Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for +publisher:"University of Toronto" +contributor:("Laboratory Medicine and Pathobiology"). Showing records 1 – 30 of 335 total matches.

[1] [2] [3] [4] [5] … [12]

Search Limiters

Last 2 Years | English Only

Levels

▼ Search Limiters

1. Li, June. Anti-GPIbα Mediated Platelet Desialylation and Activation: A Novel Fc-independent Platelet Clearance Mechanism and Potential Therapeutic and Diagnostic Target in ITP.

Degree: 2014, University of Toronto

Immune thrombocytopenia (ITP) is a common bleeding disorder caused primarily by autoantibodies against platelet GPIIbIIIa and/or the GPIb complex. Current theory suggests antibody-mediated platelet destruction… (more)

Subjects/Keywords: Immune thrombocytopeni; Fc-independent clearance; 0719; 0982; 0571

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Li, J. (2014). Anti-GPIbα Mediated Platelet Desialylation and Activation: A Novel Fc-independent Platelet Clearance Mechanism and Potential Therapeutic and Diagnostic Target in ITP. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/65570

Chicago Manual of Style (16th Edition):

Li, June. “Anti-GPIbα Mediated Platelet Desialylation and Activation: A Novel Fc-independent Platelet Clearance Mechanism and Potential Therapeutic and Diagnostic Target in ITP.” 2014. Masters Thesis, University of Toronto. Accessed December 10, 2018. http://hdl.handle.net/1807/65570.

MLA Handbook (7th Edition):

Li, June. “Anti-GPIbα Mediated Platelet Desialylation and Activation: A Novel Fc-independent Platelet Clearance Mechanism and Potential Therapeutic and Diagnostic Target in ITP.” 2014. Web. 10 Dec 2018.

Vancouver:

Li J. Anti-GPIbα Mediated Platelet Desialylation and Activation: A Novel Fc-independent Platelet Clearance Mechanism and Potential Therapeutic and Diagnostic Target in ITP. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/1807/65570.

Council of Science Editors:

Li J. Anti-GPIbα Mediated Platelet Desialylation and Activation: A Novel Fc-independent Platelet Clearance Mechanism and Potential Therapeutic and Diagnostic Target in ITP. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/65570

2. Munteanu, Alexander. Characterizing the Role of Podocalyxin in Breast Carcinoma.

Degree: 2015, University of Toronto

Lymphatic invasion (LVI), the presence of tumour emboli within the peritumoural lymphatics of the breast, is an independent prognostic marker for axillary node negative breast… (more)

Subjects/Keywords: 3D culture; acini; Breast Cancer; Lymphatic Invasion; Podocalyxin; TGF-β; 0307

Page 1 Page 2 Page 3 Page 4 Page 5 Page 6 Page 7

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Munteanu, A. (2015). Characterizing the Role of Podocalyxin in Breast Carcinoma. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/69151

Chicago Manual of Style (16th Edition):

Munteanu, Alexander. “Characterizing the Role of Podocalyxin in Breast Carcinoma.” 2015. Masters Thesis, University of Toronto. Accessed December 10, 2018. http://hdl.handle.net/1807/69151.

MLA Handbook (7th Edition):

Munteanu, Alexander. “Characterizing the Role of Podocalyxin in Breast Carcinoma.” 2015. Web. 10 Dec 2018.

Vancouver:

Munteanu A. Characterizing the Role of Podocalyxin in Breast Carcinoma. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/1807/69151.

Council of Science Editors:

Munteanu A. Characterizing the Role of Podocalyxin in Breast Carcinoma. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/69151

3. Sobhani, Mona. PRIMA-1met Induces Apoptosis in Waldenstrรถm's Macroglobulinemia Independent of p53, Alone and in Combination with Bortezomib.

Degree: 2015, University of Toronto

PRIMA-1met has shown promising preclinical activity in various cancer types. However, its effect on Waldenstrรถm's Macroglobulinemia (WM) as well as its exact mechanism of action… (more)

Subjects/Keywords: p53; p73; PRIMA-1met; Waldenström's Macroglobulinemia; 0992

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sobhani, M. (2015). PRIMA-1met Induces Apoptosis in Waldenstrรถm's Macroglobulinemia Independent of p53, Alone and in Combination with Bortezomib. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/69162

Chicago Manual of Style (16th Edition):

Sobhani, Mona. “PRIMA-1met Induces Apoptosis in Waldenstrรถm's Macroglobulinemia Independent of p53, Alone and in Combination with Bortezomib.” 2015. Masters Thesis, University of Toronto. Accessed December 10, 2018. http://hdl.handle.net/1807/69162.

MLA Handbook (7th Edition):

Sobhani, Mona. “PRIMA-1met Induces Apoptosis in Waldenstrรถm's Macroglobulinemia Independent of p53, Alone and in Combination with Bortezomib.” 2015. Web. 10 Dec 2018.

Vancouver:

Sobhani M. PRIMA-1met Induces Apoptosis in Waldenstrรถm's Macroglobulinemia Independent of p53, Alone and in Combination with Bortezomib. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/1807/69162.

Council of Science Editors:

Sobhani M. PRIMA-1met Induces Apoptosis in Waldenstrรถm's Macroglobulinemia Independent of p53, Alone and in Combination with Bortezomib. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/69162

4. Chen, Xi. Murine Model of Autoimmune Hemolytic Anemia: Red Blood Cell Clearance Mechanisms and Treatment Efficacy.

Degree: 2012, University of Toronto

Antibodies against erythrocyte-specific antigens can cause immune red blood cell (RBC) destruction, resulting in autoimmune hemolytic anemia (AHA). Therapy for patients with AHA remains a… (more)

Subjects/Keywords: autoimmunity; red blood cell; anemia; antibody; 0982; 0571

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chen, X. (2012). Murine Model of Autoimmune Hemolytic Anemia: Red Blood Cell Clearance Mechanisms and Treatment Efficacy. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/67232

Chicago Manual of Style (16th Edition):

Chen, Xi. “Murine Model of Autoimmune Hemolytic Anemia: Red Blood Cell Clearance Mechanisms and Treatment Efficacy.” 2012. Masters Thesis, University of Toronto. Accessed December 10, 2018. http://hdl.handle.net/1807/67232.

MLA Handbook (7th Edition):

Chen, Xi. “Murine Model of Autoimmune Hemolytic Anemia: Red Blood Cell Clearance Mechanisms and Treatment Efficacy.” 2012. Web. 10 Dec 2018.

Vancouver:

Chen X. Murine Model of Autoimmune Hemolytic Anemia: Red Blood Cell Clearance Mechanisms and Treatment Efficacy. [Internet] [Masters thesis]. University of Toronto; 2012. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/1807/67232.

Council of Science Editors:

Chen X. Murine Model of Autoimmune Hemolytic Anemia: Red Blood Cell Clearance Mechanisms and Treatment Efficacy. [Masters Thesis]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/67232

5. Picard, Daniel J. Integrated Genomic Analyses of Childhood Central Nervous System-Ppimitive Neuro-ectodermal Tumours (CNS-PNETs).

Degree: 2014, University of Toronto

CNS-PNETs are rare, aggressive, paediatric embryonal brain tumours that are poorly studied. We recently identified an aggressive sub-group of CNS-PNETs characterized by the amplification of… (more)

Subjects/Keywords: Primitive Neuro-ectodermal Tumours; LIN28; OLIG2; CNS-PNET; 0992

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Picard, D. J. (2014). Integrated Genomic Analyses of Childhood Central Nervous System-Ppimitive Neuro-ectodermal Tumours (CNS-PNETs). (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/44053

Chicago Manual of Style (16th Edition):

Picard, Daniel J. “Integrated Genomic Analyses of Childhood Central Nervous System-Ppimitive Neuro-ectodermal Tumours (CNS-PNETs).” 2014. Masters Thesis, University of Toronto. Accessed December 10, 2018. http://hdl.handle.net/1807/44053.

MLA Handbook (7th Edition):

Picard, Daniel J. “Integrated Genomic Analyses of Childhood Central Nervous System-Ppimitive Neuro-ectodermal Tumours (CNS-PNETs).” 2014. Web. 10 Dec 2018.

Vancouver:

Picard DJ. Integrated Genomic Analyses of Childhood Central Nervous System-Ppimitive Neuro-ectodermal Tumours (CNS-PNETs). [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/1807/44053.

Council of Science Editors:

Picard DJ. Integrated Genomic Analyses of Childhood Central Nervous System-Ppimitive Neuro-ectodermal Tumours (CNS-PNETs). [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/44053

6. Luft, Olga. Targeted Deletion of Fgl2 Enhances Anti-viral T Cell Responses and Mediates Viral Clearance in a Murine Model of Chronic Viral Infection.

Degree: 2014, University of Toronto

Chronic viral infection is a significant burden on healthcare systems worldwide. Robust anti-viral immune responses are essential for viral clearance. Persistent viruses use a variety… (more)

Subjects/Keywords: Chronic viral infection; FGL2; LCMV clone 13; viral clearance; 0720

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Luft, O. (2014). Targeted Deletion of Fgl2 Enhances Anti-viral T Cell Responses and Mediates Viral Clearance in a Murine Model of Chronic Viral Infection. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/44040

Chicago Manual of Style (16th Edition):

Luft, Olga. “Targeted Deletion of Fgl2 Enhances Anti-viral T Cell Responses and Mediates Viral Clearance in a Murine Model of Chronic Viral Infection.” 2014. Masters Thesis, University of Toronto. Accessed December 10, 2018. http://hdl.handle.net/1807/44040.

MLA Handbook (7th Edition):

Luft, Olga. “Targeted Deletion of Fgl2 Enhances Anti-viral T Cell Responses and Mediates Viral Clearance in a Murine Model of Chronic Viral Infection.” 2014. Web. 10 Dec 2018.

Vancouver:

Luft O. Targeted Deletion of Fgl2 Enhances Anti-viral T Cell Responses and Mediates Viral Clearance in a Murine Model of Chronic Viral Infection. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/1807/44040.

Council of Science Editors:

Luft O. Targeted Deletion of Fgl2 Enhances Anti-viral T Cell Responses and Mediates Viral Clearance in a Murine Model of Chronic Viral Infection. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/44040

7. Celebre, Angela Maria Louisa. ID1 Mediates Glioblastoma Chemoresistance to Temozolomide.

Degree: 2016, University of Toronto

Glioblastoma (GBM) is the most lethal primary brain tumour in adults, and represents a therapeutic challenge. We investigated the role of inhibitor-of-DNA-binding-1 (ID1), a transcriptional… (more)

Subjects/Keywords: cancer; chemoresistance; inhibitor of DNA binding 1 (ID1); tumour recurrence; 0379

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Celebre, A. M. L. (2016). ID1 Mediates Glioblastoma Chemoresistance to Temozolomide. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/74528

Chicago Manual of Style (16th Edition):

Celebre, Angela Maria Louisa. “ID1 Mediates Glioblastoma Chemoresistance to Temozolomide.” 2016. Masters Thesis, University of Toronto. Accessed December 10, 2018. http://hdl.handle.net/1807/74528.

MLA Handbook (7th Edition):

Celebre, Angela Maria Louisa. “ID1 Mediates Glioblastoma Chemoresistance to Temozolomide.” 2016. Web. 10 Dec 2018.

Vancouver:

Celebre AML. ID1 Mediates Glioblastoma Chemoresistance to Temozolomide. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/1807/74528.

Council of Science Editors:

Celebre AML. ID1 Mediates Glioblastoma Chemoresistance to Temozolomide. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/74528

8. Organ, Shawna L. c-MET and KRAS: Signalling and Clinical Implications in Colorectal Cancer.

Degree: 2013, University of Toronto

Colorectal cancer (CRC) is the third leading cause of death from cancer in North America. The KRAS gene is mutated in approximately 40-50% of all… (more)

Subjects/Keywords: c-MET; KRAS; 0379

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Organ, S. L. (2013). c-MET and KRAS: Signalling and Clinical Implications in Colorectal Cancer. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/43691

Chicago Manual of Style (16th Edition):

Organ, Shawna L. “c-MET and KRAS: Signalling and Clinical Implications in Colorectal Cancer.” 2013. Doctoral Dissertation, University of Toronto. Accessed December 10, 2018. http://hdl.handle.net/1807/43691.

MLA Handbook (7th Edition):

Organ, Shawna L. “c-MET and KRAS: Signalling and Clinical Implications in Colorectal Cancer.” 2013. Web. 10 Dec 2018.

Vancouver:

Organ SL. c-MET and KRAS: Signalling and Clinical Implications in Colorectal Cancer. [Internet] [Doctoral dissertation]. University of Toronto; 2013. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/1807/43691.

Council of Science Editors:

Organ SL. c-MET and KRAS: Signalling and Clinical Implications in Colorectal Cancer. [Doctoral Dissertation]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/43691

9. Eissa, Azza. Characterization of Kallikrein-related Peptidase-8 in Normal Human Epidermis and Psoriasis.

Degree: 2013, University of Toronto

Kallikrein-8 (KLK8) is a relatively-uncharacterized epidermal protease. Although proposed to regulate wound-healing and barrier repair in KLK8-deficient mouse skin, KLK8-catalytic activity was never demonstrated in… (more)

Subjects/Keywords: Kallikrein; Epidermis; Stratum corneum; Psoriasis; 0487; 0307

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Eissa, A. (2013). Characterization of Kallikrein-related Peptidase-8 in Normal Human Epidermis and Psoriasis. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/43602

Chicago Manual of Style (16th Edition):

Eissa, Azza. “Characterization of Kallikrein-related Peptidase-8 in Normal Human Epidermis and Psoriasis.” 2013. Doctoral Dissertation, University of Toronto. Accessed December 10, 2018. http://hdl.handle.net/1807/43602.

MLA Handbook (7th Edition):

Eissa, Azza. “Characterization of Kallikrein-related Peptidase-8 in Normal Human Epidermis and Psoriasis.” 2013. Web. 10 Dec 2018.

Vancouver:

Eissa A. Characterization of Kallikrein-related Peptidase-8 in Normal Human Epidermis and Psoriasis. [Internet] [Doctoral dissertation]. University of Toronto; 2013. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/1807/43602.

Council of Science Editors:

Eissa A. Characterization of Kallikrein-related Peptidase-8 in Normal Human Epidermis and Psoriasis. [Doctoral Dissertation]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/43602

10. Wang, Jenny Jing. Discovery of a VEGF-A Responsive lincRNA in Human Endothelial Cells with Disease Relevance and Anti-angiogenic Therapeutic Potential in Glioblastoma Multiforme.

Degree: 2014, University of Toronto

Large intergenic RNAs (lincRNA) are involved in numerous cellular processes, including many relevant to normal development and cancer progression. In my doctoral research, we hypothesized… (more)

Subjects/Keywords: lincRNAs; Glioblastoma; 0564; 0992; 0379

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wang, J. J. (2014). Discovery of a VEGF-A Responsive lincRNA in Human Endothelial Cells with Disease Relevance and Anti-angiogenic Therapeutic Potential in Glioblastoma Multiforme. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/44140

Chicago Manual of Style (16th Edition):

Wang, Jenny Jing. “Discovery of a VEGF-A Responsive lincRNA in Human Endothelial Cells with Disease Relevance and Anti-angiogenic Therapeutic Potential in Glioblastoma Multiforme.” 2014. Doctoral Dissertation, University of Toronto. Accessed December 10, 2018. http://hdl.handle.net/1807/44140.

MLA Handbook (7th Edition):

Wang, Jenny Jing. “Discovery of a VEGF-A Responsive lincRNA in Human Endothelial Cells with Disease Relevance and Anti-angiogenic Therapeutic Potential in Glioblastoma Multiforme.” 2014. Web. 10 Dec 2018.

Vancouver:

Wang JJ. Discovery of a VEGF-A Responsive lincRNA in Human Endothelial Cells with Disease Relevance and Anti-angiogenic Therapeutic Potential in Glioblastoma Multiforme. [Internet] [Doctoral dissertation]. University of Toronto; 2014. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/1807/44140.

Council of Science Editors:

Wang JJ. Discovery of a VEGF-A Responsive lincRNA in Human Endothelial Cells with Disease Relevance and Anti-angiogenic Therapeutic Potential in Glioblastoma Multiforme. [Doctoral Dissertation]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/44140

11. Mack, Christopher Stephen. The Genetic and Epigenetic Basis of Posterior Fossa Ependymoma.

Degree: PhD, 2014, University of Toronto

 Ependymomas are childhood brain tumors that occur throughout the central nervous system, but are most common in the hindbrain, also known as the posterior fossa… (more)

Subjects/Keywords: Brain Tumor; Cancer; Ependymoma; Epigenetics; Genetics; Pediatrics; 0566

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mack, C. S. (2014). The Genetic and Epigenetic Basis of Posterior Fossa Ependymoma. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/68135

Chicago Manual of Style (16th Edition):

Mack, Christopher Stephen. “The Genetic and Epigenetic Basis of Posterior Fossa Ependymoma.” 2014. Doctoral Dissertation, University of Toronto. Accessed December 10, 2018. http://hdl.handle.net/1807/68135.

MLA Handbook (7th Edition):

Mack, Christopher Stephen. “The Genetic and Epigenetic Basis of Posterior Fossa Ependymoma.” 2014. Web. 10 Dec 2018.

Vancouver:

Mack CS. The Genetic and Epigenetic Basis of Posterior Fossa Ependymoma. [Internet] [Doctoral dissertation]. University of Toronto; 2014. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/1807/68135.

Council of Science Editors:

Mack CS. The Genetic and Epigenetic Basis of Posterior Fossa Ependymoma. [Doctoral Dissertation]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/68135

12. Chow, Man Chi Edith. Destabilization of IL-8 mRNA by Anthrax Lethal Toxin: Demonstration of the Requirement for TTP and Examination of its Cellular Interactions.

Degree: 2012, University of Toronto

Control of mRNA stability is an important aspect in the regulation of gene expression. A well studied signal for rapid transcript decay in mammalian cells… (more)

Subjects/Keywords: Anthrax lethal toxin; mRNA destabilization; Tristetraprolin; 0410; 0307

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chow, M. C. E. (2012). Destabilization of IL-8 mRNA by Anthrax Lethal Toxin: Demonstration of the Requirement for TTP and Examination of its Cellular Interactions. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/33880

Chicago Manual of Style (16th Edition):

Chow, Man Chi Edith. “Destabilization of IL-8 mRNA by Anthrax Lethal Toxin: Demonstration of the Requirement for TTP and Examination of its Cellular Interactions.” 2012. Doctoral Dissertation, University of Toronto. Accessed December 10, 2018. http://hdl.handle.net/1807/33880.

MLA Handbook (7th Edition):

Chow, Man Chi Edith. “Destabilization of IL-8 mRNA by Anthrax Lethal Toxin: Demonstration of the Requirement for TTP and Examination of its Cellular Interactions.” 2012. Web. 10 Dec 2018.

Vancouver:

Chow MCE. Destabilization of IL-8 mRNA by Anthrax Lethal Toxin: Demonstration of the Requirement for TTP and Examination of its Cellular Interactions. [Internet] [Doctoral dissertation]. University of Toronto; 2012. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/1807/33880.

Council of Science Editors:

Chow MCE. Destabilization of IL-8 mRNA by Anthrax Lethal Toxin: Demonstration of the Requirement for TTP and Examination of its Cellular Interactions. [Doctoral Dissertation]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/33880

13. Weerasekera, Rasanjala Kumari. The Development of Novel Protein Topology Mapping Strategies using Crosslinking, Cyanogen Bromide Cleavage, and Mass Spectrometry.

Degree: 2011, University of Toronto

Advances in protein topology mapping methods are urgently needed to complement the wealth of interactome data that is presently being generated at a rapid pace.… (more)

Subjects/Keywords: protein structure; mass spectrometry; chemical crosslinking; interface mapping; proteomics; protein topology mapping; cyanogen bromide; interactome; method development; HPLC; 0307

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Weerasekera, R. K. (2011). The Development of Novel Protein Topology Mapping Strategies using Crosslinking, Cyanogen Bromide Cleavage, and Mass Spectrometry. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/31969

Chicago Manual of Style (16th Edition):

Weerasekera, Rasanjala Kumari. “The Development of Novel Protein Topology Mapping Strategies using Crosslinking, Cyanogen Bromide Cleavage, and Mass Spectrometry.” 2011. Doctoral Dissertation, University of Toronto. Accessed December 10, 2018. http://hdl.handle.net/1807/31969.

MLA Handbook (7th Edition):

Weerasekera, Rasanjala Kumari. “The Development of Novel Protein Topology Mapping Strategies using Crosslinking, Cyanogen Bromide Cleavage, and Mass Spectrometry.” 2011. Web. 10 Dec 2018.

Vancouver:

Weerasekera RK. The Development of Novel Protein Topology Mapping Strategies using Crosslinking, Cyanogen Bromide Cleavage, and Mass Spectrometry. [Internet] [Doctoral dissertation]. University of Toronto; 2011. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/1807/31969.

Council of Science Editors:

Weerasekera RK. The Development of Novel Protein Topology Mapping Strategies using Crosslinking, Cyanogen Bromide Cleavage, and Mass Spectrometry. [Doctoral Dissertation]. University of Toronto; 2011. Available from: http://hdl.handle.net/1807/31969

14. Mamaghani, Shadi. GSK-3 Inhibition: A Novel Approach to Sensitization of Chemo-resistant Pancreatic Cancer Cells.

Degree: 2011, University of Toronto

The aggressive nature of pancreatic cancer, characterized by invasiveness, resistance to treatment, rapid progression, and its high prevalence in the population urges the need for… (more)

Subjects/Keywords: Oncology; Molecular therapeutics; Pancreas; GSK-3; 0992; 0307; 0379

Page 1 Page 2 Page 3 Page 4 Page 5 Page 6 Page 7

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mamaghani, S. (2011). GSK-3 Inhibition: A Novel Approach to Sensitization of Chemo-resistant Pancreatic Cancer Cells. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/29802

Chicago Manual of Style (16th Edition):

Mamaghani, Shadi. “GSK-3 Inhibition: A Novel Approach to Sensitization of Chemo-resistant Pancreatic Cancer Cells.” 2011. Doctoral Dissertation, University of Toronto. Accessed December 10, 2018. http://hdl.handle.net/1807/29802.

MLA Handbook (7th Edition):

Mamaghani, Shadi. “GSK-3 Inhibition: A Novel Approach to Sensitization of Chemo-resistant Pancreatic Cancer Cells.” 2011. Web. 10 Dec 2018.

Vancouver:

Mamaghani S. GSK-3 Inhibition: A Novel Approach to Sensitization of Chemo-resistant Pancreatic Cancer Cells. [Internet] [Doctoral dissertation]. University of Toronto; 2011. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/1807/29802.

Council of Science Editors:

Mamaghani S. GSK-3 Inhibition: A Novel Approach to Sensitization of Chemo-resistant Pancreatic Cancer Cells. [Doctoral Dissertation]. University of Toronto; 2011. Available from: http://hdl.handle.net/1807/29802

15. Bikopoulos, George. Novel Molecular Mechanisms Controlling Pancreatic β-cell Function and Hepatic Glucose Homeostasis.

Degree: 2012, University of Toronto

The key defects characteristic of hyperglycemia in T2D include increased hepatic glucose production, a diminution of insulin secretion, and an absolute impairment in peripheral insulin… (more)

Subjects/Keywords: Pancreas; Liver; Transcription; Free fatty acids; 0307

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bikopoulos, G. (2012). Novel Molecular Mechanisms Controlling Pancreatic β-cell Function and Hepatic Glucose Homeostasis. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/42482

Chicago Manual of Style (16th Edition):

Bikopoulos, George. “Novel Molecular Mechanisms Controlling Pancreatic β-cell Function and Hepatic Glucose Homeostasis.” 2012. Doctoral Dissertation, University of Toronto. Accessed December 10, 2018. http://hdl.handle.net/1807/42482.

MLA Handbook (7th Edition):

Bikopoulos, George. “Novel Molecular Mechanisms Controlling Pancreatic β-cell Function and Hepatic Glucose Homeostasis.” 2012. Web. 10 Dec 2018.

Vancouver:

Bikopoulos G. Novel Molecular Mechanisms Controlling Pancreatic β-cell Function and Hepatic Glucose Homeostasis. [Internet] [Doctoral dissertation]. University of Toronto; 2012. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/1807/42482.

Council of Science Editors:

Bikopoulos G. Novel Molecular Mechanisms Controlling Pancreatic β-cell Function and Hepatic Glucose Homeostasis. [Doctoral Dissertation]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/42482

16. Dubuc, Adrian. Mining the Medulloblastoma Genome and Transcriptome.

Degree: 2013, University of Toronto

Medulloblastoma is a devastating disease of the cerebellum, and the most common solid pediatric malignancy of the central nervous system. Recently, transcriptome-wide profiling has dissected… (more)

Subjects/Keywords: medulloblastoma; cancer; brain tumor; epigenetics; genomics; 0992; 0571; 0564

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dubuc, A. (2013). Mining the Medulloblastoma Genome and Transcriptome. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/43530

Chicago Manual of Style (16th Edition):

Dubuc, Adrian. “Mining the Medulloblastoma Genome and Transcriptome.” 2013. Doctoral Dissertation, University of Toronto. Accessed December 10, 2018. http://hdl.handle.net/1807/43530.

MLA Handbook (7th Edition):

Dubuc, Adrian. “Mining the Medulloblastoma Genome and Transcriptome.” 2013. Web. 10 Dec 2018.

Vancouver:

Dubuc A. Mining the Medulloblastoma Genome and Transcriptome. [Internet] [Doctoral dissertation]. University of Toronto; 2013. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/1807/43530.

Council of Science Editors:

Dubuc A. Mining the Medulloblastoma Genome and Transcriptome. [Doctoral Dissertation]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/43530

17. Rubino, Stephen. New Insights into the Regulation of Intestinal Immunity by Nod1 and Nod2.

Degree: 2014, University of Toronto

Nod1 and Nod2 are intracellular pattern recognition receptors that detect specific moieties of peptidoglycan, a critical component of the bacterial cell wall, to initiate host… (more)

Subjects/Keywords: Innate Immunity; Th17; 0982

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rubino, S. (2014). New Insights into the Regulation of Intestinal Immunity by Nod1 and Nod2. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/44135

Chicago Manual of Style (16th Edition):

Rubino, Stephen. “New Insights into the Regulation of Intestinal Immunity by Nod1 and Nod2.” 2014. Doctoral Dissertation, University of Toronto. Accessed December 10, 2018. http://hdl.handle.net/1807/44135.

MLA Handbook (7th Edition):

Rubino, Stephen. “New Insights into the Regulation of Intestinal Immunity by Nod1 and Nod2.” 2014. Web. 10 Dec 2018.

Vancouver:

Rubino S. New Insights into the Regulation of Intestinal Immunity by Nod1 and Nod2. [Internet] [Doctoral dissertation]. University of Toronto; 2014. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/1807/44135.

Council of Science Editors:

Rubino S. New Insights into the Regulation of Intestinal Immunity by Nod1 and Nod2. [Doctoral Dissertation]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/44135

18. Baker, Amy. Disruption of Lymphatic Function following Lymph Node Excision and Irradiation: Integrating Natural Compensatory Responses with Potential Therapeutic Approaches to Facilitate Lymph Flow Restoration.

Degree: 2014, University of Toronto

Introduction: The removal or irradiation of one or more lymph nodes during cancer surgery appears to be a significant causative factor in lymphedema development. In… (more)

Subjects/Keywords: Lymphedema; Breast Cancer; Lymphatics; Radiation Therapy; Lymph node; 0571, 0574, 0719, 0564, 0433

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Baker, A. (2014). Disruption of Lymphatic Function following Lymph Node Excision and Irradiation: Integrating Natural Compensatory Responses with Potential Therapeutic Approaches to Facilitate Lymph Flow Restoration. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/69049

Chicago Manual of Style (16th Edition):

Baker, Amy. “Disruption of Lymphatic Function following Lymph Node Excision and Irradiation: Integrating Natural Compensatory Responses with Potential Therapeutic Approaches to Facilitate Lymph Flow Restoration.” 2014. Doctoral Dissertation, University of Toronto. Accessed December 10, 2018. http://hdl.handle.net/1807/69049.

MLA Handbook (7th Edition):

Baker, Amy. “Disruption of Lymphatic Function following Lymph Node Excision and Irradiation: Integrating Natural Compensatory Responses with Potential Therapeutic Approaches to Facilitate Lymph Flow Restoration.” 2014. Web. 10 Dec 2018.

Vancouver:

Baker A. Disruption of Lymphatic Function following Lymph Node Excision and Irradiation: Integrating Natural Compensatory Responses with Potential Therapeutic Approaches to Facilitate Lymph Flow Restoration. [Internet] [Doctoral dissertation]. University of Toronto; 2014. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/1807/69049.

Council of Science Editors:

Baker A. Disruption of Lymphatic Function following Lymph Node Excision and Irradiation: Integrating Natural Compensatory Responses with Potential Therapeutic Approaches to Facilitate Lymph Flow Restoration. [Doctoral Dissertation]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/69049

19. Kuzmanov, Uros. Characterization of Kallikrein 6 N-glycosylation Patterns and Identification of Sialylated Glycoproteins in Ovarian Cancer.

Degree: 2013, University of Toronto

Ovarian cancer is the leading cause of death among all gynecological disorders. Aberrant glycosylation, or more specifically, increased sialylation of proteins has been observed in… (more)

Subjects/Keywords: glycosylation; ovarian cancer; 0487

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kuzmanov, U. (2013). Characterization of Kallikrein 6 N-glycosylation Patterns and Identification of Sialylated Glycoproteins in Ovarian Cancer. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/35871

Chicago Manual of Style (16th Edition):

Kuzmanov, Uros. “Characterization of Kallikrein 6 N-glycosylation Patterns and Identification of Sialylated Glycoproteins in Ovarian Cancer.” 2013. Doctoral Dissertation, University of Toronto. Accessed December 10, 2018. http://hdl.handle.net/1807/35871.

MLA Handbook (7th Edition):

Kuzmanov, Uros. “Characterization of Kallikrein 6 N-glycosylation Patterns and Identification of Sialylated Glycoproteins in Ovarian Cancer.” 2013. Web. 10 Dec 2018.

Vancouver:

Kuzmanov U. Characterization of Kallikrein 6 N-glycosylation Patterns and Identification of Sialylated Glycoproteins in Ovarian Cancer. [Internet] [Doctoral dissertation]. University of Toronto; 2013. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/1807/35871.

Council of Science Editors:

Kuzmanov U. Characterization of Kallikrein 6 N-glycosylation Patterns and Identification of Sialylated Glycoproteins in Ovarian Cancer. [Doctoral Dissertation]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/35871

20. Lee, Jooeun. Sensing of Bacterial Peptidoglycan by Peptidoglycan Recognition Molecules.

Degree: 2013, University of Toronto

Peptidoglycan is an essential component of bacteria that provide structure and integrity of the cell. The main goal of this thesis has been to study… (more)

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lee, J. (2013). Sensing of Bacterial Peptidoglycan by Peptidoglycan Recognition Molecules. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/35877

Chicago Manual of Style (16th Edition):

Lee, Jooeun. “Sensing of Bacterial Peptidoglycan by Peptidoglycan Recognition Molecules.” 2013. Doctoral Dissertation, University of Toronto. Accessed December 10, 2018. http://hdl.handle.net/1807/35877.

MLA Handbook (7th Edition):

Lee, Jooeun. “Sensing of Bacterial Peptidoglycan by Peptidoglycan Recognition Molecules.” 2013. Web. 10 Dec 2018.

Vancouver:

Lee J. Sensing of Bacterial Peptidoglycan by Peptidoglycan Recognition Molecules. [Internet] [Doctoral dissertation]. University of Toronto; 2013. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/1807/35877.

Council of Science Editors:

Lee J. Sensing of Bacterial Peptidoglycan by Peptidoglycan Recognition Molecules. [Doctoral Dissertation]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/35877

21. Jeon, Amy Hye Won. Comparative Interactome Investigation of γ-secretase Complex in Alzheimer’s Disease.

Degree: 2012, University of Toronto

γ-Secretase plays a pivotal role in the production of neurotoxic amyloid β-peptide (Aβ), the principal component of amyloid plaques present in Alzheimer’s disease. It consists… (more)

Subjects/Keywords: Alzheimer's Disease; Gamma-secretase; Amyloid beta peptide; Proteomics; 0317

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jeon, A. H. W. (2012). Comparative Interactome Investigation of γ-secretase Complex in Alzheimer’s Disease. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/43389

Chicago Manual of Style (16th Edition):

Jeon, Amy Hye Won. “Comparative Interactome Investigation of γ-secretase Complex in Alzheimer’s Disease.” 2012. Doctoral Dissertation, University of Toronto. Accessed December 10, 2018. http://hdl.handle.net/1807/43389.

MLA Handbook (7th Edition):

Jeon, Amy Hye Won. “Comparative Interactome Investigation of γ-secretase Complex in Alzheimer’s Disease.” 2012. Web. 10 Dec 2018.

Vancouver:

Jeon AHW. Comparative Interactome Investigation of γ-secretase Complex in Alzheimer’s Disease. [Internet] [Doctoral dissertation]. University of Toronto; 2012. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/1807/43389.

Council of Science Editors:

Jeon AHW. Comparative Interactome Investigation of γ-secretase Complex in Alzheimer’s Disease. [Doctoral Dissertation]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/43389

22. Ho, Nathan. Identifying Mechanisms by which Escherichia coli O157:H7 Subverts Interferon-gamma Mediated Signal Transducer and Activator of Transcription-1 Activation.

Degree: 2012, University of Toronto

Enterohemorrhagic Escherichia coli (EHEC) serotype O157:H7 is a foodborne pathogen that causes significant morbidity and mortality in developing and industrialized nations. EHEC infection of host… (more)

Subjects/Keywords: escherichia coli; stat1; gamma interferon; subversion; 0571; 0379; 0410; 0307

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ho, N. (2012). Identifying Mechanisms by which Escherichia coli O157:H7 Subverts Interferon-gamma Mediated Signal Transducer and Activator of Transcription-1 Activation. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/34056

Chicago Manual of Style (16th Edition):

Ho, Nathan. “Identifying Mechanisms by which Escherichia coli O157:H7 Subverts Interferon-gamma Mediated Signal Transducer and Activator of Transcription-1 Activation.” 2012. Doctoral Dissertation, University of Toronto. Accessed December 10, 2018. http://hdl.handle.net/1807/34056.

MLA Handbook (7th Edition):

Ho, Nathan. “Identifying Mechanisms by which Escherichia coli O157:H7 Subverts Interferon-gamma Mediated Signal Transducer and Activator of Transcription-1 Activation.” 2012. Web. 10 Dec 2018.

Vancouver:

Ho N. Identifying Mechanisms by which Escherichia coli O157:H7 Subverts Interferon-gamma Mediated Signal Transducer and Activator of Transcription-1 Activation. [Internet] [Doctoral dissertation]. University of Toronto; 2012. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/1807/34056.

Council of Science Editors:

Ho N. Identifying Mechanisms by which Escherichia coli O157:H7 Subverts Interferon-gamma Mediated Signal Transducer and Activator of Transcription-1 Activation. [Doctoral Dissertation]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/34056

23. Yun, James Jungwon. LKB1/AMPK Pathway as a Potential Target for Cancer Treatment and its Role in TGFB Signaling.

Degree: 2014, University of Toronto

Although the “Warburg Effect” was reported >50 years ago, in recent years, there has been much greater appreciation for the importance of understanding perturbations in… (more)

Subjects/Keywords: LKB1; STK11; AMPK; Metformin; MK-2206; AKT; mTOR; Cancer Metabolism; Drug Synergy; Combination Treatment; Tumor Suppressor; TGFB; Lung Cancer; Cancer; 0307; 0379

Page 1 Page 2 Page 3 Page 4 Page 5 Page 6 Page 7

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yun, J. J. (2014). LKB1/AMPK Pathway as a Potential Target for Cancer Treatment and its Role in TGFB Signaling. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/67321

Chicago Manual of Style (16th Edition):

Yun, James Jungwon. “LKB1/AMPK Pathway as a Potential Target for Cancer Treatment and its Role in TGFB Signaling.” 2014. Doctoral Dissertation, University of Toronto. Accessed December 10, 2018. http://hdl.handle.net/1807/67321.

MLA Handbook (7th Edition):

Yun, James Jungwon. “LKB1/AMPK Pathway as a Potential Target for Cancer Treatment and its Role in TGFB Signaling.” 2014. Web. 10 Dec 2018.

Vancouver:

Yun JJ. LKB1/AMPK Pathway as a Potential Target for Cancer Treatment and its Role in TGFB Signaling. [Internet] [Doctoral dissertation]. University of Toronto; 2014. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/1807/67321.

Council of Science Editors:

Yun JJ. LKB1/AMPK Pathway as a Potential Target for Cancer Treatment and its Role in TGFB Signaling. [Doctoral Dissertation]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/67321

24. Bielefeld, Kirsten Adele. A Regulatory Loop between β-Catenin and Extra-cellular Matrix Components during the Proliferative Phase of Cutaneous Wound Healing.

Degree: 2014, University of Toronto

β-Catenin is an important modulator of the cutaneous wound phenotype, where it is activated in dermal fibroblasts during the proliferative stage of repair. Despite its… (more)

Subjects/Keywords: wound repair; beta-catenin; extracellular matrix; fibroblast; 0306; 0379; 0307

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bielefeld, K. A. (2014). A Regulatory Loop between β-Catenin and Extra-cellular Matrix Components during the Proliferative Phase of Cutaneous Wound Healing. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/67258

Chicago Manual of Style (16th Edition):

Bielefeld, Kirsten Adele. “A Regulatory Loop between β-Catenin and Extra-cellular Matrix Components during the Proliferative Phase of Cutaneous Wound Healing.” 2014. Doctoral Dissertation, University of Toronto. Accessed December 10, 2018. http://hdl.handle.net/1807/67258.

MLA Handbook (7th Edition):

Bielefeld, Kirsten Adele. “A Regulatory Loop between β-Catenin and Extra-cellular Matrix Components during the Proliferative Phase of Cutaneous Wound Healing.” 2014. Web. 10 Dec 2018.

Vancouver:

Bielefeld KA. A Regulatory Loop between β-Catenin and Extra-cellular Matrix Components during the Proliferative Phase of Cutaneous Wound Healing. [Internet] [Doctoral dissertation]. University of Toronto; 2014. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/1807/67258.

Council of Science Editors:

Bielefeld KA. A Regulatory Loop between β-Catenin and Extra-cellular Matrix Components during the Proliferative Phase of Cutaneous Wound Healing. [Doctoral Dissertation]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/67258

25. Ho, Louisa. Modulators of Hedgehog Signaling in Neoplasia.

Degree: 2012, University of Toronto

The Hedgehog (Hh) signaling pathway plays a critical role in modulating various developmental processes that requires fine tuning of the Hh signal, such that dysregulation… (more)

Subjects/Keywords: Hedgehog; Chondrosarcoma; 0369; 0992

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ho, L. (2012). Modulators of Hedgehog Signaling in Neoplasia. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/34055

Chicago Manual of Style (16th Edition):

Ho, Louisa. “Modulators of Hedgehog Signaling in Neoplasia.” 2012. Doctoral Dissertation, University of Toronto. Accessed December 10, 2018. http://hdl.handle.net/1807/34055.

MLA Handbook (7th Edition):

Ho, Louisa. “Modulators of Hedgehog Signaling in Neoplasia.” 2012. Web. 10 Dec 2018.

Vancouver:

Ho L. Modulators of Hedgehog Signaling in Neoplasia. [Internet] [Doctoral dissertation]. University of Toronto; 2012. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/1807/34055.

Council of Science Editors:

Ho L. Modulators of Hedgehog Signaling in Neoplasia. [Doctoral Dissertation]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/34055

26. Ehsani, Sepehr. The Significance of the Evolutionary Relationship of Prion Proteins and ZIP Transporters in Health and Disease.

Degree: 2012, University of Toronto

The cellular prion protein (PrPC) is unique amongst mammalian proteins in that it not only has the capacity to aggregate (in the form of scrapie… (more)

Subjects/Keywords: Prion protein; ZIP transporters; Zinc; Metal biology; 0715; 0379; 0369; 0317

Page 1 Page 2 Page 3 Page 4 Page 5 Page 6 Page 7 Sample image

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ehsani, S. (2012). The Significance of the Evolutionary Relationship of Prion Proteins and ZIP Transporters in Health and Disease. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/33986

Chicago Manual of Style (16th Edition):

Ehsani, Sepehr. “The Significance of the Evolutionary Relationship of Prion Proteins and ZIP Transporters in Health and Disease.” 2012. Doctoral Dissertation, University of Toronto. Accessed December 10, 2018. http://hdl.handle.net/1807/33986.

MLA Handbook (7th Edition):

Ehsani, Sepehr. “The Significance of the Evolutionary Relationship of Prion Proteins and ZIP Transporters in Health and Disease.” 2012. Web. 10 Dec 2018.

Vancouver:

Ehsani S. The Significance of the Evolutionary Relationship of Prion Proteins and ZIP Transporters in Health and Disease. [Internet] [Doctoral dissertation]. University of Toronto; 2012. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/1807/33986.

Council of Science Editors:

Ehsani S. The Significance of the Evolutionary Relationship of Prion Proteins and ZIP Transporters in Health and Disease. [Doctoral Dissertation]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/33986

27. Gordanpour, Aida. MicroRNAs as Prognostic Biomarkers in Prostate Cancer.

Degree: 2012, University of Toronto

Prostate cancer, one of the most common cancers among men, can be relatively harmless or extremely aggressive. The most widely used biomarker for the disease,… (more)

Subjects/Keywords: prostate cancer; MicroRNA

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gordanpour, A. (2012). MicroRNAs as Prognostic Biomarkers in Prostate Cancer. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/34019

Chicago Manual of Style (16th Edition):

Gordanpour, Aida. “MicroRNAs as Prognostic Biomarkers in Prostate Cancer.” 2012. Doctoral Dissertation, University of Toronto. Accessed December 10, 2018. http://hdl.handle.net/1807/34019.

MLA Handbook (7th Edition):

Gordanpour, Aida. “MicroRNAs as Prognostic Biomarkers in Prostate Cancer.” 2012. Web. 10 Dec 2018.

Vancouver:

Gordanpour A. MicroRNAs as Prognostic Biomarkers in Prostate Cancer. [Internet] [Doctoral dissertation]. University of Toronto; 2012. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/1807/34019.

Council of Science Editors:

Gordanpour A. MicroRNAs as Prognostic Biomarkers in Prostate Cancer. [Doctoral Dissertation]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/34019

28. Kulasingam, Vathany. Identification and Validation of Candidate Breast Cancer Biomarkers: A Mass Spectrometric Approach.

Degree: 2008, University of Toronto

One of the best ways to diagnose breast cancer early or to predict therapeutic response is to use serum biomarkers. Unfortunately, for breast cancer, we… (more)

Subjects/Keywords: proteomics; mass spectrometry; breast cancer; diagnostics; 0992

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kulasingam, V. (2008). Identification and Validation of Candidate Breast Cancer Biomarkers: A Mass Spectrometric Approach. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/32347

Chicago Manual of Style (16th Edition):

Kulasingam, Vathany. “Identification and Validation of Candidate Breast Cancer Biomarkers: A Mass Spectrometric Approach.” 2008. Doctoral Dissertation, University of Toronto. Accessed December 10, 2018. http://hdl.handle.net/1807/32347.

MLA Handbook (7th Edition):

Kulasingam, Vathany. “Identification and Validation of Candidate Breast Cancer Biomarkers: A Mass Spectrometric Approach.” 2008. Web. 10 Dec 2018.

Vancouver:

Kulasingam V. Identification and Validation of Candidate Breast Cancer Biomarkers: A Mass Spectrometric Approach. [Internet] [Doctoral dissertation]. University of Toronto; 2008. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/1807/32347.

Council of Science Editors:

Kulasingam V. Identification and Validation of Candidate Breast Cancer Biomarkers: A Mass Spectrometric Approach. [Doctoral Dissertation]. University of Toronto; 2008. Available from: http://hdl.handle.net/1807/32347

29. Petruzziello, Tania Nadia. Molecular Mechanisms of E. coli Shiga Toxin Pathogenesis.

Degree: 2012, University of Toronto

Shiga toxin-producing E. coli (STEC) comprise a group of pathogenic organisms that elaborate a family of protein exotoxins known as Shiga toxins (Stxs). Intestinal infection… (more)

Subjects/Keywords: Shiga toxin; endothelium; CXCR4; VEGF; SDF-1; O157:H7; E. coli; 0307; 0410; 0566

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Petruzziello, T. N. (2012). Molecular Mechanisms of E. coli Shiga Toxin Pathogenesis. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/32863

Chicago Manual of Style (16th Edition):

Petruzziello, Tania Nadia. “Molecular Mechanisms of E. coli Shiga Toxin Pathogenesis.” 2012. Doctoral Dissertation, University of Toronto. Accessed December 10, 2018. http://hdl.handle.net/1807/32863.

MLA Handbook (7th Edition):

Petruzziello, Tania Nadia. “Molecular Mechanisms of E. coli Shiga Toxin Pathogenesis.” 2012. Web. 10 Dec 2018.

Vancouver:

Petruzziello TN. Molecular Mechanisms of E. coli Shiga Toxin Pathogenesis. [Internet] [Doctoral dissertation]. University of Toronto; 2012. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/1807/32863.

Council of Science Editors:

Petruzziello TN. Molecular Mechanisms of E. coli Shiga Toxin Pathogenesis. [Doctoral Dissertation]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/32863

30. Garlick, Kristopher M. Interactions between the Actin Cytoskeleton and Anthrax Toxin Receptor 1 (ANTXR1).

Degree: 2012, University of Toronto

Interactions Between the Actin Cytoskeleton and Anthrax Toxin Receptor 1 (ANTXR1) Kristopher M. Garlick, Doctor of Philosophy, 2012, Department of Laboratory Medicine and Pathobiology, University(more)

Subjects/Keywords: anthrax; cytoskeleton

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Garlick, K. M. (2012). Interactions between the Actin Cytoskeleton and Anthrax Toxin Receptor 1 (ANTXR1). (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/34008

Chicago Manual of Style (16th Edition):

Garlick, Kristopher M. “Interactions between the Actin Cytoskeleton and Anthrax Toxin Receptor 1 (ANTXR1).” 2012. Doctoral Dissertation, University of Toronto. Accessed December 10, 2018. http://hdl.handle.net/1807/34008.

MLA Handbook (7th Edition):

Garlick, Kristopher M. “Interactions between the Actin Cytoskeleton and Anthrax Toxin Receptor 1 (ANTXR1).” 2012. Web. 10 Dec 2018.

Vancouver:

Garlick KM. Interactions between the Actin Cytoskeleton and Anthrax Toxin Receptor 1 (ANTXR1). [Internet] [Doctoral dissertation]. University of Toronto; 2012. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/1807/34008.

Council of Science Editors:

Garlick KM. Interactions between the Actin Cytoskeleton and Anthrax Toxin Receptor 1 (ANTXR1). [Doctoral Dissertation]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/34008

[1] [2] [3] [4] [5] … [12]

.