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You searched for subject:( Tip60). Showing records 1 – 26 of 26 total matches.

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University of Southern California

1. Lee, Janet M. The mechanism of recruitment of Tip60 to ER target genes.

Degree: MS, Biochemistry & Molecular Biology, 2011, University of Southern California

 The steroid receptors in the nuclear receptor superfamily are responsible for regulating many different functions including transcription. However, in order to completely and correctly activate… (more)

Subjects/Keywords: Tip60; coactivator; nuclear receptor

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APA (6th Edition):

Lee, J. M. (2011). The mechanism of recruitment of Tip60 to ER target genes. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/441052/rec/6975

Chicago Manual of Style (16th Edition):

Lee, Janet M. “The mechanism of recruitment of Tip60 to ER target genes.” 2011. Masters Thesis, University of Southern California. Accessed February 17, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/441052/rec/6975.

MLA Handbook (7th Edition):

Lee, Janet M. “The mechanism of recruitment of Tip60 to ER target genes.” 2011. Web. 17 Feb 2020.

Vancouver:

Lee JM. The mechanism of recruitment of Tip60 to ER target genes. [Internet] [Masters thesis]. University of Southern California; 2011. [cited 2020 Feb 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/441052/rec/6975.

Council of Science Editors:

Lee JM. The mechanism of recruitment of Tip60 to ER target genes. [Masters Thesis]. University of Southern California; 2011. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/441052/rec/6975


University of Southern California

2. Ashton, Charles. Molecular mechanism of Stat3-mediated self-renewal in mouse embryonic stem cells.

Degree: MS, Molecular Microbiology and Immunology, 2011, University of Southern California

 Mouse embryonic stem (mES) cells are principally defined by two properties, pluripotency and self-renewal. In the conventional mES cell culture conditions, cytokine receptor-mediated activation of… (more)

Subjects/Keywords: Stat3; ES; embryonic; Tip60; self-renewal

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APA (6th Edition):

Ashton, C. (2011). Molecular mechanism of Stat3-mediated self-renewal in mouse embryonic stem cells. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/667447/rec/4189

Chicago Manual of Style (16th Edition):

Ashton, Charles. “Molecular mechanism of Stat3-mediated self-renewal in mouse embryonic stem cells.” 2011. Masters Thesis, University of Southern California. Accessed February 17, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/667447/rec/4189.

MLA Handbook (7th Edition):

Ashton, Charles. “Molecular mechanism of Stat3-mediated self-renewal in mouse embryonic stem cells.” 2011. Web. 17 Feb 2020.

Vancouver:

Ashton C. Molecular mechanism of Stat3-mediated self-renewal in mouse embryonic stem cells. [Internet] [Masters thesis]. University of Southern California; 2011. [cited 2020 Feb 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/667447/rec/4189.

Council of Science Editors:

Ashton C. Molecular mechanism of Stat3-mediated self-renewal in mouse embryonic stem cells. [Masters Thesis]. University of Southern California; 2011. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/667447/rec/4189


Boston University

3. Pan, Gilbert. Lysine acetyltransferase 5 in EGFR mutated non-small cell lung cancer.

Degree: MS, Medical Sciences, 2018, Boston University

 Histone modifications are crucial in activities such as transcriptional activation, gene silencing, and epigenetic cellular memory. In particular, lysine acetylation via lysine (K) acetyltransferases (KATs)… (more)

Subjects/Keywords: Molecular biology; EGFR; KAT5; NSCLC; Tip60

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APA (6th Edition):

Pan, G. (2018). Lysine acetyltransferase 5 in EGFR mutated non-small cell lung cancer. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/31272

Chicago Manual of Style (16th Edition):

Pan, Gilbert. “Lysine acetyltransferase 5 in EGFR mutated non-small cell lung cancer.” 2018. Masters Thesis, Boston University. Accessed February 17, 2020. http://hdl.handle.net/2144/31272.

MLA Handbook (7th Edition):

Pan, Gilbert. “Lysine acetyltransferase 5 in EGFR mutated non-small cell lung cancer.” 2018. Web. 17 Feb 2020.

Vancouver:

Pan G. Lysine acetyltransferase 5 in EGFR mutated non-small cell lung cancer. [Internet] [Masters thesis]. Boston University; 2018. [cited 2020 Feb 17]. Available from: http://hdl.handle.net/2144/31272.

Council of Science Editors:

Pan G. Lysine acetyltransferase 5 in EGFR mutated non-small cell lung cancer. [Masters Thesis]. Boston University; 2018. Available from: http://hdl.handle.net/2144/31272


University of Iowa

4. Reed, Sara Marie. NIAM, a novel activator of p53 and potential tumor suppressor.

Degree: PhD, Pharmacology, 2015, University of Iowa

  Cancer is the second leading cause of death in the United States, and it results from genetic alterations that promote the survival and proliferation… (more)

Subjects/Keywords: lymphoma; Mdm2; NIAM; p53; Tip60; Pharmacology

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APA (6th Edition):

Reed, S. M. (2015). NIAM, a novel activator of p53 and potential tumor suppressor. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/1735

Chicago Manual of Style (16th Edition):

Reed, Sara Marie. “NIAM, a novel activator of p53 and potential tumor suppressor.” 2015. Doctoral Dissertation, University of Iowa. Accessed February 17, 2020. https://ir.uiowa.edu/etd/1735.

MLA Handbook (7th Edition):

Reed, Sara Marie. “NIAM, a novel activator of p53 and potential tumor suppressor.” 2015. Web. 17 Feb 2020.

Vancouver:

Reed SM. NIAM, a novel activator of p53 and potential tumor suppressor. [Internet] [Doctoral dissertation]. University of Iowa; 2015. [cited 2020 Feb 17]. Available from: https://ir.uiowa.edu/etd/1735.

Council of Science Editors:

Reed SM. NIAM, a novel activator of p53 and potential tumor suppressor. [Doctoral Dissertation]. University of Iowa; 2015. Available from: https://ir.uiowa.edu/etd/1735

5. Aggeletopoulou, Ioanna. Ο ρόλος της Tip60 στη μεταγραφική ρύθμιση γονιδίων κυτταροκινών των Τ βοηθητικών λεμφοκυττάρων.

Degree: 2019, University of Patras; Πανεπιστήμιο Πατρών

 IL-2 is the first cytokine produced when naive T helper (Th) cells are activated and differentiate into proliferating pre-Th0 precursors. Transcriptional regulation of IL-2 gene… (more)

Subjects/Keywords: Ακετυλοτρανσφεράση; Tip60; Κυτταροκίνες; Τ βοηθητικά λεμφοκύτταρα; Acetyltransferase; Tip60; Cytokines; T helper cells

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APA (6th Edition):

Aggeletopoulou, I. (2019). Ο ρόλος της Tip60 στη μεταγραφική ρύθμιση γονιδίων κυτταροκινών των Τ βοηθητικών λεμφοκυττάρων. (Thesis). University of Patras; Πανεπιστήμιο Πατρών. Retrieved from http://hdl.handle.net/10442/hedi/46506

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Aggeletopoulou, Ioanna. “Ο ρόλος της Tip60 στη μεταγραφική ρύθμιση γονιδίων κυτταροκινών των Τ βοηθητικών λεμφοκυττάρων.” 2019. Thesis, University of Patras; Πανεπιστήμιο Πατρών. Accessed February 17, 2020. http://hdl.handle.net/10442/hedi/46506.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Aggeletopoulou, Ioanna. “Ο ρόλος της Tip60 στη μεταγραφική ρύθμιση γονιδίων κυτταροκινών των Τ βοηθητικών λεμφοκυττάρων.” 2019. Web. 17 Feb 2020.

Vancouver:

Aggeletopoulou I. Ο ρόλος της Tip60 στη μεταγραφική ρύθμιση γονιδίων κυτταροκινών των Τ βοηθητικών λεμφοκυττάρων. [Internet] [Thesis]. University of Patras; Πανεπιστήμιο Πατρών; 2019. [cited 2020 Feb 17]. Available from: http://hdl.handle.net/10442/hedi/46506.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Aggeletopoulou I. Ο ρόλος της Tip60 στη μεταγραφική ρύθμιση γονιδίων κυτταροκινών των Τ βοηθητικών λεμφοκυττάρων. [Thesis]. University of Patras; Πανεπιστήμιο Πατρών; 2019. Available from: http://hdl.handle.net/10442/hedi/46506

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

6. Ravens, Sarina. A genome-wide characterization of Mof or Tip60 containing complexes in mouse embryonic stem cells : L'analyse génomique des complexes contenant les acétyltransférases Mof ou Tip60 révèle des fonctions à la fois redondantes mais aussi spécifiques dans les cellules souches embryonnaire de souris.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2014, Université de Strasbourg

L’acétylation des histones est associée à une activation transcriptionnelle. Cette acétylation est mise en place par des histone acétyltransférases (HATs) qui sont le plus souvent… (more)

Subjects/Keywords: Histone acétyltransférases; Epigénetique; Mof; Tip60; Cellules souches embryonnaires de souris; Histone acetyltransferases; Epigenetic control; Mof; Tip60; Mouse embryonic stem cells; 572.8

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APA (6th Edition):

Ravens, S. (2014). A genome-wide characterization of Mof or Tip60 containing complexes in mouse embryonic stem cells : L'analyse génomique des complexes contenant les acétyltransférases Mof ou Tip60 révèle des fonctions à la fois redondantes mais aussi spécifiques dans les cellules souches embryonnaire de souris. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2014STRAJ097

Chicago Manual of Style (16th Edition):

Ravens, Sarina. “A genome-wide characterization of Mof or Tip60 containing complexes in mouse embryonic stem cells : L'analyse génomique des complexes contenant les acétyltransférases Mof ou Tip60 révèle des fonctions à la fois redondantes mais aussi spécifiques dans les cellules souches embryonnaire de souris.” 2014. Doctoral Dissertation, Université de Strasbourg. Accessed February 17, 2020. http://www.theses.fr/2014STRAJ097.

MLA Handbook (7th Edition):

Ravens, Sarina. “A genome-wide characterization of Mof or Tip60 containing complexes in mouse embryonic stem cells : L'analyse génomique des complexes contenant les acétyltransférases Mof ou Tip60 révèle des fonctions à la fois redondantes mais aussi spécifiques dans les cellules souches embryonnaire de souris.” 2014. Web. 17 Feb 2020.

Vancouver:

Ravens S. A genome-wide characterization of Mof or Tip60 containing complexes in mouse embryonic stem cells : L'analyse génomique des complexes contenant les acétyltransférases Mof ou Tip60 révèle des fonctions à la fois redondantes mais aussi spécifiques dans les cellules souches embryonnaire de souris. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2014. [cited 2020 Feb 17]. Available from: http://www.theses.fr/2014STRAJ097.

Council of Science Editors:

Ravens S. A genome-wide characterization of Mof or Tip60 containing complexes in mouse embryonic stem cells : L'analyse génomique des complexes contenant les acétyltransférases Mof ou Tip60 révèle des fonctions à la fois redondantes mais aussi spécifiques dans les cellules souches embryonnaire de souris. [Doctoral Dissertation]. Université de Strasbourg; 2014. Available from: http://www.theses.fr/2014STRAJ097

7. Rifai, Khaldoun. Etude des modifications épigénétiques en fonction de l'agressivité du cancer sporadique du sein : l'implication de l'histone désacétylase SIRT1 dans la progression tumorale : Study of Epigenetic Modifications depending on Sporadic Breast Cancer Virulence : Involvement of Histone Deacetylase SIRT1 in Tumor Progression.

Degree: Docteur es, Physiologie et biologie des organismes - populations - interactions, 2018, Clermont Auvergne

Avec 59 000 nouveaux cas en 2017, le cancer du sein est le cancer le plus fréquemment diagnostiqué chez les femmes françaises, et pose un… (more)

Subjects/Keywords: Cancer du Sein; Épi-drogues; Épigénétique; SIRT1; TIP60; H3K4ac; Breast cancer; Epi-drugs; H3K4ac; TIP60; SIRT1; Epigenetics; 616.994

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APA (6th Edition):

Rifai, K. (2018). Etude des modifications épigénétiques en fonction de l'agressivité du cancer sporadique du sein : l'implication de l'histone désacétylase SIRT1 dans la progression tumorale : Study of Epigenetic Modifications depending on Sporadic Breast Cancer Virulence : Involvement of Histone Deacetylase SIRT1 in Tumor Progression. (Doctoral Dissertation). Clermont Auvergne. Retrieved from http://www.theses.fr/2018CLFAS011

Chicago Manual of Style (16th Edition):

Rifai, Khaldoun. “Etude des modifications épigénétiques en fonction de l'agressivité du cancer sporadique du sein : l'implication de l'histone désacétylase SIRT1 dans la progression tumorale : Study of Epigenetic Modifications depending on Sporadic Breast Cancer Virulence : Involvement of Histone Deacetylase SIRT1 in Tumor Progression.” 2018. Doctoral Dissertation, Clermont Auvergne. Accessed February 17, 2020. http://www.theses.fr/2018CLFAS011.

MLA Handbook (7th Edition):

Rifai, Khaldoun. “Etude des modifications épigénétiques en fonction de l'agressivité du cancer sporadique du sein : l'implication de l'histone désacétylase SIRT1 dans la progression tumorale : Study of Epigenetic Modifications depending on Sporadic Breast Cancer Virulence : Involvement of Histone Deacetylase SIRT1 in Tumor Progression.” 2018. Web. 17 Feb 2020.

Vancouver:

Rifai K. Etude des modifications épigénétiques en fonction de l'agressivité du cancer sporadique du sein : l'implication de l'histone désacétylase SIRT1 dans la progression tumorale : Study of Epigenetic Modifications depending on Sporadic Breast Cancer Virulence : Involvement of Histone Deacetylase SIRT1 in Tumor Progression. [Internet] [Doctoral dissertation]. Clermont Auvergne; 2018. [cited 2020 Feb 17]. Available from: http://www.theses.fr/2018CLFAS011.

Council of Science Editors:

Rifai K. Etude des modifications épigénétiques en fonction de l'agressivité du cancer sporadique du sein : l'implication de l'histone désacétylase SIRT1 dans la progression tumorale : Study of Epigenetic Modifications depending on Sporadic Breast Cancer Virulence : Involvement of Histone Deacetylase SIRT1 in Tumor Progression. [Doctoral Dissertation]. Clermont Auvergne; 2018. Available from: http://www.theses.fr/2018CLFAS011

8. Αγγελετοπούλου, Ιωάννα. Η έκφραση της ακετυλοτρανσφεράσης Tip60 σε υποπληθυσμούς Τ λεμφοκυττάρων και ο ρόλος της στη μεταγραφή του γονιδίου της ιντερλευκίνης-2 και του ιού HIV-1.

Degree: 2015, University of Patras

 Η πρωτεΐνη Tip60 (Tat-interactive protein, 60 kDa) είναι μέλος της οικογένειας πρωτεϊνών MYST και αρχικά προσδιορίστηκε ως αλληλεπιδρώσα πρωτεΐνη με την HIV-1 ΤΑΤ πρωτεΐνη. Πολλά… (more)

Subjects/Keywords: Ιντερλευκίνη 2; 572.645; Interleukin-2 (IL-2); HIV-1; Tip60

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APA (6th Edition):

Αγγελετοπούλου, . (2015). Η έκφραση της ακετυλοτρανσφεράσης Tip60 σε υποπληθυσμούς Τ λεμφοκυττάρων και ο ρόλος της στη μεταγραφή του γονιδίου της ιντερλευκίνης-2 και του ιού HIV-1. (Masters Thesis). University of Patras. Retrieved from http://hdl.handle.net/10889/8699

Chicago Manual of Style (16th Edition):

Αγγελετοπούλου, Ιωάννα. “Η έκφραση της ακετυλοτρανσφεράσης Tip60 σε υποπληθυσμούς Τ λεμφοκυττάρων και ο ρόλος της στη μεταγραφή του γονιδίου της ιντερλευκίνης-2 και του ιού HIV-1.” 2015. Masters Thesis, University of Patras. Accessed February 17, 2020. http://hdl.handle.net/10889/8699.

MLA Handbook (7th Edition):

Αγγελετοπούλου, Ιωάννα. “Η έκφραση της ακετυλοτρανσφεράσης Tip60 σε υποπληθυσμούς Τ λεμφοκυττάρων και ο ρόλος της στη μεταγραφή του γονιδίου της ιντερλευκίνης-2 και του ιού HIV-1.” 2015. Web. 17 Feb 2020.

Vancouver:

Αγγελετοπούλου . Η έκφραση της ακετυλοτρανσφεράσης Tip60 σε υποπληθυσμούς Τ λεμφοκυττάρων και ο ρόλος της στη μεταγραφή του γονιδίου της ιντερλευκίνης-2 και του ιού HIV-1. [Internet] [Masters thesis]. University of Patras; 2015. [cited 2020 Feb 17]. Available from: http://hdl.handle.net/10889/8699.

Council of Science Editors:

Αγγελετοπούλου . Η έκφραση της ακετυλοτρανσφεράσης Tip60 σε υποπληθυσμούς Τ λεμφοκυττάρων και ο ρόλος της στη μεταγραφή του γονιδίου της ιντερλευκίνης-2 και του ιού HIV-1. [Masters Thesis]. University of Patras; 2015. Available from: http://hdl.handle.net/10889/8699


University of Saskatchewan

9. Arrafi, Sifa 1988-. Structural and Functional Characterization of the Tip60 Chromodomain.

Degree: 2016, University of Saskatchewan

 The Tat-interactive protein of 60 kDa (Tip60) is a histone acetyltransferase enzyme that appears to have a wide range of acetylation targets, which include core… (more)

Subjects/Keywords: Tip60; Chromodomain; Histone; X-ray crystallography; ITC; SPR

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APA (6th Edition):

Arrafi, S. 1. (2016). Structural and Functional Characterization of the Tip60 Chromodomain. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/7536

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Arrafi, Sifa 1988-. “Structural and Functional Characterization of the Tip60 Chromodomain.” 2016. Thesis, University of Saskatchewan. Accessed February 17, 2020. http://hdl.handle.net/10388/7536.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Arrafi, Sifa 1988-. “Structural and Functional Characterization of the Tip60 Chromodomain.” 2016. Web. 17 Feb 2020.

Vancouver:

Arrafi S1. Structural and Functional Characterization of the Tip60 Chromodomain. [Internet] [Thesis]. University of Saskatchewan; 2016. [cited 2020 Feb 17]. Available from: http://hdl.handle.net/10388/7536.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Arrafi S1. Structural and Functional Characterization of the Tip60 Chromodomain. [Thesis]. University of Saskatchewan; 2016. Available from: http://hdl.handle.net/10388/7536

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. Grézy, Aude. Implication de l'acétyltransférase TIP60 dans le maintien de l'hétérochromatine péricentromérique chez les mammifères : Implication of the TIP60 acetyltransferase in pericentrometric heterochromatin maintenance in mammals.

Degree: Docteur es, Génétique moléculaire, 2015, Université Toulouse III – Paul Sabatier

Au sein du noyau des cellules eucaryotes, la molécule d'ADN s'enroule autour de protéines histones, formant la chromatine. Ce mécanisme de compaction est dynamique selon… (more)

Subjects/Keywords: Chromatine; Hétérochromatine; Péricentromères; Instabilité génétique; TIP60; Séquences répétées; Acétylation

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APA (6th Edition):

Grézy, A. (2015). Implication de l'acétyltransférase TIP60 dans le maintien de l'hétérochromatine péricentromérique chez les mammifères : Implication of the TIP60 acetyltransferase in pericentrometric heterochromatin maintenance in mammals. (Doctoral Dissertation). Université Toulouse III – Paul Sabatier. Retrieved from http://www.theses.fr/2015TOU30206

Chicago Manual of Style (16th Edition):

Grézy, Aude. “Implication de l'acétyltransférase TIP60 dans le maintien de l'hétérochromatine péricentromérique chez les mammifères : Implication of the TIP60 acetyltransferase in pericentrometric heterochromatin maintenance in mammals.” 2015. Doctoral Dissertation, Université Toulouse III – Paul Sabatier. Accessed February 17, 2020. http://www.theses.fr/2015TOU30206.

MLA Handbook (7th Edition):

Grézy, Aude. “Implication de l'acétyltransférase TIP60 dans le maintien de l'hétérochromatine péricentromérique chez les mammifères : Implication of the TIP60 acetyltransferase in pericentrometric heterochromatin maintenance in mammals.” 2015. Web. 17 Feb 2020.

Vancouver:

Grézy A. Implication de l'acétyltransférase TIP60 dans le maintien de l'hétérochromatine péricentromérique chez les mammifères : Implication of the TIP60 acetyltransferase in pericentrometric heterochromatin maintenance in mammals. [Internet] [Doctoral dissertation]. Université Toulouse III – Paul Sabatier; 2015. [cited 2020 Feb 17]. Available from: http://www.theses.fr/2015TOU30206.

Council of Science Editors:

Grézy A. Implication de l'acétyltransférase TIP60 dans le maintien de l'hétérochromatine péricentromérique chez les mammifères : Implication of the TIP60 acetyltransferase in pericentrometric heterochromatin maintenance in mammals. [Doctoral Dissertation]. Université Toulouse III – Paul Sabatier; 2015. Available from: http://www.theses.fr/2015TOU30206

11. Sheel, Ankur. Identification of Essential Genes in Hepatocellular Carcinomas using CRISPR Screening.

Degree: PhD, MD/PhD Program; RNA Therapeutics Institute, 2019, U of Massachusetts : Med

  Hepatocellular carcinoma (HCC) is an aggressive subtype of liver cancer with a poor prognosis. Currently, prognosis for HCC patients remains poor as few therapies… (more)

Subjects/Keywords: Hepatocellular Carcinoma; CRISPR; Senescence; TRRAP; TIP60; P53 Independence; Cancer Biology

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APA (6th Edition):

Sheel, A. (2019). Identification of Essential Genes in Hepatocellular Carcinomas using CRISPR Screening. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/1039

Chicago Manual of Style (16th Edition):

Sheel, Ankur. “Identification of Essential Genes in Hepatocellular Carcinomas using CRISPR Screening.” 2019. Doctoral Dissertation, U of Massachusetts : Med. Accessed February 17, 2020. https://escholarship.umassmed.edu/gsbs_diss/1039.

MLA Handbook (7th Edition):

Sheel, Ankur. “Identification of Essential Genes in Hepatocellular Carcinomas using CRISPR Screening.” 2019. Web. 17 Feb 2020.

Vancouver:

Sheel A. Identification of Essential Genes in Hepatocellular Carcinomas using CRISPR Screening. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2019. [cited 2020 Feb 17]. Available from: https://escholarship.umassmed.edu/gsbs_diss/1039.

Council of Science Editors:

Sheel A. Identification of Essential Genes in Hepatocellular Carcinomas using CRISPR Screening. [Doctoral Dissertation]. U of Massachusetts : Med; 2019. Available from: https://escholarship.umassmed.edu/gsbs_diss/1039

12. Ashraf, Waseem. Mécanismes d'interaction de l'intégrateur épigénétique UHRF1 avec l'acétyltransférase TIP60 : Interaction mechanisms of epigenetic integrator UHRF1 with TIP60 acetyltransferase.

Degree: Docteur es, Pharmacologie-pharmacocinétique, 2018, Université de Strasbourg

UHRF1 est une protéine nucléaire responsable du maintien et de la régulation de l'épigénome des cellules. Elle favorise la prolifération cellulaire et est surexprimée dans… (more)

Subjects/Keywords: UHRF1; TIP60; Interactions protéine-protéine; DNMT1; FLIM; FRET; Méthylation de l’ADN; Histone acétyltransférase (HAT); UHRF1; TIP60; Protein-Protein interaction; DNMT1; FLIM; FRET; DNA methylation; Histone acetyltransferase (HAT); 572.8; 615.7

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APA (6th Edition):

Ashraf, W. (2018). Mécanismes d'interaction de l'intégrateur épigénétique UHRF1 avec l'acétyltransférase TIP60 : Interaction mechanisms of epigenetic integrator UHRF1 with TIP60 acetyltransferase. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2018STRAJ078

Chicago Manual of Style (16th Edition):

Ashraf, Waseem. “Mécanismes d'interaction de l'intégrateur épigénétique UHRF1 avec l'acétyltransférase TIP60 : Interaction mechanisms of epigenetic integrator UHRF1 with TIP60 acetyltransferase.” 2018. Doctoral Dissertation, Université de Strasbourg. Accessed February 17, 2020. http://www.theses.fr/2018STRAJ078.

MLA Handbook (7th Edition):

Ashraf, Waseem. “Mécanismes d'interaction de l'intégrateur épigénétique UHRF1 avec l'acétyltransférase TIP60 : Interaction mechanisms of epigenetic integrator UHRF1 with TIP60 acetyltransferase.” 2018. Web. 17 Feb 2020.

Vancouver:

Ashraf W. Mécanismes d'interaction de l'intégrateur épigénétique UHRF1 avec l'acétyltransférase TIP60 : Interaction mechanisms of epigenetic integrator UHRF1 with TIP60 acetyltransferase. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2018. [cited 2020 Feb 17]. Available from: http://www.theses.fr/2018STRAJ078.

Council of Science Editors:

Ashraf W. Mécanismes d'interaction de l'intégrateur épigénétique UHRF1 avec l'acétyltransférase TIP60 : Interaction mechanisms of epigenetic integrator UHRF1 with TIP60 acetyltransferase. [Doctoral Dissertation]. Université de Strasbourg; 2018. Available from: http://www.theses.fr/2018STRAJ078

13. Ngo, Liza D. Rational Design, Synthesis, and Evaluation of Inhibitors for the HIV-1 TAT Interacting Protein Tip60.

Degree: MS, Biology, 2013, Georgia State University

  The histone acetyltransferase protein, Tip60, has many important functions in epigenetic regulation of gene expression and DNA repair. Our objective is to design, synthesize,… (more)

Subjects/Keywords: : Tip60; Chromodomain; MYST; Tip60 inhibitors; Epigenetics; HAT; PCAF

…3 1.1 1.2 Diseases linked to deregulation of Tip60… …4 1.3 Previously reported inhibitors for Tip60… …5 1.4 2 The histone acetyltransferase Tip60… …9 2.3.1 Determining the suitable substrate for fl-Tip60 and cat-Tip60… …9 2.3.2 Efficiency analysis of fl-Tip60, cat-Tip60, and PCAF… 

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APA (6th Edition):

Ngo, L. D. (2013). Rational Design, Synthesis, and Evaluation of Inhibitors for the HIV-1 TAT Interacting Protein Tip60. (Thesis). Georgia State University. Retrieved from https://scholarworks.gsu.edu/biology_theses/46

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ngo, Liza D. “Rational Design, Synthesis, and Evaluation of Inhibitors for the HIV-1 TAT Interacting Protein Tip60.” 2013. Thesis, Georgia State University. Accessed February 17, 2020. https://scholarworks.gsu.edu/biology_theses/46.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ngo, Liza D. “Rational Design, Synthesis, and Evaluation of Inhibitors for the HIV-1 TAT Interacting Protein Tip60.” 2013. Web. 17 Feb 2020.

Vancouver:

Ngo LD. Rational Design, Synthesis, and Evaluation of Inhibitors for the HIV-1 TAT Interacting Protein Tip60. [Internet] [Thesis]. Georgia State University; 2013. [cited 2020 Feb 17]. Available from: https://scholarworks.gsu.edu/biology_theses/46.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ngo LD. Rational Design, Synthesis, and Evaluation of Inhibitors for the HIV-1 TAT Interacting Protein Tip60. [Thesis]. Georgia State University; 2013. Available from: https://scholarworks.gsu.edu/biology_theses/46

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Southern California

14. Jeong, KwangWon. Molecular mechanism of the recruitment of SWI/SNF chromatin remodeling complex and histone acetyltransferase to estrogen-responsive promoters.

Degree: PhD, Biochemistry & Molecular Biology, 2010, University of Southern California

 Estrogen receptor α (ER) is a member of the family of nuclear receptors and functions as a transcriptional factor to induce gene expression by binding… (more)

Subjects/Keywords: estrogen receptor; SWI/SNF; chromatin remodeling; Fli-I; Tip60; histone; coactivator; nuclear receptor; flightless

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APA (6th Edition):

Jeong, K. (2010). Molecular mechanism of the recruitment of SWI/SNF chromatin remodeling complex and histone acetyltransferase to estrogen-responsive promoters. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/314818/rec/4190

Chicago Manual of Style (16th Edition):

Jeong, KwangWon. “Molecular mechanism of the recruitment of SWI/SNF chromatin remodeling complex and histone acetyltransferase to estrogen-responsive promoters.” 2010. Doctoral Dissertation, University of Southern California. Accessed February 17, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/314818/rec/4190.

MLA Handbook (7th Edition):

Jeong, KwangWon. “Molecular mechanism of the recruitment of SWI/SNF chromatin remodeling complex and histone acetyltransferase to estrogen-responsive promoters.” 2010. Web. 17 Feb 2020.

Vancouver:

Jeong K. Molecular mechanism of the recruitment of SWI/SNF chromatin remodeling complex and histone acetyltransferase to estrogen-responsive promoters. [Internet] [Doctoral dissertation]. University of Southern California; 2010. [cited 2020 Feb 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/314818/rec/4190.

Council of Science Editors:

Jeong K. Molecular mechanism of the recruitment of SWI/SNF chromatin remodeling complex and histone acetyltransferase to estrogen-responsive promoters. [Doctoral Dissertation]. University of Southern California; 2010. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/314818/rec/4190


Massey University

15. Smith, Rebecca Jane. ATM and p400 : characterisation of a novel interaction between a DNA repair enzyme and a chromatin remodeler : a thesis presented to Massey University in partial fulfilment of the requirements for the degree of Doctor of Philosophy in Biochemistry .

Degree: 2014, Massey University

 The ability to maintain genomic integrity prevents unrestricted cell proliferation and the progression of cancer. DNA repair pathways such as the DNA double-strand break (DSB)… (more)

Subjects/Keywords: DNA repair; Ataxia telegiectasia mutated (ATM); Phosphorylation; Chromatin; TIP60; Protein kinases; Phosphoproteins

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APA (6th Edition):

Smith, R. J. (2014). ATM and p400 : characterisation of a novel interaction between a DNA repair enzyme and a chromatin remodeler : a thesis presented to Massey University in partial fulfilment of the requirements for the degree of Doctor of Philosophy in Biochemistry . (Thesis). Massey University. Retrieved from http://hdl.handle.net/10179/6960

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Smith, Rebecca Jane. “ATM and p400 : characterisation of a novel interaction between a DNA repair enzyme and a chromatin remodeler : a thesis presented to Massey University in partial fulfilment of the requirements for the degree of Doctor of Philosophy in Biochemistry .” 2014. Thesis, Massey University. Accessed February 17, 2020. http://hdl.handle.net/10179/6960.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Smith, Rebecca Jane. “ATM and p400 : characterisation of a novel interaction between a DNA repair enzyme and a chromatin remodeler : a thesis presented to Massey University in partial fulfilment of the requirements for the degree of Doctor of Philosophy in Biochemistry .” 2014. Web. 17 Feb 2020.

Vancouver:

Smith RJ. ATM and p400 : characterisation of a novel interaction between a DNA repair enzyme and a chromatin remodeler : a thesis presented to Massey University in partial fulfilment of the requirements for the degree of Doctor of Philosophy in Biochemistry . [Internet] [Thesis]. Massey University; 2014. [cited 2020 Feb 17]. Available from: http://hdl.handle.net/10179/6960.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Smith RJ. ATM and p400 : characterisation of a novel interaction between a DNA repair enzyme and a chromatin remodeler : a thesis presented to Massey University in partial fulfilment of the requirements for the degree of Doctor of Philosophy in Biochemistry . [Thesis]. Massey University; 2014. Available from: http://hdl.handle.net/10179/6960

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of South Florida

16. Bryant, Victoria. CMG Helicase Assembly and Activation: Regulation by c-Myc through Chromatin Decondensation and Novel Therapeutic Avenues for Cancer Treatment.

Degree: 2016, University of South Florida

 The CMG (Cdc45, MCM, GINS) helicase is required for cellular proliferation and functions to unwind double-stranded DNA to allow the replication machinery to duplicate the… (more)

Subjects/Keywords: DNA replication; MCM; Cdc45; GINS; GCN5; Tip60; Biology; Cell Biology; Molecular Biology

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APA (6th Edition):

Bryant, V. (2016). CMG Helicase Assembly and Activation: Regulation by c-Myc through Chromatin Decondensation and Novel Therapeutic Avenues for Cancer Treatment. (Thesis). University of South Florida. Retrieved from https://scholarcommons.usf.edu/etd/6191

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bryant, Victoria. “CMG Helicase Assembly and Activation: Regulation by c-Myc through Chromatin Decondensation and Novel Therapeutic Avenues for Cancer Treatment.” 2016. Thesis, University of South Florida. Accessed February 17, 2020. https://scholarcommons.usf.edu/etd/6191.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bryant, Victoria. “CMG Helicase Assembly and Activation: Regulation by c-Myc through Chromatin Decondensation and Novel Therapeutic Avenues for Cancer Treatment.” 2016. Web. 17 Feb 2020.

Vancouver:

Bryant V. CMG Helicase Assembly and Activation: Regulation by c-Myc through Chromatin Decondensation and Novel Therapeutic Avenues for Cancer Treatment. [Internet] [Thesis]. University of South Florida; 2016. [cited 2020 Feb 17]. Available from: https://scholarcommons.usf.edu/etd/6191.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bryant V. CMG Helicase Assembly and Activation: Regulation by c-Myc through Chromatin Decondensation and Novel Therapeutic Avenues for Cancer Treatment. [Thesis]. University of South Florida; 2016. Available from: https://scholarcommons.usf.edu/etd/6191

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. DEEPA RAJAGOPALAN. MECHANISTIC CHARACTERIZATION OF THE TUMOR SUPPRESSIVE ROLE OF TIP60 IN CANCER.

Degree: 2017, National University of Singapore

Subjects/Keywords: TIP60; HPV; TERT; ERVs; IRF7; cancer

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APA (6th Edition):

RAJAGOPALAN, D. (2017). MECHANISTIC CHARACTERIZATION OF THE TUMOR SUPPRESSIVE ROLE OF TIP60 IN CANCER. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/139421

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

RAJAGOPALAN, DEEPA. “MECHANISTIC CHARACTERIZATION OF THE TUMOR SUPPRESSIVE ROLE OF TIP60 IN CANCER.” 2017. Thesis, National University of Singapore. Accessed February 17, 2020. http://scholarbank.nus.edu.sg/handle/10635/139421.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

RAJAGOPALAN, DEEPA. “MECHANISTIC CHARACTERIZATION OF THE TUMOR SUPPRESSIVE ROLE OF TIP60 IN CANCER.” 2017. Web. 17 Feb 2020.

Vancouver:

RAJAGOPALAN D. MECHANISTIC CHARACTERIZATION OF THE TUMOR SUPPRESSIVE ROLE OF TIP60 IN CANCER. [Internet] [Thesis]. National University of Singapore; 2017. [cited 2020 Feb 17]. Available from: http://scholarbank.nus.edu.sg/handle/10635/139421.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

RAJAGOPALAN D. MECHANISTIC CHARACTERIZATION OF THE TUMOR SUPPRESSIVE ROLE OF TIP60 IN CANCER. [Thesis]. National University of Singapore; 2017. Available from: http://scholarbank.nus.edu.sg/handle/10635/139421

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. MOHAMMAD NIZAM B ABDUL KADIR. The dynamic interplay of chromatin modifying factors and DNA-bound transcription factors in the regulation of mouse gonadotropin genes via multiple signaling pathways.

Degree: 2005, National University of Singapore

Subjects/Keywords: Gonadotropin; HAT; HDAC; GCN5; Tip60; Smad4

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APA (6th Edition):

KADIR, M. N. B. A. (2005). The dynamic interplay of chromatin modifying factors and DNA-bound transcription factors in the regulation of mouse gonadotropin genes via multiple signaling pathways. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/17011

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

KADIR, MOHAMMAD NIZAM B ABDUL. “The dynamic interplay of chromatin modifying factors and DNA-bound transcription factors in the regulation of mouse gonadotropin genes via multiple signaling pathways.” 2005. Thesis, National University of Singapore. Accessed February 17, 2020. http://scholarbank.nus.edu.sg/handle/10635/17011.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

KADIR, MOHAMMAD NIZAM B ABDUL. “The dynamic interplay of chromatin modifying factors and DNA-bound transcription factors in the regulation of mouse gonadotropin genes via multiple signaling pathways.” 2005. Web. 17 Feb 2020.

Vancouver:

KADIR MNBA. The dynamic interplay of chromatin modifying factors and DNA-bound transcription factors in the regulation of mouse gonadotropin genes via multiple signaling pathways. [Internet] [Thesis]. National University of Singapore; 2005. [cited 2020 Feb 17]. Available from: http://scholarbank.nus.edu.sg/handle/10635/17011.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

KADIR MNBA. The dynamic interplay of chromatin modifying factors and DNA-bound transcription factors in the regulation of mouse gonadotropin genes via multiple signaling pathways. [Thesis]. National University of Singapore; 2005. Available from: http://scholarbank.nus.edu.sg/handle/10635/17011

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. LEE KWOK KIN. IDENTIFYING REGULATORS OF TIP60.

Degree: 2018, National University of Singapore

Subjects/Keywords: TIP60; TRIP12; metastasis; EMT; screen; ligase

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APA (6th Edition):

KIN, L. K. (2018). IDENTIFYING REGULATORS OF TIP60. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/151491

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

KIN, LEE KWOK. “IDENTIFYING REGULATORS OF TIP60.” 2018. Thesis, National University of Singapore. Accessed February 17, 2020. http://scholarbank.nus.edu.sg/handle/10635/151491.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

KIN, LEE KWOK. “IDENTIFYING REGULATORS OF TIP60.” 2018. Web. 17 Feb 2020.

Vancouver:

KIN LK. IDENTIFYING REGULATORS OF TIP60. [Internet] [Thesis]. National University of Singapore; 2018. [cited 2020 Feb 17]. Available from: http://scholarbank.nus.edu.sg/handle/10635/151491.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

KIN LK. IDENTIFYING REGULATORS OF TIP60. [Thesis]. National University of Singapore; 2018. Available from: http://scholarbank.nus.edu.sg/handle/10635/151491

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. WANG CHENG. ROLE OF SRPK1-MODULATED ALTERNATIVE SPLICING IN CISPLATIN RESISTANCE IN BREAST CANCER CELLS.

Degree: 2019, National University of Singapore

Subjects/Keywords: Cisplatin Breast Cancer Splicing SRPK1 TIP60

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APA (6th Edition):

CHENG, W. (2019). ROLE OF SRPK1-MODULATED ALTERNATIVE SPLICING IN CISPLATIN RESISTANCE IN BREAST CANCER CELLS. (Thesis). National University of Singapore. Retrieved from https://scholarbank.nus.edu.sg/handle/10635/159904

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

CHENG, WANG. “ROLE OF SRPK1-MODULATED ALTERNATIVE SPLICING IN CISPLATIN RESISTANCE IN BREAST CANCER CELLS.” 2019. Thesis, National University of Singapore. Accessed February 17, 2020. https://scholarbank.nus.edu.sg/handle/10635/159904.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

CHENG, WANG. “ROLE OF SRPK1-MODULATED ALTERNATIVE SPLICING IN CISPLATIN RESISTANCE IN BREAST CANCER CELLS.” 2019. Web. 17 Feb 2020.

Vancouver:

CHENG W. ROLE OF SRPK1-MODULATED ALTERNATIVE SPLICING IN CISPLATIN RESISTANCE IN BREAST CANCER CELLS. [Internet] [Thesis]. National University of Singapore; 2019. [cited 2020 Feb 17]. Available from: https://scholarbank.nus.edu.sg/handle/10635/159904.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

CHENG W. ROLE OF SRPK1-MODULATED ALTERNATIVE SPLICING IN CISPLATIN RESISTANCE IN BREAST CANCER CELLS. [Thesis]. National University of Singapore; 2019. Available from: https://scholarbank.nus.edu.sg/handle/10635/159904

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Georgia State University

21. Wu, Jiang. Development of Inhibitors and Assay Methods for Histone Acetyltransferases.

Degree: PhD, Chemistry, 2011, Georgia State University

  Histone acetyltransferases (HATs) are important enzymes in transcriptional control and potential targets for chemotherapeutic intervention in malignant diseases. Among different HAT members, the yeast… (more)

Subjects/Keywords: Histone acetyltransferases; Tip60; Substrate-based analogs; Virtual screening; Computer modeling; Fluorescent reporters; Chemistry

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APA (6th Edition):

Wu, J. (2011). Development of Inhibitors and Assay Methods for Histone Acetyltransferases. (Doctoral Dissertation). Georgia State University. Retrieved from https://scholarworks.gsu.edu/chemistry_diss/52

Chicago Manual of Style (16th Edition):

Wu, Jiang. “Development of Inhibitors and Assay Methods for Histone Acetyltransferases.” 2011. Doctoral Dissertation, Georgia State University. Accessed February 17, 2020. https://scholarworks.gsu.edu/chemistry_diss/52.

MLA Handbook (7th Edition):

Wu, Jiang. “Development of Inhibitors and Assay Methods for Histone Acetyltransferases.” 2011. Web. 17 Feb 2020.

Vancouver:

Wu J. Development of Inhibitors and Assay Methods for Histone Acetyltransferases. [Internet] [Doctoral dissertation]. Georgia State University; 2011. [cited 2020 Feb 17]. Available from: https://scholarworks.gsu.edu/chemistry_diss/52.

Council of Science Editors:

Wu J. Development of Inhibitors and Assay Methods for Histone Acetyltransferases. [Doctoral Dissertation]. Georgia State University; 2011. Available from: https://scholarworks.gsu.edu/chemistry_diss/52


Freie Universität Berlin

22. Kramer, Tobias. Identification of the histon-acetyl-transferaseTip60 interacting with β-Catenin and regulatory mechanism of KAI1 in oncogenesis.

Degree: 2016, Freie Universität Berlin

 Regulation of the canonical Wnt-signaling pathway and its core member β-Catenin are of great importance in oncogenesis and metastasis. Especially in colon and metastatic prostate… (more)

Subjects/Keywords: β-Catenin; Tip60/Kat5, KAI1/CD82; histon-acetyl-transferase; oncogenesis; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

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APA (6th Edition):

Kramer, T. (2016). Identification of the histon-acetyl-transferaseTip60 interacting with β-Catenin and regulatory mechanism of KAI1 in oncogenesis. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-9739

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kramer, Tobias. “Identification of the histon-acetyl-transferaseTip60 interacting with β-Catenin and regulatory mechanism of KAI1 in oncogenesis.” 2016. Thesis, Freie Universität Berlin. Accessed February 17, 2020. http://dx.doi.org/10.17169/refubium-9739.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kramer, Tobias. “Identification of the histon-acetyl-transferaseTip60 interacting with β-Catenin and regulatory mechanism of KAI1 in oncogenesis.” 2016. Web. 17 Feb 2020.

Vancouver:

Kramer T. Identification of the histon-acetyl-transferaseTip60 interacting with β-Catenin and regulatory mechanism of KAI1 in oncogenesis. [Internet] [Thesis]. Freie Universität Berlin; 2016. [cited 2020 Feb 17]. Available from: http://dx.doi.org/10.17169/refubium-9739.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kramer T. Identification of the histon-acetyl-transferaseTip60 interacting with β-Catenin and regulatory mechanism of KAI1 in oncogenesis. [Thesis]. Freie Universität Berlin; 2016. Available from: http://dx.doi.org/10.17169/refubium-9739

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. ZHANG YANZHOU. MECHANISM AND IMPLICATIONS OF DESTABILIZING TIP60, A TUMOUR SUPPRESSOR.

Degree: 2016, National University of Singapore

Subjects/Keywords: TIP60; EDD1; SNAIL2; DNMT1; HPV tumorigenesis; EMT

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APA (6th Edition):

YANZHOU, Z. (2016). MECHANISM AND IMPLICATIONS OF DESTABILIZING TIP60, A TUMOUR SUPPRESSOR. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/134938

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

YANZHOU, ZHANG. “MECHANISM AND IMPLICATIONS OF DESTABILIZING TIP60, A TUMOUR SUPPRESSOR.” 2016. Thesis, National University of Singapore. Accessed February 17, 2020. http://scholarbank.nus.edu.sg/handle/10635/134938.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

YANZHOU, ZHANG. “MECHANISM AND IMPLICATIONS OF DESTABILIZING TIP60, A TUMOUR SUPPRESSOR.” 2016. Web. 17 Feb 2020.

Vancouver:

YANZHOU Z. MECHANISM AND IMPLICATIONS OF DESTABILIZING TIP60, A TUMOUR SUPPRESSOR. [Internet] [Thesis]. National University of Singapore; 2016. [cited 2020 Feb 17]. Available from: http://scholarbank.nus.edu.sg/handle/10635/134938.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

YANZHOU Z. MECHANISM AND IMPLICATIONS OF DESTABILIZING TIP60, A TUMOUR SUPPRESSOR. [Thesis]. National University of Singapore; 2016. Available from: http://scholarbank.nus.edu.sg/handle/10635/134938

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. YEO SHU LING, NICOLE. IDENTIFYING CELLULAR TARGETS OF HIGH-RISK HPV ONCOGENES E6 AND E7.

Degree: 2018, National University of Singapore

Subjects/Keywords: Cervical Cancer; Epigenetics; ncRNA; circRNA; TIP60; HPV

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APA (6th Edition):

YEO SHU LING, N. (2018). IDENTIFYING CELLULAR TARGETS OF HIGH-RISK HPV ONCOGENES E6 AND E7. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/151357

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

YEO SHU LING, NICOLE. “IDENTIFYING CELLULAR TARGETS OF HIGH-RISK HPV ONCOGENES E6 AND E7.” 2018. Thesis, National University of Singapore. Accessed February 17, 2020. http://scholarbank.nus.edu.sg/handle/10635/151357.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

YEO SHU LING, NICOLE. “IDENTIFYING CELLULAR TARGETS OF HIGH-RISK HPV ONCOGENES E6 AND E7.” 2018. Web. 17 Feb 2020.

Vancouver:

YEO SHU LING N. IDENTIFYING CELLULAR TARGETS OF HIGH-RISK HPV ONCOGENES E6 AND E7. [Internet] [Thesis]. National University of Singapore; 2018. [cited 2020 Feb 17]. Available from: http://scholarbank.nus.edu.sg/handle/10635/151357.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

YEO SHU LING N. IDENTIFYING CELLULAR TARGETS OF HIGH-RISK HPV ONCOGENES E6 AND E7. [Thesis]. National University of Singapore; 2018. Available from: http://scholarbank.nus.edu.sg/handle/10635/151357

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Georgia State University

25. Elangwe, Emilia N. Site Directed Mutagenesis, Expression and Enzymatic Studies of the 60 kDa Human HIV-TAT 1 Interactive Protein, TIP60.

Degree: MS, Chemistry, 2009, Georgia State University

Tip60 is a 60 kDa nuclear protein which exists in three isoforms, belongs to the MYST/HAT family of proteins and was discovered after its interaction… (more)

Subjects/Keywords: Tip60; Site-directed mutagenesis; HATs; His-tagged Protein Expression; HAT assay; Post-translational modification; Tip60 catalysis; Chromatin modification; Histones; Chromatin; MYST family HATs; Histone acetylation and HAT inhibitors

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APA (6th Edition):

Elangwe, E. N. (2009). Site Directed Mutagenesis, Expression and Enzymatic Studies of the 60 kDa Human HIV-TAT 1 Interactive Protein, TIP60. (Thesis). Georgia State University. Retrieved from https://scholarworks.gsu.edu/chemistry_theses/21

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Elangwe, Emilia N. “Site Directed Mutagenesis, Expression and Enzymatic Studies of the 60 kDa Human HIV-TAT 1 Interactive Protein, TIP60.” 2009. Thesis, Georgia State University. Accessed February 17, 2020. https://scholarworks.gsu.edu/chemistry_theses/21.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Elangwe, Emilia N. “Site Directed Mutagenesis, Expression and Enzymatic Studies of the 60 kDa Human HIV-TAT 1 Interactive Protein, TIP60.” 2009. Web. 17 Feb 2020.

Vancouver:

Elangwe EN. Site Directed Mutagenesis, Expression and Enzymatic Studies of the 60 kDa Human HIV-TAT 1 Interactive Protein, TIP60. [Internet] [Thesis]. Georgia State University; 2009. [cited 2020 Feb 17]. Available from: https://scholarworks.gsu.edu/chemistry_theses/21.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Elangwe EN. Site Directed Mutagenesis, Expression and Enzymatic Studies of the 60 kDa Human HIV-TAT 1 Interactive Protein, TIP60. [Thesis]. Georgia State University; 2009. Available from: https://scholarworks.gsu.edu/chemistry_theses/21

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. Gehrking, Kristin Marie. Partial Tip60 loss slows cerebellar degeneration in a Spinocerebellar Ataxia Type 1 (SCA1) mouse model.

Degree: PhD, Biochemistry, Molecular Biology, and Biophysics, 2009, University of Minnesota

 Spinocerebellar ataxia type 1 (SCA1) is one of nine dominantly inherited neurodegenerative diseases caused by polyglutamine tract expansion. In SCA1, the expanded polyglutamine tract is… (more)

Subjects/Keywords: Ataxin-1; Cerebellum; Neurodegeneration; ROR-alpha; Spinocerebellar Ataxia Type 1; Tip60; Biochemistry, Molecular Bio, and Biophysics

…20 5 Q-RT-PCR of Tip60, in cerebella of C57BL/6 mice comparing Tip60+/+ and Tip60… …ATXN1[82Q]/+:Tip60+/- mice . . . . . . . . . . . . . 44 7 Q-RT-PCR of ATXN1 in… …ATXN1[82Q]/+:Tip60+/- mice and ATXN1[82Q]/+ littermate cerebella… …wt, Tip60+/-, ATXN1[82Q]/+, and ATXN1[82Q]/+:Tip60+/- mice at 9, 12, 16… …and 20 weeks . . . . . . . . . 47 10 CF-PC terminals at 9 weeks in wt, Tip60+/-, ATXN1… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gehrking, K. M. (2009). Partial Tip60 loss slows cerebellar degeneration in a Spinocerebellar Ataxia Type 1 (SCA1) mouse model. (Doctoral Dissertation). University of Minnesota. Retrieved from http://purl.umn.edu/53466

Chicago Manual of Style (16th Edition):

Gehrking, Kristin Marie. “Partial Tip60 loss slows cerebellar degeneration in a Spinocerebellar Ataxia Type 1 (SCA1) mouse model.” 2009. Doctoral Dissertation, University of Minnesota. Accessed February 17, 2020. http://purl.umn.edu/53466.

MLA Handbook (7th Edition):

Gehrking, Kristin Marie. “Partial Tip60 loss slows cerebellar degeneration in a Spinocerebellar Ataxia Type 1 (SCA1) mouse model.” 2009. Web. 17 Feb 2020.

Vancouver:

Gehrking KM. Partial Tip60 loss slows cerebellar degeneration in a Spinocerebellar Ataxia Type 1 (SCA1) mouse model. [Internet] [Doctoral dissertation]. University of Minnesota; 2009. [cited 2020 Feb 17]. Available from: http://purl.umn.edu/53466.

Council of Science Editors:

Gehrking KM. Partial Tip60 loss slows cerebellar degeneration in a Spinocerebellar Ataxia Type 1 (SCA1) mouse model. [Doctoral Dissertation]. University of Minnesota; 2009. Available from: http://purl.umn.edu/53466

.