Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:( Amyloid beta protein). Showing records 1 – 30 of 29022 total matches.

[1] [2] [3] [4] [5] … [968]

Search Limiters

Last 2 Years | English Only

Languages

Country

▼ Search Limiters


Drexel University

1. Nguyen, Phuong Thi Tam. Phytosterol Cyclopamine Rescues Structural and Behavioral Deficits in a Drosophila model of Alzheimer's Disease.

Degree: 2017, Drexel University

The average person spends several hours a day behind the wheel of their vehicles, which are usually equipped with on-board computers capable of collecting real-time… (more)

Subjects/Keywords: Biology; Alzheimer's disease; Drosophila; Amyloid beta-protein; Amyloid beta-protein precursor

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Nguyen, P. T. T. (2017). Phytosterol Cyclopamine Rescues Structural and Behavioral Deficits in a Drosophila model of Alzheimer's Disease. (Thesis). Drexel University. Retrieved from http://hdl.handle.net/1860/idea:7473

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nguyen, Phuong Thi Tam. “Phytosterol Cyclopamine Rescues Structural and Behavioral Deficits in a Drosophila model of Alzheimer's Disease.” 2017. Thesis, Drexel University. Accessed May 08, 2021. http://hdl.handle.net/1860/idea:7473.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nguyen, Phuong Thi Tam. “Phytosterol Cyclopamine Rescues Structural and Behavioral Deficits in a Drosophila model of Alzheimer's Disease.” 2017. Web. 08 May 2021.

Vancouver:

Nguyen PTT. Phytosterol Cyclopamine Rescues Structural and Behavioral Deficits in a Drosophila model of Alzheimer's Disease. [Internet] [Thesis]. Drexel University; 2017. [cited 2021 May 08]. Available from: http://hdl.handle.net/1860/idea:7473.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nguyen PTT. Phytosterol Cyclopamine Rescues Structural and Behavioral Deficits in a Drosophila model of Alzheimer's Disease. [Thesis]. Drexel University; 2017. Available from: http://hdl.handle.net/1860/idea:7473

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of the Western Cape

2. Teponnou, Gerard A. Kenfack. Tacrine, trolox and tryptoline as lead compounds for the design and synthesis of multi-target drugs for Alzheimer's disease therapy .

Degree: 2016, University of the Western Cape

 The cascade of neurotoxic events involved in the pathogenesis of Alzheimer's disease may explain the inefficacy of currently available treatment based on acetylcholinesterase inhibitors (AChEI… (more)

Subjects/Keywords: Amyloid Beta; Cholinesterases; Alzheimer's disease; Multifunctional drugs; Amyloid beta-protein

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Teponnou, G. A. K. (2016). Tacrine, trolox and tryptoline as lead compounds for the design and synthesis of multi-target drugs for Alzheimer's disease therapy . (Thesis). University of the Western Cape. Retrieved from http://hdl.handle.net/11394/5344

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Teponnou, Gerard A Kenfack. “Tacrine, trolox and tryptoline as lead compounds for the design and synthesis of multi-target drugs for Alzheimer's disease therapy .” 2016. Thesis, University of the Western Cape. Accessed May 08, 2021. http://hdl.handle.net/11394/5344.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Teponnou, Gerard A Kenfack. “Tacrine, trolox and tryptoline as lead compounds for the design and synthesis of multi-target drugs for Alzheimer's disease therapy .” 2016. Web. 08 May 2021.

Vancouver:

Teponnou GAK. Tacrine, trolox and tryptoline as lead compounds for the design and synthesis of multi-target drugs for Alzheimer's disease therapy . [Internet] [Thesis]. University of the Western Cape; 2016. [cited 2021 May 08]. Available from: http://hdl.handle.net/11394/5344.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Teponnou GAK. Tacrine, trolox and tryptoline as lead compounds for the design and synthesis of multi-target drugs for Alzheimer's disease therapy . [Thesis]. University of the Western Cape; 2016. Available from: http://hdl.handle.net/11394/5344

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Hong Kong University of Science and Technology

3. Tang, Siqi LIFS. Characterization of the function of EphA4 signaling in Alzheimer's disease.

Degree: 2015, Hong Kong University of Science and Technology

 Alzheimer’s disease (AD) is one most common dementia. Although there is currently no effective treatment, the reversal of amyloid beta (Aβ)-induced synaptic deficits is believed… (more)

Subjects/Keywords: Alzheimer's disease ; Amyloid beta-protein ; Erythropoietin ; Receptors

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tang, S. L. (2015). Characterization of the function of EphA4 signaling in Alzheimer's disease. (Thesis). Hong Kong University of Science and Technology. Retrieved from http://repository.ust.hk/ir/Record/1783.1-92215 ; https://doi.org/10.14711/thesis-b1514949 ; http://repository.ust.hk/ir/bitstream/1783.1-92215/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tang, Siqi LIFS. “Characterization of the function of EphA4 signaling in Alzheimer's disease.” 2015. Thesis, Hong Kong University of Science and Technology. Accessed May 08, 2021. http://repository.ust.hk/ir/Record/1783.1-92215 ; https://doi.org/10.14711/thesis-b1514949 ; http://repository.ust.hk/ir/bitstream/1783.1-92215/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tang, Siqi LIFS. “Characterization of the function of EphA4 signaling in Alzheimer's disease.” 2015. Web. 08 May 2021.

Vancouver:

Tang SL. Characterization of the function of EphA4 signaling in Alzheimer's disease. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2015. [cited 2021 May 08]. Available from: http://repository.ust.hk/ir/Record/1783.1-92215 ; https://doi.org/10.14711/thesis-b1514949 ; http://repository.ust.hk/ir/bitstream/1783.1-92215/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tang SL. Characterization of the function of EphA4 signaling in Alzheimer's disease. [Thesis]. Hong Kong University of Science and Technology; 2015. Available from: http://repository.ust.hk/ir/Record/1783.1-92215 ; https://doi.org/10.14711/thesis-b1514949 ; http://repository.ust.hk/ir/bitstream/1783.1-92215/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Drexel University

4. Utreja, Suruchi. Calcineurin regulates APP metabolism: In vitro & In vivo studies.

Degree: 2013, Drexel University

Alzheimer's disease (AD) is a complex neurodegenerative disease determined by the combination of environmental and genetic factors. AD is pathologically characterized by the accumulation of… (more)

Subjects/Keywords: Life sciences; Alzheimer's disease; Amyloid beta-protein

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Utreja, S. (2013). Calcineurin regulates APP metabolism: In vitro & In vivo studies. (Thesis). Drexel University. Retrieved from http://hdl.handle.net/1860/idea:6997

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Utreja, Suruchi. “Calcineurin regulates APP metabolism: In vitro & In vivo studies.” 2013. Thesis, Drexel University. Accessed May 08, 2021. http://hdl.handle.net/1860/idea:6997.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Utreja, Suruchi. “Calcineurin regulates APP metabolism: In vitro & In vivo studies.” 2013. Web. 08 May 2021.

Vancouver:

Utreja S. Calcineurin regulates APP metabolism: In vitro & In vivo studies. [Internet] [Thesis]. Drexel University; 2013. [cited 2021 May 08]. Available from: http://hdl.handle.net/1860/idea:6997.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Utreja S. Calcineurin regulates APP metabolism: In vitro & In vivo studies. [Thesis]. Drexel University; 2013. Available from: http://hdl.handle.net/1860/idea:6997

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Drexel University

5. Vorobyeva, Anna Gennadiy. Sonic Hedgehog Signaling Pathway in Amyloid Precursor Protein Trafficking and Proteolysis.

Degree: 2014, Drexel University

Alzheimer's disease (AD) is the most common form of dementia for which there is no cure. AD is characterized by progressive neurotoxicity resulting in cell… (more)

Subjects/Keywords: Biological sciences; Alzheimer's disease; Amyloid beta-protein

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Vorobyeva, A. G. (2014). Sonic Hedgehog Signaling Pathway in Amyloid Precursor Protein Trafficking and Proteolysis. (Thesis). Drexel University. Retrieved from http://hdl.handle.net/1860/idea:6998

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vorobyeva, Anna Gennadiy. “Sonic Hedgehog Signaling Pathway in Amyloid Precursor Protein Trafficking and Proteolysis.” 2014. Thesis, Drexel University. Accessed May 08, 2021. http://hdl.handle.net/1860/idea:6998.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vorobyeva, Anna Gennadiy. “Sonic Hedgehog Signaling Pathway in Amyloid Precursor Protein Trafficking and Proteolysis.” 2014. Web. 08 May 2021.

Vancouver:

Vorobyeva AG. Sonic Hedgehog Signaling Pathway in Amyloid Precursor Protein Trafficking and Proteolysis. [Internet] [Thesis]. Drexel University; 2014. [cited 2021 May 08]. Available from: http://hdl.handle.net/1860/idea:6998.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vorobyeva AG. Sonic Hedgehog Signaling Pathway in Amyloid Precursor Protein Trafficking and Proteolysis. [Thesis]. Drexel University; 2014. Available from: http://hdl.handle.net/1860/idea:6998

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Drexel University

6. Smith, Micholas Dean. Influence of Aqueous-Salt Conditions on the Structure and Dynamics of the Monomeric and Novel Dimeric forms of the Alzheimer's A[beta]21-30 protein fragment.

Degree: 2014, Drexel University

The behavior of the Alzheimer's related peptide Aβ is the subject of much study. In typical computational studies the environment local to the peptide is… (more)

Subjects/Keywords: Physics; Alzheimer's disease; Amyloid beta-protein

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Smith, M. D. (2014). Influence of Aqueous-Salt Conditions on the Structure and Dynamics of the Monomeric and Novel Dimeric forms of the Alzheimer's A[beta]21-30 protein fragment. (Thesis). Drexel University. Retrieved from http://hdl.handle.net/1860/4569

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Smith, Micholas Dean. “Influence of Aqueous-Salt Conditions on the Structure and Dynamics of the Monomeric and Novel Dimeric forms of the Alzheimer's A[beta]21-30 protein fragment.” 2014. Thesis, Drexel University. Accessed May 08, 2021. http://hdl.handle.net/1860/4569.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Smith, Micholas Dean. “Influence of Aqueous-Salt Conditions on the Structure and Dynamics of the Monomeric and Novel Dimeric forms of the Alzheimer's A[beta]21-30 protein fragment.” 2014. Web. 08 May 2021.

Vancouver:

Smith MD. Influence of Aqueous-Salt Conditions on the Structure and Dynamics of the Monomeric and Novel Dimeric forms of the Alzheimer's A[beta]21-30 protein fragment. [Internet] [Thesis]. Drexel University; 2014. [cited 2021 May 08]. Available from: http://hdl.handle.net/1860/4569.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Smith MD. Influence of Aqueous-Salt Conditions on the Structure and Dynamics of the Monomeric and Novel Dimeric forms of the Alzheimer's A[beta]21-30 protein fragment. [Thesis]. Drexel University; 2014. Available from: http://hdl.handle.net/1860/4569

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Rhodes University

7. Carlisle, Tanya. Molecular simulations of potential agents and targets of Alzheimer’s disease.

Degree: Faculty of Science, Biochemistry and Microbiology, 2020, Rhodes University

 The World Alzheimer Report statedin 2016 that approximately 46.8 million people were living with dementia and this figure is expected to triple by 2050. Alzheimer’s… (more)

Subjects/Keywords: Alzheimer's disease  – Treatment; Alzheimer's disease  – Molecular aspects; Amyloid beta-protein precurs; Amyloid beta-protein

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Carlisle, T. (2020). Molecular simulations of potential agents and targets of Alzheimer’s disease. (Thesis). Rhodes University. Retrieved from http://hdl.handle.net/10962/140025

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Carlisle, Tanya. “Molecular simulations of potential agents and targets of Alzheimer’s disease.” 2020. Thesis, Rhodes University. Accessed May 08, 2021. http://hdl.handle.net/10962/140025.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Carlisle, Tanya. “Molecular simulations of potential agents and targets of Alzheimer’s disease.” 2020. Web. 08 May 2021.

Vancouver:

Carlisle T. Molecular simulations of potential agents and targets of Alzheimer’s disease. [Internet] [Thesis]. Rhodes University; 2020. [cited 2021 May 08]. Available from: http://hdl.handle.net/10962/140025.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Carlisle T. Molecular simulations of potential agents and targets of Alzheimer’s disease. [Thesis]. Rhodes University; 2020. Available from: http://hdl.handle.net/10962/140025

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

8. Oliveira, Joana Machado de. Phosphorylation mediated events in biomarker discovery for Alzheimer’s disease .

Degree: 2021, Universidade de Aveiro

 Alzheimer´s disease (AD) is one of the most common forms of dementia worldwide; representing 60-70% of all dementia cases. This neurodegenerative disorder is characterized by… (more)

Subjects/Keywords: Alzheimer´s disease; Amyloid-β peptide; Okadaic acid; Protein phosphorylation; Amyloid precursor protein; Tau

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Oliveira, J. M. d. (2021). Phosphorylation mediated events in biomarker discovery for Alzheimer’s disease . (Thesis). Universidade de Aveiro. Retrieved from http://hdl.handle.net/10773/30913

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Oliveira, Joana Machado de. “Phosphorylation mediated events in biomarker discovery for Alzheimer’s disease .” 2021. Thesis, Universidade de Aveiro. Accessed May 08, 2021. http://hdl.handle.net/10773/30913.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Oliveira, Joana Machado de. “Phosphorylation mediated events in biomarker discovery for Alzheimer’s disease .” 2021. Web. 08 May 2021.

Vancouver:

Oliveira JMd. Phosphorylation mediated events in biomarker discovery for Alzheimer’s disease . [Internet] [Thesis]. Universidade de Aveiro; 2021. [cited 2021 May 08]. Available from: http://hdl.handle.net/10773/30913.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Oliveira JMd. Phosphorylation mediated events in biomarker discovery for Alzheimer’s disease . [Thesis]. Universidade de Aveiro; 2021. Available from: http://hdl.handle.net/10773/30913

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

9. Andrew, Robert. Differential Proteolysis of the Amyloid Precursor Protein Isoforms: The Role of Cellular Location and Protein-Protein Interactions.

Degree: 2015, University of Manchester

 Dementia, the most common cause of which is Alzheimer’s disease (AD), currently affects 850,000 people in the UK, a figure set to rise to over… (more)

Subjects/Keywords: Alzheimers disease; Amyloid precursor protein; Amyloid-beta; interactome; Proteolysis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Andrew, R. (2015). Differential Proteolysis of the Amyloid Precursor Protein Isoforms: The Role of Cellular Location and Protein-Protein Interactions. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:280080

Chicago Manual of Style (16th Edition):

Andrew, Robert. “Differential Proteolysis of the Amyloid Precursor Protein Isoforms: The Role of Cellular Location and Protein-Protein Interactions.” 2015. Doctoral Dissertation, University of Manchester. Accessed May 08, 2021. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:280080.

MLA Handbook (7th Edition):

Andrew, Robert. “Differential Proteolysis of the Amyloid Precursor Protein Isoforms: The Role of Cellular Location and Protein-Protein Interactions.” 2015. Web. 08 May 2021.

Vancouver:

Andrew R. Differential Proteolysis of the Amyloid Precursor Protein Isoforms: The Role of Cellular Location and Protein-Protein Interactions. [Internet] [Doctoral dissertation]. University of Manchester; 2015. [cited 2021 May 08]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:280080.

Council of Science Editors:

Andrew R. Differential Proteolysis of the Amyloid Precursor Protein Isoforms: The Role of Cellular Location and Protein-Protein Interactions. [Doctoral Dissertation]. University of Manchester; 2015. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:280080

10. Kirouac, Lisa. The Concerted Regulation of Intracellular Signaling by Amyloid Precursor Protein and Aβ Peptide.

Degree: 2016, University of South Florida

 It is widely accepted that A-beta (Aβ) generated from amyloid precursor protein (APP) oligomerizes and fibrillizes to form neuritic plaques in the Alzheimer’s disease (AD)… (more)

Subjects/Keywords: Alzheimer’s disease; amyloid-beta; amyloid precursor protein; mitosis; proliferation; Neurosciences

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kirouac, L. (2016). The Concerted Regulation of Intracellular Signaling by Amyloid Precursor Protein and Aβ Peptide. (Thesis). University of South Florida. Retrieved from https://scholarcommons.usf.edu/etd/6278

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kirouac, Lisa. “The Concerted Regulation of Intracellular Signaling by Amyloid Precursor Protein and Aβ Peptide.” 2016. Thesis, University of South Florida. Accessed May 08, 2021. https://scholarcommons.usf.edu/etd/6278.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kirouac, Lisa. “The Concerted Regulation of Intracellular Signaling by Amyloid Precursor Protein and Aβ Peptide.” 2016. Web. 08 May 2021.

Vancouver:

Kirouac L. The Concerted Regulation of Intracellular Signaling by Amyloid Precursor Protein and Aβ Peptide. [Internet] [Thesis]. University of South Florida; 2016. [cited 2021 May 08]. Available from: https://scholarcommons.usf.edu/etd/6278.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kirouac L. The Concerted Regulation of Intracellular Signaling by Amyloid Precursor Protein and Aβ Peptide. [Thesis]. University of South Florida; 2016. Available from: https://scholarcommons.usf.edu/etd/6278

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Duke University

11. Christianson, Melissa Gottron. Amyloid Precursor Protein-Dependent and -Independent Mechanisms in Hypoxia-Induced Axonopathy .

Degree: 2012, Duke University

  Hypoxia is a profound stressor of the central nervous system implicated in numerous neurodegenerative diseases. While it is increasingly evident that the early effects… (more)

Subjects/Keywords: Neurosciences; Alzheimer's disease; Amyloid Beta; Amyloid Precursor Protein; Axon; Hypoxia; Neurodegeneration

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Christianson, M. G. (2012). Amyloid Precursor Protein-Dependent and -Independent Mechanisms in Hypoxia-Induced Axonopathy . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/5427

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Christianson, Melissa Gottron. “Amyloid Precursor Protein-Dependent and -Independent Mechanisms in Hypoxia-Induced Axonopathy .” 2012. Thesis, Duke University. Accessed May 08, 2021. http://hdl.handle.net/10161/5427.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Christianson, Melissa Gottron. “Amyloid Precursor Protein-Dependent and -Independent Mechanisms in Hypoxia-Induced Axonopathy .” 2012. Web. 08 May 2021.

Vancouver:

Christianson MG. Amyloid Precursor Protein-Dependent and -Independent Mechanisms in Hypoxia-Induced Axonopathy . [Internet] [Thesis]. Duke University; 2012. [cited 2021 May 08]. Available from: http://hdl.handle.net/10161/5427.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Christianson MG. Amyloid Precursor Protein-Dependent and -Independent Mechanisms in Hypoxia-Induced Axonopathy . [Thesis]. Duke University; 2012. Available from: http://hdl.handle.net/10161/5427

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

12. Andrew, Robert. Differential proteolysis of the amyloid precursor protein isoforms : the role of cellular location and protein-protein interactions.

Degree: PhD, 2015, University of Manchester

 Dementia, the most common cause of which is Alzheimer's disease (AD), currently affects 850,000 people in the UK, a figure set to rise to over… (more)

Subjects/Keywords: 616.8; Alzheimers disease; Amyloid precursor protein; Amyloid-beta; interactome; Proteolysis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Andrew, R. (2015). Differential proteolysis of the amyloid precursor protein isoforms : the role of cellular location and protein-protein interactions. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/differential-proteolysis-of-the-amyloid-precursor-protein-isoforms-the-role-of-cellular-location-and-proteinprotein-interactions(5390e8fa-fc5e-4357-8109-cb2bb1c49212).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764346

Chicago Manual of Style (16th Edition):

Andrew, Robert. “Differential proteolysis of the amyloid precursor protein isoforms : the role of cellular location and protein-protein interactions.” 2015. Doctoral Dissertation, University of Manchester. Accessed May 08, 2021. https://www.research.manchester.ac.uk/portal/en/theses/differential-proteolysis-of-the-amyloid-precursor-protein-isoforms-the-role-of-cellular-location-and-proteinprotein-interactions(5390e8fa-fc5e-4357-8109-cb2bb1c49212).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764346.

MLA Handbook (7th Edition):

Andrew, Robert. “Differential proteolysis of the amyloid precursor protein isoforms : the role of cellular location and protein-protein interactions.” 2015. Web. 08 May 2021.

Vancouver:

Andrew R. Differential proteolysis of the amyloid precursor protein isoforms : the role of cellular location and protein-protein interactions. [Internet] [Doctoral dissertation]. University of Manchester; 2015. [cited 2021 May 08]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/differential-proteolysis-of-the-amyloid-precursor-protein-isoforms-the-role-of-cellular-location-and-proteinprotein-interactions(5390e8fa-fc5e-4357-8109-cb2bb1c49212).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764346.

Council of Science Editors:

Andrew R. Differential proteolysis of the amyloid precursor protein isoforms : the role of cellular location and protein-protein interactions. [Doctoral Dissertation]. University of Manchester; 2015. Available from: https://www.research.manchester.ac.uk/portal/en/theses/differential-proteolysis-of-the-amyloid-precursor-protein-isoforms-the-role-of-cellular-location-and-proteinprotein-interactions(5390e8fa-fc5e-4357-8109-cb2bb1c49212).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764346


Texas State University – San Marcos

13. Campbell, James. Optimization of SDD-AGE as a Method to Study Amyloid Conversion of Human Recombinant Prion Protein.

Degree: MS, Biochemistry, 2014, Texas State University – San Marcos

 Many common diseases are caused by amyloid proteins. Amyloid structures are β sheet rich, protease resistant, and can form polydisperse insoluble fibers. Due to these… (more)

Subjects/Keywords: SDD-AGE, Prion, Amyloid; Prion; Amyloid; Amyloid beta-protein; Amyloidosis; Prions; Recombinant proteins

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Campbell, J. (2014). Optimization of SDD-AGE as a Method to Study Amyloid Conversion of Human Recombinant Prion Protein. (Masters Thesis). Texas State University – San Marcos. Retrieved from https://digital.library.txstate.edu/handle/10877/5272

Chicago Manual of Style (16th Edition):

Campbell, James. “Optimization of SDD-AGE as a Method to Study Amyloid Conversion of Human Recombinant Prion Protein.” 2014. Masters Thesis, Texas State University – San Marcos. Accessed May 08, 2021. https://digital.library.txstate.edu/handle/10877/5272.

MLA Handbook (7th Edition):

Campbell, James. “Optimization of SDD-AGE as a Method to Study Amyloid Conversion of Human Recombinant Prion Protein.” 2014. Web. 08 May 2021.

Vancouver:

Campbell J. Optimization of SDD-AGE as a Method to Study Amyloid Conversion of Human Recombinant Prion Protein. [Internet] [Masters thesis]. Texas State University – San Marcos; 2014. [cited 2021 May 08]. Available from: https://digital.library.txstate.edu/handle/10877/5272.

Council of Science Editors:

Campbell J. Optimization of SDD-AGE as a Method to Study Amyloid Conversion of Human Recombinant Prion Protein. [Masters Thesis]. Texas State University – San Marcos; 2014. Available from: https://digital.library.txstate.edu/handle/10877/5272


University of Ottawa

14. Yuka, Sai. The Effect of Amyloid-Beta on the Insulin Signalling Pathway in Neuroblastoma 2a (N2a) Cells: The Characterization of Insulin Resistance in Alzheimer’s Disease .

Degree: 2016, University of Ottawa

 7Alzheimer’s disease (AD) is a progressive neurodegenerative disorder characterized by accumulation and deposition of extracellular beta-amyloid peptides (Aβ) and intra-neuronal hyperphosphorylated tau in the brain.… (more)

Subjects/Keywords: Alzheimer's disease; neurodegeneration; amyloid beta binding protein; cell signalling; amyloid beta; PI3K/Akt; mTOR

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yuka, S. (2016). The Effect of Amyloid-Beta on the Insulin Signalling Pathway in Neuroblastoma 2a (N2a) Cells: The Characterization of Insulin Resistance in Alzheimer’s Disease . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/34248

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yuka, Sai. “The Effect of Amyloid-Beta on the Insulin Signalling Pathway in Neuroblastoma 2a (N2a) Cells: The Characterization of Insulin Resistance in Alzheimer’s Disease .” 2016. Thesis, University of Ottawa. Accessed May 08, 2021. http://hdl.handle.net/10393/34248.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yuka, Sai. “The Effect of Amyloid-Beta on the Insulin Signalling Pathway in Neuroblastoma 2a (N2a) Cells: The Characterization of Insulin Resistance in Alzheimer’s Disease .” 2016. Web. 08 May 2021.

Vancouver:

Yuka S. The Effect of Amyloid-Beta on the Insulin Signalling Pathway in Neuroblastoma 2a (N2a) Cells: The Characterization of Insulin Resistance in Alzheimer’s Disease . [Internet] [Thesis]. University of Ottawa; 2016. [cited 2021 May 08]. Available from: http://hdl.handle.net/10393/34248.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yuka S. The Effect of Amyloid-Beta on the Insulin Signalling Pathway in Neuroblastoma 2a (N2a) Cells: The Characterization of Insulin Resistance in Alzheimer’s Disease . [Thesis]. University of Ottawa; 2016. Available from: http://hdl.handle.net/10393/34248

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Bath

15. Roberts, Hazel. Alpha-synuclein expression influences the processing of the amyloid precursor protein.

Degree: PhD, 2016, University of Bath

 In certain neurodegenerative diseases such Dementia with Lewy Bodies (DLB), it is hypothesised that misfolded α-synuclein (α-syn) and β-amyloid both contribute to pathology. α-Syn and… (more)

Subjects/Keywords: 616.8; alpha-synuclein; amyloid precursor protein; amyloid-beta peptide; beta-secretase; alpha-secretase

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Roberts, H. (2016). Alpha-synuclein expression influences the processing of the amyloid precursor protein. (Doctoral Dissertation). University of Bath. Retrieved from https://researchportal.bath.ac.uk/en/studentthesis/alphasynuclein-expression-influences-the-processing-of-the-amyloid-precursor-protein(81175adb-7013-402d-82e6-f309a6890030).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.707587

Chicago Manual of Style (16th Edition):

Roberts, Hazel. “Alpha-synuclein expression influences the processing of the amyloid precursor protein.” 2016. Doctoral Dissertation, University of Bath. Accessed May 08, 2021. https://researchportal.bath.ac.uk/en/studentthesis/alphasynuclein-expression-influences-the-processing-of-the-amyloid-precursor-protein(81175adb-7013-402d-82e6-f309a6890030).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.707587.

MLA Handbook (7th Edition):

Roberts, Hazel. “Alpha-synuclein expression influences the processing of the amyloid precursor protein.” 2016. Web. 08 May 2021.

Vancouver:

Roberts H. Alpha-synuclein expression influences the processing of the amyloid precursor protein. [Internet] [Doctoral dissertation]. University of Bath; 2016. [cited 2021 May 08]. Available from: https://researchportal.bath.ac.uk/en/studentthesis/alphasynuclein-expression-influences-the-processing-of-the-amyloid-precursor-protein(81175adb-7013-402d-82e6-f309a6890030).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.707587.

Council of Science Editors:

Roberts H. Alpha-synuclein expression influences the processing of the amyloid precursor protein. [Doctoral Dissertation]. University of Bath; 2016. Available from: https://researchportal.bath.ac.uk/en/studentthesis/alphasynuclein-expression-influences-the-processing-of-the-amyloid-precursor-protein(81175adb-7013-402d-82e6-f309a6890030).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.707587


Macquarie University

16. Gangoda, Sumudu V.S. Cyclic stretch of brain microvascular endothelial cells and regulation of amyloid processing and expression: evidence for contribution of vascular pulsatility in Alzheimer's disease.

Degree: 2018, Macquarie University

Thesis by publication.

Bibliography: pages 222-238.

1. Introduction  – 2. Literature review  – 3. Optimisation of mechanical stretch of endothelial cells for protein and RNA… (more)

Subjects/Keywords: Amyloid beta-protein; Alzheimer's disease; cyclic stretch; pulsatility; Alzheimer's disease; amyloid beta

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gangoda, S. V. S. (2018). Cyclic stretch of brain microvascular endothelial cells and regulation of amyloid processing and expression: evidence for contribution of vascular pulsatility in Alzheimer's disease. (Doctoral Dissertation). Macquarie University. Retrieved from http://hdl.handle.net/1959.14/1268283

Chicago Manual of Style (16th Edition):

Gangoda, Sumudu V S. “Cyclic stretch of brain microvascular endothelial cells and regulation of amyloid processing and expression: evidence for contribution of vascular pulsatility in Alzheimer's disease.” 2018. Doctoral Dissertation, Macquarie University. Accessed May 08, 2021. http://hdl.handle.net/1959.14/1268283.

MLA Handbook (7th Edition):

Gangoda, Sumudu V S. “Cyclic stretch of brain microvascular endothelial cells and regulation of amyloid processing and expression: evidence for contribution of vascular pulsatility in Alzheimer's disease.” 2018. Web. 08 May 2021.

Vancouver:

Gangoda SVS. Cyclic stretch of brain microvascular endothelial cells and regulation of amyloid processing and expression: evidence for contribution of vascular pulsatility in Alzheimer's disease. [Internet] [Doctoral dissertation]. Macquarie University; 2018. [cited 2021 May 08]. Available from: http://hdl.handle.net/1959.14/1268283.

Council of Science Editors:

Gangoda SVS. Cyclic stretch of brain microvascular endothelial cells and regulation of amyloid processing and expression: evidence for contribution of vascular pulsatility in Alzheimer's disease. [Doctoral Dissertation]. Macquarie University; 2018. Available from: http://hdl.handle.net/1959.14/1268283


Universidade de Lisboa

17. Dionísio, Pedro Elói Antunes. Using TUDCA to treat Alzheimer's disease after pathology onset in APP/PS1 mice.

Degree: 2014, Universidade de Lisboa

Tese de mestrado, Ciências Biofarmacêuticas, Universidade de Lisboa, Faculdade de Farmácia, 2014

A doença de Alzheimer (AD) é a forma mais comum de demência, constituindo… (more)

Subjects/Keywords: Amyloid-β; Amyloid precursor protein; APP/PS1 mice; Glycogen synthase kinase β; Neuroinflammation; Tau; TUDCA; Teses de mestrado - 2014

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dionísio, P. E. A. (2014). Using TUDCA to treat Alzheimer's disease after pathology onset in APP/PS1 mice. (Thesis). Universidade de Lisboa. Retrieved from https://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/15924

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dionísio, Pedro Elói Antunes. “Using TUDCA to treat Alzheimer's disease after pathology onset in APP/PS1 mice.” 2014. Thesis, Universidade de Lisboa. Accessed May 08, 2021. https://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/15924.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dionísio, Pedro Elói Antunes. “Using TUDCA to treat Alzheimer's disease after pathology onset in APP/PS1 mice.” 2014. Web. 08 May 2021.

Vancouver:

Dionísio PEA. Using TUDCA to treat Alzheimer's disease after pathology onset in APP/PS1 mice. [Internet] [Thesis]. Universidade de Lisboa; 2014. [cited 2021 May 08]. Available from: https://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/15924.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dionísio PEA. Using TUDCA to treat Alzheimer's disease after pathology onset in APP/PS1 mice. [Thesis]. Universidade de Lisboa; 2014. Available from: https://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/15924

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Columbia University

18. Williamson, Rebecca Lynn. Investigating the role of ubiquitin in endosomal sorting and processing of amyloid precursor protein.

Degree: 2017, Columbia University

Amyloid plaques, a neuropathological hallmark of Alzheimer’s disease (AD), are largely composed of amyloid beta (Aβ) peptide, derived from cleavage of amyloid precursor protein (APP)… (more)

Subjects/Keywords: Amyloid beta-protein precursor; Ubiquitin; Endosomes; Nervous system – Diseases; Amyloid beta-protein; Neurosciences; Pathology; Molecular biology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Williamson, R. L. (2017). Investigating the role of ubiquitin in endosomal sorting and processing of amyloid precursor protein. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8MW2HQZ

Chicago Manual of Style (16th Edition):

Williamson, Rebecca Lynn. “Investigating the role of ubiquitin in endosomal sorting and processing of amyloid precursor protein.” 2017. Doctoral Dissertation, Columbia University. Accessed May 08, 2021. https://doi.org/10.7916/D8MW2HQZ.

MLA Handbook (7th Edition):

Williamson, Rebecca Lynn. “Investigating the role of ubiquitin in endosomal sorting and processing of amyloid precursor protein.” 2017. Web. 08 May 2021.

Vancouver:

Williamson RL. Investigating the role of ubiquitin in endosomal sorting and processing of amyloid precursor protein. [Internet] [Doctoral dissertation]. Columbia University; 2017. [cited 2021 May 08]. Available from: https://doi.org/10.7916/D8MW2HQZ.

Council of Science Editors:

Williamson RL. Investigating the role of ubiquitin in endosomal sorting and processing of amyloid precursor protein. [Doctoral Dissertation]. Columbia University; 2017. Available from: https://doi.org/10.7916/D8MW2HQZ


East Carolina University

19. Gade, Sunitha. Cu²� Binding to A[beta] peptides : Detailed Heat Capacity Studies Provide Structural Insight into Complex Formation.

Degree: MS, Chemistry, 2012, East Carolina University

 Alzheimer's disease (AD), an irreversible, progressive and devastating neurodegenerative disease, is the most common cause of dementia. It is characterized by the extracellular amyloid plaques… (more)

Subjects/Keywords: Chemistry; Chemistry, Inorganic; Inorganic chemistry; Alzheimer's disease; Amyloid beta-protein; Amyloid beta-protein precursor; Metal ions

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gade, S. (2012). Cu²� Binding to A[beta] peptides : Detailed Heat Capacity Studies Provide Structural Insight into Complex Formation. (Masters Thesis). East Carolina University. Retrieved from http://hdl.handle.net/10342/1814

Chicago Manual of Style (16th Edition):

Gade, Sunitha. “Cu²â�º Binding to A[beta] peptides : Detailed Heat Capacity Studies Provide Structural Insight into Complex Formation.” 2012. Masters Thesis, East Carolina University. Accessed May 08, 2021. http://hdl.handle.net/10342/1814.

MLA Handbook (7th Edition):

Gade, Sunitha. “Cu²â�º Binding to A[beta] peptides : Detailed Heat Capacity Studies Provide Structural Insight into Complex Formation.” 2012. Web. 08 May 2021.

Vancouver:

Gade S. Cu²� Binding to A[beta] peptides : Detailed Heat Capacity Studies Provide Structural Insight into Complex Formation. [Internet] [Masters thesis]. East Carolina University; 2012. [cited 2021 May 08]. Available from: http://hdl.handle.net/10342/1814.

Council of Science Editors:

Gade S. Cu²� Binding to A[beta] peptides : Detailed Heat Capacity Studies Provide Structural Insight into Complex Formation. [Masters Thesis]. East Carolina University; 2012. Available from: http://hdl.handle.net/10342/1814


University of California – San Diego

20. Fong, Lauren Kristen. Elucidating Amyloid Precursor Protein Function in Human Astrocytes Derived from Pluripotent Stem Cells.

Degree: Biomedical Sciences, 2016, University of California – San Diego

 Successful development of effective therapeutics for Alzheimer’s disease (AD) requires a deep understanding of the mechanisms of disease pathogenesis. Recent human induced pluripotent stem cell… (more)

Subjects/Keywords: Biology; Neurosciences; Amyloid-beta; Amyloid Precursor Protein; Astrocyte; Cholesterol; Lipoprotein; Stem Cell

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fong, L. K. (2016). Elucidating Amyloid Precursor Protein Function in Human Astrocytes Derived from Pluripotent Stem Cells. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/5740p2b0

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fong, Lauren Kristen. “Elucidating Amyloid Precursor Protein Function in Human Astrocytes Derived from Pluripotent Stem Cells.” 2016. Thesis, University of California – San Diego. Accessed May 08, 2021. http://www.escholarship.org/uc/item/5740p2b0.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fong, Lauren Kristen. “Elucidating Amyloid Precursor Protein Function in Human Astrocytes Derived from Pluripotent Stem Cells.” 2016. Web. 08 May 2021.

Vancouver:

Fong LK. Elucidating Amyloid Precursor Protein Function in Human Astrocytes Derived from Pluripotent Stem Cells. [Internet] [Thesis]. University of California – San Diego; 2016. [cited 2021 May 08]. Available from: http://www.escholarship.org/uc/item/5740p2b0.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fong LK. Elucidating Amyloid Precursor Protein Function in Human Astrocytes Derived from Pluripotent Stem Cells. [Thesis]. University of California – San Diego; 2016. Available from: http://www.escholarship.org/uc/item/5740p2b0

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

21. S. Therin. USE OF A CELL PERMEABLE PEPTIDE TO MODULATE ADAM10 SYNAPTIC LOCALIZATION AND ACTIVITY IN A MOUSE MODEL OF ALZHEIMER'S DISEASE.

Degree: 2019, Università degli Studi di Milano

 Alzheimer’s disease (AD) is characterized by the aggregation of amyloid beta peptide (Aβ). Aβ derives from the amyloid precursor protein (APP), which can undergo two… (more)

Subjects/Keywords: Alzheimer's disease; ADAM10; Amyloid precursor protein; Amyloid beta; APP/PS1 mouse; Settore BIO/14 - Farmacologia

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Therin, S. (2019). USE OF A CELL PERMEABLE PEPTIDE TO MODULATE ADAM10 SYNAPTIC LOCALIZATION AND ACTIVITY IN A MOUSE MODEL OF ALZHEIMER'S DISEASE. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/649095

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Therin, S.. “USE OF A CELL PERMEABLE PEPTIDE TO MODULATE ADAM10 SYNAPTIC LOCALIZATION AND ACTIVITY IN A MOUSE MODEL OF ALZHEIMER'S DISEASE.” 2019. Thesis, Università degli Studi di Milano. Accessed May 08, 2021. http://hdl.handle.net/2434/649095.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Therin, S.. “USE OF A CELL PERMEABLE PEPTIDE TO MODULATE ADAM10 SYNAPTIC LOCALIZATION AND ACTIVITY IN A MOUSE MODEL OF ALZHEIMER'S DISEASE.” 2019. Web. 08 May 2021.

Vancouver:

Therin S. USE OF A CELL PERMEABLE PEPTIDE TO MODULATE ADAM10 SYNAPTIC LOCALIZATION AND ACTIVITY IN A MOUSE MODEL OF ALZHEIMER'S DISEASE. [Internet] [Thesis]. Università degli Studi di Milano; 2019. [cited 2021 May 08]. Available from: http://hdl.handle.net/2434/649095.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Therin S. USE OF A CELL PERMEABLE PEPTIDE TO MODULATE ADAM10 SYNAPTIC LOCALIZATION AND ACTIVITY IN A MOUSE MODEL OF ALZHEIMER'S DISEASE. [Thesis]. Università degli Studi di Milano; 2019. Available from: http://hdl.handle.net/2434/649095

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Michigan Technological University

22. Adhikari, Rashmi. Characterizing the physicochemical properties of TDP-43 protein and Acetylated Amyloid β peptides to discern its role in neurodegenerative diseases.

Degree: PhD, Department of Chemistry, 2019, Michigan Technological University

  Proteins are the biological macromolecules central to most cellular functions. The native structure of protein determines its function and any perturbations such as mutation,… (more)

Subjects/Keywords: Protein purification; Protein aggregation; Cytotoxicity; Neurodegenerative diseases; TDP-43; Amyloid β acetylation

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Adhikari, R. (2019). Characterizing the physicochemical properties of TDP-43 protein and Acetylated Amyloid β peptides to discern its role in neurodegenerative diseases. (Doctoral Dissertation). Michigan Technological University. Retrieved from https://digitalcommons.mtu.edu/etdr/891

Chicago Manual of Style (16th Edition):

Adhikari, Rashmi. “Characterizing the physicochemical properties of TDP-43 protein and Acetylated Amyloid β peptides to discern its role in neurodegenerative diseases.” 2019. Doctoral Dissertation, Michigan Technological University. Accessed May 08, 2021. https://digitalcommons.mtu.edu/etdr/891.

MLA Handbook (7th Edition):

Adhikari, Rashmi. “Characterizing the physicochemical properties of TDP-43 protein and Acetylated Amyloid β peptides to discern its role in neurodegenerative diseases.” 2019. Web. 08 May 2021.

Vancouver:

Adhikari R. Characterizing the physicochemical properties of TDP-43 protein and Acetylated Amyloid β peptides to discern its role in neurodegenerative diseases. [Internet] [Doctoral dissertation]. Michigan Technological University; 2019. [cited 2021 May 08]. Available from: https://digitalcommons.mtu.edu/etdr/891.

Council of Science Editors:

Adhikari R. Characterizing the physicochemical properties of TDP-43 protein and Acetylated Amyloid β peptides to discern its role in neurodegenerative diseases. [Doctoral Dissertation]. Michigan Technological University; 2019. Available from: https://digitalcommons.mtu.edu/etdr/891


University of St. Andrews

23. Taylor, Margaret Alexandra. Cellular and proteomic studies of the mitochondrial ABAD/Aβ complex : investigating its role in Alzheimer's disease.

Degree: 2012, University of St. Andrews

 The focus of this thesis is to investigate the intracellular protein-peptide complex 3-hydroxyacyl-CoA dehydrogenase (HADH), also known as ABAD (amyloid- binding alcohol dehydrogenase) and amyloid-beta(more)

Subjects/Keywords: QP552.A45T2; Amyloid beta protein – Analysis; Alzheimer Disease – Pathophysiology; Protein-protein interactions; Cell-mediated cytotoxicity

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Taylor, M. A. (2012). Cellular and proteomic studies of the mitochondrial ABAD/Aβ complex : investigating its role in Alzheimer's disease. (Doctoral Dissertation). University of St. Andrews. Retrieved from http://hdl.handle.net/10023/3632

Chicago Manual of Style (16th Edition):

Taylor, Margaret Alexandra. “Cellular and proteomic studies of the mitochondrial ABAD/Aβ complex : investigating its role in Alzheimer's disease.” 2012. Doctoral Dissertation, University of St. Andrews. Accessed May 08, 2021. http://hdl.handle.net/10023/3632.

MLA Handbook (7th Edition):

Taylor, Margaret Alexandra. “Cellular and proteomic studies of the mitochondrial ABAD/Aβ complex : investigating its role in Alzheimer's disease.” 2012. Web. 08 May 2021.

Vancouver:

Taylor MA. Cellular and proteomic studies of the mitochondrial ABAD/Aβ complex : investigating its role in Alzheimer's disease. [Internet] [Doctoral dissertation]. University of St. Andrews; 2012. [cited 2021 May 08]. Available from: http://hdl.handle.net/10023/3632.

Council of Science Editors:

Taylor MA. Cellular and proteomic studies of the mitochondrial ABAD/Aβ complex : investigating its role in Alzheimer's disease. [Doctoral Dissertation]. University of St. Andrews; 2012. Available from: http://hdl.handle.net/10023/3632


University of Lund

24. Szczepankiewicz, Olga. To Fold or To Fibrillate? Serendipity in Stability Studies.

Degree: 2011, University of Lund

 Proteins are complex structures and years of research have been spent on attempts to understand their complexity. The non-covalent interactions involved in protein folding are:… (more)

Subjects/Keywords: Physical Chemistry; Protein stability; protein reconstitution; protein aggregation; Monellin; PGB1; Split-GFP; Amyloid beta

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Szczepankiewicz, O. (2011). To Fold or To Fibrillate? Serendipity in Stability Studies. (Doctoral Dissertation). University of Lund. Retrieved from https://lup.lub.lu.se/record/2169882 ; https://portal.research.lu.se/ws/files/6319272/2169889.pdf

Chicago Manual of Style (16th Edition):

Szczepankiewicz, Olga. “To Fold or To Fibrillate? Serendipity in Stability Studies.” 2011. Doctoral Dissertation, University of Lund. Accessed May 08, 2021. https://lup.lub.lu.se/record/2169882 ; https://portal.research.lu.se/ws/files/6319272/2169889.pdf.

MLA Handbook (7th Edition):

Szczepankiewicz, Olga. “To Fold or To Fibrillate? Serendipity in Stability Studies.” 2011. Web. 08 May 2021.

Vancouver:

Szczepankiewicz O. To Fold or To Fibrillate? Serendipity in Stability Studies. [Internet] [Doctoral dissertation]. University of Lund; 2011. [cited 2021 May 08]. Available from: https://lup.lub.lu.se/record/2169882 ; https://portal.research.lu.se/ws/files/6319272/2169889.pdf.

Council of Science Editors:

Szczepankiewicz O. To Fold or To Fibrillate? Serendipity in Stability Studies. [Doctoral Dissertation]. University of Lund; 2011. Available from: https://lup.lub.lu.se/record/2169882 ; https://portal.research.lu.se/ws/files/6319272/2169889.pdf


University of Alberta

25. Mohamed, Amany. Amyloid beta peptide, Cholesterol and Isoprenoids in Alzheimer’s disease.

Degree: PhD, Department of Pharmacology, 2013, University of Alberta

 Alzheimer’s disease (AD) is the most common form of dementia in the elderly. The major pathological features of AD are extracellular amyloid plaques, intracellular neurofibrillary… (more)

Subjects/Keywords: Amyloid beta peptide; SREBP-2; Cholesterol; Protein prenylation

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mohamed, A. (2013). Amyloid beta peptide, Cholesterol and Isoprenoids in Alzheimer’s disease. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/f7623d25p

Chicago Manual of Style (16th Edition):

Mohamed, Amany. “Amyloid beta peptide, Cholesterol and Isoprenoids in Alzheimer’s disease.” 2013. Doctoral Dissertation, University of Alberta. Accessed May 08, 2021. https://era.library.ualberta.ca/files/f7623d25p.

MLA Handbook (7th Edition):

Mohamed, Amany. “Amyloid beta peptide, Cholesterol and Isoprenoids in Alzheimer’s disease.” 2013. Web. 08 May 2021.

Vancouver:

Mohamed A. Amyloid beta peptide, Cholesterol and Isoprenoids in Alzheimer’s disease. [Internet] [Doctoral dissertation]. University of Alberta; 2013. [cited 2021 May 08]. Available from: https://era.library.ualberta.ca/files/f7623d25p.

Council of Science Editors:

Mohamed A. Amyloid beta peptide, Cholesterol and Isoprenoids in Alzheimer’s disease. [Doctoral Dissertation]. University of Alberta; 2013. Available from: https://era.library.ualberta.ca/files/f7623d25p


Stony Brook University

26. Chiang, Hsueh-Chiang. Drosophila Model of Alzheimer's Disease .

Degree: 2009, Stony Brook University

Subjects/Keywords: Beta-amyloid precursor protein

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chiang, H. (2009). Drosophila Model of Alzheimer's Disease . (Thesis). Stony Brook University. Retrieved from http://hdl.handle.net/1951/52212

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chiang, Hsueh-Chiang. “Drosophila Model of Alzheimer's Disease .” 2009. Thesis, Stony Brook University. Accessed May 08, 2021. http://hdl.handle.net/1951/52212.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chiang, Hsueh-Chiang. “Drosophila Model of Alzheimer's Disease .” 2009. Web. 08 May 2021.

Vancouver:

Chiang H. Drosophila Model of Alzheimer's Disease . [Internet] [Thesis]. Stony Brook University; 2009. [cited 2021 May 08]. Available from: http://hdl.handle.net/1951/52212.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chiang H. Drosophila Model of Alzheimer's Disease . [Thesis]. Stony Brook University; 2009. Available from: http://hdl.handle.net/1951/52212

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Tasmania

27. Dawkins, E. Role of Phosphoinositides in the biology of the amyloid precursor protein.

Degree: 2014, University of Tasmania

 Alzheimer’s disease (AD) is the leading cause of dementia in the elderly. In countries with aging populations, such as Australia, the prevalence of AD is… (more)

Subjects/Keywords: Alzheimer's disease; Phosphoinositides; beta-amyloid precursor protein; Phosphatidylinositol; phosphate; heparin.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dawkins, E. (2014). Role of Phosphoinositides in the biology of the amyloid precursor protein. (Thesis). University of Tasmania. Retrieved from https://eprints.utas.edu.au/18274/1/front-Dawkins-thesis.pdf ; https://eprints.utas.edu.au/18274/2/Whole-Dawkins-thesis.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dawkins, E. “Role of Phosphoinositides in the biology of the amyloid precursor protein.” 2014. Thesis, University of Tasmania. Accessed May 08, 2021. https://eprints.utas.edu.au/18274/1/front-Dawkins-thesis.pdf ; https://eprints.utas.edu.au/18274/2/Whole-Dawkins-thesis.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dawkins, E. “Role of Phosphoinositides in the biology of the amyloid precursor protein.” 2014. Web. 08 May 2021.

Vancouver:

Dawkins E. Role of Phosphoinositides in the biology of the amyloid precursor protein. [Internet] [Thesis]. University of Tasmania; 2014. [cited 2021 May 08]. Available from: https://eprints.utas.edu.au/18274/1/front-Dawkins-thesis.pdf ; https://eprints.utas.edu.au/18274/2/Whole-Dawkins-thesis.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dawkins E. Role of Phosphoinositides in the biology of the amyloid precursor protein. [Thesis]. University of Tasmania; 2014. Available from: https://eprints.utas.edu.au/18274/1/front-Dawkins-thesis.pdf ; https://eprints.utas.edu.au/18274/2/Whole-Dawkins-thesis.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. Tran, Denise Phuong. Biological and structure characterisation of eukaryotic prefoldin.

Degree: 2018, University of Adelaide

 Prefoldin is a hexameric protein complex ubiquitously expressed and found to influence the conformation of amyloidogenic peptides. Relatively high degrees of sequence identity and conservation… (more)

Subjects/Keywords: eukaryotic prefoldin; amyloid beta (1-40); biophysical characterisation; protein structure

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tran, D. P. (2018). Biological and structure characterisation of eukaryotic prefoldin. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/115168

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tran, Denise Phuong. “Biological and structure characterisation of eukaryotic prefoldin.” 2018. Thesis, University of Adelaide. Accessed May 08, 2021. http://hdl.handle.net/2440/115168.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tran, Denise Phuong. “Biological and structure characterisation of eukaryotic prefoldin.” 2018. Web. 08 May 2021.

Vancouver:

Tran DP. Biological and structure characterisation of eukaryotic prefoldin. [Internet] [Thesis]. University of Adelaide; 2018. [cited 2021 May 08]. Available from: http://hdl.handle.net/2440/115168.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tran DP. Biological and structure characterisation of eukaryotic prefoldin. [Thesis]. University of Adelaide; 2018. Available from: http://hdl.handle.net/2440/115168

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

29. Warren, Rebekkah Lynn. Genetic Reduction of Cholesterol Synthesis in the Mouse Brain Does Not Affect Amyloid Formation in an Alzheimer’s Disease Model, but Does Extend Lifespan.

Degree: 2011, University of Texas Southwestern Medical Center

 In vitro alterations in cellular cholesterol content or synthesis affect the cleavage of amyloid precursor protein (APP) to amyloidogenic peptides characteristic of Alzheimer’s disease (AD).… (more)

Subjects/Keywords: Amyloid beta-Protein Precursor; Alzheimer Disease; Lipid Metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Warren, R. L. (2011). Genetic Reduction of Cholesterol Synthesis in the Mouse Brain Does Not Affect Amyloid Formation in an Alzheimer’s Disease Model, but Does Extend Lifespan. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/891

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Warren, Rebekkah Lynn. “Genetic Reduction of Cholesterol Synthesis in the Mouse Brain Does Not Affect Amyloid Formation in an Alzheimer’s Disease Model, but Does Extend Lifespan.” 2011. Thesis, University of Texas Southwestern Medical Center. Accessed May 08, 2021. http://hdl.handle.net/2152.5/891.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Warren, Rebekkah Lynn. “Genetic Reduction of Cholesterol Synthesis in the Mouse Brain Does Not Affect Amyloid Formation in an Alzheimer’s Disease Model, but Does Extend Lifespan.” 2011. Web. 08 May 2021.

Vancouver:

Warren RL. Genetic Reduction of Cholesterol Synthesis in the Mouse Brain Does Not Affect Amyloid Formation in an Alzheimer’s Disease Model, but Does Extend Lifespan. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2011. [cited 2021 May 08]. Available from: http://hdl.handle.net/2152.5/891.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Warren RL. Genetic Reduction of Cholesterol Synthesis in the Mouse Brain Does Not Affect Amyloid Formation in an Alzheimer’s Disease Model, but Does Extend Lifespan. [Thesis]. University of Texas Southwestern Medical Center; 2011. Available from: http://hdl.handle.net/2152.5/891

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Columbia University

30. Qu, Xiaoyi. Microtubule Dynamics in Tau-dependent Amyloid Beta Synaptotoxicity.

Degree: 2019, Columbia University

 Alzheimer’s disease is the most common form of dementia among older adults, and directly contributes to the third leading cause of death in the United… (more)

Subjects/Keywords: Cytology; Neurosciences; Amyloid beta-protein; Microtubules; Alzheimer's disease; Alzheimer's disease – Pathophysiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Qu, X. (2019). Microtubule Dynamics in Tau-dependent Amyloid Beta Synaptotoxicity. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/d8-4qaj-2923

Chicago Manual of Style (16th Edition):

Qu, Xiaoyi. “Microtubule Dynamics in Tau-dependent Amyloid Beta Synaptotoxicity.” 2019. Doctoral Dissertation, Columbia University. Accessed May 08, 2021. https://doi.org/10.7916/d8-4qaj-2923.

MLA Handbook (7th Edition):

Qu, Xiaoyi. “Microtubule Dynamics in Tau-dependent Amyloid Beta Synaptotoxicity.” 2019. Web. 08 May 2021.

Vancouver:

Qu X. Microtubule Dynamics in Tau-dependent Amyloid Beta Synaptotoxicity. [Internet] [Doctoral dissertation]. Columbia University; 2019. [cited 2021 May 08]. Available from: https://doi.org/10.7916/d8-4qaj-2923.

Council of Science Editors:

Qu X. Microtubule Dynamics in Tau-dependent Amyloid Beta Synaptotoxicity. [Doctoral Dissertation]. Columbia University; 2019. Available from: https://doi.org/10.7916/d8-4qaj-2923

[1] [2] [3] [4] [5] … [968]

.