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You searched for +publisher:"University of Southern California" +contributor:("Tokes, Zoltan A."). Showing records 1 – 30 of 94 total matches.

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University of Southern California

1. Madhav, Anisha. The noncanonical role of telomerase in prostate cancer cells: exploring a non-telomeric signaling role for telomerase protein (TERT) in a cancer cell line.

Degree: MS, Biochemistry and Molecular Biology, 2012, University of Southern California

 The telomerase reverse transcriptase (TERT) component of telomerase has reverse transcriptase activity capable of elongating telomeres during each replication cycle of the cell. TERT plays… (more)

Subjects/Keywords: telomerase; TERT; prostate cancer; cancer stem cells

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APA (6th Edition):

Madhav, A. (2012). The noncanonical role of telomerase in prostate cancer cells: exploring a non-telomeric signaling role for telomerase protein (TERT) in a cancer cell line. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/30249/rec/7025

Chicago Manual of Style (16th Edition):

Madhav, Anisha. “The noncanonical role of telomerase in prostate cancer cells: exploring a non-telomeric signaling role for telomerase protein (TERT) in a cancer cell line.” 2012. Masters Thesis, University of Southern California. Accessed June 17, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/30249/rec/7025.

MLA Handbook (7th Edition):

Madhav, Anisha. “The noncanonical role of telomerase in prostate cancer cells: exploring a non-telomeric signaling role for telomerase protein (TERT) in a cancer cell line.” 2012. Web. 17 Jun 2019.

Vancouver:

Madhav A. The noncanonical role of telomerase in prostate cancer cells: exploring a non-telomeric signaling role for telomerase protein (TERT) in a cancer cell line. [Internet] [Masters thesis]. University of Southern California; 2012. [cited 2019 Jun 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/30249/rec/7025.

Council of Science Editors:

Madhav A. The noncanonical role of telomerase in prostate cancer cells: exploring a non-telomeric signaling role for telomerase protein (TERT) in a cancer cell line. [Masters Thesis]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/30249/rec/7025


University of Southern California

2. Zhang, Mengrao. Characteristics of hydrogen peroxide inducible clone-5 and its potential role as a nuclear receptor coactivator.

Degree: MS, Biochemistry and Molecular Biology, 2013, University of Southern California

 Hydrogen peroxide inducible clone-5 (Hic-5), also named transforming growth factor-β1 induced transcript 1(TGFB1 1), TGF-β stimulated clone-5 (TSC-5) or androgen receptor associated protein 55 (ARA55),… (more)

Subjects/Keywords: hydrogen peroxide inducible clone-5; nuclear receptor coactivator

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APA (6th Edition):

Zhang, M. (2013). Characteristics of hydrogen peroxide inducible clone-5 and its potential role as a nuclear receptor coactivator. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/336409/rec/1291

Chicago Manual of Style (16th Edition):

Zhang, Mengrao. “Characteristics of hydrogen peroxide inducible clone-5 and its potential role as a nuclear receptor coactivator.” 2013. Masters Thesis, University of Southern California. Accessed June 17, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/336409/rec/1291.

MLA Handbook (7th Edition):

Zhang, Mengrao. “Characteristics of hydrogen peroxide inducible clone-5 and its potential role as a nuclear receptor coactivator.” 2013. Web. 17 Jun 2019.

Vancouver:

Zhang M. Characteristics of hydrogen peroxide inducible clone-5 and its potential role as a nuclear receptor coactivator. [Internet] [Masters thesis]. University of Southern California; 2013. [cited 2019 Jun 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/336409/rec/1291.

Council of Science Editors:

Zhang M. Characteristics of hydrogen peroxide inducible clone-5 and its potential role as a nuclear receptor coactivator. [Masters Thesis]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/336409/rec/1291


University of Southern California

3. Chang, Yin-Wei. Creating a multiple micrornia expression vector to target GRP78, an ER chaperone and signaling regulator in cancer.

Degree: MS, Biochemistry and Molecular Biology, 2012, University of Southern California

 GRP78, a major endoplasmic reticulum (ER) chaperone and signaling regulator, is commonly over-expressed in human cancer and contributes to cancer cell proliferation, survival and tumorigenesis.… (more)

Subjects/Keywords: GRP78; microRNA

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APA (6th Edition):

Chang, Y. (2012). Creating a multiple micrornia expression vector to target GRP78, an ER chaperone and signaling regulator in cancer. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/83898/rec/1684

Chicago Manual of Style (16th Edition):

Chang, Yin-Wei. “Creating a multiple micrornia expression vector to target GRP78, an ER chaperone and signaling regulator in cancer.” 2012. Masters Thesis, University of Southern California. Accessed June 17, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/83898/rec/1684.

MLA Handbook (7th Edition):

Chang, Yin-Wei. “Creating a multiple micrornia expression vector to target GRP78, an ER chaperone and signaling regulator in cancer.” 2012. Web. 17 Jun 2019.

Vancouver:

Chang Y. Creating a multiple micrornia expression vector to target GRP78, an ER chaperone and signaling regulator in cancer. [Internet] [Masters thesis]. University of Southern California; 2012. [cited 2019 Jun 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/83898/rec/1684.

Council of Science Editors:

Chang Y. Creating a multiple micrornia expression vector to target GRP78, an ER chaperone and signaling regulator in cancer. [Masters Thesis]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/83898/rec/1684


University of Southern California

4. Yu, Jiali. Preosteoblast-specific RUNX2 promotes RANKL membrane association: antagonism by sex steroids through a non-genomic mechanism.

Degree: MS, Biochemistry and Molecular Biology, 2015, University of Southern California

 Molecular mechanisms underlying the bone‐sparing effects of sex steroid hormones are not fully understood. We show that RUNX2, a master regulator of osteoblast differentiation and… (more)

Subjects/Keywords: RUNX2; RANKL; sex steroids; osteoblast differentiation

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APA (6th Edition):

Yu, J. (2015). Preosteoblast-specific RUNX2 promotes RANKL membrane association: antagonism by sex steroids through a non-genomic mechanism. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/571674/rec/5193

Chicago Manual of Style (16th Edition):

Yu, Jiali. “Preosteoblast-specific RUNX2 promotes RANKL membrane association: antagonism by sex steroids through a non-genomic mechanism.” 2015. Masters Thesis, University of Southern California. Accessed June 17, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/571674/rec/5193.

MLA Handbook (7th Edition):

Yu, Jiali. “Preosteoblast-specific RUNX2 promotes RANKL membrane association: antagonism by sex steroids through a non-genomic mechanism.” 2015. Web. 17 Jun 2019.

Vancouver:

Yu J. Preosteoblast-specific RUNX2 promotes RANKL membrane association: antagonism by sex steroids through a non-genomic mechanism. [Internet] [Masters thesis]. University of Southern California; 2015. [cited 2019 Jun 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/571674/rec/5193.

Council of Science Editors:

Yu J. Preosteoblast-specific RUNX2 promotes RANKL membrane association: antagonism by sex steroids through a non-genomic mechanism. [Masters Thesis]. University of Southern California; 2015. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/571674/rec/5193


University of Southern California

5. Hong, Dai. Studies on the role of a novel protein, TMEM 56 in tumorigenic growth for MCF-7 cells.

Degree: MS, Biochemistry and Molecular Biology, 2014, University of Southern California

 Transmembrane protein TMEM 56 was first identified as an IKK‐associated protein by mass spectrometry in Dr. Ebrahim Zandi’s lab. This 263AA protein was found to… (more)

Subjects/Keywords: protein; TMEM 56; tumorigenic growth; MCF-7 cells

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APA (6th Edition):

Hong, D. (2014). Studies on the role of a novel protein, TMEM 56 in tumorigenic growth for MCF-7 cells. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/404283/rec/6174

Chicago Manual of Style (16th Edition):

Hong, Dai. “Studies on the role of a novel protein, TMEM 56 in tumorigenic growth for MCF-7 cells.” 2014. Masters Thesis, University of Southern California. Accessed June 17, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/404283/rec/6174.

MLA Handbook (7th Edition):

Hong, Dai. “Studies on the role of a novel protein, TMEM 56 in tumorigenic growth for MCF-7 cells.” 2014. Web. 17 Jun 2019.

Vancouver:

Hong D. Studies on the role of a novel protein, TMEM 56 in tumorigenic growth for MCF-7 cells. [Internet] [Masters thesis]. University of Southern California; 2014. [cited 2019 Jun 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/404283/rec/6174.

Council of Science Editors:

Hong D. Studies on the role of a novel protein, TMEM 56 in tumorigenic growth for MCF-7 cells. [Masters Thesis]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/404283/rec/6174


University of Southern California

6. Alef-Omidy, Honey. DNA methylation markers for blood-based detection of small cell lung cancer in mouse models.

Degree: MS, Biochemistry and Molecular Biology, 2014, University of Southern California

 Lung cancer is the top cancer killer in the United States. This cancer starts in the cells lining the bronchi or other parts of the… (more)

Subjects/Keywords: small cell lung cancer; DNA methylation; biomarkers; mouse models; MethyLight; sodium bisulfite

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APA (6th Edition):

Alef-Omidy, H. (2014). DNA methylation markers for blood-based detection of small cell lung cancer in mouse models. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/514619/rec/2069

Chicago Manual of Style (16th Edition):

Alef-Omidy, Honey. “DNA methylation markers for blood-based detection of small cell lung cancer in mouse models.” 2014. Masters Thesis, University of Southern California. Accessed June 17, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/514619/rec/2069.

MLA Handbook (7th Edition):

Alef-Omidy, Honey. “DNA methylation markers for blood-based detection of small cell lung cancer in mouse models.” 2014. Web. 17 Jun 2019.

Vancouver:

Alef-Omidy H. DNA methylation markers for blood-based detection of small cell lung cancer in mouse models. [Internet] [Masters thesis]. University of Southern California; 2014. [cited 2019 Jun 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/514619/rec/2069.

Council of Science Editors:

Alef-Omidy H. DNA methylation markers for blood-based detection of small cell lung cancer in mouse models. [Masters Thesis]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/514619/rec/2069


University of Southern California

7. Wu, Jia. Wnt/β-catenin/p300 induced transcription is critical for the differentiation and maintenance of Paneth cells.

Degree: MS, Biochemistry and Molecular Biology, 2014, University of Southern California

 Our lab's previous studies have demonstrated that Wnt/β‐catenin/p300 transcription initiates cell differentiation, whereas Wnt/β‐catenin/cyclic AMP‐responsive element binding protein‐binding protein (CBP) transcription mediates cell proliferation/maintenance of… (more)

Subjects/Keywords: Wnt; β ‐Catenin/p300; Paneth cell

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APA (6th Edition):

Wu, J. (2014). Wnt/β-catenin/p300 induced transcription is critical for the differentiation and maintenance of Paneth cells. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/405481/rec/7947

Chicago Manual of Style (16th Edition):

Wu, Jia. “Wnt/β-catenin/p300 induced transcription is critical for the differentiation and maintenance of Paneth cells.” 2014. Masters Thesis, University of Southern California. Accessed June 17, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/405481/rec/7947.

MLA Handbook (7th Edition):

Wu, Jia. “Wnt/β-catenin/p300 induced transcription is critical for the differentiation and maintenance of Paneth cells.” 2014. Web. 17 Jun 2019.

Vancouver:

Wu J. Wnt/β-catenin/p300 induced transcription is critical for the differentiation and maintenance of Paneth cells. [Internet] [Masters thesis]. University of Southern California; 2014. [cited 2019 Jun 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/405481/rec/7947.

Council of Science Editors:

Wu J. Wnt/β-catenin/p300 induced transcription is critical for the differentiation and maintenance of Paneth cells. [Masters Thesis]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/405481/rec/7947


University of Southern California

8. Jhaveri, Niyati. Noscapine: a study of its effects on the glioma vasculature.

Degree: MS, Biochemistry & Molecular Biology, 2009, University of Southern California

 Noscapine has been widely used as an oral antitussive agent. More recently, studies have shown that noscapine significantly affects microtubule dynamics and has potent antitumor… (more)

Subjects/Keywords: angiogenesis; endothelial cell migration; glioma; noscapine; TMZ-resistant; vasculature

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APA (6th Edition):

Jhaveri, N. (2009). Noscapine: a study of its effects on the glioma vasculature. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/269936/rec/4452

Chicago Manual of Style (16th Edition):

Jhaveri, Niyati. “Noscapine: a study of its effects on the glioma vasculature.” 2009. Masters Thesis, University of Southern California. Accessed June 17, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/269936/rec/4452.

MLA Handbook (7th Edition):

Jhaveri, Niyati. “Noscapine: a study of its effects on the glioma vasculature.” 2009. Web. 17 Jun 2019.

Vancouver:

Jhaveri N. Noscapine: a study of its effects on the glioma vasculature. [Internet] [Masters thesis]. University of Southern California; 2009. [cited 2019 Jun 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/269936/rec/4452.

Council of Science Editors:

Jhaveri N. Noscapine: a study of its effects on the glioma vasculature. [Masters Thesis]. University of Southern California; 2009. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/269936/rec/4452


University of Southern California

9. Vikman, Susanna. The contribution of arachidonate 5-lipoxygenase to inflammatory response in coronary artery disease.

Degree: MS, Biochemistry & Molecular Biology, 2010, University of Southern California

 This study provides a review of the literature on coronary artery disease (CAD) focusing on the role of the enzyme 5-lipoxygenase (encoded by ALOX5) and… (more)

Subjects/Keywords: 5-lipoxygenase; coronary artery disease; arachidonic acid metabolism; leukotriene; atherosclerosis

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APA (6th Edition):

Vikman, S. (2010). The contribution of arachidonate 5-lipoxygenase to inflammatory response in coronary artery disease. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/426799/rec/6521

Chicago Manual of Style (16th Edition):

Vikman, Susanna. “The contribution of arachidonate 5-lipoxygenase to inflammatory response in coronary artery disease.” 2010. Masters Thesis, University of Southern California. Accessed June 17, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/426799/rec/6521.

MLA Handbook (7th Edition):

Vikman, Susanna. “The contribution of arachidonate 5-lipoxygenase to inflammatory response in coronary artery disease.” 2010. Web. 17 Jun 2019.

Vancouver:

Vikman S. The contribution of arachidonate 5-lipoxygenase to inflammatory response in coronary artery disease. [Internet] [Masters thesis]. University of Southern California; 2010. [cited 2019 Jun 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/426799/rec/6521.

Council of Science Editors:

Vikman S. The contribution of arachidonate 5-lipoxygenase to inflammatory response in coronary artery disease. [Masters Thesis]. University of Southern California; 2010. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/426799/rec/6521


University of Southern California

10. Ramanathan, Anita. Defining function of an autism pathway in human neuronal cells.

Degree: MS, Biochemistry and Molecular Biology, 2011, University of Southern California

 Autism is a neurodevelopmental disorder that affects 1% of the population and causes deficits in social interaction, communication and behavioral flexibility. Autism is highly heritable,… (more)

Subjects/Keywords: autism; luminex; PI3K signaling; PI3K Akt pathway

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APA (6th Edition):

Ramanathan, A. (2011). Defining function of an autism pathway in human neuronal cells. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/627881/rec/1816

Chicago Manual of Style (16th Edition):

Ramanathan, Anita. “Defining function of an autism pathway in human neuronal cells.” 2011. Masters Thesis, University of Southern California. Accessed June 17, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/627881/rec/1816.

MLA Handbook (7th Edition):

Ramanathan, Anita. “Defining function of an autism pathway in human neuronal cells.” 2011. Web. 17 Jun 2019.

Vancouver:

Ramanathan A. Defining function of an autism pathway in human neuronal cells. [Internet] [Masters thesis]. University of Southern California; 2011. [cited 2019 Jun 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/627881/rec/1816.

Council of Science Editors:

Ramanathan A. Defining function of an autism pathway in human neuronal cells. [Masters Thesis]. University of Southern California; 2011. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/627881/rec/1816


University of Southern California

11. Zhang, Naibo. Targeting Ryk-signaling to attenuate multiple sclerosis by promoting oligodendrocyte differentiation and myelination.

Degree: MS, Biochemistry and Molecular Biology, 2015, University of Southern California

 Multiple Sclerosis (MS) is a CNS degenerative disease. It is one of the most common chronic disabling CNS disease in young adults. In MS, oligodendrocytes(OLs)… (more)

Subjects/Keywords: multiple sclerosis; oligodendrocytes; remyelination

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APA (6th Edition):

Zhang, N. (2015). Targeting Ryk-signaling to attenuate multiple sclerosis by promoting oligodendrocyte differentiation and myelination. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/615832/rec/6322

Chicago Manual of Style (16th Edition):

Zhang, Naibo. “Targeting Ryk-signaling to attenuate multiple sclerosis by promoting oligodendrocyte differentiation and myelination.” 2015. Masters Thesis, University of Southern California. Accessed June 17, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/615832/rec/6322.

MLA Handbook (7th Edition):

Zhang, Naibo. “Targeting Ryk-signaling to attenuate multiple sclerosis by promoting oligodendrocyte differentiation and myelination.” 2015. Web. 17 Jun 2019.

Vancouver:

Zhang N. Targeting Ryk-signaling to attenuate multiple sclerosis by promoting oligodendrocyte differentiation and myelination. [Internet] [Masters thesis]. University of Southern California; 2015. [cited 2019 Jun 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/615832/rec/6322.

Council of Science Editors:

Zhang N. Targeting Ryk-signaling to attenuate multiple sclerosis by promoting oligodendrocyte differentiation and myelination. [Masters Thesis]. University of Southern California; 2015. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/615832/rec/6322


University of Southern California

12. Li, Chen. MicroRNAs involved in the regulation of Endothelin-1 gene expression in endothelial cells.

Degree: MS, Biochemistry and Molecular Biology, 2014, University of Southern California

 We showed that in human dermal microvascular endothelial cell line (HMEC-1), the stability of ET-1 mRNA was increased in response to PlGF treatment. We also… (more)

Subjects/Keywords: endothelial cells; Endothelin-1; microRNA

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APA (6th Edition):

Li, C. (2014). MicroRNAs involved in the regulation of Endothelin-1 gene expression in endothelial cells. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/86172/rec/4069

Chicago Manual of Style (16th Edition):

Li, Chen. “MicroRNAs involved in the regulation of Endothelin-1 gene expression in endothelial cells.” 2014. Masters Thesis, University of Southern California. Accessed June 17, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/86172/rec/4069.

MLA Handbook (7th Edition):

Li, Chen. “MicroRNAs involved in the regulation of Endothelin-1 gene expression in endothelial cells.” 2014. Web. 17 Jun 2019.

Vancouver:

Li C. MicroRNAs involved in the regulation of Endothelin-1 gene expression in endothelial cells. [Internet] [Masters thesis]. University of Southern California; 2014. [cited 2019 Jun 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/86172/rec/4069.

Council of Science Editors:

Li C. MicroRNAs involved in the regulation of Endothelin-1 gene expression in endothelial cells. [Masters Thesis]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/86172/rec/4069


University of Southern California

13. Chen, Chien-Yu. Quantitative evaluation of the effectiveness of an integrin antagonist, vicrostatin, in cell migration.

Degree: MS, Biochemistry and Molecular Biology, 2013, University of Southern California

 Cell migration underlies tissue formation, maintenance and regeneration as well as pathological condition such as cancer invasion and metastasis. The integrin family of cell adhesion… (more)

Subjects/Keywords: cell migration; disintegrin; integrin; metastasis; vicrostatin

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APA (6th Edition):

Chen, C. (2013). Quantitative evaluation of the effectiveness of an integrin antagonist, vicrostatin, in cell migration. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/298355/rec/5349

Chicago Manual of Style (16th Edition):

Chen, Chien-Yu. “Quantitative evaluation of the effectiveness of an integrin antagonist, vicrostatin, in cell migration.” 2013. Masters Thesis, University of Southern California. Accessed June 17, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/298355/rec/5349.

MLA Handbook (7th Edition):

Chen, Chien-Yu. “Quantitative evaluation of the effectiveness of an integrin antagonist, vicrostatin, in cell migration.” 2013. Web. 17 Jun 2019.

Vancouver:

Chen C. Quantitative evaluation of the effectiveness of an integrin antagonist, vicrostatin, in cell migration. [Internet] [Masters thesis]. University of Southern California; 2013. [cited 2019 Jun 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/298355/rec/5349.

Council of Science Editors:

Chen C. Quantitative evaluation of the effectiveness of an integrin antagonist, vicrostatin, in cell migration. [Masters Thesis]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/298355/rec/5349


University of Southern California

14. Gururaj, Sushmitha. Ethanol induced modulation of microglial P2X7 receptor expression and its role in neuroinflammation.

Degree: MS, Biochemistry and Molecular Biology, 2012, University of Southern California

 The present work builds upon a central hypothesis that ATP-gated purinergic P2X7 receptors (P2X7Rs) play an important role in causing ethanol-induced neuroinflammation and neurodegeneration. A… (more)

Subjects/Keywords: ethanol; purinergic; P2X7 receptor; neuroinflammation; neurodenegeration

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APA (6th Edition):

Gururaj, S. (2012). Ethanol induced modulation of microglial P2X7 receptor expression and its role in neuroinflammation. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/43372/rec/2511

Chicago Manual of Style (16th Edition):

Gururaj, Sushmitha. “Ethanol induced modulation of microglial P2X7 receptor expression and its role in neuroinflammation.” 2012. Masters Thesis, University of Southern California. Accessed June 17, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/43372/rec/2511.

MLA Handbook (7th Edition):

Gururaj, Sushmitha. “Ethanol induced modulation of microglial P2X7 receptor expression and its role in neuroinflammation.” 2012. Web. 17 Jun 2019.

Vancouver:

Gururaj S. Ethanol induced modulation of microglial P2X7 receptor expression and its role in neuroinflammation. [Internet] [Masters thesis]. University of Southern California; 2012. [cited 2019 Jun 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/43372/rec/2511.

Council of Science Editors:

Gururaj S. Ethanol induced modulation of microglial P2X7 receptor expression and its role in neuroinflammation. [Masters Thesis]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/43372/rec/2511


University of Southern California

15. Sura, Asmiti Vivek. Characterization of the retromer complex of proteins in gastric parietal cells.

Degree: MS, Biochemistry and Molecular Biology, 2013, University of Southern California

 Gastric acid secretion involves the regulated recycling of the H, K-ATPase to and from the apical membrane of parietal cells. All of the steps in… (more)

Subjects/Keywords: apical membrane recycling; gastric microsomes; H K-ATPase; hydrogen-potassium-adenosinetriphosphatase; retromer; tubulovesicles

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APA (6th Edition):

Sura, A. V. (2013). Characterization of the retromer complex of proteins in gastric parietal cells. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/307627/rec/1310

Chicago Manual of Style (16th Edition):

Sura, Asmiti Vivek. “Characterization of the retromer complex of proteins in gastric parietal cells.” 2013. Masters Thesis, University of Southern California. Accessed June 17, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/307627/rec/1310.

MLA Handbook (7th Edition):

Sura, Asmiti Vivek. “Characterization of the retromer complex of proteins in gastric parietal cells.” 2013. Web. 17 Jun 2019.

Vancouver:

Sura AV. Characterization of the retromer complex of proteins in gastric parietal cells. [Internet] [Masters thesis]. University of Southern California; 2013. [cited 2019 Jun 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/307627/rec/1310.

Council of Science Editors:

Sura AV. Characterization of the retromer complex of proteins in gastric parietal cells. [Masters Thesis]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/307627/rec/1310


University of Southern California

16. Yu, Xinyang. Deletion of Krüppel-like factor 4 (KLF4) affects NPCs to embark on differentiative progression and leads to delayed neurogenesis in mouse embryonic cortex.

Degree: MS, Biochemistry and Molecular Biology, 2014, University of Southern California

 The krüppel‐like factors (KLFs) transcription factor family functions by interacting with other co‐regulators to modulate transactivation or transrepression during cellular development. KLF4 is a KLF… (more)

Subjects/Keywords: Krü; ppel-like factor 4; neurogenesis; neural precursor cell; embryonic cerebral cortex; asymmetric division

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APA (6th Edition):

Yu, X. (2014). Deletion of Krüppel-like factor 4 (KLF4) affects NPCs to embark on differentiative progression and leads to delayed neurogenesis in mouse embryonic cortex. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/404435/rec/1824

Chicago Manual of Style (16th Edition):

Yu, Xinyang. “Deletion of Krüppel-like factor 4 (KLF4) affects NPCs to embark on differentiative progression and leads to delayed neurogenesis in mouse embryonic cortex.” 2014. Masters Thesis, University of Southern California. Accessed June 17, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/404435/rec/1824.

MLA Handbook (7th Edition):

Yu, Xinyang. “Deletion of Krüppel-like factor 4 (KLF4) affects NPCs to embark on differentiative progression and leads to delayed neurogenesis in mouse embryonic cortex.” 2014. Web. 17 Jun 2019.

Vancouver:

Yu X. Deletion of Krüppel-like factor 4 (KLF4) affects NPCs to embark on differentiative progression and leads to delayed neurogenesis in mouse embryonic cortex. [Internet] [Masters thesis]. University of Southern California; 2014. [cited 2019 Jun 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/404435/rec/1824.

Council of Science Editors:

Yu X. Deletion of Krüppel-like factor 4 (KLF4) affects NPCs to embark on differentiative progression and leads to delayed neurogenesis in mouse embryonic cortex. [Masters Thesis]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/404435/rec/1824


University of Southern California

17. Ravichandran, Monisha. Alcohol mediated expression of cyto-protective enzyme - NQO-1 and its post translational regulation.

Degree: MS, Biochemistry and Molecular Biology, 2012, University of Southern California

 Chronic alcohol consumption leads to liver injury and death. Studies have shown ethanol causes increased inflammation, which leads to liver damage and cirrhosis. To counteract… (more)

Subjects/Keywords: NQO-1; ethanol; miR-566; miR-518; miR-642; Nrf-2; Bach-1; Keap-1

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APA (6th Edition):

Ravichandran, M. (2012). Alcohol mediated expression of cyto-protective enzyme - NQO-1 and its post translational regulation. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/84016/rec/598

Chicago Manual of Style (16th Edition):

Ravichandran, Monisha. “Alcohol mediated expression of cyto-protective enzyme - NQO-1 and its post translational regulation.” 2012. Masters Thesis, University of Southern California. Accessed June 17, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/84016/rec/598.

MLA Handbook (7th Edition):

Ravichandran, Monisha. “Alcohol mediated expression of cyto-protective enzyme - NQO-1 and its post translational regulation.” 2012. Web. 17 Jun 2019.

Vancouver:

Ravichandran M. Alcohol mediated expression of cyto-protective enzyme - NQO-1 and its post translational regulation. [Internet] [Masters thesis]. University of Southern California; 2012. [cited 2019 Jun 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/84016/rec/598.

Council of Science Editors:

Ravichandran M. Alcohol mediated expression of cyto-protective enzyme - NQO-1 and its post translational regulation. [Masters Thesis]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/84016/rec/598


University of Southern California

18. Chen, Po-Han. The kinetic study of engineered MBD domain interactions with methylated DNA: insight into binding of methylated DNA by MBD2b.

Degree: MS, Biochemistry & Molecular Biology, 2011, University of Southern California

 Mehtyl-CpG binding domain protein 2 and 3 (MBD2 and MBD3) contain methyl-CpG binding domains (MBD) and belong to a family of MBD proteins. Methyl-CpG binding… (more)

Subjects/Keywords: MBD protein; methyl-CpG

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APA (6th Edition):

Chen, P. (2011). The kinetic study of engineered MBD domain interactions with methylated DNA: insight into binding of methylated DNA by MBD2b. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/278087/rec/6927

Chicago Manual of Style (16th Edition):

Chen, Po-Han. “The kinetic study of engineered MBD domain interactions with methylated DNA: insight into binding of methylated DNA by MBD2b.” 2011. Masters Thesis, University of Southern California. Accessed June 17, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/278087/rec/6927.

MLA Handbook (7th Edition):

Chen, Po-Han. “The kinetic study of engineered MBD domain interactions with methylated DNA: insight into binding of methylated DNA by MBD2b.” 2011. Web. 17 Jun 2019.

Vancouver:

Chen P. The kinetic study of engineered MBD domain interactions with methylated DNA: insight into binding of methylated DNA by MBD2b. [Internet] [Masters thesis]. University of Southern California; 2011. [cited 2019 Jun 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/278087/rec/6927.

Council of Science Editors:

Chen P. The kinetic study of engineered MBD domain interactions with methylated DNA: insight into binding of methylated DNA by MBD2b. [Masters Thesis]. University of Southern California; 2011. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/278087/rec/6927


University of Southern California

19. Park, Eugene. The role of survivin in drug resistant pediatric acute lymphoblastic leukemia.

Degree: MS, Biochemistry & Molecular Biology, 2010, University of Southern California

 Despite the recent advances in chemotherapy for acute lymphoblastic leukemia (ALL), drug resistance resulting in relapse and long-term side effects of current treatments warrant new… (more)

Subjects/Keywords: survivin; BIRC5; acute lymphoblastic; leukemia; IAP

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APA (6th Edition):

Park, E. (2010). The role of survivin in drug resistant pediatric acute lymphoblastic leukemia. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/366748/rec/7257

Chicago Manual of Style (16th Edition):

Park, Eugene. “The role of survivin in drug resistant pediatric acute lymphoblastic leukemia.” 2010. Masters Thesis, University of Southern California. Accessed June 17, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/366748/rec/7257.

MLA Handbook (7th Edition):

Park, Eugene. “The role of survivin in drug resistant pediatric acute lymphoblastic leukemia.” 2010. Web. 17 Jun 2019.

Vancouver:

Park E. The role of survivin in drug resistant pediatric acute lymphoblastic leukemia. [Internet] [Masters thesis]. University of Southern California; 2010. [cited 2019 Jun 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/366748/rec/7257.

Council of Science Editors:

Park E. The role of survivin in drug resistant pediatric acute lymphoblastic leukemia. [Masters Thesis]. University of Southern California; 2010. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/366748/rec/7257


University of Southern California

20. Li, Yang. Nuclear respiratory factor-1 regulation by PTEN signaling.

Degree: MS, Biochemistry & Molecular Biology, 2009, University of Southern California

 Based on many previous reports regarding cancer metabolism, it is believed that tumor cells need to obtain metabolic changes to maintain the malignant phenotype. Additionally,… (more)

Subjects/Keywords: PTEN; NRF-1; mitochondria respiration; fatty liver disease

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APA (6th Edition):

Li, Y. (2009). Nuclear respiratory factor-1 regulation by PTEN signaling. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/174310/rec/4473

Chicago Manual of Style (16th Edition):

Li, Yang. “Nuclear respiratory factor-1 regulation by PTEN signaling.” 2009. Masters Thesis, University of Southern California. Accessed June 17, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/174310/rec/4473.

MLA Handbook (7th Edition):

Li, Yang. “Nuclear respiratory factor-1 regulation by PTEN signaling.” 2009. Web. 17 Jun 2019.

Vancouver:

Li Y. Nuclear respiratory factor-1 regulation by PTEN signaling. [Internet] [Masters thesis]. University of Southern California; 2009. [cited 2019 Jun 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/174310/rec/4473.

Council of Science Editors:

Li Y. Nuclear respiratory factor-1 regulation by PTEN signaling. [Masters Thesis]. University of Southern California; 2009. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/174310/rec/4473


University of Southern California

21. Alammari, Farah. Mixed lineage leukemia proteins (MLLs), their effect as coregulators on target gene expression and global histone methylation.

Degree: MS, Biochemistry and Molecular Biology, 2013, University of Southern California

 Mixed lineage leukemia proteins (MLLs) belong to the evolutionarily conserved trithorax family of human genes that play important roles in development and HOX gene regulation.… (more)

Subjects/Keywords: MLLs; coregulators; histone modification; epigenetics

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APA (6th Edition):

Alammari, F. (2013). Mixed lineage leukemia proteins (MLLs), their effect as coregulators on target gene expression and global histone methylation. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/348907/rec/4097

Chicago Manual of Style (16th Edition):

Alammari, Farah. “Mixed lineage leukemia proteins (MLLs), their effect as coregulators on target gene expression and global histone methylation.” 2013. Masters Thesis, University of Southern California. Accessed June 17, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/348907/rec/4097.

MLA Handbook (7th Edition):

Alammari, Farah. “Mixed lineage leukemia proteins (MLLs), their effect as coregulators on target gene expression and global histone methylation.” 2013. Web. 17 Jun 2019.

Vancouver:

Alammari F. Mixed lineage leukemia proteins (MLLs), their effect as coregulators on target gene expression and global histone methylation. [Internet] [Masters thesis]. University of Southern California; 2013. [cited 2019 Jun 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/348907/rec/4097.

Council of Science Editors:

Alammari F. Mixed lineage leukemia proteins (MLLs), their effect as coregulators on target gene expression and global histone methylation. [Masters Thesis]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/348907/rec/4097


University of Southern California

22. Halabi, Waiel. A synthetic lethal screen for NF-κB-dependent plasma cell disorders.

Degree: MS, Biochemistry and Molecular Biology, 2012, University of Southern California

 Nuclear factor- kappa B (NF-κB) was first described as a transcription factor in B cells that binds to the enhancer element controlling immunoglobulin kappa light… (more)

Subjects/Keywords: hematology; plasma cell disorders

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APA (6th Edition):

Halabi, W. (2012). A synthetic lethal screen for NF-κB-dependent plasma cell disorders. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/120349/rec/424

Chicago Manual of Style (16th Edition):

Halabi, Waiel. “A synthetic lethal screen for NF-κB-dependent plasma cell disorders.” 2012. Masters Thesis, University of Southern California. Accessed June 17, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/120349/rec/424.

MLA Handbook (7th Edition):

Halabi, Waiel. “A synthetic lethal screen for NF-κB-dependent plasma cell disorders.” 2012. Web. 17 Jun 2019.

Vancouver:

Halabi W. A synthetic lethal screen for NF-κB-dependent plasma cell disorders. [Internet] [Masters thesis]. University of Southern California; 2012. [cited 2019 Jun 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/120349/rec/424.

Council of Science Editors:

Halabi W. A synthetic lethal screen for NF-κB-dependent plasma cell disorders. [Masters Thesis]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/120349/rec/424


University of Southern California

23. Bedi, Yudhishtar Singh. Characterization of mouse models of peroxisome biogenesis disorders: a study of dental enamel phenotypes.

Degree: MS, Biochemistry and Molecular Biology, 2013, University of Southern California

 This thesis describes a collaborative investigatory effort between the labs of Dr. Joseph Hacia (USC), Dr. Michael Paine (USC), Dr. Steven Steinberg (Kennedy Krieger Institute,… (more)

Subjects/Keywords: peroxisome biogenesis disorders; enamel defects; mouse model; Pex1; Pex7

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APA (6th Edition):

Bedi, Y. S. (2013). Characterization of mouse models of peroxisome biogenesis disorders: a study of dental enamel phenotypes. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/337461/rec/1304

Chicago Manual of Style (16th Edition):

Bedi, Yudhishtar Singh. “Characterization of mouse models of peroxisome biogenesis disorders: a study of dental enamel phenotypes.” 2013. Masters Thesis, University of Southern California. Accessed June 17, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/337461/rec/1304.

MLA Handbook (7th Edition):

Bedi, Yudhishtar Singh. “Characterization of mouse models of peroxisome biogenesis disorders: a study of dental enamel phenotypes.” 2013. Web. 17 Jun 2019.

Vancouver:

Bedi YS. Characterization of mouse models of peroxisome biogenesis disorders: a study of dental enamel phenotypes. [Internet] [Masters thesis]. University of Southern California; 2013. [cited 2019 Jun 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/337461/rec/1304.

Council of Science Editors:

Bedi YS. Characterization of mouse models of peroxisome biogenesis disorders: a study of dental enamel phenotypes. [Masters Thesis]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/337461/rec/1304


University of Southern California

24. Park, Leena S. Structural characterization of pseudorepeat shuffled alpha-synuclein.

Degree: MS, Biochemistry & Molecular Biology, 2010, University of Southern California

 The 140-residue alpha-synuclein plays a central role in the molecular chain of events leading to Parkinson' s disease. Alpha-synuclein is dynamically unstructured in aqueous solution,… (more)

Subjects/Keywords: alpha-synuclein

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APA (6th Edition):

Park, L. S. (2010). Structural characterization of pseudorepeat shuffled alpha-synuclein. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/80230/rec/6110

Chicago Manual of Style (16th Edition):

Park, Leena S. “Structural characterization of pseudorepeat shuffled alpha-synuclein.” 2010. Masters Thesis, University of Southern California. Accessed June 17, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/80230/rec/6110.

MLA Handbook (7th Edition):

Park, Leena S. “Structural characterization of pseudorepeat shuffled alpha-synuclein.” 2010. Web. 17 Jun 2019.

Vancouver:

Park LS. Structural characterization of pseudorepeat shuffled alpha-synuclein. [Internet] [Masters thesis]. University of Southern California; 2010. [cited 2019 Jun 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/80230/rec/6110.

Council of Science Editors:

Park LS. Structural characterization of pseudorepeat shuffled alpha-synuclein. [Masters Thesis]. University of Southern California; 2010. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/80230/rec/6110


University of Southern California

25. Sriram, Kiran. Interaction of Hic-5 with different steroid receptors and its selective coregulator activity.

Degree: MS, Biochemistry and Molecular Biology, 2015, University of Southern California

 Hic-5 (Hydrogen Peroxide Inducible clone-5) is a paxillin family protein that acts as a transcription coregulator for various steroid receptors such as Androgen Receptor (AR),… (more)

Subjects/Keywords: steroid receptors; coregulator; transcription

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APA (6th Edition):

Sriram, K. (2015). Interaction of Hic-5 with different steroid receptors and its selective coregulator activity. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/587097/rec/3567

Chicago Manual of Style (16th Edition):

Sriram, Kiran. “Interaction of Hic-5 with different steroid receptors and its selective coregulator activity.” 2015. Masters Thesis, University of Southern California. Accessed June 17, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/587097/rec/3567.

MLA Handbook (7th Edition):

Sriram, Kiran. “Interaction of Hic-5 with different steroid receptors and its selective coregulator activity.” 2015. Web. 17 Jun 2019.

Vancouver:

Sriram K. Interaction of Hic-5 with different steroid receptors and its selective coregulator activity. [Internet] [Masters thesis]. University of Southern California; 2015. [cited 2019 Jun 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/587097/rec/3567.

Council of Science Editors:

Sriram K. Interaction of Hic-5 with different steroid receptors and its selective coregulator activity. [Masters Thesis]. University of Southern California; 2015. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/587097/rec/3567


University of Southern California

26. Pattamatta, Preeti. Discovery of novel small-molecule compounds with anticancer properties.

Degree: MS, Biochemistry & Molecular Biology, 2010, University of Southern California

 Cancer has emerged as one of the deadliest diseases in the recent past. According to the current statistics, one in every four deaths in the… (more)

Subjects/Keywords: anticancer; small molecules; drug discovery

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APA (6th Edition):

Pattamatta, P. (2010). Discovery of novel small-molecule compounds with anticancer properties. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/146657/rec/2028

Chicago Manual of Style (16th Edition):

Pattamatta, Preeti. “Discovery of novel small-molecule compounds with anticancer properties.” 2010. Masters Thesis, University of Southern California. Accessed June 17, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/146657/rec/2028.

MLA Handbook (7th Edition):

Pattamatta, Preeti. “Discovery of novel small-molecule compounds with anticancer properties.” 2010. Web. 17 Jun 2019.

Vancouver:

Pattamatta P. Discovery of novel small-molecule compounds with anticancer properties. [Internet] [Masters thesis]. University of Southern California; 2010. [cited 2019 Jun 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/146657/rec/2028.

Council of Science Editors:

Pattamatta P. Discovery of novel small-molecule compounds with anticancer properties. [Masters Thesis]. University of Southern California; 2010. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/146657/rec/2028


University of Southern California

27. Jia, Qi. Neuroendocrine regulation of the transcription factor SKN-1/Nrf2 in oxidative stress response.

Degree: MS, Biochemistry and Molecular Biology, 2015, University of Southern California

 The Nrf2 transcription factor plays a critical role in mediating adaptive responses to cellular stress and defending against neurodegeneration, aging, and cancer. Nrf2 activity is… (more)

Subjects/Keywords: C. elegans; oxidative stress; neuropeptide; SKN-1; FLP-1

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APA (6th Edition):

Jia, Q. (2015). Neuroendocrine regulation of the transcription factor SKN-1/Nrf2 in oxidative stress response. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/589898/rec/4370

Chicago Manual of Style (16th Edition):

Jia, Qi. “Neuroendocrine regulation of the transcription factor SKN-1/Nrf2 in oxidative stress response.” 2015. Masters Thesis, University of Southern California. Accessed June 17, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/589898/rec/4370.

MLA Handbook (7th Edition):

Jia, Qi. “Neuroendocrine regulation of the transcription factor SKN-1/Nrf2 in oxidative stress response.” 2015. Web. 17 Jun 2019.

Vancouver:

Jia Q. Neuroendocrine regulation of the transcription factor SKN-1/Nrf2 in oxidative stress response. [Internet] [Masters thesis]. University of Southern California; 2015. [cited 2019 Jun 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/589898/rec/4370.

Council of Science Editors:

Jia Q. Neuroendocrine regulation of the transcription factor SKN-1/Nrf2 in oxidative stress response. [Masters Thesis]. University of Southern California; 2015. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/589898/rec/4370


University of Southern California

28. Tseng, Chun-Chih. Connexins in feather morphogenesis.

Degree: MS, Biochemistry & Molecular Biology, 2013, University of Southern California

 Gap junctions are formed by the direct docking of two hexamers of connexins (Cxs) which allow the exchange of cellular contents, such as ions, second… (more)

Subjects/Keywords: connexin; feather; gap junction; GJIC; morphogenesis

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APA (6th Edition):

Tseng, C. (2013). Connexins in feather morphogenesis. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/416056/rec/1590

Chicago Manual of Style (16th Edition):

Tseng, Chun-Chih. “Connexins in feather morphogenesis.” 2013. Masters Thesis, University of Southern California. Accessed June 17, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/416056/rec/1590.

MLA Handbook (7th Edition):

Tseng, Chun-Chih. “Connexins in feather morphogenesis.” 2013. Web. 17 Jun 2019.

Vancouver:

Tseng C. Connexins in feather morphogenesis. [Internet] [Masters thesis]. University of Southern California; 2013. [cited 2019 Jun 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/416056/rec/1590.

Council of Science Editors:

Tseng C. Connexins in feather morphogenesis. [Masters Thesis]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/416056/rec/1590


University of Southern California

29. Lee, Jo. Differential effect of ethanol and r-sulforaphane on regulation of heme oxygenase-1 in endothelial cells.

Degree: MS, Biochemistry and Molecular Biology, 2014, University of Southern California

 Alcoholism is one of the leading causes of liver disease in the United States. The liver metabolizes 80% of ethanol consumed. Ethanol and its metabolite,… (more)

Subjects/Keywords: r-sulforaphane; heme oxygenase-1

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lee, J. (2014). Differential effect of ethanol and r-sulforaphane on regulation of heme oxygenase-1 in endothelial cells. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/121882/rec/1983

Chicago Manual of Style (16th Edition):

Lee, Jo. “Differential effect of ethanol and r-sulforaphane on regulation of heme oxygenase-1 in endothelial cells.” 2014. Masters Thesis, University of Southern California. Accessed June 17, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/121882/rec/1983.

MLA Handbook (7th Edition):

Lee, Jo. “Differential effect of ethanol and r-sulforaphane on regulation of heme oxygenase-1 in endothelial cells.” 2014. Web. 17 Jun 2019.

Vancouver:

Lee J. Differential effect of ethanol and r-sulforaphane on regulation of heme oxygenase-1 in endothelial cells. [Internet] [Masters thesis]. University of Southern California; 2014. [cited 2019 Jun 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/121882/rec/1983.

Council of Science Editors:

Lee J. Differential effect of ethanol and r-sulforaphane on regulation of heme oxygenase-1 in endothelial cells. [Masters Thesis]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/121882/rec/1983


University of Southern California

30. Reilly-Rhoten, Heather Noelle. Identification and characterization of post-translational modifications on histones: an on-line top-down mass spectrometry workflow for analysis using ProSight PTM.

Degree: MS, Biochemistry and Molecular Biology, 2012, University of Southern California

 The importance of histones for DNA management cannot be understated. From rearrangement to regulation of expression, histones act as the principle effectors of DNA availability… (more)

Subjects/Keywords: mass spectrometry; histones; post-translational modifications

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Reilly-Rhoten, H. N. (2012). Identification and characterization of post-translational modifications on histones: an on-line top-down mass spectrometry workflow for analysis using ProSight PTM. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/8330/rec/3309

Chicago Manual of Style (16th Edition):

Reilly-Rhoten, Heather Noelle. “Identification and characterization of post-translational modifications on histones: an on-line top-down mass spectrometry workflow for analysis using ProSight PTM.” 2012. Masters Thesis, University of Southern California. Accessed June 17, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/8330/rec/3309.

MLA Handbook (7th Edition):

Reilly-Rhoten, Heather Noelle. “Identification and characterization of post-translational modifications on histones: an on-line top-down mass spectrometry workflow for analysis using ProSight PTM.” 2012. Web. 17 Jun 2019.

Vancouver:

Reilly-Rhoten HN. Identification and characterization of post-translational modifications on histones: an on-line top-down mass spectrometry workflow for analysis using ProSight PTM. [Internet] [Masters thesis]. University of Southern California; 2012. [cited 2019 Jun 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/8330/rec/3309.

Council of Science Editors:

Reilly-Rhoten HN. Identification and characterization of post-translational modifications on histones: an on-line top-down mass spectrometry workflow for analysis using ProSight PTM. [Masters Thesis]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/8330/rec/3309

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