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You searched for +publisher:"University of Southern California" +contributor:("Aparicio, Oscar M."). Showing records 1 – 10 of 10 total matches.

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University of Southern California

1. Zhang, Zheng. The mechanism of mammalian immunoglobulin class switch recombination.

Degree: PhD, Molecular Biology, 2015, University of Southern California

 Class switch recombination (CSR) is the process by which B cells switch from the default IgM production to the IgA, IgE or IgG production to… (more)

Subjects/Keywords: class switch recombination; activation-induced deaminase; RNA:DNA hybrid; immunoglobulin; isotype switch; B cell; antibody; secondary response; genetic instability; chromosomal rearrangement; gene rearrangement

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhang, Z. (2015). The mechanism of mammalian immunoglobulin class switch recombination. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/453588/rec/6972

Chicago Manual of Style (16th Edition):

Zhang, Zheng. “The mechanism of mammalian immunoglobulin class switch recombination.” 2015. Doctoral Dissertation, University of Southern California. Accessed January 19, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/453588/rec/6972.

MLA Handbook (7th Edition):

Zhang, Zheng. “The mechanism of mammalian immunoglobulin class switch recombination.” 2015. Web. 19 Jan 2020.

Vancouver:

Zhang Z. The mechanism of mammalian immunoglobulin class switch recombination. [Internet] [Doctoral dissertation]. University of Southern California; 2015. [cited 2020 Jan 19]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/453588/rec/6972.

Council of Science Editors:

Zhang Z. The mechanism of mammalian immunoglobulin class switch recombination. [Doctoral Dissertation]. University of Southern California; 2015. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/453588/rec/6972


University of Southern California

2. Mu, Yunxiang. DNA deamination and binding properties of wild type and HIGM2 mutants of activation induced cytidine daminase.

Degree: PhD, Molecular Biology, 2012, University of Southern California

 AID is required for SHM and CSR as an initiator protein. Hyper IgM syndrome 2, a primary immunological deficiency disorder caused by AICDA gene (encoding… (more)

Subjects/Keywords: deaminase; apobec; hyper IgM syndrome; class switch recombination; somatic hypermutation

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APA (6th Edition):

Mu, Y. (2012). DNA deamination and binding properties of wild type and HIGM2 mutants of activation induced cytidine daminase. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/1674/rec/2062

Chicago Manual of Style (16th Edition):

Mu, Yunxiang. “DNA deamination and binding properties of wild type and HIGM2 mutants of activation induced cytidine daminase.” 2012. Doctoral Dissertation, University of Southern California. Accessed January 19, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/1674/rec/2062.

MLA Handbook (7th Edition):

Mu, Yunxiang. “DNA deamination and binding properties of wild type and HIGM2 mutants of activation induced cytidine daminase.” 2012. Web. 19 Jan 2020.

Vancouver:

Mu Y. DNA deamination and binding properties of wild type and HIGM2 mutants of activation induced cytidine daminase. [Internet] [Doctoral dissertation]. University of Southern California; 2012. [cited 2020 Jan 19]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/1674/rec/2062.

Council of Science Editors:

Mu Y. DNA deamination and binding properties of wild type and HIGM2 mutants of activation induced cytidine daminase. [Doctoral Dissertation]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/1674/rec/2062


University of Southern California

3. Nellimoottil, Tittu Thomas. Computational analysis of DNA replication timing and fork dynamics in S. cerevisiae.

Degree: PhD, Computational Biology and Bioinformatics, 2014, University of Southern California

 In the first project, I show that in Saccharomyces cerevisiae, the rate of replication fork progression is inversely related to the number of origins that… (more)

Subjects/Keywords: DNA replication; timing; fork; budding yeast; forkhead; Fkh; Saccharomyces cerevisiae; fork speed; ORC; RNA binding proteins; RIP-chip

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APA (6th Edition):

Nellimoottil, T. T. (2014). Computational analysis of DNA replication timing and fork dynamics in S. cerevisiae. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/325648/rec/1536

Chicago Manual of Style (16th Edition):

Nellimoottil, Tittu Thomas. “Computational analysis of DNA replication timing and fork dynamics in S. cerevisiae.” 2014. Doctoral Dissertation, University of Southern California. Accessed January 19, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/325648/rec/1536.

MLA Handbook (7th Edition):

Nellimoottil, Tittu Thomas. “Computational analysis of DNA replication timing and fork dynamics in S. cerevisiae.” 2014. Web. 19 Jan 2020.

Vancouver:

Nellimoottil TT. Computational analysis of DNA replication timing and fork dynamics in S. cerevisiae. [Internet] [Doctoral dissertation]. University of Southern California; 2014. [cited 2020 Jan 19]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/325648/rec/1536.

Council of Science Editors:

Nellimoottil TT. Computational analysis of DNA replication timing and fork dynamics in S. cerevisiae. [Doctoral Dissertation]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/325648/rec/1536


University of Southern California

4. Butuči, Melina. Zygotic genome activation triggers chromosome damage and checkpoint signaling in Caenorhabditis elegans primordial germ cells.

Degree: PhD, Molecular Biology, 2015, University of Southern California

 During early development in many animals, embryonic cells transition from periods of transcriptional quiescence to abrupt activation of RNA polymerase II (RNAPII)‐dependent transcription on a… (more)

Subjects/Keywords: C. elegans; primordial germ cells; PGC; Z2/Z3; DNA damage; transcription; X chromosome; TOPOII

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APA (6th Edition):

Butuči, M. (2015). Zygotic genome activation triggers chromosome damage and checkpoint signaling in Caenorhabditis elegans primordial germ cells. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/637935/rec/7988

Chicago Manual of Style (16th Edition):

Butuči, Melina. “Zygotic genome activation triggers chromosome damage and checkpoint signaling in Caenorhabditis elegans primordial germ cells.” 2015. Doctoral Dissertation, University of Southern California. Accessed January 19, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/637935/rec/7988.

MLA Handbook (7th Edition):

Butuči, Melina. “Zygotic genome activation triggers chromosome damage and checkpoint signaling in Caenorhabditis elegans primordial germ cells.” 2015. Web. 19 Jan 2020.

Vancouver:

Butuči M. Zygotic genome activation triggers chromosome damage and checkpoint signaling in Caenorhabditis elegans primordial germ cells. [Internet] [Doctoral dissertation]. University of Southern California; 2015. [cited 2020 Jan 19]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/637935/rec/7988.

Council of Science Editors:

Butuči M. Zygotic genome activation triggers chromosome damage and checkpoint signaling in Caenorhabditis elegans primordial germ cells. [Doctoral Dissertation]. University of Southern California; 2015. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/637935/rec/7988


University of Southern California

5. Ostrow, Andrew Zachary. Forkhead transcription factors regulate replication origin firing through dimerization and cell cycle-dependent chromatin binding in S. cerevisiae.

Degree: PhD, Molecular Biology, 2015, University of Southern California

 Forkhead box (FOX) transcription factors regulate a wide variety of cellular functions in higher eukaryotes, including cell cycle control and developmental regulation. In Saccharomyces cerevisiae,… (more)

Subjects/Keywords: replication timing; chromatin; Fkh1; Fkh2; Forkhead; transcription factors; dimer; dimers; dimerization; replication foci; genome architecture; genome structure; chromatin organization; replication origins

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APA (6th Edition):

Ostrow, A. Z. (2015). Forkhead transcription factors regulate replication origin firing through dimerization and cell cycle-dependent chromatin binding in S. cerevisiae. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/520750/rec/2870

Chicago Manual of Style (16th Edition):

Ostrow, Andrew Zachary. “Forkhead transcription factors regulate replication origin firing through dimerization and cell cycle-dependent chromatin binding in S. cerevisiae.” 2015. Doctoral Dissertation, University of Southern California. Accessed January 19, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/520750/rec/2870.

MLA Handbook (7th Edition):

Ostrow, Andrew Zachary. “Forkhead transcription factors regulate replication origin firing through dimerization and cell cycle-dependent chromatin binding in S. cerevisiae.” 2015. Web. 19 Jan 2020.

Vancouver:

Ostrow AZ. Forkhead transcription factors regulate replication origin firing through dimerization and cell cycle-dependent chromatin binding in S. cerevisiae. [Internet] [Doctoral dissertation]. University of Southern California; 2015. [cited 2020 Jan 19]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/520750/rec/2870.

Council of Science Editors:

Ostrow AZ. Forkhead transcription factors regulate replication origin firing through dimerization and cell cycle-dependent chromatin binding in S. cerevisiae. [Doctoral Dissertation]. University of Southern California; 2015. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/520750/rec/2870


University of Southern California

6. Kalhor, Reza. Exploring three-dimensional organization of the genome by mapping chromatin contacts and population modeling.

Degree: PhD, Genetic, Molecular and Cellular Biology, 2012, University of Southern California

 The genetic information in most organisms is stored in a double-helical fiber molecule known as DNA. The total DNA fiber inside every single human nucleus,… (more)

Subjects/Keywords: chromosome conformation capture; genome architecture; chromatin structure; TCC; Tethered Conformation Capture; Hi-C; Population-based modeling; population modeling; chromosome structure

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APA (6th Edition):

Kalhor, R. (2012). Exploring three-dimensional organization of the genome by mapping chromatin contacts and population modeling. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/44451/rec/2678

Chicago Manual of Style (16th Edition):

Kalhor, Reza. “Exploring three-dimensional organization of the genome by mapping chromatin contacts and population modeling.” 2012. Doctoral Dissertation, University of Southern California. Accessed January 19, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/44451/rec/2678.

MLA Handbook (7th Edition):

Kalhor, Reza. “Exploring three-dimensional organization of the genome by mapping chromatin contacts and population modeling.” 2012. Web. 19 Jan 2020.

Vancouver:

Kalhor R. Exploring three-dimensional organization of the genome by mapping chromatin contacts and population modeling. [Internet] [Doctoral dissertation]. University of Southern California; 2012. [cited 2020 Jan 19]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/44451/rec/2678.

Council of Science Editors:

Kalhor R. Exploring three-dimensional organization of the genome by mapping chromatin contacts and population modeling. [Doctoral Dissertation]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/44451/rec/2678


University of Southern California

7. Ding, Lin. Essential and non-essential helicases maintain genome stability in Schizosaccharomyces pombe.

Degree: PhD, Molecular Biology, 2014, University of Southern California

 A healthy cell needs to accurately duplicate its genome and pass one copy to each of its daughter cells. The DNA double helix is accessed… (more)

Subjects/Keywords: replication fork; helicases; MCM; S. pombe; ubiquitin ligase; fork regression; fork recovery; homologous recombination

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APA (6th Edition):

Ding, L. (2014). Essential and non-essential helicases maintain genome stability in Schizosaccharomyces pombe. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/360582/rec/2492

Chicago Manual of Style (16th Edition):

Ding, Lin. “Essential and non-essential helicases maintain genome stability in Schizosaccharomyces pombe.” 2014. Doctoral Dissertation, University of Southern California. Accessed January 19, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/360582/rec/2492.

MLA Handbook (7th Edition):

Ding, Lin. “Essential and non-essential helicases maintain genome stability in Schizosaccharomyces pombe.” 2014. Web. 19 Jan 2020.

Vancouver:

Ding L. Essential and non-essential helicases maintain genome stability in Schizosaccharomyces pombe. [Internet] [Doctoral dissertation]. University of Southern California; 2014. [cited 2020 Jan 19]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/360582/rec/2492.

Council of Science Editors:

Ding L. Essential and non-essential helicases maintain genome stability in Schizosaccharomyces pombe. [Doctoral Dissertation]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/360582/rec/2492


University of Southern California

8. Peace, Jared Michael. Forkhead transcription factors control genome wide dynamics of the S. cerevisiae replication timing program.

Degree: PhD, Molecular Biology, 2014, University of Southern California

 Eukaryotic cells initiate DNA replication from hundreds to thousands of origins genome wide. The coordinated firing of these origins across a range of times throughout… (more)

Subjects/Keywords: Forkhead; Fkh1; Fkh2; Cdc45; Rif1; Rap1; Pfa4; Mec1; Cdc7; Dbf4; Dbf4 Dependent Kinase (DDK); Orc1; replication origin timing; replication fork rate; chromatin; centromere; telomere; epigenetics; transcription; nuclear architecture; S. cerevisiae

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APA (6th Edition):

Peace, J. M. (2014). Forkhead transcription factors control genome wide dynamics of the S. cerevisiae replication timing program. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/514469/rec/2869

Chicago Manual of Style (16th Edition):

Peace, Jared Michael. “Forkhead transcription factors control genome wide dynamics of the S. cerevisiae replication timing program.” 2014. Doctoral Dissertation, University of Southern California. Accessed January 19, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/514469/rec/2869.

MLA Handbook (7th Edition):

Peace, Jared Michael. “Forkhead transcription factors control genome wide dynamics of the S. cerevisiae replication timing program.” 2014. Web. 19 Jan 2020.

Vancouver:

Peace JM. Forkhead transcription factors control genome wide dynamics of the S. cerevisiae replication timing program. [Internet] [Doctoral dissertation]. University of Southern California; 2014. [cited 2020 Jan 19]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/514469/rec/2869.

Council of Science Editors:

Peace JM. Forkhead transcription factors control genome wide dynamics of the S. cerevisiae replication timing program. [Doctoral Dissertation]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/514469/rec/2869


University of Southern California

9. Li, Pao-Chen. The centromere: replication, recombination, reassembly.

Degree: PhD, Molecular Biology, 2012, University of Southern California

 Normal cell division requires faithful DNA replication and proper DNA segregation in order to generate two identical daughter cells. Within cells DNA is always assembled… (more)

Subjects/Keywords: centromere; replication; heterochromatin; recombination; chromosome segregation; hp1; swi6; cdc6; rad51

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APA (6th Edition):

Li, P. (2012). The centromere: replication, recombination, reassembly. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/213298/rec/6487

Chicago Manual of Style (16th Edition):

Li, Pao-Chen. “The centromere: replication, recombination, reassembly.” 2012. Doctoral Dissertation, University of Southern California. Accessed January 19, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/213298/rec/6487.

MLA Handbook (7th Edition):

Li, Pao-Chen. “The centromere: replication, recombination, reassembly.” 2012. Web. 19 Jan 2020.

Vancouver:

Li P. The centromere: replication, recombination, reassembly. [Internet] [Doctoral dissertation]. University of Southern California; 2012. [cited 2020 Jan 19]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/213298/rec/6487.

Council of Science Editors:

Li P. The centromere: replication, recombination, reassembly. [Doctoral Dissertation]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/213298/rec/6487


University of Southern California

10. Zhong, Yuan. The role of Cdc7 in replication fork progression in response to DNA damage.

Degree: PhD, Molecular Biology, 2012, University of Southern California

 Cdc7-Dbf4 is an essential protein kinase complex required for every single origin firing. As a target of the intra-S checkpoint, Cdc7 kinase activity has also… (more)

Subjects/Keywords: Cdc7; replication fork; DNA damage; fork progression; MMS

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APA (6th Edition):

Zhong, Y. (2012). The role of Cdc7 in replication fork progression in response to DNA damage. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/18011/rec/7199

Chicago Manual of Style (16th Edition):

Zhong, Yuan. “The role of Cdc7 in replication fork progression in response to DNA damage.” 2012. Doctoral Dissertation, University of Southern California. Accessed January 19, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/18011/rec/7199.

MLA Handbook (7th Edition):

Zhong, Yuan. “The role of Cdc7 in replication fork progression in response to DNA damage.” 2012. Web. 19 Jan 2020.

Vancouver:

Zhong Y. The role of Cdc7 in replication fork progression in response to DNA damage. [Internet] [Doctoral dissertation]. University of Southern California; 2012. [cited 2020 Jan 19]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/18011/rec/7199.

Council of Science Editors:

Zhong Y. The role of Cdc7 in replication fork progression in response to DNA damage. [Doctoral Dissertation]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/18011/rec/7199

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