DB : Bioinformatic analysis of BCL-2 proteins and development of the dedicated knowledge database, BCL2DB. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2013. Available from: http://www.theses.fr/2013LYO10273

18. Faustino, Viviane Dias. Inibição simultânea dos genes antiapoptóticos Bcl-2 e Bcl-XL em células de leucemia  linfoide aguda e células de linfoma do manto mediante RNA de interferência.

Degree: Mestrado, Distúrbios do Crescimento Celular, Hemodinâmicos e da Hemostasia, 2012, University of São Paulo

As estatísticas relacionadas aos cânceres hematológicos indicam que a incidência e mortalidade dessas doenças têm aumentado ao longo dos anos. Embora a maioria dos casos… (more)

Subjects/Keywords: Apoptose; Apoptosis; Bcl-2 gene; Gene therapy; Genes Bcl-2; Inibição simultânea; Interferência de RNA; Neoplasias; Neoplasms; Protein Bcl-XL; Proteína Bcl-XL; RNA interference; Simultaneous inhibition; Terapia de genes

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APA (6th Edition):

Faustino, V. D. (2012). Inibição simultânea dos genes antiapoptóticos Bcl-2 e Bcl-XL em células de leucemia  linfoide aguda e células de linfoma do manto mediante RNA de interferência. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5167/tde-22012013-154327/ ;

Chicago Manual of Style (16th Edition):

Faustino, Viviane Dias. “Inibição simultânea dos genes antiapoptóticos Bcl-2 e Bcl-XL em células de leucemia  linfoide aguda e células de linfoma do manto mediante RNA de interferência.” 2012. Masters Thesis, University of São Paulo. Accessed January 21, 2020. http://www.teses.usp.br/teses/disponiveis/5/5167/tde-22012013-154327/ ;.

MLA Handbook (7th Edition):

Faustino, Viviane Dias. “Inibição simultânea dos genes antiapoptóticos Bcl-2 e Bcl-XL em células de leucemia  linfoide aguda e células de linfoma do manto mediante RNA de interferência.” 2012. Web. 21 Jan 2020.

Vancouver:

Faustino VD. Inibição simultânea dos genes antiapoptóticos Bcl-2 e Bcl-XL em células de leucemia  linfoide aguda e células de linfoma do manto mediante RNA de interferência. [Internet] [Masters thesis]. University of São Paulo; 2012. [cited 2020 Jan 21]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5167/tde-22012013-154327/ ;.

Council of Science Editors:

Faustino VD. Inibição simultânea dos genes antiapoptóticos Bcl-2 e Bcl-XL em células de leucemia  linfoide aguda e células de linfoma do manto mediante RNA de interferência. [Masters Thesis]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/5/5167/tde-22012013-154327/ ;

19. Cheetham, Chad Christopher. Pathophysiology Of Dyt1 Dystonia: Targeted Mouse Models.

Degree: PhD, 2011, University of Alabama – Birmingham

DYT1 dystonia is an inherited movement disorder caused by a trinucleotide deletion (DeltaGAG) in the DYT1 (TOR1A) gene, which codes for the torsinA protein. Dr.… (more)

Subjects/Keywords: Adrenergic Neurons – physiology.<; br>; Adrenergic Uptake Inhibitors – pharmacology.<; br>; Antidepressive Agents – pharmacology<; br>; Antidepressive Agents, Tricyclic – pharmacology.<; br>; Arrestins – physiology.<; br>; Desipramine – pharmacology.<; br>; Microfilament Proteins – physiology.<; br>; Mitogen-Activated Protein Kinase 1 – biosynthesis.<; br>; Mitogen-Activated Protein Kinase 3 – biosynthesis.<; br>; Nerve Tissue Proteins – physiology<; br>; Receptors, Adrenergic, alpha-2 – physiology.

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APA (6th Edition):

Cheetham, C. C. (2011). Pathophysiology Of Dyt1 Dystonia: Targeted Mouse Models. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1433

Chicago Manual of Style (16th Edition):

Cheetham, Chad Christopher. “Pathophysiology Of Dyt1 Dystonia: Targeted Mouse Models.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1433.

MLA Handbook (7th Edition):

Cheetham, Chad Christopher. “Pathophysiology Of Dyt1 Dystonia: Targeted Mouse Models.” 2011. Web. 21 Jan 2020.

Vancouver:

Cheetham CC. Pathophysiology Of Dyt1 Dystonia: Targeted Mouse Models. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1433.

Council of Science Editors:

Cheetham CC. Pathophysiology Of Dyt1 Dystonia: Targeted Mouse Models. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1433


University of Melbourne

20. Cowan, Angus. Structural investigations of pro‑apoptotic Bcl‑2 family proteins.

Degree: 2017, University of Melbourne

 The Bcl‑2 protein family regulates the intrinsic apoptotic pathway through an intricate network of protein:protein and protein:membrane interactions. The pathway culminates in the permeabilisation of… (more)

Subjects/Keywords: apoptosis; Bak; Bax; Bok; X-ray crystallography; MOMP; Bcl-2 family; cell death

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APA (6th Edition):

Cowan, A. (2017). Structural investigations of pro‑apoptotic Bcl‑2 family proteins. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/192663

Chicago Manual of Style (16th Edition):

Cowan, Angus. “Structural investigations of pro‑apoptotic Bcl‑2 family proteins.” 2017. Doctoral Dissertation, University of Melbourne. Accessed January 21, 2020. http://hdl.handle.net/11343/192663.

MLA Handbook (7th Edition):

Cowan, Angus. “Structural investigations of pro‑apoptotic Bcl‑2 family proteins.” 2017. Web. 21 Jan 2020.

Vancouver:

Cowan A. Structural investigations of pro‑apoptotic Bcl‑2 family proteins. [Internet] [Doctoral dissertation]. University of Melbourne; 2017. [cited 2020 Jan 21]. Available from: http://hdl.handle.net/11343/192663.

Council of Science Editors:

Cowan A. Structural investigations of pro‑apoptotic Bcl‑2 family proteins. [Doctoral Dissertation]. University of Melbourne; 2017. Available from: http://hdl.handle.net/11343/192663


University of Melbourne

21. Roy, Michael James. Towards novel BH3-mimetics: structure-guided development of small molecule inhibitors targeting prosurvival BCL-2 family proteins.

Degree: 2016, University of Melbourne

 Defects in apoptosis (programmed cell death) due to overexpression of prosurvival members of the BCL‑2 family (eg. BCL‑2, MCL‑1 and BCL‑XL) are a genetic feature… (more)

Subjects/Keywords: apoptosis; BCL-2 family; BH3 mimetic; medicinal chemistry; X-ray crystallography; structure-guided drug design

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APA (6th Edition):

Roy, M. J. (2016). Towards novel BH3-mimetics: structure-guided development of small molecule inhibitors targeting prosurvival BCL-2 family proteins. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/129237

Chicago Manual of Style (16th Edition):

Roy, Michael James. “Towards novel BH3-mimetics: structure-guided development of small molecule inhibitors targeting prosurvival BCL-2 family proteins.” 2016. Doctoral Dissertation, University of Melbourne. Accessed January 21, 2020. http://hdl.handle.net/11343/129237.

MLA Handbook (7th Edition):

Roy, Michael James. “Towards novel BH3-mimetics: structure-guided development of small molecule inhibitors targeting prosurvival BCL-2 family proteins.” 2016. Web. 21 Jan 2020.

Vancouver:

Roy MJ. Towards novel BH3-mimetics: structure-guided development of small molecule inhibitors targeting prosurvival BCL-2 family proteins. [Internet] [Doctoral dissertation]. University of Melbourne; 2016. [cited 2020 Jan 21]. Available from: http://hdl.handle.net/11343/129237.

Council of Science Editors:

Roy MJ. Towards novel BH3-mimetics: structure-guided development of small molecule inhibitors targeting prosurvival BCL-2 family proteins. [Doctoral Dissertation]. University of Melbourne; 2016. Available from: http://hdl.handle.net/11343/129237

22. Thal, Melissa Ann. Characterization of the induction and regulation of early B cell development.

Degree: PhD, 2009, University of Alabama – Birmingham

Hematopoiesis is a highly regulated process directed by the microenvironment or niche in the bone marrow and the transcription factors those signals activate. Gene knockout… (more)

Subjects/Keywords: B-Lymphocytes  – cytology<; br>; Down-Regulation<; br>; Inhibitor of Differentiation Protein 2  – genetics<; br>; Inhibitor of Differentiation Proteins  – genetics<; br>; Receptors, Interleukin-7  – deficiency<; br>; TCF Transcription Factors  – physiology<; br>; Trans-Activators  – physiology

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APA (6th Edition):

Thal, M. A. (2009). Characterization of the induction and regulation of early B cell development. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,578

Chicago Manual of Style (16th Edition):

Thal, Melissa Ann. “Characterization of the induction and regulation of early B cell development.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,578.

MLA Handbook (7th Edition):

Thal, Melissa Ann. “Characterization of the induction and regulation of early B cell development.” 2009. Web. 21 Jan 2020.

Vancouver:

Thal MA. Characterization of the induction and regulation of early B cell development. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,578.

Council of Science Editors:

Thal MA. Characterization of the induction and regulation of early B cell development. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,578

23. Maitra, Sushmit. The AU-rich element mRNA decay-promoting activity of BRF1 is regulated by mitogen-activated protein kinase activated protein kinase.

Degree: PhD, 2008, University of Alabama – Birmingham

Regulated mRNA decay is a highly important process for the tight control of gene expression. Inherently unstable mRNAs contain AU-rich elements (AREs) in the 3’… (more)

Subjects/Keywords: Intracellular Signaling Peptides and Proteins  – metabolism <; br>; Protein-Serine-Threonine Kinases  – metabolism <; br>; RNA, Messenger  – metabolism <; br>; RNA Stability  – physiology <; br>; RNA-Binding Proteins  – metabolism <; br>; TATA-Binding Protein Associated Factors  – metabolism

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APA (6th Edition):

Maitra, S. (2008). The AU-rich element mRNA decay-promoting activity of BRF1 is regulated by mitogen-activated protein kinase activated protein kinase. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,379

Chicago Manual of Style (16th Edition):

Maitra, Sushmit. “The AU-rich element mRNA decay-promoting activity of BRF1 is regulated by mitogen-activated protein kinase activated protein kinase.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,379.

MLA Handbook (7th Edition):

Maitra, Sushmit. “The AU-rich element mRNA decay-promoting activity of BRF1 is regulated by mitogen-activated protein kinase activated protein kinase.” 2008. Web. 21 Jan 2020.

Vancouver:

Maitra S. The AU-rich element mRNA decay-promoting activity of BRF1 is regulated by mitogen-activated protein kinase activated protein kinase. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,379.

Council of Science Editors:

Maitra S. The AU-rich element mRNA decay-promoting activity of BRF1 is regulated by mitogen-activated protein kinase activated protein kinase. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,379

24. Harms, Kelly Lynn. Mechanisms of P53-mediated apoptosis.

Degree: PhD, 2007, University of Alabama – Birmingham

The p53 tumor suppressor is the most commonly inactivated gene in human cancers. The ability of p53, a transcription factor, to regulate genes that mediate… (more)

Subjects/Keywords: Apoptosis  – genetics<; br>; DNA, Neoplasm  – metabolism<; br>; Gene Expression Regulation  – physiology<; br>; Histone Deacetylases  – metabolism<; br>; Insulin-Like Growth Factor Binding Protein 3  – genetics<; br>; Repressor Proteins  – metabolism<; br>; Transcriptional Activation  – physiology<; br>; Tumor Suppressor Protein p53

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APA (6th Edition):

Harms, K. L. (2007). Mechanisms of P53-mediated apoptosis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,446

Chicago Manual of Style (16th Edition):

Harms, Kelly Lynn. “Mechanisms of P53-mediated apoptosis.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,446.

MLA Handbook (7th Edition):

Harms, Kelly Lynn. “Mechanisms of P53-mediated apoptosis.” 2007. Web. 21 Jan 2020.

Vancouver:

Harms KL. Mechanisms of P53-mediated apoptosis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,446.

Council of Science Editors:

Harms KL. Mechanisms of P53-mediated apoptosis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,446

25. Kosek, David J. (David Jeffrey). Aging differences in mechanisms of human skeletal muscle hypertrophy.

Degree: PhD, 2007, University of Alabama – Birmingham

Sarcopenia, the age-associated loss of muscle mass, affects all individuals to varying degrees as years advance, and leads to decreases in strength, power and agility… (more)

Subjects/Keywords: Aging  – physiology<; br>; Dystrophin-Associated Proteins  – physiology<; br>; Exercise  – physiology<; br>; Muscle Development  – physiology<; br>; Muscle, Skeletal  – cytology<; br>; Physical Fitness  – physiology<; br>; Satellite Cells, Skeletal Muscle  – physiology

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APA (6th Edition):

Kosek, D. J. (. J. (2007). Aging differences in mechanisms of human skeletal muscle hypertrophy. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,603

Chicago Manual of Style (16th Edition):

Kosek, David J (David Jeffrey). “Aging differences in mechanisms of human skeletal muscle hypertrophy.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,603.

MLA Handbook (7th Edition):

Kosek, David J (David Jeffrey). “Aging differences in mechanisms of human skeletal muscle hypertrophy.” 2007. Web. 21 Jan 2020.

Vancouver:

Kosek DJ(J. Aging differences in mechanisms of human skeletal muscle hypertrophy. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,603.

Council of Science Editors:

Kosek DJ(J. Aging differences in mechanisms of human skeletal muscle hypertrophy. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,603


University of Manchester

26. Rodriguez-Enriquez, Ricardo. Analysis of Bcl-2 family protein interactions in live cells by fluorescence recovery after photobleaching.

Degree: 2014, University of Manchester

 The Bcl-2 family of proteins strictly regulates the intrinsic pathway of apoptosis.Direct physical interactions between Bcl-2 proteins regulate mitochondrial outerpermeabilisation (MOMP), which occurs in response… (more)

Subjects/Keywords: Bcl-2 family protein; FRAP

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APA (6th Edition):

Rodriguez-Enriquez, R. (2014). Analysis of Bcl-2 family protein interactions in live cells by fluorescence recovery after photobleaching. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:228609

Chicago Manual of Style (16th Edition):

Rodriguez-Enriquez, Ricardo. “Analysis of Bcl-2 family protein interactions in live cells by fluorescence recovery after photobleaching.” 2014. Doctoral Dissertation, University of Manchester. Accessed January 21, 2020. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:228609.

MLA Handbook (7th Edition):

Rodriguez-Enriquez, Ricardo. “Analysis of Bcl-2 family protein interactions in live cells by fluorescence recovery after photobleaching.” 2014. Web. 21 Jan 2020.

Vancouver:

Rodriguez-Enriquez R. Analysis of Bcl-2 family protein interactions in live cells by fluorescence recovery after photobleaching. [Internet] [Doctoral dissertation]. University of Manchester; 2014. [cited 2020 Jan 21]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:228609.

Council of Science Editors:

Rodriguez-Enriquez R. Analysis of Bcl-2 family protein interactions in live cells by fluorescence recovery after photobleaching. [Doctoral Dissertation]. University of Manchester; 2014. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:228609

27. Merwin, Liz A. A C-terminus Mitochondrial-localization Region and BH3 Domain of Puma are Required for Apoptotic Function.

Degree: 2013, University of Western Ontario

Apoptosis is known to contribute to the loss of neurological function in brain injury and several neurodegenerative diseases. The Bcl-2 family of proteins consist of… (more)

Subjects/Keywords: Apoptosis; Bax; Bcl-2; Bcl-xl; BH3-only; Puma; Cellular and Molecular Physiology

…regulating target Bax – Bcl-2 associated X-protein Bcl-2 – B-cell lymphoma type 2 Bcl-xl – B-cell… …it to be such an effective and essential mediator of apoptosis. 1.5 – Bcl-2-associated x… …protein (Bax) Bcl-2-associated x protein (Bax) is a multi-domain pro… …members of the Bcl-2 protein family are key 14 regulators of neuronal apoptosis in a number of… …x29;. 1.3 – Bcl-2 family members in neuronal apoptosis The wide variety of apoptotic… 

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APA (6th Edition):

Merwin, L. A. (2013). A C-terminus Mitochondrial-localization Region and BH3 Domain of Puma are Required for Apoptotic Function. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/1283

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Merwin, Liz A. “A C-terminus Mitochondrial-localization Region and BH3 Domain of Puma are Required for Apoptotic Function.” 2013. Thesis, University of Western Ontario. Accessed January 21, 2020. https://ir.lib.uwo.ca/etd/1283.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Merwin, Liz A. “A C-terminus Mitochondrial-localization Region and BH3 Domain of Puma are Required for Apoptotic Function.” 2013. Web. 21 Jan 2020.

Vancouver:

Merwin LA. A C-terminus Mitochondrial-localization Region and BH3 Domain of Puma are Required for Apoptotic Function. [Internet] [Thesis]. University of Western Ontario; 2013. [cited 2020 Jan 21]. Available from: https://ir.lib.uwo.ca/etd/1283.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Merwin LA. A C-terminus Mitochondrial-localization Region and BH3 Domain of Puma are Required for Apoptotic Function. [Thesis]. University of Western Ontario; 2013. Available from: https://ir.lib.uwo.ca/etd/1283

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. Souza, Walkiria de Araújo. Análise da expressão de genes da família Bcl-2 em macrófagos infectados por Mycobacterium tuberculosis uni e multi-drogas resistentes.

Degree: Mestrado, Análises Clínicas, 2012, University of São Paulo

A Tuberculose é uma das doenças infecciosas mais antiga e bem descrita. Entretanto, ainda permanece como um dos principais problemas de saúde pública a ser… (more)

Subjects/Keywords: Apoptose; Apoptosis; Bcl-2; Bcl-2; Mycobacterium tuberculosis; Mycobacterium tuberculosis

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APA (6th Edition):

Souza, W. d. A. (2012). Análise da expressão de genes da família Bcl-2 em macrófagos infectados por Mycobacterium tuberculosis uni e multi-drogas resistentes. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/9/9136/tde-10092012-141753/ ;

Chicago Manual of Style (16th Edition):

Souza, Walkiria de Araújo. “Análise da expressão de genes da família Bcl-2 em macrófagos infectados por Mycobacterium tuberculosis uni e multi-drogas resistentes.” 2012. Masters Thesis, University of São Paulo. Accessed January 21, 2020. http://www.teses.usp.br/teses/disponiveis/9/9136/tde-10092012-141753/ ;.

MLA Handbook (7th Edition):

Souza, Walkiria de Araújo. “Análise da expressão de genes da família Bcl-2 em macrófagos infectados por Mycobacterium tuberculosis uni e multi-drogas resistentes.” 2012. Web. 21 Jan 2020.

Vancouver:

Souza WdA. Análise da expressão de genes da família Bcl-2 em macrófagos infectados por Mycobacterium tuberculosis uni e multi-drogas resistentes. [Internet] [Masters thesis]. University of São Paulo; 2012. [cited 2020 Jan 21]. Available from: http://www.teses.usp.br/teses/disponiveis/9/9136/tde-10092012-141753/ ;.

Council of Science Editors:

Souza WdA. Análise da expressão de genes da família Bcl-2 em macrófagos infectados por Mycobacterium tuberculosis uni e multi-drogas resistentes. [Masters Thesis]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/9/9136/tde-10092012-141753/ ;

29. McCoy, Portia Anne. Intracellular signaling mechanisms underlying muscarinic dependent plasticity in visual cortex and the impact of cholinergic degeneration.

Degree: PhD, 2008, University of Alabama – Birmingham

Cholinergic innervation to visual cortex is essential for spatial learning and memory as well as normal visual function. Long-term depression (LTD) or potentiation (LTP) dependent… (more)

Subjects/Keywords: Adrenergic Fibers  – physiology<; br>; Cholinergic Fibers  – physiology<; br>; Extracellular Signal-Regulated MAP Kinases  – physiology<; br>; Long-Term Synaptic Depression<; br>; Neuronal Plasticity  – physiology<; br>; Protein Kinase C  – physiology<; br>; Receptors, Muscarinic  – physiology<; br>; Sympathetic Fibers, Postganglionic  – physiology<; br>; Visual Cortex  – physiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

McCoy, P. A. (2008). Intracellular signaling mechanisms underlying muscarinic dependent plasticity in visual cortex and the impact of cholinergic degeneration. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,750

Chicago Manual of Style (16th Edition):

McCoy, Portia Anne. “Intracellular signaling mechanisms underlying muscarinic dependent plasticity in visual cortex and the impact of cholinergic degeneration.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,750.

MLA Handbook (7th Edition):

McCoy, Portia Anne. “Intracellular signaling mechanisms underlying muscarinic dependent plasticity in visual cortex and the impact of cholinergic degeneration.” 2008. Web. 21 Jan 2020.

Vancouver:

McCoy PA. Intracellular signaling mechanisms underlying muscarinic dependent plasticity in visual cortex and the impact of cholinergic degeneration. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,750.

Council of Science Editors:

McCoy PA. Intracellular signaling mechanisms underlying muscarinic dependent plasticity in visual cortex and the impact of cholinergic degeneration. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,750


Marshall University

30. Gadde, Murali K. Effects of aging on regulators of muscle apoptosis in the female F344BN rat.

Degree: 2009, Marshall University

 Age-related muscle atrophy is a consequence of normal aging characterized by decreases in muscle mass and strength. The mechanism(s) underlying the loss of muscle mass… (more)

Subjects/Keywords: aging; Apoptosis; F344BN; Female; Bax; Bcl-2; Muscle; <; p>; Apoptosis.<; /p>; <; p>; Cell death. <; /p>; <; p>; Aging.<; /p>;

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gadde, M. K. (2009). Effects of aging on regulators of muscle apoptosis in the female F344BN rat. (Thesis). Marshall University. Retrieved from http://mds.marshall.edu/etd/596

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gadde, Murali K. “Effects of aging on regulators of muscle apoptosis in the female F344BN rat.” 2009. Thesis, Marshall University. Accessed January 21, 2020. http://mds.marshall.edu/etd/596.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gadde, Murali K. “Effects of aging on regulators of muscle apoptosis in the female F344BN rat.” 2009. Web. 21 Jan 2020.

Vancouver:

Gadde MK. Effects of aging on regulators of muscle apoptosis in the female F344BN rat. [Internet] [Thesis]. Marshall University; 2009. [cited 2020 Jan 21]. Available from: http://mds.marshall.edu/etd/596.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gadde MK. Effects of aging on regulators of muscle apoptosis in the female F344BN rat. [Thesis]. Marshall University; 2009. Available from: http://mds.marshall.edu/etd/596

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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