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You searched for +publisher:"Vanderbilt University" +contributor:("Jonathan L Haines"). Showing records 1 – 18 of 18 total matches.

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Vanderbilt University

1. Cummings, Anna Christine. Power and type 1 error for large pedigree analyses of binary traits.

Degree: MS, Interdisciplinary Studies: Applied Statistics, 2012, Vanderbilt University

 Studying population isolates with large, complex pedigrees has many advantages for discovering genetic susceptibility loci; however, statistical analyses can be computationally challenging. Allelic association tests… (more)

Subjects/Keywords: association; linkage; simulations; power; type 1 error; pedigrees

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APA (6th Edition):

Cummings, A. C. (2012). Power and type 1 error for large pedigree analyses of binary traits. (Thesis). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14871

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cummings, Anna Christine. “Power and type 1 error for large pedigree analyses of binary traits.” 2012. Thesis, Vanderbilt University. Accessed April 13, 2021. http://hdl.handle.net/1803/14871.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cummings, Anna Christine. “Power and type 1 error for large pedigree analyses of binary traits.” 2012. Web. 13 Apr 2021.

Vancouver:

Cummings AC. Power and type 1 error for large pedigree analyses of binary traits. [Internet] [Thesis]. Vanderbilt University; 2012. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1803/14871.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cummings AC. Power and type 1 error for large pedigree analyses of binary traits. [Thesis]. Vanderbilt University; 2012. Available from: http://hdl.handle.net/1803/14871

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

2. Davis, Mary Feller. Parkinson Disease Loci in the Mid-Western Amish.

Degree: MS, Interdisciplinary Studies: Applied Statistics, 2013, Vanderbilt University

 Previous evidence has shown that Parkinson disease (PD) has a heritable component, but only a small proportion of the total genetic contribution to PD has… (more)

Subjects/Keywords: parkinson disease; Amish; genetics; linkage; association

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APA (6th Edition):

Davis, M. F. (2013). Parkinson Disease Loci in the Mid-Western Amish. (Thesis). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11436

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Davis, Mary Feller. “Parkinson Disease Loci in the Mid-Western Amish.” 2013. Thesis, Vanderbilt University. Accessed April 13, 2021. http://hdl.handle.net/1803/11436.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Davis, Mary Feller. “Parkinson Disease Loci in the Mid-Western Amish.” 2013. Web. 13 Apr 2021.

Vancouver:

Davis MF. Parkinson Disease Loci in the Mid-Western Amish. [Internet] [Thesis]. Vanderbilt University; 2013. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1803/11436.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Davis MF. Parkinson Disease Loci in the Mid-Western Amish. [Thesis]. Vanderbilt University; 2013. Available from: http://hdl.handle.net/1803/11436

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

3. D'Aoust, Laura Nicole. Examination of Candidate Exonic Variants that Confer Susceptibility to Alzheimer Disease in the Amish.

Degree: PhD, Human Genetics, 2015, Vanderbilt University

 Alzheimer disease (AD) is the most common cause of dementia. As with many complex diseases, the identified variants do not explain the total expected genetic… (more)

Subjects/Keywords: Alzheimer; family-based study; genetics

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APA (6th Edition):

D'Aoust, L. N. (2015). Examination of Candidate Exonic Variants that Confer Susceptibility to Alzheimer Disease in the Amish. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10861

Chicago Manual of Style (16th Edition):

D'Aoust, Laura Nicole. “Examination of Candidate Exonic Variants that Confer Susceptibility to Alzheimer Disease in the Amish.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed April 13, 2021. http://hdl.handle.net/1803/10861.

MLA Handbook (7th Edition):

D'Aoust, Laura Nicole. “Examination of Candidate Exonic Variants that Confer Susceptibility to Alzheimer Disease in the Amish.” 2015. Web. 13 Apr 2021.

Vancouver:

D'Aoust LN. Examination of Candidate Exonic Variants that Confer Susceptibility to Alzheimer Disease in the Amish. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1803/10861.

Council of Science Editors:

D'Aoust LN. Examination of Candidate Exonic Variants that Confer Susceptibility to Alzheimer Disease in the Amish. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://hdl.handle.net/1803/10861


Vanderbilt University

4. Veatch, Olivia Jean. Identifying biological pathways implicated in defined subgroups of phenotypic expression for Autism Spectrum Disorders and evaluating small molecule effects on expression of ASMT.

Degree: PhD, Human Genetics, 2013, Vanderbilt University

 Autism Spectrum Disorder is a neurodevelopmental condition with evidence for genetic susceptibility. However, effect sizes for implicated loci are small, and current evidence does not… (more)

Subjects/Keywords: Autism Spectrum Disorder; Human Genetics; Multivariate Statistics; Pharmacogenetics; Phenotyping; Ne

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APA (6th Edition):

Veatch, O. J. (2013). Identifying biological pathways implicated in defined subgroups of phenotypic expression for Autism Spectrum Disorders and evaluating small molecule effects on expression of ASMT. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/15041

Chicago Manual of Style (16th Edition):

Veatch, Olivia Jean. “Identifying biological pathways implicated in defined subgroups of phenotypic expression for Autism Spectrum Disorders and evaluating small molecule effects on expression of ASMT.” 2013. Doctoral Dissertation, Vanderbilt University. Accessed April 13, 2021. http://hdl.handle.net/1803/15041.

MLA Handbook (7th Edition):

Veatch, Olivia Jean. “Identifying biological pathways implicated in defined subgroups of phenotypic expression for Autism Spectrum Disorders and evaluating small molecule effects on expression of ASMT.” 2013. Web. 13 Apr 2021.

Vancouver:

Veatch OJ. Identifying biological pathways implicated in defined subgroups of phenotypic expression for Autism Spectrum Disorders and evaluating small molecule effects on expression of ASMT. [Internet] [Doctoral dissertation]. Vanderbilt University; 2013. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1803/15041.

Council of Science Editors:

Veatch OJ. Identifying biological pathways implicated in defined subgroups of phenotypic expression for Autism Spectrum Disorders and evaluating small molecule effects on expression of ASMT. [Doctoral Dissertation]. Vanderbilt University; 2013. Available from: http://hdl.handle.net/1803/15041


Vanderbilt University

5. Sobota, Rafal Sebastian. Genetics of Tuberculosis Resistance.

Degree: PhD, Human Genetics, 2015, Vanderbilt University

 One third of the world’s population has been infected with Mycobacterium tuberculosis (MTB). Most of those exposed develop an asymptomatic latent infection. In the absence… (more)

Subjects/Keywords: il9; tuberculosis; il12b; gwas

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APA (6th Edition):

Sobota, R. S. (2015). Genetics of Tuberculosis Resistance. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12103

Chicago Manual of Style (16th Edition):

Sobota, Rafal Sebastian. “Genetics of Tuberculosis Resistance.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed April 13, 2021. http://hdl.handle.net/1803/12103.

MLA Handbook (7th Edition):

Sobota, Rafal Sebastian. “Genetics of Tuberculosis Resistance.” 2015. Web. 13 Apr 2021.

Vancouver:

Sobota RS. Genetics of Tuberculosis Resistance. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1803/12103.

Council of Science Editors:

Sobota RS. Genetics of Tuberculosis Resistance. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://hdl.handle.net/1803/12103


Vanderbilt University

6. Hall, Jacob B. The genetics of age-related macular degeneration: exploring pathway and epistatic effects.

Degree: PhD, Human Genetics, 2016, Vanderbilt University

 Age-related macular degeneration (AMD) is a neurodegenerative disease that leads to a loss of central vision and is the leading cause of blindness in the… (more)

Subjects/Keywords: mixed linear models; macular degeneration; heritability; genetics; interactions; epistasis

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APA (6th Edition):

Hall, J. B. (2016). The genetics of age-related macular degeneration: exploring pathway and epistatic effects. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11621

Chicago Manual of Style (16th Edition):

Hall, Jacob B. “The genetics of age-related macular degeneration: exploring pathway and epistatic effects.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed April 13, 2021. http://hdl.handle.net/1803/11621.

MLA Handbook (7th Edition):

Hall, Jacob B. “The genetics of age-related macular degeneration: exploring pathway and epistatic effects.” 2016. Web. 13 Apr 2021.

Vancouver:

Hall JB. The genetics of age-related macular degeneration: exploring pathway and epistatic effects. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1803/11621.

Council of Science Editors:

Hall JB. The genetics of age-related macular degeneration: exploring pathway and epistatic effects. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://hdl.handle.net/1803/11621


Vanderbilt University

7. Cummings, Anna Christine. Exploring the genetic architecture of late-onset Alzheimer disease in an Amish population.

Degree: PhD, Human Genetics, 2012, Vanderbilt University

 Late-onset Alzheimer disease (LOAD) is a complex neurodegenerative disorder with a strong genetic component. APOE is a well-established risk gene for LOAD, and several other… (more)

Subjects/Keywords: Amish; Alzheimer disease; genetics

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APA (6th Edition):

Cummings, A. C. (2012). Exploring the genetic architecture of late-onset Alzheimer disease in an Amish population. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14870

Chicago Manual of Style (16th Edition):

Cummings, Anna Christine. “Exploring the genetic architecture of late-onset Alzheimer disease in an Amish population.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed April 13, 2021. http://hdl.handle.net/1803/14870.

MLA Handbook (7th Edition):

Cummings, Anna Christine. “Exploring the genetic architecture of late-onset Alzheimer disease in an Amish population.” 2012. Web. 13 Apr 2021.

Vancouver:

Cummings AC. Exploring the genetic architecture of late-onset Alzheimer disease in an Amish population. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1803/14870.

Council of Science Editors:

Cummings AC. Exploring the genetic architecture of late-onset Alzheimer disease in an Amish population. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://hdl.handle.net/1803/14870


Vanderbilt University

8. Hoffman, Joshua David. Modeling Macular Degeneration Using Quantitative Phenotypes.

Degree: PhD, Human Genetics, 2015, Vanderbilt University

 Age-related macular degeneration (AMD) is one of the most common causes of visual impairment in the United States (US). Although a multitude of studies have… (more)

Subjects/Keywords: AMD; genetics; Age-Related Macular Degeneration; genomics; assocation analysis; linkage analysis

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APA (6th Edition):

Hoffman, J. D. (2015). Modeling Macular Degeneration Using Quantitative Phenotypes. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10680

Chicago Manual of Style (16th Edition):

Hoffman, Joshua David. “Modeling Macular Degeneration Using Quantitative Phenotypes.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed April 13, 2021. http://hdl.handle.net/1803/10680.

MLA Handbook (7th Edition):

Hoffman, Joshua David. “Modeling Macular Degeneration Using Quantitative Phenotypes.” 2015. Web. 13 Apr 2021.

Vancouver:

Hoffman JD. Modeling Macular Degeneration Using Quantitative Phenotypes. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1803/10680.

Council of Science Editors:

Hoffman JD. Modeling Macular Degeneration Using Quantitative Phenotypes. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://hdl.handle.net/1803/10680


Vanderbilt University

9. Restrepo, Nicole Ann. Investigation of the genetic epidemiology of age-related macular degeneration, primary open-angle glaucoma, and diabetic retinopathy in diverse populations.

Degree: PhD, Human Genetics, 2015, Vanderbilt University

 Common age, related eye diseases are a major driving force behind vision disability and blindness. The three most common diseases afflicting Americans today are age-related… (more)

Subjects/Keywords: African American; diabetic retinopathy; primary open angle glaucoma; age-related macular degeneration; Mexican American

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APA (6th Edition):

Restrepo, N. A. (2015). Investigation of the genetic epidemiology of age-related macular degeneration, primary open-angle glaucoma, and diabetic retinopathy in diverse populations. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10956

Chicago Manual of Style (16th Edition):

Restrepo, Nicole Ann. “Investigation of the genetic epidemiology of age-related macular degeneration, primary open-angle glaucoma, and diabetic retinopathy in diverse populations.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed April 13, 2021. http://hdl.handle.net/1803/10956.

MLA Handbook (7th Edition):

Restrepo, Nicole Ann. “Investigation of the genetic epidemiology of age-related macular degeneration, primary open-angle glaucoma, and diabetic retinopathy in diverse populations.” 2015. Web. 13 Apr 2021.

Vancouver:

Restrepo NA. Investigation of the genetic epidemiology of age-related macular degeneration, primary open-angle glaucoma, and diabetic retinopathy in diverse populations. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1803/10956.

Council of Science Editors:

Restrepo NA. Investigation of the genetic epidemiology of age-related macular degeneration, primary open-angle glaucoma, and diabetic retinopathy in diverse populations. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://hdl.handle.net/1803/10956


Vanderbilt University

10. Davis, Mary Feller. Determining the Use of Electronic Medical Records in Genetic Studies of Multiple Sclerosis.

Degree: PhD, Human Genetics, 2013, Vanderbilt University

 The clinical course of multiple sclerosis (MS) is highly variable, and research data collection is costly and time-consuming. Much is known about the genetic risk… (more)

Subjects/Keywords: genetic association; natural language processing

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APA (6th Edition):

Davis, M. F. (2013). Determining the Use of Electronic Medical Records in Genetic Studies of Multiple Sclerosis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14936

Chicago Manual of Style (16th Edition):

Davis, Mary Feller. “Determining the Use of Electronic Medical Records in Genetic Studies of Multiple Sclerosis.” 2013. Doctoral Dissertation, Vanderbilt University. Accessed April 13, 2021. http://hdl.handle.net/1803/14936.

MLA Handbook (7th Edition):

Davis, Mary Feller. “Determining the Use of Electronic Medical Records in Genetic Studies of Multiple Sclerosis.” 2013. Web. 13 Apr 2021.

Vancouver:

Davis MF. Determining the Use of Electronic Medical Records in Genetic Studies of Multiple Sclerosis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2013. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1803/14936.

Council of Science Editors:

Davis MF. Determining the Use of Electronic Medical Records in Genetic Studies of Multiple Sclerosis. [Doctoral Dissertation]. Vanderbilt University; 2013. Available from: http://hdl.handle.net/1803/14936


Vanderbilt University

11. Thornton-Wells, Tricia Ann. Confronting Complexity: A Comprehensive Statistical and Computational Strategy for Identifying the Missing Link between Genotype and Phenotype.

Degree: PhD, Neuroscience, 2006, Vanderbilt University

 Common diseases with a genetic basis are likely to have a very complex etiology, in which the mapping between genotype and phenotype is far from… (more)

Subjects/Keywords: genetics; statistical genetics; gene-gene interactions; heterogeneity; Alzheimer disease; Epistasis; cluster analysis; computational analysis; Genetics  – Statistical methods; Phenotype; Alzheimer's disease  – Genetic aspects

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APA (6th Edition):

Thornton-Wells, T. A. (2006). Confronting Complexity: A Comprehensive Statistical and Computational Strategy for Identifying the Missing Link between Genotype and Phenotype. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14241

Chicago Manual of Style (16th Edition):

Thornton-Wells, Tricia Ann. “Confronting Complexity: A Comprehensive Statistical and Computational Strategy for Identifying the Missing Link between Genotype and Phenotype.” 2006. Doctoral Dissertation, Vanderbilt University. Accessed April 13, 2021. http://hdl.handle.net/1803/14241.

MLA Handbook (7th Edition):

Thornton-Wells, Tricia Ann. “Confronting Complexity: A Comprehensive Statistical and Computational Strategy for Identifying the Missing Link between Genotype and Phenotype.” 2006. Web. 13 Apr 2021.

Vancouver:

Thornton-Wells TA. Confronting Complexity: A Comprehensive Statistical and Computational Strategy for Identifying the Missing Link between Genotype and Phenotype. [Internet] [Doctoral dissertation]. Vanderbilt University; 2006. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1803/14241.

Council of Science Editors:

Thornton-Wells TA. Confronting Complexity: A Comprehensive Statistical and Computational Strategy for Identifying the Missing Link between Genotype and Phenotype. [Doctoral Dissertation]. Vanderbilt University; 2006. Available from: http://hdl.handle.net/1803/14241


Vanderbilt University

12. Bush, William Scott. A Knowledge-Driven Multi-Locus Analysis of Multiple Sclerosis Susceptiblity.

Degree: PhD, Human Genetics, 2009, Vanderbilt University

 Evaluating epistasis in whole-genome association studies is an important challenge in human genetics, as many common diseases are thought to have complex underlying genetic architectures… (more)

Subjects/Keywords: Genomics  – Methodology; Inositol  – Pathophysiology; Epistasis (Genetics); Multiple sclerosis  – Genetic aspects; disease gene discovery; knowledge-based analysis; Disease susceptibility  – Genetic aspects  – Data processing

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APA (6th Edition):

Bush, W. S. (2009). A Knowledge-Driven Multi-Locus Analysis of Multiple Sclerosis Susceptiblity. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10678

Chicago Manual of Style (16th Edition):

Bush, William Scott. “A Knowledge-Driven Multi-Locus Analysis of Multiple Sclerosis Susceptiblity.” 2009. Doctoral Dissertation, Vanderbilt University. Accessed April 13, 2021. http://hdl.handle.net/1803/10678.

MLA Handbook (7th Edition):

Bush, William Scott. “A Knowledge-Driven Multi-Locus Analysis of Multiple Sclerosis Susceptiblity.” 2009. Web. 13 Apr 2021.

Vancouver:

Bush WS. A Knowledge-Driven Multi-Locus Analysis of Multiple Sclerosis Susceptiblity. [Internet] [Doctoral dissertation]. Vanderbilt University; 2009. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1803/10678.

Council of Science Editors:

Bush WS. A Knowledge-Driven Multi-Locus Analysis of Multiple Sclerosis Susceptiblity. [Doctoral Dissertation]. Vanderbilt University; 2009. Available from: http://hdl.handle.net/1803/10678


Vanderbilt University

13. Thornton-Wells, Tricia Ann. Comparison of Three Clustering Methods for Dissecting Trait Heterogeneity in Genotypic Data.

Degree: MS, Biomedical Informatics, 2005, Vanderbilt University

 Trait heterogeneity, which exists when a trait has been defined with insufficient specificity such that it is actually two or more distinct traits, has been… (more)

Subjects/Keywords: unsupervised learning; simulation study; method comparison; clustering; complex disease; genetics; trait heterogenetiy; locus heterogeneity

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APA (6th Edition):

Thornton-Wells, T. A. (2005). Comparison of Three Clustering Methods for Dissecting Trait Heterogeneity in Genotypic Data. (Thesis). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/13157

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Thornton-Wells, Tricia Ann. “Comparison of Three Clustering Methods for Dissecting Trait Heterogeneity in Genotypic Data.” 2005. Thesis, Vanderbilt University. Accessed April 13, 2021. http://hdl.handle.net/1803/13157.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Thornton-Wells, Tricia Ann. “Comparison of Three Clustering Methods for Dissecting Trait Heterogeneity in Genotypic Data.” 2005. Web. 13 Apr 2021.

Vancouver:

Thornton-Wells TA. Comparison of Three Clustering Methods for Dissecting Trait Heterogeneity in Genotypic Data. [Internet] [Thesis]. Vanderbilt University; 2005. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1803/13157.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Thornton-Wells TA. Comparison of Three Clustering Methods for Dissecting Trait Heterogeneity in Genotypic Data. [Thesis]. Vanderbilt University; 2005. Available from: http://hdl.handle.net/1803/13157

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

14. Liang, Xueying. Investigation of genetic susceptibility to late-onset Alzheimer disease through genomic convergence.

Degree: PhD, Human Genetics, 2007, Vanderbilt University

 With the exception of ApoE gene, no universally accepted genetic association has been identified with the complex Late-onset Alzheimer Disease (LOAD). A broad region of… (more)

Subjects/Keywords: Alzheimer Disease; gene-gene interaction; genomic convergence; association; candidate gene; chromosome 10; linkage; SNP; genetics; Alzheimer's disease  – Susceptibility

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APA (6th Edition):

Liang, X. (2007). Investigation of genetic susceptibility to late-onset Alzheimer disease through genomic convergence. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11992

Chicago Manual of Style (16th Edition):

Liang, Xueying. “Investigation of genetic susceptibility to late-onset Alzheimer disease through genomic convergence.” 2007. Doctoral Dissertation, Vanderbilt University. Accessed April 13, 2021. http://hdl.handle.net/1803/11992.

MLA Handbook (7th Edition):

Liang, Xueying. “Investigation of genetic susceptibility to late-onset Alzheimer disease through genomic convergence.” 2007. Web. 13 Apr 2021.

Vancouver:

Liang X. Investigation of genetic susceptibility to late-onset Alzheimer disease through genomic convergence. [Internet] [Doctoral dissertation]. Vanderbilt University; 2007. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1803/11992.

Council of Science Editors:

Liang X. Investigation of genetic susceptibility to late-onset Alzheimer disease through genomic convergence. [Doctoral Dissertation]. Vanderbilt University; 2007. Available from: http://hdl.handle.net/1803/11992


Vanderbilt University

15. Ryckman, Kelli Kae. The Genetics and Epidemiology of Reproductive Disorders.

Degree: PhD, Human Genetics, 2009, Vanderbilt University

 Each year in the United States about one million (17%) of all pregnancies experience complications that result in fetal loss. Of the five million pregnancies… (more)

Subjects/Keywords: bacterial vaginosis; pregnancy complications; preterm birth; Pregnancy  – Complications  – Epidemiology; Cytokines  – Physiological effect; Genetic disorders in pregnancy; Premature labor  – Etiology

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APA (6th Edition):

Ryckman, K. K. (2009). The Genetics and Epidemiology of Reproductive Disorders. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10423

Chicago Manual of Style (16th Edition):

Ryckman, Kelli Kae. “The Genetics and Epidemiology of Reproductive Disorders.” 2009. Doctoral Dissertation, Vanderbilt University. Accessed April 13, 2021. http://hdl.handle.net/1803/10423.

MLA Handbook (7th Edition):

Ryckman, Kelli Kae. “The Genetics and Epidemiology of Reproductive Disorders.” 2009. Web. 13 Apr 2021.

Vancouver:

Ryckman KK. The Genetics and Epidemiology of Reproductive Disorders. [Internet] [Doctoral dissertation]. Vanderbilt University; 2009. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1803/10423.

Council of Science Editors:

Ryckman KK. The Genetics and Epidemiology of Reproductive Disorders. [Doctoral Dissertation]. Vanderbilt University; 2009. Available from: http://hdl.handle.net/1803/10423


Vanderbilt University

16. Spencer, Kylee L. Variants in complement factor pathway genes and a novel locus on chromosome 16p12 influence age-related macular degeneration susceptibility.

Degree: PhD, Human Genetics, 2007, Vanderbilt University

 Age-related macular degeneration (AMD) is a complex, late-onset disease that is the leading cause of blindness in the elderly. Age, smoking, and variants in complement… (more)

Subjects/Keywords: complex genetic disease

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APA (6th Edition):

Spencer, K. L. (2007). Variants in complement factor pathway genes and a novel locus on chromosome 16p12 influence age-related macular degeneration susceptibility. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14024

Chicago Manual of Style (16th Edition):

Spencer, Kylee L. “Variants in complement factor pathway genes and a novel locus on chromosome 16p12 influence age-related macular degeneration susceptibility.” 2007. Doctoral Dissertation, Vanderbilt University. Accessed April 13, 2021. http://hdl.handle.net/1803/14024.

MLA Handbook (7th Edition):

Spencer, Kylee L. “Variants in complement factor pathway genes and a novel locus on chromosome 16p12 influence age-related macular degeneration susceptibility.” 2007. Web. 13 Apr 2021.

Vancouver:

Spencer KL. Variants in complement factor pathway genes and a novel locus on chromosome 16p12 influence age-related macular degeneration susceptibility. [Internet] [Doctoral dissertation]. Vanderbilt University; 2007. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1803/14024.

Council of Science Editors:

Spencer KL. Variants in complement factor pathway genes and a novel locus on chromosome 16p12 influence age-related macular degeneration susceptibility. [Doctoral Dissertation]. Vanderbilt University; 2007. Available from: http://hdl.handle.net/1803/14024


Vanderbilt University

17. Kenealy, Shannon. Investigating the genetic susceptibility to multiple sclerosis: a genomic convergence approach.

Degree: PhD, Molecular Physiology and Biophysics, 2006, Vanderbilt University

 Multiple sclerosis (MS) is a debilitating neuroimmunological and neuro-degenerative disease. Despite substantial evidence for polygenic inheritance, the MHC is the only region that clearly and… (more)

Subjects/Keywords: chromosome 1q; genetic linkage; allelic association; SNPs; Multiple sclerosis  – Genetic aspects

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kenealy, S. (2006). Investigating the genetic susceptibility to multiple sclerosis: a genomic convergence approach. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11989

Chicago Manual of Style (16th Edition):

Kenealy, Shannon. “Investigating the genetic susceptibility to multiple sclerosis: a genomic convergence approach.” 2006. Doctoral Dissertation, Vanderbilt University. Accessed April 13, 2021. http://hdl.handle.net/1803/11989.

MLA Handbook (7th Edition):

Kenealy, Shannon. “Investigating the genetic susceptibility to multiple sclerosis: a genomic convergence approach.” 2006. Web. 13 Apr 2021.

Vancouver:

Kenealy S. Investigating the genetic susceptibility to multiple sclerosis: a genomic convergence approach. [Internet] [Doctoral dissertation]. Vanderbilt University; 2006. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1803/11989.

Council of Science Editors:

Kenealy S. Investigating the genetic susceptibility to multiple sclerosis: a genomic convergence approach. [Doctoral Dissertation]. Vanderbilt University; 2006. Available from: http://hdl.handle.net/1803/11989


Vanderbilt University

18. McCauley, Jacob Lee. Genetic and phenotypic dissection of autism susceptibility.

Degree: PhD, Molecular Physiology and Biophysics, 2005, Vanderbilt University

 Autism is a severe neurodevelopmental disorder characterized by deficits in language and social interaction, and patterns of repetitive and stereotyped behaviors, interests and activities. Evidence… (more)

Subjects/Keywords: linkage; serotonin; GABA; chromosome 17; chromosome 19; chromosome 15q11-q13; association; autism; genetics

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

McCauley, J. L. (2005). Genetic and phenotypic dissection of autism susceptibility. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10664

Chicago Manual of Style (16th Edition):

McCauley, Jacob Lee. “Genetic and phenotypic dissection of autism susceptibility.” 2005. Doctoral Dissertation, Vanderbilt University. Accessed April 13, 2021. http://hdl.handle.net/1803/10664.

MLA Handbook (7th Edition):

McCauley, Jacob Lee. “Genetic and phenotypic dissection of autism susceptibility.” 2005. Web. 13 Apr 2021.

Vancouver:

McCauley JL. Genetic and phenotypic dissection of autism susceptibility. [Internet] [Doctoral dissertation]. Vanderbilt University; 2005. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1803/10664.

Council of Science Editors:

McCauley JL. Genetic and phenotypic dissection of autism susceptibility. [Doctoral Dissertation]. Vanderbilt University; 2005. Available from: http://hdl.handle.net/1803/10664

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