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You searched for +publisher:"University of Toronto" +contributor:("Physiology"). Showing records 1 – 30 of 429 total matches.

[1] [2] [3] [4] [5] … [15]

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University of Toronto

1. Mah, Henry Ka-Hey. Existence of P/Q-type Calcium Channels in Chicken Presynaptic Nerve Terminals?.

Degree: 2018, University of Toronto

Transmitter release at fast-transmitting mammalian synapses are predominately gated by CaV2.2 and CaV2.1. This was primarily determined due to pharmacological characterizations of these synapses with… (more)

Subjects/Keywords: CaV2.1; Chicken; Exon44; Immunocytochemistry; Presynaptic; Synaptosomes; 0317

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APA (6th Edition):

Mah, H. K. (2018). Existence of P/Q-type Calcium Channels in Chicken Presynaptic Nerve Terminals?. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/91407

Chicago Manual of Style (16th Edition):

Mah, Henry Ka-Hey. “Existence of P/Q-type Calcium Channels in Chicken Presynaptic Nerve Terminals?.” 2018. Masters Thesis, University of Toronto. Accessed April 26, 2019. http://hdl.handle.net/1807/91407.

MLA Handbook (7th Edition):

Mah, Henry Ka-Hey. “Existence of P/Q-type Calcium Channels in Chicken Presynaptic Nerve Terminals?.” 2018. Web. 26 Apr 2019.

Vancouver:

Mah HK. Existence of P/Q-type Calcium Channels in Chicken Presynaptic Nerve Terminals?. [Internet] [Masters thesis]. University of Toronto; 2018. [cited 2019 Apr 26]. Available from: http://hdl.handle.net/1807/91407.

Council of Science Editors:

Mah HK. Existence of P/Q-type Calcium Channels in Chicken Presynaptic Nerve Terminals?. [Masters Thesis]. University of Toronto; 2018. Available from: http://hdl.handle.net/1807/91407


University of Toronto

2. Ahn, Kyoungheon. Targeting kidney inflammation in a murine model of Alport Syndrome.

Degree: 2018, University of Toronto

Alport Syndrome is a rare inherited kidney disease that is due to genetic mutations of type IV collagen. The mutations interfere with normal assembly of… (more)

Subjects/Keywords: 0719

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APA (6th Edition):

Ahn, K. (2018). Targeting kidney inflammation in a murine model of Alport Syndrome. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/91686

Chicago Manual of Style (16th Edition):

Ahn, Kyoungheon. “Targeting kidney inflammation in a murine model of Alport Syndrome.” 2018. Masters Thesis, University of Toronto. Accessed April 26, 2019. http://hdl.handle.net/1807/91686.

MLA Handbook (7th Edition):

Ahn, Kyoungheon. “Targeting kidney inflammation in a murine model of Alport Syndrome.” 2018. Web. 26 Apr 2019.

Vancouver:

Ahn K. Targeting kidney inflammation in a murine model of Alport Syndrome. [Internet] [Masters thesis]. University of Toronto; 2018. [cited 2019 Apr 26]. Available from: http://hdl.handle.net/1807/91686.

Council of Science Editors:

Ahn K. Targeting kidney inflammation in a murine model of Alport Syndrome. [Masters Thesis]. University of Toronto; 2018. Available from: http://hdl.handle.net/1807/91686


University of Toronto

3. Farine, Tali. Mechanisms responsible for the activation of maternal peripheral leukocytes during term labour.

Degree: 2018, University of Toronto

Infiltration of maternal peripheral leukocytes into the uterine tissues has been demonstrated to be a critical event occurring prior to and during active term labour… (more)

Subjects/Keywords: cytokines; labour; leukocytes; preterm birth; 0380

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APA (6th Edition):

Farine, T. (2018). Mechanisms responsible for the activation of maternal peripheral leukocytes during term labour. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/91551

Chicago Manual of Style (16th Edition):

Farine, Tali. “Mechanisms responsible for the activation of maternal peripheral leukocytes during term labour.” 2018. Masters Thesis, University of Toronto. Accessed April 26, 2019. http://hdl.handle.net/1807/91551.

MLA Handbook (7th Edition):

Farine, Tali. “Mechanisms responsible for the activation of maternal peripheral leukocytes during term labour.” 2018. Web. 26 Apr 2019.

Vancouver:

Farine T. Mechanisms responsible for the activation of maternal peripheral leukocytes during term labour. [Internet] [Masters thesis]. University of Toronto; 2018. [cited 2019 Apr 26]. Available from: http://hdl.handle.net/1807/91551.

Council of Science Editors:

Farine T. Mechanisms responsible for the activation of maternal peripheral leukocytes during term labour. [Masters Thesis]. University of Toronto; 2018. Available from: http://hdl.handle.net/1807/91551


University of Toronto

4. Joseph, Michael Jason Emmanuel. After Intracerebral Hemorrhage, Oligodendrocyte Precursors Proliferate and Differentiate Inside White-matter Tracts in the Rat Striatum.

Degree: 2016, University of Toronto

Using a rat model of ICH, I quantified survival, proliferation and differentiation of oligodendrocyte-lineage cells (Olig2+) in the peri-hematoma region, surrounding striatum and contralateral striatum… (more)

Subjects/Keywords: hemorrhagic stroke; myelinated axon tracts; oligodendrocyte maturation; peri-hematoma recovery; post-stroke recovery; 0317

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APA (6th Edition):

Joseph, M. J. E. (2016). After Intracerebral Hemorrhage, Oligodendrocyte Precursors Proliferate and Differentiate Inside White-matter Tracts in the Rat Striatum. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/91214

Chicago Manual of Style (16th Edition):

Joseph, Michael Jason Emmanuel. “After Intracerebral Hemorrhage, Oligodendrocyte Precursors Proliferate and Differentiate Inside White-matter Tracts in the Rat Striatum.” 2016. Masters Thesis, University of Toronto. Accessed April 26, 2019. http://hdl.handle.net/1807/91214.

MLA Handbook (7th Edition):

Joseph, Michael Jason Emmanuel. “After Intracerebral Hemorrhage, Oligodendrocyte Precursors Proliferate and Differentiate Inside White-matter Tracts in the Rat Striatum.” 2016. Web. 26 Apr 2019.

Vancouver:

Joseph MJE. After Intracerebral Hemorrhage, Oligodendrocyte Precursors Proliferate and Differentiate Inside White-matter Tracts in the Rat Striatum. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2019 Apr 26]. Available from: http://hdl.handle.net/1807/91214.

Council of Science Editors:

Joseph MJE. After Intracerebral Hemorrhage, Oligodendrocyte Precursors Proliferate and Differentiate Inside White-matter Tracts in the Rat Striatum. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/91214


University of Toronto

5. Tse, Erika Koppert. Effects of the Saturated Fatty Acid Palmitate on Neuroinflammation, ER Stress, and Pomc mRNA Expression in Hypothalamic mHypoA-POMC/GFP Neurons.

Degree: 2017, University of Toronto

Dietary fats can modulate brain function. How free fatty acids (FFAs) alter hypothalamic pro-opiomelanocortin (POMC) neurons remain undefined. The saturated FFA, palmitate, increased neuroinflammatory and… (more)

Subjects/Keywords: ER stress; hypothalamus; JNK; neuroinflammation; palmitate; Pro-opiomelanocortin (POMC); 0719

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APA (6th Edition):

Tse, E. K. (2017). Effects of the Saturated Fatty Acid Palmitate on Neuroinflammation, ER Stress, and Pomc mRNA Expression in Hypothalamic mHypoA-POMC/GFP Neurons. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/91258

Chicago Manual of Style (16th Edition):

Tse, Erika Koppert. “Effects of the Saturated Fatty Acid Palmitate on Neuroinflammation, ER Stress, and Pomc mRNA Expression in Hypothalamic mHypoA-POMC/GFP Neurons.” 2017. Masters Thesis, University of Toronto. Accessed April 26, 2019. http://hdl.handle.net/1807/91258.

MLA Handbook (7th Edition):

Tse, Erika Koppert. “Effects of the Saturated Fatty Acid Palmitate on Neuroinflammation, ER Stress, and Pomc mRNA Expression in Hypothalamic mHypoA-POMC/GFP Neurons.” 2017. Web. 26 Apr 2019.

Vancouver:

Tse EK. Effects of the Saturated Fatty Acid Palmitate on Neuroinflammation, ER Stress, and Pomc mRNA Expression in Hypothalamic mHypoA-POMC/GFP Neurons. [Internet] [Masters thesis]. University of Toronto; 2017. [cited 2019 Apr 26]. Available from: http://hdl.handle.net/1807/91258.

Council of Science Editors:

Tse EK. Effects of the Saturated Fatty Acid Palmitate on Neuroinflammation, ER Stress, and Pomc mRNA Expression in Hypothalamic mHypoA-POMC/GFP Neurons. [Masters Thesis]. University of Toronto; 2017. Available from: http://hdl.handle.net/1807/91258


University of Toronto

6. Eversley, Judith Andrea. Organic Anion Transporter Blockade to Improve Beta Cell Function in Diabetes.

Degree: 2016, University of Toronto

The furan fatty acid metabolite 3-carboxy-4-methyl-5-propyl-2-furanpropanoic acid (CMPF) is elevated in Type 2 Diabetes. CMPF delivery to diabetic levels in mice causes glucose intolerance, impairing… (more)

Subjects/Keywords: Beta Cell; CMPF; Diabetes; OAT3; Organic Anion Transporter; Probenecid; 0719

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APA (6th Edition):

Eversley, J. A. (2016). Organic Anion Transporter Blockade to Improve Beta Cell Function in Diabetes. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/82402

Chicago Manual of Style (16th Edition):

Eversley, Judith Andrea. “Organic Anion Transporter Blockade to Improve Beta Cell Function in Diabetes.” 2016. Masters Thesis, University of Toronto. Accessed April 26, 2019. http://hdl.handle.net/1807/82402.

MLA Handbook (7th Edition):

Eversley, Judith Andrea. “Organic Anion Transporter Blockade to Improve Beta Cell Function in Diabetes.” 2016. Web. 26 Apr 2019.

Vancouver:

Eversley JA. Organic Anion Transporter Blockade to Improve Beta Cell Function in Diabetes. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2019 Apr 26]. Available from: http://hdl.handle.net/1807/82402.

Council of Science Editors:

Eversley JA. Organic Anion Transporter Blockade to Improve Beta Cell Function in Diabetes. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/82402


University of Toronto

7. Kim, Ji-Sun. The Role of Ryanodine Receptor on Neurite Outgrowth under Physiologic and Oxygen and Glucose-deprived Conditions.

Degree: 2016, University of Toronto

Ca2+ can enter a neuron from outside or can be released from internal stores, including endoplasmic reticulum (ER). Ryanodine receptor (RyR) is one of Ca2+… (more)

Subjects/Keywords: 0379

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APA (6th Edition):

Kim, J. (2016). The Role of Ryanodine Receptor on Neurite Outgrowth under Physiologic and Oxygen and Glucose-deprived Conditions. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/82405

Chicago Manual of Style (16th Edition):

Kim, Ji-Sun. “The Role of Ryanodine Receptor on Neurite Outgrowth under Physiologic and Oxygen and Glucose-deprived Conditions.” 2016. Masters Thesis, University of Toronto. Accessed April 26, 2019. http://hdl.handle.net/1807/82405.

MLA Handbook (7th Edition):

Kim, Ji-Sun. “The Role of Ryanodine Receptor on Neurite Outgrowth under Physiologic and Oxygen and Glucose-deprived Conditions.” 2016. Web. 26 Apr 2019.

Vancouver:

Kim J. The Role of Ryanodine Receptor on Neurite Outgrowth under Physiologic and Oxygen and Glucose-deprived Conditions. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2019 Apr 26]. Available from: http://hdl.handle.net/1807/82405.

Council of Science Editors:

Kim J. The Role of Ryanodine Receptor on Neurite Outgrowth under Physiologic and Oxygen and Glucose-deprived Conditions. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/82405


University of Toronto

8. Brathwaite, Shakira. The Role of Toll-like Receptor 4 in the Pathogenesis of Experimental Subarachnoid Hemorrhage.

Degree: 2016, University of Toronto

Subarachnoid hemorrhage (SAH) accounts for 5-7% of strokes worldwide and is associated with high mortality and morbidity. In addition to brain damage from the initial… (more)

Subjects/Keywords: Delayed Cerebral Ischemia; Inflammation; Subarachnoid hemorrhage; Toll-like receptor 4; 0317

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APA (6th Edition):

Brathwaite, S. (2016). The Role of Toll-like Receptor 4 in the Pathogenesis of Experimental Subarachnoid Hemorrhage. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/82406

Chicago Manual of Style (16th Edition):

Brathwaite, Shakira. “The Role of Toll-like Receptor 4 in the Pathogenesis of Experimental Subarachnoid Hemorrhage.” 2016. Masters Thesis, University of Toronto. Accessed April 26, 2019. http://hdl.handle.net/1807/82406.

MLA Handbook (7th Edition):

Brathwaite, Shakira. “The Role of Toll-like Receptor 4 in the Pathogenesis of Experimental Subarachnoid Hemorrhage.” 2016. Web. 26 Apr 2019.

Vancouver:

Brathwaite S. The Role of Toll-like Receptor 4 in the Pathogenesis of Experimental Subarachnoid Hemorrhage. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2019 Apr 26]. Available from: http://hdl.handle.net/1807/82406.

Council of Science Editors:

Brathwaite S. The Role of Toll-like Receptor 4 in the Pathogenesis of Experimental Subarachnoid Hemorrhage. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/82406


University of Toronto

9. Tahmasebi, Melika Zadeh. Ileal Fatty Acid Sensing Mechanisms Regulate Glucose Homeostasis In Vivo.

Degree: 2015, University of Toronto

Long-chain-fatty acids (LCFAs) are found in the diet, whereas short-chain fatty acids (SCFAs) are byproducts of fermentation in the distal intestine. It remains to be… (more)

Subjects/Keywords: Diabetes; glucagon-like peptide-1; glucose production; ileum; intestine; rats; 0719

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APA (6th Edition):

Tahmasebi, M. Z. (2015). Ileal Fatty Acid Sensing Mechanisms Regulate Glucose Homeostasis In Vivo. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/81088

Chicago Manual of Style (16th Edition):

Tahmasebi, Melika Zadeh. “Ileal Fatty Acid Sensing Mechanisms Regulate Glucose Homeostasis In Vivo.” 2015. Masters Thesis, University of Toronto. Accessed April 26, 2019. http://hdl.handle.net/1807/81088.

MLA Handbook (7th Edition):

Tahmasebi, Melika Zadeh. “Ileal Fatty Acid Sensing Mechanisms Regulate Glucose Homeostasis In Vivo.” 2015. Web. 26 Apr 2019.

Vancouver:

Tahmasebi MZ. Ileal Fatty Acid Sensing Mechanisms Regulate Glucose Homeostasis In Vivo. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2019 Apr 26]. Available from: http://hdl.handle.net/1807/81088.

Council of Science Editors:

Tahmasebi MZ. Ileal Fatty Acid Sensing Mechanisms Regulate Glucose Homeostasis In Vivo. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/81088


University of Toronto

10. Tahmasebi, Melika Zadeh. Ileal Fatty Acid Sensing Mechanisms Regulate Glucose Homeostasis In Vivo.

Degree: 2015, University of Toronto

Long-chain-fatty acids (LCFAs) are found in the diet, whereas short-chain fatty acids (SCFAs) are byproducts of fermentation in the distal intestine. It remains to be… (more)

Subjects/Keywords: Diabetes; glucagon-like peptide-1; glucose production; ileum; intestine; rats; 0719

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APA (6th Edition):

Tahmasebi, M. Z. (2015). Ileal Fatty Acid Sensing Mechanisms Regulate Glucose Homeostasis In Vivo. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/81087

Chicago Manual of Style (16th Edition):

Tahmasebi, Melika Zadeh. “Ileal Fatty Acid Sensing Mechanisms Regulate Glucose Homeostasis In Vivo.” 2015. Masters Thesis, University of Toronto. Accessed April 26, 2019. http://hdl.handle.net/1807/81087.

MLA Handbook (7th Edition):

Tahmasebi, Melika Zadeh. “Ileal Fatty Acid Sensing Mechanisms Regulate Glucose Homeostasis In Vivo.” 2015. Web. 26 Apr 2019.

Vancouver:

Tahmasebi MZ. Ileal Fatty Acid Sensing Mechanisms Regulate Glucose Homeostasis In Vivo. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2019 Apr 26]. Available from: http://hdl.handle.net/1807/81087.

Council of Science Editors:

Tahmasebi MZ. Ileal Fatty Acid Sensing Mechanisms Regulate Glucose Homeostasis In Vivo. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/81087


University of Toronto

11. Nahle, Ashraf. Nicotinamide Mononucleotide in the Context of Elevated Plasma Levels of Free Fatty Acids Improves Glucose Tolerance by Decreasing Insulin Clearance.

Degree: 2016, University of Toronto

The NAD-dependent deacetylase SIRT1 has been shown to be beneficial to beta cell function. Nicotinamide Mononucleotide (NMN), the product of the rate-limiting enzyme in NAD… (more)

Subjects/Keywords: beta cell dysfunction; insulin clearance; lipotoxicity; nicotinamide mononucleotide; SIRT; type 2 diabetes; 0409

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APA (6th Edition):

Nahle, A. (2016). Nicotinamide Mononucleotide in the Context of Elevated Plasma Levels of Free Fatty Acids Improves Glucose Tolerance by Decreasing Insulin Clearance. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/89209

Chicago Manual of Style (16th Edition):

Nahle, Ashraf. “Nicotinamide Mononucleotide in the Context of Elevated Plasma Levels of Free Fatty Acids Improves Glucose Tolerance by Decreasing Insulin Clearance.” 2016. Masters Thesis, University of Toronto. Accessed April 26, 2019. http://hdl.handle.net/1807/89209.

MLA Handbook (7th Edition):

Nahle, Ashraf. “Nicotinamide Mononucleotide in the Context of Elevated Plasma Levels of Free Fatty Acids Improves Glucose Tolerance by Decreasing Insulin Clearance.” 2016. Web. 26 Apr 2019.

Vancouver:

Nahle A. Nicotinamide Mononucleotide in the Context of Elevated Plasma Levels of Free Fatty Acids Improves Glucose Tolerance by Decreasing Insulin Clearance. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2019 Apr 26]. Available from: http://hdl.handle.net/1807/89209.

Council of Science Editors:

Nahle A. Nicotinamide Mononucleotide in the Context of Elevated Plasma Levels of Free Fatty Acids Improves Glucose Tolerance by Decreasing Insulin Clearance. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/89209


University of Toronto

12. Lee, Simon. Uncoupling Protein-2 Modulation of Reactive Oxygen Species and Cell Viability in the Pancreatic Beta Cell.

Degree: 2008, University of Toronto

Uncoupling protein-2 (UCP2) may be linked to the attenuation of reactive oxygen species (ROS), but it is unclear whether this phenomenon pertains to the pancreatic… (more)

Subjects/Keywords: uncoupling protein-2; diabetes; beta cell; reactive oxygen species; oxidative stress; cell viability; 0719

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APA (6th Edition):

Lee, S. (2008). Uncoupling Protein-2 Modulation of Reactive Oxygen Species and Cell Viability in the Pancreatic Beta Cell. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/11150

Chicago Manual of Style (16th Edition):

Lee, Simon. “Uncoupling Protein-2 Modulation of Reactive Oxygen Species and Cell Viability in the Pancreatic Beta Cell.” 2008. Masters Thesis, University of Toronto. Accessed April 26, 2019. http://hdl.handle.net/1807/11150.

MLA Handbook (7th Edition):

Lee, Simon. “Uncoupling Protein-2 Modulation of Reactive Oxygen Species and Cell Viability in the Pancreatic Beta Cell.” 2008. Web. 26 Apr 2019.

Vancouver:

Lee S. Uncoupling Protein-2 Modulation of Reactive Oxygen Species and Cell Viability in the Pancreatic Beta Cell. [Internet] [Masters thesis]. University of Toronto; 2008. [cited 2019 Apr 26]. Available from: http://hdl.handle.net/1807/11150.

Council of Science Editors:

Lee S. Uncoupling Protein-2 Modulation of Reactive Oxygen Species and Cell Viability in the Pancreatic Beta Cell. [Masters Thesis]. University of Toronto; 2008. Available from: http://hdl.handle.net/1807/11150


University of Toronto

13. Kena-Cohen, Veronique. Defining a Model of Classical Activation in Microglia.

Degree: 2008, University of Toronto

Microglia, the resident immune cells of the central nervous system, can become activated following injury, disease, or infection. In vitro, they can be activated by… (more)

Subjects/Keywords: Neuroinflammation; Microglia; Cytokine; cAMP Response Element Binding Protein; 0719

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APA (6th Edition):

Kena-Cohen, V. (2008). Defining a Model of Classical Activation in Microglia. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/17186

Chicago Manual of Style (16th Edition):

Kena-Cohen, Veronique. “Defining a Model of Classical Activation in Microglia.” 2008. Masters Thesis, University of Toronto. Accessed April 26, 2019. http://hdl.handle.net/1807/17186.

MLA Handbook (7th Edition):

Kena-Cohen, Veronique. “Defining a Model of Classical Activation in Microglia.” 2008. Web. 26 Apr 2019.

Vancouver:

Kena-Cohen V. Defining a Model of Classical Activation in Microglia. [Internet] [Masters thesis]. University of Toronto; 2008. [cited 2019 Apr 26]. Available from: http://hdl.handle.net/1807/17186.

Council of Science Editors:

Kena-Cohen V. Defining a Model of Classical Activation in Microglia. [Masters Thesis]. University of Toronto; 2008. Available from: http://hdl.handle.net/1807/17186


University of Toronto

14. Haufler, Darrell. On the Generation of Subthreshold Membrane Potential Fluctuations in Hippocampal CA1 Interneurons.

Degree: 2008, University of Toronto

A class of hippocampal interneurons in CA1, bordering the lacunosum-moleculare and radiatum hippocampal layers (the LM/R cell), has been shown to exhibit membrane potential oscillations… (more)

Subjects/Keywords: hippocampus; theta frequency oscillations; 0317

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APA (6th Edition):

Haufler, D. (2008). On the Generation of Subthreshold Membrane Potential Fluctuations in Hippocampal CA1 Interneurons. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/17177

Chicago Manual of Style (16th Edition):

Haufler, Darrell. “On the Generation of Subthreshold Membrane Potential Fluctuations in Hippocampal CA1 Interneurons.” 2008. Masters Thesis, University of Toronto. Accessed April 26, 2019. http://hdl.handle.net/1807/17177.

MLA Handbook (7th Edition):

Haufler, Darrell. “On the Generation of Subthreshold Membrane Potential Fluctuations in Hippocampal CA1 Interneurons.” 2008. Web. 26 Apr 2019.

Vancouver:

Haufler D. On the Generation of Subthreshold Membrane Potential Fluctuations in Hippocampal CA1 Interneurons. [Internet] [Masters thesis]. University of Toronto; 2008. [cited 2019 Apr 26]. Available from: http://hdl.handle.net/1807/17177.

Council of Science Editors:

Haufler D. On the Generation of Subthreshold Membrane Potential Fluctuations in Hippocampal CA1 Interneurons. [Masters Thesis]. University of Toronto; 2008. Available from: http://hdl.handle.net/1807/17177


University of Toronto

15. Mihic, Anton. Interaction of hERG Channels and Syntaxin 1A.

Degree: 2009, University of Toronto

The human ether-à-go-go related gene (hERG) encodes the pore-forming voltage-gated K+ channel that is essential for cardiac repolarization. Dr. Tsushima’s laboratory has previously characterized the… (more)

Subjects/Keywords: cardiovascular; molecular biology; electrophysiology; protein trafficking; ion channel; protein-protein interaction; patch clamp; hERG; SNARE; syntaxin; 0719

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APA (6th Edition):

Mihic, A. (2009). Interaction of hERG Channels and Syntaxin 1A. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/17438

Chicago Manual of Style (16th Edition):

Mihic, Anton. “Interaction of hERG Channels and Syntaxin 1A.” 2009. Masters Thesis, University of Toronto. Accessed April 26, 2019. http://hdl.handle.net/1807/17438.

MLA Handbook (7th Edition):

Mihic, Anton. “Interaction of hERG Channels and Syntaxin 1A.” 2009. Web. 26 Apr 2019.

Vancouver:

Mihic A. Interaction of hERG Channels and Syntaxin 1A. [Internet] [Masters thesis]. University of Toronto; 2009. [cited 2019 Apr 26]. Available from: http://hdl.handle.net/1807/17438.

Council of Science Editors:

Mihic A. Interaction of hERG Channels and Syntaxin 1A. [Masters Thesis]. University of Toronto; 2009. Available from: http://hdl.handle.net/1807/17438


University of Toronto

16. Pabari, Reena. ICAM-1 as a Novel Binding Partner for LPS to Mediate TLR4-Independent Cell Activation.

Degree: 2009, University of Toronto

Introduction: The mechanism of cell activation by LPS in the absence of surface Toll-like receptor 4 (TLR4) is unclear. We hypothesize that ICAM-1 binds LPS… (more)

Subjects/Keywords: Lipopolysaccharide; ICAM-1; TLR4-Independent; 0719

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APA (6th Edition):

Pabari, R. (2009). ICAM-1 as a Novel Binding Partner for LPS to Mediate TLR4-Independent Cell Activation. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/17702

Chicago Manual of Style (16th Edition):

Pabari, Reena. “ICAM-1 as a Novel Binding Partner for LPS to Mediate TLR4-Independent Cell Activation.” 2009. Masters Thesis, University of Toronto. Accessed April 26, 2019. http://hdl.handle.net/1807/17702.

MLA Handbook (7th Edition):

Pabari, Reena. “ICAM-1 as a Novel Binding Partner for LPS to Mediate TLR4-Independent Cell Activation.” 2009. Web. 26 Apr 2019.

Vancouver:

Pabari R. ICAM-1 as a Novel Binding Partner for LPS to Mediate TLR4-Independent Cell Activation. [Internet] [Masters thesis]. University of Toronto; 2009. [cited 2019 Apr 26]. Available from: http://hdl.handle.net/1807/17702.

Council of Science Editors:

Pabari R. ICAM-1 as a Novel Binding Partner for LPS to Mediate TLR4-Independent Cell Activation. [Masters Thesis]. University of Toronto; 2009. Available from: http://hdl.handle.net/1807/17702


University of Toronto

17. Manwani, Neetu. Role of Distal Airway Epithelial Glucocorticoid-Glucocorticoid Receptor Signalling in Mouse Lung in Late Gestation.

Degree: 2009, University of Toronto

Glucocorticoid (GC) signalling via the GC receptor (GR) regulates many aspects of lung development. To determine the need for epithelial GC-GR signalling, triple transgenic (TT)… (more)

Subjects/Keywords: Lung; Development; Glucocorticoid Receptor; 0758

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APA (6th Edition):

Manwani, N. (2009). Role of Distal Airway Epithelial Glucocorticoid-Glucocorticoid Receptor Signalling in Mouse Lung in Late Gestation. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/17697

Chicago Manual of Style (16th Edition):

Manwani, Neetu. “Role of Distal Airway Epithelial Glucocorticoid-Glucocorticoid Receptor Signalling in Mouse Lung in Late Gestation.” 2009. Masters Thesis, University of Toronto. Accessed April 26, 2019. http://hdl.handle.net/1807/17697.

MLA Handbook (7th Edition):

Manwani, Neetu. “Role of Distal Airway Epithelial Glucocorticoid-Glucocorticoid Receptor Signalling in Mouse Lung in Late Gestation.” 2009. Web. 26 Apr 2019.

Vancouver:

Manwani N. Role of Distal Airway Epithelial Glucocorticoid-Glucocorticoid Receptor Signalling in Mouse Lung in Late Gestation. [Internet] [Masters thesis]. University of Toronto; 2009. [cited 2019 Apr 26]. Available from: http://hdl.handle.net/1807/17697.

Council of Science Editors:

Manwani N. Role of Distal Airway Epithelial Glucocorticoid-Glucocorticoid Receptor Signalling in Mouse Lung in Late Gestation. [Masters Thesis]. University of Toronto; 2009. Available from: http://hdl.handle.net/1807/17697


University of Toronto

18. Liang, Philip. Novel Formulations of Antioxidant and Anti-inflammatory Drugs to Ameliorate Ischemic Damage Measured In Vitro.

Degree: 2009, University of Toronto

The two of major pathways that cause ischemic damage are oxidative stress and inflammation. To decreasing oxidative stress and inflammation, new anti-oxidant and anti-inflammatory agents… (more)

Subjects/Keywords: Neuroinflammation; Ischemia; 0719

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APA (6th Edition):

Liang, P. (2009). Novel Formulations of Antioxidant and Anti-inflammatory Drugs to Ameliorate Ischemic Damage Measured In Vitro. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/17434

Chicago Manual of Style (16th Edition):

Liang, Philip. “Novel Formulations of Antioxidant and Anti-inflammatory Drugs to Ameliorate Ischemic Damage Measured In Vitro.” 2009. Masters Thesis, University of Toronto. Accessed April 26, 2019. http://hdl.handle.net/1807/17434.

MLA Handbook (7th Edition):

Liang, Philip. “Novel Formulations of Antioxidant and Anti-inflammatory Drugs to Ameliorate Ischemic Damage Measured In Vitro.” 2009. Web. 26 Apr 2019.

Vancouver:

Liang P. Novel Formulations of Antioxidant and Anti-inflammatory Drugs to Ameliorate Ischemic Damage Measured In Vitro. [Internet] [Masters thesis]. University of Toronto; 2009. [cited 2019 Apr 26]. Available from: http://hdl.handle.net/1807/17434.

Council of Science Editors:

Liang P. Novel Formulations of Antioxidant and Anti-inflammatory Drugs to Ameliorate Ischemic Damage Measured In Vitro. [Masters Thesis]. University of Toronto; 2009. Available from: http://hdl.handle.net/1807/17434


University of Toronto

19. Sheppy, Evan Anthony. Behavioral and Electrographic Abnormalities due to Repeated Hypoglycemic Episodes in Mice.

Degree: 2009, University of Toronto

Severe hypoglycemia poses the greatest challenge to glycemic control in diabetic patients, especially children with type 1 diabetes mellitus. Although in vivo animal models exist… (more)

Subjects/Keywords: Hypoglycemia; Physiology; Electroencephalography; Diabetes; 0719

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APA (6th Edition):

Sheppy, E. A. (2009). Behavioral and Electrographic Abnormalities due to Repeated Hypoglycemic Episodes in Mice. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/17714

Chicago Manual of Style (16th Edition):

Sheppy, Evan Anthony. “Behavioral and Electrographic Abnormalities due to Repeated Hypoglycemic Episodes in Mice.” 2009. Masters Thesis, University of Toronto. Accessed April 26, 2019. http://hdl.handle.net/1807/17714.

MLA Handbook (7th Edition):

Sheppy, Evan Anthony. “Behavioral and Electrographic Abnormalities due to Repeated Hypoglycemic Episodes in Mice.” 2009. Web. 26 Apr 2019.

Vancouver:

Sheppy EA. Behavioral and Electrographic Abnormalities due to Repeated Hypoglycemic Episodes in Mice. [Internet] [Masters thesis]. University of Toronto; 2009. [cited 2019 Apr 26]. Available from: http://hdl.handle.net/1807/17714.

Council of Science Editors:

Sheppy EA. Behavioral and Electrographic Abnormalities due to Repeated Hypoglycemic Episodes in Mice. [Masters Thesis]. University of Toronto; 2009. Available from: http://hdl.handle.net/1807/17714


University of Toronto

20. Ahmed, Mijhgan. Developmental Regulation of the type-A Gamma-Aminobutyric Acid Receptor (GABA-AR) Signaling in the Fetal Rat Lung.

Degree: 2009, University of Toronto

The fetal lung epithelium secretes fluid into the potential pulmonary air-spaces by actively transporting chloride (Cl¯) into the lung lumen. This Cl¯-driven fluid secretion declines… (more)

Subjects/Keywords: Lung Development; Type-A Gamma Aminobutyric Acid Receptor (GABA-AR) and Glutamic Acid Decarboxylase (GAD) in Fetal Lungs; Fluid Secretion in the Mammalian Lungs; Cell and Molecular Biology; 0719; 0307; 0306; 0433; 0379; 0566; 0564; 0317; 0571; 0350

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APA (6th Edition):

Ahmed, M. (2009). Developmental Regulation of the type-A Gamma-Aminobutyric Acid Receptor (GABA-AR) Signaling in the Fetal Rat Lung. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/17491

Chicago Manual of Style (16th Edition):

Ahmed, Mijhgan. “Developmental Regulation of the type-A Gamma-Aminobutyric Acid Receptor (GABA-AR) Signaling in the Fetal Rat Lung.” 2009. Masters Thesis, University of Toronto. Accessed April 26, 2019. http://hdl.handle.net/1807/17491.

MLA Handbook (7th Edition):

Ahmed, Mijhgan. “Developmental Regulation of the type-A Gamma-Aminobutyric Acid Receptor (GABA-AR) Signaling in the Fetal Rat Lung.” 2009. Web. 26 Apr 2019.

Vancouver:

Ahmed M. Developmental Regulation of the type-A Gamma-Aminobutyric Acid Receptor (GABA-AR) Signaling in the Fetal Rat Lung. [Internet] [Masters thesis]. University of Toronto; 2009. [cited 2019 Apr 26]. Available from: http://hdl.handle.net/1807/17491.

Council of Science Editors:

Ahmed M. Developmental Regulation of the type-A Gamma-Aminobutyric Acid Receptor (GABA-AR) Signaling in the Fetal Rat Lung. [Masters Thesis]. University of Toronto; 2009. Available from: http://hdl.handle.net/1807/17491


University of Toronto

21. Diec, Diana. The Effects of Oxidative Stress on Calcineurin Activity and DJ-1 Subcellular Localization.

Degree: 2009, University of Toronto

Oxidative stress and mutations in DJ-1, a redox sensitive protein, are linked to Parkinson's Disease. The protective mechanism of DJ-1 is unclear. I hypothesized that:… (more)

Subjects/Keywords: oxidative stress; DJ-1, calcineurin; 0317

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APA (6th Edition):

Diec, D. (2009). The Effects of Oxidative Stress on Calcineurin Activity and DJ-1 Subcellular Localization. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/18279

Chicago Manual of Style (16th Edition):

Diec, Diana. “The Effects of Oxidative Stress on Calcineurin Activity and DJ-1 Subcellular Localization.” 2009. Masters Thesis, University of Toronto. Accessed April 26, 2019. http://hdl.handle.net/1807/18279.

MLA Handbook (7th Edition):

Diec, Diana. “The Effects of Oxidative Stress on Calcineurin Activity and DJ-1 Subcellular Localization.” 2009. Web. 26 Apr 2019.

Vancouver:

Diec D. The Effects of Oxidative Stress on Calcineurin Activity and DJ-1 Subcellular Localization. [Internet] [Masters thesis]. University of Toronto; 2009. [cited 2019 Apr 26]. Available from: http://hdl.handle.net/1807/18279.

Council of Science Editors:

Diec D. The Effects of Oxidative Stress on Calcineurin Activity and DJ-1 Subcellular Localization. [Masters Thesis]. University of Toronto; 2009. Available from: http://hdl.handle.net/1807/18279


University of Toronto

22. Henriques, Melanie Dawn. Human Neutrophil Peptides: A Novel Agonist of Platelet Activation and Aggregation.

Degree: 2009, University of Toronto

INTRODUCTION: Platelets are involved in the inflammatory and thrombotic complications associated with atherosclerosis. Human neutrophil peptides (HNP), released from activated neutrophils, demonstrate inflammatory effects related… (more)

Subjects/Keywords: atherosclerosis; inflammation; alpha-defensins; 0719

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APA (6th Edition):

Henriques, M. D. (2009). Human Neutrophil Peptides: A Novel Agonist of Platelet Activation and Aggregation. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/18343

Chicago Manual of Style (16th Edition):

Henriques, Melanie Dawn. “Human Neutrophil Peptides: A Novel Agonist of Platelet Activation and Aggregation.” 2009. Masters Thesis, University of Toronto. Accessed April 26, 2019. http://hdl.handle.net/1807/18343.

MLA Handbook (7th Edition):

Henriques, Melanie Dawn. “Human Neutrophil Peptides: A Novel Agonist of Platelet Activation and Aggregation.” 2009. Web. 26 Apr 2019.

Vancouver:

Henriques MD. Human Neutrophil Peptides: A Novel Agonist of Platelet Activation and Aggregation. [Internet] [Masters thesis]. University of Toronto; 2009. [cited 2019 Apr 26]. Available from: http://hdl.handle.net/1807/18343.

Council of Science Editors:

Henriques MD. Human Neutrophil Peptides: A Novel Agonist of Platelet Activation and Aggregation. [Masters Thesis]. University of Toronto; 2009. Available from: http://hdl.handle.net/1807/18343


University of Toronto

23. Sellan, Michael. The Eelectrophysiological Effects of Iron Overload on the Heart.

Degree: 2009, University of Toronto

Chronic iron overload (CIO) in patients leads to a cardiomyopathy characterized by conduction defects, including bradyarrhythmias. Using a murine model of CIO, we explored the… (more)

Subjects/Keywords: Cardiac; Electrophysiology; Iron Overload; Calcium; 0719

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APA (6th Edition):

Sellan, M. (2009). The Eelectrophysiological Effects of Iron Overload on the Heart. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/18852

Chicago Manual of Style (16th Edition):

Sellan, Michael. “The Eelectrophysiological Effects of Iron Overload on the Heart.” 2009. Masters Thesis, University of Toronto. Accessed April 26, 2019. http://hdl.handle.net/1807/18852.

MLA Handbook (7th Edition):

Sellan, Michael. “The Eelectrophysiological Effects of Iron Overload on the Heart.” 2009. Web. 26 Apr 2019.

Vancouver:

Sellan M. The Eelectrophysiological Effects of Iron Overload on the Heart. [Internet] [Masters thesis]. University of Toronto; 2009. [cited 2019 Apr 26]. Available from: http://hdl.handle.net/1807/18852.

Council of Science Editors:

Sellan M. The Eelectrophysiological Effects of Iron Overload on the Heart. [Masters Thesis]. University of Toronto; 2009. Available from: http://hdl.handle.net/1807/18852


University of Toronto

24. Leen, Jason. The Effect of Glucagon-like Peptide-2 on Insulin-like Growth Factor-1 in Murine Intestinal Subepithelial Myofibroblasts.

Degree: 2009, University of Toronto

Insulin-like growth factor-1 (IGF-1), a known secretory product of intestinal subepithelial myofibroblasts (ISEMF), is essential for the intestinotrophic effects of glucagon-like peptide-2(GLP-2). I hypothesized that… (more)

Subjects/Keywords: Cell cultures; Glucagon-like peptide-2; Subepithelial myofibroblasts; Insulin-like growth factor-1; PI3K/AKT signaling; Intestinal growth; Real time RT-PCR; Western blots; Immunocytochemistry; RIA; 0719

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APA (6th Edition):

Leen, J. (2009). The Effect of Glucagon-like Peptide-2 on Insulin-like Growth Factor-1 in Murine Intestinal Subepithelial Myofibroblasts. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/18828

Chicago Manual of Style (16th Edition):

Leen, Jason. “The Effect of Glucagon-like Peptide-2 on Insulin-like Growth Factor-1 in Murine Intestinal Subepithelial Myofibroblasts.” 2009. Masters Thesis, University of Toronto. Accessed April 26, 2019. http://hdl.handle.net/1807/18828.

MLA Handbook (7th Edition):

Leen, Jason. “The Effect of Glucagon-like Peptide-2 on Insulin-like Growth Factor-1 in Murine Intestinal Subepithelial Myofibroblasts.” 2009. Web. 26 Apr 2019.

Vancouver:

Leen J. The Effect of Glucagon-like Peptide-2 on Insulin-like Growth Factor-1 in Murine Intestinal Subepithelial Myofibroblasts. [Internet] [Masters thesis]. University of Toronto; 2009. [cited 2019 Apr 26]. Available from: http://hdl.handle.net/1807/18828.

Council of Science Editors:

Leen J. The Effect of Glucagon-like Peptide-2 on Insulin-like Growth Factor-1 in Murine Intestinal Subepithelial Myofibroblasts. [Masters Thesis]. University of Toronto; 2009. Available from: http://hdl.handle.net/1807/18828


University of Toronto

25. Thomas, Garry. Insulin Sensitivity is Enhanced by CGMP-mediated MAPK Inhibition in Rat Adipocytes.

Degree: 2009, University of Toronto

Bradykinin (BK) acts through eNOS to reduce MAPK-mediated feedback inhibition of insulin signalling. Preliminary data suggest that the sGC-cGMP-PKG pathway, a prominent NO target, is… (more)

Subjects/Keywords: ANP; insulin resistance; bradykinin; diabetes; MAPK; AKT; PKB; cGMP; PKG; 0307

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APA (6th Edition):

Thomas, G. (2009). Insulin Sensitivity is Enhanced by CGMP-mediated MAPK Inhibition in Rat Adipocytes. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/18962

Chicago Manual of Style (16th Edition):

Thomas, Garry. “Insulin Sensitivity is Enhanced by CGMP-mediated MAPK Inhibition in Rat Adipocytes.” 2009. Masters Thesis, University of Toronto. Accessed April 26, 2019. http://hdl.handle.net/1807/18962.

MLA Handbook (7th Edition):

Thomas, Garry. “Insulin Sensitivity is Enhanced by CGMP-mediated MAPK Inhibition in Rat Adipocytes.” 2009. Web. 26 Apr 2019.

Vancouver:

Thomas G. Insulin Sensitivity is Enhanced by CGMP-mediated MAPK Inhibition in Rat Adipocytes. [Internet] [Masters thesis]. University of Toronto; 2009. [cited 2019 Apr 26]. Available from: http://hdl.handle.net/1807/18962.

Council of Science Editors:

Thomas G. Insulin Sensitivity is Enhanced by CGMP-mediated MAPK Inhibition in Rat Adipocytes. [Masters Thesis]. University of Toronto; 2009. Available from: http://hdl.handle.net/1807/18962


University of Toronto

26. Khalid, Sarah. The Relationship Between Insulin Resistance and Hyperinsulinemia on Mammary Cancer Growth and Development.

Degree: 2009, University of Toronto

Insulin resistance associated with obesity has been suggested to contribute to an increased risk and poor prognosis for breast cancer. In this study, a HER2/Neu… (more)

Subjects/Keywords: Hyperinsulinemia; Breast Cancer; 0307

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APA (6th Edition):

Khalid, S. (2009). The Relationship Between Insulin Resistance and Hyperinsulinemia on Mammary Cancer Growth and Development. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/19312

Chicago Manual of Style (16th Edition):

Khalid, Sarah. “The Relationship Between Insulin Resistance and Hyperinsulinemia on Mammary Cancer Growth and Development.” 2009. Masters Thesis, University of Toronto. Accessed April 26, 2019. http://hdl.handle.net/1807/19312.

MLA Handbook (7th Edition):

Khalid, Sarah. “The Relationship Between Insulin Resistance and Hyperinsulinemia on Mammary Cancer Growth and Development.” 2009. Web. 26 Apr 2019.

Vancouver:

Khalid S. The Relationship Between Insulin Resistance and Hyperinsulinemia on Mammary Cancer Growth and Development. [Internet] [Masters thesis]. University of Toronto; 2009. [cited 2019 Apr 26]. Available from: http://hdl.handle.net/1807/19312.

Council of Science Editors:

Khalid S. The Relationship Between Insulin Resistance and Hyperinsulinemia on Mammary Cancer Growth and Development. [Masters Thesis]. University of Toronto; 2009. Available from: http://hdl.handle.net/1807/19312


University of Toronto

27. Alves, Nyresa. Sex Differences in Nicotinic Currents of Layer VI Neurons of Prefrontal Cortex During Development.

Degree: 2009, University of Toronto

There is a large sex difference in the prevalence of attention deficit disorder; yet, little is known about sex differences in prefrontal attention circuitry. We… (more)

Subjects/Keywords: prefrontal cortex; nicotine; sex difference; electrophysiology; development; progesterone; 0719

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APA (6th Edition):

Alves, N. (2009). Sex Differences in Nicotinic Currents of Layer VI Neurons of Prefrontal Cortex During Development. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/18137

Chicago Manual of Style (16th Edition):

Alves, Nyresa. “Sex Differences in Nicotinic Currents of Layer VI Neurons of Prefrontal Cortex During Development.” 2009. Masters Thesis, University of Toronto. Accessed April 26, 2019. http://hdl.handle.net/1807/18137.

MLA Handbook (7th Edition):

Alves, Nyresa. “Sex Differences in Nicotinic Currents of Layer VI Neurons of Prefrontal Cortex During Development.” 2009. Web. 26 Apr 2019.

Vancouver:

Alves N. Sex Differences in Nicotinic Currents of Layer VI Neurons of Prefrontal Cortex During Development. [Internet] [Masters thesis]. University of Toronto; 2009. [cited 2019 Apr 26]. Available from: http://hdl.handle.net/1807/18137.

Council of Science Editors:

Alves N. Sex Differences in Nicotinic Currents of Layer VI Neurons of Prefrontal Cortex During Development. [Masters Thesis]. University of Toronto; 2009. Available from: http://hdl.handle.net/1807/18137


University of Toronto

28. Gaisano, Gregory. Identifying Novel Protein Interactors of the Glucagon Superfamily of Receptors.

Degree: 2009, University of Toronto

G-protein coupled receptors (GPCRs) have been shown to act as part of GPCR associated protein complexes (GAPCs) which are required to appropriately transduce downstream signaling… (more)

Subjects/Keywords: GPCR; protein-protein interaction; glucagon; B class GCPR; GLP-1; GIPR; GLP-2R; GCGR; 0719; 0307; 0369

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APA (6th Edition):

Gaisano, G. (2009). Identifying Novel Protein Interactors of the Glucagon Superfamily of Receptors. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/18304

Chicago Manual of Style (16th Edition):

Gaisano, Gregory. “Identifying Novel Protein Interactors of the Glucagon Superfamily of Receptors.” 2009. Masters Thesis, University of Toronto. Accessed April 26, 2019. http://hdl.handle.net/1807/18304.

MLA Handbook (7th Edition):

Gaisano, Gregory. “Identifying Novel Protein Interactors of the Glucagon Superfamily of Receptors.” 2009. Web. 26 Apr 2019.

Vancouver:

Gaisano G. Identifying Novel Protein Interactors of the Glucagon Superfamily of Receptors. [Internet] [Masters thesis]. University of Toronto; 2009. [cited 2019 Apr 26]. Available from: http://hdl.handle.net/1807/18304.

Council of Science Editors:

Gaisano G. Identifying Novel Protein Interactors of the Glucagon Superfamily of Receptors. [Masters Thesis]. University of Toronto; 2009. Available from: http://hdl.handle.net/1807/18304


University of Toronto

29. Jiang, Tiandan T. J. Roles of Sec5 in the Regulation of Dense-Core Vesicle Secretion in PC12 Cells.

Degree: 2009, University of Toronto

The exocyst is thought to tether secretory vesicles to specific sites on the plasma membrane. As a member of the exocyst, Sec5 is implicated in… (more)

Subjects/Keywords: dense-core vesicle; exocytosis; docking; tethering; exocyst; Sec5; 0719

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APA (6th Edition):

Jiang, T. T. J. (2009). Roles of Sec5 in the Regulation of Dense-Core Vesicle Secretion in PC12 Cells. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/25705

Chicago Manual of Style (16th Edition):

Jiang, Tiandan T J. “Roles of Sec5 in the Regulation of Dense-Core Vesicle Secretion in PC12 Cells.” 2009. Masters Thesis, University of Toronto. Accessed April 26, 2019. http://hdl.handle.net/1807/25705.

MLA Handbook (7th Edition):

Jiang, Tiandan T J. “Roles of Sec5 in the Regulation of Dense-Core Vesicle Secretion in PC12 Cells.” 2009. Web. 26 Apr 2019.

Vancouver:

Jiang TTJ. Roles of Sec5 in the Regulation of Dense-Core Vesicle Secretion in PC12 Cells. [Internet] [Masters thesis]. University of Toronto; 2009. [cited 2019 Apr 26]. Available from: http://hdl.handle.net/1807/25705.

Council of Science Editors:

Jiang TTJ. Roles of Sec5 in the Regulation of Dense-Core Vesicle Secretion in PC12 Cells. [Masters Thesis]. University of Toronto; 2009. Available from: http://hdl.handle.net/1807/25705


University of Toronto

30. Regan, Rosemary. Change in Middle Cerebral Artery Velocity over Time to an Acute and Sustained Stimulus.

Degree: 2009, University of Toronto

Little is known of the temporal cerebral blood flow response to a chemical stimulus consisting of increased PCO2 measured over time. Currently, there is only… (more)

Subjects/Keywords: Cerebral Blood Flow; Hypercapnia; Blood Pressure; 0719

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APA (6th Edition):

Regan, R. (2009). Change in Middle Cerebral Artery Velocity over Time to an Acute and Sustained Stimulus. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/18844

Chicago Manual of Style (16th Edition):

Regan, Rosemary. “Change in Middle Cerebral Artery Velocity over Time to an Acute and Sustained Stimulus.” 2009. Masters Thesis, University of Toronto. Accessed April 26, 2019. http://hdl.handle.net/1807/18844.

MLA Handbook (7th Edition):

Regan, Rosemary. “Change in Middle Cerebral Artery Velocity over Time to an Acute and Sustained Stimulus.” 2009. Web. 26 Apr 2019.

Vancouver:

Regan R. Change in Middle Cerebral Artery Velocity over Time to an Acute and Sustained Stimulus. [Internet] [Masters thesis]. University of Toronto; 2009. [cited 2019 Apr 26]. Available from: http://hdl.handle.net/1807/18844.

Council of Science Editors:

Regan R. Change in Middle Cerebral Artery Velocity over Time to an Acute and Sustained Stimulus. [Masters Thesis]. University of Toronto; 2009. Available from: http://hdl.handle.net/1807/18844

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