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You searched for +publisher:"Vanderbilt University" +contributor:("Alyssa Hasty"). Showing records 1 – 19 of 19 total matches.

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Vanderbilt University

1. Day, Charles Asher. Role of Cytoskeleton in Cholera Toxin Diffusion and Endocytosis.

Degree: PhD, Molecular Physiology and Biophysics, 2013, Vanderbilt University

 The plasma membrane is highly organized as a means to regulate various processes including sorting of cargo for endocytosis. However, the mechanisms responsible for this… (more)

Subjects/Keywords: microtubule; cytoplasmic dynein; actin; caveolae; diffusion; cholera toxin; lipid raft; endocytosis

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APA (6th Edition):

Day, C. A. (2013). Role of Cytoskeleton in Cholera Toxin Diffusion and Endocytosis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12181

Chicago Manual of Style (16th Edition):

Day, Charles Asher. “Role of Cytoskeleton in Cholera Toxin Diffusion and Endocytosis.” 2013. Doctoral Dissertation, Vanderbilt University. Accessed April 15, 2021. http://hdl.handle.net/1803/12181.

MLA Handbook (7th Edition):

Day, Charles Asher. “Role of Cytoskeleton in Cholera Toxin Diffusion and Endocytosis.” 2013. Web. 15 Apr 2021.

Vancouver:

Day CA. Role of Cytoskeleton in Cholera Toxin Diffusion and Endocytosis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2013. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1803/12181.

Council of Science Editors:

Day CA. Role of Cytoskeleton in Cholera Toxin Diffusion and Endocytosis. [Doctoral Dissertation]. Vanderbilt University; 2013. Available from: http://hdl.handle.net/1803/12181


Vanderbilt University

2. Hickman, Francis Edward. Mechanisms of p75 neurotrophin receptor signaling.

Degree: PhD, Neuroscience, 2018, Vanderbilt University

 The p75 neurotrophin receptor (p75NTR) plays an integral role in patterning the sympathetic nervous system during development. Initially, p75NTR is expressed at low levels as… (more)

Subjects/Keywords: p75; neurotrophin; receptor trafficking; NGF; NT3; TrkA

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APA (6th Edition):

Hickman, F. E. (2018). Mechanisms of p75 neurotrophin receptor signaling. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12800

Chicago Manual of Style (16th Edition):

Hickman, Francis Edward. “Mechanisms of p75 neurotrophin receptor signaling.” 2018. Doctoral Dissertation, Vanderbilt University. Accessed April 15, 2021. http://hdl.handle.net/1803/12800.

MLA Handbook (7th Edition):

Hickman, Francis Edward. “Mechanisms of p75 neurotrophin receptor signaling.” 2018. Web. 15 Apr 2021.

Vancouver:

Hickman FE. Mechanisms of p75 neurotrophin receptor signaling. [Internet] [Doctoral dissertation]. Vanderbilt University; 2018. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1803/12800.

Council of Science Editors:

Hickman FE. Mechanisms of p75 neurotrophin receptor signaling. [Doctoral Dissertation]. Vanderbilt University; 2018. Available from: http://hdl.handle.net/1803/12800


Vanderbilt University

3. Barke, Theresa Leigh. The Intersection of Maternal Inflammatory Stress and the Developmental Origins of Health and Disease.

Degree: PhD, Microbiology and Immunology, 2019, Vanderbilt University

 The most important factor influencing the prenatal environment is the maternal environment. There is a growing body of evidence suggesting that the maternal and intrauterine… (more)

Subjects/Keywords: Sexual Dimorphism; Placental Macrophage; Gestational Diabetes; Maternal Immune Activation; Reproductive Immunology

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APA (6th Edition):

Barke, T. L. (2019). The Intersection of Maternal Inflammatory Stress and the Developmental Origins of Health and Disease. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10970

Chicago Manual of Style (16th Edition):

Barke, Theresa Leigh. “The Intersection of Maternal Inflammatory Stress and the Developmental Origins of Health and Disease.” 2019. Doctoral Dissertation, Vanderbilt University. Accessed April 15, 2021. http://hdl.handle.net/1803/10970.

MLA Handbook (7th Edition):

Barke, Theresa Leigh. “The Intersection of Maternal Inflammatory Stress and the Developmental Origins of Health and Disease.” 2019. Web. 15 Apr 2021.

Vancouver:

Barke TL. The Intersection of Maternal Inflammatory Stress and the Developmental Origins of Health and Disease. [Internet] [Doctoral dissertation]. Vanderbilt University; 2019. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1803/10970.

Council of Science Editors:

Barke TL. The Intersection of Maternal Inflammatory Stress and the Developmental Origins of Health and Disease. [Doctoral Dissertation]. Vanderbilt University; 2019. Available from: http://hdl.handle.net/1803/10970


Vanderbilt University

4. Stouch, Ashley Nicole. NF-kappa B signaling and inflammasome activation in developing fetal lung macrophages.

Degree: PhD, Cell and Developmental Biology, 2014, Vanderbilt University

 Bronchopulmonary dysplasia is a life-threatening lung disease affecting low birth weight preterm infants. While the occurrence of BPD is correlated with chorioamnionitis, the origination and… (more)

Subjects/Keywords: macrophage; inflammasome; inflammation

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APA (6th Edition):

Stouch, A. N. (2014). NF-kappa B signaling and inflammasome activation in developing fetal lung macrophages. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/15353

Chicago Manual of Style (16th Edition):

Stouch, Ashley Nicole. “NF-kappa B signaling and inflammasome activation in developing fetal lung macrophages.” 2014. Doctoral Dissertation, Vanderbilt University. Accessed April 15, 2021. http://hdl.handle.net/1803/15353.

MLA Handbook (7th Edition):

Stouch, Ashley Nicole. “NF-kappa B signaling and inflammasome activation in developing fetal lung macrophages.” 2014. Web. 15 Apr 2021.

Vancouver:

Stouch AN. NF-kappa B signaling and inflammasome activation in developing fetal lung macrophages. [Internet] [Doctoral dissertation]. Vanderbilt University; 2014. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1803/15353.

Council of Science Editors:

Stouch AN. NF-kappa B signaling and inflammasome activation in developing fetal lung macrophages. [Doctoral Dissertation]. Vanderbilt University; 2014. Available from: http://hdl.handle.net/1803/15353


Vanderbilt University

5. Norlander, Allison Elizabeth. SGK1, influenced by salt and IL-17A, promotes hypertension and end-organ damage.

Degree: PhD, Molecular Physiology and Biophysics, 2017, Vanderbilt University

 Hypertension is a leading cause of cardiovascular morbidity and mortality, leading to myocardial infarction, heart failure, stroke, and chronic kidney disease. We have previously shown… (more)

Subjects/Keywords: immunology; hypertension; kidney

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APA (6th Edition):

Norlander, A. E. (2017). SGK1, influenced by salt and IL-17A, promotes hypertension and end-organ damage. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11007

Chicago Manual of Style (16th Edition):

Norlander, Allison Elizabeth. “SGK1, influenced by salt and IL-17A, promotes hypertension and end-organ damage.” 2017. Doctoral Dissertation, Vanderbilt University. Accessed April 15, 2021. http://hdl.handle.net/1803/11007.

MLA Handbook (7th Edition):

Norlander, Allison Elizabeth. “SGK1, influenced by salt and IL-17A, promotes hypertension and end-organ damage.” 2017. Web. 15 Apr 2021.

Vancouver:

Norlander AE. SGK1, influenced by salt and IL-17A, promotes hypertension and end-organ damage. [Internet] [Doctoral dissertation]. Vanderbilt University; 2017. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1803/11007.

Council of Science Editors:

Norlander AE. SGK1, influenced by salt and IL-17A, promotes hypertension and end-organ damage. [Doctoral Dissertation]. Vanderbilt University; 2017. Available from: http://hdl.handle.net/1803/11007


Vanderbilt University

6. Dale, Bethany Lyn. The Influence of Interleukin 21 and T Follicular Helper Cells in Hypertension and Vascular Dysfunction.

Degree: PhD, Molecular Physiology and Biophysics, 2019, Vanderbilt University

 The role of T and B cells of the adaptive immune have been implicated in hypertension, but the mechanisms by which they produce a coordinated… (more)

Subjects/Keywords: hypertension; vascular dysfunction; interleukin 21; T follicular helper; blood pressure

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APA (6th Edition):

Dale, B. L. (2019). The Influence of Interleukin 21 and T Follicular Helper Cells in Hypertension and Vascular Dysfunction. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/15413

Chicago Manual of Style (16th Edition):

Dale, Bethany Lyn. “The Influence of Interleukin 21 and T Follicular Helper Cells in Hypertension and Vascular Dysfunction.” 2019. Doctoral Dissertation, Vanderbilt University. Accessed April 15, 2021. http://hdl.handle.net/1803/15413.

MLA Handbook (7th Edition):

Dale, Bethany Lyn. “The Influence of Interleukin 21 and T Follicular Helper Cells in Hypertension and Vascular Dysfunction.” 2019. Web. 15 Apr 2021.

Vancouver:

Dale BL. The Influence of Interleukin 21 and T Follicular Helper Cells in Hypertension and Vascular Dysfunction. [Internet] [Doctoral dissertation]. Vanderbilt University; 2019. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1803/15413.

Council of Science Editors:

Dale BL. The Influence of Interleukin 21 and T Follicular Helper Cells in Hypertension and Vascular Dysfunction. [Doctoral Dissertation]. Vanderbilt University; 2019. Available from: http://hdl.handle.net/1803/15413


Vanderbilt University

7. Potter, Leah Ashley. Transcriptional Profiling of Pancreatic Progenitor Cells.

Degree: PhD, Molecular Physiology and Biophysics, 2011, Vanderbilt University

 Pluripotent stem cells, because of their ability to differentiate into any cell type, have been widely advocated as a means of producing a nearly unlimited… (more)

Subjects/Keywords: pancreas; genomics; mouse development; diabetes; RNA-Sequencing; progenitor cell

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APA (6th Edition):

Potter, L. A. (2011). Transcriptional Profiling of Pancreatic Progenitor Cells. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14974

Chicago Manual of Style (16th Edition):

Potter, Leah Ashley. “Transcriptional Profiling of Pancreatic Progenitor Cells.” 2011. Doctoral Dissertation, Vanderbilt University. Accessed April 15, 2021. http://hdl.handle.net/1803/14974.

MLA Handbook (7th Edition):

Potter, Leah Ashley. “Transcriptional Profiling of Pancreatic Progenitor Cells.” 2011. Web. 15 Apr 2021.

Vancouver:

Potter LA. Transcriptional Profiling of Pancreatic Progenitor Cells. [Internet] [Doctoral dissertation]. Vanderbilt University; 2011. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1803/14974.

Council of Science Editors:

Potter LA. Transcriptional Profiling of Pancreatic Progenitor Cells. [Doctoral Dissertation]. Vanderbilt University; 2011. Available from: http://hdl.handle.net/1803/14974


Vanderbilt University

8. Jeff, Janina Maria. The Genetics of Quantitative Traits Associated with Cardiovascular Disease in African Americans.

Degree: PhD, Human Genetics, 2012, Vanderbilt University

 Cardiovascular disease (CVD) is the leading cause of death in most developed countries. In addition to environmental risk factors, such as diet and physical activity,… (more)

Subjects/Keywords: electrocardiographic traits; fibrinogen; African Americans; quantitative traits

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APA (6th Edition):

Jeff, J. M. (2012). The Genetics of Quantitative Traits Associated with Cardiovascular Disease in African Americans. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10690

Chicago Manual of Style (16th Edition):

Jeff, Janina Maria. “The Genetics of Quantitative Traits Associated with Cardiovascular Disease in African Americans.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed April 15, 2021. http://hdl.handle.net/1803/10690.

MLA Handbook (7th Edition):

Jeff, Janina Maria. “The Genetics of Quantitative Traits Associated with Cardiovascular Disease in African Americans.” 2012. Web. 15 Apr 2021.

Vancouver:

Jeff JM. The Genetics of Quantitative Traits Associated with Cardiovascular Disease in African Americans. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1803/10690.

Council of Science Editors:

Jeff JM. The Genetics of Quantitative Traits Associated with Cardiovascular Disease in African Americans. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://hdl.handle.net/1803/10690


Vanderbilt University

9. Mendonsa, Alisha Maria. Cellular and Molecular Changes Impacting Cancer Progression in Non-Alcoholic Fatty Liver Disease.

Degree: PhD, Cancer Biology, 2014, Vanderbilt University

 Non alcoholic fatty liver disease (NAFLD), is recognized as the one of the most common causes of liver disease in the United States and worldwide.… (more)

Subjects/Keywords: NAFLD; liver metastasis; MMP13

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APA (6th Edition):

Mendonsa, A. M. (2014). Cellular and Molecular Changes Impacting Cancer Progression in Non-Alcoholic Fatty Liver Disease. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/15352

Chicago Manual of Style (16th Edition):

Mendonsa, Alisha Maria. “Cellular and Molecular Changes Impacting Cancer Progression in Non-Alcoholic Fatty Liver Disease.” 2014. Doctoral Dissertation, Vanderbilt University. Accessed April 15, 2021. http://hdl.handle.net/1803/15352.

MLA Handbook (7th Edition):

Mendonsa, Alisha Maria. “Cellular and Molecular Changes Impacting Cancer Progression in Non-Alcoholic Fatty Liver Disease.” 2014. Web. 15 Apr 2021.

Vancouver:

Mendonsa AM. Cellular and Molecular Changes Impacting Cancer Progression in Non-Alcoholic Fatty Liver Disease. [Internet] [Doctoral dissertation]. Vanderbilt University; 2014. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1803/15352.

Council of Science Editors:

Mendonsa AM. Cellular and Molecular Changes Impacting Cancer Progression in Non-Alcoholic Fatty Liver Disease. [Doctoral Dissertation]. Vanderbilt University; 2014. Available from: http://hdl.handle.net/1803/15352


Vanderbilt University

10. Wylezinski, Lukasz Szczepan. Activation and Disruption of Brain Microvascular Endothelial Cells by Interleukin 2.

Degree: PhD, Molecular Physiology and Biophysics, 2016, Vanderbilt University

 The pleiotropic cytokine interleukin 2 (IL2) disrupts the blood-brain barrier and alters brain microcirculation, underlying vascular leak syndrome that complicates cancer immunotherapy with IL2. The… (more)

Subjects/Keywords: immunotherapy; permeability; brain microvascular endothelial cells; interleukin 2; adherens junction

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APA (6th Edition):

Wylezinski, L. S. (2016). Activation and Disruption of Brain Microvascular Endothelial Cells by Interleukin 2. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14708

Chicago Manual of Style (16th Edition):

Wylezinski, Lukasz Szczepan. “Activation and Disruption of Brain Microvascular Endothelial Cells by Interleukin 2.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed April 15, 2021. http://hdl.handle.net/1803/14708.

MLA Handbook (7th Edition):

Wylezinski, Lukasz Szczepan. “Activation and Disruption of Brain Microvascular Endothelial Cells by Interleukin 2.” 2016. Web. 15 Apr 2021.

Vancouver:

Wylezinski LS. Activation and Disruption of Brain Microvascular Endothelial Cells by Interleukin 2. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1803/14708.

Council of Science Editors:

Wylezinski LS. Activation and Disruption of Brain Microvascular Endothelial Cells by Interleukin 2. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://hdl.handle.net/1803/14708


Vanderbilt University

11. Sullivan, Chelsea Suzanne. The mechanisms by which apoptotic neurons in the developing dorsal root ganglia are engulfed.

Degree: PhD, Biochemistry, 2014, Vanderbilt University

 During development of the nervous system, approximately 50% of the neurons generated undergo apoptosis as part of a normal pruning process. The neuronal corpses must… (more)

Subjects/Keywords: autoimmunity; satellite glial cells; engulfment; efferocytosis

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APA (6th Edition):

Sullivan, C. S. (2014). The mechanisms by which apoptotic neurons in the developing dorsal root ganglia are engulfed. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14221

Chicago Manual of Style (16th Edition):

Sullivan, Chelsea Suzanne. “The mechanisms by which apoptotic neurons in the developing dorsal root ganglia are engulfed.” 2014. Doctoral Dissertation, Vanderbilt University. Accessed April 15, 2021. http://hdl.handle.net/1803/14221.

MLA Handbook (7th Edition):

Sullivan, Chelsea Suzanne. “The mechanisms by which apoptotic neurons in the developing dorsal root ganglia are engulfed.” 2014. Web. 15 Apr 2021.

Vancouver:

Sullivan CS. The mechanisms by which apoptotic neurons in the developing dorsal root ganglia are engulfed. [Internet] [Doctoral dissertation]. Vanderbilt University; 2014. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1803/14221.

Council of Science Editors:

Sullivan CS. The mechanisms by which apoptotic neurons in the developing dorsal root ganglia are engulfed. [Doctoral Dissertation]. Vanderbilt University; 2014. Available from: http://hdl.handle.net/1803/14221


Vanderbilt University

12. Sedgeman, Leslie Roteta. The pathophysiology and functionality of metabolic microRNAs in Type 2 Diabetes Mellitus.

Degree: PhD, Molecular Physiology and Biophysics, 2018, Vanderbilt University

 microRNAs (miRNA) have been shown to be critical players in metabolism. While most miRNAs studies have focused on their functional role within cells, miRNAs are… (more)

Subjects/Keywords: microRNAs; high-density lipoproteins; diabetes; islet; extracellular RNA

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APA (6th Edition):

Sedgeman, L. R. (2018). The pathophysiology and functionality of metabolic microRNAs in Type 2 Diabetes Mellitus. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14403

Chicago Manual of Style (16th Edition):

Sedgeman, Leslie Roteta. “The pathophysiology and functionality of metabolic microRNAs in Type 2 Diabetes Mellitus.” 2018. Doctoral Dissertation, Vanderbilt University. Accessed April 15, 2021. http://hdl.handle.net/1803/14403.

MLA Handbook (7th Edition):

Sedgeman, Leslie Roteta. “The pathophysiology and functionality of metabolic microRNAs in Type 2 Diabetes Mellitus.” 2018. Web. 15 Apr 2021.

Vancouver:

Sedgeman LR. The pathophysiology and functionality of metabolic microRNAs in Type 2 Diabetes Mellitus. [Internet] [Doctoral dissertation]. Vanderbilt University; 2018. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1803/14403.

Council of Science Editors:

Sedgeman LR. The pathophysiology and functionality of metabolic microRNAs in Type 2 Diabetes Mellitus. [Doctoral Dissertation]. Vanderbilt University; 2018. Available from: http://hdl.handle.net/1803/14403


Vanderbilt University

13. Buckman, Laura Beth. Studies on the Development and Consequences of Neuroinflammation in Obesity.

Degree: PhD, Molecular Physiology and Biophysics, 2014, Vanderbilt University

 In the past decade, evidence has emerged that obesity induces a neuroinflammatory response in the hypothalamus, a part of the brain that contains neuronal circuitry… (more)

Subjects/Keywords: neuroinflammation; diet-induced obesity; CNS; obesity; microglia; astrocytes; high-fat diet

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APA (6th Edition):

Buckman, L. B. (2014). Studies on the Development and Consequences of Neuroinflammation in Obesity. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12699

Chicago Manual of Style (16th Edition):

Buckman, Laura Beth. “Studies on the Development and Consequences of Neuroinflammation in Obesity.” 2014. Doctoral Dissertation, Vanderbilt University. Accessed April 15, 2021. http://hdl.handle.net/1803/12699.

MLA Handbook (7th Edition):

Buckman, Laura Beth. “Studies on the Development and Consequences of Neuroinflammation in Obesity.” 2014. Web. 15 Apr 2021.

Vancouver:

Buckman LB. Studies on the Development and Consequences of Neuroinflammation in Obesity. [Internet] [Doctoral dissertation]. Vanderbilt University; 2014. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1803/12699.

Council of Science Editors:

Buckman LB. Studies on the Development and Consequences of Neuroinflammation in Obesity. [Doctoral Dissertation]. Vanderbilt University; 2014. Available from: http://hdl.handle.net/1803/12699


Vanderbilt University

14. Anderson, Emily Kathleen. The Effects of Obesity, Weight Loss, and Weight Cycling on the Immunometabolic Properties of Mouse Adipose Tissue.

Degree: PhD, Molecular Physiology and Biophysics, 2013, Vanderbilt University

 Obesity is a prevalent disorder that increases the risk of developing many metabolic diseases, including insulin resistance (IR) and type 2 diabetes. Research conducted in… (more)

Subjects/Keywords: obesity; diabetes; adipose tissue; macrophage; t cell

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APA (6th Edition):

Anderson, E. K. (2013). The Effects of Obesity, Weight Loss, and Weight Cycling on the Immunometabolic Properties of Mouse Adipose Tissue. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10929

Chicago Manual of Style (16th Edition):

Anderson, Emily Kathleen. “The Effects of Obesity, Weight Loss, and Weight Cycling on the Immunometabolic Properties of Mouse Adipose Tissue.” 2013. Doctoral Dissertation, Vanderbilt University. Accessed April 15, 2021. http://hdl.handle.net/1803/10929.

MLA Handbook (7th Edition):

Anderson, Emily Kathleen. “The Effects of Obesity, Weight Loss, and Weight Cycling on the Immunometabolic Properties of Mouse Adipose Tissue.” 2013. Web. 15 Apr 2021.

Vancouver:

Anderson EK. The Effects of Obesity, Weight Loss, and Weight Cycling on the Immunometabolic Properties of Mouse Adipose Tissue. [Internet] [Doctoral dissertation]. Vanderbilt University; 2013. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1803/10929.

Council of Science Editors:

Anderson EK. The Effects of Obesity, Weight Loss, and Weight Cycling on the Immunometabolic Properties of Mouse Adipose Tissue. [Doctoral Dissertation]. Vanderbilt University; 2013. Available from: http://hdl.handle.net/1803/10929


Vanderbilt University

15. Peterson, Kristin Rose. Immunometabolism and the role of complement factor 5 in insulin action.

Degree: PhD, Pharmacology, 2019, Vanderbilt University

 Adipose tissue (AT) is a dynamic endocrine organ that contributes to metabolic health. Proper function of resident immune cells is vital to AT function, and… (more)

Subjects/Keywords: obesity; complement factor 5; insulin action; immunometabolism; adipose tissue

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APA (6th Edition):

Peterson, K. R. (2019). Immunometabolism and the role of complement factor 5 in insulin action. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10760

Chicago Manual of Style (16th Edition):

Peterson, Kristin Rose. “Immunometabolism and the role of complement factor 5 in insulin action.” 2019. Doctoral Dissertation, Vanderbilt University. Accessed April 15, 2021. http://hdl.handle.net/1803/10760.

MLA Handbook (7th Edition):

Peterson, Kristin Rose. “Immunometabolism and the role of complement factor 5 in insulin action.” 2019. Web. 15 Apr 2021.

Vancouver:

Peterson KR. Immunometabolism and the role of complement factor 5 in insulin action. [Internet] [Doctoral dissertation]. Vanderbilt University; 2019. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1803/10760.

Council of Science Editors:

Peterson KR. Immunometabolism and the role of complement factor 5 in insulin action. [Doctoral Dissertation]. Vanderbilt University; 2019. Available from: http://hdl.handle.net/1803/10760


Vanderbilt University

16. Panaro, Brandon Lee. Roles of the Melanocortin-4 Receptor in Gut-Brain Communication.

Degree: PhD, Molecular Physiology and Biophysics, 2014, Vanderbilt University

 Loss-of-function mutations causing haploinsufficiency of the MC4R are the most common monogenic cause of severe early-onset obesity in humans. Research has primarily focused on how… (more)

Subjects/Keywords: obesity; melanocortin-4 receptor; mc4r; gut hormones; gut-brain axis; food preference

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APA (6th Edition):

Panaro, B. L. (2014). Roles of the Melanocortin-4 Receptor in Gut-Brain Communication. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/13084

Chicago Manual of Style (16th Edition):

Panaro, Brandon Lee. “Roles of the Melanocortin-4 Receptor in Gut-Brain Communication.” 2014. Doctoral Dissertation, Vanderbilt University. Accessed April 15, 2021. http://hdl.handle.net/1803/13084.

MLA Handbook (7th Edition):

Panaro, Brandon Lee. “Roles of the Melanocortin-4 Receptor in Gut-Brain Communication.” 2014. Web. 15 Apr 2021.

Vancouver:

Panaro BL. Roles of the Melanocortin-4 Receptor in Gut-Brain Communication. [Internet] [Doctoral dissertation]. Vanderbilt University; 2014. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1803/13084.

Council of Science Editors:

Panaro BL. Roles of the Melanocortin-4 Receptor in Gut-Brain Communication. [Doctoral Dissertation]. Vanderbilt University; 2014. Available from: http://hdl.handle.net/1803/13084


Vanderbilt University

17. Johnson, Kathryn Mercedes Stettler. The role of physiological elevations of glucagon-like peptide-one in glucose regulation in the dog in vivo.

Degree: PhD, Molecular Physiology and Biophysics, 2008, Vanderbilt University

 Glucagon-like peptide-one (GLP-1) secreted from the endocrine L cell in the gut after a meal results in elevations of the peptide in arterial blood, the… (more)

Subjects/Keywords: hepatic portal vein; Glucagon-like peptide-1  – Physiological effect; Glucose  – Metabolism; GLP-1

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APA (6th Edition):

Johnson, K. M. S. (2008). The role of physiological elevations of glucagon-like peptide-one in glucose regulation in the dog in vivo. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12078

Chicago Manual of Style (16th Edition):

Johnson, Kathryn Mercedes Stettler. “The role of physiological elevations of glucagon-like peptide-one in glucose regulation in the dog in vivo.” 2008. Doctoral Dissertation, Vanderbilt University. Accessed April 15, 2021. http://hdl.handle.net/1803/12078.

MLA Handbook (7th Edition):

Johnson, Kathryn Mercedes Stettler. “The role of physiological elevations of glucagon-like peptide-one in glucose regulation in the dog in vivo.” 2008. Web. 15 Apr 2021.

Vancouver:

Johnson KMS. The role of physiological elevations of glucagon-like peptide-one in glucose regulation in the dog in vivo. [Internet] [Doctoral dissertation]. Vanderbilt University; 2008. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1803/12078.

Council of Science Editors:

Johnson KMS. The role of physiological elevations of glucagon-like peptide-one in glucose regulation in the dog in vivo. [Doctoral Dissertation]. Vanderbilt University; 2008. Available from: http://hdl.handle.net/1803/12078


Vanderbilt University

18. Ullery, Jody Christine. The Effects of Triglyceride on Lysosomal Function in Macrophage Foam Cells.

Degree: PhD, Pathology, 2009, Vanderbilt University

 Macrophage foam cells are prominent in atherosclerotic lesions. In late stage disease, much of the cholesterol accumulation within foam cells is found in large, swollen,… (more)

Subjects/Keywords: atherosclerosis; cholesterol; triglyceride; macrophage; foam cell; lipids

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ullery, J. C. (2009). The Effects of Triglyceride on Lysosomal Function in Macrophage Foam Cells. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/13538

Chicago Manual of Style (16th Edition):

Ullery, Jody Christine. “The Effects of Triglyceride on Lysosomal Function in Macrophage Foam Cells.” 2009. Doctoral Dissertation, Vanderbilt University. Accessed April 15, 2021. http://hdl.handle.net/1803/13538.

MLA Handbook (7th Edition):

Ullery, Jody Christine. “The Effects of Triglyceride on Lysosomal Function in Macrophage Foam Cells.” 2009. Web. 15 Apr 2021.

Vancouver:

Ullery JC. The Effects of Triglyceride on Lysosomal Function in Macrophage Foam Cells. [Internet] [Doctoral dissertation]. Vanderbilt University; 2009. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1803/13538.

Council of Science Editors:

Ullery JC. The Effects of Triglyceride on Lysosomal Function in Macrophage Foam Cells. [Doctoral Dissertation]. Vanderbilt University; 2009. Available from: http://hdl.handle.net/1803/13538


Vanderbilt University

19. Everett Grueter, Carrie Alicia. The effect of lipids on glucoregulatory hormone actions.

Degree: PhD, Molecular Physiology and Biophysics, 2006, Vanderbilt University

 Glucoregulatory hormones tightly control glucose production and uptake by their direct actions on glucose metabolism and indirect actions on both lipid and protein metabolism. Our… (more)

Subjects/Keywords: Insulin resistance; liver triglycerides; FFA; glucagon; gluconeogenesis; glycogenolysis; Lipids  – Physiological effect; Glucose  – Metabolism

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Everett Grueter, C. A. (2006). The effect of lipids on glucoregulatory hormone actions. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14447

Chicago Manual of Style (16th Edition):

Everett Grueter, Carrie Alicia. “The effect of lipids on glucoregulatory hormone actions.” 2006. Doctoral Dissertation, Vanderbilt University. Accessed April 15, 2021. http://hdl.handle.net/1803/14447.

MLA Handbook (7th Edition):

Everett Grueter, Carrie Alicia. “The effect of lipids on glucoregulatory hormone actions.” 2006. Web. 15 Apr 2021.

Vancouver:

Everett Grueter CA. The effect of lipids on glucoregulatory hormone actions. [Internet] [Doctoral dissertation]. Vanderbilt University; 2006. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1803/14447.

Council of Science Editors:

Everett Grueter CA. The effect of lipids on glucoregulatory hormone actions. [Doctoral Dissertation]. Vanderbilt University; 2006. Available from: http://hdl.handle.net/1803/14447

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