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You searched for +publisher:"University of Toronto" +contributor:("Physiology"). Showing records 1 – 30 of 399 total matches.

[1] [2] [3] [4] [5] … [14]

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University of Toronto

1. Tian, Michael Kexin. Controlling Prefrontal Attention Circuits: Neuromodulation of Cortical Layer 6 and its Local Outputs.

Degree: PhD, 2017, University of Toronto

 The prefrontal cortex is critical for mediating attention. Neuromodulation by acetylcholine and serotonin exerts opposing effects on attention. Layer 6 of prefrontal cortex is an… (more)

Subjects/Keywords: acetylcholine; attention; electrophysiology; layer 6; prefrontal cortex; serotonin; 0719

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APA (6th Edition):

Tian, M. K. (2017). Controlling Prefrontal Attention Circuits: Neuromodulation of Cortical Layer 6 and its Local Outputs. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/79532

Chicago Manual of Style (16th Edition):

Tian, Michael Kexin. “Controlling Prefrontal Attention Circuits: Neuromodulation of Cortical Layer 6 and its Local Outputs.” 2017. Doctoral Dissertation, University of Toronto. Accessed August 15, 2018. http://hdl.handle.net/1807/79532.

MLA Handbook (7th Edition):

Tian, Michael Kexin. “Controlling Prefrontal Attention Circuits: Neuromodulation of Cortical Layer 6 and its Local Outputs.” 2017. Web. 15 Aug 2018.

Vancouver:

Tian MK. Controlling Prefrontal Attention Circuits: Neuromodulation of Cortical Layer 6 and its Local Outputs. [Internet] [Doctoral dissertation]. University of Toronto; 2017. [cited 2018 Aug 15]. Available from: http://hdl.handle.net/1807/79532.

Council of Science Editors:

Tian MK. Controlling Prefrontal Attention Circuits: Neuromodulation of Cortical Layer 6 and its Local Outputs. [Doctoral Dissertation]. University of Toronto; 2017. Available from: http://hdl.handle.net/1807/79532


University of Toronto

2. Ferguson, Katie A.E. Mathematical models provide insight into the role of interneurons in hippocampal CA1 network theta rhythm generation.

Degree: PhD, 2015, University of Toronto

 The theta rhythm, a network rhythm oscillating between 3-12 Hz, is one of the most prominent clocking mechanisms detected in the mammalian brain. These rhythms… (more)

Subjects/Keywords: Computational Neuroscience; Fast-Spiking Interneurons; Hippocampal Microcircuits; Hippocampal Oscillations; Network Models; 0317

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APA (6th Edition):

Ferguson, K. A. E. (2015). Mathematical models provide insight into the role of interneurons in hippocampal CA1 network theta rhythm generation. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/79721

Chicago Manual of Style (16th Edition):

Ferguson, Katie A E. “Mathematical models provide insight into the role of interneurons in hippocampal CA1 network theta rhythm generation.” 2015. Doctoral Dissertation, University of Toronto. Accessed August 15, 2018. http://hdl.handle.net/1807/79721.

MLA Handbook (7th Edition):

Ferguson, Katie A E. “Mathematical models provide insight into the role of interneurons in hippocampal CA1 network theta rhythm generation.” 2015. Web. 15 Aug 2018.

Vancouver:

Ferguson KAE. Mathematical models provide insight into the role of interneurons in hippocampal CA1 network theta rhythm generation. [Internet] [Doctoral dissertation]. University of Toronto; 2015. [cited 2018 Aug 15]. Available from: http://hdl.handle.net/1807/79721.

Council of Science Editors:

Ferguson KAE. Mathematical models provide insight into the role of interneurons in hippocampal CA1 network theta rhythm generation. [Doctoral Dissertation]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/79721


University of Toronto

3. D'Alessandro, Lisa. Developmental Plasticity of Tonotopic Maps in Chinchilla Auditory Midbrain.

Degree: PhD, 2015, University of Toronto

Sensory areas of the brain have the remarkable ability to reorganize following changes in peripheral input, especially during early development. Cortex of altricious species (post-natal… (more)

Subjects/Keywords: c-fos immunohistochemistry; enhanced acoustic environment; inferior colliculus; multi-unit electrophysiology; neuroplasticity; precocious animal model; 0317

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APA (6th Edition):

D'Alessandro, L. (2015). Developmental Plasticity of Tonotopic Maps in Chinchilla Auditory Midbrain. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/81467

Chicago Manual of Style (16th Edition):

D'Alessandro, Lisa. “Developmental Plasticity of Tonotopic Maps in Chinchilla Auditory Midbrain.” 2015. Doctoral Dissertation, University of Toronto. Accessed August 15, 2018. http://hdl.handle.net/1807/81467.

MLA Handbook (7th Edition):

D'Alessandro, Lisa. “Developmental Plasticity of Tonotopic Maps in Chinchilla Auditory Midbrain.” 2015. Web. 15 Aug 2018.

Vancouver:

D'Alessandro L. Developmental Plasticity of Tonotopic Maps in Chinchilla Auditory Midbrain. [Internet] [Doctoral dissertation]. University of Toronto; 2015. [cited 2018 Aug 15]. Available from: http://hdl.handle.net/1807/81467.

Council of Science Editors:

D'Alessandro L. Developmental Plasticity of Tonotopic Maps in Chinchilla Auditory Midbrain. [Doctoral Dissertation]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/81467


University of Toronto

4. D'Alessandro, Lisa. Developmental Plasticity of Tonotopic Maps in Chinchilla Auditory Midbrain.

Degree: PhD, 2015, University of Toronto

Sensory areas of the brain have the remarkable ability to reorganize following changes in peripheral input, especially during early development. Cortex of altricious species (post-natal… (more)

Subjects/Keywords: c-fos immunohistochemistry; enhanced acoustic environment; inferior colliculus; multi-unit electrophysiology; neuroplasticity; precocious animal model; 0317

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APA (6th Edition):

D'Alessandro, L. (2015). Developmental Plasticity of Tonotopic Maps in Chinchilla Auditory Midbrain. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/81468

Chicago Manual of Style (16th Edition):

D'Alessandro, Lisa. “Developmental Plasticity of Tonotopic Maps in Chinchilla Auditory Midbrain.” 2015. Doctoral Dissertation, University of Toronto. Accessed August 15, 2018. http://hdl.handle.net/1807/81468.

MLA Handbook (7th Edition):

D'Alessandro, Lisa. “Developmental Plasticity of Tonotopic Maps in Chinchilla Auditory Midbrain.” 2015. Web. 15 Aug 2018.

Vancouver:

D'Alessandro L. Developmental Plasticity of Tonotopic Maps in Chinchilla Auditory Midbrain. [Internet] [Doctoral dissertation]. University of Toronto; 2015. [cited 2018 Aug 15]. Available from: http://hdl.handle.net/1807/81468.

Council of Science Editors:

D'Alessandro L. Developmental Plasticity of Tonotopic Maps in Chinchilla Auditory Midbrain. [Doctoral Dissertation]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/81468


University of Toronto

5. Eversley, Judith Andrea. Organic Anion Transporter Blockade to Improve Beta Cell Function in Diabetes.

Degree: 2016, University of Toronto

The furan fatty acid metabolite 3-carboxy-4-methyl-5-propyl-2-furanpropanoic acid (CMPF) is elevated in Type 2 Diabetes. CMPF delivery to diabetic levels in mice causes glucose intolerance, impairing… (more)

Subjects/Keywords: Beta Cell; CMPF; Diabetes; OAT3; Organic Anion Transporter; Probenecid; 0719

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APA (6th Edition):

Eversley, J. A. (2016). Organic Anion Transporter Blockade to Improve Beta Cell Function in Diabetes. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/82402

Chicago Manual of Style (16th Edition):

Eversley, Judith Andrea. “Organic Anion Transporter Blockade to Improve Beta Cell Function in Diabetes.” 2016. Masters Thesis, University of Toronto. Accessed August 15, 2018. http://hdl.handle.net/1807/82402.

MLA Handbook (7th Edition):

Eversley, Judith Andrea. “Organic Anion Transporter Blockade to Improve Beta Cell Function in Diabetes.” 2016. Web. 15 Aug 2018.

Vancouver:

Eversley JA. Organic Anion Transporter Blockade to Improve Beta Cell Function in Diabetes. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2018 Aug 15]. Available from: http://hdl.handle.net/1807/82402.

Council of Science Editors:

Eversley JA. Organic Anion Transporter Blockade to Improve Beta Cell Function in Diabetes. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/82402


University of Toronto

6. Kim, Ji-Sun. The Role of Ryanodine Receptor on Neurite Outgrowth under Physiologic and Oxygen and Glucose-deprived Conditions.

Degree: 2016, University of Toronto

Ca2+ can enter a neuron from outside or can be released from internal stores, including endoplasmic reticulum (ER). Ryanodine receptor (RyR) is one of Ca2+… (more)

Subjects/Keywords: 0379

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APA (6th Edition):

Kim, J. (2016). The Role of Ryanodine Receptor on Neurite Outgrowth under Physiologic and Oxygen and Glucose-deprived Conditions. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/82405

Chicago Manual of Style (16th Edition):

Kim, Ji-Sun. “The Role of Ryanodine Receptor on Neurite Outgrowth under Physiologic and Oxygen and Glucose-deprived Conditions.” 2016. Masters Thesis, University of Toronto. Accessed August 15, 2018. http://hdl.handle.net/1807/82405.

MLA Handbook (7th Edition):

Kim, Ji-Sun. “The Role of Ryanodine Receptor on Neurite Outgrowth under Physiologic and Oxygen and Glucose-deprived Conditions.” 2016. Web. 15 Aug 2018.

Vancouver:

Kim J. The Role of Ryanodine Receptor on Neurite Outgrowth under Physiologic and Oxygen and Glucose-deprived Conditions. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2018 Aug 15]. Available from: http://hdl.handle.net/1807/82405.

Council of Science Editors:

Kim J. The Role of Ryanodine Receptor on Neurite Outgrowth under Physiologic and Oxygen and Glucose-deprived Conditions. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/82405


University of Toronto

7. Brathwaite, Shakira. The Role of Toll-like Receptor 4 in the Pathogenesis of Experimental Subarachnoid Hemorrhage.

Degree: 2016, University of Toronto

Subarachnoid hemorrhage (SAH) accounts for 5-7% of strokes worldwide and is associated with high mortality and morbidity. In addition to brain damage from the initial… (more)

Subjects/Keywords: Delayed Cerebral Ischemia; Inflammation; Subarachnoid hemorrhage; Toll-like receptor 4; 0317

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APA (6th Edition):

Brathwaite, S. (2016). The Role of Toll-like Receptor 4 in the Pathogenesis of Experimental Subarachnoid Hemorrhage. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/82406

Chicago Manual of Style (16th Edition):

Brathwaite, Shakira. “The Role of Toll-like Receptor 4 in the Pathogenesis of Experimental Subarachnoid Hemorrhage.” 2016. Masters Thesis, University of Toronto. Accessed August 15, 2018. http://hdl.handle.net/1807/82406.

MLA Handbook (7th Edition):

Brathwaite, Shakira. “The Role of Toll-like Receptor 4 in the Pathogenesis of Experimental Subarachnoid Hemorrhage.” 2016. Web. 15 Aug 2018.

Vancouver:

Brathwaite S. The Role of Toll-like Receptor 4 in the Pathogenesis of Experimental Subarachnoid Hemorrhage. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2018 Aug 15]. Available from: http://hdl.handle.net/1807/82406.

Council of Science Editors:

Brathwaite S. The Role of Toll-like Receptor 4 in the Pathogenesis of Experimental Subarachnoid Hemorrhage. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/82406


University of Toronto

8. Chan, Allen. Mechanisms of Presynaptic CaV2.2 (N-type) Modulation.

Degree: 2008, University of Toronto

Neurotransmitter release at presynaptic terminals is a complex process involving calcium ion influx through voltage-gated calcium channels (CaV). In addition to their role as entry… (more)

Subjects/Keywords: calcium channel; g-protein; voltage-clamp; DRG; presynaptic; CaV2.2; chick; patch-clamp; Munc18; modulation; ion channel; 0719

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APA (6th Edition):

Chan, A. (2008). Mechanisms of Presynaptic CaV2.2 (N-type) Modulation. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/19479

Chicago Manual of Style (16th Edition):

Chan, Allen. “Mechanisms of Presynaptic CaV2.2 (N-type) Modulation.” 2008. Doctoral Dissertation, University of Toronto. Accessed August 15, 2018. http://hdl.handle.net/1807/19479.

MLA Handbook (7th Edition):

Chan, Allen. “Mechanisms of Presynaptic CaV2.2 (N-type) Modulation.” 2008. Web. 15 Aug 2018.

Vancouver:

Chan A. Mechanisms of Presynaptic CaV2.2 (N-type) Modulation. [Internet] [Doctoral dissertation]. University of Toronto; 2008. [cited 2018 Aug 15]. Available from: http://hdl.handle.net/1807/19479.

Council of Science Editors:

Chan A. Mechanisms of Presynaptic CaV2.2 (N-type) Modulation. [Doctoral Dissertation]. University of Toronto; 2008. Available from: http://hdl.handle.net/1807/19479


University of Toronto

9. Venkateswararao, Lakshminarayan Chinta. Adaptive Optimal-control Algorithms for Brainlike Networks.

Degree: 2010, University of Toronto

Many neural control systems are at least roughly optimized, but how is optimal control learned in the brain? There are algorithms for this purpose, but… (more)

Subjects/Keywords: 0719

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APA (6th Edition):

Venkateswararao, L. C. (2010). Adaptive Optimal-control Algorithms for Brainlike Networks. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/24706

Chicago Manual of Style (16th Edition):

Venkateswararao, Lakshminarayan Chinta. “Adaptive Optimal-control Algorithms for Brainlike Networks.” 2010. Doctoral Dissertation, University of Toronto. Accessed August 15, 2018. http://hdl.handle.net/1807/24706.

MLA Handbook (7th Edition):

Venkateswararao, Lakshminarayan Chinta. “Adaptive Optimal-control Algorithms for Brainlike Networks.” 2010. Web. 15 Aug 2018.

Vancouver:

Venkateswararao LC. Adaptive Optimal-control Algorithms for Brainlike Networks. [Internet] [Doctoral dissertation]. University of Toronto; 2010. [cited 2018 Aug 15]. Available from: http://hdl.handle.net/1807/24706.

Council of Science Editors:

Venkateswararao LC. Adaptive Optimal-control Algorithms for Brainlike Networks. [Doctoral Dissertation]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/24706


University of Toronto

10. Han, Lu. Molecular Mechanisms of Glycine Primed NMDA Receptor Internalization.

Degree: 2012, University of Toronto

N-Methyl-D-aspartate receptors (NMDARs) are a principal subtype of excitatory ligandgated ion channel with prominent roles in physiology and disease in the mammalian central nervous system… (more)

Subjects/Keywords: NMDA receptor; Neuroscience; Glycine; Endocytosis; 0317

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APA (6th Edition):

Han, L. (2012). Molecular Mechanisms of Glycine Primed NMDA Receptor Internalization. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/34027

Chicago Manual of Style (16th Edition):

Han, Lu. “Molecular Mechanisms of Glycine Primed NMDA Receptor Internalization.” 2012. Doctoral Dissertation, University of Toronto. Accessed August 15, 2018. http://hdl.handle.net/1807/34027.

MLA Handbook (7th Edition):

Han, Lu. “Molecular Mechanisms of Glycine Primed NMDA Receptor Internalization.” 2012. Web. 15 Aug 2018.

Vancouver:

Han L. Molecular Mechanisms of Glycine Primed NMDA Receptor Internalization. [Internet] [Doctoral dissertation]. University of Toronto; 2012. [cited 2018 Aug 15]. Available from: http://hdl.handle.net/1807/34027.

Council of Science Editors:

Han L. Molecular Mechanisms of Glycine Primed NMDA Receptor Internalization. [Doctoral Dissertation]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/34027


University of Toronto

11. Shathasivam, Premalatha. VEPH1 Regulation of TGF-beta Signalling.

Degree: 2013, University of Toronto

Epithelial ovarian cancer is the leading cause of gynecologic cancer-related mortality. Altered transforming growth factor-β (TGF-β) and wingless and integration 1 (Wnt) signalling are associated… (more)

Subjects/Keywords: VEPH1; ovarian cancer; TGF-beta; 0719

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APA (6th Edition):

Shathasivam, P. (2013). VEPH1 Regulation of TGF-beta Signalling. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/68964

Chicago Manual of Style (16th Edition):

Shathasivam, Premalatha. “VEPH1 Regulation of TGF-beta Signalling.” 2013. Doctoral Dissertation, University of Toronto. Accessed August 15, 2018. http://hdl.handle.net/1807/68964.

MLA Handbook (7th Edition):

Shathasivam, Premalatha. “VEPH1 Regulation of TGF-beta Signalling.” 2013. Web. 15 Aug 2018.

Vancouver:

Shathasivam P. VEPH1 Regulation of TGF-beta Signalling. [Internet] [Doctoral dissertation]. University of Toronto; 2013. [cited 2018 Aug 15]. Available from: http://hdl.handle.net/1807/68964.

Council of Science Editors:

Shathasivam P. VEPH1 Regulation of TGF-beta Signalling. [Doctoral Dissertation]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/68964


University of Toronto

12. Pereira, Sandra M. Differential Effects of Prolonged and Short-term Lipid Infusion on Hepatic Insulin Sensitivity.

Degree: 2013, University of Toronto

To investigate if the mechanisms of free fatty acid (FFA)-induced hepatic insulin resistance differ depending on duration of plasma FFA elevation, i.v. infusion of Intralipid… (more)

Subjects/Keywords: free fatty acids; insulin sensitivity; liver; 0719

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APA (6th Edition):

Pereira, S. M. (2013). Differential Effects of Prolonged and Short-term Lipid Infusion on Hepatic Insulin Sensitivity. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/68983

Chicago Manual of Style (16th Edition):

Pereira, Sandra M. “Differential Effects of Prolonged and Short-term Lipid Infusion on Hepatic Insulin Sensitivity.” 2013. Doctoral Dissertation, University of Toronto. Accessed August 15, 2018. http://hdl.handle.net/1807/68983.

MLA Handbook (7th Edition):

Pereira, Sandra M. “Differential Effects of Prolonged and Short-term Lipid Infusion on Hepatic Insulin Sensitivity.” 2013. Web. 15 Aug 2018.

Vancouver:

Pereira SM. Differential Effects of Prolonged and Short-term Lipid Infusion on Hepatic Insulin Sensitivity. [Internet] [Doctoral dissertation]. University of Toronto; 2013. [cited 2018 Aug 15]. Available from: http://hdl.handle.net/1807/68983.

Council of Science Editors:

Pereira SM. Differential Effects of Prolonged and Short-term Lipid Infusion on Hepatic Insulin Sensitivity. [Doctoral Dissertation]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/68983


University of Toronto

13. Kim, Jung-Eun Jinny. The Role of Suppressor of Fused During Murine Cerebellar Morphogenesis.

Degree: 2013, University of Toronto

Suppressor of Fused (SuFu) is an intracellular mediator of the Hedgehog signaling pathway with diverse biological roles, including a tumour suppressor function. Recent data suggest… (more)

Subjects/Keywords: Cerebellum; Suppressor of Fused; Sonic hedgehog; Differentiation; 0307; 0317

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APA (6th Edition):

Kim, J. J. (2013). The Role of Suppressor of Fused During Murine Cerebellar Morphogenesis. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/68891

Chicago Manual of Style (16th Edition):

Kim, Jung-Eun Jinny. “The Role of Suppressor of Fused During Murine Cerebellar Morphogenesis.” 2013. Doctoral Dissertation, University of Toronto. Accessed August 15, 2018. http://hdl.handle.net/1807/68891.

MLA Handbook (7th Edition):

Kim, Jung-Eun Jinny. “The Role of Suppressor of Fused During Murine Cerebellar Morphogenesis.” 2013. Web. 15 Aug 2018.

Vancouver:

Kim JJ. The Role of Suppressor of Fused During Murine Cerebellar Morphogenesis. [Internet] [Doctoral dissertation]. University of Toronto; 2013. [cited 2018 Aug 15]. Available from: http://hdl.handle.net/1807/68891.

Council of Science Editors:

Kim JJ. The Role of Suppressor of Fused During Murine Cerebellar Morphogenesis. [Doctoral Dissertation]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/68891


University of Toronto

14. Han, Gayoung A. Molecular insights into the mechanisms underlying the pleiotropic functions of Munc18-1 in dense-core vesicle exocytosis.

Degree: 2013, University of Toronto

Exocytosis refers to a cellular process in which an intracellular vesicle fuses its membrane with the plasma membrane to release its contents to the extracellular… (more)

Subjects/Keywords: Munc18-1; exocytosis; dense-core vesicle; 0719

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APA (6th Edition):

Han, G. A. (2013). Molecular insights into the mechanisms underlying the pleiotropic functions of Munc18-1 in dense-core vesicle exocytosis. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/70083

Chicago Manual of Style (16th Edition):

Han, Gayoung A. “Molecular insights into the mechanisms underlying the pleiotropic functions of Munc18-1 in dense-core vesicle exocytosis.” 2013. Doctoral Dissertation, University of Toronto. Accessed August 15, 2018. http://hdl.handle.net/1807/70083.

MLA Handbook (7th Edition):

Han, Gayoung A. “Molecular insights into the mechanisms underlying the pleiotropic functions of Munc18-1 in dense-core vesicle exocytosis.” 2013. Web. 15 Aug 2018.

Vancouver:

Han GA. Molecular insights into the mechanisms underlying the pleiotropic functions of Munc18-1 in dense-core vesicle exocytosis. [Internet] [Doctoral dissertation]. University of Toronto; 2013. [cited 2018 Aug 15]. Available from: http://hdl.handle.net/1807/70083.

Council of Science Editors:

Han GA. Molecular insights into the mechanisms underlying the pleiotropic functions of Munc18-1 in dense-core vesicle exocytosis. [Doctoral Dissertation]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/70083


University of Toronto

15. Wong, Fiona Kar-Mun. The Presynaptic Transmitter Release Site Within the Calcium Nanodomain: Calcium Channel and Synaptic Vesicle Tethering.

Degree: 2014, University of Toronto

Fast neurotransmitter release (NTR) is triggered when the presynaptic membrane is depolarized, opening voltage-sensitive calcium channels (Cavs) and allowing calcium (Ca2+) ions into the terminal… (more)

Subjects/Keywords: presynaptic; calcium channel; synaptic vesicle; tethering; tether; Cav2.2; transmitter release; PDZ; SV-PD; 0317; 0307

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APA (6th Edition):

Wong, F. K. (2014). The Presynaptic Transmitter Release Site Within the Calcium Nanodomain: Calcium Channel and Synaptic Vesicle Tethering. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/72594

Chicago Manual of Style (16th Edition):

Wong, Fiona Kar-Mun. “The Presynaptic Transmitter Release Site Within the Calcium Nanodomain: Calcium Channel and Synaptic Vesicle Tethering.” 2014. Doctoral Dissertation, University of Toronto. Accessed August 15, 2018. http://hdl.handle.net/1807/72594.

MLA Handbook (7th Edition):

Wong, Fiona Kar-Mun. “The Presynaptic Transmitter Release Site Within the Calcium Nanodomain: Calcium Channel and Synaptic Vesicle Tethering.” 2014. Web. 15 Aug 2018.

Vancouver:

Wong FK. The Presynaptic Transmitter Release Site Within the Calcium Nanodomain: Calcium Channel and Synaptic Vesicle Tethering. [Internet] [Doctoral dissertation]. University of Toronto; 2014. [cited 2018 Aug 15]. Available from: http://hdl.handle.net/1807/72594.

Council of Science Editors:

Wong FK. The Presynaptic Transmitter Release Site Within the Calcium Nanodomain: Calcium Channel and Synaptic Vesicle Tethering. [Doctoral Dissertation]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/72594

16. Prescott, Ian A. Synaptic Plasticity of Basal Ganglia Output Neurons in Movement Disorders.

Degree: PhD, 2014, University of Toronto

 Information storage in the central nervous system is believed to emerge from changes in the functional efficacy of synaptic connections, a property known as synaptic… (more)

Subjects/Keywords: 0317

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APA (6th Edition):

Prescott, I. A. (2014). Synaptic Plasticity of Basal Ganglia Output Neurons in Movement Disorders. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/68154

Chicago Manual of Style (16th Edition):

Prescott, Ian A. “Synaptic Plasticity of Basal Ganglia Output Neurons in Movement Disorders.” 2014. Doctoral Dissertation, University of Toronto. Accessed August 15, 2018. http://hdl.handle.net/1807/68154.

MLA Handbook (7th Edition):

Prescott, Ian A. “Synaptic Plasticity of Basal Ganglia Output Neurons in Movement Disorders.” 2014. Web. 15 Aug 2018.

Vancouver:

Prescott IA. Synaptic Plasticity of Basal Ganglia Output Neurons in Movement Disorders. [Internet] [Doctoral dissertation]. University of Toronto; 2014. [cited 2018 Aug 15]. Available from: http://hdl.handle.net/1807/68154.

Council of Science Editors:

Prescott IA. Synaptic Plasticity of Basal Ganglia Output Neurons in Movement Disorders. [Doctoral Dissertation]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/68154


University of Toronto

17. Askin, Nesime. Metabolic Regulation of Fast- and Slow-twitch Skeletal Muscles by Estradiol and Progesterone in Females and their Impact during Ischemia.

Degree: PhD, 2014, University of Toronto

 The roles of estradiol and progesterone were investigated to determine the effect of these hormones on metabolism in skeletal muscle composed of different fiber types… (more)

Subjects/Keywords: estradiol; estrogen receptor; fiber types; ischemia; progesterone; skeletal muscle; 0719

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APA (6th Edition):

Askin, N. (2014). Metabolic Regulation of Fast- and Slow-twitch Skeletal Muscles by Estradiol and Progesterone in Females and their Impact during Ischemia. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/68380

Chicago Manual of Style (16th Edition):

Askin, Nesime. “Metabolic Regulation of Fast- and Slow-twitch Skeletal Muscles by Estradiol and Progesterone in Females and their Impact during Ischemia.” 2014. Doctoral Dissertation, University of Toronto. Accessed August 15, 2018. http://hdl.handle.net/1807/68380.

MLA Handbook (7th Edition):

Askin, Nesime. “Metabolic Regulation of Fast- and Slow-twitch Skeletal Muscles by Estradiol and Progesterone in Females and their Impact during Ischemia.” 2014. Web. 15 Aug 2018.

Vancouver:

Askin N. Metabolic Regulation of Fast- and Slow-twitch Skeletal Muscles by Estradiol and Progesterone in Females and their Impact during Ischemia. [Internet] [Doctoral dissertation]. University of Toronto; 2014. [cited 2018 Aug 15]. Available from: http://hdl.handle.net/1807/68380.

Council of Science Editors:

Askin N. Metabolic Regulation of Fast- and Slow-twitch Skeletal Muscles by Estradiol and Progesterone in Females and their Impact during Ischemia. [Doctoral Dissertation]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/68380


University of Toronto

18. Malik, Firhan. The Modulation of the Cystic Fibrosis Transmembrane Conductance Regulator Protein by Sphingosine-1-Phosphate in the Cerebral Microvascular System.

Degree: PhD, 2014, University of Toronto

 In the vascular system, peripheral resistance is regulated by the myogenic response (MR), which is the innate ability of a resistance artery to adapt its… (more)

Subjects/Keywords: Cerebral autoregulation; Cystic fibrosis; Cystic Fibrosis Transmembrane Conductance Regulator; Heart failure; Microvascular; Sphingosine-1-phosphate; 0719

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APA (6th Edition):

Malik, F. (2014). The Modulation of the Cystic Fibrosis Transmembrane Conductance Regulator Protein by Sphingosine-1-Phosphate in the Cerebral Microvascular System. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/68446

Chicago Manual of Style (16th Edition):

Malik, Firhan. “The Modulation of the Cystic Fibrosis Transmembrane Conductance Regulator Protein by Sphingosine-1-Phosphate in the Cerebral Microvascular System.” 2014. Doctoral Dissertation, University of Toronto. Accessed August 15, 2018. http://hdl.handle.net/1807/68446.

MLA Handbook (7th Edition):

Malik, Firhan. “The Modulation of the Cystic Fibrosis Transmembrane Conductance Regulator Protein by Sphingosine-1-Phosphate in the Cerebral Microvascular System.” 2014. Web. 15 Aug 2018.

Vancouver:

Malik F. The Modulation of the Cystic Fibrosis Transmembrane Conductance Regulator Protein by Sphingosine-1-Phosphate in the Cerebral Microvascular System. [Internet] [Doctoral dissertation]. University of Toronto; 2014. [cited 2018 Aug 15]. Available from: http://hdl.handle.net/1807/68446.

Council of Science Editors:

Malik F. The Modulation of the Cystic Fibrosis Transmembrane Conductance Regulator Protein by Sphingosine-1-Phosphate in the Cerebral Microvascular System. [Doctoral Dissertation]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/68446


University of Toronto

19. Rasmussen, Brittany Anne. Intestinal Cholecystokinin and Leptin Signaling and the Regulation of Glucose Production.

Degree: PhD, 2015, University of Toronto

 The number of individuals affected by diabetes is on the rise due in part to lifestyle and/or genetic factors. Diabetes and obesity are characterized by… (more)

Subjects/Keywords: Bypass Surgery; Cholecystokinin; Diabetes; Glucose production; Leptin; Obesity; 0719

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APA (6th Edition):

Rasmussen, B. A. (2015). Intestinal Cholecystokinin and Leptin Signaling and the Regulation of Glucose Production. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/71315

Chicago Manual of Style (16th Edition):

Rasmussen, Brittany Anne. “Intestinal Cholecystokinin and Leptin Signaling and the Regulation of Glucose Production.” 2015. Doctoral Dissertation, University of Toronto. Accessed August 15, 2018. http://hdl.handle.net/1807/71315.

MLA Handbook (7th Edition):

Rasmussen, Brittany Anne. “Intestinal Cholecystokinin and Leptin Signaling and the Regulation of Glucose Production.” 2015. Web. 15 Aug 2018.

Vancouver:

Rasmussen BA. Intestinal Cholecystokinin and Leptin Signaling and the Regulation of Glucose Production. [Internet] [Doctoral dissertation]. University of Toronto; 2015. [cited 2018 Aug 15]. Available from: http://hdl.handle.net/1807/71315.

Council of Science Editors:

Rasmussen BA. Intestinal Cholecystokinin and Leptin Signaling and the Regulation of Glucose Production. [Doctoral Dissertation]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/71315


University of Toronto

20. Shah, Anu. The Role of Thioredoxin Interacting Protein (TxNIP) in the Development of Diabetic Nephropathy.

Degree: PhD, 2015, University of Toronto

 Thioredoxin-interacting protein (TxNIP), an endogenous inhibitor of the thiol oxidoreductase - thioredoxin, is augmented by high glucose (HG) and promotes oxidative stress. The objective of… (more)

Subjects/Keywords: Diabetic Nephropathy; Mitochondria; Reactive Oxygen Species; Thioredoxin Interacting Protein; 0719

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APA (6th Edition):

Shah, A. (2015). The Role of Thioredoxin Interacting Protein (TxNIP) in the Development of Diabetic Nephropathy. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/71327

Chicago Manual of Style (16th Edition):

Shah, Anu. “The Role of Thioredoxin Interacting Protein (TxNIP) in the Development of Diabetic Nephropathy.” 2015. Doctoral Dissertation, University of Toronto. Accessed August 15, 2018. http://hdl.handle.net/1807/71327.

MLA Handbook (7th Edition):

Shah, Anu. “The Role of Thioredoxin Interacting Protein (TxNIP) in the Development of Diabetic Nephropathy.” 2015. Web. 15 Aug 2018.

Vancouver:

Shah A. The Role of Thioredoxin Interacting Protein (TxNIP) in the Development of Diabetic Nephropathy. [Internet] [Doctoral dissertation]. University of Toronto; 2015. [cited 2018 Aug 15]. Available from: http://hdl.handle.net/1807/71327.

Council of Science Editors:

Shah A. The Role of Thioredoxin Interacting Protein (TxNIP) in the Development of Diabetic Nephropathy. [Doctoral Dissertation]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/71327


University of Toronto

21. Omari, Shakib. MCL-1 - Guardian of the Ovarian Reserve: Characterization of the Contribution of MCL-1 to Oocyte Survival.

Degree: PhD, 2014, University of Toronto

Oocyte quality and the maintenance of the ovarian reserve are essential factors in reproductive competence and fertility. The predominant portion of ovarian follicles are lost… (more)

Subjects/Keywords: Bcl-2; Mcl-1; Mitochondria; Oocyte; Ovary; 0758

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APA (6th Edition):

Omari, S. (2014). MCL-1 - Guardian of the Ovarian Reserve: Characterization of the Contribution of MCL-1 to Oocyte Survival. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/72836

Chicago Manual of Style (16th Edition):

Omari, Shakib. “MCL-1 - Guardian of the Ovarian Reserve: Characterization of the Contribution of MCL-1 to Oocyte Survival.” 2014. Doctoral Dissertation, University of Toronto. Accessed August 15, 2018. http://hdl.handle.net/1807/72836.

MLA Handbook (7th Edition):

Omari, Shakib. “MCL-1 - Guardian of the Ovarian Reserve: Characterization of the Contribution of MCL-1 to Oocyte Survival.” 2014. Web. 15 Aug 2018.

Vancouver:

Omari S. MCL-1 - Guardian of the Ovarian Reserve: Characterization of the Contribution of MCL-1 to Oocyte Survival. [Internet] [Doctoral dissertation]. University of Toronto; 2014. [cited 2018 Aug 15]. Available from: http://hdl.handle.net/1807/72836.

Council of Science Editors:

Omari S. MCL-1 - Guardian of the Ovarian Reserve: Characterization of the Contribution of MCL-1 to Oocyte Survival. [Doctoral Dissertation]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/72836

22. Goligher, Ewan Christopher. Diaphragm Activity and Function During Mechanical Ventilation.

Degree: PhD, 2016, University of Toronto

 Previous studies suggest that mechanical ventilation (MV) can injure the diaphragm, potentially prolonging ventilator dependence. However, there are many potential insults to diaphragm function during… (more)

Subjects/Keywords: Diaphragm; Mechanical ventilation; Muscle function; Ultrasound; Weaning; 0719

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APA (6th Edition):

Goligher, E. C. (2016). Diaphragm Activity and Function During Mechanical Ventilation. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/73004

Chicago Manual of Style (16th Edition):

Goligher, Ewan Christopher. “Diaphragm Activity and Function During Mechanical Ventilation.” 2016. Doctoral Dissertation, University of Toronto. Accessed August 15, 2018. http://hdl.handle.net/1807/73004.

MLA Handbook (7th Edition):

Goligher, Ewan Christopher. “Diaphragm Activity and Function During Mechanical Ventilation.” 2016. Web. 15 Aug 2018.

Vancouver:

Goligher EC. Diaphragm Activity and Function During Mechanical Ventilation. [Internet] [Doctoral dissertation]. University of Toronto; 2016. [cited 2018 Aug 15]. Available from: http://hdl.handle.net/1807/73004.

Council of Science Editors:

Goligher EC. Diaphragm Activity and Function During Mechanical Ventilation. [Doctoral Dissertation]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/73004


University of Toronto

23. Kim, Hyunhee. The Cellular and Molecular Mechanisms of Ischemia-reperfusion induced Lung injury.

Degree: PhD, 2016, University of Toronto

 Lung transplantation is the last option to treat patients with end-stage lung diseases. One of the main problems of lung transplantation is ischemia-reperfusion-induced injury, which… (more)

Subjects/Keywords: acute lung injury; cell death; ischemia reperfusion; lung transplantation; 0719

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APA (6th Edition):

Kim, H. (2016). The Cellular and Molecular Mechanisms of Ischemia-reperfusion induced Lung injury. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/73054

Chicago Manual of Style (16th Edition):

Kim, Hyunhee. “The Cellular and Molecular Mechanisms of Ischemia-reperfusion induced Lung injury.” 2016. Doctoral Dissertation, University of Toronto. Accessed August 15, 2018. http://hdl.handle.net/1807/73054.

MLA Handbook (7th Edition):

Kim, Hyunhee. “The Cellular and Molecular Mechanisms of Ischemia-reperfusion induced Lung injury.” 2016. Web. 15 Aug 2018.

Vancouver:

Kim H. The Cellular and Molecular Mechanisms of Ischemia-reperfusion induced Lung injury. [Internet] [Doctoral dissertation]. University of Toronto; 2016. [cited 2018 Aug 15]. Available from: http://hdl.handle.net/1807/73054.

Council of Science Editors:

Kim H. The Cellular and Molecular Mechanisms of Ischemia-reperfusion induced Lung injury. [Doctoral Dissertation]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/73054


University of Toronto

24. Rozanski, Gabriela Maria. The Sandwich Synapse: Trans-glial Signaling Between Neuronal Somata in the Dorsal Root Ganglion.

Degree: PhD, 2014, University of Toronto

 The dorsal root ganglion (DRG) contains the cell bodies of primary somatosensory neurons that form axonal connections for the transfer of sensory input from the… (more)

Subjects/Keywords: dorsal root ganglion; glutamatergic; patch clamp; purinergic; trans-glial; T-type channel; 0317

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APA (6th Edition):

Rozanski, G. M. (2014). The Sandwich Synapse: Trans-glial Signaling Between Neuronal Somata in the Dorsal Root Ganglion. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/73784

Chicago Manual of Style (16th Edition):

Rozanski, Gabriela Maria. “The Sandwich Synapse: Trans-glial Signaling Between Neuronal Somata in the Dorsal Root Ganglion.” 2014. Doctoral Dissertation, University of Toronto. Accessed August 15, 2018. http://hdl.handle.net/1807/73784.

MLA Handbook (7th Edition):

Rozanski, Gabriela Maria. “The Sandwich Synapse: Trans-glial Signaling Between Neuronal Somata in the Dorsal Root Ganglion.” 2014. Web. 15 Aug 2018.

Vancouver:

Rozanski GM. The Sandwich Synapse: Trans-glial Signaling Between Neuronal Somata in the Dorsal Root Ganglion. [Internet] [Doctoral dissertation]. University of Toronto; 2014. [cited 2018 Aug 15]. Available from: http://hdl.handle.net/1807/73784.

Council of Science Editors:

Rozanski GM. The Sandwich Synapse: Trans-glial Signaling Between Neuronal Somata in the Dorsal Root Ganglion. [Doctoral Dissertation]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/73784


University of Toronto

25. Guo, June. The Effects of Insulin and Insulin Sensitizers on Neointimal Formation after Arterial Injuries in Rodent Models.

Degree: PhD, 2014, University of Toronto

Restenosis, a common complication following angioplasty, is exacerbated in insulin resistant patients with or without diabetes. Our laboratory has previously shown that insulin decreases arterial… (more)

Subjects/Keywords: 0719

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APA (6th Edition):

Guo, J. (2014). The Effects of Insulin and Insulin Sensitizers on Neointimal Formation after Arterial Injuries in Rodent Models. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/74799

Chicago Manual of Style (16th Edition):

Guo, June. “The Effects of Insulin and Insulin Sensitizers on Neointimal Formation after Arterial Injuries in Rodent Models.” 2014. Doctoral Dissertation, University of Toronto. Accessed August 15, 2018. http://hdl.handle.net/1807/74799.

MLA Handbook (7th Edition):

Guo, June. “The Effects of Insulin and Insulin Sensitizers on Neointimal Formation after Arterial Injuries in Rodent Models.” 2014. Web. 15 Aug 2018.

Vancouver:

Guo J. The Effects of Insulin and Insulin Sensitizers on Neointimal Formation after Arterial Injuries in Rodent Models. [Internet] [Doctoral dissertation]. University of Toronto; 2014. [cited 2018 Aug 15]. Available from: http://hdl.handle.net/1807/74799.

Council of Science Editors:

Guo J. The Effects of Insulin and Insulin Sensitizers on Neointimal Formation after Arterial Injuries in Rodent Models. [Doctoral Dissertation]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/74799


University of Toronto

26. Proulx, Eliane. Mechanisms of Cholinergic Neuromodulation of Prefrontal Cortex in Health and Disease: A Combined Whole-Cell Electrophysiology and Multiphoton Calcium Imaging Approach.

Degree: PhD, 2015, University of Toronto

The neuromodulation of the prefrontal cortex (PFC) by acetylcholine is essential for optimal executive function, where it exerts robust excitatory actions together with complex calcium… (more)

Subjects/Keywords: Acetylcholine; Calcium imaging; Electrophysiology; Muscarinic; Nicotinic; Prefrontal cortex; 0719

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APA (6th Edition):

Proulx, E. (2015). Mechanisms of Cholinergic Neuromodulation of Prefrontal Cortex in Health and Disease: A Combined Whole-Cell Electrophysiology and Multiphoton Calcium Imaging Approach. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/75680

Chicago Manual of Style (16th Edition):

Proulx, Eliane. “Mechanisms of Cholinergic Neuromodulation of Prefrontal Cortex in Health and Disease: A Combined Whole-Cell Electrophysiology and Multiphoton Calcium Imaging Approach.” 2015. Doctoral Dissertation, University of Toronto. Accessed August 15, 2018. http://hdl.handle.net/1807/75680.

MLA Handbook (7th Edition):

Proulx, Eliane. “Mechanisms of Cholinergic Neuromodulation of Prefrontal Cortex in Health and Disease: A Combined Whole-Cell Electrophysiology and Multiphoton Calcium Imaging Approach.” 2015. Web. 15 Aug 2018.

Vancouver:

Proulx E. Mechanisms of Cholinergic Neuromodulation of Prefrontal Cortex in Health and Disease: A Combined Whole-Cell Electrophysiology and Multiphoton Calcium Imaging Approach. [Internet] [Doctoral dissertation]. University of Toronto; 2015. [cited 2018 Aug 15]. Available from: http://hdl.handle.net/1807/75680.

Council of Science Editors:

Proulx E. Mechanisms of Cholinergic Neuromodulation of Prefrontal Cortex in Health and Disease: A Combined Whole-Cell Electrophysiology and Multiphoton Calcium Imaging Approach. [Doctoral Dissertation]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/75680


University of Toronto

27. Sauvé, Meghan Geraldine Lillian. The TNFα/S1P Signalling Axis Mediates Elevated Myogenic Tone in Diabetes Mellitus.

Degree: PhD, 2015, University of Toronto

Diabetes causes changes in microvascular function that can ultimately promote multi-organ failure. Microvascular resistance arteries, integral to blood pressure, tissue perfusion and capillary integrity, are… (more)

Subjects/Keywords: 0719

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APA (6th Edition):

Sauvé, M. G. L. (2015). The TNFα/S1P Signalling Axis Mediates Elevated Myogenic Tone in Diabetes Mellitus. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/75683

Chicago Manual of Style (16th Edition):

Sauvé, Meghan Geraldine Lillian. “The TNFα/S1P Signalling Axis Mediates Elevated Myogenic Tone in Diabetes Mellitus.” 2015. Doctoral Dissertation, University of Toronto. Accessed August 15, 2018. http://hdl.handle.net/1807/75683.

MLA Handbook (7th Edition):

Sauvé, Meghan Geraldine Lillian. “The TNFα/S1P Signalling Axis Mediates Elevated Myogenic Tone in Diabetes Mellitus.” 2015. Web. 15 Aug 2018.

Vancouver:

Sauvé MGL. The TNFα/S1P Signalling Axis Mediates Elevated Myogenic Tone in Diabetes Mellitus. [Internet] [Doctoral dissertation]. University of Toronto; 2015. [cited 2018 Aug 15]. Available from: http://hdl.handle.net/1807/75683.

Council of Science Editors:

Sauvé MGL. The TNFα/S1P Signalling Axis Mediates Elevated Myogenic Tone in Diabetes Mellitus. [Doctoral Dissertation]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/75683


University of Toronto

28. Mundil, Dhanwantee. Cardiovascular Effects and Mechanisms of Action of Glucagon-like Peptide-1 Metabolite, GLP-1(28-36).

Degree: PhD, 2015, University of Toronto

The incretin hormone, glucagon-like peptide-1 (GLP-1), is a cardioprotective peptide. GLP-1(28-36) is a C-terminal nonapeptide derived from the cleavage of GLP-1 by the endopeptidase NEP… (more)

Subjects/Keywords: Cardiovascular; Glucagon-like peptide-1; Incretins; Ischemia; Myocardial Infarction; 0719

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APA (6th Edition):

Mundil, D. (2015). Cardiovascular Effects and Mechanisms of Action of Glucagon-like Peptide-1 Metabolite, GLP-1(28-36). (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/75686

Chicago Manual of Style (16th Edition):

Mundil, Dhanwantee. “Cardiovascular Effects and Mechanisms of Action of Glucagon-like Peptide-1 Metabolite, GLP-1(28-36).” 2015. Doctoral Dissertation, University of Toronto. Accessed August 15, 2018. http://hdl.handle.net/1807/75686.

MLA Handbook (7th Edition):

Mundil, Dhanwantee. “Cardiovascular Effects and Mechanisms of Action of Glucagon-like Peptide-1 Metabolite, GLP-1(28-36).” 2015. Web. 15 Aug 2018.

Vancouver:

Mundil D. Cardiovascular Effects and Mechanisms of Action of Glucagon-like Peptide-1 Metabolite, GLP-1(28-36). [Internet] [Doctoral dissertation]. University of Toronto; 2015. [cited 2018 Aug 15]. Available from: http://hdl.handle.net/1807/75686.

Council of Science Editors:

Mundil D. Cardiovascular Effects and Mechanisms of Action of Glucagon-like Peptide-1 Metabolite, GLP-1(28-36). [Doctoral Dissertation]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/75686


University of Toronto

29. Prentice, Kacey June. Identification and Characterization of the Role of the Circulating Metabolite 3-Carboxy-4-Methyl-5-Propyl-2-Furanpropanoic Acid (CMPF) in Diabetes.

Degree: PhD, 2015, University of Toronto

Both gestational diabetes (GDM) and type 2 diabetes (T2D) are caused by a failure of the pancreatic beta cell to compensate for increased insulin demand.… (more)

Subjects/Keywords: Beta Cell; Diabetes; Gestational Diabetes; Insulin; Metabolomics; Steatosis; 0719

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APA (6th Edition):

Prentice, K. J. (2015). Identification and Characterization of the Role of the Circulating Metabolite 3-Carboxy-4-Methyl-5-Propyl-2-Furanpropanoic Acid (CMPF) in Diabetes. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/75688

Chicago Manual of Style (16th Edition):

Prentice, Kacey June. “Identification and Characterization of the Role of the Circulating Metabolite 3-Carboxy-4-Methyl-5-Propyl-2-Furanpropanoic Acid (CMPF) in Diabetes.” 2015. Doctoral Dissertation, University of Toronto. Accessed August 15, 2018. http://hdl.handle.net/1807/75688.

MLA Handbook (7th Edition):

Prentice, Kacey June. “Identification and Characterization of the Role of the Circulating Metabolite 3-Carboxy-4-Methyl-5-Propyl-2-Furanpropanoic Acid (CMPF) in Diabetes.” 2015. Web. 15 Aug 2018.

Vancouver:

Prentice KJ. Identification and Characterization of the Role of the Circulating Metabolite 3-Carboxy-4-Methyl-5-Propyl-2-Furanpropanoic Acid (CMPF) in Diabetes. [Internet] [Doctoral dissertation]. University of Toronto; 2015. [cited 2018 Aug 15]. Available from: http://hdl.handle.net/1807/75688.

Council of Science Editors:

Prentice KJ. Identification and Characterization of the Role of the Circulating Metabolite 3-Carboxy-4-Methyl-5-Propyl-2-Furanpropanoic Acid (CMPF) in Diabetes. [Doctoral Dissertation]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/75688


University of Toronto

30. Hui, Sonya. Translation of Sphingosine-1-phosphate-dependent Regulation of Myogenic Tone in Human Resistance Arteries with Heart Failure.

Degree: PhD, 2015, University of Toronto

Small resistance arteries critically influence tissue perfusion, capillary integrity, total peripheral resistance and systemic blood pressure. They do this in part by adapting diameter to… (more)

Subjects/Keywords: 0564

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APA (6th Edition):

Hui, S. (2015). Translation of Sphingosine-1-phosphate-dependent Regulation of Myogenic Tone in Human Resistance Arteries with Heart Failure. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/76260

Chicago Manual of Style (16th Edition):

Hui, Sonya. “Translation of Sphingosine-1-phosphate-dependent Regulation of Myogenic Tone in Human Resistance Arteries with Heart Failure.” 2015. Doctoral Dissertation, University of Toronto. Accessed August 15, 2018. http://hdl.handle.net/1807/76260.

MLA Handbook (7th Edition):

Hui, Sonya. “Translation of Sphingosine-1-phosphate-dependent Regulation of Myogenic Tone in Human Resistance Arteries with Heart Failure.” 2015. Web. 15 Aug 2018.

Vancouver:

Hui S. Translation of Sphingosine-1-phosphate-dependent Regulation of Myogenic Tone in Human Resistance Arteries with Heart Failure. [Internet] [Doctoral dissertation]. University of Toronto; 2015. [cited 2018 Aug 15]. Available from: http://hdl.handle.net/1807/76260.

Council of Science Editors:

Hui S. Translation of Sphingosine-1-phosphate-dependent Regulation of Myogenic Tone in Human Resistance Arteries with Heart Failure. [Doctoral Dissertation]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/76260

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