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You searched for +publisher:"Vanderbilt University" +contributor:("Martin J. Gallagher"). Showing records 1 – 4 of 4 total matches.

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Vanderbilt University

1. Deel, Megan Elizabeth. Alterations in GABAA receptor expression and physiology in a mouse model of idiopathic generalized epilepsy.

Degree: MS, Neuroscience, 2012, Vanderbilt University

 The proper function of the nervous system is dependent upon a delicate balance between excitatory and inhibitory activity in the brain. GABAA receptors are extremely… (more)

Subjects/Keywords: childhood absence epilepsy; seizure; epilepsy; GABA; compensation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Deel, M. E. (2012). Alterations in GABAA receptor expression and physiology in a mouse model of idiopathic generalized epilepsy. (Thesis). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14874

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Deel, Megan Elizabeth. “Alterations in GABAA receptor expression and physiology in a mouse model of idiopathic generalized epilepsy.” 2012. Thesis, Vanderbilt University. Accessed January 16, 2021. http://hdl.handle.net/1803/14874.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Deel, Megan Elizabeth. “Alterations in GABAA receptor expression and physiology in a mouse model of idiopathic generalized epilepsy.” 2012. Web. 16 Jan 2021.

Vancouver:

Deel ME. Alterations in GABAA receptor expression and physiology in a mouse model of idiopathic generalized epilepsy. [Internet] [Thesis]. Vanderbilt University; 2012. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/1803/14874.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Deel ME. Alterations in GABAA receptor expression and physiology in a mouse model of idiopathic generalized epilepsy. [Thesis]. Vanderbilt University; 2012. Available from: http://hdl.handle.net/1803/14874

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

2. Shen, Dingding. Characterization of GABAA receptor subunit mutations associated with epileptic encephalopathies.

Degree: PhD, Neuroscience, 2017, Vanderbilt University

 Epileptic encephalopathies (EEs) are a devastating group of severe childhood onset epilepsies with medication resistant seizures and poor developmental outcomes. Many EEs have a genetic… (more)

Subjects/Keywords: Epileptic encephalopathies; GABAA receptor

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Shen, D. (2017). Characterization of GABAA receptor subunit mutations associated with epileptic encephalopathies. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14271

Chicago Manual of Style (16th Edition):

Shen, Dingding. “Characterization of GABAA receptor subunit mutations associated with epileptic encephalopathies.” 2017. Doctoral Dissertation, Vanderbilt University. Accessed January 16, 2021. http://hdl.handle.net/1803/14271.

MLA Handbook (7th Edition):

Shen, Dingding. “Characterization of GABAA receptor subunit mutations associated with epileptic encephalopathies.” 2017. Web. 16 Jan 2021.

Vancouver:

Shen D. Characterization of GABAA receptor subunit mutations associated with epileptic encephalopathies. [Internet] [Doctoral dissertation]. Vanderbilt University; 2017. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/1803/14271.

Council of Science Editors:

Shen D. Characterization of GABAA receptor subunit mutations associated with epileptic encephalopathies. [Doctoral Dissertation]. Vanderbilt University; 2017. Available from: http://hdl.handle.net/1803/14271


Vanderbilt University

3. Arain, Fazal Manzoor. Two Human Epilepsy Mutations Cause Developmentally Dependant Changes in Seizure Phenotype and GABAA Receptor Expression in Genetically Modified Mice.

Degree: PhD, Neuroscience, 2014, Vanderbilt University

 Epilepsy is a disease characterized by two or more unprovoked seizures. Two mutations, S326fs328X and A322D, in the α1 subunit of GABAA receptor (Gabra1), are… (more)

Subjects/Keywords: GABA; Epilepsy; Development

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Arain, F. M. (2014). Two Human Epilepsy Mutations Cause Developmentally Dependant Changes in Seizure Phenotype and GABAA Receptor Expression in Genetically Modified Mice. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10463

Chicago Manual of Style (16th Edition):

Arain, Fazal Manzoor. “Two Human Epilepsy Mutations Cause Developmentally Dependant Changes in Seizure Phenotype and GABAA Receptor Expression in Genetically Modified Mice.” 2014. Doctoral Dissertation, Vanderbilt University. Accessed January 16, 2021. http://hdl.handle.net/1803/10463.

MLA Handbook (7th Edition):

Arain, Fazal Manzoor. “Two Human Epilepsy Mutations Cause Developmentally Dependant Changes in Seizure Phenotype and GABAA Receptor Expression in Genetically Modified Mice.” 2014. Web. 16 Jan 2021.

Vancouver:

Arain FM. Two Human Epilepsy Mutations Cause Developmentally Dependant Changes in Seizure Phenotype and GABAA Receptor Expression in Genetically Modified Mice. [Internet] [Doctoral dissertation]. Vanderbilt University; 2014. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/1803/10463.

Council of Science Editors:

Arain FM. Two Human Epilepsy Mutations Cause Developmentally Dependant Changes in Seizure Phenotype and GABAA Receptor Expression in Genetically Modified Mice. [Doctoral Dissertation]. Vanderbilt University; 2014. Available from: http://hdl.handle.net/1803/10463


Vanderbilt University

4. Gamble-George, Joyonna Carrie. Endocannabinoid Augmentation Through Substrate-Selective COX-2 Inhibition: Behavioral and Synaptic Effects In An Animal Model of Stress-Induced Anxiety.

Degree: PhD, Neuroscience, 2016, Vanderbilt University

 Cannabinoid receptors have been examined as potential targets to alleviate the negative consequences of anxiety, trauma-related, and stress-related disorders. However, in preclinical animal studies, synthetic… (more)

Subjects/Keywords: dopamine; depression; anandamide; stress; cannabinoid receptor; anxiety; PTSD; COX-2; endocannabinoid; SK channel

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gamble-George, J. C. (2016). Endocannabinoid Augmentation Through Substrate-Selective COX-2 Inhibition: Behavioral and Synaptic Effects In An Animal Model of Stress-Induced Anxiety. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/13270

Chicago Manual of Style (16th Edition):

Gamble-George, Joyonna Carrie. “Endocannabinoid Augmentation Through Substrate-Selective COX-2 Inhibition: Behavioral and Synaptic Effects In An Animal Model of Stress-Induced Anxiety.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed January 16, 2021. http://hdl.handle.net/1803/13270.

MLA Handbook (7th Edition):

Gamble-George, Joyonna Carrie. “Endocannabinoid Augmentation Through Substrate-Selective COX-2 Inhibition: Behavioral and Synaptic Effects In An Animal Model of Stress-Induced Anxiety.” 2016. Web. 16 Jan 2021.

Vancouver:

Gamble-George JC. Endocannabinoid Augmentation Through Substrate-Selective COX-2 Inhibition: Behavioral and Synaptic Effects In An Animal Model of Stress-Induced Anxiety. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/1803/13270.

Council of Science Editors:

Gamble-George JC. Endocannabinoid Augmentation Through Substrate-Selective COX-2 Inhibition: Behavioral and Synaptic Effects In An Animal Model of Stress-Induced Anxiety. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://hdl.handle.net/1803/13270

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