Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for +publisher:"University of Toronto" +contributor:("Physiology"). Showing records 1 – 30 of 345 total matches.

[1] [2] [3] [4] [5] … [12]

Search Limiters

Last 2 Years | English Only

Levels

▼ Search Limiters


University of Toronto

1. Restrepo, Cristian Castro. Impact of Time of Day Feeding, High Fat Diet, and Exercise on Adult Hippocampal Neurogenesis in Rats.

Degree: 2015, University of Toronto

Experiences like education and exercise buffer against age-related cognitive decline; however, the neurological underpinnings of this process are unknown. Adult neurogenesis in the hippocampus, a… (more)

Subjects/Keywords: 0719

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Restrepo, C. C. (2015). Impact of Time of Day Feeding, High Fat Diet, and Exercise on Adult Hippocampal Neurogenesis in Rats. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/74675

Chicago Manual of Style (16th Edition):

Restrepo, Cristian Castro. “Impact of Time of Day Feeding, High Fat Diet, and Exercise on Adult Hippocampal Neurogenesis in Rats.” 2015. Masters Thesis, University of Toronto. Accessed October 18, 2017. http://hdl.handle.net/1807/74675.

MLA Handbook (7th Edition):

Restrepo, Cristian Castro. “Impact of Time of Day Feeding, High Fat Diet, and Exercise on Adult Hippocampal Neurogenesis in Rats.” 2015. Web. 18 Oct 2017.

Vancouver:

Restrepo CC. Impact of Time of Day Feeding, High Fat Diet, and Exercise on Adult Hippocampal Neurogenesis in Rats. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2017 Oct 18]. Available from: http://hdl.handle.net/1807/74675.

Council of Science Editors:

Restrepo CC. Impact of Time of Day Feeding, High Fat Diet, and Exercise on Adult Hippocampal Neurogenesis in Rats. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/74675


University of Toronto

2. Dinh, Danny Duy. Recovery of Skeletal Muscle Microvascular Myogenic Reactivity After Smooth Muscle Cell-TNF Knockout.

Degree: 2015, University of Toronto

Resistance arteries possess an intrinsic ability, termed the myogenic response, which enables them to adapt to changes in blood pressure, allowing for the regulation of… (more)

Subjects/Keywords: Ca2+ sensitivity; compensation; resistance artery; sphingosine-1-phosphate; tumor necrosis factor; vascular smooth muscle; 0719

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dinh, D. D. (2015). Recovery of Skeletal Muscle Microvascular Myogenic Reactivity After Smooth Muscle Cell-TNF Knockout. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/74688

Chicago Manual of Style (16th Edition):

Dinh, Danny Duy. “Recovery of Skeletal Muscle Microvascular Myogenic Reactivity After Smooth Muscle Cell-TNF Knockout.” 2015. Masters Thesis, University of Toronto. Accessed October 18, 2017. http://hdl.handle.net/1807/74688.

MLA Handbook (7th Edition):

Dinh, Danny Duy. “Recovery of Skeletal Muscle Microvascular Myogenic Reactivity After Smooth Muscle Cell-TNF Knockout.” 2015. Web. 18 Oct 2017.

Vancouver:

Dinh DD. Recovery of Skeletal Muscle Microvascular Myogenic Reactivity After Smooth Muscle Cell-TNF Knockout. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2017 Oct 18]. Available from: http://hdl.handle.net/1807/74688.

Council of Science Editors:

Dinh DD. Recovery of Skeletal Muscle Microvascular Myogenic Reactivity After Smooth Muscle Cell-TNF Knockout. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/74688


University of Toronto

3. Fares, Jessica. Therapeutically Targeting the Cystic Fibrosis Transmembrane Conductance Regulator in Cerebrovascular Dysfunction associated with Subarachnoid Hemorrhage.

Degree: 2015, University of Toronto

Subarachnoid hemorrhage (SAH) is a devastating type of stroke consisting of an initial intracranial bleed followed by delayed cerebral ischemia (DCI). Current therapeutic strategies are… (more)

Subjects/Keywords: Ischemia; Myogenic response; Neuronal injury; Resistance arteries; Sphingosine-1-phosphate; Vascular smooth muscle; 0719

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fares, J. (2015). Therapeutically Targeting the Cystic Fibrosis Transmembrane Conductance Regulator in Cerebrovascular Dysfunction associated with Subarachnoid Hemorrhage. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/74718

Chicago Manual of Style (16th Edition):

Fares, Jessica. “Therapeutically Targeting the Cystic Fibrosis Transmembrane Conductance Regulator in Cerebrovascular Dysfunction associated with Subarachnoid Hemorrhage.” 2015. Masters Thesis, University of Toronto. Accessed October 18, 2017. http://hdl.handle.net/1807/74718.

MLA Handbook (7th Edition):

Fares, Jessica. “Therapeutically Targeting the Cystic Fibrosis Transmembrane Conductance Regulator in Cerebrovascular Dysfunction associated with Subarachnoid Hemorrhage.” 2015. Web. 18 Oct 2017.

Vancouver:

Fares J. Therapeutically Targeting the Cystic Fibrosis Transmembrane Conductance Regulator in Cerebrovascular Dysfunction associated with Subarachnoid Hemorrhage. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2017 Oct 18]. Available from: http://hdl.handle.net/1807/74718.

Council of Science Editors:

Fares J. Therapeutically Targeting the Cystic Fibrosis Transmembrane Conductance Regulator in Cerebrovascular Dysfunction associated with Subarachnoid Hemorrhage. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/74718


University of Toronto

4. Haffey, Sean Christopher. Probing the Role of Astrocytes in Postanesthetic Memory Deficits.

Degree: 2015, University of Toronto

Many patients who undergo surgery and general anesthesia suffer from memory impairments for weeks to months afterwards. However, the mechanisms underlying this phenomenon remain poorly… (more)

Subjects/Keywords: Astrocytes; Electrophysiology; Etomidate; GABA; Hippocampus; Tonic Inhibition; 0719

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Haffey, S. C. (2015). Probing the Role of Astrocytes in Postanesthetic Memory Deficits. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/74727

Chicago Manual of Style (16th Edition):

Haffey, Sean Christopher. “Probing the Role of Astrocytes in Postanesthetic Memory Deficits.” 2015. Masters Thesis, University of Toronto. Accessed October 18, 2017. http://hdl.handle.net/1807/74727.

MLA Handbook (7th Edition):

Haffey, Sean Christopher. “Probing the Role of Astrocytes in Postanesthetic Memory Deficits.” 2015. Web. 18 Oct 2017.

Vancouver:

Haffey SC. Probing the Role of Astrocytes in Postanesthetic Memory Deficits. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2017 Oct 18]. Available from: http://hdl.handle.net/1807/74727.

Council of Science Editors:

Haffey SC. Probing the Role of Astrocytes in Postanesthetic Memory Deficits. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/74727


University of Toronto

5. Polidovitch, Nazari. The Roles of Phosphodiesterase (PDE) 3A and 3B as Regulators of Cardiac Function in Disease.

Degree: 2014, University of Toronto

Cyclic nucleotide phosphodiesterases (PDEs) are enzymes that hydrolyze the 3'-5'-phosphodiester bonds of cAMP and/or cGMP. PDE3 family variants (PDE3A, PDE3B) are highly expressed in the… (more)

Subjects/Keywords: cAMP/cGMP signaling; Cardiac hypertrophy; Heart failure; Phosphodiesterases; 0719

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Polidovitch, N. (2014). The Roles of Phosphodiesterase (PDE) 3A and 3B as Regulators of Cardiac Function in Disease. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/74621

Chicago Manual of Style (16th Edition):

Polidovitch, Nazari. “The Roles of Phosphodiesterase (PDE) 3A and 3B as Regulators of Cardiac Function in Disease.” 2014. Masters Thesis, University of Toronto. Accessed October 18, 2017. http://hdl.handle.net/1807/74621.

MLA Handbook (7th Edition):

Polidovitch, Nazari. “The Roles of Phosphodiesterase (PDE) 3A and 3B as Regulators of Cardiac Function in Disease.” 2014. Web. 18 Oct 2017.

Vancouver:

Polidovitch N. The Roles of Phosphodiesterase (PDE) 3A and 3B as Regulators of Cardiac Function in Disease. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2017 Oct 18]. Available from: http://hdl.handle.net/1807/74621.

Council of Science Editors:

Polidovitch N. The Roles of Phosphodiesterase (PDE) 3A and 3B as Regulators of Cardiac Function in Disease. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/74621


University of Toronto

6. Lu, Zhen Qi. Proteomic Analyses of Human Fetal Atria and Ventricles.

Degree: 2014, University of Toronto

In this study we carried out a mass spectrometry-based proteome analysis of human fetal atria and ventricles. Heart protein lysates were analyzed on the Q-Exactive… (more)

Subjects/Keywords: Bioinformatics; Chamber specificity; Fetal tissue; Mass spectrometry; Q-Exactive; Ventricle; 0719

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lu, Z. Q. (2014). Proteomic Analyses of Human Fetal Atria and Ventricles. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/74628

Chicago Manual of Style (16th Edition):

Lu, Zhen Qi. “Proteomic Analyses of Human Fetal Atria and Ventricles.” 2014. Masters Thesis, University of Toronto. Accessed October 18, 2017. http://hdl.handle.net/1807/74628.

MLA Handbook (7th Edition):

Lu, Zhen Qi. “Proteomic Analyses of Human Fetal Atria and Ventricles.” 2014. Web. 18 Oct 2017.

Vancouver:

Lu ZQ. Proteomic Analyses of Human Fetal Atria and Ventricles. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2017 Oct 18]. Available from: http://hdl.handle.net/1807/74628.

Council of Science Editors:

Lu ZQ. Proteomic Analyses of Human Fetal Atria and Ventricles. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/74628


University of Toronto

7. Wong, Raymond. PKA Reduces the Rat and Human KCa3.1 Current, CaM Binding and Ca2+ Signaling, which Requires Ser332/ 334 in the CaM-binding C Terminus.

Degree: 2014, University of Toronto

The Ca2+-dependent K+ channel, KCa3.1 (KCNN4/IK/SK4), is a therapeutic target for several CNS disorders involving microglial activation. While PKA signaling is important in many cells… (more)

Subjects/Keywords: 0786

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wong, R. (2014). PKA Reduces the Rat and Human KCa3.1 Current, CaM Binding and Ca2+ Signaling, which Requires Ser332/ 334 in the CaM-binding C Terminus. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/74629

Chicago Manual of Style (16th Edition):

Wong, Raymond. “PKA Reduces the Rat and Human KCa3.1 Current, CaM Binding and Ca2+ Signaling, which Requires Ser332/ 334 in the CaM-binding C Terminus.” 2014. Masters Thesis, University of Toronto. Accessed October 18, 2017. http://hdl.handle.net/1807/74629.

MLA Handbook (7th Edition):

Wong, Raymond. “PKA Reduces the Rat and Human KCa3.1 Current, CaM Binding and Ca2+ Signaling, which Requires Ser332/ 334 in the CaM-binding C Terminus.” 2014. Web. 18 Oct 2017.

Vancouver:

Wong R. PKA Reduces the Rat and Human KCa3.1 Current, CaM Binding and Ca2+ Signaling, which Requires Ser332/ 334 in the CaM-binding C Terminus. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2017 Oct 18]. Available from: http://hdl.handle.net/1807/74629.

Council of Science Editors:

Wong R. PKA Reduces the Rat and Human KCa3.1 Current, CaM Binding and Ca2+ Signaling, which Requires Ser332/ 334 in the CaM-binding C Terminus. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/74629


University of Toronto

8. Bashiri, Amir. Cholesterol Plays a Crucial Role in High-fat-diet Induced ER stress and Non-alcoholic Fatty Liver Disease.

Degree: 2014, University of Toronto

By comparing C57 mice with two dyslipidemic models, LDL-receptor null (LDLR-/-) and LCAT/LDL-receptor null mice (LCAT-/-xLDLR-/-), our lab showed an observation of ER-membrane cholesterol being… (more)

Subjects/Keywords: Cholesterol; ER-stress; High fat diet; Murine; NAFLD/NASH; Obesity; 0719

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bashiri, A. (2014). Cholesterol Plays a Crucial Role in High-fat-diet Induced ER stress and Non-alcoholic Fatty Liver Disease. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/74632

Chicago Manual of Style (16th Edition):

Bashiri, Amir. “Cholesterol Plays a Crucial Role in High-fat-diet Induced ER stress and Non-alcoholic Fatty Liver Disease.” 2014. Masters Thesis, University of Toronto. Accessed October 18, 2017. http://hdl.handle.net/1807/74632.

MLA Handbook (7th Edition):

Bashiri, Amir. “Cholesterol Plays a Crucial Role in High-fat-diet Induced ER stress and Non-alcoholic Fatty Liver Disease.” 2014. Web. 18 Oct 2017.

Vancouver:

Bashiri A. Cholesterol Plays a Crucial Role in High-fat-diet Induced ER stress and Non-alcoholic Fatty Liver Disease. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2017 Oct 18]. Available from: http://hdl.handle.net/1807/74632.

Council of Science Editors:

Bashiri A. Cholesterol Plays a Crucial Role in High-fat-diet Induced ER stress and Non-alcoholic Fatty Liver Disease. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/74632


University of Toronto

9. Treen, Alice Klytie. The Effects of the Novel Reproductive Peptide Phoenixin-20 Amide on GnRH and Kisspeptin Hypothalamic Cell Models.

Degree: 2014, University of Toronto

Reproductive function is coordinated by the actions of specific neuropeptides and hormones, which converge on GnRH neurons in the hypothalamus. Phoenixin-20 amide (PNX-20) is a… (more)

Subjects/Keywords: gonadotropin-releasing hormone; kisspeptin; phoenixin; 0719

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Treen, A. K. (2014). The Effects of the Novel Reproductive Peptide Phoenixin-20 Amide on GnRH and Kisspeptin Hypothalamic Cell Models. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/74635

Chicago Manual of Style (16th Edition):

Treen, Alice Klytie. “The Effects of the Novel Reproductive Peptide Phoenixin-20 Amide on GnRH and Kisspeptin Hypothalamic Cell Models.” 2014. Masters Thesis, University of Toronto. Accessed October 18, 2017. http://hdl.handle.net/1807/74635.

MLA Handbook (7th Edition):

Treen, Alice Klytie. “The Effects of the Novel Reproductive Peptide Phoenixin-20 Amide on GnRH and Kisspeptin Hypothalamic Cell Models.” 2014. Web. 18 Oct 2017.

Vancouver:

Treen AK. The Effects of the Novel Reproductive Peptide Phoenixin-20 Amide on GnRH and Kisspeptin Hypothalamic Cell Models. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2017 Oct 18]. Available from: http://hdl.handle.net/1807/74635.

Council of Science Editors:

Treen AK. The Effects of the Novel Reproductive Peptide Phoenixin-20 Amide on GnRH and Kisspeptin Hypothalamic Cell Models. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/74635


University of Toronto

10. Korogyi, Adam S. Mechanisms of Atrial Fibrillation in Mice with Genetic Ablation of the α1D L-type Calcium Channel.

Degree: 2014, University of Toronto

Atrial fibrillation (AF) is the most common supraventricular arrhythmia with a multifactorial pathophysiology. Reductions in α1 L-type calcium channel (LTCC) current (ICaL) and action potential… (more)

Subjects/Keywords: atrial fibrillation; cav1.3; arrhythmia; atria; alternans; electrophysiology; heart; L-type calcium channel; 0719

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Korogyi, A. S. (2014). Mechanisms of Atrial Fibrillation in Mice with Genetic Ablation of the α1D L-type Calcium Channel. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/71517

Chicago Manual of Style (16th Edition):

Korogyi, Adam S. “Mechanisms of Atrial Fibrillation in Mice with Genetic Ablation of the α1D L-type Calcium Channel.” 2014. Masters Thesis, University of Toronto. Accessed October 18, 2017. http://hdl.handle.net/1807/71517.

MLA Handbook (7th Edition):

Korogyi, Adam S. “Mechanisms of Atrial Fibrillation in Mice with Genetic Ablation of the α1D L-type Calcium Channel.” 2014. Web. 18 Oct 2017.

Vancouver:

Korogyi AS. Mechanisms of Atrial Fibrillation in Mice with Genetic Ablation of the α1D L-type Calcium Channel. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2017 Oct 18]. Available from: http://hdl.handle.net/1807/71517.

Council of Science Editors:

Korogyi AS. Mechanisms of Atrial Fibrillation in Mice with Genetic Ablation of the α1D L-type Calcium Channel. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/71517


University of Toronto

11. Lloyd-Kuzik, Clifford Andrew. Investigations of the Functional Expression of SLC6A14 in Non-CF and CF Airway Epithelial Cells.

Degree: 2014, University of Toronto

Recent studies have found single nucleotide polymorphisms significantly associated with more severe cystic fibrosis (CF) in the promoter region of the SLC6A14 gene, encoding a… (more)

Subjects/Keywords: amino acid transport; cystic fibrosis; epithelial physiology; SLC6A14; 0719

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lloyd-Kuzik, C. A. (2014). Investigations of the Functional Expression of SLC6A14 in Non-CF and CF Airway Epithelial Cells. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/67842

Chicago Manual of Style (16th Edition):

Lloyd-Kuzik, Clifford Andrew. “Investigations of the Functional Expression of SLC6A14 in Non-CF and CF Airway Epithelial Cells.” 2014. Masters Thesis, University of Toronto. Accessed October 18, 2017. http://hdl.handle.net/1807/67842.

MLA Handbook (7th Edition):

Lloyd-Kuzik, Clifford Andrew. “Investigations of the Functional Expression of SLC6A14 in Non-CF and CF Airway Epithelial Cells.” 2014. Web. 18 Oct 2017.

Vancouver:

Lloyd-Kuzik CA. Investigations of the Functional Expression of SLC6A14 in Non-CF and CF Airway Epithelial Cells. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2017 Oct 18]. Available from: http://hdl.handle.net/1807/67842.

Council of Science Editors:

Lloyd-Kuzik CA. Investigations of the Functional Expression of SLC6A14 in Non-CF and CF Airway Epithelial Cells. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/67842


University of Toronto

12. Isaac, Michelle Sarah. The Role of Ovarian and Fetal Factors in Fetoplacental Microvascular Growth and Placental Development in Late Gestation in Mice.

Degree: 2014, University of Toronto

In late gestation, the rate of fetal growth increases significantly which must be matched by proper nutrient delivery. Although there is little change in placental… (more)

Subjects/Keywords: Estradiol; Fetectomy; Fetoplacental capillaries; Late gestation; Ovariectomy; Placenta; 0719

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Isaac, M. S. (2014). The Role of Ovarian and Fetal Factors in Fetoplacental Microvascular Growth and Placental Development in Late Gestation in Mice. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/67903

Chicago Manual of Style (16th Edition):

Isaac, Michelle Sarah. “The Role of Ovarian and Fetal Factors in Fetoplacental Microvascular Growth and Placental Development in Late Gestation in Mice.” 2014. Masters Thesis, University of Toronto. Accessed October 18, 2017. http://hdl.handle.net/1807/67903.

MLA Handbook (7th Edition):

Isaac, Michelle Sarah. “The Role of Ovarian and Fetal Factors in Fetoplacental Microvascular Growth and Placental Development in Late Gestation in Mice.” 2014. Web. 18 Oct 2017.

Vancouver:

Isaac MS. The Role of Ovarian and Fetal Factors in Fetoplacental Microvascular Growth and Placental Development in Late Gestation in Mice. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2017 Oct 18]. Available from: http://hdl.handle.net/1807/67903.

Council of Science Editors:

Isaac MS. The Role of Ovarian and Fetal Factors in Fetoplacental Microvascular Growth and Placental Development in Late Gestation in Mice. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/67903


University of Toronto

13. Menchella, Jonathan. The Role of AMP-activated Protein Kinase in Alleviating Insulin Resistance in Hypothalamic Neurons.

Degree: 2014, University of Toronto

Insulin signaling in the brain is vital to maintain whole-body glucose and energy homeostasis. AMP-activated protein kinase (AMPK) is a key enzyme that acts as… (more)

Subjects/Keywords: Energy Homeostasis; Hypothalamus; Insulin Resistance; Obesity; Type 2 Diabetes; 0719

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Menchella, J. (2014). The Role of AMP-activated Protein Kinase in Alleviating Insulin Resistance in Hypothalamic Neurons. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/68006

Chicago Manual of Style (16th Edition):

Menchella, Jonathan. “The Role of AMP-activated Protein Kinase in Alleviating Insulin Resistance in Hypothalamic Neurons.” 2014. Masters Thesis, University of Toronto. Accessed October 18, 2017. http://hdl.handle.net/1807/68006.

MLA Handbook (7th Edition):

Menchella, Jonathan. “The Role of AMP-activated Protein Kinase in Alleviating Insulin Resistance in Hypothalamic Neurons.” 2014. Web. 18 Oct 2017.

Vancouver:

Menchella J. The Role of AMP-activated Protein Kinase in Alleviating Insulin Resistance in Hypothalamic Neurons. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2017 Oct 18]. Available from: http://hdl.handle.net/1807/68006.

Council of Science Editors:

Menchella J. The Role of AMP-activated Protein Kinase in Alleviating Insulin Resistance in Hypothalamic Neurons. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/68006


University of Toronto

14. Hu, Xiaochen. Conditional p53 Deletion Promotes Adult Neurogenesis and Improves the Acquisition and Clearance of Contextual Fear Memory.

Degree: 2014, University of Toronto

We crossed Nestin-CreERT2 mice and p53 flox mice to produce mice with heterozygous or homozygous p53 deletion in a tamoxifen (TAM)-dependent manner (named iKO-p53+/- and… (more)

Subjects/Keywords: adult neurogenesis; conditional p53 deletion; contextual fear memory; dentate gyrus; immunohistochemistry; 0317

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hu, X. (2014). Conditional p53 Deletion Promotes Adult Neurogenesis and Improves the Acquisition and Clearance of Contextual Fear Memory. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/68511

Chicago Manual of Style (16th Edition):

Hu, Xiaochen. “Conditional p53 Deletion Promotes Adult Neurogenesis and Improves the Acquisition and Clearance of Contextual Fear Memory.” 2014. Masters Thesis, University of Toronto. Accessed October 18, 2017. http://hdl.handle.net/1807/68511.

MLA Handbook (7th Edition):

Hu, Xiaochen. “Conditional p53 Deletion Promotes Adult Neurogenesis and Improves the Acquisition and Clearance of Contextual Fear Memory.” 2014. Web. 18 Oct 2017.

Vancouver:

Hu X. Conditional p53 Deletion Promotes Adult Neurogenesis and Improves the Acquisition and Clearance of Contextual Fear Memory. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2017 Oct 18]. Available from: http://hdl.handle.net/1807/68511.

Council of Science Editors:

Hu X. Conditional p53 Deletion Promotes Adult Neurogenesis and Improves the Acquisition and Clearance of Contextual Fear Memory. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/68511


University of Toronto

15. Cunningham, Ceilidh Morgan. Mechanisms of Septin-mediated Inhibition of Neurotransmitter Release.

Degree: 2014, University of Toronto

Neurons communicate at chemical synapses via exocytosis of synaptic vesicles containing neurotransmitter, a process mediated by SNARE proteins. SEPT5, a predominantly brain-specific member of the… (more)

Subjects/Keywords: 0719

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cunningham, C. M. (2014). Mechanisms of Septin-mediated Inhibition of Neurotransmitter Release. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/68530

Chicago Manual of Style (16th Edition):

Cunningham, Ceilidh Morgan. “Mechanisms of Septin-mediated Inhibition of Neurotransmitter Release.” 2014. Masters Thesis, University of Toronto. Accessed October 18, 2017. http://hdl.handle.net/1807/68530.

MLA Handbook (7th Edition):

Cunningham, Ceilidh Morgan. “Mechanisms of Septin-mediated Inhibition of Neurotransmitter Release.” 2014. Web. 18 Oct 2017.

Vancouver:

Cunningham CM. Mechanisms of Septin-mediated Inhibition of Neurotransmitter Release. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2017 Oct 18]. Available from: http://hdl.handle.net/1807/68530.

Council of Science Editors:

Cunningham CM. Mechanisms of Septin-mediated Inhibition of Neurotransmitter Release. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/68530


University of Toronto

16. Mosa, Adam Jonathan. Optimization of Organotypic Hippocampal Slice Cultures for Adult Neurogenesis Research: Exploring Pharmacological Interactions in Newborn Dentate Granule Cells.

Degree: 2014, University of Toronto

The organotypic hippocampal slice culture technique is an in vitro system for explanting the hippocampus and incubating tissue for up to four weeks. This method… (more)

Subjects/Keywords: in vitro; neurogenesis; organotypic; 0317

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mosa, A. J. (2014). Optimization of Organotypic Hippocampal Slice Cultures for Adult Neurogenesis Research: Exploring Pharmacological Interactions in Newborn Dentate Granule Cells. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/68851

Chicago Manual of Style (16th Edition):

Mosa, Adam Jonathan. “Optimization of Organotypic Hippocampal Slice Cultures for Adult Neurogenesis Research: Exploring Pharmacological Interactions in Newborn Dentate Granule Cells.” 2014. Masters Thesis, University of Toronto. Accessed October 18, 2017. http://hdl.handle.net/1807/68851.

MLA Handbook (7th Edition):

Mosa, Adam Jonathan. “Optimization of Organotypic Hippocampal Slice Cultures for Adult Neurogenesis Research: Exploring Pharmacological Interactions in Newborn Dentate Granule Cells.” 2014. Web. 18 Oct 2017.

Vancouver:

Mosa AJ. Optimization of Organotypic Hippocampal Slice Cultures for Adult Neurogenesis Research: Exploring Pharmacological Interactions in Newborn Dentate Granule Cells. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2017 Oct 18]. Available from: http://hdl.handle.net/1807/68851.

Council of Science Editors:

Mosa AJ. Optimization of Organotypic Hippocampal Slice Cultures for Adult Neurogenesis Research: Exploring Pharmacological Interactions in Newborn Dentate Granule Cells. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/68851


University of Toronto

17. Nosak, Courtney. Examining the Role of Jagn1 in Pancreatic Beta Cells.

Degree: 2014, University of Toronto

Endoplasmic Reticulum (ER) stress and activation of the Unfolded Protein Response (UPR) has been implicated in causing pancreatic beta cell dysfunction leading to the development… (more)

Subjects/Keywords: 0719

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Nosak, C. (2014). Examining the Role of Jagn1 in Pancreatic Beta Cells. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/69073

Chicago Manual of Style (16th Edition):

Nosak, Courtney. “Examining the Role of Jagn1 in Pancreatic Beta Cells.” 2014. Masters Thesis, University of Toronto. Accessed October 18, 2017. http://hdl.handle.net/1807/69073.

MLA Handbook (7th Edition):

Nosak, Courtney. “Examining the Role of Jagn1 in Pancreatic Beta Cells.” 2014. Web. 18 Oct 2017.

Vancouver:

Nosak C. Examining the Role of Jagn1 in Pancreatic Beta Cells. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2017 Oct 18]. Available from: http://hdl.handle.net/1807/69073.

Council of Science Editors:

Nosak C. Examining the Role of Jagn1 in Pancreatic Beta Cells. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/69073


University of Toronto

18. Jiang, Jia Xin. DNA Methylation Mediates the Bladder's Response to Obstructive Stimuli.

Degree: 2015, University of Toronto

Partial bladder obstruction arises insidiously and compromises quality of life often by perpetuating urinary tract infections, incontinence and kidney failure. Obstruction leads to prolonged over-distension… (more)

Subjects/Keywords: 0719

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jiang, J. X. (2015). DNA Methylation Mediates the Bladder's Response to Obstructive Stimuli. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/69649

Chicago Manual of Style (16th Edition):

Jiang, Jia Xin. “DNA Methylation Mediates the Bladder's Response to Obstructive Stimuli.” 2015. Masters Thesis, University of Toronto. Accessed October 18, 2017. http://hdl.handle.net/1807/69649.

MLA Handbook (7th Edition):

Jiang, Jia Xin. “DNA Methylation Mediates the Bladder's Response to Obstructive Stimuli.” 2015. Web. 18 Oct 2017.

Vancouver:

Jiang JX. DNA Methylation Mediates the Bladder's Response to Obstructive Stimuli. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2017 Oct 18]. Available from: http://hdl.handle.net/1807/69649.

Council of Science Editors:

Jiang JX. DNA Methylation Mediates the Bladder's Response to Obstructive Stimuli. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/69649


University of Toronto

19. Pacheco, Shaun. Nanoscale formulation strategy for therapeutic agents in the prevention of acute lung injury.

Degree: 2014, University of Toronto

Acute lung injury is a morbid complication with numerous causes and currently no effective clinical treatment. Studies have shown that Src protein tyrosine kinases (PTK's)… (more)

Subjects/Keywords: Acute lung injury; Drug delivery; High-throughput screen; Hydrophobic drug; Nanotechnology; Self-assembling peptide; 0719

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pacheco, S. (2014). Nanoscale formulation strategy for therapeutic agents in the prevention of acute lung injury. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/69966

Chicago Manual of Style (16th Edition):

Pacheco, Shaun. “Nanoscale formulation strategy for therapeutic agents in the prevention of acute lung injury.” 2014. Masters Thesis, University of Toronto. Accessed October 18, 2017. http://hdl.handle.net/1807/69966.

MLA Handbook (7th Edition):

Pacheco, Shaun. “Nanoscale formulation strategy for therapeutic agents in the prevention of acute lung injury.” 2014. Web. 18 Oct 2017.

Vancouver:

Pacheco S. Nanoscale formulation strategy for therapeutic agents in the prevention of acute lung injury. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2017 Oct 18]. Available from: http://hdl.handle.net/1807/69966.

Council of Science Editors:

Pacheco S. Nanoscale formulation strategy for therapeutic agents in the prevention of acute lung injury. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/69966


University of Toronto

20. Guet-McCreight, Alexandre Thomas Liam. Predicting Cell-type Specific Active Properties by Developing Multi-compartment Models using Databases and Electrophysiological Feature Constraints: Application to Interneuron Specific 3 (IS3) Cells in the Hippocampus.

Degree: 2015, University of Toronto

In hippocampus, interneuron specific 3 (IS3) cells have been shown to make inhibitory synapses onto specific types of inhibitory interneuron dendrites with the ability to… (more)

Subjects/Keywords: Computational Neuroscience; Dendrites; Multi-Compartment Modeling; Voltage-Gated Channels; 0317

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Guet-McCreight, A. T. L. (2015). Predicting Cell-type Specific Active Properties by Developing Multi-compartment Models using Databases and Electrophysiological Feature Constraints: Application to Interneuron Specific 3 (IS3) Cells in the Hippocampus. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/70331

Chicago Manual of Style (16th Edition):

Guet-McCreight, Alexandre Thomas Liam. “Predicting Cell-type Specific Active Properties by Developing Multi-compartment Models using Databases and Electrophysiological Feature Constraints: Application to Interneuron Specific 3 (IS3) Cells in the Hippocampus.” 2015. Masters Thesis, University of Toronto. Accessed October 18, 2017. http://hdl.handle.net/1807/70331.

MLA Handbook (7th Edition):

Guet-McCreight, Alexandre Thomas Liam. “Predicting Cell-type Specific Active Properties by Developing Multi-compartment Models using Databases and Electrophysiological Feature Constraints: Application to Interneuron Specific 3 (IS3) Cells in the Hippocampus.” 2015. Web. 18 Oct 2017.

Vancouver:

Guet-McCreight ATL. Predicting Cell-type Specific Active Properties by Developing Multi-compartment Models using Databases and Electrophysiological Feature Constraints: Application to Interneuron Specific 3 (IS3) Cells in the Hippocampus. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2017 Oct 18]. Available from: http://hdl.handle.net/1807/70331.

Council of Science Editors:

Guet-McCreight ATL. Predicting Cell-type Specific Active Properties by Developing Multi-compartment Models using Databases and Electrophysiological Feature Constraints: Application to Interneuron Specific 3 (IS3) Cells in the Hippocampus. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/70331


University of Toronto

21. Ellis, Shane Luke. Role of the Human Globus Pallidus in Tremorgenesis.

Degree: 2015, University of Toronto

The GPi is a nucleus that serves as an output of the basal ganglia; a collection of nuclei which function in selecting movements to be… (more)

Subjects/Keywords: Globus Pallidus Internus; Microelectrode Recordings; Movement Disorders; Parkinson's Disease; Theta Oscillations; Tremor; 0317

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ellis, S. L. (2015). Role of the Human Globus Pallidus in Tremorgenesis. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/70403

Chicago Manual of Style (16th Edition):

Ellis, Shane Luke. “Role of the Human Globus Pallidus in Tremorgenesis.” 2015. Masters Thesis, University of Toronto. Accessed October 18, 2017. http://hdl.handle.net/1807/70403.

MLA Handbook (7th Edition):

Ellis, Shane Luke. “Role of the Human Globus Pallidus in Tremorgenesis.” 2015. Web. 18 Oct 2017.

Vancouver:

Ellis SL. Role of the Human Globus Pallidus in Tremorgenesis. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2017 Oct 18]. Available from: http://hdl.handle.net/1807/70403.

Council of Science Editors:

Ellis SL. Role of the Human Globus Pallidus in Tremorgenesis. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/70403


University of Toronto

22. Guan, Sihui. The Anatomic Connectome and Functional Dissection of the Caenorhabditis elegans First-stage Larvae (L1) Motor Circuit.

Degree: 2015, University of Toronto

During development, neural networks exhibits anatomic and functional modifications yet the activity patterns change remains poorly understood. The neural circuit controlling locomotion of C. elegans… (more)

Subjects/Keywords: calcium imaging; c elegans; development; locomotion; 0758

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Guan, S. (2015). The Anatomic Connectome and Functional Dissection of the Caenorhabditis elegans First-stage Larvae (L1) Motor Circuit. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/70415

Chicago Manual of Style (16th Edition):

Guan, Sihui. “The Anatomic Connectome and Functional Dissection of the Caenorhabditis elegans First-stage Larvae (L1) Motor Circuit.” 2015. Masters Thesis, University of Toronto. Accessed October 18, 2017. http://hdl.handle.net/1807/70415.

MLA Handbook (7th Edition):

Guan, Sihui. “The Anatomic Connectome and Functional Dissection of the Caenorhabditis elegans First-stage Larvae (L1) Motor Circuit.” 2015. Web. 18 Oct 2017.

Vancouver:

Guan S. The Anatomic Connectome and Functional Dissection of the Caenorhabditis elegans First-stage Larvae (L1) Motor Circuit. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2017 Oct 18]. Available from: http://hdl.handle.net/1807/70415.

Council of Science Editors:

Guan S. The Anatomic Connectome and Functional Dissection of the Caenorhabditis elegans First-stage Larvae (L1) Motor Circuit. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/70415


University of Toronto

23. Froese, Sean. Characterization of Novel Glucagon Receptor Interactors that Modify Receptor Activity.

Degree: 2015, University of Toronto

Glucagon helps maintain blood glucose homeostasis by stimulating gluconeogenesis and glycogenolysis. Elevated glucagon levels have been reported in type 2 diabetics, and may be in… (more)

Subjects/Keywords: Glucagon receptor; Glucose production; Hepatocytes; Interactome; Membrane proteins; 0719

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Froese, S. (2015). Characterization of Novel Glucagon Receptor Interactors that Modify Receptor Activity. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/70419

Chicago Manual of Style (16th Edition):

Froese, Sean. “Characterization of Novel Glucagon Receptor Interactors that Modify Receptor Activity.” 2015. Masters Thesis, University of Toronto. Accessed October 18, 2017. http://hdl.handle.net/1807/70419.

MLA Handbook (7th Edition):

Froese, Sean. “Characterization of Novel Glucagon Receptor Interactors that Modify Receptor Activity.” 2015. Web. 18 Oct 2017.

Vancouver:

Froese S. Characterization of Novel Glucagon Receptor Interactors that Modify Receptor Activity. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2017 Oct 18]. Available from: http://hdl.handle.net/1807/70419.

Council of Science Editors:

Froese S. Characterization of Novel Glucagon Receptor Interactors that Modify Receptor Activity. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/70419


University of Toronto

24. Kim, Bomin. Glypican-1 Modulates CSPG inhibition on Growing Axons.

Degree: 2015, University of Toronto

During development, guidance cues mediate axon guidance such that synaptic connections made give rise to a properly functioning central nervous system. Chondroitin sulfate proteoglycans (CSPGs)… (more)

Subjects/Keywords: 0317

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kim, B. (2015). Glypican-1 Modulates CSPG inhibition on Growing Axons. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/70420

Chicago Manual of Style (16th Edition):

Kim, Bomin. “Glypican-1 Modulates CSPG inhibition on Growing Axons.” 2015. Masters Thesis, University of Toronto. Accessed October 18, 2017. http://hdl.handle.net/1807/70420.

MLA Handbook (7th Edition):

Kim, Bomin. “Glypican-1 Modulates CSPG inhibition on Growing Axons.” 2015. Web. 18 Oct 2017.

Vancouver:

Kim B. Glypican-1 Modulates CSPG inhibition on Growing Axons. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2017 Oct 18]. Available from: http://hdl.handle.net/1807/70420.

Council of Science Editors:

Kim B. Glypican-1 Modulates CSPG inhibition on Growing Axons. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/70420


University of Toronto

25. Nguyen, Tina Tu-Thu Ngoc. Matrix Metalloproteinase Expression and Regulation During Pregnancy, Term Labour, and Postpartum.

Degree: 2015, University of Toronto

It is widely accepted that pregnancy, spontaneous term labour (TL), and postpartum (PP) involution is associated with changes in the cellular and extracellular composition of… (more)

Subjects/Keywords: matrix metalloproteinases; myometrium; postpartum; pregnancy; term labour; 0719

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Nguyen, T. T. N. (2015). Matrix Metalloproteinase Expression and Regulation During Pregnancy, Term Labour, and Postpartum. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/70507

Chicago Manual of Style (16th Edition):

Nguyen, Tina Tu-Thu Ngoc. “Matrix Metalloproteinase Expression and Regulation During Pregnancy, Term Labour, and Postpartum.” 2015. Masters Thesis, University of Toronto. Accessed October 18, 2017. http://hdl.handle.net/1807/70507.

MLA Handbook (7th Edition):

Nguyen, Tina Tu-Thu Ngoc. “Matrix Metalloproteinase Expression and Regulation During Pregnancy, Term Labour, and Postpartum.” 2015. Web. 18 Oct 2017.

Vancouver:

Nguyen TTN. Matrix Metalloproteinase Expression and Regulation During Pregnancy, Term Labour, and Postpartum. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2017 Oct 18]. Available from: http://hdl.handle.net/1807/70507.

Council of Science Editors:

Nguyen TTN. Matrix Metalloproteinase Expression and Regulation During Pregnancy, Term Labour, and Postpartum. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/70507


University of Toronto

26. Raileanu, Vanessa. Characterization of Mesenchymal Stromal Cells Derived from Human Umbilical Cord Tissue.

Degree: 2015, University of Toronto

Mesenchymal stromal cells (MSCs) have emerged as candidates with therapeutic potential to treat different pathologies. MSCs isolated from the bone marrow are most commonly used,… (more)

Subjects/Keywords: Characterization; Diabetes; Excisional Murine Model; Immunophenotype; Mesenchymal Stromal Cells; TSG-6; 0719

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Raileanu, V. (2015). Characterization of Mesenchymal Stromal Cells Derived from Human Umbilical Cord Tissue. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/70533

Chicago Manual of Style (16th Edition):

Raileanu, Vanessa. “Characterization of Mesenchymal Stromal Cells Derived from Human Umbilical Cord Tissue.” 2015. Masters Thesis, University of Toronto. Accessed October 18, 2017. http://hdl.handle.net/1807/70533.

MLA Handbook (7th Edition):

Raileanu, Vanessa. “Characterization of Mesenchymal Stromal Cells Derived from Human Umbilical Cord Tissue.” 2015. Web. 18 Oct 2017.

Vancouver:

Raileanu V. Characterization of Mesenchymal Stromal Cells Derived from Human Umbilical Cord Tissue. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2017 Oct 18]. Available from: http://hdl.handle.net/1807/70533.

Council of Science Editors:

Raileanu V. Characterization of Mesenchymal Stromal Cells Derived from Human Umbilical Cord Tissue. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/70533


University of Toronto

27. Sattar, Joobin. Investigating the Putative Mechanism and Functional Role of RGS5 Upregulation in Vascular Smooth Muscle Cells Following Statin Treatment.

Degree: 2015, University of Toronto

RGS5 within vascular smooth muscle cells (VSMCs) is capable of inhibiting G-protein signaling involved in VSMC recruitment. Based on the suggested ability of statin to… (more)

Subjects/Keywords: Atherosclerosis; G-Protein Signaling; Neointimal Hyperplasia; Pleiotropic; Statins; Vascular Smooth Muscle Cell; 0719

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sattar, J. (2015). Investigating the Putative Mechanism and Functional Role of RGS5 Upregulation in Vascular Smooth Muscle Cells Following Statin Treatment. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/70645

Chicago Manual of Style (16th Edition):

Sattar, Joobin. “Investigating the Putative Mechanism and Functional Role of RGS5 Upregulation in Vascular Smooth Muscle Cells Following Statin Treatment.” 2015. Masters Thesis, University of Toronto. Accessed October 18, 2017. http://hdl.handle.net/1807/70645.

MLA Handbook (7th Edition):

Sattar, Joobin. “Investigating the Putative Mechanism and Functional Role of RGS5 Upregulation in Vascular Smooth Muscle Cells Following Statin Treatment.” 2015. Web. 18 Oct 2017.

Vancouver:

Sattar J. Investigating the Putative Mechanism and Functional Role of RGS5 Upregulation in Vascular Smooth Muscle Cells Following Statin Treatment. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2017 Oct 18]. Available from: http://hdl.handle.net/1807/70645.

Council of Science Editors:

Sattar J. Investigating the Putative Mechanism and Functional Role of RGS5 Upregulation in Vascular Smooth Muscle Cells Following Statin Treatment. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/70645


University of Toronto

28. Vu, Michael. TASK Channels on Basal Forebrain Cholinergic Neurons Modulate Electrocortical Signatures of Arousal by Histamine.

Degree: 2015, University of Toronto

Activation of basal forebrain cholinergic neurons by histamine elicits cortical arousal. TWIK-like acid sensitive K+ (TASK) channels modulate neuronal excitability, but the role of TASK… (more)

Subjects/Keywords: Basal Forebrain; Histamine; Sleep; TASK Channels; Wakefulness; 0719

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Vu, M. (2015). TASK Channels on Basal Forebrain Cholinergic Neurons Modulate Electrocortical Signatures of Arousal by Histamine. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/70702

Chicago Manual of Style (16th Edition):

Vu, Michael. “TASK Channels on Basal Forebrain Cholinergic Neurons Modulate Electrocortical Signatures of Arousal by Histamine.” 2015. Masters Thesis, University of Toronto. Accessed October 18, 2017. http://hdl.handle.net/1807/70702.

MLA Handbook (7th Edition):

Vu, Michael. “TASK Channels on Basal Forebrain Cholinergic Neurons Modulate Electrocortical Signatures of Arousal by Histamine.” 2015. Web. 18 Oct 2017.

Vancouver:

Vu M. TASK Channels on Basal Forebrain Cholinergic Neurons Modulate Electrocortical Signatures of Arousal by Histamine. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2017 Oct 18]. Available from: http://hdl.handle.net/1807/70702.

Council of Science Editors:

Vu M. TASK Channels on Basal Forebrain Cholinergic Neurons Modulate Electrocortical Signatures of Arousal by Histamine. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/70702


University of Toronto

29. Stacey, Holly Maryanne. The Role of Syntaxin-1a in Glucagon-like Peptide-1 Secretion from the Adult Mouse Intestinal L-Cell.

Degree: 2015, University of Toronto

Glucagon-like Peptide-1 (GLP-1) is an incretin secreted from intestinal L-cells. Although pathways promoting GLP-1 secretion have been characterized, little is known about the mechanism of… (more)

Subjects/Keywords: Exocytosis; Glucagon-like Peptide-1 (GLP-1); in vivo; Primary L-Cell; Secretion; Syntaxin-1a; 0719

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Stacey, H. M. (2015). The Role of Syntaxin-1a in Glucagon-like Peptide-1 Secretion from the Adult Mouse Intestinal L-Cell. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/70774

Chicago Manual of Style (16th Edition):

Stacey, Holly Maryanne. “The Role of Syntaxin-1a in Glucagon-like Peptide-1 Secretion from the Adult Mouse Intestinal L-Cell.” 2015. Masters Thesis, University of Toronto. Accessed October 18, 2017. http://hdl.handle.net/1807/70774.

MLA Handbook (7th Edition):

Stacey, Holly Maryanne. “The Role of Syntaxin-1a in Glucagon-like Peptide-1 Secretion from the Adult Mouse Intestinal L-Cell.” 2015. Web. 18 Oct 2017.

Vancouver:

Stacey HM. The Role of Syntaxin-1a in Glucagon-like Peptide-1 Secretion from the Adult Mouse Intestinal L-Cell. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2017 Oct 18]. Available from: http://hdl.handle.net/1807/70774.

Council of Science Editors:

Stacey HM. The Role of Syntaxin-1a in Glucagon-like Peptide-1 Secretion from the Adult Mouse Intestinal L-Cell. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/70774


University of Toronto

30. Dang, Frances. miR-142-3p Directly Regulates Autophagy Dependent Gene ATG16L1.

Degree: 2014, University of Toronto

Numerous genome-wide association studies demonstrate that a variant in the autophagy-dependent gene ATG16L1 is associated with Crohn's Disease (CD). miRNA regulation has been shown to… (more)

Subjects/Keywords: ATG16L1; Autophagy; Crohn's Disease; Inflammatory Bowel Disease; microRNA; miR-142-3p; 0719

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dang, F. (2014). miR-142-3p Directly Regulates Autophagy Dependent Gene ATG16L1. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/73105

Chicago Manual of Style (16th Edition):

Dang, Frances. “miR-142-3p Directly Regulates Autophagy Dependent Gene ATG16L1.” 2014. Masters Thesis, University of Toronto. Accessed October 18, 2017. http://hdl.handle.net/1807/73105.

MLA Handbook (7th Edition):

Dang, Frances. “miR-142-3p Directly Regulates Autophagy Dependent Gene ATG16L1.” 2014. Web. 18 Oct 2017.

Vancouver:

Dang F. miR-142-3p Directly Regulates Autophagy Dependent Gene ATG16L1. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2017 Oct 18]. Available from: http://hdl.handle.net/1807/73105.

Council of Science Editors:

Dang F. miR-142-3p Directly Regulates Autophagy Dependent Gene ATG16L1. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/73105

[1] [2] [3] [4] [5] … [12]

.