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You searched for +publisher:"University of Texas Southwestern Medical Center" +contributor:("Minna, John D."). Showing records 1 – 26 of 26 total matches.

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University of Texas Southwestern Medical Center

1. Sullivan, James Patrick. Identification and Characterization of Non Small Cell Lung Cancer Stem Cells.

Degree: PhD, Cancer Biology, 2011, University of Texas Southwestern Medical Center

 The discovery of rare tumor cells with stem cell features first in myeloproliferative disease and later in solid tumors has emerged as an important area… (more)

Subjects/Keywords: Lung Neoplasms; Aldehyde Dehydrogenase; Carcinoma, Non-Small-Cell Lung; Receptors, Notch

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sullivan, J. P. (2011). Identification and Characterization of Non Small Cell Lung Cancer Stem Cells. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/849

Chicago Manual of Style (16th Edition):

Sullivan, James Patrick. “Identification and Characterization of Non Small Cell Lung Cancer Stem Cells.” 2011. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/849.

MLA Handbook (7th Edition):

Sullivan, James Patrick. “Identification and Characterization of Non Small Cell Lung Cancer Stem Cells.” 2011. Web. 09 Jul 2020.

Vancouver:

Sullivan JP. Identification and Characterization of Non Small Cell Lung Cancer Stem Cells. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2011. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/849.

Council of Science Editors:

Sullivan JP. Identification and Characterization of Non Small Cell Lung Cancer Stem Cells. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2011. Available from: http://hdl.handle.net/2152.5/849


University of Texas Southwestern Medical Center

2. Greer, Rachel Marie. Mechanisms of Sensitization to Chemotherapy in Non-Small Cell Lung Cancer.

Degree: PhD, Cell Regulation, 2012, University of Texas Southwestern Medical Center

 Lung cancer is the leading cause of cancer-related deaths world-wide for men and women, due in part to late detection of disease and inherent resistance… (more)

Subjects/Keywords: Carcinoma, Non-Small-Cell Lung; Tumor Necrosis Factor-alpha; Antineoplastic Combined Chemotherapy Protocols

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APA (6th Edition):

Greer, R. M. (2012). Mechanisms of Sensitization to Chemotherapy in Non-Small Cell Lung Cancer. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1025

Chicago Manual of Style (16th Edition):

Greer, Rachel Marie. “Mechanisms of Sensitization to Chemotherapy in Non-Small Cell Lung Cancer.” 2012. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/1025.

MLA Handbook (7th Edition):

Greer, Rachel Marie. “Mechanisms of Sensitization to Chemotherapy in Non-Small Cell Lung Cancer.” 2012. Web. 09 Jul 2020.

Vancouver:

Greer RM. Mechanisms of Sensitization to Chemotherapy in Non-Small Cell Lung Cancer. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2012. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/1025.

Council of Science Editors:

Greer RM. Mechanisms of Sensitization to Chemotherapy in Non-Small Cell Lung Cancer. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2012. Available from: http://hdl.handle.net/2152.5/1025


University of Texas Southwestern Medical Center

3. Guo, Wei. Roles of MicroRNAs in Fetal Lung Development.

Degree: 2016, University of Texas Southwestern Medical Center

 Lung alveolar type II cells uniquely synthesize surfactant, a developmentally-regulated lipoprotein that is essential for breathing. Expression of the major surfactant protein, SP-A, in midgestation… (more)

Subjects/Keywords: Alveolar Epithelial Cells; Lung; MicroRNAs; Nuclear Proteins; Transcription Factors

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APA (6th Edition):

Guo, W. (2016). Roles of MicroRNAs in Fetal Lung Development. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/5729

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Guo, Wei. “Roles of MicroRNAs in Fetal Lung Development.” 2016. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/5729.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Guo, Wei. “Roles of MicroRNAs in Fetal Lung Development.” 2016. Web. 09 Jul 2020.

Vancouver:

Guo W. Roles of MicroRNAs in Fetal Lung Development. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2016. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/5729.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Guo W. Roles of MicroRNAs in Fetal Lung Development. [Thesis]. University of Texas Southwestern Medical Center; 2016. Available from: http://hdl.handle.net/2152.5/5729

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

4. Aguilera, Kristina Yolanda. Inhibition of Discoidin Domain Receptor 1 Reduces Collagen-Mediated Tumorigenicity in Pancreatic Ductal Adenocarcinoma.

Degree: 2015, University of Texas Southwestern Medical Center

 An extracellular matrix (ECM) rich in fibrillar collagens is a principal component of pancreatic ductal adenocarcinoma (PDA). The ECM provides structural support for the tumor… (more)

Subjects/Keywords: Carcinoma, Pancreatic Ductal; Collagen; Discoidin Domain Receptor 1; Pancreatic Neoplasms

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APA (6th Edition):

Aguilera, K. Y. (2015). Inhibition of Discoidin Domain Receptor 1 Reduces Collagen-Mediated Tumorigenicity in Pancreatic Ductal Adenocarcinoma. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4220

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Aguilera, Kristina Yolanda. “Inhibition of Discoidin Domain Receptor 1 Reduces Collagen-Mediated Tumorigenicity in Pancreatic Ductal Adenocarcinoma.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/4220.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Aguilera, Kristina Yolanda. “Inhibition of Discoidin Domain Receptor 1 Reduces Collagen-Mediated Tumorigenicity in Pancreatic Ductal Adenocarcinoma.” 2015. Web. 09 Jul 2020.

Vancouver:

Aguilera KY. Inhibition of Discoidin Domain Receptor 1 Reduces Collagen-Mediated Tumorigenicity in Pancreatic Ductal Adenocarcinoma. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/4220.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Aguilera KY. Inhibition of Discoidin Domain Receptor 1 Reduces Collagen-Mediated Tumorigenicity in Pancreatic Ductal Adenocarcinoma. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4220

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

5. Ou, Yi-Hung 1977-. Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation.

Degree: 2013, University of Texas Southwestern Medical Center

 An essential kinase in innate immune signaling, TBK1 couples pathogen surveillance to induction of host defense mechanisms. The pathological activation of TBK1 in cancer can… (more)

Subjects/Keywords: Immunity, Innate; Neoplasms; Protein-Serine-Threonine Kinases; Proto-Oncogene Proteins c-akt

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APA (6th Edition):

Ou, Y. 1. (2013). Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-125

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ou, Yi-Hung 1977-. “Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-125.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ou, Yi-Hung 1977-. “Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation.” 2013. Web. 09 Jul 2020.

Vancouver:

Ou Y1. Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-125.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ou Y1. Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-125

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

6. Britt, Rebecca. Characterization of mTOR Inhibition and Autophagy Inhibition in Non-Small Cell Lung Cancer.

Degree: 2014, University of Texas Southwestern Medical Center

 Lung cancer continues to be the leading cause of cancer related death in both men and women. Pre-clinical studies of targeted therapies are needed in… (more)

Subjects/Keywords: Autophagy; Carcinoma, Non-Small-Cell Lung; Lung Neoplasms; TOR Serine-Threonine Kinases

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APA (6th Edition):

Britt, R. (2014). Characterization of mTOR Inhibition and Autophagy Inhibition in Non-Small Cell Lung Cancer. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3304

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Britt, Rebecca. “Characterization of mTOR Inhibition and Autophagy Inhibition in Non-Small Cell Lung Cancer.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/3304.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Britt, Rebecca. “Characterization of mTOR Inhibition and Autophagy Inhibition in Non-Small Cell Lung Cancer.” 2014. Web. 09 Jul 2020.

Vancouver:

Britt R. Characterization of mTOR Inhibition and Autophagy Inhibition in Non-Small Cell Lung Cancer. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/3304.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Britt R. Characterization of mTOR Inhibition and Autophagy Inhibition in Non-Small Cell Lung Cancer. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3304

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

7. Cheng, Wing Yin. Macrophage PPAR-Gamma Inhibits Gpr132 to Mediate the Anti-Tumor Effects of Rosiglitazone.

Degree: 2016, University of Texas Southwestern Medical Center

 Tumor-associated macrophage (TAM) significantly contributes to tumorigenesis. Human cancer is enhanced by PPARgamma loss-of-function mutations, and inhibited by the thiazolidinedione (TZD) class of synthetic PPARgamma… (more)

Subjects/Keywords: Antineoplastic Agents; Cell Cycle Proteins; Macrophages; Mammary Neoplasms, Experimental; PPAR gamma; Receptors, G-Protein-Coupled; Thiazolidinediones

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APA (6th Edition):

Cheng, W. Y. (2016). Macrophage PPAR-Gamma Inhibits Gpr132 to Mediate the Anti-Tumor Effects of Rosiglitazone. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/5295

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cheng, Wing Yin. “Macrophage PPAR-Gamma Inhibits Gpr132 to Mediate the Anti-Tumor Effects of Rosiglitazone.” 2016. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/5295.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cheng, Wing Yin. “Macrophage PPAR-Gamma Inhibits Gpr132 to Mediate the Anti-Tumor Effects of Rosiglitazone.” 2016. Web. 09 Jul 2020.

Vancouver:

Cheng WY. Macrophage PPAR-Gamma Inhibits Gpr132 to Mediate the Anti-Tumor Effects of Rosiglitazone. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2016. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/5295.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cheng WY. Macrophage PPAR-Gamma Inhibits Gpr132 to Mediate the Anti-Tumor Effects of Rosiglitazone. [Thesis]. University of Texas Southwestern Medical Center; 2016. Available from: http://hdl.handle.net/2152.5/5295

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

8. McMillan, Elizabeth Anne. Identification of Biomarker Driven Intervention Opportunities and Advancement of Mechanism of Action Predictions for Anti-Cancer Therapeutics.

Degree: 2017, University of Texas Southwestern Medical Center

 Oncogenic lesions arising during cancer progression provide an attractive target for chemical intervention strategies. The extreme molecular heterogeneity of tumors, however, makes it difficult to… (more)

Subjects/Keywords: Carcinoma, Non-Small-Cell Lung; High-Throughput Screening Assays; Lung Neoplasms; NF-E2-Related Factor 2

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APA (6th Edition):

McMillan, E. A. (2017). Identification of Biomarker Driven Intervention Opportunities and Advancement of Mechanism of Action Predictions for Anti-Cancer Therapeutics. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/6616

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

McMillan, Elizabeth Anne. “Identification of Biomarker Driven Intervention Opportunities and Advancement of Mechanism of Action Predictions for Anti-Cancer Therapeutics.” 2017. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/6616.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

McMillan, Elizabeth Anne. “Identification of Biomarker Driven Intervention Opportunities and Advancement of Mechanism of Action Predictions for Anti-Cancer Therapeutics.” 2017. Web. 09 Jul 2020.

Vancouver:

McMillan EA. Identification of Biomarker Driven Intervention Opportunities and Advancement of Mechanism of Action Predictions for Anti-Cancer Therapeutics. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2017. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/6616.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

McMillan EA. Identification of Biomarker Driven Intervention Opportunities and Advancement of Mechanism of Action Predictions for Anti-Cancer Therapeutics. [Thesis]. University of Texas Southwestern Medical Center; 2017. Available from: http://hdl.handle.net/2152.5/6616

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

9. Kulak, Ozlem. Chemical Disruption of Wnt Signaling and Telomere Length Maintenance.

Degree: 2015, University of Texas Southwestern Medical Center

Pages xvii-xviii of the dissertation are incorrectly numbered as pages xvi-xvii.

A nearly universal feature of colorectal cancer (CRC) incidents is the presence of genetic… (more)

Subjects/Keywords: Enzyme Inhibitors; Tankyrases; Telomere Shortening; Wnt Signaling Pathway

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APA (6th Edition):

Kulak, O. (2015). Chemical Disruption of Wnt Signaling and Telomere Length Maintenance. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4110

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kulak, Ozlem. “Chemical Disruption of Wnt Signaling and Telomere Length Maintenance.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/4110.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kulak, Ozlem. “Chemical Disruption of Wnt Signaling and Telomere Length Maintenance.” 2015. Web. 09 Jul 2020.

Vancouver:

Kulak O. Chemical Disruption of Wnt Signaling and Telomere Length Maintenance. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/4110.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kulak O. Chemical Disruption of Wnt Signaling and Telomere Length Maintenance. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4110

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

10. Connelly, Sarah Elkin. Regulation of Clathrin Mediated Endocytosis and Its Role in Cancer Progression.

Degree: 2017, University of Texas Southwestern Medical Center

 Metastasis is a multistep process requiring cancer cell signaling, invasion, migration, survival, and proliferation. These processes require dynamic modulation of cell surface proteins by endocytosis.… (more)

Subjects/Keywords: Carcinoma, Non-Small-Cell Lung; Clathrin; Endocytosis; Lactams; Lung Neoplasms

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APA (6th Edition):

Connelly, S. E. (2017). Regulation of Clathrin Mediated Endocytosis and Its Role in Cancer Progression. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/7182

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Connelly, Sarah Elkin. “Regulation of Clathrin Mediated Endocytosis and Its Role in Cancer Progression.” 2017. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/7182.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Connelly, Sarah Elkin. “Regulation of Clathrin Mediated Endocytosis and Its Role in Cancer Progression.” 2017. Web. 09 Jul 2020.

Vancouver:

Connelly SE. Regulation of Clathrin Mediated Endocytosis and Its Role in Cancer Progression. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2017. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/7182.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Connelly SE. Regulation of Clathrin Mediated Endocytosis and Its Role in Cancer Progression. [Thesis]. University of Texas Southwestern Medical Center; 2017. Available from: http://hdl.handle.net/2152.5/7182

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

11. Hight, Suzie K. An In Vivo Functional Genomics Screen Identifies New Regulators of Tumorigenesis in Non-Small Cell Lung Cancer.

Degree: 2015, University of Texas Southwestern Medical Center

 Cancer cells are characterized by the aberrant regulation of signaling pathways that govern responses to growth stimuli, resulting in dysregulated cellular proliferation. The accumulated genomic… (more)

Subjects/Keywords: Carcinoma, Non-Small-Cell Lung; Gene Expression Regulation, Neoplastic; Lung Neoplasms; Receptors, Cytoplasmic and Nuclear

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APA (6th Edition):

Hight, S. K. (2015). An In Vivo Functional Genomics Screen Identifies New Regulators of Tumorigenesis in Non-Small Cell Lung Cancer. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4462

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hight, Suzie K. “An In Vivo Functional Genomics Screen Identifies New Regulators of Tumorigenesis in Non-Small Cell Lung Cancer.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/4462.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hight, Suzie K. “An In Vivo Functional Genomics Screen Identifies New Regulators of Tumorigenesis in Non-Small Cell Lung Cancer.” 2015. Web. 09 Jul 2020.

Vancouver:

Hight SK. An In Vivo Functional Genomics Screen Identifies New Regulators of Tumorigenesis in Non-Small Cell Lung Cancer. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/4462.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hight SK. An In Vivo Functional Genomics Screen Identifies New Regulators of Tumorigenesis in Non-Small Cell Lung Cancer. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4462

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

12. Tagal, Vural. SMARCA4/BRG1-Inactivating Mutations as Potential Predictive Markers for Aurora Kinase A-Targeted Therapy in NSCLCs.

Degree: 2014, University of Texas Southwestern Medical Center

 SMARCA4 encodes a catalytic subunit of the SWI/SNF chromatin remodeling complex, BRG1. Frequent occurrence of SMARCA4/BRG1-inactivating mutations and their mutually exclusive nature from EGFR and… (more)

Subjects/Keywords: Aurora Kinase A; Carcinoma, Non-Small-Cell Lung; DNA Helicases; Nuclear Proteins; Transcription Factors

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APA (6th Edition):

Tagal, V. (2014). SMARCA4/BRG1-Inactivating Mutations as Potential Predictive Markers for Aurora Kinase A-Targeted Therapy in NSCLCs. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3570

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tagal, Vural. “SMARCA4/BRG1-Inactivating Mutations as Potential Predictive Markers for Aurora Kinase A-Targeted Therapy in NSCLCs.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/3570.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tagal, Vural. “SMARCA4/BRG1-Inactivating Mutations as Potential Predictive Markers for Aurora Kinase A-Targeted Therapy in NSCLCs.” 2014. Web. 09 Jul 2020.

Vancouver:

Tagal V. SMARCA4/BRG1-Inactivating Mutations as Potential Predictive Markers for Aurora Kinase A-Targeted Therapy in NSCLCs. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/3570.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tagal V. SMARCA4/BRG1-Inactivating Mutations as Potential Predictive Markers for Aurora Kinase A-Targeted Therapy in NSCLCs. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3570

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

13. Augustyn, Alexander. Characterizing ASCL1-Dependent Neuroendocrine Non-Small Cell Lung Cancers.

Degree: 2013, University of Texas Southwestern Medical Center

 In order to achieve personalized medicine for the treatment of lung cancer, it is important to accurately classify tumors using a combination of factors, including… (more)

Subjects/Keywords: Basic Helix-Loop-Helix Transcription Factors; Carcinoma, Non-Small-Cell Lung; Cell Lineage; Neuroendocrine Tumors; Oncogenes; Small Cell Lung Carcinoma

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APA (6th Edition):

Augustyn, A. (2013). Characterizing ASCL1-Dependent Neuroendocrine Non-Small Cell Lung Cancers. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3336

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Augustyn, Alexander. “Characterizing ASCL1-Dependent Neuroendocrine Non-Small Cell Lung Cancers.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/3336.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Augustyn, Alexander. “Characterizing ASCL1-Dependent Neuroendocrine Non-Small Cell Lung Cancers.” 2013. Web. 09 Jul 2020.

Vancouver:

Augustyn A. Characterizing ASCL1-Dependent Neuroendocrine Non-Small Cell Lung Cancers. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/3336.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Augustyn A. Characterizing ASCL1-Dependent Neuroendocrine Non-Small Cell Lung Cancers. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/3336

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

14. DeSevo, Christopher Gerard. MiR-10a Regulation of Drug Response and Cancer Stem Cell Populations in Non-Small Cell Lung Carcinomas.

Degree: 2013, University of Texas Southwestern Medical Center

 Phosphatidylinositol 3-kinases (PI3Ks) are enzymes involved in diverse cellular functions including cell growth, proliferation, differentiation, motility, survival and apoptosis. Many of these functions relate to… (more)

Subjects/Keywords: Carcinoma, Non-Small-Cell Lung; MicroRNAs; Phosphatidylinositol 3-Kinases

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APA (6th Edition):

DeSevo, C. G. (2013). MiR-10a Regulation of Drug Response and Cancer Stem Cell Populations in Non-Small Cell Lung Carcinomas. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/2717

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

DeSevo, Christopher Gerard. “MiR-10a Regulation of Drug Response and Cancer Stem Cell Populations in Non-Small Cell Lung Carcinomas.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/2717.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

DeSevo, Christopher Gerard. “MiR-10a Regulation of Drug Response and Cancer Stem Cell Populations in Non-Small Cell Lung Carcinomas.” 2013. Web. 09 Jul 2020.

Vancouver:

DeSevo CG. MiR-10a Regulation of Drug Response and Cancer Stem Cell Populations in Non-Small Cell Lung Carcinomas. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/2717.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

DeSevo CG. MiR-10a Regulation of Drug Response and Cancer Stem Cell Populations in Non-Small Cell Lung Carcinomas. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/2717

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

15. Chen, Pei-Hsuan. Metabolic Diversity in Human Non-Small Cell Lung Cancer Cells.

Degree: 2015, University of Texas Southwestern Medical Center

 Cancer cells display oncogene-driven rewiring of metabolism to produce energy and macromolecules for growth. Inhibition of growth-promoting metabolic pathways may prove to be a useful… (more)

Subjects/Keywords: Antineoplastic Agents; Carcinoma, Non-Small-Cell Lung; Lung Neoplasms

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APA (6th Edition):

Chen, P. (2015). Metabolic Diversity in Human Non-Small Cell Lung Cancer Cells. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4451

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Pei-Hsuan. “Metabolic Diversity in Human Non-Small Cell Lung Cancer Cells.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/4451.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Pei-Hsuan. “Metabolic Diversity in Human Non-Small Cell Lung Cancer Cells.” 2015. Web. 09 Jul 2020.

Vancouver:

Chen P. Metabolic Diversity in Human Non-Small Cell Lung Cancer Cells. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/4451.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen P. Metabolic Diversity in Human Non-Small Cell Lung Cancer Cells. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4451

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

16. Zhong, Rui. Tackling Computational Challenges in High-Throughput RNA Interference Screening.

Degree: 2014, University of Texas Southwestern Medical Center

 Since the discovery of RNAi decades ago, it has been increasingly used in biomedical and biological research. The success of analyzing single genes using siRNAs… (more)

Subjects/Keywords: High-Throughput Screening Assays; RNA Interference; Software

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APA (6th Edition):

Zhong, R. (2014). Tackling Computational Challenges in High-Throughput RNA Interference Screening. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3335

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhong, Rui. “Tackling Computational Challenges in High-Throughput RNA Interference Screening.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/3335.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhong, Rui. “Tackling Computational Challenges in High-Throughput RNA Interference Screening.” 2014. Web. 09 Jul 2020.

Vancouver:

Zhong R. Tackling Computational Challenges in High-Throughput RNA Interference Screening. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/3335.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhong R. Tackling Computational Challenges in High-Throughput RNA Interference Screening. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3335

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

17. Frink, Robin Elizabeth. Preclinical Studies of Telomerase Inhibitor Imetelstat in Non-small Cell Lung Cancer.

Degree: 2013, University of Texas Southwestern Medical Center

 Telomerase is expressed in ~90% of all cancers but is not expressed in most somatic cells making it an attractive target for cancer therapy. Telomerase… (more)

Subjects/Keywords: Carcinoma, Non-Small-Cell Lung; Lung Neoplasms; Oligonucleotides; Telomerase

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APA (6th Edition):

Frink, R. E. (2013). Preclinical Studies of Telomerase Inhibitor Imetelstat in Non-small Cell Lung Cancer. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-126

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Frink, Robin Elizabeth. “Preclinical Studies of Telomerase Inhibitor Imetelstat in Non-small Cell Lung Cancer.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-126.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Frink, Robin Elizabeth. “Preclinical Studies of Telomerase Inhibitor Imetelstat in Non-small Cell Lung Cancer.” 2013. Web. 09 Jul 2020.

Vancouver:

Frink RE. Preclinical Studies of Telomerase Inhibitor Imetelstat in Non-small Cell Lung Cancer. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-126.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Frink RE. Preclinical Studies of Telomerase Inhibitor Imetelstat in Non-small Cell Lung Cancer. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-126

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

18. Cupka, Dorothy Lynn. Development of an αᵥβ₆-Binding Peptide for In Vivo Applications: Modulation of Serum Stability and Biodistribution.

Degree: 2015, University of Texas Southwestern Medical Center

 Targeted delivery of imaging and therapeutic agents to tumors improves detection, characterization, and treatment of many types of cancers. Peptides are capable of efficient and… (more)

Subjects/Keywords: Antigens, Neoplasm; Integrins; Neoplasms; Peptides

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APA (6th Edition):

Cupka, D. L. (2015). Development of an αᵥβ₆-Binding Peptide for In Vivo Applications: Modulation of Serum Stability and Biodistribution. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4208

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cupka, Dorothy Lynn. “Development of an αᵥβ₆-Binding Peptide for In Vivo Applications: Modulation of Serum Stability and Biodistribution.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/4208.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cupka, Dorothy Lynn. “Development of an αᵥβ₆-Binding Peptide for In Vivo Applications: Modulation of Serum Stability and Biodistribution.” 2015. Web. 09 Jul 2020.

Vancouver:

Cupka DL. Development of an αᵥβ₆-Binding Peptide for In Vivo Applications: Modulation of Serum Stability and Biodistribution. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/4208.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cupka DL. Development of an αᵥβ₆-Binding Peptide for In Vivo Applications: Modulation of Serum Stability and Biodistribution. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4208

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

19. Carstens, Ryan Murray. Exploration of Nuclear Receptor Activity and siRNA-Derived Phenotypes as Therapeutics in Non-Small Cell Lung Cancer.

Degree: 2015, University of Texas Southwestern Medical Center

 Nuclear hormone receptors are master regulators of diverse cellular functions implicated in tumor pathogenesis and as oncogenic drivers of many human cancers. To better understand… (more)

Subjects/Keywords: Carcinoma, Non-Small-Cell Lung; Receptors, Cytoplasmic and Nuclear; RNA, Small Interfering

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APA (6th Edition):

Carstens, R. M. (2015). Exploration of Nuclear Receptor Activity and siRNA-Derived Phenotypes as Therapeutics in Non-Small Cell Lung Cancer. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4115

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Carstens, Ryan Murray. “Exploration of Nuclear Receptor Activity and siRNA-Derived Phenotypes as Therapeutics in Non-Small Cell Lung Cancer.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/4115.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Carstens, Ryan Murray. “Exploration of Nuclear Receptor Activity and siRNA-Derived Phenotypes as Therapeutics in Non-Small Cell Lung Cancer.” 2015. Web. 09 Jul 2020.

Vancouver:

Carstens RM. Exploration of Nuclear Receptor Activity and siRNA-Derived Phenotypes as Therapeutics in Non-Small Cell Lung Cancer. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/4115.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Carstens RM. Exploration of Nuclear Receptor Activity and siRNA-Derived Phenotypes as Therapeutics in Non-Small Cell Lung Cancer. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4115

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

20. Dalvi, Maithili Prafulla. Targeting Taxane-Platin Resistant Non-Small Cell Lung Cancers with Jumonji C Histone Lysine Demethylase Inhibitors.

Degree: 2015, University of Texas Southwestern Medical Center

 Taxane-platin doublet therapy provides benefit as front-line chemotherapy in advanced and localized non-small cell lung cancer (NSCLC); however the majority of patients relapse with drug… (more)

Subjects/Keywords: Antineoplastic Agents; Carcinoma, Non-Small-Cell Lung; Drug Resistance, Neoplasm; Jumonji Domain-Containing Histone Demethylases; Taxoids

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APA (6th Edition):

Dalvi, M. P. (2015). Targeting Taxane-Platin Resistant Non-Small Cell Lung Cancers with Jumonji C Histone Lysine Demethylase Inhibitors. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4459

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dalvi, Maithili Prafulla. “Targeting Taxane-Platin Resistant Non-Small Cell Lung Cancers with Jumonji C Histone Lysine Demethylase Inhibitors.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/4459.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dalvi, Maithili Prafulla. “Targeting Taxane-Platin Resistant Non-Small Cell Lung Cancers with Jumonji C Histone Lysine Demethylase Inhibitors.” 2015. Web. 09 Jul 2020.

Vancouver:

Dalvi MP. Targeting Taxane-Platin Resistant Non-Small Cell Lung Cancers with Jumonji C Histone Lysine Demethylase Inhibitors. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/4459.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dalvi MP. Targeting Taxane-Platin Resistant Non-Small Cell Lung Cancers with Jumonji C Histone Lysine Demethylase Inhibitors. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4459

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

21. Moss, Lacy Reynolds. Elucidating the Anti-Cancer Mechanism of Low Density Lipoprotein-Mediated Delivery of Docosahexaenoic Acid to Hepatocellular Carcinoma Cells.

Degree: 2015, University of Texas Southwestern Medical Center

 Hepatocellular carcinoma is a lethal malignancy with few effective therapy options. New selective treatments are urgently needed to destroy hepatocellular carcinoma cells without harming the… (more)

Subjects/Keywords: Cell Death; Docosahexaenoic Acids; Lipoproteins, LDL; Liver Neoplasms, Experimental

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APA (6th Edition):

Moss, L. R. (2015). Elucidating the Anti-Cancer Mechanism of Low Density Lipoprotein-Mediated Delivery of Docosahexaenoic Acid to Hepatocellular Carcinoma Cells. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4215

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Moss, Lacy Reynolds. “Elucidating the Anti-Cancer Mechanism of Low Density Lipoprotein-Mediated Delivery of Docosahexaenoic Acid to Hepatocellular Carcinoma Cells.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/4215.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Moss, Lacy Reynolds. “Elucidating the Anti-Cancer Mechanism of Low Density Lipoprotein-Mediated Delivery of Docosahexaenoic Acid to Hepatocellular Carcinoma Cells.” 2015. Web. 09 Jul 2020.

Vancouver:

Moss LR. Elucidating the Anti-Cancer Mechanism of Low Density Lipoprotein-Mediated Delivery of Docosahexaenoic Acid to Hepatocellular Carcinoma Cells. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/4215.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Moss LR. Elucidating the Anti-Cancer Mechanism of Low Density Lipoprotein-Mediated Delivery of Docosahexaenoic Acid to Hepatocellular Carcinoma Cells. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4215

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

22. Dospoy, Patrick. Characterizing c-Myc Dependent Lung Cancers.

Degree: 2015, University of Texas Southwestern Medical Center

 MYC is one of the most commonly deregulated oncogenes in human cancer, including breast, colorectal and lung. While mutations in myc are rare, MYC is… (more)

Subjects/Keywords: Lung Neoplasms; Peptide Fragments; Proto-Oncogene Proteins c-myc

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APA (6th Edition):

Dospoy, P. (2015). Characterizing c-Myc Dependent Lung Cancers. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4225

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dospoy, Patrick. “Characterizing c-Myc Dependent Lung Cancers.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/4225.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dospoy, Patrick. “Characterizing c-Myc Dependent Lung Cancers.” 2015. Web. 09 Jul 2020.

Vancouver:

Dospoy P. Characterizing c-Myc Dependent Lung Cancers. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/4225.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dospoy P. Characterizing c-Myc Dependent Lung Cancers. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4225

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

23. Osborne, Jihan K. The Developmental Transcription Factor Neurogenic Differentiation 1 in Migration and Survival of Neuroendocrine Carcinomas.

Degree: 2013, University of Texas Southwestern Medical Center

 Differentiation and determination of cell fate during embryogenesis is decided by a collection of transcription factors, including the large family of basic-helix-loop-helix (bHLH) transcription factors.… (more)

Subjects/Keywords: Basic Helix-Loop-Helix Transcription Factors; Cell Movement; Cell Survival; Lung Neoplasms; Neural Cell Adhesion Molecules

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APA (6th Edition):

Osborne, J. K. (2013). The Developmental Transcription Factor Neurogenic Differentiation 1 in Migration and Survival of Neuroendocrine Carcinomas. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-105

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Osborne, Jihan K. “The Developmental Transcription Factor Neurogenic Differentiation 1 in Migration and Survival of Neuroendocrine Carcinomas.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-105.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Osborne, Jihan K. “The Developmental Transcription Factor Neurogenic Differentiation 1 in Migration and Survival of Neuroendocrine Carcinomas.” 2013. Web. 09 Jul 2020.

Vancouver:

Osborne JK. The Developmental Transcription Factor Neurogenic Differentiation 1 in Migration and Survival of Neuroendocrine Carcinomas. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-105.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Osborne JK. The Developmental Transcription Factor Neurogenic Differentiation 1 in Migration and Survival of Neuroendocrine Carcinomas. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-105

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

24. Deb, Dhruba. Oncogene-Induced Signaling Heterogeneity in Lung Cancer.

Degree: 2015, University of Texas Southwestern Medical Center

 Lung cancer causes the maximum number of cancer related deaths worldwide. In recent years, the cancer genome atlas (TCGA) initiative has identified 138 frequently occurring… (more)

Subjects/Keywords: Carcinoma, Squamous Cell; Lung Neoplasms; Proto-Oncogene Proteins c-myc; Smad2 Protein; Transforming Growth Factor beta

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APA (6th Edition):

Deb, D. (2015). Oncogene-Induced Signaling Heterogeneity in Lung Cancer. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4213

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Deb, Dhruba. “Oncogene-Induced Signaling Heterogeneity in Lung Cancer.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/4213.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Deb, Dhruba. “Oncogene-Induced Signaling Heterogeneity in Lung Cancer.” 2015. Web. 09 Jul 2020.

Vancouver:

Deb D. Oncogene-Induced Signaling Heterogeneity in Lung Cancer. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/4213.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Deb D. Oncogene-Induced Signaling Heterogeneity in Lung Cancer. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4213

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

25. Shah, Jyoti Khetsi. To develop a small interfering Rna (siRNA) design and information resource to facilitate genetic manipulaton of human cells.

Degree: MS, Biomedical Engineering, 2004, University of Texas Southwestern Medical Center

 Part I: Small interfering RNAs (siRNAs) have revolutionized our ability to study the effects of altering the expression of single genes in mammalian (and other)… (more)

Subjects/Keywords: RNA, Small Interfering; Software; Gene Expression

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APA (6th Edition):

Shah, J. K. (2004). To develop a small interfering Rna (siRNA) design and information resource to facilitate genetic manipulaton of human cells. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/773

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shah, Jyoti Khetsi. “To develop a small interfering Rna (siRNA) design and information resource to facilitate genetic manipulaton of human cells.” 2004. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/773.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shah, Jyoti Khetsi. “To develop a small interfering Rna (siRNA) design and information resource to facilitate genetic manipulaton of human cells.” 2004. Web. 09 Jul 2020.

Vancouver:

Shah JK. To develop a small interfering Rna (siRNA) design and information resource to facilitate genetic manipulaton of human cells. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2004. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/773.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shah JK. To develop a small interfering Rna (siRNA) design and information resource to facilitate genetic manipulaton of human cells. [Thesis]. University of Texas Southwestern Medical Center; 2004. Available from: http://hdl.handle.net/2152.5/773

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

26. Shames, David S. Aberrant DNA Methylation and Cancer: A Global Analysis of Promoter Hypermethylation in Human Lung Cancers.

Degree: PhD, Cell Regulation, 2006, University of Texas Southwestern Medical Center

 Tumor-acquired alterations in DNA methylation include both genome-wide hypomethylation and locus specific hypermethylation. Global loss of DNA methylation destabilizes chromatin architecture, augments genomic instability, and… (more)

Subjects/Keywords: Promoter Regions, Genetics; ATP-Binding Cassette Transporters; Gene Expression Profiling; Adenosine Triphosphate

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Shames, D. S. (2006). Aberrant DNA Methylation and Cancer: A Global Analysis of Promoter Hypermethylation in Human Lung Cancers. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/334

Chicago Manual of Style (16th Edition):

Shames, David S. “Aberrant DNA Methylation and Cancer: A Global Analysis of Promoter Hypermethylation in Human Lung Cancers.” 2006. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/334.

MLA Handbook (7th Edition):

Shames, David S. “Aberrant DNA Methylation and Cancer: A Global Analysis of Promoter Hypermethylation in Human Lung Cancers.” 2006. Web. 09 Jul 2020.

Vancouver:

Shames DS. Aberrant DNA Methylation and Cancer: A Global Analysis of Promoter Hypermethylation in Human Lung Cancers. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2006. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/334.

Council of Science Editors:

Shames DS. Aberrant DNA Methylation and Cancer: A Global Analysis of Promoter Hypermethylation in Human Lung Cancers. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2006. Available from: http://hdl.handle.net/2152.5/334

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