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You searched for +publisher:"Vanderbilt University" +contributor:("Andrea Page-McCaw"). Showing records 1 – 18 of 18 total matches.

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Vanderbilt University

1. Merkle, Julie Ann. Regulation of Drosophila early embryogenesis and genome maintenance by the E3 ubiquitin ligase no poles.

Degree: PhD, Cell and Developmental Biology, 2011, Vanderbilt University

 In a screen for cell-cycle regulators, we identified a Drosophila maternal effect-lethal mutant that we named âno polesâ (nopo). Embryos from nopo females undergo mitotic… (more)

Subjects/Keywords: translesion synthesis; embryogensis; Drosophila; E3 ubiquitin ligase; genome maintenance

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APA (6th Edition):

Merkle, J. A. (2011). Regulation of Drosophila early embryogenesis and genome maintenance by the E3 ubiquitin ligase no poles. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-07212011-151147/ ;

Chicago Manual of Style (16th Edition):

Merkle, Julie Ann. “Regulation of Drosophila early embryogenesis and genome maintenance by the E3 ubiquitin ligase no poles.” 2011. Doctoral Dissertation, Vanderbilt University. Accessed January 17, 2020. http://etd.library.vanderbilt.edu/available/etd-07212011-151147/ ;.

MLA Handbook (7th Edition):

Merkle, Julie Ann. “Regulation of Drosophila early embryogenesis and genome maintenance by the E3 ubiquitin ligase no poles.” 2011. Web. 17 Jan 2020.

Vancouver:

Merkle JA. Regulation of Drosophila early embryogenesis and genome maintenance by the E3 ubiquitin ligase no poles. [Internet] [Doctoral dissertation]. Vanderbilt University; 2011. [cited 2020 Jan 17]. Available from: http://etd.library.vanderbilt.edu/available/etd-07212011-151147/ ;.

Council of Science Editors:

Merkle JA. Regulation of Drosophila early embryogenesis and genome maintenance by the E3 ubiquitin ligase no poles. [Doctoral Dissertation]. Vanderbilt University; 2011. Available from: http://etd.library.vanderbilt.edu/available/etd-07212011-151147/ ;


Vanderbilt University

2. Stevens, Laura Jeanette. The Regulation and Essential Functions of Matrix Metalloproteinases during Wound Healing.

Degree: PhD, Cell and Developmental Biology, 2012, Vanderbilt University

 Wound healing, an essential function to the survival of all multicellular organisms, requires the precise orchestration of multiple cell types in order to repair the… (more)

Subjects/Keywords: MMP; JNK; re-epithelialization; cell migration

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APA (6th Edition):

Stevens, L. J. (2012). The Regulation and Essential Functions of Matrix Metalloproteinases during Wound Healing. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-03132012-101745/ ;

Chicago Manual of Style (16th Edition):

Stevens, Laura Jeanette. “The Regulation and Essential Functions of Matrix Metalloproteinases during Wound Healing.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed January 17, 2020. http://etd.library.vanderbilt.edu/available/etd-03132012-101745/ ;.

MLA Handbook (7th Edition):

Stevens, Laura Jeanette. “The Regulation and Essential Functions of Matrix Metalloproteinases during Wound Healing.” 2012. Web. 17 Jan 2020.

Vancouver:

Stevens LJ. The Regulation and Essential Functions of Matrix Metalloproteinases during Wound Healing. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2020 Jan 17]. Available from: http://etd.library.vanderbilt.edu/available/etd-03132012-101745/ ;.

Council of Science Editors:

Stevens LJ. The Regulation and Essential Functions of Matrix Metalloproteinases during Wound Healing. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://etd.library.vanderbilt.edu/available/etd-03132012-101745/ ;


Vanderbilt University

3. Guyer, Richard Allen. Tumor suppressor mechanisms of the polarity protein Par3.

Degree: PhD, Cell and Developmental Biology, 2015, Vanderbilt University

 Proteins that regulate cell polarity are fundamental for metazoan biology and are necessary for proper development of tissues and organs. In light of polarity genesâ… (more)

Subjects/Keywords: aPKC; polarity; Par3; Stat3

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APA (6th Edition):

Guyer, R. A. (2015). Tumor suppressor mechanisms of the polarity protein Par3. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-07172015-083313/ ;

Chicago Manual of Style (16th Edition):

Guyer, Richard Allen. “Tumor suppressor mechanisms of the polarity protein Par3.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed January 17, 2020. http://etd.library.vanderbilt.edu/available/etd-07172015-083313/ ;.

MLA Handbook (7th Edition):

Guyer, Richard Allen. “Tumor suppressor mechanisms of the polarity protein Par3.” 2015. Web. 17 Jan 2020.

Vancouver:

Guyer RA. Tumor suppressor mechanisms of the polarity protein Par3. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2020 Jan 17]. Available from: http://etd.library.vanderbilt.edu/available/etd-07172015-083313/ ;.

Council of Science Editors:

Guyer RA. Tumor suppressor mechanisms of the polarity protein Par3. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://etd.library.vanderbilt.edu/available/etd-07172015-083313/ ;


Vanderbilt University

4. Sanders, Lehanna Nalani. The role of the BMP antagonist Gremlin 2 during cardiac tissue repair.

Degree: PhD, Cell and Developmental Biology, 2016, Vanderbilt University

 CELL AND DEVELOPMENTAL BIOLOGY The role of the BMP antagonist Gremlin2 during cardiac tissue repair Lehanna Nalani Sanders Dissertation under the direction of Professor Antonis… (more)

Subjects/Keywords: BMP signaling; acute MI; Gremlin 2

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APA (6th Edition):

Sanders, L. N. (2016). The role of the BMP antagonist Gremlin 2 during cardiac tissue repair. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu//available/etd-06012016-112723/ ;

Chicago Manual of Style (16th Edition):

Sanders, Lehanna Nalani. “The role of the BMP antagonist Gremlin 2 during cardiac tissue repair.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed January 17, 2020. http://etd.library.vanderbilt.edu//available/etd-06012016-112723/ ;.

MLA Handbook (7th Edition):

Sanders, Lehanna Nalani. “The role of the BMP antagonist Gremlin 2 during cardiac tissue repair.” 2016. Web. 17 Jan 2020.

Vancouver:

Sanders LN. The role of the BMP antagonist Gremlin 2 during cardiac tissue repair. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2020 Jan 17]. Available from: http://etd.library.vanderbilt.edu//available/etd-06012016-112723/ ;.

Council of Science Editors:

Sanders LN. The role of the BMP antagonist Gremlin 2 during cardiac tissue repair. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://etd.library.vanderbilt.edu//available/etd-06012016-112723/ ;


Vanderbilt University

5. LePage, Daniel Paul. The Molecular Basis of Wolbachia-induced Cytoplasmic Incompatibility.

Degree: PhD, Biological Sciences, 2016, Vanderbilt University

 Wolbachia pipientis arguably constitute the largest pandemic of the animal kingdom. Estimated to infect 40% of all arthropod species, these obligate intracellular bacteria are masterful… (more)

Subjects/Keywords: Wolbachia pipientis; cytoplasmic incompatibility; Drosophila melanogaster

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APA (6th Edition):

LePage, D. P. (2016). The Molecular Basis of Wolbachia-induced Cytoplasmic Incompatibility. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu//available/etd-07072016-120006/ ;

Chicago Manual of Style (16th Edition):

LePage, Daniel Paul. “The Molecular Basis of Wolbachia-induced Cytoplasmic Incompatibility.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed January 17, 2020. http://etd.library.vanderbilt.edu//available/etd-07072016-120006/ ;.

MLA Handbook (7th Edition):

LePage, Daniel Paul. “The Molecular Basis of Wolbachia-induced Cytoplasmic Incompatibility.” 2016. Web. 17 Jan 2020.

Vancouver:

LePage DP. The Molecular Basis of Wolbachia-induced Cytoplasmic Incompatibility. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2020 Jan 17]. Available from: http://etd.library.vanderbilt.edu//available/etd-07072016-120006/ ;.

Council of Science Editors:

LePage DP. The Molecular Basis of Wolbachia-induced Cytoplasmic Incompatibility. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://etd.library.vanderbilt.edu//available/etd-07072016-120006/ ;


Vanderbilt University

6. Howard, Angela Marie. Basement Membrane Homeostasis and Repair.

Degree: PhD, Cell and Developmental Biology, 2019, Vanderbilt University

 The basement membrane is a sheet-like extracellular matrix that wraps around muscle fibers and underlies epithelia. Although the basement membrane is often considered to be… (more)

Subjects/Keywords: Basement Membrane; Dextran Sodium Sulfate; Drosophila

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APA (6th Edition):

Howard, A. M. (2019). Basement Membrane Homeostasis and Repair. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-03222019-152412/ ;

Chicago Manual of Style (16th Edition):

Howard, Angela Marie. “Basement Membrane Homeostasis and Repair.” 2019. Doctoral Dissertation, Vanderbilt University. Accessed January 17, 2020. http://etd.library.vanderbilt.edu/available/etd-03222019-152412/ ;.

MLA Handbook (7th Edition):

Howard, Angela Marie. “Basement Membrane Homeostasis and Repair.” 2019. Web. 17 Jan 2020.

Vancouver:

Howard AM. Basement Membrane Homeostasis and Repair. [Internet] [Doctoral dissertation]. Vanderbilt University; 2019. [cited 2020 Jan 17]. Available from: http://etd.library.vanderbilt.edu/available/etd-03222019-152412/ ;.

Council of Science Editors:

Howard AM. Basement Membrane Homeostasis and Repair. [Doctoral Dissertation]. Vanderbilt University; 2019. Available from: http://etd.library.vanderbilt.edu/available/etd-03222019-152412/ ;


Vanderbilt University

7. Liu, Jing. Ngn3-expressing progenitor heterogeneity drives endocrine lineage allocation in pancreas development.

Degree: PhD, Cell and Developmental Biology, 2015, Vanderbilt University

 Diabetes is a worldwide health issue. In both type I and late stage type II diabetes, significant beta-cell loss causes insulin deficiency and hyperglycemia. Replenishing… (more)

Subjects/Keywords: Neurogenin 3/Ngn3; beta cell; Cre/loxP; lineage tracing

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APA (6th Edition):

Liu, J. (2015). Ngn3-expressing progenitor heterogeneity drives endocrine lineage allocation in pancreas development. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-11152015-162440/ ;

Chicago Manual of Style (16th Edition):

Liu, Jing. “Ngn3-expressing progenitor heterogeneity drives endocrine lineage allocation in pancreas development.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed January 17, 2020. http://etd.library.vanderbilt.edu/available/etd-11152015-162440/ ;.

MLA Handbook (7th Edition):

Liu, Jing. “Ngn3-expressing progenitor heterogeneity drives endocrine lineage allocation in pancreas development.” 2015. Web. 17 Jan 2020.

Vancouver:

Liu J. Ngn3-expressing progenitor heterogeneity drives endocrine lineage allocation in pancreas development. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2020 Jan 17]. Available from: http://etd.library.vanderbilt.edu/available/etd-11152015-162440/ ;.

Council of Science Editors:

Liu J. Ngn3-expressing progenitor heterogeneity drives endocrine lineage allocation in pancreas development. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://etd.library.vanderbilt.edu/available/etd-11152015-162440/ ;


Vanderbilt University

8. Armstrong, Laura Craig. Modeling Tuberous Sclerosis Complex Using Patient-Derived Cells.

Degree: PhD, Cell and Developmental Biology, 2017, Vanderbilt University

 Tuberous Sclerosis Complex (TSC) is a pediatric disorder of dysregulated growth and differentiation caused by loss of function mutations in either the TSC1 or TSC2… (more)

Subjects/Keywords: p53; induced pluripotent stem cells; tuberous sclerosis complex; mTOR

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APA (6th Edition):

Armstrong, L. C. (2017). Modeling Tuberous Sclerosis Complex Using Patient-Derived Cells. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-09162017-134651/ ;

Chicago Manual of Style (16th Edition):

Armstrong, Laura Craig. “Modeling Tuberous Sclerosis Complex Using Patient-Derived Cells.” 2017. Doctoral Dissertation, Vanderbilt University. Accessed January 17, 2020. http://etd.library.vanderbilt.edu/available/etd-09162017-134651/ ;.

MLA Handbook (7th Edition):

Armstrong, Laura Craig. “Modeling Tuberous Sclerosis Complex Using Patient-Derived Cells.” 2017. Web. 17 Jan 2020.

Vancouver:

Armstrong LC. Modeling Tuberous Sclerosis Complex Using Patient-Derived Cells. [Internet] [Doctoral dissertation]. Vanderbilt University; 2017. [cited 2020 Jan 17]. Available from: http://etd.library.vanderbilt.edu/available/etd-09162017-134651/ ;.

Council of Science Editors:

Armstrong LC. Modeling Tuberous Sclerosis Complex Using Patient-Derived Cells. [Doctoral Dissertation]. Vanderbilt University; 2017. Available from: http://etd.library.vanderbilt.edu/available/etd-09162017-134651/ ;


Vanderbilt University

9. Saito-Diaz, Vicente Kenyi. Regulation of Wnt Receptor Activation by the Tumor Suppressor APC.

Degree: PhD, Cell and Developmental Biology, 2017, Vanderbilt University

 The Wnt pathway is a highly-conserved pathway that controls many developmental processes and is mutated in many human diseases (e.g., cancer). The tumor suppressor adenomatous… (more)

Subjects/Keywords: endocytosis; beta-catenin; LRP6; APC; Wnt signaling; clathrin; caveolin; colorectal cancer

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APA (6th Edition):

Saito-Diaz, V. K. (2017). Regulation of Wnt Receptor Activation by the Tumor Suppressor APC. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-03272017-113107/ ;

Chicago Manual of Style (16th Edition):

Saito-Diaz, Vicente Kenyi. “Regulation of Wnt Receptor Activation by the Tumor Suppressor APC.” 2017. Doctoral Dissertation, Vanderbilt University. Accessed January 17, 2020. http://etd.library.vanderbilt.edu/available/etd-03272017-113107/ ;.

MLA Handbook (7th Edition):

Saito-Diaz, Vicente Kenyi. “Regulation of Wnt Receptor Activation by the Tumor Suppressor APC.” 2017. Web. 17 Jan 2020.

Vancouver:

Saito-Diaz VK. Regulation of Wnt Receptor Activation by the Tumor Suppressor APC. [Internet] [Doctoral dissertation]. Vanderbilt University; 2017. [cited 2020 Jan 17]. Available from: http://etd.library.vanderbilt.edu/available/etd-03272017-113107/ ;.

Council of Science Editors:

Saito-Diaz VK. Regulation of Wnt Receptor Activation by the Tumor Suppressor APC. [Doctoral Dissertation]. Vanderbilt University; 2017. Available from: http://etd.library.vanderbilt.edu/available/etd-03272017-113107/ ;


Vanderbilt University

10. Diggins, Nicole Lee. AAPPL1 inhibits cell migration by modulating integrin trafficking and Rac signaling.

Degree: PhD, Biological Sciences, 2018, Vanderbilt University

 Cell migration is vital to numerous biological processes, and is misregulated in pathological processes, such as cancer metastasis. Cell migration is a tightly, spatiotemporally regulated… (more)

Subjects/Keywords: adaptor protein; integrin; cell migration; endosome

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APA (6th Edition):

Diggins, N. L. (2018). AAPPL1 inhibits cell migration by modulating integrin trafficking and Rac signaling. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-02132018-120305/ ;

Chicago Manual of Style (16th Edition):

Diggins, Nicole Lee. “AAPPL1 inhibits cell migration by modulating integrin trafficking and Rac signaling.” 2018. Doctoral Dissertation, Vanderbilt University. Accessed January 17, 2020. http://etd.library.vanderbilt.edu/available/etd-02132018-120305/ ;.

MLA Handbook (7th Edition):

Diggins, Nicole Lee. “AAPPL1 inhibits cell migration by modulating integrin trafficking and Rac signaling.” 2018. Web. 17 Jan 2020.

Vancouver:

Diggins NL. AAPPL1 inhibits cell migration by modulating integrin trafficking and Rac signaling. [Internet] [Doctoral dissertation]. Vanderbilt University; 2018. [cited 2020 Jan 17]. Available from: http://etd.library.vanderbilt.edu/available/etd-02132018-120305/ ;.

Council of Science Editors:

Diggins NL. AAPPL1 inhibits cell migration by modulating integrin trafficking and Rac signaling. [Doctoral Dissertation]. Vanderbilt University; 2018. Available from: http://etd.library.vanderbilt.edu/available/etd-02132018-120305/ ;


Vanderbilt University

11. Nielsen, Casey Paulasue. Regulation of WNT pathway specification by DUB-E3 interactions.

Degree: PhD, Cell and Developmental Biology, 2019, Vanderbilt University

 The WNT signaling network is comprised of multiple receptors that relay various input signals via distinct transduction pathways to execute multiple complex and context-specific output… (more)

Subjects/Keywords: Ubiquitin proteasome system; Cell signaling; WNT pathway; ubiquitin rheostat; breast cancer cells; deubiquitylases

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APA (6th Edition):

Nielsen, C. P. (2019). Regulation of WNT pathway specification by DUB-E3 interactions. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-08192019-173731/ ;

Chicago Manual of Style (16th Edition):

Nielsen, Casey Paulasue. “Regulation of WNT pathway specification by DUB-E3 interactions.” 2019. Doctoral Dissertation, Vanderbilt University. Accessed January 17, 2020. http://etd.library.vanderbilt.edu/available/etd-08192019-173731/ ;.

MLA Handbook (7th Edition):

Nielsen, Casey Paulasue. “Regulation of WNT pathway specification by DUB-E3 interactions.” 2019. Web. 17 Jan 2020.

Vancouver:

Nielsen CP. Regulation of WNT pathway specification by DUB-E3 interactions. [Internet] [Doctoral dissertation]. Vanderbilt University; 2019. [cited 2020 Jan 17]. Available from: http://etd.library.vanderbilt.edu/available/etd-08192019-173731/ ;.

Council of Science Editors:

Nielsen CP. Regulation of WNT pathway specification by DUB-E3 interactions. [Doctoral Dissertation]. Vanderbilt University; 2019. Available from: http://etd.library.vanderbilt.edu/available/etd-08192019-173731/ ;


Vanderbilt University

12. Fleming, Jonathan Tyler. The sonic hedgehog pathway mediates central regulation of cerebellar development and sarcoma phenotypic outcome.

Degree: PhD, Cell and Developmental Biology, 2014, Vanderbilt University

 Sonic hedgehog (Shh) signaling regulates critical processes during embryonic development and in homeostasis of adult tissues. Deregulated pathway activity is a major factor underlying the… (more)

Subjects/Keywords: Sonic hedgehog cerebellum sarcoma

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APA (6th Edition):

Fleming, J. T. (2014). The sonic hedgehog pathway mediates central regulation of cerebellar development and sarcoma phenotypic outcome. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-04162014-131844/ ;

Chicago Manual of Style (16th Edition):

Fleming, Jonathan Tyler. “The sonic hedgehog pathway mediates central regulation of cerebellar development and sarcoma phenotypic outcome.” 2014. Doctoral Dissertation, Vanderbilt University. Accessed January 17, 2020. http://etd.library.vanderbilt.edu/available/etd-04162014-131844/ ;.

MLA Handbook (7th Edition):

Fleming, Jonathan Tyler. “The sonic hedgehog pathway mediates central regulation of cerebellar development and sarcoma phenotypic outcome.” 2014. Web. 17 Jan 2020.

Vancouver:

Fleming JT. The sonic hedgehog pathway mediates central regulation of cerebellar development and sarcoma phenotypic outcome. [Internet] [Doctoral dissertation]. Vanderbilt University; 2014. [cited 2020 Jan 17]. Available from: http://etd.library.vanderbilt.edu/available/etd-04162014-131844/ ;.

Council of Science Editors:

Fleming JT. The sonic hedgehog pathway mediates central regulation of cerebellar development and sarcoma phenotypic outcome. [Doctoral Dissertation]. Vanderbilt University; 2014. Available from: http://etd.library.vanderbilt.edu/available/etd-04162014-131844/ ;


Vanderbilt University

13. Hainline, Sarah Grace. Regulation and Requirement of MCPH1 and Rough Deal during Drosophila Early Embryogenesis.

Degree: PhD, Cell and Developmental Biology, 2014, Vanderbilt University

 In a biochemical screen Anaphase-Promoting Complex (APC) substrates, we identified Drosophila MCPH1 isoform B (dMCPH1-B) as a substrate of APCCdh1. dMCPH1-B undergoes Cdh1-dependent degradation in… (more)

Subjects/Keywords: Spindle Assembly Checkpoint; Anaphase Promoting Complex; cell cycle; Drosophila

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APA (6th Edition):

Hainline, S. G. (2014). Regulation and Requirement of MCPH1 and Rough Deal during Drosophila Early Embryogenesis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu//available/etd-08132014-132348/ ;

Chicago Manual of Style (16th Edition):

Hainline, Sarah Grace. “Regulation and Requirement of MCPH1 and Rough Deal during Drosophila Early Embryogenesis.” 2014. Doctoral Dissertation, Vanderbilt University. Accessed January 17, 2020. http://etd.library.vanderbilt.edu//available/etd-08132014-132348/ ;.

MLA Handbook (7th Edition):

Hainline, Sarah Grace. “Regulation and Requirement of MCPH1 and Rough Deal during Drosophila Early Embryogenesis.” 2014. Web. 17 Jan 2020.

Vancouver:

Hainline SG. Regulation and Requirement of MCPH1 and Rough Deal during Drosophila Early Embryogenesis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2014. [cited 2020 Jan 17]. Available from: http://etd.library.vanderbilt.edu//available/etd-08132014-132348/ ;.

Council of Science Editors:

Hainline SG. Regulation and Requirement of MCPH1 and Rough Deal during Drosophila Early Embryogenesis. [Doctoral Dissertation]. Vanderbilt University; 2014. Available from: http://etd.library.vanderbilt.edu//available/etd-08132014-132348/ ;


Vanderbilt University

14. League, Garrett Philip. The Metamorphosis of Mosquito Immune and Circulatory Physiology: Comparative Analyses in Larval and Adult Anopheles gambiae.

Degree: PhD, Biological Sciences, 2017, Vanderbilt University

 Mosquitoes encounter pathogens throughout their holometabolous life cycle, be it in the microbe-rich pools of the aquatic larva stage or the infected blood meals of… (more)

Subjects/Keywords: phagocytosis; heart; contraction; circulation; hemocyte; melanization

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APA (6th Edition):

League, G. P. (2017). The Metamorphosis of Mosquito Immune and Circulatory Physiology: Comparative Analyses in Larval and Adult Anopheles gambiae. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu//available/etd-03242017-105726/ ;

Chicago Manual of Style (16th Edition):

League, Garrett Philip. “The Metamorphosis of Mosquito Immune and Circulatory Physiology: Comparative Analyses in Larval and Adult Anopheles gambiae.” 2017. Doctoral Dissertation, Vanderbilt University. Accessed January 17, 2020. http://etd.library.vanderbilt.edu//available/etd-03242017-105726/ ;.

MLA Handbook (7th Edition):

League, Garrett Philip. “The Metamorphosis of Mosquito Immune and Circulatory Physiology: Comparative Analyses in Larval and Adult Anopheles gambiae.” 2017. Web. 17 Jan 2020.

Vancouver:

League GP. The Metamorphosis of Mosquito Immune and Circulatory Physiology: Comparative Analyses in Larval and Adult Anopheles gambiae. [Internet] [Doctoral dissertation]. Vanderbilt University; 2017. [cited 2020 Jan 17]. Available from: http://etd.library.vanderbilt.edu//available/etd-03242017-105726/ ;.

Council of Science Editors:

League GP. The Metamorphosis of Mosquito Immune and Circulatory Physiology: Comparative Analyses in Larval and Adult Anopheles gambiae. [Doctoral Dissertation]. Vanderbilt University; 2017. Available from: http://etd.library.vanderbilt.edu//available/etd-03242017-105726/ ;

15. Riley, Kimberly Ann Gooding. Modulation of Beta-Cell Intrinsic and Extrinsic Characteristics by CTGF to Promote Beta-Cell Mass Regeneration.

Degree: PhD, Cell and Developmental Biology, 2015, Vanderbilt University

 As diabetes continues to affect millions of people within the United States, identification of novel factors that may enhance beta-cell proliferation and mass regeneration in… (more)

Subjects/Keywords: diabetes; regeneration; beta-cell; CTGF; pancreas; proliferation

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APA (6th Edition):

Riley, K. A. G. (2015). Modulation of Beta-Cell Intrinsic and Extrinsic Characteristics by CTGF to Promote Beta-Cell Mass Regeneration. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu//available/etd-03102015-132801/ ;

Chicago Manual of Style (16th Edition):

Riley, Kimberly Ann Gooding. “Modulation of Beta-Cell Intrinsic and Extrinsic Characteristics by CTGF to Promote Beta-Cell Mass Regeneration.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed January 17, 2020. http://etd.library.vanderbilt.edu//available/etd-03102015-132801/ ;.

MLA Handbook (7th Edition):

Riley, Kimberly Ann Gooding. “Modulation of Beta-Cell Intrinsic and Extrinsic Characteristics by CTGF to Promote Beta-Cell Mass Regeneration.” 2015. Web. 17 Jan 2020.

Vancouver:

Riley KAG. Modulation of Beta-Cell Intrinsic and Extrinsic Characteristics by CTGF to Promote Beta-Cell Mass Regeneration. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2020 Jan 17]. Available from: http://etd.library.vanderbilt.edu//available/etd-03102015-132801/ ;.

Council of Science Editors:

Riley KAG. Modulation of Beta-Cell Intrinsic and Extrinsic Characteristics by CTGF to Promote Beta-Cell Mass Regeneration. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://etd.library.vanderbilt.edu//available/etd-03102015-132801/ ;

16. Walter, Teagan Jo. Investigating the roles of Notch and Vascular Endothelial Growth Factor in Hepatic Cell Fate Decisions and Architectural Establishment in Development and Disease.

Degree: PhD, Cell and Developmental Biology, 2013, Vanderbilt University

 During liver development, precise signaling is required to direct cell fate decisions and architectural establishment. It is hypothesized that the same signaling pathways are important… (more)

Subjects/Keywords: liver; intrahepatic bile duct; portal vein; hepatic artery; progenitor cells

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APA (6th Edition):

Walter, T. J. (2013). Investigating the roles of Notch and Vascular Endothelial Growth Factor in Hepatic Cell Fate Decisions and Architectural Establishment in Development and Disease. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-12022013-130113/ ;

Chicago Manual of Style (16th Edition):

Walter, Teagan Jo. “Investigating the roles of Notch and Vascular Endothelial Growth Factor in Hepatic Cell Fate Decisions and Architectural Establishment in Development and Disease.” 2013. Doctoral Dissertation, Vanderbilt University. Accessed January 17, 2020. http://etd.library.vanderbilt.edu/available/etd-12022013-130113/ ;.

MLA Handbook (7th Edition):

Walter, Teagan Jo. “Investigating the roles of Notch and Vascular Endothelial Growth Factor in Hepatic Cell Fate Decisions and Architectural Establishment in Development and Disease.” 2013. Web. 17 Jan 2020.

Vancouver:

Walter TJ. Investigating the roles of Notch and Vascular Endothelial Growth Factor in Hepatic Cell Fate Decisions and Architectural Establishment in Development and Disease. [Internet] [Doctoral dissertation]. Vanderbilt University; 2013. [cited 2020 Jan 17]. Available from: http://etd.library.vanderbilt.edu/available/etd-12022013-130113/ ;.

Council of Science Editors:

Walter TJ. Investigating the roles of Notch and Vascular Endothelial Growth Factor in Hepatic Cell Fate Decisions and Architectural Establishment in Development and Disease. [Doctoral Dissertation]. Vanderbilt University; 2013. Available from: http://etd.library.vanderbilt.edu/available/etd-12022013-130113/ ;

17. Broderick, Sarah M. Genetic Analysis of Ninjurin A, A stress-regulated potein that induces nonapoptotic cell death.

Degree: PhD, Cell and Developmental Biology, 2013, Vanderbilt University

 Ninjurins are a conserved family of transmembrane proteins that increase expression in response to injury and stress. There are few in vivo studies of Ninjurin,… (more)

Subjects/Keywords: Cell Biology

…Wang, Nicholas Simms, Andrea Page-­‐McCaw Abstract: Ninjurins… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Broderick, S. M. (2013). Genetic Analysis of Ninjurin A, A stress-regulated potein that induces nonapoptotic cell death. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-04182013-154535/ ;

Chicago Manual of Style (16th Edition):

Broderick, Sarah M. “Genetic Analysis of Ninjurin A, A stress-regulated potein that induces nonapoptotic cell death.” 2013. Doctoral Dissertation, Vanderbilt University. Accessed January 17, 2020. http://etd.library.vanderbilt.edu/available/etd-04182013-154535/ ;.

MLA Handbook (7th Edition):

Broderick, Sarah M. “Genetic Analysis of Ninjurin A, A stress-regulated potein that induces nonapoptotic cell death.” 2013. Web. 17 Jan 2020.

Vancouver:

Broderick SM. Genetic Analysis of Ninjurin A, A stress-regulated potein that induces nonapoptotic cell death. [Internet] [Doctoral dissertation]. Vanderbilt University; 2013. [cited 2020 Jan 17]. Available from: http://etd.library.vanderbilt.edu/available/etd-04182013-154535/ ;.

Council of Science Editors:

Broderick SM. Genetic Analysis of Ninjurin A, A stress-regulated potein that induces nonapoptotic cell death. [Doctoral Dissertation]. Vanderbilt University; 2013. Available from: http://etd.library.vanderbilt.edu/available/etd-04182013-154535/ ;

18. Bennett, Monica Elaine. Balancing act: investigating the coordinated mechanics of germband and amnioserosa in Drosophila morphogenesis.

Degree: PhD, Physics, 2017, Vanderbilt University

 In the series of morphogenetic events that are required for Drosophila development, some of the most dramatic are those that involve the coordinated movement of… (more)

Subjects/Keywords: biophysics; Drosophila; embryogenesis; tissue mechanics; amnioserosa; germband; retraction; reverse modeling

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bennett, M. E. (2017). Balancing act: investigating the coordinated mechanics of germband and amnioserosa in Drosophila morphogenesis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-03272017-112322/ ;

Chicago Manual of Style (16th Edition):

Bennett, Monica Elaine. “Balancing act: investigating the coordinated mechanics of germband and amnioserosa in Drosophila morphogenesis.” 2017. Doctoral Dissertation, Vanderbilt University. Accessed January 17, 2020. http://etd.library.vanderbilt.edu/available/etd-03272017-112322/ ;.

MLA Handbook (7th Edition):

Bennett, Monica Elaine. “Balancing act: investigating the coordinated mechanics of germband and amnioserosa in Drosophila morphogenesis.” 2017. Web. 17 Jan 2020.

Vancouver:

Bennett ME. Balancing act: investigating the coordinated mechanics of germband and amnioserosa in Drosophila morphogenesis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2017. [cited 2020 Jan 17]. Available from: http://etd.library.vanderbilt.edu/available/etd-03272017-112322/ ;.

Council of Science Editors:

Bennett ME. Balancing act: investigating the coordinated mechanics of germband and amnioserosa in Drosophila morphogenesis. [Doctoral Dissertation]. Vanderbilt University; 2017. Available from: http://etd.library.vanderbilt.edu/available/etd-03272017-112322/ ;

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