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You searched for +publisher:"University of Southern California" +contributor:("Tokes, Zoltan A."). Showing records 1 – 30 of 93 total matches.

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University of Southern California

1. Madhav, Anisha. The noncanonical role of telomerase in prostate cancer cells: exploring a non-telomeric signaling role for telomerase protein (TERT) in a cancer cell line.

Degree: MS, Biochemistry and Molecular Biology, 2012, University of Southern California

 The telomerase reverse transcriptase (TERT) component of telomerase has reverse transcriptase activity capable of elongating telomeres during each replication cycle of the cell. TERT plays… (more)

Subjects/Keywords: telomerase; TERT; prostate cancer; cancer stem cells

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APA (6th Edition):

Madhav, A. (2012). The noncanonical role of telomerase in prostate cancer cells: exploring a non-telomeric signaling role for telomerase protein (TERT) in a cancer cell line. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/30249/rec/7034

Chicago Manual of Style (16th Edition):

Madhav, Anisha. “The noncanonical role of telomerase in prostate cancer cells: exploring a non-telomeric signaling role for telomerase protein (TERT) in a cancer cell line.” 2012. Masters Thesis, University of Southern California. Accessed October 25, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/30249/rec/7034.

MLA Handbook (7th Edition):

Madhav, Anisha. “The noncanonical role of telomerase in prostate cancer cells: exploring a non-telomeric signaling role for telomerase protein (TERT) in a cancer cell line.” 2012. Web. 25 Oct 2020.

Vancouver:

Madhav A. The noncanonical role of telomerase in prostate cancer cells: exploring a non-telomeric signaling role for telomerase protein (TERT) in a cancer cell line. [Internet] [Masters thesis]. University of Southern California; 2012. [cited 2020 Oct 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/30249/rec/7034.

Council of Science Editors:

Madhav A. The noncanonical role of telomerase in prostate cancer cells: exploring a non-telomeric signaling role for telomerase protein (TERT) in a cancer cell line. [Masters Thesis]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/30249/rec/7034


University of Southern California

2. Zhang, Mengrao. Characteristics of hydrogen peroxide inducible clone-5 and its potential role as a nuclear receptor coactivator.

Degree: MS, Biochemistry and Molecular Biology, 2013, University of Southern California

 Hydrogen peroxide inducible clone-5 (Hic-5), also named transforming growth factor-β1 induced transcript 1(TGFB1 1), TGF-β stimulated clone-5 (TSC-5) or androgen receptor associated protein 55 (ARA55),… (more)

Subjects/Keywords: hydrogen peroxide inducible clone-5; nuclear receptor coactivator

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APA (6th Edition):

Zhang, M. (2013). Characteristics of hydrogen peroxide inducible clone-5 and its potential role as a nuclear receptor coactivator. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/336409/rec/1293

Chicago Manual of Style (16th Edition):

Zhang, Mengrao. “Characteristics of hydrogen peroxide inducible clone-5 and its potential role as a nuclear receptor coactivator.” 2013. Masters Thesis, University of Southern California. Accessed October 25, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/336409/rec/1293.

MLA Handbook (7th Edition):

Zhang, Mengrao. “Characteristics of hydrogen peroxide inducible clone-5 and its potential role as a nuclear receptor coactivator.” 2013. Web. 25 Oct 2020.

Vancouver:

Zhang M. Characteristics of hydrogen peroxide inducible clone-5 and its potential role as a nuclear receptor coactivator. [Internet] [Masters thesis]. University of Southern California; 2013. [cited 2020 Oct 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/336409/rec/1293.

Council of Science Editors:

Zhang M. Characteristics of hydrogen peroxide inducible clone-5 and its potential role as a nuclear receptor coactivator. [Masters Thesis]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/336409/rec/1293


University of Southern California

3. Alef-Omidy, Honey. DNA methylation markers for blood-based detection of small cell lung cancer in mouse models.

Degree: MS, Biochemistry and Molecular Biology, 2014, University of Southern California

 Lung cancer is the top cancer killer in the United States. This cancer starts in the cells lining the bronchi or other parts of the… (more)

Subjects/Keywords: small cell lung cancer; DNA methylation; biomarkers; mouse models; MethyLight; sodium bisulfite

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APA (6th Edition):

Alef-Omidy, H. (2014). DNA methylation markers for blood-based detection of small cell lung cancer in mouse models. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/514619/rec/2072

Chicago Manual of Style (16th Edition):

Alef-Omidy, Honey. “DNA methylation markers for blood-based detection of small cell lung cancer in mouse models.” 2014. Masters Thesis, University of Southern California. Accessed October 25, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/514619/rec/2072.

MLA Handbook (7th Edition):

Alef-Omidy, Honey. “DNA methylation markers for blood-based detection of small cell lung cancer in mouse models.” 2014. Web. 25 Oct 2020.

Vancouver:

Alef-Omidy H. DNA methylation markers for blood-based detection of small cell lung cancer in mouse models. [Internet] [Masters thesis]. University of Southern California; 2014. [cited 2020 Oct 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/514619/rec/2072.

Council of Science Editors:

Alef-Omidy H. DNA methylation markers for blood-based detection of small cell lung cancer in mouse models. [Masters Thesis]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/514619/rec/2072


University of Southern California

4. Chung, Paul J. Neurogenic placodes provide migratory enteric sensory neural progenitors in response to endothelin signaling pathway.

Degree: MS, Biochemistry and Molecular Biology, 2013, University of Southern California

 The enteric nervous system (ENS) is the neuronal regulatory network, which maintains homeostasis and peristalsis of the gastrointestinal (GI) tract. Abnormal development of ENS may… (more)

Subjects/Keywords: neurogenic placode; endothelin signaling

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APA (6th Edition):

Chung, P. J. (2013). Neurogenic placodes provide migratory enteric sensory neural progenitors in response to endothelin signaling pathway. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/310482/rec/4380

Chicago Manual of Style (16th Edition):

Chung, Paul J. “Neurogenic placodes provide migratory enteric sensory neural progenitors in response to endothelin signaling pathway.” 2013. Masters Thesis, University of Southern California. Accessed October 25, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/310482/rec/4380.

MLA Handbook (7th Edition):

Chung, Paul J. “Neurogenic placodes provide migratory enteric sensory neural progenitors in response to endothelin signaling pathway.” 2013. Web. 25 Oct 2020.

Vancouver:

Chung PJ. Neurogenic placodes provide migratory enteric sensory neural progenitors in response to endothelin signaling pathway. [Internet] [Masters thesis]. University of Southern California; 2013. [cited 2020 Oct 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/310482/rec/4380.

Council of Science Editors:

Chung PJ. Neurogenic placodes provide migratory enteric sensory neural progenitors in response to endothelin signaling pathway. [Masters Thesis]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/310482/rec/4380


University of Southern California

5. Doshi, Nemi. Development of computational tools to assist high content screening to identify drug therapies for peroxisome biogenesis disorders.

Degree: MS, Biochemistry and Molecular Biology, 2014, University of Southern California

 Peroxisome biogenesis disorders are genetic disorders due to mutations in one or more of the PEX genes, resulting in impaired transport of peroxisomal proteins or… (more)

Subjects/Keywords: peroxisome biogenesis disorders; PEX1; drug screen; SIGMA LOPAC

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APA (6th Edition):

Doshi, N. (2014). Development of computational tools to assist high content screening to identify drug therapies for peroxisome biogenesis disorders. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/404342/rec/1934

Chicago Manual of Style (16th Edition):

Doshi, Nemi. “Development of computational tools to assist high content screening to identify drug therapies for peroxisome biogenesis disorders.” 2014. Masters Thesis, University of Southern California. Accessed October 25, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/404342/rec/1934.

MLA Handbook (7th Edition):

Doshi, Nemi. “Development of computational tools to assist high content screening to identify drug therapies for peroxisome biogenesis disorders.” 2014. Web. 25 Oct 2020.

Vancouver:

Doshi N. Development of computational tools to assist high content screening to identify drug therapies for peroxisome biogenesis disorders. [Internet] [Masters thesis]. University of Southern California; 2014. [cited 2020 Oct 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/404342/rec/1934.

Council of Science Editors:

Doshi N. Development of computational tools to assist high content screening to identify drug therapies for peroxisome biogenesis disorders. [Masters Thesis]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/404342/rec/1934


University of Southern California

6. Zhang, Naibo. Targeting Ryk-signaling to attenuate multiple sclerosis by promoting oligodendrocyte differentiation and myelination.

Degree: MS, Biochemistry and Molecular Biology, 2015, University of Southern California

 Multiple Sclerosis (MS) is a CNS degenerative disease. It is one of the most common chronic disabling CNS disease in young adults. In MS, oligodendrocytes(OLs)… (more)

Subjects/Keywords: multiple sclerosis; oligodendrocytes; remyelination

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APA (6th Edition):

Zhang, N. (2015). Targeting Ryk-signaling to attenuate multiple sclerosis by promoting oligodendrocyte differentiation and myelination. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/615832/rec/6331

Chicago Manual of Style (16th Edition):

Zhang, Naibo. “Targeting Ryk-signaling to attenuate multiple sclerosis by promoting oligodendrocyte differentiation and myelination.” 2015. Masters Thesis, University of Southern California. Accessed October 25, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/615832/rec/6331.

MLA Handbook (7th Edition):

Zhang, Naibo. “Targeting Ryk-signaling to attenuate multiple sclerosis by promoting oligodendrocyte differentiation and myelination.” 2015. Web. 25 Oct 2020.

Vancouver:

Zhang N. Targeting Ryk-signaling to attenuate multiple sclerosis by promoting oligodendrocyte differentiation and myelination. [Internet] [Masters thesis]. University of Southern California; 2015. [cited 2020 Oct 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/615832/rec/6331.

Council of Science Editors:

Zhang N. Targeting Ryk-signaling to attenuate multiple sclerosis by promoting oligodendrocyte differentiation and myelination. [Masters Thesis]. University of Southern California; 2015. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/615832/rec/6331


University of Southern California

7. Chang, Yin-Wei. Creating a multiple micrornia expression vector to target GRP78, an ER chaperone and signaling regulator in cancer.

Degree: MS, Biochemistry and Molecular Biology, 2012, University of Southern California

 GRP78, a major endoplasmic reticulum (ER) chaperone and signaling regulator, is commonly over-expressed in human cancer and contributes to cancer cell proliferation, survival and tumorigenesis.… (more)

Subjects/Keywords: GRP78; microRNA

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APA (6th Edition):

Chang, Y. (2012). Creating a multiple micrornia expression vector to target GRP78, an ER chaperone and signaling regulator in cancer. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/83898/rec/1687

Chicago Manual of Style (16th Edition):

Chang, Yin-Wei. “Creating a multiple micrornia expression vector to target GRP78, an ER chaperone and signaling regulator in cancer.” 2012. Masters Thesis, University of Southern California. Accessed October 25, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/83898/rec/1687.

MLA Handbook (7th Edition):

Chang, Yin-Wei. “Creating a multiple micrornia expression vector to target GRP78, an ER chaperone and signaling regulator in cancer.” 2012. Web. 25 Oct 2020.

Vancouver:

Chang Y. Creating a multiple micrornia expression vector to target GRP78, an ER chaperone and signaling regulator in cancer. [Internet] [Masters thesis]. University of Southern California; 2012. [cited 2020 Oct 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/83898/rec/1687.

Council of Science Editors:

Chang Y. Creating a multiple micrornia expression vector to target GRP78, an ER chaperone and signaling regulator in cancer. [Masters Thesis]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/83898/rec/1687


University of Southern California

8. Kelsom, Corey Sayuri Kauai. Asymmetric cell division during neurogenesis, and the mechanisms behind GABAergic cortical interneuron development and specification.

Degree: MS, Biochemistry and Molecular Biology, 2012, University of Southern California

 On the most basic level, an asymmetric division is a developmental process that produces two daughter cells, each possessing a different identity or fate. Progenitor… (more)

Subjects/Keywords: neuroblast; asymmetric; GABA; cortical; interneuron

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APA (6th Edition):

Kelsom, C. S. K. (2012). Asymmetric cell division during neurogenesis, and the mechanisms behind GABAergic cortical interneuron development and specification. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/116106/rec/975

Chicago Manual of Style (16th Edition):

Kelsom, Corey Sayuri Kauai. “Asymmetric cell division during neurogenesis, and the mechanisms behind GABAergic cortical interneuron development and specification.” 2012. Masters Thesis, University of Southern California. Accessed October 25, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/116106/rec/975.

MLA Handbook (7th Edition):

Kelsom, Corey Sayuri Kauai. “Asymmetric cell division during neurogenesis, and the mechanisms behind GABAergic cortical interneuron development and specification.” 2012. Web. 25 Oct 2020.

Vancouver:

Kelsom CSK. Asymmetric cell division during neurogenesis, and the mechanisms behind GABAergic cortical interneuron development and specification. [Internet] [Masters thesis]. University of Southern California; 2012. [cited 2020 Oct 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/116106/rec/975.

Council of Science Editors:

Kelsom CSK. Asymmetric cell division during neurogenesis, and the mechanisms behind GABAergic cortical interneuron development and specification. [Masters Thesis]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/116106/rec/975


University of Southern California

9. Si, Yuchen. Determinination of the causal potential of histone modifications on transcription and chromatin structure.

Degree: MS, Biochemistry and Molecular Biology, 2012, University of Southern California

 Histone modification is a major epigenetic regulatory mechanism that controls chromatin structure and gene expression potential. However, the causal potential of individual histone modifications remains… (more)

Subjects/Keywords: histone modification; G9a; Suv39h1; Suv39h2; Pr-set7; transcription; chromatin structure

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APA (6th Edition):

Si, Y. (2012). Determinination of the causal potential of histone modifications on transcription and chromatin structure. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/62768/rec/1894

Chicago Manual of Style (16th Edition):

Si, Yuchen. “Determinination of the causal potential of histone modifications on transcription and chromatin structure.” 2012. Masters Thesis, University of Southern California. Accessed October 25, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/62768/rec/1894.

MLA Handbook (7th Edition):

Si, Yuchen. “Determinination of the causal potential of histone modifications on transcription and chromatin structure.” 2012. Web. 25 Oct 2020.

Vancouver:

Si Y. Determinination of the causal potential of histone modifications on transcription and chromatin structure. [Internet] [Masters thesis]. University of Southern California; 2012. [cited 2020 Oct 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/62768/rec/1894.

Council of Science Editors:

Si Y. Determinination of the causal potential of histone modifications on transcription and chromatin structure. [Masters Thesis]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/62768/rec/1894


University of Southern California

10. Reilly-Rhoten, Heather Noelle. Identification and characterization of post-translational modifications on histones: an on-line top-down mass spectrometry workflow for analysis using ProSight PTM.

Degree: MS, Biochemistry and Molecular Biology, 2012, University of Southern California

 The importance of histones for DNA management cannot be understated. From rearrangement to regulation of expression, histones act as the principle effectors of DNA availability… (more)

Subjects/Keywords: mass spectrometry; histones; post-translational modifications

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APA (6th Edition):

Reilly-Rhoten, H. N. (2012). Identification and characterization of post-translational modifications on histones: an on-line top-down mass spectrometry workflow for analysis using ProSight PTM. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/8330/rec/3314

Chicago Manual of Style (16th Edition):

Reilly-Rhoten, Heather Noelle. “Identification and characterization of post-translational modifications on histones: an on-line top-down mass spectrometry workflow for analysis using ProSight PTM.” 2012. Masters Thesis, University of Southern California. Accessed October 25, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/8330/rec/3314.

MLA Handbook (7th Edition):

Reilly-Rhoten, Heather Noelle. “Identification and characterization of post-translational modifications on histones: an on-line top-down mass spectrometry workflow for analysis using ProSight PTM.” 2012. Web. 25 Oct 2020.

Vancouver:

Reilly-Rhoten HN. Identification and characterization of post-translational modifications on histones: an on-line top-down mass spectrometry workflow for analysis using ProSight PTM. [Internet] [Masters thesis]. University of Southern California; 2012. [cited 2020 Oct 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/8330/rec/3314.

Council of Science Editors:

Reilly-Rhoten HN. Identification and characterization of post-translational modifications on histones: an on-line top-down mass spectrometry workflow for analysis using ProSight PTM. [Masters Thesis]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/8330/rec/3314


University of Southern California

11. Davis, Gabrielle B. Topical adipose-derived stem cell therapy ameliorates radiation-induced delayed wound healing.

Degree: MS, Biochemistry and Molecular Biology, 2013, University of Southern California

 Recent literature supports the notion that Adipose Derived Stem Cells (ADSCs) in lipoaspirated fat could significantly improve radiation-induced wounds. However the mechanism of action is… (more)

Subjects/Keywords: adipose-derived stem cells; radiation skin injury

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APA (6th Edition):

Davis, G. B. (2013). Topical adipose-derived stem cell therapy ameliorates radiation-induced delayed wound healing. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/304435/rec/7510

Chicago Manual of Style (16th Edition):

Davis, Gabrielle B. “Topical adipose-derived stem cell therapy ameliorates radiation-induced delayed wound healing.” 2013. Masters Thesis, University of Southern California. Accessed October 25, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/304435/rec/7510.

MLA Handbook (7th Edition):

Davis, Gabrielle B. “Topical adipose-derived stem cell therapy ameliorates radiation-induced delayed wound healing.” 2013. Web. 25 Oct 2020.

Vancouver:

Davis GB. Topical adipose-derived stem cell therapy ameliorates radiation-induced delayed wound healing. [Internet] [Masters thesis]. University of Southern California; 2013. [cited 2020 Oct 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/304435/rec/7510.

Council of Science Editors:

Davis GB. Topical adipose-derived stem cell therapy ameliorates radiation-induced delayed wound healing. [Masters Thesis]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/304435/rec/7510


University of Southern California

12. Yu, Xinyang. Deletion of Krüppel-like factor 4 (KLF4) affects NPCs to embark on differentiative progression and leads to delayed neurogenesis in mouse embryonic cortex.

Degree: MS, Biochemistry and Molecular Biology, 2014, University of Southern California

 The krüppel‐like factors (KLFs) transcription factor family functions by interacting with other co‐regulators to modulate transactivation or transrepression during cellular development. KLF4 is a KLF… (more)

Subjects/Keywords: Krü; ppel-like factor 4; neurogenesis; neural precursor cell; embryonic cerebral cortex; asymmetric division

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APA (6th Edition):

Yu, X. (2014). Deletion of Krüppel-like factor 4 (KLF4) affects NPCs to embark on differentiative progression and leads to delayed neurogenesis in mouse embryonic cortex. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/404435/rec/1827

Chicago Manual of Style (16th Edition):

Yu, Xinyang. “Deletion of Krüppel-like factor 4 (KLF4) affects NPCs to embark on differentiative progression and leads to delayed neurogenesis in mouse embryonic cortex.” 2014. Masters Thesis, University of Southern California. Accessed October 25, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/404435/rec/1827.

MLA Handbook (7th Edition):

Yu, Xinyang. “Deletion of Krüppel-like factor 4 (KLF4) affects NPCs to embark on differentiative progression and leads to delayed neurogenesis in mouse embryonic cortex.” 2014. Web. 25 Oct 2020.

Vancouver:

Yu X. Deletion of Krüppel-like factor 4 (KLF4) affects NPCs to embark on differentiative progression and leads to delayed neurogenesis in mouse embryonic cortex. [Internet] [Masters thesis]. University of Southern California; 2014. [cited 2020 Oct 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/404435/rec/1827.

Council of Science Editors:

Yu X. Deletion of Krüppel-like factor 4 (KLF4) affects NPCs to embark on differentiative progression and leads to delayed neurogenesis in mouse embryonic cortex. [Masters Thesis]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/404435/rec/1827


University of Southern California

13. Li, Chen. MicroRNAs involved in the regulation of Endothelin-1 gene expression in endothelial cells.

Degree: MS, Biochemistry and Molecular Biology, 2014, University of Southern California

 We showed that in human dermal microvascular endothelial cell line (HMEC-1), the stability of ET-1 mRNA was increased in response to PlGF treatment. We also… (more)

Subjects/Keywords: endothelial cells; Endothelin-1; microRNA

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APA (6th Edition):

Li, C. (2014). MicroRNAs involved in the regulation of Endothelin-1 gene expression in endothelial cells. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/86172/rec/4076

Chicago Manual of Style (16th Edition):

Li, Chen. “MicroRNAs involved in the regulation of Endothelin-1 gene expression in endothelial cells.” 2014. Masters Thesis, University of Southern California. Accessed October 25, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/86172/rec/4076.

MLA Handbook (7th Edition):

Li, Chen. “MicroRNAs involved in the regulation of Endothelin-1 gene expression in endothelial cells.” 2014. Web. 25 Oct 2020.

Vancouver:

Li C. MicroRNAs involved in the regulation of Endothelin-1 gene expression in endothelial cells. [Internet] [Masters thesis]. University of Southern California; 2014. [cited 2020 Oct 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/86172/rec/4076.

Council of Science Editors:

Li C. MicroRNAs involved in the regulation of Endothelin-1 gene expression in endothelial cells. [Masters Thesis]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/86172/rec/4076


University of Southern California

14. Jung, Eunson. Visualization and characterization of rat lymphatic vasculature in a Prox1-EGFP bacterial artificial chromosome (BAC) transgenic rat: a novel animal model for physiological and pathological lymphangiogenesis.

Degree: MS, Biochemistry and Molecular Biology, 2015, University of Southern California

 In the human body there are two fundamental circulatory networks, the blood and the lymphatic system, which are anatomically similar and complementary in function. Despite… (more)

Subjects/Keywords: lymphatic reporter; Prox1-EGFP; Prox1 reporter rat; bacterial artificial chromosome; BAC; transgenic rat

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APA (6th Edition):

Jung, E. (2015). Visualization and characterization of rat lymphatic vasculature in a Prox1-EGFP bacterial artificial chromosome (BAC) transgenic rat: a novel animal model for physiological and pathological lymphangiogenesis. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/592422/rec/7849

Chicago Manual of Style (16th Edition):

Jung, Eunson. “Visualization and characterization of rat lymphatic vasculature in a Prox1-EGFP bacterial artificial chromosome (BAC) transgenic rat: a novel animal model for physiological and pathological lymphangiogenesis.” 2015. Masters Thesis, University of Southern California. Accessed October 25, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/592422/rec/7849.

MLA Handbook (7th Edition):

Jung, Eunson. “Visualization and characterization of rat lymphatic vasculature in a Prox1-EGFP bacterial artificial chromosome (BAC) transgenic rat: a novel animal model for physiological and pathological lymphangiogenesis.” 2015. Web. 25 Oct 2020.

Vancouver:

Jung E. Visualization and characterization of rat lymphatic vasculature in a Prox1-EGFP bacterial artificial chromosome (BAC) transgenic rat: a novel animal model for physiological and pathological lymphangiogenesis. [Internet] [Masters thesis]. University of Southern California; 2015. [cited 2020 Oct 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/592422/rec/7849.

Council of Science Editors:

Jung E. Visualization and characterization of rat lymphatic vasculature in a Prox1-EGFP bacterial artificial chromosome (BAC) transgenic rat: a novel animal model for physiological and pathological lymphangiogenesis. [Masters Thesis]. University of Southern California; 2015. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/592422/rec/7849


University of Southern California

15. Chen, Po-Han. The kinetic study of engineered MBD domain interactions with methylated DNA: insight into binding of methylated DNA by MBD2b.

Degree: MS, Biochemistry & Molecular Biology, 2011, University of Southern California

 Mehtyl-CpG binding domain protein 2 and 3 (MBD2 and MBD3) contain methyl-CpG binding domains (MBD) and belong to a family of MBD proteins. Methyl-CpG binding… (more)

Subjects/Keywords: MBD protein; methyl-CpG

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APA (6th Edition):

Chen, P. (2011). The kinetic study of engineered MBD domain interactions with methylated DNA: insight into binding of methylated DNA by MBD2b. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/278087/rec/6936

Chicago Manual of Style (16th Edition):

Chen, Po-Han. “The kinetic study of engineered MBD domain interactions with methylated DNA: insight into binding of methylated DNA by MBD2b.” 2011. Masters Thesis, University of Southern California. Accessed October 25, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/278087/rec/6936.

MLA Handbook (7th Edition):

Chen, Po-Han. “The kinetic study of engineered MBD domain interactions with methylated DNA: insight into binding of methylated DNA by MBD2b.” 2011. Web. 25 Oct 2020.

Vancouver:

Chen P. The kinetic study of engineered MBD domain interactions with methylated DNA: insight into binding of methylated DNA by MBD2b. [Internet] [Masters thesis]. University of Southern California; 2011. [cited 2020 Oct 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/278087/rec/6936.

Council of Science Editors:

Chen P. The kinetic study of engineered MBD domain interactions with methylated DNA: insight into binding of methylated DNA by MBD2b. [Masters Thesis]. University of Southern California; 2011. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/278087/rec/6936


University of Southern California

16. Waite, Jamie Lynn. Characterization of cytochrome c peroxidase of Marinobacter aquaeolei.

Degree: MS, Biochemistry and Molecular Biology, 2012, University of Southern California

 The molecular biology and biochemistry of biological iron oxidation at neutral pH is poorly understood. In part this is due to the fact that most… (more)

Subjects/Keywords: iron oxidation; cytochrome c peroxidase; Marinobacter aquaeolei; plasmid construct; mutagenesis

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APA (6th Edition):

Waite, J. L. (2012). Characterization of cytochrome c peroxidase of Marinobacter aquaeolei. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/4495/rec/1299

Chicago Manual of Style (16th Edition):

Waite, Jamie Lynn. “Characterization of cytochrome c peroxidase of Marinobacter aquaeolei.” 2012. Masters Thesis, University of Southern California. Accessed October 25, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/4495/rec/1299.

MLA Handbook (7th Edition):

Waite, Jamie Lynn. “Characterization of cytochrome c peroxidase of Marinobacter aquaeolei.” 2012. Web. 25 Oct 2020.

Vancouver:

Waite JL. Characterization of cytochrome c peroxidase of Marinobacter aquaeolei. [Internet] [Masters thesis]. University of Southern California; 2012. [cited 2020 Oct 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/4495/rec/1299.

Council of Science Editors:

Waite JL. Characterization of cytochrome c peroxidase of Marinobacter aquaeolei. [Masters Thesis]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/4495/rec/1299


University of Southern California

17. Rengarajan, Srinivas. Regulation of potassium homeostasis during acute potassium loading.

Degree: MS, Biochemistry and Molecular Biology, 2013, University of Southern California

 Potassium is important for the maintenance of acid-base balance, electrolyte homeostasis and optimal cellular excitability. It is vital that the plasma K⁺ level is maintained… (more)

Subjects/Keywords: homeostasis; potassium; ion transport; NCC; SPAK; phosphorylation; metabolic cage; plasma potassium; kidney

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APA (6th Edition):

Rengarajan, S. (2013). Regulation of potassium homeostasis during acute potassium loading. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/318608/rec/5501

Chicago Manual of Style (16th Edition):

Rengarajan, Srinivas. “Regulation of potassium homeostasis during acute potassium loading.” 2013. Masters Thesis, University of Southern California. Accessed October 25, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/318608/rec/5501.

MLA Handbook (7th Edition):

Rengarajan, Srinivas. “Regulation of potassium homeostasis during acute potassium loading.” 2013. Web. 25 Oct 2020.

Vancouver:

Rengarajan S. Regulation of potassium homeostasis during acute potassium loading. [Internet] [Masters thesis]. University of Southern California; 2013. [cited 2020 Oct 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/318608/rec/5501.

Council of Science Editors:

Rengarajan S. Regulation of potassium homeostasis during acute potassium loading. [Masters Thesis]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/318608/rec/5501


University of Southern California

18. Qian, Junjie. Discovery of small-molecule compounds targeting TREX1.

Degree: MS, Biochemistry and Molecular Biology, 2012, University of Southern California

 Three prime repair exonuclease 1 (TREX1) plays an important role in human DNA repair. More importantly, it has been recently discovered that the 3’→5’ DNA… (more)

Subjects/Keywords: HIV1; TREX1; Small Molecule Compounds; Inhibitor

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APA (6th Edition):

Qian, J. (2012). Discovery of small-molecule compounds targeting TREX1. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/74180/rec/2034

Chicago Manual of Style (16th Edition):

Qian, Junjie. “Discovery of small-molecule compounds targeting TREX1.” 2012. Masters Thesis, University of Southern California. Accessed October 25, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/74180/rec/2034.

MLA Handbook (7th Edition):

Qian, Junjie. “Discovery of small-molecule compounds targeting TREX1.” 2012. Web. 25 Oct 2020.

Vancouver:

Qian J. Discovery of small-molecule compounds targeting TREX1. [Internet] [Masters thesis]. University of Southern California; 2012. [cited 2020 Oct 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/74180/rec/2034.

Council of Science Editors:

Qian J. Discovery of small-molecule compounds targeting TREX1. [Masters Thesis]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/74180/rec/2034


University of Southern California

19. Hong, Dai. Studies on the role of a novel protein, TMEM 56 in tumorigenic growth for MCF-7 cells.

Degree: MS, Biochemistry and Molecular Biology, 2014, University of Southern California

 Transmembrane protein TMEM 56 was first identified as an IKK‐associated protein by mass spectrometry in Dr. Ebrahim Zandi’s lab. This 263AA protein was found to… (more)

Subjects/Keywords: protein; TMEM 56; tumorigenic growth; MCF-7 cells

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APA (6th Edition):

Hong, D. (2014). Studies on the role of a novel protein, TMEM 56 in tumorigenic growth for MCF-7 cells. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/404283/rec/6183

Chicago Manual of Style (16th Edition):

Hong, Dai. “Studies on the role of a novel protein, TMEM 56 in tumorigenic growth for MCF-7 cells.” 2014. Masters Thesis, University of Southern California. Accessed October 25, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/404283/rec/6183.

MLA Handbook (7th Edition):

Hong, Dai. “Studies on the role of a novel protein, TMEM 56 in tumorigenic growth for MCF-7 cells.” 2014. Web. 25 Oct 2020.

Vancouver:

Hong D. Studies on the role of a novel protein, TMEM 56 in tumorigenic growth for MCF-7 cells. [Internet] [Masters thesis]. University of Southern California; 2014. [cited 2020 Oct 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/404283/rec/6183.

Council of Science Editors:

Hong D. Studies on the role of a novel protein, TMEM 56 in tumorigenic growth for MCF-7 cells. [Masters Thesis]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/404283/rec/6183


University of Southern California

20. Ramanathan, Anita. Defining function of an autism pathway in human neuronal cells.

Degree: MS, Biochemistry and Molecular Biology, 2011, University of Southern California

 Autism is a neurodevelopmental disorder that affects 1% of the population and causes deficits in social interaction, communication and behavioral flexibility. Autism is highly heritable,… (more)

Subjects/Keywords: autism; luminex; PI3K signaling; PI3K Akt pathway

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APA (6th Edition):

Ramanathan, A. (2011). Defining function of an autism pathway in human neuronal cells. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/627881/rec/1819

Chicago Manual of Style (16th Edition):

Ramanathan, Anita. “Defining function of an autism pathway in human neuronal cells.” 2011. Masters Thesis, University of Southern California. Accessed October 25, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/627881/rec/1819.

MLA Handbook (7th Edition):

Ramanathan, Anita. “Defining function of an autism pathway in human neuronal cells.” 2011. Web. 25 Oct 2020.

Vancouver:

Ramanathan A. Defining function of an autism pathway in human neuronal cells. [Internet] [Masters thesis]. University of Southern California; 2011. [cited 2020 Oct 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/627881/rec/1819.

Council of Science Editors:

Ramanathan A. Defining function of an autism pathway in human neuronal cells. [Masters Thesis]. University of Southern California; 2011. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/627881/rec/1819


University of Southern California

21. Sanghani, Dhvani. TWIST1 functions in both mesoderm and neural crest derived cranial tissues to establish and maintain coronal suture patency.

Degree: MS, Biochemistry and Molecular Biology, 2013, University of Southern California

 The coronal suture is a non-osteogenic tissue boundary located between neural crest and mesoderm derived skull bones. Failure in the formation of this boundary can… (more)

Subjects/Keywords: TWIST1; craniosynostosis; coronal suture

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APA (6th Edition):

Sanghani, D. (2013). TWIST1 functions in both mesoderm and neural crest derived cranial tissues to establish and maintain coronal suture patency. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/313936/rec/7628

Chicago Manual of Style (16th Edition):

Sanghani, Dhvani. “TWIST1 functions in both mesoderm and neural crest derived cranial tissues to establish and maintain coronal suture patency.” 2013. Masters Thesis, University of Southern California. Accessed October 25, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/313936/rec/7628.

MLA Handbook (7th Edition):

Sanghani, Dhvani. “TWIST1 functions in both mesoderm and neural crest derived cranial tissues to establish and maintain coronal suture patency.” 2013. Web. 25 Oct 2020.

Vancouver:

Sanghani D. TWIST1 functions in both mesoderm and neural crest derived cranial tissues to establish and maintain coronal suture patency. [Internet] [Masters thesis]. University of Southern California; 2013. [cited 2020 Oct 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/313936/rec/7628.

Council of Science Editors:

Sanghani D. TWIST1 functions in both mesoderm and neural crest derived cranial tissues to establish and maintain coronal suture patency. [Masters Thesis]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/313936/rec/7628


University of Southern California

22. Wu, Jia. Wnt/β-catenin/p300 induced transcription is critical for the differentiation and maintenance of Paneth cells.

Degree: MS, Biochemistry and Molecular Biology, 2014, University of Southern California

 Our lab's previous studies have demonstrated that Wnt/β‐catenin/p300 transcription initiates cell differentiation, whereas Wnt/β‐catenin/cyclic AMP‐responsive element binding protein‐binding protein (CBP) transcription mediates cell proliferation/maintenance of… (more)

Subjects/Keywords: Wnt; β ‐Catenin/p300; Paneth cell

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APA (6th Edition):

Wu, J. (2014). Wnt/β-catenin/p300 induced transcription is critical for the differentiation and maintenance of Paneth cells. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/405481/rec/7958

Chicago Manual of Style (16th Edition):

Wu, Jia. “Wnt/β-catenin/p300 induced transcription is critical for the differentiation and maintenance of Paneth cells.” 2014. Masters Thesis, University of Southern California. Accessed October 25, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/405481/rec/7958.

MLA Handbook (7th Edition):

Wu, Jia. “Wnt/β-catenin/p300 induced transcription is critical for the differentiation and maintenance of Paneth cells.” 2014. Web. 25 Oct 2020.

Vancouver:

Wu J. Wnt/β-catenin/p300 induced transcription is critical for the differentiation and maintenance of Paneth cells. [Internet] [Masters thesis]. University of Southern California; 2014. [cited 2020 Oct 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/405481/rec/7958.

Council of Science Editors:

Wu J. Wnt/β-catenin/p300 induced transcription is critical for the differentiation and maintenance of Paneth cells. [Masters Thesis]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/405481/rec/7958


University of Southern California

23. Yu, Jiali. Preosteoblast-specific RUNX2 promotes RANKL membrane association: antagonism by sex steroids through a non-genomic mechanism.

Degree: MS, Biochemistry and Molecular Biology, 2015, University of Southern California

 Molecular mechanisms underlying the bone‐sparing effects of sex steroid hormones are not fully understood. We show that RUNX2, a master regulator of osteoblast differentiation and… (more)

Subjects/Keywords: RUNX2; RANKL; sex steroids; osteoblast differentiation

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APA (6th Edition):

Yu, J. (2015). Preosteoblast-specific RUNX2 promotes RANKL membrane association: antagonism by sex steroids through a non-genomic mechanism. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/571674/rec/5188

Chicago Manual of Style (16th Edition):

Yu, Jiali. “Preosteoblast-specific RUNX2 promotes RANKL membrane association: antagonism by sex steroids through a non-genomic mechanism.” 2015. Masters Thesis, University of Southern California. Accessed October 25, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/571674/rec/5188.

MLA Handbook (7th Edition):

Yu, Jiali. “Preosteoblast-specific RUNX2 promotes RANKL membrane association: antagonism by sex steroids through a non-genomic mechanism.” 2015. Web. 25 Oct 2020.

Vancouver:

Yu J. Preosteoblast-specific RUNX2 promotes RANKL membrane association: antagonism by sex steroids through a non-genomic mechanism. [Internet] [Masters thesis]. University of Southern California; 2015. [cited 2020 Oct 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/571674/rec/5188.

Council of Science Editors:

Yu J. Preosteoblast-specific RUNX2 promotes RANKL membrane association: antagonism by sex steroids through a non-genomic mechanism. [Masters Thesis]. University of Southern California; 2015. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/571674/rec/5188


University of Southern California

24. Jia, Qi. Neuroendocrine regulation of the transcription factor SKN-1/Nrf2 in oxidative stress response.

Degree: MS, Biochemistry and Molecular Biology, 2015, University of Southern California

 The Nrf2 transcription factor plays a critical role in mediating adaptive responses to cellular stress and defending against neurodegeneration, aging, and cancer. Nrf2 activity is… (more)

Subjects/Keywords: C. elegans; oxidative stress; neuropeptide; SKN-1; FLP-1

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APA (6th Edition):

Jia, Q. (2015). Neuroendocrine regulation of the transcription factor SKN-1/Nrf2 in oxidative stress response. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/589898/rec/4377

Chicago Manual of Style (16th Edition):

Jia, Qi. “Neuroendocrine regulation of the transcription factor SKN-1/Nrf2 in oxidative stress response.” 2015. Masters Thesis, University of Southern California. Accessed October 25, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/589898/rec/4377.

MLA Handbook (7th Edition):

Jia, Qi. “Neuroendocrine regulation of the transcription factor SKN-1/Nrf2 in oxidative stress response.” 2015. Web. 25 Oct 2020.

Vancouver:

Jia Q. Neuroendocrine regulation of the transcription factor SKN-1/Nrf2 in oxidative stress response. [Internet] [Masters thesis]. University of Southern California; 2015. [cited 2020 Oct 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/589898/rec/4377.

Council of Science Editors:

Jia Q. Neuroendocrine regulation of the transcription factor SKN-1/Nrf2 in oxidative stress response. [Masters Thesis]. University of Southern California; 2015. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/589898/rec/4377


University of Southern California

25. Lee, Jo. Differential effect of ethanol and r-sulforaphane on regulation of heme oxygenase-1 in endothelial cells.

Degree: MS, Biochemistry and Molecular Biology, 2014, University of Southern California

 Alcoholism is one of the leading causes of liver disease in the United States. The liver metabolizes 80% of ethanol consumed. Ethanol and its metabolite,… (more)

Subjects/Keywords: r-sulforaphane; heme oxygenase-1

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APA (6th Edition):

Lee, J. (2014). Differential effect of ethanol and r-sulforaphane on regulation of heme oxygenase-1 in endothelial cells. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/121882/rec/1986

Chicago Manual of Style (16th Edition):

Lee, Jo. “Differential effect of ethanol and r-sulforaphane on regulation of heme oxygenase-1 in endothelial cells.” 2014. Masters Thesis, University of Southern California. Accessed October 25, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/121882/rec/1986.

MLA Handbook (7th Edition):

Lee, Jo. “Differential effect of ethanol and r-sulforaphane on regulation of heme oxygenase-1 in endothelial cells.” 2014. Web. 25 Oct 2020.

Vancouver:

Lee J. Differential effect of ethanol and r-sulforaphane on regulation of heme oxygenase-1 in endothelial cells. [Internet] [Masters thesis]. University of Southern California; 2014. [cited 2020 Oct 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/121882/rec/1986.

Council of Science Editors:

Lee J. Differential effect of ethanol and r-sulforaphane on regulation of heme oxygenase-1 in endothelial cells. [Masters Thesis]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/121882/rec/1986


University of Southern California

26. Ybanez, Maria Cecilia D. Protein kinase C (PKC) participates in acetaminophen hepatotoxicity through JNK dependent and independent pathways.

Degree: MS, Biochemistry and Molecular Biology, 2013, University of Southern California

 Our previous studies have shown that acetaminophen (APAP)-induced hepatocyte necrosis is mediated by JNK. In the present study we show that protein kinase C (PKC)… (more)

Subjects/Keywords: acetaminophen (APAP); AMPK; p70S6K; necrosis

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APA (6th Edition):

Ybanez, M. C. D. (2013). Protein kinase C (PKC) participates in acetaminophen hepatotoxicity through JNK dependent and independent pathways. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/247992/rec/5301

Chicago Manual of Style (16th Edition):

Ybanez, Maria Cecilia D. “Protein kinase C (PKC) participates in acetaminophen hepatotoxicity through JNK dependent and independent pathways.” 2013. Masters Thesis, University of Southern California. Accessed October 25, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/247992/rec/5301.

MLA Handbook (7th Edition):

Ybanez, Maria Cecilia D. “Protein kinase C (PKC) participates in acetaminophen hepatotoxicity through JNK dependent and independent pathways.” 2013. Web. 25 Oct 2020.

Vancouver:

Ybanez MCD. Protein kinase C (PKC) participates in acetaminophen hepatotoxicity through JNK dependent and independent pathways. [Internet] [Masters thesis]. University of Southern California; 2013. [cited 2020 Oct 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/247992/rec/5301.

Council of Science Editors:

Ybanez MCD. Protein kinase C (PKC) participates in acetaminophen hepatotoxicity through JNK dependent and independent pathways. [Masters Thesis]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/247992/rec/5301


University of Southern California

27. Halabi, Waiel. A synthetic lethal screen for NF-κB-dependent plasma cell disorders.

Degree: MS, Biochemistry and Molecular Biology, 2012, University of Southern California

 Nuclear factor- kappa B (NF-κB) was first described as a transcription factor in B cells that binds to the enhancer element controlling immunoglobulin kappa light… (more)

Subjects/Keywords: hematology; plasma cell disorders

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APA (6th Edition):

Halabi, W. (2012). A synthetic lethal screen for NF-κB-dependent plasma cell disorders. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/120349/rec/424

Chicago Manual of Style (16th Edition):

Halabi, Waiel. “A synthetic lethal screen for NF-κB-dependent plasma cell disorders.” 2012. Masters Thesis, University of Southern California. Accessed October 25, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/120349/rec/424.

MLA Handbook (7th Edition):

Halabi, Waiel. “A synthetic lethal screen for NF-κB-dependent plasma cell disorders.” 2012. Web. 25 Oct 2020.

Vancouver:

Halabi W. A synthetic lethal screen for NF-κB-dependent plasma cell disorders. [Internet] [Masters thesis]. University of Southern California; 2012. [cited 2020 Oct 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/120349/rec/424.

Council of Science Editors:

Halabi W. A synthetic lethal screen for NF-κB-dependent plasma cell disorders. [Masters Thesis]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/120349/rec/424


University of Southern California

28. Guo, Yu (Phoebe). The relationship between DNA methylation and transcription factor binding in colon cancer cells.

Degree: MS, Biochemistry and Molecular Biology, 2014, University of Southern California

 DNA methylation is an important epigenetic mark in the human genome. It is known as a repressive mark that associates with stably silenced genes. Hypotheses… (more)

Subjects/Keywords: transcription factor binding; DNA methylation; DKO1; RNAPII; CEBPB; ZNF274; MAX

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APA (6th Edition):

Guo, Y. (. (2014). The relationship between DNA methylation and transcription factor binding in colon cancer cells. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/479522/rec/7145

Chicago Manual of Style (16th Edition):

Guo, Yu (Phoebe). “The relationship between DNA methylation and transcription factor binding in colon cancer cells.” 2014. Masters Thesis, University of Southern California. Accessed October 25, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/479522/rec/7145.

MLA Handbook (7th Edition):

Guo, Yu (Phoebe). “The relationship between DNA methylation and transcription factor binding in colon cancer cells.” 2014. Web. 25 Oct 2020.

Vancouver:

Guo Y(. The relationship between DNA methylation and transcription factor binding in colon cancer cells. [Internet] [Masters thesis]. University of Southern California; 2014. [cited 2020 Oct 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/479522/rec/7145.

Council of Science Editors:

Guo Y(. The relationship between DNA methylation and transcription factor binding in colon cancer cells. [Masters Thesis]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/479522/rec/7145


University of Southern California

29. Xiong, Jian. RUNX2 stimulates osteoblast-driven osteoclastogenesis by RANKL presentation.

Degree: MS, Biochemistry and Molecular Biology, 2013, University of Southern California

 In mammalian bodies, bones are continuously remodeled via two processes: bone formation and bone resorption. RUNX2, used to be recognized as activator of osteoblast differentiation… (more)

Subjects/Keywords: RUNX2; RANKL; osteoclastogenesis; mouse primary osteoblasts

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Xiong, J. (2013). RUNX2 stimulates osteoblast-driven osteoclastogenesis by RANKL presentation. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/316502/rec/5669

Chicago Manual of Style (16th Edition):

Xiong, Jian. “RUNX2 stimulates osteoblast-driven osteoclastogenesis by RANKL presentation.” 2013. Masters Thesis, University of Southern California. Accessed October 25, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/316502/rec/5669.

MLA Handbook (7th Edition):

Xiong, Jian. “RUNX2 stimulates osteoblast-driven osteoclastogenesis by RANKL presentation.” 2013. Web. 25 Oct 2020.

Vancouver:

Xiong J. RUNX2 stimulates osteoblast-driven osteoclastogenesis by RANKL presentation. [Internet] [Masters thesis]. University of Southern California; 2013. [cited 2020 Oct 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/316502/rec/5669.

Council of Science Editors:

Xiong J. RUNX2 stimulates osteoblast-driven osteoclastogenesis by RANKL presentation. [Masters Thesis]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/316502/rec/5669


University of Southern California

30. Yang, Kai-Ting. Pten deletion in adult pancreatic beta-cells induces cell proliferation and G1/S cell cycle progression.

Degree: MS, Biochemistry and Molecular Biology, 2013, University of Southern California

 β-cells, which produce and release insulin play a major role in maintaining glucose homeostasis. PTEN is a lipid phosphatase that antagonizes PI3K/AKT signaling pathway and… (more)

Subjects/Keywords: Pten; beta-cell; cell proliferation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yang, K. (2013). Pten deletion in adult pancreatic beta-cells induces cell proliferation and G1/S cell cycle progression. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/316863/rec/5321

Chicago Manual of Style (16th Edition):

Yang, Kai-Ting. “Pten deletion in adult pancreatic beta-cells induces cell proliferation and G1/S cell cycle progression.” 2013. Masters Thesis, University of Southern California. Accessed October 25, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/316863/rec/5321.

MLA Handbook (7th Edition):

Yang, Kai-Ting. “Pten deletion in adult pancreatic beta-cells induces cell proliferation and G1/S cell cycle progression.” 2013. Web. 25 Oct 2020.

Vancouver:

Yang K. Pten deletion in adult pancreatic beta-cells induces cell proliferation and G1/S cell cycle progression. [Internet] [Masters thesis]. University of Southern California; 2013. [cited 2020 Oct 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/316863/rec/5321.

Council of Science Editors:

Yang K. Pten deletion in adult pancreatic beta-cells induces cell proliferation and G1/S cell cycle progression. [Masters Thesis]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/316863/rec/5321

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