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You searched for +publisher:"University of Southern California" +contributor:("Schauwecker, P. Elyse"). Showing records 1 – 6 of 6 total matches.

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University of Southern California

1. Bali, Namrata. Progesterone receptors in the rat brain and their role in steroidal regulation of neurite outgrowth.

Degree: PhD, Molecular Biology, 2012, University of Southern California

 Estrogen (E2) and progesterone (P4) regulate synaptic plasticity in the adult rat hippocampus during the normal rat estrous cycle and in response to deafferenting lesions.… (more)

Subjects/Keywords: microglia; Pgrmc1; progesterone; neurite outgrowth; progesterone receptor

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bali, N. (2012). Progesterone receptors in the rat brain and their role in steroidal regulation of neurite outgrowth. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/46403/rec/5268

Chicago Manual of Style (16th Edition):

Bali, Namrata. “Progesterone receptors in the rat brain and their role in steroidal regulation of neurite outgrowth.” 2012. Doctoral Dissertation, University of Southern California. Accessed January 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/46403/rec/5268.

MLA Handbook (7th Edition):

Bali, Namrata. “Progesterone receptors in the rat brain and their role in steroidal regulation of neurite outgrowth.” 2012. Web. 21 Jan 2021.

Vancouver:

Bali N. Progesterone receptors in the rat brain and their role in steroidal regulation of neurite outgrowth. [Internet] [Doctoral dissertation]. University of Southern California; 2012. [cited 2021 Jan 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/46403/rec/5268.

Council of Science Editors:

Bali N. Progesterone receptors in the rat brain and their role in steroidal regulation of neurite outgrowth. [Doctoral Dissertation]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/46403/rec/5268


University of Southern California

2. Tao, Litao. Investigation of the molecular mechanisms of ototoxicity.

Degree: PhD, Genetic, Molecular and Cellular Biology, 2014, University of Southern California

 Sensory hair cells are essential for transforming the mechanical vibrations of sound into electric signals that our nervous system can interpret. However, sensory hair cells… (more)

Subjects/Keywords: aminoglycoside antibiotics; ototoxicity; cyclin-dependent kinase 2; c-Jun; RNA sequencing

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tao, L. (2014). Investigation of the molecular mechanisms of ototoxicity. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/426847/rec/3651

Chicago Manual of Style (16th Edition):

Tao, Litao. “Investigation of the molecular mechanisms of ototoxicity.” 2014. Doctoral Dissertation, University of Southern California. Accessed January 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/426847/rec/3651.

MLA Handbook (7th Edition):

Tao, Litao. “Investigation of the molecular mechanisms of ototoxicity.” 2014. Web. 21 Jan 2021.

Vancouver:

Tao L. Investigation of the molecular mechanisms of ototoxicity. [Internet] [Doctoral dissertation]. University of Southern California; 2014. [cited 2021 Jan 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/426847/rec/3651.

Council of Science Editors:

Tao L. Investigation of the molecular mechanisms of ototoxicity. [Doctoral Dissertation]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/426847/rec/3651


University of Southern California

3. Liu, Laura. Morphological and functional evaluation of hESC-RPE cell transplantation in RCS rats.

Degree: PhD, Cell and Neurobiology, 2015, University of Southern California

 Age-related Macular Degeneration (AMD) is the leading cause of blindness among the elderly in United States. It is categorized into two types: neovascular (wet) AMD,… (more)

Subjects/Keywords: human embryonic stem cells; retinal pigment epithelium; retinal degeneration; Royal College of Surgeon rats

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APA (6th Edition):

Liu, L. (2015). Morphological and functional evaluation of hESC-RPE cell transplantation in RCS rats. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/290592/rec/4224

Chicago Manual of Style (16th Edition):

Liu, Laura. “Morphological and functional evaluation of hESC-RPE cell transplantation in RCS rats.” 2015. Doctoral Dissertation, University of Southern California. Accessed January 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/290592/rec/4224.

MLA Handbook (7th Edition):

Liu, Laura. “Morphological and functional evaluation of hESC-RPE cell transplantation in RCS rats.” 2015. Web. 21 Jan 2021.

Vancouver:

Liu L. Morphological and functional evaluation of hESC-RPE cell transplantation in RCS rats. [Internet] [Doctoral dissertation]. University of Southern California; 2015. [cited 2021 Jan 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/290592/rec/4224.

Council of Science Editors:

Liu L. Morphological and functional evaluation of hESC-RPE cell transplantation in RCS rats. [Doctoral Dissertation]. University of Southern California; 2015. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/290592/rec/4224


University of Southern California

4. Kong, Seogkyoung. Variation in Galr1 expression determines the susceptibility to excitotoxin-induced neuronal death in mice.

Degree: PhD, Biochemistry & Molecular Biology, 2009, University of Southern California

 Glutamate excitotoxicity plays a role in neuronal death in diverse neurodegenerative diseases. Inbred strains of mice differ in their susceptibility to excitotoxin-induced cell death, but… (more)

Subjects/Keywords: glutamate excitotoxicity; genetic variation; Galr1; inbred and congenic mouse models

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APA (6th Edition):

Kong, S. (2009). Variation in Galr1 expression determines the susceptibility to excitotoxin-induced neuronal death in mice. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/322194/rec/7813

Chicago Manual of Style (16th Edition):

Kong, Seogkyoung. “Variation in Galr1 expression determines the susceptibility to excitotoxin-induced neuronal death in mice.” 2009. Doctoral Dissertation, University of Southern California. Accessed January 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/322194/rec/7813.

MLA Handbook (7th Edition):

Kong, Seogkyoung. “Variation in Galr1 expression determines the susceptibility to excitotoxin-induced neuronal death in mice.” 2009. Web. 21 Jan 2021.

Vancouver:

Kong S. Variation in Galr1 expression determines the susceptibility to excitotoxin-induced neuronal death in mice. [Internet] [Doctoral dissertation]. University of Southern California; 2009. [cited 2021 Jan 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/322194/rec/7813.

Council of Science Editors:

Kong S. Variation in Galr1 expression determines the susceptibility to excitotoxin-induced neuronal death in mice. [Doctoral Dissertation]. University of Southern California; 2009. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/322194/rec/7813


University of Southern California

5. Thomas, Stefani Nicole Cottrell. Functional proteomic analysis of altered protein signaling modules in Alzheimer's disease.

Degree: PhD, Pharmaceutical Sciences, 2007, University of Southern California

 Neuritic plaques comprised of amyloid ß (Aß) are one of the primary neuropathological hallmarks of Alzheimer's disease (AD). However, Aß plaque deposition is preceded by… (more)

Subjects/Keywords: Vps4b; HNK-1/NCAM; LC-MS/MS; proteomics; amyloid beta; Alzheimer'; s disease

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APA (6th Edition):

Thomas, S. N. C. (2007). Functional proteomic analysis of altered protein signaling modules in Alzheimer's disease. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/329545/rec/2945

Chicago Manual of Style (16th Edition):

Thomas, Stefani Nicole Cottrell. “Functional proteomic analysis of altered protein signaling modules in Alzheimer's disease.” 2007. Doctoral Dissertation, University of Southern California. Accessed January 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/329545/rec/2945.

MLA Handbook (7th Edition):

Thomas, Stefani Nicole Cottrell. “Functional proteomic analysis of altered protein signaling modules in Alzheimer's disease.” 2007. Web. 21 Jan 2021.

Vancouver:

Thomas SNC. Functional proteomic analysis of altered protein signaling modules in Alzheimer's disease. [Internet] [Doctoral dissertation]. University of Southern California; 2007. [cited 2021 Jan 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/329545/rec/2945.

Council of Science Editors:

Thomas SNC. Functional proteomic analysis of altered protein signaling modules in Alzheimer's disease. [Doctoral Dissertation]. University of Southern California; 2007. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/329545/rec/2945


University of Southern California

6. Davis, Elizabeth J.H. Striatal Dopamine and functional recovery after cortical lesion.

Degree: PhD, Neuroscience, 2006, University of Southern California

 Brain injury research utilizes animal models to examine the neuronal and neurological effects of damage, including reparative or compensatory cellular and molecular responses to injury.… (more)

Subjects/Keywords: dopamine; strain; brain injury

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Davis, E. J. H. (2006). Striatal Dopamine and functional recovery after cortical lesion. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/26184/rec/6109

Chicago Manual of Style (16th Edition):

Davis, Elizabeth J H. “Striatal Dopamine and functional recovery after cortical lesion.” 2006. Doctoral Dissertation, University of Southern California. Accessed January 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/26184/rec/6109.

MLA Handbook (7th Edition):

Davis, Elizabeth J H. “Striatal Dopamine and functional recovery after cortical lesion.” 2006. Web. 21 Jan 2021.

Vancouver:

Davis EJH. Striatal Dopamine and functional recovery after cortical lesion. [Internet] [Doctoral dissertation]. University of Southern California; 2006. [cited 2021 Jan 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/26184/rec/6109.

Council of Science Editors:

Davis EJH. Striatal Dopamine and functional recovery after cortical lesion. [Doctoral Dissertation]. University of Southern California; 2006. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/26184/rec/6109

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