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You searched for +publisher:"Vanderbilt University" +contributor:("Jin Chen"). Showing records 1 – 30 of 32 total matches.

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Vanderbilt University

1. Ardestani, Shidrokh. New insights into tumor necrosis factor-alpha in cancer: distinct isoforms exert opposing effects on tumor associated myeloid cells and tumorigenesis.

Degree: PhD, Pathology, 2013, Vanderbilt University

 TNF-α, produced by most malignant cells, orchestrates the interplay between malignant cells and myeloid cells, which have been linked to tumor growth and metastasis. Although… (more)

Subjects/Keywords: ROS; Myeloid cells; TNF-alpha

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APA (6th Edition):

Ardestani, S. (2013). New insights into tumor necrosis factor-alpha in cancer: distinct isoforms exert opposing effects on tumor associated myeloid cells and tumorigenesis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/13448

Chicago Manual of Style (16th Edition):

Ardestani, Shidrokh. “New insights into tumor necrosis factor-alpha in cancer: distinct isoforms exert opposing effects on tumor associated myeloid cells and tumorigenesis.” 2013. Doctoral Dissertation, Vanderbilt University. Accessed January 26, 2021. http://hdl.handle.net/1803/13448.

MLA Handbook (7th Edition):

Ardestani, Shidrokh. “New insights into tumor necrosis factor-alpha in cancer: distinct isoforms exert opposing effects on tumor associated myeloid cells and tumorigenesis.” 2013. Web. 26 Jan 2021.

Vancouver:

Ardestani S. New insights into tumor necrosis factor-alpha in cancer: distinct isoforms exert opposing effects on tumor associated myeloid cells and tumorigenesis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2013. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1803/13448.

Council of Science Editors:

Ardestani S. New insights into tumor necrosis factor-alpha in cancer: distinct isoforms exert opposing effects on tumor associated myeloid cells and tumorigenesis. [Doctoral Dissertation]. Vanderbilt University; 2013. Available from: http://hdl.handle.net/1803/13448


Vanderbilt University

2. Violette, Katie. A Role for Tie1 in Late Gestational Semilunar Valve Development.

Degree: PhD, Cell and Developmental Biology, 2011, Vanderbilt University

 Evaluation of late events in cardiovascular development is precluded mid-gestational embryonic lethality associated with most traditional endothelial specific gene knockouts. Thus, it has not been… (more)

Subjects/Keywords: valve remodeling; heart; Tie1; NFATc1 P2Cre

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APA (6th Edition):

Violette, K. (2011). A Role for Tie1 in Late Gestational Semilunar Valve Development. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/13855

Chicago Manual of Style (16th Edition):

Violette, Katie. “A Role for Tie1 in Late Gestational Semilunar Valve Development.” 2011. Doctoral Dissertation, Vanderbilt University. Accessed January 26, 2021. http://hdl.handle.net/1803/13855.

MLA Handbook (7th Edition):

Violette, Katie. “A Role for Tie1 in Late Gestational Semilunar Valve Development.” 2011. Web. 26 Jan 2021.

Vancouver:

Violette K. A Role for Tie1 in Late Gestational Semilunar Valve Development. [Internet] [Doctoral dissertation]. Vanderbilt University; 2011. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1803/13855.

Council of Science Editors:

Violette K. A Role for Tie1 in Late Gestational Semilunar Valve Development. [Doctoral Dissertation]. Vanderbilt University; 2011. Available from: http://hdl.handle.net/1803/13855


Vanderbilt University

3. Williams, Andrew John. TGFbeta signaling enhances wound healing inflammation in post-partum breast cancer.

Degree: MS, Cancer Biology, 2014, Vanderbilt University

 Breast cancer is the leading cancer diagnosis in pre-menopausal women in the U.S. Of these breast cancers, 25% are diagnosed 2-5 years post-partum. Unfortunately, post-partum… (more)

Subjects/Keywords: breast cancer; wound healing; inflammation; post-partum; TGFbeta

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APA (6th Edition):

Williams, A. J. (2014). TGFbeta signaling enhances wound healing inflammation in post-partum breast cancer. (Thesis). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/15048

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Williams, Andrew John. “TGFbeta signaling enhances wound healing inflammation in post-partum breast cancer.” 2014. Thesis, Vanderbilt University. Accessed January 26, 2021. http://hdl.handle.net/1803/15048.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Williams, Andrew John. “TGFbeta signaling enhances wound healing inflammation in post-partum breast cancer.” 2014. Web. 26 Jan 2021.

Vancouver:

Williams AJ. TGFbeta signaling enhances wound healing inflammation in post-partum breast cancer. [Internet] [Thesis]. Vanderbilt University; 2014. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1803/15048.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Williams AJ. TGFbeta signaling enhances wound healing inflammation in post-partum breast cancer. [Thesis]. Vanderbilt University; 2014. Available from: http://hdl.handle.net/1803/15048

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

4. Morrison, Meghan Melinda. The role of mTORC2 in mammary morphogenesis and HER2-mediated tumorigenesis.

Degree: PhD, Cancer Biology, 2015, Vanderbilt University

 CANCER BIOLOGY The role of mTORC2 in mammary morphogenesis and HER2-mediated tumorigenesis Meghan Melinda Morrison Dissertation under the direction of Professor Rebecca Cook The phosphatidyl… (more)

Subjects/Keywords: mTORC2; mTOR; HER2; lapatinib; breast cancer

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APA (6th Edition):

Morrison, M. M. (2015). The role of mTORC2 in mammary morphogenesis and HER2-mediated tumorigenesis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12845

Chicago Manual of Style (16th Edition):

Morrison, Meghan Melinda. “The role of mTORC2 in mammary morphogenesis and HER2-mediated tumorigenesis.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed January 26, 2021. http://hdl.handle.net/1803/12845.

MLA Handbook (7th Edition):

Morrison, Meghan Melinda. “The role of mTORC2 in mammary morphogenesis and HER2-mediated tumorigenesis.” 2015. Web. 26 Jan 2021.

Vancouver:

Morrison MM. The role of mTORC2 in mammary morphogenesis and HER2-mediated tumorigenesis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1803/12845.

Council of Science Editors:

Morrison MM. The role of mTORC2 in mammary morphogenesis and HER2-mediated tumorigenesis. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://hdl.handle.net/1803/12845


Vanderbilt University

5. Evans, Justin D. Dissecting Location-Specific Signaling Pathway Activity in the Neurogenic Niche.

Degree: MS, Cancer Biology, 2014, Vanderbilt University

 The adult mammalian brain hosts two regions of quiescent neural stem cells that continually generate new neurons throughout life. One of these regions, the subventricular… (more)

Subjects/Keywords: Indentity; Sonic Hedgehog; SVZ; Neural Stem Cells

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APA (6th Edition):

Evans, J. D. (2014). Dissecting Location-Specific Signaling Pathway Activity in the Neurogenic Niche. (Thesis). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11326

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Evans, Justin D. “Dissecting Location-Specific Signaling Pathway Activity in the Neurogenic Niche.” 2014. Thesis, Vanderbilt University. Accessed January 26, 2021. http://hdl.handle.net/1803/11326.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Evans, Justin D. “Dissecting Location-Specific Signaling Pathway Activity in the Neurogenic Niche.” 2014. Web. 26 Jan 2021.

Vancouver:

Evans JD. Dissecting Location-Specific Signaling Pathway Activity in the Neurogenic Niche. [Internet] [Thesis]. Vanderbilt University; 2014. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1803/11326.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Evans JD. Dissecting Location-Specific Signaling Pathway Activity in the Neurogenic Niche. [Thesis]. Vanderbilt University; 2014. Available from: http://hdl.handle.net/1803/11326

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

6. Amato, Katherine Renee. Targeting the EPHA2 receptor tyrosine kinase in KRAS and EGFR mutant lung cancer.

Degree: PhD, Cancer Biology, 2015, Vanderbilt University

 Lung cancer remains the leading cause of cancer related deaths in the United States despite a significant number of advancements in the molecular diagnosis and… (more)

Subjects/Keywords: TKI; EPH; KRAS; EGFR

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APA (6th Edition):

Amato, K. R. (2015). Targeting the EPHA2 receptor tyrosine kinase in KRAS and EGFR mutant lung cancer. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11130

Chicago Manual of Style (16th Edition):

Amato, Katherine Renee. “Targeting the EPHA2 receptor tyrosine kinase in KRAS and EGFR mutant lung cancer.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed January 26, 2021. http://hdl.handle.net/1803/11130.

MLA Handbook (7th Edition):

Amato, Katherine Renee. “Targeting the EPHA2 receptor tyrosine kinase in KRAS and EGFR mutant lung cancer.” 2015. Web. 26 Jan 2021.

Vancouver:

Amato KR. Targeting the EPHA2 receptor tyrosine kinase in KRAS and EGFR mutant lung cancer. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1803/11130.

Council of Science Editors:

Amato KR. Targeting the EPHA2 receptor tyrosine kinase in KRAS and EGFR mutant lung cancer. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://hdl.handle.net/1803/11130


Vanderbilt University

7. Youngblood, Victoria Marie. The Ephrin-A1/EPHA2 Signaling Axis Regulates Glutaminolysis in HER2-positive Breast Cancer.

Degree: PhD, Cancer Biology, 2016, Vanderbilt University

 Dysregulation of receptor tyrosine kinases (RTKs) contributes to cellular transformation and cancer progression by disrupting key metabolic signaling pathways. The EPHA2 RTK is overexpressed in… (more)

Subjects/Keywords: breast cancer; ephrin-A1; EPHA2; glutaminolysis; glutamine metabolism; tumor metabolism

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APA (6th Edition):

Youngblood, V. M. (2016). The Ephrin-A1/EPHA2 Signaling Axis Regulates Glutaminolysis in HER2-positive Breast Cancer. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10841

Chicago Manual of Style (16th Edition):

Youngblood, Victoria Marie. “The Ephrin-A1/EPHA2 Signaling Axis Regulates Glutaminolysis in HER2-positive Breast Cancer.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed January 26, 2021. http://hdl.handle.net/1803/10841.

MLA Handbook (7th Edition):

Youngblood, Victoria Marie. “The Ephrin-A1/EPHA2 Signaling Axis Regulates Glutaminolysis in HER2-positive Breast Cancer.” 2016. Web. 26 Jan 2021.

Vancouver:

Youngblood VM. The Ephrin-A1/EPHA2 Signaling Axis Regulates Glutaminolysis in HER2-positive Breast Cancer. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1803/10841.

Council of Science Editors:

Youngblood VM. The Ephrin-A1/EPHA2 Signaling Axis Regulates Glutaminolysis in HER2-positive Breast Cancer. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://hdl.handle.net/1803/10841


Vanderbilt University

8. Vaught, David Bryan. EphA2 receptor tyrosine kinase in mammary gland development and breast cancer induced osteolysis.

Degree: PhD, Cancer Biology, 2011, Vanderbilt University

 Eph receptor tyrosine kinases are membrane bound receptors often expressed by normal epithelial cells but are frequently overexpressed in many human cancers. Of the many… (more)

Subjects/Keywords: Branching Morphogenesis; Mammary Gland; Bone Metastasis; Breast Cancer; EphA2; IL-6

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APA (6th Edition):

Vaught, D. B. (2011). EphA2 receptor tyrosine kinase in mammary gland development and breast cancer induced osteolysis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11610

Chicago Manual of Style (16th Edition):

Vaught, David Bryan. “EphA2 receptor tyrosine kinase in mammary gland development and breast cancer induced osteolysis.” 2011. Doctoral Dissertation, Vanderbilt University. Accessed January 26, 2021. http://hdl.handle.net/1803/11610.

MLA Handbook (7th Edition):

Vaught, David Bryan. “EphA2 receptor tyrosine kinase in mammary gland development and breast cancer induced osteolysis.” 2011. Web. 26 Jan 2021.

Vancouver:

Vaught DB. EphA2 receptor tyrosine kinase in mammary gland development and breast cancer induced osteolysis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2011. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1803/11610.

Council of Science Editors:

Vaught DB. EphA2 receptor tyrosine kinase in mammary gland development and breast cancer induced osteolysis. [Doctoral Dissertation]. Vanderbilt University; 2011. Available from: http://hdl.handle.net/1803/11610


Vanderbilt University

9. Rao, Meghana Nallamala. The Role of VAPB in Breast Cancer.

Degree: PhD, Cancer Biology, 2012, Vanderbilt University

 VAPB (VAMP-associated protein B) is an endoplasmic reticulum protein that regulates multiple biological functions. VAPB protein expression is elevated in human breast cancers and correlates… (more)

Subjects/Keywords: cancer; breast; VAPB; secretion; AKT

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APA (6th Edition):

Rao, M. N. (2012). The Role of VAPB in Breast Cancer. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14873

Chicago Manual of Style (16th Edition):

Rao, Meghana Nallamala. “The Role of VAPB in Breast Cancer.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed January 26, 2021. http://hdl.handle.net/1803/14873.

MLA Handbook (7th Edition):

Rao, Meghana Nallamala. “The Role of VAPB in Breast Cancer.” 2012. Web. 26 Jan 2021.

Vancouver:

Rao MN. The Role of VAPB in Breast Cancer. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1803/14873.

Council of Science Editors:

Rao MN. The Role of VAPB in Breast Cancer. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://hdl.handle.net/1803/14873


Vanderbilt University

10. Mackert, John Rodway. Dual Negative Roles of C/EBPα in the Expansion and Pro-angiogenic Function of Myeloid-Derived Suppressor Cells.

Degree: PhD, Cell and Developmental Biology, 2012, Vanderbilt University

 Myeloid-derived suppressor cells (MDSCs) play an important role in cancer progression. Elucidating the mechanisms involved in the expansion and function of these cells is important… (more)

Subjects/Keywords: MDSCs; C/EBPα; myeloid-derived suprressor cells; angiogenesis

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APA (6th Edition):

Mackert, J. R. (2012). Dual Negative Roles of C/EBPα in the Expansion and Pro-angiogenic Function of Myeloid-Derived Suppressor Cells. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/15280

Chicago Manual of Style (16th Edition):

Mackert, John Rodway. “Dual Negative Roles of C/EBPα in the Expansion and Pro-angiogenic Function of Myeloid-Derived Suppressor Cells.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed January 26, 2021. http://hdl.handle.net/1803/15280.

MLA Handbook (7th Edition):

Mackert, John Rodway. “Dual Negative Roles of C/EBPα in the Expansion and Pro-angiogenic Function of Myeloid-Derived Suppressor Cells.” 2012. Web. 26 Jan 2021.

Vancouver:

Mackert JR. Dual Negative Roles of C/EBPα in the Expansion and Pro-angiogenic Function of Myeloid-Derived Suppressor Cells. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1803/15280.

Council of Science Editors:

Mackert JR. Dual Negative Roles of C/EBPα in the Expansion and Pro-angiogenic Function of Myeloid-Derived Suppressor Cells. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://hdl.handle.net/1803/15280


Vanderbilt University

11. Saito-Diaz, Vicente Kenyi. Regulation of Wnt Receptor Activation by the Tumor Suppressor APC.

Degree: PhD, Cell and Developmental Biology, 2017, Vanderbilt University

 The Wnt pathway is a highly-conserved pathway that controls many developmental processes and is mutated in many human diseases (e.g., cancer). The tumor suppressor adenomatous… (more)

Subjects/Keywords: endocytosis; beta-catenin; LRP6; APC; Wnt signaling; clathrin; caveolin; colorectal cancer

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APA (6th Edition):

Saito-Diaz, V. K. (2017). Regulation of Wnt Receptor Activation by the Tumor Suppressor APC. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11541

Chicago Manual of Style (16th Edition):

Saito-Diaz, Vicente Kenyi. “Regulation of Wnt Receptor Activation by the Tumor Suppressor APC.” 2017. Doctoral Dissertation, Vanderbilt University. Accessed January 26, 2021. http://hdl.handle.net/1803/11541.

MLA Handbook (7th Edition):

Saito-Diaz, Vicente Kenyi. “Regulation of Wnt Receptor Activation by the Tumor Suppressor APC.” 2017. Web. 26 Jan 2021.

Vancouver:

Saito-Diaz VK. Regulation of Wnt Receptor Activation by the Tumor Suppressor APC. [Internet] [Doctoral dissertation]. Vanderbilt University; 2017. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1803/11541.

Council of Science Editors:

Saito-Diaz VK. Regulation of Wnt Receptor Activation by the Tumor Suppressor APC. [Doctoral Dissertation]. Vanderbilt University; 2017. Available from: http://hdl.handle.net/1803/11541


Vanderbilt University

12. Hutchinson, Katherine Emily. Identification of Novel Targets for Therapy in Solid Tumors.

Degree: PhD, Cancer Biology, 2015, Vanderbilt University

 Solid tumor treatment paradigms have drastically improved in recent decades through direct targeting of the protein products of somatic, constitutively active “driver” mutations and their… (more)

Subjects/Keywords: next-generation sequencing; targeted therapy; cancer; melanoma

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APA (6th Edition):

Hutchinson, K. E. (2015). Identification of Novel Targets for Therapy in Solid Tumors. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/15255

Chicago Manual of Style (16th Edition):

Hutchinson, Katherine Emily. “Identification of Novel Targets for Therapy in Solid Tumors.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed January 26, 2021. http://hdl.handle.net/1803/15255.

MLA Handbook (7th Edition):

Hutchinson, Katherine Emily. “Identification of Novel Targets for Therapy in Solid Tumors.” 2015. Web. 26 Jan 2021.

Vancouver:

Hutchinson KE. Identification of Novel Targets for Therapy in Solid Tumors. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1803/15255.

Council of Science Editors:

Hutchinson KE. Identification of Novel Targets for Therapy in Solid Tumors. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://hdl.handle.net/1803/15255


Vanderbilt University

13. Mulcrone, Patrick Louis. Influence of Stress on Bone Vasculature and Breast Cancer Bone Metastasis.

Degree: PhD, Cancer Biology, 2017, Vanderbilt University

 The skeleton is a common site for breast cancer metastasis. Although significant progress has been made to manage osteolytic bone lesions caused by breast tumors,… (more)

Subjects/Keywords: SNS; Osteoblasts; Breast cancer; Bone Vasculature

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APA (6th Edition):

Mulcrone, P. L. (2017). Influence of Stress on Bone Vasculature and Breast Cancer Bone Metastasis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/13591

Chicago Manual of Style (16th Edition):

Mulcrone, Patrick Louis. “Influence of Stress on Bone Vasculature and Breast Cancer Bone Metastasis.” 2017. Doctoral Dissertation, Vanderbilt University. Accessed January 26, 2021. http://hdl.handle.net/1803/13591.

MLA Handbook (7th Edition):

Mulcrone, Patrick Louis. “Influence of Stress on Bone Vasculature and Breast Cancer Bone Metastasis.” 2017. Web. 26 Jan 2021.

Vancouver:

Mulcrone PL. Influence of Stress on Bone Vasculature and Breast Cancer Bone Metastasis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2017. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1803/13591.

Council of Science Editors:

Mulcrone PL. Influence of Stress on Bone Vasculature and Breast Cancer Bone Metastasis. [Doctoral Dissertation]. Vanderbilt University; 2017. Available from: http://hdl.handle.net/1803/13591


Vanderbilt University

14. Samanta, Debangshu. Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer.

Degree: PhD, Cancer Biology, 2012, Vanderbilt University

 Inactivating mutations in TGF-©¬ receptors and Smad signal transducers that contribute to resistance to TGF-©¬, are associated with only very small number of NSCLC. The… (more)

Subjects/Keywords: Smoking; Lung cancer; Smad3; Chemoresistance

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APA (6th Edition):

Samanta, D. (2012). Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12131

Chicago Manual of Style (16th Edition):

Samanta, Debangshu. “Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed January 26, 2021. http://hdl.handle.net/1803/12131.

MLA Handbook (7th Edition):

Samanta, Debangshu. “Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer.” 2012. Web. 26 Jan 2021.

Vancouver:

Samanta D. Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1803/12131.

Council of Science Editors:

Samanta D. Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://hdl.handle.net/1803/12131


Vanderbilt University

15. Yu, Huapeng. p120-catenin controls contractility along the vertical axis of epithelial lateral membranes.

Degree: PhD, Cancer Biology, 2015, Vanderbilt University

 In vertebrate epithelia, p120-catenin mediates E-cadherin stability and suppression of RhoA. Genetic ablation of p120 in various epithelial tissues typically causes striking alterations in tissue… (more)

Subjects/Keywords: contractility; cadherin; catenin

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APA (6th Edition):

Yu, H. (2015). p120-catenin controls contractility along the vertical axis of epithelial lateral membranes. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/15207

Chicago Manual of Style (16th Edition):

Yu, Huapeng. “p120-catenin controls contractility along the vertical axis of epithelial lateral membranes.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed January 26, 2021. http://hdl.handle.net/1803/15207.

MLA Handbook (7th Edition):

Yu, Huapeng. “p120-catenin controls contractility along the vertical axis of epithelial lateral membranes.” 2015. Web. 26 Jan 2021.

Vancouver:

Yu H. p120-catenin controls contractility along the vertical axis of epithelial lateral membranes. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1803/15207.

Council of Science Editors:

Yu H. p120-catenin controls contractility along the vertical axis of epithelial lateral membranes. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://hdl.handle.net/1803/15207


Vanderbilt University

16. Williams, Michelle Marie. Mcl-1 Drives Resistance of Estrogen Receptor-α Positive Breast Cancers to Targeted Therapies.

Degree: PhD, Cancer Biology, 2017, Vanderbilt University

 Evasion of cell death is essential to every step of tumorigenesis. Anti-apoptotic Bcl-2 family proteins are master inhibitors of cell death, and thus are often… (more)

Subjects/Keywords: Mcl-1; therapeutic resistance; Bcl-2 family proteins; luminal breast cancers

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APA (6th Edition):

Williams, M. M. (2017). Mcl-1 Drives Resistance of Estrogen Receptor-α Positive Breast Cancers to Targeted Therapies. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/13213

Chicago Manual of Style (16th Edition):

Williams, Michelle Marie. “Mcl-1 Drives Resistance of Estrogen Receptor-α Positive Breast Cancers to Targeted Therapies.” 2017. Doctoral Dissertation, Vanderbilt University. Accessed January 26, 2021. http://hdl.handle.net/1803/13213.

MLA Handbook (7th Edition):

Williams, Michelle Marie. “Mcl-1 Drives Resistance of Estrogen Receptor-α Positive Breast Cancers to Targeted Therapies.” 2017. Web. 26 Jan 2021.

Vancouver:

Williams MM. Mcl-1 Drives Resistance of Estrogen Receptor-α Positive Breast Cancers to Targeted Therapies. [Internet] [Doctoral dissertation]. Vanderbilt University; 2017. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1803/13213.

Council of Science Editors:

Williams MM. Mcl-1 Drives Resistance of Estrogen Receptor-α Positive Breast Cancers to Targeted Therapies. [Doctoral Dissertation]. Vanderbilt University; 2017. Available from: http://hdl.handle.net/1803/13213


Vanderbilt University

17. Hang, Brian I. Kinase Regulation of XIAP in Wnt Signaling.

Degree: PhD, Cell and Developmental Biology, 2016, Vanderbilt University

 The Wnt signaling pathway plays essential roles in a wide variety of biological processes including early animal development, cell fate determination, cell proliferation, organogenesis, and… (more)

Subjects/Keywords: Wnt; XIAP; phosphorylation; ubiquitination

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APA (6th Edition):

Hang, B. I. (2016). Kinase Regulation of XIAP in Wnt Signaling. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12849

Chicago Manual of Style (16th Edition):

Hang, Brian I. “Kinase Regulation of XIAP in Wnt Signaling.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed January 26, 2021. http://hdl.handle.net/1803/12849.

MLA Handbook (7th Edition):

Hang, Brian I. “Kinase Regulation of XIAP in Wnt Signaling.” 2016. Web. 26 Jan 2021.

Vancouver:

Hang BI. Kinase Regulation of XIAP in Wnt Signaling. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1803/12849.

Council of Science Editors:

Hang BI. Kinase Regulation of XIAP in Wnt Signaling. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://hdl.handle.net/1803/12849


Vanderbilt University

18. Tran, Thuy Thanh Thi. Roles of the alpha2beta1 Integrin in Cancer Progression and Metastasis.

Degree: PhD, Pathology, 2011, Vanderbilt University

 In this dissertation, I examined the function of the alpha2beta1 integrin in epithelial tumor cells as well as in the tumor microenvironment. The alpha2beta1 integrin… (more)

Subjects/Keywords: squamous cell carcinoma; lymphatic metastasis; a2b1 integrin; breast cancer

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APA (6th Edition):

Tran, T. T. T. (2011). Roles of the alpha2beta1 Integrin in Cancer Progression and Metastasis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/15151

Chicago Manual of Style (16th Edition):

Tran, Thuy Thanh Thi. “Roles of the alpha2beta1 Integrin in Cancer Progression and Metastasis.” 2011. Doctoral Dissertation, Vanderbilt University. Accessed January 26, 2021. http://hdl.handle.net/1803/15151.

MLA Handbook (7th Edition):

Tran, Thuy Thanh Thi. “Roles of the alpha2beta1 Integrin in Cancer Progression and Metastasis.” 2011. Web. 26 Jan 2021.

Vancouver:

Tran TTT. Roles of the alpha2beta1 Integrin in Cancer Progression and Metastasis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2011. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1803/15151.

Council of Science Editors:

Tran TTT. Roles of the alpha2beta1 Integrin in Cancer Progression and Metastasis. [Doctoral Dissertation]. Vanderbilt University; 2011. Available from: http://hdl.handle.net/1803/15151


Vanderbilt University

19. Short, Sarah Palmer. A Gatekeeper Function for p120 and the E-cadherin Complex in Intestinal Tumorigenesis.

Degree: PhD, Cancer Biology, 2015, Vanderbilt University

 E-cadherin is widely recognized as a tumor- and/or metastasis suppressor, with its activity as a cell-cell adhesion receptor is dependent on tightly coupled interactions with… (more)

Subjects/Keywords: cell adhesion; cadherin complex; Kaiso; p120-catenin; adherens junction

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APA (6th Edition):

Short, S. P. (2015). A Gatekeeper Function for p120 and the E-cadherin Complex in Intestinal Tumorigenesis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10950

Chicago Manual of Style (16th Edition):

Short, Sarah Palmer. “A Gatekeeper Function for p120 and the E-cadherin Complex in Intestinal Tumorigenesis.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed January 26, 2021. http://hdl.handle.net/1803/10950.

MLA Handbook (7th Edition):

Short, Sarah Palmer. “A Gatekeeper Function for p120 and the E-cadherin Complex in Intestinal Tumorigenesis.” 2015. Web. 26 Jan 2021.

Vancouver:

Short SP. A Gatekeeper Function for p120 and the E-cadherin Complex in Intestinal Tumorigenesis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1803/10950.

Council of Science Editors:

Short SP. A Gatekeeper Function for p120 and the E-cadherin Complex in Intestinal Tumorigenesis. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://hdl.handle.net/1803/10950


Vanderbilt University

20. Leelatian, Nalin. Stratification of Adult Glioblastoma with Signaling and Single Cell Biology.

Degree: PhD, Cancer Biology, 2018, Vanderbilt University

 Genomic and transcriptomic profiling have revealed extensive cellular diversity in adult glioblastoma, a rapidly fatal brain tumor. These approaches, however, have neither successfully stratified patient… (more)

Subjects/Keywords: Glioblastoma; Cancer; Single cell; Signaling; Proteomics; Systems biology

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APA (6th Edition):

Leelatian, N. (2018). Stratification of Adult Glioblastoma with Signaling and Single Cell Biology. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10874

Chicago Manual of Style (16th Edition):

Leelatian, Nalin. “Stratification of Adult Glioblastoma with Signaling and Single Cell Biology.” 2018. Doctoral Dissertation, Vanderbilt University. Accessed January 26, 2021. http://hdl.handle.net/1803/10874.

MLA Handbook (7th Edition):

Leelatian, Nalin. “Stratification of Adult Glioblastoma with Signaling and Single Cell Biology.” 2018. Web. 26 Jan 2021.

Vancouver:

Leelatian N. Stratification of Adult Glioblastoma with Signaling and Single Cell Biology. [Internet] [Doctoral dissertation]. Vanderbilt University; 2018. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1803/10874.

Council of Science Editors:

Leelatian N. Stratification of Adult Glioblastoma with Signaling and Single Cell Biology. [Doctoral Dissertation]. Vanderbilt University; 2018. Available from: http://hdl.handle.net/1803/10874


Vanderbilt University

21. Hutchens, Troy John. Novel Regulators of Glucagon Secretion from α-Cells.

Degree: PhD, Chemical and Physical Biology, 2015, Vanderbilt University

 The loss of inhibition of glucagon secretion exacerbates hyperglycemia in types 1 and 2 diabetes. However, the molecular mechanisms that regulate glucagon secretion in unaffected… (more)

Subjects/Keywords: α-cell; glucagon; EphA receptors; ephrin-A ligands; brown adipose tissue

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APA (6th Edition):

Hutchens, T. J. (2015). Novel Regulators of Glucagon Secretion from α-Cells. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/13895

Chicago Manual of Style (16th Edition):

Hutchens, Troy John. “Novel Regulators of Glucagon Secretion from α-Cells.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed January 26, 2021. http://hdl.handle.net/1803/13895.

MLA Handbook (7th Edition):

Hutchens, Troy John. “Novel Regulators of Glucagon Secretion from α-Cells.” 2015. Web. 26 Jan 2021.

Vancouver:

Hutchens TJ. Novel Regulators of Glucagon Secretion from α-Cells. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1803/13895.

Council of Science Editors:

Hutchens TJ. Novel Regulators of Glucagon Secretion from α-Cells. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://hdl.handle.net/1803/13895


Vanderbilt University

22. Madamanchi, Aasakiran. α2β1 integrin in Retinopathy and Sprouting Angiogenesis.

Degree: PhD, Cancer Biology, 2016, Vanderbilt University

 Angiogenesis expands the vascular network during normal development and in response to angiogenic stress. Dysregulation of this dynamic process contributes to the pathogenesis of many… (more)

Subjects/Keywords: α2β1 Integrin; Retinopathy; Notch; Mathematical Models

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APA (6th Edition):

Madamanchi, A. (2016). α2β1 integrin in Retinopathy and Sprouting Angiogenesis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10549

Chicago Manual of Style (16th Edition):

Madamanchi, Aasakiran. “α2β1 integrin in Retinopathy and Sprouting Angiogenesis.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed January 26, 2021. http://hdl.handle.net/1803/10549.

MLA Handbook (7th Edition):

Madamanchi, Aasakiran. “α2β1 integrin in Retinopathy and Sprouting Angiogenesis.” 2016. Web. 26 Jan 2021.

Vancouver:

Madamanchi A. α2β1 integrin in Retinopathy and Sprouting Angiogenesis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1803/10549.

Council of Science Editors:

Madamanchi A. α2β1 integrin in Retinopathy and Sprouting Angiogenesis. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://hdl.handle.net/1803/10549


Vanderbilt University

23. Duell, Stephanie. Kaposi's Sarcoma Associated Herpesvirus-Encoded Cyclin, K-cyclin Enhance NF-kappa B Dependent Transcription And Interacts With Latency-Associated Nuclear Antigen In Virally and Non-Virally Infected Cells.

Degree: MS, Cancer Biology, 2007, Vanderbilt University

 Kaposi's sarcoma Herpesvirus (KSHV) is the causative agent of Kaposi's sarcoma, primary effusion lymphoma (PEL) and multicentric Castlemen's disease (MCD). This ƒ×-herpesvirus infects endothelial cells… (more)

Subjects/Keywords: Transcription factors; Viral antigens; cyclin regulated transcription; HHV-8; viral oncology; Cyclins  – Physiological effect

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APA (6th Edition):

Duell, S. (2007). Kaposi's Sarcoma Associated Herpesvirus-Encoded Cyclin, K-cyclin Enhance NF-kappa B Dependent Transcription And Interacts With Latency-Associated Nuclear Antigen In Virally and Non-Virally Infected Cells. (Thesis). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12658

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Duell, Stephanie. “Kaposi's Sarcoma Associated Herpesvirus-Encoded Cyclin, K-cyclin Enhance NF-kappa B Dependent Transcription And Interacts With Latency-Associated Nuclear Antigen In Virally and Non-Virally Infected Cells.” 2007. Thesis, Vanderbilt University. Accessed January 26, 2021. http://hdl.handle.net/1803/12658.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Duell, Stephanie. “Kaposi's Sarcoma Associated Herpesvirus-Encoded Cyclin, K-cyclin Enhance NF-kappa B Dependent Transcription And Interacts With Latency-Associated Nuclear Antigen In Virally and Non-Virally Infected Cells.” 2007. Web. 26 Jan 2021.

Vancouver:

Duell S. Kaposi's Sarcoma Associated Herpesvirus-Encoded Cyclin, K-cyclin Enhance NF-kappa B Dependent Transcription And Interacts With Latency-Associated Nuclear Antigen In Virally and Non-Virally Infected Cells. [Internet] [Thesis]. Vanderbilt University; 2007. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1803/12658.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Duell S. Kaposi's Sarcoma Associated Herpesvirus-Encoded Cyclin, K-cyclin Enhance NF-kappa B Dependent Transcription And Interacts With Latency-Associated Nuclear Antigen In Virally and Non-Virally Infected Cells. [Thesis]. Vanderbilt University; 2007. Available from: http://hdl.handle.net/1803/12658

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

24. Fang, Wei Bin. The Role of EphA2 RTK in Breast Cancer Cell Malignancy and Tumor Angiogenesis.

Degree: PhD, Cancer Biology, 2008, Vanderbilt University

 The EphA2 receptor belongs to the recently cloned Eph family of receptor tyrosine kinase (RTK). High levels of EphA2 RTK have been detected in 60-90%… (more)

Subjects/Keywords: Receptor Tyrosine Kinase; Breast Cancer; EphA2; Neovascularization  – Regulation; Protein-tyrosine kinase  – Receptors

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APA (6th Edition):

Fang, W. B. (2008). The Role of EphA2 RTK in Breast Cancer Cell Malignancy and Tumor Angiogenesis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/13970

Chicago Manual of Style (16th Edition):

Fang, Wei Bin. “The Role of EphA2 RTK in Breast Cancer Cell Malignancy and Tumor Angiogenesis.” 2008. Doctoral Dissertation, Vanderbilt University. Accessed January 26, 2021. http://hdl.handle.net/1803/13970.

MLA Handbook (7th Edition):

Fang, Wei Bin. “The Role of EphA2 RTK in Breast Cancer Cell Malignancy and Tumor Angiogenesis.” 2008. Web. 26 Jan 2021.

Vancouver:

Fang WB. The Role of EphA2 RTK in Breast Cancer Cell Malignancy and Tumor Angiogenesis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2008. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1803/13970.

Council of Science Editors:

Fang WB. The Role of EphA2 RTK in Breast Cancer Cell Malignancy and Tumor Angiogenesis. [Doctoral Dissertation]. Vanderbilt University; 2008. Available from: http://hdl.handle.net/1803/13970


Vanderbilt University

25. Atwood, Allison Annette. Regulation of C/EBPbeta1 in transformed mammary epithelial cells and the role of C/EBPbeta1 in oncogene-induced senescence.

Degree: PhD, Cancer Biology, 2010, Vanderbilt University

 Overexpression of activated oncogenes such as Ras(V12) in primary cells induces senescence rather than transformation, thus suppressing tumorigenesis. C/EBPbeta is required for Ras(V12)-induced senescence in… (more)

Subjects/Keywords: Ras; breast cancer; IL6; senescence; C/EBPbeta; sumoylation; degradation; phosphorylation

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APA (6th Edition):

Atwood, A. A. (2010). Regulation of C/EBPbeta1 in transformed mammary epithelial cells and the role of C/EBPbeta1 in oncogene-induced senescence. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/15241

Chicago Manual of Style (16th Edition):

Atwood, Allison Annette. “Regulation of C/EBPbeta1 in transformed mammary epithelial cells and the role of C/EBPbeta1 in oncogene-induced senescence.” 2010. Doctoral Dissertation, Vanderbilt University. Accessed January 26, 2021. http://hdl.handle.net/1803/15241.

MLA Handbook (7th Edition):

Atwood, Allison Annette. “Regulation of C/EBPbeta1 in transformed mammary epithelial cells and the role of C/EBPbeta1 in oncogene-induced senescence.” 2010. Web. 26 Jan 2021.

Vancouver:

Atwood AA. Regulation of C/EBPbeta1 in transformed mammary epithelial cells and the role of C/EBPbeta1 in oncogene-induced senescence. [Internet] [Doctoral dissertation]. Vanderbilt University; 2010. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1803/15241.

Council of Science Editors:

Atwood AA. Regulation of C/EBPbeta1 in transformed mammary epithelial cells and the role of C/EBPbeta1 in oncogene-induced senescence. [Doctoral Dissertation]. Vanderbilt University; 2010. Available from: http://hdl.handle.net/1803/15241


Vanderbilt University

26. Westmoreland, Joby Jackson. The effect of post-translational modifications on Xlefty function.

Degree: PhD, Cell and Developmental Biology, 2007, Vanderbilt University

 The Nodal and Nodal-related morphogens are utilized for the specification of distinct cellular identity throughout development by activating discrete target genes in a concentration-dependant manner.… (more)

Subjects/Keywords: Post-translational modification; Xenopus; Xlefty; mesoderm; Transforming growth factors-beta; Morphogenesis  – Molecular aspects

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APA (6th Edition):

Westmoreland, J. J. (2007). The effect of post-translational modifications on Xlefty function. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14809

Chicago Manual of Style (16th Edition):

Westmoreland, Joby Jackson. “The effect of post-translational modifications on Xlefty function.” 2007. Doctoral Dissertation, Vanderbilt University. Accessed January 26, 2021. http://hdl.handle.net/1803/14809.

MLA Handbook (7th Edition):

Westmoreland, Joby Jackson. “The effect of post-translational modifications on Xlefty function.” 2007. Web. 26 Jan 2021.

Vancouver:

Westmoreland JJ. The effect of post-translational modifications on Xlefty function. [Internet] [Doctoral dissertation]. Vanderbilt University; 2007. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1803/14809.

Council of Science Editors:

Westmoreland JJ. The effect of post-translational modifications on Xlefty function. [Doctoral Dissertation]. Vanderbilt University; 2007. Available from: http://hdl.handle.net/1803/14809


Vanderbilt University

27. DeBusk, Laura M. AKT/IKKα/VAV1 signaling in endothelial cell survival and angiogenesis.

Degree: PhD, Cancer Biology, 2008, Vanderbilt University

 We have identified a novel signaling pathway, AKT/IKKα/VAV1, which induces endothelial cell survival and motility. Ang1/Tie2 and VEGF/VEGFR signaling activates Akt and induces cell survival.… (more)

Subjects/Keywords: angiogenesis; Neovascularization  – Regulation; Vascular endothelial growth factors  – Pathophysiology; Cellular control mechanisms

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APA (6th Edition):

DeBusk, L. M. (2008). AKT/IKKα/VAV1 signaling in endothelial cell survival and angiogenesis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11831

Chicago Manual of Style (16th Edition):

DeBusk, Laura M. “AKT/IKKα/VAV1 signaling in endothelial cell survival and angiogenesis.” 2008. Doctoral Dissertation, Vanderbilt University. Accessed January 26, 2021. http://hdl.handle.net/1803/11831.

MLA Handbook (7th Edition):

DeBusk, Laura M. “AKT/IKKα/VAV1 signaling in endothelial cell survival and angiogenesis.” 2008. Web. 26 Jan 2021.

Vancouver:

DeBusk LM. AKT/IKKα/VAV1 signaling in endothelial cell survival and angiogenesis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2008. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1803/11831.

Council of Science Editors:

DeBusk LM. AKT/IKKα/VAV1 signaling in endothelial cell survival and angiogenesis. [Doctoral Dissertation]. Vanderbilt University; 2008. Available from: http://hdl.handle.net/1803/11831


Vanderbilt University

28. Cheng, Hua. Analysis of Eph receptor signaling during oocyte meiotic maturation in Caenorhabditis elegans.

Degree: PhD, cell and developmental biology, 2008, Vanderbilt University

 A conserved biological feature of sexual reproduction in animals is that oocytes arrest in meiotic prophase and resume meiosis in response to extra-ovarian signals. In… (more)

Subjects/Keywords: receptor trafficking; meiotic maturation; Caenorhabditis elegans  – Reproduction  – Regulation; Protein-tyrosine kinase  – Receptors; Eph receptor; G proteins  – Physiological effect

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APA (6th Edition):

Cheng, H. (2008). Analysis of Eph receptor signaling during oocyte meiotic maturation in Caenorhabditis elegans. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14843

Chicago Manual of Style (16th Edition):

Cheng, Hua. “Analysis of Eph receptor signaling during oocyte meiotic maturation in Caenorhabditis elegans.” 2008. Doctoral Dissertation, Vanderbilt University. Accessed January 26, 2021. http://hdl.handle.net/1803/14843.

MLA Handbook (7th Edition):

Cheng, Hua. “Analysis of Eph receptor signaling during oocyte meiotic maturation in Caenorhabditis elegans.” 2008. Web. 26 Jan 2021.

Vancouver:

Cheng H. Analysis of Eph receptor signaling during oocyte meiotic maturation in Caenorhabditis elegans. [Internet] [Doctoral dissertation]. Vanderbilt University; 2008. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1803/14843.

Council of Science Editors:

Cheng H. Analysis of Eph receptor signaling during oocyte meiotic maturation in Caenorhabditis elegans. [Doctoral Dissertation]. Vanderbilt University; 2008. Available from: http://hdl.handle.net/1803/14843


Vanderbilt University

29. Werdich, Xiang Qi. Characterization of Src family kinases as potential targets for intervention in vascular endothelial growth factor-mediated retinal neovascularization.

Degree: PhD, Cell and Developmental Biology, 2005, Vanderbilt University

 Hypoxia inducible vascular endothelial growth factor (VEGF) plays a major role in initiation and regulation of retinal neovascularization, which is the leading cause of severe… (more)

Subjects/Keywords: signal transduction; RNA interference; retinal ischemia; cellular function; receptor

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APA (6th Edition):

Werdich, X. Q. (2005). Characterization of Src family kinases as potential targets for intervention in vascular endothelial growth factor-mediated retinal neovascularization. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11671

Chicago Manual of Style (16th Edition):

Werdich, Xiang Qi. “Characterization of Src family kinases as potential targets for intervention in vascular endothelial growth factor-mediated retinal neovascularization.” 2005. Doctoral Dissertation, Vanderbilt University. Accessed January 26, 2021. http://hdl.handle.net/1803/11671.

MLA Handbook (7th Edition):

Werdich, Xiang Qi. “Characterization of Src family kinases as potential targets for intervention in vascular endothelial growth factor-mediated retinal neovascularization.” 2005. Web. 26 Jan 2021.

Vancouver:

Werdich XQ. Characterization of Src family kinases as potential targets for intervention in vascular endothelial growth factor-mediated retinal neovascularization. [Internet] [Doctoral dissertation]. Vanderbilt University; 2005. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1803/11671.

Council of Science Editors:

Werdich XQ. Characterization of Src family kinases as potential targets for intervention in vascular endothelial growth factor-mediated retinal neovascularization. [Doctoral Dissertation]. Vanderbilt University; 2005. Available from: http://hdl.handle.net/1803/11671


Vanderbilt University

30. Everhart, Michael Brett. The role of Nuclear Factor-kappa B (NF-kB) in the regulation of lung inflammation.

Degree: PhD, Cell and Developmental Biology, 2004, Vanderbilt University

 The NF-kappaB pathway has been shown to play a critical role in both adaptive and innate immunity and has been implicated as a focal point… (more)

Subjects/Keywords: inflammation; cell signaling; nuclear factor-kappa B; lung

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APA (6th Edition):

Everhart, M. B. (2004). The role of Nuclear Factor-kappa B (NF-kB) in the regulation of lung inflammation. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14942

Chicago Manual of Style (16th Edition):

Everhart, Michael Brett. “The role of Nuclear Factor-kappa B (NF-kB) in the regulation of lung inflammation.” 2004. Doctoral Dissertation, Vanderbilt University. Accessed January 26, 2021. http://hdl.handle.net/1803/14942.

MLA Handbook (7th Edition):

Everhart, Michael Brett. “The role of Nuclear Factor-kappa B (NF-kB) in the regulation of lung inflammation.” 2004. Web. 26 Jan 2021.

Vancouver:

Everhart MB. The role of Nuclear Factor-kappa B (NF-kB) in the regulation of lung inflammation. [Internet] [Doctoral dissertation]. Vanderbilt University; 2004. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1803/14942.

Council of Science Editors:

Everhart MB. The role of Nuclear Factor-kappa B (NF-kB) in the regulation of lung inflammation. [Doctoral Dissertation]. Vanderbilt University; 2004. Available from: http://hdl.handle.net/1803/14942

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