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You searched for +publisher:"University of Southern California" +contributor:("Watanabe, Richard M."). Showing records 1 – 23 of 23 total matches.

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University of Southern California

1. Chen, Yen-Fu. Genetic variations in gene from the cytochrome P450 family may account for differential response to pioglitazone therapy in the Hispanic women.

Degree: MS, Biostatistics, 2009, University of Southern California

 Studies have shown that in addition to treating T2DM, TZDs may be effective in the prevention of T2DM as first demonstrated in the TRIPOD and… (more)

Subjects/Keywords: SNP; PIPOD; diabetes; principal component analysis; pioglitazone; cytochrome P450; TZD; CYP2C8; CYP2C9; CYP3A4; T2DM

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APA (6th Edition):

Chen, Y. (2009). Genetic variations in gene from the cytochrome P450 family may account for differential response to pioglitazone therapy in the Hispanic women. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/278329/rec/3009

Chicago Manual of Style (16th Edition):

Chen, Yen-Fu. “Genetic variations in gene from the cytochrome P450 family may account for differential response to pioglitazone therapy in the Hispanic women.” 2009. Masters Thesis, University of Southern California. Accessed October 14, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/278329/rec/3009.

MLA Handbook (7th Edition):

Chen, Yen-Fu. “Genetic variations in gene from the cytochrome P450 family may account for differential response to pioglitazone therapy in the Hispanic women.” 2009. Web. 14 Oct 2019.

Vancouver:

Chen Y. Genetic variations in gene from the cytochrome P450 family may account for differential response to pioglitazone therapy in the Hispanic women. [Internet] [Masters thesis]. University of Southern California; 2009. [cited 2019 Oct 14]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/278329/rec/3009.

Council of Science Editors:

Chen Y. Genetic variations in gene from the cytochrome P450 family may account for differential response to pioglitazone therapy in the Hispanic women. [Masters Thesis]. University of Southern California; 2009. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/278329/rec/3009


University of Southern California

2. Chiu, Jenny Dong-Nee. Hemodynamic factors in the insulin resistance of obesity and hyperlipidemia.

Degree: PhD, Physiology & Biophysics, 2009, University of Southern California

 Insulin resistance is an important risk factor for the development of type 2 diabetes, obesity, cardiovascular disease, hypertension (24;73;241), and certain cancers (95). Although it… (more)

Subjects/Keywords: diabetes; endothelium; free fatty acids; insulin; interstitial; transendothelial transport

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APA (6th Edition):

Chiu, J. D. (2009). Hemodynamic factors in the insulin resistance of obesity and hyperlipidemia. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/566298/rec/3146

Chicago Manual of Style (16th Edition):

Chiu, Jenny Dong-Nee. “Hemodynamic factors in the insulin resistance of obesity and hyperlipidemia.” 2009. Doctoral Dissertation, University of Southern California. Accessed October 14, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/566298/rec/3146.

MLA Handbook (7th Edition):

Chiu, Jenny Dong-Nee. “Hemodynamic factors in the insulin resistance of obesity and hyperlipidemia.” 2009. Web. 14 Oct 2019.

Vancouver:

Chiu JD. Hemodynamic factors in the insulin resistance of obesity and hyperlipidemia. [Internet] [Doctoral dissertation]. University of Southern California; 2009. [cited 2019 Oct 14]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/566298/rec/3146.

Council of Science Editors:

Chiu JD. Hemodynamic factors in the insulin resistance of obesity and hyperlipidemia. [Doctoral Dissertation]. University of Southern California; 2009. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/566298/rec/3146


University of Southern California

3. Beale, Elizabeth Ogden. Lessons learned conducting a clinical study.

Degree: MS, Preventive Medicine (Health Behavior), 2009, University of Southern California

 Background. The aim of the Masters of Science in Clinical and Biomedical Investigations is for the candidate to develop the ability to conduct a real-world… (more)

Subjects/Keywords: type 2 diabetes; genetics; HNF4a; Mexican American; clinical study

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APA (6th Edition):

Beale, E. O. (2009). Lessons learned conducting a clinical study. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/206052/rec/3801

Chicago Manual of Style (16th Edition):

Beale, Elizabeth Ogden. “Lessons learned conducting a clinical study.” 2009. Masters Thesis, University of Southern California. Accessed October 14, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/206052/rec/3801.

MLA Handbook (7th Edition):

Beale, Elizabeth Ogden. “Lessons learned conducting a clinical study.” 2009. Web. 14 Oct 2019.

Vancouver:

Beale EO. Lessons learned conducting a clinical study. [Internet] [Masters thesis]. University of Southern California; 2009. [cited 2019 Oct 14]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/206052/rec/3801.

Council of Science Editors:

Beale EO. Lessons learned conducting a clinical study. [Masters Thesis]. University of Southern California; 2009. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/206052/rec/3801


University of Southern California

4. Zhang, Yaping. Systems pharmacology for acute myeloid leukemia and feedback control pharmacodynamic models.

Degree: PhD, Biomedical Engineering, 2015, University of Southern California

 Systems pharmacology is an emerging discipline with the goal of understanding how drugs modulate cellular networks in space and time and how they impact human… (more)

Subjects/Keywords: FLT3 inhibitors; CDK4/6 inhibitors; acute myeloid leukemia; STAT5; Rb; subcutaneous tumor; orthotopic tumor; feedback control; indirect response models; proportional feedback; integral feedback; derivative feedback

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APA (6th Edition):

Zhang, Y. (2015). Systems pharmacology for acute myeloid leukemia and feedback control pharmacodynamic models. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/524108/rec/6302

Chicago Manual of Style (16th Edition):

Zhang, Yaping. “Systems pharmacology for acute myeloid leukemia and feedback control pharmacodynamic models.” 2015. Doctoral Dissertation, University of Southern California. Accessed October 14, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/524108/rec/6302.

MLA Handbook (7th Edition):

Zhang, Yaping. “Systems pharmacology for acute myeloid leukemia and feedback control pharmacodynamic models.” 2015. Web. 14 Oct 2019.

Vancouver:

Zhang Y. Systems pharmacology for acute myeloid leukemia and feedback control pharmacodynamic models. [Internet] [Doctoral dissertation]. University of Southern California; 2015. [cited 2019 Oct 14]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/524108/rec/6302.

Council of Science Editors:

Zhang Y. Systems pharmacology for acute myeloid leukemia and feedback control pharmacodynamic models. [Doctoral Dissertation]. University of Southern California; 2015. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/524108/rec/6302


University of Southern California

5. Walker, Ryan William. Genetic and dietary determinants of nonalcoholic fatty liver disease in Hispanic children.

Degree: PhD, Integrative Biology of Disease, 2013, University of Southern California

 The purpose of this dissertation is to summarize our recent findings that have examined dietary, genetic and inflammatory factors that contribute to fat accumulation and… (more)

Subjects/Keywords: NAFLD; obesity; liver fat; HFCS; sugar; children; Hispanic; liver; fat; genetic; SNP; polymorphism; dietary

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APA (6th Edition):

Walker, R. W. (2013). Genetic and dietary determinants of nonalcoholic fatty liver disease in Hispanic children. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/310859/rec/2991

Chicago Manual of Style (16th Edition):

Walker, Ryan William. “Genetic and dietary determinants of nonalcoholic fatty liver disease in Hispanic children.” 2013. Doctoral Dissertation, University of Southern California. Accessed October 14, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/310859/rec/2991.

MLA Handbook (7th Edition):

Walker, Ryan William. “Genetic and dietary determinants of nonalcoholic fatty liver disease in Hispanic children.” 2013. Web. 14 Oct 2019.

Vancouver:

Walker RW. Genetic and dietary determinants of nonalcoholic fatty liver disease in Hispanic children. [Internet] [Doctoral dissertation]. University of Southern California; 2013. [cited 2019 Oct 14]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/310859/rec/2991.

Council of Science Editors:

Walker RW. Genetic and dietary determinants of nonalcoholic fatty liver disease in Hispanic children. [Doctoral Dissertation]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/310859/rec/2991


University of Southern California

6. Gyllenhammer, Lauren Elizabeth. Quantity versus quality: how adipose tissue accumulation and immune cell profile associate with risk for type 2 diabetes in minority children and adults.

Degree: PhD, Integrative Biology of Disease, 2015, University of Southern California

 Obesity affects an estimated 78.6 million adults in the US and 396 million globally, and is associated with type 2 diabetes, cardiovascular disease, non-alcoholic fatty… (more)

Subjects/Keywords: adipose tissue; ectopic fat; type 2 diabetes; liver fat; visceral fat; inflammation; cortisol; T regulatory cells

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APA (6th Edition):

Gyllenhammer, L. E. (2015). Quantity versus quality: how adipose tissue accumulation and immune cell profile associate with risk for type 2 diabetes in minority children and adults. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/526587/rec/5353

Chicago Manual of Style (16th Edition):

Gyllenhammer, Lauren Elizabeth. “Quantity versus quality: how adipose tissue accumulation and immune cell profile associate with risk for type 2 diabetes in minority children and adults.” 2015. Doctoral Dissertation, University of Southern California. Accessed October 14, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/526587/rec/5353.

MLA Handbook (7th Edition):

Gyllenhammer, Lauren Elizabeth. “Quantity versus quality: how adipose tissue accumulation and immune cell profile associate with risk for type 2 diabetes in minority children and adults.” 2015. Web. 14 Oct 2019.

Vancouver:

Gyllenhammer LE. Quantity versus quality: how adipose tissue accumulation and immune cell profile associate with risk for type 2 diabetes in minority children and adults. [Internet] [Doctoral dissertation]. University of Southern California; 2015. [cited 2019 Oct 14]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/526587/rec/5353.

Council of Science Editors:

Gyllenhammer LE. Quantity versus quality: how adipose tissue accumulation and immune cell profile associate with risk for type 2 diabetes in minority children and adults. [Doctoral Dissertation]. University of Southern California; 2015. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/526587/rec/5353


University of Southern California

7. O'Brien, Kathryn Elizabeth. Extracellular Hsp90α in diabetic wound healing: structure, function, and mechanism of action.

Degree: PhD, Genetic, Molecular and Cellular Biology, 2014, University of Southern California

 Chronic and non-healing skin wounds represent a significant clinical, economic and social problem in both developing and developed countries. The current cost for care of… (more)

Subjects/Keywords: wound healing; chronic wounds; diabetic foot ulcers; diabetic porcine model; porcine wound healing model; secreted Hsp90 alpha; Hsp90 alpha; dfu

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APA (6th Edition):

O'Brien, K. E. (2014). Extracellular Hsp90α in diabetic wound healing: structure, function, and mechanism of action. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/506946/rec/2690

Chicago Manual of Style (16th Edition):

O'Brien, Kathryn Elizabeth. “Extracellular Hsp90α in diabetic wound healing: structure, function, and mechanism of action.” 2014. Doctoral Dissertation, University of Southern California. Accessed October 14, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/506946/rec/2690.

MLA Handbook (7th Edition):

O'Brien, Kathryn Elizabeth. “Extracellular Hsp90α in diabetic wound healing: structure, function, and mechanism of action.” 2014. Web. 14 Oct 2019.

Vancouver:

O'Brien KE. Extracellular Hsp90α in diabetic wound healing: structure, function, and mechanism of action. [Internet] [Doctoral dissertation]. University of Southern California; 2014. [cited 2019 Oct 14]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/506946/rec/2690.

Council of Science Editors:

O'Brien KE. Extracellular Hsp90α in diabetic wound healing: structure, function, and mechanism of action. [Doctoral Dissertation]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/506946/rec/2690


University of Southern California

8. Tomooka, Beren H. Flipped zinc transporter 8 (ZnT8): a novel approach to characterize zinc transport and its possible relevance to type 2 diabetes.

Degree: MS, Physiology & Biophysics, 2009, University of Southern California

 Zinc is an essential trace element in living organisms, and zinc content in pancreatic β-cells is amongst the highest in the body. Zinc transporter 8… (more)

Subjects/Keywords: zinc transport; zinc transporter 8; slc30a8; diabetes; fluozin; insulin; SNP

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APA (6th Edition):

Tomooka, B. H. (2009). Flipped zinc transporter 8 (ZnT8): a novel approach to characterize zinc transport and its possible relevance to type 2 diabetes. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/252558/rec/2843

Chicago Manual of Style (16th Edition):

Tomooka, Beren H. “Flipped zinc transporter 8 (ZnT8): a novel approach to characterize zinc transport and its possible relevance to type 2 diabetes.” 2009. Masters Thesis, University of Southern California. Accessed October 14, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/252558/rec/2843.

MLA Handbook (7th Edition):

Tomooka, Beren H. “Flipped zinc transporter 8 (ZnT8): a novel approach to characterize zinc transport and its possible relevance to type 2 diabetes.” 2009. Web. 14 Oct 2019.

Vancouver:

Tomooka BH. Flipped zinc transporter 8 (ZnT8): a novel approach to characterize zinc transport and its possible relevance to type 2 diabetes. [Internet] [Masters thesis]. University of Southern California; 2009. [cited 2019 Oct 14]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/252558/rec/2843.

Council of Science Editors:

Tomooka BH. Flipped zinc transporter 8 (ZnT8): a novel approach to characterize zinc transport and its possible relevance to type 2 diabetes. [Masters Thesis]. University of Southern California; 2009. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/252558/rec/2843


University of Southern California

9. Stefanovski, Darko. Nutrients as signal for insulin resistance induced hyperinsulinemic compensation.

Degree: PhD, Physiology & Biophysics, 2009, University of Southern California

 In normal individuals, the product of insulin sensitivity and insulin secretion is approximately a constant, referred to as disposition index (DI) and represents a quantitative… (more)

Subjects/Keywords: disposition index; hyperinsulinemic compensation; insulin resistance; metabolic syndrome; obesity; Type 2 diabetes

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APA (6th Edition):

Stefanovski, D. (2009). Nutrients as signal for insulin resistance induced hyperinsulinemic compensation. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/267683/rec/4488

Chicago Manual of Style (16th Edition):

Stefanovski, Darko. “Nutrients as signal for insulin resistance induced hyperinsulinemic compensation.” 2009. Doctoral Dissertation, University of Southern California. Accessed October 14, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/267683/rec/4488.

MLA Handbook (7th Edition):

Stefanovski, Darko. “Nutrients as signal for insulin resistance induced hyperinsulinemic compensation.” 2009. Web. 14 Oct 2019.

Vancouver:

Stefanovski D. Nutrients as signal for insulin resistance induced hyperinsulinemic compensation. [Internet] [Doctoral dissertation]. University of Southern California; 2009. [cited 2019 Oct 14]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/267683/rec/4488.

Council of Science Editors:

Stefanovski D. Nutrients as signal for insulin resistance induced hyperinsulinemic compensation. [Doctoral Dissertation]. University of Southern California; 2009. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/267683/rec/4488


University of Southern California

10. Nakamura, Kyle J. Route-specific selection during mother-to-child transmission of HIV-1.

Degree: PhD, Systems Biology and Disease, 2011, University of Southern California

 The struggle to contain and extinguish the worldwide HIV/AIDS pandemic, now in its 30th year, may represent the single greatest challenge faced by modern medical… (more)

Subjects/Keywords: HIV; AIDS; mother-to-child transmission

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APA (6th Edition):

Nakamura, K. J. (2011). Route-specific selection during mother-to-child transmission of HIV-1. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/619869/rec/5653

Chicago Manual of Style (16th Edition):

Nakamura, Kyle J. “Route-specific selection during mother-to-child transmission of HIV-1.” 2011. Doctoral Dissertation, University of Southern California. Accessed October 14, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/619869/rec/5653.

MLA Handbook (7th Edition):

Nakamura, Kyle J. “Route-specific selection during mother-to-child transmission of HIV-1.” 2011. Web. 14 Oct 2019.

Vancouver:

Nakamura KJ. Route-specific selection during mother-to-child transmission of HIV-1. [Internet] [Doctoral dissertation]. University of Southern California; 2011. [cited 2019 Oct 14]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/619869/rec/5653.

Council of Science Editors:

Nakamura KJ. Route-specific selection during mother-to-child transmission of HIV-1. [Doctoral Dissertation]. University of Southern California; 2011. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/619869/rec/5653


University of Southern California

11. Black, Mary Helen. Multivariate methods for extracting genetic associations from correlated data.

Degree: PhD, Statistical Genetics & Genetic Epidemiology, 2010, University of Southern California

 Multivariate methods ranging from traditional joint SNP methods to principal components analysis (PCA) have been developed for testing multiple markers in a region for association… (more)

Subjects/Keywords: genetic epidemiology; principal components; cluster analysis; association studies; candidate gene

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APA (6th Edition):

Black, M. H. (2010). Multivariate methods for extracting genetic associations from correlated data. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/179175/rec/4281

Chicago Manual of Style (16th Edition):

Black, Mary Helen. “Multivariate methods for extracting genetic associations from correlated data.” 2010. Doctoral Dissertation, University of Southern California. Accessed October 14, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/179175/rec/4281.

MLA Handbook (7th Edition):

Black, Mary Helen. “Multivariate methods for extracting genetic associations from correlated data.” 2010. Web. 14 Oct 2019.

Vancouver:

Black MH. Multivariate methods for extracting genetic associations from correlated data. [Internet] [Doctoral dissertation]. University of Southern California; 2010. [cited 2019 Oct 14]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/179175/rec/4281.

Council of Science Editors:

Black MH. Multivariate methods for extracting genetic associations from correlated data. [Doctoral Dissertation]. University of Southern California; 2010. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/179175/rec/4281


University of Southern California

12. Toledo-Corral, Claudia M. Subclinical atherosclerosis in overweight Latino youth: influence of cardiometabolic risk factors.

Degree: PhD, Preventive Medicine (Health Behavior), 2010, University of Southern California

 Cardiovascular disease is the leading cause of death in the general population. Arterial inflammation and damage manifests as arterial thickening and is the pathological basis… (more)

Subjects/Keywords: childhood obesity; carotid intima media thickness; subclinical atherosclerosis; insulin resistance; adiposity; LDL-cholesterol; high blood pressure; impaired fasting glucose

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APA (6th Edition):

Toledo-Corral, C. M. (2010). Subclinical atherosclerosis in overweight Latino youth: influence of cardiometabolic risk factors. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/408621/rec/6186

Chicago Manual of Style (16th Edition):

Toledo-Corral, Claudia M. “Subclinical atherosclerosis in overweight Latino youth: influence of cardiometabolic risk factors.” 2010. Doctoral Dissertation, University of Southern California. Accessed October 14, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/408621/rec/6186.

MLA Handbook (7th Edition):

Toledo-Corral, Claudia M. “Subclinical atherosclerosis in overweight Latino youth: influence of cardiometabolic risk factors.” 2010. Web. 14 Oct 2019.

Vancouver:

Toledo-Corral CM. Subclinical atherosclerosis in overweight Latino youth: influence of cardiometabolic risk factors. [Internet] [Doctoral dissertation]. University of Southern California; 2010. [cited 2019 Oct 14]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/408621/rec/6186.

Council of Science Editors:

Toledo-Corral CM. Subclinical atherosclerosis in overweight Latino youth: influence of cardiometabolic risk factors. [Doctoral Dissertation]. University of Southern California; 2010. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/408621/rec/6186


University of Southern California

13. Tsai, Pei-Jung. The development of type 2 diabetes mellitus (type 2 DM) in California twins.

Degree: PhD, Epidemiology, 2011, University of Southern California

 Previous studies have shown that the development of type 2 diabetes mellitus (Type 2 DM) is affected by multiple genetic and environmental factors. Numerous family… (more)

Subjects/Keywords: type 2 diabetes; type 2 diabetes mellitus; genetic susceptibility; monozygotic; MZ; dizygotic; DZ; disease discordant probability; genetic susceptibility; body mass index; smoking; monozygotic; MZ; dizygotic; DZ; adjusted odds ratio; adjusted OR

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APA (6th Edition):

Tsai, P. (2011). The development of type 2 diabetes mellitus (type 2 DM) in California twins. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/470530/rec/6557

Chicago Manual of Style (16th Edition):

Tsai, Pei-Jung. “The development of type 2 diabetes mellitus (type 2 DM) in California twins.” 2011. Doctoral Dissertation, University of Southern California. Accessed October 14, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/470530/rec/6557.

MLA Handbook (7th Edition):

Tsai, Pei-Jung. “The development of type 2 diabetes mellitus (type 2 DM) in California twins.” 2011. Web. 14 Oct 2019.

Vancouver:

Tsai P. The development of type 2 diabetes mellitus (type 2 DM) in California twins. [Internet] [Doctoral dissertation]. University of Southern California; 2011. [cited 2019 Oct 14]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/470530/rec/6557.

Council of Science Editors:

Tsai P. The development of type 2 diabetes mellitus (type 2 DM) in California twins. [Doctoral Dissertation]. University of Southern California; 2011. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/470530/rec/6557


University of Southern California

14. Waters, Kevin M. Examining the relationship between common genetic variation, type 2 diabetes and prostate cancer risk in the multiethnic cohort.

Degree: PhD, Epidemiology, 2010, University of Southern California

 This thesis is comprised of five studies that examine the relationship between common genetic variation, type 2 diabetes and prostate cancer risk.; Generalizability of Associations… (more)

Subjects/Keywords: prostate cancer; type 2 diabetes; common genetic variation; genetic susceptibility; multiethnic cohort; MSMB

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APA (6th Edition):

Waters, K. M. (2010). Examining the relationship between common genetic variation, type 2 diabetes and prostate cancer risk in the multiethnic cohort. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/373007/rec/2593

Chicago Manual of Style (16th Edition):

Waters, Kevin M. “Examining the relationship between common genetic variation, type 2 diabetes and prostate cancer risk in the multiethnic cohort.” 2010. Doctoral Dissertation, University of Southern California. Accessed October 14, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/373007/rec/2593.

MLA Handbook (7th Edition):

Waters, Kevin M. “Examining the relationship between common genetic variation, type 2 diabetes and prostate cancer risk in the multiethnic cohort.” 2010. Web. 14 Oct 2019.

Vancouver:

Waters KM. Examining the relationship between common genetic variation, type 2 diabetes and prostate cancer risk in the multiethnic cohort. [Internet] [Doctoral dissertation]. University of Southern California; 2010. [cited 2019 Oct 14]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/373007/rec/2593.

Council of Science Editors:

Waters KM. Examining the relationship between common genetic variation, type 2 diabetes and prostate cancer risk in the multiethnic cohort. [Doctoral Dissertation]. University of Southern California; 2010. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/373007/rec/2593


University of Southern California

15. Wang, Jian. ABCB1 and IMPDH functional and genetic contributions to tacrolimus and mycophenolic acid therapy in transplant patients.

Degree: PhD, Molecular Pharmacology & Toxicology, 2009, University of Southern California

 Tacrolimus and mycophenolic acid (MPA) are commonly used in clinic to prevent rejection in transplant recipients. The influence of ABCB1 polymorphisms on tacrolimus dosing has… (more)

Subjects/Keywords: pharmacogenetics; ABCB1; IMPDH; transplantation; polymorphism

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APA (6th Edition):

Wang, J. (2009). ABCB1 and IMPDH functional and genetic contributions to tacrolimus and mycophenolic acid therapy in transplant patients. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/535363/rec/458

Chicago Manual of Style (16th Edition):

Wang, Jian. “ABCB1 and IMPDH functional and genetic contributions to tacrolimus and mycophenolic acid therapy in transplant patients.” 2009. Doctoral Dissertation, University of Southern California. Accessed October 14, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/535363/rec/458.

MLA Handbook (7th Edition):

Wang, Jian. “ABCB1 and IMPDH functional and genetic contributions to tacrolimus and mycophenolic acid therapy in transplant patients.” 2009. Web. 14 Oct 2019.

Vancouver:

Wang J. ABCB1 and IMPDH functional and genetic contributions to tacrolimus and mycophenolic acid therapy in transplant patients. [Internet] [Doctoral dissertation]. University of Southern California; 2009. [cited 2019 Oct 14]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/535363/rec/458.

Council of Science Editors:

Wang J. ABCB1 and IMPDH functional and genetic contributions to tacrolimus and mycophenolic acid therapy in transplant patients. [Doctoral Dissertation]. University of Southern California; 2009. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/535363/rec/458


University of Southern California

16. Chen, Zhanghua. Evaluating the associations between the baseline and other exposure variables with the longitudinal trajectory when responses are measured with error.

Degree: PhD, Biostatistics, 2013, University of Southern California

 Evaluating the associations between the baseline value and other exposure variables with the longitudinal trajectory constantly occurs in the medical research. Traditional analyses using the… (more)

Subjects/Keywords: baseline adjustment; baseline prediction; longitudinal analysis; measurement error; mixed effects model; regression to the mean

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APA (6th Edition):

Chen, Z. (2013). Evaluating the associations between the baseline and other exposure variables with the longitudinal trajectory when responses are measured with error. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/351704/rec/2526

Chicago Manual of Style (16th Edition):

Chen, Zhanghua. “Evaluating the associations between the baseline and other exposure variables with the longitudinal trajectory when responses are measured with error.” 2013. Doctoral Dissertation, University of Southern California. Accessed October 14, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/351704/rec/2526.

MLA Handbook (7th Edition):

Chen, Zhanghua. “Evaluating the associations between the baseline and other exposure variables with the longitudinal trajectory when responses are measured with error.” 2013. Web. 14 Oct 2019.

Vancouver:

Chen Z. Evaluating the associations between the baseline and other exposure variables with the longitudinal trajectory when responses are measured with error. [Internet] [Doctoral dissertation]. University of Southern California; 2013. [cited 2019 Oct 14]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/351704/rec/2526.

Council of Science Editors:

Chen Z. Evaluating the associations between the baseline and other exposure variables with the longitudinal trajectory when responses are measured with error. [Doctoral Dissertation]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/351704/rec/2526


University of Southern California

17. Hsu, Isabel R. The physiologic and pathophysiologic role of sympathetic nervous system induced pulsatile lipolysis in metabolism.

Degree: PhD, Physiology & Biophysics, 2010, University of Southern California

 Insulin resistance is a major risk factor for the development of type 2 diabetes and the metabolic syndrome. Activation of the sympathetic nervous system (SNS)… (more)

Subjects/Keywords: lipolysis; sympathetic nervous system; free fatty acids; pulsatile

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APA (6th Edition):

Hsu, I. R. (2010). The physiologic and pathophysiologic role of sympathetic nervous system induced pulsatile lipolysis in metabolism. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/193569/rec/7078

Chicago Manual of Style (16th Edition):

Hsu, Isabel R. “The physiologic and pathophysiologic role of sympathetic nervous system induced pulsatile lipolysis in metabolism.” 2010. Doctoral Dissertation, University of Southern California. Accessed October 14, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/193569/rec/7078.

MLA Handbook (7th Edition):

Hsu, Isabel R. “The physiologic and pathophysiologic role of sympathetic nervous system induced pulsatile lipolysis in metabolism.” 2010. Web. 14 Oct 2019.

Vancouver:

Hsu IR. The physiologic and pathophysiologic role of sympathetic nervous system induced pulsatile lipolysis in metabolism. [Internet] [Doctoral dissertation]. University of Southern California; 2010. [cited 2019 Oct 14]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/193569/rec/7078.

Council of Science Editors:

Hsu IR. The physiologic and pathophysiologic role of sympathetic nervous system induced pulsatile lipolysis in metabolism. [Doctoral Dissertation]. University of Southern California; 2010. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/193569/rec/7078


University of Southern California

18. Veerappan, Chendhore Sai. Epigenetic plasticity of cultured female human embryonic stem cells and regulation of gene expression and chromatin by PR-SET7 mediated H4K20me1.

Degree: PhD, Genetic, Molecular and Cellular Biology, 2012, University of Southern California

 Epigenetics is the study of changes in gene expression that occur in cells without alterations to DNA sequence. Epigenetic modifications are critical components of eukaryotic… (more)

Subjects/Keywords: epigenetics; embryonic stem cells; X-chromosome inactivation; H4K4me3; H3K27me3; heterogeneity

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APA (6th Edition):

Veerappan, C. S. (2012). Epigenetic plasticity of cultured female human embryonic stem cells and regulation of gene expression and chromatin by PR-SET7 mediated H4K20me1. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/112300/rec/2411

Chicago Manual of Style (16th Edition):

Veerappan, Chendhore Sai. “Epigenetic plasticity of cultured female human embryonic stem cells and regulation of gene expression and chromatin by PR-SET7 mediated H4K20me1.” 2012. Doctoral Dissertation, University of Southern California. Accessed October 14, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/112300/rec/2411.

MLA Handbook (7th Edition):

Veerappan, Chendhore Sai. “Epigenetic plasticity of cultured female human embryonic stem cells and regulation of gene expression and chromatin by PR-SET7 mediated H4K20me1.” 2012. Web. 14 Oct 2019.

Vancouver:

Veerappan CS. Epigenetic plasticity of cultured female human embryonic stem cells and regulation of gene expression and chromatin by PR-SET7 mediated H4K20me1. [Internet] [Doctoral dissertation]. University of Southern California; 2012. [cited 2019 Oct 14]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/112300/rec/2411.

Council of Science Editors:

Veerappan CS. Epigenetic plasticity of cultured female human embryonic stem cells and regulation of gene expression and chromatin by PR-SET7 mediated H4K20me1. [Doctoral Dissertation]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/112300/rec/2411


University of Southern California

19. Li, Xia. Variation in insulin-like growth factor-2 binding protein 2 interacts with adiposity to alter insulin sensitivity in Mexican Americans.

Degree: MS, Applied Biostatistics & Epidemiology, 2008, University of Southern California

 GWA studies have shown that variation in IGF2BP2 is associated with T2DM. We examined a 20 kb region of IGF2BP2 for association with T2DM-related quantitative… (more)

Subjects/Keywords: adiposity; genetics; IGF2BP2; insulin resistance; Mexican American; type 2 diabetes

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APA (6th Edition):

Li, X. (2008). Variation in insulin-like growth factor-2 binding protein 2 interacts with adiposity to alter insulin sensitivity in Mexican Americans. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/97796/rec/7803

Chicago Manual of Style (16th Edition):

Li, Xia. “Variation in insulin-like growth factor-2 binding protein 2 interacts with adiposity to alter insulin sensitivity in Mexican Americans.” 2008. Masters Thesis, University of Southern California. Accessed October 14, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/97796/rec/7803.

MLA Handbook (7th Edition):

Li, Xia. “Variation in insulin-like growth factor-2 binding protein 2 interacts with adiposity to alter insulin sensitivity in Mexican Americans.” 2008. Web. 14 Oct 2019.

Vancouver:

Li X. Variation in insulin-like growth factor-2 binding protein 2 interacts with adiposity to alter insulin sensitivity in Mexican Americans. [Internet] [Masters thesis]. University of Southern California; 2008. [cited 2019 Oct 14]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/97796/rec/7803.

Council of Science Editors:

Li X. Variation in insulin-like growth factor-2 binding protein 2 interacts with adiposity to alter insulin sensitivity in Mexican Americans. [Masters Thesis]. University of Southern California; 2008. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/97796/rec/7803


University of Southern California

20. Du, Lingyun. A hierarchical physiologically-based pharmacokinetic modeling platform for genetic and exposure effects in metabolic pathways.

Degree: MS, Biostatistics, 2007, University of Southern California

 Metabolic pathways are candidates for examining gene-exposure interaction effects on complex diseases. We present a hierarchical modeling platform integrating physiologically based pharmocokinetic models and Bayesian… (more)

Subjects/Keywords: metabolic pathways; physiologically based pharmacokinetic models; Bayesian approaches; hierarchical modeling; exposure; genotype; Michaelis-Menten; intermediate measurements; enzyme activities; metabolite concentrations; Markov chain Monte Carlo

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APA (6th Edition):

Du, L. (2007). A hierarchical physiologically-based pharmacokinetic modeling platform for genetic and exposure effects in metabolic pathways. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/516458/rec/215

Chicago Manual of Style (16th Edition):

Du, Lingyun. “A hierarchical physiologically-based pharmacokinetic modeling platform for genetic and exposure effects in metabolic pathways.” 2007. Masters Thesis, University of Southern California. Accessed October 14, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/516458/rec/215.

MLA Handbook (7th Edition):

Du, Lingyun. “A hierarchical physiologically-based pharmacokinetic modeling platform for genetic and exposure effects in metabolic pathways.” 2007. Web. 14 Oct 2019.

Vancouver:

Du L. A hierarchical physiologically-based pharmacokinetic modeling platform for genetic and exposure effects in metabolic pathways. [Internet] [Masters thesis]. University of Southern California; 2007. [cited 2019 Oct 14]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/516458/rec/215.

Council of Science Editors:

Du L. A hierarchical physiologically-based pharmacokinetic modeling platform for genetic and exposure effects in metabolic pathways. [Masters Thesis]. University of Southern California; 2007. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/516458/rec/215


University of Southern California

21. Kim, Stella P. Mechanisms relating obesity to insulin resistance and the metabolic syndrome.

Degree: PhD, Physiology & Biophysics, 2007, University of Southern California

 Insulin resistance has long been a distinguishing characteristic and major contributor in the pathogenesis of type 2 diabetes. Although there is no single cause for… (more)

Subjects/Keywords: insulin resistance; obesity; diabetes

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APA (6th Edition):

Kim, S. P. (2007). Mechanisms relating obesity to insulin resistance and the metabolic syndrome. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/492414/rec/4016

Chicago Manual of Style (16th Edition):

Kim, Stella P. “Mechanisms relating obesity to insulin resistance and the metabolic syndrome.” 2007. Doctoral Dissertation, University of Southern California. Accessed October 14, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/492414/rec/4016.

MLA Handbook (7th Edition):

Kim, Stella P. “Mechanisms relating obesity to insulin resistance and the metabolic syndrome.” 2007. Web. 14 Oct 2019.

Vancouver:

Kim SP. Mechanisms relating obesity to insulin resistance and the metabolic syndrome. [Internet] [Doctoral dissertation]. University of Southern California; 2007. [cited 2019 Oct 14]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/492414/rec/4016.

Council of Science Editors:

Kim SP. Mechanisms relating obesity to insulin resistance and the metabolic syndrome. [Doctoral Dissertation]. University of Southern California; 2007. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/492414/rec/4016


University of Southern California

22. Lottati, Maya. Omentectomy in the canine: a unique model to investigate the physiologic relationship between visceral fat and insulin action.

Degree: PhD, Physiology & Biophysics, 2008, University of Southern California

 Visceral obesity is associated with a variety of chronic diseases including type 2 diabetes and cardiovascular disease. A reduction in visceral fat by omentectomy improves… (more)

Subjects/Keywords: visceral fat; insulin resistance; omentectomy

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APA (6th Edition):

Lottati, M. (2008). Omentectomy in the canine: a unique model to investigate the physiologic relationship between visceral fat and insulin action. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/72231/rec/4512

Chicago Manual of Style (16th Edition):

Lottati, Maya. “Omentectomy in the canine: a unique model to investigate the physiologic relationship between visceral fat and insulin action.” 2008. Doctoral Dissertation, University of Southern California. Accessed October 14, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/72231/rec/4512.

MLA Handbook (7th Edition):

Lottati, Maya. “Omentectomy in the canine: a unique model to investigate the physiologic relationship between visceral fat and insulin action.” 2008. Web. 14 Oct 2019.

Vancouver:

Lottati M. Omentectomy in the canine: a unique model to investigate the physiologic relationship between visceral fat and insulin action. [Internet] [Doctoral dissertation]. University of Southern California; 2008. [cited 2019 Oct 14]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/72231/rec/4512.

Council of Science Editors:

Lottati M. Omentectomy in the canine: a unique model to investigate the physiologic relationship between visceral fat and insulin action. [Doctoral Dissertation]. University of Southern California; 2008. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/72231/rec/4512


University of Southern California

23. Zheng, Dan. Exenatide, an incretin hormone & AMP-activated protein kinase, an energy sensor, emerge as novel insulin-sensitizing agents.

Degree: PhD, Physiology & Biophysics, 2008, University of Southern California

 The present dissertation focuses on two novel metabolic players: exenatide, an incretin mimetic, and AMP-activated protein kinase, a cell energy sensor.; Exenatide is a long-acting… (more)

Subjects/Keywords: exenatide; insulin action; AMP-activated protein kinase

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APA (6th Edition):

Zheng, D. (2008). Exenatide, an incretin hormone & AMP-activated protein kinase, an energy sensor, emerge as novel insulin-sensitizing agents. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/124122/rec/2604

Chicago Manual of Style (16th Edition):

Zheng, Dan. “Exenatide, an incretin hormone & AMP-activated protein kinase, an energy sensor, emerge as novel insulin-sensitizing agents.” 2008. Doctoral Dissertation, University of Southern California. Accessed October 14, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/124122/rec/2604.

MLA Handbook (7th Edition):

Zheng, Dan. “Exenatide, an incretin hormone & AMP-activated protein kinase, an energy sensor, emerge as novel insulin-sensitizing agents.” 2008. Web. 14 Oct 2019.

Vancouver:

Zheng D. Exenatide, an incretin hormone & AMP-activated protein kinase, an energy sensor, emerge as novel insulin-sensitizing agents. [Internet] [Doctoral dissertation]. University of Southern California; 2008. [cited 2019 Oct 14]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/124122/rec/2604.

Council of Science Editors:

Zheng D. Exenatide, an incretin hormone & AMP-activated protein kinase, an energy sensor, emerge as novel insulin-sensitizing agents. [Doctoral Dissertation]. University of Southern California; 2008. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/124122/rec/2604

.