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You searched for +publisher:"Vanderbilt University" +contributor:("Ann Richmond"). Showing records 1 – 27 of 27 total matches.

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Vanderbilt University

1. Thu, Yee Mon. The role of NF-kB inducing kinase (NIK) in modulating melanoma tumorigenesis.

Degree: PhD, Cancer Biology, 2011, Vanderbilt University

 Nuclear factor-êB (NF-êB) inducing kinase (NIK) is a MAP3K that regulates activation of NF-êB. NIK is often over-expressed in tumor cells, including melanoma, but the… (more)

Subjects/Keywords: melanoma; cancer; NF-kB inducing kinase; NF-kB

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APA (6th Edition):

Thu, Y. M. (2011). The role of NF-kB inducing kinase (NIK) in modulating melanoma tumorigenesis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14261

Chicago Manual of Style (16th Edition):

Thu, Yee Mon. “The role of NF-kB inducing kinase (NIK) in modulating melanoma tumorigenesis.” 2011. Doctoral Dissertation, Vanderbilt University. Accessed September 19, 2020. http://hdl.handle.net/1803/14261.

MLA Handbook (7th Edition):

Thu, Yee Mon. “The role of NF-kB inducing kinase (NIK) in modulating melanoma tumorigenesis.” 2011. Web. 19 Sep 2020.

Vancouver:

Thu YM. The role of NF-kB inducing kinase (NIK) in modulating melanoma tumorigenesis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2011. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1803/14261.

Council of Science Editors:

Thu YM. The role of NF-kB inducing kinase (NIK) in modulating melanoma tumorigenesis. [Doctoral Dissertation]. Vanderbilt University; 2011. Available from: http://hdl.handle.net/1803/14261


Vanderbilt University

2. Doxie, Deon Bryant. Quantitative Single Cell Analysis of Cancerous Cells During Therapy.

Degree: PhD, Cell and Developmental Biology, 2018, Vanderbilt University

 Single cell tools have great potential to characterize mechanisms of oncogenesis and treatment resistance. To date, single cell quantitative cytometry has been widely applied in… (more)

Subjects/Keywords: single cell; solid tumor; targeted therapy; melanoma; mass cytometry

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APA (6th Edition):

Doxie, D. B. (2018). Quantitative Single Cell Analysis of Cancerous Cells During Therapy. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12624

Chicago Manual of Style (16th Edition):

Doxie, Deon Bryant. “Quantitative Single Cell Analysis of Cancerous Cells During Therapy.” 2018. Doctoral Dissertation, Vanderbilt University. Accessed September 19, 2020. http://hdl.handle.net/1803/12624.

MLA Handbook (7th Edition):

Doxie, Deon Bryant. “Quantitative Single Cell Analysis of Cancerous Cells During Therapy.” 2018. Web. 19 Sep 2020.

Vancouver:

Doxie DB. Quantitative Single Cell Analysis of Cancerous Cells During Therapy. [Internet] [Doctoral dissertation]. Vanderbilt University; 2018. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1803/12624.

Council of Science Editors:

Doxie DB. Quantitative Single Cell Analysis of Cancerous Cells During Therapy. [Doctoral Dissertation]. Vanderbilt University; 2018. Available from: http://hdl.handle.net/1803/12624


Vanderbilt University

3. Reiner, Jennifer Elisabeth. Oncogenic STRAP regulates c-Jun stability and persistent migration.

Degree: PhD, Cancer Biology, 2011, Vanderbilt University

 The serine threonine kinase receptor-associated protein (STRAP) is a WD40 domain protein that regulates a wide array of biological processes. Previous reports suggest that STRAP… (more)

Subjects/Keywords: proliferation; ubiquitylation; cancer; STRAP; motility

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APA (6th Edition):

Reiner, J. E. (2011). Oncogenic STRAP regulates c-Jun stability and persistent migration. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12548

Chicago Manual of Style (16th Edition):

Reiner, Jennifer Elisabeth. “Oncogenic STRAP regulates c-Jun stability and persistent migration.” 2011. Doctoral Dissertation, Vanderbilt University. Accessed September 19, 2020. http://hdl.handle.net/1803/12548.

MLA Handbook (7th Edition):

Reiner, Jennifer Elisabeth. “Oncogenic STRAP regulates c-Jun stability and persistent migration.” 2011. Web. 19 Sep 2020.

Vancouver:

Reiner JE. Oncogenic STRAP regulates c-Jun stability and persistent migration. [Internet] [Doctoral dissertation]. Vanderbilt University; 2011. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1803/12548.

Council of Science Editors:

Reiner JE. Oncogenic STRAP regulates c-Jun stability and persistent migration. [Doctoral Dissertation]. Vanderbilt University; 2011. Available from: http://hdl.handle.net/1803/12548


Vanderbilt University

4. Saxon, Jamie Ausborn. Investigation of epithelial canonical and non-canonical NF-κB signaling in lung adenocarcinoma.

Degree: PhD, Cancer Biology, 2016, Vanderbilt University

 Nuclear factor κ-light chain enhancer of activated B cells (NF-κB) is a transcription factor that can be activated through canonical or non-canonical signaling pathways, and… (more)

Subjects/Keywords: macrophages; cell cycle; inflammation; lung cancer; mouse models; ARDS; apoptosis

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APA (6th Edition):

Saxon, J. A. (2016). Investigation of epithelial canonical and non-canonical NF-κB signaling in lung adenocarcinoma. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10550

Chicago Manual of Style (16th Edition):

Saxon, Jamie Ausborn. “Investigation of epithelial canonical and non-canonical NF-κB signaling in lung adenocarcinoma.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed September 19, 2020. http://hdl.handle.net/1803/10550.

MLA Handbook (7th Edition):

Saxon, Jamie Ausborn. “Investigation of epithelial canonical and non-canonical NF-κB signaling in lung adenocarcinoma.” 2016. Web. 19 Sep 2020.

Vancouver:

Saxon JA. Investigation of epithelial canonical and non-canonical NF-κB signaling in lung adenocarcinoma. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1803/10550.

Council of Science Editors:

Saxon JA. Investigation of epithelial canonical and non-canonical NF-κB signaling in lung adenocarcinoma. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://hdl.handle.net/1803/10550


Vanderbilt University

5. Perry, Allyson Gail. The Role of Nuclear Factor Kappa B in Myeloid Cells During Lung Carcinogenesis.

Degree: PhD, Cancer Biology, 2016, Vanderbilt University

 Despite recent progress, non-small cell lung cancer (NSCLC) remains the leading cause of cancer-related mortality in the United States and new therapeutic approaches are needed.… (more)

Subjects/Keywords: lung cancer; myeloid cell; neutrophil; interleukin-1 beta; nuclear factor kappa B; cathepsin G; inflammation

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APA (6th Edition):

Perry, A. G. (2016). The Role of Nuclear Factor Kappa B in Myeloid Cells During Lung Carcinogenesis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14295

Chicago Manual of Style (16th Edition):

Perry, Allyson Gail. “The Role of Nuclear Factor Kappa B in Myeloid Cells During Lung Carcinogenesis.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed September 19, 2020. http://hdl.handle.net/1803/14295.

MLA Handbook (7th Edition):

Perry, Allyson Gail. “The Role of Nuclear Factor Kappa B in Myeloid Cells During Lung Carcinogenesis.” 2016. Web. 19 Sep 2020.

Vancouver:

Perry AG. The Role of Nuclear Factor Kappa B in Myeloid Cells During Lung Carcinogenesis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1803/14295.

Council of Science Editors:

Perry AG. The Role of Nuclear Factor Kappa B in Myeloid Cells During Lung Carcinogenesis. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://hdl.handle.net/1803/14295


Vanderbilt University

6. Greenplate, Allison Rae. A Comprehensive Framework for Systems Immune Monitoring in Patients with Cancer.

Degree: PhD, Microbiology and Immunology, 2018, Vanderbilt University

 The immune system is a complex network of cells spread throughout the body, spanning dozens of tissue types and locations. A complete understanding of a… (more)

Subjects/Keywords: immunology; melanoma; cancer; systems immunology; T cells

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APA (6th Edition):

Greenplate, A. R. (2018). A Comprehensive Framework for Systems Immune Monitoring in Patients with Cancer. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11081

Chicago Manual of Style (16th Edition):

Greenplate, Allison Rae. “A Comprehensive Framework for Systems Immune Monitoring in Patients with Cancer.” 2018. Doctoral Dissertation, Vanderbilt University. Accessed September 19, 2020. http://hdl.handle.net/1803/11081.

MLA Handbook (7th Edition):

Greenplate, Allison Rae. “A Comprehensive Framework for Systems Immune Monitoring in Patients with Cancer.” 2018. Web. 19 Sep 2020.

Vancouver:

Greenplate AR. A Comprehensive Framework for Systems Immune Monitoring in Patients with Cancer. [Internet] [Doctoral dissertation]. Vanderbilt University; 2018. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1803/11081.

Council of Science Editors:

Greenplate AR. A Comprehensive Framework for Systems Immune Monitoring in Patients with Cancer. [Doctoral Dissertation]. Vanderbilt University; 2018. Available from: http://hdl.handle.net/1803/11081


Vanderbilt University

7. Gordy, Laura Elizabeth. Repertoire selection and effector differentiation during NKT cell development.

Degree: PhD, Microbiology and Immunology, 2012, Vanderbilt University

 Natural killer T (NKT) cells are innate-like lymphocytes that develop and mature in the thymus. From there, they home to the peripheral lymphoid tissues where,… (more)

Subjects/Keywords: T-bet; IL-15; Bcl-xL; development; survival; NKT cells; Nur77; negative selection

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APA (6th Edition):

Gordy, L. E. (2012). Repertoire selection and effector differentiation during NKT cell development. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11710

Chicago Manual of Style (16th Edition):

Gordy, Laura Elizabeth. “Repertoire selection and effector differentiation during NKT cell development.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed September 19, 2020. http://hdl.handle.net/1803/11710.

MLA Handbook (7th Edition):

Gordy, Laura Elizabeth. “Repertoire selection and effector differentiation during NKT cell development.” 2012. Web. 19 Sep 2020.

Vancouver:

Gordy LE. Repertoire selection and effector differentiation during NKT cell development. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1803/11710.

Council of Science Editors:

Gordy LE. Repertoire selection and effector differentiation during NKT cell development. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://hdl.handle.net/1803/11710


Vanderbilt University

8. Lu, Xinyuan. LZAP and PPM phosphatases: reciprocal regulation and shared mechanisms altering tumor behavior.

Degree: PhD, Cancer Biology, 2013, Vanderbilt University

 LZAP (Cdk5rap3, C53) is a putative tumor suppressor lost in 30% of human head and neck squamous cell carcinoma. LZAP depletion enhances cell invasion, xenograft… (more)

Subjects/Keywords: post-translational modification; phosphatase; tumor suppressor; NF kappa B; metastasis; PPM1A

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APA (6th Edition):

Lu, X. (2013). LZAP and PPM phosphatases: reciprocal regulation and shared mechanisms altering tumor behavior. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12947

Chicago Manual of Style (16th Edition):

Lu, Xinyuan. “LZAP and PPM phosphatases: reciprocal regulation and shared mechanisms altering tumor behavior.” 2013. Doctoral Dissertation, Vanderbilt University. Accessed September 19, 2020. http://hdl.handle.net/1803/12947.

MLA Handbook (7th Edition):

Lu, Xinyuan. “LZAP and PPM phosphatases: reciprocal regulation and shared mechanisms altering tumor behavior.” 2013. Web. 19 Sep 2020.

Vancouver:

Lu X. LZAP and PPM phosphatases: reciprocal regulation and shared mechanisms altering tumor behavior. [Internet] [Doctoral dissertation]. Vanderbilt University; 2013. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1803/12947.

Council of Science Editors:

Lu X. LZAP and PPM phosphatases: reciprocal regulation and shared mechanisms altering tumor behavior. [Doctoral Dissertation]. Vanderbilt University; 2013. Available from: http://hdl.handle.net/1803/12947


Vanderbilt University

9. Rabacal, Whitney Amber Sachi. The Role of Transcription Factor Krüppel-like Factor 2 in Lymphocyte Migration and Homeostasis.

Degree: PhD, Microbiology and Immunology, 2016, Vanderbilt University

 The immune system is charged with the task of protecting the body from pathogens and clearing apoptotic or cancerous cells to prevent chronic disease. Controlled… (more)

Subjects/Keywords: IL-15; natural killer cells; lymphocyte migration; KLF2; cancer therapies

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APA (6th Edition):

Rabacal, W. A. S. (2016). The Role of Transcription Factor Krüppel-like Factor 2 in Lymphocyte Migration and Homeostasis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/13925

Chicago Manual of Style (16th Edition):

Rabacal, Whitney Amber Sachi. “The Role of Transcription Factor Krüppel-like Factor 2 in Lymphocyte Migration and Homeostasis.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed September 19, 2020. http://hdl.handle.net/1803/13925.

MLA Handbook (7th Edition):

Rabacal, Whitney Amber Sachi. “The Role of Transcription Factor Krüppel-like Factor 2 in Lymphocyte Migration and Homeostasis.” 2016. Web. 19 Sep 2020.

Vancouver:

Rabacal WAS. The Role of Transcription Factor Krüppel-like Factor 2 in Lymphocyte Migration and Homeostasis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1803/13925.

Council of Science Editors:

Rabacal WAS. The Role of Transcription Factor Krüppel-like Factor 2 in Lymphocyte Migration and Homeostasis. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://hdl.handle.net/1803/13925


Vanderbilt University

10. Loomans, Holli Ann. Loss of ACVRIB-dependent Activin A signaling induces esophageal and head and neck carcinoma aggressiveness.

Degree: PhD, Cancer Biology, 2017, Vanderbilt University

 It was been well established that the Activin A signaling pathway plays a pivotal role in the developing, adult, and diseased organism, commonly acting as… (more)

Subjects/Keywords: adhesion; three-dimensional culture; dysplasia; fibroblast; cell invasion; extracellular matrix

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APA (6th Edition):

Loomans, H. A. (2017). Loss of ACVRIB-dependent Activin A signaling induces esophageal and head and neck carcinoma aggressiveness. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12234

Chicago Manual of Style (16th Edition):

Loomans, Holli Ann. “Loss of ACVRIB-dependent Activin A signaling induces esophageal and head and neck carcinoma aggressiveness.” 2017. Doctoral Dissertation, Vanderbilt University. Accessed September 19, 2020. http://hdl.handle.net/1803/12234.

MLA Handbook (7th Edition):

Loomans, Holli Ann. “Loss of ACVRIB-dependent Activin A signaling induces esophageal and head and neck carcinoma aggressiveness.” 2017. Web. 19 Sep 2020.

Vancouver:

Loomans HA. Loss of ACVRIB-dependent Activin A signaling induces esophageal and head and neck carcinoma aggressiveness. [Internet] [Doctoral dissertation]. Vanderbilt University; 2017. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1803/12234.

Council of Science Editors:

Loomans HA. Loss of ACVRIB-dependent Activin A signaling induces esophageal and head and neck carcinoma aggressiveness. [Doctoral Dissertation]. Vanderbilt University; 2017. Available from: http://hdl.handle.net/1803/12234


Vanderbilt University

11. Hutchinson, Katherine Emily. Identification of Novel Targets for Therapy in Solid Tumors.

Degree: PhD, Cancer Biology, 2015, Vanderbilt University

 Solid tumor treatment paradigms have drastically improved in recent decades through direct targeting of the protein products of somatic, constitutively active “driver” mutations and their… (more)

Subjects/Keywords: next-generation sequencing; targeted therapy; cancer; melanoma

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APA (6th Edition):

Hutchinson, K. E. (2015). Identification of Novel Targets for Therapy in Solid Tumors. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/15255

Chicago Manual of Style (16th Edition):

Hutchinson, Katherine Emily. “Identification of Novel Targets for Therapy in Solid Tumors.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed September 19, 2020. http://hdl.handle.net/1803/15255.

MLA Handbook (7th Edition):

Hutchinson, Katherine Emily. “Identification of Novel Targets for Therapy in Solid Tumors.” 2015. Web. 19 Sep 2020.

Vancouver:

Hutchinson KE. Identification of Novel Targets for Therapy in Solid Tumors. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1803/15255.

Council of Science Editors:

Hutchinson KE. Identification of Novel Targets for Therapy in Solid Tumors. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://hdl.handle.net/1803/15255


Vanderbilt University

12. Crowder, Spencer William. Multiplex biomaterial matrix cues regulate redox status and stemness in human mesenchymal stem cells.

Degree: PhD, Biomedical Engineering, 2014, Vanderbilt University

 Human mesenchymal stem cells (hMSCs) offer therapeutic potential for clinical applications but exhibit a decline in overall health when isolated from aging patients or serially… (more)

Subjects/Keywords: stemness; copolymers; biomaterials; cell-material interactions; stem cells

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APA (6th Edition):

Crowder, S. W. (2014). Multiplex biomaterial matrix cues regulate redox status and stemness in human mesenchymal stem cells. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11315

Chicago Manual of Style (16th Edition):

Crowder, Spencer William. “Multiplex biomaterial matrix cues regulate redox status and stemness in human mesenchymal stem cells.” 2014. Doctoral Dissertation, Vanderbilt University. Accessed September 19, 2020. http://hdl.handle.net/1803/11315.

MLA Handbook (7th Edition):

Crowder, Spencer William. “Multiplex biomaterial matrix cues regulate redox status and stemness in human mesenchymal stem cells.” 2014. Web. 19 Sep 2020.

Vancouver:

Crowder SW. Multiplex biomaterial matrix cues regulate redox status and stemness in human mesenchymal stem cells. [Internet] [Doctoral dissertation]. Vanderbilt University; 2014. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1803/11315.

Council of Science Editors:

Crowder SW. Multiplex biomaterial matrix cues regulate redox status and stemness in human mesenchymal stem cells. [Doctoral Dissertation]. Vanderbilt University; 2014. Available from: http://hdl.handle.net/1803/11315


Vanderbilt University

13. Sakrikar, Dhananjay. Paying attention to the details: rare genetic variation in the dopamine transporter and ADHD.

Degree: PhD, Neuroscience, 2012, Vanderbilt University

 Attention-Deficit Hyperactivity Disorder (ADHD) is the most commonly diagnosed disorder of school-age children. Although genetic and brain imaging studies suggest a contribution of altered dopamine… (more)

Subjects/Keywords: Dopamine Transporter; ADHD

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APA (6th Edition):

Sakrikar, D. (2012). Paying attention to the details: rare genetic variation in the dopamine transporter and ADHD. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11416

Chicago Manual of Style (16th Edition):

Sakrikar, Dhananjay. “Paying attention to the details: rare genetic variation in the dopamine transporter and ADHD.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed September 19, 2020. http://hdl.handle.net/1803/11416.

MLA Handbook (7th Edition):

Sakrikar, Dhananjay. “Paying attention to the details: rare genetic variation in the dopamine transporter and ADHD.” 2012. Web. 19 Sep 2020.

Vancouver:

Sakrikar D. Paying attention to the details: rare genetic variation in the dopamine transporter and ADHD. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1803/11416.

Council of Science Editors:

Sakrikar D. Paying attention to the details: rare genetic variation in the dopamine transporter and ADHD. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://hdl.handle.net/1803/11416


Vanderbilt University

14. Austin, David C. The Role of Nuclear Factor Kappa B in Benign Prostatic Hyperplasia.

Degree: PhD, Cancer Biology, 2016, Vanderbilt University

 Benign prostatic hyperplasia (BPH) is a common, progressive chronic disease. Inflammation is associated with prostatic enlargement and resistance to 5?-reductase inhibitor (5ARI) therapy. Activation of… (more)

Subjects/Keywords: Androgen Receptor Variant 7; Inflammation; Androgen Receptor

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APA (6th Edition):

Austin, D. C. (2016). The Role of Nuclear Factor Kappa B in Benign Prostatic Hyperplasia. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10843

Chicago Manual of Style (16th Edition):

Austin, David C. “The Role of Nuclear Factor Kappa B in Benign Prostatic Hyperplasia.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed September 19, 2020. http://hdl.handle.net/1803/10843.

MLA Handbook (7th Edition):

Austin, David C. “The Role of Nuclear Factor Kappa B in Benign Prostatic Hyperplasia.” 2016. Web. 19 Sep 2020.

Vancouver:

Austin DC. The Role of Nuclear Factor Kappa B in Benign Prostatic Hyperplasia. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1803/10843.

Council of Science Editors:

Austin DC. The Role of Nuclear Factor Kappa B in Benign Prostatic Hyperplasia. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://hdl.handle.net/1803/10843


Vanderbilt University

15. Jowhar, Dawit Kamil. Characterizing the mechanisms that regulate cell polarity during eukaryotic chemotaxis.

Degree: PhD, Biological Sciences, 2013, Vanderbilt University

 Cells that are migrating in a chemical gradient display a polarized distribution of signaling and cytoskeletal components. We have investigated the mechanism of polarity establishment… (more)

Subjects/Keywords: Dictyostelium; cell polarity; Ras; PTEN. microtubules; microfluidics; actin; PIP2

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APA (6th Edition):

Jowhar, D. K. (2013). Characterizing the mechanisms that regulate cell polarity during eukaryotic chemotaxis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/13186

Chicago Manual of Style (16th Edition):

Jowhar, Dawit Kamil. “Characterizing the mechanisms that regulate cell polarity during eukaryotic chemotaxis.” 2013. Doctoral Dissertation, Vanderbilt University. Accessed September 19, 2020. http://hdl.handle.net/1803/13186.

MLA Handbook (7th Edition):

Jowhar, Dawit Kamil. “Characterizing the mechanisms that regulate cell polarity during eukaryotic chemotaxis.” 2013. Web. 19 Sep 2020.

Vancouver:

Jowhar DK. Characterizing the mechanisms that regulate cell polarity during eukaryotic chemotaxis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2013. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1803/13186.

Council of Science Editors:

Jowhar DK. Characterizing the mechanisms that regulate cell polarity during eukaryotic chemotaxis. [Doctoral Dissertation]. Vanderbilt University; 2013. Available from: http://hdl.handle.net/1803/13186


Vanderbilt University

16. Vollbrecht, Peter John. A Role for Astrocytes in Dopamine-Glutamate Interactions of the Prefrontal Cortex.

Degree: PhD, Neuroscience, 2014, Vanderbilt University

 Both dopamine and glutamate are critically involved in cognitive processes such as working memory. Evidence has demonstrated that manipulation of either neurotransmitter can lead to… (more)

Subjects/Keywords: Schizophrenia; EAAT2; GLT-1; Glutamate; Dopamine; Prefrontal Cortex

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APA (6th Edition):

Vollbrecht, P. J. (2014). A Role for Astrocytes in Dopamine-Glutamate Interactions of the Prefrontal Cortex. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11230

Chicago Manual of Style (16th Edition):

Vollbrecht, Peter John. “A Role for Astrocytes in Dopamine-Glutamate Interactions of the Prefrontal Cortex.” 2014. Doctoral Dissertation, Vanderbilt University. Accessed September 19, 2020. http://hdl.handle.net/1803/11230.

MLA Handbook (7th Edition):

Vollbrecht, Peter John. “A Role for Astrocytes in Dopamine-Glutamate Interactions of the Prefrontal Cortex.” 2014. Web. 19 Sep 2020.

Vancouver:

Vollbrecht PJ. A Role for Astrocytes in Dopamine-Glutamate Interactions of the Prefrontal Cortex. [Internet] [Doctoral dissertation]. Vanderbilt University; 2014. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1803/11230.

Council of Science Editors:

Vollbrecht PJ. A Role for Astrocytes in Dopamine-Glutamate Interactions of the Prefrontal Cortex. [Doctoral Dissertation]. Vanderbilt University; 2014. Available from: http://hdl.handle.net/1803/11230


Vanderbilt University

17. Hardeman, Keisha Nicole. Cellular Metabolism Contributes To Therapeutic Responses in BRAF-Mutated Melanomas.

Degree: PhD, Cancer Biology, 2017, Vanderbilt University

 Melanoma is the deadliest form of skin cancer, and virtually all patients progress on targeted therapies. Dysregulated metabolism has been shown to affect therapy response,… (more)

Subjects/Keywords: metabolic reprogramming; melanoma; cellular metabolism; glycolysis

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APA (6th Edition):

Hardeman, K. N. (2017). Cellular Metabolism Contributes To Therapeutic Responses in BRAF-Mutated Melanomas. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12123

Chicago Manual of Style (16th Edition):

Hardeman, Keisha Nicole. “Cellular Metabolism Contributes To Therapeutic Responses in BRAF-Mutated Melanomas.” 2017. Doctoral Dissertation, Vanderbilt University. Accessed September 19, 2020. http://hdl.handle.net/1803/12123.

MLA Handbook (7th Edition):

Hardeman, Keisha Nicole. “Cellular Metabolism Contributes To Therapeutic Responses in BRAF-Mutated Melanomas.” 2017. Web. 19 Sep 2020.

Vancouver:

Hardeman KN. Cellular Metabolism Contributes To Therapeutic Responses in BRAF-Mutated Melanomas. [Internet] [Doctoral dissertation]. Vanderbilt University; 2017. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1803/12123.

Council of Science Editors:

Hardeman KN. Cellular Metabolism Contributes To Therapeutic Responses in BRAF-Mutated Melanomas. [Doctoral Dissertation]. Vanderbilt University; 2017. Available from: http://hdl.handle.net/1803/12123


Vanderbilt University

18. Capozzi, Megan Elise. The Role of Epoxygenated Fatty Acids in Diabetic Retinopathy.

Degree: PhD, Molecular Physiology and Biophysics, 2016, Vanderbilt University

 Diabetic retinopathy (DR) is the leading cause of blindness in working age Americans. The early stages of DR pathogenesis include chronic inflammation, which can eventually… (more)

Subjects/Keywords: diabetic retinopathy

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APA (6th Edition):

Capozzi, M. E. (2016). The Role of Epoxygenated Fatty Acids in Diabetic Retinopathy. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/15289

Chicago Manual of Style (16th Edition):

Capozzi, Megan Elise. “The Role of Epoxygenated Fatty Acids in Diabetic Retinopathy.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed September 19, 2020. http://hdl.handle.net/1803/15289.

MLA Handbook (7th Edition):

Capozzi, Megan Elise. “The Role of Epoxygenated Fatty Acids in Diabetic Retinopathy.” 2016. Web. 19 Sep 2020.

Vancouver:

Capozzi ME. The Role of Epoxygenated Fatty Acids in Diabetic Retinopathy. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1803/15289.

Council of Science Editors:

Capozzi ME. The Role of Epoxygenated Fatty Acids in Diabetic Retinopathy. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://hdl.handle.net/1803/15289


Vanderbilt University

19. He, Wenjuan. Examination of the Cytoprotective Role of Sirtuin-1 in the Renal Medulla.

Degree: PhD, Molecular Physiology and Biophysics, 2010, Vanderbilt University

 Sirtuin 1 (Sirt1) is a NAD+-dependent deacetylase that exerts many of the pleiotropic effects of oxidative metabolism. Due to local hypoxia and hypertonicity, the renal… (more)

Subjects/Keywords: Oxidative stress; renal medulla; sirtuin

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APA (6th Edition):

He, W. (2010). Examination of the Cytoprotective Role of Sirtuin-1 in the Renal Medulla. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12836

Chicago Manual of Style (16th Edition):

He, Wenjuan. “Examination of the Cytoprotective Role of Sirtuin-1 in the Renal Medulla.” 2010. Doctoral Dissertation, Vanderbilt University. Accessed September 19, 2020. http://hdl.handle.net/1803/12836.

MLA Handbook (7th Edition):

He, Wenjuan. “Examination of the Cytoprotective Role of Sirtuin-1 in the Renal Medulla.” 2010. Web. 19 Sep 2020.

Vancouver:

He W. Examination of the Cytoprotective Role of Sirtuin-1 in the Renal Medulla. [Internet] [Doctoral dissertation]. Vanderbilt University; 2010. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1803/12836.

Council of Science Editors:

He W. Examination of the Cytoprotective Role of Sirtuin-1 in the Renal Medulla. [Doctoral Dissertation]. Vanderbilt University; 2010. Available from: http://hdl.handle.net/1803/12836


Vanderbilt University

20. Fang, Wei Bin. The Role of EphA2 RTK in Breast Cancer Cell Malignancy and Tumor Angiogenesis.

Degree: PhD, Cancer Biology, 2008, Vanderbilt University

 The EphA2 receptor belongs to the recently cloned Eph family of receptor tyrosine kinase (RTK). High levels of EphA2 RTK have been detected in 60-90%… (more)

Subjects/Keywords: Receptor Tyrosine Kinase; Breast Cancer; EphA2; Neovascularization  – Regulation; Protein-tyrosine kinase  – Receptors

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APA (6th Edition):

Fang, W. B. (2008). The Role of EphA2 RTK in Breast Cancer Cell Malignancy and Tumor Angiogenesis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/13970

Chicago Manual of Style (16th Edition):

Fang, Wei Bin. “The Role of EphA2 RTK in Breast Cancer Cell Malignancy and Tumor Angiogenesis.” 2008. Doctoral Dissertation, Vanderbilt University. Accessed September 19, 2020. http://hdl.handle.net/1803/13970.

MLA Handbook (7th Edition):

Fang, Wei Bin. “The Role of EphA2 RTK in Breast Cancer Cell Malignancy and Tumor Angiogenesis.” 2008. Web. 19 Sep 2020.

Vancouver:

Fang WB. The Role of EphA2 RTK in Breast Cancer Cell Malignancy and Tumor Angiogenesis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2008. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1803/13970.

Council of Science Editors:

Fang WB. The Role of EphA2 RTK in Breast Cancer Cell Malignancy and Tumor Angiogenesis. [Doctoral Dissertation]. Vanderbilt University; 2008. Available from: http://hdl.handle.net/1803/13970


Vanderbilt University

21. Zhuang, Guanglei. Pro and Anti Tumorigenic Effects of EPHA Receptor Signaling in Breast and Lung Cancer.

Degree: PhD, Cancer Biology, 2010, Vanderbilt University

 The Eph family of receptor tyrosine kinases has important roles in multiple aspects of cancer development and progression. Substantial advances have been made in understanding… (more)

Subjects/Keywords: EphA2; EphA3; cancer; HER2

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APA (6th Edition):

Zhuang, G. (2010). Pro and Anti Tumorigenic Effects of EPHA Receptor Signaling in Breast and Lung Cancer. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11279

Chicago Manual of Style (16th Edition):

Zhuang, Guanglei. “Pro and Anti Tumorigenic Effects of EPHA Receptor Signaling in Breast and Lung Cancer.” 2010. Doctoral Dissertation, Vanderbilt University. Accessed September 19, 2020. http://hdl.handle.net/1803/11279.

MLA Handbook (7th Edition):

Zhuang, Guanglei. “Pro and Anti Tumorigenic Effects of EPHA Receptor Signaling in Breast and Lung Cancer.” 2010. Web. 19 Sep 2020.

Vancouver:

Zhuang G. Pro and Anti Tumorigenic Effects of EPHA Receptor Signaling in Breast and Lung Cancer. [Internet] [Doctoral dissertation]. Vanderbilt University; 2010. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1803/11279.

Council of Science Editors:

Zhuang G. Pro and Anti Tumorigenic Effects of EPHA Receptor Signaling in Breast and Lung Cancer. [Doctoral Dissertation]. Vanderbilt University; 2010. Available from: http://hdl.handle.net/1803/11279


Vanderbilt University

22. Russell, Alisha Joy. The role of CCAAT/enhancer binding protein beta2 in mammary epithelial cells.

Degree: PhD, Cancer Biology, 2010, Vanderbilt University

 C/EBPbeta is essential for mammary gland growth and development and has been associated with poor prognosis in breast cancer. Overexpression of C/EBPbeta2 in MCF10A cells,… (more)

Subjects/Keywords: C/EBPbeta; interleukin-1beta; bioinformatics; breast cancer

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APA (6th Edition):

Russell, A. J. (2010). The role of CCAAT/enhancer binding protein beta2 in mammary epithelial cells. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/13981

Chicago Manual of Style (16th Edition):

Russell, Alisha Joy. “The role of CCAAT/enhancer binding protein beta2 in mammary epithelial cells.” 2010. Doctoral Dissertation, Vanderbilt University. Accessed September 19, 2020. http://hdl.handle.net/1803/13981.

MLA Handbook (7th Edition):

Russell, Alisha Joy. “The role of CCAAT/enhancer binding protein beta2 in mammary epithelial cells.” 2010. Web. 19 Sep 2020.

Vancouver:

Russell AJ. The role of CCAAT/enhancer binding protein beta2 in mammary epithelial cells. [Internet] [Doctoral dissertation]. Vanderbilt University; 2010. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1803/13981.

Council of Science Editors:

Russell AJ. The role of CCAAT/enhancer binding protein beta2 in mammary epithelial cells. [Doctoral Dissertation]. Vanderbilt University; 2010. Available from: http://hdl.handle.net/1803/13981


Vanderbilt University

23. DeBusk, Laura M. AKT/IKKα/VAV1 signaling in endothelial cell survival and angiogenesis.

Degree: PhD, Cancer Biology, 2008, Vanderbilt University

 We have identified a novel signaling pathway, AKT/IKKα/VAV1, which induces endothelial cell survival and motility. Ang1/Tie2 and VEGF/VEGFR signaling activates Akt and induces cell survival.… (more)

Subjects/Keywords: angiogenesis; Neovascularization  – Regulation; Vascular endothelial growth factors  – Pathophysiology; Cellular control mechanisms

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APA (6th Edition):

DeBusk, L. M. (2008). AKT/IKKα/VAV1 signaling in endothelial cell survival and angiogenesis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11831

Chicago Manual of Style (16th Edition):

DeBusk, Laura M. “AKT/IKKα/VAV1 signaling in endothelial cell survival and angiogenesis.” 2008. Doctoral Dissertation, Vanderbilt University. Accessed September 19, 2020. http://hdl.handle.net/1803/11831.

MLA Handbook (7th Edition):

DeBusk, Laura M. “AKT/IKKα/VAV1 signaling in endothelial cell survival and angiogenesis.” 2008. Web. 19 Sep 2020.

Vancouver:

DeBusk LM. AKT/IKKα/VAV1 signaling in endothelial cell survival and angiogenesis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2008. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1803/11831.

Council of Science Editors:

DeBusk LM. AKT/IKKα/VAV1 signaling in endothelial cell survival and angiogenesis. [Doctoral Dissertation]. Vanderbilt University; 2008. Available from: http://hdl.handle.net/1803/11831


Vanderbilt University

24. Neel, Nicole Fowler. Regulation of CXC Chemokine Receptor Function Through Intracellular Trafficking and Novel Receptor-Interacting Proteins.

Degree: PhD, Cancer Biology, 2008, Vanderbilt University

 Chemokines are a family of small chemotactic cytokines that bind seven transmembrane G protein-coupled receptors. Roles for CXC chemokines and the receptors CXCR2 and CXCR4… (more)

Subjects/Keywords: VASP; breast cancer; proteomics; RhoB; intracellular trafficking; chemokine receptor; CXCR4; CXCR2

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APA (6th Edition):

Neel, N. F. (2008). Regulation of CXC Chemokine Receptor Function Through Intracellular Trafficking and Novel Receptor-Interacting Proteins. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10677

Chicago Manual of Style (16th Edition):

Neel, Nicole Fowler. “Regulation of CXC Chemokine Receptor Function Through Intracellular Trafficking and Novel Receptor-Interacting Proteins.” 2008. Doctoral Dissertation, Vanderbilt University. Accessed September 19, 2020. http://hdl.handle.net/1803/10677.

MLA Handbook (7th Edition):

Neel, Nicole Fowler. “Regulation of CXC Chemokine Receptor Function Through Intracellular Trafficking and Novel Receptor-Interacting Proteins.” 2008. Web. 19 Sep 2020.

Vancouver:

Neel NF. Regulation of CXC Chemokine Receptor Function Through Intracellular Trafficking and Novel Receptor-Interacting Proteins. [Internet] [Doctoral dissertation]. Vanderbilt University; 2008. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1803/10677.

Council of Science Editors:

Neel NF. Regulation of CXC Chemokine Receptor Function Through Intracellular Trafficking and Novel Receptor-Interacting Proteins. [Doctoral Dissertation]. Vanderbilt University; 2008. Available from: http://hdl.handle.net/1803/10677


Vanderbilt University

25. Werdich, Xiang Qi. Characterization of Src family kinases as potential targets for intervention in vascular endothelial growth factor-mediated retinal neovascularization.

Degree: PhD, Cell and Developmental Biology, 2005, Vanderbilt University

 Hypoxia inducible vascular endothelial growth factor (VEGF) plays a major role in initiation and regulation of retinal neovascularization, which is the leading cause of severe… (more)

Subjects/Keywords: signal transduction; RNA interference; retinal ischemia; cellular function; receptor

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APA (6th Edition):

Werdich, X. Q. (2005). Characterization of Src family kinases as potential targets for intervention in vascular endothelial growth factor-mediated retinal neovascularization. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11671

Chicago Manual of Style (16th Edition):

Werdich, Xiang Qi. “Characterization of Src family kinases as potential targets for intervention in vascular endothelial growth factor-mediated retinal neovascularization.” 2005. Doctoral Dissertation, Vanderbilt University. Accessed September 19, 2020. http://hdl.handle.net/1803/11671.

MLA Handbook (7th Edition):

Werdich, Xiang Qi. “Characterization of Src family kinases as potential targets for intervention in vascular endothelial growth factor-mediated retinal neovascularization.” 2005. Web. 19 Sep 2020.

Vancouver:

Werdich XQ. Characterization of Src family kinases as potential targets for intervention in vascular endothelial growth factor-mediated retinal neovascularization. [Internet] [Doctoral dissertation]. Vanderbilt University; 2005. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1803/11671.

Council of Science Editors:

Werdich XQ. Characterization of Src family kinases as potential targets for intervention in vascular endothelial growth factor-mediated retinal neovascularization. [Doctoral Dissertation]. Vanderbilt University; 2005. Available from: http://hdl.handle.net/1803/11671


Vanderbilt University

26. Everhart, Michael Brett. The role of Nuclear Factor-kappa B (NF-kB) in the regulation of lung inflammation.

Degree: PhD, Cell and Developmental Biology, 2004, Vanderbilt University

 The NF-kappaB pathway has been shown to play a critical role in both adaptive and innate immunity and has been implicated as a focal point… (more)

Subjects/Keywords: inflammation; cell signaling; nuclear factor-kappa B; lung

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APA (6th Edition):

Everhart, M. B. (2004). The role of Nuclear Factor-kappa B (NF-kB) in the regulation of lung inflammation. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14942

Chicago Manual of Style (16th Edition):

Everhart, Michael Brett. “The role of Nuclear Factor-kappa B (NF-kB) in the regulation of lung inflammation.” 2004. Doctoral Dissertation, Vanderbilt University. Accessed September 19, 2020. http://hdl.handle.net/1803/14942.

MLA Handbook (7th Edition):

Everhart, Michael Brett. “The role of Nuclear Factor-kappa B (NF-kB) in the regulation of lung inflammation.” 2004. Web. 19 Sep 2020.

Vancouver:

Everhart MB. The role of Nuclear Factor-kappa B (NF-kB) in the regulation of lung inflammation. [Internet] [Doctoral dissertation]. Vanderbilt University; 2004. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1803/14942.

Council of Science Editors:

Everhart MB. The role of Nuclear Factor-kappa B (NF-kB) in the regulation of lung inflammation. [Doctoral Dissertation]. Vanderbilt University; 2004. Available from: http://hdl.handle.net/1803/14942


Vanderbilt University

27. Ducharme, Nicole Annette. The Role of Rab11-FIP2 in Epithelial Cells.

Degree: PhD, Cell and Developmental Biology, 2007, Vanderbilt University

 The small GTPase Rab11 family proteins have been implicated in the plasma membrane recycling system in such diverse model systems as H/K-ATPase trafficking in parietal… (more)

Subjects/Keywords: polarized cells; Rab11; Rab11-FIP2; epithelial cells

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APA (6th Edition):

Ducharme, N. A. (2007). The Role of Rab11-FIP2 in Epithelial Cells. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11100

Chicago Manual of Style (16th Edition):

Ducharme, Nicole Annette. “The Role of Rab11-FIP2 in Epithelial Cells.” 2007. Doctoral Dissertation, Vanderbilt University. Accessed September 19, 2020. http://hdl.handle.net/1803/11100.

MLA Handbook (7th Edition):

Ducharme, Nicole Annette. “The Role of Rab11-FIP2 in Epithelial Cells.” 2007. Web. 19 Sep 2020.

Vancouver:

Ducharme NA. The Role of Rab11-FIP2 in Epithelial Cells. [Internet] [Doctoral dissertation]. Vanderbilt University; 2007. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1803/11100.

Council of Science Editors:

Ducharme NA. The Role of Rab11-FIP2 in Epithelial Cells. [Doctoral Dissertation]. Vanderbilt University; 2007. Available from: http://hdl.handle.net/1803/11100

.