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You searched for +publisher:"University of Texas Southwestern Medical Center" +contributor:("Corey, David R."). Showing records 1 – 12 of 12 total matches.

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University of Texas Southwestern Medical Center

1. Younger, Scott Thomas. Transcriptional Gene Silencing in Mammalian Cells by MicroRNAs That Target Gene Promoters.

Degree: PhD, 2011, University of Texas Southwestern Medical Center

 A rich history exists for RNA-based regulation of gene transcription. It was reported more than a decade ago that RNA is capable of inducing DNA… (more)

Subjects/Keywords: MicroRNAs; Gene Silencing; Transcription, Genetic

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Younger, S. T. (2011). Transcriptional Gene Silencing in Mammalian Cells by MicroRNAs That Target Gene Promoters. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/894

Chicago Manual of Style (16th Edition):

Younger, Scott Thomas. “Transcriptional Gene Silencing in Mammalian Cells by MicroRNAs That Target Gene Promoters.” 2011. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 07, 2020. http://hdl.handle.net/2152.5/894.

MLA Handbook (7th Edition):

Younger, Scott Thomas. “Transcriptional Gene Silencing in Mammalian Cells by MicroRNAs That Target Gene Promoters.” 2011. Web. 07 Jul 2020.

Vancouver:

Younger ST. Transcriptional Gene Silencing in Mammalian Cells by MicroRNAs That Target Gene Promoters. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2011. [cited 2020 Jul 07]. Available from: http://hdl.handle.net/2152.5/894.

Council of Science Editors:

Younger ST. Transcriptional Gene Silencing in Mammalian Cells by MicroRNAs That Target Gene Promoters. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2011. Available from: http://hdl.handle.net/2152.5/894


University of Texas Southwestern Medical Center

2. Sei, Emi. Characterization of the Interactions Between Kaposi’s Sarcoma-Associated Herpesvirus ORF57 and Its RNA Partners.

Degree: 2014, University of Texas Southwestern Medical Center

 Kaposi's sarcoma-associated herpesvirus (KSHV; HHV-8) is a human gammaherpesvirus and the etiological agent of Kaposi's sarcoma (KS), the most common AIDS-associated malignancy. Like all herpesviruses,… (more)

Subjects/Keywords: Gene Expression Regulation, Viral; Herpesviridae Infections; Herpesvirus 8, Human; RNA Precursors; Viral Regulatory and Accessory Proteins; Virus Activation

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APA (6th Edition):

Sei, E. (2014). Characterization of the Interactions Between Kaposi’s Sarcoma-Associated Herpesvirus ORF57 and Its RNA Partners. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3600

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sei, Emi. “Characterization of the Interactions Between Kaposi’s Sarcoma-Associated Herpesvirus ORF57 and Its RNA Partners.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed July 07, 2020. http://hdl.handle.net/2152.5/3600.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sei, Emi. “Characterization of the Interactions Between Kaposi’s Sarcoma-Associated Herpesvirus ORF57 and Its RNA Partners.” 2014. Web. 07 Jul 2020.

Vancouver:

Sei E. Characterization of the Interactions Between Kaposi’s Sarcoma-Associated Herpesvirus ORF57 and Its RNA Partners. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2020 Jul 07]. Available from: http://hdl.handle.net/2152.5/3600.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sei E. Characterization of the Interactions Between Kaposi’s Sarcoma-Associated Herpesvirus ORF57 and Its RNA Partners. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3600

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

3. Yu, Dongbo 1984-. Using Chemically Modified Oligonucleotides to Modulate Gene Expression, Treat Genetic Diseases, and Probe Novel Mechanisms of RNA Interference.

Degree: 2013, University of Texas Southwestern Medical Center

 A number of inherited neurological disorders remain incurable despite having well-defined monogenic etiologies. One example is Huntington's disease (HD), which is caused by CAG trinucleotide… (more)

Subjects/Keywords: Huntington Disease; Nerve Tissue Proteins; RNA Interference; RNA, Small Interfering

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APA (6th Edition):

Yu, D. 1. (2013). Using Chemically Modified Oligonucleotides to Modulate Gene Expression, Treat Genetic Diseases, and Probe Novel Mechanisms of RNA Interference. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/ETD-UTSWMED-2014-05-59

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yu, Dongbo 1984-. “Using Chemically Modified Oligonucleotides to Modulate Gene Expression, Treat Genetic Diseases, and Probe Novel Mechanisms of RNA Interference.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed July 07, 2020. http://hdl.handle.net/2152.5/ETD-UTSWMED-2014-05-59.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yu, Dongbo 1984-. “Using Chemically Modified Oligonucleotides to Modulate Gene Expression, Treat Genetic Diseases, and Probe Novel Mechanisms of RNA Interference.” 2013. Web. 07 Jul 2020.

Vancouver:

Yu D1. Using Chemically Modified Oligonucleotides to Modulate Gene Expression, Treat Genetic Diseases, and Probe Novel Mechanisms of RNA Interference. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Jul 07]. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2014-05-59.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yu D1. Using Chemically Modified Oligonucleotides to Modulate Gene Expression, Treat Genetic Diseases, and Probe Novel Mechanisms of RNA Interference. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2014-05-59

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

4. Adebesin, Adeniyi Michael. Studies Toward the Syntheses of Antiarrythmic, Anti-Virulence, and Anticancer Small Molecules.

Degree: 2015, University of Texas Southwestern Medical Center

 This work is comprised of three projects: a) the development of antiarrythmic analogs of 17(R),18(S)-epoxyeicosatetraenoic acid for the treatment of atrial fibrillation, b) the development… (more)

Subjects/Keywords: Anti-Bacterial Agents; Gram-Negative Bacteria; Protein Kinase Inhibitors; Protein Kinases; Quorum Sensing; Sulfonamides

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APA (6th Edition):

Adebesin, A. M. (2015). Studies Toward the Syntheses of Antiarrythmic, Anti-Virulence, and Anticancer Small Molecules. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4460

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Adebesin, Adeniyi Michael. “Studies Toward the Syntheses of Antiarrythmic, Anti-Virulence, and Anticancer Small Molecules.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed July 07, 2020. http://hdl.handle.net/2152.5/4460.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Adebesin, Adeniyi Michael. “Studies Toward the Syntheses of Antiarrythmic, Anti-Virulence, and Anticancer Small Molecules.” 2015. Web. 07 Jul 2020.

Vancouver:

Adebesin AM. Studies Toward the Syntheses of Antiarrythmic, Anti-Virulence, and Anticancer Small Molecules. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Jul 07]. Available from: http://hdl.handle.net/2152.5/4460.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Adebesin AM. Studies Toward the Syntheses of Antiarrythmic, Anti-Virulence, and Anticancer Small Molecules. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4460

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

5. Nguyen, Liem Hieu. The Functional Roles of the Lin28/let-7 Axis in Tissue Regeneration and Cancer.

Degree: 2017, University of Texas Southwestern Medical Center

 The let-7 microRNAs and their antagonists, the Lin28 RNA-binding proteins (Lin28a and Lin28b), are post-transcriptional regulators well known for their initially discovered role in controlling… (more)

Subjects/Keywords: Liver Neoplasms; Liver Regeneration; MicroRNAs; RNA-Binding Proteins

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APA (6th Edition):

Nguyen, L. H. (2017). The Functional Roles of the Lin28/let-7 Axis in Tissue Regeneration and Cancer. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/6599

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nguyen, Liem Hieu. “The Functional Roles of the Lin28/let-7 Axis in Tissue Regeneration and Cancer.” 2017. Thesis, University of Texas Southwestern Medical Center. Accessed July 07, 2020. http://hdl.handle.net/2152.5/6599.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nguyen, Liem Hieu. “The Functional Roles of the Lin28/let-7 Axis in Tissue Regeneration and Cancer.” 2017. Web. 07 Jul 2020.

Vancouver:

Nguyen LH. The Functional Roles of the Lin28/let-7 Axis in Tissue Regeneration and Cancer. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2017. [cited 2020 Jul 07]. Available from: http://hdl.handle.net/2152.5/6599.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nguyen LH. The Functional Roles of the Lin28/let-7 Axis in Tissue Regeneration and Cancer. [Thesis]. University of Texas Southwestern Medical Center; 2017. Available from: http://hdl.handle.net/2152.5/6599

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

6. Pendleton, Kathryn Elizabeth. The U6 snRNA m6A Methyltransferase METTL16 Regulates MAT2A Intron Retention Through Co-Transcriptional Splicing.

Degree: 2017, University of Texas Southwestern Medical Center

 Maintenance of proper levels of the methyl donor S-adenosylmethionine (SAM) is critical for a wide variety of biological processes. We demonstrate that the N6-adenosine methyltransferase… (more)

Subjects/Keywords: Introns; Methionine Adenosyltransferase; Methyltransferases; RNA Splicing; S-Adenosylmethionine

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APA (6th Edition):

Pendleton, K. E. (2017). The U6 snRNA m6A Methyltransferase METTL16 Regulates MAT2A Intron Retention Through Co-Transcriptional Splicing. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/7194

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pendleton, Kathryn Elizabeth. “The U6 snRNA m6A Methyltransferase METTL16 Regulates MAT2A Intron Retention Through Co-Transcriptional Splicing.” 2017. Thesis, University of Texas Southwestern Medical Center. Accessed July 07, 2020. http://hdl.handle.net/2152.5/7194.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pendleton, Kathryn Elizabeth. “The U6 snRNA m6A Methyltransferase METTL16 Regulates MAT2A Intron Retention Through Co-Transcriptional Splicing.” 2017. Web. 07 Jul 2020.

Vancouver:

Pendleton KE. The U6 snRNA m6A Methyltransferase METTL16 Regulates MAT2A Intron Retention Through Co-Transcriptional Splicing. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2017. [cited 2020 Jul 07]. Available from: http://hdl.handle.net/2152.5/7194.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pendleton KE. The U6 snRNA m6A Methyltransferase METTL16 Regulates MAT2A Intron Retention Through Co-Transcriptional Splicing. [Thesis]. University of Texas Southwestern Medical Center; 2017. Available from: http://hdl.handle.net/2152.5/7194

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

7. Oswald, Nathaniel Walter. Using Multiple Screening Strategies to Biologically and Chemically Characterize Natural Products.

Degree: 2016, University of Texas Southwestern Medical Center

 Natural products play an important role in the discovery and development of therapeutics and biological probes. However, in recent decades therapeutic screening efforts have moved… (more)

Subjects/Keywords: Biological Factors; Biological Products; Computational Biology; Lung Neoplasms; Technology, Pharmaceutical

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APA (6th Edition):

Oswald, N. W. (2016). Using Multiple Screening Strategies to Biologically and Chemically Characterize Natural Products. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/5730

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Oswald, Nathaniel Walter. “Using Multiple Screening Strategies to Biologically and Chemically Characterize Natural Products.” 2016. Thesis, University of Texas Southwestern Medical Center. Accessed July 07, 2020. http://hdl.handle.net/2152.5/5730.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Oswald, Nathaniel Walter. “Using Multiple Screening Strategies to Biologically and Chemically Characterize Natural Products.” 2016. Web. 07 Jul 2020.

Vancouver:

Oswald NW. Using Multiple Screening Strategies to Biologically and Chemically Characterize Natural Products. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2016. [cited 2020 Jul 07]. Available from: http://hdl.handle.net/2152.5/5730.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Oswald NW. Using Multiple Screening Strategies to Biologically and Chemically Characterize Natural Products. [Thesis]. University of Texas Southwestern Medical Center; 2016. Available from: http://hdl.handle.net/2152.5/5730

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

8. Borkowski, Robert John. Identifying Context-Specific Synthetic Lethal miRNA Inhibition in Non-Small Cell Lung Cancer.

Degree: 2013, University of Texas Southwestern Medical Center

 Non-small cell lung cancer (NSCLC) is the leading cause of cancer-related fatalities in the US. This is due in part to a lack of highly… (more)

Subjects/Keywords: Carcinoma, Non-Small-Cell Lung; MicroRNAs; Gene Targeting

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APA (6th Edition):

Borkowski, R. J. (2013). Identifying Context-Specific Synthetic Lethal miRNA Inhibition in Non-Small Cell Lung Cancer. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1726

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Borkowski, Robert John. “Identifying Context-Specific Synthetic Lethal miRNA Inhibition in Non-Small Cell Lung Cancer.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed July 07, 2020. http://hdl.handle.net/2152.5/1726.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Borkowski, Robert John. “Identifying Context-Specific Synthetic Lethal miRNA Inhibition in Non-Small Cell Lung Cancer.” 2013. Web. 07 Jul 2020.

Vancouver:

Borkowski RJ. Identifying Context-Specific Synthetic Lethal miRNA Inhibition in Non-Small Cell Lung Cancer. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Jul 07]. Available from: http://hdl.handle.net/2152.5/1726.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Borkowski RJ. Identifying Context-Specific Synthetic Lethal miRNA Inhibition in Non-Small Cell Lung Cancer. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1726

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

9. Kalantari, Roya. Building the Human Argonaute 2 Interaction Network.

Degree: 2015, University of Texas Southwestern Medical Center

 RNA interference (RNAi) is a system that has been largely studied and defined by its ability to affect gene expression and translation in the cytoplasm.… (more)

Subjects/Keywords: Argonaute Proteins; Immunoprecipitation; RNA-Induced Silencing Complex

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APA (6th Edition):

Kalantari, R. (2015). Building the Human Argonaute 2 Interaction Network. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1737

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kalantari, Roya. “Building the Human Argonaute 2 Interaction Network.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed July 07, 2020. http://hdl.handle.net/2152.5/1737.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kalantari, Roya. “Building the Human Argonaute 2 Interaction Network.” 2015. Web. 07 Jul 2020.

Vancouver:

Kalantari R. Building the Human Argonaute 2 Interaction Network. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Jul 07]. Available from: http://hdl.handle.net/2152.5/1737.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kalantari R. Building the Human Argonaute 2 Interaction Network. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/1737

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

10. Hicks, Jessica Anne. Human GW182 Paralogs Are the Central Organizers for RNA-Mediate Control of Transcription.

Degree: 2017, University of Texas Southwestern Medical Center

 RNA interference (RNAi) is an endogenous mechanism for regulating gene expression that can be manipulated for experimental or therapeutic purposes to knockdown protein expression. In… (more)

Subjects/Keywords: Argonaute Proteins; Autoantigens; Cell Nucleus; RNA Interference; RNA-Binding Proteins

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APA (6th Edition):

Hicks, J. A. (2017). Human GW182 Paralogs Are the Central Organizers for RNA-Mediate Control of Transcription. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4229

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hicks, Jessica Anne. “Human GW182 Paralogs Are the Central Organizers for RNA-Mediate Control of Transcription.” 2017. Thesis, University of Texas Southwestern Medical Center. Accessed July 07, 2020. http://hdl.handle.net/2152.5/4229.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hicks, Jessica Anne. “Human GW182 Paralogs Are the Central Organizers for RNA-Mediate Control of Transcription.” 2017. Web. 07 Jul 2020.

Vancouver:

Hicks JA. Human GW182 Paralogs Are the Central Organizers for RNA-Mediate Control of Transcription. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2017. [cited 2020 Jul 07]. Available from: http://hdl.handle.net/2152.5/4229.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hicks JA. Human GW182 Paralogs Are the Central Organizers for RNA-Mediate Control of Transcription. [Thesis]. University of Texas Southwestern Medical Center; 2017. Available from: http://hdl.handle.net/2152.5/4229

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

11. Holohan, Brody Christopher. Identifying, Characterizing and Inhibiting the Telomerase Regulatory Network.

Degree: 2015, University of Texas Southwestern Medical Center

 Telomeres, which are structures that cap the ends of linear chromosomes are maintained by telomerase, a reverse transcriptase. Telomere length limits the self-renewal capacity for… (more)

Subjects/Keywords: Enzyme Inhibitors; Neoplasms; Phosphorylcholine; Telomerase

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APA (6th Edition):

Holohan, B. C. (2015). Identifying, Characterizing and Inhibiting the Telomerase Regulatory Network. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/5288

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Holohan, Brody Christopher. “Identifying, Characterizing and Inhibiting the Telomerase Regulatory Network.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed July 07, 2020. http://hdl.handle.net/2152.5/5288.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Holohan, Brody Christopher. “Identifying, Characterizing and Inhibiting the Telomerase Regulatory Network.” 2015. Web. 07 Jul 2020.

Vancouver:

Holohan BC. Identifying, Characterizing and Inhibiting the Telomerase Regulatory Network. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Jul 07]. Available from: http://hdl.handle.net/2152.5/5288.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Holohan BC. Identifying, Characterizing and Inhibiting the Telomerase Regulatory Network. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/5288

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

12. Nulf, Christopher J. Peptide Nucleic Acid (PNA) Hybridization to Nucleic Acid Targets.

Degree: PhD, 2004, University of Texas Southwestern Medical Center

 Peptide nucleic acid (PNA) is a DNA/RNA mimic that offers many advantages for hybridization to nucleic acid targets. The simple premise of Watson- Crick base-pairing… (more)

Subjects/Keywords: Mimicry, DNA; Oligonucleotides; Peptide Nucleic Acids

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APA (6th Edition):

Nulf, C. J. (2004). Peptide Nucleic Acid (PNA) Hybridization to Nucleic Acid Targets. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/348

Chicago Manual of Style (16th Edition):

Nulf, Christopher J. “Peptide Nucleic Acid (PNA) Hybridization to Nucleic Acid Targets.” 2004. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 07, 2020. http://hdl.handle.net/2152.5/348.

MLA Handbook (7th Edition):

Nulf, Christopher J. “Peptide Nucleic Acid (PNA) Hybridization to Nucleic Acid Targets.” 2004. Web. 07 Jul 2020.

Vancouver:

Nulf CJ. Peptide Nucleic Acid (PNA) Hybridization to Nucleic Acid Targets. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2004. [cited 2020 Jul 07]. Available from: http://hdl.handle.net/2152.5/348.

Council of Science Editors:

Nulf CJ. Peptide Nucleic Acid (PNA) Hybridization to Nucleic Acid Targets. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2004. Available from: http://hdl.handle.net/2152.5/348

.