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You searched for subject:( TGF Smad3 pirin ). Showing records 1 – 30 of 630 total matches.

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1. 山岡, 華児. 細胞の浸潤・増殖・遊走を制御する新規低分子化合物による創傷治癒促進機序の検討 : サイボウ ノ シンジュン ゾウショク ユウソウ オ セイギョ スル シンキ テイブンシ カゴウブツ ニヨル ソウショウ チユ ソクシン キジョ ノ ケントウ.

Degree: 博士(医学), 2014, Tokai University Repository / 東海大学

Subjects/Keywords: 創傷治癒, TGF-β/Smad3シグナル, pirin, 表皮角化細胞, 真皮線維芽細胞; wound healing, TGF-β/Smad3 signal, pirin, Artificial dermis graft

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APA (6th Edition):

山岡, . (2014). 細胞の浸潤・増殖・遊走を制御する新規低分子化合物による創傷治癒促進機序の検討 : サイボウ ノ シンジュン ゾウショク ユウソウ オ セイギョ スル シンキ テイブンシ カゴウブツ ニヨル ソウショウ チユ ソクシン キジョ ノ ケントウ. (Thesis). Tokai University Repository / 東海大学. Retrieved from https://opac.time.u-tokai.ac.jp/webopac/TD00000121

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

山岡, 華児. “細胞の浸潤・増殖・遊走を制御する新規低分子化合物による創傷治癒促進機序の検討 : サイボウ ノ シンジュン ゾウショク ユウソウ オ セイギョ スル シンキ テイブンシ カゴウブツ ニヨル ソウショウ チユ ソクシン キジョ ノ ケントウ.” 2014. Thesis, Tokai University Repository / 東海大学. Accessed January 26, 2020. https://opac.time.u-tokai.ac.jp/webopac/TD00000121.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

山岡, 華児. “細胞の浸潤・増殖・遊走を制御する新規低分子化合物による創傷治癒促進機序の検討 : サイボウ ノ シンジュン ゾウショク ユウソウ オ セイギョ スル シンキ テイブンシ カゴウブツ ニヨル ソウショウ チユ ソクシン キジョ ノ ケントウ.” 2014. Web. 26 Jan 2020.

Vancouver:

山岡 . 細胞の浸潤・増殖・遊走を制御する新規低分子化合物による創傷治癒促進機序の検討 : サイボウ ノ シンジュン ゾウショク ユウソウ オ セイギョ スル シンキ テイブンシ カゴウブツ ニヨル ソウショウ チユ ソクシン キジョ ノ ケントウ. [Internet] [Thesis]. Tokai University Repository / 東海大学; 2014. [cited 2020 Jan 26]. Available from: https://opac.time.u-tokai.ac.jp/webopac/TD00000121.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

山岡 . 細胞の浸潤・増殖・遊走を制御する新規低分子化合物による創傷治癒促進機序の検討 : サイボウ ノ シンジュン ゾウショク ユウソウ オ セイギョ スル シンキ テイブンシ カゴウブツ ニヨル ソウショウ チユ ソクシン キジョ ノ ケントウ. [Thesis]. Tokai University Repository / 東海大学; 2014. Available from: https://opac.time.u-tokai.ac.jp/webopac/TD00000121

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Case Western Reserve University

2. Wang, Hui. The Roles of a LIM Domain Protein, Hic-5/ARA55, in TGF-ß Signaling in Prostate Cancer Cells.

Degree: PhD, Pharmacology, 2008, Case Western Reserve University

  Findings from our laboratory as well as from others support that transforming growth factor beta (TGF-ß) functions as a critical tumor suppressor of the… (more)

Subjects/Keywords: Biomedical Research; TGF-&946; Smad3; Smad2; Smad7; Hic-5; ARA55

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APA (6th Edition):

Wang, H. (2008). The Roles of a LIM Domain Protein, Hic-5/ARA55, in TGF-ß Signaling in Prostate Cancer Cells. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1220931692

Chicago Manual of Style (16th Edition):

Wang, Hui. “The Roles of a LIM Domain Protein, Hic-5/ARA55, in TGF-ß Signaling in Prostate Cancer Cells.” 2008. Doctoral Dissertation, Case Western Reserve University. Accessed January 26, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=case1220931692.

MLA Handbook (7th Edition):

Wang, Hui. “The Roles of a LIM Domain Protein, Hic-5/ARA55, in TGF-ß Signaling in Prostate Cancer Cells.” 2008. Web. 26 Jan 2020.

Vancouver:

Wang H. The Roles of a LIM Domain Protein, Hic-5/ARA55, in TGF-ß Signaling in Prostate Cancer Cells. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2008. [cited 2020 Jan 26]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1220931692.

Council of Science Editors:

Wang H. The Roles of a LIM Domain Protein, Hic-5/ARA55, in TGF-ß Signaling in Prostate Cancer Cells. [Doctoral Dissertation]. Case Western Reserve University; 2008. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1220931692


University of Toronto

3. Aref, Donya. Canonical TGF-β Pathway Activity is a Predictor of Medulloblastoma Survival and Delineates Putative Precursors in Cerebellar Development.

Degree: 2012, University of Toronto

Medulloblastoma (MB) is the most common pediatric malignant brain tumor. Little is known about aggressive forms of this disease. In order to identify pathways mediating… (more)

Subjects/Keywords: medulloblastoma; smad3; TGF-beta; prognosis; cerebellar development; 0571; 0992

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APA (6th Edition):

Aref, D. (2012). Canonical TGF-β Pathway Activity is a Predictor of Medulloblastoma Survival and Delineates Putative Precursors in Cerebellar Development. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/33325

Chicago Manual of Style (16th Edition):

Aref, Donya. “Canonical TGF-β Pathway Activity is a Predictor of Medulloblastoma Survival and Delineates Putative Precursors in Cerebellar Development.” 2012. Masters Thesis, University of Toronto. Accessed January 26, 2020. http://hdl.handle.net/1807/33325.

MLA Handbook (7th Edition):

Aref, Donya. “Canonical TGF-β Pathway Activity is a Predictor of Medulloblastoma Survival and Delineates Putative Precursors in Cerebellar Development.” 2012. Web. 26 Jan 2020.

Vancouver:

Aref D. Canonical TGF-β Pathway Activity is a Predictor of Medulloblastoma Survival and Delineates Putative Precursors in Cerebellar Development. [Internet] [Masters thesis]. University of Toronto; 2012. [cited 2020 Jan 26]. Available from: http://hdl.handle.net/1807/33325.

Council of Science Editors:

Aref D. Canonical TGF-β Pathway Activity is a Predictor of Medulloblastoma Survival and Delineates Putative Precursors in Cerebellar Development. [Masters Thesis]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/33325


Université Paris-Sud – Paris XI

4. Cailloux, Jérémy. Rôle du système générateur d’espèces réactives de l’oxygène NOX4-p22phox dans la thyroïde humaine : implication dans la prolifération et la différenciation thyroïdienne : Role of the NOX4-p22phox ROS Producing System in the Human Thyroid : Implication in Thyroid Proliferation and Differenciation.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2014, Université Paris-Sud – Paris XI

 Rôle de la NADPH oxydase NOX4 dans la régulation de l'expression du symporteur sodium/iode (NIS) dans le cas du cancer papillaire de la thyroïde (PTC).… (more)

Subjects/Keywords: Thyroïde; BRAF; NIS; ROS; NOX4; Cancer; Captation d’iode; Dédifférenciation; TGF-β; Smad3; PTC; Thyroid; BRAF; NIS; ROS; NOX4; Cancer; Iodide uptake; Dedifferenciation; TGF-β; Smad3; PTC

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APA (6th Edition):

Cailloux, J. (2014). Rôle du système générateur d’espèces réactives de l’oxygène NOX4-p22phox dans la thyroïde humaine : implication dans la prolifération et la différenciation thyroïdienne : Role of the NOX4-p22phox ROS Producing System in the Human Thyroid : Implication in Thyroid Proliferation and Differenciation. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2014PA11T064

Chicago Manual of Style (16th Edition):

Cailloux, Jérémy. “Rôle du système générateur d’espèces réactives de l’oxygène NOX4-p22phox dans la thyroïde humaine : implication dans la prolifération et la différenciation thyroïdienne : Role of the NOX4-p22phox ROS Producing System in the Human Thyroid : Implication in Thyroid Proliferation and Differenciation.” 2014. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed January 26, 2020. http://www.theses.fr/2014PA11T064.

MLA Handbook (7th Edition):

Cailloux, Jérémy. “Rôle du système générateur d’espèces réactives de l’oxygène NOX4-p22phox dans la thyroïde humaine : implication dans la prolifération et la différenciation thyroïdienne : Role of the NOX4-p22phox ROS Producing System in the Human Thyroid : Implication in Thyroid Proliferation and Differenciation.” 2014. Web. 26 Jan 2020.

Vancouver:

Cailloux J. Rôle du système générateur d’espèces réactives de l’oxygène NOX4-p22phox dans la thyroïde humaine : implication dans la prolifération et la différenciation thyroïdienne : Role of the NOX4-p22phox ROS Producing System in the Human Thyroid : Implication in Thyroid Proliferation and Differenciation. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2014. [cited 2020 Jan 26]. Available from: http://www.theses.fr/2014PA11T064.

Council of Science Editors:

Cailloux J. Rôle du système générateur d’espèces réactives de l’oxygène NOX4-p22phox dans la thyroïde humaine : implication dans la prolifération et la différenciation thyroïdienne : Role of the NOX4-p22phox ROS Producing System in the Human Thyroid : Implication in Thyroid Proliferation and Differenciation. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2014. Available from: http://www.theses.fr/2014PA11T064


Case Western Reserve University

5. Ye, Fang. PPAR¿ and Smad2 Mediate Ski Induced Energy Metabolism Shift and Oncogenic Transformation.

Degree: PhD, Biochemistry, 2010, Case Western Reserve University

  Ski is a versatile nuclear oncoprotein that regulates transcriptions of genes involved in multiple biological processes. Ski induces oncogenic transformation of chicken embryo fibroblasts… (more)

Subjects/Keywords: Biochemistry; Ski, TGF-&946; , Smad2, Smad3, PPAR&947; , &946; -oxidation, transformation, metabolism, mitochondria, oxygen consumption

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APA (6th Edition):

Ye, F. (2010). PPAR¿ and Smad2 Mediate Ski Induced Energy Metabolism Shift and Oncogenic Transformation. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1280930750

Chicago Manual of Style (16th Edition):

Ye, Fang. “PPAR¿ and Smad2 Mediate Ski Induced Energy Metabolism Shift and Oncogenic Transformation.” 2010. Doctoral Dissertation, Case Western Reserve University. Accessed January 26, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=case1280930750.

MLA Handbook (7th Edition):

Ye, Fang. “PPAR¿ and Smad2 Mediate Ski Induced Energy Metabolism Shift and Oncogenic Transformation.” 2010. Web. 26 Jan 2020.

Vancouver:

Ye F. PPAR¿ and Smad2 Mediate Ski Induced Energy Metabolism Shift and Oncogenic Transformation. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2010. [cited 2020 Jan 26]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1280930750.

Council of Science Editors:

Ye F. PPAR¿ and Smad2 Mediate Ski Induced Energy Metabolism Shift and Oncogenic Transformation. [Doctoral Dissertation]. Case Western Reserve University; 2010. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1280930750


Case Western Reserve University

6. Yang, Jiayi. TGF-ß/Smad Signaling in Growth Control of Prostate Epithelial Cells.

Degree: PhD, Biochemistry, 2009, Case Western Reserve University

  Several reports from various laboratories, including ours, find that transforming growth factor-ß (TGF-ß) functions as a tumor suppressor of the prostate. Tumor suppression mediated… (more)

Subjects/Keywords: Biochemistry; Biomedical Research; Cellular Biology; Molecular Biology; TGF-¿¿¿¿/SMAD; Survivin; Smad2; Smads; PROSTATE; Smad3; TGF-¿¿¿¿1

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APA (6th Edition):

Yang, J. (2009). TGF-ß/Smad Signaling in Growth Control of Prostate Epithelial Cells. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1228406147

Chicago Manual of Style (16th Edition):

Yang, Jiayi. “TGF-ß/Smad Signaling in Growth Control of Prostate Epithelial Cells.” 2009. Doctoral Dissertation, Case Western Reserve University. Accessed January 26, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=case1228406147.

MLA Handbook (7th Edition):

Yang, Jiayi. “TGF-ß/Smad Signaling in Growth Control of Prostate Epithelial Cells.” 2009. Web. 26 Jan 2020.

Vancouver:

Yang J. TGF-ß/Smad Signaling in Growth Control of Prostate Epithelial Cells. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2009. [cited 2020 Jan 26]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1228406147.

Council of Science Editors:

Yang J. TGF-ß/Smad Signaling in Growth Control of Prostate Epithelial Cells. [Doctoral Dissertation]. Case Western Reserve University; 2009. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1228406147

7. Donato, Tatiani Ayako Goto. Aspectos moleculares do efeito do fator de transformação de crescimento-beta1 (TGF-β1) nas vias de sinalização na biomineralização in vitro.

Degree: PhD, Biologia Celular e Tecidual, 2014, University of São Paulo

Este estudo in vitro teve como objetivo avaliar os efeitos moleculares do TGF-β1, com diferentes períodos de suplementação, sobre a formação do fenótipo osteogênico das… (more)

Subjects/Keywords: In vitro mineralization; Linhagem celular MC3T3-E1; MC3T3-E1 cell line; Mineralização in vitro; Neutralização de receptores de TGF-β 1; Neutralization of TGF-β 1 receptors; Noncollagenous proteins; Proteínas não colágenas; Smad3; Smad3; TGF-β 1; TGF-β 1

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APA (6th Edition):

Donato, T. A. G. (2014). Aspectos moleculares do efeito do fator de transformação de crescimento-beta1 (TGF-β1) nas vias de sinalização na biomineralização in vitro. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/42/42134/tde-26062014-201523/ ;

Chicago Manual of Style (16th Edition):

Donato, Tatiani Ayako Goto. “Aspectos moleculares do efeito do fator de transformação de crescimento-beta1 (TGF-β1) nas vias de sinalização na biomineralização in vitro.” 2014. Doctoral Dissertation, University of São Paulo. Accessed January 26, 2020. http://www.teses.usp.br/teses/disponiveis/42/42134/tde-26062014-201523/ ;.

MLA Handbook (7th Edition):

Donato, Tatiani Ayako Goto. “Aspectos moleculares do efeito do fator de transformação de crescimento-beta1 (TGF-β1) nas vias de sinalização na biomineralização in vitro.” 2014. Web. 26 Jan 2020.

Vancouver:

Donato TAG. Aspectos moleculares do efeito do fator de transformação de crescimento-beta1 (TGF-β1) nas vias de sinalização na biomineralização in vitro. [Internet] [Doctoral dissertation]. University of São Paulo; 2014. [cited 2020 Jan 26]. Available from: http://www.teses.usp.br/teses/disponiveis/42/42134/tde-26062014-201523/ ;.

Council of Science Editors:

Donato TAG. Aspectos moleculares do efeito do fator de transformação de crescimento-beta1 (TGF-β1) nas vias de sinalização na biomineralização in vitro. [Doctoral Dissertation]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/42/42134/tde-26062014-201523/ ;


Case Western Reserve University

8. Chou, Yu-Ting. Cited2, an autoregulated transcriptional modulator, in TGF-beta signaling.

Degree: PhD, Pharmacology, 2006, Case Western Reserve University

 Cited2 [CBP/p300 interacting transactivator with E (Glutamic acid) and D (Aspartic acid) rich C-terminal domain, 2] is a CBP/p300 binding transcription factor without typical DNA… (more)

Subjects/Keywords: Biology, Molecular; Cited2; TGF-beta; MMP9; Smad3; p300

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APA (6th Edition):

Chou, Y. (2006). Cited2, an autoregulated transcriptional modulator, in TGF-beta signaling. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1147147222

Chicago Manual of Style (16th Edition):

Chou, Yu-Ting. “Cited2, an autoregulated transcriptional modulator, in TGF-beta signaling.” 2006. Doctoral Dissertation, Case Western Reserve University. Accessed January 26, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=case1147147222.

MLA Handbook (7th Edition):

Chou, Yu-Ting. “Cited2, an autoregulated transcriptional modulator, in TGF-beta signaling.” 2006. Web. 26 Jan 2020.

Vancouver:

Chou Y. Cited2, an autoregulated transcriptional modulator, in TGF-beta signaling. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2006. [cited 2020 Jan 26]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1147147222.

Council of Science Editors:

Chou Y. Cited2, an autoregulated transcriptional modulator, in TGF-beta signaling. [Doctoral Dissertation]. Case Western Reserve University; 2006. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1147147222


University of Ottawa

9. Putos, Samantha. Repurposing 13-Cis-Retinoic Acid: A Potential Treatment for Aneurysms-Osteoarthritis Syndrome .

Degree: 2015, University of Ottawa

 Approximately 7000 rare disorders exist, affecting 2 percent of Canadians and millions of people worldwide. Given that for many rare diseases only one allele is… (more)

Subjects/Keywords: Loeys-Dietz Syndrome Type 3; Aneurysms-Osteoarthritis Syndrome; SMAD3; 13-cis-retinoic acid; Isotretinoin; Care for Rare; TGF-beta

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APA (6th Edition):

Putos, S. (2015). Repurposing 13-Cis-Retinoic Acid: A Potential Treatment for Aneurysms-Osteoarthritis Syndrome . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/32427

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Putos, Samantha. “Repurposing 13-Cis-Retinoic Acid: A Potential Treatment for Aneurysms-Osteoarthritis Syndrome .” 2015. Thesis, University of Ottawa. Accessed January 26, 2020. http://hdl.handle.net/10393/32427.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Putos, Samantha. “Repurposing 13-Cis-Retinoic Acid: A Potential Treatment for Aneurysms-Osteoarthritis Syndrome .” 2015. Web. 26 Jan 2020.

Vancouver:

Putos S. Repurposing 13-Cis-Retinoic Acid: A Potential Treatment for Aneurysms-Osteoarthritis Syndrome . [Internet] [Thesis]. University of Ottawa; 2015. [cited 2020 Jan 26]. Available from: http://hdl.handle.net/10393/32427.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Putos S. Repurposing 13-Cis-Retinoic Acid: A Potential Treatment for Aneurysms-Osteoarthritis Syndrome . [Thesis]. University of Ottawa; 2015. Available from: http://hdl.handle.net/10393/32427

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. Denis, Jean-Francois. Rôle des Smads lors du processus de régénération chez Ambystoma mexicanum .

Degree: 2017, Université de Montréal

 Les capacités de guérison humaine étant limitées et grandement associées à la fibrose, la possibilité de régénérer tous tissus contribueraient grandement à l’amélioration de la… (more)

Subjects/Keywords: Axolotl; TGF-β; Smad2; Smad3; Régénération

…61 3.3 Texte de l’article - Activation of Smad2 but not Smad3 is required to mediate TGF-β… …signalisation TGF-β/p-Smad3, dont l’expression augmente avec l’âge du tissu musculaire et qui cause de… …2 1.1.1 Voie canonique des TGF-β… …11 1.3 Signalisation TGF-β, essentielle la guérison et pour la régénération… …16 1.3.1 Importance de la signalisation TGF-β lors de la guérison chez les vertébrés… 

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APA (6th Edition):

Denis, J. (2017). Rôle des Smads lors du processus de régénération chez Ambystoma mexicanum . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/19320

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Denis, Jean-Francois. “Rôle des Smads lors du processus de régénération chez Ambystoma mexicanum .” 2017. Thesis, Université de Montréal. Accessed January 26, 2020. http://hdl.handle.net/1866/19320.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Denis, Jean-Francois. “Rôle des Smads lors du processus de régénération chez Ambystoma mexicanum .” 2017. Web. 26 Jan 2020.

Vancouver:

Denis J. Rôle des Smads lors du processus de régénération chez Ambystoma mexicanum . [Internet] [Thesis]. Université de Montréal; 2017. [cited 2020 Jan 26]. Available from: http://hdl.handle.net/1866/19320.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Denis J. Rôle des Smads lors du processus de régénération chez Ambystoma mexicanum . [Thesis]. Université de Montréal; 2017. Available from: http://hdl.handle.net/1866/19320

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

11. Brkic, Jelena. The Role of miR-218-5p and TGF-B Signaling in Human Placenta from Normal and Comprised Pregnancies.

Degree: PhD, Biology, 2017, York University

 Placenta is critical during pregnancy, forming the interface between mother and fetus. A key process in placental development is the differentiation and invasion of extravillous… (more)

Subjects/Keywords: Pathology; Biology; Molecular biology; Pathology; Placenta; Preeclampsia; Trophoblasts; MicroRNA; TGF-beta; Gestational disease; Endovascular trophoblast; Spiral artery remodelling; Smad2; Smad3

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APA (6th Edition):

Brkic, J. (2017). The Role of miR-218-5p and TGF-B Signaling in Human Placenta from Normal and Comprised Pregnancies. (Doctoral Dissertation). York University. Retrieved from http://hdl.handle.net/10315/33520

Chicago Manual of Style (16th Edition):

Brkic, Jelena. “The Role of miR-218-5p and TGF-B Signaling in Human Placenta from Normal and Comprised Pregnancies.” 2017. Doctoral Dissertation, York University. Accessed January 26, 2020. http://hdl.handle.net/10315/33520.

MLA Handbook (7th Edition):

Brkic, Jelena. “The Role of miR-218-5p and TGF-B Signaling in Human Placenta from Normal and Comprised Pregnancies.” 2017. Web. 26 Jan 2020.

Vancouver:

Brkic J. The Role of miR-218-5p and TGF-B Signaling in Human Placenta from Normal and Comprised Pregnancies. [Internet] [Doctoral dissertation]. York University; 2017. [cited 2020 Jan 26]. Available from: http://hdl.handle.net/10315/33520.

Council of Science Editors:

Brkic J. The Role of miR-218-5p and TGF-B Signaling in Human Placenta from Normal and Comprised Pregnancies. [Doctoral Dissertation]. York University; 2017. Available from: http://hdl.handle.net/10315/33520


The Ohio State University

12. Liu, Ye. Generation and utilization of knockout mice to elucidate the functions of the TGF-ß pathway in mammalian endodermal specification and placental development.

Degree: PhD, Molecular, Cellular, and Developmental Biology, 2006, The Ohio State University

  Ligands of the transforming growth factor beta (TGF-ß) superfamily are involved in numerous developmental and disease processes. TGF-ß, Activins, and Nodal ligands operate through… (more)

Subjects/Keywords: Biology, Genetics; TGF-&946; , Smad2, Smad3, Craniofacial, liver, Smif, mRNA decapping

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APA (6th Edition):

Liu, Y. (2006). Generation and utilization of knockout mice to elucidate the functions of the TGF-ß pathway in mammalian endodermal specification and placental development. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1158673346

Chicago Manual of Style (16th Edition):

Liu, Ye. “Generation and utilization of knockout mice to elucidate the functions of the TGF-ß pathway in mammalian endodermal specification and placental development.” 2006. Doctoral Dissertation, The Ohio State University. Accessed January 26, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1158673346.

MLA Handbook (7th Edition):

Liu, Ye. “Generation and utilization of knockout mice to elucidate the functions of the TGF-ß pathway in mammalian endodermal specification and placental development.” 2006. Web. 26 Jan 2020.

Vancouver:

Liu Y. Generation and utilization of knockout mice to elucidate the functions of the TGF-ß pathway in mammalian endodermal specification and placental development. [Internet] [Doctoral dissertation]. The Ohio State University; 2006. [cited 2020 Jan 26]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1158673346.

Council of Science Editors:

Liu Y. Generation and utilization of knockout mice to elucidate the functions of the TGF-ß pathway in mammalian endodermal specification and placental development. [Doctoral Dissertation]. The Ohio State University; 2006. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1158673346

13. ONG HONGQIAN ESTHER. DISTINGUISHING SMAD2 and SMAD3-DRIVEN EFFECTS OF THE TGF-? SIGNALLING IN A METASTATIC BREAST CANCER MODEL.

Degree: 2017, National University of Singapore

Subjects/Keywords: TGF-B; Smad2; Smad3; breast cancer; rna-seq; chip-seq

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APA (6th Edition):

ESTHER, O. H. (2017). DISTINGUISHING SMAD2 and SMAD3-DRIVEN EFFECTS OF THE TGF-? SIGNALLING IN A METASTATIC BREAST CANCER MODEL. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/135499

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

ESTHER, ONG HONGQIAN. “DISTINGUISHING SMAD2 and SMAD3-DRIVEN EFFECTS OF THE TGF-? SIGNALLING IN A METASTATIC BREAST CANCER MODEL.” 2017. Thesis, National University of Singapore. Accessed January 26, 2020. http://scholarbank.nus.edu.sg/handle/10635/135499.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

ESTHER, ONG HONGQIAN. “DISTINGUISHING SMAD2 and SMAD3-DRIVEN EFFECTS OF THE TGF-? SIGNALLING IN A METASTATIC BREAST CANCER MODEL.” 2017. Web. 26 Jan 2020.

Vancouver:

ESTHER OH. DISTINGUISHING SMAD2 and SMAD3-DRIVEN EFFECTS OF THE TGF-? SIGNALLING IN A METASTATIC BREAST CANCER MODEL. [Internet] [Thesis]. National University of Singapore; 2017. [cited 2020 Jan 26]. Available from: http://scholarbank.nus.edu.sg/handle/10635/135499.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

ESTHER OH. DISTINGUISHING SMAD2 and SMAD3-DRIVEN EFFECTS OF THE TGF-? SIGNALLING IN A METASTATIC BREAST CANCER MODEL. [Thesis]. National University of Singapore; 2017. Available from: http://scholarbank.nus.edu.sg/handle/10635/135499

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

14. Samanta, Debangshu. Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer.

Degree: PhD, Cancer Biology, 2012, Vanderbilt University

 Inactivating mutations in TGF-©¬ receptors and Smad signal transducers that contribute to resistance to TGF-©¬, are associated with only very small number of NSCLC. The… (more)

Subjects/Keywords: Smoking; Lung cancer; Smad3; Chemoresistance

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APA (6th Edition):

Samanta, D. (2012). Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-04122012-114031/ ;

Chicago Manual of Style (16th Edition):

Samanta, Debangshu. “Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed January 26, 2020. http://etd.library.vanderbilt.edu/available/etd-04122012-114031/ ;.

MLA Handbook (7th Edition):

Samanta, Debangshu. “Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer.” 2012. Web. 26 Jan 2020.

Vancouver:

Samanta D. Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2020 Jan 26]. Available from: http://etd.library.vanderbilt.edu/available/etd-04122012-114031/ ;.

Council of Science Editors:

Samanta D. Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://etd.library.vanderbilt.edu/available/etd-04122012-114031/ ;


Universidad Andrés Bello

15. Mosquera Solorza, Javiera Andrea. Expresión de Pirin en tumores de distintas localizaciones anatómicas y su relación con la presencia del virus papiloma humano (HPV) .

Degree: 2015, Universidad Andrés Bello

 En Chile, el cáncer es una patología muy relevante ocupando el segundo lugar como causa de muerte en la población. Existen múltiples factores de riesgo… (more)

Subjects/Keywords: Papillomavirus; Carcinoma de Células Escamosas; Pirin; HPV; Chile

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APA (6th Edition):

Mosquera Solorza, J. A. (2015). Expresión de Pirin en tumores de distintas localizaciones anatómicas y su relación con la presencia del virus papiloma humano (HPV) . (Thesis). Universidad Andrés Bello. Retrieved from http://repositorio.unab.cl/xmlui/handle/ria/3330

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mosquera Solorza, Javiera Andrea. “Expresión de Pirin en tumores de distintas localizaciones anatómicas y su relación con la presencia del virus papiloma humano (HPV) .” 2015. Thesis, Universidad Andrés Bello. Accessed January 26, 2020. http://repositorio.unab.cl/xmlui/handle/ria/3330.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mosquera Solorza, Javiera Andrea. “Expresión de Pirin en tumores de distintas localizaciones anatómicas y su relación con la presencia del virus papiloma humano (HPV) .” 2015. Web. 26 Jan 2020.

Vancouver:

Mosquera Solorza JA. Expresión de Pirin en tumores de distintas localizaciones anatómicas y su relación con la presencia del virus papiloma humano (HPV) . [Internet] [Thesis]. Universidad Andrés Bello; 2015. [cited 2020 Jan 26]. Available from: http://repositorio.unab.cl/xmlui/handle/ria/3330.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mosquera Solorza JA. Expresión de Pirin en tumores de distintas localizaciones anatómicas y su relación con la presencia del virus papiloma humano (HPV) . [Thesis]. Universidad Andrés Bello; 2015. Available from: http://repositorio.unab.cl/xmlui/handle/ria/3330

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Georgia State University

16. Rehmani, Imran J. Studying the DNA Binding and Conformation of Metal-Binding Site Mutations in Pirin.

Degree: MS, Chemistry, 2012, Georgia State University

  The transcription factor NF-κB interacts with many other co-regulator proteins that modulate its binding and transcriptional activity. One of these co-regulators, Pirin, is an… (more)

Subjects/Keywords: Pirin; NF-κB; Bcl-3; DNA; Metalloproteins; Transcription regulation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rehmani, I. J. (2012). Studying the DNA Binding and Conformation of Metal-Binding Site Mutations in Pirin. (Thesis). Georgia State University. Retrieved from https://scholarworks.gsu.edu/chemistry_theses/53

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rehmani, Imran J. “Studying the DNA Binding and Conformation of Metal-Binding Site Mutations in Pirin.” 2012. Thesis, Georgia State University. Accessed January 26, 2020. https://scholarworks.gsu.edu/chemistry_theses/53.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rehmani, Imran J. “Studying the DNA Binding and Conformation of Metal-Binding Site Mutations in Pirin.” 2012. Web. 26 Jan 2020.

Vancouver:

Rehmani IJ. Studying the DNA Binding and Conformation of Metal-Binding Site Mutations in Pirin. [Internet] [Thesis]. Georgia State University; 2012. [cited 2020 Jan 26]. Available from: https://scholarworks.gsu.edu/chemistry_theses/53.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rehmani IJ. Studying the DNA Binding and Conformation of Metal-Binding Site Mutations in Pirin. [Thesis]. Georgia State University; 2012. Available from: https://scholarworks.gsu.edu/chemistry_theses/53

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Georgia State University

17. Adeniran, Charles. Pirin Allosterically Modulates The Dynamics And Interactions Of The κB DNA In The NF-κB Supramolecular Complex.

Degree: MS, Chemistry, 2017, Georgia State University

  The NF-κB family of transcription factors controls a number of essential cellular functions. Pirin is a non-heme iron (Fe) redox specific co-regulator of NF-κB… (more)

Subjects/Keywords: NF-κB; Pirin; DNA; Transcription Factor; Precursor Protein; Iron

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APA (6th Edition):

Adeniran, C. (2017). Pirin Allosterically Modulates The Dynamics And Interactions Of The κB DNA In The NF-κB Supramolecular Complex. (Thesis). Georgia State University. Retrieved from https://scholarworks.gsu.edu/chemistry_theses/108

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Adeniran, Charles. “Pirin Allosterically Modulates The Dynamics And Interactions Of The κB DNA In The NF-κB Supramolecular Complex.” 2017. Thesis, Georgia State University. Accessed January 26, 2020. https://scholarworks.gsu.edu/chemistry_theses/108.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Adeniran, Charles. “Pirin Allosterically Modulates The Dynamics And Interactions Of The κB DNA In The NF-κB Supramolecular Complex.” 2017. Web. 26 Jan 2020.

Vancouver:

Adeniran C. Pirin Allosterically Modulates The Dynamics And Interactions Of The κB DNA In The NF-κB Supramolecular Complex. [Internet] [Thesis]. Georgia State University; 2017. [cited 2020 Jan 26]. Available from: https://scholarworks.gsu.edu/chemistry_theses/108.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Adeniran C. Pirin Allosterically Modulates The Dynamics And Interactions Of The κB DNA In The NF-κB Supramolecular Complex. [Thesis]. Georgia State University; 2017. Available from: https://scholarworks.gsu.edu/chemistry_theses/108

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Diego

18. Warner, Lindsey Marie. A Non-redundant Role for Thymic Epithelial MicroRNA-155 in the Generation of Tregs.

Degree: Biology, 2017, University of California – San Diego

 miR-155 is a non-coding RNA that is highly upregulated in regulatory T-cells (Tregs), the adaptive immune cells specialized in suppressing autoreactive T cells in order… (more)

Subjects/Keywords: Immunology; Development; miR-155; mTEC; SMAD3; Treg

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Warner, L. M. (2017). A Non-redundant Role for Thymic Epithelial MicroRNA-155 in the Generation of Tregs. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/2q68h4j3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Warner, Lindsey Marie. “A Non-redundant Role for Thymic Epithelial MicroRNA-155 in the Generation of Tregs.” 2017. Thesis, University of California – San Diego. Accessed January 26, 2020. http://www.escholarship.org/uc/item/2q68h4j3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Warner, Lindsey Marie. “A Non-redundant Role for Thymic Epithelial MicroRNA-155 in the Generation of Tregs.” 2017. Web. 26 Jan 2020.

Vancouver:

Warner LM. A Non-redundant Role for Thymic Epithelial MicroRNA-155 in the Generation of Tregs. [Internet] [Thesis]. University of California – San Diego; 2017. [cited 2020 Jan 26]. Available from: http://www.escholarship.org/uc/item/2q68h4j3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Warner LM. A Non-redundant Role for Thymic Epithelial MicroRNA-155 in the Generation of Tregs. [Thesis]. University of California – San Diego; 2017. Available from: http://www.escholarship.org/uc/item/2q68h4j3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Sherbrooke

19. Arsenault, Dominique. Rôle du microenvironnement hypoxique dans la formation des métastases : impact de la relocalisation intracellulaire de la furine dans l'invasion cellulaire .

Degree: 2013, Université de Sherbrooke

 La compréhension des mécanismes impliqués dans la formation des métastases est l’un des défis majeurs de la recherche sur le cancer. En effet, la formation… (more)

Subjects/Keywords: TGFß; Smad3; Invasion; Invadopode; Hypoxie; HDAC6; Furine

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APA (6th Edition):

Arsenault, D. (2013). Rôle du microenvironnement hypoxique dans la formation des métastases : impact de la relocalisation intracellulaire de la furine dans l'invasion cellulaire . (Doctoral Dissertation). Université de Sherbrooke. Retrieved from http://hdl.handle.net/11143/6221

Chicago Manual of Style (16th Edition):

Arsenault, Dominique. “Rôle du microenvironnement hypoxique dans la formation des métastases : impact de la relocalisation intracellulaire de la furine dans l'invasion cellulaire .” 2013. Doctoral Dissertation, Université de Sherbrooke. Accessed January 26, 2020. http://hdl.handle.net/11143/6221.

MLA Handbook (7th Edition):

Arsenault, Dominique. “Rôle du microenvironnement hypoxique dans la formation des métastases : impact de la relocalisation intracellulaire de la furine dans l'invasion cellulaire .” 2013. Web. 26 Jan 2020.

Vancouver:

Arsenault D. Rôle du microenvironnement hypoxique dans la formation des métastases : impact de la relocalisation intracellulaire de la furine dans l'invasion cellulaire . [Internet] [Doctoral dissertation]. Université de Sherbrooke; 2013. [cited 2020 Jan 26]. Available from: http://hdl.handle.net/11143/6221.

Council of Science Editors:

Arsenault D. Rôle du microenvironnement hypoxique dans la formation des métastases : impact de la relocalisation intracellulaire de la furine dans l'invasion cellulaire . [Doctoral Dissertation]. Université de Sherbrooke; 2013. Available from: http://hdl.handle.net/11143/6221


University of Toronto

20. Tram, Eric. Identification of Germline Alterations in the Mad-homology 2 (MH2) Domain of SMAD3 and SMAD4 In Breast Cancer Susceptibility.

Degree: 2012, University of Toronto

A feature of neoplastic cells is that mutations in the key intermediates of TGF-β signaling contribute to the loss of sensitivity to its anti-tumor effects.… (more)

Subjects/Keywords: Breast cancer; germline mutations; SMAD3; SMAD4; Bioinformatics

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APA (6th Edition):

Tram, E. (2012). Identification of Germline Alterations in the Mad-homology 2 (MH2) Domain of SMAD3 and SMAD4 In Breast Cancer Susceptibility. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/33713

Chicago Manual of Style (16th Edition):

Tram, Eric. “Identification of Germline Alterations in the Mad-homology 2 (MH2) Domain of SMAD3 and SMAD4 In Breast Cancer Susceptibility.” 2012. Masters Thesis, University of Toronto. Accessed January 26, 2020. http://hdl.handle.net/1807/33713.

MLA Handbook (7th Edition):

Tram, Eric. “Identification of Germline Alterations in the Mad-homology 2 (MH2) Domain of SMAD3 and SMAD4 In Breast Cancer Susceptibility.” 2012. Web. 26 Jan 2020.

Vancouver:

Tram E. Identification of Germline Alterations in the Mad-homology 2 (MH2) Domain of SMAD3 and SMAD4 In Breast Cancer Susceptibility. [Internet] [Masters thesis]. University of Toronto; 2012. [cited 2020 Jan 26]. Available from: http://hdl.handle.net/1807/33713.

Council of Science Editors:

Tram E. Identification of Germline Alterations in the Mad-homology 2 (MH2) Domain of SMAD3 and SMAD4 In Breast Cancer Susceptibility. [Masters Thesis]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/33713


University of Illinois – Chicago

21. Orozco-Nunnelly, Danielle A. Determining the Role of Pirin1 in Light-Regulated Growth and Cellular Responses in Arabidopsis thaliana.

Degree: 2015, University of Illinois – Chicago

 As photoautotrophic and sessile organisms, plants are both reliant upon, and vulnerable to abiotic signals. Different wavelengths of light provide information for plant development, but… (more)

Subjects/Keywords: Arabidopsis; G protein pathway; Light signaling; Photomorphogenesis; Pirin; Plant transgenes; Posttranslational modification; Quercetin; Regulation of transcript and protein; Seedling development

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APA (6th Edition):

Orozco-Nunnelly, D. A. (2015). Determining the Role of Pirin1 in Light-Regulated Growth and Cellular Responses in Arabidopsis thaliana. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/19577

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Orozco-Nunnelly, Danielle A. “Determining the Role of Pirin1 in Light-Regulated Growth and Cellular Responses in Arabidopsis thaliana.” 2015. Thesis, University of Illinois – Chicago. Accessed January 26, 2020. http://hdl.handle.net/10027/19577.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Orozco-Nunnelly, Danielle A. “Determining the Role of Pirin1 in Light-Regulated Growth and Cellular Responses in Arabidopsis thaliana.” 2015. Web. 26 Jan 2020.

Vancouver:

Orozco-Nunnelly DA. Determining the Role of Pirin1 in Light-Regulated Growth and Cellular Responses in Arabidopsis thaliana. [Internet] [Thesis]. University of Illinois – Chicago; 2015. [cited 2020 Jan 26]. Available from: http://hdl.handle.net/10027/19577.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Orozco-Nunnelly DA. Determining the Role of Pirin1 in Light-Regulated Growth and Cellular Responses in Arabidopsis thaliana. [Thesis]. University of Illinois – Chicago; 2015. Available from: http://hdl.handle.net/10027/19577

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

22. Turumtay, Halbay. Cell wall engineering for better conversion of lignocellulosic biomass.

Degree: 2014, Ghent University

 The objective of our research was to use the flexibility of the cell wall to its extremes by modifying its composition while avoiding deleterious effects… (more)

Subjects/Keywords: Biology and Life Sciences; Cell Wall; Bio-fuels; Cell wall degrading enzymes; Galactanase; Lignocellulosic biomass; Pirin; NODC

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APA (6th Edition):

Turumtay, H. (2014). Cell wall engineering for better conversion of lignocellulosic biomass. (Thesis). Ghent University. Retrieved from http://hdl.handle.net/1854/LU-5750077

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Turumtay, Halbay. “Cell wall engineering for better conversion of lignocellulosic biomass.” 2014. Thesis, Ghent University. Accessed January 26, 2020. http://hdl.handle.net/1854/LU-5750077.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Turumtay, Halbay. “Cell wall engineering for better conversion of lignocellulosic biomass.” 2014. Web. 26 Jan 2020.

Vancouver:

Turumtay H. Cell wall engineering for better conversion of lignocellulosic biomass. [Internet] [Thesis]. Ghent University; 2014. [cited 2020 Jan 26]. Available from: http://hdl.handle.net/1854/LU-5750077.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Turumtay H. Cell wall engineering for better conversion of lignocellulosic biomass. [Thesis]. Ghent University; 2014. Available from: http://hdl.handle.net/1854/LU-5750077

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Connecticut

23. Parikh, Shan. Investigation of miRNAs Expression in a Citron-Kinase Mutant Model of Microcephaly.

Degree: MS, Physiology and Neurobiology, 2011, University of Connecticut

  Mutation of Citron-Kinase (Cit-K) in rodents causes substantial reductions in the number of neurons generated in the CNS and results in a primary microcephaly-like… (more)

Subjects/Keywords: Investigation of miRNAs Expression; Citron-Kinase SMAD3; Nr3c1

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Parikh, S. (2011). Investigation of miRNAs Expression in a Citron-Kinase Mutant Model of Microcephaly. (Masters Thesis). University of Connecticut. Retrieved from https://opencommons.uconn.edu/gs_theses/99

Chicago Manual of Style (16th Edition):

Parikh, Shan. “Investigation of miRNAs Expression in a Citron-Kinase Mutant Model of Microcephaly.” 2011. Masters Thesis, University of Connecticut. Accessed January 26, 2020. https://opencommons.uconn.edu/gs_theses/99.

MLA Handbook (7th Edition):

Parikh, Shan. “Investigation of miRNAs Expression in a Citron-Kinase Mutant Model of Microcephaly.” 2011. Web. 26 Jan 2020.

Vancouver:

Parikh S. Investigation of miRNAs Expression in a Citron-Kinase Mutant Model of Microcephaly. [Internet] [Masters thesis]. University of Connecticut; 2011. [cited 2020 Jan 26]. Available from: https://opencommons.uconn.edu/gs_theses/99.

Council of Science Editors:

Parikh S. Investigation of miRNAs Expression in a Citron-Kinase Mutant Model of Microcephaly. [Masters Thesis]. University of Connecticut; 2011. Available from: https://opencommons.uconn.edu/gs_theses/99


Universitat Pompeu Fabra

24. Bosch Gutiérrez, Almudena. Role of Ring1B in ephitelial to mesenchimal transition, invasion and migration of mammary epithelial cells.

Degree: Departament de Ciències Experimentals i de la Salut, 2009, Universitat Pompeu Fabra

 Las proteínas del grupo Polycomb (PcG) forman complejos modificadores de la cromatina esenciales en el desarrollo embrionario y en la renovación de las células madre,… (more)

Subjects/Keywords: Tgf-;

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bosch Gutiérrez, A. (2009). Role of Ring1B in ephitelial to mesenchimal transition, invasion and migration of mammary epithelial cells. (Thesis). Universitat Pompeu Fabra. Retrieved from http://hdl.handle.net/10803/7230

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bosch Gutiérrez, Almudena. “Role of Ring1B in ephitelial to mesenchimal transition, invasion and migration of mammary epithelial cells.” 2009. Thesis, Universitat Pompeu Fabra. Accessed January 26, 2020. http://hdl.handle.net/10803/7230.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bosch Gutiérrez, Almudena. “Role of Ring1B in ephitelial to mesenchimal transition, invasion and migration of mammary epithelial cells.” 2009. Web. 26 Jan 2020.

Vancouver:

Bosch Gutiérrez A. Role of Ring1B in ephitelial to mesenchimal transition, invasion and migration of mammary epithelial cells. [Internet] [Thesis]. Universitat Pompeu Fabra; 2009. [cited 2020 Jan 26]. Available from: http://hdl.handle.net/10803/7230.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bosch Gutiérrez A. Role of Ring1B in ephitelial to mesenchimal transition, invasion and migration of mammary epithelial cells. [Thesis]. Universitat Pompeu Fabra; 2009. Available from: http://hdl.handle.net/10803/7230

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Rochester

25. Shen, Jie; Chen, Di (1955 - ). Loss of TGF-B Signaling Leads to Up-Regulation of Mmp13 and Adamts5 and the Development of Osteoarthritis.

Degree: PhD, 2012, University of Rochester

 Osteoarthritis (OA) is a common degenerative joint disease affecting more than 20% of American adults. The pathogenesis of OA is poorly understood and currently there… (more)

Subjects/Keywords: TGF-B; Mmp13; Osteoarthritis; Adamts5

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APA (6th Edition):

Shen, Jie; Chen, D. (. -. ). (2012). Loss of TGF-B Signaling Leads to Up-Regulation of Mmp13 and Adamts5 and the Development of Osteoarthritis. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/25525

Chicago Manual of Style (16th Edition):

Shen, Jie; Chen, Di (1955 - ). “Loss of TGF-B Signaling Leads to Up-Regulation of Mmp13 and Adamts5 and the Development of Osteoarthritis.” 2012. Doctoral Dissertation, University of Rochester. Accessed January 26, 2020. http://hdl.handle.net/1802/25525.

MLA Handbook (7th Edition):

Shen, Jie; Chen, Di (1955 - ). “Loss of TGF-B Signaling Leads to Up-Regulation of Mmp13 and Adamts5 and the Development of Osteoarthritis.” 2012. Web. 26 Jan 2020.

Vancouver:

Shen, Jie; Chen D(-). Loss of TGF-B Signaling Leads to Up-Regulation of Mmp13 and Adamts5 and the Development of Osteoarthritis. [Internet] [Doctoral dissertation]. University of Rochester; 2012. [cited 2020 Jan 26]. Available from: http://hdl.handle.net/1802/25525.

Council of Science Editors:

Shen, Jie; Chen D(-). Loss of TGF-B Signaling Leads to Up-Regulation of Mmp13 and Adamts5 and the Development of Osteoarthritis. [Doctoral Dissertation]. University of Rochester; 2012. Available from: http://hdl.handle.net/1802/25525


NSYSU

26. Huang, Sz-yang. Identification of target genes of SMAD4 signaling network inhibit pancreatic tumor metastasis and chemoresistance.

Degree: Master, Institute of Biomedical Sciences, 2010, NSYSU

 Pancreatic ductal adenocarcinoma (PDAC) is one of the most insidious forms of cancer whose incidence nearly equals its death rate. Despite extensive research studies, no… (more)

Subjects/Keywords: TGF; PDAC; HIF-1

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APA (6th Edition):

Huang, S. (2010). Identification of target genes of SMAD4 signaling network inhibit pancreatic tumor metastasis and chemoresistance. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0708110-141641

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Huang, Sz-yang. “Identification of target genes of SMAD4 signaling network inhibit pancreatic tumor metastasis and chemoresistance.” 2010. Thesis, NSYSU. Accessed January 26, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0708110-141641.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Huang, Sz-yang. “Identification of target genes of SMAD4 signaling network inhibit pancreatic tumor metastasis and chemoresistance.” 2010. Web. 26 Jan 2020.

Vancouver:

Huang S. Identification of target genes of SMAD4 signaling network inhibit pancreatic tumor metastasis and chemoresistance. [Internet] [Thesis]. NSYSU; 2010. [cited 2020 Jan 26]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0708110-141641.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Huang S. Identification of target genes of SMAD4 signaling network inhibit pancreatic tumor metastasis and chemoresistance. [Thesis]. NSYSU; 2010. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0708110-141641

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université Catholique de Louvain

27. Gauthy, Emilie. GARP : how it controls TGF-b1 production in human regulatory T cells, and how its expression is regulated at transcriptional and post-transcriptional levels.

Degree: 2013, Université Catholique de Louvain

Regulatory T lymphocytes (Tregs) are a subset of CD4+ T cells specialised in the inhibition of immune responses. Tregs are required for the maintenance of… (more)

Subjects/Keywords: GARP; TGF-b; miRNA; Treg

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gauthy, E. (2013). GARP : how it controls TGF-b1 production in human regulatory T cells, and how its expression is regulated at transcriptional and post-transcriptional levels. (Thesis). Université Catholique de Louvain. Retrieved from http://hdl.handle.net/2078.1/135384

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gauthy, Emilie. “GARP : how it controls TGF-b1 production in human regulatory T cells, and how its expression is regulated at transcriptional and post-transcriptional levels.” 2013. Thesis, Université Catholique de Louvain. Accessed January 26, 2020. http://hdl.handle.net/2078.1/135384.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gauthy, Emilie. “GARP : how it controls TGF-b1 production in human regulatory T cells, and how its expression is regulated at transcriptional and post-transcriptional levels.” 2013. Web. 26 Jan 2020.

Vancouver:

Gauthy E. GARP : how it controls TGF-b1 production in human regulatory T cells, and how its expression is regulated at transcriptional and post-transcriptional levels. [Internet] [Thesis]. Université Catholique de Louvain; 2013. [cited 2020 Jan 26]. Available from: http://hdl.handle.net/2078.1/135384.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gauthy E. GARP : how it controls TGF-b1 production in human regulatory T cells, and how its expression is regulated at transcriptional and post-transcriptional levels. [Thesis]. Université Catholique de Louvain; 2013. Available from: http://hdl.handle.net/2078.1/135384

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

28. Deheshi, Benjamin Michael. Alterations in Tgf-β Signaling Mediate the Biologic Behaviour of Osteosarcoma Cell Lines.

Degree: 2010, University of Toronto

Osteosarcoma is a mesenchymal tumour of bone common among children and young adults. Prognosis is poor if metastases are present at diagnosis. The transforming growth… (more)

Subjects/Keywords: Osteosarcoma; TGF-β; 0379

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APA (6th Edition):

Deheshi, B. M. (2010). Alterations in Tgf-β Signaling Mediate the Biologic Behaviour of Osteosarcoma Cell Lines. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/25551

Chicago Manual of Style (16th Edition):

Deheshi, Benjamin Michael. “Alterations in Tgf-β Signaling Mediate the Biologic Behaviour of Osteosarcoma Cell Lines.” 2010. Masters Thesis, University of Toronto. Accessed January 26, 2020. http://hdl.handle.net/1807/25551.

MLA Handbook (7th Edition):

Deheshi, Benjamin Michael. “Alterations in Tgf-β Signaling Mediate the Biologic Behaviour of Osteosarcoma Cell Lines.” 2010. Web. 26 Jan 2020.

Vancouver:

Deheshi BM. Alterations in Tgf-β Signaling Mediate the Biologic Behaviour of Osteosarcoma Cell Lines. [Internet] [Masters thesis]. University of Toronto; 2010. [cited 2020 Jan 26]. Available from: http://hdl.handle.net/1807/25551.

Council of Science Editors:

Deheshi BM. Alterations in Tgf-β Signaling Mediate the Biologic Behaviour of Osteosarcoma Cell Lines. [Masters Thesis]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/25551


University of Guelph

29. Avery-Cooper, Geordon James. The Potential Role of Integrin Regulation by Par6 in TGF-beta-induced Apoptosis .

Degree: 2011, University of Guelph

 The Par6-polarity pathway regulates breast cancer metastasis, and more recently has been shown to regulate transforming growth factor β (TGFβ)-induced apoptosis. Integrins may mediate the… (more)

Subjects/Keywords: Par6; Polarity; apoptosis; TGF-beta

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APA (6th Edition):

Avery-Cooper, G. J. (2011). The Potential Role of Integrin Regulation by Par6 in TGF-beta-induced Apoptosis . (Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/2874

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Avery-Cooper, Geordon James. “The Potential Role of Integrin Regulation by Par6 in TGF-beta-induced Apoptosis .” 2011. Thesis, University of Guelph. Accessed January 26, 2020. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/2874.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Avery-Cooper, Geordon James. “The Potential Role of Integrin Regulation by Par6 in TGF-beta-induced Apoptosis .” 2011. Web. 26 Jan 2020.

Vancouver:

Avery-Cooper GJ. The Potential Role of Integrin Regulation by Par6 in TGF-beta-induced Apoptosis . [Internet] [Thesis]. University of Guelph; 2011. [cited 2020 Jan 26]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/2874.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Avery-Cooper GJ. The Potential Role of Integrin Regulation by Par6 in TGF-beta-induced Apoptosis . [Thesis]. University of Guelph; 2011. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/2874

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

30. ZAIATZ BITTENCOURT, VANESSA. Investigating the impact of natural killer (NK) cell metabolism on NK cell effector function.

Degree: School of Biochemistry & Immunology. Discipline of Biochemistry, 2019, Trinity College Dublin

 It is now known that metabolism, in addition to providing energy and biochemical building blocks, also regulates immune cell function. Over the last few years,… (more)

Subjects/Keywords: immunometabolism; nk cells; tgf beta

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APA (6th Edition):

ZAIATZ BITTENCOURT, V. (2019). Investigating the impact of natural killer (NK) cell metabolism on NK cell effector function. (Thesis). Trinity College Dublin. Retrieved from http://hdl.handle.net/2262/85996

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

ZAIATZ BITTENCOURT, VANESSA. “Investigating the impact of natural killer (NK) cell metabolism on NK cell effector function.” 2019. Thesis, Trinity College Dublin. Accessed January 26, 2020. http://hdl.handle.net/2262/85996.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

ZAIATZ BITTENCOURT, VANESSA. “Investigating the impact of natural killer (NK) cell metabolism on NK cell effector function.” 2019. Web. 26 Jan 2020.

Vancouver:

ZAIATZ BITTENCOURT V. Investigating the impact of natural killer (NK) cell metabolism on NK cell effector function. [Internet] [Thesis]. Trinity College Dublin; 2019. [cited 2020 Jan 26]. Available from: http://hdl.handle.net/2262/85996.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

ZAIATZ BITTENCOURT V. Investigating the impact of natural killer (NK) cell metabolism on NK cell effector function. [Thesis]. Trinity College Dublin; 2019. Available from: http://hdl.handle.net/2262/85996

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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