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You searched for publisher:("Texas Medical Center"). Showing records 1 – 30 of 974 total matches.

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Texas Medical Center

1. Tonthat, Nam K. Molecular mechanism by which the Escherichia coli nucleoid occlusion factor, SlmA, keeps cytokinesis in check.

Degree: PhD, 2011, Texas Medical Center

 Cell division or cytokinesis is one of the most fundamental processes in biology and is essential for the propagation of all living species. In Escherichia… (more)

Subjects/Keywords: bacterial cell division; chromosome segregation; FtsZ Z-ring formation; nucleoid occlusion; SlmA; Biochemistry, Biophysics, and Structural Biology

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APA (6th Edition):

Tonthat, N. K. (2011). Molecular mechanism by which the Escherichia coli nucleoid occlusion factor, SlmA, keeps cytokinesis in check. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/107

Chicago Manual of Style (16th Edition):

Tonthat, Nam K. “Molecular mechanism by which the Escherichia coli nucleoid occlusion factor, SlmA, keeps cytokinesis in check.” 2011. Doctoral Dissertation, Texas Medical Center. Accessed February 26, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/107.

MLA Handbook (7th Edition):

Tonthat, Nam K. “Molecular mechanism by which the Escherichia coli nucleoid occlusion factor, SlmA, keeps cytokinesis in check.” 2011. Web. 26 Feb 2021.

Vancouver:

Tonthat NK. Molecular mechanism by which the Escherichia coli nucleoid occlusion factor, SlmA, keeps cytokinesis in check. [Internet] [Doctoral dissertation]. Texas Medical Center; 2011. [cited 2021 Feb 26]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/107.

Council of Science Editors:

Tonthat NK. Molecular mechanism by which the Escherichia coli nucleoid occlusion factor, SlmA, keeps cytokinesis in check. [Doctoral Dissertation]. Texas Medical Center; 2011. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/107


Texas Medical Center

2. Jung, Youngsin. Analysis of Band 4.1B in Integrin-Mediated Cell Adhesion and Signaling.

Degree: PhD, 2011, Texas Medical Center

  Band 4.1B is a cytoskeletal adaptor protein that regulates various cellular behavior; however, the mechanisms by which Band 4.1B contributes to intracellular signaling are… (more)

Subjects/Keywords: Band 4.1B; Integrins; Development; Cell adhesion; Cell migration; Cell Biology; Developmental Biology

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APA (6th Edition):

Jung, Y. (2011). Analysis of Band 4.1B in Integrin-Mediated Cell Adhesion and Signaling. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/108

Chicago Manual of Style (16th Edition):

Jung, Youngsin. “Analysis of Band 4.1B in Integrin-Mediated Cell Adhesion and Signaling.” 2011. Doctoral Dissertation, Texas Medical Center. Accessed February 26, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/108.

MLA Handbook (7th Edition):

Jung, Youngsin. “Analysis of Band 4.1B in Integrin-Mediated Cell Adhesion and Signaling.” 2011. Web. 26 Feb 2021.

Vancouver:

Jung Y. Analysis of Band 4.1B in Integrin-Mediated Cell Adhesion and Signaling. [Internet] [Doctoral dissertation]. Texas Medical Center; 2011. [cited 2021 Feb 26]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/108.

Council of Science Editors:

Jung Y. Analysis of Band 4.1B in Integrin-Mediated Cell Adhesion and Signaling. [Doctoral Dissertation]. Texas Medical Center; 2011. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/108


Texas Medical Center

3. Maili, Lorena. COCAINE DEPENDENCE: THE ROLE OF SEROTONIN GENES IN.

Degree: MS, 2011, Texas Medical Center

  Animal studies have shown that behavioral responses to cocaine-related cues are altered by serotonergic medications. The effects of pharmacological agents on serotonin receptors 2a… (more)

Subjects/Keywords: cocaine dependence attentional bias serotonin polymorphisms impulsivity; Behavioral Neurobiology; Cognitive Neuroscience; Genetics

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APA (6th Edition):

Maili, L. (2011). COCAINE DEPENDENCE: THE ROLE OF SEROTONIN GENES IN. (Thesis). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/109

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Maili, Lorena. “COCAINE DEPENDENCE: THE ROLE OF SEROTONIN GENES IN.” 2011. Thesis, Texas Medical Center. Accessed February 26, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/109.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Maili, Lorena. “COCAINE DEPENDENCE: THE ROLE OF SEROTONIN GENES IN.” 2011. Web. 26 Feb 2021.

Vancouver:

Maili L. COCAINE DEPENDENCE: THE ROLE OF SEROTONIN GENES IN. [Internet] [Thesis]. Texas Medical Center; 2011. [cited 2021 Feb 26]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/109.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Maili L. COCAINE DEPENDENCE: THE ROLE OF SEROTONIN GENES IN. [Thesis]. Texas Medical Center; 2011. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/109

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas Medical Center

4. Hansen, Bryan J. POPULATION CODING IN LAMINAR CORTICAL CIRCUITS.

Degree: PhD, 2012, Texas Medical Center

  One of the fundamental questions in neuroscience is to understand how encoding of sensory inputs is distributed across neuronal networks in cerebral cortex to… (more)

Subjects/Keywords: Cortical layers Synchronization Correlated varability Visual cortex Adaptation; Medicine and Health Sciences; Systems Neuroscience

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APA (6th Edition):

Hansen, B. J. (2012). POPULATION CODING IN LAMINAR CORTICAL CIRCUITS. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/225

Chicago Manual of Style (16th Edition):

Hansen, Bryan J. “POPULATION CODING IN LAMINAR CORTICAL CIRCUITS.” 2012. Doctoral Dissertation, Texas Medical Center. Accessed February 26, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/225.

MLA Handbook (7th Edition):

Hansen, Bryan J. “POPULATION CODING IN LAMINAR CORTICAL CIRCUITS.” 2012. Web. 26 Feb 2021.

Vancouver:

Hansen BJ. POPULATION CODING IN LAMINAR CORTICAL CIRCUITS. [Internet] [Doctoral dissertation]. Texas Medical Center; 2012. [cited 2021 Feb 26]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/225.

Council of Science Editors:

Hansen BJ. POPULATION CODING IN LAMINAR CORTICAL CIRCUITS. [Doctoral Dissertation]. Texas Medical Center; 2012. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/225


Texas Medical Center

5. Popnoe, Dawn Olivia. Feasibility of Using Virtual Unenhanced Images to Replace Pre-contrast Images in Multiphase Renal CT Examinations.

Degree: MS, 2015, Texas Medical Center

  Multiphase renal CT exams are a commonly used imaging technique for the diagnosis of renal masses. The pre-contrast, or true unenhanced (TUE), image provides… (more)

Subjects/Keywords: CT Dual Energy CT Multi-material decomposition Renal CT Virtual Unenhanced Imaging; Medicine and Health Sciences; Physics

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APA (6th Edition):

Popnoe, D. O. (2015). Feasibility of Using Virtual Unenhanced Images to Replace Pre-contrast Images in Multiphase Renal CT Examinations. (Thesis). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/600

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Popnoe, Dawn Olivia. “Feasibility of Using Virtual Unenhanced Images to Replace Pre-contrast Images in Multiphase Renal CT Examinations.” 2015. Thesis, Texas Medical Center. Accessed February 26, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/600.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Popnoe, Dawn Olivia. “Feasibility of Using Virtual Unenhanced Images to Replace Pre-contrast Images in Multiphase Renal CT Examinations.” 2015. Web. 26 Feb 2021.

Vancouver:

Popnoe DO. Feasibility of Using Virtual Unenhanced Images to Replace Pre-contrast Images in Multiphase Renal CT Examinations. [Internet] [Thesis]. Texas Medical Center; 2015. [cited 2021 Feb 26]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/600.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Popnoe DO. Feasibility of Using Virtual Unenhanced Images to Replace Pre-contrast Images in Multiphase Renal CT Examinations. [Thesis]. Texas Medical Center; 2015. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/600

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas Medical Center

6. Zhang, Lin. MICROENVIRONMENT-INDUCED PTEN LOSS BY EXOSOMAL MICRORNA PRIMES BRAIN METASTASIS OUTGROWTH.

Degree: PhD, 2016, Texas Medical Center

  Development of life-threatening cancer metastases at distant organs requires disseminated tumor cells’ adaptation to and co-evolution with the drastically different microenvironments of metastatic sites.… (more)

Subjects/Keywords: metastasis; tumor microenvironment; exosome; PTEN; microRNAs; brain metastasis; epigenetics; Biology; Cancer Biology; Cell Biology; Medicine and Health Sciences; Neoplasms

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APA (6th Edition):

Zhang, L. (2016). MICROENVIRONMENT-INDUCED PTEN LOSS BY EXOSOMAL MICRORNA PRIMES BRAIN METASTASIS OUTGROWTH. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/710

Chicago Manual of Style (16th Edition):

Zhang, Lin. “MICROENVIRONMENT-INDUCED PTEN LOSS BY EXOSOMAL MICRORNA PRIMES BRAIN METASTASIS OUTGROWTH.” 2016. Doctoral Dissertation, Texas Medical Center. Accessed February 26, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/710.

MLA Handbook (7th Edition):

Zhang, Lin. “MICROENVIRONMENT-INDUCED PTEN LOSS BY EXOSOMAL MICRORNA PRIMES BRAIN METASTASIS OUTGROWTH.” 2016. Web. 26 Feb 2021.

Vancouver:

Zhang L. MICROENVIRONMENT-INDUCED PTEN LOSS BY EXOSOMAL MICRORNA PRIMES BRAIN METASTASIS OUTGROWTH. [Internet] [Doctoral dissertation]. Texas Medical Center; 2016. [cited 2021 Feb 26]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/710.

Council of Science Editors:

Zhang L. MICROENVIRONMENT-INDUCED PTEN LOSS BY EXOSOMAL MICRORNA PRIMES BRAIN METASTASIS OUTGROWTH. [Doctoral Dissertation]. Texas Medical Center; 2016. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/710


Texas Medical Center

7. Espejo, Alexsandra B. Role of phosphorylation in the regulation of PRMT5.

Degree: PhD, 2016, Texas Medical Center

  PRMT5 is a member of a group of proteins that mediate arginine methylation. It is involved in diverse cellular processes, including cell differentiation, splicing,… (more)

Subjects/Keywords: PRMT5; phosphorylation; interactions; domain; protein microarray; SH2; PDZ; 14-3-3; FHA; Molecular Biology

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APA (6th Edition):

Espejo, A. B. (2016). Role of phosphorylation in the regulation of PRMT5. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/711

Chicago Manual of Style (16th Edition):

Espejo, Alexsandra B. “Role of phosphorylation in the regulation of PRMT5.” 2016. Doctoral Dissertation, Texas Medical Center. Accessed February 26, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/711.

MLA Handbook (7th Edition):

Espejo, Alexsandra B. “Role of phosphorylation in the regulation of PRMT5.” 2016. Web. 26 Feb 2021.

Vancouver:

Espejo AB. Role of phosphorylation in the regulation of PRMT5. [Internet] [Doctoral dissertation]. Texas Medical Center; 2016. [cited 2021 Feb 26]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/711.

Council of Science Editors:

Espejo AB. Role of phosphorylation in the regulation of PRMT5. [Doctoral Dissertation]. Texas Medical Center; 2016. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/711


Texas Medical Center

8. Walker, Christopher M. Novel Simulation to Avoid Bias in Measurement of Hyperpolarized Pyruvate: Demonstrated in Phantom and In Vivo.

Degree: PhD, 2016, Texas Medical Center

  Dynamic nuclear polarization creates a transient hyperpolarized nuclear state that can dramatically increase the signal detected by magnetic resonance imaging. This signal increase allows… (more)

Subjects/Keywords: hyperpolarized; pyruvate; magnetic resoance; simulation; Analytical, Diagnostic and Therapeutic Techniques and Equipment; Medicine and Health Sciences; Other Physics

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APA (6th Edition):

Walker, C. M. (2016). Novel Simulation to Avoid Bias in Measurement of Hyperpolarized Pyruvate: Demonstrated in Phantom and In Vivo. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/712

Chicago Manual of Style (16th Edition):

Walker, Christopher M. “Novel Simulation to Avoid Bias in Measurement of Hyperpolarized Pyruvate: Demonstrated in Phantom and In Vivo.” 2016. Doctoral Dissertation, Texas Medical Center. Accessed February 26, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/712.

MLA Handbook (7th Edition):

Walker, Christopher M. “Novel Simulation to Avoid Bias in Measurement of Hyperpolarized Pyruvate: Demonstrated in Phantom and In Vivo.” 2016. Web. 26 Feb 2021.

Vancouver:

Walker CM. Novel Simulation to Avoid Bias in Measurement of Hyperpolarized Pyruvate: Demonstrated in Phantom and In Vivo. [Internet] [Doctoral dissertation]. Texas Medical Center; 2016. [cited 2021 Feb 26]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/712.

Council of Science Editors:

Walker CM. Novel Simulation to Avoid Bias in Measurement of Hyperpolarized Pyruvate: Demonstrated in Phantom and In Vivo. [Doctoral Dissertation]. Texas Medical Center; 2016. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/712


Texas Medical Center

9. Niedzielski, Joshua S. INVESTIGATION OF RADIATION INJURY IN THE ESOPHAGUS FROM DEFINITIVE CHEMORADIATION THERAPY USING NOVEL IMAGING BIOMARKERS.

Degree: PhD, 2016, Texas Medical Center

  Radiation injury in the esophagus occurs with high frequency from the treatment of non-small cell lung cancer (NSCLC). Radiation esophagitis is an acute normal… (more)

Subjects/Keywords: Radiation therapy; normal tissue toxicity; esophagitis; non small cell lung cancer; NTCP modelling; imaging biomarker; machine learning; functional imaging; 4DCT; FDG-PET; Other Medical Sciences

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APA (6th Edition):

Niedzielski, J. S. (2016). INVESTIGATION OF RADIATION INJURY IN THE ESOPHAGUS FROM DEFINITIVE CHEMORADIATION THERAPY USING NOVEL IMAGING BIOMARKERS. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/713

Chicago Manual of Style (16th Edition):

Niedzielski, Joshua S. “INVESTIGATION OF RADIATION INJURY IN THE ESOPHAGUS FROM DEFINITIVE CHEMORADIATION THERAPY USING NOVEL IMAGING BIOMARKERS.” 2016. Doctoral Dissertation, Texas Medical Center. Accessed February 26, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/713.

MLA Handbook (7th Edition):

Niedzielski, Joshua S. “INVESTIGATION OF RADIATION INJURY IN THE ESOPHAGUS FROM DEFINITIVE CHEMORADIATION THERAPY USING NOVEL IMAGING BIOMARKERS.” 2016. Web. 26 Feb 2021.

Vancouver:

Niedzielski JS. INVESTIGATION OF RADIATION INJURY IN THE ESOPHAGUS FROM DEFINITIVE CHEMORADIATION THERAPY USING NOVEL IMAGING BIOMARKERS. [Internet] [Doctoral dissertation]. Texas Medical Center; 2016. [cited 2021 Feb 26]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/713.

Council of Science Editors:

Niedzielski JS. INVESTIGATION OF RADIATION INJURY IN THE ESOPHAGUS FROM DEFINITIVE CHEMORADIATION THERAPY USING NOVEL IMAGING BIOMARKERS. [Doctoral Dissertation]. Texas Medical Center; 2016. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/713


Texas Medical Center

10. Turner, Heather N. Cellular and genetic bases of cold nociception and nociceptive sensitization in Drosophila larvae.

Degree: PhD, 2016, Texas Medical Center

  Organisms from flies to mammals utilize thermoreceptors to detect and respond to noxious thermal stimuli. Although much is understood about noxious heat avoidance, our… (more)

Subjects/Keywords: Drosophila; nociception; pain; thermosensation; cold; sensitization; neurons; TRP channels; genetics; neuroscience; Molecular and Cellular Neuroscience

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APA (6th Edition):

Turner, H. N. (2016). Cellular and genetic bases of cold nociception and nociceptive sensitization in Drosophila larvae. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/714

Chicago Manual of Style (16th Edition):

Turner, Heather N. “Cellular and genetic bases of cold nociception and nociceptive sensitization in Drosophila larvae.” 2016. Doctoral Dissertation, Texas Medical Center. Accessed February 26, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/714.

MLA Handbook (7th Edition):

Turner, Heather N. “Cellular and genetic bases of cold nociception and nociceptive sensitization in Drosophila larvae.” 2016. Web. 26 Feb 2021.

Vancouver:

Turner HN. Cellular and genetic bases of cold nociception and nociceptive sensitization in Drosophila larvae. [Internet] [Doctoral dissertation]. Texas Medical Center; 2016. [cited 2021 Feb 26]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/714.

Council of Science Editors:

Turner HN. Cellular and genetic bases of cold nociception and nociceptive sensitization in Drosophila larvae. [Doctoral Dissertation]. Texas Medical Center; 2016. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/714


Texas Medical Center

11. MacLellan, Christopher James. Determination of Thermal Dose Model Parameters Using Magnetic Resonance Imaging.

Degree: PhD, 2016, Texas Medical Center

  Magnetic Resonance Temperature Imaging (MRTI) is a powerful technique for noninvasively monitoring temperature during minimally invasive thermal therapy procedures. When coupled with thermal dose… (more)

Subjects/Keywords: Magnetic Resonance Imaging; Thermal Dose Modeling; Thermal Ablation; Laser Ablation; Arrhenius Parameters; Thermal Damage; Medicine and Health Sciences; Radiology

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APA (6th Edition):

MacLellan, C. J. (2016). Determination of Thermal Dose Model Parameters Using Magnetic Resonance Imaging. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/715

Chicago Manual of Style (16th Edition):

MacLellan, Christopher James. “Determination of Thermal Dose Model Parameters Using Magnetic Resonance Imaging.” 2016. Doctoral Dissertation, Texas Medical Center. Accessed February 26, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/715.

MLA Handbook (7th Edition):

MacLellan, Christopher James. “Determination of Thermal Dose Model Parameters Using Magnetic Resonance Imaging.” 2016. Web. 26 Feb 2021.

Vancouver:

MacLellan CJ. Determination of Thermal Dose Model Parameters Using Magnetic Resonance Imaging. [Internet] [Doctoral dissertation]. Texas Medical Center; 2016. [cited 2021 Feb 26]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/715.

Council of Science Editors:

MacLellan CJ. Determination of Thermal Dose Model Parameters Using Magnetic Resonance Imaging. [Doctoral Dissertation]. Texas Medical Center; 2016. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/715


Texas Medical Center

12. Glitza Oliva, Isabella C. LONG-TERM SURVIVAL IN METASTATIC MELANOMA PATIENTS WITH LEPTOMENINGEAL DISEASE TREATED WITH INTRATHECAL INTERLEUKIN-2.

Degree: MS, 2016, Texas Medical Center

  BACKGROUND: Metastatic melanoma patients with leptomeningeal disease (LMD) have an extremely poor prognosis and a paucity of effective treatment options. We assessed the safety… (more)

Subjects/Keywords: melanoma; interleukin-2; intrathecal; leptomeningeal disease; Medicine and Health Sciences

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APA (6th Edition):

Glitza Oliva, I. C. (2016). LONG-TERM SURVIVAL IN METASTATIC MELANOMA PATIENTS WITH LEPTOMENINGEAL DISEASE TREATED WITH INTRATHECAL INTERLEUKIN-2. (Thesis). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/716

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Glitza Oliva, Isabella C. “LONG-TERM SURVIVAL IN METASTATIC MELANOMA PATIENTS WITH LEPTOMENINGEAL DISEASE TREATED WITH INTRATHECAL INTERLEUKIN-2.” 2016. Thesis, Texas Medical Center. Accessed February 26, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/716.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Glitza Oliva, Isabella C. “LONG-TERM SURVIVAL IN METASTATIC MELANOMA PATIENTS WITH LEPTOMENINGEAL DISEASE TREATED WITH INTRATHECAL INTERLEUKIN-2.” 2016. Web. 26 Feb 2021.

Vancouver:

Glitza Oliva IC. LONG-TERM SURVIVAL IN METASTATIC MELANOMA PATIENTS WITH LEPTOMENINGEAL DISEASE TREATED WITH INTRATHECAL INTERLEUKIN-2. [Internet] [Thesis]. Texas Medical Center; 2016. [cited 2021 Feb 26]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/716.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Glitza Oliva IC. LONG-TERM SURVIVAL IN METASTATIC MELANOMA PATIENTS WITH LEPTOMENINGEAL DISEASE TREATED WITH INTRATHECAL INTERLEUKIN-2. [Thesis]. Texas Medical Center; 2016. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/716

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas Medical Center

13. Gloston, Gabrielle F. The Role of Two Homologous E3 Ligases in Muscle Physiology.

Degree: MS, 2016, Texas Medical Center

  Ubiquitin-mediated proteasomal degradation is an essential cellular function that is coordinated by three key components: E1 ubiquitin activating enzyme, E2 ubiquitin conjugating enzyme, and… (more)

Subjects/Keywords: FBXL3; FBXL21; E3 ligases; circadian; muscle; protein degradation; Life Sciences; Medicine and Health Sciences

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APA (6th Edition):

Gloston, G. F. (2016). The Role of Two Homologous E3 Ligases in Muscle Physiology. (Thesis). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/717

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gloston, Gabrielle F. “The Role of Two Homologous E3 Ligases in Muscle Physiology.” 2016. Thesis, Texas Medical Center. Accessed February 26, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/717.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gloston, Gabrielle F. “The Role of Two Homologous E3 Ligases in Muscle Physiology.” 2016. Web. 26 Feb 2021.

Vancouver:

Gloston GF. The Role of Two Homologous E3 Ligases in Muscle Physiology. [Internet] [Thesis]. Texas Medical Center; 2016. [cited 2021 Feb 26]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/717.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gloston GF. The Role of Two Homologous E3 Ligases in Muscle Physiology. [Thesis]. Texas Medical Center; 2016. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/717

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas Medical Center

14. Herold, Jennifer L. THE ROLE OF STREPTOCOCCUS GALLOLYTICUS SUBSPECIES GALLOLYTICUS IN COLON CANCER DEVELOPMENT.

Degree: PhD, 2016, Texas Medical Center

  Colorectal cancer (CRC) is the third most common cancer in men and women and is also the third most common cause of cancer death.… (more)

Subjects/Keywords: Colorectal cancer; microbiome; Streptococcus gallolyticus; Streptococcus bovis; β-catenin; tumorigenesis; Bacteriology; Cancer Biology; Medicine and Health Sciences

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APA (6th Edition):

Herold, J. L. (2016). THE ROLE OF STREPTOCOCCUS GALLOLYTICUS SUBSPECIES GALLOLYTICUS IN COLON CANCER DEVELOPMENT. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/718

Chicago Manual of Style (16th Edition):

Herold, Jennifer L. “THE ROLE OF STREPTOCOCCUS GALLOLYTICUS SUBSPECIES GALLOLYTICUS IN COLON CANCER DEVELOPMENT.” 2016. Doctoral Dissertation, Texas Medical Center. Accessed February 26, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/718.

MLA Handbook (7th Edition):

Herold, Jennifer L. “THE ROLE OF STREPTOCOCCUS GALLOLYTICUS SUBSPECIES GALLOLYTICUS IN COLON CANCER DEVELOPMENT.” 2016. Web. 26 Feb 2021.

Vancouver:

Herold JL. THE ROLE OF STREPTOCOCCUS GALLOLYTICUS SUBSPECIES GALLOLYTICUS IN COLON CANCER DEVELOPMENT. [Internet] [Doctoral dissertation]. Texas Medical Center; 2016. [cited 2021 Feb 26]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/718.

Council of Science Editors:

Herold JL. THE ROLE OF STREPTOCOCCUS GALLOLYTICUS SUBSPECIES GALLOLYTICUS IN COLON CANCER DEVELOPMENT. [Doctoral Dissertation]. Texas Medical Center; 2016. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/718


Texas Medical Center

15. Tito, Antonio Joel, Jr. Characterization of Vesicular Monoamine Transporter 2 and its role in Parkinson's Disease Pathogenesis using Drosophila.

Degree: PhD, 2016, Texas Medical Center

  Parkinson’s disease (PD) is a progressive neurodegenerative disorder caused by the selective loss of the dopaminergic neurons in the Substantia nigra pars compacta region… (more)

Subjects/Keywords: Neuroscience; Neurodegenerative Disease; Drosophila; Brain; Dopamine Neurons; Dopamine; Parkinson's Disease; Parkin; VMAT; Cell Biology; Medicine and Health Sciences; Molecular and Cellular Neuroscience; Molecular Biology; Molecular Genetics

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APA (6th Edition):

Tito, Antonio Joel, J. (2016). Characterization of Vesicular Monoamine Transporter 2 and its role in Parkinson's Disease Pathogenesis using Drosophila. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/719

Chicago Manual of Style (16th Edition):

Tito, Antonio Joel, Jr. “Characterization of Vesicular Monoamine Transporter 2 and its role in Parkinson's Disease Pathogenesis using Drosophila.” 2016. Doctoral Dissertation, Texas Medical Center. Accessed February 26, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/719.

MLA Handbook (7th Edition):

Tito, Antonio Joel, Jr. “Characterization of Vesicular Monoamine Transporter 2 and its role in Parkinson's Disease Pathogenesis using Drosophila.” 2016. Web. 26 Feb 2021.

Vancouver:

Tito, Antonio Joel J. Characterization of Vesicular Monoamine Transporter 2 and its role in Parkinson's Disease Pathogenesis using Drosophila. [Internet] [Doctoral dissertation]. Texas Medical Center; 2016. [cited 2021 Feb 26]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/719.

Council of Science Editors:

Tito, Antonio Joel J. Characterization of Vesicular Monoamine Transporter 2 and its role in Parkinson's Disease Pathogenesis using Drosophila. [Doctoral Dissertation]. Texas Medical Center; 2016. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/719


Texas Medical Center

16. Dominguez, Emily. USING MOUSE MODELS TO DEFINE HOW THE P53 R72P POLYMORPHISM IMPACTS THE ADVERSE EFFECTS OF DOXORUBICIN AND IONIZING RADIATION.

Degree: PhD, 2016, Texas Medical Center

  The single nucleotide polymorphism (SNP) at codon 72 of the tumor suppressor gene p53 codes for either an arginine (R) or proline (P) (p53… (more)

Subjects/Keywords: mouse models; p53; single nucleotide polymorphism; genotoxins; Cell Biology; Genetics and Genomics; Medicine and Health Sciences; Other Pharmacology, Toxicology and Environmental Health

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APA (6th Edition):

Dominguez, E. (2016). USING MOUSE MODELS TO DEFINE HOW THE P53 R72P POLYMORPHISM IMPACTS THE ADVERSE EFFECTS OF DOXORUBICIN AND IONIZING RADIATION. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/720

Chicago Manual of Style (16th Edition):

Dominguez, Emily. “USING MOUSE MODELS TO DEFINE HOW THE P53 R72P POLYMORPHISM IMPACTS THE ADVERSE EFFECTS OF DOXORUBICIN AND IONIZING RADIATION.” 2016. Doctoral Dissertation, Texas Medical Center. Accessed February 26, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/720.

MLA Handbook (7th Edition):

Dominguez, Emily. “USING MOUSE MODELS TO DEFINE HOW THE P53 R72P POLYMORPHISM IMPACTS THE ADVERSE EFFECTS OF DOXORUBICIN AND IONIZING RADIATION.” 2016. Web. 26 Feb 2021.

Vancouver:

Dominguez E. USING MOUSE MODELS TO DEFINE HOW THE P53 R72P POLYMORPHISM IMPACTS THE ADVERSE EFFECTS OF DOXORUBICIN AND IONIZING RADIATION. [Internet] [Doctoral dissertation]. Texas Medical Center; 2016. [cited 2021 Feb 26]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/720.

Council of Science Editors:

Dominguez E. USING MOUSE MODELS TO DEFINE HOW THE P53 R72P POLYMORPHISM IMPACTS THE ADVERSE EFFECTS OF DOXORUBICIN AND IONIZING RADIATION. [Doctoral Dissertation]. Texas Medical Center; 2016. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/720


Texas Medical Center

17. Dhupkar, Pooja, 3012359. TARGETING PD-1/PDL-1 SIGNALING IN THE TREATMENT OF OSTEOSARCOMA LUNG METASTASIS.

Degree: PhD, 2016, Texas Medical Center

  Osteosarcoma (OS) manifests itself as pulmonary metastasis, which is a major cause of death in OS patients. Novel treatments like immunotherapy hold promise for… (more)

Subjects/Keywords: Programmed death ligand 1 (PDL-1); osteosarcoma; lung metastasis; immunotherapy; macrophages; Life Sciences

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APA (6th Edition):

Dhupkar, Pooja, 3. (2016). TARGETING PD-1/PDL-1 SIGNALING IN THE TREATMENT OF OSTEOSARCOMA LUNG METASTASIS. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/721

Chicago Manual of Style (16th Edition):

Dhupkar, Pooja, 3012359. “TARGETING PD-1/PDL-1 SIGNALING IN THE TREATMENT OF OSTEOSARCOMA LUNG METASTASIS.” 2016. Doctoral Dissertation, Texas Medical Center. Accessed February 26, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/721.

MLA Handbook (7th Edition):

Dhupkar, Pooja, 3012359. “TARGETING PD-1/PDL-1 SIGNALING IN THE TREATMENT OF OSTEOSARCOMA LUNG METASTASIS.” 2016. Web. 26 Feb 2021.

Vancouver:

Dhupkar, Pooja 3. TARGETING PD-1/PDL-1 SIGNALING IN THE TREATMENT OF OSTEOSARCOMA LUNG METASTASIS. [Internet] [Doctoral dissertation]. Texas Medical Center; 2016. [cited 2021 Feb 26]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/721.

Council of Science Editors:

Dhupkar, Pooja 3. TARGETING PD-1/PDL-1 SIGNALING IN THE TREATMENT OF OSTEOSARCOMA LUNG METASTASIS. [Doctoral Dissertation]. Texas Medical Center; 2016. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/721


Texas Medical Center

18. HUANG, KAI-LIEH. THE ROLE OF PHOSPHORYLATION IN PAM2 MOTIF-CONTAINING PROTEINS MEDIATED MESSENGER RNA DEADENYLATION.

Degree: PhD, 2016, Texas Medical Center

  Phosphorylation regulates many cellular processes. However, its role in mRNA deadenylation, a process to remove poly adenosines from the mature mRNA 3’ end tail,… (more)

Subjects/Keywords: mRNA deadenylation; PAM2 motif; Phosphorylation; Intrinsically disordered regions; PABP; Tob2; Pan3; Tnrc6c; JNK; CDK; Biochemistry; Molecular Biology

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APA (6th Edition):

HUANG, K. (2016). THE ROLE OF PHOSPHORYLATION IN PAM2 MOTIF-CONTAINING PROTEINS MEDIATED MESSENGER RNA DEADENYLATION. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/722

Chicago Manual of Style (16th Edition):

HUANG, KAI-LIEH. “THE ROLE OF PHOSPHORYLATION IN PAM2 MOTIF-CONTAINING PROTEINS MEDIATED MESSENGER RNA DEADENYLATION.” 2016. Doctoral Dissertation, Texas Medical Center. Accessed February 26, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/722.

MLA Handbook (7th Edition):

HUANG, KAI-LIEH. “THE ROLE OF PHOSPHORYLATION IN PAM2 MOTIF-CONTAINING PROTEINS MEDIATED MESSENGER RNA DEADENYLATION.” 2016. Web. 26 Feb 2021.

Vancouver:

HUANG K. THE ROLE OF PHOSPHORYLATION IN PAM2 MOTIF-CONTAINING PROTEINS MEDIATED MESSENGER RNA DEADENYLATION. [Internet] [Doctoral dissertation]. Texas Medical Center; 2016. [cited 2021 Feb 26]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/722.

Council of Science Editors:

HUANG K. THE ROLE OF PHOSPHORYLATION IN PAM2 MOTIF-CONTAINING PROTEINS MEDIATED MESSENGER RNA DEADENYLATION. [Doctoral Dissertation]. Texas Medical Center; 2016. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/722


Texas Medical Center

19. Fletcher, Eliot S. ORGAN SPECIFIC VASCULAR RESPONSE TO FIBROSIS AFFECTS BREAST CANCER METASTATIC ORGANOTROPISM.

Degree: PhD, 2016, Texas Medical Center

  The solid tumor microenvironment, pre-metastatic niche, and fibrotic environment are known to have significant biochemical and biomechanical similarities to the fibrotic environment. All have… (more)

Subjects/Keywords: Medicine and Health Sciences

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APA (6th Edition):

Fletcher, E. S. (2016). ORGAN SPECIFIC VASCULAR RESPONSE TO FIBROSIS AFFECTS BREAST CANCER METASTATIC ORGANOTROPISM. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/723

Chicago Manual of Style (16th Edition):

Fletcher, Eliot S. “ORGAN SPECIFIC VASCULAR RESPONSE TO FIBROSIS AFFECTS BREAST CANCER METASTATIC ORGANOTROPISM.” 2016. Doctoral Dissertation, Texas Medical Center. Accessed February 26, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/723.

MLA Handbook (7th Edition):

Fletcher, Eliot S. “ORGAN SPECIFIC VASCULAR RESPONSE TO FIBROSIS AFFECTS BREAST CANCER METASTATIC ORGANOTROPISM.” 2016. Web. 26 Feb 2021.

Vancouver:

Fletcher ES. ORGAN SPECIFIC VASCULAR RESPONSE TO FIBROSIS AFFECTS BREAST CANCER METASTATIC ORGANOTROPISM. [Internet] [Doctoral dissertation]. Texas Medical Center; 2016. [cited 2021 Feb 26]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/723.

Council of Science Editors:

Fletcher ES. ORGAN SPECIFIC VASCULAR RESPONSE TO FIBROSIS AFFECTS BREAST CANCER METASTATIC ORGANOTROPISM. [Doctoral Dissertation]. Texas Medical Center; 2016. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/723


Texas Medical Center

20. RIvera-Valentin, Rocio K. Her4 Promotes a Stem-like Phenotype in Osteosarcoma.

Degree: PhD, 2016, Texas Medical Center

  Metastatic disease to the lungs is the primary cause of death for patients with pediatric osteosarcoma (OS). OS has a high degree of heterogeneity… (more)

Subjects/Keywords: osteosarcoma; stem-like phenotype; Her4; ERBB4; Oncology; Translational Medical Research

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APA (6th Edition):

RIvera-Valentin, R. K. (2016). Her4 Promotes a Stem-like Phenotype in Osteosarcoma. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/724

Chicago Manual of Style (16th Edition):

RIvera-Valentin, Rocio K. “Her4 Promotes a Stem-like Phenotype in Osteosarcoma.” 2016. Doctoral Dissertation, Texas Medical Center. Accessed February 26, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/724.

MLA Handbook (7th Edition):

RIvera-Valentin, Rocio K. “Her4 Promotes a Stem-like Phenotype in Osteosarcoma.” 2016. Web. 26 Feb 2021.

Vancouver:

RIvera-Valentin RK. Her4 Promotes a Stem-like Phenotype in Osteosarcoma. [Internet] [Doctoral dissertation]. Texas Medical Center; 2016. [cited 2021 Feb 26]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/724.

Council of Science Editors:

RIvera-Valentin RK. Her4 Promotes a Stem-like Phenotype in Osteosarcoma. [Doctoral Dissertation]. Texas Medical Center; 2016. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/724


Texas Medical Center

21. Mananadhar, Mandira. NUCLEOTIDE EXCISION REPAIR, CROSSLINK REPAIR AND TRANSCRIPTIONAL FUNCTION OF XPA IN HUMAN CELLS.

Degree: PhD, 2016, Texas Medical Center

  Nucleotide excision repair (NER) in mammalian cells includes xeroderma pigmentosum group A protein (XPA) as a core factor. XPA and other NER proteins have… (more)

Subjects/Keywords: Nucleotide excision repair; XPA; Interstrand crosslink; Transcription; DNA repair; Medicine and Health Sciences

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APA (6th Edition):

Mananadhar, M. (2016). NUCLEOTIDE EXCISION REPAIR, CROSSLINK REPAIR AND TRANSCRIPTIONAL FUNCTION OF XPA IN HUMAN CELLS. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/726

Chicago Manual of Style (16th Edition):

Mananadhar, Mandira. “NUCLEOTIDE EXCISION REPAIR, CROSSLINK REPAIR AND TRANSCRIPTIONAL FUNCTION OF XPA IN HUMAN CELLS.” 2016. Doctoral Dissertation, Texas Medical Center. Accessed February 26, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/726.

MLA Handbook (7th Edition):

Mananadhar, Mandira. “NUCLEOTIDE EXCISION REPAIR, CROSSLINK REPAIR AND TRANSCRIPTIONAL FUNCTION OF XPA IN HUMAN CELLS.” 2016. Web. 26 Feb 2021.

Vancouver:

Mananadhar M. NUCLEOTIDE EXCISION REPAIR, CROSSLINK REPAIR AND TRANSCRIPTIONAL FUNCTION OF XPA IN HUMAN CELLS. [Internet] [Doctoral dissertation]. Texas Medical Center; 2016. [cited 2021 Feb 26]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/726.

Council of Science Editors:

Mananadhar M. NUCLEOTIDE EXCISION REPAIR, CROSSLINK REPAIR AND TRANSCRIPTIONAL FUNCTION OF XPA IN HUMAN CELLS. [Doctoral Dissertation]. Texas Medical Center; 2016. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/726


Texas Medical Center

22. Vila, Ph.D., Alejandro. MAGUK SCAFFOLDS ORGANIZE A KEY SYNAPTIC COMPLEX IN HORIZONTAL CELL PROCESSES CONTACTING PHOTORECEPTORS.

Degree: PhD, 2016, Texas Medical Center

  Synaptic processes and plasticity of synapses are mediated by large suites of proteins. In most cases, many of these proteins are tethered together by… (more)

Subjects/Keywords: Dlg3; Dlg2; Kcnj2; Grik2; synaptic signaling; proximity ligation assays; Medicine and Health Sciences; Molecular and Cellular Neuroscience; Systems Neuroscience

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APA (6th Edition):

Vila, Ph.D., A. (2016). MAGUK SCAFFOLDS ORGANIZE A KEY SYNAPTIC COMPLEX IN HORIZONTAL CELL PROCESSES CONTACTING PHOTORECEPTORS. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/727

Chicago Manual of Style (16th Edition):

Vila, Ph.D., Alejandro. “MAGUK SCAFFOLDS ORGANIZE A KEY SYNAPTIC COMPLEX IN HORIZONTAL CELL PROCESSES CONTACTING PHOTORECEPTORS.” 2016. Doctoral Dissertation, Texas Medical Center. Accessed February 26, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/727.

MLA Handbook (7th Edition):

Vila, Ph.D., Alejandro. “MAGUK SCAFFOLDS ORGANIZE A KEY SYNAPTIC COMPLEX IN HORIZONTAL CELL PROCESSES CONTACTING PHOTORECEPTORS.” 2016. Web. 26 Feb 2021.

Vancouver:

Vila, Ph.D. A. MAGUK SCAFFOLDS ORGANIZE A KEY SYNAPTIC COMPLEX IN HORIZONTAL CELL PROCESSES CONTACTING PHOTORECEPTORS. [Internet] [Doctoral dissertation]. Texas Medical Center; 2016. [cited 2021 Feb 26]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/727.

Council of Science Editors:

Vila, Ph.D. A. MAGUK SCAFFOLDS ORGANIZE A KEY SYNAPTIC COMPLEX IN HORIZONTAL CELL PROCESSES CONTACTING PHOTORECEPTORS. [Doctoral Dissertation]. Texas Medical Center; 2016. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/727


Texas Medical Center

23. Pan, Rongqing. TARGETING APOPTOTIC PATHWAYS TO OVERCOME DRUG RESISTANCE IN ACUTE MYELOID LEUKEMIA.

Degree: PhD, 2017, Texas Medical Center

  Evasion of apoptosis is integral to tumorigenesis and drug resistance. BCL-2 and p53 proteins represent two focal nodes in convergent apoptosis signaling. Upregulation of… (more)

Subjects/Keywords: Mitochondrial apoptosis; Bcl-2 family proteins; Drug resistance; p53; MDM2; Mcl-1; G1 arrest; BH3 profiling; Acute myeloid leukemia; Biology; Cancer Biology; Cell and Developmental Biology; Life Sciences; Medicine and Health Sciences

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APA (6th Edition):

Pan, R. (2017). TARGETING APOPTOTIC PATHWAYS TO OVERCOME DRUG RESISTANCE IN ACUTE MYELOID LEUKEMIA. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/728

Chicago Manual of Style (16th Edition):

Pan, Rongqing. “TARGETING APOPTOTIC PATHWAYS TO OVERCOME DRUG RESISTANCE IN ACUTE MYELOID LEUKEMIA.” 2017. Doctoral Dissertation, Texas Medical Center. Accessed February 26, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/728.

MLA Handbook (7th Edition):

Pan, Rongqing. “TARGETING APOPTOTIC PATHWAYS TO OVERCOME DRUG RESISTANCE IN ACUTE MYELOID LEUKEMIA.” 2017. Web. 26 Feb 2021.

Vancouver:

Pan R. TARGETING APOPTOTIC PATHWAYS TO OVERCOME DRUG RESISTANCE IN ACUTE MYELOID LEUKEMIA. [Internet] [Doctoral dissertation]. Texas Medical Center; 2017. [cited 2021 Feb 26]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/728.

Council of Science Editors:

Pan R. TARGETING APOPTOTIC PATHWAYS TO OVERCOME DRUG RESISTANCE IN ACUTE MYELOID LEUKEMIA. [Doctoral Dissertation]. Texas Medical Center; 2017. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/728


Texas Medical Center

24. hu, Xin. INTEGRATIVE ANALYSIS OF OMICS DATA IN ADULT GLIOMA AND OTHER TCGA CANCERS TO GUIDE PRECISION MEDICINE.

Degree: PhD, 2017, Texas Medical Center

  Transcriptomic profiling and gene expression signatures have been widely applied as effective approaches for enhancing the molecular classification, diagnosis, prognosis or prediction of therapeutic… (more)

Subjects/Keywords: Cancer genomics; Omics data mining; Transcriptomics profiling; Gene signature; Risk prediction; Tumor microenvironment; Precision medicine; Glioma; Pan-cancer; Fusion transcripts; Genomic rearrangement; Next-generation sequencing; Prediction model; Disease Modeling; Health Information Technology; Medicine and Health Sciences; Translational Medical Research

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APA (6th Edition):

hu, X. (2017). INTEGRATIVE ANALYSIS OF OMICS DATA IN ADULT GLIOMA AND OTHER TCGA CANCERS TO GUIDE PRECISION MEDICINE. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/729

Chicago Manual of Style (16th Edition):

hu, Xin. “INTEGRATIVE ANALYSIS OF OMICS DATA IN ADULT GLIOMA AND OTHER TCGA CANCERS TO GUIDE PRECISION MEDICINE.” 2017. Doctoral Dissertation, Texas Medical Center. Accessed February 26, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/729.

MLA Handbook (7th Edition):

hu, Xin. “INTEGRATIVE ANALYSIS OF OMICS DATA IN ADULT GLIOMA AND OTHER TCGA CANCERS TO GUIDE PRECISION MEDICINE.” 2017. Web. 26 Feb 2021.

Vancouver:

hu X. INTEGRATIVE ANALYSIS OF OMICS DATA IN ADULT GLIOMA AND OTHER TCGA CANCERS TO GUIDE PRECISION MEDICINE. [Internet] [Doctoral dissertation]. Texas Medical Center; 2017. [cited 2021 Feb 26]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/729.

Council of Science Editors:

hu X. INTEGRATIVE ANALYSIS OF OMICS DATA IN ADULT GLIOMA AND OTHER TCGA CANCERS TO GUIDE PRECISION MEDICINE. [Doctoral Dissertation]. Texas Medical Center; 2017. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/729


Texas Medical Center

25. Wang, Pingping. PROTEOMIC IDENTIFICATION OF HISTONE POST-TRANSLATIONAL MODIFICATIONS INDUCED BY DNA DOUBLE-STRAND BREAKS AND NOVEL PROTEINS INVOLVED IN THE DNA DAMAGE RESPONSE.

Degree: PhD, 2017, Texas Medical Center

  Inaccurate repair of DNA double-strand breaks (DSBs) can lead to DNA mutation and chromosome rearrangements, causing human diseases such as cancer. Although we know… (more)

Subjects/Keywords: DSB; PTM; Affinity Purification; Mass Spectrometry; Yeast; DNA repair; Checkpoint; Biology; Cell Biology; Life Sciences; Medicine and Health Sciences; Microbiology; Molecular Genetics

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APA (6th Edition):

Wang, P. (2017). PROTEOMIC IDENTIFICATION OF HISTONE POST-TRANSLATIONAL MODIFICATIONS INDUCED BY DNA DOUBLE-STRAND BREAKS AND NOVEL PROTEINS INVOLVED IN THE DNA DAMAGE RESPONSE. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/731

Chicago Manual of Style (16th Edition):

Wang, Pingping. “PROTEOMIC IDENTIFICATION OF HISTONE POST-TRANSLATIONAL MODIFICATIONS INDUCED BY DNA DOUBLE-STRAND BREAKS AND NOVEL PROTEINS INVOLVED IN THE DNA DAMAGE RESPONSE.” 2017. Doctoral Dissertation, Texas Medical Center. Accessed February 26, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/731.

MLA Handbook (7th Edition):

Wang, Pingping. “PROTEOMIC IDENTIFICATION OF HISTONE POST-TRANSLATIONAL MODIFICATIONS INDUCED BY DNA DOUBLE-STRAND BREAKS AND NOVEL PROTEINS INVOLVED IN THE DNA DAMAGE RESPONSE.” 2017. Web. 26 Feb 2021.

Vancouver:

Wang P. PROTEOMIC IDENTIFICATION OF HISTONE POST-TRANSLATIONAL MODIFICATIONS INDUCED BY DNA DOUBLE-STRAND BREAKS AND NOVEL PROTEINS INVOLVED IN THE DNA DAMAGE RESPONSE. [Internet] [Doctoral dissertation]. Texas Medical Center; 2017. [cited 2021 Feb 26]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/731.

Council of Science Editors:

Wang P. PROTEOMIC IDENTIFICATION OF HISTONE POST-TRANSLATIONAL MODIFICATIONS INDUCED BY DNA DOUBLE-STRAND BREAKS AND NOVEL PROTEINS INVOLVED IN THE DNA DAMAGE RESPONSE. [Doctoral Dissertation]. Texas Medical Center; 2017. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/731


Texas Medical Center

26. Hunt, Albert J, Jr. The External Globus Pallidus: Bidirectional Control Over Anxiety-Related Behavior Mediated by CRFR1.

Degree: PhD, 2017, Texas Medical Center

 <h1>Abstract</h1> THE EXTERNAL GLOBUS PALLIDUS: BIDIRECTIONAL CONTROL OVER ANXIETY-RELATED BEHAVIOR MEDIATED BY CRFR1 Albert Lee Joseph Hunt, Jr., B.S. Advisory Professor: Shane Cunha, Ph.D. Corticotropin-releasing… (more)

Subjects/Keywords: GPe; external globus pallidus; CRF; CRFR1; anxiety; behavior; mice; parkinson's disease; psychiatric; Medicine and Health Sciences

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APA (6th Edition):

Hunt, Albert J, J. (2017). The External Globus Pallidus: Bidirectional Control Over Anxiety-Related Behavior Mediated by CRFR1. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/732

Chicago Manual of Style (16th Edition):

Hunt, Albert J, Jr. “The External Globus Pallidus: Bidirectional Control Over Anxiety-Related Behavior Mediated by CRFR1.” 2017. Doctoral Dissertation, Texas Medical Center. Accessed February 26, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/732.

MLA Handbook (7th Edition):

Hunt, Albert J, Jr. “The External Globus Pallidus: Bidirectional Control Over Anxiety-Related Behavior Mediated by CRFR1.” 2017. Web. 26 Feb 2021.

Vancouver:

Hunt, Albert J J. The External Globus Pallidus: Bidirectional Control Over Anxiety-Related Behavior Mediated by CRFR1. [Internet] [Doctoral dissertation]. Texas Medical Center; 2017. [cited 2021 Feb 26]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/732.

Council of Science Editors:

Hunt, Albert J J. The External Globus Pallidus: Bidirectional Control Over Anxiety-Related Behavior Mediated by CRFR1. [Doctoral Dissertation]. Texas Medical Center; 2017. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/732


Texas Medical Center

27. Kiany, Simin. EFFECT OF ENTINOSTAT ON NK CELL-MEDIATED CYTOTOXICITY AGAINST OS CELLS AND OS LUNG METASTSIS.

Degree: PhD, 2017, Texas Medical Center

  The purpose of this study was to investigate the effect of the HDAC inhibitor entinostat on the efficacy of NK cell therapy for OS… (more)

Subjects/Keywords: Osteosarcoma; NK cell; Entinostat; Life Sciences; Medicine and Health Sciences

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APA (6th Edition):

Kiany, S. (2017). EFFECT OF ENTINOSTAT ON NK CELL-MEDIATED CYTOTOXICITY AGAINST OS CELLS AND OS LUNG METASTSIS. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/733

Chicago Manual of Style (16th Edition):

Kiany, Simin. “EFFECT OF ENTINOSTAT ON NK CELL-MEDIATED CYTOTOXICITY AGAINST OS CELLS AND OS LUNG METASTSIS.” 2017. Doctoral Dissertation, Texas Medical Center. Accessed February 26, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/733.

MLA Handbook (7th Edition):

Kiany, Simin. “EFFECT OF ENTINOSTAT ON NK CELL-MEDIATED CYTOTOXICITY AGAINST OS CELLS AND OS LUNG METASTSIS.” 2017. Web. 26 Feb 2021.

Vancouver:

Kiany S. EFFECT OF ENTINOSTAT ON NK CELL-MEDIATED CYTOTOXICITY AGAINST OS CELLS AND OS LUNG METASTSIS. [Internet] [Doctoral dissertation]. Texas Medical Center; 2017. [cited 2021 Feb 26]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/733.

Council of Science Editors:

Kiany S. EFFECT OF ENTINOSTAT ON NK CELL-MEDIATED CYTOTOXICITY AGAINST OS CELLS AND OS LUNG METASTSIS. [Doctoral Dissertation]. Texas Medical Center; 2017. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/733


Texas Medical Center

28. Philip, Kemly Mary. The Hypoxic Adenosine Response Modulates Macrophage Differentiation and Contributes to Lung Disease.

Degree: PhD, 2017, Texas Medical Center

  Idiopathic pulmonary fibrosis (IPF) is a chronic lung disease which affects 5 - 8 million individuals worldwide and 200,000 individuals in the United States… (more)

Subjects/Keywords: idiopathic pulmonary fibrosis; adenosine; alternatively-activated macrophages; hypoxia-inducible factor 1-alpha; acute lung injury; Medicine and Health Sciences

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Philip, K. M. (2017). The Hypoxic Adenosine Response Modulates Macrophage Differentiation and Contributes to Lung Disease. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/734

Chicago Manual of Style (16th Edition):

Philip, Kemly Mary. “The Hypoxic Adenosine Response Modulates Macrophage Differentiation and Contributes to Lung Disease.” 2017. Doctoral Dissertation, Texas Medical Center. Accessed February 26, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/734.

MLA Handbook (7th Edition):

Philip, Kemly Mary. “The Hypoxic Adenosine Response Modulates Macrophage Differentiation and Contributes to Lung Disease.” 2017. Web. 26 Feb 2021.

Vancouver:

Philip KM. The Hypoxic Adenosine Response Modulates Macrophage Differentiation and Contributes to Lung Disease. [Internet] [Doctoral dissertation]. Texas Medical Center; 2017. [cited 2021 Feb 26]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/734.

Council of Science Editors:

Philip KM. The Hypoxic Adenosine Response Modulates Macrophage Differentiation and Contributes to Lung Disease. [Doctoral Dissertation]. Texas Medical Center; 2017. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/734


Texas Medical Center

29. Sun, Kaiqi. ROLE AND REGULATION OF SPHINGOSINE 1-PHOSPHATE IN ERYTHROCYTE.

Degree: PhD, 2017, Texas Medical Center

  Sphingosine 1-phosphate (S1P) is a bioactive signaling sphingolipid produced in every mammalian cell. It plays a variety of important roles both in and outside… (more)

Subjects/Keywords: Erythrocyte; Sphingosine 1-Phosphate; Sphingosine Kinase 1; Adenosine Signaling; Hypoxia Adaptation; Oxygen Release; Glucose Metabolism; Sickle Cell Disease; Biochemistry; Medicine and Health Sciences

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sun, K. (2017). ROLE AND REGULATION OF SPHINGOSINE 1-PHOSPHATE IN ERYTHROCYTE. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/736

Chicago Manual of Style (16th Edition):

Sun, Kaiqi. “ROLE AND REGULATION OF SPHINGOSINE 1-PHOSPHATE IN ERYTHROCYTE.” 2017. Doctoral Dissertation, Texas Medical Center. Accessed February 26, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/736.

MLA Handbook (7th Edition):

Sun, Kaiqi. “ROLE AND REGULATION OF SPHINGOSINE 1-PHOSPHATE IN ERYTHROCYTE.” 2017. Web. 26 Feb 2021.

Vancouver:

Sun K. ROLE AND REGULATION OF SPHINGOSINE 1-PHOSPHATE IN ERYTHROCYTE. [Internet] [Doctoral dissertation]. Texas Medical Center; 2017. [cited 2021 Feb 26]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/736.

Council of Science Editors:

Sun K. ROLE AND REGULATION OF SPHINGOSINE 1-PHOSPHATE IN ERYTHROCYTE. [Doctoral Dissertation]. Texas Medical Center; 2017. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/736


Texas Medical Center

30. Kwan, Suet Ying. Cyclin B1 mediates the effect of UCHL1 in promoting cell cycle progression in uterine papillary serous carcinoma.

Degree: PhD, 2017, Texas Medical Center

  Uterine papillary serous carcinoma (UPSC) is an aggressive form of endometrial cancer with poor survival rates and a high risk of recurrence. The rarity… (more)

Subjects/Keywords: Endometrial cancer; uterine papillary serous carcinoma; UCHL1; cyclin B1; Cancer Biology; Medicine and Health Sciences; Oncology; Translational Medical Research

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kwan, S. Y. (2017). Cyclin B1 mediates the effect of UCHL1 in promoting cell cycle progression in uterine papillary serous carcinoma. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/737

Chicago Manual of Style (16th Edition):

Kwan, Suet Ying. “Cyclin B1 mediates the effect of UCHL1 in promoting cell cycle progression in uterine papillary serous carcinoma.” 2017. Doctoral Dissertation, Texas Medical Center. Accessed February 26, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/737.

MLA Handbook (7th Edition):

Kwan, Suet Ying. “Cyclin B1 mediates the effect of UCHL1 in promoting cell cycle progression in uterine papillary serous carcinoma.” 2017. Web. 26 Feb 2021.

Vancouver:

Kwan SY. Cyclin B1 mediates the effect of UCHL1 in promoting cell cycle progression in uterine papillary serous carcinoma. [Internet] [Doctoral dissertation]. Texas Medical Center; 2017. [cited 2021 Feb 26]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/737.

Council of Science Editors:

Kwan SY. Cyclin B1 mediates the effect of UCHL1 in promoting cell cycle progression in uterine papillary serous carcinoma. [Doctoral Dissertation]. Texas Medical Center; 2017. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/737

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