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You searched for +publisher:"University of Notre Dame" +contributor:("Dr. Jeffrey Schorey, Committee Member"). Showing records 1 – 5 of 5 total matches.

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University of Notre Dame

1. James W. Clancy. ARF6 Regulated Tumor Progression: Glandular Disruption and Cell Invasion</h1>.

Degree: PhD, Biological Sciences, 2014, University of Notre Dame

  Cancer progression can be described as a continuum ranging from normal, healthy tissue to advanced metastatic disease. In epithelial tissues, glandular disruption and loss… (more)

Subjects/Keywords: ARF6; microvesicle; mitotic spindle angle; cell invasion; epithelial cyst; glandular disruption

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Clancy, J. W. (2014). ARF6 Regulated Tumor Progression: Glandular Disruption and Cell Invasion</h1>. (Doctoral Dissertation). University of Notre Dame. Retrieved from https://curate.nd.edu/show/05741r68b5t

Chicago Manual of Style (16th Edition):

Clancy, James W.. “ARF6 Regulated Tumor Progression: Glandular Disruption and Cell Invasion</h1>.” 2014. Doctoral Dissertation, University of Notre Dame. Accessed December 17, 2018. https://curate.nd.edu/show/05741r68b5t.

MLA Handbook (7th Edition):

Clancy, James W.. “ARF6 Regulated Tumor Progression: Glandular Disruption and Cell Invasion</h1>.” 2014. Web. 17 Dec 2018.

Vancouver:

Clancy JW. ARF6 Regulated Tumor Progression: Glandular Disruption and Cell Invasion</h1>. [Internet] [Doctoral dissertation]. University of Notre Dame; 2014. [cited 2018 Dec 17]. Available from: https://curate.nd.edu/show/05741r68b5t.

Council of Science Editors:

Clancy JW. ARF6 Regulated Tumor Progression: Glandular Disruption and Cell Invasion</h1>. [Doctoral Dissertation]. University of Notre Dame; 2014. Available from: https://curate.nd.edu/show/05741r68b5t


University of Notre Dame

2. Michelle Abadilla Favila. Host and Pathogenic Features That Drive Cellular Immune Response to <i>Leishmania spp. </i> Infection</h1>.

Degree: PhD, Biological Sciences, 2013, University of Notre Dame

  Leishmaniases is a group of vector borne parasitic diseases that affects approximately 12 million people worldwide and results in diverse clinical pathologies, the initiating… (more)

Subjects/Keywords: host cell; leishmania; interleukin-12

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APA (6th Edition):

Favila, M. A. (2013). Host and Pathogenic Features That Drive Cellular Immune Response to <i>Leishmania spp. </i> Infection</h1>. (Doctoral Dissertation). University of Notre Dame. Retrieved from https://curate.nd.edu/show/ww72b853p7q

Chicago Manual of Style (16th Edition):

Favila, Michelle Abadilla. “Host and Pathogenic Features That Drive Cellular Immune Response to <i>Leishmania spp. </i> Infection</h1>.” 2013. Doctoral Dissertation, University of Notre Dame. Accessed December 17, 2018. https://curate.nd.edu/show/ww72b853p7q.

MLA Handbook (7th Edition):

Favila, Michelle Abadilla. “Host and Pathogenic Features That Drive Cellular Immune Response to <i>Leishmania spp. </i> Infection</h1>.” 2013. Web. 17 Dec 2018.

Vancouver:

Favila MA. Host and Pathogenic Features That Drive Cellular Immune Response to <i>Leishmania spp. </i> Infection</h1>. [Internet] [Doctoral dissertation]. University of Notre Dame; 2013. [cited 2018 Dec 17]. Available from: https://curate.nd.edu/show/ww72b853p7q.

Council of Science Editors:

Favila MA. Host and Pathogenic Features That Drive Cellular Immune Response to <i>Leishmania spp. </i> Infection</h1>. [Doctoral Dissertation]. University of Notre Dame; 2013. Available from: https://curate.nd.edu/show/ww72b853p7q


University of Notre Dame

3. Mallary Claire Greenlee-Wacker. CD93 regulates acute inflammation and innate immunity</h1>.

Degree: PhD, Biological Sciences, 2011, University of Notre Dame

  CD93 is emerging as a novel regulator of inflammation. CD93 was identified and cloned based on its ability to modulate C1q-triggered enhanced phagocytosis. However,… (more)

Subjects/Keywords: CD93; LRP; peritonitis; inflammation; C1q; phagocytosis

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APA (6th Edition):

Greenlee-Wacker, M. C. (2011). CD93 regulates acute inflammation and innate immunity</h1>. (Doctoral Dissertation). University of Notre Dame. Retrieved from https://curate.nd.edu/show/1544bp0146s

Chicago Manual of Style (16th Edition):

Greenlee-Wacker, Mallary Claire. “CD93 regulates acute inflammation and innate immunity</h1>.” 2011. Doctoral Dissertation, University of Notre Dame. Accessed December 17, 2018. https://curate.nd.edu/show/1544bp0146s.

MLA Handbook (7th Edition):

Greenlee-Wacker, Mallary Claire. “CD93 regulates acute inflammation and innate immunity</h1>.” 2011. Web. 17 Dec 2018.

Vancouver:

Greenlee-Wacker MC. CD93 regulates acute inflammation and innate immunity</h1>. [Internet] [Doctoral dissertation]. University of Notre Dame; 2011. [cited 2018 Dec 17]. Available from: https://curate.nd.edu/show/1544bp0146s.

Council of Science Editors:

Greenlee-Wacker MC. CD93 regulates acute inflammation and innate immunity</h1>. [Doctoral Dissertation]. University of Notre Dame; 2011. Available from: https://curate.nd.edu/show/1544bp0146s


University of Notre Dame

4. Cristina Ricardo Carter. Host-Pathogen Interactions:.

Degree: MS, Biological Sciences, 2008, University of Notre Dame

  Leishmania species infect approximately 2 million people each year and 350 million people live in areas where they are at risk for infection. This… (more)

Subjects/Keywords: complement receptor 3; leishmaniasis; macrophage

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APA (6th Edition):

Carter, C. R. (2008). Host-Pathogen Interactions:. (Masters Thesis). University of Notre Dame. Retrieved from https://curate.nd.edu/show/4b29b566244

Chicago Manual of Style (16th Edition):

Carter, Cristina Ricardo. “Host-Pathogen Interactions:.” 2008. Masters Thesis, University of Notre Dame. Accessed December 17, 2018. https://curate.nd.edu/show/4b29b566244.

MLA Handbook (7th Edition):

Carter, Cristina Ricardo. “Host-Pathogen Interactions:.” 2008. Web. 17 Dec 2018.

Vancouver:

Carter CR. Host-Pathogen Interactions:. [Internet] [Masters thesis]. University of Notre Dame; 2008. [cited 2018 Dec 17]. Available from: https://curate.nd.edu/show/4b29b566244.

Council of Science Editors:

Carter CR. Host-Pathogen Interactions:. [Masters Thesis]. University of Notre Dame; 2008. Available from: https://curate.nd.edu/show/4b29b566244


University of Notre Dame

5. Emma A Lynch. Role and Regulation of the Proteasome in Epithelial Cell Adhesion and Migration</h1>.

Degree: PhD, Biological Sciences, 2006, University of Notre Dame

  Epithelial cells disassemble their adherens junctions and “scatter’ during processes such as tumor cell invasion, as well as some stages of embryonic development. As… (more)

Subjects/Keywords: E-cadherin; epithelial cell scattering; proteasome; adherens junction

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lynch, E. A. (2006). Role and Regulation of the Proteasome in Epithelial Cell Adhesion and Migration</h1>. (Doctoral Dissertation). University of Notre Dame. Retrieved from https://curate.nd.edu/show/n296ww74m9w

Chicago Manual of Style (16th Edition):

Lynch, Emma A. “Role and Regulation of the Proteasome in Epithelial Cell Adhesion and Migration</h1>.” 2006. Doctoral Dissertation, University of Notre Dame. Accessed December 17, 2018. https://curate.nd.edu/show/n296ww74m9w.

MLA Handbook (7th Edition):

Lynch, Emma A. “Role and Regulation of the Proteasome in Epithelial Cell Adhesion and Migration</h1>.” 2006. Web. 17 Dec 2018.

Vancouver:

Lynch EA. Role and Regulation of the Proteasome in Epithelial Cell Adhesion and Migration</h1>. [Internet] [Doctoral dissertation]. University of Notre Dame; 2006. [cited 2018 Dec 17]. Available from: https://curate.nd.edu/show/n296ww74m9w.

Council of Science Editors:

Lynch EA. Role and Regulation of the Proteasome in Epithelial Cell Adhesion and Migration</h1>. [Doctoral Dissertation]. University of Notre Dame; 2006. Available from: https://curate.nd.edu/show/n296ww74m9w

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