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You searched for +publisher:"University of Southern California" +contributor:("Hofman, Florence M."). Showing records 1 – 30 of 30 total matches.

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University of Southern California

1. Karimi, Saman Shanaya. Suppressor cell therapy: targeting T regulatory cells in cancer.

Degree: MS, Experimental and Molecular Pathology, 2013, University of Southern California

 Tumor growth and propagation has been attributed to many important physiological processes, such as angiogenesis and epigenetic mechanisms. Immune suppression and tumor immune escape, however,… (more)

Subjects/Keywords: cancer; Tregs; suppressor cell therapy; effector cells; immunotherapy

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APA (6th Edition):

Karimi, S. S. (2013). Suppressor cell therapy: targeting T regulatory cells in cancer. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/316900/rec/6234

Chicago Manual of Style (16th Edition):

Karimi, Saman Shanaya. “Suppressor cell therapy: targeting T regulatory cells in cancer.” 2013. Masters Thesis, University of Southern California. Accessed April 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/316900/rec/6234.

MLA Handbook (7th Edition):

Karimi, Saman Shanaya. “Suppressor cell therapy: targeting T regulatory cells in cancer.” 2013. Web. 21 Apr 2021.

Vancouver:

Karimi SS. Suppressor cell therapy: targeting T regulatory cells in cancer. [Internet] [Masters thesis]. University of Southern California; 2013. [cited 2021 Apr 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/316900/rec/6234.

Council of Science Editors:

Karimi SS. Suppressor cell therapy: targeting T regulatory cells in cancer. [Masters Thesis]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/316900/rec/6234


University of Southern California

2. Marquez, Stefanie Brooke. Neurogenesis in diseases of oxidative stress.

Degree: PhD, Pathobiology, 2012, University of Southern California

 Transient hypoxia induces cell death in area CA1 of the rat hippocampus, but spares cells in CA2/3. Activation of stem cells in the subgranular zone… (more)

Subjects/Keywords: stroke; hypoxia; rat hippocampus; Alzheimer'; s Disease; stem cells; human brain; neurodegenerative disease

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APA (6th Edition):

Marquez, S. B. (2012). Neurogenesis in diseases of oxidative stress. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/626588/rec/4379

Chicago Manual of Style (16th Edition):

Marquez, Stefanie Brooke. “Neurogenesis in diseases of oxidative stress.” 2012. Doctoral Dissertation, University of Southern California. Accessed April 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/626588/rec/4379.

MLA Handbook (7th Edition):

Marquez, Stefanie Brooke. “Neurogenesis in diseases of oxidative stress.” 2012. Web. 21 Apr 2021.

Vancouver:

Marquez SB. Neurogenesis in diseases of oxidative stress. [Internet] [Doctoral dissertation]. University of Southern California; 2012. [cited 2021 Apr 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/626588/rec/4379.

Council of Science Editors:

Marquez SB. Neurogenesis in diseases of oxidative stress. [Doctoral Dissertation]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/626588/rec/4379


University of Southern California

3. Andini, Nadya. trans-translation in mycobacteria: expression, regulation, and the effects of ribosome inhibitors.

Degree: PhD, Pathobiology, 2012, University of Southern California

 Unique bacterial process, trans-translation, has been studied to be an important process in many bacteria. However, there was nothing known about the significance of this… (more)

Subjects/Keywords: trans-translation; mycobacteria; tmRNA; macrolides.

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APA (6th Edition):

Andini, N. (2012). trans-translation in mycobacteria: expression, regulation, and the effects of ribosome inhibitors. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/97093/rec/7569

Chicago Manual of Style (16th Edition):

Andini, Nadya. “trans-translation in mycobacteria: expression, regulation, and the effects of ribosome inhibitors.” 2012. Doctoral Dissertation, University of Southern California. Accessed April 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/97093/rec/7569.

MLA Handbook (7th Edition):

Andini, Nadya. “trans-translation in mycobacteria: expression, regulation, and the effects of ribosome inhibitors.” 2012. Web. 21 Apr 2021.

Vancouver:

Andini N. trans-translation in mycobacteria: expression, regulation, and the effects of ribosome inhibitors. [Internet] [Doctoral dissertation]. University of Southern California; 2012. [cited 2021 Apr 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/97093/rec/7569.

Council of Science Editors:

Andini N. trans-translation in mycobacteria: expression, regulation, and the effects of ribosome inhibitors. [Doctoral Dissertation]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/97093/rec/7569


University of Southern California

4. Barath, Manasi. Increased anti-cancer efficacy of a novel lipid modified analog of temozolomide in breast cancer cells.

Degree: MS, Molecular Microbiology and Immunology, 2013, University of Southern California

 Temozolomide (TMZ; an alkylating agent that is able to cross the blood brain barrier) is used for the treatment of malignant gliomas, but much less… (more)

Subjects/Keywords: temozolomide; DNA damage; breast cancer; gamma H2AX; MGMT; TMZ-POH; brain metastases; DNA alkylating agents

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APA (6th Edition):

Barath, M. (2013). Increased anti-cancer efficacy of a novel lipid modified analog of temozolomide in breast cancer cells. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/243872/rec/3449

Chicago Manual of Style (16th Edition):

Barath, Manasi. “Increased anti-cancer efficacy of a novel lipid modified analog of temozolomide in breast cancer cells.” 2013. Masters Thesis, University of Southern California. Accessed April 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/243872/rec/3449.

MLA Handbook (7th Edition):

Barath, Manasi. “Increased anti-cancer efficacy of a novel lipid modified analog of temozolomide in breast cancer cells.” 2013. Web. 21 Apr 2021.

Vancouver:

Barath M. Increased anti-cancer efficacy of a novel lipid modified analog of temozolomide in breast cancer cells. [Internet] [Masters thesis]. University of Southern California; 2013. [cited 2021 Apr 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/243872/rec/3449.

Council of Science Editors:

Barath M. Increased anti-cancer efficacy of a novel lipid modified analog of temozolomide in breast cancer cells. [Masters Thesis]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/243872/rec/3449


University of Southern California

5. Osborne, Andrea. Integrin mediated cellular adhesion may alter the cytokine profile in acute lymphoblastic leukemia.

Degree: MS, Experimental and Molecular Pathology, 2015, University of Southern California

 Although survival rates are promising in children with Acute Lymphoblastic Leukemia, in relapsed childhood ALL, survival rates remain a problem. More than 80% of first… (more)

Subjects/Keywords: acute lymphoblastic leukemia; cytokines; adhesion; integrin; integrin alpha6; hemoresistance; bone marrow; relapsed; childhood ALL; cell adhesion mediated drug resistance; soluble factor mediated drug resistance

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APA (6th Edition):

Osborne, A. (2015). Integrin mediated cellular adhesion may alter the cytokine profile in acute lymphoblastic leukemia. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/589960/rec/3558

Chicago Manual of Style (16th Edition):

Osborne, Andrea. “Integrin mediated cellular adhesion may alter the cytokine profile in acute lymphoblastic leukemia.” 2015. Masters Thesis, University of Southern California. Accessed April 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/589960/rec/3558.

MLA Handbook (7th Edition):

Osborne, Andrea. “Integrin mediated cellular adhesion may alter the cytokine profile in acute lymphoblastic leukemia.” 2015. Web. 21 Apr 2021.

Vancouver:

Osborne A. Integrin mediated cellular adhesion may alter the cytokine profile in acute lymphoblastic leukemia. [Internet] [Masters thesis]. University of Southern California; 2015. [cited 2021 Apr 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/589960/rec/3558.

Council of Science Editors:

Osborne A. Integrin mediated cellular adhesion may alter the cytokine profile in acute lymphoblastic leukemia. [Masters Thesis]. University of Southern California; 2015. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/589960/rec/3558


University of Southern California

6. Park, Ryan. Investigation of immune escape paths in the tumor microenvironment.

Degree: MS, Experimental and Molecular Pathology, 2013, University of Southern California

 Fold changes in 113 genes were measured using RT-qPCR in B16, RENCA, and LLC tumors of syngeneic mice for mechanisms of immune escape. Fold changes… (more)

Subjects/Keywords: immune; tumor; microenvironment; RT-qPCR; B16 melanoma; RENCA renal carcinoma; LLC Lewis lung carcinoma; fold change

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APA (6th Edition):

Park, R. (2013). Investigation of immune escape paths in the tumor microenvironment. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/252101/rec/3644

Chicago Manual of Style (16th Edition):

Park, Ryan. “Investigation of immune escape paths in the tumor microenvironment.” 2013. Masters Thesis, University of Southern California. Accessed April 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/252101/rec/3644.

MLA Handbook (7th Edition):

Park, Ryan. “Investigation of immune escape paths in the tumor microenvironment.” 2013. Web. 21 Apr 2021.

Vancouver:

Park R. Investigation of immune escape paths in the tumor microenvironment. [Internet] [Masters thesis]. University of Southern California; 2013. [cited 2021 Apr 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/252101/rec/3644.

Council of Science Editors:

Park R. Investigation of immune escape paths in the tumor microenvironment. [Masters Thesis]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/252101/rec/3644


University of Southern California

7. Ashish Anshu, Fnu. Anti-cancer effects of novel glidobactin type proteasome inhibitors.

Degree: MS, Molecular Microbiology & Immunology, 2011, University of Southern California

 Proteasome inhibitors are widely used today as an important tool for anti-cancer treatment, which has led to a wide search of novel proteasome inhibitors. Proteasome… (more)

Subjects/Keywords: apoptosis; autophagy; endoplasmic reticulum stress; glidobactin A; hematological malignancy; proteasome

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APA (6th Edition):

Ashish Anshu, F. (2011). Anti-cancer effects of novel glidobactin type proteasome inhibitors. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/456326/rec/839

Chicago Manual of Style (16th Edition):

Ashish Anshu, Fnu. “Anti-cancer effects of novel glidobactin type proteasome inhibitors.” 2011. Masters Thesis, University of Southern California. Accessed April 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/456326/rec/839.

MLA Handbook (7th Edition):

Ashish Anshu, Fnu. “Anti-cancer effects of novel glidobactin type proteasome inhibitors.” 2011. Web. 21 Apr 2021.

Vancouver:

Ashish Anshu F. Anti-cancer effects of novel glidobactin type proteasome inhibitors. [Internet] [Masters thesis]. University of Southern California; 2011. [cited 2021 Apr 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/456326/rec/839.

Council of Science Editors:

Ashish Anshu F. Anti-cancer effects of novel glidobactin type proteasome inhibitors. [Masters Thesis]. University of Southern California; 2011. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/456326/rec/839


University of Southern California

8. Russell, Darren Jason. Investigating the roles of BMI-1 and IGF-1 in promoting ESFT survival.

Degree: MS, Experimental & Molecular Pathology, 2011, University of Southern California

 Purpose: Ewing sarcoma family tumors (ESFT) are highly aggressive pediatric and young adult stem cell tumors for which prognostic biomarkers and novel treatments are needed.… (more)

Subjects/Keywords: Ewing'; s sarcoma; ESFT; cancer; BMI-1, IFG-1; IGF-1R

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APA (6th Edition):

Russell, D. J. (2011). Investigating the roles of BMI-1 and IGF-1 in promoting ESFT survival. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/427668/rec/3641

Chicago Manual of Style (16th Edition):

Russell, Darren Jason. “Investigating the roles of BMI-1 and IGF-1 in promoting ESFT survival.” 2011. Masters Thesis, University of Southern California. Accessed April 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/427668/rec/3641.

MLA Handbook (7th Edition):

Russell, Darren Jason. “Investigating the roles of BMI-1 and IGF-1 in promoting ESFT survival.” 2011. Web. 21 Apr 2021.

Vancouver:

Russell DJ. Investigating the roles of BMI-1 and IGF-1 in promoting ESFT survival. [Internet] [Masters thesis]. University of Southern California; 2011. [cited 2021 Apr 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/427668/rec/3641.

Council of Science Editors:

Russell DJ. Investigating the roles of BMI-1 and IGF-1 in promoting ESFT survival. [Masters Thesis]. University of Southern California; 2011. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/427668/rec/3641


University of Southern California

9. Bergfeld, Scott Anthony. Bone marrow derived mesenchymal stem cells promote survival and drug resistance in tumor cells.

Degree: PhD, Pathobiology, 2013, University of Southern California

 Bone marrow mesenchymal stem cells (BMMSC) are recruited to primary tumors and have been reported to display pro-tumorigenic as well as anti-tumorigenic activities. We hypothesize… (more)

Subjects/Keywords: mesenchymal stem cells; tumor microenvironment; drug resistance; apoptosis

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APA (6th Edition):

Bergfeld, S. A. (2013). Bone marrow derived mesenchymal stem cells promote survival and drug resistance in tumor cells. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/335114/rec/1154

Chicago Manual of Style (16th Edition):

Bergfeld, Scott Anthony. “Bone marrow derived mesenchymal stem cells promote survival and drug resistance in tumor cells.” 2013. Doctoral Dissertation, University of Southern California. Accessed April 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/335114/rec/1154.

MLA Handbook (7th Edition):

Bergfeld, Scott Anthony. “Bone marrow derived mesenchymal stem cells promote survival and drug resistance in tumor cells.” 2013. Web. 21 Apr 2021.

Vancouver:

Bergfeld SA. Bone marrow derived mesenchymal stem cells promote survival and drug resistance in tumor cells. [Internet] [Doctoral dissertation]. University of Southern California; 2013. [cited 2021 Apr 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/335114/rec/1154.

Council of Science Editors:

Bergfeld SA. Bone marrow derived mesenchymal stem cells promote survival and drug resistance in tumor cells. [Doctoral Dissertation]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/335114/rec/1154


University of Southern California

10. Shah, Shrenik Rajesh. Effect of vicrostatin on integrin based signaling molecules in cancer.

Degree: MS, Biochemistry and Molecular Biology, 2013, University of Southern California

 Cell adhesion molecules have been found to play a pivotal role in progression of cancer. Integrins are one the molecules among these various cell adhesion… (more)

Subjects/Keywords: beta 1 &; talin; cancer; disintegrins; integrins; signaling; vicrostatin

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APA (6th Edition):

Shah, S. R. (2013). Effect of vicrostatin on integrin based signaling molecules in cancer. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/315398/rec/2186

Chicago Manual of Style (16th Edition):

Shah, Shrenik Rajesh. “Effect of vicrostatin on integrin based signaling molecules in cancer.” 2013. Masters Thesis, University of Southern California. Accessed April 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/315398/rec/2186.

MLA Handbook (7th Edition):

Shah, Shrenik Rajesh. “Effect of vicrostatin on integrin based signaling molecules in cancer.” 2013. Web. 21 Apr 2021.

Vancouver:

Shah SR. Effect of vicrostatin on integrin based signaling molecules in cancer. [Internet] [Masters thesis]. University of Southern California; 2013. [cited 2021 Apr 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/315398/rec/2186.

Council of Science Editors:

Shah SR. Effect of vicrostatin on integrin based signaling molecules in cancer. [Masters Thesis]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/315398/rec/2186


University of Southern California

11. Wang, Xinyi. The roles of type VII collagen in wound healing and scar reduction.

Degree: PhD, Pathobiology, 2014, University of Southern California

 Dystrophic epidermolysis bullosa (DEB) is a family of rare inherited mechano-bullous disorders caused by mutations in the gene encoding for type VII collagen (C7), the… (more)

Subjects/Keywords: type VII collagen; C7; dystrophic epidermolysis bullosa; DEB; wound healing; scar reduction; TGF beta pathways; protein-based therapeutics; rare disease

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APA (6th Edition):

Wang, X. (2014). The roles of type VII collagen in wound healing and scar reduction. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/506591/rec/7286

Chicago Manual of Style (16th Edition):

Wang, Xinyi. “The roles of type VII collagen in wound healing and scar reduction.” 2014. Doctoral Dissertation, University of Southern California. Accessed April 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/506591/rec/7286.

MLA Handbook (7th Edition):

Wang, Xinyi. “The roles of type VII collagen in wound healing and scar reduction.” 2014. Web. 21 Apr 2021.

Vancouver:

Wang X. The roles of type VII collagen in wound healing and scar reduction. [Internet] [Doctoral dissertation]. University of Southern California; 2014. [cited 2021 Apr 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/506591/rec/7286.

Council of Science Editors:

Wang X. The roles of type VII collagen in wound healing and scar reduction. [Doctoral Dissertation]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/506591/rec/7286


University of Southern California

12. Kasman, Alex Michael. A putative drug efflux pump in mycobacteria: examining promoter structure and regulation of the novel protein mmpS-3.

Degree: MS, Experimental and Molecular Pathology, 2011, University of Southern California

 Mycobacterium tuberculosis, the causative agent of tuberculosis (TB), caused nearly 2 million deaths in 2009 while one third of the world population is likely infected… (more)

Subjects/Keywords: mycobacteria; efflux; promoter; tuberculosis

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APA (6th Edition):

Kasman, A. M. (2011). A putative drug efflux pump in mycobacteria: examining promoter structure and regulation of the novel protein mmpS-3. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/616427/rec/321

Chicago Manual of Style (16th Edition):

Kasman, Alex Michael. “A putative drug efflux pump in mycobacteria: examining promoter structure and regulation of the novel protein mmpS-3.” 2011. Masters Thesis, University of Southern California. Accessed April 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/616427/rec/321.

MLA Handbook (7th Edition):

Kasman, Alex Michael. “A putative drug efflux pump in mycobacteria: examining promoter structure and regulation of the novel protein mmpS-3.” 2011. Web. 21 Apr 2021.

Vancouver:

Kasman AM. A putative drug efflux pump in mycobacteria: examining promoter structure and regulation of the novel protein mmpS-3. [Internet] [Masters thesis]. University of Southern California; 2011. [cited 2021 Apr 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/616427/rec/321.

Council of Science Editors:

Kasman AM. A putative drug efflux pump in mycobacteria: examining promoter structure and regulation of the novel protein mmpS-3. [Masters Thesis]. University of Southern California; 2011. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/616427/rec/321


University of Southern California

13. Vaikari, Vijaya Pooja. Molecular targets for treatment of glioblastoma multiforme.

Degree: MS, Molecular Microbiology and Immunology, 2016, University of Southern California

 In this study we have tried to understand mechanisms by which tumor suppression of glioblastoma mutliforme (GBM) can be enhanced. To do so we analyzed… (more)

Subjects/Keywords: glioblastoma multiforme; bevacizumab; temozolomide; angiogenesis; macrophages M1 and M2; dendritic cells; serotonin; glioblastoma multiforme (GBM) treatment

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APA (6th Edition):

Vaikari, V. P. (2016). Molecular targets for treatment of glioblastoma multiforme. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/506490/rec/4207

Chicago Manual of Style (16th Edition):

Vaikari, Vijaya Pooja. “Molecular targets for treatment of glioblastoma multiforme.” 2016. Masters Thesis, University of Southern California. Accessed April 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/506490/rec/4207.

MLA Handbook (7th Edition):

Vaikari, Vijaya Pooja. “Molecular targets for treatment of glioblastoma multiforme.” 2016. Web. 21 Apr 2021.

Vancouver:

Vaikari VP. Molecular targets for treatment of glioblastoma multiforme. [Internet] [Masters thesis]. University of Southern California; 2016. [cited 2021 Apr 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/506490/rec/4207.

Council of Science Editors:

Vaikari VP. Molecular targets for treatment of glioblastoma multiforme. [Masters Thesis]. University of Southern California; 2016. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/506490/rec/4207


University of Southern California

14. Hsieh, Yao-Te (Stanley). Role of integrin α4 in drug resistant acute lymphoblastic leukemia.

Degree: PhD, Pathobiology, 2013, University of Southern California

 Although cure rates for acute lymphoblastic leukemia (ALL) in children are high, relapse of ALL leads to death in 50-95%. Adult ALL patients have a… (more)

Subjects/Keywords: acute lymphoblastic leukemia; ALL; integrin; alpha4; drug resistant; Tysabri; TBC3486; Natalizumab

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APA (6th Edition):

Hsieh, Y. (. (2013). Role of integrin α4 in drug resistant acute lymphoblastic leukemia. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/250789/rec/5644

Chicago Manual of Style (16th Edition):

Hsieh, Yao-Te (Stanley). “Role of integrin α4 in drug resistant acute lymphoblastic leukemia.” 2013. Doctoral Dissertation, University of Southern California. Accessed April 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/250789/rec/5644.

MLA Handbook (7th Edition):

Hsieh, Yao-Te (Stanley). “Role of integrin α4 in drug resistant acute lymphoblastic leukemia.” 2013. Web. 21 Apr 2021.

Vancouver:

Hsieh Y(. Role of integrin α4 in drug resistant acute lymphoblastic leukemia. [Internet] [Doctoral dissertation]. University of Southern California; 2013. [cited 2021 Apr 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/250789/rec/5644.

Council of Science Editors:

Hsieh Y(. Role of integrin α4 in drug resistant acute lymphoblastic leukemia. [Doctoral Dissertation]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/250789/rec/5644


University of Southern California

15. Rubin, Nicole. Tools to study the epicardium's response during cardiac regeneration.

Degree: PhD, Pathobiology, 2013, University of Southern California

 Cardiovascular disease is the number one cause of mortality worldwide and when not fatal contributes to chronic morbidity and decreased lifespan. Treating cardiovascular disease is… (more)

Subjects/Keywords: fgf10; cardiovascular; regeneration; ischemic injury; epicardium

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APA (6th Edition):

Rubin, N. (2013). Tools to study the epicardium's response during cardiac regeneration. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/247186/rec/7509

Chicago Manual of Style (16th Edition):

Rubin, Nicole. “Tools to study the epicardium's response during cardiac regeneration.” 2013. Doctoral Dissertation, University of Southern California. Accessed April 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/247186/rec/7509.

MLA Handbook (7th Edition):

Rubin, Nicole. “Tools to study the epicardium's response during cardiac regeneration.” 2013. Web. 21 Apr 2021.

Vancouver:

Rubin N. Tools to study the epicardium's response during cardiac regeneration. [Internet] [Doctoral dissertation]. University of Southern California; 2013. [cited 2021 Apr 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/247186/rec/7509.

Council of Science Editors:

Rubin N. Tools to study the epicardium's response during cardiac regeneration. [Doctoral Dissertation]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/247186/rec/7509


University of Southern California

16. Marion, Christine M. Understanding the role of BRCA1 and Aurora A kinase in polyploidy development in ovarian carcinoma precursor cells.

Degree: PhD, Genetic, Molecular and Cellular Biology, 2015, University of Southern California

 Aurora A belongs to a family of serine/threonine kinases known to play a role in mitotic regulation. More recently, the kinase has been implicated in… (more)

Subjects/Keywords: Aurora A; BRCA1; polyploidy; ovarian carcinoma; cell cycle

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APA (6th Edition):

Marion, C. M. (2015). Understanding the role of BRCA1 and Aurora A kinase in polyploidy development in ovarian carcinoma precursor cells. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/636634/rec/7689

Chicago Manual of Style (16th Edition):

Marion, Christine M. “Understanding the role of BRCA1 and Aurora A kinase in polyploidy development in ovarian carcinoma precursor cells.” 2015. Doctoral Dissertation, University of Southern California. Accessed April 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/636634/rec/7689.

MLA Handbook (7th Edition):

Marion, Christine M. “Understanding the role of BRCA1 and Aurora A kinase in polyploidy development in ovarian carcinoma precursor cells.” 2015. Web. 21 Apr 2021.

Vancouver:

Marion CM. Understanding the role of BRCA1 and Aurora A kinase in polyploidy development in ovarian carcinoma precursor cells. [Internet] [Doctoral dissertation]. University of Southern California; 2015. [cited 2021 Apr 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/636634/rec/7689.

Council of Science Editors:

Marion CM. Understanding the role of BRCA1 and Aurora A kinase in polyploidy development in ovarian carcinoma precursor cells. [Doctoral Dissertation]. University of Southern California; 2015. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/636634/rec/7689


University of Southern California

17. Malone, Karen Emmerette. Immune responses by glia during neurotropic coronavirus induced encephalomyelitis.

Degree: PhD, Pathobiology, 2008, University of Southern California

 The data presented in this thesis is incorporated into the model of early events of coronavirus infection and focuses on the innate responses by glia… (more)

Subjects/Keywords: murine coronavirus; glia; neurosciences; interferon; immunity; major histocompatibility complex; T-cell response; anti-viral response

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APA (6th Edition):

Malone, K. E. (2008). Immune responses by glia during neurotropic coronavirus induced encephalomyelitis. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/124776/rec/3366

Chicago Manual of Style (16th Edition):

Malone, Karen Emmerette. “Immune responses by glia during neurotropic coronavirus induced encephalomyelitis.” 2008. Doctoral Dissertation, University of Southern California. Accessed April 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/124776/rec/3366.

MLA Handbook (7th Edition):

Malone, Karen Emmerette. “Immune responses by glia during neurotropic coronavirus induced encephalomyelitis.” 2008. Web. 21 Apr 2021.

Vancouver:

Malone KE. Immune responses by glia during neurotropic coronavirus induced encephalomyelitis. [Internet] [Doctoral dissertation]. University of Southern California; 2008. [cited 2021 Apr 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/124776/rec/3366.

Council of Science Editors:

Malone KE. Immune responses by glia during neurotropic coronavirus induced encephalomyelitis. [Doctoral Dissertation]. University of Southern California; 2008. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/124776/rec/3366


University of Southern California

18. Kohan, George. POLR2B and its contributions to cancer cell growth.

Degree: MS, Experimental & Molecular Pathology, 2010, University of Southern California

 Ovarian cancer incidence is highest in the western world, where it is the leading cause of death from gynecological malignancies. Numerous risk factors for ovarian… (more)

Subjects/Keywords: POLR2B; ovarian cancer; oncogene; HEY/Chr6; NM22B; RNA polymerase II; ovarian carcinoma; genetics; ovarian cancer genetics

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APA (6th Edition):

Kohan, G. (2010). POLR2B and its contributions to cancer cell growth. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/251783/rec/5103

Chicago Manual of Style (16th Edition):

Kohan, George. “POLR2B and its contributions to cancer cell growth.” 2010. Masters Thesis, University of Southern California. Accessed April 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/251783/rec/5103.

MLA Handbook (7th Edition):

Kohan, George. “POLR2B and its contributions to cancer cell growth.” 2010. Web. 21 Apr 2021.

Vancouver:

Kohan G. POLR2B and its contributions to cancer cell growth. [Internet] [Masters thesis]. University of Southern California; 2010. [cited 2021 Apr 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/251783/rec/5103.

Council of Science Editors:

Kohan G. POLR2B and its contributions to cancer cell growth. [Masters Thesis]. University of Southern California; 2010. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/251783/rec/5103


University of Southern California

19. Umetsu, Akiko. Effects of parathyroid hormone analogues on hematopoietic stem cell niche potential of bone-marrow mononuclear cells.

Degree: MS, Pathobiology, 2009, University of Southern California

 In this study we compared the significance of different intracellular pathways in bone-marrow mononuclear cells (BMMNCs) that are implicated in their hematopoietic stem cell (HSC)… (more)

Subjects/Keywords: hematopoietic stem cell; bone marrow; niche; PKA; PKC; parathyroid hormone

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APA (6th Edition):

Umetsu, A. (2009). Effects of parathyroid hormone analogues on hematopoietic stem cell niche potential of bone-marrow mononuclear cells. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/250789/rec/2226

Chicago Manual of Style (16th Edition):

Umetsu, Akiko. “Effects of parathyroid hormone analogues on hematopoietic stem cell niche potential of bone-marrow mononuclear cells.” 2009. Masters Thesis, University of Southern California. Accessed April 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/250789/rec/2226.

MLA Handbook (7th Edition):

Umetsu, Akiko. “Effects of parathyroid hormone analogues on hematopoietic stem cell niche potential of bone-marrow mononuclear cells.” 2009. Web. 21 Apr 2021.

Vancouver:

Umetsu A. Effects of parathyroid hormone analogues on hematopoietic stem cell niche potential of bone-marrow mononuclear cells. [Internet] [Masters thesis]. University of Southern California; 2009. [cited 2021 Apr 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/250789/rec/2226.

Council of Science Editors:

Umetsu A. Effects of parathyroid hormone analogues on hematopoietic stem cell niche potential of bone-marrow mononuclear cells. [Masters Thesis]. University of Southern California; 2009. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/250789/rec/2226


University of Southern California

20. Jhaveri, Niyati. Noscapine: a study of its effects on the glioma vasculature.

Degree: MS, Biochemistry & Molecular Biology, 2009, University of Southern California

 Noscapine has been widely used as an oral antitussive agent. More recently, studies have shown that noscapine significantly affects microtubule dynamics and has potent antitumor… (more)

Subjects/Keywords: angiogenesis; endothelial cell migration; glioma; noscapine; TMZ-resistant; vasculature

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APA (6th Edition):

Jhaveri, N. (2009). Noscapine: a study of its effects on the glioma vasculature. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/269936/rec/4459

Chicago Manual of Style (16th Edition):

Jhaveri, Niyati. “Noscapine: a study of its effects on the glioma vasculature.” 2009. Masters Thesis, University of Southern California. Accessed April 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/269936/rec/4459.

MLA Handbook (7th Edition):

Jhaveri, Niyati. “Noscapine: a study of its effects on the glioma vasculature.” 2009. Web. 21 Apr 2021.

Vancouver:

Jhaveri N. Noscapine: a study of its effects on the glioma vasculature. [Internet] [Masters thesis]. University of Southern California; 2009. [cited 2021 Apr 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/269936/rec/4459.

Council of Science Editors:

Jhaveri N. Noscapine: a study of its effects on the glioma vasculature. [Masters Thesis]. University of Southern California; 2009. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/269936/rec/4459


University of Southern California

21. Virrey, Jenilyn Joyce. The role of tumor angiogenesis in glioblastoma multiforme and implications for anticancer therapy.

Degree: PhD, Pathobiology, 2009, University of Southern California

 Glioblastoma multiforme (GBM) is the most malignant form of brain tumor. GBM displays characteristics of rapid invasiveness and massive vascularization. Despite modern advances in surgery,… (more)

Subjects/Keywords: angiogenesis; endothelial cells; brain tumors; glioma

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APA (6th Edition):

Virrey, J. J. (2009). The role of tumor angiogenesis in glioblastoma multiforme and implications for anticancer therapy. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/185853/rec/7279

Chicago Manual of Style (16th Edition):

Virrey, Jenilyn Joyce. “The role of tumor angiogenesis in glioblastoma multiforme and implications for anticancer therapy.” 2009. Doctoral Dissertation, University of Southern California. Accessed April 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/185853/rec/7279.

MLA Handbook (7th Edition):

Virrey, Jenilyn Joyce. “The role of tumor angiogenesis in glioblastoma multiforme and implications for anticancer therapy.” 2009. Web. 21 Apr 2021.

Vancouver:

Virrey JJ. The role of tumor angiogenesis in glioblastoma multiforme and implications for anticancer therapy. [Internet] [Doctoral dissertation]. University of Southern California; 2009. [cited 2021 Apr 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/185853/rec/7279.

Council of Science Editors:

Virrey JJ. The role of tumor angiogenesis in glioblastoma multiforme and implications for anticancer therapy. [Doctoral Dissertation]. University of Southern California; 2009. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/185853/rec/7279


University of Southern California

22. Soriano, Nathaniel Leu. Glioblastoma treatment with 2,5-dimethyl-celecoxib (DMC) in vitro: - effects of additional chemotherapeutic drugs and tumor microenvironment, - inconsistencies among commonly used in vitro cell growth and survival assays.

Degree: MS, Experimental & Molecular Pathology, 2006, University of Southern California

 2,5-dimethyl-celecoxib (DMC) is a close structural analog of the selective cyclooxygenase-2 (COX-2) inhibitor celecoxib (Celebrex®) that lacks COX-2 inhibitory function yet maintains all of celecoxib's… (more)

Subjects/Keywords: DMC; glioblastoma; drug combination

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APA (6th Edition):

Soriano, N. L. (2006). Glioblastoma treatment with 2,5-dimethyl-celecoxib (DMC) in vitro: - effects of additional chemotherapeutic drugs and tumor microenvironment, - inconsistencies among commonly used in vitro cell growth and survival assays. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/3025/rec/3061

Chicago Manual of Style (16th Edition):

Soriano, Nathaniel Leu. “Glioblastoma treatment with 2,5-dimethyl-celecoxib (DMC) in vitro: - effects of additional chemotherapeutic drugs and tumor microenvironment, - inconsistencies among commonly used in vitro cell growth and survival assays.” 2006. Masters Thesis, University of Southern California. Accessed April 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/3025/rec/3061.

MLA Handbook (7th Edition):

Soriano, Nathaniel Leu. “Glioblastoma treatment with 2,5-dimethyl-celecoxib (DMC) in vitro: - effects of additional chemotherapeutic drugs and tumor microenvironment, - inconsistencies among commonly used in vitro cell growth and survival assays.” 2006. Web. 21 Apr 2021.

Vancouver:

Soriano NL. Glioblastoma treatment with 2,5-dimethyl-celecoxib (DMC) in vitro: - effects of additional chemotherapeutic drugs and tumor microenvironment, - inconsistencies among commonly used in vitro cell growth and survival assays. [Internet] [Masters thesis]. University of Southern California; 2006. [cited 2021 Apr 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/3025/rec/3061.

Council of Science Editors:

Soriano NL. Glioblastoma treatment with 2,5-dimethyl-celecoxib (DMC) in vitro: - effects of additional chemotherapeutic drugs and tumor microenvironment, - inconsistencies among commonly used in vitro cell growth and survival assays. [Masters Thesis]. University of Southern California; 2006. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/3025/rec/3061


University of Southern California

23. Liu, Zhi. Interleukin-11: a study of its effects in glioblastoma multiforme.

Degree: MS, Biochemistry & Molecular Biology, 2010, University of Southern California

 Interleukin-11 (IL-11) is a member of the interleukin-6 (IL-6) cytokine family, which utilizes a common membrane glycoprotein 130 (gp130) for signal transduction. Previously it had… (more)

Subjects/Keywords: angiogenesis; brain endothelial cells; glioblastoma multiforme; interleukin-11; invasion; migration

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APA (6th Edition):

Liu, Z. (2010). Interleukin-11: a study of its effects in glioblastoma multiforme. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/409725/rec/3591

Chicago Manual of Style (16th Edition):

Liu, Zhi. “Interleukin-11: a study of its effects in glioblastoma multiforme.” 2010. Masters Thesis, University of Southern California. Accessed April 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/409725/rec/3591.

MLA Handbook (7th Edition):

Liu, Zhi. “Interleukin-11: a study of its effects in glioblastoma multiforme.” 2010. Web. 21 Apr 2021.

Vancouver:

Liu Z. Interleukin-11: a study of its effects in glioblastoma multiforme. [Internet] [Masters thesis]. University of Southern California; 2010. [cited 2021 Apr 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/409725/rec/3591.

Council of Science Editors:

Liu Z. Interleukin-11: a study of its effects in glioblastoma multiforme. [Masters Thesis]. University of Southern California; 2010. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/409725/rec/3591


University of Southern California

24. Guan, Shengxi. Role of SH2/SH3 adapter Nck in regulation of actin cytoskeleton and cell motility.

Degree: PhD, Biochemistry & Molecular Biology, 2007, University of Southern California

 Nck family, including Nckalpha and Nckbeta, is involved in signaling pathways that regulate the actin cytoskeleton rearrangement. Although Nckalpha; and Nckbeta were initially considered functionally… (more)

Subjects/Keywords: Nck; actin cytoskeleton; neurite outgrowth; cell migration

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APA (6th Edition):

Guan, S. (2007). Role of SH2/SH3 adapter Nck in regulation of actin cytoskeleton and cell motility. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/558414/rec/5648

Chicago Manual of Style (16th Edition):

Guan, Shengxi. “Role of SH2/SH3 adapter Nck in regulation of actin cytoskeleton and cell motility.” 2007. Doctoral Dissertation, University of Southern California. Accessed April 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/558414/rec/5648.

MLA Handbook (7th Edition):

Guan, Shengxi. “Role of SH2/SH3 adapter Nck in regulation of actin cytoskeleton and cell motility.” 2007. Web. 21 Apr 2021.

Vancouver:

Guan S. Role of SH2/SH3 adapter Nck in regulation of actin cytoskeleton and cell motility. [Internet] [Doctoral dissertation]. University of Southern California; 2007. [cited 2021 Apr 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/558414/rec/5648.

Council of Science Editors:

Guan S. Role of SH2/SH3 adapter Nck in regulation of actin cytoskeleton and cell motility. [Doctoral Dissertation]. University of Southern California; 2007. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/558414/rec/5648


University of Southern California

25. Kardosh, Adel. Identification of the targets required for the anti-tumor effect of celecoxib and its non-COX-2 inhibitory derivative.

Degree: PhD, Molecular Microbiology & Immunology, 2007, University of Southern California

 The non-steroidal anti-inflammatory drug (NSAID) celecoxib (Celebrex®) was specifically developed as a selective cyclooxygenase-2 (COX-2) inhibitor. It is well established that COX-2 plays a role… (more)

Subjects/Keywords: celecoxib; Celebrex; NSAIDs; cell cycle; CDK; DMC; 2,5-dimethyl-celecoxib; survivin; apoptosis; ER Stress; CHOP; GRP78; SERCA; Caspase 4

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APA (6th Edition):

Kardosh, A. (2007). Identification of the targets required for the anti-tumor effect of celecoxib and its non-COX-2 inhibitory derivative. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/508464/rec/3326

Chicago Manual of Style (16th Edition):

Kardosh, Adel. “Identification of the targets required for the anti-tumor effect of celecoxib and its non-COX-2 inhibitory derivative.” 2007. Doctoral Dissertation, University of Southern California. Accessed April 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/508464/rec/3326.

MLA Handbook (7th Edition):

Kardosh, Adel. “Identification of the targets required for the anti-tumor effect of celecoxib and its non-COX-2 inhibitory derivative.” 2007. Web. 21 Apr 2021.

Vancouver:

Kardosh A. Identification of the targets required for the anti-tumor effect of celecoxib and its non-COX-2 inhibitory derivative. [Internet] [Doctoral dissertation]. University of Southern California; 2007. [cited 2021 Apr 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/508464/rec/3326.

Council of Science Editors:

Kardosh A. Identification of the targets required for the anti-tumor effect of celecoxib and its non-COX-2 inhibitory derivative. [Doctoral Dissertation]. University of Southern California; 2007. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/508464/rec/3326


University of Southern California

26. Pyrko, Peter. Targeting ER stress for malignant glioma therapy.

Degree: PhD, Pathobiology, 2007, University of Southern California

 Poor chemosensitivity and the development of chemoresistance remain major obstacles to successful chemotherapy of malignant gliomas. Recently, a new goal has been set: changing malignant… (more)

Subjects/Keywords: glioma

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APA (6th Edition):

Pyrko, P. (2007). Targeting ER stress for malignant glioma therapy. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/324413/rec/6326

Chicago Manual of Style (16th Edition):

Pyrko, Peter. “Targeting ER stress for malignant glioma therapy.” 2007. Doctoral Dissertation, University of Southern California. Accessed April 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/324413/rec/6326.

MLA Handbook (7th Edition):

Pyrko, Peter. “Targeting ER stress for malignant glioma therapy.” 2007. Web. 21 Apr 2021.

Vancouver:

Pyrko P. Targeting ER stress for malignant glioma therapy. [Internet] [Doctoral dissertation]. University of Southern California; 2007. [cited 2021 Apr 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/324413/rec/6326.

Council of Science Editors:

Pyrko P. Targeting ER stress for malignant glioma therapy. [Doctoral Dissertation]. University of Southern California; 2007. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/324413/rec/6326


University of Southern California

27. Golden, Encouse B. Utilizing chemotherapies to alter the balance between life and death in the ER stress response of cancer.

Degree: PhD, Pathobiology, 2009, University of Southern California

 When the protein load is lower than the endoplasmic reticulum (ER) folding capacity, the ER master regulator, GRP78, suppresses ER unfolded protein response (UPR) signaling… (more)

Subjects/Keywords: endoplasmic reticulum stress; celecoxib; chloroquine; green tea; epigallocatechin gallate; bortezomib; proteasome; autophagy; glioblastoma; breast cancer; multiple myeloma

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APA (6th Edition):

Golden, E. B. (2009). Utilizing chemotherapies to alter the balance between life and death in the ER stress response of cancer. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/183258/rec/7791

Chicago Manual of Style (16th Edition):

Golden, Encouse B. “Utilizing chemotherapies to alter the balance between life and death in the ER stress response of cancer.” 2009. Doctoral Dissertation, University of Southern California. Accessed April 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/183258/rec/7791.

MLA Handbook (7th Edition):

Golden, Encouse B. “Utilizing chemotherapies to alter the balance between life and death in the ER stress response of cancer.” 2009. Web. 21 Apr 2021.

Vancouver:

Golden EB. Utilizing chemotherapies to alter the balance between life and death in the ER stress response of cancer. [Internet] [Doctoral dissertation]. University of Southern California; 2009. [cited 2021 Apr 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/183258/rec/7791.

Council of Science Editors:

Golden EB. Utilizing chemotherapies to alter the balance between life and death in the ER stress response of cancer. [Doctoral Dissertation]. University of Southern California; 2009. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/183258/rec/7791


University of Southern California

28. Parsa, Sara. Role of FGFR2b signaling pathway in the development of ectodermal derivatives.

Degree: PhD, Pathobiology, 2010, University of Southern California

 The Fibroblast growth factor (FGF) family consists of 23 members, which play important roles during development, homeostasis and pathogenesis by controlling proliferation, migration and differentiation… (more)

Subjects/Keywords: FGF signaling; FGF10; FGFr2b; apical ectodermal ridge; limb; incisor; cervical loop; mammary gland; bipotent progenitor cells; luminal progenitor cells; terminal end buds

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APA (6th Edition):

Parsa, S. (2010). Role of FGFR2b signaling pathway in the development of ectodermal derivatives. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/437776/rec/5640

Chicago Manual of Style (16th Edition):

Parsa, Sara. “Role of FGFR2b signaling pathway in the development of ectodermal derivatives.” 2010. Doctoral Dissertation, University of Southern California. Accessed April 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/437776/rec/5640.

MLA Handbook (7th Edition):

Parsa, Sara. “Role of FGFR2b signaling pathway in the development of ectodermal derivatives.” 2010. Web. 21 Apr 2021.

Vancouver:

Parsa S. Role of FGFR2b signaling pathway in the development of ectodermal derivatives. [Internet] [Doctoral dissertation]. University of Southern California; 2010. [cited 2021 Apr 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/437776/rec/5640.

Council of Science Editors:

Parsa S. Role of FGFR2b signaling pathway in the development of ectodermal derivatives. [Doctoral Dissertation]. University of Southern California; 2010. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/437776/rec/5640


University of Southern California

29. Sadun, Rebecca Eli. Rationalized immunotherapy by immune signature characterization.

Degree: PhD, Pathobiology, 2007, University of Southern California

 Great strides in the understanding of cancer biology have provided a multitude of approaches to the medical treatment of cancer. While chemotherapy alone has succeeded… (more)

Subjects/Keywords: cancer; oncology; tumor escape; immunosurveillance; tolerance; immunoeditting; biomarkers; real-time PCR

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sadun, R. E. (2007). Rationalized immunotherapy by immune signature characterization. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/501913/rec/5411

Chicago Manual of Style (16th Edition):

Sadun, Rebecca Eli. “Rationalized immunotherapy by immune signature characterization.” 2007. Doctoral Dissertation, University of Southern California. Accessed April 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/501913/rec/5411.

MLA Handbook (7th Edition):

Sadun, Rebecca Eli. “Rationalized immunotherapy by immune signature characterization.” 2007. Web. 21 Apr 2021.

Vancouver:

Sadun RE. Rationalized immunotherapy by immune signature characterization. [Internet] [Doctoral dissertation]. University of Southern California; 2007. [cited 2021 Apr 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/501913/rec/5411.

Council of Science Editors:

Sadun RE. Rationalized immunotherapy by immune signature characterization. [Doctoral Dissertation]. University of Southern California; 2007. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/501913/rec/5411


University of Southern California

30. Khankan, Rima Rachad. Extracellular matrix regulation in retinal pigment epithelial cells and role in retinal fibrosis.

Degree: PhD, Pathobiology, 2008, University of Southern California

 The research in our laboratory is focused on the response of the retinal pigment epithelium (RPE) to injury and how RPE structure and function are… (more)

Subjects/Keywords: fibronectin-EDA; retinal pigment epithelial cells; proliferative vitreoretinopathy

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Khankan, R. R. (2008). Extracellular matrix regulation in retinal pigment epithelial cells and role in retinal fibrosis. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/38498/rec/2693

Chicago Manual of Style (16th Edition):

Khankan, Rima Rachad. “Extracellular matrix regulation in retinal pigment epithelial cells and role in retinal fibrosis.” 2008. Doctoral Dissertation, University of Southern California. Accessed April 21, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/38498/rec/2693.

MLA Handbook (7th Edition):

Khankan, Rima Rachad. “Extracellular matrix regulation in retinal pigment epithelial cells and role in retinal fibrosis.” 2008. Web. 21 Apr 2021.

Vancouver:

Khankan RR. Extracellular matrix regulation in retinal pigment epithelial cells and role in retinal fibrosis. [Internet] [Doctoral dissertation]. University of Southern California; 2008. [cited 2021 Apr 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/38498/rec/2693.

Council of Science Editors:

Khankan RR. Extracellular matrix regulation in retinal pigment epithelial cells and role in retinal fibrosis. [Doctoral Dissertation]. University of Southern California; 2008. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/38498/rec/2693

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