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You searched for publisher:"Texas Medical Center". Showing records 1 – 30 of 804 total matches.

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Texas Medical Center

1. Ganguly, Dipyaman. Immune recognition of self nucleic acids driven by endogenous antimicrobial peptides: role in autoimmunity.

Degree: PhD, 2010, Texas Medical Center

 Innate immune recognition of extracellular host-derived self-DNA and self-RNA is prevented by endosomal seclusion of the Toll-like receptors (TLRs) in the dendritic cells (DCs). However,… (more)

Subjects/Keywords: pDC; DNA; RNA; LL37; dendritic cells; TLR; psoriasis; Biological Phenomena, Cell Phenomena, and Immunity; Cell Biology; Immune System Diseases; Immunity; Immunopathology; Medical Cell Biology; Medical Immunology; Medical Pathology; Pathological Conditions, Signs and Symptoms

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APA (6th Edition):

Ganguly, D. (2010). Immune recognition of self nucleic acids driven by endogenous antimicrobial peptides: role in autoimmunity. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/53

Chicago Manual of Style (16th Edition):

Ganguly, Dipyaman. “Immune recognition of self nucleic acids driven by endogenous antimicrobial peptides: role in autoimmunity.” 2010. Doctoral Dissertation, Texas Medical Center. Accessed November 18, 2017. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/53.

MLA Handbook (7th Edition):

Ganguly, Dipyaman. “Immune recognition of self nucleic acids driven by endogenous antimicrobial peptides: role in autoimmunity.” 2010. Web. 18 Nov 2017.

Vancouver:

Ganguly D. Immune recognition of self nucleic acids driven by endogenous antimicrobial peptides: role in autoimmunity. [Internet] [Doctoral dissertation]. Texas Medical Center; 2010. [cited 2017 Nov 18]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/53.

Council of Science Editors:

Ganguly D. Immune recognition of self nucleic acids driven by endogenous antimicrobial peptides: role in autoimmunity. [Doctoral Dissertation]. Texas Medical Center; 2010. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/53


Texas Medical Center

2. O'Day, Diana. GENETIC ANALYSIS OF THE FUNCTION OF THE DROSOPHILA DOUBLESEX-RELATED FACTOR dmrt93B.

Degree: PhD, 2010, Texas Medical Center

 DMRT (Doublesex and Mab-3 related transcription factor) proteins generally associated with sexual differentiation in many organisms share a common DNA binding domain and are often… (more)

Subjects/Keywords: dmrt93B; Doublesex and Mab-3 related transcription factor; Drosophila; GAL4 knock-in; Capillary Feeder (CAFÉ) assay; ends-out gene replacement; homologous recombination <; em>; in vivo<; /em>;

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APA (6th Edition):

O'Day, D. (2010). GENETIC ANALYSIS OF THE FUNCTION OF THE DROSOPHILA DOUBLESEX-RELATED FACTOR dmrt93B. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/54

Chicago Manual of Style (16th Edition):

O'Day, Diana. “GENETIC ANALYSIS OF THE FUNCTION OF THE DROSOPHILA DOUBLESEX-RELATED FACTOR dmrt93B.” 2010. Doctoral Dissertation, Texas Medical Center. Accessed November 18, 2017. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/54.

MLA Handbook (7th Edition):

O'Day, Diana. “GENETIC ANALYSIS OF THE FUNCTION OF THE DROSOPHILA DOUBLESEX-RELATED FACTOR dmrt93B.” 2010. Web. 18 Nov 2017.

Vancouver:

O'Day D. GENETIC ANALYSIS OF THE FUNCTION OF THE DROSOPHILA DOUBLESEX-RELATED FACTOR dmrt93B. [Internet] [Doctoral dissertation]. Texas Medical Center; 2010. [cited 2017 Nov 18]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/54.

Council of Science Editors:

O'Day D. GENETIC ANALYSIS OF THE FUNCTION OF THE DROSOPHILA DOUBLESEX-RELATED FACTOR dmrt93B. [Doctoral Dissertation]. Texas Medical Center; 2010. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/54


Texas Medical Center

3. Hennessey, Violeta G. A BAYESIAN APPROACH TO DOSE-RESPONSE ASSESSMENT AND DRUG-DRUG INTERACTION ANALYSIS: APPLICATION TO IN VITRO STUDIES.

Degree: PhD, 2010, Texas Medical Center

 The considerable search for synergistic agents in cancer research is motivated by the therapeutic benefits achieved by combining anti-cancer agents. Synergistic agents make it possible… (more)

Subjects/Keywords: Combination Index method; Drug interaction; Emax model; Interaction index; Median-effect principle; Loewe additivity model; Non-parametric regression methods; Monotone regression splines; Biometry; Biostatistics; Other Pharmacy and Pharmaceutical Sciences; Statistical Models

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APA (6th Edition):

Hennessey, V. G. (2010). A BAYESIAN APPROACH TO DOSE-RESPONSE ASSESSMENT AND DRUG-DRUG INTERACTION ANALYSIS: APPLICATION TO IN VITRO STUDIES. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/55

Chicago Manual of Style (16th Edition):

Hennessey, Violeta G. “A BAYESIAN APPROACH TO DOSE-RESPONSE ASSESSMENT AND DRUG-DRUG INTERACTION ANALYSIS: APPLICATION TO IN VITRO STUDIES.” 2010. Doctoral Dissertation, Texas Medical Center. Accessed November 18, 2017. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/55.

MLA Handbook (7th Edition):

Hennessey, Violeta G. “A BAYESIAN APPROACH TO DOSE-RESPONSE ASSESSMENT AND DRUG-DRUG INTERACTION ANALYSIS: APPLICATION TO IN VITRO STUDIES.” 2010. Web. 18 Nov 2017.

Vancouver:

Hennessey VG. A BAYESIAN APPROACH TO DOSE-RESPONSE ASSESSMENT AND DRUG-DRUG INTERACTION ANALYSIS: APPLICATION TO IN VITRO STUDIES. [Internet] [Doctoral dissertation]. Texas Medical Center; 2010. [cited 2017 Nov 18]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/55.

Council of Science Editors:

Hennessey VG. A BAYESIAN APPROACH TO DOSE-RESPONSE ASSESSMENT AND DRUG-DRUG INTERACTION ANALYSIS: APPLICATION TO IN VITRO STUDIES. [Doctoral Dissertation]. Texas Medical Center; 2010. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/55


Texas Medical Center

4. Taylor, Brian A. Dynamic Chemical Shift Imaging for Image-Guided Thermal Therapy.

Degree: PhD, 2010, Texas Medical Center

 Magnetic resonance temperature imaging (MRTI) is recognized as a noninvasive means to provide temperature imaging for guidance in thermal therapies. The most common method of… (more)

Subjects/Keywords: Magnetic Resonance; Chemical Shift Imaging; Thermal Therapy; Steighlitz-McBride; Autoregressive Moving Average (ARMA); k-means; Other Physics; Radiology

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APA (6th Edition):

Taylor, B. A. (2010). Dynamic Chemical Shift Imaging for Image-Guided Thermal Therapy. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/57

Chicago Manual of Style (16th Edition):

Taylor, Brian A. “Dynamic Chemical Shift Imaging for Image-Guided Thermal Therapy.” 2010. Doctoral Dissertation, Texas Medical Center. Accessed November 18, 2017. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/57.

MLA Handbook (7th Edition):

Taylor, Brian A. “Dynamic Chemical Shift Imaging for Image-Guided Thermal Therapy.” 2010. Web. 18 Nov 2017.

Vancouver:

Taylor BA. Dynamic Chemical Shift Imaging for Image-Guided Thermal Therapy. [Internet] [Doctoral dissertation]. Texas Medical Center; 2010. [cited 2017 Nov 18]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/57.

Council of Science Editors:

Taylor BA. Dynamic Chemical Shift Imaging for Image-Guided Thermal Therapy. [Doctoral Dissertation]. Texas Medical Center; 2010. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/57


Texas Medical Center

5. Ozawa, Michael G. Targeting the Blood-brain Barrier with a Non-canonical Iron-mimicry Mechanism.

Degree: PhD, 2010, Texas Medical Center

 Treatment of central nervous system (CNS) diseases is limited by the blood-brain barrier (BBB), a selective vascular interface restricting passage of most molecules from blood… (more)

Subjects/Keywords: Glioblastoma; angiogenesis; gene therapy; HSVtk; phage display; molecular imaging

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APA (6th Edition):

Ozawa, M. G. (2010). Targeting the Blood-brain Barrier with a Non-canonical Iron-mimicry Mechanism. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/58

Chicago Manual of Style (16th Edition):

Ozawa, Michael G. “Targeting the Blood-brain Barrier with a Non-canonical Iron-mimicry Mechanism.” 2010. Doctoral Dissertation, Texas Medical Center. Accessed November 18, 2017. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/58.

MLA Handbook (7th Edition):

Ozawa, Michael G. “Targeting the Blood-brain Barrier with a Non-canonical Iron-mimicry Mechanism.” 2010. Web. 18 Nov 2017.

Vancouver:

Ozawa MG. Targeting the Blood-brain Barrier with a Non-canonical Iron-mimicry Mechanism. [Internet] [Doctoral dissertation]. Texas Medical Center; 2010. [cited 2017 Nov 18]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/58.

Council of Science Editors:

Ozawa MG. Targeting the Blood-brain Barrier with a Non-canonical Iron-mimicry Mechanism. [Doctoral Dissertation]. Texas Medical Center; 2010. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/58


Texas Medical Center

6. Lee, Alessandro K. A NEW TUMOR SUPPRESSOR GENE CANDIDATE REGULATED BY THE NON-CODING RNA PCA3 IN HUMAN PROSTATE CANCER.

Degree: PhD, 2010, Texas Medical Center

 Prostate cancer is the second leading cause of cancer-related death and the most common non-skin cancer in men in the USA. Considerable advancements in the… (more)

Subjects/Keywords: PCA3; PC-TSGC; prostate cancer; non coding RNA; PRUNE2; BMCC1; tumor suppressor; Biology; Genomics; Male Urogenital Diseases; Molecular Biology; Oncology

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APA (6th Edition):

Lee, A. K. (2010). A NEW TUMOR SUPPRESSOR GENE CANDIDATE REGULATED BY THE NON-CODING RNA PCA3 IN HUMAN PROSTATE CANCER. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/22

Chicago Manual of Style (16th Edition):

Lee, Alessandro K. “A NEW TUMOR SUPPRESSOR GENE CANDIDATE REGULATED BY THE NON-CODING RNA PCA3 IN HUMAN PROSTATE CANCER.” 2010. Doctoral Dissertation, Texas Medical Center. Accessed November 18, 2017. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/22.

MLA Handbook (7th Edition):

Lee, Alessandro K. “A NEW TUMOR SUPPRESSOR GENE CANDIDATE REGULATED BY THE NON-CODING RNA PCA3 IN HUMAN PROSTATE CANCER.” 2010. Web. 18 Nov 2017.

Vancouver:

Lee AK. A NEW TUMOR SUPPRESSOR GENE CANDIDATE REGULATED BY THE NON-CODING RNA PCA3 IN HUMAN PROSTATE CANCER. [Internet] [Doctoral dissertation]. Texas Medical Center; 2010. [cited 2017 Nov 18]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/22.

Council of Science Editors:

Lee AK. A NEW TUMOR SUPPRESSOR GENE CANDIDATE REGULATED BY THE NON-CODING RNA PCA3 IN HUMAN PROSTATE CANCER. [Doctoral Dissertation]. Texas Medical Center; 2010. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/22


Texas Medical Center

7. Zhou, Yang. THE ROLE OF A2B ADENOSINE RECEPTOR SIGNALING IN ADENOSINE DEPENDENT LUNG DISEASE.

Degree: PhD, 2010, Texas Medical Center

 Chronic lung diseases and acute lung injuries are two distinctive pulmonary disorders that result in significant morbidity and mortality. Adenosine is a signaling nucleoside generated… (more)

Subjects/Keywords: Adenosine; Lung; COPD; Pulmonary Fibrosis; Biochemistry

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APA (6th Edition):

Zhou, Y. (2010). THE ROLE OF A2B ADENOSINE RECEPTOR SIGNALING IN ADENOSINE DEPENDENT LUNG DISEASE. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/13

Chicago Manual of Style (16th Edition):

Zhou, Yang. “THE ROLE OF A2B ADENOSINE RECEPTOR SIGNALING IN ADENOSINE DEPENDENT LUNG DISEASE.” 2010. Doctoral Dissertation, Texas Medical Center. Accessed November 18, 2017. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/13.

MLA Handbook (7th Edition):

Zhou, Yang. “THE ROLE OF A2B ADENOSINE RECEPTOR SIGNALING IN ADENOSINE DEPENDENT LUNG DISEASE.” 2010. Web. 18 Nov 2017.

Vancouver:

Zhou Y. THE ROLE OF A2B ADENOSINE RECEPTOR SIGNALING IN ADENOSINE DEPENDENT LUNG DISEASE. [Internet] [Doctoral dissertation]. Texas Medical Center; 2010. [cited 2017 Nov 18]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/13.

Council of Science Editors:

Zhou Y. THE ROLE OF A2B ADENOSINE RECEPTOR SIGNALING IN ADENOSINE DEPENDENT LUNG DISEASE. [Doctoral Dissertation]. Texas Medical Center; 2010. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/13


Texas Medical Center

8. Latham, (Leigh) Beth T. Protein-Protein Interactions That Regulate Neurotransmitter Release from Retinal Ribbon Synapses.

Degree: PhD, 2010, Texas Medical Center

 Protein-Protein Interactions That Regulate Neurotransmitter Release from Retinal Ribbon Synapses Photoreceptors and bipolar cells in the retina form specialized chemical synapses called ribbon synapses. This… (more)

Subjects/Keywords: syntaxin 3B; retinal ribbon synapses; Molecular and Cellular Neuroscience

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APA (6th Edition):

Latham, (. B. T. (2010). Protein-Protein Interactions That Regulate Neurotransmitter Release from Retinal Ribbon Synapses. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/29

Chicago Manual of Style (16th Edition):

Latham, (Leigh) Beth T. “Protein-Protein Interactions That Regulate Neurotransmitter Release from Retinal Ribbon Synapses.” 2010. Doctoral Dissertation, Texas Medical Center. Accessed November 18, 2017. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/29.

MLA Handbook (7th Edition):

Latham, (Leigh) Beth T. “Protein-Protein Interactions That Regulate Neurotransmitter Release from Retinal Ribbon Synapses.” 2010. Web. 18 Nov 2017.

Vancouver:

Latham (BT. Protein-Protein Interactions That Regulate Neurotransmitter Release from Retinal Ribbon Synapses. [Internet] [Doctoral dissertation]. Texas Medical Center; 2010. [cited 2017 Nov 18]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/29.

Council of Science Editors:

Latham (BT. Protein-Protein Interactions That Regulate Neurotransmitter Release from Retinal Ribbon Synapses. [Doctoral Dissertation]. Texas Medical Center; 2010. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/29


Texas Medical Center

9. Newman, Leslie A. Attitudes of Parents at risk of inheriting Li-Fraumeni Syndrome towards predictive genetic testing in their minor-aged children.

Degree: MS, 2010, Texas Medical Center

 Li-Fraumeni Syndrome (LFS) is a hereditary cancer syndrome which predisposes individuals to cancer beginning in childhood. These risks are spread across a lifetime, from early… (more)

Subjects/Keywords: Li-Fraumeni Syndrome; genetic testing; children; Bioethics and Medical Ethics; Genetics

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APA (6th Edition):

Newman, L. A. (2010). Attitudes of Parents at risk of inheriting Li-Fraumeni Syndrome towards predictive genetic testing in their minor-aged children. (Masters Thesis). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/38

Chicago Manual of Style (16th Edition):

Newman, Leslie A. “Attitudes of Parents at risk of inheriting Li-Fraumeni Syndrome towards predictive genetic testing in their minor-aged children.” 2010. Masters Thesis, Texas Medical Center. Accessed November 18, 2017. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/38.

MLA Handbook (7th Edition):

Newman, Leslie A. “Attitudes of Parents at risk of inheriting Li-Fraumeni Syndrome towards predictive genetic testing in their minor-aged children.” 2010. Web. 18 Nov 2017.

Vancouver:

Newman LA. Attitudes of Parents at risk of inheriting Li-Fraumeni Syndrome towards predictive genetic testing in their minor-aged children. [Internet] [Masters thesis]. Texas Medical Center; 2010. [cited 2017 Nov 18]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/38.

Council of Science Editors:

Newman LA. Attitudes of Parents at risk of inheriting Li-Fraumeni Syndrome towards predictive genetic testing in their minor-aged children. [Masters Thesis]. Texas Medical Center; 2010. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/38


Texas Medical Center

10. Schaeffer, Daneen. THE DOMAINS OF THE CATALYTIC SUBUNIT OF THE EUKARYOTIC RNA DEGRADING EXOSOME, RRP44P, HAVE DISTINCT FUNCTIONS.

Degree: PhD, 2010, Texas Medical Center

 The exosome is a 3’ to 5’ exoribonuclease complex that consists of ten essential subunits. In the cytoplasm, the exosome degrades mRNA in a general… (more)

Subjects/Keywords: S. cerevisiae; mRNA degradation; eukaryotic gene expression; Molecular Genetics; Other Microbiology

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APA (6th Edition):

Schaeffer, D. (2010). THE DOMAINS OF THE CATALYTIC SUBUNIT OF THE EUKARYOTIC RNA DEGRADING EXOSOME, RRP44P, HAVE DISTINCT FUNCTIONS. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/56

Chicago Manual of Style (16th Edition):

Schaeffer, Daneen. “THE DOMAINS OF THE CATALYTIC SUBUNIT OF THE EUKARYOTIC RNA DEGRADING EXOSOME, RRP44P, HAVE DISTINCT FUNCTIONS.” 2010. Doctoral Dissertation, Texas Medical Center. Accessed November 18, 2017. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/56.

MLA Handbook (7th Edition):

Schaeffer, Daneen. “THE DOMAINS OF THE CATALYTIC SUBUNIT OF THE EUKARYOTIC RNA DEGRADING EXOSOME, RRP44P, HAVE DISTINCT FUNCTIONS.” 2010. Web. 18 Nov 2017.

Vancouver:

Schaeffer D. THE DOMAINS OF THE CATALYTIC SUBUNIT OF THE EUKARYOTIC RNA DEGRADING EXOSOME, RRP44P, HAVE DISTINCT FUNCTIONS. [Internet] [Doctoral dissertation]. Texas Medical Center; 2010. [cited 2017 Nov 18]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/56.

Council of Science Editors:

Schaeffer D. THE DOMAINS OF THE CATALYTIC SUBUNIT OF THE EUKARYOTIC RNA DEGRADING EXOSOME, RRP44P, HAVE DISTINCT FUNCTIONS. [Doctoral Dissertation]. Texas Medical Center; 2010. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/56


Texas Medical Center

11. Ester, Audrey R. Analysis of Variation in Clubfoot Candidate Genes.

Degree: PhD, 2010, Texas Medical Center

 Isolated clubfoot, a common birth defect occurring in more than 135,000 livebirths worldwide each year, is associated with significant health care and financial burdens. Clubfoot… (more)

Subjects/Keywords: genotyping; clubfoot; complex disease; limb development; Genetics; Molecular Genetics

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APA (6th Edition):

Ester, A. R. (2010). Analysis of Variation in Clubfoot Candidate Genes. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/7

Chicago Manual of Style (16th Edition):

Ester, Audrey R. “Analysis of Variation in Clubfoot Candidate Genes.” 2010. Doctoral Dissertation, Texas Medical Center. Accessed November 18, 2017. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/7.

MLA Handbook (7th Edition):

Ester, Audrey R. “Analysis of Variation in Clubfoot Candidate Genes.” 2010. Web. 18 Nov 2017.

Vancouver:

Ester AR. Analysis of Variation in Clubfoot Candidate Genes. [Internet] [Doctoral dissertation]. Texas Medical Center; 2010. [cited 2017 Nov 18]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/7.

Council of Science Editors:

Ester AR. Analysis of Variation in Clubfoot Candidate Genes. [Doctoral Dissertation]. Texas Medical Center; 2010. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/7


Texas Medical Center

12. Kurinna, Svitlana M. NEW TARGET GENES FOR TUMOR SUPPRESSORS p53 AND p73 IN REGENERATING LIVER.

Degree: PhD, 2010, Texas Medical Center

 The p53-family of proteins regulates expression of target genes during tissue development and differentiation. Within the p53-family, p53 and p73 have hepatic-specific functions in development… (more)

Subjects/Keywords: p53; p73; forkhead; transcription; liver; regeneration; Biology; Cell and Developmental Biology; Laboratory and Basic Science Research; Medicine and Health Sciences

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APA (6th Edition):

Kurinna, S. M. (2010). NEW TARGET GENES FOR TUMOR SUPPRESSORS p53 AND p73 IN REGENERATING LIVER. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/20

Chicago Manual of Style (16th Edition):

Kurinna, Svitlana M. “NEW TARGET GENES FOR TUMOR SUPPRESSORS p53 AND p73 IN REGENERATING LIVER.” 2010. Doctoral Dissertation, Texas Medical Center. Accessed November 18, 2017. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/20.

MLA Handbook (7th Edition):

Kurinna, Svitlana M. “NEW TARGET GENES FOR TUMOR SUPPRESSORS p53 AND p73 IN REGENERATING LIVER.” 2010. Web. 18 Nov 2017.

Vancouver:

Kurinna SM. NEW TARGET GENES FOR TUMOR SUPPRESSORS p53 AND p73 IN REGENERATING LIVER. [Internet] [Doctoral dissertation]. Texas Medical Center; 2010. [cited 2017 Nov 18]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/20.

Council of Science Editors:

Kurinna SM. NEW TARGET GENES FOR TUMOR SUPPRESSORS p53 AND p73 IN REGENERATING LIVER. [Doctoral Dissertation]. Texas Medical Center; 2010. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/20


Texas Medical Center

13. Vishwamitra, Deeksha. TYPE I INSULIN-LIKE GROWTH FACTOR RECEPTOR TYROSINE KINASE AS A MOLECULAR TARGET IN MANTLE CELL LYMPHOMA.

Degree: MS, 2010, Texas Medical Center

 Mantle cell lymphoma (MCL) is an aggressive B-cell lymphoid malignancy representing 5-10% of all non-Hodgkin’s lymphomas. It is distinguished by the t(11;14)(q13;q32) chromosomal translocation that… (more)

Subjects/Keywords: IGF-IR; mantle cell lymphoma; cyclin D1; picropodophyllin; Medical Pathology

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APA (6th Edition):

Vishwamitra, D. (2010). TYPE I INSULIN-LIKE GROWTH FACTOR RECEPTOR TYROSINE KINASE AS A MOLECULAR TARGET IN MANTLE CELL LYMPHOMA. (Masters Thesis). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/21

Chicago Manual of Style (16th Edition):

Vishwamitra, Deeksha. “TYPE I INSULIN-LIKE GROWTH FACTOR RECEPTOR TYROSINE KINASE AS A MOLECULAR TARGET IN MANTLE CELL LYMPHOMA.” 2010. Masters Thesis, Texas Medical Center. Accessed November 18, 2017. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/21.

MLA Handbook (7th Edition):

Vishwamitra, Deeksha. “TYPE I INSULIN-LIKE GROWTH FACTOR RECEPTOR TYROSINE KINASE AS A MOLECULAR TARGET IN MANTLE CELL LYMPHOMA.” 2010. Web. 18 Nov 2017.

Vancouver:

Vishwamitra D. TYPE I INSULIN-LIKE GROWTH FACTOR RECEPTOR TYROSINE KINASE AS A MOLECULAR TARGET IN MANTLE CELL LYMPHOMA. [Internet] [Masters thesis]. Texas Medical Center; 2010. [cited 2017 Nov 18]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/21.

Council of Science Editors:

Vishwamitra D. TYPE I INSULIN-LIKE GROWTH FACTOR RECEPTOR TYROSINE KINASE AS A MOLECULAR TARGET IN MANTLE CELL LYMPHOMA. [Masters Thesis]. Texas Medical Center; 2010. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/21


Texas Medical Center

14. Jen, Emily. IL-2 REGULATION OF IL-24 EXPRESSION LEADS TO GROWTH SUPPRESSION OF MELANOMA CELLS.

Degree: PhD, 2010, Texas Medical Center

 Melanoma is known to be highly resistant to chemotherapy. Treatment with high dose IL-2 has shown significant clinical benefit in a minority of metastatic melanoma… (more)

Subjects/Keywords: IL-24; melanoma; IL-2; predictive marker; growth suppression; Medical Cell Biology; Oncology

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APA (6th Edition):

Jen, E. (2010). IL-2 REGULATION OF IL-24 EXPRESSION LEADS TO GROWTH SUPPRESSION OF MELANOMA CELLS. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/32

Chicago Manual of Style (16th Edition):

Jen, Emily. “IL-2 REGULATION OF IL-24 EXPRESSION LEADS TO GROWTH SUPPRESSION OF MELANOMA CELLS.” 2010. Doctoral Dissertation, Texas Medical Center. Accessed November 18, 2017. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/32.

MLA Handbook (7th Edition):

Jen, Emily. “IL-2 REGULATION OF IL-24 EXPRESSION LEADS TO GROWTH SUPPRESSION OF MELANOMA CELLS.” 2010. Web. 18 Nov 2017.

Vancouver:

Jen E. IL-2 REGULATION OF IL-24 EXPRESSION LEADS TO GROWTH SUPPRESSION OF MELANOMA CELLS. [Internet] [Doctoral dissertation]. Texas Medical Center; 2010. [cited 2017 Nov 18]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/32.

Council of Science Editors:

Jen E. IL-2 REGULATION OF IL-24 EXPRESSION LEADS TO GROWTH SUPPRESSION OF MELANOMA CELLS. [Doctoral Dissertation]. Texas Medical Center; 2010. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/32


Texas Medical Center

15. Sommer, Amy M. Xenobiotic Metabolism Genes and Clubfoot.

Degree: MS, 2010, Texas Medical Center

 Idiopathic or isolated clubfoot is a common orthopedic birth defect that affects approximately 135,000 children worldwide. It is characterized by equinus, varus and adductus deformities… (more)

Subjects/Keywords: clubfoot; xenobiotic metabolism; tobacco smoke; birth defects; talipes; Genetics; Medical Genetics; Molecular Genetics

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APA (6th Edition):

Sommer, A. M. (2010). Xenobiotic Metabolism Genes and Clubfoot. (Masters Thesis). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/35

Chicago Manual of Style (16th Edition):

Sommer, Amy M. “Xenobiotic Metabolism Genes and Clubfoot.” 2010. Masters Thesis, Texas Medical Center. Accessed November 18, 2017. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/35.

MLA Handbook (7th Edition):

Sommer, Amy M. “Xenobiotic Metabolism Genes and Clubfoot.” 2010. Web. 18 Nov 2017.

Vancouver:

Sommer AM. Xenobiotic Metabolism Genes and Clubfoot. [Internet] [Masters thesis]. Texas Medical Center; 2010. [cited 2017 Nov 18]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/35.

Council of Science Editors:

Sommer AM. Xenobiotic Metabolism Genes and Clubfoot. [Masters Thesis]. Texas Medical Center; 2010. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/35


Texas Medical Center

16. Nguyen, Vuvi H. THE EXPRESSION AND CELLULAR LOCALIZATION OF CC-CHEMOKINE RECEPTOR 5 (CCR5) AFTER TRAUMATIC BRAIN INJURY.

Degree: MS, 2010, Texas Medical Center

 Traumatic brain injury results from a primary insult and secondary events that together result in tissue injury. This primary injury occurs at the moment of… (more)

Subjects/Keywords: CCR5; CC-Chemokine Receptor 5; Chemokines; Traumatic Brain Injury; Neuroinflammation; Molecular and Cellular Neuroscience; Neuroscience and Neurobiology

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APA (6th Edition):

Nguyen, V. H. (2010). THE EXPRESSION AND CELLULAR LOCALIZATION OF CC-CHEMOKINE RECEPTOR 5 (CCR5) AFTER TRAUMATIC BRAIN INJURY. (Masters Thesis). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/37

Chicago Manual of Style (16th Edition):

Nguyen, Vuvi H. “THE EXPRESSION AND CELLULAR LOCALIZATION OF CC-CHEMOKINE RECEPTOR 5 (CCR5) AFTER TRAUMATIC BRAIN INJURY.” 2010. Masters Thesis, Texas Medical Center. Accessed November 18, 2017. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/37.

MLA Handbook (7th Edition):

Nguyen, Vuvi H. “THE EXPRESSION AND CELLULAR LOCALIZATION OF CC-CHEMOKINE RECEPTOR 5 (CCR5) AFTER TRAUMATIC BRAIN INJURY.” 2010. Web. 18 Nov 2017.

Vancouver:

Nguyen VH. THE EXPRESSION AND CELLULAR LOCALIZATION OF CC-CHEMOKINE RECEPTOR 5 (CCR5) AFTER TRAUMATIC BRAIN INJURY. [Internet] [Masters thesis]. Texas Medical Center; 2010. [cited 2017 Nov 18]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/37.

Council of Science Editors:

Nguyen VH. THE EXPRESSION AND CELLULAR LOCALIZATION OF CC-CHEMOKINE RECEPTOR 5 (CCR5) AFTER TRAUMATIC BRAIN INJURY. [Masters Thesis]. Texas Medical Center; 2010. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/37


Texas Medical Center

17. Crain, Carrie A. An assessment of obesity and hyperphagia in individuals with Smith-Magenis syndrome.

Degree: MS, 2010, Texas Medical Center

 Smith-Magenis syndrome (SMS;OMIM# 182290) is a multiple congenital anomalies and mental retardation syndrome caused by a 3.7- Mb deletion on chromosome 17p11.2 or a mutation… (more)

Subjects/Keywords: Smith Magenis syndrome; obesity; hyperphagia; BMI; food seeking; behavior; genetic; Congenital, Hereditary, and Neonatal Diseases and Abnormalities; Medical Genetics

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APA (6th Edition):

Crain, C. A. (2010). An assessment of obesity and hyperphagia in individuals with Smith-Magenis syndrome. (Masters Thesis). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/39

Chicago Manual of Style (16th Edition):

Crain, Carrie A. “An assessment of obesity and hyperphagia in individuals with Smith-Magenis syndrome.” 2010. Masters Thesis, Texas Medical Center. Accessed November 18, 2017. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/39.

MLA Handbook (7th Edition):

Crain, Carrie A. “An assessment of obesity and hyperphagia in individuals with Smith-Magenis syndrome.” 2010. Web. 18 Nov 2017.

Vancouver:

Crain CA. An assessment of obesity and hyperphagia in individuals with Smith-Magenis syndrome. [Internet] [Masters thesis]. Texas Medical Center; 2010. [cited 2017 Nov 18]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/39.

Council of Science Editors:

Crain CA. An assessment of obesity and hyperphagia in individuals with Smith-Magenis syndrome. [Masters Thesis]. Texas Medical Center; 2010. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/39


Texas Medical Center

18. Melancon, Adam D. RANGE ADAPTIVE PROTON THERAPY FOR PROSTATE CANCER.

Degree: PhD, 2010, Texas Medical Center

 Purpose: The rapid distal falloff of a proton beam allows for sparing of normal tissues distal to the target. However proton beams that aim directly… (more)

Subjects/Keywords: adaptive radiotherapy; proton therapy; in vivo dosimetry; image-guided radiation therapy; scintillation dosimetry; Other Physics

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APA (6th Edition):

Melancon, A. D. (2010). RANGE ADAPTIVE PROTON THERAPY FOR PROSTATE CANCER. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/44

Chicago Manual of Style (16th Edition):

Melancon, Adam D. “RANGE ADAPTIVE PROTON THERAPY FOR PROSTATE CANCER.” 2010. Doctoral Dissertation, Texas Medical Center. Accessed November 18, 2017. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/44.

MLA Handbook (7th Edition):

Melancon, Adam D. “RANGE ADAPTIVE PROTON THERAPY FOR PROSTATE CANCER.” 2010. Web. 18 Nov 2017.

Vancouver:

Melancon AD. RANGE ADAPTIVE PROTON THERAPY FOR PROSTATE CANCER. [Internet] [Doctoral dissertation]. Texas Medical Center; 2010. [cited 2017 Nov 18]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/44.

Council of Science Editors:

Melancon AD. RANGE ADAPTIVE PROTON THERAPY FOR PROSTATE CANCER. [Doctoral Dissertation]. Texas Medical Center; 2010. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/44


Texas Medical Center

19. Nabilsi, Nancy H. Regulation of survivin gene expression in the human endometrium and endometrial cancer.

Degree: PhD, 2009, Texas Medical Center

 In the United States, endometrial cancer is the leading cancer of the female reproductive tract. There are 40,100 new cases and 7,470 deaths from endometrial… (more)

Subjects/Keywords: Endometrial Cancer; Genes; Molecular biomarkers; Survivin; Medical Genetics; Medicine and Health Sciences; Oncology

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APA (6th Edition):

Nabilsi, N. H. (2009). Regulation of survivin gene expression in the human endometrium and endometrial cancer. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/1

Chicago Manual of Style (16th Edition):

Nabilsi, Nancy H. “Regulation of survivin gene expression in the human endometrium and endometrial cancer.” 2009. Doctoral Dissertation, Texas Medical Center. Accessed November 18, 2017. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/1.

MLA Handbook (7th Edition):

Nabilsi, Nancy H. “Regulation of survivin gene expression in the human endometrium and endometrial cancer.” 2009. Web. 18 Nov 2017.

Vancouver:

Nabilsi NH. Regulation of survivin gene expression in the human endometrium and endometrial cancer. [Internet] [Doctoral dissertation]. Texas Medical Center; 2009. [cited 2017 Nov 18]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/1.

Council of Science Editors:

Nabilsi NH. Regulation of survivin gene expression in the human endometrium and endometrial cancer. [Doctoral Dissertation]. Texas Medical Center; 2009. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/1


Texas Medical Center

20. Ragan, Dustin K. Measurement of the vascular input function in mice for DCE-MRI.

Degree: PhD, 2010, Texas Medical Center

 DCE-MRI is an important technique in the study of small animal cancer models because its sensitivity to vascular changes opens the possibility of quantitative assessment… (more)

Subjects/Keywords: magnetic resonance imaging; DCE-MRI; VIF; AIF; small animal imaging; constrained reconstruction; flow enhancement; reproducibility; Medical Biophysics

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APA (6th Edition):

Ragan, D. K. (2010). Measurement of the vascular input function in mice for DCE-MRI. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/11

Chicago Manual of Style (16th Edition):

Ragan, Dustin K. “Measurement of the vascular input function in mice for DCE-MRI.” 2010. Doctoral Dissertation, Texas Medical Center. Accessed November 18, 2017. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/11.

MLA Handbook (7th Edition):

Ragan, Dustin K. “Measurement of the vascular input function in mice for DCE-MRI.” 2010. Web. 18 Nov 2017.

Vancouver:

Ragan DK. Measurement of the vascular input function in mice for DCE-MRI. [Internet] [Doctoral dissertation]. Texas Medical Center; 2010. [cited 2017 Nov 18]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/11.

Council of Science Editors:

Ragan DK. Measurement of the vascular input function in mice for DCE-MRI. [Doctoral Dissertation]. Texas Medical Center; 2010. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/11


Texas Medical Center

21. Grant, Ryan L. Implementation of an Anthropomorphic Phantom for the Evaluation of Proton Therapy Treatment Procedures.

Degree: MS, 2010, Texas Medical Center

 With an increasing number of institutions offering proton therapy, the number of multi-institutional clinical trials involving proton therapy will also increase in the coming years.… (more)

Subjects/Keywords: proton therapy; dosimetry; phantoms; quality assurance; Medical Sciences; Physics

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APA (6th Edition):

Grant, R. L. (2010). Implementation of an Anthropomorphic Phantom for the Evaluation of Proton Therapy Treatment Procedures. (Masters Thesis). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/8

Chicago Manual of Style (16th Edition):

Grant, Ryan L. “Implementation of an Anthropomorphic Phantom for the Evaluation of Proton Therapy Treatment Procedures.” 2010. Masters Thesis, Texas Medical Center. Accessed November 18, 2017. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/8.

MLA Handbook (7th Edition):

Grant, Ryan L. “Implementation of an Anthropomorphic Phantom for the Evaluation of Proton Therapy Treatment Procedures.” 2010. Web. 18 Nov 2017.

Vancouver:

Grant RL. Implementation of an Anthropomorphic Phantom for the Evaluation of Proton Therapy Treatment Procedures. [Internet] [Masters thesis]. Texas Medical Center; 2010. [cited 2017 Nov 18]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/8.

Council of Science Editors:

Grant RL. Implementation of an Anthropomorphic Phantom for the Evaluation of Proton Therapy Treatment Procedures. [Masters Thesis]. Texas Medical Center; 2010. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/8


Texas Medical Center

22. Stewart, Keri L. Delta like ligand 4 is a critical regulator of bone marrow cell differentiation into pericytes/vascular smooth muscle cells and is essential for the vasculogenesis that supports the growth of Ewing’s sarcoma.

Degree: PhD, 2010, Texas Medical Center

 We have previously shown that vasculogenesis, the process by which bone marrow-derived cells are recruited to the tumor and organized to form a blood vessel… (more)

Subjects/Keywords: Notch; DLL4; vasculogenesis; Ewing's sarcoma; pericytes; vascular smooth muscle cells; Cancer Biology

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APA (6th Edition):

Stewart, K. L. (2010). Delta like ligand 4 is a critical regulator of bone marrow cell differentiation into pericytes/vascular smooth muscle cells and is essential for the vasculogenesis that supports the growth of Ewing’s sarcoma. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/30

Chicago Manual of Style (16th Edition):

Stewart, Keri L. “Delta like ligand 4 is a critical regulator of bone marrow cell differentiation into pericytes/vascular smooth muscle cells and is essential for the vasculogenesis that supports the growth of Ewing’s sarcoma.” 2010. Doctoral Dissertation, Texas Medical Center. Accessed November 18, 2017. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/30.

MLA Handbook (7th Edition):

Stewart, Keri L. “Delta like ligand 4 is a critical regulator of bone marrow cell differentiation into pericytes/vascular smooth muscle cells and is essential for the vasculogenesis that supports the growth of Ewing’s sarcoma.” 2010. Web. 18 Nov 2017.

Vancouver:

Stewart KL. Delta like ligand 4 is a critical regulator of bone marrow cell differentiation into pericytes/vascular smooth muscle cells and is essential for the vasculogenesis that supports the growth of Ewing’s sarcoma. [Internet] [Doctoral dissertation]. Texas Medical Center; 2010. [cited 2017 Nov 18]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/30.

Council of Science Editors:

Stewart KL. Delta like ligand 4 is a critical regulator of bone marrow cell differentiation into pericytes/vascular smooth muscle cells and is essential for the vasculogenesis that supports the growth of Ewing’s sarcoma. [Doctoral Dissertation]. Texas Medical Center; 2010. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/30


Texas Medical Center

23. Babcock, Daniel T. DAMAGE-INDUCED INFLAMMATION AND NOCICEPTIVE HYPERSENSITIVITY IN DROSOPHILA LARVAE.

Degree: PhD, 2010, Texas Medical Center

 Mounting an effective response to tissue damage requires a concerted effort from a number of systems, including both the immune and nervous systems. Immune-responsive blood… (more)

Subjects/Keywords: Drosophila; nociception; inflammation; damage; allodynia; hyperalgesia; ultraviolet; Genetics; Molecular and Cellular Neuroscience; Neuroscience and Neurobiology

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APA (6th Edition):

Babcock, D. T. (2010). DAMAGE-INDUCED INFLAMMATION AND NOCICEPTIVE HYPERSENSITIVITY IN DROSOPHILA LARVAE. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/6

Chicago Manual of Style (16th Edition):

Babcock, Daniel T. “DAMAGE-INDUCED INFLAMMATION AND NOCICEPTIVE HYPERSENSITIVITY IN DROSOPHILA LARVAE.” 2010. Doctoral Dissertation, Texas Medical Center. Accessed November 18, 2017. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/6.

MLA Handbook (7th Edition):

Babcock, Daniel T. “DAMAGE-INDUCED INFLAMMATION AND NOCICEPTIVE HYPERSENSITIVITY IN DROSOPHILA LARVAE.” 2010. Web. 18 Nov 2017.

Vancouver:

Babcock DT. DAMAGE-INDUCED INFLAMMATION AND NOCICEPTIVE HYPERSENSITIVITY IN DROSOPHILA LARVAE. [Internet] [Doctoral dissertation]. Texas Medical Center; 2010. [cited 2017 Nov 18]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/6.

Council of Science Editors:

Babcock DT. DAMAGE-INDUCED INFLAMMATION AND NOCICEPTIVE HYPERSENSITIVITY IN DROSOPHILA LARVAE. [Doctoral Dissertation]. Texas Medical Center; 2010. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/6


Texas Medical Center

24. Nguyen, Vuvi H. THE EXPRESSION AND CELLULAR LOCALIZATION OF CC-CHEMOKINE RECEPTOR 5 (CCR5) AFTER TRAUMATIC BRAIN INJURY.

Degree: MS, 2010, Texas Medical Center

 Traumatic brain injury results from a primary insult and secondary events that together result in tissue injury. This primary injury occurs at the moment of… (more)

Subjects/Keywords: CCR5; chemokine receptor; traumatic brain injury; neuroinflammation; Molecular and Cellular Neuroscience; Neuroscience and Neurobiology

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APA (6th Edition):

Nguyen, V. H. (2010). THE EXPRESSION AND CELLULAR LOCALIZATION OF CC-CHEMOKINE RECEPTOR 5 (CCR5) AFTER TRAUMATIC BRAIN INJURY. (Masters Thesis). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/36

Chicago Manual of Style (16th Edition):

Nguyen, Vuvi H. “THE EXPRESSION AND CELLULAR LOCALIZATION OF CC-CHEMOKINE RECEPTOR 5 (CCR5) AFTER TRAUMATIC BRAIN INJURY.” 2010. Masters Thesis, Texas Medical Center. Accessed November 18, 2017. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/36.

MLA Handbook (7th Edition):

Nguyen, Vuvi H. “THE EXPRESSION AND CELLULAR LOCALIZATION OF CC-CHEMOKINE RECEPTOR 5 (CCR5) AFTER TRAUMATIC BRAIN INJURY.” 2010. Web. 18 Nov 2017.

Vancouver:

Nguyen VH. THE EXPRESSION AND CELLULAR LOCALIZATION OF CC-CHEMOKINE RECEPTOR 5 (CCR5) AFTER TRAUMATIC BRAIN INJURY. [Internet] [Masters thesis]. Texas Medical Center; 2010. [cited 2017 Nov 18]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/36.

Council of Science Editors:

Nguyen VH. THE EXPRESSION AND CELLULAR LOCALIZATION OF CC-CHEMOKINE RECEPTOR 5 (CCR5) AFTER TRAUMATIC BRAIN INJURY. [Masters Thesis]. Texas Medical Center; 2010. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/36


Texas Medical Center

25. gomez, fabiola c. MECHANISM-BASED STRATEGIES TO ENHANCE THE ACTIONS OF A.

Degree: PhD, 2010, Texas Medical Center

 Heat shock protein 90 (HSP90) is an abundant molecular chaperone that regulates the functional stability of client oncoproteins, such as STAT3, Raf-1 and Akt, which… (more)

Subjects/Keywords: 17-allylamino-17demethoxygeldanamycin; 8-chloro-adenosine; actinomycin D; heat shock proteins; heat shock factor; multiple myeloma; transcription inhibitor; Cancer Biology; Therapeutics

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APA (6th Edition):

gomez, f. c. (2010). MECHANISM-BASED STRATEGIES TO ENHANCE THE ACTIONS OF A. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/31

Chicago Manual of Style (16th Edition):

gomez, fabiola c. “MECHANISM-BASED STRATEGIES TO ENHANCE THE ACTIONS OF A.” 2010. Doctoral Dissertation, Texas Medical Center. Accessed November 18, 2017. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/31.

MLA Handbook (7th Edition):

gomez, fabiola c. “MECHANISM-BASED STRATEGIES TO ENHANCE THE ACTIONS OF A.” 2010. Web. 18 Nov 2017.

Vancouver:

gomez fc. MECHANISM-BASED STRATEGIES TO ENHANCE THE ACTIONS OF A. [Internet] [Doctoral dissertation]. Texas Medical Center; 2010. [cited 2017 Nov 18]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/31.

Council of Science Editors:

gomez fc. MECHANISM-BASED STRATEGIES TO ENHANCE THE ACTIONS OF A. [Doctoral Dissertation]. Texas Medical Center; 2010. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/31


Texas Medical Center

26. Gonzalez-McGehee, Jennifer. AMPA RECEPTOR ALLOSTERISM: MEASUREMENT OF THE CONFORMATIONAL CHANGES IN THE LIGAND BINDING DOMAIN OF A FUNCTIONAL RECEPTOR.

Degree: PhD, 2010, Texas Medical Center

 Ionotropic glutamate receptors are important excitatory neurotransmitter receptors in the mammalian central nervous system that have been implicated in a number of neuropathologies such as… (more)

Subjects/Keywords: AMPA receptor; Luminescence Resonance Energy Transfer; structure-function; ion channel; Biochemistry, Biophysics, and Structural Biology

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APA (6th Edition):

Gonzalez-McGehee, J. (2010). AMPA RECEPTOR ALLOSTERISM: MEASUREMENT OF THE CONFORMATIONAL CHANGES IN THE LIGAND BINDING DOMAIN OF A FUNCTIONAL RECEPTOR. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/33

Chicago Manual of Style (16th Edition):

Gonzalez-McGehee, Jennifer. “AMPA RECEPTOR ALLOSTERISM: MEASUREMENT OF THE CONFORMATIONAL CHANGES IN THE LIGAND BINDING DOMAIN OF A FUNCTIONAL RECEPTOR.” 2010. Doctoral Dissertation, Texas Medical Center. Accessed November 18, 2017. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/33.

MLA Handbook (7th Edition):

Gonzalez-McGehee, Jennifer. “AMPA RECEPTOR ALLOSTERISM: MEASUREMENT OF THE CONFORMATIONAL CHANGES IN THE LIGAND BINDING DOMAIN OF A FUNCTIONAL RECEPTOR.” 2010. Web. 18 Nov 2017.

Vancouver:

Gonzalez-McGehee J. AMPA RECEPTOR ALLOSTERISM: MEASUREMENT OF THE CONFORMATIONAL CHANGES IN THE LIGAND BINDING DOMAIN OF A FUNCTIONAL RECEPTOR. [Internet] [Doctoral dissertation]. Texas Medical Center; 2010. [cited 2017 Nov 18]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/33.

Council of Science Editors:

Gonzalez-McGehee J. AMPA RECEPTOR ALLOSTERISM: MEASUREMENT OF THE CONFORMATIONAL CHANGES IN THE LIGAND BINDING DOMAIN OF A FUNCTIONAL RECEPTOR. [Doctoral Dissertation]. Texas Medical Center; 2010. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/33


Texas Medical Center

27. Greathouse, Kristen L. XENOESTROGEN-SPECIFIC MECHANISMS OF DEVELOPMENTAL REPROGRAMMING CORRELATE WITH GENE EXPRESSION AND TUMOR DEVELOPMENT.

Degree: PhD, 2010, Texas Medical Center

 Environmental exposures during sensitive windows of development can reprogram normal physiological responses and alter disease susceptibility later in life in a process known as developmental… (more)

Subjects/Keywords: Xenoestrogens; developmental reprogramming; uterine leiomyoma; epigenetics; non-genomic signaling; estrogen receptor; Cancer Biology; Developmental Biology; Molecular Biology; Other Pharmacology, Toxicology and Environmental Health

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APA (6th Edition):

Greathouse, K. L. (2010). XENOESTROGEN-SPECIFIC MECHANISMS OF DEVELOPMENTAL REPROGRAMMING CORRELATE WITH GENE EXPRESSION AND TUMOR DEVELOPMENT. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/51

Chicago Manual of Style (16th Edition):

Greathouse, Kristen L. “XENOESTROGEN-SPECIFIC MECHANISMS OF DEVELOPMENTAL REPROGRAMMING CORRELATE WITH GENE EXPRESSION AND TUMOR DEVELOPMENT.” 2010. Doctoral Dissertation, Texas Medical Center. Accessed November 18, 2017. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/51.

MLA Handbook (7th Edition):

Greathouse, Kristen L. “XENOESTROGEN-SPECIFIC MECHANISMS OF DEVELOPMENTAL REPROGRAMMING CORRELATE WITH GENE EXPRESSION AND TUMOR DEVELOPMENT.” 2010. Web. 18 Nov 2017.

Vancouver:

Greathouse KL. XENOESTROGEN-SPECIFIC MECHANISMS OF DEVELOPMENTAL REPROGRAMMING CORRELATE WITH GENE EXPRESSION AND TUMOR DEVELOPMENT. [Internet] [Doctoral dissertation]. Texas Medical Center; 2010. [cited 2017 Nov 18]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/51.

Council of Science Editors:

Greathouse KL. XENOESTROGEN-SPECIFIC MECHANISMS OF DEVELOPMENTAL REPROGRAMMING CORRELATE WITH GENE EXPRESSION AND TUMOR DEVELOPMENT. [Doctoral Dissertation]. Texas Medical Center; 2010. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/51


Texas Medical Center

28. Kapuria, Vaibhav. Inhibition of Deubiquitinase Activity and Ubiquitination of Jak2 Blocks Cytokine Signaling and Induces Tumor Cell Apoptosis.

Degree: PhD, 2010, Texas Medical Center

 The Jak-stat pathway is critical for cellular proliferation and is commonly found to be deregulated in many solid tumors as well as hematological malignancies. Such… (more)

Subjects/Keywords: Ubiquitination; Deubiquitination; Small molecule inhibitor; Jak-Stat pathway; Deubiquitinating Enzymes; Cancer Biology; Cell Biology

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APA (6th Edition):

Kapuria, V. (2010). Inhibition of Deubiquitinase Activity and Ubiquitination of Jak2 Blocks Cytokine Signaling and Induces Tumor Cell Apoptosis. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/42

Chicago Manual of Style (16th Edition):

Kapuria, Vaibhav. “Inhibition of Deubiquitinase Activity and Ubiquitination of Jak2 Blocks Cytokine Signaling and Induces Tumor Cell Apoptosis.” 2010. Doctoral Dissertation, Texas Medical Center. Accessed November 18, 2017. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/42.

MLA Handbook (7th Edition):

Kapuria, Vaibhav. “Inhibition of Deubiquitinase Activity and Ubiquitination of Jak2 Blocks Cytokine Signaling and Induces Tumor Cell Apoptosis.” 2010. Web. 18 Nov 2017.

Vancouver:

Kapuria V. Inhibition of Deubiquitinase Activity and Ubiquitination of Jak2 Blocks Cytokine Signaling and Induces Tumor Cell Apoptosis. [Internet] [Doctoral dissertation]. Texas Medical Center; 2010. [cited 2017 Nov 18]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/42.

Council of Science Editors:

Kapuria V. Inhibition of Deubiquitinase Activity and Ubiquitination of Jak2 Blocks Cytokine Signaling and Induces Tumor Cell Apoptosis. [Doctoral Dissertation]. Texas Medical Center; 2010. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/42


Texas Medical Center

29. Broadway, Melissa M. Molecular basis of Corynebacterium diphtheriae virulence and infection in the Caenorhabditis elegans model host.

Degree: MS, 2010, Texas Medical Center

 Corynebacterium diphtheriae is the causative agent of cutaneous and pharyngeal diphtheria in humans. While lethality is certainly caused by diphtheria toxin, corynebacterial colonization may primarily… (more)

Subjects/Keywords: C. diphtheriae; pili; bacterial pathogenesis; C. elegans; Microbiology; Pathogenic Microbiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Broadway, M. M. (2010). Molecular basis of Corynebacterium diphtheriae virulence and infection in the Caenorhabditis elegans model host. (Masters Thesis). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/41

Chicago Manual of Style (16th Edition):

Broadway, Melissa M. “Molecular basis of Corynebacterium diphtheriae virulence and infection in the Caenorhabditis elegans model host.” 2010. Masters Thesis, Texas Medical Center. Accessed November 18, 2017. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/41.

MLA Handbook (7th Edition):

Broadway, Melissa M. “Molecular basis of Corynebacterium diphtheriae virulence and infection in the Caenorhabditis elegans model host.” 2010. Web. 18 Nov 2017.

Vancouver:

Broadway MM. Molecular basis of Corynebacterium diphtheriae virulence and infection in the Caenorhabditis elegans model host. [Internet] [Masters thesis]. Texas Medical Center; 2010. [cited 2017 Nov 18]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/41.

Council of Science Editors:

Broadway MM. Molecular basis of Corynebacterium diphtheriae virulence and infection in the Caenorhabditis elegans model host. [Masters Thesis]. Texas Medical Center; 2010. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/41


Texas Medical Center

30. Darnes, Deanna. First Trimester Screening and its Impact on Uptake of Diagnostic Testing.

Degree: MS, 2010, Texas Medical Center

 Introduction: First Trimester Screening (FTS) combines maternal age with fetal nuchal translucency (NT) and maternal analytes to identify pregnancies at an increased risk for Down… (more)

Subjects/Keywords: first trimester screening; diagnostic testing; impact; Obstetrics and Gynecology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Darnes, D. (2010). First Trimester Screening and its Impact on Uptake of Diagnostic Testing. (Masters Thesis). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/52

Chicago Manual of Style (16th Edition):

Darnes, Deanna. “First Trimester Screening and its Impact on Uptake of Diagnostic Testing.” 2010. Masters Thesis, Texas Medical Center. Accessed November 18, 2017. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/52.

MLA Handbook (7th Edition):

Darnes, Deanna. “First Trimester Screening and its Impact on Uptake of Diagnostic Testing.” 2010. Web. 18 Nov 2017.

Vancouver:

Darnes D. First Trimester Screening and its Impact on Uptake of Diagnostic Testing. [Internet] [Masters thesis]. Texas Medical Center; 2010. [cited 2017 Nov 18]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/52.

Council of Science Editors:

Darnes D. First Trimester Screening and its Impact on Uptake of Diagnostic Testing. [Masters Thesis]. Texas Medical Center; 2010. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/52

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