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You searched for +publisher:"Vanderbilt University" +contributor:("Dan Roden"). Showing records 1 – 10 of 10 total matches.

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Vanderbilt University

1. Parikh, Shan S. Maturation and implementation of human induced pluripotent stem cell derived cardiomyocytes for modeling cardiac disease.

Degree: PhD, Pharmacology, 2018, Vanderbilt University

 Mutations in lamin A/C (LMNA) are the second most common contributor to genetic cases of dilated cardiomyopathy (DCM). Despite the autosomal-dominant inheritance of LMNA DCM,… (more)

Subjects/Keywords: ipsc; calcium; lamin; dilated cardiomyopathy; t-tubules; calcium hanlding; crispr; hipsc-cm; excitation-contraction coupling; electrophysiology

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APA (6th Edition):

Parikh, S. S. (2018). Maturation and implementation of human induced pluripotent stem cell derived cardiomyocytes for modeling cardiac disease. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12262

Chicago Manual of Style (16th Edition):

Parikh, Shan S. “Maturation and implementation of human induced pluripotent stem cell derived cardiomyocytes for modeling cardiac disease.” 2018. Doctoral Dissertation, Vanderbilt University. Accessed April 12, 2021. http://hdl.handle.net/1803/12262.

MLA Handbook (7th Edition):

Parikh, Shan S. “Maturation and implementation of human induced pluripotent stem cell derived cardiomyocytes for modeling cardiac disease.” 2018. Web. 12 Apr 2021.

Vancouver:

Parikh SS. Maturation and implementation of human induced pluripotent stem cell derived cardiomyocytes for modeling cardiac disease. [Internet] [Doctoral dissertation]. Vanderbilt University; 2018. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/1803/12262.

Council of Science Editors:

Parikh SS. Maturation and implementation of human induced pluripotent stem cell derived cardiomyocytes for modeling cardiac disease. [Doctoral Dissertation]. Vanderbilt University; 2018. Available from: http://hdl.handle.net/1803/12262


Vanderbilt University

2. Shuey, Megan Marie. Pharmacogenetics of Resistant Hypertension: Leveraging the Electronic Medical Record.

Degree: PhD, Pharmacology, 2018, Vanderbilt University

 Patients with resistant hypertension have uncontrolled blood pressure despite concurrent treatment with three or more antihypertensive medications including a thiazide diuretic. Compared to patients with… (more)

Subjects/Keywords: CYP4A11; Blood Pressure response; Resistant Hypertension; Antihypertensive Medications; Electronic Medical Records; Spironolactone

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APA (6th Edition):

Shuey, M. M. (2018). Pharmacogenetics of Resistant Hypertension: Leveraging the Electronic Medical Record. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/15419

Chicago Manual of Style (16th Edition):

Shuey, Megan Marie. “Pharmacogenetics of Resistant Hypertension: Leveraging the Electronic Medical Record.” 2018. Doctoral Dissertation, Vanderbilt University. Accessed April 12, 2021. http://hdl.handle.net/1803/15419.

MLA Handbook (7th Edition):

Shuey, Megan Marie. “Pharmacogenetics of Resistant Hypertension: Leveraging the Electronic Medical Record.” 2018. Web. 12 Apr 2021.

Vancouver:

Shuey MM. Pharmacogenetics of Resistant Hypertension: Leveraging the Electronic Medical Record. [Internet] [Doctoral dissertation]. Vanderbilt University; 2018. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/1803/15419.

Council of Science Editors:

Shuey MM. Pharmacogenetics of Resistant Hypertension: Leveraging the Electronic Medical Record. [Doctoral Dissertation]. Vanderbilt University; 2018. Available from: http://hdl.handle.net/1803/15419


Vanderbilt University

3. Moore, Carrie Colleen Buchanan. A biologically informed method for detecting associations with rare variants.

Degree: PhD, Human Genetics, 2013, Vanderbilt University

 Many recent studies have identified rare variants that contribute to common, complex disease. It is believed that rare variants likely have a larger effect size… (more)

Subjects/Keywords: collapsing methods; 1000 Genomes Project data; next-generation sequencing; genomics; rare variants

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APA (6th Edition):

Moore, C. C. B. (2013). A biologically informed method for detecting associations with rare variants. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14835

Chicago Manual of Style (16th Edition):

Moore, Carrie Colleen Buchanan. “A biologically informed method for detecting associations with rare variants.” 2013. Doctoral Dissertation, Vanderbilt University. Accessed April 12, 2021. http://hdl.handle.net/1803/14835.

MLA Handbook (7th Edition):

Moore, Carrie Colleen Buchanan. “A biologically informed method for detecting associations with rare variants.” 2013. Web. 12 Apr 2021.

Vancouver:

Moore CCB. A biologically informed method for detecting associations with rare variants. [Internet] [Doctoral dissertation]. Vanderbilt University; 2013. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/1803/14835.

Council of Science Editors:

Moore CCB. A biologically informed method for detecting associations with rare variants. [Doctoral Dissertation]. Vanderbilt University; 2013. Available from: http://hdl.handle.net/1803/14835


Vanderbilt University

4. Wiley, Laura Katherine. Addressing Barriers to Clinical Implementation of Pharmacogenomics.

Degree: PhD, Human Genetics, 2016, Vanderbilt University

 Pharmacogenomics offers one of the best use cases for widespread clinical implementation of genomic medicine, as variants tend to have moderate allele frequencies, many of… (more)

Subjects/Keywords: warfarin; precision medicine; translational bioinformatics; policy

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APA (6th Edition):

Wiley, L. K. (2016). Addressing Barriers to Clinical Implementation of Pharmacogenomics. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11632

Chicago Manual of Style (16th Edition):

Wiley, Laura Katherine. “Addressing Barriers to Clinical Implementation of Pharmacogenomics.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed April 12, 2021. http://hdl.handle.net/1803/11632.

MLA Handbook (7th Edition):

Wiley, Laura Katherine. “Addressing Barriers to Clinical Implementation of Pharmacogenomics.” 2016. Web. 12 Apr 2021.

Vancouver:

Wiley LK. Addressing Barriers to Clinical Implementation of Pharmacogenomics. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/1803/11632.

Council of Science Editors:

Wiley LK. Addressing Barriers to Clinical Implementation of Pharmacogenomics. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://hdl.handle.net/1803/11632


Vanderbilt University

5. Bennett, Jeffrey Scott. A biphasic role for the voltage-gated sodium channel scn5Lab in cardiac development of zebrafish.

Degree: PhD, Human Genetics, 2013, Vanderbilt University

 A BIPHASIC ROLE FOR THE VOLTAGE-GATED SODIUM CHANNEL SCN5LAB IN CARDIAC DEVELOPMENT OF ZEBRAFISH JEFFREY S. BENNETT Dissertation under direction of Professor Dan M. Roden,… (more)

Subjects/Keywords: heart development; genetics; sodium channels; proliferation; zebrafish

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APA (6th Edition):

Bennett, J. S. (2013). A biphasic role for the voltage-gated sodium channel scn5Lab in cardiac development of zebrafish. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12607

Chicago Manual of Style (16th Edition):

Bennett, Jeffrey Scott. “A biphasic role for the voltage-gated sodium channel scn5Lab in cardiac development of zebrafish.” 2013. Doctoral Dissertation, Vanderbilt University. Accessed April 12, 2021. http://hdl.handle.net/1803/12607.

MLA Handbook (7th Edition):

Bennett, Jeffrey Scott. “A biphasic role for the voltage-gated sodium channel scn5Lab in cardiac development of zebrafish.” 2013. Web. 12 Apr 2021.

Vancouver:

Bennett JS. A biphasic role for the voltage-gated sodium channel scn5Lab in cardiac development of zebrafish. [Internet] [Doctoral dissertation]. Vanderbilt University; 2013. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/1803/12607.

Council of Science Editors:

Bennett JS. A biphasic role for the voltage-gated sodium channel scn5Lab in cardiac development of zebrafish. [Doctoral Dissertation]. Vanderbilt University; 2013. Available from: http://hdl.handle.net/1803/12607


Vanderbilt University

6. Beckermann, Thomas Martin. Mutant Cardiac Sodium Channel Dysfunction Associated with Cardiomyopathy.

Degree: PhD, Pharmacology, 2014, Vanderbilt University

 The goal of this project is to better understand the relationship between cardiac sodium channel dysfunction and cardiomyopathy. Mutations in the gene SCN5A, encoding the… (more)

Subjects/Keywords: amiodarone; cardiac electrophysiology; dilated cardiomyopathy

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APA (6th Edition):

Beckermann, T. M. (2014). Mutant Cardiac Sodium Channel Dysfunction Associated with Cardiomyopathy. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12694

Chicago Manual of Style (16th Edition):

Beckermann, Thomas Martin. “Mutant Cardiac Sodium Channel Dysfunction Associated with Cardiomyopathy.” 2014. Doctoral Dissertation, Vanderbilt University. Accessed April 12, 2021. http://hdl.handle.net/1803/12694.

MLA Handbook (7th Edition):

Beckermann, Thomas Martin. “Mutant Cardiac Sodium Channel Dysfunction Associated with Cardiomyopathy.” 2014. Web. 12 Apr 2021.

Vancouver:

Beckermann TM. Mutant Cardiac Sodium Channel Dysfunction Associated with Cardiomyopathy. [Internet] [Doctoral dissertation]. Vanderbilt University; 2014. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/1803/12694.

Council of Science Editors:

Beckermann TM. Mutant Cardiac Sodium Channel Dysfunction Associated with Cardiomyopathy. [Doctoral Dissertation]. Vanderbilt University; 2014. Available from: http://hdl.handle.net/1803/12694


Vanderbilt University

7. Jeff, Janina Maria. The Genetics of Quantitative Traits Associated with Cardiovascular Disease in African Americans.

Degree: PhD, Human Genetics, 2012, Vanderbilt University

 Cardiovascular disease (CVD) is the leading cause of death in most developed countries. In addition to environmental risk factors, such as diet and physical activity,… (more)

Subjects/Keywords: electrocardiographic traits; fibrinogen; African Americans; quantitative traits

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APA (6th Edition):

Jeff, J. M. (2012). The Genetics of Quantitative Traits Associated with Cardiovascular Disease in African Americans. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10690

Chicago Manual of Style (16th Edition):

Jeff, Janina Maria. “The Genetics of Quantitative Traits Associated with Cardiovascular Disease in African Americans.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed April 12, 2021. http://hdl.handle.net/1803/10690.

MLA Handbook (7th Edition):

Jeff, Janina Maria. “The Genetics of Quantitative Traits Associated with Cardiovascular Disease in African Americans.” 2012. Web. 12 Apr 2021.

Vancouver:

Jeff JM. The Genetics of Quantitative Traits Associated with Cardiovascular Disease in African Americans. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/1803/10690.

Council of Science Editors:

Jeff JM. The Genetics of Quantitative Traits Associated with Cardiovascular Disease in African Americans. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://hdl.handle.net/1803/10690


Vanderbilt University

8. Thiel, William Howard. Proarrhythmic defects in Timothy Syndrome require calmodulin kinase II.

Degree: PhD, Pharmacology, 2008, Vanderbilt University

 Ca2+ activated signaling pathways coordinate contraction in the heart, but these pathways cause disease upon excessive activation. Intracellular Ca2+ activates the multifunctional Ca2+ and calmodulin… (more)

Subjects/Keywords: CaMKII; Calcium Channel; cardiology; arrhythmia

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APA (6th Edition):

Thiel, W. H. (2008). Proarrhythmic defects in Timothy Syndrome require calmodulin kinase II. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14564

Chicago Manual of Style (16th Edition):

Thiel, William Howard. “Proarrhythmic defects in Timothy Syndrome require calmodulin kinase II.” 2008. Doctoral Dissertation, Vanderbilt University. Accessed April 12, 2021. http://hdl.handle.net/1803/14564.

MLA Handbook (7th Edition):

Thiel, William Howard. “Proarrhythmic defects in Timothy Syndrome require calmodulin kinase II.” 2008. Web. 12 Apr 2021.

Vancouver:

Thiel WH. Proarrhythmic defects in Timothy Syndrome require calmodulin kinase II. [Internet] [Doctoral dissertation]. Vanderbilt University; 2008. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/1803/14564.

Council of Science Editors:

Thiel WH. Proarrhythmic defects in Timothy Syndrome require calmodulin kinase II. [Doctoral Dissertation]. Vanderbilt University; 2008. Available from: http://hdl.handle.net/1803/14564


Vanderbilt University

9. English, Brett Alan. Cardiovascular and Neuropsychiatric Consequences of a Genetic Loss of the High-Affinity Choline Transporter (CHT).

Degree: PhD, Pharmacology, 2010, Vanderbilt University

 Acetylcholine (ACh) was one of the first neurotransmitters discovered and has been implicated in regulating a number of physiologic processes within the CNS and the… (more)

Subjects/Keywords: heart rate; polymorphism; cardiovascular; choline transporter; baroreceptor reflex; autonomic; parasympathetic

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APA (6th Edition):

English, B. A. (2010). Cardiovascular and Neuropsychiatric Consequences of a Genetic Loss of the High-Affinity Choline Transporter (CHT). (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11766

Chicago Manual of Style (16th Edition):

English, Brett Alan. “Cardiovascular and Neuropsychiatric Consequences of a Genetic Loss of the High-Affinity Choline Transporter (CHT).” 2010. Doctoral Dissertation, Vanderbilt University. Accessed April 12, 2021. http://hdl.handle.net/1803/11766.

MLA Handbook (7th Edition):

English, Brett Alan. “Cardiovascular and Neuropsychiatric Consequences of a Genetic Loss of the High-Affinity Choline Transporter (CHT).” 2010. Web. 12 Apr 2021.

Vancouver:

English BA. Cardiovascular and Neuropsychiatric Consequences of a Genetic Loss of the High-Affinity Choline Transporter (CHT). [Internet] [Doctoral dissertation]. Vanderbilt University; 2010. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/1803/11766.

Council of Science Editors:

English BA. Cardiovascular and Neuropsychiatric Consequences of a Genetic Loss of the High-Affinity Choline Transporter (CHT). [Doctoral Dissertation]. Vanderbilt University; 2010. Available from: http://hdl.handle.net/1803/11766


Vanderbilt University

10. Chopra, Sameer. Sodium channels are required for cardiac cell-fate specification via a novel, non-electrogenic mechanism in zebrafish.

Degree: PhD, Pharmacology, 2008, Vanderbilt University

 Electrical signaling events are required for each human thought, feeling, and perception, the movement of our limbs, and the beat of our hearts. As the… (more)

Subjects/Keywords: heart; cardiac development; cardiomyopathy; zebrafish; Sodium channels; electrophysiology; animal models of disease; arrhythmia; Zebra danio  – Development; Heart  – Differentiation

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APA (6th Edition):

Chopra, S. (2008). Sodium channels are required for cardiac cell-fate specification via a novel, non-electrogenic mechanism in zebrafish. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14896

Chicago Manual of Style (16th Edition):

Chopra, Sameer. “Sodium channels are required for cardiac cell-fate specification via a novel, non-electrogenic mechanism in zebrafish.” 2008. Doctoral Dissertation, Vanderbilt University. Accessed April 12, 2021. http://hdl.handle.net/1803/14896.

MLA Handbook (7th Edition):

Chopra, Sameer. “Sodium channels are required for cardiac cell-fate specification via a novel, non-electrogenic mechanism in zebrafish.” 2008. Web. 12 Apr 2021.

Vancouver:

Chopra S. Sodium channels are required for cardiac cell-fate specification via a novel, non-electrogenic mechanism in zebrafish. [Internet] [Doctoral dissertation]. Vanderbilt University; 2008. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/1803/14896.

Council of Science Editors:

Chopra S. Sodium channels are required for cardiac cell-fate specification via a novel, non-electrogenic mechanism in zebrafish. [Doctoral Dissertation]. Vanderbilt University; 2008. Available from: http://hdl.handle.net/1803/14896

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