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You searched for subject:( catenin). Showing records 1 – 30 of 373 total matches.

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Vanderbilt University

1. Yu, Huapeng. p120-catenin controls contractility along the vertical axis of epithelial lateral membranes.

Degree: PhD, Cancer Biology, 2015, Vanderbilt University

 In vertebrate epithelia, p120-catenin mediates E-cadherin stability and suppression of RhoA. Genetic ablation of p120 in various epithelial tissues typically causes striking alterations in tissue… (more)

Subjects/Keywords: contractility; cadherin; catenin

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APA (6th Edition):

Yu, H. (2015). p120-catenin controls contractility along the vertical axis of epithelial lateral membranes. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-12072015-193917/ ;

Chicago Manual of Style (16th Edition):

Yu, Huapeng. “p120-catenin controls contractility along the vertical axis of epithelial lateral membranes.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed September 23, 2019. http://etd.library.vanderbilt.edu/available/etd-12072015-193917/ ;.

MLA Handbook (7th Edition):

Yu, Huapeng. “p120-catenin controls contractility along the vertical axis of epithelial lateral membranes.” 2015. Web. 23 Sep 2019.

Vancouver:

Yu H. p120-catenin controls contractility along the vertical axis of epithelial lateral membranes. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2019 Sep 23]. Available from: http://etd.library.vanderbilt.edu/available/etd-12072015-193917/ ;.

Council of Science Editors:

Yu H. p120-catenin controls contractility along the vertical axis of epithelial lateral membranes. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://etd.library.vanderbilt.edu/available/etd-12072015-193917/ ;


University of Toronto

2. Shipstone, Arun. Analysis of Alpha-catenin Mediated Intercellular Adhesion in Drosophila.

Degree: 2015, University of Toronto

Dynamic linkage between the cadherin-catenin complex (CCC) and the actin cytoskeleton at Adherens Junctions is essential for cell-cell adhesion in epithelial cells. Alpha-catenin is a… (more)

Subjects/Keywords: alpha-catenin; Beta-catenin; catenin; cell adhesion; Drosophila; E-Cadherin; 0379

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APA (6th Edition):

Shipstone, A. (2015). Analysis of Alpha-catenin Mediated Intercellular Adhesion in Drosophila. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/88582

Chicago Manual of Style (16th Edition):

Shipstone, Arun. “Analysis of Alpha-catenin Mediated Intercellular Adhesion in Drosophila.” 2015. Masters Thesis, University of Toronto. Accessed September 23, 2019. http://hdl.handle.net/1807/88582.

MLA Handbook (7th Edition):

Shipstone, Arun. “Analysis of Alpha-catenin Mediated Intercellular Adhesion in Drosophila.” 2015. Web. 23 Sep 2019.

Vancouver:

Shipstone A. Analysis of Alpha-catenin Mediated Intercellular Adhesion in Drosophila. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2019 Sep 23]. Available from: http://hdl.handle.net/1807/88582.

Council of Science Editors:

Shipstone A. Analysis of Alpha-catenin Mediated Intercellular Adhesion in Drosophila. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/88582


University of Western Australia

3. Sng, Joo Li Natasha. The role of β-catenin in activated EGFR-driven lung tumourigenesis.

Degree: M.Med.Sc., 2011, University of Western Australia

Lung cancer is associated with a high mortality rate, with Non-Small Cell Lung Cancer (NSCLC) accounting for the majority of cases. Mutations in the epidermal… (more)

Subjects/Keywords: EGFR; Lung tumour; Beta-catenin

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APA (6th Edition):

Sng, J. L. N. (2011). The role of β-catenin in activated EGFR-driven lung tumourigenesis. (Masters Thesis). University of Western Australia. Retrieved from http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=32218&local_base=GEN01-INS01

Chicago Manual of Style (16th Edition):

Sng, Joo Li Natasha. “The role of β-catenin in activated EGFR-driven lung tumourigenesis.” 2011. Masters Thesis, University of Western Australia. Accessed September 23, 2019. http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=32218&local_base=GEN01-INS01.

MLA Handbook (7th Edition):

Sng, Joo Li Natasha. “The role of β-catenin in activated EGFR-driven lung tumourigenesis.” 2011. Web. 23 Sep 2019.

Vancouver:

Sng JLN. The role of β-catenin in activated EGFR-driven lung tumourigenesis. [Internet] [Masters thesis]. University of Western Australia; 2011. [cited 2019 Sep 23]. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=32218&local_base=GEN01-INS01.

Council of Science Editors:

Sng JLN. The role of β-catenin in activated EGFR-driven lung tumourigenesis. [Masters Thesis]. University of Western Australia; 2011. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=32218&local_base=GEN01-INS01

4. 神宮司, 健太郎. DIF-1 inhibits the Wnt/β-catenin signaling pathway by inhibiting TCF7L2 expression in colon cancer cell lines : DIF-1は大腸がん細胞株においてTCF7L2の発現を抑制することにより、Wnt/β-cateninシグナル伝達経路を阻害する.

Degree: 博士(医学), 2013, Kyushu University / 九州大学

我々はこれまでに細胞性粘菌Dictyostelium discoideumが分泌する分化誘導因子Differentiation-inducing factor-1(DIF-1)がβ-cateninタンパク質の分解を誘導することでWnt/β-cateninシグナル伝達経路を阻害し、ヒトがん細胞株の増殖を抑制することを報告してきた。β-cateninタンパク質の分解がDIF-1の効果に不可欠であるか否かを明らかにするために、我々はWnt/β-cateninシグナル伝達経路が恒常的に活性化されているヒト由来大腸がん細胞株(HCT-116、SW-620、DLD-1)に対するDIF-1の効果を検討した。DIF-1はβ-cateninタンパク質の分解非依存性にcyclin D1の発現をmRNA、タンパク質レベル共に減少させ、G_0/G_1期における細胞周期拘束を起こすことにより、強力に細胞増殖を抑制した。DIF-1によるtranscription factor 7-like 2(TCF7L2)発現量の抑制によって、TCF依存性転写活性及びcyclin D1プロモーター活性が抑制されることが明らかとなった。ルシフェラーゼレポーター活性測定及びTCF7L2プロモーター断片を用いたEMSAにより、翻訳開始点を起点として-609から-601塩基上流に位置する転写因子early growth response-1(Egr-1)結合部位が、DIF-1の効果に関与していることが示された。さらに、RNAi法による内在性TCF7L2の欠失はcyclin D1プロモーター活性及びタンパク質量の減少につながり、そして、TCF7L2の強制発現はDIF-1によるTCF依存性転写活性及びcyclin D1プロモーター活性の低下効果を減弱させた。したがって、DIF-1は大腸がん細胞株において、Egr-1依存性のTCF7L2転写活性を減少させることによりTCF7L2発現を抑制し、Wnt/β-cateninシグナル伝達経路を阻害していることが示唆された。我々の結果は、DIF-1によるWnt/β-cateninシグナル伝達経路の阻害機構に新たな知見をもたらすものである。

We previously reported that differentiation-inducing factor-1 (DIF-1), a morphogen in Dictyostelium discoideum, inhibits… (more)

Subjects/Keywords: DIF-1; Wnt/β-catenin

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APA (6th Edition):

神宮司, . (2013). DIF-1 inhibits the Wnt/β-catenin signaling pathway by inhibiting TCF7L2 expression in colon cancer cell lines : DIF-1は大腸がん細胞株においてTCF7L2の発現を抑制することにより、Wnt/β-cateninシグナル伝達経路を阻害する. (Thesis). Kyushu University / 九州大学. Retrieved from http://hdl.handle.net/2324/21727 ; http://dx.doi.org/10.15017/21727

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

神宮司, 健太郎. “DIF-1 inhibits the Wnt/β-catenin signaling pathway by inhibiting TCF7L2 expression in colon cancer cell lines : DIF-1は大腸がん細胞株においてTCF7L2の発現を抑制することにより、Wnt/β-cateninシグナル伝達経路を阻害する.” 2013. Thesis, Kyushu University / 九州大学. Accessed September 23, 2019. http://hdl.handle.net/2324/21727 ; http://dx.doi.org/10.15017/21727.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

神宮司, 健太郎. “DIF-1 inhibits the Wnt/β-catenin signaling pathway by inhibiting TCF7L2 expression in colon cancer cell lines : DIF-1は大腸がん細胞株においてTCF7L2の発現を抑制することにより、Wnt/β-cateninシグナル伝達経路を阻害する.” 2013. Web. 23 Sep 2019.

Vancouver:

神宮司 . DIF-1 inhibits the Wnt/β-catenin signaling pathway by inhibiting TCF7L2 expression in colon cancer cell lines : DIF-1は大腸がん細胞株においてTCF7L2の発現を抑制することにより、Wnt/β-cateninシグナル伝達経路を阻害する. [Internet] [Thesis]. Kyushu University / 九州大学; 2013. [cited 2019 Sep 23]. Available from: http://hdl.handle.net/2324/21727 ; http://dx.doi.org/10.15017/21727.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

神宮司 . DIF-1 inhibits the Wnt/β-catenin signaling pathway by inhibiting TCF7L2 expression in colon cancer cell lines : DIF-1は大腸がん細胞株においてTCF7L2の発現を抑制することにより、Wnt/β-cateninシグナル伝達経路を阻害する. [Thesis]. Kyushu University / 九州大学; 2013. Available from: http://hdl.handle.net/2324/21727 ; http://dx.doi.org/10.15017/21727

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McMaster University

5. Abdulla, Solen. INVESTIGATING NOVEL β-CATENIN SIGNALLING MECHANISMS IN AN EMBRYONIC STEM CELL MODEL.

Degree: MSc, 2017, McMaster University

The Wnt/β-catenin pathway is a fundamental regulator of embryonic development and adult tissue homeostasis. The key effector, β-catenin, is a multifunctional protein that occupies dual… (more)

Subjects/Keywords: β-catenin; Stem Cells

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APA (6th Edition):

Abdulla, S. (2017). INVESTIGATING NOVEL β-CATENIN SIGNALLING MECHANISMS IN AN EMBRYONIC STEM CELL MODEL. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/23461

Chicago Manual of Style (16th Edition):

Abdulla, Solen. “INVESTIGATING NOVEL β-CATENIN SIGNALLING MECHANISMS IN AN EMBRYONIC STEM CELL MODEL.” 2017. Masters Thesis, McMaster University. Accessed September 23, 2019. http://hdl.handle.net/11375/23461.

MLA Handbook (7th Edition):

Abdulla, Solen. “INVESTIGATING NOVEL β-CATENIN SIGNALLING MECHANISMS IN AN EMBRYONIC STEM CELL MODEL.” 2017. Web. 23 Sep 2019.

Vancouver:

Abdulla S. INVESTIGATING NOVEL β-CATENIN SIGNALLING MECHANISMS IN AN EMBRYONIC STEM CELL MODEL. [Internet] [Masters thesis]. McMaster University; 2017. [cited 2019 Sep 23]. Available from: http://hdl.handle.net/11375/23461.

Council of Science Editors:

Abdulla S. INVESTIGATING NOVEL β-CATENIN SIGNALLING MECHANISMS IN AN EMBRYONIC STEM CELL MODEL. [Masters Thesis]. McMaster University; 2017. Available from: http://hdl.handle.net/11375/23461

6. Marcato, Vasco. La β-caténine : un activateur de l’expression du gène codant pour l’interféron-béta et une cible du Virus de la Fièvre de la Vallée du Rift : Magnetismo cooperativo en compuestos de coordinación basados en Cu(II), Ni(II) y Co(II) con ligandos imidazol carboxílicos.

Degree: Docteur es, Génétique, 2015, Sorbonne Paris Cité

La réponse antivirale innée constitue la première réaction d’une cellule à une infection virale. Il s’agit d’une réponse rapide, transitoire, non-spécifique, ubiquitaire qui a été… (more)

Subjects/Keywords: Β-caténine; Β-catenin; 616.042

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APA (6th Edition):

Marcato, V. (2015). La β-caténine : un activateur de l’expression du gène codant pour l’interféron-béta et une cible du Virus de la Fièvre de la Vallée du Rift : Magnetismo cooperativo en compuestos de coordinación basados en Cu(II), Ni(II) y Co(II) con ligandos imidazol carboxílicos. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2015USPCB103

Chicago Manual of Style (16th Edition):

Marcato, Vasco. “La β-caténine : un activateur de l’expression du gène codant pour l’interféron-béta et une cible du Virus de la Fièvre de la Vallée du Rift : Magnetismo cooperativo en compuestos de coordinación basados en Cu(II), Ni(II) y Co(II) con ligandos imidazol carboxílicos.” 2015. Doctoral Dissertation, Sorbonne Paris Cité. Accessed September 23, 2019. http://www.theses.fr/2015USPCB103.

MLA Handbook (7th Edition):

Marcato, Vasco. “La β-caténine : un activateur de l’expression du gène codant pour l’interféron-béta et une cible du Virus de la Fièvre de la Vallée du Rift : Magnetismo cooperativo en compuestos de coordinación basados en Cu(II), Ni(II) y Co(II) con ligandos imidazol carboxílicos.” 2015. Web. 23 Sep 2019.

Vancouver:

Marcato V. La β-caténine : un activateur de l’expression du gène codant pour l’interféron-béta et une cible du Virus de la Fièvre de la Vallée du Rift : Magnetismo cooperativo en compuestos de coordinación basados en Cu(II), Ni(II) y Co(II) con ligandos imidazol carboxílicos. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2015. [cited 2019 Sep 23]. Available from: http://www.theses.fr/2015USPCB103.

Council of Science Editors:

Marcato V. La β-caténine : un activateur de l’expression du gène codant pour l’interféron-béta et une cible du Virus de la Fièvre de la Vallée du Rift : Magnetismo cooperativo en compuestos de coordinación basados en Cu(II), Ni(II) y Co(II) con ligandos imidazol carboxílicos. [Doctoral Dissertation]. Sorbonne Paris Cité; 2015. Available from: http://www.theses.fr/2015USPCB103


Vanderbilt University

7. Markham, Nicholas Owen. DIPA is a novel, isoform-specific binding partner of the tumor-associated p120 isoform 1.

Degree: PhD, Cancer Biology, 2012, Vanderbilt University

 p120-catenin (p120) is a master regulator of cellular adherens junctions and is important for epithelial homeostasis, development, tumorigenesis, and metastasis. Relatively little is known about… (more)

Subjects/Keywords: adherens junctions; DIPA; p120-catenin

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APA (6th Edition):

Markham, N. O. (2012). DIPA is a novel, isoform-specific binding partner of the tumor-associated p120 isoform 1. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu//available/etd-07192012-173715/ ;

Chicago Manual of Style (16th Edition):

Markham, Nicholas Owen. “DIPA is a novel, isoform-specific binding partner of the tumor-associated p120 isoform 1.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed September 23, 2019. http://etd.library.vanderbilt.edu//available/etd-07192012-173715/ ;.

MLA Handbook (7th Edition):

Markham, Nicholas Owen. “DIPA is a novel, isoform-specific binding partner of the tumor-associated p120 isoform 1.” 2012. Web. 23 Sep 2019.

Vancouver:

Markham NO. DIPA is a novel, isoform-specific binding partner of the tumor-associated p120 isoform 1. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2019 Sep 23]. Available from: http://etd.library.vanderbilt.edu//available/etd-07192012-173715/ ;.

Council of Science Editors:

Markham NO. DIPA is a novel, isoform-specific binding partner of the tumor-associated p120 isoform 1. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://etd.library.vanderbilt.edu//available/etd-07192012-173715/ ;


Colorado State University

8. Nip, Kaila Akemi Ka`ohinani. AUTISM-ASSOCIATED δ-CATENIN G34S MUTATION PROMOTES GSK3β-MEDIATED PREMATURE δ-CATENIN DEGRADATION INDUCING NEURONAL DYSFUNCTION.

Degree: MS(M.S.), Cell and Molecular Biology (Graduate Degree Program), 2019, Colorado State University

 δ-catenin is a crucial component of a synaptic scaffolding complex, which regulates synaptic structure and function in neurons. Loss of δ-catenin function is strongly associated… (more)

Subjects/Keywords: delta-catenin; autism; GSK3β

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APA (6th Edition):

Nip, K. A. K. (2019). AUTISM-ASSOCIATED δ-CATENIN G34S MUTATION PROMOTES GSK3β-MEDIATED PREMATURE δ-CATENIN DEGRADATION INDUCING NEURONAL DYSFUNCTION. (Masters Thesis). Colorado State University. Retrieved from http://hdl.handle.net/10217/195416

Chicago Manual of Style (16th Edition):

Nip, Kaila Akemi Ka`ohinani. “AUTISM-ASSOCIATED δ-CATENIN G34S MUTATION PROMOTES GSK3β-MEDIATED PREMATURE δ-CATENIN DEGRADATION INDUCING NEURONAL DYSFUNCTION.” 2019. Masters Thesis, Colorado State University. Accessed September 23, 2019. http://hdl.handle.net/10217/195416.

MLA Handbook (7th Edition):

Nip, Kaila Akemi Ka`ohinani. “AUTISM-ASSOCIATED δ-CATENIN G34S MUTATION PROMOTES GSK3β-MEDIATED PREMATURE δ-CATENIN DEGRADATION INDUCING NEURONAL DYSFUNCTION.” 2019. Web. 23 Sep 2019.

Vancouver:

Nip KAK. AUTISM-ASSOCIATED δ-CATENIN G34S MUTATION PROMOTES GSK3β-MEDIATED PREMATURE δ-CATENIN DEGRADATION INDUCING NEURONAL DYSFUNCTION. [Internet] [Masters thesis]. Colorado State University; 2019. [cited 2019 Sep 23]. Available from: http://hdl.handle.net/10217/195416.

Council of Science Editors:

Nip KAK. AUTISM-ASSOCIATED δ-CATENIN G34S MUTATION PROMOTES GSK3β-MEDIATED PREMATURE δ-CATENIN DEGRADATION INDUCING NEURONAL DYSFUNCTION. [Masters Thesis]. Colorado State University; 2019. Available from: http://hdl.handle.net/10217/195416


Universitat Pompeu Fabra

9. Aulicino, Francesco, 1987-. Investigating the role of Wnt/β-catenin pathway in pluripotency and somatic cell reprogramming.

Degree: Departament de Ciències Experimentals i de la Salut, 2016, Universitat Pompeu Fabra

 La respuesta adaptativa de las células a estímulos externos es un mecanismo fundamental de la existencia de la vida en sí misma. Para este fin,… (more)

Subjects/Keywords: Wnt/ß-catenin; Pluripotency; Reprogramming; ß-catenin; TCF1; Pluripotencia; Reprogramación; 576

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APA (6th Edition):

Aulicino, Francesco, 1. (2016). Investigating the role of Wnt/β-catenin pathway in pluripotency and somatic cell reprogramming. (Thesis). Universitat Pompeu Fabra. Retrieved from http://hdl.handle.net/10803/552942

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Aulicino, Francesco, 1987-. “Investigating the role of Wnt/β-catenin pathway in pluripotency and somatic cell reprogramming.” 2016. Thesis, Universitat Pompeu Fabra. Accessed September 23, 2019. http://hdl.handle.net/10803/552942.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Aulicino, Francesco, 1987-. “Investigating the role of Wnt/β-catenin pathway in pluripotency and somatic cell reprogramming.” 2016. Web. 23 Sep 2019.

Vancouver:

Aulicino, Francesco 1. Investigating the role of Wnt/β-catenin pathway in pluripotency and somatic cell reprogramming. [Internet] [Thesis]. Universitat Pompeu Fabra; 2016. [cited 2019 Sep 23]. Available from: http://hdl.handle.net/10803/552942.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Aulicino, Francesco 1. Investigating the role of Wnt/β-catenin pathway in pluripotency and somatic cell reprogramming. [Thesis]. Universitat Pompeu Fabra; 2016. Available from: http://hdl.handle.net/10803/552942

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

10. Chiang, Chi-Hsiang. Regulation of microRNA-182 and its functional role in breast cancer.

Degree: PhD, Institute of Biomedical Sciences, 2013, NSYSU

 MicroRNAs (MiRNAs) are endogenous small non-coding RNAs which negatively regulate gene expression by inducing translation repression or mRNA cleavage. MiR-182 is a member of the… (more)

Subjects/Keywords: RECK; β-catenin; microRNA; matrix metalloproteinase

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APA (6th Edition):

Chiang, C. (2013). Regulation of microRNA-182 and its functional role in breast cancer. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0624113-191725

Chicago Manual of Style (16th Edition):

Chiang, Chi-Hsiang. “Regulation of microRNA-182 and its functional role in breast cancer.” 2013. Doctoral Dissertation, NSYSU. Accessed September 23, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0624113-191725.

MLA Handbook (7th Edition):

Chiang, Chi-Hsiang. “Regulation of microRNA-182 and its functional role in breast cancer.” 2013. Web. 23 Sep 2019.

Vancouver:

Chiang C. Regulation of microRNA-182 and its functional role in breast cancer. [Internet] [Doctoral dissertation]. NSYSU; 2013. [cited 2019 Sep 23]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0624113-191725.

Council of Science Editors:

Chiang C. Regulation of microRNA-182 and its functional role in breast cancer. [Doctoral Dissertation]. NSYSU; 2013. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0624113-191725


Universiteit Utrecht

11. Pustjens, M.F. α-catenin in the adherens junction complex and the possible implications in cancer.

Degree: 2012, Universiteit Utrecht

 The most common malignancy in women of the Western World is breast cancer, which develops from mammary gland epithelium. 30-40% of the patients develop metastatic… (more)

Subjects/Keywords: alpha-catenin; adherens junctions; breast cancer

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APA (6th Edition):

Pustjens, M. F. (2012). α-catenin in the adherens junction complex and the possible implications in cancer. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/250410

Chicago Manual of Style (16th Edition):

Pustjens, M F. “α-catenin in the adherens junction complex and the possible implications in cancer.” 2012. Masters Thesis, Universiteit Utrecht. Accessed September 23, 2019. http://dspace.library.uu.nl:8080/handle/1874/250410.

MLA Handbook (7th Edition):

Pustjens, M F. “α-catenin in the adherens junction complex and the possible implications in cancer.” 2012. Web. 23 Sep 2019.

Vancouver:

Pustjens MF. α-catenin in the adherens junction complex and the possible implications in cancer. [Internet] [Masters thesis]. Universiteit Utrecht; 2012. [cited 2019 Sep 23]. Available from: http://dspace.library.uu.nl:8080/handle/1874/250410.

Council of Science Editors:

Pustjens MF. α-catenin in the adherens junction complex and the possible implications in cancer. [Masters Thesis]. Universiteit Utrecht; 2012. Available from: http://dspace.library.uu.nl:8080/handle/1874/250410


University of Manchester

12. Giles, Adam Alexander. Wnt signalling in oestrogen-induced lactotroph proliferation.

Degree: PhD, 2011, University of Manchester

 The anterior pituitary gland is the major hormonal regulator in the body. The gland contains five secretory cell types whose emergence during development is defined… (more)

Subjects/Keywords: 616.4; Wnt; Pituitary; Lactotroph; Beta-Catenin; Prolactin

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Giles, A. A. (2011). Wnt signalling in oestrogen-induced lactotroph proliferation. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/wnt-signalling-in-oestrogeninduced-lactotroph-proliferation(35c1ba30-0755-4583-bdce-69dce1382721).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.538396

Chicago Manual of Style (16th Edition):

Giles, Adam Alexander. “Wnt signalling in oestrogen-induced lactotroph proliferation.” 2011. Doctoral Dissertation, University of Manchester. Accessed September 23, 2019. https://www.research.manchester.ac.uk/portal/en/theses/wnt-signalling-in-oestrogeninduced-lactotroph-proliferation(35c1ba30-0755-4583-bdce-69dce1382721).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.538396.

MLA Handbook (7th Edition):

Giles, Adam Alexander. “Wnt signalling in oestrogen-induced lactotroph proliferation.” 2011. Web. 23 Sep 2019.

Vancouver:

Giles AA. Wnt signalling in oestrogen-induced lactotroph proliferation. [Internet] [Doctoral dissertation]. University of Manchester; 2011. [cited 2019 Sep 23]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/wnt-signalling-in-oestrogeninduced-lactotroph-proliferation(35c1ba30-0755-4583-bdce-69dce1382721).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.538396.

Council of Science Editors:

Giles AA. Wnt signalling in oestrogen-induced lactotroph proliferation. [Doctoral Dissertation]. University of Manchester; 2011. Available from: https://www.research.manchester.ac.uk/portal/en/theses/wnt-signalling-in-oestrogeninduced-lactotroph-proliferation(35c1ba30-0755-4583-bdce-69dce1382721).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.538396


University of Manchester

13. Hidalgo Sastre, Ana. Crosstalk between Notch and Wnt signalling pathways in vertebrates.

Degree: PhD, 2012, University of Manchester

 The development of complex metazoans depends on the integration of a handful of signalling pathways that eventually modulate precise patterns of gene expression. The fact… (more)

Subjects/Keywords: 612.015; Notch; Wnt; ß-catenin; crosstalk; inhibition

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hidalgo Sastre, A. (2012). Crosstalk between Notch and Wnt signalling pathways in vertebrates. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/crosstalk-between-notch-and-wnt-signalling-pathways-in-vertebrates(9b4411a3-cd03-4af3-b3b5-8c432c7a2c68).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559348

Chicago Manual of Style (16th Edition):

Hidalgo Sastre, Ana. “Crosstalk between Notch and Wnt signalling pathways in vertebrates.” 2012. Doctoral Dissertation, University of Manchester. Accessed September 23, 2019. https://www.research.manchester.ac.uk/portal/en/theses/crosstalk-between-notch-and-wnt-signalling-pathways-in-vertebrates(9b4411a3-cd03-4af3-b3b5-8c432c7a2c68).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559348.

MLA Handbook (7th Edition):

Hidalgo Sastre, Ana. “Crosstalk between Notch and Wnt signalling pathways in vertebrates.” 2012. Web. 23 Sep 2019.

Vancouver:

Hidalgo Sastre A. Crosstalk between Notch and Wnt signalling pathways in vertebrates. [Internet] [Doctoral dissertation]. University of Manchester; 2012. [cited 2019 Sep 23]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/crosstalk-between-notch-and-wnt-signalling-pathways-in-vertebrates(9b4411a3-cd03-4af3-b3b5-8c432c7a2c68).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559348.

Council of Science Editors:

Hidalgo Sastre A. Crosstalk between Notch and Wnt signalling pathways in vertebrates. [Doctoral Dissertation]. University of Manchester; 2012. Available from: https://www.research.manchester.ac.uk/portal/en/theses/crosstalk-between-notch-and-wnt-signalling-pathways-in-vertebrates(9b4411a3-cd03-4af3-b3b5-8c432c7a2c68).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559348


University of Southern California

14. Wu, Jia. Wnt/β-catenin/p300 induced transcription is critical for the differentiation and maintenance of Paneth cells.

Degree: MS, Biochemistry and Molecular Biology, 2014, University of Southern California

 Our lab's previous studies have demonstrated that Wnt/β‐catenin/p300 transcription initiates cell differentiation, whereas Wnt/β‐catenin/cyclic AMP‐responsive element binding protein‐binding protein (CBP) transcription mediates cell proliferation/maintenance of… (more)

Subjects/Keywords: Wnt; β ‐Catenin/p300; Paneth cell

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wu, J. (2014). Wnt/β-catenin/p300 induced transcription is critical for the differentiation and maintenance of Paneth cells. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/405481/rec/7947

Chicago Manual of Style (16th Edition):

Wu, Jia. “Wnt/β-catenin/p300 induced transcription is critical for the differentiation and maintenance of Paneth cells.” 2014. Masters Thesis, University of Southern California. Accessed September 23, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/405481/rec/7947.

MLA Handbook (7th Edition):

Wu, Jia. “Wnt/β-catenin/p300 induced transcription is critical for the differentiation and maintenance of Paneth cells.” 2014. Web. 23 Sep 2019.

Vancouver:

Wu J. Wnt/β-catenin/p300 induced transcription is critical for the differentiation and maintenance of Paneth cells. [Internet] [Masters thesis]. University of Southern California; 2014. [cited 2019 Sep 23]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/405481/rec/7947.

Council of Science Editors:

Wu J. Wnt/β-catenin/p300 induced transcription is critical for the differentiation and maintenance of Paneth cells. [Masters Thesis]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/405481/rec/7947


University of Illinois – Chicago

15. Vandenbroucke St Amant, Emily E. The Role of PKCAlpha-Mediated p120-Catenin Phosphorylation on Endothelial Permeability.

Degree: 2013, University of Illinois – Chicago

 Adherens junctions are the primary cell-cell junction that maintains the integrity of the endothelium. Vascular leakage resulting from inflammation contributes to a variety of pathological… (more)

Subjects/Keywords: endothelial permeability; p120-catenin; Serine 879; PKCalpha

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APA (6th Edition):

Vandenbroucke St Amant, E. E. (2013). The Role of PKCAlpha-Mediated p120-Catenin Phosphorylation on Endothelial Permeability. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/9728

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vandenbroucke St Amant, Emily E. “The Role of PKCAlpha-Mediated p120-Catenin Phosphorylation on Endothelial Permeability.” 2013. Thesis, University of Illinois – Chicago. Accessed September 23, 2019. http://hdl.handle.net/10027/9728.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vandenbroucke St Amant, Emily E. “The Role of PKCAlpha-Mediated p120-Catenin Phosphorylation on Endothelial Permeability.” 2013. Web. 23 Sep 2019.

Vancouver:

Vandenbroucke St Amant EE. The Role of PKCAlpha-Mediated p120-Catenin Phosphorylation on Endothelial Permeability. [Internet] [Thesis]. University of Illinois – Chicago; 2013. [cited 2019 Sep 23]. Available from: http://hdl.handle.net/10027/9728.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vandenbroucke St Amant EE. The Role of PKCAlpha-Mediated p120-Catenin Phosphorylation on Endothelial Permeability. [Thesis]. University of Illinois – Chicago; 2013. Available from: http://hdl.handle.net/10027/9728

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Chicago

16. Pham, Thao. Protein Kinase C Alpha Mediates FOXC2 Transcriptional Repression Of p120-Catenin In Human Breast Cancer.

Degree: 2016, University of Illinois – Chicago

 Breast cancer is the most commonly diagnosed malignancy in women worldwide. Despite recent advances in prevention, diagnosis, and treatment, metastasis remains a roadblock to successful… (more)

Subjects/Keywords: PKC; p120-catenin; FOXC2; breast cancer

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APA (6th Edition):

Pham, T. (2016). Protein Kinase C Alpha Mediates FOXC2 Transcriptional Repression Of p120-Catenin In Human Breast Cancer. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/21604

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pham, Thao. “Protein Kinase C Alpha Mediates FOXC2 Transcriptional Repression Of p120-Catenin In Human Breast Cancer.” 2016. Thesis, University of Illinois – Chicago. Accessed September 23, 2019. http://hdl.handle.net/10027/21604.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pham, Thao. “Protein Kinase C Alpha Mediates FOXC2 Transcriptional Repression Of p120-Catenin In Human Breast Cancer.” 2016. Web. 23 Sep 2019.

Vancouver:

Pham T. Protein Kinase C Alpha Mediates FOXC2 Transcriptional Repression Of p120-Catenin In Human Breast Cancer. [Internet] [Thesis]. University of Illinois – Chicago; 2016. [cited 2019 Sep 23]. Available from: http://hdl.handle.net/10027/21604.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pham T. Protein Kinase C Alpha Mediates FOXC2 Transcriptional Repression Of p120-Catenin In Human Breast Cancer. [Thesis]. University of Illinois – Chicago; 2016. Available from: http://hdl.handle.net/10027/21604

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Washington

17. Conrad, William. Resolving mechanisms of apoptosis in response to WNT3A in Melanoma.

Degree: PhD, 2013, University of Washington

 Wnt/β-catenin and ERK/MAPK signaling regulate the balance of differentiation and proliferation in melanoma. Aberrant activation of ERK/MAPK signaling results from the BRAFV600E or NRASQ61X mutations… (more)

Subjects/Keywords: BRAF; catenin; FAM129B; NRAS; WNT; Pharmacology; pharmacology

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APA (6th Edition):

Conrad, W. (2013). Resolving mechanisms of apoptosis in response to WNT3A in Melanoma. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/22448

Chicago Manual of Style (16th Edition):

Conrad, William. “Resolving mechanisms of apoptosis in response to WNT3A in Melanoma.” 2013. Doctoral Dissertation, University of Washington. Accessed September 23, 2019. http://hdl.handle.net/1773/22448.

MLA Handbook (7th Edition):

Conrad, William. “Resolving mechanisms of apoptosis in response to WNT3A in Melanoma.” 2013. Web. 23 Sep 2019.

Vancouver:

Conrad W. Resolving mechanisms of apoptosis in response to WNT3A in Melanoma. [Internet] [Doctoral dissertation]. University of Washington; 2013. [cited 2019 Sep 23]. Available from: http://hdl.handle.net/1773/22448.

Council of Science Editors:

Conrad W. Resolving mechanisms of apoptosis in response to WNT3A in Melanoma. [Doctoral Dissertation]. University of Washington; 2013. Available from: http://hdl.handle.net/1773/22448


University of Manchester

18. Giles, Adam Alexander. Wnt signalling in oestrogen-induced lactotroph proliferation.

Degree: 2011, University of Manchester

 The University of ManchesterAdam GilesDoctor of Philosophy – PhDWnt signalling in oestrogen-induced lactotroph hyperplasia2011The anterior pituitary gland is the major hormonal regulator in the body.… (more)

Subjects/Keywords: Wnt; Pituitary; Lactotroph; Beta-Catenin; Prolactin

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Giles, A. A. (2011). Wnt signalling in oestrogen-induced lactotroph proliferation. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:121389

Chicago Manual of Style (16th Edition):

Giles, Adam Alexander. “Wnt signalling in oestrogen-induced lactotroph proliferation.” 2011. Doctoral Dissertation, University of Manchester. Accessed September 23, 2019. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:121389.

MLA Handbook (7th Edition):

Giles, Adam Alexander. “Wnt signalling in oestrogen-induced lactotroph proliferation.” 2011. Web. 23 Sep 2019.

Vancouver:

Giles AA. Wnt signalling in oestrogen-induced lactotroph proliferation. [Internet] [Doctoral dissertation]. University of Manchester; 2011. [cited 2019 Sep 23]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:121389.

Council of Science Editors:

Giles AA. Wnt signalling in oestrogen-induced lactotroph proliferation. [Doctoral Dissertation]. University of Manchester; 2011. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:121389


Boston University

19. Gowda, Asha. The effect of protein kinase C and Beta-catenin inhibitors on uveal melanoma cells.

Degree: MS, Medical Sciences, 2014, Boston University

 PURPOSE: Uveal melanoma (UM) is the most common intraocular malignancy in adults with an incidence of six per one million individuals each year. Globe conserving… (more)

Subjects/Keywords: Ophthalmology; Beta-catenin; Melanoma; Protein kinase C

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APA (6th Edition):

Gowda, A. (2014). The effect of protein kinase C and Beta-catenin inhibitors on uveal melanoma cells. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/15046

Chicago Manual of Style (16th Edition):

Gowda, Asha. “The effect of protein kinase C and Beta-catenin inhibitors on uveal melanoma cells.” 2014. Masters Thesis, Boston University. Accessed September 23, 2019. http://hdl.handle.net/2144/15046.

MLA Handbook (7th Edition):

Gowda, Asha. “The effect of protein kinase C and Beta-catenin inhibitors on uveal melanoma cells.” 2014. Web. 23 Sep 2019.

Vancouver:

Gowda A. The effect of protein kinase C and Beta-catenin inhibitors on uveal melanoma cells. [Internet] [Masters thesis]. Boston University; 2014. [cited 2019 Sep 23]. Available from: http://hdl.handle.net/2144/15046.

Council of Science Editors:

Gowda A. The effect of protein kinase C and Beta-catenin inhibitors on uveal melanoma cells. [Masters Thesis]. Boston University; 2014. Available from: http://hdl.handle.net/2144/15046

20. Manring, Heather Renee. COMPARATIVE CELL BASED FUNCTIONAL ANALYSIS OF KEY WNT/BETA-CATENIN PATHWAY GENES AND GENE MUTATIONS.

Degree: 2012, Wake Forest University

 The Wnt/Beta-catenin signaling pathway has many roles including regulation of developmental pathways, cell proliferation, and homeostasis of adult tissues. Wnt/Beta-catenin signaling activity depends on the… (more)

Subjects/Keywords: Beta-catenin

…OF FIGURES Page Figure 1.1. Schematic of the Wnt/β-catenin pathway in the inactive (A… …3 Figure 1.2. β-catenin function is partially mediated by its location within the cell… …and interaction domains for proteins of the Wnt/β-catenin pathway and other signaling… …pathways. .......... 8 Figure 1.4. Mechanisms of regulating Wnt/β-catenin signaling activity… …22 Figure 2.1. Increased β-catenin protein expression and β-catenin mediated transcription… 

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APA (6th Edition):

Manring, H. R. (2012). COMPARATIVE CELL BASED FUNCTIONAL ANALYSIS OF KEY WNT/BETA-CATENIN PATHWAY GENES AND GENE MUTATIONS. (Thesis). Wake Forest University. Retrieved from http://hdl.handle.net/10339/37663

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Manring, Heather Renee. “COMPARATIVE CELL BASED FUNCTIONAL ANALYSIS OF KEY WNT/BETA-CATENIN PATHWAY GENES AND GENE MUTATIONS.” 2012. Thesis, Wake Forest University. Accessed September 23, 2019. http://hdl.handle.net/10339/37663.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Manring, Heather Renee. “COMPARATIVE CELL BASED FUNCTIONAL ANALYSIS OF KEY WNT/BETA-CATENIN PATHWAY GENES AND GENE MUTATIONS.” 2012. Web. 23 Sep 2019.

Vancouver:

Manring HR. COMPARATIVE CELL BASED FUNCTIONAL ANALYSIS OF KEY WNT/BETA-CATENIN PATHWAY GENES AND GENE MUTATIONS. [Internet] [Thesis]. Wake Forest University; 2012. [cited 2019 Sep 23]. Available from: http://hdl.handle.net/10339/37663.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Manring HR. COMPARATIVE CELL BASED FUNCTIONAL ANALYSIS OF KEY WNT/BETA-CATENIN PATHWAY GENES AND GENE MUTATIONS. [Thesis]. Wake Forest University; 2012. Available from: http://hdl.handle.net/10339/37663

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

21. Dimitrova, Yoana Nantcheva. Biochemical and structural analyses of TBL1: insights into the function of a transcriptional regulator.

Degree: PhD, Biochemistry, 2010, Vanderbilt University

 The mechanism controlling the switch between gene activation and repression is critically important for understanding the process of transcriptional regulation. Gene expression is highly controlled… (more)

Subjects/Keywords: transcriptional regulation; ubiquitination; beta-catenin; TBL1

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APA (6th Edition):

Dimitrova, Y. N. (2010). Biochemical and structural analyses of TBL1: insights into the function of a transcriptional regulator. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-10282010-173834/ ;

Chicago Manual of Style (16th Edition):

Dimitrova, Yoana Nantcheva. “Biochemical and structural analyses of TBL1: insights into the function of a transcriptional regulator.” 2010. Doctoral Dissertation, Vanderbilt University. Accessed September 23, 2019. http://etd.library.vanderbilt.edu/available/etd-10282010-173834/ ;.

MLA Handbook (7th Edition):

Dimitrova, Yoana Nantcheva. “Biochemical and structural analyses of TBL1: insights into the function of a transcriptional regulator.” 2010. Web. 23 Sep 2019.

Vancouver:

Dimitrova YN. Biochemical and structural analyses of TBL1: insights into the function of a transcriptional regulator. [Internet] [Doctoral dissertation]. Vanderbilt University; 2010. [cited 2019 Sep 23]. Available from: http://etd.library.vanderbilt.edu/available/etd-10282010-173834/ ;.

Council of Science Editors:

Dimitrova YN. Biochemical and structural analyses of TBL1: insights into the function of a transcriptional regulator. [Doctoral Dissertation]. Vanderbilt University; 2010. Available from: http://etd.library.vanderbilt.edu/available/etd-10282010-173834/ ;


Vanderbilt University

22. Smith, Andrew Leslie. ReCLIP (Reversible Cross-Link Immuno-Precipitation) reveals a novel interaction between p120-catenin and p160 Rho Kinase.

Degree: PhD, Cancer Biology, 2011, Vanderbilt University

 p120 catenin (p120) binds and stabilizes classical cadherins, making it a critical regulator of cell-cell adhesion. Here, we report an efficient technique (designated ReCLIP for… (more)

Subjects/Keywords: crosslink; Rho Kinase; cadherin; proteomics; p120-catenin

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APA (6th Edition):

Smith, A. L. (2011). ReCLIP (Reversible Cross-Link Immuno-Precipitation) reveals a novel interaction between p120-catenin and p160 Rho Kinase. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-03222011-133954/ ;

Chicago Manual of Style (16th Edition):

Smith, Andrew Leslie. “ReCLIP (Reversible Cross-Link Immuno-Precipitation) reveals a novel interaction between p120-catenin and p160 Rho Kinase.” 2011. Doctoral Dissertation, Vanderbilt University. Accessed September 23, 2019. http://etd.library.vanderbilt.edu/available/etd-03222011-133954/ ;.

MLA Handbook (7th Edition):

Smith, Andrew Leslie. “ReCLIP (Reversible Cross-Link Immuno-Precipitation) reveals a novel interaction between p120-catenin and p160 Rho Kinase.” 2011. Web. 23 Sep 2019.

Vancouver:

Smith AL. ReCLIP (Reversible Cross-Link Immuno-Precipitation) reveals a novel interaction between p120-catenin and p160 Rho Kinase. [Internet] [Doctoral dissertation]. Vanderbilt University; 2011. [cited 2019 Sep 23]. Available from: http://etd.library.vanderbilt.edu/available/etd-03222011-133954/ ;.

Council of Science Editors:

Smith AL. ReCLIP (Reversible Cross-Link Immuno-Precipitation) reveals a novel interaction between p120-catenin and p160 Rho Kinase. [Doctoral Dissertation]. Vanderbilt University; 2011. Available from: http://etd.library.vanderbilt.edu/available/etd-03222011-133954/ ;


Wayne State University

23. Guan, Xiaoqing. P120 Catenin Regulates Inflammation In Macrophage.

Degree: PhD, Biochemistry and Molecular Biology, 2017, Wayne State University

  Objective: p120 catenin (p120ctn) has been reported to play a critical role in maintenance of the stability of adherens junctions. It also has potential… (more)

Subjects/Keywords: Autophagy; Inflammation; Macrophage; p120 catenin; Biochemistry

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APA (6th Edition):

Guan, X. (2017). P120 Catenin Regulates Inflammation In Macrophage. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/1706

Chicago Manual of Style (16th Edition):

Guan, Xiaoqing. “P120 Catenin Regulates Inflammation In Macrophage.” 2017. Doctoral Dissertation, Wayne State University. Accessed September 23, 2019. https://digitalcommons.wayne.edu/oa_dissertations/1706.

MLA Handbook (7th Edition):

Guan, Xiaoqing. “P120 Catenin Regulates Inflammation In Macrophage.” 2017. Web. 23 Sep 2019.

Vancouver:

Guan X. P120 Catenin Regulates Inflammation In Macrophage. [Internet] [Doctoral dissertation]. Wayne State University; 2017. [cited 2019 Sep 23]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/1706.

Council of Science Editors:

Guan X. P120 Catenin Regulates Inflammation In Macrophage. [Doctoral Dissertation]. Wayne State University; 2017. Available from: https://digitalcommons.wayne.edu/oa_dissertations/1706


University of Rochester

24. Wang, Meina; Chen, Di (1955 - ). β-Catenin and Mmp13 Play Key Roles in Osteoarthritis and Degenerative Disc Disease.

Degree: PhD, 2011, University of Rochester

 Osteoarthritis (OA) is the most common arthritis affecting more than 10% of American adults. The pathogenic mechanism of this highly prevalent disease is not clear… (more)

Subjects/Keywords: B-Catenin; Mmp13; Osteoarthritis; Degenerative Disc Disease

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APA (6th Edition):

Wang, Meina; Chen, D. (. -. ). (2011). β-Catenin and Mmp13 Play Key Roles in Osteoarthritis and Degenerative Disc Disease. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/15855

Chicago Manual of Style (16th Edition):

Wang, Meina; Chen, Di (1955 - ). “β-Catenin and Mmp13 Play Key Roles in Osteoarthritis and Degenerative Disc Disease.” 2011. Doctoral Dissertation, University of Rochester. Accessed September 23, 2019. http://hdl.handle.net/1802/15855.

MLA Handbook (7th Edition):

Wang, Meina; Chen, Di (1955 - ). “β-Catenin and Mmp13 Play Key Roles in Osteoarthritis and Degenerative Disc Disease.” 2011. Web. 23 Sep 2019.

Vancouver:

Wang, Meina; Chen D(-). β-Catenin and Mmp13 Play Key Roles in Osteoarthritis and Degenerative Disc Disease. [Internet] [Doctoral dissertation]. University of Rochester; 2011. [cited 2019 Sep 23]. Available from: http://hdl.handle.net/1802/15855.

Council of Science Editors:

Wang, Meina; Chen D(-). β-Catenin and Mmp13 Play Key Roles in Osteoarthritis and Degenerative Disc Disease. [Doctoral Dissertation]. University of Rochester; 2011. Available from: http://hdl.handle.net/1802/15855


University of Rochester

25. Zhang, Yongchun. B-CATENIN Signaling Integrates with BMP and CCN1 Signaling to Regulate Chondrocyte Differentiation and Cartilage Development ate.

Degree: PhD, 2015, University of Rochester

 This work addresses the central question of how B-CATENIN signaling regulates chondrocyte differentiation during endochondral bone formation and cartilage development through integration with BMP and… (more)

Subjects/Keywords: beta-CATENIN; CCN1; BMP2; Chondrocyte; Cartilage

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APA (6th Edition):

Zhang, Y. (2015). B-CATENIN Signaling Integrates with BMP and CCN1 Signaling to Regulate Chondrocyte Differentiation and Cartilage Development ate. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/30113

Chicago Manual of Style (16th Edition):

Zhang, Yongchun. “B-CATENIN Signaling Integrates with BMP and CCN1 Signaling to Regulate Chondrocyte Differentiation and Cartilage Development ate.” 2015. Doctoral Dissertation, University of Rochester. Accessed September 23, 2019. http://hdl.handle.net/1802/30113.

MLA Handbook (7th Edition):

Zhang, Yongchun. “B-CATENIN Signaling Integrates with BMP and CCN1 Signaling to Regulate Chondrocyte Differentiation and Cartilage Development ate.” 2015. Web. 23 Sep 2019.

Vancouver:

Zhang Y. B-CATENIN Signaling Integrates with BMP and CCN1 Signaling to Regulate Chondrocyte Differentiation and Cartilage Development ate. [Internet] [Doctoral dissertation]. University of Rochester; 2015. [cited 2019 Sep 23]. Available from: http://hdl.handle.net/1802/30113.

Council of Science Editors:

Zhang Y. B-CATENIN Signaling Integrates with BMP and CCN1 Signaling to Regulate Chondrocyte Differentiation and Cartilage Development ate. [Doctoral Dissertation]. University of Rochester; 2015. Available from: http://hdl.handle.net/1802/30113


University of Cincinnati

26. Kendall, Jed. Targeting ß-catenin in MPNSTs.

Degree: PhD, Medicine: Molecular and Developmental Biology, 2017, University of Cincinnati

 Malignant peripheral nerve sheath tumors (MPNSTs) are soft tissue sarcomas thatare a major cause of mortality of Neurofibromatosis type 1 (NF1) patients. MPNSTpatients have few… (more)

Subjects/Keywords: Biology; MPNST; B-catenin; NF1; CK2; PITX2

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APA (6th Edition):

Kendall, J. (2017). Targeting ß-catenin in MPNSTs. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin149155952770148

Chicago Manual of Style (16th Edition):

Kendall, Jed. “Targeting ß-catenin in MPNSTs.” 2017. Doctoral Dissertation, University of Cincinnati. Accessed September 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=ucin149155952770148.

MLA Handbook (7th Edition):

Kendall, Jed. “Targeting ß-catenin in MPNSTs.” 2017. Web. 23 Sep 2019.

Vancouver:

Kendall J. Targeting ß-catenin in MPNSTs. [Internet] [Doctoral dissertation]. University of Cincinnati; 2017. [cited 2019 Sep 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin149155952770148.

Council of Science Editors:

Kendall J. Targeting ß-catenin in MPNSTs. [Doctoral Dissertation]. University of Cincinnati; 2017. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin149155952770148


University of New South Wales

27. Ben Hmeda, Imad. Investigation of the roles of IQGAP1 and Activated β-Catenin in epithelial ovarian cancer metastasis and patient outcome.

Degree: Clinical School - Prince of Wales Hospital, 2013, University of New South Wales

 Epithelial ovarian cancer (EOC) represents 90% of all ovarian cancer types and is a leading cause of death among gynaecological cancers in developed countries.Despite long… (more)

Subjects/Keywords: Activated β; -catenin; Epithelial ovarian cancer; IQGAP

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APA (6th Edition):

Ben Hmeda, I. (2013). Investigation of the roles of IQGAP1 and Activated β-Catenin in epithelial ovarian cancer metastasis and patient outcome. (Masters Thesis). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/52774 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11447/SOURCE01?view=true

Chicago Manual of Style (16th Edition):

Ben Hmeda, Imad. “Investigation of the roles of IQGAP1 and Activated β-Catenin in epithelial ovarian cancer metastasis and patient outcome.” 2013. Masters Thesis, University of New South Wales. Accessed September 23, 2019. http://handle.unsw.edu.au/1959.4/52774 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11447/SOURCE01?view=true.

MLA Handbook (7th Edition):

Ben Hmeda, Imad. “Investigation of the roles of IQGAP1 and Activated β-Catenin in epithelial ovarian cancer metastasis and patient outcome.” 2013. Web. 23 Sep 2019.

Vancouver:

Ben Hmeda I. Investigation of the roles of IQGAP1 and Activated β-Catenin in epithelial ovarian cancer metastasis and patient outcome. [Internet] [Masters thesis]. University of New South Wales; 2013. [cited 2019 Sep 23]. Available from: http://handle.unsw.edu.au/1959.4/52774 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11447/SOURCE01?view=true.

Council of Science Editors:

Ben Hmeda I. Investigation of the roles of IQGAP1 and Activated β-Catenin in epithelial ovarian cancer metastasis and patient outcome. [Masters Thesis]. University of New South Wales; 2013. Available from: http://handle.unsw.edu.au/1959.4/52774 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11447/SOURCE01?view=true


University of New South Wales

28. Dietrich, Philipp. Identification of targetable components of beta-catenin signalling in Acute Myeloid Leukaemic Stem Cells.

Degree: Women's & Children's Health, 2015, University of New South Wales

 Acute myeloid leukaemia (AML) remains the leading cause of leukaemia-related death with a relative five-year survival rate of only 24% in Australia. This poor prognosis… (more)

Subjects/Keywords: Acute Myeloid Leukaemia; GPR84; MLLAF9; beta-catenin

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APA (6th Edition):

Dietrich, P. (2015). Identification of targetable components of beta-catenin signalling in Acute Myeloid Leukaemic Stem Cells. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/55087 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:36505/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Dietrich, Philipp. “Identification of targetable components of beta-catenin signalling in Acute Myeloid Leukaemic Stem Cells.” 2015. Doctoral Dissertation, University of New South Wales. Accessed September 23, 2019. http://handle.unsw.edu.au/1959.4/55087 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:36505/SOURCE02?view=true.

MLA Handbook (7th Edition):

Dietrich, Philipp. “Identification of targetable components of beta-catenin signalling in Acute Myeloid Leukaemic Stem Cells.” 2015. Web. 23 Sep 2019.

Vancouver:

Dietrich P. Identification of targetable components of beta-catenin signalling in Acute Myeloid Leukaemic Stem Cells. [Internet] [Doctoral dissertation]. University of New South Wales; 2015. [cited 2019 Sep 23]. Available from: http://handle.unsw.edu.au/1959.4/55087 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:36505/SOURCE02?view=true.

Council of Science Editors:

Dietrich P. Identification of targetable components of beta-catenin signalling in Acute Myeloid Leukaemic Stem Cells. [Doctoral Dissertation]. University of New South Wales; 2015. Available from: http://handle.unsw.edu.au/1959.4/55087 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:36505/SOURCE02?view=true


University of Alberta

29. Ha, Jacqueline R. β-catenin is O-GlcNAc modified at Serine 23: Implications for β-catenin’s Subcellular Distribution and Transcriptional Activity.

Degree: MS, Medical Sciences-Paediatrics, 2012, University of Alberta

 β-catenin is a potent oncoprotein that serves as a structural anchor at the adherens junctions and as a transcriptional co-activator of the Wnt Signaling pathway.… (more)

Subjects/Keywords: β-catenin; β-catenin is O-GlcNAc modified at Serine 23; O-GlcNAcylation

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APA (6th Edition):

Ha, J. R. (2012). β-catenin is O-GlcNAc modified at Serine 23: Implications for β-catenin’s Subcellular Distribution and Transcriptional Activity. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/jd472x88t

Chicago Manual of Style (16th Edition):

Ha, Jacqueline R. “β-catenin is O-GlcNAc modified at Serine 23: Implications for β-catenin’s Subcellular Distribution and Transcriptional Activity.” 2012. Masters Thesis, University of Alberta. Accessed September 23, 2019. https://era.library.ualberta.ca/files/jd472x88t.

MLA Handbook (7th Edition):

Ha, Jacqueline R. “β-catenin is O-GlcNAc modified at Serine 23: Implications for β-catenin’s Subcellular Distribution and Transcriptional Activity.” 2012. Web. 23 Sep 2019.

Vancouver:

Ha JR. β-catenin is O-GlcNAc modified at Serine 23: Implications for β-catenin’s Subcellular Distribution and Transcriptional Activity. [Internet] [Masters thesis]. University of Alberta; 2012. [cited 2019 Sep 23]. Available from: https://era.library.ualberta.ca/files/jd472x88t.

Council of Science Editors:

Ha JR. β-catenin is O-GlcNAc modified at Serine 23: Implications for β-catenin’s Subcellular Distribution and Transcriptional Activity. [Masters Thesis]. University of Alberta; 2012. Available from: https://era.library.ualberta.ca/files/jd472x88t

30. Sacco, Sonia. Rôle de la signalisation Rspondin dans le développement et l’homéostasie de la glande surrénale : Rspondin signaling in adrenal gland development and homeostasis.

Degree: Docteur es, Sciences de la vie et de la santé, 2016, Côte d'Azur

La glande surrénale est un organe endocrinien d’une importance vitale de par son rôle dans le maintien de l’homéostasie corporelle. Pour assurer cette fonction, le… (more)

Subjects/Keywords: Glande surrénale; Rspondin; Β-catenin; Zonation; Homéostasie; Adrenal gland; Rspondin; Β-catenin; Zonation; Homeostasis

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APA (6th Edition):

Sacco, S. (2016). Rôle de la signalisation Rspondin dans le développement et l’homéostasie de la glande surrénale : Rspondin signaling in adrenal gland development and homeostasis. (Doctoral Dissertation). Côte d'Azur. Retrieved from http://www.theses.fr/2016AZUR4125

Chicago Manual of Style (16th Edition):

Sacco, Sonia. “Rôle de la signalisation Rspondin dans le développement et l’homéostasie de la glande surrénale : Rspondin signaling in adrenal gland development and homeostasis.” 2016. Doctoral Dissertation, Côte d'Azur. Accessed September 23, 2019. http://www.theses.fr/2016AZUR4125.

MLA Handbook (7th Edition):

Sacco, Sonia. “Rôle de la signalisation Rspondin dans le développement et l’homéostasie de la glande surrénale : Rspondin signaling in adrenal gland development and homeostasis.” 2016. Web. 23 Sep 2019.

Vancouver:

Sacco S. Rôle de la signalisation Rspondin dans le développement et l’homéostasie de la glande surrénale : Rspondin signaling in adrenal gland development and homeostasis. [Internet] [Doctoral dissertation]. Côte d'Azur; 2016. [cited 2019 Sep 23]. Available from: http://www.theses.fr/2016AZUR4125.

Council of Science Editors:

Sacco S. Rôle de la signalisation Rspondin dans le développement et l’homéostasie de la glande surrénale : Rspondin signaling in adrenal gland development and homeostasis. [Doctoral Dissertation]. Côte d'Azur; 2016. Available from: http://www.theses.fr/2016AZUR4125

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