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You searched for +publisher:"University of Toronto" +contributor:("Molecular and Medical Genetics"). Showing records 1 – 30 of 179 total matches.

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University of Toronto

1. Jiao, Wei. Machine Learning for Variant Detection and Population Analysis in Heterogenerous Cancer Sample.

Degree: 2013, University of Toronto

Cancer is a complex and deadly disease that is caused by genetic lesions in somatic cells. Further research in computational methodology for detecting and characterizing… (more)

Subjects/Keywords: Single nucleotide variant; Machine learning; Cancer heterogeneity; 0715

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APA (6th Edition):

Jiao, W. (2013). Machine Learning for Variant Detection and Population Analysis in Heterogenerous Cancer Sample. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/42971

Chicago Manual of Style (16th Edition):

Jiao, Wei. “Machine Learning for Variant Detection and Population Analysis in Heterogenerous Cancer Sample.” 2013. Masters Thesis, University of Toronto. Accessed July 18, 2019. http://hdl.handle.net/1807/42971.

MLA Handbook (7th Edition):

Jiao, Wei. “Machine Learning for Variant Detection and Population Analysis in Heterogenerous Cancer Sample.” 2013. Web. 18 Jul 2019.

Vancouver:

Jiao W. Machine Learning for Variant Detection and Population Analysis in Heterogenerous Cancer Sample. [Internet] [Masters thesis]. University of Toronto; 2013. [cited 2019 Jul 18]. Available from: http://hdl.handle.net/1807/42971.

Council of Science Editors:

Jiao W. Machine Learning for Variant Detection and Population Analysis in Heterogenerous Cancer Sample. [Masters Thesis]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/42971


University of Toronto

2. Sekikawa, Akiko. Coordinated Post-transcriptional Regulation by MicroRNAs and RNA- binding Proteins.

Degree: 2013, University of Toronto

Both microRNAs (miRNAs) and RNA-binding proteins (RBPs) regulate post- transcriptional events, but the post-transcriptional contribution to the global mammalian transcriptomes is still not well understood.… (more)

Subjects/Keywords: microRNA; HuR; RNA-binding protein; evolution; post-transcriptional regulation; RNA-seq; normalization; 0369; 0715

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APA (6th Edition):

Sekikawa, A. (2013). Coordinated Post-transcriptional Regulation by MicroRNAs and RNA- binding Proteins. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/42912

Chicago Manual of Style (16th Edition):

Sekikawa, Akiko. “Coordinated Post-transcriptional Regulation by MicroRNAs and RNA- binding Proteins.” 2013. Masters Thesis, University of Toronto. Accessed July 18, 2019. http://hdl.handle.net/1807/42912.

MLA Handbook (7th Edition):

Sekikawa, Akiko. “Coordinated Post-transcriptional Regulation by MicroRNAs and RNA- binding Proteins.” 2013. Web. 18 Jul 2019.

Vancouver:

Sekikawa A. Coordinated Post-transcriptional Regulation by MicroRNAs and RNA- binding Proteins. [Internet] [Masters thesis]. University of Toronto; 2013. [cited 2019 Jul 18]. Available from: http://hdl.handle.net/1807/42912.

Council of Science Editors:

Sekikawa A. Coordinated Post-transcriptional Regulation by MicroRNAs and RNA- binding Proteins. [Masters Thesis]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/42912


University of Toronto

3. Lau, Esther. Generation of Human Synthetic Antibodies to Investigate the Biological Functions of the Receptor Tyrosine Kinases HER3 and PTK7.

Degree: 2013, University of Toronto

Receptor tyrosine kinases or RTKs are an important class of signaling proteins that are frequently deregulated in cancer and other diseases. Based on a series… (more)

Subjects/Keywords: HER3; PTK7; synthetic antibodies; cancer

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APA (6th Edition):

Lau, E. (2013). Generation of Human Synthetic Antibodies to Investigate the Biological Functions of the Receptor Tyrosine Kinases HER3 and PTK7. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/70019

Chicago Manual of Style (16th Edition):

Lau, Esther. “Generation of Human Synthetic Antibodies to Investigate the Biological Functions of the Receptor Tyrosine Kinases HER3 and PTK7.” 2013. Masters Thesis, University of Toronto. Accessed July 18, 2019. http://hdl.handle.net/1807/70019.

MLA Handbook (7th Edition):

Lau, Esther. “Generation of Human Synthetic Antibodies to Investigate the Biological Functions of the Receptor Tyrosine Kinases HER3 and PTK7.” 2013. Web. 18 Jul 2019.

Vancouver:

Lau E. Generation of Human Synthetic Antibodies to Investigate the Biological Functions of the Receptor Tyrosine Kinases HER3 and PTK7. [Internet] [Masters thesis]. University of Toronto; 2013. [cited 2019 Jul 18]. Available from: http://hdl.handle.net/1807/70019.

Council of Science Editors:

Lau E. Generation of Human Synthetic Antibodies to Investigate the Biological Functions of the Receptor Tyrosine Kinases HER3 and PTK7. [Masters Thesis]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/70019


University of Toronto

4. Yuan, Fang. Characterization of the Interactome of the Hippo Tumour Suppressor Pathway using Mass Spectrometry.

Degree: 2013, University of Toronto

The Hippo signaling pathway offers an intrinsic mechanism to control organ sizes, and dysfunction of this pathway can often lead to cancer. Great advancement has… (more)

Subjects/Keywords: hippo pathway; mass spectrometry; 0307

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APA (6th Edition):

Yuan, F. (2013). Characterization of the Interactome of the Hippo Tumour Suppressor Pathway using Mass Spectrometry. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/43372

Chicago Manual of Style (16th Edition):

Yuan, Fang. “Characterization of the Interactome of the Hippo Tumour Suppressor Pathway using Mass Spectrometry.” 2013. Masters Thesis, University of Toronto. Accessed July 18, 2019. http://hdl.handle.net/1807/43372.

MLA Handbook (7th Edition):

Yuan, Fang. “Characterization of the Interactome of the Hippo Tumour Suppressor Pathway using Mass Spectrometry.” 2013. Web. 18 Jul 2019.

Vancouver:

Yuan F. Characterization of the Interactome of the Hippo Tumour Suppressor Pathway using Mass Spectrometry. [Internet] [Masters thesis]. University of Toronto; 2013. [cited 2019 Jul 18]. Available from: http://hdl.handle.net/1807/43372.

Council of Science Editors:

Yuan F. Characterization of the Interactome of the Hippo Tumour Suppressor Pathway using Mass Spectrometry. [Masters Thesis]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/43372


University of Toronto

5. Brimble, Nicole Fabris Elise. Investigating the Function of GTF2I and its Contribution to the Williams-Beuren Syndrome Neurological Profile.

Degree: 2014, University of Toronto

Williams-Beuren Syndrome (WBS) is a genetic neurodevelopmental disorder caused by the deletion of 25 protein-coding genes at chromosome 7q11.23. It is associated with a highly… (more)

Subjects/Keywords: 7q11.23; GTF2I; iPSC; neurogenesis; WBS; Williams; 0369

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APA (6th Edition):

Brimble, N. F. E. (2014). Investigating the Function of GTF2I and its Contribution to the Williams-Beuren Syndrome Neurological Profile. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/67620

Chicago Manual of Style (16th Edition):

Brimble, Nicole Fabris Elise. “Investigating the Function of GTF2I and its Contribution to the Williams-Beuren Syndrome Neurological Profile.” 2014. Masters Thesis, University of Toronto. Accessed July 18, 2019. http://hdl.handle.net/1807/67620.

MLA Handbook (7th Edition):

Brimble, Nicole Fabris Elise. “Investigating the Function of GTF2I and its Contribution to the Williams-Beuren Syndrome Neurological Profile.” 2014. Web. 18 Jul 2019.

Vancouver:

Brimble NFE. Investigating the Function of GTF2I and its Contribution to the Williams-Beuren Syndrome Neurological Profile. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2019 Jul 18]. Available from: http://hdl.handle.net/1807/67620.

Council of Science Editors:

Brimble NFE. Investigating the Function of GTF2I and its Contribution to the Williams-Beuren Syndrome Neurological Profile. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/67620


University of Toronto

6. Ma, Xiangyuan. Identification of Genes and Pathways Linking Cancer Metabolism to Cell Surface Dynamics through Protein N-glycosylation.

Degree: 2014, University of Toronto

N-glycosylation is a co-translational modification that covalently attaches oligosaccharide to and regulates proper folding, trafficking, and surface residency of secreted proteins. MGAT5 encodes a glycosyltransferase… (more)

Subjects/Keywords: Cell Signaling; Metabolism; Mgat5; N-glycosylation; Pooled Lentiviral shRNA Drop-out Screen; 0307

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APA (6th Edition):

Ma, X. (2014). Identification of Genes and Pathways Linking Cancer Metabolism to Cell Surface Dynamics through Protein N-glycosylation. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/67885

Chicago Manual of Style (16th Edition):

Ma, Xiangyuan. “Identification of Genes and Pathways Linking Cancer Metabolism to Cell Surface Dynamics through Protein N-glycosylation.” 2014. Masters Thesis, University of Toronto. Accessed July 18, 2019. http://hdl.handle.net/1807/67885.

MLA Handbook (7th Edition):

Ma, Xiangyuan. “Identification of Genes and Pathways Linking Cancer Metabolism to Cell Surface Dynamics through Protein N-glycosylation.” 2014. Web. 18 Jul 2019.

Vancouver:

Ma X. Identification of Genes and Pathways Linking Cancer Metabolism to Cell Surface Dynamics through Protein N-glycosylation. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2019 Jul 18]. Available from: http://hdl.handle.net/1807/67885.

Council of Science Editors:

Ma X. Identification of Genes and Pathways Linking Cancer Metabolism to Cell Surface Dynamics through Protein N-glycosylation. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/67885


University of Toronto

7. Wang, Miling. Phosphorylation of Neuralized at Serine 94 and 96 is Required for Notch Signaling during Drosophila Development.

Degree: 2014, University of Toronto

The Notch (N) signaling pathway defines one of the most fundamental signaling pathways that govern metazoan development. Neuralized (Neur) and Mindbomb (Mib), E3 ubiquitin ligases,… (more)

Subjects/Keywords: Delta endocytosis; Drosophila; lateral inhibition; Neuralized; Phosphorylation; third instar larvae; 0307

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APA (6th Edition):

Wang, M. (2014). Phosphorylation of Neuralized at Serine 94 and 96 is Required for Notch Signaling during Drosophila Development. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/67913

Chicago Manual of Style (16th Edition):

Wang, Miling. “Phosphorylation of Neuralized at Serine 94 and 96 is Required for Notch Signaling during Drosophila Development.” 2014. Masters Thesis, University of Toronto. Accessed July 18, 2019. http://hdl.handle.net/1807/67913.

MLA Handbook (7th Edition):

Wang, Miling. “Phosphorylation of Neuralized at Serine 94 and 96 is Required for Notch Signaling during Drosophila Development.” 2014. Web. 18 Jul 2019.

Vancouver:

Wang M. Phosphorylation of Neuralized at Serine 94 and 96 is Required for Notch Signaling during Drosophila Development. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2019 Jul 18]. Available from: http://hdl.handle.net/1807/67913.

Council of Science Editors:

Wang M. Phosphorylation of Neuralized at Serine 94 and 96 is Required for Notch Signaling during Drosophila Development. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/67913


University of Toronto

8. Ehman, Dylan Alexander. Investigating Differential Expression of MicroRNAs Related to Recurrence in the Luminal Breast Cancer Subtype.

Degree: 2014, University of Toronto

Breast cancer is a heterogeneous disease, and patients with the luminal subtype have a range of outcomes unable to be predicted by current clinical methods.… (more)

Subjects/Keywords: breast; expression; luminal; microRNA; prognosis; recurrence; 0369

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APA (6th Edition):

Ehman, D. A. (2014). Investigating Differential Expression of MicroRNAs Related to Recurrence in the Luminal Breast Cancer Subtype. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/67924

Chicago Manual of Style (16th Edition):

Ehman, Dylan Alexander. “Investigating Differential Expression of MicroRNAs Related to Recurrence in the Luminal Breast Cancer Subtype.” 2014. Masters Thesis, University of Toronto. Accessed July 18, 2019. http://hdl.handle.net/1807/67924.

MLA Handbook (7th Edition):

Ehman, Dylan Alexander. “Investigating Differential Expression of MicroRNAs Related to Recurrence in the Luminal Breast Cancer Subtype.” 2014. Web. 18 Jul 2019.

Vancouver:

Ehman DA. Investigating Differential Expression of MicroRNAs Related to Recurrence in the Luminal Breast Cancer Subtype. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2019 Jul 18]. Available from: http://hdl.handle.net/1807/67924.

Council of Science Editors:

Ehman DA. Investigating Differential Expression of MicroRNAs Related to Recurrence in the Luminal Breast Cancer Subtype. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/67924


University of Toronto

9. Lai, Wesley. MECP2e1 Mutation Reduces the Dendritic Complexity of Rett Syndrome Patient iPS Cell-derived Neurons.

Degree: 2014, University of Toronto

The gene MECP2 that is commonly mutated in Rett Syndrome (RTT) can be alternatively spliced in neurons into MeCP2e1 and MECP2e2 isoforms. Fibroblasts from a… (more)

Subjects/Keywords: dendrite complexity; induced pluripotent stem cells; MECP2e1; Ngn2; Rett Syndrome; 0369

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APA (6th Edition):

Lai, W. (2014). MECP2e1 Mutation Reduces the Dendritic Complexity of Rett Syndrome Patient iPS Cell-derived Neurons. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/67925

Chicago Manual of Style (16th Edition):

Lai, Wesley. “MECP2e1 Mutation Reduces the Dendritic Complexity of Rett Syndrome Patient iPS Cell-derived Neurons.” 2014. Masters Thesis, University of Toronto. Accessed July 18, 2019. http://hdl.handle.net/1807/67925.

MLA Handbook (7th Edition):

Lai, Wesley. “MECP2e1 Mutation Reduces the Dendritic Complexity of Rett Syndrome Patient iPS Cell-derived Neurons.” 2014. Web. 18 Jul 2019.

Vancouver:

Lai W. MECP2e1 Mutation Reduces the Dendritic Complexity of Rett Syndrome Patient iPS Cell-derived Neurons. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2019 Jul 18]. Available from: http://hdl.handle.net/1807/67925.

Council of Science Editors:

Lai W. MECP2e1 Mutation Reduces the Dendritic Complexity of Rett Syndrome Patient iPS Cell-derived Neurons. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/67925


University of Toronto

10. Jayakumaran, Gowtham. Molecular Mechanisms Regulating Somatic Reprogramming.

Degree: 2014, University of Toronto

In reprogramming, cellular transition to pluripotency only occurs in few cells. My thesisis focused on exploring the mechanisms underlying the successful transition of somatic cells… (more)

Subjects/Keywords: Cell plasticity; NGS; Reprogramming; Stem cells; 0307

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APA (6th Edition):

Jayakumaran, G. (2014). Molecular Mechanisms Regulating Somatic Reprogramming. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/67955

Chicago Manual of Style (16th Edition):

Jayakumaran, Gowtham. “Molecular Mechanisms Regulating Somatic Reprogramming.” 2014. Masters Thesis, University of Toronto. Accessed July 18, 2019. http://hdl.handle.net/1807/67955.

MLA Handbook (7th Edition):

Jayakumaran, Gowtham. “Molecular Mechanisms Regulating Somatic Reprogramming.” 2014. Web. 18 Jul 2019.

Vancouver:

Jayakumaran G. Molecular Mechanisms Regulating Somatic Reprogramming. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2019 Jul 18]. Available from: http://hdl.handle.net/1807/67955.

Council of Science Editors:

Jayakumaran G. Molecular Mechanisms Regulating Somatic Reprogramming. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/67955


University of Toronto

11. Naghdigheshlaghi, Zahra. Exploring Mechanisms Underlying Retinitis Pigmentosa by Using Temperature Sensitive Alleles in Yeast.

Degree: 2014, University of Toronto

Retinitis pigmentosa (RP) is characterized by progressive degeneration of the photoreceptors and visual field defects. No drug treatment has yet been found for this disease.… (more)

Subjects/Keywords: Genetic suppression; Retinitis Pigmentosa; Spliceosome; Temperature sensitive alleles; 0307

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APA (6th Edition):

Naghdigheshlaghi, Z. (2014). Exploring Mechanisms Underlying Retinitis Pigmentosa by Using Temperature Sensitive Alleles in Yeast. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/67994

Chicago Manual of Style (16th Edition):

Naghdigheshlaghi, Zahra. “Exploring Mechanisms Underlying Retinitis Pigmentosa by Using Temperature Sensitive Alleles in Yeast.” 2014. Masters Thesis, University of Toronto. Accessed July 18, 2019. http://hdl.handle.net/1807/67994.

MLA Handbook (7th Edition):

Naghdigheshlaghi, Zahra. “Exploring Mechanisms Underlying Retinitis Pigmentosa by Using Temperature Sensitive Alleles in Yeast.” 2014. Web. 18 Jul 2019.

Vancouver:

Naghdigheshlaghi Z. Exploring Mechanisms Underlying Retinitis Pigmentosa by Using Temperature Sensitive Alleles in Yeast. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2019 Jul 18]. Available from: http://hdl.handle.net/1807/67994.

Council of Science Editors:

Naghdigheshlaghi Z. Exploring Mechanisms Underlying Retinitis Pigmentosa by Using Temperature Sensitive Alleles in Yeast. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/67994


University of Toronto

12. Gazzellone, Joseph Matthew. Copy Number Variation in Han Chinese Individuals with Autism Spectrum Disorder.

Degree: 2014, University of Toronto

Autism spectrum disorders (ASDs) are a group of neurodevelopmental conditions with a demonstrated genetic etiology. Rare copy number variations (CNVs) account for a proportion of… (more)

Subjects/Keywords: Autism spectrum disorder (ASD); Copy number variations (CNVs); Han Chinese; Microarray diagnostic testing; 0369

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APA (6th Edition):

Gazzellone, J. M. (2014). Copy Number Variation in Han Chinese Individuals with Autism Spectrum Disorder. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/68053

Chicago Manual of Style (16th Edition):

Gazzellone, Joseph Matthew. “Copy Number Variation in Han Chinese Individuals with Autism Spectrum Disorder.” 2014. Masters Thesis, University of Toronto. Accessed July 18, 2019. http://hdl.handle.net/1807/68053.

MLA Handbook (7th Edition):

Gazzellone, Joseph Matthew. “Copy Number Variation in Han Chinese Individuals with Autism Spectrum Disorder.” 2014. Web. 18 Jul 2019.

Vancouver:

Gazzellone JM. Copy Number Variation in Han Chinese Individuals with Autism Spectrum Disorder. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2019 Jul 18]. Available from: http://hdl.handle.net/1807/68053.

Council of Science Editors:

Gazzellone JM. Copy Number Variation in Han Chinese Individuals with Autism Spectrum Disorder. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/68053


University of Toronto

13. Mador-House, Kelly Rachel. Investigation of the Epigenetic Landscape at Disease-causing Polymorphic Repeat Loci.

Degree: 2014, University of Toronto

There are over 40 genetic diseases caused by repeat locus-specific instability. The causes of repeat instability are poorly understood and a mechanism that explains their… (more)

Subjects/Keywords: 0369

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APA (6th Edition):

Mador-House, K. R. (2014). Investigation of the Epigenetic Landscape at Disease-causing Polymorphic Repeat Loci. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/68079

Chicago Manual of Style (16th Edition):

Mador-House, Kelly Rachel. “Investigation of the Epigenetic Landscape at Disease-causing Polymorphic Repeat Loci.” 2014. Masters Thesis, University of Toronto. Accessed July 18, 2019. http://hdl.handle.net/1807/68079.

MLA Handbook (7th Edition):

Mador-House, Kelly Rachel. “Investigation of the Epigenetic Landscape at Disease-causing Polymorphic Repeat Loci.” 2014. Web. 18 Jul 2019.

Vancouver:

Mador-House KR. Investigation of the Epigenetic Landscape at Disease-causing Polymorphic Repeat Loci. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2019 Jul 18]. Available from: http://hdl.handle.net/1807/68079.

Council of Science Editors:

Mador-House KR. Investigation of the Epigenetic Landscape at Disease-causing Polymorphic Repeat Loci. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/68079


University of Toronto

14. Li, Siyang. Identifying Protein Interactions Amongst DNA Damage Repair Proteins in Yeast.

Degree: 2014, University of Toronto

Mapping protein-protein interactions (PPIs) is crucial for understanding cellular systems. PPIs can be studied with binary or co-complex methods. A major economical binary method is… (more)

Subjects/Keywords: High-throughput; Protein interactions; Yeast two-hybrid; 0307

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APA (6th Edition):

Li, S. (2014). Identifying Protein Interactions Amongst DNA Damage Repair Proteins in Yeast. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/68505

Chicago Manual of Style (16th Edition):

Li, Siyang. “Identifying Protein Interactions Amongst DNA Damage Repair Proteins in Yeast.” 2014. Masters Thesis, University of Toronto. Accessed July 18, 2019. http://hdl.handle.net/1807/68505.

MLA Handbook (7th Edition):

Li, Siyang. “Identifying Protein Interactions Amongst DNA Damage Repair Proteins in Yeast.” 2014. Web. 18 Jul 2019.

Vancouver:

Li S. Identifying Protein Interactions Amongst DNA Damage Repair Proteins in Yeast. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2019 Jul 18]. Available from: http://hdl.handle.net/1807/68505.

Council of Science Editors:

Li S. Identifying Protein Interactions Amongst DNA Damage Repair Proteins in Yeast. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/68505


University of Toronto

15. Soo, Jeremy. Examining the Differences in Silencing Properties between H-NS and its paralog, StpA.

Degree: 2015, University of Toronto

Horizontal gene transfer (HGT) is a ubiquitous process that facilitates bacterial evolution. Horizontally acquired genes typically display a lower AT-content than the average of the… (more)

Subjects/Keywords: Genetics; H-NS; Microbiology; Salmonella; StpA; Xenogeneic silencer; 0410

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APA (6th Edition):

Soo, J. (2015). Examining the Differences in Silencing Properties between H-NS and its paralog, StpA. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/69709

Chicago Manual of Style (16th Edition):

Soo, Jeremy. “Examining the Differences in Silencing Properties between H-NS and its paralog, StpA.” 2015. Masters Thesis, University of Toronto. Accessed July 18, 2019. http://hdl.handle.net/1807/69709.

MLA Handbook (7th Edition):

Soo, Jeremy. “Examining the Differences in Silencing Properties between H-NS and its paralog, StpA.” 2015. Web. 18 Jul 2019.

Vancouver:

Soo J. Examining the Differences in Silencing Properties between H-NS and its paralog, StpA. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2019 Jul 18]. Available from: http://hdl.handle.net/1807/69709.

Council of Science Editors:

Soo J. Examining the Differences in Silencing Properties between H-NS and its paralog, StpA. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/69709


University of Toronto

16. Lee, Richard Heungki. An In Vivo System for the Identification of Ligands for Peroxisome Proliferator-activated Receptors.

Degree: 2015, University of Toronto

Peroxisome proliferator-activated receptors (PPARs) are a group of nuclear receptors (NRs) that play major roles in metabolism and development and, when disrupted, lead to disorders… (more)

Subjects/Keywords: Coenzyme Q10; Nuclear Receptor; PPAR; 0307

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APA (6th Edition):

Lee, R. H. (2015). An In Vivo System for the Identification of Ligands for Peroxisome Proliferator-activated Receptors. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/70369

Chicago Manual of Style (16th Edition):

Lee, Richard Heungki. “An In Vivo System for the Identification of Ligands for Peroxisome Proliferator-activated Receptors.” 2015. Masters Thesis, University of Toronto. Accessed July 18, 2019. http://hdl.handle.net/1807/70369.

MLA Handbook (7th Edition):

Lee, Richard Heungki. “An In Vivo System for the Identification of Ligands for Peroxisome Proliferator-activated Receptors.” 2015. Web. 18 Jul 2019.

Vancouver:

Lee RH. An In Vivo System for the Identification of Ligands for Peroxisome Proliferator-activated Receptors. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2019 Jul 18]. Available from: http://hdl.handle.net/1807/70369.

Council of Science Editors:

Lee RH. An In Vivo System for the Identification of Ligands for Peroxisome Proliferator-activated Receptors. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/70369


University of Toronto

17. Li, Xinliu. Regulation of Fungal Drug Resistance and Morphogenesis by Lysine Deacetylases.

Degree: 2015, University of Toronto

Hsp90 is a molecular chaperone that governs drug resistance, morphogenesis, and virulence in the leading human fungal pathogen Candida albicans. Previous work with Saccharomyces cerevisiae… (more)

Subjects/Keywords: Acetylation; Candida albicans; Deacetylases; Drug resistance; Hsp90; Morphogenesis; 0410

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APA (6th Edition):

Li, X. (2015). Regulation of Fungal Drug Resistance and Morphogenesis by Lysine Deacetylases. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/70439

Chicago Manual of Style (16th Edition):

Li, Xinliu. “Regulation of Fungal Drug Resistance and Morphogenesis by Lysine Deacetylases.” 2015. Masters Thesis, University of Toronto. Accessed July 18, 2019. http://hdl.handle.net/1807/70439.

MLA Handbook (7th Edition):

Li, Xinliu. “Regulation of Fungal Drug Resistance and Morphogenesis by Lysine Deacetylases.” 2015. Web. 18 Jul 2019.

Vancouver:

Li X. Regulation of Fungal Drug Resistance and Morphogenesis by Lysine Deacetylases. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2019 Jul 18]. Available from: http://hdl.handle.net/1807/70439.

Council of Science Editors:

Li X. Regulation of Fungal Drug Resistance and Morphogenesis by Lysine Deacetylases. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/70439


University of Toronto

18. Naccarato, Alessandra. Phosphorylation of Neuralized at Serine 73 and 74 is Required for Proper Regulation of Neuralized and Notch Signaling during Drosophila development.

Degree: 2014, University of Toronto

The Notch (N) pathway plays a crucial role in metazoan development. Neuralized (Neur), an E3 ubiquitin ligase, is responsible for ubiquitination and endocytosis of the… (more)

Subjects/Keywords: aPKC; Drosophila; Neuralized; 0307

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APA (6th Edition):

Naccarato, A. (2014). Phosphorylation of Neuralized at Serine 73 and 74 is Required for Proper Regulation of Neuralized and Notch Signaling during Drosophila development. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/70451

Chicago Manual of Style (16th Edition):

Naccarato, Alessandra. “Phosphorylation of Neuralized at Serine 73 and 74 is Required for Proper Regulation of Neuralized and Notch Signaling during Drosophila development.” 2014. Masters Thesis, University of Toronto. Accessed July 18, 2019. http://hdl.handle.net/1807/70451.

MLA Handbook (7th Edition):

Naccarato, Alessandra. “Phosphorylation of Neuralized at Serine 73 and 74 is Required for Proper Regulation of Neuralized and Notch Signaling during Drosophila development.” 2014. Web. 18 Jul 2019.

Vancouver:

Naccarato A. Phosphorylation of Neuralized at Serine 73 and 74 is Required for Proper Regulation of Neuralized and Notch Signaling during Drosophila development. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2019 Jul 18]. Available from: http://hdl.handle.net/1807/70451.

Council of Science Editors:

Naccarato A. Phosphorylation of Neuralized at Serine 73 and 74 is Required for Proper Regulation of Neuralized and Notch Signaling during Drosophila development. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/70451


University of Toronto

19. Magnan, Chantal Nicole. Investigating the Determinants of Host Range Specificity of Pseudomonas aeruginosa Phage.

Degree: 2015, University of Toronto

Phage therapy, or the use of bacteriophages (phages) to kill pathogenic bacteria, represents an alternative strategy to antibiotics in combating bacterial disease in the face… (more)

Subjects/Keywords: aeruginosa; Bacteriophage; fibers; Pseudomonas; Specificity; tail; 0410

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APA (6th Edition):

Magnan, C. N. (2015). Investigating the Determinants of Host Range Specificity of Pseudomonas aeruginosa Phage. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/70489

Chicago Manual of Style (16th Edition):

Magnan, Chantal Nicole. “Investigating the Determinants of Host Range Specificity of Pseudomonas aeruginosa Phage.” 2015. Masters Thesis, University of Toronto. Accessed July 18, 2019. http://hdl.handle.net/1807/70489.

MLA Handbook (7th Edition):

Magnan, Chantal Nicole. “Investigating the Determinants of Host Range Specificity of Pseudomonas aeruginosa Phage.” 2015. Web. 18 Jul 2019.

Vancouver:

Magnan CN. Investigating the Determinants of Host Range Specificity of Pseudomonas aeruginosa Phage. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2019 Jul 18]. Available from: http://hdl.handle.net/1807/70489.

Council of Science Editors:

Magnan CN. Investigating the Determinants of Host Range Specificity of Pseudomonas aeruginosa Phage. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/70489


University of Toronto

20. Nhat Nguyen, Huy Huynh. Characterization of Symbiotic Interactions between Pancreatic Cancer Cells and Tumor-associated Fibroblasts.

Degree: 2015, University of Toronto

Pancreatic cancer, one of the most lethal of cancers, is characterized by extensive fibrotic stroma. It is unknown whether this extensive stroma promotes or prevents… (more)

Subjects/Keywords: co-culture; desmoplasia; fibroblasts; fibrosis; pancreatic cancer; tumor microenvironment; 0307

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APA (6th Edition):

Nhat Nguyen, H. H. (2015). Characterization of Symbiotic Interactions between Pancreatic Cancer Cells and Tumor-associated Fibroblasts. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/70505

Chicago Manual of Style (16th Edition):

Nhat Nguyen, Huy Huynh. “Characterization of Symbiotic Interactions between Pancreatic Cancer Cells and Tumor-associated Fibroblasts.” 2015. Masters Thesis, University of Toronto. Accessed July 18, 2019. http://hdl.handle.net/1807/70505.

MLA Handbook (7th Edition):

Nhat Nguyen, Huy Huynh. “Characterization of Symbiotic Interactions between Pancreatic Cancer Cells and Tumor-associated Fibroblasts.” 2015. Web. 18 Jul 2019.

Vancouver:

Nhat Nguyen HH. Characterization of Symbiotic Interactions between Pancreatic Cancer Cells and Tumor-associated Fibroblasts. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2019 Jul 18]. Available from: http://hdl.handle.net/1807/70505.

Council of Science Editors:

Nhat Nguyen HH. Characterization of Symbiotic Interactions between Pancreatic Cancer Cells and Tumor-associated Fibroblasts. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/70505


University of Toronto

21. Nachman, Emil Natanson. Functional Consequences of Embryonic Stem Cell-Specific Alternative Splicing.

Degree: 2015, University of Toronto

Alternative splicing (AS) is a powerful mechanism for the generation of functional and regulatory diversity. It is present in transcripts from 95% of human multi-exon… (more)

Subjects/Keywords: alternative splicing; differentiation; embryonic stem cells; Mta1; 0307

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APA (6th Edition):

Nachman, E. N. (2015). Functional Consequences of Embryonic Stem Cell-Specific Alternative Splicing. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/70540

Chicago Manual of Style (16th Edition):

Nachman, Emil Natanson. “Functional Consequences of Embryonic Stem Cell-Specific Alternative Splicing.” 2015. Masters Thesis, University of Toronto. Accessed July 18, 2019. http://hdl.handle.net/1807/70540.

MLA Handbook (7th Edition):

Nachman, Emil Natanson. “Functional Consequences of Embryonic Stem Cell-Specific Alternative Splicing.” 2015. Web. 18 Jul 2019.

Vancouver:

Nachman EN. Functional Consequences of Embryonic Stem Cell-Specific Alternative Splicing. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2019 Jul 18]. Available from: http://hdl.handle.net/1807/70540.

Council of Science Editors:

Nachman EN. Functional Consequences of Embryonic Stem Cell-Specific Alternative Splicing. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/70540


University of Toronto

22. Wu, Yingzhou. Evaluating the Correspondence between Genetic Interactions and Gene Order of Action in S. cerevisiae.

Degree: 2015, University of Toronto

Although a molecular function has been revealed for over 80% of Saccharomyces cerevisiae genes, the specific position of genes within ordered biological pathways remains elusive.… (more)

Subjects/Keywords: Epistasis; Gene order; 0715

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APA (6th Edition):

Wu, Y. (2015). Evaluating the Correspondence between Genetic Interactions and Gene Order of Action in S. cerevisiae. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/70701

Chicago Manual of Style (16th Edition):

Wu, Yingzhou. “Evaluating the Correspondence between Genetic Interactions and Gene Order of Action in S. cerevisiae.” 2015. Masters Thesis, University of Toronto. Accessed July 18, 2019. http://hdl.handle.net/1807/70701.

MLA Handbook (7th Edition):

Wu, Yingzhou. “Evaluating the Correspondence between Genetic Interactions and Gene Order of Action in S. cerevisiae.” 2015. Web. 18 Jul 2019.

Vancouver:

Wu Y. Evaluating the Correspondence between Genetic Interactions and Gene Order of Action in S. cerevisiae. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2019 Jul 18]. Available from: http://hdl.handle.net/1807/70701.

Council of Science Editors:

Wu Y. Evaluating the Correspondence between Genetic Interactions and Gene Order of Action in S. cerevisiae. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/70701


University of Toronto

23. McSheffrey, Gordon Gerald. Host Cellular Responses to Neisseria gonorrhoeae.

Degree: 2016, University of Toronto

Neisseria gonorrhoeae is a human-restricted pathogen associated with increasing morbidity as antibiotic-resistant strains spread globally. N. gonorrhoeae possesses sophisticated mechanisms to survive the host immune… (more)

Subjects/Keywords: gonorrhea; macrophage; phosphoinositides; 0410

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APA (6th Edition):

McSheffrey, G. G. (2016). Host Cellular Responses to Neisseria gonorrhoeae. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/71686

Chicago Manual of Style (16th Edition):

McSheffrey, Gordon Gerald. “Host Cellular Responses to Neisseria gonorrhoeae.” 2016. Masters Thesis, University of Toronto. Accessed July 18, 2019. http://hdl.handle.net/1807/71686.

MLA Handbook (7th Edition):

McSheffrey, Gordon Gerald. “Host Cellular Responses to Neisseria gonorrhoeae.” 2016. Web. 18 Jul 2019.

Vancouver:

McSheffrey GG. Host Cellular Responses to Neisseria gonorrhoeae. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2019 Jul 18]. Available from: http://hdl.handle.net/1807/71686.

Council of Science Editors:

McSheffrey GG. Host Cellular Responses to Neisseria gonorrhoeae. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/71686


University of Toronto

24. Koh, Eun Jee. Exploring Context-specific Genetic Vulnerabilities in Human Cancer Cell Lines.

Degree: 2016, University of Toronto

Synthetic lethality arises when a combination of mutations in multiple genes leads to cell death, whereas a mutation in only one of them retains cell… (more)

Subjects/Keywords: alpha-ketoglutarate dehydrogenase; context-specific vulnerability; exonuclease1; fitness genes; genetic interaction; oxidative phosphorylation; 0992

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APA (6th Edition):

Koh, E. J. (2016). Exploring Context-specific Genetic Vulnerabilities in Human Cancer Cell Lines. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/72730

Chicago Manual of Style (16th Edition):

Koh, Eun Jee. “Exploring Context-specific Genetic Vulnerabilities in Human Cancer Cell Lines.” 2016. Masters Thesis, University of Toronto. Accessed July 18, 2019. http://hdl.handle.net/1807/72730.

MLA Handbook (7th Edition):

Koh, Eun Jee. “Exploring Context-specific Genetic Vulnerabilities in Human Cancer Cell Lines.” 2016. Web. 18 Jul 2019.

Vancouver:

Koh EJ. Exploring Context-specific Genetic Vulnerabilities in Human Cancer Cell Lines. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2019 Jul 18]. Available from: http://hdl.handle.net/1807/72730.

Council of Science Editors:

Koh EJ. Exploring Context-specific Genetic Vulnerabilities in Human Cancer Cell Lines. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/72730


University of Toronto

25. Lee, Eva I-Hua. Distinctive Roles for FANCD2 and FANCJ in the Resection of Radiation-induced DNA Double-strand Breaks in Human Cells.

Degree: 2016, University of Toronto

Fanconi anemia (FA) patients are hypersensitive to ionizing radiation and other agents that generate DNA double-strand breaks (DSBs). The major error-free DSB repair pathway in… (more)

Subjects/Keywords: DNA repair; Double strand breaks; DSB resection; FANCD2; FANCJ; Fanconi Anemia; 0369

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APA (6th Edition):

Lee, E. I. (2016). Distinctive Roles for FANCD2 and FANCJ in the Resection of Radiation-induced DNA Double-strand Breaks in Human Cells. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/72732

Chicago Manual of Style (16th Edition):

Lee, Eva I-Hua. “Distinctive Roles for FANCD2 and FANCJ in the Resection of Radiation-induced DNA Double-strand Breaks in Human Cells.” 2016. Masters Thesis, University of Toronto. Accessed July 18, 2019. http://hdl.handle.net/1807/72732.

MLA Handbook (7th Edition):

Lee, Eva I-Hua. “Distinctive Roles for FANCD2 and FANCJ in the Resection of Radiation-induced DNA Double-strand Breaks in Human Cells.” 2016. Web. 18 Jul 2019.

Vancouver:

Lee EI. Distinctive Roles for FANCD2 and FANCJ in the Resection of Radiation-induced DNA Double-strand Breaks in Human Cells. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2019 Jul 18]. Available from: http://hdl.handle.net/1807/72732.

Council of Science Editors:

Lee EI. Distinctive Roles for FANCD2 and FANCJ in the Resection of Radiation-induced DNA Double-strand Breaks in Human Cells. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/72732


University of Toronto

26. Balachandran, Ahalya. Identification of Novel Compounds That Inhibit HIV-1 Gene Expression by Targeting Viral RNA Processing.

Degree: 2015, University of Toronto

Novel strategies targeting different stages of the HIV lifecycle are vital for continued success in combating viral infection. Since HIV gene expression is dependent upon… (more)

Subjects/Keywords: anti-viral therapy; HIV; RNA processing; splicing; 0720

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APA (6th Edition):

Balachandran, A. (2015). Identification of Novel Compounds That Inhibit HIV-1 Gene Expression by Targeting Viral RNA Processing. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/74639

Chicago Manual of Style (16th Edition):

Balachandran, Ahalya. “Identification of Novel Compounds That Inhibit HIV-1 Gene Expression by Targeting Viral RNA Processing.” 2015. Masters Thesis, University of Toronto. Accessed July 18, 2019. http://hdl.handle.net/1807/74639.

MLA Handbook (7th Edition):

Balachandran, Ahalya. “Identification of Novel Compounds That Inhibit HIV-1 Gene Expression by Targeting Viral RNA Processing.” 2015. Web. 18 Jul 2019.

Vancouver:

Balachandran A. Identification of Novel Compounds That Inhibit HIV-1 Gene Expression by Targeting Viral RNA Processing. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2019 Jul 18]. Available from: http://hdl.handle.net/1807/74639.

Council of Science Editors:

Balachandran A. Identification of Novel Compounds That Inhibit HIV-1 Gene Expression by Targeting Viral RNA Processing. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/74639


University of Toronto

27. Lee, Timothy Mendel. Surveying the Human Genome for Genetic Markers Associated with the Mutator Phenotype and Chromosomal Instability.

Degree: 2016, University of Toronto

Cancer is a genetic disease that develops as a result of the acquisition of stable somatic mutations. Many cancers are believed to acquire mutations at… (more)

Subjects/Keywords: 0369

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APA (6th Edition):

Lee, T. M. (2016). Surveying the Human Genome for Genetic Markers Associated with the Mutator Phenotype and Chromosomal Instability. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/75109

Chicago Manual of Style (16th Edition):

Lee, Timothy Mendel. “Surveying the Human Genome for Genetic Markers Associated with the Mutator Phenotype and Chromosomal Instability.” 2016. Masters Thesis, University of Toronto. Accessed July 18, 2019. http://hdl.handle.net/1807/75109.

MLA Handbook (7th Edition):

Lee, Timothy Mendel. “Surveying the Human Genome for Genetic Markers Associated with the Mutator Phenotype and Chromosomal Instability.” 2016. Web. 18 Jul 2019.

Vancouver:

Lee TM. Surveying the Human Genome for Genetic Markers Associated with the Mutator Phenotype and Chromosomal Instability. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2019 Jul 18]. Available from: http://hdl.handle.net/1807/75109.

Council of Science Editors:

Lee TM. Surveying the Human Genome for Genetic Markers Associated with the Mutator Phenotype and Chromosomal Instability. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/75109


University of Toronto

28. He, Xinwen. Initial Molecular Characterization of Planarian Pigment Cells, a New Model for Studying Lineage Specification and Gut Excretion.

Degree: 2016, University of Toronto

ASCs play crucial roles in tissue homeostasis and regeneration. Here, I established pigment cells in Schmidtea mediterranea as a novel model to study ASC lineage… (more)

Subjects/Keywords: Lineage Specification; Photosensitivity; Pigment; Planarian; Regeneration; Stem Cell; 0369

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APA (6th Edition):

He, X. (2016). Initial Molecular Characterization of Planarian Pigment Cells, a New Model for Studying Lineage Specification and Gut Excretion. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/75963

Chicago Manual of Style (16th Edition):

He, Xinwen. “Initial Molecular Characterization of Planarian Pigment Cells, a New Model for Studying Lineage Specification and Gut Excretion.” 2016. Masters Thesis, University of Toronto. Accessed July 18, 2019. http://hdl.handle.net/1807/75963.

MLA Handbook (7th Edition):

He, Xinwen. “Initial Molecular Characterization of Planarian Pigment Cells, a New Model for Studying Lineage Specification and Gut Excretion.” 2016. Web. 18 Jul 2019.

Vancouver:

He X. Initial Molecular Characterization of Planarian Pigment Cells, a New Model for Studying Lineage Specification and Gut Excretion. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2019 Jul 18]. Available from: http://hdl.handle.net/1807/75963.

Council of Science Editors:

He X. Initial Molecular Characterization of Planarian Pigment Cells, a New Model for Studying Lineage Specification and Gut Excretion. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/75963


University of Toronto

29. Smith, Sarah. Investigating Lysosomes and Protein Degradation Pathways for the Treatment of Congenital Muscular Dystrophy Type 1A.

Degree: 2016, University of Toronto

Congenital muscular dystrophy type 1A (MDC1A) is the most common congenital muscular dystrophy in Western countries. The goal of our lab is to identify potential… (more)

Subjects/Keywords: autophagy; congenital muscular dystrophies; lysosomes; MDC1A; therapy development; zebrafish; 0307

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APA (6th Edition):

Smith, S. (2016). Investigating Lysosomes and Protein Degradation Pathways for the Treatment of Congenital Muscular Dystrophy Type 1A. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/76035

Chicago Manual of Style (16th Edition):

Smith, Sarah. “Investigating Lysosomes and Protein Degradation Pathways for the Treatment of Congenital Muscular Dystrophy Type 1A.” 2016. Masters Thesis, University of Toronto. Accessed July 18, 2019. http://hdl.handle.net/1807/76035.

MLA Handbook (7th Edition):

Smith, Sarah. “Investigating Lysosomes and Protein Degradation Pathways for the Treatment of Congenital Muscular Dystrophy Type 1A.” 2016. Web. 18 Jul 2019.

Vancouver:

Smith S. Investigating Lysosomes and Protein Degradation Pathways for the Treatment of Congenital Muscular Dystrophy Type 1A. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2019 Jul 18]. Available from: http://hdl.handle.net/1807/76035.

Council of Science Editors:

Smith S. Investigating Lysosomes and Protein Degradation Pathways for the Treatment of Congenital Muscular Dystrophy Type 1A. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/76035


University of Toronto

30. Yu, Emily Pui Yun. Identifying Novel Genetic Causes of Primary Ciliary Dyskinesia.

Degree: 2016, University of Toronto

Primary ciliary dyskinesia (PCD) is a disease that affects the function of respiratory cilia that helps protect against airway infections. Diagnosis is difficult with current… (more)

Subjects/Keywords: cilia; PCD; spdl1; zebrafish; 0758

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APA (6th Edition):

Yu, E. P. Y. (2016). Identifying Novel Genetic Causes of Primary Ciliary Dyskinesia. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/76125

Chicago Manual of Style (16th Edition):

Yu, Emily Pui Yun. “Identifying Novel Genetic Causes of Primary Ciliary Dyskinesia.” 2016. Masters Thesis, University of Toronto. Accessed July 18, 2019. http://hdl.handle.net/1807/76125.

MLA Handbook (7th Edition):

Yu, Emily Pui Yun. “Identifying Novel Genetic Causes of Primary Ciliary Dyskinesia.” 2016. Web. 18 Jul 2019.

Vancouver:

Yu EPY. Identifying Novel Genetic Causes of Primary Ciliary Dyskinesia. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2019 Jul 18]. Available from: http://hdl.handle.net/1807/76125.

Council of Science Editors:

Yu EPY. Identifying Novel Genetic Causes of Primary Ciliary Dyskinesia. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/76125

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