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You searched for +publisher:"University of Texas Southwestern Medical Center" +contributor:("Cobb, Melanie H."). Showing records 1 – 30 of 75 total matches.

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University of Texas Southwestern Medical Center

1. Duan, Lingling. MAPK Signaling Pathways in Pancreatic Beta Cells: The Regulation of RAF Activation by Nutrient Stimuli.

Degree: 2011, University of Texas Southwestern Medical Center

 In pancreatic β cells cells, ERK1 and ERK2 participate in nutrient sensing and their activities rise and fall as a function of glucose concentration over… (more)

Subjects/Keywords: Mitogen-Activated Protein Kinase 1; Insulin-Secreting Cells; Calcineurin; Glucose

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APA (6th Edition):

Duan, L. (2011). MAPK Signaling Pathways in Pancreatic Beta Cells: The Regulation of RAF Activation by Nutrient Stimuli. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/833

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Duan, Lingling. “MAPK Signaling Pathways in Pancreatic Beta Cells: The Regulation of RAF Activation by Nutrient Stimuli.” 2011. Thesis, University of Texas Southwestern Medical Center. Accessed November 26, 2020. http://hdl.handle.net/2152.5/833.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Duan, Lingling. “MAPK Signaling Pathways in Pancreatic Beta Cells: The Regulation of RAF Activation by Nutrient Stimuli.” 2011. Web. 26 Nov 2020.

Vancouver:

Duan L. MAPK Signaling Pathways in Pancreatic Beta Cells: The Regulation of RAF Activation by Nutrient Stimuli. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2011. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2152.5/833.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Duan L. MAPK Signaling Pathways in Pancreatic Beta Cells: The Regulation of RAF Activation by Nutrient Stimuli. [Thesis]. University of Texas Southwestern Medical Center; 2011. Available from: http://hdl.handle.net/2152.5/833

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

2. Sengupta, Samarpita. A WNK and a Nudge Towards Kinase Biology.

Degree: 2013, University of Texas Southwestern Medical Center

 A family of four atypical protein kinases; WNKs are characterized by a non-canonical position of the catalytic lysine. The significance of WNKs was first realized… (more)

Subjects/Keywords: Protein-Serine-Threonine Kinases; Sodium-Potassium-Chloride Symporters; Intracellular Signaling Peptides and Proteins

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APA (6th Edition):

Sengupta, S. (2013). A WNK and a Nudge Towards Kinase Biology. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1256

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sengupta, Samarpita. “A WNK and a Nudge Towards Kinase Biology.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed November 26, 2020. http://hdl.handle.net/2152.5/1256.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sengupta, Samarpita. “A WNK and a Nudge Towards Kinase Biology.” 2013. Web. 26 Nov 2020.

Vancouver:

Sengupta S. A WNK and a Nudge Towards Kinase Biology. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2152.5/1256.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sengupta S. A WNK and a Nudge Towards Kinase Biology. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1256

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

3. Wedin, Kyle Edward. With No Lysine (WNK) Family Proteins and Their Interaction with Downstream Kinases.

Degree: 2011, University of Texas Southwestern Medical Center

 With no lysine (WNK) kinases are a family of protein kinases characterized by unusual kinase domain architecture. These large proteins, divergent outside of a kinase… (more)

Subjects/Keywords: Protein-Serine-Threonine Kinases; Hypertension; Transcription Factors

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APA (6th Edition):

Wedin, K. E. (2011). With No Lysine (WNK) Family Proteins and Their Interaction with Downstream Kinases. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/892

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wedin, Kyle Edward. “With No Lysine (WNK) Family Proteins and Their Interaction with Downstream Kinases.” 2011. Thesis, University of Texas Southwestern Medical Center. Accessed November 26, 2020. http://hdl.handle.net/2152.5/892.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wedin, Kyle Edward. “With No Lysine (WNK) Family Proteins and Their Interaction with Downstream Kinases.” 2011. Web. 26 Nov 2020.

Vancouver:

Wedin KE. With No Lysine (WNK) Family Proteins and Their Interaction with Downstream Kinases. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2011. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2152.5/892.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wedin KE. With No Lysine (WNK) Family Proteins and Their Interaction with Downstream Kinases. [Thesis]. University of Texas Southwestern Medical Center; 2011. Available from: http://hdl.handle.net/2152.5/892

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

4. Zhang, Zhenyu. The Mechanism of Small Interfering RNA Biogenesis in Neurospora Crassa.

Degree: 2014, University of Texas Southwestern Medical Center

 RNA interference is a well-conserved post-transcriptional gene silencing mechanism that regulates various biological processes including development, genome defense, and heterochromatin formation. In filamentous fungus Neurospora… (more)

Subjects/Keywords: Homologous Recombination; Neurospora; Repetitive Sequences, Nucleic Acid; RNA Interference; RNA, Small Interfering

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APA (6th Edition):

Zhang, Z. (2014). The Mechanism of Small Interfering RNA Biogenesis in Neurospora Crassa. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3334

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhang, Zhenyu. “The Mechanism of Small Interfering RNA Biogenesis in Neurospora Crassa.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed November 26, 2020. http://hdl.handle.net/2152.5/3334.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhang, Zhenyu. “The Mechanism of Small Interfering RNA Biogenesis in Neurospora Crassa.” 2014. Web. 26 Nov 2020.

Vancouver:

Zhang Z. The Mechanism of Small Interfering RNA Biogenesis in Neurospora Crassa. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2152.5/3334.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhang Z. The Mechanism of Small Interfering RNA Biogenesis in Neurospora Crassa. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3334

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

5. Selyunin, Andrey S. Identification and Characterization of a Bacterial Catalytic Scaffold with Specificity for Host Endomembrane Traffic.

Degree: 2013, University of Texas Southwestern Medical Center

 The fidelity and specificity of information flow within a cell is controlled by scaffolding proteins that assemble and link enzymes into signaling circuits. These circuits… (more)

Subjects/Keywords: ADP-Ribosylation Factors; Escherichia coli Proteins; rab GTP-Binding Proteins; Signal Transduction

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APA (6th Edition):

Selyunin, A. S. (2013). Identification and Characterization of a Bacterial Catalytic Scaffold with Specificity for Host Endomembrane Traffic. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/2729

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Selyunin, Andrey S. “Identification and Characterization of a Bacterial Catalytic Scaffold with Specificity for Host Endomembrane Traffic.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed November 26, 2020. http://hdl.handle.net/2152.5/2729.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Selyunin, Andrey S. “Identification and Characterization of a Bacterial Catalytic Scaffold with Specificity for Host Endomembrane Traffic.” 2013. Web. 26 Nov 2020.

Vancouver:

Selyunin AS. Identification and Characterization of a Bacterial Catalytic Scaffold with Specificity for Host Endomembrane Traffic. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2152.5/2729.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Selyunin AS. Identification and Characterization of a Bacterial Catalytic Scaffold with Specificity for Host Endomembrane Traffic. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/2729

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

6. An, Zhenyi. The Regulation of Autophagy and Its Role in Mitotic Exit.

Degree: 2014, University of Texas Southwestern Medical Center

 Autophagy is an evolutionarily conserved pathway in which cells enclose cytoplasmic contents in double membrane vesicles and deliver them to the lysosome for degradation. Autophagy… (more)

Subjects/Keywords: Apoptosis Regulatory Proteins; Autophagy; Mitogen-Activated Protein Kinases; Phosphorylation

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APA (6th Edition):

An, Z. (2014). The Regulation of Autophagy and Its Role in Mitotic Exit. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3582

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

An, Zhenyi. “The Regulation of Autophagy and Its Role in Mitotic Exit.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed November 26, 2020. http://hdl.handle.net/2152.5/3582.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

An, Zhenyi. “The Regulation of Autophagy and Its Role in Mitotic Exit.” 2014. Web. 26 Nov 2020.

Vancouver:

An Z. The Regulation of Autophagy and Its Role in Mitotic Exit. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2152.5/3582.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

An Z. The Regulation of Autophagy and Its Role in Mitotic Exit. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3582

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

7. McNamara, Ryan Philip. Enzymatic Disassembly of Promoter Bound 7SK snRNP Drives Transcription Elongation of HIV and Cellular Genes.

Degree: 2015, University of Texas Southwestern Medical Center

 Gene expression of the human immunodeficiency virus (HIV) and cellular primary responsive genes (PRG’s) is regulated at the step of transcription elongation. Shortly after transcription… (more)

Subjects/Keywords: Phosphoprotein Phosphatases; Positive Transcriptional Elongation Factor B; Promoter Regions, Genetic; Ribonucleoproteins, Small Nuclear; Transcription Elongation, Genetic

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APA (6th Edition):

McNamara, R. P. (2015). Enzymatic Disassembly of Promoter Bound 7SK snRNP Drives Transcription Elongation of HIV and Cellular Genes. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/2724

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

McNamara, Ryan Philip. “Enzymatic Disassembly of Promoter Bound 7SK snRNP Drives Transcription Elongation of HIV and Cellular Genes.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed November 26, 2020. http://hdl.handle.net/2152.5/2724.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

McNamara, Ryan Philip. “Enzymatic Disassembly of Promoter Bound 7SK snRNP Drives Transcription Elongation of HIV and Cellular Genes.” 2015. Web. 26 Nov 2020.

Vancouver:

McNamara RP. Enzymatic Disassembly of Promoter Bound 7SK snRNP Drives Transcription Elongation of HIV and Cellular Genes. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2152.5/2724.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

McNamara RP. Enzymatic Disassembly of Promoter Bound 7SK snRNP Drives Transcription Elongation of HIV and Cellular Genes. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/2724

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

8. Torres, Michael Jason 1982-. The Sec6/8 (a.k.a. Exocyst) Complex Supports DNA Repair Fidelity.

Degree: 2014, University of Texas Southwestern Medical Center

 The exocyst complex, first described in yeast, is a heterooctomeric complex that serves as a signaling platform to mediate cellular responses to diverse spatial and… (more)

Subjects/Keywords: DNA Repair; Genomic Instability; Vesicular Transport Proteins

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APA (6th Edition):

Torres, M. J. 1. (2014). The Sec6/8 (a.k.a. Exocyst) Complex Supports DNA Repair Fidelity. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3327

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Torres, Michael Jason 1982-. “The Sec6/8 (a.k.a. Exocyst) Complex Supports DNA Repair Fidelity.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed November 26, 2020. http://hdl.handle.net/2152.5/3327.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Torres, Michael Jason 1982-. “The Sec6/8 (a.k.a. Exocyst) Complex Supports DNA Repair Fidelity.” 2014. Web. 26 Nov 2020.

Vancouver:

Torres MJ1. The Sec6/8 (a.k.a. Exocyst) Complex Supports DNA Repair Fidelity. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2152.5/3327.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Torres MJ1. The Sec6/8 (a.k.a. Exocyst) Complex Supports DNA Repair Fidelity. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3327

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

9. Boldt, Clayton Ryan. Identification of Developmental Signaling Pathways with a Novel Role in Regulating Zebrafish Primordial Germ Cell Migration.

Degree: 2013, University of Texas Southwestern Medical Center

The file named "BOLDT-DISSERTATION-2013.pdf" is the primary dissertation file. Four (4) supplemental videos are also provided (in Audio Video Interleaved format).

Normal cell migration is… (more)

Subjects/Keywords: Cell Movement; Germ Cells; Signal Transduction; Tankyrases; Zebrafish

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APA (6th Edition):

Boldt, C. R. (2013). Identification of Developmental Signaling Pathways with a Novel Role in Regulating Zebrafish Primordial Germ Cell Migration. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1725

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Boldt, Clayton Ryan. “Identification of Developmental Signaling Pathways with a Novel Role in Regulating Zebrafish Primordial Germ Cell Migration.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed November 26, 2020. http://hdl.handle.net/2152.5/1725.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Boldt, Clayton Ryan. “Identification of Developmental Signaling Pathways with a Novel Role in Regulating Zebrafish Primordial Germ Cell Migration.” 2013. Web. 26 Nov 2020.

Vancouver:

Boldt CR. Identification of Developmental Signaling Pathways with a Novel Role in Regulating Zebrafish Primordial Germ Cell Migration. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2152.5/1725.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Boldt CR. Identification of Developmental Signaling Pathways with a Novel Role in Regulating Zebrafish Primordial Germ Cell Migration. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1725

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

10. Topalovski, Mary. Fibulin-5 Promotes Pancreatic Tumor Growth through Inhibition of Integrin-induced ROS: Insights into Tumor-Matrix Signaling.

Degree: 2016, University of Texas Southwestern Medical Center

 Elevated oxidative stress is an aberration seen in many solid tumors, and exploiting this biochemical difference has the potential to enhance the efficacy of anti-cancer… (more)

Subjects/Keywords: Carcinoma, Pancreatic Ductal; Extracellular Matrix Proteins; Gene Expression Regulation, Neoplastic; Neoplasms, Experimental; Pancreatic Neoplasms; Reactive Oxygen Species; Recombinant Proteins; Signal Transduction; Transforming Growth Factor beta

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APA (6th Edition):

Topalovski, M. (2016). Fibulin-5 Promotes Pancreatic Tumor Growth through Inhibition of Integrin-induced ROS: Insights into Tumor-Matrix Signaling. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/5737

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Topalovski, Mary. “Fibulin-5 Promotes Pancreatic Tumor Growth through Inhibition of Integrin-induced ROS: Insights into Tumor-Matrix Signaling.” 2016. Thesis, University of Texas Southwestern Medical Center. Accessed November 26, 2020. http://hdl.handle.net/2152.5/5737.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Topalovski, Mary. “Fibulin-5 Promotes Pancreatic Tumor Growth through Inhibition of Integrin-induced ROS: Insights into Tumor-Matrix Signaling.” 2016. Web. 26 Nov 2020.

Vancouver:

Topalovski M. Fibulin-5 Promotes Pancreatic Tumor Growth through Inhibition of Integrin-induced ROS: Insights into Tumor-Matrix Signaling. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2016. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2152.5/5737.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Topalovski M. Fibulin-5 Promotes Pancreatic Tumor Growth through Inhibition of Integrin-induced ROS: Insights into Tumor-Matrix Signaling. [Thesis]. University of Texas Southwestern Medical Center; 2016. Available from: http://hdl.handle.net/2152.5/5737

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

11. McReynolds, Andrea Christine. Functional Significance of Extracellular Signal Regulated Kinase (ERK2) Phosphorylation States: Implications for DNA Binding.

Degree: 2013, University of Texas Southwestern Medical Center

 The protein kinase extracellular signal-regulated kinase 2 (ERK2) has been well understood structurally for nearly twenty years. New insight is emerging about its structure and… (more)

Subjects/Keywords: DNA; Extracellular Signal-Regulated MAP Kinases; Phosphorylation

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APA (6th Edition):

McReynolds, A. C. (2013). Functional Significance of Extracellular Signal Regulated Kinase (ERK2) Phosphorylation States: Implications for DNA Binding. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/2725

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

McReynolds, Andrea Christine. “Functional Significance of Extracellular Signal Regulated Kinase (ERK2) Phosphorylation States: Implications for DNA Binding.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed November 26, 2020. http://hdl.handle.net/2152.5/2725.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

McReynolds, Andrea Christine. “Functional Significance of Extracellular Signal Regulated Kinase (ERK2) Phosphorylation States: Implications for DNA Binding.” 2013. Web. 26 Nov 2020.

Vancouver:

McReynolds AC. Functional Significance of Extracellular Signal Regulated Kinase (ERK2) Phosphorylation States: Implications for DNA Binding. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2152.5/2725.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

McReynolds AC. Functional Significance of Extracellular Signal Regulated Kinase (ERK2) Phosphorylation States: Implications for DNA Binding. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/2725

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

12. McMillan, Elizabeth Anne. Identification of Biomarker Driven Intervention Opportunities and Advancement of Mechanism of Action Predictions for Anti-Cancer Therapeutics.

Degree: 2017, University of Texas Southwestern Medical Center

 Oncogenic lesions arising during cancer progression provide an attractive target for chemical intervention strategies. The extreme molecular heterogeneity of tumors, however, makes it difficult to… (more)

Subjects/Keywords: Carcinoma, Non-Small-Cell Lung; High-Throughput Screening Assays; Lung Neoplasms; NF-E2-Related Factor 2

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APA (6th Edition):

McMillan, E. A. (2017). Identification of Biomarker Driven Intervention Opportunities and Advancement of Mechanism of Action Predictions for Anti-Cancer Therapeutics. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/6616

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

McMillan, Elizabeth Anne. “Identification of Biomarker Driven Intervention Opportunities and Advancement of Mechanism of Action Predictions for Anti-Cancer Therapeutics.” 2017. Thesis, University of Texas Southwestern Medical Center. Accessed November 26, 2020. http://hdl.handle.net/2152.5/6616.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

McMillan, Elizabeth Anne. “Identification of Biomarker Driven Intervention Opportunities and Advancement of Mechanism of Action Predictions for Anti-Cancer Therapeutics.” 2017. Web. 26 Nov 2020.

Vancouver:

McMillan EA. Identification of Biomarker Driven Intervention Opportunities and Advancement of Mechanism of Action Predictions for Anti-Cancer Therapeutics. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2017. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2152.5/6616.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

McMillan EA. Identification of Biomarker Driven Intervention Opportunities and Advancement of Mechanism of Action Predictions for Anti-Cancer Therapeutics. [Thesis]. University of Texas Southwestern Medical Center; 2017. Available from: http://hdl.handle.net/2152.5/6616

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

13. Farley, Demetra Dannielle. Role for NIP45 in Telomere Recruitment to PML Bodies in ALT Cancer Cells.

Degree: 2013, University of Texas Southwestern Medical Center

 Telomere length maintenance is critical for continued cell proliferation. The SMC5/6 complex, required for double-strand break (DSB) repair in both yeast and humans, has been… (more)

Subjects/Keywords: Telomere Homeostasis; Carrier Proteins; Neoplasms

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APA (6th Edition):

Farley, D. D. (2013). Role for NIP45 in Telomere Recruitment to PML Bodies in ALT Cancer Cells. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1348

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Farley, Demetra Dannielle. “Role for NIP45 in Telomere Recruitment to PML Bodies in ALT Cancer Cells.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed November 26, 2020. http://hdl.handle.net/2152.5/1348.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Farley, Demetra Dannielle. “Role for NIP45 in Telomere Recruitment to PML Bodies in ALT Cancer Cells.” 2013. Web. 26 Nov 2020.

Vancouver:

Farley DD. Role for NIP45 in Telomere Recruitment to PML Bodies in ALT Cancer Cells. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2152.5/1348.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Farley DD. Role for NIP45 in Telomere Recruitment to PML Bodies in ALT Cancer Cells. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1348

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

14. Szabla, Kristen Lynn. Molecular Mechanisms Underlying Fast-Acting Antidepressant Efficacy.

Degree: 2015, University of Texas Southwestern Medical Center

 Major Depressive Disorder is a devastating mental illness with a profound disease burden, particularly in the United States. Major Depressive Disorder is a heterogeneous disorder… (more)

Subjects/Keywords: Antidepressive Agents; Brain-Derived Neurotrophic Factor; Depression; Ketamine

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APA (6th Edition):

Szabla, K. L. (2015). Molecular Mechanisms Underlying Fast-Acting Antidepressant Efficacy. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4127

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Szabla, Kristen Lynn. “Molecular Mechanisms Underlying Fast-Acting Antidepressant Efficacy.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed November 26, 2020. http://hdl.handle.net/2152.5/4127.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Szabla, Kristen Lynn. “Molecular Mechanisms Underlying Fast-Acting Antidepressant Efficacy.” 2015. Web. 26 Nov 2020.

Vancouver:

Szabla KL. Molecular Mechanisms Underlying Fast-Acting Antidepressant Efficacy. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2152.5/4127.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Szabla KL. Molecular Mechanisms Underlying Fast-Acting Antidepressant Efficacy. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4127

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

15. Shi, Lei. Metabolic Regulation of Quiescence Entry and Exit in Saccharomyces cerevisiae.

Degree: 2014, University of Texas Southwestern Medical Center

 Unicellular microorganisms often enter a state called quiescence when they encounter harsh environmental conditions. They stop growth and proliferation until conditions improve. In quiescence, the… (more)

Subjects/Keywords: Acetyl Coenzyme A; Cell Cycle; Cyclins; Saccharomyces cerevisiae; Saccharomyces cerevisiae Proteins; Transcription, Genetic

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APA (6th Edition):

Shi, L. (2014). Metabolic Regulation of Quiescence Entry and Exit in Saccharomyces cerevisiae. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3955

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shi, Lei. “Metabolic Regulation of Quiescence Entry and Exit in Saccharomyces cerevisiae.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed November 26, 2020. http://hdl.handle.net/2152.5/3955.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shi, Lei. “Metabolic Regulation of Quiescence Entry and Exit in Saccharomyces cerevisiae.” 2014. Web. 26 Nov 2020.

Vancouver:

Shi L. Metabolic Regulation of Quiescence Entry and Exit in Saccharomyces cerevisiae. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2152.5/3955.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shi L. Metabolic Regulation of Quiescence Entry and Exit in Saccharomyces cerevisiae. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3955

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

16. Owen, Joshua Lee. Insulin Stimulation of Sterol Regulatory Element-Binding Protein-1c Processing in Liver: Lessons from Transgenic Rats.

Degree: 2015, University of Texas Southwestern Medical Center

 Transcriptional control of hepatic fatty acid (FA) and triglyceride (TG) synthesis is mediated by SREBP-1c, one of three sterol regulatory element-binding protein (SREBP) isoforms. SREBPs… (more)

Subjects/Keywords: Insulin; Lipogenesis; Liver; Ribosomal Protein S6 Kinases, 70-kDa; Sterol Regulatory Element Binding Protein 1

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APA (6th Edition):

Owen, J. L. (2015). Insulin Stimulation of Sterol Regulatory Element-Binding Protein-1c Processing in Liver: Lessons from Transgenic Rats. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1584

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Owen, Joshua Lee. “Insulin Stimulation of Sterol Regulatory Element-Binding Protein-1c Processing in Liver: Lessons from Transgenic Rats.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed November 26, 2020. http://hdl.handle.net/2152.5/1584.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Owen, Joshua Lee. “Insulin Stimulation of Sterol Regulatory Element-Binding Protein-1c Processing in Liver: Lessons from Transgenic Rats.” 2015. Web. 26 Nov 2020.

Vancouver:

Owen JL. Insulin Stimulation of Sterol Regulatory Element-Binding Protein-1c Processing in Liver: Lessons from Transgenic Rats. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2152.5/1584.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Owen JL. Insulin Stimulation of Sterol Regulatory Element-Binding Protein-1c Processing in Liver: Lessons from Transgenic Rats. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/1584

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

17. Cormier, Kevin William. Exploration of Chemical and Biochemical Mechanisms of Catalysis.

Degree: 2013, University of Texas Southwestern Medical Center

 Nature uses proteins to catalyze a wide range of chemical processes that control cellular signaling. One such enzyme is ERK2 which plays a role in… (more)

Subjects/Keywords: MAP Kinase Signaling System; Pyrroles; Vanadium

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APA (6th Edition):

Cormier, K. W. (2013). Exploration of Chemical and Biochemical Mechanisms of Catalysis. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1730

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cormier, Kevin William. “Exploration of Chemical and Biochemical Mechanisms of Catalysis.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed November 26, 2020. http://hdl.handle.net/2152.5/1730.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cormier, Kevin William. “Exploration of Chemical and Biochemical Mechanisms of Catalysis.” 2013. Web. 26 Nov 2020.

Vancouver:

Cormier KW. Exploration of Chemical and Biochemical Mechanisms of Catalysis. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2152.5/1730.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cormier KW. Exploration of Chemical and Biochemical Mechanisms of Catalysis. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1730

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

18. Skaug, Brian. Mechanisms Governing NF-κB Regulation by the Anti-Inflammatory Protein A20.

Degree: 2013, University of Texas Southwestern Medical Center

 A20 is a potent anti-inflammatory protein that inhibits NF-ΚB, and A20 dysfunction is associated with autoimmunity and B-cell lymphoma. A20 harbors a deubiquitination enzyme domain… (more)

Subjects/Keywords: I-kappa B Kinase; Inflammation; Intracellular Signaling Peptides and Proteins; NF-kappa B

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APA (6th Edition):

Skaug, B. (2013). Mechanisms Governing NF-κB Regulation by the Anti-Inflammatory Protein A20. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1706

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Skaug, Brian. “Mechanisms Governing NF-κB Regulation by the Anti-Inflammatory Protein A20.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed November 26, 2020. http://hdl.handle.net/2152.5/1706.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Skaug, Brian. “Mechanisms Governing NF-κB Regulation by the Anti-Inflammatory Protein A20.” 2013. Web. 26 Nov 2020.

Vancouver:

Skaug B. Mechanisms Governing NF-κB Regulation by the Anti-Inflammatory Protein A20. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2152.5/1706.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Skaug B. Mechanisms Governing NF-κB Regulation by the Anti-Inflammatory Protein A20. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1706

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

19. Long, Chengzu. Prevention of Muscular Dystrophy in Mice by Gene Editing.

Degree: 2014, University of Texas Southwestern Medical Center

 Duchenne muscular dystrophy (DMD) is an inherited X-linked disease caused by mutations in the gene encoding dystrophin, a protein required for muscle fiber integrity. DMD… (more)

Subjects/Keywords: CRISPR-Cas Systems; Dystrophin; Gene Targeting; Muscular Dystrophy, Duchenne

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APA (6th Edition):

Long, C. (2014). Prevention of Muscular Dystrophy in Mice by Gene Editing. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3953

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Long, Chengzu. “Prevention of Muscular Dystrophy in Mice by Gene Editing.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed November 26, 2020. http://hdl.handle.net/2152.5/3953.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Long, Chengzu. “Prevention of Muscular Dystrophy in Mice by Gene Editing.” 2014. Web. 26 Nov 2020.

Vancouver:

Long C. Prevention of Muscular Dystrophy in Mice by Gene Editing. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2152.5/3953.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Long C. Prevention of Muscular Dystrophy in Mice by Gene Editing. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3953

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

20. Kim, Soonjoung. Spindle Checkpoint at Kinetochores.

Degree: 2014, University of Texas Southwestern Medical Center

 The kinetochore—a large protein assembly on centromeric chromatin—functions as the docking site for spindle microtubules and as a signaling hub for the spindle checkpoint. The… (more)

Subjects/Keywords: Cell Cycle Proteins; Kinetochores; M Phase Cell Cycle Checkpoints; Mad2 Proteins; Phosphorylation; Spindle Apparatus

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APA (6th Edition):

Kim, S. (2014). Spindle Checkpoint at Kinetochores. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3590

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kim, Soonjoung. “Spindle Checkpoint at Kinetochores.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed November 26, 2020. http://hdl.handle.net/2152.5/3590.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kim, Soonjoung. “Spindle Checkpoint at Kinetochores.” 2014. Web. 26 Nov 2020.

Vancouver:

Kim S. Spindle Checkpoint at Kinetochores. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2152.5/3590.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kim S. Spindle Checkpoint at Kinetochores. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3590

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

21. Kim, Ji Mi. Identification of Oncogenic KRAS-Associated Vulnerabilities in Non-Small Cell Lung Cancer.

Degree: 2016, University of Texas Southwestern Medical Center

 Activating mutations in KRAS are frequently involved in the pathogenesis of non-small cell lung cancer (NSCLC), the disease responsible for the most cancer-related deaths in… (more)

Subjects/Keywords: Active Transport, Cell Nucleus; Cell Nucleus; Karyopherins; Lung Neoplasms; Proto-Oncogene Proteins p21(ras); Receptors, Cytoplasmic and Nuclear

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APA (6th Edition):

Kim, J. M. (2016). Identification of Oncogenic KRAS-Associated Vulnerabilities in Non-Small Cell Lung Cancer. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/5726

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kim, Ji Mi. “Identification of Oncogenic KRAS-Associated Vulnerabilities in Non-Small Cell Lung Cancer.” 2016. Thesis, University of Texas Southwestern Medical Center. Accessed November 26, 2020. http://hdl.handle.net/2152.5/5726.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kim, Ji Mi. “Identification of Oncogenic KRAS-Associated Vulnerabilities in Non-Small Cell Lung Cancer.” 2016. Web. 26 Nov 2020.

Vancouver:

Kim JM. Identification of Oncogenic KRAS-Associated Vulnerabilities in Non-Small Cell Lung Cancer. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2016. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2152.5/5726.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kim JM. Identification of Oncogenic KRAS-Associated Vulnerabilities in Non-Small Cell Lung Cancer. [Thesis]. University of Texas Southwestern Medical Center; 2016. Available from: http://hdl.handle.net/2152.5/5726

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

22. Ou, Yi-Hung 1977-. Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation.

Degree: 2013, University of Texas Southwestern Medical Center

 An essential kinase in innate immune signaling, TBK1 couples pathogen surveillance to induction of host defense mechanisms. The pathological activation of TBK1 in cancer can… (more)

Subjects/Keywords: Immunity, Innate; Neoplasms; Protein-Serine-Threonine Kinases; Proto-Oncogene Proteins c-akt

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APA (6th Edition):

Ou, Y. 1. (2013). Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-125

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ou, Yi-Hung 1977-. “Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed November 26, 2020. http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-125.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ou, Yi-Hung 1977-. “Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation.” 2013. Web. 26 Nov 2020.

Vancouver:

Ou Y1. Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-125.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ou Y1. Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-125

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

23. He, Min. Analysis of ASCL1 in Neuroendocrine Lung Cancer.

Degree: 2015, University of Texas Southwestern Medical Center

 Small cell lung cancer (SCLC) is an understudied tumor subset with aggressive neuroendocrine carcinoma features. Previous studies have determined that the basic helix-loop-helix (bHLH) transcription… (more)

Subjects/Keywords: Basic Helix-Loop-Helix Transcription Factors; Carcinoma, Non-Small-Cell Lung; Cell Lineage; Neuroendocrine Tumors; Oncogenes; Small Cell Lung Carcinoma

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APA (6th Edition):

He, M. (2015). Analysis of ASCL1 in Neuroendocrine Lung Cancer. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4227

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

He, Min. “Analysis of ASCL1 in Neuroendocrine Lung Cancer.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed November 26, 2020. http://hdl.handle.net/2152.5/4227.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

He, Min. “Analysis of ASCL1 in Neuroendocrine Lung Cancer.” 2015. Web. 26 Nov 2020.

Vancouver:

He M. Analysis of ASCL1 in Neuroendocrine Lung Cancer. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2152.5/4227.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

He M. Analysis of ASCL1 in Neuroendocrine Lung Cancer. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4227

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

24. Gallolu Kankanamalage, Sachith Sandaruwan Perera. Identifying Novel Functions of the WNK Pathway.

Degree: 2017, University of Texas Southwestern Medical Center

 The with no lysine [K] (WNK) pathway consists of WNK kinases, their downstream target kinases, oxidative stress responsive (OSR)1 and SPS/Ste20-related proline-alanine-rich kinase (SPAK), and… (more)

Subjects/Keywords: AMP-Activated Protein Kinases; Autophagy; Signal Transduction; WNK Lysine-Deficient Protein Kinase 1

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APA (6th Edition):

Gallolu Kankanamalage, S. S. P. (2017). Identifying Novel Functions of the WNK Pathway. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/7738

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gallolu Kankanamalage, Sachith Sandaruwan Perera. “Identifying Novel Functions of the WNK Pathway.” 2017. Thesis, University of Texas Southwestern Medical Center. Accessed November 26, 2020. http://hdl.handle.net/2152.5/7738.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gallolu Kankanamalage, Sachith Sandaruwan Perera. “Identifying Novel Functions of the WNK Pathway.” 2017. Web. 26 Nov 2020.

Vancouver:

Gallolu Kankanamalage SSP. Identifying Novel Functions of the WNK Pathway. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2017. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2152.5/7738.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gallolu Kankanamalage SSP. Identifying Novel Functions of the WNK Pathway. [Thesis]. University of Texas Southwestern Medical Center; 2017. Available from: http://hdl.handle.net/2152.5/7738

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

25. Jia, Luying. The Multifunctional Kinase Bub1 Acts as a Signaling Hub for the Spindle Checkpoint.

Degree: 2015, University of Texas Southwestern Medical Center

 The spindle checkpoint is an essential mechanism to ensure accurate chromosome segregation during mitosis. The checkpoint signal originates from the kinetochore, which is a huge… (more)

Subjects/Keywords: Anaphase-Promoting Complex-Cyclosome; Cdc20 Proteins; Cell Cycle Proteins; M Phase Cell Cycle Checkpoints; Protein-Serine-Threonine Kinases; Proto-Oncogene Proteins

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APA (6th Edition):

Jia, L. (2015). The Multifunctional Kinase Bub1 Acts as a Signaling Hub for the Spindle Checkpoint. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4456

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jia, Luying. “The Multifunctional Kinase Bub1 Acts as a Signaling Hub for the Spindle Checkpoint.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed November 26, 2020. http://hdl.handle.net/2152.5/4456.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jia, Luying. “The Multifunctional Kinase Bub1 Acts as a Signaling Hub for the Spindle Checkpoint.” 2015. Web. 26 Nov 2020.

Vancouver:

Jia L. The Multifunctional Kinase Bub1 Acts as a Signaling Hub for the Spindle Checkpoint. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2152.5/4456.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jia L. The Multifunctional Kinase Bub1 Acts as a Signaling Hub for the Spindle Checkpoint. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4456

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

26. Zaganjor, Elma 1981-. Regulation by ERK1/2 of Novel Substrates, Kinesins KIF2A and KIF2C.

Degree: 2013, University of Texas Southwestern Medical Center

 The kinesin-like protein KIF2A is a microtubule-associated motor protein thatauses microtubule depolymerization by inducing a conformational change in tubulin. The depolymerase function of KIF2A is… (more)

Subjects/Keywords: Cell Transformation, Neoplastic; Extracellular Signal-Regulated MAP Kinases; Kinesin

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APA (6th Edition):

Zaganjor, E. 1. (2013). Regulation by ERK1/2 of Novel Substrates, Kinesins KIF2A and KIF2C. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-124

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zaganjor, Elma 1981-. “Regulation by ERK1/2 of Novel Substrates, Kinesins KIF2A and KIF2C.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed November 26, 2020. http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-124.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zaganjor, Elma 1981-. “Regulation by ERK1/2 of Novel Substrates, Kinesins KIF2A and KIF2C.” 2013. Web. 26 Nov 2020.

Vancouver:

Zaganjor E1. Regulation by ERK1/2 of Novel Substrates, Kinesins KIF2A and KIF2C. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-124.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zaganjor E1. Regulation by ERK1/2 of Novel Substrates, Kinesins KIF2A and KIF2C. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-124

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

27. Estrada, Armando, III 1980-. Regulation of Cell Migration by WNK1.

Degree: 2012, University of Texas Southwestern Medical Center

 Cell motility is an immensely complex process that involves reorganization of the cytoskeleton, and consequent membrane deformation, triggered by a variety of motogenic stimuli, including… (more)

Subjects/Keywords: Cell Movement; Cytoskeleton; Protein-Serine-Threonine Kinases

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APA (6th Edition):

Estrada, Armando, I. 1. (2012). Regulation of Cell Migration by WNK1. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/ETD-UTSWMED-2012-12-65

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Estrada, Armando, III 1980-. “Regulation of Cell Migration by WNK1.” 2012. Thesis, University of Texas Southwestern Medical Center. Accessed November 26, 2020. http://hdl.handle.net/2152.5/ETD-UTSWMED-2012-12-65.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Estrada, Armando, III 1980-. “Regulation of Cell Migration by WNK1.” 2012. Web. 26 Nov 2020.

Vancouver:

Estrada, Armando I1. Regulation of Cell Migration by WNK1. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2012. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2012-12-65.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Estrada, Armando I1. Regulation of Cell Migration by WNK1. [Thesis]. University of Texas Southwestern Medical Center; 2012. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2012-12-65

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

28. Koo, Yeon Seung. Blood Vessel Development.

Degree: 2014, University of Texas Southwestern Medical Center

 Cardiovascular system is the first developing functional organ in vertebrates, and one of the fundamental organ systems through adulthoods. Cardiovascular function depends on patent blood… (more)

Subjects/Keywords: Blood Vessels; Endothelium, Vascular; GTPase-Activating Proteins; Intracellular Signaling Peptides and Proteins

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APA (6th Edition):

Koo, Y. S. (2014). Blood Vessel Development. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3949

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Koo, Yeon Seung. “Blood Vessel Development.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed November 26, 2020. http://hdl.handle.net/2152.5/3949.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Koo, Yeon Seung. “Blood Vessel Development.” 2014. Web. 26 Nov 2020.

Vancouver:

Koo YS. Blood Vessel Development. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2152.5/3949.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Koo YS. Blood Vessel Development. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3949

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

29. Woolery, Andrew Ryan. Fic-Mediated AMPylation in Bacterial Infection and Endoplasmic Reticulum Stress.

Degree: 2015, University of Texas Southwestern Medical Center

 The post-translational modification AMPylation is emerging as a significant regulatory mechanism in both prokaryotic and eukaryotic biology. This process involves the covalent addition of an… (more)

Subjects/Keywords: Adenosine Monophosphate; Endoplasmic Reticulum; Nucleotidyltransferases; Unfolded Protein Response; Vibrio parahaemolyticus

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APA (6th Edition):

Woolery, A. R. (2015). Fic-Mediated AMPylation in Bacterial Infection and Endoplasmic Reticulum Stress. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4132

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Woolery, Andrew Ryan. “Fic-Mediated AMPylation in Bacterial Infection and Endoplasmic Reticulum Stress.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed November 26, 2020. http://hdl.handle.net/2152.5/4132.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Woolery, Andrew Ryan. “Fic-Mediated AMPylation in Bacterial Infection and Endoplasmic Reticulum Stress.” 2015. Web. 26 Nov 2020.

Vancouver:

Woolery AR. Fic-Mediated AMPylation in Bacterial Infection and Endoplasmic Reticulum Stress. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2152.5/4132.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Woolery AR. Fic-Mediated AMPylation in Bacterial Infection and Endoplasmic Reticulum Stress. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4132

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

30. Sanchez, Angelica. BMP Signaling Regulates Germ Cell Pluripotency, Sexual Differentiation, and Cancer Susceptibility.

Degree: 2016, University of Texas Southwestern Medical Center

 Testicular germ cell tumors are the most common malignancy found in young men between the ages of 14-40. While these tumors are highly curable with… (more)

Subjects/Keywords: Bone Morphogenetic Proteins; Cell Differentiation; Germ Cells; Neoplasms, Germ Cell and Embryonal; Sex Differentiation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sanchez, A. (2016). BMP Signaling Regulates Germ Cell Pluripotency, Sexual Differentiation, and Cancer Susceptibility. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/6149

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sanchez, Angelica. “BMP Signaling Regulates Germ Cell Pluripotency, Sexual Differentiation, and Cancer Susceptibility.” 2016. Thesis, University of Texas Southwestern Medical Center. Accessed November 26, 2020. http://hdl.handle.net/2152.5/6149.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sanchez, Angelica. “BMP Signaling Regulates Germ Cell Pluripotency, Sexual Differentiation, and Cancer Susceptibility.” 2016. Web. 26 Nov 2020.

Vancouver:

Sanchez A. BMP Signaling Regulates Germ Cell Pluripotency, Sexual Differentiation, and Cancer Susceptibility. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2016. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2152.5/6149.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sanchez A. BMP Signaling Regulates Germ Cell Pluripotency, Sexual Differentiation, and Cancer Susceptibility. [Thesis]. University of Texas Southwestern Medical Center; 2016. Available from: http://hdl.handle.net/2152.5/6149

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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