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You searched for subject:( Catenin). Showing records 1 – 30 of 389 total matches.

[1] [2] [3] [4] [5] … [13]

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Vanderbilt University

1. Yu, Huapeng. p120-catenin controls contractility along the vertical axis of epithelial lateral membranes.

Degree: PhD, Cancer Biology, 2015, Vanderbilt University

 In vertebrate epithelia, p120-catenin mediates E-cadherin stability and suppression of RhoA. Genetic ablation of p120 in various epithelial tissues typically causes striking alterations in tissue… (more)

Subjects/Keywords: contractility; cadherin; catenin

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APA (6th Edition):

Yu, H. (2015). p120-catenin controls contractility along the vertical axis of epithelial lateral membranes. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-12072015-193917/ ;

Chicago Manual of Style (16th Edition):

Yu, Huapeng. “p120-catenin controls contractility along the vertical axis of epithelial lateral membranes.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed January 22, 2020. http://etd.library.vanderbilt.edu/available/etd-12072015-193917/ ;.

MLA Handbook (7th Edition):

Yu, Huapeng. “p120-catenin controls contractility along the vertical axis of epithelial lateral membranes.” 2015. Web. 22 Jan 2020.

Vancouver:

Yu H. p120-catenin controls contractility along the vertical axis of epithelial lateral membranes. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2020 Jan 22]. Available from: http://etd.library.vanderbilt.edu/available/etd-12072015-193917/ ;.

Council of Science Editors:

Yu H. p120-catenin controls contractility along the vertical axis of epithelial lateral membranes. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://etd.library.vanderbilt.edu/available/etd-12072015-193917/ ;


University of Toronto

2. Shipstone, Arun. Analysis of Alpha-catenin Mediated Intercellular Adhesion in Drosophila.

Degree: 2015, University of Toronto

Dynamic linkage between the cadherin-catenin complex (CCC) and the actin cytoskeleton at Adherens Junctions is essential for cell-cell adhesion in epithelial cells. Alpha-catenin is a… (more)

Subjects/Keywords: alpha-catenin; Beta-catenin; catenin; cell adhesion; Drosophila; E-Cadherin; 0379

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APA (6th Edition):

Shipstone, A. (2015). Analysis of Alpha-catenin Mediated Intercellular Adhesion in Drosophila. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/88582

Chicago Manual of Style (16th Edition):

Shipstone, Arun. “Analysis of Alpha-catenin Mediated Intercellular Adhesion in Drosophila.” 2015. Masters Thesis, University of Toronto. Accessed January 22, 2020. http://hdl.handle.net/1807/88582.

MLA Handbook (7th Edition):

Shipstone, Arun. “Analysis of Alpha-catenin Mediated Intercellular Adhesion in Drosophila.” 2015. Web. 22 Jan 2020.

Vancouver:

Shipstone A. Analysis of Alpha-catenin Mediated Intercellular Adhesion in Drosophila. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/1807/88582.

Council of Science Editors:

Shipstone A. Analysis of Alpha-catenin Mediated Intercellular Adhesion in Drosophila. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/88582

3. 神宮司, 健太郎. DIF-1 inhibits the Wnt/β-catenin signaling pathway by inhibiting TCF7L2 expression in colon cancer cell lines : DIF-1は大腸がん細胞株においてTCF7L2の発現を抑制することにより、Wnt/β-cateninシグナル伝達経路を阻害する.

Degree: 博士(医学), 2013, Kyushu University / 九州大学

我々はこれまでに細胞性粘菌Dictyostelium discoideumが分泌する分化誘導因子Differentiation-inducing factor-1(DIF-1)がβ-cateninタンパク質の分解を誘導することでWnt/β-cateninシグナル伝達経路を阻害し、ヒトがん細胞株の増殖を抑制することを報告してきた。β-cateninタンパク質の分解がDIF-1の効果に不可欠であるか否かを明らかにするために、我々はWnt/β-cateninシグナル伝達経路が恒常的に活性化されているヒト由来大腸がん細胞株(HCT-116、SW-620、DLD-1)に対するDIF-1の効果を検討した。DIF-1はβ-cateninタンパク質の分解非依存性にcyclin D1の発現をmRNA、タンパク質レベル共に減少させ、G_0/G_1期における細胞周期拘束を起こすことにより、強力に細胞増殖を抑制した。DIF-1によるtranscription factor 7-like 2(TCF7L2)発現量の抑制によって、TCF依存性転写活性及びcyclin D1プロモーター活性が抑制されることが明らかとなった。ルシフェラーゼレポーター活性測定及びTCF7L2プロモーター断片を用いたEMSAにより、翻訳開始点を起点として-609から-601塩基上流に位置する転写因子early growth response-1(Egr-1)結合部位が、DIF-1の効果に関与していることが示された。さらに、RNAi法による内在性TCF7L2の欠失はcyclin D1プロモーター活性及びタンパク質量の減少につながり、そして、TCF7L2の強制発現はDIF-1によるTCF依存性転写活性及びcyclin D1プロモーター活性の低下効果を減弱させた。したがって、DIF-1は大腸がん細胞株において、Egr-1依存性のTCF7L2転写活性を減少させることによりTCF7L2発現を抑制し、Wnt/β-cateninシグナル伝達経路を阻害していることが示唆された。我々の結果は、DIF-1によるWnt/β-cateninシグナル伝達経路の阻害機構に新たな知見をもたらすものである。

We previously reported that differentiation-inducing factor-1 (DIF-1), a morphogen in Dictyostelium discoideum, inhibits… (more)

Subjects/Keywords: DIF-1; Wnt/β-catenin

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APA (6th Edition):

神宮司, . (2013). DIF-1 inhibits the Wnt/β-catenin signaling pathway by inhibiting TCF7L2 expression in colon cancer cell lines : DIF-1は大腸がん細胞株においてTCF7L2の発現を抑制することにより、Wnt/β-cateninシグナル伝達経路を阻害する. (Thesis). Kyushu University / 九州大学. Retrieved from http://hdl.handle.net/2324/21727 ; http://dx.doi.org/10.15017/21727

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

神宮司, 健太郎. “DIF-1 inhibits the Wnt/β-catenin signaling pathway by inhibiting TCF7L2 expression in colon cancer cell lines : DIF-1は大腸がん細胞株においてTCF7L2の発現を抑制することにより、Wnt/β-cateninシグナル伝達経路を阻害する.” 2013. Thesis, Kyushu University / 九州大学. Accessed January 22, 2020. http://hdl.handle.net/2324/21727 ; http://dx.doi.org/10.15017/21727.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

神宮司, 健太郎. “DIF-1 inhibits the Wnt/β-catenin signaling pathway by inhibiting TCF7L2 expression in colon cancer cell lines : DIF-1は大腸がん細胞株においてTCF7L2の発現を抑制することにより、Wnt/β-cateninシグナル伝達経路を阻害する.” 2013. Web. 22 Jan 2020.

Vancouver:

神宮司 . DIF-1 inhibits the Wnt/β-catenin signaling pathway by inhibiting TCF7L2 expression in colon cancer cell lines : DIF-1は大腸がん細胞株においてTCF7L2の発現を抑制することにより、Wnt/β-cateninシグナル伝達経路を阻害する. [Internet] [Thesis]. Kyushu University / 九州大学; 2013. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/2324/21727 ; http://dx.doi.org/10.15017/21727.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

神宮司 . DIF-1 inhibits the Wnt/β-catenin signaling pathway by inhibiting TCF7L2 expression in colon cancer cell lines : DIF-1は大腸がん細胞株においてTCF7L2の発現を抑制することにより、Wnt/β-cateninシグナル伝達経路を阻害する. [Thesis]. Kyushu University / 九州大学; 2013. Available from: http://hdl.handle.net/2324/21727 ; http://dx.doi.org/10.15017/21727

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Australia

4. Sng, Joo Li Natasha. The role of β-catenin in activated EGFR-driven lung tumourigenesis.

Degree: M.Med.Sc., 2011, University of Western Australia

Lung cancer is associated with a high mortality rate, with Non-Small Cell Lung Cancer (NSCLC) accounting for the majority of cases. Mutations in the epidermal… (more)

Subjects/Keywords: EGFR; Lung tumour; Beta-catenin

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APA (6th Edition):

Sng, J. L. N. (2011). The role of β-catenin in activated EGFR-driven lung tumourigenesis. (Masters Thesis). University of Western Australia. Retrieved from http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=32218&local_base=GEN01-INS01

Chicago Manual of Style (16th Edition):

Sng, Joo Li Natasha. “The role of β-catenin in activated EGFR-driven lung tumourigenesis.” 2011. Masters Thesis, University of Western Australia. Accessed January 22, 2020. http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=32218&local_base=GEN01-INS01.

MLA Handbook (7th Edition):

Sng, Joo Li Natasha. “The role of β-catenin in activated EGFR-driven lung tumourigenesis.” 2011. Web. 22 Jan 2020.

Vancouver:

Sng JLN. The role of β-catenin in activated EGFR-driven lung tumourigenesis. [Internet] [Masters thesis]. University of Western Australia; 2011. [cited 2020 Jan 22]. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=32218&local_base=GEN01-INS01.

Council of Science Editors:

Sng JLN. The role of β-catenin in activated EGFR-driven lung tumourigenesis. [Masters Thesis]. University of Western Australia; 2011. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=32218&local_base=GEN01-INS01


McMaster University

5. Abdulla, Solen. INVESTIGATING NOVEL β-CATENIN SIGNALLING MECHANISMS IN AN EMBRYONIC STEM CELL MODEL.

Degree: MSc, 2017, McMaster University

The Wnt/β-catenin pathway is a fundamental regulator of embryonic development and adult tissue homeostasis. The key effector, β-catenin, is a multifunctional protein that occupies dual… (more)

Subjects/Keywords: β-catenin; Stem Cells

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APA (6th Edition):

Abdulla, S. (2017). INVESTIGATING NOVEL β-CATENIN SIGNALLING MECHANISMS IN AN EMBRYONIC STEM CELL MODEL. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/23461

Chicago Manual of Style (16th Edition):

Abdulla, Solen. “INVESTIGATING NOVEL β-CATENIN SIGNALLING MECHANISMS IN AN EMBRYONIC STEM CELL MODEL.” 2017. Masters Thesis, McMaster University. Accessed January 22, 2020. http://hdl.handle.net/11375/23461.

MLA Handbook (7th Edition):

Abdulla, Solen. “INVESTIGATING NOVEL β-CATENIN SIGNALLING MECHANISMS IN AN EMBRYONIC STEM CELL MODEL.” 2017. Web. 22 Jan 2020.

Vancouver:

Abdulla S. INVESTIGATING NOVEL β-CATENIN SIGNALLING MECHANISMS IN AN EMBRYONIC STEM CELL MODEL. [Internet] [Masters thesis]. McMaster University; 2017. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/11375/23461.

Council of Science Editors:

Abdulla S. INVESTIGATING NOVEL β-CATENIN SIGNALLING MECHANISMS IN AN EMBRYONIC STEM CELL MODEL. [Masters Thesis]. McMaster University; 2017. Available from: http://hdl.handle.net/11375/23461

6. Marcato, Vasco. La β-caténine : un activateur de l’expression du gène codant pour l’interféron-béta et une cible du Virus de la Fièvre de la Vallée du Rift : Magnetismo cooperativo en compuestos de coordinación basados en Cu(II), Ni(II) y Co(II) con ligandos imidazol carboxílicos.

Degree: Docteur es, Génétique, 2015, Sorbonne Paris Cité

La réponse antivirale innée constitue la première réaction d’une cellule à une infection virale. Il s’agit d’une réponse rapide, transitoire, non-spécifique, ubiquitaire qui a été… (more)

Subjects/Keywords: Β-caténine; Β-catenin; 616.042

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APA (6th Edition):

Marcato, V. (2015). La β-caténine : un activateur de l’expression du gène codant pour l’interféron-béta et une cible du Virus de la Fièvre de la Vallée du Rift : Magnetismo cooperativo en compuestos de coordinación basados en Cu(II), Ni(II) y Co(II) con ligandos imidazol carboxílicos. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2015USPCB103

Chicago Manual of Style (16th Edition):

Marcato, Vasco. “La β-caténine : un activateur de l’expression du gène codant pour l’interféron-béta et une cible du Virus de la Fièvre de la Vallée du Rift : Magnetismo cooperativo en compuestos de coordinación basados en Cu(II), Ni(II) y Co(II) con ligandos imidazol carboxílicos.” 2015. Doctoral Dissertation, Sorbonne Paris Cité. Accessed January 22, 2020. http://www.theses.fr/2015USPCB103.

MLA Handbook (7th Edition):

Marcato, Vasco. “La β-caténine : un activateur de l’expression du gène codant pour l’interféron-béta et une cible du Virus de la Fièvre de la Vallée du Rift : Magnetismo cooperativo en compuestos de coordinación basados en Cu(II), Ni(II) y Co(II) con ligandos imidazol carboxílicos.” 2015. Web. 22 Jan 2020.

Vancouver:

Marcato V. La β-caténine : un activateur de l’expression du gène codant pour l’interféron-béta et une cible du Virus de la Fièvre de la Vallée du Rift : Magnetismo cooperativo en compuestos de coordinación basados en Cu(II), Ni(II) y Co(II) con ligandos imidazol carboxílicos. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2015. [cited 2020 Jan 22]. Available from: http://www.theses.fr/2015USPCB103.

Council of Science Editors:

Marcato V. La β-caténine : un activateur de l’expression du gène codant pour l’interféron-béta et une cible du Virus de la Fièvre de la Vallée du Rift : Magnetismo cooperativo en compuestos de coordinación basados en Cu(II), Ni(II) y Co(II) con ligandos imidazol carboxílicos. [Doctoral Dissertation]. Sorbonne Paris Cité; 2015. Available from: http://www.theses.fr/2015USPCB103


Vanderbilt University

7. Markham, Nicholas Owen. DIPA is a novel, isoform-specific binding partner of the tumor-associated p120 isoform 1.

Degree: PhD, Cancer Biology, 2012, Vanderbilt University

 p120-catenin (p120) is a master regulator of cellular adherens junctions and is important for epithelial homeostasis, development, tumorigenesis, and metastasis. Relatively little is known about… (more)

Subjects/Keywords: adherens junctions; DIPA; p120-catenin

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APA (6th Edition):

Markham, N. O. (2012). DIPA is a novel, isoform-specific binding partner of the tumor-associated p120 isoform 1. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu//available/etd-07192012-173715/ ;

Chicago Manual of Style (16th Edition):

Markham, Nicholas Owen. “DIPA is a novel, isoform-specific binding partner of the tumor-associated p120 isoform 1.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed January 22, 2020. http://etd.library.vanderbilt.edu//available/etd-07192012-173715/ ;.

MLA Handbook (7th Edition):

Markham, Nicholas Owen. “DIPA is a novel, isoform-specific binding partner of the tumor-associated p120 isoform 1.” 2012. Web. 22 Jan 2020.

Vancouver:

Markham NO. DIPA is a novel, isoform-specific binding partner of the tumor-associated p120 isoform 1. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2020 Jan 22]. Available from: http://etd.library.vanderbilt.edu//available/etd-07192012-173715/ ;.

Council of Science Editors:

Markham NO. DIPA is a novel, isoform-specific binding partner of the tumor-associated p120 isoform 1. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://etd.library.vanderbilt.edu//available/etd-07192012-173715/ ;


Colorado State University

8. Nip, Kaila Akemi Ka`ohinani. AUTISM-ASSOCIATED δ-CATENIN G34S MUTATION PROMOTES GSK3β-MEDIATED PREMATURE δ-CATENIN DEGRADATION INDUCING NEURONAL DYSFUNCTION.

Degree: MS(M.S.), Cell and Molecular Biology (Graduate Degree Program), 2019, Colorado State University

 δ-catenin is a crucial component of a synaptic scaffolding complex, which regulates synaptic structure and function in neurons. Loss of δ-catenin function is strongly associated… (more)

Subjects/Keywords: delta-catenin; autism; GSK3β

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APA (6th Edition):

Nip, K. A. K. (2019). AUTISM-ASSOCIATED δ-CATENIN G34S MUTATION PROMOTES GSK3β-MEDIATED PREMATURE δ-CATENIN DEGRADATION INDUCING NEURONAL DYSFUNCTION. (Masters Thesis). Colorado State University. Retrieved from http://hdl.handle.net/10217/195416

Chicago Manual of Style (16th Edition):

Nip, Kaila Akemi Ka`ohinani. “AUTISM-ASSOCIATED δ-CATENIN G34S MUTATION PROMOTES GSK3β-MEDIATED PREMATURE δ-CATENIN DEGRADATION INDUCING NEURONAL DYSFUNCTION.” 2019. Masters Thesis, Colorado State University. Accessed January 22, 2020. http://hdl.handle.net/10217/195416.

MLA Handbook (7th Edition):

Nip, Kaila Akemi Ka`ohinani. “AUTISM-ASSOCIATED δ-CATENIN G34S MUTATION PROMOTES GSK3β-MEDIATED PREMATURE δ-CATENIN DEGRADATION INDUCING NEURONAL DYSFUNCTION.” 2019. Web. 22 Jan 2020.

Vancouver:

Nip KAK. AUTISM-ASSOCIATED δ-CATENIN G34S MUTATION PROMOTES GSK3β-MEDIATED PREMATURE δ-CATENIN DEGRADATION INDUCING NEURONAL DYSFUNCTION. [Internet] [Masters thesis]. Colorado State University; 2019. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/10217/195416.

Council of Science Editors:

Nip KAK. AUTISM-ASSOCIATED δ-CATENIN G34S MUTATION PROMOTES GSK3β-MEDIATED PREMATURE δ-CATENIN DEGRADATION INDUCING NEURONAL DYSFUNCTION. [Masters Thesis]. Colorado State University; 2019. Available from: http://hdl.handle.net/10217/195416


Universitat Pompeu Fabra

9. Aulicino, Francesco, 1987-. Investigating the role of Wnt/β-catenin pathway in pluripotency and somatic cell reprogramming.

Degree: Departament de Ciències Experimentals i de la Salut, 2016, Universitat Pompeu Fabra

 La respuesta adaptativa de las células a estímulos externos es un mecanismo fundamental de la existencia de la vida en sí misma. Para este fin,… (more)

Subjects/Keywords: Wnt/ß-catenin; Pluripotency; Reprogramming; ß-catenin; TCF1; Pluripotencia; Reprogramación; 576

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APA (6th Edition):

Aulicino, Francesco, 1. (2016). Investigating the role of Wnt/β-catenin pathway in pluripotency and somatic cell reprogramming. (Thesis). Universitat Pompeu Fabra. Retrieved from http://hdl.handle.net/10803/552942

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Aulicino, Francesco, 1987-. “Investigating the role of Wnt/β-catenin pathway in pluripotency and somatic cell reprogramming.” 2016. Thesis, Universitat Pompeu Fabra. Accessed January 22, 2020. http://hdl.handle.net/10803/552942.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Aulicino, Francesco, 1987-. “Investigating the role of Wnt/β-catenin pathway in pluripotency and somatic cell reprogramming.” 2016. Web. 22 Jan 2020.

Vancouver:

Aulicino, Francesco 1. Investigating the role of Wnt/β-catenin pathway in pluripotency and somatic cell reprogramming. [Internet] [Thesis]. Universitat Pompeu Fabra; 2016. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/10803/552942.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Aulicino, Francesco 1. Investigating the role of Wnt/β-catenin pathway in pluripotency and somatic cell reprogramming. [Thesis]. Universitat Pompeu Fabra; 2016. Available from: http://hdl.handle.net/10803/552942

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Rochester

10. Wang, Meina; Chen, Di (1955 - ). β-Catenin and Mmp13 Play Key Roles in Osteoarthritis and Degenerative Disc Disease.

Degree: PhD, 2011, University of Rochester

 Osteoarthritis (OA) is the most common arthritis affecting more than 10% of American adults. The pathogenic mechanism of this highly prevalent disease is not clear… (more)

Subjects/Keywords: B-Catenin; Mmp13; Osteoarthritis; Degenerative Disc Disease

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APA (6th Edition):

Wang, Meina; Chen, D. (. -. ). (2011). β-Catenin and Mmp13 Play Key Roles in Osteoarthritis and Degenerative Disc Disease. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/15855

Chicago Manual of Style (16th Edition):

Wang, Meina; Chen, Di (1955 - ). “β-Catenin and Mmp13 Play Key Roles in Osteoarthritis and Degenerative Disc Disease.” 2011. Doctoral Dissertation, University of Rochester. Accessed January 22, 2020. http://hdl.handle.net/1802/15855.

MLA Handbook (7th Edition):

Wang, Meina; Chen, Di (1955 - ). “β-Catenin and Mmp13 Play Key Roles in Osteoarthritis and Degenerative Disc Disease.” 2011. Web. 22 Jan 2020.

Vancouver:

Wang, Meina; Chen D(-). β-Catenin and Mmp13 Play Key Roles in Osteoarthritis and Degenerative Disc Disease. [Internet] [Doctoral dissertation]. University of Rochester; 2011. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/1802/15855.

Council of Science Editors:

Wang, Meina; Chen D(-). β-Catenin and Mmp13 Play Key Roles in Osteoarthritis and Degenerative Disc Disease. [Doctoral Dissertation]. University of Rochester; 2011. Available from: http://hdl.handle.net/1802/15855


University of Rochester

11. Zhang, Yongchun. B-CATENIN Signaling Integrates with BMP and CCN1 Signaling to Regulate Chondrocyte Differentiation and Cartilage Development ate.

Degree: PhD, 2015, University of Rochester

 This work addresses the central question of how B-CATENIN signaling regulates chondrocyte differentiation during endochondral bone formation and cartilage development through integration with BMP and… (more)

Subjects/Keywords: beta-CATENIN; CCN1; BMP2; Chondrocyte; Cartilage

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APA (6th Edition):

Zhang, Y. (2015). B-CATENIN Signaling Integrates with BMP and CCN1 Signaling to Regulate Chondrocyte Differentiation and Cartilage Development ate. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/30113

Chicago Manual of Style (16th Edition):

Zhang, Yongchun. “B-CATENIN Signaling Integrates with BMP and CCN1 Signaling to Regulate Chondrocyte Differentiation and Cartilage Development ate.” 2015. Doctoral Dissertation, University of Rochester. Accessed January 22, 2020. http://hdl.handle.net/1802/30113.

MLA Handbook (7th Edition):

Zhang, Yongchun. “B-CATENIN Signaling Integrates with BMP and CCN1 Signaling to Regulate Chondrocyte Differentiation and Cartilage Development ate.” 2015. Web. 22 Jan 2020.

Vancouver:

Zhang Y. B-CATENIN Signaling Integrates with BMP and CCN1 Signaling to Regulate Chondrocyte Differentiation and Cartilage Development ate. [Internet] [Doctoral dissertation]. University of Rochester; 2015. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/1802/30113.

Council of Science Editors:

Zhang Y. B-CATENIN Signaling Integrates with BMP and CCN1 Signaling to Regulate Chondrocyte Differentiation and Cartilage Development ate. [Doctoral Dissertation]. University of Rochester; 2015. Available from: http://hdl.handle.net/1802/30113


NSYSU

12. Chiang, Chi-Hsiang. Regulation of microRNA-182 and its functional role in breast cancer.

Degree: PhD, Institute of Biomedical Sciences, 2013, NSYSU

 MicroRNAs (MiRNAs) are endogenous small non-coding RNAs which negatively regulate gene expression by inducing translation repression or mRNA cleavage. MiR-182 is a member of the… (more)

Subjects/Keywords: RECK; β-catenin; microRNA; matrix metalloproteinase

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APA (6th Edition):

Chiang, C. (2013). Regulation of microRNA-182 and its functional role in breast cancer. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0624113-191725

Chicago Manual of Style (16th Edition):

Chiang, Chi-Hsiang. “Regulation of microRNA-182 and its functional role in breast cancer.” 2013. Doctoral Dissertation, NSYSU. Accessed January 22, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0624113-191725.

MLA Handbook (7th Edition):

Chiang, Chi-Hsiang. “Regulation of microRNA-182 and its functional role in breast cancer.” 2013. Web. 22 Jan 2020.

Vancouver:

Chiang C. Regulation of microRNA-182 and its functional role in breast cancer. [Internet] [Doctoral dissertation]. NSYSU; 2013. [cited 2020 Jan 22]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0624113-191725.

Council of Science Editors:

Chiang C. Regulation of microRNA-182 and its functional role in breast cancer. [Doctoral Dissertation]. NSYSU; 2013. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0624113-191725


Universiteit Utrecht

13. Pustjens, M.F. α-catenin in the adherens junction complex and the possible implications in cancer.

Degree: 2012, Universiteit Utrecht

 The most common malignancy in women of the Western World is breast cancer, which develops from mammary gland epithelium. 30-40% of the patients develop metastatic… (more)

Subjects/Keywords: alpha-catenin; adherens junctions; breast cancer

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APA (6th Edition):

Pustjens, M. F. (2012). α-catenin in the adherens junction complex and the possible implications in cancer. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/250410

Chicago Manual of Style (16th Edition):

Pustjens, M F. “α-catenin in the adherens junction complex and the possible implications in cancer.” 2012. Masters Thesis, Universiteit Utrecht. Accessed January 22, 2020. http://dspace.library.uu.nl:8080/handle/1874/250410.

MLA Handbook (7th Edition):

Pustjens, M F. “α-catenin in the adherens junction complex and the possible implications in cancer.” 2012. Web. 22 Jan 2020.

Vancouver:

Pustjens MF. α-catenin in the adherens junction complex and the possible implications in cancer. [Internet] [Masters thesis]. Universiteit Utrecht; 2012. [cited 2020 Jan 22]. Available from: http://dspace.library.uu.nl:8080/handle/1874/250410.

Council of Science Editors:

Pustjens MF. α-catenin in the adherens junction complex and the possible implications in cancer. [Masters Thesis]. Universiteit Utrecht; 2012. Available from: http://dspace.library.uu.nl:8080/handle/1874/250410


University of Manchester

14. Giles, Adam Alexander. Wnt signalling in oestrogen-induced lactotroph proliferation.

Degree: PhD, 2011, University of Manchester

 The anterior pituitary gland is the major hormonal regulator in the body. The gland contains five secretory cell types whose emergence during development is defined… (more)

Subjects/Keywords: 616.4; Wnt; Pituitary; Lactotroph; Beta-Catenin; Prolactin

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APA (6th Edition):

Giles, A. A. (2011). Wnt signalling in oestrogen-induced lactotroph proliferation. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/wnt-signalling-in-oestrogeninduced-lactotroph-proliferation(35c1ba30-0755-4583-bdce-69dce1382721).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.538396

Chicago Manual of Style (16th Edition):

Giles, Adam Alexander. “Wnt signalling in oestrogen-induced lactotroph proliferation.” 2011. Doctoral Dissertation, University of Manchester. Accessed January 22, 2020. https://www.research.manchester.ac.uk/portal/en/theses/wnt-signalling-in-oestrogeninduced-lactotroph-proliferation(35c1ba30-0755-4583-bdce-69dce1382721).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.538396.

MLA Handbook (7th Edition):

Giles, Adam Alexander. “Wnt signalling in oestrogen-induced lactotroph proliferation.” 2011. Web. 22 Jan 2020.

Vancouver:

Giles AA. Wnt signalling in oestrogen-induced lactotroph proliferation. [Internet] [Doctoral dissertation]. University of Manchester; 2011. [cited 2020 Jan 22]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/wnt-signalling-in-oestrogeninduced-lactotroph-proliferation(35c1ba30-0755-4583-bdce-69dce1382721).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.538396.

Council of Science Editors:

Giles AA. Wnt signalling in oestrogen-induced lactotroph proliferation. [Doctoral Dissertation]. University of Manchester; 2011. Available from: https://www.research.manchester.ac.uk/portal/en/theses/wnt-signalling-in-oestrogeninduced-lactotroph-proliferation(35c1ba30-0755-4583-bdce-69dce1382721).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.538396


University of Manchester

15. Hidalgo Sastre, Ana. Crosstalk between Notch and Wnt signalling pathways in vertebrates.

Degree: PhD, 2012, University of Manchester

 The development of complex metazoans depends on the integration of a handful of signalling pathways that eventually modulate precise patterns of gene expression. The fact… (more)

Subjects/Keywords: 612.015; Notch; Wnt; ß-catenin; crosstalk; inhibition

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APA (6th Edition):

Hidalgo Sastre, A. (2012). Crosstalk between Notch and Wnt signalling pathways in vertebrates. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/crosstalk-between-notch-and-wnt-signalling-pathways-in-vertebrates(9b4411a3-cd03-4af3-b3b5-8c432c7a2c68).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559348

Chicago Manual of Style (16th Edition):

Hidalgo Sastre, Ana. “Crosstalk between Notch and Wnt signalling pathways in vertebrates.” 2012. Doctoral Dissertation, University of Manchester. Accessed January 22, 2020. https://www.research.manchester.ac.uk/portal/en/theses/crosstalk-between-notch-and-wnt-signalling-pathways-in-vertebrates(9b4411a3-cd03-4af3-b3b5-8c432c7a2c68).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559348.

MLA Handbook (7th Edition):

Hidalgo Sastre, Ana. “Crosstalk between Notch and Wnt signalling pathways in vertebrates.” 2012. Web. 22 Jan 2020.

Vancouver:

Hidalgo Sastre A. Crosstalk between Notch and Wnt signalling pathways in vertebrates. [Internet] [Doctoral dissertation]. University of Manchester; 2012. [cited 2020 Jan 22]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/crosstalk-between-notch-and-wnt-signalling-pathways-in-vertebrates(9b4411a3-cd03-4af3-b3b5-8c432c7a2c68).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559348.

Council of Science Editors:

Hidalgo Sastre A. Crosstalk between Notch and Wnt signalling pathways in vertebrates. [Doctoral Dissertation]. University of Manchester; 2012. Available from: https://www.research.manchester.ac.uk/portal/en/theses/crosstalk-between-notch-and-wnt-signalling-pathways-in-vertebrates(9b4411a3-cd03-4af3-b3b5-8c432c7a2c68).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559348


University of Southern California

16. Wu, Jia. Wnt/β-catenin/p300 induced transcription is critical for the differentiation and maintenance of Paneth cells.

Degree: MS, Biochemistry and Molecular Biology, 2014, University of Southern California

 Our lab's previous studies have demonstrated that Wnt/β‐catenin/p300 transcription initiates cell differentiation, whereas Wnt/β‐catenin/cyclic AMP‐responsive element binding protein‐binding protein (CBP) transcription mediates cell proliferation/maintenance of… (more)

Subjects/Keywords: Wnt; β ‐Catenin/p300; Paneth cell

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APA (6th Edition):

Wu, J. (2014). Wnt/β-catenin/p300 induced transcription is critical for the differentiation and maintenance of Paneth cells. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/405481/rec/7947

Chicago Manual of Style (16th Edition):

Wu, Jia. “Wnt/β-catenin/p300 induced transcription is critical for the differentiation and maintenance of Paneth cells.” 2014. Masters Thesis, University of Southern California. Accessed January 22, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/405481/rec/7947.

MLA Handbook (7th Edition):

Wu, Jia. “Wnt/β-catenin/p300 induced transcription is critical for the differentiation and maintenance of Paneth cells.” 2014. Web. 22 Jan 2020.

Vancouver:

Wu J. Wnt/β-catenin/p300 induced transcription is critical for the differentiation and maintenance of Paneth cells. [Internet] [Masters thesis]. University of Southern California; 2014. [cited 2020 Jan 22]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/405481/rec/7947.

Council of Science Editors:

Wu J. Wnt/β-catenin/p300 induced transcription is critical for the differentiation and maintenance of Paneth cells. [Masters Thesis]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/405481/rec/7947


University of Manchester

17. Giles, Adam Alexander. Wnt signalling in oestrogen-induced lactotroph proliferation.

Degree: 2011, University of Manchester

 The University of ManchesterAdam GilesDoctor of Philosophy – PhDWnt signalling in oestrogen-induced lactotroph hyperplasia2011The anterior pituitary gland is the major hormonal regulator in the body.… (more)

Subjects/Keywords: Wnt; Pituitary; Lactotroph; Beta-Catenin; Prolactin

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APA (6th Edition):

Giles, A. A. (2011). Wnt signalling in oestrogen-induced lactotroph proliferation. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:121389

Chicago Manual of Style (16th Edition):

Giles, Adam Alexander. “Wnt signalling in oestrogen-induced lactotroph proliferation.” 2011. Doctoral Dissertation, University of Manchester. Accessed January 22, 2020. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:121389.

MLA Handbook (7th Edition):

Giles, Adam Alexander. “Wnt signalling in oestrogen-induced lactotroph proliferation.” 2011. Web. 22 Jan 2020.

Vancouver:

Giles AA. Wnt signalling in oestrogen-induced lactotroph proliferation. [Internet] [Doctoral dissertation]. University of Manchester; 2011. [cited 2020 Jan 22]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:121389.

Council of Science Editors:

Giles AA. Wnt signalling in oestrogen-induced lactotroph proliferation. [Doctoral Dissertation]. University of Manchester; 2011. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:121389


Boston University

18. Gowda, Asha. The effect of protein kinase C and Beta-catenin inhibitors on uveal melanoma cells.

Degree: MS, Medical Sciences, 2014, Boston University

 PURPOSE: Uveal melanoma (UM) is the most common intraocular malignancy in adults with an incidence of six per one million individuals each year. Globe conserving… (more)

Subjects/Keywords: Ophthalmology; Beta-catenin; Melanoma; Protein kinase C

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APA (6th Edition):

Gowda, A. (2014). The effect of protein kinase C and Beta-catenin inhibitors on uveal melanoma cells. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/15046

Chicago Manual of Style (16th Edition):

Gowda, Asha. “The effect of protein kinase C and Beta-catenin inhibitors on uveal melanoma cells.” 2014. Masters Thesis, Boston University. Accessed January 22, 2020. http://hdl.handle.net/2144/15046.

MLA Handbook (7th Edition):

Gowda, Asha. “The effect of protein kinase C and Beta-catenin inhibitors on uveal melanoma cells.” 2014. Web. 22 Jan 2020.

Vancouver:

Gowda A. The effect of protein kinase C and Beta-catenin inhibitors on uveal melanoma cells. [Internet] [Masters thesis]. Boston University; 2014. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/2144/15046.

Council of Science Editors:

Gowda A. The effect of protein kinase C and Beta-catenin inhibitors on uveal melanoma cells. [Masters Thesis]. Boston University; 2014. Available from: http://hdl.handle.net/2144/15046

19. Manring, Heather Renee. COMPARATIVE CELL BASED FUNCTIONAL ANALYSIS OF KEY WNT/BETA-CATENIN PATHWAY GENES AND GENE MUTATIONS.

Degree: 2012, Wake Forest University

 The Wnt/Beta-catenin signaling pathway has many roles including regulation of developmental pathways, cell proliferation, and homeostasis of adult tissues. Wnt/Beta-catenin signaling activity depends on the… (more)

Subjects/Keywords: Beta-catenin

…OF FIGURES Page Figure 1.1. Schematic of the Wnt/β-catenin pathway in the inactive (A… …3 Figure 1.2. β-catenin function is partially mediated by its location within the cell… …and interaction domains for proteins of the Wnt/β-catenin pathway and other signaling… …pathways. .......... 8 Figure 1.4. Mechanisms of regulating Wnt/β-catenin signaling activity… …22 Figure 2.1. Increased β-catenin protein expression and β-catenin mediated transcription… 

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APA (6th Edition):

Manring, H. R. (2012). COMPARATIVE CELL BASED FUNCTIONAL ANALYSIS OF KEY WNT/BETA-CATENIN PATHWAY GENES AND GENE MUTATIONS. (Thesis). Wake Forest University. Retrieved from http://hdl.handle.net/10339/37663

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Manring, Heather Renee. “COMPARATIVE CELL BASED FUNCTIONAL ANALYSIS OF KEY WNT/BETA-CATENIN PATHWAY GENES AND GENE MUTATIONS.” 2012. Thesis, Wake Forest University. Accessed January 22, 2020. http://hdl.handle.net/10339/37663.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Manring, Heather Renee. “COMPARATIVE CELL BASED FUNCTIONAL ANALYSIS OF KEY WNT/BETA-CATENIN PATHWAY GENES AND GENE MUTATIONS.” 2012. Web. 22 Jan 2020.

Vancouver:

Manring HR. COMPARATIVE CELL BASED FUNCTIONAL ANALYSIS OF KEY WNT/BETA-CATENIN PATHWAY GENES AND GENE MUTATIONS. [Internet] [Thesis]. Wake Forest University; 2012. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/10339/37663.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Manring HR. COMPARATIVE CELL BASED FUNCTIONAL ANALYSIS OF KEY WNT/BETA-CATENIN PATHWAY GENES AND GENE MUTATIONS. [Thesis]. Wake Forest University; 2012. Available from: http://hdl.handle.net/10339/37663

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

20. Dimitrova, Yoana Nantcheva. Biochemical and structural analyses of TBL1: insights into the function of a transcriptional regulator.

Degree: PhD, Biochemistry, 2010, Vanderbilt University

 The mechanism controlling the switch between gene activation and repression is critically important for understanding the process of transcriptional regulation. Gene expression is highly controlled… (more)

Subjects/Keywords: transcriptional regulation; ubiquitination; beta-catenin; TBL1

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APA (6th Edition):

Dimitrova, Y. N. (2010). Biochemical and structural analyses of TBL1: insights into the function of a transcriptional regulator. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-10282010-173834/ ;

Chicago Manual of Style (16th Edition):

Dimitrova, Yoana Nantcheva. “Biochemical and structural analyses of TBL1: insights into the function of a transcriptional regulator.” 2010. Doctoral Dissertation, Vanderbilt University. Accessed January 22, 2020. http://etd.library.vanderbilt.edu/available/etd-10282010-173834/ ;.

MLA Handbook (7th Edition):

Dimitrova, Yoana Nantcheva. “Biochemical and structural analyses of TBL1: insights into the function of a transcriptional regulator.” 2010. Web. 22 Jan 2020.

Vancouver:

Dimitrova YN. Biochemical and structural analyses of TBL1: insights into the function of a transcriptional regulator. [Internet] [Doctoral dissertation]. Vanderbilt University; 2010. [cited 2020 Jan 22]. Available from: http://etd.library.vanderbilt.edu/available/etd-10282010-173834/ ;.

Council of Science Editors:

Dimitrova YN. Biochemical and structural analyses of TBL1: insights into the function of a transcriptional regulator. [Doctoral Dissertation]. Vanderbilt University; 2010. Available from: http://etd.library.vanderbilt.edu/available/etd-10282010-173834/ ;


Vanderbilt University

21. Smith, Andrew Leslie. ReCLIP (Reversible Cross-Link Immuno-Precipitation) reveals a novel interaction between p120-catenin and p160 Rho Kinase.

Degree: PhD, Cancer Biology, 2011, Vanderbilt University

 p120 catenin (p120) binds and stabilizes classical cadherins, making it a critical regulator of cell-cell adhesion. Here, we report an efficient technique (designated ReCLIP for… (more)

Subjects/Keywords: crosslink; Rho Kinase; cadherin; proteomics; p120-catenin

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APA (6th Edition):

Smith, A. L. (2011). ReCLIP (Reversible Cross-Link Immuno-Precipitation) reveals a novel interaction between p120-catenin and p160 Rho Kinase. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-03222011-133954/ ;

Chicago Manual of Style (16th Edition):

Smith, Andrew Leslie. “ReCLIP (Reversible Cross-Link Immuno-Precipitation) reveals a novel interaction between p120-catenin and p160 Rho Kinase.” 2011. Doctoral Dissertation, Vanderbilt University. Accessed January 22, 2020. http://etd.library.vanderbilt.edu/available/etd-03222011-133954/ ;.

MLA Handbook (7th Edition):

Smith, Andrew Leslie. “ReCLIP (Reversible Cross-Link Immuno-Precipitation) reveals a novel interaction between p120-catenin and p160 Rho Kinase.” 2011. Web. 22 Jan 2020.

Vancouver:

Smith AL. ReCLIP (Reversible Cross-Link Immuno-Precipitation) reveals a novel interaction between p120-catenin and p160 Rho Kinase. [Internet] [Doctoral dissertation]. Vanderbilt University; 2011. [cited 2020 Jan 22]. Available from: http://etd.library.vanderbilt.edu/available/etd-03222011-133954/ ;.

Council of Science Editors:

Smith AL. ReCLIP (Reversible Cross-Link Immuno-Precipitation) reveals a novel interaction between p120-catenin and p160 Rho Kinase. [Doctoral Dissertation]. Vanderbilt University; 2011. Available from: http://etd.library.vanderbilt.edu/available/etd-03222011-133954/ ;


Wayne State University

22. Guan, Xiaoqing. P120 Catenin Regulates Inflammation In Macrophage.

Degree: PhD, Biochemistry and Molecular Biology, 2017, Wayne State University

  Objective: p120 catenin (p120ctn) has been reported to play a critical role in maintenance of the stability of adherens junctions. It also has potential… (more)

Subjects/Keywords: Autophagy; Inflammation; Macrophage; p120 catenin; Biochemistry

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APA (6th Edition):

Guan, X. (2017). P120 Catenin Regulates Inflammation In Macrophage. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/1706

Chicago Manual of Style (16th Edition):

Guan, Xiaoqing. “P120 Catenin Regulates Inflammation In Macrophage.” 2017. Doctoral Dissertation, Wayne State University. Accessed January 22, 2020. https://digitalcommons.wayne.edu/oa_dissertations/1706.

MLA Handbook (7th Edition):

Guan, Xiaoqing. “P120 Catenin Regulates Inflammation In Macrophage.” 2017. Web. 22 Jan 2020.

Vancouver:

Guan X. P120 Catenin Regulates Inflammation In Macrophage. [Internet] [Doctoral dissertation]. Wayne State University; 2017. [cited 2020 Jan 22]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/1706.

Council of Science Editors:

Guan X. P120 Catenin Regulates Inflammation In Macrophage. [Doctoral Dissertation]. Wayne State University; 2017. Available from: https://digitalcommons.wayne.edu/oa_dissertations/1706

23. Kendall, Jed. Targeting ß-catenin in MPNSTs.

Degree: PhD, Medicine: Molecular and Developmental Biology, 2017, University of Cincinnati

 Malignant peripheral nerve sheath tumors (MPNSTs) are soft tissue sarcomas thatare a major cause of mortality of Neurofibromatosis type 1 (NF1) patients. MPNSTpatients have few… (more)

Subjects/Keywords: Biology; MPNST; B-catenin; NF1; CK2; PITX2

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APA (6th Edition):

Kendall, J. (2017). Targeting ß-catenin in MPNSTs. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin149155952770148

Chicago Manual of Style (16th Edition):

Kendall, Jed. “Targeting ß-catenin in MPNSTs.” 2017. Doctoral Dissertation, University of Cincinnati. Accessed January 22, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=ucin149155952770148.

MLA Handbook (7th Edition):

Kendall, Jed. “Targeting ß-catenin in MPNSTs.” 2017. Web. 22 Jan 2020.

Vancouver:

Kendall J. Targeting ß-catenin in MPNSTs. [Internet] [Doctoral dissertation]. University of Cincinnati; 2017. [cited 2020 Jan 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin149155952770148.

Council of Science Editors:

Kendall J. Targeting ß-catenin in MPNSTs. [Doctoral Dissertation]. University of Cincinnati; 2017. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin149155952770148


University of New South Wales

24. Ben Hmeda, Imad. Investigation of the roles of IQGAP1 and Activated β-Catenin in epithelial ovarian cancer metastasis and patient outcome.

Degree: Clinical School - Prince of Wales Hospital, 2013, University of New South Wales

 Epithelial ovarian cancer (EOC) represents 90% of all ovarian cancer types and is a leading cause of death among gynaecological cancers in developed countries.Despite long… (more)

Subjects/Keywords: Activated β; -catenin; Epithelial ovarian cancer; IQGAP

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APA (6th Edition):

Ben Hmeda, I. (2013). Investigation of the roles of IQGAP1 and Activated β-Catenin in epithelial ovarian cancer metastasis and patient outcome. (Masters Thesis). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/52774 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11447/SOURCE01?view=true

Chicago Manual of Style (16th Edition):

Ben Hmeda, Imad. “Investigation of the roles of IQGAP1 and Activated β-Catenin in epithelial ovarian cancer metastasis and patient outcome.” 2013. Masters Thesis, University of New South Wales. Accessed January 22, 2020. http://handle.unsw.edu.au/1959.4/52774 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11447/SOURCE01?view=true.

MLA Handbook (7th Edition):

Ben Hmeda, Imad. “Investigation of the roles of IQGAP1 and Activated β-Catenin in epithelial ovarian cancer metastasis and patient outcome.” 2013. Web. 22 Jan 2020.

Vancouver:

Ben Hmeda I. Investigation of the roles of IQGAP1 and Activated β-Catenin in epithelial ovarian cancer metastasis and patient outcome. [Internet] [Masters thesis]. University of New South Wales; 2013. [cited 2020 Jan 22]. Available from: http://handle.unsw.edu.au/1959.4/52774 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11447/SOURCE01?view=true.

Council of Science Editors:

Ben Hmeda I. Investigation of the roles of IQGAP1 and Activated β-Catenin in epithelial ovarian cancer metastasis and patient outcome. [Masters Thesis]. University of New South Wales; 2013. Available from: http://handle.unsw.edu.au/1959.4/52774 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11447/SOURCE01?view=true


University of New South Wales

25. Dietrich, Philipp. Identification of targetable components of beta-catenin signalling in Acute Myeloid Leukaemic Stem Cells.

Degree: Women's & Children's Health, 2015, University of New South Wales

 Acute myeloid leukaemia (AML) remains the leading cause of leukaemia-related death with a relative five-year survival rate of only 24% in Australia. This poor prognosis… (more)

Subjects/Keywords: Acute Myeloid Leukaemia; GPR84; MLLAF9; beta-catenin

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APA (6th Edition):

Dietrich, P. (2015). Identification of targetable components of beta-catenin signalling in Acute Myeloid Leukaemic Stem Cells. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/55087 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:36505/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Dietrich, Philipp. “Identification of targetable components of beta-catenin signalling in Acute Myeloid Leukaemic Stem Cells.” 2015. Doctoral Dissertation, University of New South Wales. Accessed January 22, 2020. http://handle.unsw.edu.au/1959.4/55087 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:36505/SOURCE02?view=true.

MLA Handbook (7th Edition):

Dietrich, Philipp. “Identification of targetable components of beta-catenin signalling in Acute Myeloid Leukaemic Stem Cells.” 2015. Web. 22 Jan 2020.

Vancouver:

Dietrich P. Identification of targetable components of beta-catenin signalling in Acute Myeloid Leukaemic Stem Cells. [Internet] [Doctoral dissertation]. University of New South Wales; 2015. [cited 2020 Jan 22]. Available from: http://handle.unsw.edu.au/1959.4/55087 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:36505/SOURCE02?view=true.

Council of Science Editors:

Dietrich P. Identification of targetable components of beta-catenin signalling in Acute Myeloid Leukaemic Stem Cells. [Doctoral Dissertation]. University of New South Wales; 2015. Available from: http://handle.unsw.edu.au/1959.4/55087 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:36505/SOURCE02?view=true


University of Illinois – Chicago

26. Vandenbroucke St Amant, Emily E. The Role of PKCAlpha-Mediated p120-Catenin Phosphorylation on Endothelial Permeability.

Degree: 2013, University of Illinois – Chicago

 Adherens junctions are the primary cell-cell junction that maintains the integrity of the endothelium. Vascular leakage resulting from inflammation contributes to a variety of pathological… (more)

Subjects/Keywords: endothelial permeability; p120-catenin; Serine 879; PKCalpha

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APA (6th Edition):

Vandenbroucke St Amant, E. E. (2013). The Role of PKCAlpha-Mediated p120-Catenin Phosphorylation on Endothelial Permeability. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/9728

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vandenbroucke St Amant, Emily E. “The Role of PKCAlpha-Mediated p120-Catenin Phosphorylation on Endothelial Permeability.” 2013. Thesis, University of Illinois – Chicago. Accessed January 22, 2020. http://hdl.handle.net/10027/9728.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vandenbroucke St Amant, Emily E. “The Role of PKCAlpha-Mediated p120-Catenin Phosphorylation on Endothelial Permeability.” 2013. Web. 22 Jan 2020.

Vancouver:

Vandenbroucke St Amant EE. The Role of PKCAlpha-Mediated p120-Catenin Phosphorylation on Endothelial Permeability. [Internet] [Thesis]. University of Illinois – Chicago; 2013. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/10027/9728.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vandenbroucke St Amant EE. The Role of PKCAlpha-Mediated p120-Catenin Phosphorylation on Endothelial Permeability. [Thesis]. University of Illinois – Chicago; 2013. Available from: http://hdl.handle.net/10027/9728

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Chicago

27. Pham, Thao. Protein Kinase C Alpha Mediates FOXC2 Transcriptional Repression Of p120-Catenin In Human Breast Cancer.

Degree: 2016, University of Illinois – Chicago

 Breast cancer is the most commonly diagnosed malignancy in women worldwide. Despite recent advances in prevention, diagnosis, and treatment, metastasis remains a roadblock to successful… (more)

Subjects/Keywords: PKC; p120-catenin; FOXC2; breast cancer

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APA (6th Edition):

Pham, T. (2016). Protein Kinase C Alpha Mediates FOXC2 Transcriptional Repression Of p120-Catenin In Human Breast Cancer. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/21604

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pham, Thao. “Protein Kinase C Alpha Mediates FOXC2 Transcriptional Repression Of p120-Catenin In Human Breast Cancer.” 2016. Thesis, University of Illinois – Chicago. Accessed January 22, 2020. http://hdl.handle.net/10027/21604.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pham, Thao. “Protein Kinase C Alpha Mediates FOXC2 Transcriptional Repression Of p120-Catenin In Human Breast Cancer.” 2016. Web. 22 Jan 2020.

Vancouver:

Pham T. Protein Kinase C Alpha Mediates FOXC2 Transcriptional Repression Of p120-Catenin In Human Breast Cancer. [Internet] [Thesis]. University of Illinois – Chicago; 2016. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/10027/21604.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pham T. Protein Kinase C Alpha Mediates FOXC2 Transcriptional Repression Of p120-Catenin In Human Breast Cancer. [Thesis]. University of Illinois – Chicago; 2016. Available from: http://hdl.handle.net/10027/21604

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McMaster University

28. Cunanan, Joanna. Quercetin Inhibits β-catenin Transcriptional Activity During Kidney Development and Reduces the Severity of Renal Dysplasia.

Degree: MSMS, 2019, McMaster University

M.Sc. Thesis Dissertation, August 2019, McMaster University

Renal dysplasia, defined as the abnormal development of kidney tissue, is the leading cause of kidney disease in… (more)

Subjects/Keywords: kidney development; beta-catenin; renal dysplasia; quercetin

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APA (6th Edition):

Cunanan, J. (2019). Quercetin Inhibits β-catenin Transcriptional Activity During Kidney Development and Reduces the Severity of Renal Dysplasia. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/24793

Chicago Manual of Style (16th Edition):

Cunanan, Joanna. “Quercetin Inhibits β-catenin Transcriptional Activity During Kidney Development and Reduces the Severity of Renal Dysplasia.” 2019. Masters Thesis, McMaster University. Accessed January 22, 2020. http://hdl.handle.net/11375/24793.

MLA Handbook (7th Edition):

Cunanan, Joanna. “Quercetin Inhibits β-catenin Transcriptional Activity During Kidney Development and Reduces the Severity of Renal Dysplasia.” 2019. Web. 22 Jan 2020.

Vancouver:

Cunanan J. Quercetin Inhibits β-catenin Transcriptional Activity During Kidney Development and Reduces the Severity of Renal Dysplasia. [Internet] [Masters thesis]. McMaster University; 2019. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/11375/24793.

Council of Science Editors:

Cunanan J. Quercetin Inhibits β-catenin Transcriptional Activity During Kidney Development and Reduces the Severity of Renal Dysplasia. [Masters Thesis]. McMaster University; 2019. Available from: http://hdl.handle.net/11375/24793


Georgia State University

29. Hobson, Katherine. METHIONINE RESTRICTION INHIBITS NON-SMALL CELL LUNG CANCER GROWTH BY TARGETING THE BETA-CATENIN PATHWAY.

Degree: MS, Nutrition, 2018, Georgia State University

  Background: Lung cancer is the leading cause of cancer related death for both men and women. Non-small cell lung cancer (NSCLC) accounts for 80%… (more)

Subjects/Keywords: Lung Cancer; Methionine Restriction; Beta-catenin

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APA (6th Edition):

Hobson, K. (2018). METHIONINE RESTRICTION INHIBITS NON-SMALL CELL LUNG CANCER GROWTH BY TARGETING THE BETA-CATENIN PATHWAY. (Thesis). Georgia State University. Retrieved from https://scholarworks.gsu.edu/nutrition_theses/99

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hobson, Katherine. “METHIONINE RESTRICTION INHIBITS NON-SMALL CELL LUNG CANCER GROWTH BY TARGETING THE BETA-CATENIN PATHWAY.” 2018. Thesis, Georgia State University. Accessed January 22, 2020. https://scholarworks.gsu.edu/nutrition_theses/99.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hobson, Katherine. “METHIONINE RESTRICTION INHIBITS NON-SMALL CELL LUNG CANCER GROWTH BY TARGETING THE BETA-CATENIN PATHWAY.” 2018. Web. 22 Jan 2020.

Vancouver:

Hobson K. METHIONINE RESTRICTION INHIBITS NON-SMALL CELL LUNG CANCER GROWTH BY TARGETING THE BETA-CATENIN PATHWAY. [Internet] [Thesis]. Georgia State University; 2018. [cited 2020 Jan 22]. Available from: https://scholarworks.gsu.edu/nutrition_theses/99.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hobson K. METHIONINE RESTRICTION INHIBITS NON-SMALL CELL LUNG CANCER GROWTH BY TARGETING THE BETA-CATENIN PATHWAY. [Thesis]. Georgia State University; 2018. Available from: https://scholarworks.gsu.edu/nutrition_theses/99

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Washington

30. Conrad, William. Resolving mechanisms of apoptosis in response to WNT3A in Melanoma.

Degree: PhD, 2013, University of Washington

 Wnt/β-catenin and ERK/MAPK signaling regulate the balance of differentiation and proliferation in melanoma. Aberrant activation of ERK/MAPK signaling results from the BRAFV600E or NRASQ61X mutations… (more)

Subjects/Keywords: BRAF; catenin; FAM129B; NRAS; WNT; Pharmacology; pharmacology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Conrad, W. (2013). Resolving mechanisms of apoptosis in response to WNT3A in Melanoma. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/22448

Chicago Manual of Style (16th Edition):

Conrad, William. “Resolving mechanisms of apoptosis in response to WNT3A in Melanoma.” 2013. Doctoral Dissertation, University of Washington. Accessed January 22, 2020. http://hdl.handle.net/1773/22448.

MLA Handbook (7th Edition):

Conrad, William. “Resolving mechanisms of apoptosis in response to WNT3A in Melanoma.” 2013. Web. 22 Jan 2020.

Vancouver:

Conrad W. Resolving mechanisms of apoptosis in response to WNT3A in Melanoma. [Internet] [Doctoral dissertation]. University of Washington; 2013. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/1773/22448.

Council of Science Editors:

Conrad W. Resolving mechanisms of apoptosis in response to WNT3A in Melanoma. [Doctoral Dissertation]. University of Washington; 2013. Available from: http://hdl.handle.net/1773/22448

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