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Dept: Microbiology and Immunology

You searched for subject:( 1 5 ). Showing records 1 – 30 of 43 total matches.

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Vanderbilt University

1. Yufenyuy, Ernest Limnyuy. The Role Of Intersubunit Interfaces In HIV-1 Capsid Assembly And Stability.

Degree: PhD, Microbiology and Immunology, 2013, Vanderbilt University

 This dissertation discusses the role of intersubunit interfaces in HIV-1 capsid assembly and stability. In this study I showed the presence of both the NTD-CTD… (more)

Subjects/Keywords: Assembly; HIV-1; Uncoating; Capsid

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APA (6th Edition):

Yufenyuy, E. L. (2013). The Role Of Intersubunit Interfaces In HIV-1 Capsid Assembly And Stability. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-03262013-155906/ ;

Chicago Manual of Style (16th Edition):

Yufenyuy, Ernest Limnyuy. “The Role Of Intersubunit Interfaces In HIV-1 Capsid Assembly And Stability.” 2013. Doctoral Dissertation, Vanderbilt University. Accessed October 23, 2019. http://etd.library.vanderbilt.edu/available/etd-03262013-155906/ ;.

MLA Handbook (7th Edition):

Yufenyuy, Ernest Limnyuy. “The Role Of Intersubunit Interfaces In HIV-1 Capsid Assembly And Stability.” 2013. Web. 23 Oct 2019.

Vancouver:

Yufenyuy EL. The Role Of Intersubunit Interfaces In HIV-1 Capsid Assembly And Stability. [Internet] [Doctoral dissertation]. Vanderbilt University; 2013. [cited 2019 Oct 23]. Available from: http://etd.library.vanderbilt.edu/available/etd-03262013-155906/ ;.

Council of Science Editors:

Yufenyuy EL. The Role Of Intersubunit Interfaces In HIV-1 Capsid Assembly And Stability. [Doctoral Dissertation]. Vanderbilt University; 2013. Available from: http://etd.library.vanderbilt.edu/available/etd-03262013-155906/ ;


Penn State University

2. O'Regan, Kevin Joseph. INSIGHTS INTO HERPES SIMPLEX VIRUS TYPE 1 ASSEMBLY: PROTEIN INTERACTIONS AND VIRION INCORPORATION DETERMINANTS OF THE TEGUMENT PROTEIN VP22.

Degree: PhD, Microbiology and Immunology, 2008, Penn State University

 Herpes simplex virus type 1 (HSV-1) assembly involves a complex sequence of events occurring within numerous cellular compartments that culminates in the formation of virions… (more)

Subjects/Keywords: Herpes simplex virus type 1: HSV-1: VP22: VP16: Gl

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APA (6th Edition):

O'Regan, K. J. (2008). INSIGHTS INTO HERPES SIMPLEX VIRUS TYPE 1 ASSEMBLY: PROTEIN INTERACTIONS AND VIRION INCORPORATION DETERMINANTS OF THE TEGUMENT PROTEIN VP22. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/8115

Chicago Manual of Style (16th Edition):

O'Regan, Kevin Joseph. “INSIGHTS INTO HERPES SIMPLEX VIRUS TYPE 1 ASSEMBLY: PROTEIN INTERACTIONS AND VIRION INCORPORATION DETERMINANTS OF THE TEGUMENT PROTEIN VP22.” 2008. Doctoral Dissertation, Penn State University. Accessed October 23, 2019. https://etda.libraries.psu.edu/catalog/8115.

MLA Handbook (7th Edition):

O'Regan, Kevin Joseph. “INSIGHTS INTO HERPES SIMPLEX VIRUS TYPE 1 ASSEMBLY: PROTEIN INTERACTIONS AND VIRION INCORPORATION DETERMINANTS OF THE TEGUMENT PROTEIN VP22.” 2008. Web. 23 Oct 2019.

Vancouver:

O'Regan KJ. INSIGHTS INTO HERPES SIMPLEX VIRUS TYPE 1 ASSEMBLY: PROTEIN INTERACTIONS AND VIRION INCORPORATION DETERMINANTS OF THE TEGUMENT PROTEIN VP22. [Internet] [Doctoral dissertation]. Penn State University; 2008. [cited 2019 Oct 23]. Available from: https://etda.libraries.psu.edu/catalog/8115.

Council of Science Editors:

O'Regan KJ. INSIGHTS INTO HERPES SIMPLEX VIRUS TYPE 1 ASSEMBLY: PROTEIN INTERACTIONS AND VIRION INCORPORATION DETERMINANTS OF THE TEGUMENT PROTEIN VP22. [Doctoral Dissertation]. Penn State University; 2008. Available from: https://etda.libraries.psu.edu/catalog/8115


Wright State University

3. Alhazmi, Amani Mohammed. The Response of M0, M1, and M2 RAW246.7 Macrophage Cell Line to HSV-1 Infection in vitro.

Degree: MS, Microbiology and Immunology, 2019, Wright State University

 Herpes Simplex Virus Type 1 (HSV-1) infection occurs through the epithelial cells of the skin or mucous membranes. The beginning of the primary infection is… (more)

Subjects/Keywords: Immunology; Herpes Simplex Virus Type 1; HSV-1; HSV-1 infection; macrophage; macrophage recruitment; macrophage differentiation

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APA (6th Edition):

Alhazmi, A. M. (2019). The Response of M0, M1, and M2 RAW246.7 Macrophage Cell Line to HSV-1 Infection in vitro. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1557759403892368

Chicago Manual of Style (16th Edition):

Alhazmi, Amani Mohammed. “The Response of M0, M1, and M2 RAW246.7 Macrophage Cell Line to HSV-1 Infection in vitro.” 2019. Masters Thesis, Wright State University. Accessed October 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=wright1557759403892368.

MLA Handbook (7th Edition):

Alhazmi, Amani Mohammed. “The Response of M0, M1, and M2 RAW246.7 Macrophage Cell Line to HSV-1 Infection in vitro.” 2019. Web. 23 Oct 2019.

Vancouver:

Alhazmi AM. The Response of M0, M1, and M2 RAW246.7 Macrophage Cell Line to HSV-1 Infection in vitro. [Internet] [Masters thesis]. Wright State University; 2019. [cited 2019 Oct 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1557759403892368.

Council of Science Editors:

Alhazmi AM. The Response of M0, M1, and M2 RAW246.7 Macrophage Cell Line to HSV-1 Infection in vitro. [Masters Thesis]. Wright State University; 2019. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1557759403892368


Wright State University

4. Alamri, Badrieah Mohammad. Effects of Myrrh on HSV-1 Using Plaque Assay.

Degree: MS, Microbiology and Immunology, 2017, Wright State University

 Herpes simplex virus type 1 (HSV-1) is a highly infective human pathogen which infects a wide range of population in North America and worldwide. HSV-1(more)

Subjects/Keywords: Immunology; Myrrh; HSV-1; Vero cells

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APA (6th Edition):

Alamri, B. M. (2017). Effects of Myrrh on HSV-1 Using Plaque Assay. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1481628828816542

Chicago Manual of Style (16th Edition):

Alamri, Badrieah Mohammad. “Effects of Myrrh on HSV-1 Using Plaque Assay.” 2017. Masters Thesis, Wright State University. Accessed October 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=wright1481628828816542.

MLA Handbook (7th Edition):

Alamri, Badrieah Mohammad. “Effects of Myrrh on HSV-1 Using Plaque Assay.” 2017. Web. 23 Oct 2019.

Vancouver:

Alamri BM. Effects of Myrrh on HSV-1 Using Plaque Assay. [Internet] [Masters thesis]. Wright State University; 2017. [cited 2019 Oct 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1481628828816542.

Council of Science Editors:

Alamri BM. Effects of Myrrh on HSV-1 Using Plaque Assay. [Masters Thesis]. Wright State University; 2017. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1481628828816542


Wright State University

5. Alanazi, Yousef Nifaj. HSV-1 Replication in different RAW 264.7 and J774.1 macrophage Phenotypes and Macrophage viability following HSV-1 infection.

Degree: MS, Microbiology and Immunology, 2018, Wright State University

 HSV-1 is a ubiquitous virus capable of causing lifelong latent infection. The virus contains a large double strand DNA genome covered by an icosahedral capsid.… (more)

Subjects/Keywords: Immunology; Microbiology; HSV-1; phenotype; macrophage

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APA (6th Edition):

Alanazi, Y. N. (2018). HSV-1 Replication in different RAW 264.7 and J774.1 macrophage Phenotypes and Macrophage viability following HSV-1 infection. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1525211483024617

Chicago Manual of Style (16th Edition):

Alanazi, Yousef Nifaj. “HSV-1 Replication in different RAW 264.7 and J774.1 macrophage Phenotypes and Macrophage viability following HSV-1 infection.” 2018. Masters Thesis, Wright State University. Accessed October 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=wright1525211483024617.

MLA Handbook (7th Edition):

Alanazi, Yousef Nifaj. “HSV-1 Replication in different RAW 264.7 and J774.1 macrophage Phenotypes and Macrophage viability following HSV-1 infection.” 2018. Web. 23 Oct 2019.

Vancouver:

Alanazi YN. HSV-1 Replication in different RAW 264.7 and J774.1 macrophage Phenotypes and Macrophage viability following HSV-1 infection. [Internet] [Masters thesis]. Wright State University; 2018. [cited 2019 Oct 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1525211483024617.

Council of Science Editors:

Alanazi YN. HSV-1 Replication in different RAW 264.7 and J774.1 macrophage Phenotypes and Macrophage viability following HSV-1 infection. [Masters Thesis]. Wright State University; 2018. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1525211483024617


Vanderbilt University

6. Wilson, Christopher Scott. New Modes of B lymphocyte Regulation in Autoimmune Disease.

Degree: PhD, Microbiology and Immunology, 2019, Vanderbilt University

 New Modes of B lymphocyte Regulation in Autoimmune Disease Christopher Scott Wilson Dissertation under the direction of Professor Daniel J. Moore The deleterious roles that… (more)

Subjects/Keywords: B cells; Type 1 Diabetes; Tolerance; Immunology

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APA (6th Edition):

Wilson, C. S. (2019). New Modes of B lymphocyte Regulation in Autoimmune Disease. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-01162019-091326/ ;

Chicago Manual of Style (16th Edition):

Wilson, Christopher Scott. “New Modes of B lymphocyte Regulation in Autoimmune Disease.” 2019. Doctoral Dissertation, Vanderbilt University. Accessed October 23, 2019. http://etd.library.vanderbilt.edu/available/etd-01162019-091326/ ;.

MLA Handbook (7th Edition):

Wilson, Christopher Scott. “New Modes of B lymphocyte Regulation in Autoimmune Disease.” 2019. Web. 23 Oct 2019.

Vancouver:

Wilson CS. New Modes of B lymphocyte Regulation in Autoimmune Disease. [Internet] [Doctoral dissertation]. Vanderbilt University; 2019. [cited 2019 Oct 23]. Available from: http://etd.library.vanderbilt.edu/available/etd-01162019-091326/ ;.

Council of Science Editors:

Wilson CS. New Modes of B lymphocyte Regulation in Autoimmune Disease. [Doctoral Dissertation]. Vanderbilt University; 2019. Available from: http://etd.library.vanderbilt.edu/available/etd-01162019-091326/ ;


Queens University

7. Alghamdi, Farah. NEURAMINIDASE-1 SIALIDASE AND MATRIX METALLOPROTEINASE-9 CROSSTALK IN ALLIANCE WITH INSULIN RECEPTORS IS AN ESSENTIAL MOLECULAR SIGNALING PLATFORM FOR INSULIN-INDUCED RECEPTOR ACTIVATION .

Degree: Microbiology and Immunology, 2013, Queens University

 Molecular-targeting therapeutics directed towards growth factor receptors have become promising interventions in cancer. They include the family of mammalian receptor tyrosine kinases such as epidermal… (more)

Subjects/Keywords: Insulin receptors; MMP-9; NEURAMINIDASE-1

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APA (6th Edition):

Alghamdi, F. (2013). NEURAMINIDASE-1 SIALIDASE AND MATRIX METALLOPROTEINASE-9 CROSSTALK IN ALLIANCE WITH INSULIN RECEPTORS IS AN ESSENTIAL MOLECULAR SIGNALING PLATFORM FOR INSULIN-INDUCED RECEPTOR ACTIVATION . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/7820

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Alghamdi, Farah. “NEURAMINIDASE-1 SIALIDASE AND MATRIX METALLOPROTEINASE-9 CROSSTALK IN ALLIANCE WITH INSULIN RECEPTORS IS AN ESSENTIAL MOLECULAR SIGNALING PLATFORM FOR INSULIN-INDUCED RECEPTOR ACTIVATION .” 2013. Thesis, Queens University. Accessed October 23, 2019. http://hdl.handle.net/1974/7820.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Alghamdi, Farah. “NEURAMINIDASE-1 SIALIDASE AND MATRIX METALLOPROTEINASE-9 CROSSTALK IN ALLIANCE WITH INSULIN RECEPTORS IS AN ESSENTIAL MOLECULAR SIGNALING PLATFORM FOR INSULIN-INDUCED RECEPTOR ACTIVATION .” 2013. Web. 23 Oct 2019.

Vancouver:

Alghamdi F. NEURAMINIDASE-1 SIALIDASE AND MATRIX METALLOPROTEINASE-9 CROSSTALK IN ALLIANCE WITH INSULIN RECEPTORS IS AN ESSENTIAL MOLECULAR SIGNALING PLATFORM FOR INSULIN-INDUCED RECEPTOR ACTIVATION . [Internet] [Thesis]. Queens University; 2013. [cited 2019 Oct 23]. Available from: http://hdl.handle.net/1974/7820.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Alghamdi F. NEURAMINIDASE-1 SIALIDASE AND MATRIX METALLOPROTEINASE-9 CROSSTALK IN ALLIANCE WITH INSULIN RECEPTORS IS AN ESSENTIAL MOLECULAR SIGNALING PLATFORM FOR INSULIN-INDUCED RECEPTOR ACTIVATION . [Thesis]. Queens University; 2013. Available from: http://hdl.handle.net/1974/7820

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queens University

8. Plater, Samantha. Development of models to study Salmonella enterica infection of THP-1 cells pre-treated with IL-27 .

Degree: Microbiology and Immunology, 2015, Queens University

 IL-27 has been shown to have important pro- and anti-inflammatory roles in the adaptive immune response. However, its role in the innate immune response is… (more)

Subjects/Keywords: THP-1 cells; Salmonella pathogenicity island 1; IL-27; Innate immunity; Salmonella enterica

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APA (6th Edition):

Plater, S. (2015). Development of models to study Salmonella enterica infection of THP-1 cells pre-treated with IL-27 . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/13820

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Plater, Samantha. “Development of models to study Salmonella enterica infection of THP-1 cells pre-treated with IL-27 .” 2015. Thesis, Queens University. Accessed October 23, 2019. http://hdl.handle.net/1974/13820.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Plater, Samantha. “Development of models to study Salmonella enterica infection of THP-1 cells pre-treated with IL-27 .” 2015. Web. 23 Oct 2019.

Vancouver:

Plater S. Development of models to study Salmonella enterica infection of THP-1 cells pre-treated with IL-27 . [Internet] [Thesis]. Queens University; 2015. [cited 2019 Oct 23]. Available from: http://hdl.handle.net/1974/13820.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Plater S. Development of models to study Salmonella enterica infection of THP-1 cells pre-treated with IL-27 . [Thesis]. Queens University; 2015. Available from: http://hdl.handle.net/1974/13820

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Wright State University

9. Hey, Jessica Renee. The Effects of SOCS1, SOCS3 and HSV-1 Infection on Morphology, Cell Viability and Rab7 Expression in Polarized M1 and M2 Raw 264.7 Murine Macrophages.

Degree: MS, Microbiology and Immunology, 2018, Wright State University

 HSV-1 causes a life-long infection in its host and has evolved multiple strategies to facilitate infection and evade the immune response. This virus has been… (more)

Subjects/Keywords: Biology; Biomedical Research; Immunology; Microbiology; Rab7; HSV-1; Herpes Simplex 1; Rab7; F-actin; SOCS1; SOCS3

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APA (6th Edition):

Hey, J. R. (2018). The Effects of SOCS1, SOCS3 and HSV-1 Infection on Morphology, Cell Viability and Rab7 Expression in Polarized M1 and M2 Raw 264.7 Murine Macrophages. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1526826716666845

Chicago Manual of Style (16th Edition):

Hey, Jessica Renee. “The Effects of SOCS1, SOCS3 and HSV-1 Infection on Morphology, Cell Viability and Rab7 Expression in Polarized M1 and M2 Raw 264.7 Murine Macrophages.” 2018. Masters Thesis, Wright State University. Accessed October 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=wright1526826716666845.

MLA Handbook (7th Edition):

Hey, Jessica Renee. “The Effects of SOCS1, SOCS3 and HSV-1 Infection on Morphology, Cell Viability and Rab7 Expression in Polarized M1 and M2 Raw 264.7 Murine Macrophages.” 2018. Web. 23 Oct 2019.

Vancouver:

Hey JR. The Effects of SOCS1, SOCS3 and HSV-1 Infection on Morphology, Cell Viability and Rab7 Expression in Polarized M1 and M2 Raw 264.7 Murine Macrophages. [Internet] [Masters thesis]. Wright State University; 2018. [cited 2019 Oct 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1526826716666845.

Council of Science Editors:

Hey JR. The Effects of SOCS1, SOCS3 and HSV-1 Infection on Morphology, Cell Viability and Rab7 Expression in Polarized M1 and M2 Raw 264.7 Murine Macrophages. [Masters Thesis]. Wright State University; 2018. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1526826716666845


Wright State University

10. Van Buren, Lauren Kay. HSV-1 Infection of C3H Central Nervous System Cell Lines.

Degree: MS, Microbiology and Immunology, 2007, Wright State University

 Herpes Simplex Virus Type 1 (HSV-1) can infect the nervous system, resulting in a disease known as herpes encephalitis (HSE). Herpes encephalitis affects thousands of… (more)

Subjects/Keywords: Biology, Microbiology; HSV-1 (Herpes Simplex Virus) Type 1; herpes; neurons; astrocytes; fibroblast-like cells; herpes encephalitis; HSE; microglia

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APA (6th Edition):

Van Buren, L. K. (2007). HSV-1 Infection of C3H Central Nervous System Cell Lines. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1189746668

Chicago Manual of Style (16th Edition):

Van Buren, Lauren Kay. “HSV-1 Infection of C3H Central Nervous System Cell Lines.” 2007. Masters Thesis, Wright State University. Accessed October 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=wright1189746668.

MLA Handbook (7th Edition):

Van Buren, Lauren Kay. “HSV-1 Infection of C3H Central Nervous System Cell Lines.” 2007. Web. 23 Oct 2019.

Vancouver:

Van Buren LK. HSV-1 Infection of C3H Central Nervous System Cell Lines. [Internet] [Masters thesis]. Wright State University; 2007. [cited 2019 Oct 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1189746668.

Council of Science Editors:

Van Buren LK. HSV-1 Infection of C3H Central Nervous System Cell Lines. [Masters Thesis]. Wright State University; 2007. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1189746668


Wright State University

11. McNichol, Ryan Matthew. Characteristics of a Foamy Virus-Derived Vector that allow for safe Autologous Gene Therapy to correct Leukocyte Adhesion Deficiency Type 1.

Degree: MS, Microbiology and Immunology, 2007, Wright State University

 The hematopoeitic stem cell is a prime target for gene therapy in the attempt to correct a number of single gene inherited genetic defects that… (more)

Subjects/Keywords: Biology, Microbiology; LAD-1; Gene Therapy; Foamy Virus; Spumavirus

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APA (6th Edition):

McNichol, R. M. (2007). Characteristics of a Foamy Virus-Derived Vector that allow for safe Autologous Gene Therapy to correct Leukocyte Adhesion Deficiency Type 1. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1189781066

Chicago Manual of Style (16th Edition):

McNichol, Ryan Matthew. “Characteristics of a Foamy Virus-Derived Vector that allow for safe Autologous Gene Therapy to correct Leukocyte Adhesion Deficiency Type 1.” 2007. Masters Thesis, Wright State University. Accessed October 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=wright1189781066.

MLA Handbook (7th Edition):

McNichol, Ryan Matthew. “Characteristics of a Foamy Virus-Derived Vector that allow for safe Autologous Gene Therapy to correct Leukocyte Adhesion Deficiency Type 1.” 2007. Web. 23 Oct 2019.

Vancouver:

McNichol RM. Characteristics of a Foamy Virus-Derived Vector that allow for safe Autologous Gene Therapy to correct Leukocyte Adhesion Deficiency Type 1. [Internet] [Masters thesis]. Wright State University; 2007. [cited 2019 Oct 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1189781066.

Council of Science Editors:

McNichol RM. Characteristics of a Foamy Virus-Derived Vector that allow for safe Autologous Gene Therapy to correct Leukocyte Adhesion Deficiency Type 1. [Masters Thesis]. Wright State University; 2007. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1189781066


Wright State University

12. Abbas, Asma A. HSV-1 INFECTION IN KERATINOCYTE CELL LINES TREATED WITH MITOTIC INHIBITORS.

Degree: MS, Microbiology and Immunology, 2011, Wright State University

 The hypothesis for this research was: Herpes simplex virus type 1 (HSV-1) infection of murine keratinocyte cell lines (HEL-30 and PAM-212) treated with mitotic inhibitors… (more)

Subjects/Keywords: Immunology; HSV-1 infection; keratinocyte cell lines; mitotic inhibitors

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APA (6th Edition):

Abbas, A. A. (2011). HSV-1 INFECTION IN KERATINOCYTE CELL LINES TREATED WITH MITOTIC INHIBITORS. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1303839668

Chicago Manual of Style (16th Edition):

Abbas, Asma A. “HSV-1 INFECTION IN KERATINOCYTE CELL LINES TREATED WITH MITOTIC INHIBITORS.” 2011. Masters Thesis, Wright State University. Accessed October 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=wright1303839668.

MLA Handbook (7th Edition):

Abbas, Asma A. “HSV-1 INFECTION IN KERATINOCYTE CELL LINES TREATED WITH MITOTIC INHIBITORS.” 2011. Web. 23 Oct 2019.

Vancouver:

Abbas AA. HSV-1 INFECTION IN KERATINOCYTE CELL LINES TREATED WITH MITOTIC INHIBITORS. [Internet] [Masters thesis]. Wright State University; 2011. [cited 2019 Oct 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1303839668.

Council of Science Editors:

Abbas AA. HSV-1 INFECTION IN KERATINOCYTE CELL LINES TREATED WITH MITOTIC INHIBITORS. [Masters Thesis]. Wright State University; 2011. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1303839668


Wright State University

13. Alhejely, Amani Saud. Amino acid substitutions created in Reverse Transcriptase and their influence on HIV-1 mutation frequencies.

Degree: MS, Microbiology and Immunology, 2011, Wright State University

 As research has shown, a high mutation rate occursduring the reverse transcription RT process because HIVreverse transcriptase fails to correct erroneouslyincorporated nucleotides during the reverse… (more)

Subjects/Keywords: Immunology; Microbiology; HIV-1 mutation rate; amino acid; Reverse Transcriptase

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APA (6th Edition):

Alhejely, A. S. (2011). Amino acid substitutions created in Reverse Transcriptase and their influence on HIV-1 mutation frequencies. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1309968759

Chicago Manual of Style (16th Edition):

Alhejely, Amani Saud. “Amino acid substitutions created in Reverse Transcriptase and their influence on HIV-1 mutation frequencies.” 2011. Masters Thesis, Wright State University. Accessed October 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=wright1309968759.

MLA Handbook (7th Edition):

Alhejely, Amani Saud. “Amino acid substitutions created in Reverse Transcriptase and their influence on HIV-1 mutation frequencies.” 2011. Web. 23 Oct 2019.

Vancouver:

Alhejely AS. Amino acid substitutions created in Reverse Transcriptase and their influence on HIV-1 mutation frequencies. [Internet] [Masters thesis]. Wright State University; 2011. [cited 2019 Oct 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1309968759.

Council of Science Editors:

Alhejely AS. Amino acid substitutions created in Reverse Transcriptase and their influence on HIV-1 mutation frequencies. [Masters Thesis]. Wright State University; 2011. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1309968759


Wright State University

14. Aldreiwish, Allolo Dreiwish. The Effect of HSV-1 Infection on Differentiated and Polarized U937 cells.

Degree: MS, Microbiology and Immunology, 2013, Wright State University

 Herpes simplex virus type 1 (HSV-1) challenges the host immune systemthrough several mechanisms (Frey, et al., 2009). In vitro, U937 cells (humanmacrophage-like precursor cell line)… (more)

Subjects/Keywords: Immunology; Microbiology; U937; HSV-1; Macrophages polarization; Differentiation of U937

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APA (6th Edition):

Aldreiwish, A. D. (2013). The Effect of HSV-1 Infection on Differentiated and Polarized U937 cells. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1389610801

Chicago Manual of Style (16th Edition):

Aldreiwish, Allolo Dreiwish. “The Effect of HSV-1 Infection on Differentiated and Polarized U937 cells.” 2013. Masters Thesis, Wright State University. Accessed October 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=wright1389610801.

MLA Handbook (7th Edition):

Aldreiwish, Allolo Dreiwish. “The Effect of HSV-1 Infection on Differentiated and Polarized U937 cells.” 2013. Web. 23 Oct 2019.

Vancouver:

Aldreiwish AD. The Effect of HSV-1 Infection on Differentiated and Polarized U937 cells. [Internet] [Masters thesis]. Wright State University; 2013. [cited 2019 Oct 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1389610801.

Council of Science Editors:

Aldreiwish AD. The Effect of HSV-1 Infection on Differentiated and Polarized U937 cells. [Masters Thesis]. Wright State University; 2013. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1389610801


Vanderbilt University

15. Yang, Ruifeng. Identification of Novel Determinants of HIV-1 Uncoating in the Capsid Protein.

Degree: PhD, Microbiology and Immunology, 2009, Vanderbilt University

 Uncoating of the viral core following penetration into the target cell represents a fundamentally obscure step in the HIV-1 life cycle. Our laboratory has previously… (more)

Subjects/Keywords: Uncoating; Postentry; Capsid; HIV-1

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APA (6th Edition):

Yang, R. (2009). Identification of Novel Determinants of HIV-1 Uncoating in the Capsid Protein. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu//available/etd-02092009-163700/ ;

Chicago Manual of Style (16th Edition):

Yang, Ruifeng. “Identification of Novel Determinants of HIV-1 Uncoating in the Capsid Protein.” 2009. Doctoral Dissertation, Vanderbilt University. Accessed October 23, 2019. http://etd.library.vanderbilt.edu//available/etd-02092009-163700/ ;.

MLA Handbook (7th Edition):

Yang, Ruifeng. “Identification of Novel Determinants of HIV-1 Uncoating in the Capsid Protein.” 2009. Web. 23 Oct 2019.

Vancouver:

Yang R. Identification of Novel Determinants of HIV-1 Uncoating in the Capsid Protein. [Internet] [Doctoral dissertation]. Vanderbilt University; 2009. [cited 2019 Oct 23]. Available from: http://etd.library.vanderbilt.edu//available/etd-02092009-163700/ ;.

Council of Science Editors:

Yang R. Identification of Novel Determinants of HIV-1 Uncoating in the Capsid Protein. [Doctoral Dissertation]. Vanderbilt University; 2009. Available from: http://etd.library.vanderbilt.edu//available/etd-02092009-163700/ ;


Vanderbilt University

16. Burse, Mallori Jacole. Inhibitory Control of HIV-1 by Cyclophilin A.

Degree: PhD, Microbiology and Immunology, 2017, Vanderbilt University

 The host protein cyclophilin A (CypA) can both stimulate and inhibit HIV-1 infection through its interaction with the viral capsid (CA). CypA enhances the early… (more)

Subjects/Keywords: restriction factors; virus-host Interactions; HIV-1; cyclophilin A; CypA

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APA (6th Edition):

Burse, M. J. (2017). Inhibitory Control of HIV-1 by Cyclophilin A. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-12042017-090452/ ;

Chicago Manual of Style (16th Edition):

Burse, Mallori Jacole. “Inhibitory Control of HIV-1 by Cyclophilin A.” 2017. Doctoral Dissertation, Vanderbilt University. Accessed October 23, 2019. http://etd.library.vanderbilt.edu/available/etd-12042017-090452/ ;.

MLA Handbook (7th Edition):

Burse, Mallori Jacole. “Inhibitory Control of HIV-1 by Cyclophilin A.” 2017. Web. 23 Oct 2019.

Vancouver:

Burse MJ. Inhibitory Control of HIV-1 by Cyclophilin A. [Internet] [Doctoral dissertation]. Vanderbilt University; 2017. [cited 2019 Oct 23]. Available from: http://etd.library.vanderbilt.edu/available/etd-12042017-090452/ ;.

Council of Science Editors:

Burse MJ. Inhibitory Control of HIV-1 by Cyclophilin A. [Doctoral Dissertation]. Vanderbilt University; 2017. Available from: http://etd.library.vanderbilt.edu/available/etd-12042017-090452/ ;


Vanderbilt University

17. Erickson, John Joseph. CD8+ T cells are impaired during viral acute respiratory infection by coordinated inhibitory pathways.

Degree: PhD, Microbiology and Immunology, 2013, Vanderbilt University

 My thesis project explores mechanisms controlling CD8+ T cell impairment in the lung during viral acute lower respiratory infection. I show that viral infection is… (more)

Subjects/Keywords: PD-1; impairment; CD8+ T cell; influenza virus; human metapneumovirus

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APA (6th Edition):

Erickson, J. J. (2013). CD8+ T cells are impaired during viral acute respiratory infection by coordinated inhibitory pathways. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu//available/etd-08072013-164449/ ;

Chicago Manual of Style (16th Edition):

Erickson, John Joseph. “CD8+ T cells are impaired during viral acute respiratory infection by coordinated inhibitory pathways.” 2013. Doctoral Dissertation, Vanderbilt University. Accessed October 23, 2019. http://etd.library.vanderbilt.edu//available/etd-08072013-164449/ ;.

MLA Handbook (7th Edition):

Erickson, John Joseph. “CD8+ T cells are impaired during viral acute respiratory infection by coordinated inhibitory pathways.” 2013. Web. 23 Oct 2019.

Vancouver:

Erickson JJ. CD8+ T cells are impaired during viral acute respiratory infection by coordinated inhibitory pathways. [Internet] [Doctoral dissertation]. Vanderbilt University; 2013. [cited 2019 Oct 23]. Available from: http://etd.library.vanderbilt.edu//available/etd-08072013-164449/ ;.

Council of Science Editors:

Erickson JJ. CD8+ T cells are impaired during viral acute respiratory infection by coordinated inhibitory pathways. [Doctoral Dissertation]. Vanderbilt University; 2013. Available from: http://etd.library.vanderbilt.edu//available/etd-08072013-164449/ ;


Vanderbilt University

18. Stocks, Blair Taylor. Dysregulated T-B Lymphocyte Collaboration in Autoimmunity Poses a Barrier to Transplant Tolerance.

Degree: PhD, Microbiology and Immunology, 2016, Vanderbilt University

 Achieving transplant tolerance in the autoimmune environment will require targeting multiple immunologic dysregulations in T-B lymphocyte collaboration that drive the aggressive anti-graft response. At a… (more)

Subjects/Keywords: T Regulatory Cells; Transplantation; Autoimmunity; Type 1 Diabetes; Lupus; Tolerance

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APA (6th Edition):

Stocks, B. T. (2016). Dysregulated T-B Lymphocyte Collaboration in Autoimmunity Poses a Barrier to Transplant Tolerance. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-07132016-074224/ ;

Chicago Manual of Style (16th Edition):

Stocks, Blair Taylor. “Dysregulated T-B Lymphocyte Collaboration in Autoimmunity Poses a Barrier to Transplant Tolerance.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed October 23, 2019. http://etd.library.vanderbilt.edu/available/etd-07132016-074224/ ;.

MLA Handbook (7th Edition):

Stocks, Blair Taylor. “Dysregulated T-B Lymphocyte Collaboration in Autoimmunity Poses a Barrier to Transplant Tolerance.” 2016. Web. 23 Oct 2019.

Vancouver:

Stocks BT. Dysregulated T-B Lymphocyte Collaboration in Autoimmunity Poses a Barrier to Transplant Tolerance. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2019 Oct 23]. Available from: http://etd.library.vanderbilt.edu/available/etd-07132016-074224/ ;.

Council of Science Editors:

Stocks BT. Dysregulated T-B Lymphocyte Collaboration in Autoimmunity Poses a Barrier to Transplant Tolerance. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://etd.library.vanderbilt.edu/available/etd-07132016-074224/ ;

19. Williams, Jonathan Murphy. Peripheral Tolerance in Anti-Insulin B Lymphocytes.

Degree: PhD, Microbiology and Immunology, 2015, Vanderbilt University

 Overall, these studies examine how peripheral tolerance is governed for autoreactive B lymphocytes that bind the relevant autoAg, insulin. CSR-competent anti-insulin B cells enter mature… (more)

Subjects/Keywords: Autoimmunity; Tolerance; B cells; Insulin; Type 1 Diabetes

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APA (6th Edition):

Williams, J. M. (2015). Peripheral Tolerance in Anti-Insulin B Lymphocytes. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu//available/etd-11092015-092347/ ;

Chicago Manual of Style (16th Edition):

Williams, Jonathan Murphy. “Peripheral Tolerance in Anti-Insulin B Lymphocytes.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed October 23, 2019. http://etd.library.vanderbilt.edu//available/etd-11092015-092347/ ;.

MLA Handbook (7th Edition):

Williams, Jonathan Murphy. “Peripheral Tolerance in Anti-Insulin B Lymphocytes.” 2015. Web. 23 Oct 2019.

Vancouver:

Williams JM. Peripheral Tolerance in Anti-Insulin B Lymphocytes. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2019 Oct 23]. Available from: http://etd.library.vanderbilt.edu//available/etd-11092015-092347/ ;.

Council of Science Editors:

Williams JM. Peripheral Tolerance in Anti-Insulin B Lymphocytes. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://etd.library.vanderbilt.edu//available/etd-11092015-092347/ ;

20. Rinehart, Erica Marie. Inhibition of Nectin-1 and Herpes Virus Entry Mediator (HVEM) Using Monoclonal Antibodies Decreases HSV-1 Entry into Neuro-2A Cells.

Degree: MS, Microbiology and Immunology, 2015, Wright State University

 This study examines the effects of blocking the nectin-1 and Herpes Virus Entry Mediator (HVEM) receptors on Neuro-2a (N2a) cells in order to prevent HSV-1(more)

Subjects/Keywords: Immunology; HSV -1; Nectin-1; HVEM; Neuro-2A; PAM212

…cells with Receptors Blocked with Nectin-1 (F-10)/HVEM (D-5) Monoclonal… …counts of N2a cells with Receptors Blocked with Nectin-1(F-10)/HVEM (D-5)… …Normal Mouse IgM, or Control Cells in 5% Serum that were infected with .01 MOI HSV-1 at 0 hours… …with Nectin-1 (F-10) Monoclonal Antibody, Normal Mouse IgM, or Control Cells in 5… …experiment to demonstrate the level of HSV-1 infection in N2a cells grown in 5% serum and blocked… 

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APA (6th Edition):

Rinehart, E. M. (2015). Inhibition of Nectin-1 and Herpes Virus Entry Mediator (HVEM) Using Monoclonal Antibodies Decreases HSV-1 Entry into Neuro-2A Cells. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1438508507

Chicago Manual of Style (16th Edition):

Rinehart, Erica Marie. “Inhibition of Nectin-1 and Herpes Virus Entry Mediator (HVEM) Using Monoclonal Antibodies Decreases HSV-1 Entry into Neuro-2A Cells.” 2015. Masters Thesis, Wright State University. Accessed October 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=wright1438508507.

MLA Handbook (7th Edition):

Rinehart, Erica Marie. “Inhibition of Nectin-1 and Herpes Virus Entry Mediator (HVEM) Using Monoclonal Antibodies Decreases HSV-1 Entry into Neuro-2A Cells.” 2015. Web. 23 Oct 2019.

Vancouver:

Rinehart EM. Inhibition of Nectin-1 and Herpes Virus Entry Mediator (HVEM) Using Monoclonal Antibodies Decreases HSV-1 Entry into Neuro-2A Cells. [Internet] [Masters thesis]. Wright State University; 2015. [cited 2019 Oct 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1438508507.

Council of Science Editors:

Rinehart EM. Inhibition of Nectin-1 and Herpes Virus Entry Mediator (HVEM) Using Monoclonal Antibodies Decreases HSV-1 Entry into Neuro-2A Cells. [Masters Thesis]. Wright State University; 2015. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1438508507


Wright State University

21. Marathe, Jai Govind. Susceptibility of Primary Eosinophils to Infection with HIV-1 Strain HTLV-IIIB.

Degree: MS, Microbiology and Immunology, 2006, Wright State University

 Over the past two decades, much research has been done in the field of human immunodeficiency virus (HIV) and acquired immunodeficiency syndrome (AIDS). However, many… (more)

Subjects/Keywords: Biology, Microbiology; Hiv-1; Eosinophils; HIV-1 infection of eosinophils

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APA (6th Edition):

Marathe, J. G. (2006). Susceptibility of Primary Eosinophils to Infection with HIV-1 Strain HTLV-IIIB. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1158586306

Chicago Manual of Style (16th Edition):

Marathe, Jai Govind. “Susceptibility of Primary Eosinophils to Infection with HIV-1 Strain HTLV-IIIB.” 2006. Masters Thesis, Wright State University. Accessed October 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=wright1158586306.

MLA Handbook (7th Edition):

Marathe, Jai Govind. “Susceptibility of Primary Eosinophils to Infection with HIV-1 Strain HTLV-IIIB.” 2006. Web. 23 Oct 2019.

Vancouver:

Marathe JG. Susceptibility of Primary Eosinophils to Infection with HIV-1 Strain HTLV-IIIB. [Internet] [Masters thesis]. Wright State University; 2006. [cited 2019 Oct 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1158586306.

Council of Science Editors:

Marathe JG. Susceptibility of Primary Eosinophils to Infection with HIV-1 Strain HTLV-IIIB. [Masters Thesis]. Wright State University; 2006. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1158586306

22. Atem, Jude N. Human Immunodeficiency Virus-1 Productively Infects Mature Terminally Differentiated Eosinophils in HIV/AIDS Patients.

Degree: MS, Microbiology and Immunology, 2008, Wright State University

 Eosinophils express membrane CD4 protein and can bind HIV-1 glycoprotein (gp) 120. Therefore, eosinophils could serve as host cells for HIV-1 infection in vivo, especially… (more)

Subjects/Keywords: Biology; Microbiology; HIV-1; Eosinophils; HIV-1 infection of eosinophils

…eotaxin, IL-5 CD11a/CD18 CD11b/CD18 ICAM-1, fibrinogen, C3bi CD11c/CD18 Fibrinogen, C3bi… …LIST OF TABLES TABLE 1. PAGE Eosinophil adhesion molecules and their ligands vi 14… …every step of the way. vii INTRODUCTION The degree or extent of HIV-1 infection depends on… …host cell. Since eosinophils express membrane CD4 and CXCR4 molecules and can bind to HIV-1… …Therefore infections cells for HIV-1 eosinophilia reported in productive that HIV/AIDS… 

Page 1 Page 2 Page 3 Page 4 Page 5 Sample image Sample image

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APA (6th Edition):

Atem, J. N. (2008). Human Immunodeficiency Virus-1 Productively Infects Mature Terminally Differentiated Eosinophils in HIV/AIDS Patients. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1208724964

Chicago Manual of Style (16th Edition):

Atem, Jude N. “Human Immunodeficiency Virus-1 Productively Infects Mature Terminally Differentiated Eosinophils in HIV/AIDS Patients.” 2008. Masters Thesis, Wright State University. Accessed October 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=wright1208724964.

MLA Handbook (7th Edition):

Atem, Jude N. “Human Immunodeficiency Virus-1 Productively Infects Mature Terminally Differentiated Eosinophils in HIV/AIDS Patients.” 2008. Web. 23 Oct 2019.

Vancouver:

Atem JN. Human Immunodeficiency Virus-1 Productively Infects Mature Terminally Differentiated Eosinophils in HIV/AIDS Patients. [Internet] [Masters thesis]. Wright State University; 2008. [cited 2019 Oct 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1208724964.

Council of Science Editors:

Atem JN. Human Immunodeficiency Virus-1 Productively Infects Mature Terminally Differentiated Eosinophils in HIV/AIDS Patients. [Masters Thesis]. Wright State University; 2008. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1208724964


Wright State University

23. Doran, Diane Michelle. HYPOXIC INDUCTION AND THE ROLE OF HIFS IN THE ACTIVATION OF LUCIFERASE CONSTITUTIVE REPORTERS IN PLACENTAL STEM CELLS.

Degree: MS, Microbiology and Immunology, 2007, Wright State University

 Hypoxia is critically important to the development of the embryo and placenta. Proper placental development is critical for normal fetal growth and embryonic survival. Abnormal… (more)

Subjects/Keywords: Hypoxia Inducible Factors,; Luciferase,; Placental Formation; Differentiation; Trophoblast Stem Cells; Rcho-1 Trophoblasts

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APA (6th Edition):

Doran, D. M. (2007). HYPOXIC INDUCTION AND THE ROLE OF HIFS IN THE ACTIVATION OF LUCIFERASE CONSTITUTIVE REPORTERS IN PLACENTAL STEM CELLS. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1190149635

Chicago Manual of Style (16th Edition):

Doran, Diane Michelle. “HYPOXIC INDUCTION AND THE ROLE OF HIFS IN THE ACTIVATION OF LUCIFERASE CONSTITUTIVE REPORTERS IN PLACENTAL STEM CELLS.” 2007. Masters Thesis, Wright State University. Accessed October 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=wright1190149635.

MLA Handbook (7th Edition):

Doran, Diane Michelle. “HYPOXIC INDUCTION AND THE ROLE OF HIFS IN THE ACTIVATION OF LUCIFERASE CONSTITUTIVE REPORTERS IN PLACENTAL STEM CELLS.” 2007. Web. 23 Oct 2019.

Vancouver:

Doran DM. HYPOXIC INDUCTION AND THE ROLE OF HIFS IN THE ACTIVATION OF LUCIFERASE CONSTITUTIVE REPORTERS IN PLACENTAL STEM CELLS. [Internet] [Masters thesis]. Wright State University; 2007. [cited 2019 Oct 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1190149635.

Council of Science Editors:

Doran DM. HYPOXIC INDUCTION AND THE ROLE OF HIFS IN THE ACTIVATION OF LUCIFERASE CONSTITUTIVE REPORTERS IN PLACENTAL STEM CELLS. [Masters Thesis]. Wright State University; 2007. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1190149635


Penn State University

24. Berry, Gregory John. Mechanisms of insulin-dependent diabetes locus 9-mediated protection from type 1 diabetes.

Degree: PhD, Microbiology and Immunology, 2014, Penn State University

 There is currently no cure for type 1 diabetes (T1D), a chronic autoimmune disease typically diagnosed in children that results in destruction of the insulin-producing… (more)

Subjects/Keywords: Type 1 Diabetes; NOD mouse; Insulin-dependent diabetes locus 9; B cells

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APA (6th Edition):

Berry, G. J. (2014). Mechanisms of insulin-dependent diabetes locus 9-mediated protection from type 1 diabetes. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/22546

Chicago Manual of Style (16th Edition):

Berry, Gregory John. “Mechanisms of insulin-dependent diabetes locus 9-mediated protection from type 1 diabetes.” 2014. Doctoral Dissertation, Penn State University. Accessed October 23, 2019. https://etda.libraries.psu.edu/catalog/22546.

MLA Handbook (7th Edition):

Berry, Gregory John. “Mechanisms of insulin-dependent diabetes locus 9-mediated protection from type 1 diabetes.” 2014. Web. 23 Oct 2019.

Vancouver:

Berry GJ. Mechanisms of insulin-dependent diabetes locus 9-mediated protection from type 1 diabetes. [Internet] [Doctoral dissertation]. Penn State University; 2014. [cited 2019 Oct 23]. Available from: https://etda.libraries.psu.edu/catalog/22546.

Council of Science Editors:

Berry GJ. Mechanisms of insulin-dependent diabetes locus 9-mediated protection from type 1 diabetes. [Doctoral Dissertation]. Penn State University; 2014. Available from: https://etda.libraries.psu.edu/catalog/22546


Vanderbilt University

25. Floyd, Kyle Anthony. Dissecting Protein Stratification and Regulation in Uropathogenic <i>Escherichia coli<i> Biofilms.

Degree: PhD, Microbiology and Immunology, 2017, Vanderbilt University

 Catheter-associated urinary tract infections (CAUTIs) account for ~30% of hospital-acquired infections. Biofilm formation, a process where bacteria assume a temporary multicellular lifestyle, serves as a… (more)

Subjects/Keywords: UPEC; Biofilms; Imaging Mass Spectrometry; MALDI-TOF; Type 1 Pili; Phase Variation; Oxygen

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APA (6th Edition):

Floyd, K. A. (2017). Dissecting Protein Stratification and Regulation in Uropathogenic <i>Escherichia coli<i> Biofilms. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu//available/etd-02272017-161756/ ;

Chicago Manual of Style (16th Edition):

Floyd, Kyle Anthony. “Dissecting Protein Stratification and Regulation in Uropathogenic <i>Escherichia coli<i> Biofilms.” 2017. Doctoral Dissertation, Vanderbilt University. Accessed October 23, 2019. http://etd.library.vanderbilt.edu//available/etd-02272017-161756/ ;.

MLA Handbook (7th Edition):

Floyd, Kyle Anthony. “Dissecting Protein Stratification and Regulation in Uropathogenic <i>Escherichia coli<i> Biofilms.” 2017. Web. 23 Oct 2019.

Vancouver:

Floyd KA. Dissecting Protein Stratification and Regulation in Uropathogenic <i>Escherichia coli<i> Biofilms. [Internet] [Doctoral dissertation]. Vanderbilt University; 2017. [cited 2019 Oct 23]. Available from: http://etd.library.vanderbilt.edu//available/etd-02272017-161756/ ;.

Council of Science Editors:

Floyd KA. Dissecting Protein Stratification and Regulation in Uropathogenic <i>Escherichia coli<i> Biofilms. [Doctoral Dissertation]. Vanderbilt University; 2017. Available from: http://etd.library.vanderbilt.edu//available/etd-02272017-161756/ ;

26. McCarthy, Mary Katherine. Host Inflammatory Responses to Adenovirus Respiratory Infection.

Degree: PhD, Microbiology and Immunology, 2014, University of Michigan

 Adenoviruses are common causes of acute respiratory infection and myocarditis. It is unclear if manifestations of adenovirus disease are mediated by virus-induced tissue damage or… (more)

Subjects/Keywords: virus; pathogenesis; respiratory infection; myocarditis; mouse adenovirus type 1; Microbiology and Immunology; Health Sciences

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APA (6th Edition):

McCarthy, M. K. (2014). Host Inflammatory Responses to Adenovirus Respiratory Infection. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/110390

Chicago Manual of Style (16th Edition):

McCarthy, Mary Katherine. “Host Inflammatory Responses to Adenovirus Respiratory Infection.” 2014. Doctoral Dissertation, University of Michigan. Accessed October 23, 2019. http://hdl.handle.net/2027.42/110390.

MLA Handbook (7th Edition):

McCarthy, Mary Katherine. “Host Inflammatory Responses to Adenovirus Respiratory Infection.” 2014. Web. 23 Oct 2019.

Vancouver:

McCarthy MK. Host Inflammatory Responses to Adenovirus Respiratory Infection. [Internet] [Doctoral dissertation]. University of Michigan; 2014. [cited 2019 Oct 23]. Available from: http://hdl.handle.net/2027.42/110390.

Council of Science Editors:

McCarthy MK. Host Inflammatory Responses to Adenovirus Respiratory Infection. [Doctoral Dissertation]. University of Michigan; 2014. Available from: http://hdl.handle.net/2027.42/110390


Penn State University

27. Cole, Sara L. The DNA damage response of rodent fibroblasts expressing all or part of Simian Virus 40 large T antigen.

Degree: PhD, Microbiology and Immunology, 2001, Penn State University

 ABSTRACT The Simian Virus 40 large tumor (T) antigen is sufficient to transform cells in culture and induce tumors in experimental animals. T antigen has… (more)

Subjects/Keywords: cell stress; Rb; Bcl-2; J-domain; SV40; 5-fluorouracil; p53; apoptosis; DNA damage; large t antigen; transformation

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APA (6th Edition):

Cole, S. L. (2001). The DNA damage response of rodent fibroblasts expressing all or part of Simian Virus 40 large T antigen. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/5887

Chicago Manual of Style (16th Edition):

Cole, Sara L. “The DNA damage response of rodent fibroblasts expressing all or part of Simian Virus 40 large T antigen.” 2001. Doctoral Dissertation, Penn State University. Accessed October 23, 2019. https://etda.libraries.psu.edu/catalog/5887.

MLA Handbook (7th Edition):

Cole, Sara L. “The DNA damage response of rodent fibroblasts expressing all or part of Simian Virus 40 large T antigen.” 2001. Web. 23 Oct 2019.

Vancouver:

Cole SL. The DNA damage response of rodent fibroblasts expressing all or part of Simian Virus 40 large T antigen. [Internet] [Doctoral dissertation]. Penn State University; 2001. [cited 2019 Oct 23]. Available from: https://etda.libraries.psu.edu/catalog/5887.

Council of Science Editors:

Cole SL. The DNA damage response of rodent fibroblasts expressing all or part of Simian Virus 40 large T antigen. [Doctoral Dissertation]. Penn State University; 2001. Available from: https://etda.libraries.psu.edu/catalog/5887


Wright State University

28. Svitlova, Olena B. Six-Nine Months Long Term Culture of Mouse Bone Marrow Cells Differentiated to Macrophages and Eosinophils.

Degree: MS, Microbiology and Immunology, 2019, Wright State University

 Mouse models of eosinophil-associated diseases have been used to study the mechanisms of disease pathogenesis. In this study, mouse-derived bone marrow cells were used in… (more)

Subjects/Keywords: Microbiology; Immunology; eosinophil extracellular trap; IL-5; bone marrow derived eosinophils; long term culture; long term differentiation of bone marrow cells; growth factors; bi-lobed nucleus of eosinophils; eosinophils dendrite-like appendixes of cytoplasm

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APA (6th Edition):

Svitlova, O. B. (2019). Six-Nine Months Long Term Culture of Mouse Bone Marrow Cells Differentiated to Macrophages and Eosinophils. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1567524586159929

Chicago Manual of Style (16th Edition):

Svitlova, Olena B. “Six-Nine Months Long Term Culture of Mouse Bone Marrow Cells Differentiated to Macrophages and Eosinophils.” 2019. Masters Thesis, Wright State University. Accessed October 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=wright1567524586159929.

MLA Handbook (7th Edition):

Svitlova, Olena B. “Six-Nine Months Long Term Culture of Mouse Bone Marrow Cells Differentiated to Macrophages and Eosinophils.” 2019. Web. 23 Oct 2019.

Vancouver:

Svitlova OB. Six-Nine Months Long Term Culture of Mouse Bone Marrow Cells Differentiated to Macrophages and Eosinophils. [Internet] [Masters thesis]. Wright State University; 2019. [cited 2019 Oct 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1567524586159929.

Council of Science Editors:

Svitlova OB. Six-Nine Months Long Term Culture of Mouse Bone Marrow Cells Differentiated to Macrophages and Eosinophils. [Masters Thesis]. Wright State University; 2019. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1567524586159929


Vanderbilt University

29. Campbell, Jacquelyn Andrea. Structure-function analysis of reovirus binding to junctional adhesion molecule-A.

Degree: PhD, Microbiology and Immunology, 2005, Vanderbilt University

 Mammalian reoviruses are nonenveloped, double-stranded RNA viruses that serve as important models for studies of viral neuropathogenesis. Reovirus disease is initiated by binding of the… (more)

Subjects/Keywords: zo-1; junctional adhesion molecule; reovirus; tight junction

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Campbell, J. A. (2005). Structure-function analysis of reovirus binding to junctional adhesion molecule-A. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-11282005-150051/ ;

Chicago Manual of Style (16th Edition):

Campbell, Jacquelyn Andrea. “Structure-function analysis of reovirus binding to junctional adhesion molecule-A.” 2005. Doctoral Dissertation, Vanderbilt University. Accessed October 23, 2019. http://etd.library.vanderbilt.edu/available/etd-11282005-150051/ ;.

MLA Handbook (7th Edition):

Campbell, Jacquelyn Andrea. “Structure-function analysis of reovirus binding to junctional adhesion molecule-A.” 2005. Web. 23 Oct 2019.

Vancouver:

Campbell JA. Structure-function analysis of reovirus binding to junctional adhesion molecule-A. [Internet] [Doctoral dissertation]. Vanderbilt University; 2005. [cited 2019 Oct 23]. Available from: http://etd.library.vanderbilt.edu/available/etd-11282005-150051/ ;.

Council of Science Editors:

Campbell JA. Structure-function analysis of reovirus binding to junctional adhesion molecule-A. [Doctoral Dissertation]. Vanderbilt University; 2005. Available from: http://etd.library.vanderbilt.edu/available/etd-11282005-150051/ ;

30. Rold, Christopher James. The role of the cellular proteasome and ubiquitin in post-entry restriction of retroviruses by TRIM5α.

Degree: PhD, Microbiology and Immunology, 2009, Vanderbilt University

 The host protein TRIM5α inhibits retroviral infection at an early post-penetration stage by binding to the incoming viral capsid. Interaction with TRIM5α results in a… (more)

Subjects/Keywords: post-entry restriction; TRIM5á; HIV-1; retrovirus

…9 1-5 Diagram of HIV-1 reverse transcription… …49 2-5 Destabilization of primate TRIM5α proteins is correlated with restriction of HIV-1… …p24 CCR5 CC chemokine receptor 5 CD4+ Protein marker on surface of HIV-1 target cells… …directly from HIV-1 infection. 5 Host Genus Virus Species Humans Lentivirus… …to DNA. Reverse transcription is a discontinuous 10 Figure 1-5. Diagram of HIV-1 reverse… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rold, C. J. (2009). The role of the cellular proteasome and ubiquitin in post-entry restriction of retroviruses by TRIM5α. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu//available/etd-03302009-150129/ ;

Chicago Manual of Style (16th Edition):

Rold, Christopher James. “The role of the cellular proteasome and ubiquitin in post-entry restriction of retroviruses by TRIM5α.” 2009. Doctoral Dissertation, Vanderbilt University. Accessed October 23, 2019. http://etd.library.vanderbilt.edu//available/etd-03302009-150129/ ;.

MLA Handbook (7th Edition):

Rold, Christopher James. “The role of the cellular proteasome and ubiquitin in post-entry restriction of retroviruses by TRIM5α.” 2009. Web. 23 Oct 2019.

Vancouver:

Rold CJ. The role of the cellular proteasome and ubiquitin in post-entry restriction of retroviruses by TRIM5α. [Internet] [Doctoral dissertation]. Vanderbilt University; 2009. [cited 2019 Oct 23]. Available from: http://etd.library.vanderbilt.edu//available/etd-03302009-150129/ ;.

Council of Science Editors:

Rold CJ. The role of the cellular proteasome and ubiquitin in post-entry restriction of retroviruses by TRIM5α. [Doctoral Dissertation]. Vanderbilt University; 2009. Available from: http://etd.library.vanderbilt.edu//available/etd-03302009-150129/ ;

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