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You searched for +publisher:"University of Southern California" +contributor:("Johnson, Deborah L."). Showing records 1 – 17 of 17 total matches.

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University of Southern California

1. Rohira, Aarti D. SUMOylation regulates RNA polymerase III  – dependent transcripton via MAF1.

Degree: PhD, Genetic, Molecular and Cellular Biology, 2013, University of Southern California

 Our studies have identified a role for sumoylation in mediating the ability of Maf1 to repress RNA pol III-dependent transcription. As increased expression of either… (more)

Subjects/Keywords: RNA polymerase III; Maf1; SUMOylation; transcriptional regulation; CDK8

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APA (6th Edition):

Rohira, A. D. (2013). SUMOylation regulates RNA polymerase III  – dependent transcripton via MAF1. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/240896/rec/6202

Chicago Manual of Style (16th Edition):

Rohira, Aarti D. “SUMOylation regulates RNA polymerase III  – dependent transcripton via MAF1.” 2013. Doctoral Dissertation, University of Southern California. Accessed June 20, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/240896/rec/6202.

MLA Handbook (7th Edition):

Rohira, Aarti D. “SUMOylation regulates RNA polymerase III  – dependent transcripton via MAF1.” 2013. Web. 20 Jun 2019.

Vancouver:

Rohira AD. SUMOylation regulates RNA polymerase III  – dependent transcripton via MAF1. [Internet] [Doctoral dissertation]. University of Southern California; 2013. [cited 2019 Jun 20]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/240896/rec/6202.

Council of Science Editors:

Rohira AD. SUMOylation regulates RNA polymerase III  – dependent transcripton via MAF1. [Doctoral Dissertation]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/240896/rec/6202


University of Southern California

2. Wang, Min-chuan. RNA polymerase III-dependent transcription is repressed under prolonged hypoxic conditions.

Degree: MS, Biochemistry & Molecular Biology, 2010, University of Southern California

 RNA polymerase III is important for the transcription of non-protein coding RNAs which are important for protein synthesis, RNA processing and protein trafficking. In recent… (more)

Subjects/Keywords: hypoxia; RNA pol III; HIF-1

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APA (6th Edition):

Wang, M. (2010). RNA polymerase III-dependent transcription is repressed under prolonged hypoxic conditions. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/397293/rec/5602

Chicago Manual of Style (16th Edition):

Wang, Min-chuan. “RNA polymerase III-dependent transcription is repressed under prolonged hypoxic conditions.” 2010. Masters Thesis, University of Southern California. Accessed June 20, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/397293/rec/5602.

MLA Handbook (7th Edition):

Wang, Min-chuan. “RNA polymerase III-dependent transcription is repressed under prolonged hypoxic conditions.” 2010. Web. 20 Jun 2019.

Vancouver:

Wang M. RNA polymerase III-dependent transcription is repressed under prolonged hypoxic conditions. [Internet] [Masters thesis]. University of Southern California; 2010. [cited 2019 Jun 20]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/397293/rec/5602.

Council of Science Editors:

Wang M. RNA polymerase III-dependent transcription is repressed under prolonged hypoxic conditions. [Masters Thesis]. University of Southern California; 2010. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/397293/rec/5602


University of Southern California

3. Tsai, Steven C. Investigating the role of OCT4 isoforms in human embryonic stem cell self-renewal and differentiation.

Degree: PhD, Systems Biology and Disease, 2011, University of Southern California

 Human embryonic stem cells (hESC) derived from the inner cell mass of pre-implantation human blastocysts have two unique properties—indefinite self-renewal in culture and pluripotency, or… (more)

Subjects/Keywords: human embryonic stem cells; self-renewal; pluripotency; OCT4; OCT4 isoforms; OCT4 splice variants

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APA (6th Edition):

Tsai, S. C. (2011). Investigating the role of OCT4 isoforms in human embryonic stem cell self-renewal and differentiation. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/618552/rec/3632

Chicago Manual of Style (16th Edition):

Tsai, Steven C. “Investigating the role of OCT4 isoforms in human embryonic stem cell self-renewal and differentiation.” 2011. Doctoral Dissertation, University of Southern California. Accessed June 20, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/618552/rec/3632.

MLA Handbook (7th Edition):

Tsai, Steven C. “Investigating the role of OCT4 isoforms in human embryonic stem cell self-renewal and differentiation.” 2011. Web. 20 Jun 2019.

Vancouver:

Tsai SC. Investigating the role of OCT4 isoforms in human embryonic stem cell self-renewal and differentiation. [Internet] [Doctoral dissertation]. University of Southern California; 2011. [cited 2019 Jun 20]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/618552/rec/3632.

Council of Science Editors:

Tsai SC. Investigating the role of OCT4 isoforms in human embryonic stem cell self-renewal and differentiation. [Doctoral Dissertation]. University of Southern California; 2011. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/618552/rec/3632


University of Southern California

4. Zeng, Ni. PTEN loss antagonizes aging through promoting regeneration and prevents oxidative stress induced cell death.

Degree: PhD, Molecular Pharmacology and Toxicology, 2012, University of Southern California

 PTEN is a dual lipid and protein phosphatase that antagonizes the PI3K/AKT signaling cascade and controls multi cellular activities including proliferation, survival and metabolism. Here,… (more)

Subjects/Keywords: aging; AKT; beta cell; IGF-1; PTEN; regeneration

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APA (6th Edition):

Zeng, N. (2012). PTEN loss antagonizes aging through promoting regeneration and prevents oxidative stress induced cell death. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/118946/rec/5316

Chicago Manual of Style (16th Edition):

Zeng, Ni. “PTEN loss antagonizes aging through promoting regeneration and prevents oxidative stress induced cell death.” 2012. Doctoral Dissertation, University of Southern California. Accessed June 20, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/118946/rec/5316.

MLA Handbook (7th Edition):

Zeng, Ni. “PTEN loss antagonizes aging through promoting regeneration and prevents oxidative stress induced cell death.” 2012. Web. 20 Jun 2019.

Vancouver:

Zeng N. PTEN loss antagonizes aging through promoting regeneration and prevents oxidative stress induced cell death. [Internet] [Doctoral dissertation]. University of Southern California; 2012. [cited 2019 Jun 20]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/118946/rec/5316.

Council of Science Editors:

Zeng N. PTEN loss antagonizes aging through promoting regeneration and prevents oxidative stress induced cell death. [Doctoral Dissertation]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/118946/rec/5316


University of Southern California

5. Rostamiani, Karoline. Brain-derived neurotrophic factor (BDNF)-dependent activation of calpain: involvement of ERK pathway and regulation of AMPA receptor function and stability.

Degree: PhD, Genetic, Molecular & Cellular Biology, 2011, University of Southern California

 Ionotropic glutamate receptors mediate most excitatory synaptic transmission in mammalian brain, and play important roles in neuronal development and synaptic plasticity. In particular, long-term potentiation… (more)

Subjects/Keywords: glutamate receptors; stargazin; learning and memory; calpain; LTP; LTD

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APA (6th Edition):

Rostamiani, K. (2011). Brain-derived neurotrophic factor (BDNF)-dependent activation of calpain: involvement of ERK pathway and regulation of AMPA receptor function and stability. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/434278/rec/1156

Chicago Manual of Style (16th Edition):

Rostamiani, Karoline. “Brain-derived neurotrophic factor (BDNF)-dependent activation of calpain: involvement of ERK pathway and regulation of AMPA receptor function and stability.” 2011. Doctoral Dissertation, University of Southern California. Accessed June 20, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/434278/rec/1156.

MLA Handbook (7th Edition):

Rostamiani, Karoline. “Brain-derived neurotrophic factor (BDNF)-dependent activation of calpain: involvement of ERK pathway and regulation of AMPA receptor function and stability.” 2011. Web. 20 Jun 2019.

Vancouver:

Rostamiani K. Brain-derived neurotrophic factor (BDNF)-dependent activation of calpain: involvement of ERK pathway and regulation of AMPA receptor function and stability. [Internet] [Doctoral dissertation]. University of Southern California; 2011. [cited 2019 Jun 20]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/434278/rec/1156.

Council of Science Editors:

Rostamiani K. Brain-derived neurotrophic factor (BDNF)-dependent activation of calpain: involvement of ERK pathway and regulation of AMPA receptor function and stability. [Doctoral Dissertation]. University of Southern California; 2011. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/434278/rec/1156


University of Southern California

6. Tolani, Bhairavi (Vivi). Molecular strategies towards inhibition of KSHV-associated malignancies.

Degree: PhD, Molecular Biology, 2014, University of Southern California

 Primary Effusion Lymphoma (PEL) is an aggressive form of non-Hodgkin’s B cell lymphoma associated with infection by Kaposi’s sarcoma associated herpesvirus (KSHV). To perpetuate viral… (more)

Subjects/Keywords: Myc; PEL; BET; Bromodomain; KSHV; NEMO; K13; IKK

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APA (6th Edition):

Tolani, B. (. (2014). Molecular strategies towards inhibition of KSHV-associated malignancies. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/128105/rec/4199

Chicago Manual of Style (16th Edition):

Tolani, Bhairavi (Vivi). “Molecular strategies towards inhibition of KSHV-associated malignancies.” 2014. Doctoral Dissertation, University of Southern California. Accessed June 20, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/128105/rec/4199.

MLA Handbook (7th Edition):

Tolani, Bhairavi (Vivi). “Molecular strategies towards inhibition of KSHV-associated malignancies.” 2014. Web. 20 Jun 2019.

Vancouver:

Tolani B(. Molecular strategies towards inhibition of KSHV-associated malignancies. [Internet] [Doctoral dissertation]. University of Southern California; 2014. [cited 2019 Jun 20]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/128105/rec/4199.

Council of Science Editors:

Tolani B(. Molecular strategies towards inhibition of KSHV-associated malignancies. [Doctoral Dissertation]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/128105/rec/4199


University of Southern California

7. Guo, Qiuyu. Mechanism of repression of RNA polymerase III-dependent transcription under hypoxia.

Degree: MS, Biochemistry and Molecular Biology, 2012, University of Southern California

 Deregulated RNA Polymerase III (Pol III)-dependent genes transcription is linked to oncogenesis. In this study, we investigated the response of RNA Pol III-dependent gene transcription… (more)

Subjects/Keywords: RNA polymerase III; cancer; hypoxia; HIF-1alpha

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APA (6th Edition):

Guo, Q. (2012). Mechanism of repression of RNA polymerase III-dependent transcription under hypoxia. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/124902/rec/4004

Chicago Manual of Style (16th Edition):

Guo, Qiuyu. “Mechanism of repression of RNA polymerase III-dependent transcription under hypoxia.” 2012. Doctoral Dissertation, University of Southern California. Accessed June 20, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/124902/rec/4004.

MLA Handbook (7th Edition):

Guo, Qiuyu. “Mechanism of repression of RNA polymerase III-dependent transcription under hypoxia.” 2012. Web. 20 Jun 2019.

Vancouver:

Guo Q. Mechanism of repression of RNA polymerase III-dependent transcription under hypoxia. [Internet] [Doctoral dissertation]. University of Southern California; 2012. [cited 2019 Jun 20]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/124902/rec/4004.

Council of Science Editors:

Guo Q. Mechanism of repression of RNA polymerase III-dependent transcription under hypoxia. [Doctoral Dissertation]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/124902/rec/4004


University of Southern California

8. Kent, Kelly Ann. Bidirectional plasticity supports learning and post-ischemic functional recovery in the rat striatum.

Degree: PhD, Neuroscience, 2011, University of Southern California

 While stroke-related deaths have decreased in recent years, stroke is still the leading cause of long-term disability in the United States. Adequate rehabilitation is dependant… (more)

Subjects/Keywords: ischemia; learning; plasticity

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APA (6th Edition):

Kent, K. A. (2011). Bidirectional plasticity supports learning and post-ischemic functional recovery in the rat striatum. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/652454/rec/1096

Chicago Manual of Style (16th Edition):

Kent, Kelly Ann. “Bidirectional plasticity supports learning and post-ischemic functional recovery in the rat striatum.” 2011. Doctoral Dissertation, University of Southern California. Accessed June 20, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/652454/rec/1096.

MLA Handbook (7th Edition):

Kent, Kelly Ann. “Bidirectional plasticity supports learning and post-ischemic functional recovery in the rat striatum.” 2011. Web. 20 Jun 2019.

Vancouver:

Kent KA. Bidirectional plasticity supports learning and post-ischemic functional recovery in the rat striatum. [Internet] [Doctoral dissertation]. University of Southern California; 2011. [cited 2019 Jun 20]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/652454/rec/1096.

Council of Science Editors:

Kent KA. Bidirectional plasticity supports learning and post-ischemic functional recovery in the rat striatum. [Doctoral Dissertation]. University of Southern California; 2011. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/652454/rec/1096


University of Southern California

9. Fromm, Jody Arlene. Mechanisms for regulating the expression of the TATA-binding protein.

Degree: PhD, Biochemistry & Molecular Biology, 2009, University of Southern California

 The Epidermal Growth Factor Receptor (EGFR) family regulates essential biological processes upon receptor activation and deregulation of these receptors is detrimental to cellular homeostasis. HER2… (more)

Subjects/Keywords: oncogenes; transcriptional regulation; gene regulation; Elk-1; AP-1; TBP; EGFR

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APA (6th Edition):

Fromm, J. A. (2009). Mechanisms for regulating the expression of the TATA-binding protein. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/506625/rec/4007

Chicago Manual of Style (16th Edition):

Fromm, Jody Arlene. “Mechanisms for regulating the expression of the TATA-binding protein.” 2009. Doctoral Dissertation, University of Southern California. Accessed June 20, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/506625/rec/4007.

MLA Handbook (7th Edition):

Fromm, Jody Arlene. “Mechanisms for regulating the expression of the TATA-binding protein.” 2009. Web. 20 Jun 2019.

Vancouver:

Fromm JA. Mechanisms for regulating the expression of the TATA-binding protein. [Internet] [Doctoral dissertation]. University of Southern California; 2009. [cited 2019 Jun 20]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/506625/rec/4007.

Council of Science Editors:

Fromm JA. Mechanisms for regulating the expression of the TATA-binding protein. [Doctoral Dissertation]. University of Southern California; 2009. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/506625/rec/4007


University of Southern California

10. Palian, Beth Marie. Maf1 is a novel target of the tumor suppressor PTEN and a negative regulator of lipid metabolism.

Degree: PhD, Genetic, Molecular and Cellular Biology, 2012, University of Southern California

 Although Maf1 has been identified as a central negative regulator of transcription, little is known about its regulation and it is likely that many of… (more)

Subjects/Keywords: Maf1; PTEN; FASN; de novo lipogenesis

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APA (6th Edition):

Palian, B. M. (2012). Maf1 is a novel target of the tumor suppressor PTEN and a negative regulator of lipid metabolism. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/48553/rec/3901

Chicago Manual of Style (16th Edition):

Palian, Beth Marie. “Maf1 is a novel target of the tumor suppressor PTEN and a negative regulator of lipid metabolism.” 2012. Doctoral Dissertation, University of Southern California. Accessed June 20, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/48553/rec/3901.

MLA Handbook (7th Edition):

Palian, Beth Marie. “Maf1 is a novel target of the tumor suppressor PTEN and a negative regulator of lipid metabolism.” 2012. Web. 20 Jun 2019.

Vancouver:

Palian BM. Maf1 is a novel target of the tumor suppressor PTEN and a negative regulator of lipid metabolism. [Internet] [Doctoral dissertation]. University of Southern California; 2012. [cited 2019 Jun 20]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/48553/rec/3901.

Council of Science Editors:

Palian BM. Maf1 is a novel target of the tumor suppressor PTEN and a negative regulator of lipid metabolism. [Doctoral Dissertation]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/48553/rec/3901


University of Southern California

11. Tran, Nancy. Synergism via reactive oxygen species of fenretinide combined with other anti-neoplastic agents in preclinical models of recurrent neuroblastoma.

Degree: PhD, Systems Biology & Disease, 2012, University of Southern California

 Neuroblastoma is the most common extracranial solid tumor of childhood and the most frequently diagnosed malignancy during infancy. It accounts for more than 7% of… (more)

Subjects/Keywords: neuroblastoma; fenretinide; microtubule inhibitors; cancer therapeutics

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APA (6th Edition):

Tran, N. (2012). Synergism via reactive oxygen species of fenretinide combined with other anti-neoplastic agents in preclinical models of recurrent neuroblastoma. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/371093/rec/6263

Chicago Manual of Style (16th Edition):

Tran, Nancy. “Synergism via reactive oxygen species of fenretinide combined with other anti-neoplastic agents in preclinical models of recurrent neuroblastoma.” 2012. Doctoral Dissertation, University of Southern California. Accessed June 20, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/371093/rec/6263.

MLA Handbook (7th Edition):

Tran, Nancy. “Synergism via reactive oxygen species of fenretinide combined with other anti-neoplastic agents in preclinical models of recurrent neuroblastoma.” 2012. Web. 20 Jun 2019.

Vancouver:

Tran N. Synergism via reactive oxygen species of fenretinide combined with other anti-neoplastic agents in preclinical models of recurrent neuroblastoma. [Internet] [Doctoral dissertation]. University of Southern California; 2012. [cited 2019 Jun 20]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/371093/rec/6263.

Council of Science Editors:

Tran N. Synergism via reactive oxygen species of fenretinide combined with other anti-neoplastic agents in preclinical models of recurrent neuroblastoma. [Doctoral Dissertation]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/371093/rec/6263


University of Southern California

12. Hsu, Hao-Ru Jessie. Harnessing the power of stem cell self-renewal pathways in cancer: dissecting the role of BMI-1 in Ewing’s sarcoma initiation and maintenance.

Degree: PhD, Genetic, Molecular & Cellular Biology, 2010, University of Southern California

 The stem cell self-renewal pathway is often hijacked and over-activated in cancer cells. The polycomb group protein BMI-1 is a known regulator of stemness and… (more)

Subjects/Keywords: BMI-1; Ewing sarcoma; polycomb group protein, CDKN2A, p16; YAP; Yes-associated protein; contact inhibition; stem cell pathways; EWS-FLI1

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APA (6th Edition):

Hsu, H. J. (2010). Harnessing the power of stem cell self-renewal pathways in cancer: dissecting the role of BMI-1 in Ewing’s sarcoma initiation and maintenance. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/334683/rec/3123

Chicago Manual of Style (16th Edition):

Hsu, Hao-Ru Jessie. “Harnessing the power of stem cell self-renewal pathways in cancer: dissecting the role of BMI-1 in Ewing’s sarcoma initiation and maintenance.” 2010. Doctoral Dissertation, University of Southern California. Accessed June 20, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/334683/rec/3123.

MLA Handbook (7th Edition):

Hsu, Hao-Ru Jessie. “Harnessing the power of stem cell self-renewal pathways in cancer: dissecting the role of BMI-1 in Ewing’s sarcoma initiation and maintenance.” 2010. Web. 20 Jun 2019.

Vancouver:

Hsu HJ. Harnessing the power of stem cell self-renewal pathways in cancer: dissecting the role of BMI-1 in Ewing’s sarcoma initiation and maintenance. [Internet] [Doctoral dissertation]. University of Southern California; 2010. [cited 2019 Jun 20]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/334683/rec/3123.

Council of Science Editors:

Hsu HJ. Harnessing the power of stem cell self-renewal pathways in cancer: dissecting the role of BMI-1 in Ewing’s sarcoma initiation and maintenance. [Doctoral Dissertation]. University of Southern California; 2010. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/334683/rec/3123


University of Southern California

13. Wu, Shumin. Characterization and functional study of a novel human protein SFMBT, and PR-Set7 histone methyltransferase.

Degree: PhD, Biochemistry & Molecular Biology, 2010, University of Southern California

 Eukaryotic chromatin, a dynamic structure composed of DNA and chromatin-associated proteins, plays an essential role in key biological processes including gene transcription, cell cycle progression… (more)

Subjects/Keywords: PR-Set7; APC; Cdk1; Cdc14; cell cycle; chromatin

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APA (6th Edition):

Wu, S. (2010). Characterization and functional study of a novel human protein SFMBT, and PR-Set7 histone methyltransferase. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/410757/rec/1292

Chicago Manual of Style (16th Edition):

Wu, Shumin. “Characterization and functional study of a novel human protein SFMBT, and PR-Set7 histone methyltransferase.” 2010. Doctoral Dissertation, University of Southern California. Accessed June 20, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/410757/rec/1292.

MLA Handbook (7th Edition):

Wu, Shumin. “Characterization and functional study of a novel human protein SFMBT, and PR-Set7 histone methyltransferase.” 2010. Web. 20 Jun 2019.

Vancouver:

Wu S. Characterization and functional study of a novel human protein SFMBT, and PR-Set7 histone methyltransferase. [Internet] [Doctoral dissertation]. University of Southern California; 2010. [cited 2019 Jun 20]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/410757/rec/1292.

Council of Science Editors:

Wu S. Characterization and functional study of a novel human protein SFMBT, and PR-Set7 histone methyltransferase. [Doctoral Dissertation]. University of Southern California; 2010. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/410757/rec/1292


University of Southern California

14. Baumeister, Peter J. The transcriptional regulation of the pro-survival protein Grp78 by activating transcription factors and chromatin-modifying enzymes: its upregulation in response to HDAC-inhibitor treatment and the therapeutic consequences.

Degree: PhD, Biochemistry & Molecular Biology, 2008, University of Southern California

 The unfolded protein response (UPR) is an evolutionarily conserved mechanism whereby cells respond to stress conditions that target the endoplasmic reticulum (ER). One of the… (more)

Subjects/Keywords: transcription; histone; deacetylases; grp78; endoplasmic reticulum stress

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APA (6th Edition):

Baumeister, P. J. (2008). The transcriptional regulation of the pro-survival protein Grp78 by activating transcription factors and chromatin-modifying enzymes: its upregulation in response to HDAC-inhibitor treatment and the therapeutic consequences. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/130806/rec/7356

Chicago Manual of Style (16th Edition):

Baumeister, Peter J. “The transcriptional regulation of the pro-survival protein Grp78 by activating transcription factors and chromatin-modifying enzymes: its upregulation in response to HDAC-inhibitor treatment and the therapeutic consequences.” 2008. Doctoral Dissertation, University of Southern California. Accessed June 20, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/130806/rec/7356.

MLA Handbook (7th Edition):

Baumeister, Peter J. “The transcriptional regulation of the pro-survival protein Grp78 by activating transcription factors and chromatin-modifying enzymes: its upregulation in response to HDAC-inhibitor treatment and the therapeutic consequences.” 2008. Web. 20 Jun 2019.

Vancouver:

Baumeister PJ. The transcriptional regulation of the pro-survival protein Grp78 by activating transcription factors and chromatin-modifying enzymes: its upregulation in response to HDAC-inhibitor treatment and the therapeutic consequences. [Internet] [Doctoral dissertation]. University of Southern California; 2008. [cited 2019 Jun 20]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/130806/rec/7356.

Council of Science Editors:

Baumeister PJ. The transcriptional regulation of the pro-survival protein Grp78 by activating transcription factors and chromatin-modifying enzymes: its upregulation in response to HDAC-inhibitor treatment and the therapeutic consequences. [Doctoral Dissertation]. University of Southern California; 2008. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/130806/rec/7356


University of Southern California

15. Woiwode, Annette. Repression of RNA polymerase III-dependent transcription by the tumor suppressors p53 and PTEN.

Degree: PhD, Molecular Pharmacology & Toxicology, 2007, University of Southern California

 For cells to grow and proliferate they must be capable of protein synthesis. As RNA polymerase (pol) III transcription products are required for protein synthesis,… (more)

Subjects/Keywords: RNA polymerase III; p53; PTEN; tumor suppressors; gene transcription

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APA (6th Edition):

Woiwode, A. (2007). Repression of RNA polymerase III-dependent transcription by the tumor suppressors p53 and PTEN. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/493020/rec/5539

Chicago Manual of Style (16th Edition):

Woiwode, Annette. “Repression of RNA polymerase III-dependent transcription by the tumor suppressors p53 and PTEN.” 2007. Doctoral Dissertation, University of Southern California. Accessed June 20, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/493020/rec/5539.

MLA Handbook (7th Edition):

Woiwode, Annette. “Repression of RNA polymerase III-dependent transcription by the tumor suppressors p53 and PTEN.” 2007. Web. 20 Jun 2019.

Vancouver:

Woiwode A. Repression of RNA polymerase III-dependent transcription by the tumor suppressors p53 and PTEN. [Internet] [Doctoral dissertation]. University of Southern California; 2007. [cited 2019 Jun 20]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/493020/rec/5539.

Council of Science Editors:

Woiwode A. Repression of RNA polymerase III-dependent transcription by the tumor suppressors p53 and PTEN. [Doctoral Dissertation]. University of Southern California; 2007. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/493020/rec/5539


University of Southern California

16. Xu, Wei. An orchestra of glutamate receptors and transporters in synaptic transmission, plasticity and excitotoxicity.

Degree: PhD, Neuroscience, 2008, University of Southern California

 Glutamate is the major excitatory neurotransmitter in the mammalian central nervous system. Synaptic transmission at glutamatergic synapses mediates and regulates basically all aspects of brain… (more)

Subjects/Keywords: synaptic transmission; synaptic plasticity; glutamate receptor; transporter

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Xu, W. (2008). An orchestra of glutamate receptors and transporters in synaptic transmission, plasticity and excitotoxicity. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/41520/rec/791

Chicago Manual of Style (16th Edition):

Xu, Wei. “An orchestra of glutamate receptors and transporters in synaptic transmission, plasticity and excitotoxicity.” 2008. Doctoral Dissertation, University of Southern California. Accessed June 20, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/41520/rec/791.

MLA Handbook (7th Edition):

Xu, Wei. “An orchestra of glutamate receptors and transporters in synaptic transmission, plasticity and excitotoxicity.” 2008. Web. 20 Jun 2019.

Vancouver:

Xu W. An orchestra of glutamate receptors and transporters in synaptic transmission, plasticity and excitotoxicity. [Internet] [Doctoral dissertation]. University of Southern California; 2008. [cited 2019 Jun 20]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/41520/rec/791.

Council of Science Editors:

Xu W. An orchestra of glutamate receptors and transporters in synaptic transmission, plasticity and excitotoxicity. [Doctoral Dissertation]. University of Southern California; 2008. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/41520/rec/791


University of Southern California

17. Anglim, Paul P. Development of DNA methylation based biomarkers for the early detection of squamous cell lung cancer.

Degree: PhD, Biochemistry & Molecular Biology, 2008, University of Southern California

 Lung cancer is the number one cancer killer in the United States. This disease is divided into two sub-types, small cell lung cancer, (10-15% of… (more)

Subjects/Keywords: lung cancer; early detection; squamous; DNA methylation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Anglim, P. P. (2008). Development of DNA methylation based biomarkers for the early detection of squamous cell lung cancer. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/142191/rec/1932

Chicago Manual of Style (16th Edition):

Anglim, Paul P. “Development of DNA methylation based biomarkers for the early detection of squamous cell lung cancer.” 2008. Doctoral Dissertation, University of Southern California. Accessed June 20, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/142191/rec/1932.

MLA Handbook (7th Edition):

Anglim, Paul P. “Development of DNA methylation based biomarkers for the early detection of squamous cell lung cancer.” 2008. Web. 20 Jun 2019.

Vancouver:

Anglim PP. Development of DNA methylation based biomarkers for the early detection of squamous cell lung cancer. [Internet] [Doctoral dissertation]. University of Southern California; 2008. [cited 2019 Jun 20]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/142191/rec/1932.

Council of Science Editors:

Anglim PP. Development of DNA methylation based biomarkers for the early detection of squamous cell lung cancer. [Doctoral Dissertation]. University of Southern California; 2008. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/142191/rec/1932

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