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You searched for +publisher:"University of Texas Southwestern Medical Center" +contributor:("White, Michael A."). Showing records 1 – 30 of 40 total matches.

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University of Texas Southwestern Medical Center

1. Ram, Rosalyn Ruanga. The RASSF1A Tumor Suppressor Regulates a Cascade of Oncogenic Signals That Are Restrained by G1.

Degree: PhD, Cancer Biology, 2012, University of Texas Southwestern Medical Center

 The RASSF1A tumor suppressor is one of the most commonly inactivated genes in cancer. To understand why epigenetic silencing of RASSF1A promotes tumorigenesis, I employed… (more)

Subjects/Keywords: Tumor Suppressor Proteins; Ubiquitin-Protein Ligase Complexes; Cell Cycle

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ram, R. R. (2012). The RASSF1A Tumor Suppressor Regulates a Cascade of Oncogenic Signals That Are Restrained by G1. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1004

Chicago Manual of Style (16th Edition):

Ram, Rosalyn Ruanga. “The RASSF1A Tumor Suppressor Regulates a Cascade of Oncogenic Signals That Are Restrained by G1.” 2012. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/1004.

MLA Handbook (7th Edition):

Ram, Rosalyn Ruanga. “The RASSF1A Tumor Suppressor Regulates a Cascade of Oncogenic Signals That Are Restrained by G1.” 2012. Web. 09 Jul 2020.

Vancouver:

Ram RR. The RASSF1A Tumor Suppressor Regulates a Cascade of Oncogenic Signals That Are Restrained by G1. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2012. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/1004.

Council of Science Editors:

Ram RR. The RASSF1A Tumor Suppressor Regulates a Cascade of Oncogenic Signals That Are Restrained by G1. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2012. Available from: http://hdl.handle.net/2152.5/1004


University of Texas Southwestern Medical Center

2. Ward, Samuel Enoch. Host Modulagtors of the Death Response to Influenza a Infection.

Degree: PhD, Cell Regulation, 2012, University of Texas Southwestern Medical Center

 Influenza A virus infects 5-20% of the population annually, resulting in ~35,000 deaths and significant morbidity. Current treatments include vaccines and drugs that target viral… (more)

Subjects/Keywords: Immunity, Innate; Influenza A Virus; RNA Interference

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APA (6th Edition):

Ward, S. E. (2012). Host Modulagtors of the Death Response to Influenza a Infection. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1008

Chicago Manual of Style (16th Edition):

Ward, Samuel Enoch. “Host Modulagtors of the Death Response to Influenza a Infection.” 2012. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/1008.

MLA Handbook (7th Edition):

Ward, Samuel Enoch. “Host Modulagtors of the Death Response to Influenza a Infection.” 2012. Web. 09 Jul 2020.

Vancouver:

Ward SE. Host Modulagtors of the Death Response to Influenza a Infection. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2012. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/1008.

Council of Science Editors:

Ward SE. Host Modulagtors of the Death Response to Influenza a Infection. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2012. Available from: http://hdl.handle.net/2152.5/1008


University of Texas Southwestern Medical Center

3. Bodemann, Brian Oliver. Ral G-Proteins and the Exocyst Complex are Mediators of the Cellular Response to Nutrients.

Degree: PhD, Cell Regulation, 2012, University of Texas Southwestern Medical Center

 The small G-proteins, RalA and RalB, are important mediators of cellular responses to viral infection and nutrient availability. Prior work has demonstrated that the exocyst… (more)

Subjects/Keywords: ral GTP-Binding Proteins; Gene Expression Regulation, Enzymologic; Neoplasms

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APA (6th Edition):

Bodemann, B. O. (2012). Ral G-Proteins and the Exocyst Complex are Mediators of the Cellular Response to Nutrients. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1015

Chicago Manual of Style (16th Edition):

Bodemann, Brian Oliver. “Ral G-Proteins and the Exocyst Complex are Mediators of the Cellular Response to Nutrients.” 2012. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/1015.

MLA Handbook (7th Edition):

Bodemann, Brian Oliver. “Ral G-Proteins and the Exocyst Complex are Mediators of the Cellular Response to Nutrients.” 2012. Web. 09 Jul 2020.

Vancouver:

Bodemann BO. Ral G-Proteins and the Exocyst Complex are Mediators of the Cellular Response to Nutrients. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2012. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/1015.

Council of Science Editors:

Bodemann BO. Ral G-Proteins and the Exocyst Complex are Mediators of the Cellular Response to Nutrients. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2012. Available from: http://hdl.handle.net/2152.5/1015


University of Texas Southwestern Medical Center

4. An, Zhenyi. The Regulation of Autophagy and Its Role in Mitotic Exit.

Degree: 2014, University of Texas Southwestern Medical Center

 Autophagy is an evolutionarily conserved pathway in which cells enclose cytoplasmic contents in double membrane vesicles and deliver them to the lysosome for degradation. Autophagy… (more)

Subjects/Keywords: Apoptosis Regulatory Proteins; Autophagy; Mitogen-Activated Protein Kinases; Phosphorylation

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APA (6th Edition):

An, Z. (2014). The Regulation of Autophagy and Its Role in Mitotic Exit. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3582

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

An, Zhenyi. “The Regulation of Autophagy and Its Role in Mitotic Exit.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/3582.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

An, Zhenyi. “The Regulation of Autophagy and Its Role in Mitotic Exit.” 2014. Web. 09 Jul 2020.

Vancouver:

An Z. The Regulation of Autophagy and Its Role in Mitotic Exit. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/3582.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

An Z. The Regulation of Autophagy and Its Role in Mitotic Exit. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3582

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

5. Kim, Ji Mi. Identification of Oncogenic KRAS-Associated Vulnerabilities in Non-Small Cell Lung Cancer.

Degree: 2016, University of Texas Southwestern Medical Center

 Activating mutations in KRAS are frequently involved in the pathogenesis of non-small cell lung cancer (NSCLC), the disease responsible for the most cancer-related deaths in… (more)

Subjects/Keywords: Active Transport, Cell Nucleus; Cell Nucleus; Karyopherins; Lung Neoplasms; Proto-Oncogene Proteins p21(ras); Receptors, Cytoplasmic and Nuclear

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APA (6th Edition):

Kim, J. M. (2016). Identification of Oncogenic KRAS-Associated Vulnerabilities in Non-Small Cell Lung Cancer. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/5726

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kim, Ji Mi. “Identification of Oncogenic KRAS-Associated Vulnerabilities in Non-Small Cell Lung Cancer.” 2016. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/5726.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kim, Ji Mi. “Identification of Oncogenic KRAS-Associated Vulnerabilities in Non-Small Cell Lung Cancer.” 2016. Web. 09 Jul 2020.

Vancouver:

Kim JM. Identification of Oncogenic KRAS-Associated Vulnerabilities in Non-Small Cell Lung Cancer. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2016. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/5726.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kim JM. Identification of Oncogenic KRAS-Associated Vulnerabilities in Non-Small Cell Lung Cancer. [Thesis]. University of Texas Southwestern Medical Center; 2016. Available from: http://hdl.handle.net/2152.5/5726

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

6. Ou, Yi-Hung 1977-. Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation.

Degree: 2013, University of Texas Southwestern Medical Center

 An essential kinase in innate immune signaling, TBK1 couples pathogen surveillance to induction of host defense mechanisms. The pathological activation of TBK1 in cancer can… (more)

Subjects/Keywords: Immunity, Innate; Neoplasms; Protein-Serine-Threonine Kinases; Proto-Oncogene Proteins c-akt

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APA (6th Edition):

Ou, Y. 1. (2013). Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-125

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ou, Yi-Hung 1977-. “Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-125.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ou, Yi-Hung 1977-. “Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation.” 2013. Web. 09 Jul 2020.

Vancouver:

Ou Y1. Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-125.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ou Y1. Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-125

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

7. He, Min. Analysis of ASCL1 in Neuroendocrine Lung Cancer.

Degree: 2015, University of Texas Southwestern Medical Center

 Small cell lung cancer (SCLC) is an understudied tumor subset with aggressive neuroendocrine carcinoma features. Previous studies have determined that the basic helix-loop-helix (bHLH) transcription… (more)

Subjects/Keywords: Basic Helix-Loop-Helix Transcription Factors; Carcinoma, Non-Small-Cell Lung; Cell Lineage; Neuroendocrine Tumors; Oncogenes; Small Cell Lung Carcinoma

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APA (6th Edition):

He, M. (2015). Analysis of ASCL1 in Neuroendocrine Lung Cancer. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4227

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

He, Min. “Analysis of ASCL1 in Neuroendocrine Lung Cancer.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/4227.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

He, Min. “Analysis of ASCL1 in Neuroendocrine Lung Cancer.” 2015. Web. 09 Jul 2020.

Vancouver:

He M. Analysis of ASCL1 in Neuroendocrine Lung Cancer. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/4227.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

He M. Analysis of ASCL1 in Neuroendocrine Lung Cancer. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4227

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

8. Jia, Luying. The Multifunctional Kinase Bub1 Acts as a Signaling Hub for the Spindle Checkpoint.

Degree: 2015, University of Texas Southwestern Medical Center

 The spindle checkpoint is an essential mechanism to ensure accurate chromosome segregation during mitosis. The checkpoint signal originates from the kinetochore, which is a huge… (more)

Subjects/Keywords: Anaphase-Promoting Complex-Cyclosome; Cdc20 Proteins; Cell Cycle Proteins; M Phase Cell Cycle Checkpoints; Protein-Serine-Threonine Kinases; Proto-Oncogene Proteins

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APA (6th Edition):

Jia, L. (2015). The Multifunctional Kinase Bub1 Acts as a Signaling Hub for the Spindle Checkpoint. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4456

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jia, Luying. “The Multifunctional Kinase Bub1 Acts as a Signaling Hub for the Spindle Checkpoint.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/4456.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jia, Luying. “The Multifunctional Kinase Bub1 Acts as a Signaling Hub for the Spindle Checkpoint.” 2015. Web. 09 Jul 2020.

Vancouver:

Jia L. The Multifunctional Kinase Bub1 Acts as a Signaling Hub for the Spindle Checkpoint. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/4456.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jia L. The Multifunctional Kinase Bub1 Acts as a Signaling Hub for the Spindle Checkpoint. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4456

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

9. Zaganjor, Elma 1981-. Regulation by ERK1/2 of Novel Substrates, Kinesins KIF2A and KIF2C.

Degree: 2013, University of Texas Southwestern Medical Center

 The kinesin-like protein KIF2A is a microtubule-associated motor protein thatauses microtubule depolymerization by inducing a conformational change in tubulin. The depolymerase function of KIF2A is… (more)

Subjects/Keywords: Cell Transformation, Neoplastic; Extracellular Signal-Regulated MAP Kinases; Kinesin

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APA (6th Edition):

Zaganjor, E. 1. (2013). Regulation by ERK1/2 of Novel Substrates, Kinesins KIF2A and KIF2C. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-124

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zaganjor, Elma 1981-. “Regulation by ERK1/2 of Novel Substrates, Kinesins KIF2A and KIF2C.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-124.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zaganjor, Elma 1981-. “Regulation by ERK1/2 of Novel Substrates, Kinesins KIF2A and KIF2C.” 2013. Web. 09 Jul 2020.

Vancouver:

Zaganjor E1. Regulation by ERK1/2 of Novel Substrates, Kinesins KIF2A and KIF2C. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-124.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zaganjor E1. Regulation by ERK1/2 of Novel Substrates, Kinesins KIF2A and KIF2C. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-124

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

10. Gil del Alcazar, Carlos Rodrigo. Targeting DNA Double-Strand Break Repair to Potentiate Radio- and Chemo Therapy of Glioblastoma.

Degree: 2015, University of Texas Southwestern Medical Center

 Surgical resection followed by radiation and adjuvant temozolomide (TMZ) is the standard of care for glioblastoma (GBM). Not all GBMs respond to therapy, and most… (more)

Subjects/Keywords: DNA Breaks, Double-Stranded; DNA Repair; Glioblastoma; Radiation-Sensitizing Agents

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APA (6th Edition):

Gil del Alcazar, C. R. (2015). Targeting DNA Double-Strand Break Repair to Potentiate Radio- and Chemo Therapy of Glioblastoma. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4218

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gil del Alcazar, Carlos Rodrigo. “Targeting DNA Double-Strand Break Repair to Potentiate Radio- and Chemo Therapy of Glioblastoma.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/4218.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gil del Alcazar, Carlos Rodrigo. “Targeting DNA Double-Strand Break Repair to Potentiate Radio- and Chemo Therapy of Glioblastoma.” 2015. Web. 09 Jul 2020.

Vancouver:

Gil del Alcazar CR. Targeting DNA Double-Strand Break Repair to Potentiate Radio- and Chemo Therapy of Glioblastoma. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/4218.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gil del Alcazar CR. Targeting DNA Double-Strand Break Repair to Potentiate Radio- and Chemo Therapy of Glioblastoma. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4218

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

11. Rifki, Oktay Feridun. RalGDS-Dependent Cardiomyocyte Autophagy Is Necessary for Load-Induced Ventricular Hypertrophy.

Degree: 2014, University of Texas Southwestern Medical Center

 Recent work has demonstrated that autophagy, a phylogenetically conserved, lysosome-mediated pathway of protein degradation, is a key participant in pathological cardiac remodeling. One common feature… (more)

Subjects/Keywords: Autophagy; Cardiomegaly; Myocytes, Cardiac; ral Guanine Nucleotide Exchange Factor

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rifki, O. F. (2014). RalGDS-Dependent Cardiomyocyte Autophagy Is Necessary for Load-Induced Ventricular Hypertrophy. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3324

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rifki, Oktay Feridun. “RalGDS-Dependent Cardiomyocyte Autophagy Is Necessary for Load-Induced Ventricular Hypertrophy.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/3324.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rifki, Oktay Feridun. “RalGDS-Dependent Cardiomyocyte Autophagy Is Necessary for Load-Induced Ventricular Hypertrophy.” 2014. Web. 09 Jul 2020.

Vancouver:

Rifki OF. RalGDS-Dependent Cardiomyocyte Autophagy Is Necessary for Load-Induced Ventricular Hypertrophy. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/3324.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rifki OF. RalGDS-Dependent Cardiomyocyte Autophagy Is Necessary for Load-Induced Ventricular Hypertrophy. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3324

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

12. Pineda, Carlos Tyler. The Cancer Specific Ubiquitin Ligase MAGE-A3/6-TRIM28 Drives Tumorigenesis by Ubiquitination and Proteasomal Degradation of AMPK.

Degree: 2015, University of Texas Southwestern Medical Center

 The genes MAGE-A3 and MAGE-A6 (MAGE-A3/6) have a unique expression pattern in which they are normally expressed in the adult testis but are aberrantly expressed… (more)

Subjects/Keywords: AMP-Activated Protein Kinases; Antigens, Neoplasm; Neoplasm Proteins; Neoplasms; Ubiquitination; Ubiquitin-Protein Ligases

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APA (6th Edition):

Pineda, C. T. (2015). The Cancer Specific Ubiquitin Ligase MAGE-A3/6-TRIM28 Drives Tumorigenesis by Ubiquitination and Proteasomal Degradation of AMPK. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4447

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pineda, Carlos Tyler. “The Cancer Specific Ubiquitin Ligase MAGE-A3/6-TRIM28 Drives Tumorigenesis by Ubiquitination and Proteasomal Degradation of AMPK.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/4447.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pineda, Carlos Tyler. “The Cancer Specific Ubiquitin Ligase MAGE-A3/6-TRIM28 Drives Tumorigenesis by Ubiquitination and Proteasomal Degradation of AMPK.” 2015. Web. 09 Jul 2020.

Vancouver:

Pineda CT. The Cancer Specific Ubiquitin Ligase MAGE-A3/6-TRIM28 Drives Tumorigenesis by Ubiquitination and Proteasomal Degradation of AMPK. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/4447.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pineda CT. The Cancer Specific Ubiquitin Ligase MAGE-A3/6-TRIM28 Drives Tumorigenesis by Ubiquitination and Proteasomal Degradation of AMPK. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4447

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

13. McMillan, Elizabeth Anne. Identification of Biomarker Driven Intervention Opportunities and Advancement of Mechanism of Action Predictions for Anti-Cancer Therapeutics.

Degree: 2017, University of Texas Southwestern Medical Center

 Oncogenic lesions arising during cancer progression provide an attractive target for chemical intervention strategies. The extreme molecular heterogeneity of tumors, however, makes it difficult to… (more)

Subjects/Keywords: Carcinoma, Non-Small-Cell Lung; High-Throughput Screening Assays; Lung Neoplasms; NF-E2-Related Factor 2

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APA (6th Edition):

McMillan, E. A. (2017). Identification of Biomarker Driven Intervention Opportunities and Advancement of Mechanism of Action Predictions for Anti-Cancer Therapeutics. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/6616

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

McMillan, Elizabeth Anne. “Identification of Biomarker Driven Intervention Opportunities and Advancement of Mechanism of Action Predictions for Anti-Cancer Therapeutics.” 2017. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/6616.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

McMillan, Elizabeth Anne. “Identification of Biomarker Driven Intervention Opportunities and Advancement of Mechanism of Action Predictions for Anti-Cancer Therapeutics.” 2017. Web. 09 Jul 2020.

Vancouver:

McMillan EA. Identification of Biomarker Driven Intervention Opportunities and Advancement of Mechanism of Action Predictions for Anti-Cancer Therapeutics. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2017. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/6616.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

McMillan EA. Identification of Biomarker Driven Intervention Opportunities and Advancement of Mechanism of Action Predictions for Anti-Cancer Therapeutics. [Thesis]. University of Texas Southwestern Medical Center; 2017. Available from: http://hdl.handle.net/2152.5/6616

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

14. Kulak, Ozlem. Chemical Disruption of Wnt Signaling and Telomere Length Maintenance.

Degree: 2015, University of Texas Southwestern Medical Center

Pages xvii-xviii of the dissertation are incorrectly numbered as pages xvi-xvii.

A nearly universal feature of colorectal cancer (CRC) incidents is the presence of genetic… (more)

Subjects/Keywords: Enzyme Inhibitors; Tankyrases; Telomere Shortening; Wnt Signaling Pathway

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APA (6th Edition):

Kulak, O. (2015). Chemical Disruption of Wnt Signaling and Telomere Length Maintenance. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4110

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kulak, Ozlem. “Chemical Disruption of Wnt Signaling and Telomere Length Maintenance.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/4110.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kulak, Ozlem. “Chemical Disruption of Wnt Signaling and Telomere Length Maintenance.” 2015. Web. 09 Jul 2020.

Vancouver:

Kulak O. Chemical Disruption of Wnt Signaling and Telomere Length Maintenance. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/4110.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kulak O. Chemical Disruption of Wnt Signaling and Telomere Length Maintenance. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4110

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

15. Mata, Miguel Angel. Chemical Inhibition of RNA Viruses Reveals REDD1 as a Host Defense Factor.

Degree: 2013, University of Texas Southwestern Medical Center

 Influenza (flu) is a contagious infectious respiratory illness. The flu can cause from mild to life-threatening illness. The current therapeutic intervention strategies to prevent or… (more)

Subjects/Keywords: Antiviral Agents; Naphthalimides; Orthomyxoviridae; Transcription Factors; Vesiculovirus

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APA (6th Edition):

Mata, M. A. (2013). Chemical Inhibition of RNA Viruses Reveals REDD1 as a Host Defense Factor. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/2723

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mata, Miguel Angel. “Chemical Inhibition of RNA Viruses Reveals REDD1 as a Host Defense Factor.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/2723.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mata, Miguel Angel. “Chemical Inhibition of RNA Viruses Reveals REDD1 as a Host Defense Factor.” 2013. Web. 09 Jul 2020.

Vancouver:

Mata MA. Chemical Inhibition of RNA Viruses Reveals REDD1 as a Host Defense Factor. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/2723.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mata MA. Chemical Inhibition of RNA Viruses Reveals REDD1 as a Host Defense Factor. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/2723

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

16. Zaman, Aubhishek. Dynamic Modulation of Exocyst Interaction Networks Integrates Hippo and mTOR Pathway Response to Pathogen Detection.

Degree: 2017, University of Texas Southwestern Medical Center

 Monomeric RALGTPases, via direct binding to the exocyst (a.k.a Sec6/8 complex), help mount productive cell autonomous responses to trophic and immunogenic signals. However, RAL-exocyst downstream… (more)

Subjects/Keywords: Protein-Serine-Threonine Kinases; Signal Transduction; TOR Serine-Threonine Kinases

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APA (6th Edition):

Zaman, A. (2017). Dynamic Modulation of Exocyst Interaction Networks Integrates Hippo and mTOR Pathway Response to Pathogen Detection. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/7737

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zaman, Aubhishek. “Dynamic Modulation of Exocyst Interaction Networks Integrates Hippo and mTOR Pathway Response to Pathogen Detection.” 2017. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/7737.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zaman, Aubhishek. “Dynamic Modulation of Exocyst Interaction Networks Integrates Hippo and mTOR Pathway Response to Pathogen Detection.” 2017. Web. 09 Jul 2020.

Vancouver:

Zaman A. Dynamic Modulation of Exocyst Interaction Networks Integrates Hippo and mTOR Pathway Response to Pathogen Detection. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2017. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/7737.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zaman A. Dynamic Modulation of Exocyst Interaction Networks Integrates Hippo and mTOR Pathway Response to Pathogen Detection. [Thesis]. University of Texas Southwestern Medical Center; 2017. Available from: http://hdl.handle.net/2152.5/7737

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

17. Chau, Vincent. Novel Small Molecule Induces Apoptosis in Malignant Peripheral Nerve Sheath Tumors of Neurofibromatosis Type I.

Degree: 2013, University of Texas Southwestern Medical Center

 Neurofibromatosis Type 1 (NF1) is an autosomal disease that affects neural crest-derived tissues, leading to a wide spectrum of clinical presentations. Patients commonly present with… (more)

Subjects/Keywords: Antineoplastic Agents; Apoptosis; Nerve Sheath Neoplasms

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APA (6th Edition):

Chau, V. (2013). Novel Small Molecule Induces Apoptosis in Malignant Peripheral Nerve Sheath Tumors of Neurofibromatosis Type I. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4105

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chau, Vincent. “Novel Small Molecule Induces Apoptosis in Malignant Peripheral Nerve Sheath Tumors of Neurofibromatosis Type I.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/4105.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chau, Vincent. “Novel Small Molecule Induces Apoptosis in Malignant Peripheral Nerve Sheath Tumors of Neurofibromatosis Type I.” 2013. Web. 09 Jul 2020.

Vancouver:

Chau V. Novel Small Molecule Induces Apoptosis in Malignant Peripheral Nerve Sheath Tumors of Neurofibromatosis Type I. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/4105.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chau V. Novel Small Molecule Induces Apoptosis in Malignant Peripheral Nerve Sheath Tumors of Neurofibromatosis Type I. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/4105

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

18. Hight, Suzie K. An In Vivo Functional Genomics Screen Identifies New Regulators of Tumorigenesis in Non-Small Cell Lung Cancer.

Degree: 2015, University of Texas Southwestern Medical Center

 Cancer cells are characterized by the aberrant regulation of signaling pathways that govern responses to growth stimuli, resulting in dysregulated cellular proliferation. The accumulated genomic… (more)

Subjects/Keywords: Carcinoma, Non-Small-Cell Lung; Gene Expression Regulation, Neoplastic; Lung Neoplasms; Receptors, Cytoplasmic and Nuclear

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APA (6th Edition):

Hight, S. K. (2015). An In Vivo Functional Genomics Screen Identifies New Regulators of Tumorigenesis in Non-Small Cell Lung Cancer. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4462

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hight, Suzie K. “An In Vivo Functional Genomics Screen Identifies New Regulators of Tumorigenesis in Non-Small Cell Lung Cancer.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/4462.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hight, Suzie K. “An In Vivo Functional Genomics Screen Identifies New Regulators of Tumorigenesis in Non-Small Cell Lung Cancer.” 2015. Web. 09 Jul 2020.

Vancouver:

Hight SK. An In Vivo Functional Genomics Screen Identifies New Regulators of Tumorigenesis in Non-Small Cell Lung Cancer. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/4462.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hight SK. An In Vivo Functional Genomics Screen Identifies New Regulators of Tumorigenesis in Non-Small Cell Lung Cancer. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4462

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

19. Eskiocak, Banu. Biomarker Accessible and Chemically Addressable Mechanistic Subtypes of BRAF Melanoma.

Degree: 2016, University of Texas Southwestern Medical Center

 Genomic diversity and adaptive plasticity of melanoma tumors limit durable control with conventional and targeted therapies. Nevertheless, pathological activation of the ERK1/2 regulatory system is… (more)

Subjects/Keywords: Biomarkers; DNA Copy Number Variations; MAP Kinase Signaling System; Melanoma; Skin Neoplasms

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APA (6th Edition):

Eskiocak, B. (2016). Biomarker Accessible and Chemically Addressable Mechanistic Subtypes of BRAF Melanoma. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/5733

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Eskiocak, Banu. “Biomarker Accessible and Chemically Addressable Mechanistic Subtypes of BRAF Melanoma.” 2016. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/5733.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Eskiocak, Banu. “Biomarker Accessible and Chemically Addressable Mechanistic Subtypes of BRAF Melanoma.” 2016. Web. 09 Jul 2020.

Vancouver:

Eskiocak B. Biomarker Accessible and Chemically Addressable Mechanistic Subtypes of BRAF Melanoma. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2016. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/5733.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Eskiocak B. Biomarker Accessible and Chemically Addressable Mechanistic Subtypes of BRAF Melanoma. [Thesis]. University of Texas Southwestern Medical Center; 2016. Available from: http://hdl.handle.net/2152.5/5733

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

20. Oswald, Nathaniel Walter. Using Multiple Screening Strategies to Biologically and Chemically Characterize Natural Products.

Degree: 2016, University of Texas Southwestern Medical Center

 Natural products play an important role in the discovery and development of therapeutics and biological probes. However, in recent decades therapeutic screening efforts have moved… (more)

Subjects/Keywords: Biological Factors; Biological Products; Computational Biology; Lung Neoplasms; Technology, Pharmaceutical

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APA (6th Edition):

Oswald, N. W. (2016). Using Multiple Screening Strategies to Biologically and Chemically Characterize Natural Products. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/5730

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Oswald, Nathaniel Walter. “Using Multiple Screening Strategies to Biologically and Chemically Characterize Natural Products.” 2016. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/5730.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Oswald, Nathaniel Walter. “Using Multiple Screening Strategies to Biologically and Chemically Characterize Natural Products.” 2016. Web. 09 Jul 2020.

Vancouver:

Oswald NW. Using Multiple Screening Strategies to Biologically and Chemically Characterize Natural Products. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2016. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/5730.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Oswald NW. Using Multiple Screening Strategies to Biologically and Chemically Characterize Natural Products. [Thesis]. University of Texas Southwestern Medical Center; 2016. Available from: http://hdl.handle.net/2152.5/5730

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

21. Tagal, Vural. SMARCA4/BRG1-Inactivating Mutations as Potential Predictive Markers for Aurora Kinase A-Targeted Therapy in NSCLCs.

Degree: 2014, University of Texas Southwestern Medical Center

 SMARCA4 encodes a catalytic subunit of the SWI/SNF chromatin remodeling complex, BRG1. Frequent occurrence of SMARCA4/BRG1-inactivating mutations and their mutually exclusive nature from EGFR and… (more)

Subjects/Keywords: Aurora Kinase A; Carcinoma, Non-Small-Cell Lung; DNA Helicases; Nuclear Proteins; Transcription Factors

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APA (6th Edition):

Tagal, V. (2014). SMARCA4/BRG1-Inactivating Mutations as Potential Predictive Markers for Aurora Kinase A-Targeted Therapy in NSCLCs. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3570

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tagal, Vural. “SMARCA4/BRG1-Inactivating Mutations as Potential Predictive Markers for Aurora Kinase A-Targeted Therapy in NSCLCs.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/3570.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tagal, Vural. “SMARCA4/BRG1-Inactivating Mutations as Potential Predictive Markers for Aurora Kinase A-Targeted Therapy in NSCLCs.” 2014. Web. 09 Jul 2020.

Vancouver:

Tagal V. SMARCA4/BRG1-Inactivating Mutations as Potential Predictive Markers for Aurora Kinase A-Targeted Therapy in NSCLCs. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/3570.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tagal V. SMARCA4/BRG1-Inactivating Mutations as Potential Predictive Markers for Aurora Kinase A-Targeted Therapy in NSCLCs. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3570

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

22. Steininger, Robert Joseph, III. Investigating Roles for Cellular Heterogeneity in Cancer.

Degree: 2014, University of Texas Southwestern Medical Center

 Cell populations, even those derived from a single clone, can exhibit a high degree of phenotypic variability. However, most biological studies take measurements as averages… (more)

Subjects/Keywords: Genetic Heterogeneity; Neoplasms; Physiological Processes

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APA (6th Edition):

Steininger, Robert Joseph, I. (2014). Investigating Roles for Cellular Heterogeneity in Cancer. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3589

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Steininger, Robert Joseph, III. “Investigating Roles for Cellular Heterogeneity in Cancer.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/3589.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Steininger, Robert Joseph, III. “Investigating Roles for Cellular Heterogeneity in Cancer.” 2014. Web. 09 Jul 2020.

Vancouver:

Steininger, Robert Joseph I. Investigating Roles for Cellular Heterogeneity in Cancer. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/3589.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Steininger, Robert Joseph I. Investigating Roles for Cellular Heterogeneity in Cancer. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3589

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

23. Zhang, Lu. Identification of Small Molecule Inhibitors Targeting Truncated Adenomatous Polyposis Coli (APC) as a Novel Therapeutic Strategy in Colorectal Cancer.

Degree: 2014, University of Texas Southwestern Medical Center

 Adenomatous polyposis coli (APC) is a tumor suppressor gene that is mutated in the vast majority of colorectal tumors. Inactivation of this gene is a… (more)

Subjects/Keywords: Adenomatous Polyposis Coli Protein; Colorectal Neoplasms; Genes, APC

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APA (6th Edition):

Zhang, L. (2014). Identification of Small Molecule Inhibitors Targeting Truncated Adenomatous Polyposis Coli (APC) as a Novel Therapeutic Strategy in Colorectal Cancer. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3585

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhang, Lu. “Identification of Small Molecule Inhibitors Targeting Truncated Adenomatous Polyposis Coli (APC) as a Novel Therapeutic Strategy in Colorectal Cancer.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/3585.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhang, Lu. “Identification of Small Molecule Inhibitors Targeting Truncated Adenomatous Polyposis Coli (APC) as a Novel Therapeutic Strategy in Colorectal Cancer.” 2014. Web. 09 Jul 2020.

Vancouver:

Zhang L. Identification of Small Molecule Inhibitors Targeting Truncated Adenomatous Polyposis Coli (APC) as a Novel Therapeutic Strategy in Colorectal Cancer. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/3585.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhang L. Identification of Small Molecule Inhibitors Targeting Truncated Adenomatous Polyposis Coli (APC) as a Novel Therapeutic Strategy in Colorectal Cancer. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3585

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

24. Augustyn, Alexander. Characterizing ASCL1-Dependent Neuroendocrine Non-Small Cell Lung Cancers.

Degree: 2013, University of Texas Southwestern Medical Center

 In order to achieve personalized medicine for the treatment of lung cancer, it is important to accurately classify tumors using a combination of factors, including… (more)

Subjects/Keywords: Basic Helix-Loop-Helix Transcription Factors; Carcinoma, Non-Small-Cell Lung; Cell Lineage; Neuroendocrine Tumors; Oncogenes; Small Cell Lung Carcinoma

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APA (6th Edition):

Augustyn, A. (2013). Characterizing ASCL1-Dependent Neuroendocrine Non-Small Cell Lung Cancers. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3336

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Augustyn, Alexander. “Characterizing ASCL1-Dependent Neuroendocrine Non-Small Cell Lung Cancers.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/3336.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Augustyn, Alexander. “Characterizing ASCL1-Dependent Neuroendocrine Non-Small Cell Lung Cancers.” 2013. Web. 09 Jul 2020.

Vancouver:

Augustyn A. Characterizing ASCL1-Dependent Neuroendocrine Non-Small Cell Lung Cancers. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/3336.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Augustyn A. Characterizing ASCL1-Dependent Neuroendocrine Non-Small Cell Lung Cancers. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/3336

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

25. Borkowski, Robert John. Identifying Context-Specific Synthetic Lethal miRNA Inhibition in Non-Small Cell Lung Cancer.

Degree: 2013, University of Texas Southwestern Medical Center

 Non-small cell lung cancer (NSCLC) is the leading cause of cancer-related fatalities in the US. This is due in part to a lack of highly… (more)

Subjects/Keywords: Carcinoma, Non-Small-Cell Lung; MicroRNAs; Gene Targeting

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APA (6th Edition):

Borkowski, R. J. (2013). Identifying Context-Specific Synthetic Lethal miRNA Inhibition in Non-Small Cell Lung Cancer. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1726

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Borkowski, Robert John. “Identifying Context-Specific Synthetic Lethal miRNA Inhibition in Non-Small Cell Lung Cancer.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/1726.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Borkowski, Robert John. “Identifying Context-Specific Synthetic Lethal miRNA Inhibition in Non-Small Cell Lung Cancer.” 2013. Web. 09 Jul 2020.

Vancouver:

Borkowski RJ. Identifying Context-Specific Synthetic Lethal miRNA Inhibition in Non-Small Cell Lung Cancer. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/1726.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Borkowski RJ. Identifying Context-Specific Synthetic Lethal miRNA Inhibition in Non-Small Cell Lung Cancer. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1726

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

26. Chen, Pei-Hsuan. Metabolic Diversity in Human Non-Small Cell Lung Cancer Cells.

Degree: 2015, University of Texas Southwestern Medical Center

 Cancer cells display oncogene-driven rewiring of metabolism to produce energy and macromolecules for growth. Inhibition of growth-promoting metabolic pathways may prove to be a useful… (more)

Subjects/Keywords: Antineoplastic Agents; Carcinoma, Non-Small-Cell Lung; Lung Neoplasms

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chen, P. (2015). Metabolic Diversity in Human Non-Small Cell Lung Cancer Cells. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4451

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Pei-Hsuan. “Metabolic Diversity in Human Non-Small Cell Lung Cancer Cells.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/4451.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Pei-Hsuan. “Metabolic Diversity in Human Non-Small Cell Lung Cancer Cells.” 2015. Web. 09 Jul 2020.

Vancouver:

Chen P. Metabolic Diversity in Human Non-Small Cell Lung Cancer Cells. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/4451.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen P. Metabolic Diversity in Human Non-Small Cell Lung Cancer Cells. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4451

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

27. Wang, Chensu. Illuminating Endocytic Organelles with pH-Resposive [sic] Nanomaterials.

Degree: 2017, University of Texas Southwestern Medical Center

 Endosomes, lysosomes and related catabolic organelles are a dynamic continuum of vacuolar structures that impact a number of key cell physiological processes that include protein/lipid… (more)

Subjects/Keywords: Endocytosis; Endosomes; Fluorescent Dyes; Lysosomes; Nanoparticles

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wang, C. (2017). Illuminating Endocytic Organelles with pH-Resposive [sic] Nanomaterials. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/6595

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wang, Chensu. “Illuminating Endocytic Organelles with pH-Resposive [sic] Nanomaterials.” 2017. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/6595.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wang, Chensu. “Illuminating Endocytic Organelles with pH-Resposive [sic] Nanomaterials.” 2017. Web. 09 Jul 2020.

Vancouver:

Wang C. Illuminating Endocytic Organelles with pH-Resposive [sic] Nanomaterials. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2017. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/6595.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wang C. Illuminating Endocytic Organelles with pH-Resposive [sic] Nanomaterials. [Thesis]. University of Texas Southwestern Medical Center; 2017. Available from: http://hdl.handle.net/2152.5/6595

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

28. Shields, Benjamin Baker. Mapping the Landscape of Acquired Vulnerabilities in Ovarian Cancer.

Degree: 2013, University of Texas Southwestern Medical Center

 Recent undertakings to identify the genetic lesions associated with ovarian cancer have noted the striking diversity of mutations occurring in this disease. This genetic diversity… (more)

Subjects/Keywords: Cell Differentiation; MicroRNAs; Ovarian Neoplasms

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Shields, B. B. (2013). Mapping the Landscape of Acquired Vulnerabilities in Ovarian Cancer. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1589

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shields, Benjamin Baker. “Mapping the Landscape of Acquired Vulnerabilities in Ovarian Cancer.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/1589.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shields, Benjamin Baker. “Mapping the Landscape of Acquired Vulnerabilities in Ovarian Cancer.” 2013. Web. 09 Jul 2020.

Vancouver:

Shields BB. Mapping the Landscape of Acquired Vulnerabilities in Ovarian Cancer. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/1589.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shields BB. Mapping the Landscape of Acquired Vulnerabilities in Ovarian Cancer. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1589

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

29. Zhong, Rui. Tackling Computational Challenges in High-Throughput RNA Interference Screening.

Degree: 2014, University of Texas Southwestern Medical Center

 Since the discovery of RNAi decades ago, it has been increasingly used in biomedical and biological research. The success of analyzing single genes using siRNAs… (more)

Subjects/Keywords: High-Throughput Screening Assays; RNA Interference; Software

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhong, R. (2014). Tackling Computational Challenges in High-Throughput RNA Interference Screening. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3335

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhong, Rui. “Tackling Computational Challenges in High-Throughput RNA Interference Screening.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/3335.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhong, Rui. “Tackling Computational Challenges in High-Throughput RNA Interference Screening.” 2014. Web. 09 Jul 2020.

Vancouver:

Zhong R. Tackling Computational Challenges in High-Throughput RNA Interference Screening. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/3335.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhong R. Tackling Computational Challenges in High-Throughput RNA Interference Screening. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3335

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

30. Frink, Robin Elizabeth. Preclinical Studies of Telomerase Inhibitor Imetelstat in Non-small Cell Lung Cancer.

Degree: 2013, University of Texas Southwestern Medical Center

 Telomerase is expressed in ~90% of all cancers but is not expressed in most somatic cells making it an attractive target for cancer therapy. Telomerase… (more)

Subjects/Keywords: Carcinoma, Non-Small-Cell Lung; Lung Neoplasms; Oligonucleotides; Telomerase

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Frink, R. E. (2013). Preclinical Studies of Telomerase Inhibitor Imetelstat in Non-small Cell Lung Cancer. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-126

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Frink, Robin Elizabeth. “Preclinical Studies of Telomerase Inhibitor Imetelstat in Non-small Cell Lung Cancer.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-126.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Frink, Robin Elizabeth. “Preclinical Studies of Telomerase Inhibitor Imetelstat in Non-small Cell Lung Cancer.” 2013. Web. 09 Jul 2020.

Vancouver:

Frink RE. Preclinical Studies of Telomerase Inhibitor Imetelstat in Non-small Cell Lung Cancer. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-126.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Frink RE. Preclinical Studies of Telomerase Inhibitor Imetelstat in Non-small Cell Lung Cancer. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-126

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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