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You searched for +publisher:"University of Toronto" +contributor:("Pharmacology"). Showing records 1 – 30 of 189 total matches.

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University of Toronto

1. Vecchio, Laura Marie. Investigating Monoamine Regulation: The Transport Of The Dopamine Transporter To Striatal Terminals And The Effects Of Tyrosine Hydroxylase Overexpression On Dopaminergic and Noradrenergic-Related Phenotypes.

Degree: PhD, 2016, University of Toronto

Monoamines are a class of neurotransmitter responsible for a variety of functions, from the orchestration of movement to the feeling of reward. The maintenance of… (more)

Subjects/Keywords: catecholamines; dopamine; dopamine transporter; Parkinson's disease; transgenic mice; tyrosine hydroxylase; 0317

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APA (6th Edition):

Vecchio, L. M. (2016). Investigating Monoamine Regulation: The Transport Of The Dopamine Transporter To Striatal Terminals And The Effects Of Tyrosine Hydroxylase Overexpression On Dopaminergic and Noradrenergic-Related Phenotypes. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/82399

Chicago Manual of Style (16th Edition):

Vecchio, Laura Marie. “Investigating Monoamine Regulation: The Transport Of The Dopamine Transporter To Striatal Terminals And The Effects Of Tyrosine Hydroxylase Overexpression On Dopaminergic and Noradrenergic-Related Phenotypes.” 2016. Doctoral Dissertation, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/82399.

MLA Handbook (7th Edition):

Vecchio, Laura Marie. “Investigating Monoamine Regulation: The Transport Of The Dopamine Transporter To Striatal Terminals And The Effects Of Tyrosine Hydroxylase Overexpression On Dopaminergic and Noradrenergic-Related Phenotypes.” 2016. Web. 20 Jul 2018.

Vancouver:

Vecchio LM. Investigating Monoamine Regulation: The Transport Of The Dopamine Transporter To Striatal Terminals And The Effects Of Tyrosine Hydroxylase Overexpression On Dopaminergic and Noradrenergic-Related Phenotypes. [Internet] [Doctoral dissertation]. University of Toronto; 2016. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/82399.

Council of Science Editors:

Vecchio LM. Investigating Monoamine Regulation: The Transport Of The Dopamine Transporter To Striatal Terminals And The Effects Of Tyrosine Hydroxylase Overexpression On Dopaminergic and Noradrenergic-Related Phenotypes. [Doctoral Dissertation]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/82399


University of Toronto

2. Baber, Marta. Therapeutic and Substance Abuse Biomarkers in the Immediate Postpartum Period.

Degree: PhD, 2017, University of Toronto

 The aim of this thesis was to develop the use of therapeutic and substance abuse biomarkers relevant to the immediate postpartum period, to reduce the… (more)

Subjects/Keywords: fatty acid ethyl esters; fetal alcohol spectrum disorder; neonatal hair; opioids; pharmacogenetics; post-cesarean; 0419

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APA (6th Edition):

Baber, M. (2017). Therapeutic and Substance Abuse Biomarkers in the Immediate Postpartum Period. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/77980

Chicago Manual of Style (16th Edition):

Baber, Marta. “Therapeutic and Substance Abuse Biomarkers in the Immediate Postpartum Period.” 2017. Doctoral Dissertation, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/77980.

MLA Handbook (7th Edition):

Baber, Marta. “Therapeutic and Substance Abuse Biomarkers in the Immediate Postpartum Period.” 2017. Web. 20 Jul 2018.

Vancouver:

Baber M. Therapeutic and Substance Abuse Biomarkers in the Immediate Postpartum Period. [Internet] [Doctoral dissertation]. University of Toronto; 2017. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/77980.

Council of Science Editors:

Baber M. Therapeutic and Substance Abuse Biomarkers in the Immediate Postpartum Period. [Doctoral Dissertation]. University of Toronto; 2017. Available from: http://hdl.handle.net/1807/77980


University of Toronto

3. Beerepoot, Pieter Claus. Pharmacological Chaperones of the Dopamine Transporter: A Strategy for Increasing Function of Wild Type and Mutant Transporter.

Degree: PhD, 2017, University of Toronto

 The dopamine transporter (DAT) is a membrane protein that is essential for regulating signaling and intracellular stores of the neurotransmitter dopamine. An array of pathological… (more)

Subjects/Keywords: Dopamine; Dopamine transporter deficiency syndrome; Pharmacological chaperones; Protein folding; 0419

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APA (6th Edition):

Beerepoot, P. C. (2017). Pharmacological Chaperones of the Dopamine Transporter: A Strategy for Increasing Function of Wild Type and Mutant Transporter. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/78039

Chicago Manual of Style (16th Edition):

Beerepoot, Pieter Claus. “Pharmacological Chaperones of the Dopamine Transporter: A Strategy for Increasing Function of Wild Type and Mutant Transporter.” 2017. Doctoral Dissertation, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/78039.

MLA Handbook (7th Edition):

Beerepoot, Pieter Claus. “Pharmacological Chaperones of the Dopamine Transporter: A Strategy for Increasing Function of Wild Type and Mutant Transporter.” 2017. Web. 20 Jul 2018.

Vancouver:

Beerepoot PC. Pharmacological Chaperones of the Dopamine Transporter: A Strategy for Increasing Function of Wild Type and Mutant Transporter. [Internet] [Doctoral dissertation]. University of Toronto; 2017. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/78039.

Council of Science Editors:

Beerepoot PC. Pharmacological Chaperones of the Dopamine Transporter: A Strategy for Increasing Function of Wild Type and Mutant Transporter. [Doctoral Dissertation]. University of Toronto; 2017. Available from: http://hdl.handle.net/1807/78039


University of Toronto

4. Rui, Xianliang. Transcriptional Regulation of the Mouse Adrenal Cyclase Type 4 (Adcy4) in Y1 Adrenocortical Tumor Cells.

Degree: 2010, University of Toronto

Adenylyl cyclase (Adcy) is an important early effector of adrenocorticotrophin (ACTH) on the adrenal cortex; however, this enzyme consists of ten isozymes in mammalian cells… (more)

Subjects/Keywords: Adenylyl cyclase; Steroidogenesis; Steroidogenic factor 1 (SF1); Gene regulation; Pharmacology 0419

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APA (6th Edition):

Rui, X. (2010). Transcriptional Regulation of the Mouse Adrenal Cyclase Type 4 (Adcy4) in Y1 Adrenocortical Tumor Cells. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/24451

Chicago Manual of Style (16th Edition):

Rui, Xianliang. “Transcriptional Regulation of the Mouse Adrenal Cyclase Type 4 (Adcy4) in Y1 Adrenocortical Tumor Cells.” 2010. Doctoral Dissertation, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/24451.

MLA Handbook (7th Edition):

Rui, Xianliang. “Transcriptional Regulation of the Mouse Adrenal Cyclase Type 4 (Adcy4) in Y1 Adrenocortical Tumor Cells.” 2010. Web. 20 Jul 2018.

Vancouver:

Rui X. Transcriptional Regulation of the Mouse Adrenal Cyclase Type 4 (Adcy4) in Y1 Adrenocortical Tumor Cells. [Internet] [Doctoral dissertation]. University of Toronto; 2010. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/24451.

Council of Science Editors:

Rui X. Transcriptional Regulation of the Mouse Adrenal Cyclase Type 4 (Adcy4) in Y1 Adrenocortical Tumor Cells. [Doctoral Dissertation]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/24451


University of Toronto

5. Siu, Eric C. K. An Investigation of Nicotine Metabolism in Mice: The Impact of Pharmacological Inhibition and Genetic Influences on Nicotine Pharmacology.

Degree: 2010, University of Toronto

INTRODUCTION: Smoking is one of the single greatest causes of numerous preventable diseases. We were interested in developing an animal model of nicotine metabolism that… (more)

Subjects/Keywords: Nicotine; Mice; CYP2A5; CYP2A6; Selegiline; Methoxsalen; Nicotine Self-administration; Genetic; Tail-flick; Hot-plate; Mechanism-based Inhibition; Smoking; Metabolism; 0419

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APA (6th Edition):

Siu, E. C. K. (2010). An Investigation of Nicotine Metabolism in Mice: The Impact of Pharmacological Inhibition and Genetic Influences on Nicotine Pharmacology. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/24879

Chicago Manual of Style (16th Edition):

Siu, Eric C K. “An Investigation of Nicotine Metabolism in Mice: The Impact of Pharmacological Inhibition and Genetic Influences on Nicotine Pharmacology.” 2010. Doctoral Dissertation, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/24879.

MLA Handbook (7th Edition):

Siu, Eric C K. “An Investigation of Nicotine Metabolism in Mice: The Impact of Pharmacological Inhibition and Genetic Influences on Nicotine Pharmacology.” 2010. Web. 20 Jul 2018.

Vancouver:

Siu ECK. An Investigation of Nicotine Metabolism in Mice: The Impact of Pharmacological Inhibition and Genetic Influences on Nicotine Pharmacology. [Internet] [Doctoral dissertation]. University of Toronto; 2010. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/24879.

Council of Science Editors:

Siu ECK. An Investigation of Nicotine Metabolism in Mice: The Impact of Pharmacological Inhibition and Genetic Influences on Nicotine Pharmacology. [Doctoral Dissertation]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/24879


University of Toronto

6. Ptak, Carolyn. Genetic-epigenetic Interactions: Sequence-dependent and Independent DNA Nethylation.

Degree: 2013, University of Toronto

The field of human epigenetics has become widely accepted, yet many basic principles remain unclear. It is important to determine the extent to which DNA… (more)

Subjects/Keywords: epigenetics; genetics; twins; DNA methylation; microarray; allele-specific methylation; major psychosis; epigenetic inheritance; 0307

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APA (6th Edition):

Ptak, C. (2013). Genetic-epigenetic Interactions: Sequence-dependent and Independent DNA Nethylation. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/69006

Chicago Manual of Style (16th Edition):

Ptak, Carolyn. “Genetic-epigenetic Interactions: Sequence-dependent and Independent DNA Nethylation.” 2013. Doctoral Dissertation, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/69006.

MLA Handbook (7th Edition):

Ptak, Carolyn. “Genetic-epigenetic Interactions: Sequence-dependent and Independent DNA Nethylation.” 2013. Web. 20 Jul 2018.

Vancouver:

Ptak C. Genetic-epigenetic Interactions: Sequence-dependent and Independent DNA Nethylation. [Internet] [Doctoral dissertation]. University of Toronto; 2013. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/69006.

Council of Science Editors:

Ptak C. Genetic-epigenetic Interactions: Sequence-dependent and Independent DNA Nethylation. [Doctoral Dissertation]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/69006


University of Toronto

7. Trepanier, Catherine Helene. Modulation of N-methyl-D-aspartate receptors by Gαs- and Gαi/o-coupled receptors.

Degree: 2012, University of Toronto

The induction of synaptic plasticity at CA1 synapses requires NMDAR activation. Modulation of NMDAR function by various GPCRs can shift the thresholds for LTP and… (more)

Subjects/Keywords: N-methyl-D-aspartate receptors; synaptic plasticity; metaplasticity; G-protein-coupled receptor; schizophrenia; dopamine D1-like receptor; metabotropic glutamate receptors 2 and 3; 0317; 0719; 0419

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APA (6th Edition):

Trepanier, C. H. (2012). Modulation of N-methyl-D-aspartate receptors by Gαs- and Gαi/o-coupled receptors. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/34946

Chicago Manual of Style (16th Edition):

Trepanier, Catherine Helene. “Modulation of N-methyl-D-aspartate receptors by Gαs- and Gαi/o-coupled receptors.” 2012. Doctoral Dissertation, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/34946.

MLA Handbook (7th Edition):

Trepanier, Catherine Helene. “Modulation of N-methyl-D-aspartate receptors by Gαs- and Gαi/o-coupled receptors.” 2012. Web. 20 Jul 2018.

Vancouver:

Trepanier CH. Modulation of N-methyl-D-aspartate receptors by Gαs- and Gαi/o-coupled receptors. [Internet] [Doctoral dissertation]. University of Toronto; 2012. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/34946.

Council of Science Editors:

Trepanier CH. Modulation of N-methyl-D-aspartate receptors by Gαs- and Gαi/o-coupled receptors. [Doctoral Dissertation]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/34946


University of Toronto

8. Creed, Meaghan Claire. Deep Brain Stimulation of the Subthalamic and Entopeduncular Nuclei in an Animal Model of Tardive Dyskinesia.

Degree: 2012, University of Toronto

Deep brain stimulation (DBS) has emerged as a potential intervention for treatment-resistant tardive dyskinesia (TD). Despite promising case reports, no consensus exists regarding optimal stimulation… (more)

Subjects/Keywords: deep brain stimulation; subthalamic; entopeduncular; tardive dyskinesia; haloperidol; serotonin; dopamine; 0419

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APA (6th Edition):

Creed, M. C. (2012). Deep Brain Stimulation of the Subthalamic and Entopeduncular Nuclei in an Animal Model of Tardive Dyskinesia. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/43383

Chicago Manual of Style (16th Edition):

Creed, Meaghan Claire. “Deep Brain Stimulation of the Subthalamic and Entopeduncular Nuclei in an Animal Model of Tardive Dyskinesia.” 2012. Doctoral Dissertation, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/43383.

MLA Handbook (7th Edition):

Creed, Meaghan Claire. “Deep Brain Stimulation of the Subthalamic and Entopeduncular Nuclei in an Animal Model of Tardive Dyskinesia.” 2012. Web. 20 Jul 2018.

Vancouver:

Creed MC. Deep Brain Stimulation of the Subthalamic and Entopeduncular Nuclei in an Animal Model of Tardive Dyskinesia. [Internet] [Doctoral dissertation]. University of Toronto; 2012. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/43383.

Council of Science Editors:

Creed MC. Deep Brain Stimulation of the Subthalamic and Entopeduncular Nuclei in an Animal Model of Tardive Dyskinesia. [Doctoral Dissertation]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/43383


University of Toronto

9. Oh, Gabriel. Epigenomic Studies of Twins and Brain Diseases.

Degree: 2014, University of Toronto

It has been generally accepted that complex diseases are caused by genetic and environmental factors; however, both genetic and epidemiological research programs are facing significant… (more)

Subjects/Keywords: Epigenetics; Major Depressive Disorder; Alzheimer's Disease; Monozygotic Twins; DNA modification; Complex Diseases; 0317

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APA (6th Edition):

Oh, G. (2014). Epigenomic Studies of Twins and Brain Diseases. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/72575

Chicago Manual of Style (16th Edition):

Oh, Gabriel. “Epigenomic Studies of Twins and Brain Diseases.” 2014. Doctoral Dissertation, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/72575.

MLA Handbook (7th Edition):

Oh, Gabriel. “Epigenomic Studies of Twins and Brain Diseases.” 2014. Web. 20 Jul 2018.

Vancouver:

Oh G. Epigenomic Studies of Twins and Brain Diseases. [Internet] [Doctoral dissertation]. University of Toronto; 2014. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/72575.

Council of Science Editors:

Oh G. Epigenomic Studies of Twins and Brain Diseases. [Doctoral Dissertation]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/72575


University of Toronto

10. Miller, Lutfiya. Oxidative Stress Mechanisms in Alcohol Developmental Toxicity.

Degree: 2014, University of Toronto

Consumption of alcohol (ethanol, EtOH) during pregnancy can result in a spectrum of anomalies termed the Fetal Alcohol Spectrum Disorders (FASD), characterized by structural and… (more)

Subjects/Keywords: oxidative stress; fetal alcohol spectrum disorder; teratogenesis; embryology; methanol; ethanol; 0419; 0383; 0758

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APA (6th Edition):

Miller, L. (2014). Oxidative Stress Mechanisms in Alcohol Developmental Toxicity. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/72528

Chicago Manual of Style (16th Edition):

Miller, Lutfiya. “Oxidative Stress Mechanisms in Alcohol Developmental Toxicity.” 2014. Doctoral Dissertation, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/72528.

MLA Handbook (7th Edition):

Miller, Lutfiya. “Oxidative Stress Mechanisms in Alcohol Developmental Toxicity.” 2014. Web. 20 Jul 2018.

Vancouver:

Miller L. Oxidative Stress Mechanisms in Alcohol Developmental Toxicity. [Internet] [Doctoral dissertation]. University of Toronto; 2014. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/72528.

Council of Science Editors:

Miller L. Oxidative Stress Mechanisms in Alcohol Developmental Toxicity. [Doctoral Dissertation]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/72528

11. Zhu, Zixing. The Interaction Between Genetics and Tobacco Consumption in Light Smokers.

Degree: PhD, 2014, University of Toronto

 Smoking is the largest preventable cause of death globally. In North America, although the overall smoking prevalence has been declining, an increasing number of smokers… (more)

Subjects/Keywords: CHRNA5-A3-B4; Cotinine; CYP2A6; Smokeless tobacco; Smoking; 0419

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APA (6th Edition):

Zhu, Z. (2014). The Interaction Between Genetics and Tobacco Consumption in Light Smokers. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/68098

Chicago Manual of Style (16th Edition):

Zhu, Zixing. “The Interaction Between Genetics and Tobacco Consumption in Light Smokers.” 2014. Doctoral Dissertation, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/68098.

MLA Handbook (7th Edition):

Zhu, Zixing. “The Interaction Between Genetics and Tobacco Consumption in Light Smokers.” 2014. Web. 20 Jul 2018.

Vancouver:

Zhu Z. The Interaction Between Genetics and Tobacco Consumption in Light Smokers. [Internet] [Doctoral dissertation]. University of Toronto; 2014. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/68098.

Council of Science Editors:

Zhu Z. The Interaction Between Genetics and Tobacco Consumption in Light Smokers. [Doctoral Dissertation]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/68098


University of Toronto

12. Wu, Alex Man Lai. Regulation of ABCG2 Expression in the Lactating Mammary Gland.

Degree: PhD, 2014, University of Toronto

 The Breast Cancer Resistance Protein (ABCG2) is a multidrug efflux transporter that is upregulated in certain drug-resistant cancer cells and in the mammary gland during… (more)

Subjects/Keywords: ABCG2; BCRP; epigenetics; lactation; prolactin; Stat5; 0419

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APA (6th Edition):

Wu, A. M. L. (2014). Regulation of ABCG2 Expression in the Lactating Mammary Gland. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/68245

Chicago Manual of Style (16th Edition):

Wu, Alex Man Lai. “Regulation of ABCG2 Expression in the Lactating Mammary Gland.” 2014. Doctoral Dissertation, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/68245.

MLA Handbook (7th Edition):

Wu, Alex Man Lai. “Regulation of ABCG2 Expression in the Lactating Mammary Gland.” 2014. Web. 20 Jul 2018.

Vancouver:

Wu AML. Regulation of ABCG2 Expression in the Lactating Mammary Gland. [Internet] [Doctoral dissertation]. University of Toronto; 2014. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/68245.

Council of Science Editors:

Wu AML. Regulation of ABCG2 Expression in the Lactating Mammary Gland. [Doctoral Dissertation]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/68245


University of Toronto

13. Maruf, Abdullah Al. Evaluation of Drug-Induced Liver Injury in an In Vitro Oxidative Stress Inflammation System.

Degree: PhD, 2014, University of Toronto

 Drug-induced liver injury (DILI) is a major concern in clinical studies as well as in post-marketing surveillance of drugs. Previous evidence suggests that drug exposure… (more)

Subjects/Keywords: Drug-induced liver injury; Inflammation; In vitro toxicology; Oxidative stress; 0419

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APA (6th Edition):

Maruf, A. A. (2014). Evaluation of Drug-Induced Liver Injury in an In Vitro Oxidative Stress Inflammation System. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/68249

Chicago Manual of Style (16th Edition):

Maruf, Abdullah Al. “Evaluation of Drug-Induced Liver Injury in an In Vitro Oxidative Stress Inflammation System.” 2014. Doctoral Dissertation, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/68249.

MLA Handbook (7th Edition):

Maruf, Abdullah Al. “Evaluation of Drug-Induced Liver Injury in an In Vitro Oxidative Stress Inflammation System.” 2014. Web. 20 Jul 2018.

Vancouver:

Maruf AA. Evaluation of Drug-Induced Liver Injury in an In Vitro Oxidative Stress Inflammation System. [Internet] [Doctoral dissertation]. University of Toronto; 2014. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/68249.

Council of Science Editors:

Maruf AA. Evaluation of Drug-Induced Liver Injury in an In Vitro Oxidative Stress Inflammation System. [Doctoral Dissertation]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/68249


University of Toronto

14. Jeffrey, Allison Melanie. Progesterone and its Metabolites: Anticonvulsant and Behavioral Studies.

Degree: PhD, 2014, University of Toronto

 AbstractProgesterone is a well-known anticonvulsant neurosteroid. Progesterone's inhibitory effects in the brain have long been attributed to its secondary metabolite, 3-alpha,5-alpha-tetrahydroprogesterone (THP, also known as… (more)

Subjects/Keywords: Behavioral; Electrophysiology; Epilepsy; Kindling; Progesterone; Seizures; 0419

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APA (6th Edition):

Jeffrey, A. M. (2014). Progesterone and its Metabolites: Anticonvulsant and Behavioral Studies. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/68368

Chicago Manual of Style (16th Edition):

Jeffrey, Allison Melanie. “Progesterone and its Metabolites: Anticonvulsant and Behavioral Studies.” 2014. Doctoral Dissertation, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/68368.

MLA Handbook (7th Edition):

Jeffrey, Allison Melanie. “Progesterone and its Metabolites: Anticonvulsant and Behavioral Studies.” 2014. Web. 20 Jul 2018.

Vancouver:

Jeffrey AM. Progesterone and its Metabolites: Anticonvulsant and Behavioral Studies. [Internet] [Doctoral dissertation]. University of Toronto; 2014. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/68368.

Council of Science Editors:

Jeffrey AM. Progesterone and its Metabolites: Anticonvulsant and Behavioral Studies. [Doctoral Dissertation]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/68368


University of Toronto

15. Lam, Jessica Fung Chi. Risk Factors Associated With Opioid-Induced Toxicity: Ontogeny, Pharmacogenetics, and Drug Interactions.

Degree: PhD, 2014, University of Toronto

 Opioids are an important class of drugs used for the treatment of pain. The analgesic response and opioid-induced toxicity vary widely among patients. Of serious… (more)

Subjects/Keywords: Drug Interaction; Lactation; Neonate; Opioid; P-glycoprotein; Pharmacogenetics; 0419

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APA (6th Edition):

Lam, J. F. C. (2014). Risk Factors Associated With Opioid-Induced Toxicity: Ontogeny, Pharmacogenetics, and Drug Interactions. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/68430

Chicago Manual of Style (16th Edition):

Lam, Jessica Fung Chi. “Risk Factors Associated With Opioid-Induced Toxicity: Ontogeny, Pharmacogenetics, and Drug Interactions.” 2014. Doctoral Dissertation, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/68430.

MLA Handbook (7th Edition):

Lam, Jessica Fung Chi. “Risk Factors Associated With Opioid-Induced Toxicity: Ontogeny, Pharmacogenetics, and Drug Interactions.” 2014. Web. 20 Jul 2018.

Vancouver:

Lam JFC. Risk Factors Associated With Opioid-Induced Toxicity: Ontogeny, Pharmacogenetics, and Drug Interactions. [Internet] [Doctoral dissertation]. University of Toronto; 2014. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/68430.

Council of Science Editors:

Lam JFC. Risk Factors Associated With Opioid-Induced Toxicity: Ontogeny, Pharmacogenetics, and Drug Interactions. [Doctoral Dissertation]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/68430


University of Toronto

16. Wang, Shuang. Mechanisms of 4-aminobiphenyl-induced Liver Carcinogenesis in the Mouse.

Degree: PhD, 2014, University of Toronto

 4-Aminobiphenyl (ABP) is a human carcinogen commonly found in cigarette smoke and hair dyes. In a tumor study carried out previously in our laboratory using… (more)

Subjects/Keywords: aromatic amine; drug metabolism; liver cancer; mouse; mutations; oxidative stress; 0419

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APA (6th Edition):

Wang, S. (2014). Mechanisms of 4-aminobiphenyl-induced Liver Carcinogenesis in the Mouse. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/68448

Chicago Manual of Style (16th Edition):

Wang, Shuang. “Mechanisms of 4-aminobiphenyl-induced Liver Carcinogenesis in the Mouse.” 2014. Doctoral Dissertation, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/68448.

MLA Handbook (7th Edition):

Wang, Shuang. “Mechanisms of 4-aminobiphenyl-induced Liver Carcinogenesis in the Mouse.” 2014. Web. 20 Jul 2018.

Vancouver:

Wang S. Mechanisms of 4-aminobiphenyl-induced Liver Carcinogenesis in the Mouse. [Internet] [Doctoral dissertation]. University of Toronto; 2014. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/68448.

Council of Science Editors:

Wang S. Mechanisms of 4-aminobiphenyl-induced Liver Carcinogenesis in the Mouse. [Doctoral Dissertation]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/68448

17. Wassenaar, Catherine Anne. Variation in Nicotine and Nitrosamine Pharmacology Genes and Smoking-associated Lung Cancer.

Degree: PhD, 2015, University of Toronto

 Lung cancer is the leading cause of death from cancer in North America. While the vast majority of lung cancer cases are attributable to cigarette… (more)

Subjects/Keywords: 0419

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APA (6th Edition):

Wassenaar, C. A. (2015). Variation in Nicotine and Nitrosamine Pharmacology Genes and Smoking-associated Lung Cancer. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/69050

Chicago Manual of Style (16th Edition):

Wassenaar, Catherine Anne. “Variation in Nicotine and Nitrosamine Pharmacology Genes and Smoking-associated Lung Cancer.” 2015. Doctoral Dissertation, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/69050.

MLA Handbook (7th Edition):

Wassenaar, Catherine Anne. “Variation in Nicotine and Nitrosamine Pharmacology Genes and Smoking-associated Lung Cancer.” 2015. Web. 20 Jul 2018.

Vancouver:

Wassenaar CA. Variation in Nicotine and Nitrosamine Pharmacology Genes and Smoking-associated Lung Cancer. [Internet] [Doctoral dissertation]. University of Toronto; 2015. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/69050.

Council of Science Editors:

Wassenaar CA. Variation in Nicotine and Nitrosamine Pharmacology Genes and Smoking-associated Lung Cancer. [Doctoral Dissertation]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/69050


University of Toronto

18. Delano, Kaitlyn E. Utilizing Biomarkers to Assess Prevalence and Trends of Substance Use During Pregnancy in Canada.

Degree: PhD, 2015, University of Toronto

 Substance use during pregnancy is associated with numerous risks to both mother and fetus. Studies of the prevalence and trends of substance use during pregnancy… (more)

Subjects/Keywords: Alcohol; Biomarkers; Epidemiology; Illicit Drugs; Pregnancy; Substance Use; 0419

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APA (6th Edition):

Delano, K. E. (2015). Utilizing Biomarkers to Assess Prevalence and Trends of Substance Use During Pregnancy in Canada. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/70928

Chicago Manual of Style (16th Edition):

Delano, Kaitlyn E. “Utilizing Biomarkers to Assess Prevalence and Trends of Substance Use During Pregnancy in Canada.” 2015. Doctoral Dissertation, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/70928.

MLA Handbook (7th Edition):

Delano, Kaitlyn E. “Utilizing Biomarkers to Assess Prevalence and Trends of Substance Use During Pregnancy in Canada.” 2015. Web. 20 Jul 2018.

Vancouver:

Delano KE. Utilizing Biomarkers to Assess Prevalence and Trends of Substance Use During Pregnancy in Canada. [Internet] [Doctoral dissertation]. University of Toronto; 2015. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/70928.

Council of Science Editors:

Delano KE. Utilizing Biomarkers to Assess Prevalence and Trends of Substance Use During Pregnancy in Canada. [Doctoral Dissertation]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/70928


University of Toronto

19. Poon, Shirley. Hair as a Matrix for Assessing Polybrominated Diphenyl Ether (PBDE) Exposure in Animals and Humans.

Degree: PhD, 2015, University of Toronto

 Polybrominated diphenyl ethers (PBDEs) are synthetic chemicals added to various consumer products as flame retardants. They persist in the environment and their potential endocrine disrupting… (more)

Subjects/Keywords: Flame retardant; GCMS; Hair; Hypospadias; PBDE; 0419

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APA (6th Edition):

Poon, S. (2015). Hair as a Matrix for Assessing Polybrominated Diphenyl Ether (PBDE) Exposure in Animals and Humans. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/71575

Chicago Manual of Style (16th Edition):

Poon, Shirley. “Hair as a Matrix for Assessing Polybrominated Diphenyl Ether (PBDE) Exposure in Animals and Humans.” 2015. Doctoral Dissertation, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/71575.

MLA Handbook (7th Edition):

Poon, Shirley. “Hair as a Matrix for Assessing Polybrominated Diphenyl Ether (PBDE) Exposure in Animals and Humans.” 2015. Web. 20 Jul 2018.

Vancouver:

Poon S. Hair as a Matrix for Assessing Polybrominated Diphenyl Ether (PBDE) Exposure in Animals and Humans. [Internet] [Doctoral dissertation]. University of Toronto; 2015. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/71575.

Council of Science Editors:

Poon S. Hair as a Matrix for Assessing Polybrominated Diphenyl Ether (PBDE) Exposure in Animals and Humans. [Doctoral Dissertation]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/71575


University of Toronto

20. Pal, Mrinal. The HLA Complex Group 9 Gene Modification Study in Major Psychosis.

Degree: PhD, 2015, University of Toronto

 Epigenetic mechanisms can offer new insights into the non-Mendelian features of complex psychiatric disorders. The first epigenome-wide scan in major psychosis, conducted by our group,… (more)

Subjects/Keywords: bipolar disorder; bisulfite sequencing; DNA modification; epigenetics; micorarray; schizophrenia; 0317

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APA (6th Edition):

Pal, M. (2015). The HLA Complex Group 9 Gene Modification Study in Major Psychosis. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/71602

Chicago Manual of Style (16th Edition):

Pal, Mrinal. “The HLA Complex Group 9 Gene Modification Study in Major Psychosis.” 2015. Doctoral Dissertation, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/71602.

MLA Handbook (7th Edition):

Pal, Mrinal. “The HLA Complex Group 9 Gene Modification Study in Major Psychosis.” 2015. Web. 20 Jul 2018.

Vancouver:

Pal M. The HLA Complex Group 9 Gene Modification Study in Major Psychosis. [Internet] [Doctoral dissertation]. University of Toronto; 2015. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/71602.

Council of Science Editors:

Pal M. The HLA Complex Group 9 Gene Modification Study in Major Psychosis. [Doctoral Dissertation]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/71602


University of Toronto

21. Chenoweth, Meghan Jo-Ann. Sources of Variation in Nicotine Metabolism and Associations with Smoking Abstinence in Adolescents and Adults.

Degree: PhD, 2016, University of Toronto

 Smoking remains a major public health concern; worldwide, approximately one billion people smoke. Despite the fact that many smokers are motivated to quit, only a… (more)

Subjects/Keywords: 0419

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APA (6th Edition):

Chenoweth, M. J. (2016). Sources of Variation in Nicotine Metabolism and Associations with Smoking Abstinence in Adolescents and Adults. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/72924

Chicago Manual of Style (16th Edition):

Chenoweth, Meghan Jo-Ann. “Sources of Variation in Nicotine Metabolism and Associations with Smoking Abstinence in Adolescents and Adults.” 2016. Doctoral Dissertation, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/72924.

MLA Handbook (7th Edition):

Chenoweth, Meghan Jo-Ann. “Sources of Variation in Nicotine Metabolism and Associations with Smoking Abstinence in Adolescents and Adults.” 2016. Web. 20 Jul 2018.

Vancouver:

Chenoweth MJ. Sources of Variation in Nicotine Metabolism and Associations with Smoking Abstinence in Adolescents and Adults. [Internet] [Doctoral dissertation]. University of Toronto; 2016. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/72924.

Council of Science Editors:

Chenoweth MJ. Sources of Variation in Nicotine Metabolism and Associations with Smoking Abstinence in Adolescents and Adults. [Doctoral Dissertation]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/72924

22. Garcia, Kristine. The Effect of Altering Brain CYP2B Activity on Nicotine Self-administration Behaviour and Nicotine Levels in the Brain.

Degree: PhD, 2016, University of Toronto

 Cytochrome P450 (CYP) enzymes play an important role in drug metabolism. While CYPs are abundantly expressed in the liver, where CYP-mediated drug metabolism typically occurs,… (more)

Subjects/Keywords: 0419

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APA (6th Edition):

Garcia, K. (2016). The Effect of Altering Brain CYP2B Activity on Nicotine Self-administration Behaviour and Nicotine Levels in the Brain. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/72988

Chicago Manual of Style (16th Edition):

Garcia, Kristine. “The Effect of Altering Brain CYP2B Activity on Nicotine Self-administration Behaviour and Nicotine Levels in the Brain.” 2016. Doctoral Dissertation, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/72988.

MLA Handbook (7th Edition):

Garcia, Kristine. “The Effect of Altering Brain CYP2B Activity on Nicotine Self-administration Behaviour and Nicotine Levels in the Brain.” 2016. Web. 20 Jul 2018.

Vancouver:

Garcia K. The Effect of Altering Brain CYP2B Activity on Nicotine Self-administration Behaviour and Nicotine Levels in the Brain. [Internet] [Doctoral dissertation]. University of Toronto; 2016. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/72988.

Council of Science Editors:

Garcia K. The Effect of Altering Brain CYP2B Activity on Nicotine Self-administration Behaviour and Nicotine Levels in the Brain. [Doctoral Dissertation]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/72988


University of Toronto

23. Bapat, Priya. Placental Pharmacology â Implications for Therapy in Pregnancy.

Degree: PhD, 2016, University of Toronto

 Medication use during pregnancy requires a balance between treating a condition in the mother, and minimizing potential risks in the fetus. In recent years, it… (more)

Subjects/Keywords: anticoagulants; drug safety; placenta; pregnancy; 0419

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APA (6th Edition):

Bapat, P. (2016). Placental Pharmacology â Implications for Therapy in Pregnancy. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/76299

Chicago Manual of Style (16th Edition):

Bapat, Priya. “Placental Pharmacology â Implications for Therapy in Pregnancy.” 2016. Doctoral Dissertation, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/76299.

MLA Handbook (7th Edition):

Bapat, Priya. “Placental Pharmacology â Implications for Therapy in Pregnancy.” 2016. Web. 20 Jul 2018.

Vancouver:

Bapat P. Placental Pharmacology â Implications for Therapy in Pregnancy. [Internet] [Doctoral dissertation]. University of Toronto; 2016. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/76299.

Council of Science Editors:

Bapat P. Placental Pharmacology â Implications for Therapy in Pregnancy. [Doctoral Dissertation]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/76299


University of Toronto

24. Nona, Christina. Glutamatergic mechanisms in behavioural sensitization to ethanol.

Degree: PhD, 2016, University of Toronto

 Repeated exposure to ethanol (EtOH) in mice produces behavioural sensitization. However, not all mice sensitize to EtOH and in any cohort high-sensitizers (HS) can readily… (more)

Subjects/Keywords: Addiction; Ethanol; Glutamate; Locomotor activity; NMDA receptor; Sensitization; 0317

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APA (6th Edition):

Nona, C. (2016). Glutamatergic mechanisms in behavioural sensitization to ethanol. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/76581

Chicago Manual of Style (16th Edition):

Nona, Christina. “Glutamatergic mechanisms in behavioural sensitization to ethanol.” 2016. Doctoral Dissertation, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/76581.

MLA Handbook (7th Edition):

Nona, Christina. “Glutamatergic mechanisms in behavioural sensitization to ethanol.” 2016. Web. 20 Jul 2018.

Vancouver:

Nona C. Glutamatergic mechanisms in behavioural sensitization to ethanol. [Internet] [Doctoral dissertation]. University of Toronto; 2016. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/76581.

Council of Science Editors:

Nona C. Glutamatergic mechanisms in behavioural sensitization to ethanol. [Doctoral Dissertation]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/76581


University of Toronto

25. Pushparaj, Abhiram Pierre. Examining the Role of the Insular Cortex in Addiction-relevant Behaviours.

Degree: PhD, 2016, University of Toronto

 INTRODUCTION: Recently recognized as a critical brain region underlying the neurocircuitry of addiction, the body of evidence supporting the role of the insular cortex across… (more)

Subjects/Keywords: 0317

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APA (6th Edition):

Pushparaj, A. P. (2016). Examining the Role of the Insular Cortex in Addiction-relevant Behaviours. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/76797

Chicago Manual of Style (16th Edition):

Pushparaj, Abhiram Pierre. “Examining the Role of the Insular Cortex in Addiction-relevant Behaviours.” 2016. Doctoral Dissertation, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/76797.

MLA Handbook (7th Edition):

Pushparaj, Abhiram Pierre. “Examining the Role of the Insular Cortex in Addiction-relevant Behaviours.” 2016. Web. 20 Jul 2018.

Vancouver:

Pushparaj AP. Examining the Role of the Insular Cortex in Addiction-relevant Behaviours. [Internet] [Doctoral dissertation]. University of Toronto; 2016. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/76797.

Council of Science Editors:

Pushparaj AP. Examining the Role of the Insular Cortex in Addiction-relevant Behaviours. [Doctoral Dissertation]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/76797


University of Toronto

26. Shram, Megan Joyce. Age Differences in the Vulnerability to Nicotine Addiction: Evidence from a Rat Model of Adolescent Nicotine Taking.

Degree: 2008, University of Toronto

Rationale: Peak initiation of smoking occurs during adolescence and early onset of smoking is associated with a reduced probability of quitting and greater risk of… (more)

Subjects/Keywords: adolescent; nicotine; rat; reinforcement; reward; self-administration; withdrawal; c-fos mRNA; acquisition of drug taking; maintenance of drug taking; motivation; mecamylamine; behaviour; extinction; reinstatement; conditioned place preference; conditioned taste avoidance; 0419

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APA (6th Edition):

Shram, M. J. (2008). Age Differences in the Vulnerability to Nicotine Addiction: Evidence from a Rat Model of Adolescent Nicotine Taking. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/11260

Chicago Manual of Style (16th Edition):

Shram, Megan Joyce. “Age Differences in the Vulnerability to Nicotine Addiction: Evidence from a Rat Model of Adolescent Nicotine Taking.” 2008. Doctoral Dissertation, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/11260.

MLA Handbook (7th Edition):

Shram, Megan Joyce. “Age Differences in the Vulnerability to Nicotine Addiction: Evidence from a Rat Model of Adolescent Nicotine Taking.” 2008. Web. 20 Jul 2018.

Vancouver:

Shram MJ. Age Differences in the Vulnerability to Nicotine Addiction: Evidence from a Rat Model of Adolescent Nicotine Taking. [Internet] [Doctoral dissertation]. University of Toronto; 2008. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/11260.

Council of Science Editors:

Shram MJ. Age Differences in the Vulnerability to Nicotine Addiction: Evidence from a Rat Model of Adolescent Nicotine Taking. [Doctoral Dissertation]. University of Toronto; 2008. Available from: http://hdl.handle.net/1807/11260


University of Toronto

27. Moffat, Ivy D. Mechanisms of Genetic Resistance To Dioxin-induced Lethality.

Degree: 2008, University of Toronto

Dioxins are environmental contaminants that raise concern because they are potent and persistent. The most potent dioxin congener, 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD), causes a wide variety of… (more)

Subjects/Keywords: Dioxin; aryl hydrocarbon receptor; expression array; 0383

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APA (6th Edition):

Moffat, I. D. (2008). Mechanisms of Genetic Resistance To Dioxin-induced Lethality. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/11119

Chicago Manual of Style (16th Edition):

Moffat, Ivy D. “Mechanisms of Genetic Resistance To Dioxin-induced Lethality.” 2008. Doctoral Dissertation, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/11119.

MLA Handbook (7th Edition):

Moffat, Ivy D. “Mechanisms of Genetic Resistance To Dioxin-induced Lethality.” 2008. Web. 20 Jul 2018.

Vancouver:

Moffat ID. Mechanisms of Genetic Resistance To Dioxin-induced Lethality. [Internet] [Doctoral dissertation]. University of Toronto; 2008. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/11119.

Council of Science Editors:

Moffat ID. Mechanisms of Genetic Resistance To Dioxin-induced Lethality. [Doctoral Dissertation]. University of Toronto; 2008. Available from: http://hdl.handle.net/1807/11119


University of Toronto

28. Novak, Gabriela. Upregulation of CaMKIIβ and Nogo-C mRNA in Schizophrenia and the Prevalence of CAA Insert in the 3’UTR of the Nogo Gene.

Degree: 2008, University of Toronto

Schizophrenia may result from altered gene expression leading to abnormal neurodevelopment. In a search for genes with altered expression in schizophrenia, cDNA library subtractive hybridization… (more)

Subjects/Keywords: CaMKII; schizophrenia; Nogo; RTN4; human; frontal cortex; gene variant; gene expression; 0419

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APA (6th Edition):

Novak, G. (2008). Upregulation of CaMKIIβ and Nogo-C mRNA in Schizophrenia and the Prevalence of CAA Insert in the 3’UTR of the Nogo Gene. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/11240

Chicago Manual of Style (16th Edition):

Novak, Gabriela. “Upregulation of CaMKIIβ and Nogo-C mRNA in Schizophrenia and the Prevalence of CAA Insert in the 3’UTR of the Nogo Gene.” 2008. Doctoral Dissertation, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/11240.

MLA Handbook (7th Edition):

Novak, Gabriela. “Upregulation of CaMKIIβ and Nogo-C mRNA in Schizophrenia and the Prevalence of CAA Insert in the 3’UTR of the Nogo Gene.” 2008. Web. 20 Jul 2018.

Vancouver:

Novak G. Upregulation of CaMKIIβ and Nogo-C mRNA in Schizophrenia and the Prevalence of CAA Insert in the 3’UTR of the Nogo Gene. [Internet] [Doctoral dissertation]. University of Toronto; 2008. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/11240.

Council of Science Editors:

Novak G. Upregulation of CaMKIIβ and Nogo-C mRNA in Schizophrenia and the Prevalence of CAA Insert in the 3’UTR of the Nogo Gene. [Doctoral Dissertation]. University of Toronto; 2008. Available from: http://hdl.handle.net/1807/11240


University of Toronto

29. Tan, Kah Poh. Nuclear Factor (Erythroid 2-like) Factor 2 (Nrf2) as Cellular Protector in Bile Acid and Retinoid Toxicities.

Degree: 2008, University of Toronto

Exposure to toxic bile acids (BA) and retinoic acids (RA) is implicated in toxicities related to excessive oxidative stress. This thesis examined roles and mechanisms… (more)

Subjects/Keywords: oxidative stress; glutathione; bile acid; Nrf2; retinoic acid; cholestasis; ATP-binding cassette transporters; mitogen activated protein kinase; multidrug resistance associated proteins; adaptive response; antioxidants; thioredoxin reductase; 4-hydroxynonenal; lipid peroxidation; glutathione S-transferase; liver toxicity; antioxidant response element; cell defense; 0383

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APA (6th Edition):

Tan, K. P. (2008). Nuclear Factor (Erythroid 2-like) Factor 2 (Nrf2) as Cellular Protector in Bile Acid and Retinoid Toxicities. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/17287

Chicago Manual of Style (16th Edition):

Tan, Kah Poh. “Nuclear Factor (Erythroid 2-like) Factor 2 (Nrf2) as Cellular Protector in Bile Acid and Retinoid Toxicities.” 2008. Doctoral Dissertation, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/17287.

MLA Handbook (7th Edition):

Tan, Kah Poh. “Nuclear Factor (Erythroid 2-like) Factor 2 (Nrf2) as Cellular Protector in Bile Acid and Retinoid Toxicities.” 2008. Web. 20 Jul 2018.

Vancouver:

Tan KP. Nuclear Factor (Erythroid 2-like) Factor 2 (Nrf2) as Cellular Protector in Bile Acid and Retinoid Toxicities. [Internet] [Doctoral dissertation]. University of Toronto; 2008. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/17287.

Council of Science Editors:

Tan KP. Nuclear Factor (Erythroid 2-like) Factor 2 (Nrf2) as Cellular Protector in Bile Acid and Retinoid Toxicities. [Doctoral Dissertation]. University of Toronto; 2008. Available from: http://hdl.handle.net/1807/17287


University of Toronto

30. Gill, Simerpal. Investigating Sources of Variability in Pharmacological Response to Nausea and Vomiting of Pregnancy.

Degree: 2010, University of Toronto

Nausea and vomiting of pregnancy (NVP) is the most common medical condition in pregnancy, and, unfortunately, variability exists among pregnant women in the therapeutic effect… (more)

Subjects/Keywords: nausea and vomiting of pregnancy; pregnancy; 0419; 0383

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APA (6th Edition):

Gill, S. (2010). Investigating Sources of Variability in Pharmacological Response to Nausea and Vomiting of Pregnancy. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/24348

Chicago Manual of Style (16th Edition):

Gill, Simerpal. “Investigating Sources of Variability in Pharmacological Response to Nausea and Vomiting of Pregnancy.” 2010. Doctoral Dissertation, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/24348.

MLA Handbook (7th Edition):

Gill, Simerpal. “Investigating Sources of Variability in Pharmacological Response to Nausea and Vomiting of Pregnancy.” 2010. Web. 20 Jul 2018.

Vancouver:

Gill S. Investigating Sources of Variability in Pharmacological Response to Nausea and Vomiting of Pregnancy. [Internet] [Doctoral dissertation]. University of Toronto; 2010. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/24348.

Council of Science Editors:

Gill S. Investigating Sources of Variability in Pharmacological Response to Nausea and Vomiting of Pregnancy. [Doctoral Dissertation]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/24348

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