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You searched for +publisher:"University of Texas Southwestern Medical Center" +contributor:("Pasare, Chandrashekhar"). Showing records 1 – 10 of 10 total matches.

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University of Texas Southwestern Medical Center

1. Castro Rojas, Cyd Marie. Poly I:C Induced Gene Ablation Negates Potentially Beneficial Effects of A4-Integrin Depletion on Experimental Autoimmune Encephalomyelitis.

Degree: 2016, University of Texas Southwestern Medical Center

 In the experimental autoimmune encephalomyelitis (EAE) murine model of multiple sclerosis, leukocytes transmigrate through the blood brain barrier into the perivascular space and enter the… (more)

Subjects/Keywords: Central Nervous System; Encephalomyelitis, Autoimmune, Experimental; Integrin alpha4; Integrin alpha4beta1; Poly I-C

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Castro Rojas, C. M. (2016). Poly I:C Induced Gene Ablation Negates Potentially Beneficial Effects of A4-Integrin Depletion on Experimental Autoimmune Encephalomyelitis. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/5309

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Castro Rojas, Cyd Marie. “Poly I:C Induced Gene Ablation Negates Potentially Beneficial Effects of A4-Integrin Depletion on Experimental Autoimmune Encephalomyelitis.” 2016. Thesis, University of Texas Southwestern Medical Center. Accessed January 19, 2020. http://hdl.handle.net/2152.5/5309.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Castro Rojas, Cyd Marie. “Poly I:C Induced Gene Ablation Negates Potentially Beneficial Effects of A4-Integrin Depletion on Experimental Autoimmune Encephalomyelitis.” 2016. Web. 19 Jan 2020.

Vancouver:

Castro Rojas CM. Poly I:C Induced Gene Ablation Negates Potentially Beneficial Effects of A4-Integrin Depletion on Experimental Autoimmune Encephalomyelitis. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2016. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/2152.5/5309.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Castro Rojas CM. Poly I:C Induced Gene Ablation Negates Potentially Beneficial Effects of A4-Integrin Depletion on Experimental Autoimmune Encephalomyelitis. [Thesis]. University of Texas Southwestern Medical Center; 2016. Available from: http://hdl.handle.net/2152.5/5309

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

2. Gao, Daxing. Roles of Cyclic GMP-AMP Synthase in Immune Defense Against Retroviruses and Autoimmunity.

Degree: 2015, University of Texas Southwestern Medical Center

 The presence of DNA in the cytosol is known to trigger robust innate immunity. Cyclic GMP-AMP synthase (cGAS) is the sensor of cytosolic DNA and… (more)

Subjects/Keywords: Autoimmune Diseases; DNA; Nucleotidyltransferases

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APA (6th Edition):

Gao, D. (2015). Roles of Cyclic GMP-AMP Synthase in Immune Defense Against Retroviruses and Autoimmunity. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4465

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gao, Daxing. “Roles of Cyclic GMP-AMP Synthase in Immune Defense Against Retroviruses and Autoimmunity.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed January 19, 2020. http://hdl.handle.net/2152.5/4465.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gao, Daxing. “Roles of Cyclic GMP-AMP Synthase in Immune Defense Against Retroviruses and Autoimmunity.” 2015. Web. 19 Jan 2020.

Vancouver:

Gao D. Roles of Cyclic GMP-AMP Synthase in Immune Defense Against Retroviruses and Autoimmunity. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/2152.5/4465.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gao D. Roles of Cyclic GMP-AMP Synthase in Immune Defense Against Retroviruses and Autoimmunity. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4465

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

3. Roybal, Kole Thomas 1982-. Actin Regulatory Dynamics Required for T Cell Activation: A Quantitative and Systems-Level Perspective.

Degree: 2013, University of Texas Southwestern Medical Center

 T cell activation occurs through interaction with an antigen-presenting cell (APC). Upon activation, signaling ensues with the coordination of dozens of diverse signaling molecules in… (more)

Subjects/Keywords: Actins; Signal Transduction; T-Lymphocytes

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APA (6th Edition):

Roybal, K. T. 1. (2013). Actin Regulatory Dynamics Required for T Cell Activation: A Quantitative and Systems-Level Perspective. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-91

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Roybal, Kole Thomas 1982-. “Actin Regulatory Dynamics Required for T Cell Activation: A Quantitative and Systems-Level Perspective.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed January 19, 2020. http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-91.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Roybal, Kole Thomas 1982-. “Actin Regulatory Dynamics Required for T Cell Activation: A Quantitative and Systems-Level Perspective.” 2013. Web. 19 Jan 2020.

Vancouver:

Roybal KT1. Actin Regulatory Dynamics Required for T Cell Activation: A Quantitative and Systems-Level Perspective. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-91.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Roybal KT1. Actin Regulatory Dynamics Required for T Cell Activation: A Quantitative and Systems-Level Perspective. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-91

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

4. Chowdhury, Fatema Zahra. Regulation of Human Cytotoxic T Lymphocyte Functions by IL-12.

Degree: 2013, University of Texas Southwestern Medical Center

 CD8+ T cells or cytotoxic T lymphocytes (CTLs) play a major role in our defense against intracellular pathogens by secreting effector cytokines and directly killing… (more)

Subjects/Keywords: Gene Expression Regulation; CD8-Positive T-Lymphocytes; Interferon Type I

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APA (6th Edition):

Chowdhury, F. Z. (2013). Regulation of Human Cytotoxic T Lymphocyte Functions by IL-12. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1729

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chowdhury, Fatema Zahra. “Regulation of Human Cytotoxic T Lymphocyte Functions by IL-12.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed January 19, 2020. http://hdl.handle.net/2152.5/1729.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chowdhury, Fatema Zahra. “Regulation of Human Cytotoxic T Lymphocyte Functions by IL-12.” 2013. Web. 19 Jan 2020.

Vancouver:

Chowdhury FZ. Regulation of Human Cytotoxic T Lymphocyte Functions by IL-12. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/2152.5/1729.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chowdhury FZ. Regulation of Human Cytotoxic T Lymphocyte Functions by IL-12. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1729

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

5. Zlatkov, Clare Marie. Serum Amyloid A is a Retinol Binding Protein that Transports Retinol during Bacterial Infection.

Degree: 2014, University of Texas Southwestern Medical Center

 Retinol plays a vital role in the immune response to infection, however it remains unclear which proteins mediate retinol transport during infection. Serum amyloid A… (more)

Subjects/Keywords: Retinol-Binding Proteins; Salmonella Infections; Serum Amyloid A Protein; Vitamin A

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APA (6th Edition):

Zlatkov, C. M. (2014). Serum Amyloid A is a Retinol Binding Protein that Transports Retinol during Bacterial Infection. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3584

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zlatkov, Clare Marie. “Serum Amyloid A is a Retinol Binding Protein that Transports Retinol during Bacterial Infection.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed January 19, 2020. http://hdl.handle.net/2152.5/3584.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zlatkov, Clare Marie. “Serum Amyloid A is a Retinol Binding Protein that Transports Retinol during Bacterial Infection.” 2014. Web. 19 Jan 2020.

Vancouver:

Zlatkov CM. Serum Amyloid A is a Retinol Binding Protein that Transports Retinol during Bacterial Infection. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/2152.5/3584.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zlatkov CM. Serum Amyloid A is a Retinol Binding Protein that Transports Retinol during Bacterial Infection. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3584

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

6. Tyler, Andrew Farley. CD8+ T Cells Are Required for the Therapeutic Action of Glatiramer Acetate in Autoimmune Demyelinating Disease.

Degree: 2013, University of Texas Southwestern Medical Center

 Glatiramer acetate (GA, Copaxone®) is an FDA-approved immunomodulatory therapy for multiple sclerosis (MS), an immune-mediated demyelinating disease of the central nervous system. Our group has… (more)

Subjects/Keywords: CD8-Positive T-Lymphocytes; Immunologic Factors; Multiple Sclerosis, Relapsing-Remitting

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APA (6th Edition):

Tyler, A. F. (2013). CD8+ T Cells Are Required for the Therapeutic Action of Glatiramer Acetate in Autoimmune Demyelinating Disease. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1594

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tyler, Andrew Farley. “CD8+ T Cells Are Required for the Therapeutic Action of Glatiramer Acetate in Autoimmune Demyelinating Disease.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed January 19, 2020. http://hdl.handle.net/2152.5/1594.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tyler, Andrew Farley. “CD8+ T Cells Are Required for the Therapeutic Action of Glatiramer Acetate in Autoimmune Demyelinating Disease.” 2013. Web. 19 Jan 2020.

Vancouver:

Tyler AF. CD8+ T Cells Are Required for the Therapeutic Action of Glatiramer Acetate in Autoimmune Demyelinating Disease. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/2152.5/1594.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tyler AF. CD8+ T Cells Are Required for the Therapeutic Action of Glatiramer Acetate in Autoimmune Demyelinating Disease. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1594

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

7. Hu, Wei. Toll Like Receptor Dependent Control of T Helper 17 Cell Differentiation and Inflammasome Activation.

Degree: 2014, University of Texas Southwestern Medical Center

 Activation of pattern recognition receptors on dendritic cells (DCs) and macrophages leads to secretion of cytokines that control activation and differentiation of CD4+ T cells.… (more)

Subjects/Keywords: Carrier Proteins; Inflammasomes; Interleukin-1 Receptor-Associated Kinases; Th17 Cells; Toll-Like Receptors

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APA (6th Edition):

Hu, W. (2014). Toll Like Receptor Dependent Control of T Helper 17 Cell Differentiation and Inflammasome Activation. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3315

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hu, Wei. “Toll Like Receptor Dependent Control of T Helper 17 Cell Differentiation and Inflammasome Activation.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed January 19, 2020. http://hdl.handle.net/2152.5/3315.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hu, Wei. “Toll Like Receptor Dependent Control of T Helper 17 Cell Differentiation and Inflammasome Activation.” 2014. Web. 19 Jan 2020.

Vancouver:

Hu W. Toll Like Receptor Dependent Control of T Helper 17 Cell Differentiation and Inflammasome Activation. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/2152.5/3315.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hu W. Toll Like Receptor Dependent Control of T Helper 17 Cell Differentiation and Inflammasome Activation. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3315

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

8. LaRanger, Ryan. Differentiation of Normal and Cystic Fibrosis Human Lung Epithelial Cells in a Decellularized and Reconstituted Mouse Lung.

Degree: 2017, University of Texas Southwestern Medical Center

 Engineered lung tissue may eventually address the chronic shortage of transplantable lung tissue and permit modeling of lung disease in a controlled ex vivo environment.… (more)

Subjects/Keywords: Cell Culture Techniques; Epithelial Cells; Lung; rho-Associated Kinases; Tissue Engineering

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APA (6th Edition):

LaRanger, R. (2017). Differentiation of Normal and Cystic Fibrosis Human Lung Epithelial Cells in a Decellularized and Reconstituted Mouse Lung. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/6619

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

LaRanger, Ryan. “Differentiation of Normal and Cystic Fibrosis Human Lung Epithelial Cells in a Decellularized and Reconstituted Mouse Lung.” 2017. Thesis, University of Texas Southwestern Medical Center. Accessed January 19, 2020. http://hdl.handle.net/2152.5/6619.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

LaRanger, Ryan. “Differentiation of Normal and Cystic Fibrosis Human Lung Epithelial Cells in a Decellularized and Reconstituted Mouse Lung.” 2017. Web. 19 Jan 2020.

Vancouver:

LaRanger R. Differentiation of Normal and Cystic Fibrosis Human Lung Epithelial Cells in a Decellularized and Reconstituted Mouse Lung. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2017. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/2152.5/6619.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

LaRanger R. Differentiation of Normal and Cystic Fibrosis Human Lung Epithelial Cells in a Decellularized and Reconstituted Mouse Lung. [Thesis]. University of Texas Southwestern Medical Center; 2017. Available from: http://hdl.handle.net/2152.5/6619

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

9. Troutman, Ty Dale. B-Cell Adapter for Phosphoinositide 3-Kinase Is a Signaling Adapter in the Toll-Like Receptor/Interleukin-1 Receptor Superfamily.

Degree: 2014, University of Texas Southwestern Medical Center

 Toll-like receptor (TLR)/Interleukin-1 receptor (IL1R) superfamily members share signaling components and (with the exception of TLR3) depend on the adapter myeloid differentiation primary response gene… (more)

Subjects/Keywords: Adaptor Proteins, Signal Transducing; B-Lymphocytes; Phosphatidylinositol 3-Kinases; Proto-Oncogene Proteins c-akt; Signal Transduction; Toll-Like Receptors

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APA (6th Edition):

Troutman, T. D. (2014). B-Cell Adapter for Phosphoinositide 3-Kinase Is a Signaling Adapter in the Toll-Like Receptor/Interleukin-1 Receptor Superfamily. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3328

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Troutman, Ty Dale. “B-Cell Adapter for Phosphoinositide 3-Kinase Is a Signaling Adapter in the Toll-Like Receptor/Interleukin-1 Receptor Superfamily.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed January 19, 2020. http://hdl.handle.net/2152.5/3328.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Troutman, Ty Dale. “B-Cell Adapter for Phosphoinositide 3-Kinase Is a Signaling Adapter in the Toll-Like Receptor/Interleukin-1 Receptor Superfamily.” 2014. Web. 19 Jan 2020.

Vancouver:

Troutman TD. B-Cell Adapter for Phosphoinositide 3-Kinase Is a Signaling Adapter in the Toll-Like Receptor/Interleukin-1 Receptor Superfamily. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/2152.5/3328.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Troutman TD. B-Cell Adapter for Phosphoinositide 3-Kinase Is a Signaling Adapter in the Toll-Like Receptor/Interleukin-1 Receptor Superfamily. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3328

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

10. Velmurugan, Ramraj. Using Advanced Microscopy Techniques for the Study of Macrophage-Cancer Cell Interactions in the Presence of Therapeutic Antibodies.

Degree: 2017, University of Texas Southwestern Medical Center

The file named "VELMURUGAN-DISSERTATION-2017.pdf" is the primary dissertation file. Eight (8) supplemental files are also available and may be viewed individually.

The use of monoclonal… (more)

Subjects/Keywords: Antibodies; Breast Neoplasms; Macrophages; Phagocytosis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Velmurugan, R. (2017). Using Advanced Microscopy Techniques for the Study of Macrophage-Cancer Cell Interactions in the Presence of Therapeutic Antibodies. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/7195

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Velmurugan, Ramraj. “Using Advanced Microscopy Techniques for the Study of Macrophage-Cancer Cell Interactions in the Presence of Therapeutic Antibodies.” 2017. Thesis, University of Texas Southwestern Medical Center. Accessed January 19, 2020. http://hdl.handle.net/2152.5/7195.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Velmurugan, Ramraj. “Using Advanced Microscopy Techniques for the Study of Macrophage-Cancer Cell Interactions in the Presence of Therapeutic Antibodies.” 2017. Web. 19 Jan 2020.

Vancouver:

Velmurugan R. Using Advanced Microscopy Techniques for the Study of Macrophage-Cancer Cell Interactions in the Presence of Therapeutic Antibodies. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2017. [cited 2020 Jan 19]. Available from: http://hdl.handle.net/2152.5/7195.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Velmurugan R. Using Advanced Microscopy Techniques for the Study of Macrophage-Cancer Cell Interactions in the Presence of Therapeutic Antibodies. [Thesis]. University of Texas Southwestern Medical Center; 2017. Available from: http://hdl.handle.net/2152.5/7195

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.