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You searched for +publisher:"University of Texas Southwestern Medical Center" +contributor:("Olson, Eric N."). Showing records 1 – 30 of 50 total matches.

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University of Texas Southwestern Medical Center

1. Carrer, Michele. Regulation of Metabolic Processes by Micrornas and Class I Histone Deacetylases.

Degree: 2013, University of Texas Southwestern Medical Center

 Obesity is a medical condition resulting from accumulation of excess body fat that affects more than 30% of the adult population in the U.S. Obesity-related… (more)

Subjects/Keywords: Adipocytes; MicroRNAs; Mitochondria

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Carrer, M. (2013). Regulation of Metabolic Processes by Micrornas and Class I Histone Deacetylases. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1232

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Carrer, Michele. “Regulation of Metabolic Processes by Micrornas and Class I Histone Deacetylases.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed January 28, 2021. http://hdl.handle.net/2152.5/1232.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Carrer, Michele. “Regulation of Metabolic Processes by Micrornas and Class I Histone Deacetylases.” 2013. Web. 28 Jan 2021.

Vancouver:

Carrer M. Regulation of Metabolic Processes by Micrornas and Class I Histone Deacetylases. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/2152.5/1232.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Carrer M. Regulation of Metabolic Processes by Micrornas and Class I Histone Deacetylases. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1232

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

2. Johnson, Brett A. Regulation of Exercise-Dependent Cardiac Growth by MicroRNAs.

Degree: 2013, University of Texas Southwestern Medical Center

 The heart is an adaptive organ which undergoes pathological or physiological remodeling in response to a variety of stimuli to meet the demands of the… (more)

Subjects/Keywords: MicroRNAs; Heart; Exercise

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APA (6th Edition):

Johnson, B. A. (2013). Regulation of Exercise-Dependent Cardiac Growth by MicroRNAs. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1219

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Johnson, Brett A. “Regulation of Exercise-Dependent Cardiac Growth by MicroRNAs.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed January 28, 2021. http://hdl.handle.net/2152.5/1219.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Johnson, Brett A. “Regulation of Exercise-Dependent Cardiac Growth by MicroRNAs.” 2013. Web. 28 Jan 2021.

Vancouver:

Johnson BA. Regulation of Exercise-Dependent Cardiac Growth by MicroRNAs. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/2152.5/1219.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Johnson BA. Regulation of Exercise-Dependent Cardiac Growth by MicroRNAs. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1219

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

3. Chaturvedi, Dhananjay. Investigations into the Role of Paf1 Complex Proteins in Drosophila Ovaries.

Degree: 2014, University of Texas Southwestern Medical Center

 Over the past decade considerable interest has grown in epigenetics and chromatin modifications. The ability of two cells with identical genomes to have entirely different… (more)

Subjects/Keywords: Chromatin; Drosophila; Ovary

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APA (6th Edition):

Chaturvedi, D. (2014). Investigations into the Role of Paf1 Complex Proteins in Drosophila Ovaries. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1480

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chaturvedi, Dhananjay. “Investigations into the Role of Paf1 Complex Proteins in Drosophila Ovaries.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed January 28, 2021. http://hdl.handle.net/2152.5/1480.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chaturvedi, Dhananjay. “Investigations into the Role of Paf1 Complex Proteins in Drosophila Ovaries.” 2014. Web. 28 Jan 2021.

Vancouver:

Chaturvedi D. Investigations into the Role of Paf1 Complex Proteins in Drosophila Ovaries. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/2152.5/1480.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chaturvedi D. Investigations into the Role of Paf1 Complex Proteins in Drosophila Ovaries. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/1480

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

4. Rong, Ziye. Regulation of Sister Chromatid by the Acetyltransferase Naa50.

Degree: 2016, University of Texas Southwestern Medical Center

 During the cell cycle, sister-chromatid cohesion tethers sister chromatids together from S phase to the metaphase-anaphase transition and ensures accurate chromosome segregation of chromatids into… (more)

Subjects/Keywords: Cell Cycle Proteins; Chromatids; Chromosomal Proteins, Non-Histone; Chromosomes, Human; Mitosis; N-Terminal Acetyltransferase A; N-Terminal Acetyltransferase E; S Phase

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APA (6th Edition):

Rong, Z. (2016). Regulation of Sister Chromatid by the Acetyltransferase Naa50. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/5724

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rong, Ziye. “Regulation of Sister Chromatid by the Acetyltransferase Naa50.” 2016. Thesis, University of Texas Southwestern Medical Center. Accessed January 28, 2021. http://hdl.handle.net/2152.5/5724.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rong, Ziye. “Regulation of Sister Chromatid by the Acetyltransferase Naa50.” 2016. Web. 28 Jan 2021.

Vancouver:

Rong Z. Regulation of Sister Chromatid by the Acetyltransferase Naa50. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2016. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/2152.5/5724.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rong Z. Regulation of Sister Chromatid by the Acetyltransferase Naa50. [Thesis]. University of Texas Southwestern Medical Center; 2016. Available from: http://hdl.handle.net/2152.5/5724

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

5. Azizoglu, Dicle Berfin. Afadin and RhoA Control Pancreatic Endocrine Mass via Lumen Morphogenesis.

Degree: 2018, University of Texas Southwestern Medical Center

 Pancreas is a vital organ responsible for digestion and blood glucose homeostasis in vertebrates. Pancreatic endocrine cells secrete hormones that regulate blood glucose levels, while… (more)

Subjects/Keywords: Microfilament Proteins; Morphogenesis; Pancreas; rho GTP-Binding Proteins

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APA (6th Edition):

Azizoglu, D. B. (2018). Afadin and RhoA Control Pancreatic Endocrine Mass via Lumen Morphogenesis. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/8313

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Azizoglu, Dicle Berfin. “Afadin and RhoA Control Pancreatic Endocrine Mass via Lumen Morphogenesis.” 2018. Thesis, University of Texas Southwestern Medical Center. Accessed January 28, 2021. http://hdl.handle.net/2152.5/8313.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Azizoglu, Dicle Berfin. “Afadin and RhoA Control Pancreatic Endocrine Mass via Lumen Morphogenesis.” 2018. Web. 28 Jan 2021.

Vancouver:

Azizoglu DB. Afadin and RhoA Control Pancreatic Endocrine Mass via Lumen Morphogenesis. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2018. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/2152.5/8313.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Azizoglu DB. Afadin and RhoA Control Pancreatic Endocrine Mass via Lumen Morphogenesis. [Thesis]. University of Texas Southwestern Medical Center; 2018. Available from: http://hdl.handle.net/2152.5/8313

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

6. DeLeon, Susan M. Regulation of Striated Muscle Metabolism and Function by Mediator Complex.

Degree: 2015, University of Texas Southwestern Medical Center

 Mediator is a multi-protein complex that links signal-dependent transcription factors and upstream regulators with polymerase II (Pol II) to form a stable and efficient pre-initiation… (more)

Subjects/Keywords: Mediator Complex; Muscle, Striated; Muscle Development

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APA (6th Edition):

DeLeon, S. M. (2015). Regulation of Striated Muscle Metabolism and Function by Mediator Complex. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4199

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

DeLeon, Susan M. “Regulation of Striated Muscle Metabolism and Function by Mediator Complex.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed January 28, 2021. http://hdl.handle.net/2152.5/4199.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

DeLeon, Susan M. “Regulation of Striated Muscle Metabolism and Function by Mediator Complex.” 2015. Web. 28 Jan 2021.

Vancouver:

DeLeon SM. Regulation of Striated Muscle Metabolism and Function by Mediator Complex. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/2152.5/4199.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

DeLeon SM. Regulation of Striated Muscle Metabolism and Function by Mediator Complex. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4199

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

7. Liu, Siqi 1983-. Key Roles of Ubiquitination and Phosphorylation in RIG-I/MAVS Viral Sensing Pathway.

Degree: 2013, University of Texas Southwestern Medical Center

 RNA virus infections are detected by the RIG-I family of receptors, which induce the production of type-I interferons (IFNs) and other antiviral molecules through the… (more)

Subjects/Keywords: Adaptor Proteins, Signal Transducing; Interferon Regulatory Factor-3; Signal Transduction

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APA (6th Edition):

Liu, S. 1. (2013). Key Roles of Ubiquitination and Phosphorylation in RIG-I/MAVS Viral Sensing Pathway. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-108

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Liu, Siqi 1983-. “Key Roles of Ubiquitination and Phosphorylation in RIG-I/MAVS Viral Sensing Pathway.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed January 28, 2021. http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-108.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Liu, Siqi 1983-. “Key Roles of Ubiquitination and Phosphorylation in RIG-I/MAVS Viral Sensing Pathway.” 2013. Web. 28 Jan 2021.

Vancouver:

Liu S1. Key Roles of Ubiquitination and Phosphorylation in RIG-I/MAVS Viral Sensing Pathway. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-108.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Liu S1. Key Roles of Ubiquitination and Phosphorylation in RIG-I/MAVS Viral Sensing Pathway. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-108

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

8. Ramalingam, Harini. Balancing Renewal and Differentiation of Progenitor Cells in the Developing Kidney.

Degree: 2017, University of Texas Southwestern Medical Center

 Mammalian kidneys perform the important function of blood filtration. All the filtered wastes are concentrated into urine and excreted from the body. The kidney performs… (more)

Subjects/Keywords: Cell Differentiation; Cellular Microenvironment; Gene Expression Regulation; Nephrons; Stem Cells; Urogenital System

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APA (6th Edition):

Ramalingam, H. (2017). Balancing Renewal and Differentiation of Progenitor Cells in the Developing Kidney. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/7746

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ramalingam, Harini. “Balancing Renewal and Differentiation of Progenitor Cells in the Developing Kidney.” 2017. Thesis, University of Texas Southwestern Medical Center. Accessed January 28, 2021. http://hdl.handle.net/2152.5/7746.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ramalingam, Harini. “Balancing Renewal and Differentiation of Progenitor Cells in the Developing Kidney.” 2017. Web. 28 Jan 2021.

Vancouver:

Ramalingam H. Balancing Renewal and Differentiation of Progenitor Cells in the Developing Kidney. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2017. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/2152.5/7746.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ramalingam H. Balancing Renewal and Differentiation of Progenitor Cells in the Developing Kidney. [Thesis]. University of Texas Southwestern Medical Center; 2017. Available from: http://hdl.handle.net/2152.5/7746

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

9. Long, Chengzu. Prevention of Muscular Dystrophy in Mice by Gene Editing.

Degree: 2014, University of Texas Southwestern Medical Center

 Duchenne muscular dystrophy (DMD) is an inherited X-linked disease caused by mutations in the gene encoding dystrophin, a protein required for muscle fiber integrity. DMD… (more)

Subjects/Keywords: CRISPR-Cas Systems; Dystrophin; Gene Targeting; Muscular Dystrophy, Duchenne

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APA (6th Edition):

Long, C. (2014). Prevention of Muscular Dystrophy in Mice by Gene Editing. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3953

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Long, Chengzu. “Prevention of Muscular Dystrophy in Mice by Gene Editing.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed January 28, 2021. http://hdl.handle.net/2152.5/3953.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Long, Chengzu. “Prevention of Muscular Dystrophy in Mice by Gene Editing.” 2014. Web. 28 Jan 2021.

Vancouver:

Long C. Prevention of Muscular Dystrophy in Mice by Gene Editing. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/2152.5/3953.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Long C. Prevention of Muscular Dystrophy in Mice by Gene Editing. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3953

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

10. Zhou, Huanyu. Molecular Regulation of Direct Cardiac Reprogramming.

Degree: 2017, University of Texas Southwestern Medical Center

 A heart attack (also known as myocardial infarction, MI) happens when the flow of blood to the heart is blocked. A massive heart attack can… (more)

Subjects/Keywords: Cellular Reprogramming; Fibroblasts; Myocytes, Cardiac; Proto-Oncogene Proteins c-akt; Transcription, Genetic

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APA (6th Edition):

Zhou, H. (2017). Molecular Regulation of Direct Cardiac Reprogramming. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/7208

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhou, Huanyu. “Molecular Regulation of Direct Cardiac Reprogramming.” 2017. Thesis, University of Texas Southwestern Medical Center. Accessed January 28, 2021. http://hdl.handle.net/2152.5/7208.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhou, Huanyu. “Molecular Regulation of Direct Cardiac Reprogramming.” 2017. Web. 28 Jan 2021.

Vancouver:

Zhou H. Molecular Regulation of Direct Cardiac Reprogramming. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2017. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/2152.5/7208.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhou H. Molecular Regulation of Direct Cardiac Reprogramming. [Thesis]. University of Texas Southwestern Medical Center; 2017. Available from: http://hdl.handle.net/2152.5/7208

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

11. Williams, Koriand'r. Antagonistic Roles of miR-199a-3p/miR-214 and the miR-200 Family in the Regulation of Uterine Contractility During Pregnancy and Labor.

Degree: 2014, University of Texas Southwestern Medical Center

 Progesterone (P4) and estradiol-17β (E2) play critical and opposing roles in regulating myometrial quiescence and contractility during pregnancy and labor (Kamel et al., 2010). While… (more)

Subjects/Keywords: Estrogens; Labor, Obstetric; MicroRNAs; Progesterone

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APA (6th Edition):

Williams, K. (2014). Antagonistic Roles of miR-199a-3p/miR-214 and the miR-200 Family in the Regulation of Uterine Contractility During Pregnancy and Labor. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1413

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Williams, Koriand'r. “Antagonistic Roles of miR-199a-3p/miR-214 and the miR-200 Family in the Regulation of Uterine Contractility During Pregnancy and Labor.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed January 28, 2021. http://hdl.handle.net/2152.5/1413.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Williams, Koriand'r. “Antagonistic Roles of miR-199a-3p/miR-214 and the miR-200 Family in the Regulation of Uterine Contractility During Pregnancy and Labor.” 2014. Web. 28 Jan 2021.

Vancouver:

Williams K. Antagonistic Roles of miR-199a-3p/miR-214 and the miR-200 Family in the Regulation of Uterine Contractility During Pregnancy and Labor. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/2152.5/1413.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Williams K. Antagonistic Roles of miR-199a-3p/miR-214 and the miR-200 Family in the Regulation of Uterine Contractility During Pregnancy and Labor. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/1413

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

12. Koo, Yeon Seung. Blood Vessel Development.

Degree: 2014, University of Texas Southwestern Medical Center

 Cardiovascular system is the first developing functional organ in vertebrates, and one of the fundamental organ systems through adulthoods. Cardiovascular function depends on patent blood… (more)

Subjects/Keywords: Blood Vessels; Endothelium, Vascular; GTPase-Activating Proteins; Intracellular Signaling Peptides and Proteins

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APA (6th Edition):

Koo, Y. S. (2014). Blood Vessel Development. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3949

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Koo, Yeon Seung. “Blood Vessel Development.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed January 28, 2021. http://hdl.handle.net/2152.5/3949.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Koo, Yeon Seung. “Blood Vessel Development.” 2014. Web. 28 Jan 2021.

Vancouver:

Koo YS. Blood Vessel Development. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/2152.5/3949.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Koo YS. Blood Vessel Development. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3949

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

13. Garg, Ankit. Discovery of New Regulatory Proteins and Mechanisms in Muscle Biology and Disease.

Degree: 2014, University of Texas Southwestern Medical Center

 In an effort to discover new regulators of muscle function, we identified a novel muscle-specific protein, Klhl40. Genetic deletion of Klhl40 in mice results in… (more)

Subjects/Keywords: MEF2 Transcription Factors; Microfilament Proteins; Muscle Proteins; Myofibrils; Myopathies, Nemaline

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APA (6th Edition):

Garg, A. (2014). Discovery of New Regulatory Proteins and Mechanisms in Muscle Biology and Disease. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3340

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Garg, Ankit. “Discovery of New Regulatory Proteins and Mechanisms in Muscle Biology and Disease.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed January 28, 2021. http://hdl.handle.net/2152.5/3340.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Garg, Ankit. “Discovery of New Regulatory Proteins and Mechanisms in Muscle Biology and Disease.” 2014. Web. 28 Jan 2021.

Vancouver:

Garg A. Discovery of New Regulatory Proteins and Mechanisms in Muscle Biology and Disease. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/2152.5/3340.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Garg A. Discovery of New Regulatory Proteins and Mechanisms in Muscle Biology and Disease. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3340

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

14. Jiang, Yindi. Roles of HDACs and MEF2 in Adult Hippocampal Neurogenesis.

Degree: 2014, University of Texas Southwestern Medical Center

 The maintenance of the resident adult neural stem/progenitor cell (NSPC) pool depends on the precise balance of proliferation, differentiation, and maintenance of the undifferentiated state.… (more)

Subjects/Keywords: Adult Stem Cells; CDC2 Protein Kinase; G2 Phase; Histone Deacetylases; Mitosis; Neural Stem Cells

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APA (6th Edition):

Jiang, Y. (2014). Roles of HDACs and MEF2 in Adult Hippocampal Neurogenesis. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3947

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jiang, Yindi. “Roles of HDACs and MEF2 in Adult Hippocampal Neurogenesis.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed January 28, 2021. http://hdl.handle.net/2152.5/3947.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jiang, Yindi. “Roles of HDACs and MEF2 in Adult Hippocampal Neurogenesis.” 2014. Web. 28 Jan 2021.

Vancouver:

Jiang Y. Roles of HDACs and MEF2 in Adult Hippocampal Neurogenesis. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/2152.5/3947.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jiang Y. Roles of HDACs and MEF2 in Adult Hippocampal Neurogenesis. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3947

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

15. Peyer, James Gregory. Refining Our Understanding of the Hematopoietic Stem Cell Niche.

Degree: 2015, University of Texas Southwestern Medical Center

 A major therapeutic goal of studying blood-forming hematopoietic stem cells (HSCs) is to understand the mechanisms by which HSCs are maintained in the bone marrow,… (more)

Subjects/Keywords: Bone Marrow Cells; Bone Marrow Transplantation; Hematopoietic Stem Cells; Stem Cell Niche

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APA (6th Edition):

Peyer, J. G. (2015). Refining Our Understanding of the Hematopoietic Stem Cell Niche. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4450

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Peyer, James Gregory. “Refining Our Understanding of the Hematopoietic Stem Cell Niche.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed January 28, 2021. http://hdl.handle.net/2152.5/4450.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Peyer, James Gregory. “Refining Our Understanding of the Hematopoietic Stem Cell Niche.” 2015. Web. 28 Jan 2021.

Vancouver:

Peyer JG. Refining Our Understanding of the Hematopoietic Stem Cell Niche. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/2152.5/4450.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Peyer JG. Refining Our Understanding of the Hematopoietic Stem Cell Niche. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4450

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

16. Zhang, Yu. Prevention of Duchenne Muscular Dystrophy by CRISPR/Cas Therapeutic Genome Editing.

Degree: 2020, University of Texas Southwestern Medical Center

 Skeletal muscle is one of the largest tissues in the human body and hence muscle diseases caused by genetic mutations have a profound and systemic… (more)

Subjects/Keywords: CRISPR-Cas Systems; Gene Editing; Genetic Therapy; Muscular Dystrophies; Muscular Dystrophy, Duchenne; Myocytes, Cardiac

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APA (6th Edition):

Zhang, Y. (2020). Prevention of Duchenne Muscular Dystrophy by CRISPR/Cas Therapeutic Genome Editing. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/8320

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhang, Yu. “Prevention of Duchenne Muscular Dystrophy by CRISPR/Cas Therapeutic Genome Editing.” 2020. Thesis, University of Texas Southwestern Medical Center. Accessed January 28, 2021. http://hdl.handle.net/2152.5/8320.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhang, Yu. “Prevention of Duchenne Muscular Dystrophy by CRISPR/Cas Therapeutic Genome Editing.” 2020. Web. 28 Jan 2021.

Vancouver:

Zhang Y. Prevention of Duchenne Muscular Dystrophy by CRISPR/Cas Therapeutic Genome Editing. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2020. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/2152.5/8320.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhang Y. Prevention of Duchenne Muscular Dystrophy by CRISPR/Cas Therapeutic Genome Editing. [Thesis]. University of Texas Southwestern Medical Center; 2020. Available from: http://hdl.handle.net/2152.5/8320

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

17. Rifki, Oktay Feridun. RalGDS-Dependent Cardiomyocyte Autophagy Is Necessary for Load-Induced Ventricular Hypertrophy.

Degree: 2014, University of Texas Southwestern Medical Center

 Recent work has demonstrated that autophagy, a phylogenetically conserved, lysosome-mediated pathway of protein degradation, is a key participant in pathological cardiac remodeling. One common feature… (more)

Subjects/Keywords: Autophagy; Cardiomegaly; Myocytes, Cardiac; ral Guanine Nucleotide Exchange Factor

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APA (6th Edition):

Rifki, O. F. (2014). RalGDS-Dependent Cardiomyocyte Autophagy Is Necessary for Load-Induced Ventricular Hypertrophy. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3324

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rifki, Oktay Feridun. “RalGDS-Dependent Cardiomyocyte Autophagy Is Necessary for Load-Induced Ventricular Hypertrophy.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed January 28, 2021. http://hdl.handle.net/2152.5/3324.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rifki, Oktay Feridun. “RalGDS-Dependent Cardiomyocyte Autophagy Is Necessary for Load-Induced Ventricular Hypertrophy.” 2014. Web. 28 Jan 2021.

Vancouver:

Rifki OF. RalGDS-Dependent Cardiomyocyte Autophagy Is Necessary for Load-Induced Ventricular Hypertrophy. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/2152.5/3324.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rifki OF. RalGDS-Dependent Cardiomyocyte Autophagy Is Necessary for Load-Induced Ventricular Hypertrophy. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3324

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

18. Nelson, Benjamin Rhett. Discovery and Characterization of Genes Involved in Muscle Calcium Handling.

Degree: 2015, University of Texas Southwestern Medical Center

 Muscle tissue requires continuous cycling of calcium release and clearance to generate and sustain contraction. When the plasma membrane of a muscle fiber becomes electrically… (more)

Subjects/Keywords: Muscle Contraction; Muscle, Skeletal; Myocytes, Cardiac; Peptides; Sarcoplasmic Reticulum Calcium-Transporting ATPases

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APA (6th Edition):

Nelson, B. R. (2015). Discovery and Characterization of Genes Involved in Muscle Calcium Handling. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/8298

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nelson, Benjamin Rhett. “Discovery and Characterization of Genes Involved in Muscle Calcium Handling.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed January 28, 2021. http://hdl.handle.net/2152.5/8298.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nelson, Benjamin Rhett. “Discovery and Characterization of Genes Involved in Muscle Calcium Handling.” 2015. Web. 28 Jan 2021.

Vancouver:

Nelson BR. Discovery and Characterization of Genes Involved in Muscle Calcium Handling. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/2152.5/8298.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nelson BR. Discovery and Characterization of Genes Involved in Muscle Calcium Handling. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/8298

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

19. Gui, Xiang. The Mechanism and Function of Autophagy Induction by Cytosolic DNA.

Degree: 2018, University of Texas Southwestern Medical Center

The file named "GUI-DISSERTATION-2018.pdf" is the primary dissertation file. The file named "Archive.zip" is a zipped file folder that contains six (6) movie files (AVI… (more)

Subjects/Keywords: Autophagy; Membrane Proteins; Nucleotidyltransferases; Signal Transduction

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APA (6th Edition):

Gui, X. (2018). The Mechanism and Function of Autophagy Induction by Cytosolic DNA. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/8784

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gui, Xiang. “The Mechanism and Function of Autophagy Induction by Cytosolic DNA.” 2018. Thesis, University of Texas Southwestern Medical Center. Accessed January 28, 2021. http://hdl.handle.net/2152.5/8784.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gui, Xiang. “The Mechanism and Function of Autophagy Induction by Cytosolic DNA.” 2018. Web. 28 Jan 2021.

Vancouver:

Gui X. The Mechanism and Function of Autophagy Induction by Cytosolic DNA. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2018. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/2152.5/8784.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gui X. The Mechanism and Function of Autophagy Induction by Cytosolic DNA. [Thesis]. University of Texas Southwestern Medical Center; 2018. Available from: http://hdl.handle.net/2152.5/8784

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

20. Granados, Valerie Ann. EGFR and Akt Signaling in Rhabdomyosarcoma Pathogenesis.

Degree: 2018, University of Texas Southwestern Medical Center

 Rhabdomyosarcoma is an aggressive soft-tissue malignancy comprised microscopically of neoplastic skeletal muscle-lineage precursors that fail to exit the cell-cycle and fuse into syncytial muscle -… (more)

Subjects/Keywords: Antineoplastic Agents; Cell Differentiation; Drosophila; Drosophila Proteins; Myoblasts; Rhabdomyosarcoma

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APA (6th Edition):

Granados, V. A. (2018). EGFR and Akt Signaling in Rhabdomyosarcoma Pathogenesis. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/8797

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Granados, Valerie Ann. “EGFR and Akt Signaling in Rhabdomyosarcoma Pathogenesis.” 2018. Thesis, University of Texas Southwestern Medical Center. Accessed January 28, 2021. http://hdl.handle.net/2152.5/8797.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Granados, Valerie Ann. “EGFR and Akt Signaling in Rhabdomyosarcoma Pathogenesis.” 2018. Web. 28 Jan 2021.

Vancouver:

Granados VA. EGFR and Akt Signaling in Rhabdomyosarcoma Pathogenesis. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2018. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/2152.5/8797.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Granados VA. EGFR and Akt Signaling in Rhabdomyosarcoma Pathogenesis. [Thesis]. University of Texas Southwestern Medical Center; 2018. Available from: http://hdl.handle.net/2152.5/8797

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

21. Kutluk Cenik, Bercin. Regulation of Skeletal Muscle Development and Disease by an Actin-Dependent Transcriptional Circuit.

Degree: 2018, University of Texas Southwestern Medical Center

 Congenital myopathies are a group of diseases that primarily affect skeletal muscle and cause muscle weakness that manifests at birth. With an incidence of 6… (more)

Subjects/Keywords: MEF2 Transcription Factors; Microfilament Proteins; Muscle, Skeletal; Myopathies, Nemaline; Trans-Activators; Transcription Factors

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APA (6th Edition):

Kutluk Cenik, B. (2018). Regulation of Skeletal Muscle Development and Disease by an Actin-Dependent Transcriptional Circuit. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/8783

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kutluk Cenik, Bercin. “Regulation of Skeletal Muscle Development and Disease by an Actin-Dependent Transcriptional Circuit.” 2018. Thesis, University of Texas Southwestern Medical Center. Accessed January 28, 2021. http://hdl.handle.net/2152.5/8783.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kutluk Cenik, Bercin. “Regulation of Skeletal Muscle Development and Disease by an Actin-Dependent Transcriptional Circuit.” 2018. Web. 28 Jan 2021.

Vancouver:

Kutluk Cenik B. Regulation of Skeletal Muscle Development and Disease by an Actin-Dependent Transcriptional Circuit. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2018. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/2152.5/8783.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kutluk Cenik B. Regulation of Skeletal Muscle Development and Disease by an Actin-Dependent Transcriptional Circuit. [Thesis]. University of Texas Southwestern Medical Center; 2018. Available from: http://hdl.handle.net/2152.5/8783

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

22. Bhargava, Varsha. Discovering GCNA: A Novel Regulator of Germline Genomic Stability.

Degree: 2018, University of Texas Southwestern Medical Center

 Germ cells transfer genetic information across generations. Any change in germ line DNA is inherited by succeeding generations. Therefore, germ cell DNA must be protected… (more)

Subjects/Keywords: DNA Damage; DNA Repair; DNA Replication; Fertility; Genomic Instability; Nuclear Proteins; Peptide Hydrolases

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APA (6th Edition):

Bhargava, V. (2018). Discovering GCNA: A Novel Regulator of Germline Genomic Stability. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/9299

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bhargava, Varsha. “Discovering GCNA: A Novel Regulator of Germline Genomic Stability.” 2018. Thesis, University of Texas Southwestern Medical Center. Accessed January 28, 2021. http://hdl.handle.net/2152.5/9299.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bhargava, Varsha. “Discovering GCNA: A Novel Regulator of Germline Genomic Stability.” 2018. Web. 28 Jan 2021.

Vancouver:

Bhargava V. Discovering GCNA: A Novel Regulator of Germline Genomic Stability. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2018. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/2152.5/9299.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bhargava V. Discovering GCNA: A Novel Regulator of Germline Genomic Stability. [Thesis]. University of Texas Southwestern Medical Center; 2018. Available from: http://hdl.handle.net/2152.5/9299

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

23. Chen, Jueqi. Biochemical Dissection of Innate Immune Signaling Mechanisms Mediated by MAVS and Inflammasomes.

Degree: 2014, University of Texas Southwestern Medical Center

The general metadata  – e.g., title, author, abstract, subject headings, etc.  – is publicly available, but access to the submitted files is restricted to UT… (more)

Subjects/Keywords: Adaptor Proteins, Signal Transducing; Sendai virus; Signal Transduction; Ubiquitin-Protein Ligases

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APA (6th Edition):

Chen, J. (2014). Biochemical Dissection of Innate Immune Signaling Mechanisms Mediated by MAVS and Inflammasomes. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/7063

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Jueqi. “Biochemical Dissection of Innate Immune Signaling Mechanisms Mediated by MAVS and Inflammasomes.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed January 28, 2021. http://hdl.handle.net/2152.5/7063.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Jueqi. “Biochemical Dissection of Innate Immune Signaling Mechanisms Mediated by MAVS and Inflammasomes.” 2014. Web. 28 Jan 2021.

Vancouver:

Chen J. Biochemical Dissection of Innate Immune Signaling Mechanisms Mediated by MAVS and Inflammasomes. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/2152.5/7063.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen J. Biochemical Dissection of Innate Immune Signaling Mechanisms Mediated by MAVS and Inflammasomes. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/7063

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

24. Patrick, David M. Regulation of Cellular Growth and Differentiation by MicroRNAs -21 and -451.

Degree: 2013, University of Texas Southwestern Medical Center

 MicroRNAs are small RNAs approximately 20-24 nucleotides in length that are conserved throughout evolution. MicroRNA genes are transcribed by RNA polymerase II and are processed… (more)

Subjects/Keywords: Lung Neoplasms; MicroRNAs; Polycythemia Vera

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APA (6th Edition):

Patrick, D. M. (2013). Regulation of Cellular Growth and Differentiation by MicroRNAs -21 and -451. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1705

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Patrick, David M. “Regulation of Cellular Growth and Differentiation by MicroRNAs -21 and -451.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed January 28, 2021. http://hdl.handle.net/2152.5/1705.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Patrick, David M. “Regulation of Cellular Growth and Differentiation by MicroRNAs -21 and -451.” 2013. Web. 28 Jan 2021.

Vancouver:

Patrick DM. Regulation of Cellular Growth and Differentiation by MicroRNAs -21 and -451. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/2152.5/1705.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Patrick DM. Regulation of Cellular Growth and Differentiation by MicroRNAs -21 and -451. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1705

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

25. Smith, Derek Kurtis. Small Molecules Modulate Chromatin Accessibility to Promote NEUROG2-Mediated Fibroblast-to-Neuron Reprogramming.

Degree: 2016, University of Texas Southwestern Medical Center

 The activity of pro-neural signaling molecules and transcription factors is sufficient to induce the transdifferentiation of lineage-restricted fibroblasts into functional neurons; however, a mechanistic model… (more)

Subjects/Keywords: Basic Helix-Loop-Helix Transcription Factors; Cell Transdifferentiation; Cellular Reprogramming; Chromatin Assembly and Disassembly; Fibroblasts; Nerve Tissue Proteins; Neurons

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APA (6th Edition):

Smith, D. K. (2016). Small Molecules Modulate Chromatin Accessibility to Promote NEUROG2-Mediated Fibroblast-to-Neuron Reprogramming. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/5735

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Smith, Derek Kurtis. “Small Molecules Modulate Chromatin Accessibility to Promote NEUROG2-Mediated Fibroblast-to-Neuron Reprogramming.” 2016. Thesis, University of Texas Southwestern Medical Center. Accessed January 28, 2021. http://hdl.handle.net/2152.5/5735.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Smith, Derek Kurtis. “Small Molecules Modulate Chromatin Accessibility to Promote NEUROG2-Mediated Fibroblast-to-Neuron Reprogramming.” 2016. Web. 28 Jan 2021.

Vancouver:

Smith DK. Small Molecules Modulate Chromatin Accessibility to Promote NEUROG2-Mediated Fibroblast-to-Neuron Reprogramming. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2016. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/2152.5/5735.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Smith DK. Small Molecules Modulate Chromatin Accessibility to Promote NEUROG2-Mediated Fibroblast-to-Neuron Reprogramming. [Thesis]. University of Texas Southwestern Medical Center; 2016. Available from: http://hdl.handle.net/2152.5/5735

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

26. Zeve, Daniel. The Response of White Adipose Progenitor Cells to Physiological and Genetic Changes.

Degree: 2013, University of Texas Southwestern Medical Center

 We are in the midst of a dire, unprecedented and global epidemic of obesity and secondary sequelae, most prominently diabetes and hyperlipidemia. Underlying this epidemic… (more)

Subjects/Keywords: Adipocytes; Hyperlipidemias; Stem Cell Niche

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APA (6th Edition):

Zeve, D. (2013). The Response of White Adipose Progenitor Cells to Physiological and Genetic Changes. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1358

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zeve, Daniel. “The Response of White Adipose Progenitor Cells to Physiological and Genetic Changes.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed January 28, 2021. http://hdl.handle.net/2152.5/1358.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zeve, Daniel. “The Response of White Adipose Progenitor Cells to Physiological and Genetic Changes.” 2013. Web. 28 Jan 2021.

Vancouver:

Zeve D. The Response of White Adipose Progenitor Cells to Physiological and Genetic Changes. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/2152.5/1358.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zeve D. The Response of White Adipose Progenitor Cells to Physiological and Genetic Changes. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1358

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

27. Smith, Christopher Lacey. Regulation of Cardiac Fibroblast and Coronary Vascular Smooth Muscle Development by Platelet Derived Growth Factor Receptors.

Degree: 2013, University of Texas Southwestern Medical Center

 Coronary vascular smooth muscle cells (cVSMC) and cardiac fibroblasts are essential for coronary artery development and are important mediators of myocardial pathogenesis. These cells form… (more)

Subjects/Keywords: Epithelial-Mesenchymal Transition; Muscle, Smooth, Vascular; Platelet-Derived Growth Factor; SOX9 Transcription Factor

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Smith, C. L. (2013). Regulation of Cardiac Fibroblast and Coronary Vascular Smooth Muscle Development by Platelet Derived Growth Factor Receptors. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1707

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Smith, Christopher Lacey. “Regulation of Cardiac Fibroblast and Coronary Vascular Smooth Muscle Development by Platelet Derived Growth Factor Receptors.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed January 28, 2021. http://hdl.handle.net/2152.5/1707.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Smith, Christopher Lacey. “Regulation of Cardiac Fibroblast and Coronary Vascular Smooth Muscle Development by Platelet Derived Growth Factor Receptors.” 2013. Web. 28 Jan 2021.

Vancouver:

Smith CL. Regulation of Cardiac Fibroblast and Coronary Vascular Smooth Muscle Development by Platelet Derived Growth Factor Receptors. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/2152.5/1707.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Smith CL. Regulation of Cardiac Fibroblast and Coronary Vascular Smooth Muscle Development by Platelet Derived Growth Factor Receptors. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1707

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

28. Wu, Jiaxi. Innate Immune Sensing and Signaling of Cytosolic DNA.

Degree: 2015, University of Texas Southwestern Medical Center

 In eukaryotic cells, DNA is normally confined within the nucleus and mitochondria. DNA exposed in the cytosol is a danger signal that warns the host… (more)

Subjects/Keywords: Cyclic AMP; Cyclic GMP; Cytosol; DNA; Nucleotidyltransferases; Second Messenger Systems

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wu, J. (2015). Innate Immune Sensing and Signaling of Cytosolic DNA. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4131

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wu, Jiaxi. “Innate Immune Sensing and Signaling of Cytosolic DNA.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed January 28, 2021. http://hdl.handle.net/2152.5/4131.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wu, Jiaxi. “Innate Immune Sensing and Signaling of Cytosolic DNA.” 2015. Web. 28 Jan 2021.

Vancouver:

Wu J. Innate Immune Sensing and Signaling of Cytosolic DNA. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/2152.5/4131.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wu J. Innate Immune Sensing and Signaling of Cytosolic DNA. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4131

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

29. McFarlane, Matthew Ryan. Ghrelin: The Hunger Hormone that Isn't.

Degree: 2014, University of Texas Southwestern Medical Center

 Ghrelin is a 28-amino acid acylated peptide hormone secreted by endocrine cells in the stomach. It was first identified in 1999 and shortly thereafter shown… (more)

Subjects/Keywords: Appetite; Body Weight; Diet, High-Fat; Ghrelin

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APA (6th Edition):

McFarlane, M. R. (2014). Ghrelin: The Hunger Hormone that Isn't. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/5283

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

McFarlane, Matthew Ryan. “Ghrelin: The Hunger Hormone that Isn't.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed January 28, 2021. http://hdl.handle.net/2152.5/5283.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

McFarlane, Matthew Ryan. “Ghrelin: The Hunger Hormone that Isn't.” 2014. Web. 28 Jan 2021.

Vancouver:

McFarlane MR. Ghrelin: The Hunger Hormone that Isn't. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/2152.5/5283.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

McFarlane MR. Ghrelin: The Hunger Hormone that Isn't. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/5283

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

30. He, Danyang. Epigenetic Regulation of Oligodendrocyte Development and Regeneration in the Central Nervous System.

Degree: 2016, University of Texas Southwestern Medical Center

 Oligodendrocytes (OLs) produce myelin sheaths that electrically insulate axons and promote rapid propagation of action potentials in the CNS. The onset and timing of CNS… (more)

Subjects/Keywords: Gene Expression Regulation, Developmental; Myelin Sheath; Oligodendroglia; RNA, Long Noncoding; Transcription Factors

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

He, D. (2016). Epigenetic Regulation of Oligodendrocyte Development and Regeneration in the Central Nervous System. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/6142

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

He, Danyang. “Epigenetic Regulation of Oligodendrocyte Development and Regeneration in the Central Nervous System.” 2016. Thesis, University of Texas Southwestern Medical Center. Accessed January 28, 2021. http://hdl.handle.net/2152.5/6142.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

He, Danyang. “Epigenetic Regulation of Oligodendrocyte Development and Regeneration in the Central Nervous System.” 2016. Web. 28 Jan 2021.

Vancouver:

He D. Epigenetic Regulation of Oligodendrocyte Development and Regeneration in the Central Nervous System. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2016. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/2152.5/6142.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

He D. Epigenetic Regulation of Oligodendrocyte Development and Regeneration in the Central Nervous System. [Thesis]. University of Texas Southwestern Medical Center; 2016. Available from: http://hdl.handle.net/2152.5/6142

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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