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You searched for +publisher:"Vanderbilt University" +contributor:("Deborah Lannigan"). Showing records 1 – 7 of 7 total matches.

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Vanderbilt University

1. Fey, Erin Elizabeth. Phased Whole-Genome Detection and Analysis of Structural Variants of Invasive Ductal and Lobular Breast Cancer Cell Lines.

Degree: MS, Pathology, 2019, Vanderbilt University

 Heterogeneity in estrogen receptor positive breast cancers is poorly understood. Copy number alterations and structural differences contribute to carcinogenesis. Current cell line genomes representing the… (more)

Subjects/Keywords: breast cancer; cancer genomics; whole genome sequencing

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APA (6th Edition):

Fey, E. E. (2019). Phased Whole-Genome Detection and Analysis of Structural Variants of Invasive Ductal and Lobular Breast Cancer Cell Lines. (Thesis). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14359

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fey, Erin Elizabeth. “Phased Whole-Genome Detection and Analysis of Structural Variants of Invasive Ductal and Lobular Breast Cancer Cell Lines.” 2019. Thesis, Vanderbilt University. Accessed January 24, 2021. http://hdl.handle.net/1803/14359.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fey, Erin Elizabeth. “Phased Whole-Genome Detection and Analysis of Structural Variants of Invasive Ductal and Lobular Breast Cancer Cell Lines.” 2019. Web. 24 Jan 2021.

Vancouver:

Fey EE. Phased Whole-Genome Detection and Analysis of Structural Variants of Invasive Ductal and Lobular Breast Cancer Cell Lines. [Internet] [Thesis]. Vanderbilt University; 2019. [cited 2021 Jan 24]. Available from: http://hdl.handle.net/1803/14359.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fey EE. Phased Whole-Genome Detection and Analysis of Structural Variants of Invasive Ductal and Lobular Breast Cancer Cell Lines. [Thesis]. Vanderbilt University; 2019. Available from: http://hdl.handle.net/1803/14359

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

2. Hebron, Katie Elizabeth. ALCAM Dynamically Regulates Tumor Cell Adhesion Through Differential Proteolysis and a Novel Binding Partner, Tetraspanin CD151.

Degree: PhD, Cancer Biology, 2018, Vanderbilt University

 Metastasis persists as a significant unsolved hurdle in cancer treatment, with greater than 90% of cancer-related deaths attributed to metastasis. In order for cells to… (more)

Subjects/Keywords: cancer; metastasis; bladder cancer; protein processing; alternative splicing

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APA (6th Edition):

Hebron, K. E. (2018). ALCAM Dynamically Regulates Tumor Cell Adhesion Through Differential Proteolysis and a Novel Binding Partner, Tetraspanin CD151. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11464

Chicago Manual of Style (16th Edition):

Hebron, Katie Elizabeth. “ALCAM Dynamically Regulates Tumor Cell Adhesion Through Differential Proteolysis and a Novel Binding Partner, Tetraspanin CD151.” 2018. Doctoral Dissertation, Vanderbilt University. Accessed January 24, 2021. http://hdl.handle.net/1803/11464.

MLA Handbook (7th Edition):

Hebron, Katie Elizabeth. “ALCAM Dynamically Regulates Tumor Cell Adhesion Through Differential Proteolysis and a Novel Binding Partner, Tetraspanin CD151.” 2018. Web. 24 Jan 2021.

Vancouver:

Hebron KE. ALCAM Dynamically Regulates Tumor Cell Adhesion Through Differential Proteolysis and a Novel Binding Partner, Tetraspanin CD151. [Internet] [Doctoral dissertation]. Vanderbilt University; 2018. [cited 2021 Jan 24]. Available from: http://hdl.handle.net/1803/11464.

Council of Science Editors:

Hebron KE. ALCAM Dynamically Regulates Tumor Cell Adhesion Through Differential Proteolysis and a Novel Binding Partner, Tetraspanin CD151. [Doctoral Dissertation]. Vanderbilt University; 2018. Available from: http://hdl.handle.net/1803/11464


Vanderbilt University

3. Amato, Katherine Renee. Targeting the EPHA2 receptor tyrosine kinase in KRAS and EGFR mutant lung cancer.

Degree: PhD, Cancer Biology, 2015, Vanderbilt University

 Lung cancer remains the leading cause of cancer related deaths in the United States despite a significant number of advancements in the molecular diagnosis and… (more)

Subjects/Keywords: TKI; EPH; KRAS; EGFR

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APA (6th Edition):

Amato, K. R. (2015). Targeting the EPHA2 receptor tyrosine kinase in KRAS and EGFR mutant lung cancer. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11130

Chicago Manual of Style (16th Edition):

Amato, Katherine Renee. “Targeting the EPHA2 receptor tyrosine kinase in KRAS and EGFR mutant lung cancer.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed January 24, 2021. http://hdl.handle.net/1803/11130.

MLA Handbook (7th Edition):

Amato, Katherine Renee. “Targeting the EPHA2 receptor tyrosine kinase in KRAS and EGFR mutant lung cancer.” 2015. Web. 24 Jan 2021.

Vancouver:

Amato KR. Targeting the EPHA2 receptor tyrosine kinase in KRAS and EGFR mutant lung cancer. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2021 Jan 24]. Available from: http://hdl.handle.net/1803/11130.

Council of Science Editors:

Amato KR. Targeting the EPHA2 receptor tyrosine kinase in KRAS and EGFR mutant lung cancer. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://hdl.handle.net/1803/11130


Vanderbilt University

4. Austin, David C. The Role of Nuclear Factor Kappa B in Benign Prostatic Hyperplasia.

Degree: PhD, Cancer Biology, 2016, Vanderbilt University

 Benign prostatic hyperplasia (BPH) is a common, progressive chronic disease. Inflammation is associated with prostatic enlargement and resistance to 5?-reductase inhibitor (5ARI) therapy. Activation of… (more)

Subjects/Keywords: Androgen Receptor Variant 7; Inflammation; Androgen Receptor

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APA (6th Edition):

Austin, D. C. (2016). The Role of Nuclear Factor Kappa B in Benign Prostatic Hyperplasia. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10843

Chicago Manual of Style (16th Edition):

Austin, David C. “The Role of Nuclear Factor Kappa B in Benign Prostatic Hyperplasia.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed January 24, 2021. http://hdl.handle.net/1803/10843.

MLA Handbook (7th Edition):

Austin, David C. “The Role of Nuclear Factor Kappa B in Benign Prostatic Hyperplasia.” 2016. Web. 24 Jan 2021.

Vancouver:

Austin DC. The Role of Nuclear Factor Kappa B in Benign Prostatic Hyperplasia. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2021 Jan 24]. Available from: http://hdl.handle.net/1803/10843.

Council of Science Editors:

Austin DC. The Role of Nuclear Factor Kappa B in Benign Prostatic Hyperplasia. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://hdl.handle.net/1803/10843


Vanderbilt University

5. Mrozowski, Roman Michal. Targeting the Ser/Thr protein kinase RSK to reduce breast cancer metastasis.

Degree: PhD, Pathology, 2015, Vanderbilt University

 RSK is a family of four Ser/Thr protein kinases activated by ERK1/2 signaling. RSK is often misregulated in a variety of cancers, including breast and… (more)

Subjects/Keywords: kinase inhibitor; allosteric inhibitor; ribosomal S6 kinase; RSK; cancer metabolism; SL0101

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APA (6th Edition):

Mrozowski, R. M. (2015). Targeting the Ser/Thr protein kinase RSK to reduce breast cancer metastasis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/13000

Chicago Manual of Style (16th Edition):

Mrozowski, Roman Michal. “Targeting the Ser/Thr protein kinase RSK to reduce breast cancer metastasis.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed January 24, 2021. http://hdl.handle.net/1803/13000.

MLA Handbook (7th Edition):

Mrozowski, Roman Michal. “Targeting the Ser/Thr protein kinase RSK to reduce breast cancer metastasis.” 2015. Web. 24 Jan 2021.

Vancouver:

Mrozowski RM. Targeting the Ser/Thr protein kinase RSK to reduce breast cancer metastasis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2021 Jan 24]. Available from: http://hdl.handle.net/1803/13000.

Council of Science Editors:

Mrozowski RM. Targeting the Ser/Thr protein kinase RSK to reduce breast cancer metastasis. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://hdl.handle.net/1803/13000


Vanderbilt University

6. Puccetti, Matthew Vincent. The role of DNA replication fork remodeling proteins in lymphomagenesis and hematopoietic cell replication stress.

Degree: PhD, Pathology, 2018, Vanderbilt University

 Genome maintenance is essential for the viability of eukaryotic cells and for the prevention of tumorigenesis. DNA replication stress is a significant contributor to genomic… (more)

Subjects/Keywords: DNA translocases; DNA damage response; genomic instability; replication fork remodeling; tumorigenesis

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APA (6th Edition):

Puccetti, M. V. (2018). The role of DNA replication fork remodeling proteins in lymphomagenesis and hematopoietic cell replication stress. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/15446

Chicago Manual of Style (16th Edition):

Puccetti, Matthew Vincent. “The role of DNA replication fork remodeling proteins in lymphomagenesis and hematopoietic cell replication stress.” 2018. Doctoral Dissertation, Vanderbilt University. Accessed January 24, 2021. http://hdl.handle.net/1803/15446.

MLA Handbook (7th Edition):

Puccetti, Matthew Vincent. “The role of DNA replication fork remodeling proteins in lymphomagenesis and hematopoietic cell replication stress.” 2018. Web. 24 Jan 2021.

Vancouver:

Puccetti MV. The role of DNA replication fork remodeling proteins in lymphomagenesis and hematopoietic cell replication stress. [Internet] [Doctoral dissertation]. Vanderbilt University; 2018. [cited 2021 Jan 24]. Available from: http://hdl.handle.net/1803/15446.

Council of Science Editors:

Puccetti MV. The role of DNA replication fork remodeling proteins in lymphomagenesis and hematopoietic cell replication stress. [Doctoral Dissertation]. Vanderbilt University; 2018. Available from: http://hdl.handle.net/1803/15446


Vanderbilt University

7. Ludwik, Katarzyna Anna. RSKy Business in the Mammary Gland.

Degree: PhD, Pathology, 2018, Vanderbilt University

 The family of ribosomal S6 Ser/Thr protein kinases (RSK) controls proliferation, viability and motility and, therefore, contributes to the etiology of numerous cancers, including breast.… (more)

Subjects/Keywords: breast cancer; mammary gland; RSK2; estrogen receptor

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APA (6th Edition):

Ludwik, K. A. (2018). RSKy Business in the Mammary Gland. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11180

Chicago Manual of Style (16th Edition):

Ludwik, Katarzyna Anna. “RSKy Business in the Mammary Gland.” 2018. Doctoral Dissertation, Vanderbilt University. Accessed January 24, 2021. http://hdl.handle.net/1803/11180.

MLA Handbook (7th Edition):

Ludwik, Katarzyna Anna. “RSKy Business in the Mammary Gland.” 2018. Web. 24 Jan 2021.

Vancouver:

Ludwik KA. RSKy Business in the Mammary Gland. [Internet] [Doctoral dissertation]. Vanderbilt University; 2018. [cited 2021 Jan 24]. Available from: http://hdl.handle.net/1803/11180.

Council of Science Editors:

Ludwik KA. RSKy Business in the Mammary Gland. [Doctoral Dissertation]. Vanderbilt University; 2018. Available from: http://hdl.handle.net/1803/11180

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