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You searched for +publisher:"Vanderbilt University" +contributor:("Gregg Stanwood"). Showing records 1 – 17 of 17 total matches.

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Vanderbilt University

1. Brown, Molly Nicole. GABA synthesis in developing hippocampus: SNAT1 surfaces as a dynamic regulator of inhibitory synaptic transmission.

Degree: PhD, Pharmacology, 2010, Vanderbilt University

 GABA functions as the primary inhibitory neurotransmitter in the mammalian brain. In hippocampus, GABA serves multiple roles during development and throughout adulthood, which include: 1)… (more)

Subjects/Keywords: SNAT1; System A; GABA; glutamine; hippocampus; development

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APA (6th Edition):

Brown, M. N. (2010). GABA synthesis in developing hippocampus: SNAT1 surfaces as a dynamic regulator of inhibitory synaptic transmission. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-03292010-151906/ ;

Chicago Manual of Style (16th Edition):

Brown, Molly Nicole. “GABA synthesis in developing hippocampus: SNAT1 surfaces as a dynamic regulator of inhibitory synaptic transmission.” 2010. Doctoral Dissertation, Vanderbilt University. Accessed March 31, 2020. http://etd.library.vanderbilt.edu/available/etd-03292010-151906/ ;.

MLA Handbook (7th Edition):

Brown, Molly Nicole. “GABA synthesis in developing hippocampus: SNAT1 surfaces as a dynamic regulator of inhibitory synaptic transmission.” 2010. Web. 31 Mar 2020.

Vancouver:

Brown MN. GABA synthesis in developing hippocampus: SNAT1 surfaces as a dynamic regulator of inhibitory synaptic transmission. [Internet] [Doctoral dissertation]. Vanderbilt University; 2010. [cited 2020 Mar 31]. Available from: http://etd.library.vanderbilt.edu/available/etd-03292010-151906/ ;.

Council of Science Editors:

Brown MN. GABA synthesis in developing hippocampus: SNAT1 surfaces as a dynamic regulator of inhibitory synaptic transmission. [Doctoral Dissertation]. Vanderbilt University; 2010. Available from: http://etd.library.vanderbilt.edu/available/etd-03292010-151906/ ;


Vanderbilt University

2. Speed, Nicole Kathryn. The Role of Insulin Signaling on Dopamine Transporter Trafficking.

Degree: PhD, Pharmacology, 2010, Vanderbilt University

 Dopaminergic neurotransmission is a critical component in the regulation of several behaviors, including cognition, motor function, motivation, and reward. Abnormalities in dopamine (DA) signaling have… (more)

Subjects/Keywords: dopamine; insulin; obesity

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APA (6th Edition):

Speed, N. K. (2010). The Role of Insulin Signaling on Dopamine Transporter Trafficking. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-07212010-144350/ ;

Chicago Manual of Style (16th Edition):

Speed, Nicole Kathryn. “The Role of Insulin Signaling on Dopamine Transporter Trafficking.” 2010. Doctoral Dissertation, Vanderbilt University. Accessed March 31, 2020. http://etd.library.vanderbilt.edu/available/etd-07212010-144350/ ;.

MLA Handbook (7th Edition):

Speed, Nicole Kathryn. “The Role of Insulin Signaling on Dopamine Transporter Trafficking.” 2010. Web. 31 Mar 2020.

Vancouver:

Speed NK. The Role of Insulin Signaling on Dopamine Transporter Trafficking. [Internet] [Doctoral dissertation]. Vanderbilt University; 2010. [cited 2020 Mar 31]. Available from: http://etd.library.vanderbilt.edu/available/etd-07212010-144350/ ;.

Council of Science Editors:

Speed NK. The Role of Insulin Signaling on Dopamine Transporter Trafficking. [Doctoral Dissertation]. Vanderbilt University; 2010. Available from: http://etd.library.vanderbilt.edu/available/etd-07212010-144350/ ;


Vanderbilt University

3. Anderson, Lyndsey Leigh. Pharmacological and Genetic Rescue of Idiopathic Epilepsies.

Degree: PhD, Pharmacology, 2014, Vanderbilt University

 PHARMACOLOGY Pharmacological and Genetic Rescue of Idiopathic Epilepsies Lyndsey Leigh Anderson Dissertation under the direction of Professor Alfred George, Jr. Epilepsy is a common neurological… (more)

Subjects/Keywords: ion channels; epilepsy; ion channel pharmacology

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APA (6th Edition):

Anderson, L. L. (2014). Pharmacological and Genetic Rescue of Idiopathic Epilepsies. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-03182014-152959/ ;

Chicago Manual of Style (16th Edition):

Anderson, Lyndsey Leigh. “Pharmacological and Genetic Rescue of Idiopathic Epilepsies.” 2014. Doctoral Dissertation, Vanderbilt University. Accessed March 31, 2020. http://etd.library.vanderbilt.edu/available/etd-03182014-152959/ ;.

MLA Handbook (7th Edition):

Anderson, Lyndsey Leigh. “Pharmacological and Genetic Rescue of Idiopathic Epilepsies.” 2014. Web. 31 Mar 2020.

Vancouver:

Anderson LL. Pharmacological and Genetic Rescue of Idiopathic Epilepsies. [Internet] [Doctoral dissertation]. Vanderbilt University; 2014. [cited 2020 Mar 31]. Available from: http://etd.library.vanderbilt.edu/available/etd-03182014-152959/ ;.

Council of Science Editors:

Anderson LL. Pharmacological and Genetic Rescue of Idiopathic Epilepsies. [Doctoral Dissertation]. Vanderbilt University; 2014. Available from: http://etd.library.vanderbilt.edu/available/etd-03182014-152959/ ;


Vanderbilt University

4. Holleran, Katherine Mercedes. Regulation of Basal and Ethanol Abstinence-Induced Affective Behaviors by GluN2B-containin NMDA Receptors in the Bed Nucleus of the Stria Terminalis and Endocannabinoids.

Degree: PhD, Neuroscience, 2016, Vanderbilt University

 Major depressive disorder (MDD) represents an enormous societal burden that will directly affect nearly one fifth of Americans within their lifetime. Despite its high prevalence… (more)

Subjects/Keywords: Endocannabinoids; BNST; NMDA receptors; Ketamine; Depression

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APA (6th Edition):

Holleran, K. M. (2016). Regulation of Basal and Ethanol Abstinence-Induced Affective Behaviors by GluN2B-containin NMDA Receptors in the Bed Nucleus of the Stria Terminalis and Endocannabinoids. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-02042016-121346/ ;

Chicago Manual of Style (16th Edition):

Holleran, Katherine Mercedes. “Regulation of Basal and Ethanol Abstinence-Induced Affective Behaviors by GluN2B-containin NMDA Receptors in the Bed Nucleus of the Stria Terminalis and Endocannabinoids.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed March 31, 2020. http://etd.library.vanderbilt.edu/available/etd-02042016-121346/ ;.

MLA Handbook (7th Edition):

Holleran, Katherine Mercedes. “Regulation of Basal and Ethanol Abstinence-Induced Affective Behaviors by GluN2B-containin NMDA Receptors in the Bed Nucleus of the Stria Terminalis and Endocannabinoids.” 2016. Web. 31 Mar 2020.

Vancouver:

Holleran KM. Regulation of Basal and Ethanol Abstinence-Induced Affective Behaviors by GluN2B-containin NMDA Receptors in the Bed Nucleus of the Stria Terminalis and Endocannabinoids. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2020 Mar 31]. Available from: http://etd.library.vanderbilt.edu/available/etd-02042016-121346/ ;.

Council of Science Editors:

Holleran KM. Regulation of Basal and Ethanol Abstinence-Induced Affective Behaviors by GluN2B-containin NMDA Receptors in the Bed Nucleus of the Stria Terminalis and Endocannabinoids. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://etd.library.vanderbilt.edu/available/etd-02042016-121346/ ;


Vanderbilt University

5. Muller, Christopher Louis. Dissecting the Role of the Serotonin Transporter in the Developmental and Neurobehavioral Features of Autism Spectrum Disorder.

Degree: PhD, Neuroscience, 2015, Vanderbilt University

 Approximately 25% of individuals with autism spectrum disorder (ASD) possess elevated whole blood serotonin (5-HT) levels, termed hyperserotonemia. However, the connection between this biomarker and… (more)

Subjects/Keywords: serotonin transporter; autism spectrum disorder; mouse model; neurodevelopment

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APA (6th Edition):

Muller, C. L. (2015). Dissecting the Role of the Serotonin Transporter in the Developmental and Neurobehavioral Features of Autism Spectrum Disorder. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-03122015-212404/ ;

Chicago Manual of Style (16th Edition):

Muller, Christopher Louis. “Dissecting the Role of the Serotonin Transporter in the Developmental and Neurobehavioral Features of Autism Spectrum Disorder.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed March 31, 2020. http://etd.library.vanderbilt.edu/available/etd-03122015-212404/ ;.

MLA Handbook (7th Edition):

Muller, Christopher Louis. “Dissecting the Role of the Serotonin Transporter in the Developmental and Neurobehavioral Features of Autism Spectrum Disorder.” 2015. Web. 31 Mar 2020.

Vancouver:

Muller CL. Dissecting the Role of the Serotonin Transporter in the Developmental and Neurobehavioral Features of Autism Spectrum Disorder. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2020 Mar 31]. Available from: http://etd.library.vanderbilt.edu/available/etd-03122015-212404/ ;.

Council of Science Editors:

Muller CL. Dissecting the Role of the Serotonin Transporter in the Developmental and Neurobehavioral Features of Autism Spectrum Disorder. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://etd.library.vanderbilt.edu/available/etd-03122015-212404/ ;


Vanderbilt University

6. O'Neil, Richard Thomas. Exploring the dynamics of A-to-I editing in the mammalian brain.

Degree: PhD, Neuroscience, 2015, Vanderbilt University

 Adenosine-to-Inosine RNA editing (A-to-I editing) has evolved as an important process for generating transcript diversity in metazoans. Several editing events which effect transcripts encoding proteins… (more)

Subjects/Keywords: RNA; Inosine

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APA (6th Edition):

O'Neil, R. T. (2015). Exploring the dynamics of A-to-I editing in the mammalian brain. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu//available/etd-08112015-104456/ ;

Chicago Manual of Style (16th Edition):

O'Neil, Richard Thomas. “Exploring the dynamics of A-to-I editing in the mammalian brain.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed March 31, 2020. http://etd.library.vanderbilt.edu//available/etd-08112015-104456/ ;.

MLA Handbook (7th Edition):

O'Neil, Richard Thomas. “Exploring the dynamics of A-to-I editing in the mammalian brain.” 2015. Web. 31 Mar 2020.

Vancouver:

O'Neil RT. Exploring the dynamics of A-to-I editing in the mammalian brain. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2020 Mar 31]. Available from: http://etd.library.vanderbilt.edu//available/etd-08112015-104456/ ;.

Council of Science Editors:

O'Neil RT. Exploring the dynamics of A-to-I editing in the mammalian brain. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://etd.library.vanderbilt.edu//available/etd-08112015-104456/ ;

7. Zike, Isaac Daniel. Investigating the Role of Obsessive-Compulsive Disorder Candidate Gene SLC1A1 in Basal Ganglia and Repetitive Behavior.

Degree: PhD, Pharmacology, 2017, Vanderbilt University

 Obsessive-compulsive disorder (OCD) is a chronic, disabling condition with inadequate treatment options that leave most patients with substantial residual symptoms. Structural, neurochemical, and behavioral findings… (more)

Subjects/Keywords: obsessive; compulsive; glutamate; neuroscience

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APA (6th Edition):

Zike, I. D. (2017). Investigating the Role of Obsessive-Compulsive Disorder Candidate Gene SLC1A1 in Basal Ganglia and Repetitive Behavior. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-05232017-122303/ ;

Chicago Manual of Style (16th Edition):

Zike, Isaac Daniel. “Investigating the Role of Obsessive-Compulsive Disorder Candidate Gene SLC1A1 in Basal Ganglia and Repetitive Behavior.” 2017. Doctoral Dissertation, Vanderbilt University. Accessed March 31, 2020. http://etd.library.vanderbilt.edu/available/etd-05232017-122303/ ;.

MLA Handbook (7th Edition):

Zike, Isaac Daniel. “Investigating the Role of Obsessive-Compulsive Disorder Candidate Gene SLC1A1 in Basal Ganglia and Repetitive Behavior.” 2017. Web. 31 Mar 2020.

Vancouver:

Zike ID. Investigating the Role of Obsessive-Compulsive Disorder Candidate Gene SLC1A1 in Basal Ganglia and Repetitive Behavior. [Internet] [Doctoral dissertation]. Vanderbilt University; 2017. [cited 2020 Mar 31]. Available from: http://etd.library.vanderbilt.edu/available/etd-05232017-122303/ ;.

Council of Science Editors:

Zike ID. Investigating the Role of Obsessive-Compulsive Disorder Candidate Gene SLC1A1 in Basal Ganglia and Repetitive Behavior. [Doctoral Dissertation]. Vanderbilt University; 2017. Available from: http://etd.library.vanderbilt.edu/available/etd-05232017-122303/ ;


Vanderbilt University

8. Siuta, Michael Andrew. Prefrontal and Striatal Catecholamine Dysfunction in the Neuronal Rictor Null.

Degree: PhD, Neuroscience, 2013, Vanderbilt University

 Disruption in insulin signaling is a mechanism hypothesized to underlie many neurologic and psychiatric disorders. Insulin signaling is accomplished by a complex array of intracellular… (more)

Subjects/Keywords: Akt; dopamine; norepinephrine; Rictor; mTOR; transporters; schizophrenia

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APA (6th Edition):

Siuta, M. A. (2013). Prefrontal and Striatal Catecholamine Dysfunction in the Neuronal Rictor Null. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-09162013-100352/ ;

Chicago Manual of Style (16th Edition):

Siuta, Michael Andrew. “Prefrontal and Striatal Catecholamine Dysfunction in the Neuronal Rictor Null.” 2013. Doctoral Dissertation, Vanderbilt University. Accessed March 31, 2020. http://etd.library.vanderbilt.edu/available/etd-09162013-100352/ ;.

MLA Handbook (7th Edition):

Siuta, Michael Andrew. “Prefrontal and Striatal Catecholamine Dysfunction in the Neuronal Rictor Null.” 2013. Web. 31 Mar 2020.

Vancouver:

Siuta MA. Prefrontal and Striatal Catecholamine Dysfunction in the Neuronal Rictor Null. [Internet] [Doctoral dissertation]. Vanderbilt University; 2013. [cited 2020 Mar 31]. Available from: http://etd.library.vanderbilt.edu/available/etd-09162013-100352/ ;.

Council of Science Editors:

Siuta MA. Prefrontal and Striatal Catecholamine Dysfunction in the Neuronal Rictor Null. [Doctoral Dissertation]. Vanderbilt University; 2013. Available from: http://etd.library.vanderbilt.edu/available/etd-09162013-100352/ ;


Vanderbilt University

9. Ho, Karen W. The Transient Receptor Potential Vanilloid-1 Channel in Stress-Induced Astrocyte Migration.

Degree: PhD, Pharmacology, 2014, Vanderbilt University

 Astrocytes are one type of glia in the central nervous system, and they provide metabolic and structural support to neurons. Following injury astrocytes undergo an… (more)

Subjects/Keywords: astrocyte; migration; TRPV1

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APA (6th Edition):

Ho, K. W. (2014). The Transient Receptor Potential Vanilloid-1 Channel in Stress-Induced Astrocyte Migration. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-10132014-160147/ ;

Chicago Manual of Style (16th Edition):

Ho, Karen W. “The Transient Receptor Potential Vanilloid-1 Channel in Stress-Induced Astrocyte Migration.” 2014. Doctoral Dissertation, Vanderbilt University. Accessed March 31, 2020. http://etd.library.vanderbilt.edu/available/etd-10132014-160147/ ;.

MLA Handbook (7th Edition):

Ho, Karen W. “The Transient Receptor Potential Vanilloid-1 Channel in Stress-Induced Astrocyte Migration.” 2014. Web. 31 Mar 2020.

Vancouver:

Ho KW. The Transient Receptor Potential Vanilloid-1 Channel in Stress-Induced Astrocyte Migration. [Internet] [Doctoral dissertation]. Vanderbilt University; 2014. [cited 2020 Mar 31]. Available from: http://etd.library.vanderbilt.edu/available/etd-10132014-160147/ ;.

Council of Science Editors:

Ho KW. The Transient Receptor Potential Vanilloid-1 Channel in Stress-Induced Astrocyte Migration. [Doctoral Dissertation]. Vanderbilt University; 2014. Available from: http://etd.library.vanderbilt.edu/available/etd-10132014-160147/ ;


Vanderbilt University

10. Grier, Mark Donald. Unraveling the Gene/Environment Knot in Neurodevelopmental Disease: Focus on Angelman Syndrome.

Degree: PhD, Interdisciplinary Studies: Developmental Neuropharmacology, 2015, Vanderbilt University

 Angelman Syndrome (AS) is a devastating neurodevelopmental disorder characterized by developmental delay, speech impairment, movement disorder, sleep disorders and refractory epilepsy. AS is caused by… (more)

Subjects/Keywords: Maternal Effects; Environmental Influences; Epigentics

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APA (6th Edition):

Grier, M. D. (2015). Unraveling the Gene/Environment Knot in Neurodevelopmental Disease: Focus on Angelman Syndrome. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-06012015-114009/ ;

Chicago Manual of Style (16th Edition):

Grier, Mark Donald. “Unraveling the Gene/Environment Knot in Neurodevelopmental Disease: Focus on Angelman Syndrome.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed March 31, 2020. http://etd.library.vanderbilt.edu/available/etd-06012015-114009/ ;.

MLA Handbook (7th Edition):

Grier, Mark Donald. “Unraveling the Gene/Environment Knot in Neurodevelopmental Disease: Focus on Angelman Syndrome.” 2015. Web. 31 Mar 2020.

Vancouver:

Grier MD. Unraveling the Gene/Environment Knot in Neurodevelopmental Disease: Focus on Angelman Syndrome. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2020 Mar 31]. Available from: http://etd.library.vanderbilt.edu/available/etd-06012015-114009/ ;.

Council of Science Editors:

Grier MD. Unraveling the Gene/Environment Knot in Neurodevelopmental Disease: Focus on Angelman Syndrome. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://etd.library.vanderbilt.edu/available/etd-06012015-114009/ ;


Vanderbilt University

11. Klar, Rebecca. The Role of mGlu7 in Hippocampal Synaptic Plasticity: Implications for Novel Therapeutics for Rett Syndrome.

Degree: PhD, Pharmacology, 2015, Vanderbilt University

 Of the eight metabotropic glutamate (mGlu) receptor subtypes, only metabotropic glutamate receptor 7 (mGlu7) is expressed presynaptically at the Schaffer Collateral (SC)-CA1 synapse in the… (more)

Subjects/Keywords: Synaptic Plasticity; mGluR7; Autism

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APA (6th Edition):

Klar, R. (2015). The Role of mGlu7 in Hippocampal Synaptic Plasticity: Implications for Novel Therapeutics for Rett Syndrome. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu//available/etd-08052015-080100/ ;

Chicago Manual of Style (16th Edition):

Klar, Rebecca. “The Role of mGlu7 in Hippocampal Synaptic Plasticity: Implications for Novel Therapeutics for Rett Syndrome.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed March 31, 2020. http://etd.library.vanderbilt.edu//available/etd-08052015-080100/ ;.

MLA Handbook (7th Edition):

Klar, Rebecca. “The Role of mGlu7 in Hippocampal Synaptic Plasticity: Implications for Novel Therapeutics for Rett Syndrome.” 2015. Web. 31 Mar 2020.

Vancouver:

Klar R. The Role of mGlu7 in Hippocampal Synaptic Plasticity: Implications for Novel Therapeutics for Rett Syndrome. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2020 Mar 31]. Available from: http://etd.library.vanderbilt.edu//available/etd-08052015-080100/ ;.

Council of Science Editors:

Klar R. The Role of mGlu7 in Hippocampal Synaptic Plasticity: Implications for Novel Therapeutics for Rett Syndrome. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://etd.library.vanderbilt.edu//available/etd-08052015-080100/ ;


Vanderbilt University

12. Money, Kelli Marie. Inflammation and accumulating risk for developing psychiatric disorders.

Degree: PhD, Neuroscience, 2016, Vanderbilt University

 The immune system and brain are intricately intertwined in both development and adult function. Unfortunately, this relationship can lead to dysfunction when balance in either… (more)

Subjects/Keywords: gestational diabetes; depression; inflammation; maternal infection

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APA (6th Edition):

Money, K. M. (2016). Inflammation and accumulating risk for developing psychiatric disorders. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu//available/etd-08222016-130417/ ;

Chicago Manual of Style (16th Edition):

Money, Kelli Marie. “Inflammation and accumulating risk for developing psychiatric disorders.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed March 31, 2020. http://etd.library.vanderbilt.edu//available/etd-08222016-130417/ ;.

MLA Handbook (7th Edition):

Money, Kelli Marie. “Inflammation and accumulating risk for developing psychiatric disorders.” 2016. Web. 31 Mar 2020.

Vancouver:

Money KM. Inflammation and accumulating risk for developing psychiatric disorders. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2020 Mar 31]. Available from: http://etd.library.vanderbilt.edu//available/etd-08222016-130417/ ;.

Council of Science Editors:

Money KM. Inflammation and accumulating risk for developing psychiatric disorders. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://etd.library.vanderbilt.edu//available/etd-08222016-130417/ ;


Vanderbilt University

13. Nackenoff, Alexander Greer. Determining the Role of Serotonin in Antidepressant Action, as Revealed Utilizing the SERT Met172 Mouse Model.

Degree: PhD, Pharmacology, 2016, Vanderbilt University

 The most widely prescribed antidepressant medications are serotonin selective reuptake inhibitors (SSRIs), believed to provide therapeutic benefit by antagonizing the serotonin transporter (5-HT; SERT), which… (more)

Subjects/Keywords: SERT; Depression; I172M; SERT Met172; ssri; serotonin; antidepressant; serotonin

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APA (6th Edition):

Nackenoff, A. G. (2016). Determining the Role of Serotonin in Antidepressant Action, as Revealed Utilizing the SERT Met172 Mouse Model. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu//available/etd-03252016-100138/ ;

Chicago Manual of Style (16th Edition):

Nackenoff, Alexander Greer. “Determining the Role of Serotonin in Antidepressant Action, as Revealed Utilizing the SERT Met172 Mouse Model.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed March 31, 2020. http://etd.library.vanderbilt.edu//available/etd-03252016-100138/ ;.

MLA Handbook (7th Edition):

Nackenoff, Alexander Greer. “Determining the Role of Serotonin in Antidepressant Action, as Revealed Utilizing the SERT Met172 Mouse Model.” 2016. Web. 31 Mar 2020.

Vancouver:

Nackenoff AG. Determining the Role of Serotonin in Antidepressant Action, as Revealed Utilizing the SERT Met172 Mouse Model. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2020 Mar 31]. Available from: http://etd.library.vanderbilt.edu//available/etd-03252016-100138/ ;.

Council of Science Editors:

Nackenoff AG. Determining the Role of Serotonin in Antidepressant Action, as Revealed Utilizing the SERT Met172 Mouse Model. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://etd.library.vanderbilt.edu//available/etd-03252016-100138/ ;

14. Green, Noah Hammond. Perinatal Photoperiod Affects the Serotonergic System and Affective Behaviors.

Degree: PhD, Biological Sciences, 2015, Vanderbilt University

 The serotonergic raphe nuclei of the midbrain are principal centers from which serotonin neurons project to innervate cortical and sub-cortical structures. The dorsal raphe nuclei… (more)

Subjects/Keywords: Serotonin; Melatonin; Circadian Rhythms; Photoperiod

Vanderbilt University. During my first year while I was still rotating through labs in the… …Photoperiod Paradigm. 13 performed in accordance with the Vanderbilt University Institutional… …Douglas G. McMahon1,2 1 – Department of Biological Sciences, Vanderbilt University, Nashville… …TN 37235 2 – Neuroscience Graduate Program, Vanderbilt University School of Medicine… …Nashville, Tennessee 37235. 3 – Department of Pharmacology, Vanderbilt University School of… 

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APA (6th Edition):

Green, N. H. (2015). Perinatal Photoperiod Affects the Serotonergic System and Affective Behaviors. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-06292015-154010/ ;

Chicago Manual of Style (16th Edition):

Green, Noah Hammond. “Perinatal Photoperiod Affects the Serotonergic System and Affective Behaviors.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed March 31, 2020. http://etd.library.vanderbilt.edu/available/etd-06292015-154010/ ;.

MLA Handbook (7th Edition):

Green, Noah Hammond. “Perinatal Photoperiod Affects the Serotonergic System and Affective Behaviors.” 2015. Web. 31 Mar 2020.

Vancouver:

Green NH. Perinatal Photoperiod Affects the Serotonergic System and Affective Behaviors. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2020 Mar 31]. Available from: http://etd.library.vanderbilt.edu/available/etd-06292015-154010/ ;.

Council of Science Editors:

Green NH. Perinatal Photoperiod Affects the Serotonergic System and Affective Behaviors. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://etd.library.vanderbilt.edu/available/etd-06292015-154010/ ;

15. Schmidt, Martin Jefferson. Differential impact of GAD67 suppression in distinct GABA-ergic interneuron populations: implications for mental illness.

Degree: PhD, Neuroscience, 2014, Vanderbilt University

 GABA-ergic disturbances are hallmark features of many brain disorders. Two transgenic mouse lines were generated to suppress GAD1 in non-overlapping cell types that express cholecystokinin… (more)

Subjects/Keywords: CCK; NPY; GAD1; schizophrenia; GABA; amphetamine

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Schmidt, M. J. (2014). Differential impact of GAD67 suppression in distinct GABA-ergic interneuron populations: implications for mental illness. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-12022013-064125/ ;

Chicago Manual of Style (16th Edition):

Schmidt, Martin Jefferson. “Differential impact of GAD67 suppression in distinct GABA-ergic interneuron populations: implications for mental illness.” 2014. Doctoral Dissertation, Vanderbilt University. Accessed March 31, 2020. http://etd.library.vanderbilt.edu/available/etd-12022013-064125/ ;.

MLA Handbook (7th Edition):

Schmidt, Martin Jefferson. “Differential impact of GAD67 suppression in distinct GABA-ergic interneuron populations: implications for mental illness.” 2014. Web. 31 Mar 2020.

Vancouver:

Schmidt MJ. Differential impact of GAD67 suppression in distinct GABA-ergic interneuron populations: implications for mental illness. [Internet] [Doctoral dissertation]. Vanderbilt University; 2014. [cited 2020 Mar 31]. Available from: http://etd.library.vanderbilt.edu/available/etd-12022013-064125/ ;.

Council of Science Editors:

Schmidt MJ. Differential impact of GAD67 suppression in distinct GABA-ergic interneuron populations: implications for mental illness. [Doctoral Dissertation]. Vanderbilt University; 2014. Available from: http://etd.library.vanderbilt.edu/available/etd-12022013-064125/ ;

16. Frederick, Aliya Latisha. Genetic dissection of the behavioral effects of the dopamine receptor agonist SKF83959 does not support the D1/D2 receptor heteromer model.

Degree: PhD, Neuroscience, 2013, Vanderbilt University

 SKF83959 is a high affinity dopamine D1 receptor agonist that has been reported to preferentially activate D1 receptors coupled to G(alpha)q. This pathway results in… (more)

Subjects/Keywords: Dopamine receptors; Behavior; Agonist

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Frederick, A. L. (2013). Genetic dissection of the behavioral effects of the dopamine receptor agonist SKF83959 does not support the D1/D2 receptor heteromer model. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-04112013-203459/ ;

Chicago Manual of Style (16th Edition):

Frederick, Aliya Latisha. “Genetic dissection of the behavioral effects of the dopamine receptor agonist SKF83959 does not support the D1/D2 receptor heteromer model.” 2013. Doctoral Dissertation, Vanderbilt University. Accessed March 31, 2020. http://etd.library.vanderbilt.edu/available/etd-04112013-203459/ ;.

MLA Handbook (7th Edition):

Frederick, Aliya Latisha. “Genetic dissection of the behavioral effects of the dopamine receptor agonist SKF83959 does not support the D1/D2 receptor heteromer model.” 2013. Web. 31 Mar 2020.

Vancouver:

Frederick AL. Genetic dissection of the behavioral effects of the dopamine receptor agonist SKF83959 does not support the D1/D2 receptor heteromer model. [Internet] [Doctoral dissertation]. Vanderbilt University; 2013. [cited 2020 Mar 31]. Available from: http://etd.library.vanderbilt.edu/available/etd-04112013-203459/ ;.

Council of Science Editors:

Frederick AL. Genetic dissection of the behavioral effects of the dopamine receptor agonist SKF83959 does not support the D1/D2 receptor heteromer model. [Doctoral Dissertation]. Vanderbilt University; 2013. Available from: http://etd.library.vanderbilt.edu/available/etd-04112013-203459/ ;


Vanderbilt University

17. Davis, Adeola Ronke. Role of alpha2A-adrenergic receptors in extinction of positive and negative valence learned behaviors.

Degree: PhD, Neuroscience, 2009, Vanderbilt University

 NEUROSCIENCE ROLE OF α2A ADRENERGIC RECEPTORS IN EXTINCTION OF POSITIVE AND NEGATIVE VALENCE LEARNED BEHAVIORS Adeola R. Davis Dissertation under the direction of Professor Danny… (more)

Subjects/Keywords: mice; adrenergic receptor; yohimbine; conditioned place aversion; conditioned place preference; extinction

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Davis, A. R. (2009). Role of alpha2A-adrenergic receptors in extinction of positive and negative valence learned behaviors. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-06242009-204012/ ;

Chicago Manual of Style (16th Edition):

Davis, Adeola Ronke. “Role of alpha2A-adrenergic receptors in extinction of positive and negative valence learned behaviors.” 2009. Doctoral Dissertation, Vanderbilt University. Accessed March 31, 2020. http://etd.library.vanderbilt.edu/available/etd-06242009-204012/ ;.

MLA Handbook (7th Edition):

Davis, Adeola Ronke. “Role of alpha2A-adrenergic receptors in extinction of positive and negative valence learned behaviors.” 2009. Web. 31 Mar 2020.

Vancouver:

Davis AR. Role of alpha2A-adrenergic receptors in extinction of positive and negative valence learned behaviors. [Internet] [Doctoral dissertation]. Vanderbilt University; 2009. [cited 2020 Mar 31]. Available from: http://etd.library.vanderbilt.edu/available/etd-06242009-204012/ ;.

Council of Science Editors:

Davis AR. Role of alpha2A-adrenergic receptors in extinction of positive and negative valence learned behaviors. [Doctoral Dissertation]. Vanderbilt University; 2009. Available from: http://etd.library.vanderbilt.edu/available/etd-06242009-204012/ ;

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