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You searched for +publisher:"University of Toronto" +contributor:("Immunology"). Showing records 1 – 30 of 175 total matches.

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University of Toronto

1. Shabab, Ali. Purification and Identification of Cell Surface Antigens using Lamprey Monoclonal Antibodies.

Degree: 2013, University of Toronto

The evolutionary distance of lampreys from humans in conjunction with their distinct antibody architecture is profound. Thus, lampreys may provide antibodies with specificity for antigens… (more)

Subjects/Keywords: VLR; lamprey antibody; biomaker discovery; antigen purification; antigen identification; novel human antigen; 0982

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APA (6th Edition):

Shabab, A. (2013). Purification and Identification of Cell Surface Antigens using Lamprey Monoclonal Antibodies. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/42666

Chicago Manual of Style (16th Edition):

Shabab, Ali. “Purification and Identification of Cell Surface Antigens using Lamprey Monoclonal Antibodies.” 2013. Masters Thesis, University of Toronto. Accessed October 25, 2020. http://hdl.handle.net/1807/42666.

MLA Handbook (7th Edition):

Shabab, Ali. “Purification and Identification of Cell Surface Antigens using Lamprey Monoclonal Antibodies.” 2013. Web. 25 Oct 2020.

Vancouver:

Shabab A. Purification and Identification of Cell Surface Antigens using Lamprey Monoclonal Antibodies. [Internet] [Masters thesis]. University of Toronto; 2013. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1807/42666.

Council of Science Editors:

Shabab A. Purification and Identification of Cell Surface Antigens using Lamprey Monoclonal Antibodies. [Masters Thesis]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/42666


University of Toronto

2. Minty, Gillian Eleanor Summersgill. The Role of TLR3 in the Development of Lupus-like Autoimmunity in B6.NZBc13 Mice.

Degree: 2013, University of Toronto

The New Zealand Black (NZB) mouse chromosome 13 (c13) is linked to development of autoimmunity. B6 mice containing a portion of NZBc13 (B6.NZBc13 (c13)) develop… (more)

Subjects/Keywords: Autoimmunity; mouse model; SLE; 0982

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APA (6th Edition):

Minty, G. E. S. (2013). The Role of TLR3 in the Development of Lupus-like Autoimmunity in B6.NZBc13 Mice. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/43245

Chicago Manual of Style (16th Edition):

Minty, Gillian Eleanor Summersgill. “The Role of TLR3 in the Development of Lupus-like Autoimmunity in B6.NZBc13 Mice.” 2013. Masters Thesis, University of Toronto. Accessed October 25, 2020. http://hdl.handle.net/1807/43245.

MLA Handbook (7th Edition):

Minty, Gillian Eleanor Summersgill. “The Role of TLR3 in the Development of Lupus-like Autoimmunity in B6.NZBc13 Mice.” 2013. Web. 25 Oct 2020.

Vancouver:

Minty GES. The Role of TLR3 in the Development of Lupus-like Autoimmunity in B6.NZBc13 Mice. [Internet] [Masters thesis]. University of Toronto; 2013. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1807/43245.

Council of Science Editors:

Minty GES. The Role of TLR3 in the Development of Lupus-like Autoimmunity in B6.NZBc13 Mice. [Masters Thesis]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/43245


University of Toronto

3. Sotov, Valentin. Crosstalk between VEGF and BMP9 Signalling in the Context of Preeclampsia.

Degree: 2013, University of Toronto

Preeclampsia is a pregnancy related disorder, characterized by proteinuria and hypertension. The pathogenesis of preeclampsia is poorly understood; however, two proteins, called sFlt-1 and sEng,… (more)

Subjects/Keywords: Preelcmapsia; soluble endoglin; endothelin-1; VEGF; 0982

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APA (6th Edition):

Sotov, V. (2013). Crosstalk between VEGF and BMP9 Signalling in the Context of Preeclampsia. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/42943

Chicago Manual of Style (16th Edition):

Sotov, Valentin. “Crosstalk between VEGF and BMP9 Signalling in the Context of Preeclampsia.” 2013. Masters Thesis, University of Toronto. Accessed October 25, 2020. http://hdl.handle.net/1807/42943.

MLA Handbook (7th Edition):

Sotov, Valentin. “Crosstalk between VEGF and BMP9 Signalling in the Context of Preeclampsia.” 2013. Web. 25 Oct 2020.

Vancouver:

Sotov V. Crosstalk between VEGF and BMP9 Signalling in the Context of Preeclampsia. [Internet] [Masters thesis]. University of Toronto; 2013. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1807/42943.

Council of Science Editors:

Sotov V. Crosstalk between VEGF and BMP9 Signalling in the Context of Preeclampsia. [Masters Thesis]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/42943


University of Toronto

4. Lifeso, Kimberley. Creation of a Retroviral RNAi Knockdown System to Investigate Gene Variants in SLE.

Degree: 2013, University of Toronto

Systemic lupus erythematosus (SLE) is an autoimmune disease characterized by the production of autoantibodies against self-antigens. SLE has a complex multifactorial genetic basis. Genome wide… (more)

Subjects/Keywords: Immunology; Systemic Lupus Erythematosus; 0982

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APA (6th Edition):

Lifeso, K. (2013). Creation of a Retroviral RNAi Knockdown System to Investigate Gene Variants in SLE. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/43083

Chicago Manual of Style (16th Edition):

Lifeso, Kimberley. “Creation of a Retroviral RNAi Knockdown System to Investigate Gene Variants in SLE.” 2013. Masters Thesis, University of Toronto. Accessed October 25, 2020. http://hdl.handle.net/1807/43083.

MLA Handbook (7th Edition):

Lifeso, Kimberley. “Creation of a Retroviral RNAi Knockdown System to Investigate Gene Variants in SLE.” 2013. Web. 25 Oct 2020.

Vancouver:

Lifeso K. Creation of a Retroviral RNAi Knockdown System to Investigate Gene Variants in SLE. [Internet] [Masters thesis]. University of Toronto; 2013. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1807/43083.

Council of Science Editors:

Lifeso K. Creation of a Retroviral RNAi Knockdown System to Investigate Gene Variants in SLE. [Masters Thesis]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/43083


University of Toronto

5. Chan, Kevin Daniel. The Role of the Gut Microbiome in Modulating the Pathogenesis of Kawasaki Disease.

Degree: 2014, University of Toronto

The gut microbiome consists of trillions of microorganisms in constant interaction with the body. There is increasing evidence that these microbes have immunomodulatory effects and… (more)

Subjects/Keywords: Gut Microbiome; Kawasaki Disease; LCWE; SFB; 0982

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APA (6th Edition):

Chan, K. D. (2014). The Role of the Gut Microbiome in Modulating the Pathogenesis of Kawasaki Disease. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/67897

Chicago Manual of Style (16th Edition):

Chan, Kevin Daniel. “The Role of the Gut Microbiome in Modulating the Pathogenesis of Kawasaki Disease.” 2014. Masters Thesis, University of Toronto. Accessed October 25, 2020. http://hdl.handle.net/1807/67897.

MLA Handbook (7th Edition):

Chan, Kevin Daniel. “The Role of the Gut Microbiome in Modulating the Pathogenesis of Kawasaki Disease.” 2014. Web. 25 Oct 2020.

Vancouver:

Chan KD. The Role of the Gut Microbiome in Modulating the Pathogenesis of Kawasaki Disease. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1807/67897.

Council of Science Editors:

Chan KD. The Role of the Gut Microbiome in Modulating the Pathogenesis of Kawasaki Disease. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/67897


University of Toronto

6. Lee, Korris. Dynamic Regulation of CD8+ T cell Signalling by the Adaptor Protein 3BP2.

Degree: 2014, University of Toronto

AbstractDynamic Regulation of CD8+ T Cells by the Adaptor Protein 3BP2Korris Lee © 2014 MSc Department of Immunology, University of TorontoCD8+ T cells fill an… (more)

Subjects/Keywords: 0982

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APA (6th Edition):

Lee, K. (2014). Dynamic Regulation of CD8+ T cell Signalling by the Adaptor Protein 3BP2. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/67901

Chicago Manual of Style (16th Edition):

Lee, Korris. “Dynamic Regulation of CD8+ T cell Signalling by the Adaptor Protein 3BP2.” 2014. Masters Thesis, University of Toronto. Accessed October 25, 2020. http://hdl.handle.net/1807/67901.

MLA Handbook (7th Edition):

Lee, Korris. “Dynamic Regulation of CD8+ T cell Signalling by the Adaptor Protein 3BP2.” 2014. Web. 25 Oct 2020.

Vancouver:

Lee K. Dynamic Regulation of CD8+ T cell Signalling by the Adaptor Protein 3BP2. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1807/67901.

Council of Science Editors:

Lee K. Dynamic Regulation of CD8+ T cell Signalling by the Adaptor Protein 3BP2. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/67901


University of Toronto

7. Zhou, Jun Yu. Characterization of NLRC3 and its Mechanism of Action in Regulating T cell Function and Activation.

Degree: 2014, University of Toronto

NLRC3 is a newly discovered Nod-like receptor with immune regulatory functions. Strikingly, NLRC3 is highly expressed in lymphocytes, with the highest expression in T cells.… (more)

Subjects/Keywords: NLRC3; NOD-like receptor; T cell biology; T cell function; 0982

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APA (6th Edition):

Zhou, J. Y. (2014). Characterization of NLRC3 and its Mechanism of Action in Regulating T cell Function and Activation. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/68569

Chicago Manual of Style (16th Edition):

Zhou, Jun Yu. “Characterization of NLRC3 and its Mechanism of Action in Regulating T cell Function and Activation.” 2014. Masters Thesis, University of Toronto. Accessed October 25, 2020. http://hdl.handle.net/1807/68569.

MLA Handbook (7th Edition):

Zhou, Jun Yu. “Characterization of NLRC3 and its Mechanism of Action in Regulating T cell Function and Activation.” 2014. Web. 25 Oct 2020.

Vancouver:

Zhou JY. Characterization of NLRC3 and its Mechanism of Action in Regulating T cell Function and Activation. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1807/68569.

Council of Science Editors:

Zhou JY. Characterization of NLRC3 and its Mechanism of Action in Regulating T cell Function and Activation. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/68569


University of Toronto

8. Kim, Nam Yun. Characterization of the Immune Basis for Reduced IFN-α in Serologically Active Clinically Quiescent SLE Patients.

Degree: 2015, University of Toronto

Serologically active clinically quiescent (SACQ) patients with systemic lupus erythematosus (SLE) are clinically quiescent despite the presence of autoantibodies. SACQ patients differ from serologically active… (more)

Subjects/Keywords: Interferon; SACQ; SLE; 0982

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APA (6th Edition):

Kim, N. Y. (2015). Characterization of the Immune Basis for Reduced IFN-α in Serologically Active Clinically Quiescent SLE Patients. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/70417

Chicago Manual of Style (16th Edition):

Kim, Nam Yun. “Characterization of the Immune Basis for Reduced IFN-α in Serologically Active Clinically Quiescent SLE Patients.” 2015. Masters Thesis, University of Toronto. Accessed October 25, 2020. http://hdl.handle.net/1807/70417.

MLA Handbook (7th Edition):

Kim, Nam Yun. “Characterization of the Immune Basis for Reduced IFN-α in Serologically Active Clinically Quiescent SLE Patients.” 2015. Web. 25 Oct 2020.

Vancouver:

Kim NY. Characterization of the Immune Basis for Reduced IFN-α in Serologically Active Clinically Quiescent SLE Patients. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1807/70417.

Council of Science Editors:

Kim NY. Characterization of the Immune Basis for Reduced IFN-α in Serologically Active Clinically Quiescent SLE Patients. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/70417


University of Toronto

9. Xiang, Shang. Establishment of a Working Model for the Study of HEB Transcription Factor Activity and Elucidation of the Function of the Alternative Domain of HEBAlt.

Degree: 2015, University of Toronto

T cell development in the thymus occurs in a series of tightly regulated stages and is controlled by a complex network of transcription factors. E… (more)

Subjects/Keywords: E proteins; HEBAlt; HEBCan; HEB factors; T cell development; transcription factor; 0982

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APA (6th Edition):

Xiang, S. (2015). Establishment of a Working Model for the Study of HEB Transcription Factor Activity and Elucidation of the Function of the Alternative Domain of HEBAlt. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/70733

Chicago Manual of Style (16th Edition):

Xiang, Shang. “Establishment of a Working Model for the Study of HEB Transcription Factor Activity and Elucidation of the Function of the Alternative Domain of HEBAlt.” 2015. Masters Thesis, University of Toronto. Accessed October 25, 2020. http://hdl.handle.net/1807/70733.

MLA Handbook (7th Edition):

Xiang, Shang. “Establishment of a Working Model for the Study of HEB Transcription Factor Activity and Elucidation of the Function of the Alternative Domain of HEBAlt.” 2015. Web. 25 Oct 2020.

Vancouver:

Xiang S. Establishment of a Working Model for the Study of HEB Transcription Factor Activity and Elucidation of the Function of the Alternative Domain of HEBAlt. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1807/70733.

Council of Science Editors:

Xiang S. Establishment of a Working Model for the Study of HEB Transcription Factor Activity and Elucidation of the Function of the Alternative Domain of HEBAlt. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/70733


University of Toronto

10. Cho, Lindsay Kristin. The Role of Circulating Mitochondrial DNA in Juvenile Idiopathic Arthritis.

Degree: 2016, University of Toronto

Previous studies have demonstrated mitochondrial DNA (mtDNA) activates Toll-like receptor-9 signaling, and may activate inflammasomes leading to the release of pro-inflammatory cytokine IL-1b. Chronic inflammation… (more)

Subjects/Keywords: IL-1 beta; Juvenile Idiopathic Arthritis; macrophages; mitochondrial DNA; 0982

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APA (6th Edition):

Cho, L. K. (2016). The Role of Circulating Mitochondrial DNA in Juvenile Idiopathic Arthritis. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/74567

Chicago Manual of Style (16th Edition):

Cho, Lindsay Kristin. “The Role of Circulating Mitochondrial DNA in Juvenile Idiopathic Arthritis.” 2016. Masters Thesis, University of Toronto. Accessed October 25, 2020. http://hdl.handle.net/1807/74567.

MLA Handbook (7th Edition):

Cho, Lindsay Kristin. “The Role of Circulating Mitochondrial DNA in Juvenile Idiopathic Arthritis.” 2016. Web. 25 Oct 2020.

Vancouver:

Cho LK. The Role of Circulating Mitochondrial DNA in Juvenile Idiopathic Arthritis. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1807/74567.

Council of Science Editors:

Cho LK. The Role of Circulating Mitochondrial DNA in Juvenile Idiopathic Arthritis. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/74567


University of Toronto

11. Lam Yau, Christopher Yick. The Impact of Microbiome Alterations in the NOD Mouse Model of Type 1 Diabetes.

Degree: 2014, University of Toronto

Epidemiological and experimental data suggest Type 1 Diabetes (T1D) and gut inflammatory disorders share genetic and environmental risk factors. Genetics and microbiome composition differentiate T1D-prone… (more)

Subjects/Keywords: Microbiota; NOD mouse; Type 1 Diabetes; 0982

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APA (6th Edition):

Lam Yau, C. Y. (2014). The Impact of Microbiome Alterations in the NOD Mouse Model of Type 1 Diabetes. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/74623

Chicago Manual of Style (16th Edition):

Lam Yau, Christopher Yick. “The Impact of Microbiome Alterations in the NOD Mouse Model of Type 1 Diabetes.” 2014. Masters Thesis, University of Toronto. Accessed October 25, 2020. http://hdl.handle.net/1807/74623.

MLA Handbook (7th Edition):

Lam Yau, Christopher Yick. “The Impact of Microbiome Alterations in the NOD Mouse Model of Type 1 Diabetes.” 2014. Web. 25 Oct 2020.

Vancouver:

Lam Yau CY. The Impact of Microbiome Alterations in the NOD Mouse Model of Type 1 Diabetes. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1807/74623.

Council of Science Editors:

Lam Yau CY. The Impact of Microbiome Alterations in the NOD Mouse Model of Type 1 Diabetes. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/74623


University of Toronto

12. Mikhaylov, Andrey Olegovich. Integrating Biologic and Clinical Data towards Resolving Heterogeneity in Childhood Inflammatory Diseases.

Degree: 2016, University of Toronto

Kawasaki disease (KD) is the leading cause of acquired heart disease in children from the developed world, with up to 25% risk of developing aneurysms… (more)

Subjects/Keywords: Kawasaki Disease; Similarity Network Fusion; 0982

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APA (6th Edition):

Mikhaylov, A. O. (2016). Integrating Biologic and Clinical Data towards Resolving Heterogeneity in Childhood Inflammatory Diseases. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/75362

Chicago Manual of Style (16th Edition):

Mikhaylov, Andrey Olegovich. “Integrating Biologic and Clinical Data towards Resolving Heterogeneity in Childhood Inflammatory Diseases.” 2016. Masters Thesis, University of Toronto. Accessed October 25, 2020. http://hdl.handle.net/1807/75362.

MLA Handbook (7th Edition):

Mikhaylov, Andrey Olegovich. “Integrating Biologic and Clinical Data towards Resolving Heterogeneity in Childhood Inflammatory Diseases.” 2016. Web. 25 Oct 2020.

Vancouver:

Mikhaylov AO. Integrating Biologic and Clinical Data towards Resolving Heterogeneity in Childhood Inflammatory Diseases. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1807/75362.

Council of Science Editors:

Mikhaylov AO. Integrating Biologic and Clinical Data towards Resolving Heterogeneity in Childhood Inflammatory Diseases. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/75362


University of Toronto

13. Oke, Sofia. Msh2-deficiency Affects the Differentiation and Proliferation of Small Intestinal Epithelial Cells.

Degree: 2016, University of Toronto

Mutations in mismatch repair genes, such as MSH2, are common lesions in Hereditary Nonpolyposis Colorectal Cancer and approximately 15% of all sporadic human colorectal cancers.… (more)

Subjects/Keywords: Butyrate; Colorectal Cancer; Glucose; Mismatch repair; Msh2; Organoids; 0982

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APA (6th Edition):

Oke, S. (2016). Msh2-deficiency Affects the Differentiation and Proliferation of Small Intestinal Epithelial Cells. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/75823

Chicago Manual of Style (16th Edition):

Oke, Sofia. “Msh2-deficiency Affects the Differentiation and Proliferation of Small Intestinal Epithelial Cells.” 2016. Masters Thesis, University of Toronto. Accessed October 25, 2020. http://hdl.handle.net/1807/75823.

MLA Handbook (7th Edition):

Oke, Sofia. “Msh2-deficiency Affects the Differentiation and Proliferation of Small Intestinal Epithelial Cells.” 2016. Web. 25 Oct 2020.

Vancouver:

Oke S. Msh2-deficiency Affects the Differentiation and Proliferation of Small Intestinal Epithelial Cells. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1807/75823.

Council of Science Editors:

Oke S. Msh2-deficiency Affects the Differentiation and Proliferation of Small Intestinal Epithelial Cells. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/75823


University of Toronto

14. Cen, Selena Yunhua. Differential Regulation of IgA+ B Cells in Vitro by Stromal Cells from Distinctive Anatomical Compartments.

Degree: 2017, University of Toronto

B cell development is regulated by stromal cells that form a supportive microenvironment. Bone marrow (BM), spleen (Sp), and the gut lamina propria (LP) constitute… (more)

Subjects/Keywords: B cells; gut; IgA cells; plasma cells; stromal cells; 0982

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APA (6th Edition):

Cen, S. Y. (2017). Differential Regulation of IgA+ B Cells in Vitro by Stromal Cells from Distinctive Anatomical Compartments. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/77768

Chicago Manual of Style (16th Edition):

Cen, Selena Yunhua. “Differential Regulation of IgA+ B Cells in Vitro by Stromal Cells from Distinctive Anatomical Compartments.” 2017. Masters Thesis, University of Toronto. Accessed October 25, 2020. http://hdl.handle.net/1807/77768.

MLA Handbook (7th Edition):

Cen, Selena Yunhua. “Differential Regulation of IgA+ B Cells in Vitro by Stromal Cells from Distinctive Anatomical Compartments.” 2017. Web. 25 Oct 2020.

Vancouver:

Cen SY. Differential Regulation of IgA+ B Cells in Vitro by Stromal Cells from Distinctive Anatomical Compartments. [Internet] [Masters thesis]. University of Toronto; 2017. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1807/77768.

Council of Science Editors:

Cen SY. Differential Regulation of IgA+ B Cells in Vitro by Stromal Cells from Distinctive Anatomical Compartments. [Masters Thesis]. University of Toronto; 2017. Available from: http://hdl.handle.net/1807/77768


University of Toronto

15. Green, Blerta. The Mismatch Repair Pathway Functions Normally at a non-AID Target in Germinal Center B cells.

Degree: 2011, University of Toronto

Deficiency in Msh2, a component of the mismatch repair (MMR) system, leads to a ~10-fold increase in the mutation frequency in most tissues. By contrast,… (more)

Subjects/Keywords: mismatch repair; somatic hypermutation; germinal center; activation induced cytidine deaminase; B cells; mutations; 0982

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APA (6th Edition):

Green, B. (2011). The Mismatch Repair Pathway Functions Normally at a non-AID Target in Germinal Center B cells. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/30614

Chicago Manual of Style (16th Edition):

Green, Blerta. “The Mismatch Repair Pathway Functions Normally at a non-AID Target in Germinal Center B cells.” 2011. Masters Thesis, University of Toronto. Accessed October 25, 2020. http://hdl.handle.net/1807/30614.

MLA Handbook (7th Edition):

Green, Blerta. “The Mismatch Repair Pathway Functions Normally at a non-AID Target in Germinal Center B cells.” 2011. Web. 25 Oct 2020.

Vancouver:

Green B. The Mismatch Repair Pathway Functions Normally at a non-AID Target in Germinal Center B cells. [Internet] [Masters thesis]. University of Toronto; 2011. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1807/30614.

Council of Science Editors:

Green B. The Mismatch Repair Pathway Functions Normally at a non-AID Target in Germinal Center B cells. [Masters Thesis]. University of Toronto; 2011. Available from: http://hdl.handle.net/1807/30614


University of Toronto

16. Farr, Christina. Calpain and Calpastatin in a Mouse Model of Acute Myeloid Leukemia.

Degree: 2011, University of Toronto

I have studied the calpain system in acute myeloid leukemia using the 32D and 32Dkit cell lines. Specifically, I characterized the calpain system in the… (more)

Subjects/Keywords: calpain; acute myeloid leukemia; 0982

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APA (6th Edition):

Farr, C. (2011). Calpain and Calpastatin in a Mouse Model of Acute Myeloid Leukemia. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/30589

Chicago Manual of Style (16th Edition):

Farr, Christina. “Calpain and Calpastatin in a Mouse Model of Acute Myeloid Leukemia.” 2011. Masters Thesis, University of Toronto. Accessed October 25, 2020. http://hdl.handle.net/1807/30589.

MLA Handbook (7th Edition):

Farr, Christina. “Calpain and Calpastatin in a Mouse Model of Acute Myeloid Leukemia.” 2011. Web. 25 Oct 2020.

Vancouver:

Farr C. Calpain and Calpastatin in a Mouse Model of Acute Myeloid Leukemia. [Internet] [Masters thesis]. University of Toronto; 2011. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1807/30589.

Council of Science Editors:

Farr C. Calpain and Calpastatin in a Mouse Model of Acute Myeloid Leukemia. [Masters Thesis]. University of Toronto; 2011. Available from: http://hdl.handle.net/1807/30589


University of Toronto

17. Khattar, Ramzi. Targeted Deletion of Fibrinogen-like Protein 2 (FGL2) ENHANCES Immunity in a Murine Model of Acute Viral Hepatitis Caused by Lymphocytic Choriomeningitis Virus (LCMV).

Degree: 2011, University of Toronto

Viral hepatitis infection represents a significant epidemiological and economic burden on society. Following infection, some patients mount a blunted immune response to the virus, which… (more)

Subjects/Keywords: LCMV; HCV pathogenesis; 0982

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APA (6th Edition):

Khattar, R. (2011). Targeted Deletion of Fibrinogen-like Protein 2 (FGL2) ENHANCES Immunity in a Murine Model of Acute Viral Hepatitis Caused by Lymphocytic Choriomeningitis Virus (LCMV). (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/31278

Chicago Manual of Style (16th Edition):

Khattar, Ramzi. “Targeted Deletion of Fibrinogen-like Protein 2 (FGL2) ENHANCES Immunity in a Murine Model of Acute Viral Hepatitis Caused by Lymphocytic Choriomeningitis Virus (LCMV).” 2011. Masters Thesis, University of Toronto. Accessed October 25, 2020. http://hdl.handle.net/1807/31278.

MLA Handbook (7th Edition):

Khattar, Ramzi. “Targeted Deletion of Fibrinogen-like Protein 2 (FGL2) ENHANCES Immunity in a Murine Model of Acute Viral Hepatitis Caused by Lymphocytic Choriomeningitis Virus (LCMV).” 2011. Web. 25 Oct 2020.

Vancouver:

Khattar R. Targeted Deletion of Fibrinogen-like Protein 2 (FGL2) ENHANCES Immunity in a Murine Model of Acute Viral Hepatitis Caused by Lymphocytic Choriomeningitis Virus (LCMV). [Internet] [Masters thesis]. University of Toronto; 2011. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1807/31278.

Council of Science Editors:

Khattar R. Targeted Deletion of Fibrinogen-like Protein 2 (FGL2) ENHANCES Immunity in a Murine Model of Acute Viral Hepatitis Caused by Lymphocytic Choriomeningitis Virus (LCMV). [Masters Thesis]. University of Toronto; 2011. Available from: http://hdl.handle.net/1807/31278


University of Toronto

18. Gregory, Allison. Structural and Functional Characteristics of a Soluble Form of Endoglin in the Context of Preeclampsia.

Degree: 2011, University of Toronto

Endoglin is an auxiliary receptor for ligands of TGF-β receptor superfamily, present in endothelial cells and the placental syncytiotrophoblast. The expression of placental membrane endoglin… (more)

Subjects/Keywords: preeclampsia; endoglin; 0982

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APA (6th Edition):

Gregory, A. (2011). Structural and Functional Characteristics of a Soluble Form of Endoglin in the Context of Preeclampsia. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/30615

Chicago Manual of Style (16th Edition):

Gregory, Allison. “Structural and Functional Characteristics of a Soluble Form of Endoglin in the Context of Preeclampsia.” 2011. Masters Thesis, University of Toronto. Accessed October 25, 2020. http://hdl.handle.net/1807/30615.

MLA Handbook (7th Edition):

Gregory, Allison. “Structural and Functional Characteristics of a Soluble Form of Endoglin in the Context of Preeclampsia.” 2011. Web. 25 Oct 2020.

Vancouver:

Gregory A. Structural and Functional Characteristics of a Soluble Form of Endoglin in the Context of Preeclampsia. [Internet] [Masters thesis]. University of Toronto; 2011. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1807/30615.

Council of Science Editors:

Gregory A. Structural and Functional Characteristics of a Soluble Form of Endoglin in the Context of Preeclampsia. [Masters Thesis]. University of Toronto; 2011. Available from: http://hdl.handle.net/1807/30615


University of Toronto

19. Peter, Madonna. The Role of Endoglin in the Resolution of Inflammation.

Degree: 2012, University of Toronto

Endoglin, a co-receptor of the TGF-β superfamily, is predominantly expressed in endothelial cells and in some myeloid cells and implicated as a potential modulator of… (more)

Subjects/Keywords: Inflammation; Inflammatory bowel disease; Endoglin; Transforming growth factor beta superfamily; Angiogenesis; Neutrophil; 0306; 0307; 0379

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APA (6th Edition):

Peter, M. (2012). The Role of Endoglin in the Resolution of Inflammation. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/33490

Chicago Manual of Style (16th Edition):

Peter, Madonna. “The Role of Endoglin in the Resolution of Inflammation.” 2012. Masters Thesis, University of Toronto. Accessed October 25, 2020. http://hdl.handle.net/1807/33490.

MLA Handbook (7th Edition):

Peter, Madonna. “The Role of Endoglin in the Resolution of Inflammation.” 2012. Web. 25 Oct 2020.

Vancouver:

Peter M. The Role of Endoglin in the Resolution of Inflammation. [Internet] [Masters thesis]. University of Toronto; 2012. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1807/33490.

Council of Science Editors:

Peter M. The Role of Endoglin in the Resolution of Inflammation. [Masters Thesis]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/33490


University of Toronto

20. Farrokhi, Kaveh. Fibrinogen Like Protein 2 (FGL2): A Novel Regulator of Macrophage M1 Polarization.

Degree: 2018, University of Toronto

Fibrinogen-like protein 2 (FGL2) is a potent immunosuppressive molecule. The effects of FGL2 on macrophages, however, has not been studied. Peritoneal cell accumulation in fgl2-/-,… (more)

Subjects/Keywords: FGL2; Immunology; Macrophage; Polarization; 0982

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APA (6th Edition):

Farrokhi, K. (2018). Fibrinogen Like Protein 2 (FGL2): A Novel Regulator of Macrophage M1 Polarization. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/89550

Chicago Manual of Style (16th Edition):

Farrokhi, Kaveh. “Fibrinogen Like Protein 2 (FGL2): A Novel Regulator of Macrophage M1 Polarization.” 2018. Masters Thesis, University of Toronto. Accessed October 25, 2020. http://hdl.handle.net/1807/89550.

MLA Handbook (7th Edition):

Farrokhi, Kaveh. “Fibrinogen Like Protein 2 (FGL2): A Novel Regulator of Macrophage M1 Polarization.” 2018. Web. 25 Oct 2020.

Vancouver:

Farrokhi K. Fibrinogen Like Protein 2 (FGL2): A Novel Regulator of Macrophage M1 Polarization. [Internet] [Masters thesis]. University of Toronto; 2018. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1807/89550.

Council of Science Editors:

Farrokhi K. Fibrinogen Like Protein 2 (FGL2): A Novel Regulator of Macrophage M1 Polarization. [Masters Thesis]. University of Toronto; 2018. Available from: http://hdl.handle.net/1807/89550


University of Toronto

21. Wang, Kuan Chung. Dichotomy of TNF Family Ligands Expression on Classical Dendritic Cells and Monocyte-derived Antigen Presenting Cells During Viral Infection.

Degree: 2018, University of Toronto

Tumor necrosis factor receptor (TNFR) superfamily members, including 4-1BB, OX40, GITR and CD27 contribute to T cell survival during viral infection. However, the cell types… (more)

Subjects/Keywords: Antigen presenting cell; B7 family ligands; lymphoytic choriomeningitis (LCMV) clone 13 infection; Tumor necrosis factor receptor (TNFR) superfamily members; type II interferon; type I interferon; 0982

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APA (6th Edition):

Wang, K. C. (2018). Dichotomy of TNF Family Ligands Expression on Classical Dendritic Cells and Monocyte-derived Antigen Presenting Cells During Viral Infection. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/91345

Chicago Manual of Style (16th Edition):

Wang, Kuan Chung. “Dichotomy of TNF Family Ligands Expression on Classical Dendritic Cells and Monocyte-derived Antigen Presenting Cells During Viral Infection.” 2018. Masters Thesis, University of Toronto. Accessed October 25, 2020. http://hdl.handle.net/1807/91345.

MLA Handbook (7th Edition):

Wang, Kuan Chung. “Dichotomy of TNF Family Ligands Expression on Classical Dendritic Cells and Monocyte-derived Antigen Presenting Cells During Viral Infection.” 2018. Web. 25 Oct 2020.

Vancouver:

Wang KC. Dichotomy of TNF Family Ligands Expression on Classical Dendritic Cells and Monocyte-derived Antigen Presenting Cells During Viral Infection. [Internet] [Masters thesis]. University of Toronto; 2018. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1807/91345.

Council of Science Editors:

Wang KC. Dichotomy of TNF Family Ligands Expression on Classical Dendritic Cells and Monocyte-derived Antigen Presenting Cells During Viral Infection. [Masters Thesis]. University of Toronto; 2018. Available from: http://hdl.handle.net/1807/91345


University of Toronto

22. Yao, YuChen. Altered CD8+ T cell function in Ankylosing Spondylitis: The role of perforin in enhanced inflammatory profiles.

Degree: 2018, University of Toronto

Ankylosing Spondylitis (AS) is an autoimmune disease of still unknown pathogenesis and disease mechanism. Currently the most well-known genetic factor associated with AS is one… (more)

Subjects/Keywords: Ankylosing Spondylitis; Arthritis; CD8 T cell; Immunology; Inflmmation; Perforin; 0982

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APA (6th Edition):

Yao, Y. (2018). Altered CD8+ T cell function in Ankylosing Spondylitis: The role of perforin in enhanced inflammatory profiles. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/91354

Chicago Manual of Style (16th Edition):

Yao, YuChen. “Altered CD8+ T cell function in Ankylosing Spondylitis: The role of perforin in enhanced inflammatory profiles.” 2018. Masters Thesis, University of Toronto. Accessed October 25, 2020. http://hdl.handle.net/1807/91354.

MLA Handbook (7th Edition):

Yao, YuChen. “Altered CD8+ T cell function in Ankylosing Spondylitis: The role of perforin in enhanced inflammatory profiles.” 2018. Web. 25 Oct 2020.

Vancouver:

Yao Y. Altered CD8+ T cell function in Ankylosing Spondylitis: The role of perforin in enhanced inflammatory profiles. [Internet] [Masters thesis]. University of Toronto; 2018. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1807/91354.

Council of Science Editors:

Yao Y. Altered CD8+ T cell function in Ankylosing Spondylitis: The role of perforin in enhanced inflammatory profiles. [Masters Thesis]. University of Toronto; 2018. Available from: http://hdl.handle.net/1807/91354


University of Toronto

23. Umana, Juan Mauricio. Development of TCR-engineered iNKT cells for Cancer Immunotherapy.

Degree: 2018, University of Toronto

Invariant Natural Killer T (iNKT) cells are innate T cells that respond to lipid antigens presented by the molecule CD1d. Activated iNKT cells rapidly produce… (more)

Subjects/Keywords: adoptive cell therapy; cancer; Immunotherapy; iNKT cells; innate immuninty; TCR-engineering; 0982

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APA (6th Edition):

Umana, J. M. (2018). Development of TCR-engineered iNKT cells for Cancer Immunotherapy. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/91393

Chicago Manual of Style (16th Edition):

Umana, Juan Mauricio. “Development of TCR-engineered iNKT cells for Cancer Immunotherapy.” 2018. Masters Thesis, University of Toronto. Accessed October 25, 2020. http://hdl.handle.net/1807/91393.

MLA Handbook (7th Edition):

Umana, Juan Mauricio. “Development of TCR-engineered iNKT cells for Cancer Immunotherapy.” 2018. Web. 25 Oct 2020.

Vancouver:

Umana JM. Development of TCR-engineered iNKT cells for Cancer Immunotherapy. [Internet] [Masters thesis]. University of Toronto; 2018. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1807/91393.

Council of Science Editors:

Umana JM. Development of TCR-engineered iNKT cells for Cancer Immunotherapy. [Masters Thesis]. University of Toronto; 2018. Available from: http://hdl.handle.net/1807/91393


University of Toronto

24. Shen, Jieli. Impact of the Innate Immune Checkpoint Inhibitor SIRPα-Fc on Immune Function.

Degree: 2018, University of Toronto

Signal regulatory protein α (SIRPα) is an inhibitory receptor expressed on myeloid cells that functions as a “healthy self-recognition” receptor by engagement with its ubiquitously… (more)

Subjects/Keywords: CD47; Immunotherapy; SIRPa; SIRPα; SIRPα-Fc; 0982

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APA (6th Edition):

Shen, J. (2018). Impact of the Innate Immune Checkpoint Inhibitor SIRPα-Fc on Immune Function. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/91613

Chicago Manual of Style (16th Edition):

Shen, Jieli. “Impact of the Innate Immune Checkpoint Inhibitor SIRPα-Fc on Immune Function.” 2018. Masters Thesis, University of Toronto. Accessed October 25, 2020. http://hdl.handle.net/1807/91613.

MLA Handbook (7th Edition):

Shen, Jieli. “Impact of the Innate Immune Checkpoint Inhibitor SIRPα-Fc on Immune Function.” 2018. Web. 25 Oct 2020.

Vancouver:

Shen J. Impact of the Innate Immune Checkpoint Inhibitor SIRPα-Fc on Immune Function. [Internet] [Masters thesis]. University of Toronto; 2018. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1807/91613.

Council of Science Editors:

Shen J. Impact of the Innate Immune Checkpoint Inhibitor SIRPα-Fc on Immune Function. [Masters Thesis]. University of Toronto; 2018. Available from: http://hdl.handle.net/1807/91613


University of Toronto

25. Chang, Yu-Han. Cell Specific Expression, Regulation and Consequences of GITRL Induction in a Persistent Viral Infection.

Degree: 2016, University of Toronto

Co-stimulation of T cells through Glucocorticoid-Induced Tumour Necrosis Factor Receptor-Related Protein (GITR) contributes to viral control during persistent lymphocytic choriomeningitis virus (LCMV) infection. Herein, we… (more)

Subjects/Keywords: GITR; GITRL; LCMV; TNFRSF18; TNFSF18; Type I Interferon; 0982

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APA (6th Edition):

Chang, Y. (2016). Cell Specific Expression, Regulation and Consequences of GITRL Induction in a Persistent Viral Infection. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/91899

Chicago Manual of Style (16th Edition):

Chang, Yu-Han. “Cell Specific Expression, Regulation and Consequences of GITRL Induction in a Persistent Viral Infection.” 2016. Masters Thesis, University of Toronto. Accessed October 25, 2020. http://hdl.handle.net/1807/91899.

MLA Handbook (7th Edition):

Chang, Yu-Han. “Cell Specific Expression, Regulation and Consequences of GITRL Induction in a Persistent Viral Infection.” 2016. Web. 25 Oct 2020.

Vancouver:

Chang Y. Cell Specific Expression, Regulation and Consequences of GITRL Induction in a Persistent Viral Infection. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1807/91899.

Council of Science Editors:

Chang Y. Cell Specific Expression, Regulation and Consequences of GITRL Induction in a Persistent Viral Infection. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/91899


University of Toronto

26. Yam, Jennifer Yuen-Man. Exploring the Role of CCR6/CCL20 Axis in B Cell Migration into the CNS during EAE.

Degree: 2016, University of Toronto

B cells have been implicated in the pathogenesis of multiple sclerosis (MS) but how they migrate into the central nervous system (CNS) is poorly understood.… (more)

Subjects/Keywords: 0982

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APA (6th Edition):

Yam, J. Y. (2016). Exploring the Role of CCR6/CCL20 Axis in B Cell Migration into the CNS during EAE. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/92618

Chicago Manual of Style (16th Edition):

Yam, Jennifer Yuen-Man. “Exploring the Role of CCR6/CCL20 Axis in B Cell Migration into the CNS during EAE.” 2016. Masters Thesis, University of Toronto. Accessed October 25, 2020. http://hdl.handle.net/1807/92618.

MLA Handbook (7th Edition):

Yam, Jennifer Yuen-Man. “Exploring the Role of CCR6/CCL20 Axis in B Cell Migration into the CNS during EAE.” 2016. Web. 25 Oct 2020.

Vancouver:

Yam JY. Exploring the Role of CCR6/CCL20 Axis in B Cell Migration into the CNS during EAE. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1807/92618.

Council of Science Editors:

Yam JY. Exploring the Role of CCR6/CCL20 Axis in B Cell Migration into the CNS during EAE. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/92618


University of Toronto

27. Ghazarian, Magar Danny. Type I Interferon Responses Drive Intrahepatic T cells to Promote Metabolic Syndrome.

Degree: 2016, University of Toronto

Obesity-related insulin resistance is driven by low-grade chronic inflammation of metabolic tissues mediated by innate and adaptive immunity. During obesity, the liver undergoes steatosis, leading… (more)

Subjects/Keywords: Adaptive Immunity; Immunology; Insulin Resistance; Metabolic Syndrome; Non-alcoholic Fatty Liver Disease; Obesity; 0982

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APA (6th Edition):

Ghazarian, M. D. (2016). Type I Interferon Responses Drive Intrahepatic T cells to Promote Metabolic Syndrome. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/92686

Chicago Manual of Style (16th Edition):

Ghazarian, Magar Danny. “Type I Interferon Responses Drive Intrahepatic T cells to Promote Metabolic Syndrome.” 2016. Masters Thesis, University of Toronto. Accessed October 25, 2020. http://hdl.handle.net/1807/92686.

MLA Handbook (7th Edition):

Ghazarian, Magar Danny. “Type I Interferon Responses Drive Intrahepatic T cells to Promote Metabolic Syndrome.” 2016. Web. 25 Oct 2020.

Vancouver:

Ghazarian MD. Type I Interferon Responses Drive Intrahepatic T cells to Promote Metabolic Syndrome. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1807/92686.

Council of Science Editors:

Ghazarian MD. Type I Interferon Responses Drive Intrahepatic T cells to Promote Metabolic Syndrome. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/92686


University of Toronto

28. Wasim, Laabiah. The Role of Glycosylation on the Spatiotemporal Organization and Function of B-cell Co-receptor CD22.

Degree: 2019, University of Toronto

An important inhibitory co-receptor on B-cells is CD22, a sialic-acid binding protein. CD22 is highly glycosylated with six glycans located in the first two extracellular… (more)

Subjects/Keywords: B-cells; CD22; glycosylation; signalling; 0982

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APA (6th Edition):

Wasim, L. (2019). The Role of Glycosylation on the Spatiotemporal Organization and Function of B-cell Co-receptor CD22. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/94031

Chicago Manual of Style (16th Edition):

Wasim, Laabiah. “The Role of Glycosylation on the Spatiotemporal Organization and Function of B-cell Co-receptor CD22.” 2019. Masters Thesis, University of Toronto. Accessed October 25, 2020. http://hdl.handle.net/1807/94031.

MLA Handbook (7th Edition):

Wasim, Laabiah. “The Role of Glycosylation on the Spatiotemporal Organization and Function of B-cell Co-receptor CD22.” 2019. Web. 25 Oct 2020.

Vancouver:

Wasim L. The Role of Glycosylation on the Spatiotemporal Organization and Function of B-cell Co-receptor CD22. [Internet] [Masters thesis]. University of Toronto; 2019. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1807/94031.

Council of Science Editors:

Wasim L. The Role of Glycosylation on the Spatiotemporal Organization and Function of B-cell Co-receptor CD22. [Masters Thesis]. University of Toronto; 2019. Available from: http://hdl.handle.net/1807/94031


University of Toronto

29. Galiwango, Ronald Moses. Assessing Immunological and Microbiome Determinants of HIV Susceptibility in the Male Genital Tract.

Degree: PhD, 2020, University of Toronto

 Penile circumcision (PC) is a one-time, cost-effective HIV prevention strategy. A deeper understanding of its protection-mechanisms may inform novel prevention methods for vulnerable men declining… (more)

Subjects/Keywords: antibacterials; foreskin immunology; Human Immunodeficiency virus; penile HIV prevention; penile microbiome; urethra; 0982

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APA (6th Edition):

Galiwango, R. M. (2020). Assessing Immunological and Microbiome Determinants of HIV Susceptibility in the Male Genital Tract. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/101012

Chicago Manual of Style (16th Edition):

Galiwango, Ronald Moses. “Assessing Immunological and Microbiome Determinants of HIV Susceptibility in the Male Genital Tract.” 2020. Doctoral Dissertation, University of Toronto. Accessed October 25, 2020. http://hdl.handle.net/1807/101012.

MLA Handbook (7th Edition):

Galiwango, Ronald Moses. “Assessing Immunological and Microbiome Determinants of HIV Susceptibility in the Male Genital Tract.” 2020. Web. 25 Oct 2020.

Vancouver:

Galiwango RM. Assessing Immunological and Microbiome Determinants of HIV Susceptibility in the Male Genital Tract. [Internet] [Doctoral dissertation]. University of Toronto; 2020. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1807/101012.

Council of Science Editors:

Galiwango RM. Assessing Immunological and Microbiome Determinants of HIV Susceptibility in the Male Genital Tract. [Doctoral Dissertation]. University of Toronto; 2020. Available from: http://hdl.handle.net/1807/101012


University of Toronto

30. Chu, Kuan Lun. The Role of the Costimulatory TNFR Family Member GITR in T Cell Immunity During an Acute Respiratory Infection.

Degree: PhD, 2020, University of Toronto

 T cells play a critical role in control of influenza virus, a major human pathogen. Glucocorticoid-induced TNFR-related Protein (GITR), a prosurvival member of the TNFR… (more)

Subjects/Keywords: GITR; Influenza; Signal 4; T cells; TNFR; Trm; 0982

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APA (6th Edition):

Chu, K. L. (2020). The Role of the Costimulatory TNFR Family Member GITR in T Cell Immunity During an Acute Respiratory Infection. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/101086

Chicago Manual of Style (16th Edition):

Chu, Kuan Lun. “The Role of the Costimulatory TNFR Family Member GITR in T Cell Immunity During an Acute Respiratory Infection.” 2020. Doctoral Dissertation, University of Toronto. Accessed October 25, 2020. http://hdl.handle.net/1807/101086.

MLA Handbook (7th Edition):

Chu, Kuan Lun. “The Role of the Costimulatory TNFR Family Member GITR in T Cell Immunity During an Acute Respiratory Infection.” 2020. Web. 25 Oct 2020.

Vancouver:

Chu KL. The Role of the Costimulatory TNFR Family Member GITR in T Cell Immunity During an Acute Respiratory Infection. [Internet] [Doctoral dissertation]. University of Toronto; 2020. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1807/101086.

Council of Science Editors:

Chu KL. The Role of the Costimulatory TNFR Family Member GITR in T Cell Immunity During an Acute Respiratory Infection. [Doctoral Dissertation]. University of Toronto; 2020. Available from: http://hdl.handle.net/1807/101086

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