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You searched for +publisher:"University of Southern California" +contributor:("Butler, Samantha J."). Showing records 1 – 9 of 9 total matches.

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University of Southern California

1. Hazen, Virginia M. The diverse roles of smads in dorsal spinal cord development.

Degree: PhD, Neuroscience, 2011, University of Southern California

 Bone Morphogenetic Proteins (BMPs) have disparate functions establishing neural circuitry in the dorsal spinal cord. BMPs first induce specific neuronal cell fates, including the most… (more)

Subjects/Keywords: axon guidance; cell fate; Smads; spinal cord; Bone Morphogenetic Proteins ❧

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hazen, V. M. (2011). The diverse roles of smads in dorsal spinal cord development. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/656050/rec/6569

Chicago Manual of Style (16th Edition):

Hazen, Virginia M. “The diverse roles of smads in dorsal spinal cord development.” 2011. Doctoral Dissertation, University of Southern California. Accessed January 23, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/656050/rec/6569.

MLA Handbook (7th Edition):

Hazen, Virginia M. “The diverse roles of smads in dorsal spinal cord development.” 2011. Web. 23 Jan 2020.

Vancouver:

Hazen VM. The diverse roles of smads in dorsal spinal cord development. [Internet] [Doctoral dissertation]. University of Southern California; 2011. [cited 2020 Jan 23]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/656050/rec/6569.

Council of Science Editors:

Hazen VM. The diverse roles of smads in dorsal spinal cord development. [Doctoral Dissertation]. University of Southern California; 2011. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/656050/rec/6569


University of Southern California

2. Feng, Zhihua. The role of Schwann cells in the development of the neuromuscular junction.

Degree: PhD, Neuroscience, 2009, University of Southern California

 Recent findings have demonstrated that synapse-associated glial cells are an integral and essential component of the tripartite synapse. Glial cells at the vertebrate neuromuscular junction… (more)

Subjects/Keywords: Schwann cells

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APA (6th Edition):

Feng, Z. (2009). The role of Schwann cells in the development of the neuromuscular junction. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/592504/rec/7247

Chicago Manual of Style (16th Edition):

Feng, Zhihua. “The role of Schwann cells in the development of the neuromuscular junction.” 2009. Doctoral Dissertation, University of Southern California. Accessed January 23, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/592504/rec/7247.

MLA Handbook (7th Edition):

Feng, Zhihua. “The role of Schwann cells in the development of the neuromuscular junction.” 2009. Web. 23 Jan 2020.

Vancouver:

Feng Z. The role of Schwann cells in the development of the neuromuscular junction. [Internet] [Doctoral dissertation]. University of Southern California; 2009. [cited 2020 Jan 23]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/592504/rec/7247.

Council of Science Editors:

Feng Z. The role of Schwann cells in the development of the neuromuscular junction. [Doctoral Dissertation]. University of Southern California; 2009. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/592504/rec/7247


University of Southern California

3. Lin, Ming-Yi. Synapse maintenance and function at the mouse neuromuscular junction: implications in diseases.

Degree: PhD, Biology (Neurobiology), 2011, University of Southern California

 The synapse is the fundamental building block of the brain circuitry responsible for the human behavior. An understanding of synaptic structure and function is required… (more)

Subjects/Keywords: neuromuscular junction; motoneuron; complexin; amyotrophic lateral sclerosis; spinal muscular atrophy; myostatin; muscle

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APA (6th Edition):

Lin, M. (2011). Synapse maintenance and function at the mouse neuromuscular junction: implications in diseases. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/428181/rec/6257

Chicago Manual of Style (16th Edition):

Lin, Ming-Yi. “Synapse maintenance and function at the mouse neuromuscular junction: implications in diseases.” 2011. Doctoral Dissertation, University of Southern California. Accessed January 23, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/428181/rec/6257.

MLA Handbook (7th Edition):

Lin, Ming-Yi. “Synapse maintenance and function at the mouse neuromuscular junction: implications in diseases.” 2011. Web. 23 Jan 2020.

Vancouver:

Lin M. Synapse maintenance and function at the mouse neuromuscular junction: implications in diseases. [Internet] [Doctoral dissertation]. University of Southern California; 2011. [cited 2020 Jan 23]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/428181/rec/6257.

Council of Science Editors:

Lin M. Synapse maintenance and function at the mouse neuromuscular junction: implications in diseases. [Doctoral Dissertation]. University of Southern California; 2011. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/428181/rec/6257


University of Southern California

4. Yamauchi, Ken. The role of bone morphogenetic protein receptors in commissural axon guidance and growth.

Degree: PhD, Neuroscience, 2012, University of Southern California

 A correctly functioning nervous system requires that neural circuits be precisely wired during development. A growing axon must travel through a constantly changing environment, bypassing… (more)

Subjects/Keywords: commissural; neuron; axon; guidance; growth; Bone Morphogenetic Protein; BMP; cofilin; development; spinal cord; vertebrate; embryonic

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APA (6th Edition):

Yamauchi, K. (2012). The role of bone morphogenetic protein receptors in commissural axon guidance and growth. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/656751/rec/7193

Chicago Manual of Style (16th Edition):

Yamauchi, Ken. “The role of bone morphogenetic protein receptors in commissural axon guidance and growth.” 2012. Doctoral Dissertation, University of Southern California. Accessed January 23, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/656751/rec/7193.

MLA Handbook (7th Edition):

Yamauchi, Ken. “The role of bone morphogenetic protein receptors in commissural axon guidance and growth.” 2012. Web. 23 Jan 2020.

Vancouver:

Yamauchi K. The role of bone morphogenetic protein receptors in commissural axon guidance and growth. [Internet] [Doctoral dissertation]. University of Southern California; 2012. [cited 2020 Jan 23]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/656751/rec/7193.

Council of Science Editors:

Yamauchi K. The role of bone morphogenetic protein receptors in commissural axon guidance and growth. [Doctoral Dissertation]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/656751/rec/7193


University of Southern California

5. Ling, Kar Yun Karen. Synaptic defects in spinal muscular atrophy.

Degree: PhD, Neuroscience, 2012, University of Southern California

 Spinal Muscular Atrophy (SMA), an autosomal recessive neurodegenerative disease, is a leading genetic cause of infant mortality. The major clinical symptoms are muscle weakness and… (more)

Subjects/Keywords: spinal muscular atrophy; SMA; neurodegenerative disease; neuromuscular junction; neuromuscular disease; synapse; motoneuron

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ling, K. Y. K. (2012). Synaptic defects in spinal muscular atrophy. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/666955/rec/6259

Chicago Manual of Style (16th Edition):

Ling, Kar Yun Karen. “Synaptic defects in spinal muscular atrophy.” 2012. Doctoral Dissertation, University of Southern California. Accessed January 23, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/666955/rec/6259.

MLA Handbook (7th Edition):

Ling, Kar Yun Karen. “Synaptic defects in spinal muscular atrophy.” 2012. Web. 23 Jan 2020.

Vancouver:

Ling KYK. Synaptic defects in spinal muscular atrophy. [Internet] [Doctoral dissertation]. University of Southern California; 2012. [cited 2020 Jan 23]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/666955/rec/6259.

Council of Science Editors:

Ling KYK. Synaptic defects in spinal muscular atrophy. [Doctoral Dissertation]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/666955/rec/6259


University of Southern California

6. Yoo, Young-Eun. Trichostatin A and dihydrotestosterone as potential multi-systemic drugs for amyotrophic lateral sclerosis.

Degree: PhD, Neuroscience, 2010, University of Southern California

 Amyotrophic lateral sclerosis (ALS, also known as Lou Gehrig’s disease) is a lethal neurodegenerative disease characterized by motor neuron loss, progressive muscle weakness, atrophy and… (more)

Subjects/Keywords: amyotrophic lateral sclerosis; SOD1 G93A; transgenic mice; motor neurons; motor behavior; therapy

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APA (6th Edition):

Yoo, Y. (2010). Trichostatin A and dihydrotestosterone as potential multi-systemic drugs for amyotrophic lateral sclerosis. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/288133/rec/7603

Chicago Manual of Style (16th Edition):

Yoo, Young-Eun. “Trichostatin A and dihydrotestosterone as potential multi-systemic drugs for amyotrophic lateral sclerosis.” 2010. Doctoral Dissertation, University of Southern California. Accessed January 23, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/288133/rec/7603.

MLA Handbook (7th Edition):

Yoo, Young-Eun. “Trichostatin A and dihydrotestosterone as potential multi-systemic drugs for amyotrophic lateral sclerosis.” 2010. Web. 23 Jan 2020.

Vancouver:

Yoo Y. Trichostatin A and dihydrotestosterone as potential multi-systemic drugs for amyotrophic lateral sclerosis. [Internet] [Doctoral dissertation]. University of Southern California; 2010. [cited 2020 Jan 23]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/288133/rec/7603.

Council of Science Editors:

Yoo Y. Trichostatin A and dihydrotestosterone as potential multi-systemic drugs for amyotrophic lateral sclerosis. [Doctoral Dissertation]. University of Southern California; 2010. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/288133/rec/7603


University of Southern California

7. Zhao, Zhen. Regulation of axonal development by the cGMP signaling pathway Page 1.

Degree: PhD, Neuroscience, 2009, University of Southern California

 The involvement of cyclic guanosine-3’, 5’-monophosphate (cGMP) signaling in axonal development has been proposed. However, the underlying molecular and cellular mechanism is not well understood… (more)

Subjects/Keywords: axon branching; cGMP; dorsal root ganglion; GSK3; natriuretic peptide; PrkG; bifurcation; sensory neuron

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APA (6th Edition):

Zhao, Z. (2009). Regulation of axonal development by the cGMP signaling pathway Page 1. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/15539/rec/5486

Chicago Manual of Style (16th Edition):

Zhao, Zhen. “Regulation of axonal development by the cGMP signaling pathway Page 1.” 2009. Doctoral Dissertation, University of Southern California. Accessed January 23, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/15539/rec/5486.

MLA Handbook (7th Edition):

Zhao, Zhen. “Regulation of axonal development by the cGMP signaling pathway Page 1.” 2009. Web. 23 Jan 2020.

Vancouver:

Zhao Z. Regulation of axonal development by the cGMP signaling pathway Page 1. [Internet] [Doctoral dissertation]. University of Southern California; 2009. [cited 2020 Jan 23]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/15539/rec/5486.

Council of Science Editors:

Zhao Z. Regulation of axonal development by the cGMP signaling pathway Page 1. [Doctoral Dissertation]. University of Southern California; 2009. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/15539/rec/5486


University of Southern California

8. Davis, Elizabeth J.H. Striatal Dopamine and functional recovery after cortical lesion.

Degree: PhD, Neuroscience, 2006, University of Southern California

 Brain injury research utilizes animal models to examine the neuronal and neurological effects of damage, including reparative or compensatory cellular and molecular responses to injury.… (more)

Subjects/Keywords: dopamine; strain; brain injury

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APA (6th Edition):

Davis, E. J. H. (2006). Striatal Dopamine and functional recovery after cortical lesion. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/26184/rec/6100

Chicago Manual of Style (16th Edition):

Davis, Elizabeth J H. “Striatal Dopamine and functional recovery after cortical lesion.” 2006. Doctoral Dissertation, University of Southern California. Accessed January 23, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/26184/rec/6100.

MLA Handbook (7th Edition):

Davis, Elizabeth J H. “Striatal Dopamine and functional recovery after cortical lesion.” 2006. Web. 23 Jan 2020.

Vancouver:

Davis EJH. Striatal Dopamine and functional recovery after cortical lesion. [Internet] [Doctoral dissertation]. University of Southern California; 2006. [cited 2020 Jan 23]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/26184/rec/6100.

Council of Science Editors:

Davis EJH. Striatal Dopamine and functional recovery after cortical lesion. [Doctoral Dissertation]. University of Southern California; 2006. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/26184/rec/6100


University of Southern California

9. Hu, Jia. Regulatory mechanisms of gamma-aminobutyric acid transporter 1 in the rat brain.

Degree: PhD, Neuroscience, 2007, University of Southern California

 Chemical synapses are the principal locations where signals are transmitted between neurons. The transmission of this chemical signal is largely influenced by the action of… (more)

Subjects/Keywords: phosphorylation; structure; substrate; regulation; GAT1; GABA

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hu, J. (2007). Regulatory mechanisms of gamma-aminobutyric acid transporter 1 in the rat brain. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/589012/rec/5498

Chicago Manual of Style (16th Edition):

Hu, Jia. “Regulatory mechanisms of gamma-aminobutyric acid transporter 1 in the rat brain.” 2007. Doctoral Dissertation, University of Southern California. Accessed January 23, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/589012/rec/5498.

MLA Handbook (7th Edition):

Hu, Jia. “Regulatory mechanisms of gamma-aminobutyric acid transporter 1 in the rat brain.” 2007. Web. 23 Jan 2020.

Vancouver:

Hu J. Regulatory mechanisms of gamma-aminobutyric acid transporter 1 in the rat brain. [Internet] [Doctoral dissertation]. University of Southern California; 2007. [cited 2020 Jan 23]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/589012/rec/5498.

Council of Science Editors:

Hu J. Regulatory mechanisms of gamma-aminobutyric acid transporter 1 in the rat brain. [Doctoral Dissertation]. University of Southern California; 2007. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/589012/rec/5498

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