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You searched for +publisher:"Vanderbilt University" +contributor:("Bingshan Li"). Showing records 1 – 11 of 11 total matches.

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Vanderbilt University

1. Han, Mi-Ryung. Rare coding variants in GWAS identified loci with breast cancer risk.

Degree: PhD, Epidemiology, 2016, Vanderbilt University

 To date, common genetic variants in ~ 109 loci have been identified for breast cancer risk via genome-wide association studies (GWAS). Most common variants found… (more)

Subjects/Keywords: GWAS; Rare coding variant; Breast cancer

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APA (6th Edition):

Han, M. (2016). Rare coding variants in GWAS identified loci with breast cancer risk. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10872

Chicago Manual of Style (16th Edition):

Han, Mi-Ryung. “Rare coding variants in GWAS identified loci with breast cancer risk.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed March 07, 2021. http://hdl.handle.net/1803/10872.

MLA Handbook (7th Edition):

Han, Mi-Ryung. “Rare coding variants in GWAS identified loci with breast cancer risk.” 2016. Web. 07 Mar 2021.

Vancouver:

Han M. Rare coding variants in GWAS identified loci with breast cancer risk. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1803/10872.

Council of Science Editors:

Han M. Rare coding variants in GWAS identified loci with breast cancer risk. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://hdl.handle.net/1803/10872


Vanderbilt University

2. D'Aoust, Laura Nicole. Examination of Candidate Exonic Variants that Confer Susceptibility to Alzheimer Disease in the Amish.

Degree: PhD, Human Genetics, 2015, Vanderbilt University

 Alzheimer disease (AD) is the most common cause of dementia. As with many complex diseases, the identified variants do not explain the total expected genetic… (more)

Subjects/Keywords: Alzheimer; family-based study; genetics

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APA (6th Edition):

D'Aoust, L. N. (2015). Examination of Candidate Exonic Variants that Confer Susceptibility to Alzheimer Disease in the Amish. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10861

Chicago Manual of Style (16th Edition):

D'Aoust, Laura Nicole. “Examination of Candidate Exonic Variants that Confer Susceptibility to Alzheimer Disease in the Amish.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed March 07, 2021. http://hdl.handle.net/1803/10861.

MLA Handbook (7th Edition):

D'Aoust, Laura Nicole. “Examination of Candidate Exonic Variants that Confer Susceptibility to Alzheimer Disease in the Amish.” 2015. Web. 07 Mar 2021.

Vancouver:

D'Aoust LN. Examination of Candidate Exonic Variants that Confer Susceptibility to Alzheimer Disease in the Amish. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1803/10861.

Council of Science Editors:

D'Aoust LN. Examination of Candidate Exonic Variants that Confer Susceptibility to Alzheimer Disease in the Amish. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://hdl.handle.net/1803/10861


Vanderbilt University

3. Moore, Carrie Colleen Buchanan. A biologically informed method for detecting associations with rare variants.

Degree: PhD, Human Genetics, 2013, Vanderbilt University

 Many recent studies have identified rare variants that contribute to common, complex disease. It is believed that rare variants likely have a larger effect size… (more)

Subjects/Keywords: collapsing methods; 1000 Genomes Project data; next-generation sequencing; genomics; rare variants

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APA (6th Edition):

Moore, C. C. B. (2013). A biologically informed method for detecting associations with rare variants. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14835

Chicago Manual of Style (16th Edition):

Moore, Carrie Colleen Buchanan. “A biologically informed method for detecting associations with rare variants.” 2013. Doctoral Dissertation, Vanderbilt University. Accessed March 07, 2021. http://hdl.handle.net/1803/14835.

MLA Handbook (7th Edition):

Moore, Carrie Colleen Buchanan. “A biologically informed method for detecting associations with rare variants.” 2013. Web. 07 Mar 2021.

Vancouver:

Moore CCB. A biologically informed method for detecting associations with rare variants. [Internet] [Doctoral dissertation]. Vanderbilt University; 2013. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1803/14835.

Council of Science Editors:

Moore CCB. A biologically informed method for detecting associations with rare variants. [Doctoral Dissertation]. Vanderbilt University; 2013. Available from: http://hdl.handle.net/1803/14835


Vanderbilt University

4. Zeng, Chenjie. Genetic markers of 5-fluorouracil associated-toxicity in colorectal cancer patients.

Degree: PhD, Epidemiology, 2017, Vanderbilt University

 Previous pharmacogenetics studies of 5-fluorouracil (5-FU) have focused on coding variants, and only four of these variants showed consistent association, which explained a small fraction… (more)

Subjects/Keywords: pharmacogenetics; genetics; 5-FU; toxicity; side effects; colorectal cancer

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APA (6th Edition):

Zeng, C. (2017). Genetic markers of 5-fluorouracil associated-toxicity in colorectal cancer patients. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/15548

Chicago Manual of Style (16th Edition):

Zeng, Chenjie. “Genetic markers of 5-fluorouracil associated-toxicity in colorectal cancer patients.” 2017. Doctoral Dissertation, Vanderbilt University. Accessed March 07, 2021. http://hdl.handle.net/1803/15548.

MLA Handbook (7th Edition):

Zeng, Chenjie. “Genetic markers of 5-fluorouracil associated-toxicity in colorectal cancer patients.” 2017. Web. 07 Mar 2021.

Vancouver:

Zeng C. Genetic markers of 5-fluorouracil associated-toxicity in colorectal cancer patients. [Internet] [Doctoral dissertation]. Vanderbilt University; 2017. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1803/15548.

Council of Science Editors:

Zeng C. Genetic markers of 5-fluorouracil associated-toxicity in colorectal cancer patients. [Doctoral Dissertation]. Vanderbilt University; 2017. Available from: http://hdl.handle.net/1803/15548


Vanderbilt University

5. Giri, Ayush. Modifiable and non-modifiable risk factors for pelvic organ prolapse.

Degree: PhD, Epidemiology, 2015, Vanderbilt University

 Pelvic organ prolapse (POP) is characterized by the descent of pelvic organs (uterus, bladder, and bowels) from their normal anatomical positions into the vaginal space… (more)

Subjects/Keywords: Epidemiology of POP; Admixture mapping; Gene-environment interactions; Obesity and pelvic organ prolapse; Pelvic organ prolapse (POP); Women's Health

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APA (6th Edition):

Giri, A. (2015). Modifiable and non-modifiable risk factors for pelvic organ prolapse. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14413

Chicago Manual of Style (16th Edition):

Giri, Ayush. “Modifiable and non-modifiable risk factors for pelvic organ prolapse.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed March 07, 2021. http://hdl.handle.net/1803/14413.

MLA Handbook (7th Edition):

Giri, Ayush. “Modifiable and non-modifiable risk factors for pelvic organ prolapse.” 2015. Web. 07 Mar 2021.

Vancouver:

Giri A. Modifiable and non-modifiable risk factors for pelvic organ prolapse. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1803/14413.

Council of Science Editors:

Giri A. Modifiable and non-modifiable risk factors for pelvic organ prolapse. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://hdl.handle.net/1803/14413


Vanderbilt University

6. Sobota, Rafal Sebastian. Genetics of Tuberculosis Resistance.

Degree: PhD, Human Genetics, 2015, Vanderbilt University

 One third of the world’s population has been infected with Mycobacterium tuberculosis (MTB). Most of those exposed develop an asymptomatic latent infection. In the absence… (more)

Subjects/Keywords: il9; tuberculosis; il12b; gwas

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APA (6th Edition):

Sobota, R. S. (2015). Genetics of Tuberculosis Resistance. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12103

Chicago Manual of Style (16th Edition):

Sobota, Rafal Sebastian. “Genetics of Tuberculosis Resistance.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed March 07, 2021. http://hdl.handle.net/1803/12103.

MLA Handbook (7th Edition):

Sobota, Rafal Sebastian. “Genetics of Tuberculosis Resistance.” 2015. Web. 07 Mar 2021.

Vancouver:

Sobota RS. Genetics of Tuberculosis Resistance. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1803/12103.

Council of Science Editors:

Sobota RS. Genetics of Tuberculosis Resistance. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://hdl.handle.net/1803/12103


Vanderbilt University

7. Bray, Michael Joseph. Evaluating Epidemiologic and Genetic Risk Factors for Uterine Fibroid Characteristics.

Degree: PhD, Human Genetics, 2018, Vanderbilt University

 Uterine fibroids, benign tumors of the uterus, are the most common female pelvic tumor. Fibroids are highly heterogeneous, with some women developing a single small… (more)

Subjects/Keywords: admixture mapping study; genome-wide association study; uterine fibroids

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APA (6th Edition):

Bray, M. J. (2018). Evaluating Epidemiologic and Genetic Risk Factors for Uterine Fibroid Characteristics. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10965

Chicago Manual of Style (16th Edition):

Bray, Michael Joseph. “Evaluating Epidemiologic and Genetic Risk Factors for Uterine Fibroid Characteristics.” 2018. Doctoral Dissertation, Vanderbilt University. Accessed March 07, 2021. http://hdl.handle.net/1803/10965.

MLA Handbook (7th Edition):

Bray, Michael Joseph. “Evaluating Epidemiologic and Genetic Risk Factors for Uterine Fibroid Characteristics.” 2018. Web. 07 Mar 2021.

Vancouver:

Bray MJ. Evaluating Epidemiologic and Genetic Risk Factors for Uterine Fibroid Characteristics. [Internet] [Doctoral dissertation]. Vanderbilt University; 2018. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1803/10965.

Council of Science Editors:

Bray MJ. Evaluating Epidemiologic and Genetic Risk Factors for Uterine Fibroid Characteristics. [Doctoral Dissertation]. Vanderbilt University; 2018. Available from: http://hdl.handle.net/1803/10965


Vanderbilt University

8. Cummings, Anna Christine. Exploring the genetic architecture of late-onset Alzheimer disease in an Amish population.

Degree: PhD, Human Genetics, 2012, Vanderbilt University

 Late-onset Alzheimer disease (LOAD) is a complex neurodegenerative disorder with a strong genetic component. APOE is a well-established risk gene for LOAD, and several other… (more)

Subjects/Keywords: Amish; Alzheimer disease; genetics

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APA (6th Edition):

Cummings, A. C. (2012). Exploring the genetic architecture of late-onset Alzheimer disease in an Amish population. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14870

Chicago Manual of Style (16th Edition):

Cummings, Anna Christine. “Exploring the genetic architecture of late-onset Alzheimer disease in an Amish population.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed March 07, 2021. http://hdl.handle.net/1803/14870.

MLA Handbook (7th Edition):

Cummings, Anna Christine. “Exploring the genetic architecture of late-onset Alzheimer disease in an Amish population.” 2012. Web. 07 Mar 2021.

Vancouver:

Cummings AC. Exploring the genetic architecture of late-onset Alzheimer disease in an Amish population. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1803/14870.

Council of Science Editors:

Cummings AC. Exploring the genetic architecture of late-onset Alzheimer disease in an Amish population. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://hdl.handle.net/1803/14870


Vanderbilt University

9. Restrepo, Nicole Ann. Investigation of the genetic epidemiology of age-related macular degeneration, primary open-angle glaucoma, and diabetic retinopathy in diverse populations.

Degree: PhD, Human Genetics, 2015, Vanderbilt University

 Common age, related eye diseases are a major driving force behind vision disability and blindness. The three most common diseases afflicting Americans today are age-related… (more)

Subjects/Keywords: African American; diabetic retinopathy; primary open angle glaucoma; age-related macular degeneration; Mexican American

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APA (6th Edition):

Restrepo, N. A. (2015). Investigation of the genetic epidemiology of age-related macular degeneration, primary open-angle glaucoma, and diabetic retinopathy in diverse populations. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10956

Chicago Manual of Style (16th Edition):

Restrepo, Nicole Ann. “Investigation of the genetic epidemiology of age-related macular degeneration, primary open-angle glaucoma, and diabetic retinopathy in diverse populations.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed March 07, 2021. http://hdl.handle.net/1803/10956.

MLA Handbook (7th Edition):

Restrepo, Nicole Ann. “Investigation of the genetic epidemiology of age-related macular degeneration, primary open-angle glaucoma, and diabetic retinopathy in diverse populations.” 2015. Web. 07 Mar 2021.

Vancouver:

Restrepo NA. Investigation of the genetic epidemiology of age-related macular degeneration, primary open-angle glaucoma, and diabetic retinopathy in diverse populations. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1803/10956.

Council of Science Editors:

Restrepo NA. Investigation of the genetic epidemiology of age-related macular degeneration, primary open-angle glaucoma, and diabetic retinopathy in diverse populations. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://hdl.handle.net/1803/10956


Vanderbilt University

10. Levinson, Rebecca Terrall. The Phenotypic Consequences of Distinct Genetic Variation in an Electronic Medical Record.

Degree: PhD, Human Genetics, 2016, Vanderbilt University

 While genetic association studies have been able to elucidate the importance of genetics in human disease outcomes, these studies are limited by the necessity of… (more)

Subjects/Keywords: PheWAS

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APA (6th Edition):

Levinson, R. T. (2016). The Phenotypic Consequences of Distinct Genetic Variation in an Electronic Medical Record. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14698

Chicago Manual of Style (16th Edition):

Levinson, Rebecca Terrall. “The Phenotypic Consequences of Distinct Genetic Variation in an Electronic Medical Record.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed March 07, 2021. http://hdl.handle.net/1803/14698.

MLA Handbook (7th Edition):

Levinson, Rebecca Terrall. “The Phenotypic Consequences of Distinct Genetic Variation in an Electronic Medical Record.” 2016. Web. 07 Mar 2021.

Vancouver:

Levinson RT. The Phenotypic Consequences of Distinct Genetic Variation in an Electronic Medical Record. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1803/14698.

Council of Science Editors:

Levinson RT. The Phenotypic Consequences of Distinct Genetic Variation in an Electronic Medical Record. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://hdl.handle.net/1803/14698


Vanderbilt University

11. White, Marquitta Jonisse. Genetics of Plasminogen Activator Inhibitor – 1: a potent biological effector of cardiovascular disease risk.

Degree: PhD, Human Genetics, 2014, Vanderbilt University

 Cardiovascular disease (CVD) is an inclusive term encompassing several disorders of the circulatory system that together account for the majority of global non-communicable disease (NCD)… (more)

Subjects/Keywords: population genetics; plasminogen activator inhibitor-1; cardiovascular disease

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APA (6th Edition):

White, M. J. (2014). Genetics of Plasminogen Activator Inhibitor – 1: a potent biological effector of cardiovascular disease risk. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14029

Chicago Manual of Style (16th Edition):

White, Marquitta Jonisse. “Genetics of Plasminogen Activator Inhibitor – 1: a potent biological effector of cardiovascular disease risk.” 2014. Doctoral Dissertation, Vanderbilt University. Accessed March 07, 2021. http://hdl.handle.net/1803/14029.

MLA Handbook (7th Edition):

White, Marquitta Jonisse. “Genetics of Plasminogen Activator Inhibitor – 1: a potent biological effector of cardiovascular disease risk.” 2014. Web. 07 Mar 2021.

Vancouver:

White MJ. Genetics of Plasminogen Activator Inhibitor – 1: a potent biological effector of cardiovascular disease risk. [Internet] [Doctoral dissertation]. Vanderbilt University; 2014. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1803/14029.

Council of Science Editors:

White MJ. Genetics of Plasminogen Activator Inhibitor – 1: a potent biological effector of cardiovascular disease risk. [Doctoral Dissertation]. Vanderbilt University; 2014. Available from: http://hdl.handle.net/1803/14029

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