Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for +publisher:"Vanderbilt University" +contributor:("Douglas P Mortlock"). Showing records 1 – 14 of 14 total matches.

Search Limiters

Last 2 Years | English Only

No search limiters apply to these results.

▼ Search Limiters


Vanderbilt University

1. Broeckelmann, Eva Marie. Dynamics of long-range gene regulation at the BMP2 locus.

Degree: MS, Interdisciplinary Studies: Human Genetics, 2013, Vanderbilt University

 A member of the TGF-â superfamily of cytokines, BMP2 not only plays a critical role in pattern formation and morphogenesis during early embryonic development, but… (more)

Subjects/Keywords: gene regulation; Bmp2

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Broeckelmann, E. M. (2013). Dynamics of long-range gene regulation at the BMP2 locus. (Thesis). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12090

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Broeckelmann, Eva Marie. “Dynamics of long-range gene regulation at the BMP2 locus.” 2013. Thesis, Vanderbilt University. Accessed March 07, 2021. http://hdl.handle.net/1803/12090.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Broeckelmann, Eva Marie. “Dynamics of long-range gene regulation at the BMP2 locus.” 2013. Web. 07 Mar 2021.

Vancouver:

Broeckelmann EM. Dynamics of long-range gene regulation at the BMP2 locus. [Internet] [Thesis]. Vanderbilt University; 2013. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1803/12090.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Broeckelmann EM. Dynamics of long-range gene regulation at the BMP2 locus. [Thesis]. Vanderbilt University; 2013. Available from: http://hdl.handle.net/1803/12090

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

2. Kodaman, Nuri. The Genetics of Cardiovascular Risk Factor Correlations.

Degree: PhD, Human Genetics, 2015, Vanderbilt University

 Cardiovascular disease (CVD) is the leading cause of death worldwide. The vast possibilities of interaction between genetic and environmental factors that contribute to CVD can… (more)

Subjects/Keywords: genetic epidemiology; epidemiology; GXE interaction; t-PA; West Africa; fibrinolysis; cardiovascular disease risk factors; method development; ordinal regression

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kodaman, N. (2015). The Genetics of Cardiovascular Risk Factor Correlations. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/15219

Chicago Manual of Style (16th Edition):

Kodaman, Nuri. “The Genetics of Cardiovascular Risk Factor Correlations.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed March 07, 2021. http://hdl.handle.net/1803/15219.

MLA Handbook (7th Edition):

Kodaman, Nuri. “The Genetics of Cardiovascular Risk Factor Correlations.” 2015. Web. 07 Mar 2021.

Vancouver:

Kodaman N. The Genetics of Cardiovascular Risk Factor Correlations. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1803/15219.

Council of Science Editors:

Kodaman N. The Genetics of Cardiovascular Risk Factor Correlations. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://hdl.handle.net/1803/15219


Vanderbilt University

3. Clendenning, Dawn Elizabeth. Determining the role of Growth Differentiation Factor-6 (Gdf6) in the development of the coronal suture.

Degree: PhD, Human Genetics, 2012, Vanderbilt University

 HUMAN GENETICS DETERMINING THE ROLE OF GROWTH DIFFERENTIATION FACTOR-6 (GDF6) IN THE DEVELOPMENT OF THE CORONAL SUTURE DAWN ELIZABETH CLENDENNING Dissertation under the direction of… (more)

Subjects/Keywords: Bone Morphogenetic Proteins; skeletal development; craniosynostosis; cranial sutures; Gdf6

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Clendenning, D. E. (2012). Determining the role of Growth Differentiation Factor-6 (Gdf6) in the development of the coronal suture. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12174

Chicago Manual of Style (16th Edition):

Clendenning, Dawn Elizabeth. “Determining the role of Growth Differentiation Factor-6 (Gdf6) in the development of the coronal suture.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed March 07, 2021. http://hdl.handle.net/1803/12174.

MLA Handbook (7th Edition):

Clendenning, Dawn Elizabeth. “Determining the role of Growth Differentiation Factor-6 (Gdf6) in the development of the coronal suture.” 2012. Web. 07 Mar 2021.

Vancouver:

Clendenning DE. Determining the role of Growth Differentiation Factor-6 (Gdf6) in the development of the coronal suture. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1803/12174.

Council of Science Editors:

Clendenning DE. Determining the role of Growth Differentiation Factor-6 (Gdf6) in the development of the coronal suture. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://hdl.handle.net/1803/12174


Vanderbilt University

4. Wang, Weiguang. Function of Atf4 in Endochondral Bone Formation.

Degree: PhD, Pharmacology, 2011, Vanderbilt University

 Activating transcription factor 4 (Atf4) is a leucine zip transcription factor. Atf4 mutant (Atf4-/-) mice show severe low bone mass and short stature phenotype. Atf4… (more)

Subjects/Keywords: chondrocyte; Ihh; Endochondral ossification; Atf4; osteoblast

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wang, W. (2011). Function of Atf4 in Endochondral Bone Formation. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12726

Chicago Manual of Style (16th Edition):

Wang, Weiguang. “Function of Atf4 in Endochondral Bone Formation.” 2011. Doctoral Dissertation, Vanderbilt University. Accessed March 07, 2021. http://hdl.handle.net/1803/12726.

MLA Handbook (7th Edition):

Wang, Weiguang. “Function of Atf4 in Endochondral Bone Formation.” 2011. Web. 07 Mar 2021.

Vancouver:

Wang W. Function of Atf4 in Endochondral Bone Formation. [Internet] [Doctoral dissertation]. Vanderbilt University; 2011. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1803/12726.

Council of Science Editors:

Wang W. Function of Atf4 in Endochondral Bone Formation. [Doctoral Dissertation]. Vanderbilt University; 2011. Available from: http://hdl.handle.net/1803/12726


Vanderbilt University

5. Fish, Alexandra Elizabeth. Leveraging gene expression and local ancestry to investigate regulatory epistasis in humans.

Degree: PhD, Human Genetics, 2017, Vanderbilt University

 Epistasis is a phenomenon wherein the effect of a genetic variant on a phenotype is dependent on the genomic context. Better understanding epistastic relationships between… (more)

Subjects/Keywords: epistasis; gene expression; admixed populations; eQTL; PheWAS

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fish, A. E. (2017). Leveraging gene expression and local ancestry to investigate regulatory epistasis in humans. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11250

Chicago Manual of Style (16th Edition):

Fish, Alexandra Elizabeth. “Leveraging gene expression and local ancestry to investigate regulatory epistasis in humans.” 2017. Doctoral Dissertation, Vanderbilt University. Accessed March 07, 2021. http://hdl.handle.net/1803/11250.

MLA Handbook (7th Edition):

Fish, Alexandra Elizabeth. “Leveraging gene expression and local ancestry to investigate regulatory epistasis in humans.” 2017. Web. 07 Mar 2021.

Vancouver:

Fish AE. Leveraging gene expression and local ancestry to investigate regulatory epistasis in humans. [Internet] [Doctoral dissertation]. Vanderbilt University; 2017. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1803/11250.

Council of Science Editors:

Fish AE. Leveraging gene expression and local ancestry to investigate regulatory epistasis in humans. [Doctoral Dissertation]. Vanderbilt University; 2017. Available from: http://hdl.handle.net/1803/11250


Vanderbilt University

6. Veatch, Olivia Jean. Identifying biological pathways implicated in defined subgroups of phenotypic expression for Autism Spectrum Disorders and evaluating small molecule effects on expression of ASMT.

Degree: PhD, Human Genetics, 2013, Vanderbilt University

 Autism Spectrum Disorder is a neurodevelopmental condition with evidence for genetic susceptibility. However, effect sizes for implicated loci are small, and current evidence does not… (more)

Subjects/Keywords: Autism Spectrum Disorder; Human Genetics; Multivariate Statistics; Pharmacogenetics; Phenotyping; Ne

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Veatch, O. J. (2013). Identifying biological pathways implicated in defined subgroups of phenotypic expression for Autism Spectrum Disorders and evaluating small molecule effects on expression of ASMT. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/15041

Chicago Manual of Style (16th Edition):

Veatch, Olivia Jean. “Identifying biological pathways implicated in defined subgroups of phenotypic expression for Autism Spectrum Disorders and evaluating small molecule effects on expression of ASMT.” 2013. Doctoral Dissertation, Vanderbilt University. Accessed March 07, 2021. http://hdl.handle.net/1803/15041.

MLA Handbook (7th Edition):

Veatch, Olivia Jean. “Identifying biological pathways implicated in defined subgroups of phenotypic expression for Autism Spectrum Disorders and evaluating small molecule effects on expression of ASMT.” 2013. Web. 07 Mar 2021.

Vancouver:

Veatch OJ. Identifying biological pathways implicated in defined subgroups of phenotypic expression for Autism Spectrum Disorders and evaluating small molecule effects on expression of ASMT. [Internet] [Doctoral dissertation]. Vanderbilt University; 2013. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1803/15041.

Council of Science Editors:

Veatch OJ. Identifying biological pathways implicated in defined subgroups of phenotypic expression for Autism Spectrum Disorders and evaluating small molecule effects on expression of ASMT. [Doctoral Dissertation]. Vanderbilt University; 2013. Available from: http://hdl.handle.net/1803/15041


Vanderbilt University

7. Jorge, Benjamin S. Genetic Variation in the Voltage-gated Potassium Channel Genes KCNV2 and KCNB1 Contributes to Epilepsy Susceptibility.

Degree: PhD, Neuroscience, 2014, Vanderbilt University

 Epilepsy is a common neurological disease characterized by an enduring predisposition to generate seizures. Although multiple factors contribute to epilepsy, the majority of cases are… (more)

Subjects/Keywords: potassium channel; epileptic encephalopathy; mouse model; genetics; whole-exome sequencing; epilepsy

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jorge, B. S. (2014). Genetic Variation in the Voltage-gated Potassium Channel Genes KCNV2 and KCNB1 Contributes to Epilepsy Susceptibility. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14387

Chicago Manual of Style (16th Edition):

Jorge, Benjamin S. “Genetic Variation in the Voltage-gated Potassium Channel Genes KCNV2 and KCNB1 Contributes to Epilepsy Susceptibility.” 2014. Doctoral Dissertation, Vanderbilt University. Accessed March 07, 2021. http://hdl.handle.net/1803/14387.

MLA Handbook (7th Edition):

Jorge, Benjamin S. “Genetic Variation in the Voltage-gated Potassium Channel Genes KCNV2 and KCNB1 Contributes to Epilepsy Susceptibility.” 2014. Web. 07 Mar 2021.

Vancouver:

Jorge BS. Genetic Variation in the Voltage-gated Potassium Channel Genes KCNV2 and KCNB1 Contributes to Epilepsy Susceptibility. [Internet] [Doctoral dissertation]. Vanderbilt University; 2014. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1803/14387.

Council of Science Editors:

Jorge BS. Genetic Variation in the Voltage-gated Potassium Channel Genes KCNV2 and KCNB1 Contributes to Epilepsy Susceptibility. [Doctoral Dissertation]. Vanderbilt University; 2014. Available from: http://hdl.handle.net/1803/14387


Vanderbilt University

8. Levinson, Rebecca Terrall. The Phenotypic Consequences of Distinct Genetic Variation in an Electronic Medical Record.

Degree: PhD, Human Genetics, 2016, Vanderbilt University

 While genetic association studies have been able to elucidate the importance of genetics in human disease outcomes, these studies are limited by the necessity of… (more)

Subjects/Keywords: PheWAS

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Levinson, R. T. (2016). The Phenotypic Consequences of Distinct Genetic Variation in an Electronic Medical Record. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14698

Chicago Manual of Style (16th Edition):

Levinson, Rebecca Terrall. “The Phenotypic Consequences of Distinct Genetic Variation in an Electronic Medical Record.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed March 07, 2021. http://hdl.handle.net/1803/14698.

MLA Handbook (7th Edition):

Levinson, Rebecca Terrall. “The Phenotypic Consequences of Distinct Genetic Variation in an Electronic Medical Record.” 2016. Web. 07 Mar 2021.

Vancouver:

Levinson RT. The Phenotypic Consequences of Distinct Genetic Variation in an Electronic Medical Record. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1803/14698.

Council of Science Editors:

Levinson RT. The Phenotypic Consequences of Distinct Genetic Variation in an Electronic Medical Record. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://hdl.handle.net/1803/14698


Vanderbilt University

9. Bush, William Scott. A Knowledge-Driven Multi-Locus Analysis of Multiple Sclerosis Susceptiblity.

Degree: PhD, Human Genetics, 2009, Vanderbilt University

 Evaluating epistasis in whole-genome association studies is an important challenge in human genetics, as many common diseases are thought to have complex underlying genetic architectures… (more)

Subjects/Keywords: Genomics  – Methodology; Inositol  – Pathophysiology; Epistasis (Genetics); Multiple sclerosis  – Genetic aspects; disease gene discovery; knowledge-based analysis; Disease susceptibility  – Genetic aspects  – Data processing

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bush, W. S. (2009). A Knowledge-Driven Multi-Locus Analysis of Multiple Sclerosis Susceptiblity. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10678

Chicago Manual of Style (16th Edition):

Bush, William Scott. “A Knowledge-Driven Multi-Locus Analysis of Multiple Sclerosis Susceptiblity.” 2009. Doctoral Dissertation, Vanderbilt University. Accessed March 07, 2021. http://hdl.handle.net/1803/10678.

MLA Handbook (7th Edition):

Bush, William Scott. “A Knowledge-Driven Multi-Locus Analysis of Multiple Sclerosis Susceptiblity.” 2009. Web. 07 Mar 2021.

Vancouver:

Bush WS. A Knowledge-Driven Multi-Locus Analysis of Multiple Sclerosis Susceptiblity. [Internet] [Doctoral dissertation]. Vanderbilt University; 2009. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1803/10678.

Council of Science Editors:

Bush WS. A Knowledge-Driven Multi-Locus Analysis of Multiple Sclerosis Susceptiblity. [Doctoral Dissertation]. Vanderbilt University; 2009. Available from: http://hdl.handle.net/1803/10678


Vanderbilt University

10. Liang, Xueying. Investigation of genetic susceptibility to late-onset Alzheimer disease through genomic convergence.

Degree: PhD, Human Genetics, 2007, Vanderbilt University

 With the exception of ApoE gene, no universally accepted genetic association has been identified with the complex Late-onset Alzheimer Disease (LOAD). A broad region of… (more)

Subjects/Keywords: Alzheimer Disease; gene-gene interaction; genomic convergence; association; candidate gene; chromosome 10; linkage; SNP; genetics; Alzheimer's disease  – Susceptibility

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Liang, X. (2007). Investigation of genetic susceptibility to late-onset Alzheimer disease through genomic convergence. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11992

Chicago Manual of Style (16th Edition):

Liang, Xueying. “Investigation of genetic susceptibility to late-onset Alzheimer disease through genomic convergence.” 2007. Doctoral Dissertation, Vanderbilt University. Accessed March 07, 2021. http://hdl.handle.net/1803/11992.

MLA Handbook (7th Edition):

Liang, Xueying. “Investigation of genetic susceptibility to late-onset Alzheimer disease through genomic convergence.” 2007. Web. 07 Mar 2021.

Vancouver:

Liang X. Investigation of genetic susceptibility to late-onset Alzheimer disease through genomic convergence. [Internet] [Doctoral dissertation]. Vanderbilt University; 2007. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1803/11992.

Council of Science Editors:

Liang X. Investigation of genetic susceptibility to late-onset Alzheimer disease through genomic convergence. [Doctoral Dissertation]. Vanderbilt University; 2007. Available from: http://hdl.handle.net/1803/11992


Vanderbilt University

11. Spencer, Kylee L. Variants in complement factor pathway genes and a novel locus on chromosome 16p12 influence age-related macular degeneration susceptibility.

Degree: PhD, Human Genetics, 2007, Vanderbilt University

 Age-related macular degeneration (AMD) is a complex, late-onset disease that is the leading cause of blindness in the elderly. Age, smoking, and variants in complement… (more)

Subjects/Keywords: complex genetic disease

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Spencer, K. L. (2007). Variants in complement factor pathway genes and a novel locus on chromosome 16p12 influence age-related macular degeneration susceptibility. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14024

Chicago Manual of Style (16th Edition):

Spencer, Kylee L. “Variants in complement factor pathway genes and a novel locus on chromosome 16p12 influence age-related macular degeneration susceptibility.” 2007. Doctoral Dissertation, Vanderbilt University. Accessed March 07, 2021. http://hdl.handle.net/1803/14024.

MLA Handbook (7th Edition):

Spencer, Kylee L. “Variants in complement factor pathway genes and a novel locus on chromosome 16p12 influence age-related macular degeneration susceptibility.” 2007. Web. 07 Mar 2021.

Vancouver:

Spencer KL. Variants in complement factor pathway genes and a novel locus on chromosome 16p12 influence age-related macular degeneration susceptibility. [Internet] [Doctoral dissertation]. Vanderbilt University; 2007. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1803/14024.

Council of Science Editors:

Spencer KL. Variants in complement factor pathway genes and a novel locus on chromosome 16p12 influence age-related macular degeneration susceptibility. [Doctoral Dissertation]. Vanderbilt University; 2007. Available from: http://hdl.handle.net/1803/14024


Vanderbilt University

12. Speirs, Christina Koo Yang. Prostaglandin Gbetagamma signaling stimulates gastrulation movements by limiting cell adhesion through Snail stabilization.

Degree: PhD, Biological Sciences, 2009, Vanderbilt University

 Prostaglandin E2 (PGE2) influences many processes in vertebrates, including development, homeostasis, and disease through its GPCRs EP receptors 1-4. PGE2 regulates gastrulation movements during zebrafish… (more)

Subjects/Keywords: development; cell motility; protrusion formation; E-cadherin

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Speirs, C. K. Y. (2009). Prostaglandin Gbetagamma signaling stimulates gastrulation movements by limiting cell adhesion through Snail stabilization. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14101

Chicago Manual of Style (16th Edition):

Speirs, Christina Koo Yang. “Prostaglandin Gbetagamma signaling stimulates gastrulation movements by limiting cell adhesion through Snail stabilization.” 2009. Doctoral Dissertation, Vanderbilt University. Accessed March 07, 2021. http://hdl.handle.net/1803/14101.

MLA Handbook (7th Edition):

Speirs, Christina Koo Yang. “Prostaglandin Gbetagamma signaling stimulates gastrulation movements by limiting cell adhesion through Snail stabilization.” 2009. Web. 07 Mar 2021.

Vancouver:

Speirs CKY. Prostaglandin Gbetagamma signaling stimulates gastrulation movements by limiting cell adhesion through Snail stabilization. [Internet] [Doctoral dissertation]. Vanderbilt University; 2009. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1803/14101.

Council of Science Editors:

Speirs CKY. Prostaglandin Gbetagamma signaling stimulates gastrulation movements by limiting cell adhesion through Snail stabilization. [Doctoral Dissertation]. Vanderbilt University; 2009. Available from: http://hdl.handle.net/1803/14101


Vanderbilt University

13. Kenealy, Shannon. Investigating the genetic susceptibility to multiple sclerosis: a genomic convergence approach.

Degree: PhD, Molecular Physiology and Biophysics, 2006, Vanderbilt University

 Multiple sclerosis (MS) is a debilitating neuroimmunological and neuro-degenerative disease. Despite substantial evidence for polygenic inheritance, the MHC is the only region that clearly and… (more)

Subjects/Keywords: chromosome 1q; genetic linkage; allelic association; SNPs; Multiple sclerosis  – Genetic aspects

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kenealy, S. (2006). Investigating the genetic susceptibility to multiple sclerosis: a genomic convergence approach. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11989

Chicago Manual of Style (16th Edition):

Kenealy, Shannon. “Investigating the genetic susceptibility to multiple sclerosis: a genomic convergence approach.” 2006. Doctoral Dissertation, Vanderbilt University. Accessed March 07, 2021. http://hdl.handle.net/1803/11989.

MLA Handbook (7th Edition):

Kenealy, Shannon. “Investigating the genetic susceptibility to multiple sclerosis: a genomic convergence approach.” 2006. Web. 07 Mar 2021.

Vancouver:

Kenealy S. Investigating the genetic susceptibility to multiple sclerosis: a genomic convergence approach. [Internet] [Doctoral dissertation]. Vanderbilt University; 2006. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1803/11989.

Council of Science Editors:

Kenealy S. Investigating the genetic susceptibility to multiple sclerosis: a genomic convergence approach. [Doctoral Dissertation]. Vanderbilt University; 2006. Available from: http://hdl.handle.net/1803/11989


Vanderbilt University

14. Mitchell, Sabrina L. Investigation into the molecular and physiologic relationship between peptide tyrosine tyrosine and N-acetylglutamate synthase.

Degree: PhD, Human Genetics, 2010, Vanderbilt University

 Neither genome size nor gene number is indicative of organism complexity. Complex regulation of genes within a genome likely contributes to organism complexity. Coordinate regulation… (more)

Subjects/Keywords: urea cycle disorders; nitrogen; urea cycle; protein

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mitchell, S. L. (2010). Investigation into the molecular and physiologic relationship between peptide tyrosine tyrosine and N-acetylglutamate synthase. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/15010

Chicago Manual of Style (16th Edition):

Mitchell, Sabrina L. “Investigation into the molecular and physiologic relationship between peptide tyrosine tyrosine and N-acetylglutamate synthase.” 2010. Doctoral Dissertation, Vanderbilt University. Accessed March 07, 2021. http://hdl.handle.net/1803/15010.

MLA Handbook (7th Edition):

Mitchell, Sabrina L. “Investigation into the molecular and physiologic relationship between peptide tyrosine tyrosine and N-acetylglutamate synthase.” 2010. Web. 07 Mar 2021.

Vancouver:

Mitchell SL. Investigation into the molecular and physiologic relationship between peptide tyrosine tyrosine and N-acetylglutamate synthase. [Internet] [Doctoral dissertation]. Vanderbilt University; 2010. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1803/15010.

Council of Science Editors:

Mitchell SL. Investigation into the molecular and physiologic relationship between peptide tyrosine tyrosine and N-acetylglutamate synthase. [Doctoral Dissertation]. Vanderbilt University; 2010. Available from: http://hdl.handle.net/1803/15010

.