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You searched for +publisher:"University of Texas Southwestern Medical Center" +contributor:("Elmquist, Joel"). Showing records 1 – 10 of 10 total matches.

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University of Texas Southwestern Medical Center

1. Tao, Caroline. Adiponectin and Toll-Like Receptor 4: Important Adipocyte Modulators of Systemic Glucose and Lipid Metabolism.

Degree: 2015, University of Texas Southwestern Medical Center

 As a global epidemic, the prevalence of obesity and its complications have increased rapidly over the past few decades. Obesity, characterized by excessive amount of… (more)

Subjects/Keywords: Adipocytes; Adiponectin; Adipose Tissue; Obesity; Toll-Like Receptor 4

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APA (6th Edition):

Tao, C. (2015). Adiponectin and Toll-Like Receptor 4: Important Adipocyte Modulators of Systemic Glucose and Lipid Metabolism. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4122

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tao, Caroline. “Adiponectin and Toll-Like Receptor 4: Important Adipocyte Modulators of Systemic Glucose and Lipid Metabolism.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed November 28, 2020. http://hdl.handle.net/2152.5/4122.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tao, Caroline. “Adiponectin and Toll-Like Receptor 4: Important Adipocyte Modulators of Systemic Glucose and Lipid Metabolism.” 2015. Web. 28 Nov 2020.

Vancouver:

Tao C. Adiponectin and Toll-Like Receptor 4: Important Adipocyte Modulators of Systemic Glucose and Lipid Metabolism. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/2152.5/4122.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tao C. Adiponectin and Toll-Like Receptor 4: Important Adipocyte Modulators of Systemic Glucose and Lipid Metabolism. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4122

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

2. Ryu, Keun Woo. Metabolic Regulation of Transcription Through Compartmentalized NAD+ Biosynthesis.

Degree: 2017, University of Texas Southwestern Medical Center

 Extracellular signaling and nutrient availability are major factors for the cell fate decision. Responds to extracellular information requires metabolic alterations and differential gene expression. Recently… (more)

Subjects/Keywords: Adipocytes; Adipogenesis; NAD; Nicotinamide-Nucleotide Adenylyltransferase; Transcription, Genetic

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APA (6th Edition):

Ryu, K. W. (2017). Metabolic Regulation of Transcription Through Compartmentalized NAD+ Biosynthesis. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/7742

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ryu, Keun Woo. “Metabolic Regulation of Transcription Through Compartmentalized NAD+ Biosynthesis.” 2017. Thesis, University of Texas Southwestern Medical Center. Accessed November 28, 2020. http://hdl.handle.net/2152.5/7742.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ryu, Keun Woo. “Metabolic Regulation of Transcription Through Compartmentalized NAD+ Biosynthesis.” 2017. Web. 28 Nov 2020.

Vancouver:

Ryu KW. Metabolic Regulation of Transcription Through Compartmentalized NAD+ Biosynthesis. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2017. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/2152.5/7742.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ryu KW. Metabolic Regulation of Transcription Through Compartmentalized NAD+ Biosynthesis. [Thesis]. University of Texas Southwestern Medical Center; 2017. Available from: http://hdl.handle.net/2152.5/7742

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

3. Javadi, Christopher. Hormone Regulation of NPY Neuron Activity in the Arcuate Nucleus of the Hypothalamus.

Degree: 2015, University of Texas Southwestern Medical Center

 Neuropeptide Y (NPY)-expressing neurons in the arcuate nucleus of the hypothalamus are part of a neuroendocrine feedback loop that regulates feeding behavior and glucose homeostasis.… (more)

Subjects/Keywords: Arcuate Nucleus of Hypothalamus; Metabolism; Neurons; Neuropeptide Y

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APA (6th Edition):

Javadi, C. (2015). Hormone Regulation of NPY Neuron Activity in the Arcuate Nucleus of the Hypothalamus. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4222

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Javadi, Christopher. “Hormone Regulation of NPY Neuron Activity in the Arcuate Nucleus of the Hypothalamus.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed November 28, 2020. http://hdl.handle.net/2152.5/4222.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Javadi, Christopher. “Hormone Regulation of NPY Neuron Activity in the Arcuate Nucleus of the Hypothalamus.” 2015. Web. 28 Nov 2020.

Vancouver:

Javadi C. Hormone Regulation of NPY Neuron Activity in the Arcuate Nucleus of the Hypothalamus. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/2152.5/4222.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Javadi C. Hormone Regulation of NPY Neuron Activity in the Arcuate Nucleus of the Hypothalamus. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4222

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

4. Anderson, Jason Graham. SIRT6 Overexpression Protects Against Diet-Induced Diabetes in Mice.

Degree: 2013, University of Texas Southwestern Medical Center

 Numerous studies in a variety of species indicate that caloric restriction (CR) elicits beneficial metabolic effects including reduced adiposity and improved glucose homeostasis, as well… (more)

Subjects/Keywords: Diabetes Mellitus, Type 2; Glucose; Obesity; Sirtuins

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APA (6th Edition):

Anderson, J. G. (2013). SIRT6 Overexpression Protects Against Diet-Induced Diabetes in Mice. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1724

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Anderson, Jason Graham. “SIRT6 Overexpression Protects Against Diet-Induced Diabetes in Mice.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed November 28, 2020. http://hdl.handle.net/2152.5/1724.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Anderson, Jason Graham. “SIRT6 Overexpression Protects Against Diet-Induced Diabetes in Mice.” 2013. Web. 28 Nov 2020.

Vancouver:

Anderson JG. SIRT6 Overexpression Protects Against Diet-Induced Diabetes in Mice. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/2152.5/1724.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Anderson JG. SIRT6 Overexpression Protects Against Diet-Induced Diabetes in Mice. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1724

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

5. Stubblefield, Jeremy Joseph. Nocturnin Regulates Metabolic Flux Through Maintenance of Poly(A) Tail Length Dynamics.

Degree: 2016, University of Texas Southwestern Medical Center

Pages 32-126 are incorrectly numbered as pages 31-125.

Cyclic processes in both behavior and physiology are aligned to the external environment through the circadian clock.… (more)

Subjects/Keywords: Circadian Rhythm; Nuclear Proteins; Poly A; Transcription Factors

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APA (6th Edition):

Stubblefield, J. J. (2016). Nocturnin Regulates Metabolic Flux Through Maintenance of Poly(A) Tail Length Dynamics. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/5738

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Stubblefield, Jeremy Joseph. “Nocturnin Regulates Metabolic Flux Through Maintenance of Poly(A) Tail Length Dynamics.” 2016. Thesis, University of Texas Southwestern Medical Center. Accessed November 28, 2020. http://hdl.handle.net/2152.5/5738.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Stubblefield, Jeremy Joseph. “Nocturnin Regulates Metabolic Flux Through Maintenance of Poly(A) Tail Length Dynamics.” 2016. Web. 28 Nov 2020.

Vancouver:

Stubblefield JJ. Nocturnin Regulates Metabolic Flux Through Maintenance of Poly(A) Tail Length Dynamics. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2016. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/2152.5/5738.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Stubblefield JJ. Nocturnin Regulates Metabolic Flux Through Maintenance of Poly(A) Tail Length Dynamics. [Thesis]. University of Texas Southwestern Medical Center; 2016. Available from: http://hdl.handle.net/2152.5/5738

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

6. Atkin, Stan Dean. Liver Receptor Homolog-1 Regulates Kisspeptin Expression in the Arcuate Nucleus to Promote Reproductive Axis Function.

Degree: 2014, University of Texas Southwestern Medical Center

 The timing of ovulation in mammals is set by a complex hypothalamic pituitary neuroendocrine axis. Kisspeptin neurons in the arcuate nucleus (Arc) are thought to… (more)

Subjects/Keywords: Kisspeptins; Neuropeptides; Receptors, Cytoplasmic and Nuclear; Reproduction

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APA (6th Edition):

Atkin, S. D. (2014). Liver Receptor Homolog-1 Regulates Kisspeptin Expression in the Arcuate Nucleus to Promote Reproductive Axis Function. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1425

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Atkin, Stan Dean. “Liver Receptor Homolog-1 Regulates Kisspeptin Expression in the Arcuate Nucleus to Promote Reproductive Axis Function.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed November 28, 2020. http://hdl.handle.net/2152.5/1425.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Atkin, Stan Dean. “Liver Receptor Homolog-1 Regulates Kisspeptin Expression in the Arcuate Nucleus to Promote Reproductive Axis Function.” 2014. Web. 28 Nov 2020.

Vancouver:

Atkin SD. Liver Receptor Homolog-1 Regulates Kisspeptin Expression in the Arcuate Nucleus to Promote Reproductive Axis Function. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/2152.5/1425.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Atkin SD. Liver Receptor Homolog-1 Regulates Kisspeptin Expression in the Arcuate Nucleus to Promote Reproductive Axis Function. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/1425

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

7. Udit, Swalpa 1985-. Naᵥ1.8+ Visceral Afferent Neurons: Roles in Metabolism and Inflammation.

Degree: 2013, University of Texas Southwestern Medical Center

 The increasing prevalence of obesity and its related comorbidities are major health issues facing western societies. Obesity involves a long-term, chronic perturbation of energy balance,… (more)

Subjects/Keywords: Afferent Pathways; Neurons, Afferent; Sodium Channels; Vagus Nerve

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APA (6th Edition):

Udit, S. 1. (2013). Naᵥ1.8+ Visceral Afferent Neurons: Roles in Metabolism and Inflammation. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3329

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Udit, Swalpa 1985-. “Naᵥ1.8+ Visceral Afferent Neurons: Roles in Metabolism and Inflammation.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed November 28, 2020. http://hdl.handle.net/2152.5/3329.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Udit, Swalpa 1985-. “Naᵥ1.8+ Visceral Afferent Neurons: Roles in Metabolism and Inflammation.” 2013. Web. 28 Nov 2020.

Vancouver:

Udit S1. Naᵥ1.8+ Visceral Afferent Neurons: Roles in Metabolism and Inflammation. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/2152.5/3329.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Udit S1. Naᵥ1.8+ Visceral Afferent Neurons: Roles in Metabolism and Inflammation. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/3329

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

8. McFarlane, Matthew Ryan. Ghrelin: The Hunger Hormone that Isn't.

Degree: 2014, University of Texas Southwestern Medical Center

 Ghrelin is a 28-amino acid acylated peptide hormone secreted by endocrine cells in the stomach. It was first identified in 1999 and shortly thereafter shown… (more)

Subjects/Keywords: Appetite; Body Weight; Diet, High-Fat; Ghrelin

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APA (6th Edition):

McFarlane, M. R. (2014). Ghrelin: The Hunger Hormone that Isn't. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/5283

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

McFarlane, Matthew Ryan. “Ghrelin: The Hunger Hormone that Isn't.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed November 28, 2020. http://hdl.handle.net/2152.5/5283.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

McFarlane, Matthew Ryan. “Ghrelin: The Hunger Hormone that Isn't.” 2014. Web. 28 Nov 2020.

Vancouver:

McFarlane MR. Ghrelin: The Hunger Hormone that Isn't. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/2152.5/5283.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

McFarlane MR. Ghrelin: The Hunger Hormone that Isn't. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/5283

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

9. Li, Robert Lin. Essential Function of Ghrelin in Chronic Starvation.

Degree: 2013, University of Texas Southwestern Medical Center

 Ghrelin, an octanoylated peptide hormone secreted from the stomach, stimulates the release of growth hormone (GH) from the pituitary. Ghrelin O-acyltransferase (GOAT) is the enzyme… (more)

Subjects/Keywords: Acyltransferases; Ghrelin; Growth Hormone; Receptors, Adrenergic, beta-1; Starvation

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APA (6th Edition):

Li, R. L. (2013). Essential Function of Ghrelin in Chronic Starvation. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-101

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Li, Robert Lin. “Essential Function of Ghrelin in Chronic Starvation.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed November 28, 2020. http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-101.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Li, Robert Lin. “Essential Function of Ghrelin in Chronic Starvation.” 2013. Web. 28 Nov 2020.

Vancouver:

Li RL. Essential Function of Ghrelin in Chronic Starvation. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-101.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Li RL. Essential Function of Ghrelin in Chronic Starvation. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-101

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

10. Lee, Ivan T. Cellular Basis of Behavioral Circadian Rhythms in Mammals: The Role of Neuromedin S (Nms)-Producing Cells in the Suprachiasmatic Nucleus.

Degree: 2013, University of Texas Southwestern Medical Center

 Behavioral circadian rhythms in mammals are controlled by highly heterogeneous populations of neurons located in the suprachiasmatic nucleus (SCN). Lesion and transplantation studies have established… (more)

Subjects/Keywords: Circadian Clocks; Circadian Rhythm; Neurons; Neuropeptides; Suprachiasmatic Nucleus

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APA (6th Edition):

Lee, I. T. (2013). Cellular Basis of Behavioral Circadian Rhythms in Mammals: The Role of Neuromedin S (Nms)-Producing Cells in the Suprachiasmatic Nucleus. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4106

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lee, Ivan T. “Cellular Basis of Behavioral Circadian Rhythms in Mammals: The Role of Neuromedin S (Nms)-Producing Cells in the Suprachiasmatic Nucleus.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed November 28, 2020. http://hdl.handle.net/2152.5/4106.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lee, Ivan T. “Cellular Basis of Behavioral Circadian Rhythms in Mammals: The Role of Neuromedin S (Nms)-Producing Cells in the Suprachiasmatic Nucleus.” 2013. Web. 28 Nov 2020.

Vancouver:

Lee IT. Cellular Basis of Behavioral Circadian Rhythms in Mammals: The Role of Neuromedin S (Nms)-Producing Cells in the Suprachiasmatic Nucleus. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/2152.5/4106.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lee IT. Cellular Basis of Behavioral Circadian Rhythms in Mammals: The Role of Neuromedin S (Nms)-Producing Cells in the Suprachiasmatic Nucleus. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/4106

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.